There is a relationship between obesity and allergic diseases. Adipokines, which are fat proteins that function as cytokines, chemokines and cytokine receptors have an important role in that relationship. At present, most studies on obesity, allergic diseases and asthma are based on the inflammatory and metabolic roles of leptin and adiponectin. Adiponectin is an anti-inflammatory protein that inhibits IL6, the transcription factor NFκB and TNF-α and that increases the concentration of IL1 and IL10. Adiponectin levels are decreased in obese people due to necrosis of fat tissue resulting from hypoxia, which causes infiltration of polymorphonuclear cells and macrophages that secrete IL6 and TNF-α and inhibit the synthesis of adiponectin . Lectin is a proinflammatory protein that promotes the release of IL6 and TNF-α, decreases the activity of regulatory T cells, promotes Th1 lymphocyte activity and increases the levels of IFN-γ [38, 39].