The health effects of fetal and child environmental exposure to chemicals are a major concern, and several cohort studies have been conducted worldwide (Ayotte et al. 2003; Govarts et al. 2012; James et al. 2002). Polychlorinated biphenyls (PCBs) are known to affect the endocrine systems in humans. Despite being banned in 2001 by the Stockholm Convention (UNEP 2001), PCBs widely persist in wildlife and humans because of their lipophilic properties, low water solubility, and bioaccumulation in fatty tissues (Safe 1994). PCBs are reported to have numerous adverse human health effects, including reproductive (Leoni et al. 1989), developmental (Jorissen 2007), immunological (Glynn et al. 2008), and neurological (Prince et al. 2006). Food is a major source of exposure to PCBs and dioxins, which are related compounds (Caspersen et al. 2013; Kvalem et al. 2009). It has been reported that the consumption of fish was positively associated with serum levels of total PCBs among Japanese women (Tsukino et al. 2006), suggesting that human PCB contamination levels are strongly affected by eating habits.