Medical Centre but were arrest with VF (Magnevist) that are LGE– followed at another centre; N=46 had CRT-D Time delay: 15 minutes patients with CAD, sarcoidosis, amyloidosis or Patient characteristics: LV function: The LV and RV subendocardial LGE in a and end-systolic age = 56 ± 13 years; coronary artery perfusion endocardial contours were traced territory on cine images to calculate LV male = 62%; mass, end-diastolic and history of AF = 16%; volumes, and LVEF. LV volumes and mass were QRS interval = 132 ± normalised to body surface area. 32 milliseconds; LGE diagnosis: Myocardial scar LVEF (CMR) = 29 ± 12%; was assessed while the observer NYHA class: I = 32%, II = was blinded to available diagnostic 37%, III–IV = 31% data and outcome, and was Median follow-up: 45 months considered to be present only if (IQR 23–67) LGE was visible in 2 orthogonal views. LGE was defined by signal intensity ≥35% of maximal myocardial signal intensity. Shimizu et Prospective N=60 consecutively enrolled Inclusion criteria: Scanner: 1.5-T scanner with a ICD implantation: Not described al. () cohort DCM patients who underwent consecutively enrolled DCM cardiac-dedicated phased-array Outcomes: cardiac assessment: patients who underwent coil cardiac assessment in the HR for likelihood of having a: n=11 (18%) ≥10% LGE Cardiac function sequence: cardiology department Japan NHMRC level II n=49 (82%) <10% LGE CMR studies were ECG-triggered cardiac death between February 2005 and by standard software and images hospitalisation for HF March 2006 Patient characteristics: were acquired during diastole to SIGN: high in LGE+ patients compared with those Exclusion criteria: any of minimise artefacts due to cardiac age = 59 ± 12 years; quality with a low that are LGE– the standard motion. risk of bias male = 77%; contraindications for CMR, LGE sequence: gradient-echo such as the presence of a disease duration = 2.3 ± (segmented True FISP with
arrest with VF N=46 had CRT-D Patient characteristics: age = 56 ± 13 years; male = 62%; history of AF = 16%; QRS interval = 132 ± 32 milliseconds; LVEF (CMR) = 29 ± 12%; NYHA class: I = 32%, II = 37%, III–IV = 31% Median follow-up: 45 months (IQR 23–67) Medical Centre but were followed at another centre; patients with CAD, sarcoidosis, amyloidosis or subendocardial LGE in a coronary artery perfusion territory (Magnevist) Time delay: 15 minutes LV function: The LV and RV and end-systolic endocardial contours were traced on cine images to calculate LV mass, end-diastolic and volumes, and LVEF. LV volumes and mass were normalised to body surface area. LGE diagnosis: Myocardial scar was assessed while the observer was blinded to available diagnostic data and outcome, and was considered to be present only if LGE was visible in 2 orthogonal views. LGE was defined by signal intensity ≥35% of maximal myocardial signal intensity. that are LGE– Shimizu et al. () Japan Prospective cohort NHMRC level II SIGN: high quality with a low risk of bias N=60 consecutively enrolled DCM patients who underwent cardiac assessment: n=11 (18%) ≥10% LGE n=49 (82%) <10% LGE Patient characteristics: age = 59 ± 12 years; male = 77%; disease duration = 2.3 ± Inclusion criteria: consecutively enrolled DCM patients who underwent cardiac assessment in the cardiology department between February 2005 and March 2006 Exclusion criteria: any of the standard contraindications for CMR, such as the presence of a Scanner: 1.5-T scanner with a cardiac-dedicated phased-array coil Cardiac function sequence: CMR studies were ECG-triggered by standard software and images were acquired during diastole to minimise artefacts due to cardiac motion. LGE sequence: gradient-echo (segmented True FISP with ICD implantation: Not described Outcomes: HR for likelihood of having a: cardiac death hospitalisation for HF in LGE+ patients compared with those that are LGE–
1393-CMRforDCM.docx