The Government of Alberta is currently sampling and testing fish in 6 different watersheds (Peace River, Athabasca, North Saskatchewan, Red Deer, Bow and Oldman) to see where the disease has spread. Initial sample fish were collected in 2016, and are currently being processed by the Government of Alberta and CFIA labs. Since testing began, it has been detected in the Upper Bow River, and in May 2017 it was confirmed that whirling disease had also been detected in the Oldman River Basin. The declaration does not mean that every susceptible finfish population within the Bow and Oldman River watersheds are infected with the disease.As a result of the new declaration, a domestic movement permit will be required from the CFIA for susceptible species and end uses identified in the Domestic Movement Control Program, the vector Tubifex tubifex, the disease causing agent Myxobolus cerebralis, and/or related things out of the infected and buffer areas of Alberta. Recreational and sport fishing, including fishing led by a professional guide, will not require a CFIA permit. Prevention and control
Some biologists have attempted to disarm triactinomyxon spores by making them fire prematurely. In the laboratory, only extreme acidity or basicity, moderate to high concentrations of salts, or electric current caused premature filament discharge; neurochemicals, cnidarian chemosensitizers, and trout mucus were ineffective, as were anesthetized or dead fish.
They are released into the environment when the fish decomposes or is eaten. Some recent research indicates some fish may expel viable myxospores while still alive.Myxospores are extremely tough: "it was shown that Myxobolus cerebralis spores can tolerate freezing at −20°C for at least 3 months, aging in mud at 13°C for at least 5 months, and passage through the guts of northern pike Esox lucius or mallards Anas platyrhynchos without loss of infectivity" to worms. Triactinomyxons are much shorter-lived, surviving 34 days or less, depending on temperature. Pathology
M. cerebralis infections have been reported from a wide range of salmonid species: eight species of "Atlantic" salmonids, Salmo; four species of "Pacific" salmonids, Oncorhynchus; four species of char, Salvelinus; the grayling, Thymallus thymallus; and the huchen, Hucho hucho. M. cerebralis causes damage to its fish hosts through attachment of triactinomyxon spores and the migrations of various stages through tissues and along nerves, as well as by digesting cartilage. The fishs tail may darken, but aside from lesions on cartilage, internal organs generally appear healthy. Other symptoms include skeletal deformities and "whirling" behavior (tail-chasing) in young fish, which was thought to have been caused by a loss of equilibrium, but is actually caused by damage to the spinal cord and lower brain stem. Experiments have shown that fish can kill Myxobolus in their skin (possibly using antibodies), but that the fish do not attack the parasites once they have migrated to the central nervous system.