To date, EDI-OCT has become a promising new method in clinical practice3. The changes to the choroid support the hypothesis that hemodynamic changes in the choroid may cause many ocular diseases. A thicker choroid is observed in central serous chorioretinopathy, primary angle closure, nanophthalmic eyes and uveitis compared with that in normal eyes. A thinner choroid is found in APAC-afflicted eyes with elevated IOP compared with that in eyes with normal IOP6, 7, 9–11. Choroidal thickness therefore was thought to be meaningful in the assessment of disease severity and in predicting prognosis. Tagawa et al. observed a thickened choroid prior to the recurrence of Vogt-Koyanagi-Harada disease11. SFCT increase might be an anatomic feature of angle-closure disease10, but not of open angle glaucoma12. In longer and more myopic young adult eyes, the SFCT is thinner13 and may be a useful prognostic modality in high myopia14. The significantly increased SFCT was observed throughout the 6-month follow-up after cataract surgery15. Shao et al. even found an association between thin SFCT and subcapsular cataract or cortical cataract16.