At this point, because they are learning language and other motor activities, their brain take in an overload of information and often will backtrack in language development as they try to pair linguistics to sound and syntax. Prevalence and prognosis
Preschool children usually go through a period of dysfluency as they attempt to learn linguistic and speech skills. About 10% of these children will experience a speech or language delay that is serious enough for them to benefit from early referral and assessment by a speech language pathologist. Normal dysfluency begins during a childs intensive language years and dissipates as the child continues to grow and develop. These dysfluencies are considered a normal phase of language development.Additionally, 85% of children, before preschool age will experience developmental dysfluency. These children wont require intervention because their dysfluency is a normal part of their development. 1 in 12 children, ages 3–17, will experience issues with their speech that is not considered within the normal realm of developmental dysfluency. However, only half of those individuals will receive intervention and speech therapy.Children who do not require speech therapy will often outgrow the period of dysfluency. Experts find that there is a distinction between childhood dysfluency that will likely correct itself and other disorders such as stuttering. Dysfluency disorders
The following disorders can be diagnosed following the years in which speech pattern disruptions could be the result of developmental dysfluency that is common within the age range of 2–6 years old.
Kindler syndrome (also known as "bullous acrokeratotic poikiloderma of Kindler and Weary",)  is a rare congenital disease of the skin caused by a mutation in the KIND1 gene. Symptoms and signs
Infants and young children with Kindler syndrome have a tendency to blister with minor trauma and are prone to sunburns. As individuals with Kindler syndrome age, they tend to have fewer problems with blistering and photosensitivity. However, pigment changes and thinning of the skin become more prominent. Kindler syndrome can affect various mucous tissues such as the mouth and eyes, which can lead to other health problems. Genetics
Kindler syndrome is an autosomal recessive genodermatosis. The KIND1 gene mutated in Kindler syndrome codes for the protein kindlin-1, which is thought to be active in the interactions between actin and the extracellular matrix (focal adhesion plaques). Kindler syndrome was first described in 1954 by Theresa Kindler. Diagnosis
Clinical and genetic tests are used to confirm diagnosis. Management
Treatment may involve several different types of practitioner to address the various manifestations that may occur. This multidisciplinary team  will also be involved in preventing secondary complications. See also
Rothmund–Thomson syndrome
Epidermolysis bullosa
List of cutaneous conditions

References


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