For those with severe stenosis that are at risk for impending stroke endovascular treatment is used. Depending on the individual and the location of the stenosis there are multiple treatments that can be undertaken. These include angioplasty, stent insertion, or bypass the blocked area. Related diseases
Diseases associated with cerebral atherosclerosis include:

Hypertensive arteriopathyThis pathological process involves the thickening and damage of arteriole walls. It mainly affects the ends of the arterioles which are located in the deep gray nuclei and deep white matter of the brain. It is thought that this is what causes cerebral microbleeds in deep brain regions. This small vessel damage can also reduce the clearance of amyloid-β, thereby increasing the likelihood of CAA.Diseases cerebral atherosclerosis and associated diseases can cause are:

Alzheimers diseaseAlzheimers disease is a form of dementia that entails brain atrophy. Cerebral amyloid angiopathy is found in 90% of the cases at autopsy, with 25% being severe CAA. Cerebral microbleeds (CMB)Cerebral microbleeds have been observed during recent studies on people with dementia using MRI. StrokeStrokes occur from the sudden loss of blood flow to an area of the brain. The loss of flow is generally either from a blockage or hemorrhage. Studies of postmortem stroke cases have shown that intracranial atherosclerotic plaque build up occurred in over half of the individuals and over one third of the overall cases had stenotic build up. == References ==
Transient neonatal pustular melanosis (TNPM), also known as pustular melanosis, is a transient rash common in newborns. It is vesiculopustular and made up of 1–3 mm fluid-filled lesions that rupture, leaving behind a collarette of scale and a brown macule. [3] This rash occurs only in the newborn stage, usually appearing a few days after birth[2], but is sometimes already present at birth[3]. The rash usually fades over three to four weeks  but may linger for up to three months after birth. [3] It can occur anywhere on the body, including the palms and soles. [1][2][3]The cause of TNPM is unknown. No treatment is needed except for parental reassurance. Causes
The cause of TNPM is unknown but it is a common rash in newborns. [3]

Histopathology
Gram, Wright, or Giemsa staining of the pustular contents will show polymorphic neutrophils and occasional eosinophils. [2][3] On histopathology, the pigmented macules will show basal and supra-basal increase in pigmentation without any pigmentary incontinence. [3] Bacterial culture will be negative. [4]It has been suggested that TNPM is merely a precocious form of erythema toxicum neonatorum based on the similar histopathology. [4]

Diagnosis
Transient neonatal pustular melanosis is diagnosed clinically, based on appearance alone, with no need for special testing. Proper identification is important to distinguish it from other serious, infectious neonatal diseases[3] and to help avoid unnecessary diagnostic testing and treatments. Treatment
No treatment is needed except for parental reassurance. The rash spontaneously resolves, usually in three to four weeks,  but may linger for up to three months after birth.