Diuretics: Patients treated with fenoprofen may be resistant to the effects of loop diuretics and thiazides. Hydantoins, sulfonamides, sulfonylureas: Fenoprofen may displace these drugs from their binding site. Lithium: Renal Cl of lithium may be reduced and plasma levels may be elevated, which may increase the risk of lithium toxicity. Methotrexate: May increase methotrexate levels. Phenobarbital: May decrease fenoprofen t 1⁄2 . Dosage adjustments of fenoprofen may be required if phenobarbital is added or withdrawn. SSRIs (e.g. fluoxetine, citalopram): The risk of GI effects may be increased. Laboratory test interactions
False elevation in free and total serum T 3 as measured by Amerlex-M kit. Brand names
UK - Fenopron (Typharm Limited)
See also
Ibuprofen
Ketoprofen
Thalidomide
Chirality
Chiral drugs
Chiral inversion

References
External links
Fenoprofen info from Drugs.com
And that if this were the case, then the mechanism remains unclear.Administration of nitrous oxide anesthesia can precipitate subacute combined degeneration in people with subclinical vitamin B12 deficiency, while chronic nitrous oxide exposure can cause it even in persons with normal B12 levels. Posterior column dysfunction decreases vibratory sensation and proprioception (joint sense). Lateral corticospinal tract dysfunction produces spasticity and dorsal spinocerebellar tract dysfunction causes ataxia. Cause
In general, the most common cause of this condition is a deficiency of vitamin B12. This may be due to a dietary deficiency, malabsorption in the terminal ileum, lack of intrinsic factor secreted from gastric parietal cells, or low gastric pH inhibiting attachment of intrinsic factor to ileal receptors.Vitamin E deficiency, which is associated with malabsorption disorders such as cystic fibrosis and Bassen-Kornzweig syndrome, can cause a similar presentation due to the degeneration of the dorsal columns. Diagnosis
Serum vitamin B12, methylmalonic acid, Schilling test, and a complete blood count, looking for megaloblastic anemia if there is also folic acid deficiency or macrocytic anemia. The Schilling test is no longer available in most areas.MRI-T2 images may reveal increased signal within the white matter of the spinal cord, predominantly in the posterior columns and possibly in the spinothalamic tracts. Treatment
Therapy with vitamin B12 results in partial to full recovery where SACD has been caused by vitamin B12 deficiency, depending on the duration and extent of neurodegeneration. References


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