-- As an aside, it&#x27;s a textbook example of multiple discovery&#x2F;simultaneous invention: <a href="https:&#x2F;&#x2F;en.wikipedia.org&#x2F;wiki&#x2F;Multiple_discovery" rel="nofollow">https:&#x2F;&#x2F;en.wikipedia.org&#x2F;wiki&#x2F;Multiple_discovery</a> --<p>I also completely agree that the sedative-level dose given to soldiers after stress might have worked by blocking traumatic memory formation. It has already been established that blocking or &quot;erasing&quot; stress memories may prevent PTSD (here&#x27;s just one example: <a href="http:&#x2F;&#x2F;www.medscape.com&#x2F;viewarticle&#x2F;819452" rel="nofollow">http:&#x2F;&#x2F;www.medscape.com&#x2F;viewarticle&#x2F;819452</a>).<p>In the studies in mice, ketamine was no longer in the animals&#x27; systems by the time they were stressed. So they were not sedated. And the researchers tested memory formation to confirm it was not affected. Thus, it seems that ketamine&#x27;s protective effect in mice <i>isn&#x27;t</i> just because memory is disrupted. That said, it is always possible they missed something.<p>From what I can find, it doesn&#x27;t seem like anyone has even <i>looked</i> yet to see how this ketamine-vaccine strategy effects neuroplasticity. At this stage, it&#x27;s probably best to consider the post-stress effect of ketamine in soldiers (1), the pre-stress protection in mice (2), and the neuroplastic effects of ketamine (3) as three separate things.