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Thrombocytopenia
Immune thrombocytopenias (ITP) – formerly known as immune thrombocytopenic purpura and idiopathic thrombocytopenic purpura
Splenomegaly
Gauchers disease
Familial thrombocytopenia
Chemotherapy
Babesiosis
Dengue fever
Onyalai
Thrombotic thrombocytopenic purpura
HELLP syndrome
Hemolytic–uremic syndrome
Drug-induced thrombocytopenic purpura (five known drugs – most problematic is heparin-induced thrombocytopenia (HIT)
Pregnancy-associated
Neonatal alloimmune associated
Aplastic anemia
Transfusion-associated
Pseudothrombocytopenia
Idiopathic thrombocytopenic purpura
Vaccine induced immune thrombocytopenia
Gilberts syndrome

Altered platelet function (thrombocytopathy)
Congenital
Disorders of adhesion
Bernard–Soulier syndrome
Disorders of activation
Disorders of granule amount or release
Hermansky–Pudlak syndrome
Gray platelet syndrome
ADP receptor defect
Decreased cyclooxygenase activity
Platelet storage pool deficiency
Disorders of aggregation
Glanzmanns thrombasthenia
Wiskott–Aldrich syndrome
Disorders of coagulant activity
COAT platelet defect
Scott syndrome
Acquired
Disorders of adhesion
Paroxysmal nocturnal hemoglobinuria
Asthma
Samters triad (aspirin-exacerbated respiratory disease/AERD)
Cancer
Malaria
Decreased cyclooxygenase activity

Thrombocytosis and thrombocythemia
Reactive
Chronic infection
Chronic inflammation
Malignancy
Hyposplenism (post-splenectomy)
Iron deficiency
Acute blood loss
Myeloproliferative neoplasms – platelets are both elevated and activated
Essential thrombocythemia
Polycythemia vera
Associated with other myeloid neoplasms
Congenital

Drugs affecting
Anti-inflammatory drugs
Some drugs used to treat inflammation have the unwanted side effect of suppressing normal platelet function. These are the non-steroidal anti-inflammatory drugs (NSAIDS). Aspirin irreversibly disrupts platelet function by inhibiting cyclooxygenase-1 (COX1), and hence normal hemostasis. The resulting platelets are unable to produce new cyclooxygenase because they have no DNA. Normal platelet function will not return until the use of aspirin has ceased and enough of the affected platelets have been replaced by new ones, which can take over a week. Ibuprofen, another NSAID, does not have such a long duration effect, with platelet function usually returning within 24 hours, and taking ibuprofen before aspirin prevents the irreversible effects of aspirin. Drugs that suppress platelet function
These drugs are used to prevent thrombus formation.
Avita may refer to:

Australian Green Tree Frog
Tretinoin