Symptoms are found around the body parts that sit in the hot tub - the legs, hips, chest, buttocks, and surrounding areas. Symptoms are amplified around regions that were covered by wet clothing, such as bathing suits. Sycosis vulgaris, sycosis barbae, or barbers itch is a staphylococcal infection of the hair follicles in the bearded area of the face, usually the upper lip. Shaving aggravates the condition. Gram-negative folliculitis may appear after prolonged acne treatment with antibiotics. Fungal
Tinea barbae is similar to barbers itch, but the infection is caused by the fungus T. rubrum. Malassezia folliculitis, formerly known as Pityrosporum folliculitis, is caused by yeasts (part of the fungus kingdom) of the genus Malassezia

Mites
Demodex folliculitis is usually caused by an overgrowth of Demodex folliculorum a mite that lives in human hair follicles. Although most people with D. folliculorurm have no symptoms, the mite can reproduce excessively, particularly in people with oily scalps. Viral
Herpetic folliculitis is rarer, but may occur when herpes simplex virus infection spreads to nearby hair follicles appearing in groups or clusters, mostly around the mouth. Noninfectious
Pseudofolliculitis barbae is a disorder occurring when hair curves back into the skin and causes inflammation. Eosinophilic folliculitis may appear in persons with impaired immune systems. Folliculitis decalvans or tufted folliculitis usually affects the scalp. Several hairs arise from the same hair follicle. Scarring and permanent hair loss may follow. The cause is unknown. Folliculitis keloidalis scarring on the nape of the neck is most common among males with curly hair.
Resolution within 7–10 days is the norm. Individuals with persistent (chronic) erythema multiforme will often have a lesion form at an injury site, e.g. a minor scratch or abrasion, within a week. Irritation or even pressure from clothing will cause the erythema sore to continue to expand along its margins for weeks or months, long after the original sore at the center heals. Causes
Many suspected etiologic factors have been reported to cause EM. Infections: bacterial (including Bacillus Calmette–Guérin (BCG) vaccination, haemolytic Streptococci, legionellosis, leprosy, Neisseria meningitidis, Mycobacterium, Pneumococcus, Salmonella species, Staphylococcus species, Mycoplasma pneumoniae), chlamydial. Fungal (Coccidioides immitis)
Parasitic (Trichomonas species, Toxoplasma gondii), 
Viral (especially Herpes simplex)
Drug reactions, most commonly to: antibiotics (including, sulphonamides, penicillin), anticonvulsants (phenytoin, barbiturates), aspirin, modafinil, antituberculoids, and allopurinol and many others. Physical factors: radiotherapy, cold, sunlight
Others: collagen diseases, vasculitides, non-Hodgkin lymphoma, leukaemia, multiple myeloma, myeloid metaplasia, polycythemiaEM minor is regarded as being triggered by HSV in almost all cases. A herpetic etiology also accounts for 55% of cases of EM major. Among the other infections, Mycoplasma infection appears to be a common cause. Herpes simplex virus suppression and even prophylaxis (with acyclovir) has been shown to prevent recurrent erythema multiforme eruption. Treatment
Erythema multiforme is frequently self-limiting and requires no treatment. The appropriateness of glucocorticoid therapy can be uncertain, because it is difficult to determine if the course will be a resolving one. See also
Erythema multiforme major
Erythema multiforme minor
Toxic epidermal necrolysis
Stevens–Johnson syndrome

References


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