There may also be enlargement of the liver, heart failure, and a characteristic odor resembling that of sweaty feet. Some infants with glutaric acidemia type 2 have birth defects, including multiple fluid-filled growths in the kidneys (polycystic kidneys). Glutaric acidemia type 2 is a very rare disorder. Its precise incidence is unknown. It has been reported in several different ethnic groups. Treatment
It is important for patients with MADD to strictly avoid fasting to prevent hypoglycemia and crises of metabolic acidosis; for this reason, infants and small children should eat frequent meals. Patients with MADD can experience life-threatening metabolic crises precipitated by common childhood illnesses or other stresses on the body, so avoidance of such stresses is critical. Patients may be advised to follow a diet low in fat and protein and high in carbohydrates, particularly in severe cases. Depending on the subtype, riboflavin (100-400 mg/day), coenzyme Q10 (CoQ10), L-carnitine, or glycine supplements may be used to help restore energy production. Some small, uncontrolled studies have reported that racemic salts of beta-hydroxybutyrate were helpful in patients with moderately severe disease; further research is needed. See also
Glutaric acidemia type 1
Riboflavin-responsive exercise intolerance - similar in biochemical features, also responsive to riboflavin

References

This article incorporates public domain text from The U.S. National Library of Medicine


== External links ==
Visceroptosis also is known as Glénards disease (after French physician Frantz Glénard [1848–1920]). Glénards theory – the theory that abdominal ptosis is a nutritional disease with atrophy and prolapse of the intestine
Glénards test (also called girdle test) – while standing behind the patient, the examiner places his arms around the patient, so that his hands meet in front of the patients abdomen; he squeezes, raising the viscera, and then allows them to fall suddenly; Glénards theory suggests that if the patient feels relieved by the raising pressure and experiences distress on the release, the condition is probably one of splanchnoptosis
Stillers theory – the theory that gastroptosis is due to universal asthenia characterized by weakness and laxity of the viscera

References
External links

This article incorporates text from a publication now in the public domain: Gilman, D. C.; Peck, H. T.; Colby, F. M., eds. (1905). New International Encyclopedia (1st ed.). New York: Dodd, Mead. {{cite encyclopedia}}: Missing or empty |title= (help)