Critics, on the other hand, believe that the rule does not uphold the best interests of the donors and that the rule does not effectively promote organ donation. Signs
Signs of death or strong indications that a warm-blooded animal is no longer alive are:

Respiratory arrest (no breathing)
Cardiac arrest (no pulse)
Brain death (no neuronal activity)The stages that follow after death are:

Pallor mortis, paleness which happens in 15–120 minutes after death
Algor mortis, the reduction in body temperature following death. This is generally a steady decline until matching ambient temperature
Rigor mortis, the limbs of the corpse become stiff (Latin rigor) and difficult to move or manipulate
Livor mortis, a settling of the blood in the lower (dependent) portion of the body
Putrefaction, the beginning signs of decomposition
Decomposition, the reduction into simpler forms of matter, accompanied by a strong, unpleasant odor. Skeletonization, the end of decomposition, where all soft tissues have decomposed, leaving only the skeleton. Fossilization, the natural preservation of the skeletal remains formed over a very long period

Legal
The death of a person has legal consequences that may vary between different jurisdictions. A death certificate is issued in most jurisdictions, either by a doctor, or by an administrative office upon presentation of a doctors declaration of death. Misdiagnosed
There are many anecdotal references to people being declared dead by physicians and then "coming back to life", sometimes days later in their own coffin, or when embalming procedures are about to begin.
Season – More common in summer. Hence, the name Spring catarrh is a misnomer. Recently it is being labelled as Warm weather conjunctivitis. Climate – More prevalent in the tropics. VKC cases are mostly seen in hot months of summer, therefore, more suitable term for this condition is "summer catarrh" Ref. Pathology
Conjunctival epithelium undergoes hyperplasia and sends downward projection into sub-epithelial tissue. Adenoid layer shows marked cellular infiltration by eosinophils, lymphocytes, plasma cells and histiocytes. Fibrous layer show proliferation which later undergoes hyaline changes. Conjunctival vessels also show proliferation, increased permeability and vasodilation. Diagnosis
Classification
Based on severity, authors have classified VKC into clinical grades:Grade 0 - Absence of symptoms
Grade 1 MILD - Symptoms but no corneal involvement
Grade 2 MODERATE - Symptoms with photophobia but no corneal involvement
Grade 3 SEVERE - Symptoms, photophobia, mild to moderate SPKs OR with Diffuse SPK or corneal ulcer

Treatment
Local therapy- Topical steroids are effective. Commonly used solutions are of fluorometholone, medrysone, betamethasone or dexamethasone. Mast cell stabilizers such as sodium cromoglycate (2%) drops 4–5 times a day are quite effective in controlling VKC, especially atopic ones. Azelastine eyedrops are also effective. Topical antihistamines can be used. Acetyl cysteine (0.5%) used topically has mucolytic properties and is useful in the treatment of early plaque formation. Topical Cyclosporine is reserved for unresponsive cases. Systemic therapy- Oral antihistamines and oral steroids for severe cases. Treatment of large papillae- Cryo application, surgical excision or supratarsal application of long-acting steroids.