It was also found that some people with declarative information amnesia are able to be primed.Studies were completed consistently throughout Molaisons lifetime to discover more about amnesia. Researchers did a 14-year follow-up study on Molaison. They studied him for a period of two weeks to learn more about his amnesia. After 14 years, Molaison still could not recall things that had happened since his surgery. However, he could still remember things that had happened prior to the operation. Researchers also found that, when asked, Molaison could answer questions about national or international events, but he could not remember his own personal memories. After his death Molaison donated his brain to science, where they were able to discover the areas of the brain that had the lesions which caused his amnesia, particularly the medial temporal lobe. This case study provided important insight to the areas of the brain that are affected in anterograde amnesia, as well as how amnesia works. H.M.s case showed us that memory processes are consolidated into different parts of the brain and that short-term and working memory are not usually impaired in cases of amnesia. Clive Wearing
Another famous historical case of amnesia was that of Clive Wearing. Clive Wearing was a conductor and musician who contracted herpes simplex virus. This virus affected the hippocampal regions of the brain. Because of this damage, Wearing was unable to remember information for more than a few moments. Wearings non-declarative memory was still functioning but his declarative memory was impaired.
Miosis, or myosis (from Ancient Greek  μύειν (múein) to close the eyes), is excessive constriction of the pupil. The opposite condition, mydriasis, is the dilation of the pupil. Anisocoria is the condition of one pupil being more dilated than the other. Causes
Age
Senile miosis (a reduction in the size of a persons pupil in old age)

Diseases
Horners syndrome
Hemorrhage into pons (intracranial hemorrhage)
Hereditary disorders
Cluster headaches with ptosis
Iridocyclitis
Fatal familial insomnia
Aphakia

Drugs
Opioids such as fentanyl, morphine, heroin and methadone (the notable exception being pethidine)
Products containing nicotine such as cigarettes, chewing tobacco or gum. Imidazolines such as clonidine, naphazoline, oxymetazoline and tetrahydrozoline
Antipsychotics, including risperidone, haloperidol, chlorpromazine, olanzapine, quetiapine
Cholinergic agents such as acetylcholine
Acetylcholinesterase inhibitors
Serotonin antagonists, such as Ondansetron (an anti-emetic) known by its brand name Zofran
Some cancer chemotherapy drugs, including camptothecin derivatives
Mirtazapine, a noradrenergic and specific serotonergic antidepressant (NaSSA)
Some MAO Inhibitors. Pilocarpine eye drops and all other parasympathomimetics
In some rare cases, when exposed to mustard gas
Organophosphates

Physiology of the photomotor reflex
Light entering the eye strikes three different photoreceptors in the retina: the familiar rods and cones used in image forming and the more newly discovered photosensitive ganglion cells. The ganglion cells give information about ambient light levels, and react sluggishly compared to the rods and cones. Signals from photosensitive ganglion cells have multiple functions including acute suppression of the hormone melatonin, entrainment of the bodys circadian rhythms and regulation of the size of the pupil. The retinal photoceptors convert light stimuli into electric impulses.