We know that renal dysfunction is a serious complication of coronary revascularisation with CABG and results in increased morbidity and mortality rates and prolonged hospital stay.18 The injurious action of CABG on renal function is caused by several mechanisms, including non-pulsatile perfusion and increased levels of circulating catecholamines, cytokines and free haemoglobin.19 These effects result in damage to the glomerular as well as tubular structures, which, in turn, may cause renal dysfunction, especially in the presence of additional risk factors.20-21