Full employment of the unemployed workforce, all focused toward the goal of developing more environmentally-efficient methods for production and consumption, might provide a more significant and lasting cumulative environmental benefit and reduced resource consumption.Some critics of the "culture of work" such as the anarchist Bob Black see employment as culturally overemphasized in modern countries. Such critics often propose quitting jobs when possible, working less, reassessing the cost of living to that end, creation of jobs that are "fun" as opposed to "work," and creating cultural norms in which work is seen as unhealthy. These people advocate an "anti-work" ethic for life. Decline in work hours
As a result of productivity, the work week declined considerably during the 19th century. By the 1920s, the average workweek in the US was 49 hours, but it was reduced to 40 hours (after which overtime premium was applied) as part of the 1933 National Industrial Recovery Act. During the Great Depression, the enormous productivity gains caused by electrification, mass production, and agricultural mechanization were believed to have ended the need for a large number of previously-employed workers. Remedies
Societies try a number of different measures to get as many people as possible into work, and various societies have experienced close to full employment for extended periods, particularly during the post-World War II economic expansion.
The patient can also experience damage or loss of teeth. Obvious signs
Most obvious sign is gingival overgrowth (overgrowth of the gums)
Hindered chewing efficiency and difficulties eating
Increasing mobility of teeth
Abnormally shaped teeth and abnormal movement of teeth
Inflammation and/or swelling of the gums/gingiva
Not necessarily any signs of pain but experiencing pain is possible
Difficulties in speaking, oftentimes can lead to speech disorders
Other dental and oral problems
In some cases, Hereditary Gingival Fibromatosis may cause bleeding from the gums, or gum ulcerations. Cause
Though much more research needs to be done, researchers have mostly agreed that a mutation in SOS1, son-of-sevenless gene, is responsible for this disease. SOS1 is a guanine nucleotide-exchange factor that functions in the transduction of signals that control cell growth and differentiation. A mutation in the SOS1 gene results in a single nucleotide insertion. Specific linkage studies have localized the mutation for isolated, nonsyndromic autosomal dominant forms of gingival fibromatosis to chromosomes 2 and 5, more specifically 2p21-p22 and 5q13-q22. Genetic
HGF1 - Caused by a mutation in the SOS1 gene localized on chromosome 2p21-p22HGF2 - Caused by a mutation in the SOS1 gene localized on chromosome 5q13-q22Mutations in the RE1-silencing transcription factor (REST) gene can also cause this syndrome. Non genetic
HGF may also be caused by unwanted side effects of pharmacological agents like phenytoin, ciclosporin, and some calcium-channel blockers, meaning HGF is a disease that can be drug-induced. However, there is little next to no research done in this area to support the claim.