The thinning of myelin sheaths or focal demyelination are the main consequences of the injury that lead to conduction blockage. Diagnosis
Seddon classification
There are three distinct classifications and degrees of nerve injury:

Neurotmesis is the most serious degree of nerve injury. It involves the disruption of the nerve and the nerve sheath. Axonotmesis occurs when the majority of the supporting structures of the nerve are preserved, but disruption of the nerve fibers is still observed. Wallerian degeneration often occurs in the near the proximity of the injury site. Neurapraxia is least serious form of nerve injury.There are two different forms of mechanical nerve injury involving neurapraxia. The underlying causes of transient nerve injury typically include a brief ischemic episode or any form of compression. More persistent forms of nerve injury involve demyelination and axonal constriction. In certain circumstances, diagnosing neurapraxia can be uncomfortable because of the presence of severe neuropathic pain. Neuropathic pain is an indication that the lesion of the nerve is still in progress. Diagnosis of neurapraxia is almost always followed by a quick and complete recovery period. Treatment and recovery
The entire nerve is involved in the response to traumatic injuries. The outcome of nerve repair is dependent on the degree of the nerve injury and the circumstances at the site of injury. Since neurapraxia is the least serious form of peripheral nerve injury, recovery and treatment are not extensive. Once the cause of neurapraxia is eliminated, recovery of the lesions in the nerve occurs within a short time span.
Non-operative treatment
Neurapraxia is often treated and cured by non-operative means. The primary goals of treatment are to maintain the proper nutrition of the paralyzed muscles, prevent contraction by the antagonists of the paralyzed muscles, and to consistently keep the joints mobile. A splint is often used in cases of neurapraxia because it is able to maintain a relaxed position of the paralyzed muscle. The splint prevents the paralyzed muscle from being overstretched either by the force of gravity or by other non-paralyzed antagonists. During the recovery period of neurapraxia, it is essential that the joints constantly undergo passive movement in order to preserve proper mobility. If joints are kept mobile, the limb has the best possible chance of benefit from the return of nervous function. Non-steroidal anti-inflammatory medications can also help to reduce swelling at the injury site. In addition to these non-operative remedies, it is suggested that muscles affected by neurapraxia be kept warm at all times. Circulation in the limb is stimulated with the use of heat. Once voluntary movement has returned to the muscle, recovery and treatment continues by the participation in active exercises. Physical Therapy and Occupational Therapy are common sources of treatment during these early stages of restoration of active movement. Almost all cases of neurapraxia can be completely treated by non-operative means.