Thrombin increased expression of CAMs, di-phosphorylation of MLC, actin stress-fiber formation activation of mitogen activated protein kinases (MAPKs) and decreased phosphorylation of AKT.
Thrombín jók tjáningu viðloðunarsameinda, tvífosfórun á MLC, stressþráðamyndun aktíns og virkjun mitogen activated protein (MAP) kínasa en minnkaði fosfórun á AKT.