The infection is now endemic in wildlife and a few human cases have also been reported in areas where the parasite was not originally endemic, such as New Orleans and Egypt. The disease has also arrived in Brazil, where there were 34 confirmed cases from 2006 to 2014, including one death. The giant African land snail, which can be a vector of the parasite, has been introduced to Brazil as an invasive species and is spreading the disease. There may be more undiagnosed cases, as Brazilian physicians are not familiar with the eosinophilic meningitis associated to angiostrongyliasis and misdiagnose it as bacterial or viral.The parasite is rarely seen outside of endemic areas, and in these cases patients generally have a history of travel to an endemic area. See also
Schistosomiasis, a parasitic disease also spread by snails
List of unusual deaths – lists Australian man Sam Ballard as dying from Angiostrongyliasis in 2018, as a result of eating a garden slug eight years earlier on a dare

References
Further reading
"Parasites - Angiostrongyliasis (also known as Angiostrongylus Infection)". CDC. 2015-12-28. Retrieved 2017-04-04. "DPDx - Angiostrongyliasis". CDC. 2016-10-17. Retrieved 2017-04-04. Tabs for Parasite Biology, Image Gallery, Laboratory Diagnosis, and Treatment Information. == External links ==
Oculodentodigital syndrome (ODD syndrome) is an extremely rare genetic condition that typically results in small eyes, underdeveloped teeth, and syndactyly and malformation of  the fourth and fifth fingers. It is considered a kind of ectodermal dysplasia. Signs and symptoms
People with ODD syndrome often have a characteristic appearance. Visible features of the condition include:
small teeth that are prone to dental caries because of underdeveloped tooth enamel;
a long, thin nose;
unusually small eyes; and
type III syndactyly of the fourth and fifth fingers.Iris atrophy and glaucoma are more common than average. The size of the eyes often interferes with learning to read; special eyeglasses may be required. Hair may be fine, thin, dry, or fragile; in some families, it is curly.Neurologic abnormalities may be seen in adults. The neurologic changes may appear earlier in each subsequent generation and can include abnormal white matter, conductive deafness, and various kinds of paresis, including ataxia, spastic paraplegia, difficulty controlling the eyes, and bladder and bowel disturbances. Genetics
ODD is typically an autosomal dominant condition, but can be inherited as a recessive trait. It is generally believed to be caused by a mutation in the gene GJA1, which codes for the gap junction protein connexin 43. Slightly different mutations in this gene may explain the different way the condition manifests in different families. Most people inherit this condition from one of their parents, but new cases do arise through novel mutations.