p. 244. ISBN 978-0-19-534285-7. OCLC 162501989. Hoyano, L.; Keenan C. (2007). Child Abuse: Law and Policy Across Boundaries. Oxford; New York: Oxford University Press. ISBN 978-0-19-829946-2. OCLC 79004390. Korbin, Jill E. (1983). Child abuse and neglect: cross-cultural perspectives. Berkeley, CA: University of California Press. ISBN 978-0-520-05070-9. OCLC 144570871. Miller, Alice (1990). "Thou Shalt Not Be Aware": Societys Betrayal of the Child, in series, Meridian Book[s]. Trans. by Heildegarde and Hunter Hannum. New York: Penguin. x, 329 p. Trans. from the German, titled Du sollst nicht merken. ISBN 0-452-00929-4 pbk
Turton, Jackie (2008). Child Abuse, Gender, and Society. New York: Routledge. p. 161. ISBN 978-0-415-36505-5. OCLC 144570871. Young, Leontine (1964). Wednesdays children: A study of child neglect and abuse. New York: McGraw-Hill. OCLC 192177. External links

Child abuse at Curlie
Child maltreatment, World Health Organization
World Report on Violence Against Children, Secretary-General of the United Nations
Prevent Child Abuse America
Pete (award-winning 2004 short film)
"Cold-nosed Comfort" (using a service dog to aid child abuse victims), Maryland Lawyer
nspcc.org.uk
Argyll Robertson pupils (AR pupils) are bilateral small pupils that reduce in size on a near object (i.e., they accommodate), but do not constrict when exposed to bright light (i.e., they do not react). They are a highly specific sign of neurosyphilis; however, Argyll Robertson pupils may also be a sign of diabetic neuropathy. In general, pupils that accommodate but do not react are said to show light-near dissociation (i.e., it is the absence of a miotic reaction to light, both direct and consensual, with the preservation of a miotic reaction to near stimulus (accommodation/convergence).AR pupils are extremely uncommon in the developed world. There is continued interest in the underlying pathophysiology, but the scarcity of cases makes ongoing research difficult. Pathophysiology
The two different types of near response are caused by different underlying disease processes. Adies pupil is caused by damage to peripheral pathways to the pupil (parasympathetic neurons in the ciliary ganglion that cause pupillary constriction to bright light and with near vision). The pathophysiologic mechanism which produces an Argyll Robertson pupil is unclear, but is believed to be the result of bilateral damage to the pretectal nuclei in the midbrain. Studies have failed to demonstrate a focal localising lesion. Research has implicated the rostral midbrain in the vicinity of the cerebral aqueduct of the third ventricle as the most likely region of damage.