In 1860, Bénédict Morel introduced the term dementia précoce and Langdon Down provided  in 1887 the most complete description to that date of the clinical manifestation that Charpentier described in 1890 as dementia précoce simple des enfant normaux.The description of simple schizophrenia is inter-changeable with describing symptoms as a form of dementia praecox known as simple dementing, at least in the time when the Swiss psychiatrists Otto Diem and Eugen Bleuler were studying it. In 1893, Emil Kraepelin considered there were four types of schizophrenia, and was amongst the first to identify three of them (dementia hebephrenica, dementia paranoides, dementia catatonica). The simplex type was added by Eugen Bleuler to the earlier ones identified by Kraepelin in 1899 and subsequently given a basic outline in 1903 by Otto Diem publishing a monograph on dementia praecox in the simple dementing form. This was based on a survey of two males having had a relatively normal childhood but who then fell into patterns of living tending towards vagrancy.A description of a cerebral disorder in relation to organic factors and in the context of general paralysis of the insane only; with no reference to schizophrenia, shows a disorder with features of generalized dementia (Lishman 1998). In 1951, a film was made showing the clinical characteristics of simple-type schizophrenia. Controversy
Definition of this type of schizophrenia is without unity or is controversial. The diagnosis was discontinued in the DSM system, although it was recommended for reinclusion in 1989.
The birthmark can vary in color from light pink to deep purple and is caused by an overabundance of capillaries around the ophthalmic branch of the trigeminal nerve, just under the surface of the face. There is also malformation of blood vessels in the pia mater overlying the brain on the same side of the head as the birthmark. This causes calcification of tissue and loss of nerve cells in the cerebral cortex.Neurological signs include seizures that begin in infancy and may worsen with age. Convulsions usually happen on the side of the body opposite the birthmark, and vary in severity. There may also be muscle weakness on the side of the body opposite the birthmark.Some children will have developmental delays and cognitive delays; about 50% will have glaucoma (optic neuropathy often associated with increased intraocular pressure), which can be present at birth or develop later. Glaucoma can be expressed as leukocoria, which should suggest further evaluation for retinoblastoma. Increased pressure within the eye can cause the eyeball to enlarge and bulge out of its socket (buphthalmos).Sturge–Weber syndrome rarely affects other body organs. Cause
The blood vessel formations associated with SWS start in the fetal stage. Around the sixth week of development, a network of nerves develops around the area that will become a babys head. Normally, this network goes away in the ninth week of development. In babies with SWS due to mutation of gene GNAQ, this network of nerves doesnt go away.