decreased libido, impotence Kidney stones CARDIOVASCULAR CONDITION Who is at risk? Signs/Symptoms Lab/Diagnostics Nursing Management CAD Increasing age Atherosclerosis Lipid panel CV modifiable risk factor Genetic predisposition Angina pectoris CRP – inflammation reduction Gender - male Unstable angina Homocysteine – Physical activity Diet Myocardial infarction inflammation Dietary changes Sedentary lifestyle - obesity Sudden cardiac death Intravascular US Monitor weight, exercise, food Smoking Supply & demand of myocardial Cardiac cath and intake High LDL tissue percutaneous coronary Support groups Low HDL Ischemia – ECG changes; angina intervention (PCI) Catheterization and PCI High total cholesterol pectoris Coronary angiography Balloon or stent HTN DM High CRP Angina Pectoris Same as above Sudden onset of discomfort in Ca-channel blockers chest, jaw, shoulder, back, or Nitrates Stable – predictable, arm Beta-blockers goes away with rest Activity restriction Remove precipitating factors Unstable – Supplemental O2 during pain unpredictable, may Assess: PQRST, hx, physical involve clot, findings, VS, ECG changes vasoconstriction, risk for MI Prinzmetal or vasospastic Myocardial Complications of angina (high Acute: chest pain more intense Troponin – 4-6 h post up to MONA - Dilate coronary Infarction MI risk): than angina 4-7 days; serial pattern arteries Increased angina duration, Diaphoresis Creatine kinase – (CK-NB) Thrombolytics frequency or at rest (unstable) SOB h to 48-72 h CABG ST or T wave changes Generalized weakness Myoglobin – damage to heart Prevent ACS: O2, vasodilate, Signs of HF May mistake S/S for indigestion muscle reperfuse, prevent thrombus, Pulmonary edema EKG changes – place in tele blood flow Tachycardia IHI: Hypotension early admin of aspirin aspirin @ d/c 325 mg beta-blocker @ d/c
decreased libido, impotence Kidney stones CARDIOVASCULAR CONDITION Who is at risk? Signs/Symptoms Lab/Diagnostics Nursing Management CAD Increasing age Genetic predisposition Gender - male Diet Sedentary lifestyle - obesity Smoking High LDL Low HDL High total cholesterol HTN DM High CRP Atherosclerosis Angina pectoris Unstable angina Myocardial infarction Sudden cardiac death Supply & demand of myocardial tissue Ischemia – ECG changes; angina pectoris Lipid panel CRP – inflammation Homocysteine – inflammation Intravascular US Cardiac cath and percutaneous coronary intervention (PCI) Coronary angiography CV modifiable risk factor reduction Physical activity Dietary changes Monitor weight, exercise, food intake Support groups Catheterization and PCI Balloon or stent Angina Pectoris Stable – predictable, goes away with rest Unstable – unpredictable, may involve clot, vasoconstriction, risk for MI Prinzmetal or vasospastic Same as above Sudden onset of discomfort in chest, jaw, shoulder, back, or arm Ca-channel blockers Nitrates Beta-blockers Activity restriction Remove precipitating factors Supplemental O2 during pain Assess: PQRST, hx, physical findings, VS, ECG changes Myocardial Infarction Complications of angina (high MI risk): Increased angina duration, frequency or at rest (unstable) ST or T wave changes Signs of HF Pulmonary edema Tachycardia Hypotension Acute: chest pain more intense than angina Diaphoresis SOB Generalized weakness May mistake S/S for indigestion Troponin – 4-6 h post up to 4-7 days; serial pattern Creatine kinase – (CK-NB) h to 48-72 h Myoglobin – damage to heart muscle EKG changes – place in tele MONA - Dilate coronary arteries Thrombolytics CABG Prevent ACS: O2, vasodilate, reperfuse, prevent thrombus, blood flow IHI: early admin of aspirin aspirin @ d/c 325 mg beta-blocker @ d/c
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