In the early part of the 20th century, Serratia marcescens was considered a non-pathogenic organism and was used in medical experiments and as a biological warfare test agent [1]. Since the mid-1970s, however, Serratia species have been recognized to cause a full spectrum of human clinical disease. Additionally, Serratia species may harbor multidrug-resistance mechanisms that can complicate treatment decisions. This article will review the microbiology, epidemiology, clinical features, diagnosis, and treatment of infections caused by Serratia species.
Scar tissue is the formation of fibrosis connective tissue that is created to act as a protective barrier. It replaces healthy tissues that have been damaged due to a number of reasons. Scar tissue generally forms in muscles, tendons, ligaments, fascia, and joints. It is also called an adhesion.
With keloids, the fibroblasts continue to multiply even after the wound is filled in. Thus keloids project above the surface of the skin and form large mounds of scar tissue. Keloids may form on any part of the body, although the upper chest, shoulders and upper back are especially prone to keloid formation. Symptoms include pigmentation of the skin, itchiness, redness, unusual sensations and pain.