In the fifth edition of the Diagnostic and Statistical Manual of Mental Disorders (DSM-5) it is instead considered a sub-category of the "Other Specified Obsessive-Compulsive and Related Disorder" category [300.3 (F42)], where variants of taijin kyofusho are "Shubo-kyofu [...] that is similar to body dysmorphic disorder and is characterized by excessive fear of having a bodily deformity" and "Jikoshu-kyofu [...] characterized by fear of having an offensive body odor".One source even breaks taijin kyofusho into two different subtypes: neurotic and offensive. The first subtype can be broken into two parts that are classical type and avoidant type. The classical type being afraid of being judged negatively because of physical signs of anxiety and feeling shame due to anxiety. The physical signs that can cause fear of being judged include sweating and tremors. The second subtype deals with people thinking something about them is offensive. Some of their fears include body odor, gas, excessive or insufficient eye contact, blushing, etc. In the official Japanese diagnostic system, taijin kyofusho is subdivided into the following categories:
Sekimen-kyofu (Japanese: 赤面恐怖), the phobia of blushing (ereuthophobia)
Shubo-kyofu (Japanese: 醜貌恐怖), the phobia of a deformed body, similar to body dysmorphic disorder
Jikoshisen-kyofu (Japanese: 自己視線恐怖), the phobia of eye contact
Jikoshu-kyofu (Japanese: 自己臭恐怖), the phobia of having foul body odor (also termed olfactory reference syndrome, osmophobia or bromidrosiphobia)Japan psychology also recognizes additional types of taijin kyofusho based on severity:

Transient: This type of taijin kyofusho is short-lived and moderately severe. It most commonly appears in teens, but may occur at any time.
Increases in expression of such genes as Pax-2, as well as inhibition of Pax-6, from the notochord have been implicated in normal differentiation of cephalic midline structures. Inappropriate expression of any of these genes may result in mild to severe forms of holoprosencephaly. Other candidate genes have been located, including the SHH (holoprosencephaly type 3 a.k.a. HPE3), TGIF, ZIC2, SIX3 and BOC genes.Although many children with holoprosencephaly have normal chromosomes, specific chromosomal abnormalities have been identified in some patients (trisomy of chromosome 13, also known as Patau syndrome). There is evidence that in some families, HPE is inherited (autosomal dominant as well as autosomal or X-linked recessive inheritance). Features consistent with familial transmission of the disease (e.g., a single central maxillary incisor) should be carefully assessed in parents and family members. Non-genetic factors
Numerous possible risk factors have been identified, including gestational diabetes, transplacental infections (the "TORCH complex"), first trimester bleeding, and a history of miscarriage. As well, the disorder is found twice as often in female babies. However, there appears to be no correlation between HPE and maternal age.There is evidence of a correlation between HPE and the use of various drugs classified as being potentially unsafe for pregnant and lactating mothers. These include insulin, birth control pills, aspirin, lithium, thorazine, retinoic acid, and anticonvulsants. There is also a correlation between alcohol consumption and HPE, along with nicotine, the toxins in cigarettes and toxins in cigarette smoke when used during pregnancy. Prognosis
HPE is not a condition in which the brain deteriorates over time.