Studies have demonstrated that the dietary intake of fish is a major pathway for human exposure to PCBs (Caspersen et al. 2013; Kvalem et al. 2009; Sjodin et al. 2000). Fitzgerald et al. (2004) reported that Mohawk women had high serum levels of PCBs that were associated with consumption of local fish and not with the exposure to other sources, like residential soil, lived sites, and the other foodstuff (Fitzgerald et al. 2004). Furthermore, it was reported that fish and shellfish accounted for 30–84% of the dioxin toxic equivalences from food intake by a Japanese total-diet study (Sasamoto et al. 2006), indicating that dioxin and dioxin-like PCB intake also occurs through fish and shellfish. Sasamoto et al. (2006) reported that meat and egg consumption comprised the second highest contribution to dietary intake of dioxins and dioxin-like PCBs, indicating that egg consumption may be associated with higher serum dioxin and dioxin-like PCB concentrations. The Bureau of Social Welfare and Public Health, Tokyo Metropolitan Government, showed the levels of PCBs in meat and eggs were below the detection limit in a food contamination survey (Bureau of Social Welfare and Public Health, Tokyo Metropolitan Government, 2015); however, continuous monitoring of PCBs in these foodstuffs might be needed.