Dysplasia in the myeloid series is defined by:

Granulocytic series:
Hypersegmented neutrophils (also seen in vit B12/folate deficiency)
Hyposegmented neutrophils (pseudo Pelger-Huet)
Hypogranular neutrophils or pseudo Chediak-Higashi (large azurophilic granules)
Auer rods – automatically RAEB II (if blast count < 5% in the peripheral blood and < 10% in the bone marrow aspirate); also note Auer rods may be seen in mature neutrophils in AML with translocation t(8;21)
Dimorphic granules (basophilic and eosinophilic granules) within eosinophils
Erythroid series:
Binucleated erythroid precursors and karyorrhexis
Erythroid nuclear budding
Erythroid nuclear strings or internuclear bridging (also seen in congenital dyserythropoietic anemias)
Loss of e-cadherin in normoblasts is a sign of aberrancy. PAS (globular in vacuoles or diffuse cytoplasmic staining) within erythroid precursors in the bone marrow aspirate (has no bearing on paraffin-fixed bone-marrow biopsy). Note: one can see PAS vacuolar positivity in L1 and L2 blasts (FAB classification; the L1 and L2 nomenclature is not used in the WHO classification)
Ringed sideroblasts (10 or more iron granules encircling one-third or more of the nucleus) seen on Perls Prussian blue iron stain (>15% ringed sideroblasts when counted among red cell precursors for refractory anemia with ring sideroblasts)
Megakaryocytic series (can be the most subjective):
Hyposegmented nuclear features in platelet producing megakaryocytes (lack of lobation)
Hypersegmented (osteoclastic appearing) megakaryocytes
Ballooning of the platelets (seen with interference contrast microscopy)Other stains can help in special cases (PAS and naphthol ASD chloroacetate esterase positivity) in eosinophils is a marker of abnormality seen in chronic eosinophilic leukemia and is a sign of aberrancy.
Head-to-head contact is by far the most common route of lice transmission.The United Kingdoms National Health Service, and many American health agencies, report that lice "prefer" clean hair, because its easier to attach eggs and to cling to the strands.Head lice (Pediculus humanus capitis) are not known to be vectors of diseases, unlike body lice (Pediculus humanus humanus), which are known vectors of epidemic or louse-borne typhus (Rickettsia prowazekii), trench fever (Rochalimaea quintana) and louse-borne relapsing fever (Borrelia recurrentis). Body lice
This condition is caused by body louse (Pediculus humanus humanus, sometimes called Pediculus humanus corporis), a louse which infests humans and is adapted to lay eggs in clothing, rather than at the base of hairs, and is thus of recent evolutionary origin. Pediculosis is a more serious threat due to possible contagion of diseases such as typhus. Epidemiology and treatment of human body lice is described in the article on body lice. Pubic lice
The pubic or crab louse (Pthirus pubis) is a parasitic insect which spends its entire life on human hair and feeds exclusively on blood. Humans are the only known host of this parasite, although it is more closely related to the louse parasites in other primate species, than are human head or body lice which probably evolved from it as the "original" louse infestation of humans. Epidemiology and treatment of pubic lice is discussed in the article on pubic lice. Other animals
Pediculosis is more common in cattle than any other type of domesticated animal.