Plant flowering involves a transition process from vegetative growth to reproductive development with a series of conserved underlying metabolic or external phenotypic changes taking place in the shoot apical meristem. In Arabidopsis thaliana, there are four major pathways controlling the timing of flowering, including photoperiod, vernalization, gibberellin (GA) and autonomous pathways (Mouradov et al. 2002). Major genetic elements involved in this pathways have been defined as the key switches to control flowering, such as CONSTANS (CO) in the photoperiodic pathway and FLOWERING LOCUS C (FLC) in the autonomous and vernalization pathways (Mouradov et al. 2002). The FLOWERING LOCUS T (FT) gene activates the expression of a number of flower developmental genes (Mouradov et al. 2002). In the orchid plant Phalaenopsis aphrodite, PaFT1 can suppress the delayed flowering caused by SHORT VEGATATIVE PHASE (SVP) and FRIGIDA (FRI) (Jang et al. 2015). The Cymbidium FT orthologous gene was also cloned and ectopic expression of CgFT resulted in early flowering in transgenic Arabidopsis (Huang et al. 2012). Over expression of DnAGL19, a SOC1-1/TM3-like ortholog in Arabidopsis resulted in a slightly accelerated flowering time under normal growth conditions (Liu et al. 2016).