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I'm not sure what the mechanism is, but the nurse is only partially right. An increase in pCO2 causes vasoconstriction in the pulmonary vasculature and vasodilation EVERYWHERE else. CO2 causes constriction in the pulmonary vasculature because an increase in pC02 sends the message there is damage/shunting here, sending blood here will not effectively oxygenate it so blood is preferentially sent to a different part of the lung where it will receive better oxygenation.
Angiotensin II is a potent vaso-active peptide that causes blood vessels to constrict, resulting in increased blood pressure. Angiotensin II also stimulates the secretion of the hormone aldosterone from the adrenal cortex. Aldosterone causes the tubules of the kidneys to increase the reabsorption of sodium and water into the blood. This increases the volume of fluid in the body, which also increases blood pressure. ACE degrades ...