Ramachandran was able to show that patients with TLE showed enhanced emotional responses to the religious words, diminished responses to the sexually charged words, and normal responses to the neutral words. This study was presented as an abstract at a neuroscience conference and referenced in Ramachandrans book, Phantoms in the Brain, but it has never been published in the peer-reviewed scientific press.A study in 2015, reported that intrinsic religiosity and religiosity outside of organized religion were higher in patients with epilepsy than in controls. Lower education level, abnormal background EEG activity, and hippocampal sclerosis have been found to be contributing factors for religiosity in TLE.TLE has been suggested as a materialistic explanation for the revelatory experiences of prominent religious figures such as Abraham, Moses, Jesus, Mohammed, Saint Paul, Joan of Arc, Saint Teresa of Ávila, and Joseph Smith. These experiences are described (in possibly unreliable accounts) as complex interactions with their visions; but possibly (and dependent on the reliability of historical accounts, often made by acolytes) lack the stereotypy, amnestic periods, and automatisms or generalized motor events, which are characteristic of TLE. Psychiatric conditions with psychotic spectrum symptoms might be more plausible physical explanation of these experiences. It has been suggested that Pope Pius IXs doctrine of the immaculate conception was influenced by his forensically diagnosed partial epilepsy.In 2016, a case history found that a male temporal lobe epileptic patient experienced a vision of God following a temporal lobe seizure, while undergoing EEG monitoring.
An idioventricular rhythm is a cardiac rhythm characterized by a rate of <50 beats per minute (bpm), absence of P waves and widening of the QRS complex. In cases where the heart rate is  between 50 and 110 bpm, it is known as accelerated idioventricular rhythm and ventricular tachycardia if the rate exceeds 120 bpm. Causes of idioventricular rhythms are varied and can include drugs or a heart defect at birth. It is typically benign and not life-threatening. Etiology
Various etiologies may contribute to the formation of an idioventricular rhythm, and include:
Heart block
Reperfusion after myocardial infarction
Electrolyte abnormalities
Heart diseases present at birth
Certain drugs (eg. digoxin, β-agonists, anaesthetics)

Pathophysiology
The physiological pacemaker of the heart is the sinoatrial node. If the sinoatrial node is rendered dysfunctional, the AV node may act as the pacemaker. If both of these fail, the ventricles begin to act as the dominant pacemaker in the heart. The ventricles acting as their own pacemaker gives rise to an idioventricular rhythm. Diagnosis
An ECG trace is required for diagnosis. Treatment
As this rhythm is not life-threatening, treatment has limited value for the patient. If underlying pathologies are identified, they should be treated appropriately. Antidysrhythmics may be utilised if the patient suffers from dysrhythmias. == References ==