Albinism–black lock–cell migration disorder of the neurocytes of the gut–deafness syndrome (ABCD syndrome)
Albinism–deafness syndrome (Woolf syndrome, Ziprkowski–Margolis syndrome)
Alezzandrini syndrome
Argyria
Arsenic poisoning 
Berlin syndrome
Canthaxanthin
Chédiak–Higashi syndrome
Chrysiasis
Cross–McKusick–Breen syndrome (Cross syndrome, oculocerebral-hypopigmentation syndrome)
Dermatopathia pigmentosa reticularis (dermatopathia pigmentosa reticularis hyperkeratotica et mutilans, dermatopathia pigmentosa reticularis hypohidotica et atrophica, dermatopathic pigmentosa reticularis)
Dyschromatosis symmetrica hereditaria (reticulate acropigmentation of Dohi, symmetrical dyschromatosis of the extremities)
Dyschromatosis universalis hereditaria
Elejalde syndrome (Griscelli syndrome type 1)
Eruptive hypomelanosis
Familial progressive hyperpigmentation
Galli–Galli disease
Griscelli syndrome type 2 (partial albinism with immunodeficiency)
Griscelli syndrome type 3
Hemochromatosis (bronze diabetes)
Hemosiderin hyperpigmentation
Hermansky–Pudlak syndrome
Idiopathic guttate hypomelanosis (leukopathia symmetrica progressiva)
Iron metallic discoloration
Klein–Waardenburg syndrome
Lead poisoning
Leukoderma
Melanoma-associated leukoderma
Melasma (chloasma faciei, mask of pregnancy)
Mukamel syndrome
Necklace of Venus
Nevus anemicus 
Nevus depigmentosus (nevus achromicus)
Ocular albinism
Oculocutaneous albinism
Pallister–Killian syndrome
Periorbital hyperpigmentation
Photoleukomelanodermatitis of Kobori
Phylloid hypomelanosis
Piebaldism
Pigmentatio reticularis faciei et colli
Pityriasis alba
Poikiloderma of Civatte
Poikiloderma vasculare atrophicans
Postinflammatory hyperpigmentation (postinflammatory hypermelanosis)
Postinflammatory hypopigmentation
Progressive macular hypomelanosis
Quadrichrome vitiligo
Reticular pigmented anomaly of the flexures (dark dot disease, Dowling–Degos disease)
Reticulate acropigmentation of Kitamura
Revesz syndrome
Riehl melanosis
Scratch dermatitis (flagellate pigmentation from bleomycin)
Segmental vitiligo
Shah–Waardenburg syndrome
Shiitake mushroom dermatitis (flagellate mushroom dermatitis, mushroom workers disease, shiitake-induced toxicoderma)
Tar melanosis (melanodermatitis toxica lichenoides)
Tietz syndrome
Titanium metallic discoloration
Transient neonatal pustular melanosis (transient neonatal pustulosis, lentigines neonatorum)
Trichrome vitiligo
Vagabonds leukomelanoderma
Vasospastic macule
Vitiligo
Vitiligo ponctué
Vogt–Koyanagi–Harada syndrome
Waardenburg syndrome
Wende–Bauckus syndrome (Pegum syndrome)
Woronoffs ring
X-linked reticulate pigmentary disorder (familial cutaneous amyloidosis, Partington amyloidosis, Partington cutaneous amyloidosis, Partington syndrome type II, reticulate pigmentary disorder, X-linked reticulate pigmentary disorder with systemic manifestations)
Yemenite deaf-blind hypopigmentation syndrome

Drug eruptions
Drug eruptions are adverse drug reactions that present with cutaneous manifestations.
Studies have suggested that these tumors are due to the expression of male sex hormones (i.e. androgens and progesterones), genetic factors, molecular alterations (i.e. changes in the normal characteristics of cells that lead to abnormal cell growth), and/or human papillomavirus infection.Angiofibroma of soft tissue is also named angiofibroma, not otherwise specified, by the World Health Organization, 2020. The Organization also classified these lesions as in the category of benign fibroblastic and myofibroblastic tumors. These tumors more often afflict females, typically occur in adults (median age 49 years), have a median size of ~3.5 cm, and develop in a leg near to, and may invade, a large joint. Less uncommonly, they occur in the back, abdominal wall, pelvic cavity, or breast. Angiofibroma of soft tissue tumors consist of uniform, bland, spindle-shaped cells and a prominent vascular network consisting of small thin-walled branching blood vessels in a variably collagenous tissue background. Its tumor cells contain an AHRR-NCOA2 fusion gene in 60% to 80% of cases and a GTF2I-NCOA2 or GAB1-ABL1 fusion gene in rare cases.Cellular angiofibroma is usually a small, slow-growing tumor arising in the vulva-vaginal areas of adult woman and the inguinal-scrotal areas of adult men although some of these tumors, especially in men, can grow up to 25 cm. Affected men are usually older (7th decade) than women (5th decade). Less commonly. cellular angiofibromas have occurred in various other superficial soft tissue areas throughout the body. These tumors are edematous (i.e.