&gt; <i>MP1 interacts with lipids that are abnormally distributed on the surface of cancer cells, creating gaping holes that allow molecules crucial for cell function to leak out.</i><p>This is fascinating. As IANAMD, I had no idea there were physical (or nanomechanical?) differences between normal and cancer cells. This sounds like a huge number of mistakes, maybe from the extremely rapid divisions.<p>So is <i>Polybia-MP1</i> another game changer in the Cancer world?