The test determines whether the mutated copy of SOD1 is present in the DNA sample submitted. It must be interpreted with caution by a veterinary clinician in combination with the animals clinical signs and other lab test results. The results reported are:

Normal / Normal (N/N, or clear): The dog does not have the mutation and is extremely unlikely to develop degenerative myelopathy. There have been cases, however, in which dogs that tested clear were found to have DM upon necropsy. Normal / Abnormal (N/A or carrier): The dog has one mutated copy of the gene (is heterozygous) and is a carrier but will not have degenerative myelopathy though there has now been some cases of heterozygous carriers developing DM. It will be possible for it to pass the mutation to offspring. A thorough examination of the dogs pedigree and DNA testing should be undertaken prior to breeding a dog with this result. Abnormal / Abnormal (A/A or At Risk): The dog has two copies (is homozygous) for the mutation and is at risk for degenerative myelopathy. Genetics
Breeding risks for degenerative myelopathy can be calculated using the Punnett Square:

If both parents are clear (N/N) then all of the puppies will be clear. If one parent is a carrier (N/A) and one is clear (N/N) each puppy has a 50% chance of being clear and a 50% chance of being a carrier.
taut, which is further from the Ancient Greek: τείνειν, romanized: teinein, lit. to stretch. Research
There is insufficient evidence that tetanus can be treated or prevented by vitamin C. This is at least partially due to the fact that historically trials conducted trying to look for a possible connection between vitamin C and helping tetanus patients were of poor quality. See also
Renshaw cell
Tetanized state
Tetanospasmin

References
External links

Tetanus Information from Medline Plus
Tetanus Surveillance -- United States, 1998-2000 (Data and Analysis)
"Tetanus". MedlinePlus. U.S. National Library of Medicine.