Their parents, who are often their only source of care, are into their seventies or eighties or already deceased. Often, these patients find themselves tied to their own homes and the care of their family, effectively isolated from the local community. Some welfare facilities for patients do exist. One notable example is Hot House, a vocational training centre for congenital patients as well as other disabled people in the Minamata area. Hot House members are also involved in raising awareness of Minamata disease, often attending conferences and seminars as well as making regular visits to elementary schools throughout Kumamoto Prefecture. See also
Heavy metal poisoning
Minamata Convention on Mercury
Ontario Minamata disease
Mercury in fish

References
Further reading
"Minamata Disease: The History and Measures", The Ministry of the Environment, (2002), retrieved 17 January 2007
"Minamata Disease Archives" by the National Institute for Minamata Disease, retrieved 29 October 2006
Harada, Masazumi. (1972). Minamata Disease. Kumamoto Nichinichi Shinbun Centre & Information Center/Iwanami Shoten Publishers. ISBN 4-87755-171-9 C3036
George, S. Timothy. (2001). Minamata: Pollution and the Struggle for Democracy in Postwar Japan. Harvard University Press. ISBN 0-674-00785-9
Ui, Jun. (1992). Industrial Pollution in Japan. United Nations University Press. ISBN 92-808-0548-7. Chapter 4, section IV
Smith, W. E. and Smith, A. M. (1975). Minamata. Chatto & Windus, Ltd. (London), ISBN 0-7011-2131-9
Eto, K., Marumoto, M. and Takeya, M. (2010) "The pathology of methylmercury poisoning (Minamata disease)", retrieved 7 December 2013
Oiwa, Keibo. (2001). Rowing the Eternal Sea: The Story of a Minamata Fisherman. Rowman & Littlefield Publishers. ISBN 0-7425-0021-7
Steingraber, Sandra. (2001). Having Faith: An Ecologist Journey to Motherhood. Perseus Publishing.
Autosomal dominant hypophosphatemic rickets (ADHR) is a rare hereditary disease in which excessive loss of phosphate in the urine leads to poorly formed bones (rickets), bone pain, and tooth abscesses. ADHR is caused by a mutation in the fibroblast growth factor 23 (FGF23). ADHR affects men and women equally; symptoms may become apparent at any point from childhood through early adulthood. Blood tests reveal low levels of phosphate (hypophosphatemia) and inappropriately normal levels of vitamin D. Occasionally, hypophosphatemia may improve over time as urine losses of phosphate partially correct.ADHR may be lumped in with X-linked hypophosphatemia under general terms such as hypophosphatemic rickets. Hypophosphatemic rickets are associated with at least nine other genetic mutations. Clinical management of hypophosphatemic rickets may differ depending on the specific mutations associated with an individual case, but treatments are aimed at raising phosphate levels to promote normal bone formation. References
Further reading
"Hereditary hypophosphatemic rickets". Genetics Home Reference. September 2010. Archived from the original on 17 September 2012. Retrieved 10 October 2012. "Hypophosphatemic Rickets". The Merck Manual Home Health Handbook. Merck Sharp & Dohme Corp. December 2006. Archived from the original on 13 October 2012. Retrieved 10 October 2012. == External links ==