About half of PTE cases go into remission, but cases that occur later may have a smaller chance of doing so. Epidemiology
Studies have found that the incidence of PTE ranges between 1.9 and more than 30% of those with TBI, varying by severity of injury and by the amount of time after TBI for which the studies followed subjects.Brain trauma is one of the strongest predisposing factors for epilepsy development, and is an especially important factor in young adults. Young adults, who are at the highest risk for head injury, also have the highest rate of PTE, which is the largest cause of new-onset epilepsy cases in young people. Children have a lower risk for developing epilepsy; 10% of children with severe TBI and 16–20% of similarly injured adults develop PTE. Being older than 65 is also a predictive factor in the development of epilepsy after brain trauma. One study found PTE to be more common in male TBI survivors than in females. History
Records of PTE exist from as early as 3000 BC. Trepanation, in which a hole is cut in the skull, may have been used to treat PTE in ancient cultures. In the early 19th century, the surgeons Baron Larrey and WC Wells each reported having performed the operation for PTE. The French-educated American surgeon Benjamin Winslow Dudley (1785–1870) performed six trepanations for PTE between the years of 1819 and 1832 in Kentucky and had good results despite the unavailability of antisepsis.
Hypercementosis is an idiopathic, non-neoplastic condition characterized by the excessive buildup of normal cementum (calcified tissue) on the roots of one or more teeth. A thicker layer of cementum can give the tooth an enlarged appearance, which mainly occurs at the apex or apices of the tooth. Signs and symptoms
It is experienced as an uncomfortable sensation in the tooth, followed by an aching pain.It may be shown on radiographs as a radiopaque (or lighter) mass at each root apex. Cause
Can be caused by many things. A way to remember the causes is "PIG ON TAP"Local factors- 

Occlusal Trauma
Trauma
Non-functional tooth
Unopposed tooth (and impacted teeth, embedded teeth, teeth without antagonists)Systemic factors-

Idiopathic
Pituitary Gigantism
Pagets Disease
Acromegaly
Periapical granuloma
Arthritis
Calcinosis
Rheumatic feverIt may be one of the complications of Pagets disease of bone in the form of generalized hypercementosis. It may also be a compensatory mechanism in response to attrition to increase occlusal tooth height. Complications
Such deposits form bulbous enlargements on the roots and may interfere with extractions, especially if adjacent teeth become fused (concrescence). It may also result in pulpal necrosis by blocking blood supply via the apical foramen. References


== External links ==