Promises and pitfalls of applying computational models to mental disorders
In June some scientists in Australia published their paper that links an inability to produce cortisol to psychosis:<p><a href="https:&#x2F;&#x2F;www.jcu.edu.au&#x2F;news&#x2F;releases&#x2F;2016&#x2F;june&#x2F;stress-hormone-link-with-psychosis" rel="nofollow">https:&#x2F;&#x2F;www.jcu.edu.au&#x2F;news&#x2F;releases&#x2F;2016&#x2F;june&#x2F;stress-hormon...</a><p>Cortisol is produced from Cholesterol (through a couple steps [1]). Cholesterol gets turned into pregnenolone in the mitochondria. My girlfriend did a lot of damage to her mitochondria with cocaine. 5 months after meeting me, she came to appreciate that &quot;drugs&quot; were causing her problems, and tried to quit everything cold turkey.<p>Now she&#x27;s getting professionally mistreated by Psychiatry. They are trying to suppress the symptom of &quot;psychosis&quot; without caring why it&#x27;s being exhibited.<p>[1] <a href="https:&#x2F;&#x2F;en.wikipedia.org&#x2F;wiki&#x2F;File:Steroidogenesis.svg" rel="nofollow">https:&#x2F;&#x2F;en.wikipedia.org&#x2F;wiki&#x2F;File:Steroidogenesis.svg</a><p>There is a ton of &quot;technical debt&quot; in the study of &quot;mental disorders&quot;. Most of the research just needs to be thrown out. It would be much more productive to assume that people with mental problems are exhausted, give them cytomel (a T3 analogue), Retinol (Vitamin A), pregnenolone, and sugar, then re-evaluate.