Animal studies have shown no effects on fertility or teratogenicity, but studies in humans have not been performed. The excretion of permethrin in breastmilk is unknown, and it is recommended that breastfeeding be temporarily discontinued during treatment. Skin reactions are uncommon. Excessive exposure to permethrin can cause nausea, headache, muscle weakness, excessive salivation, shortness of breath, and seizures. Worker exposure to the chemical can be monitored by measurement of the urinary metabolites, while severe overdose may be confirmed by measurement of permethrin in serum or blood plasma.Permethrin does not present any notable genotoxicity or immunotoxicity in humans and farm animals, but is classified by the EPA as a likely human carcinogen when ingested, based on reproducible studies in which mice fed permethrin developed liver and lung tumors. Pharmacokinetics
Permethrin is a chemical categorized in the pyrethroid insecticide group. The chemicals in the pyrethroid family are created to emulate the chemicals found in the chrysanthemum flower. Absorption
Absorption of topical permethrin is minimal. One in vivo study demonstrated 0.5% absorption in the first 48 hours based upon excretion of urinary metabolites. Distribution
Distribution of permethrin has been studied in rat models, with highest amounts accumulating in fat and the brain. This can be explained by the lipophilic nature of the permethrin molecule. Metabolism
Metabolism of permethrin occurs mainly in the liver, where the molecule undergoes oxidation by the cytochrome P450 system, as well as hydrolysis, into metabolites. Elimination of these metabolites occurs via urinary excretion. Stereochemistry
Permethrin has four stereoisomers (two enantiomeric pairs), arising from the two stereocenters in the cyclopropane ring.
Neonatal hypoglycaemia can be a stand-alone condition or a clinical manifestation of other conditions. Signs and symptoms
The way in which neonatal hypoglycemia symptoms may be presented is vague or hard to tell apart from other conditions. The symptoms can be confused with:

hypocalcemia
Sepsis
CNS disorders
Cardiorespiratory problemsNeonatal hypoglycemia can also show no symptoms in some newborns or may be life-threatening.Some observed symptoms are (these symptoms may be transient but reoccurring):

Jitteriness
hypothermia 
irritability 
pallor 
tremors
twitching
weak or high pitched cry
lethargy
hypotonia
generalized seizures
coma
cyanosis
apnea
rapid and irregular respirations
diaphoresis
eye rolling
refusal to feed
hunger

Causes
Risk Factors
Mother
Risk factors in the mother that increased the risk of developing hypoglycemia shortly after birth include:

Type 1 diabetes
Gestational diabetes mellitus (Transient)
Intrapartum glucose administration (Transient)
Gestational hypertension
Preeclampsia
Terbutaline administration (Transient)
Intrauterine growth restriction (Transient)
Perinatal stress or asphyxia (Transient)

Baby
Babies which have an increased risk of developing hypoglycemia shortly after birth are:

hypothermia (Transient)
polycythemia (Transient)
hyperinsulinism (Recurrent)
IEMs (Recurrent)
Beckwith-Wiedmann syndrome (Recurrent)
nesidioblastosis (Recurrent)
Rh isoimmunization (Recurrent)
Certain endocrine disorders (Recurrent)
fetal hydrops
Prematurity (Transient)
congenital malformations
small for gestational age (Transient)
Large for gestational age (Transient)

Hyperinsulinism
The most common cause on neonatal hypoglycemia is hyperinsulinism. Hyperinsulinism is also called persistent hyperinsulemic hypoglycemia of infancy (PHHI). This is seen very frequently to the neonates born from mothers with diabetes. Congenital hyperinsulinism is correlated with the abnormality of beta-cell regulation within the pancreas. Isolated islet adenoma, which is a focal disease, is often the cause of congenital hyperinsulism. Drug-induced hyperinsulisim is correlated with the administration insulin or use of hypoglycemic medication. In critical cases, a drug called Diazoxide is availed to stop any secretion of insulin. Limited glycogen stores
Limited glycogen storage occurs in premature newborns or newborns that had intrauterine growth retardation.