The individuals conducting this experiment have identified that centrosome duplication is regulated by the following proteins: PLK4, CEP192, CEP152, CEP63, and CPAP. Among these, they found that CEP63 interacts with satellite proteins, namely KIAA0753 and another protein called CCDC14, to play a role in regulating centriole duplication. Their findings conclude that KIAA0753 positively regulates CEP63 centrosome localization and centriole duplication. A second study has to do with the identification of hepatic microvascular adhesion-related genes of human colon cancer cells. This experiment was conducted with a variety of genes using a method called random homozygous gene perturbation. "KIAA0753" was identified as a gene in a silenced state when screening for nonadherent cells. The article concludes that the identified genes, including "KIAA0753", may contribute to understand the metastatic process of colon cancer and possible treatments. Ubiquitin C has shown an interaction with KIAA0753 in humans. Further analysis of this interaction reveals that it is not due to neighboring or co-occurrence, but has been discovered experimentally. The study in question showed there to be a potential for binding between the two proteins. The following two proteins have been predicted, with high confidence, to interact with KIAA0753 through physical/neighboring interactions: WRD19, C2CD3. KIAA0753, KIAA1609, and SAV1 were found to interact with KIAA0753, determined through textmining methods.