This is because the images the brain receives from each eye are always blurred. A child with severe hyperopia can never see objects in detail. If the brain never learns to see objects in detail, then there is a high chance of one eye becoming dominant. The result is that the brain will block the impulses of the non-dominant eye. In contrast, the child with myopia can see objects close to the eye in detail and does learn at an early age to see objects in detail. Classification
Hyperopia is typically classified according to clinical appearance, its severity, or how it relates to the eyes accommodative status. Clinical classification
There are three clinical categories of hyperopia. Simple hyperopia: Occurs naturally due to biological diversity. Pathological hyperopia: Caused by disease, trauma, or abnormal development. Functional hyperopia: Caused by paralysis that interferes eyes ability to accommodate. Classification according to severity
There are also three categories severity:
Low: Refractive error less than or equal to +2.00 diopters (D). Moderate: Refractive error greater than +2.00 D up to +5.00 D.
High: Refractive error greater than +5.00 D.

Components of hypermetropia
Accommodation has significant role in hyperopia. Considering accommodative status, hyperopia can be classified as:
Total hypermetropia: It is the total amount of hyperopia which is obtained after complete relaxation of accommodation using cycloplegics like atropine. Latent hyperopia: It is the amount of hyperopia normally corrected by ciliary tone (approximately 1 diopter). Manifest hyperopia: It is the amount of hyperopia not corrected by ciliary tone. Manifest hyperopia is further classified into two, facultative and absolute.
Refractive lens exchange (RLE): A variation of cataract surgery where the natural crystalline lens is replaced with an artificial intraocular lens; the difference is the existence of abnormal ocular anatomy which causes a high refractive error. Phakic IOL: Phakic intraocular lens are lenses that implanted inside eye without removing the normal crystalline lens. Phakic IOLs can be used to correct hypermetropia up to +20 diopters.Non laser procedures

Conductive keratoplasty (CK): Conductive keratoplasty is a non laser refractive procedure used to correct presbyopia and low hypermetropia (+0.75D to +3.25D) with or without astigmatism (up to 0.75D). It uses radiofrequency energy to heat and shrink corneal collagen tissue. CK is contraindicated in pregnant/breastfeeding women, central corneal dystrophies and scarring, history of herpetic keratitis, type 1 diabetes etc. Automated lamellar keratoplasty (ALK): Hyperopic automated lamellar keratoplasty (H-ALK) and  Homoplastic ALK are ALK procedures that corrects low to moderate hyperopia. Poor predictability and the risk of complications limits usefulness of these procedures. Keratophakia and epi-keratophakia are another two non laser surgical procedures used to correct hypermetropia. Keratophakia is a surgical technique developed by Barraquer for treating high hypermetropia and aphakia. Poor predictability and induced irregular astigmatism are complications of these procedures. Etymology
The term hyperopia comes from Greek ὑπέρ hyper "over" and ὤψ ōps "sight" (GEN ὠπός ōpos). References


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