A total of 31 patients were treated with Glybera, most for free in clinical trials before the drug was taken off the market. Incidence
The disorder affects about 1 out of 1,000,000 people; however, epidemiological data are limited and there are regional differences due to cofounder effect (e.g. in Canada) or intermarriage. See also
Primary hyperlipoproteinemia
Familial apoprotein CII deficiency
List of cutaneous conditions

References
Further reading
Gaudet, D; de Wal, J; Tremblay, K; Déry, S; van Deventer, S; Freidig, A; Brisson, D; Méthot, J (June 2010). "Review of the clinical development of alipogene tiparvovec gene therapy for lipoprotein lipase deficiency". Atherosclerosis. Supplements. 11 (1): 55–60. doi:10.1016/j.atherosclerosissup.2010.03.004. PMID 20427244. "LPL gene". NIH Genetics Home Reference. February 2015. Hegele, RA;  et al. (August 2014). "The polygenic nature of hypertriglyceridaemia: implications for definition, diagnosis, and management". The Lancet. Diabetes & Endocrinology. 2 (8): 655–66. doi:10.1016/S2213-8587(13)70191-8. PMC 4201123. PMID 24731657. == External links ==
Fovilles syndrome is caused by the blockage of the perforating branches of the basilar artery in the region of the brainstem known as the pons. It is most frequently caused by vascular disease or tumors involving the dorsal pons. Structures affected by the infarct are the dorsal pons(pontine tegmentum) which comprises paramedian pontine reticular formation (PPRF), nuclei of cranial nerves VI and VII, corticospinal tract, medial lemniscus, and the medial longitudinal fasciculus. There is involvement of the fifth to eighth cranial nerves, central sympathetic fibres (Horner syndrome) and horizontal gaze palsy. Presentation
This produces ipsilateral horizontal gaze palsy and facial nerve palsy and contralateral hemiparesis, hemisensory loss, and internuclear ophthalmoplegia. Diagnosis
Treatment
History
Fovilles syndrome was initially described by Achille-Louis Foville, a French physician, in 1859. References


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