The role of Botulinum toxin type A related axon transport in neuropathic pain induced by chronic constriction injury
<|begin_of_text|><|start_header_id|>system<|end_header_id|> You are a research archivist expert in dataset metadata. Answer the question truthfully.  <|eot_id|><|start_header_id|>user<|end_header_id|> Here is the Description of a dataset. Description:'The mechanism of peripheral axon transport in neuropathic pain is still unclear. Chemokine ligand 13 (CXCL13) and its receptor (C-X-C chemokine receptor type 5, CXCR5) and GABA transporter 1 (GAT-1) play an important role in the development of pain. The aim of this study was to explore the axonal transport of CXCL13/CXCR5 and GAT-1 with the aid of the analgesic effect of botulinum toxin A (BTX-A) in rats.Chronic constriction injury (CCI) rat models were established. BTX-A was administered to rats through subcutaneous injection. The pain behaviors in CCI rats were measured by paw withdrawal threshold (PWT) and paw withdrawal latencies (PWL). The levels of CXCL13/CXCR5 and GAT-1 were measured by western blots' . Please return only a json list of at most 3 Keywords corresponding to the Description:.<|eot_id|><|start_header_id|>assistant<|end_header_id|> {"Answer" : ["Axonal Transport","Botulinum Toxins, Type A","Chemokines","Colchicine","GABA Plasma Membrane Transport Proteins","Hyperalgesia","Neuralgia","Receptors, Chemokine","Sciatic Nerve"]} <|eot_id|>
 Here is the Description of a dataset. Description:'The mechanism of peripheral axon transport in neuropathic pain is still unclear. Chemokine ligand 13 (CXCL13) and its receptor (C-X-C chemokine receptor type 5, CXCR5) and GABA transporter 1 (GAT-1) play an important role in the development of pain. The aim of this study was to explore the axonal transport of CXCL13/CXCR5 and GAT-1 with the aid of the analgesic effect of botulinum toxin A (BTX-A) in rats.Chronic constriction injury (CCI) rat models were established. BTX-A was administered to rats through subcutaneous injection. The pain behaviors in CCI rats were measured by paw withdrawal threshold (PWT) and paw withdrawal latencies (PWL). The levels of CXCL13/CXCR5 and GAT-1 were measured by western blots' . Please return only a json list of at most 3 Keywords corresponding to the Description:.
The mechanism of peripheral axon transport in neuropathic pain is still unclear. Chemokine ligand 13 (CXCL13) and its receptor (C-X-C chemokine receptor type 5, CXCR5) and GABA transporter 1 (GAT-1) play an important role in the development of pain. The aim of this study was to explore the axonal transport of CXCL13/CXCR5 and GAT-1 with the aid of the analgesic effect of botulinum toxin A (BTX-A) in rats.Chronic constriction injury (CCI) rat models were established. BTX-A was administered to rats through subcutaneous injection. The pain behaviors in CCI rats were measured by paw withdrawal threshold (PWT) and paw withdrawal latencies (PWL). The levels of CXCL13/CXCR5 and GAT-1 were measured by western blots
Axonal Transport; Botulinum Toxins, Type A; Chemokines; Colchicine; GABA Plasma Membrane Transport Proteins; Hyperalgesia; Neuralgia; Receptors, Chemokine; Sciatic Nerve