For labor to end successfully, the uterus needs to produce strong, coordinated and effective contractions. Under exertion, lactic acid is produced by glycolysis in all human cells. Glycolysis mainly occurs under hypoxic conditions; but the uterus is highly glycolytic and produces lactate even under normoxic conditions [9–11]. Repeated transient hypoxia is a normal feature of labor as the uterine vessels are occluded during each contraction [9–11]. Lactate will be produced and the levels of pH in the tissue will decrease. Decreasing pH leads to intracellular acidification and an inhibition of the Ca2+ channels in the myometrial cells . A decreased inflow of Ca2+ into the muscle means that the contraction will be weaker and consequently less effective [9, 10]. New studies show a significant correlation between lactate production, hypoxia and the effect of oxytocin [13–15]. This is an important interaction if labor is to end normally. The level of lactate has been recognized as a factor in uterine activity and pathophysiological processes during labor [13–15].