The number one cause of this type of anemia, however, is iron deficiency.An insufficient intake of iron, Vitamin B12, and folic acid impairs the bone marrow function. The lack of iron within a person’s body can also stem from ulcer bacteria. These microbes live in the digestive tract and after many years cause ulcers in the lining of the stomach or small intestine. Therefore, a high percentage of patients with nutritional anemia may have a potential gastrointestinal disorder that causes chronic blood loss. This is common in immunocompromised, elderly, and diabetic people. High blood loss can also come from increased loss of blood during menstruation, childbirth, cancers of the intestines, and disorders that hinder the blood’s ability to coagulate. Medications can have adverse effects and cause nutritional anemia as well. Medications that stop the absorption of iron in the gut and cause bleeding from the gut (NSAIDs and Aspirin) can be culprits in the development of this condition. Hydrocortisones and valproic acid are also two drugs that can cause moderate bleeding from the gut. Amoxicillin and phenytoin have the ability to cause a vitamin B12 deficiency. Other common causes are thyroid disorders, lead toxicity, infectious diseases (e.g malaria), alcoholism, and vitamin E deficiency. Diagnosis
Complete blood count. Acute phase reactants
Serum iron studies 
Peripheral blood morphology

Types of nutritional anemia
Iron Deficiency Anemia
It is the last stage of iron deficiency and it is characterized by the production of small (microcytic) erythrocytes and diminished level of circulating hemoglobin. It represents the end point of a long period of iron deprivation.
If the opening pressure is increased, CSF may be removed for transient relief (see below). The CSF is examined for abnormal cells, infections, antibody levels, the glucose level, and protein levels. By definition, all of these are within their normal limits in IIH. Occasionally, the CSF pressure measurement may be normal despite very suggestive symptoms. This may be attributable to the fact that CSF pressure may fluctuate over the course of the normal day. If the suspicion of problems remains high, it may be necessary to perform more long-term monitoring of the ICP by a pressure catheter. Classification
The original criteria for IIH were described by Dandy in 1937. They were modified by Smith in 1985 to become the "modified Dandy criteria". Smith included the use of more advanced imaging: Dandy had required ventriculography, but Smith replaced this with computed tomography. In a 2001 paper, Digre and Corbett amended Dandys criteria further. They added the requirement that the person is awake and alert, as coma precludes adequate neurological assessment, and require exclusion of venous sinus thrombosis as an underlying cause. Furthermore, they added the requirement that no other cause for the raised ICP is found. In a 2002 review, Friedman and Jacobson propose an alternative set of criteria, derived from Smiths. These require the absence of symptoms that could not be explained by a diagnosis of IIH, but do not require the actual presence of any symptoms (such as headache) attributable to IIH.