Also, it may be associated with hyponatremia (hypervolemic hyponatremia). Causes
Excessive sodium and/or fluid intake:

IV therapy containing sodium
As a transfusion reaction to a rapid blood transfusion. High intake of sodiumSodium and water retention:

Heart failure
Liver cirrhosis
Nephrotic syndrome
Corticosteroid therapy
Hyperaldosteronism
Low protein intakeFluid shift into the intravascular space:

Fluid remobilization after burn treatment
Administration of hypertonic fluids, e.g. mannitol or hypertonic saline solution
Administration of plasma proteins, such as albumin

Treatment
Treatment includes diuretics, particularly loop diuretics. See also
Volume status
Volume overload
Fluid balance
Edema
Anasarca (swelling of skin)
Pleural effusion (excess fluid in the pleural cavity)
Diuretics
Aquapheresis
Low pressure receptor zones
Apsia

References


== External links ==
Aminoaciduria occurs when the urine contains abnormally high amounts of amino acids. In the healthy kidney, the glomeruli filter all amino acids out of the blood, and the renal tubules then reabsorb over 95% of the filtered amino acids back into the blood.In overflow aminoaciduria, abnormally high concentrations of amino acids in the blood plasma overwhelm the resorptive capacity of the renal tubules, resulting in high concentrations of amino acids in the urine. This may be caused by congenital disorders of amino acid metabolism, for example, phenylketonuria, or may be secondary to liver disease.In renal aminoaciduria, the renal tubules are unable to reabsorb the filtered amino acids back into the blood, causing high concentrations of amino acids in the urine. This may be caused by a defect in the transport proteins in the renal tubule, for example, as occurs in Hartnup disease, or may be due to damage to the kidney tubule, for example, as occurs in Fanconi syndrome. References


== External links ==