In addition, we compared by mixed linear regression the association of glycosylation with RA disease activity, as assessed by DAS28(3)-CRP, both between and within individuals with RA (respectively, adjusted for time point and individual) (Figure S2 and Table S6 in Supplementary Material). The increase in DAS28(3)-CRP associated with both the decrease in IgG-Fc-type galactosylation (e.g., A2S0G, βA2S0G = −0.34 ± 0.02) and the decrease in fucosylation of non-α2,3-sialylated triantennary species (A3L0F, βA3L0F = −0.13 ± 0.03). Increasing with DAS28(3)-CRP were the bisection of IgG-Fc-type glycans (A2F0S0B, βA2F0S0B = 0.25 ± 0.03) as well as the sialylation and fucosylation of tri- and tetraantennary species (e.g., A3FGS, βA3FGS = 0.26 ± 0.02; A4F, βA4F = 0.13 ± 0.03; A4GS, βA4GS = 0.17 ± 0.02). Glycosylation features associating with DAS28(3)-CRP only within (and not between) individuals throughout pregnancy were the fucosylation of (α2,6-)sialylated diantennaries (A2SF, βA2SF = −0.12 ± 0.02) and α2,6-sialylation of triantennary non-fucosylated species (A3F0GE, βA3F0GE = 0.14 ± 0.02).