Lace bite is an irritation of the tibialis anterior and toe extensor tendons. The medical term for the condition is "tibialis anterior tendinopathy." This irritation, felt on the front of the foot or ankle, is often experienced by ice hockey players and figure skaters. It is caused by friction between the tendon and tongue of the ice skate.Common symptoms of lace bite include:
Pain, tenderness, and swelling where the front of the ankle meets the foot
Discomfort with ankle motion, especially when pulling the foot up or turning it inward
A cracking sound when the front of the foot or ankle is moved or touchedTreatment options include adjusting tongue positioning, additional padding, alternative lacing, and placing an orthoplast between the leather of the boot and its padding. == References ==
Amenorrhea is the absence of a menstrual period in a woman of reproductive age. Physiological states of amenorrhoea are seen, most commonly, during pregnancy and lactation (breastfeeding). Outside the reproductive years, there is absence of menses during childhood and after menopause.Amenorrhoea is a symptom with many potential causes. Primary amenorrhea is defined as an absence of secondary sexual characteristics by age 13 with no menarche or normal secondary sexual characteristics but no menarche by 15 years of age. It may be caused by developmental problems, such as the congenital absence of the uterus, failure of the ovary to receive or maintain egg cells, or delay in pubertal development. Secondary amenorrhoea, ceasing of menstrual cycles after menarche, is defined as the absence of menses for three months in a woman with previously normal menstruation, or six months for women with a history of oligomenorrhoea. It is often caused by hormonal disturbances from the hypothalamus and the pituitary gland, premature menopause, intrauterine scar formation, or eating disorders. Pathophysiology
Although amenorrhea has multiple potential causes, ultimately, it is the result of hormonal imbalance or an anatomical abnormality.Physiologically, menstruation is controlled by the release of gonadotropin-releasing hormone (GnRH) from the hypothalamus. GnRH acts on the pituitary to stimulate the release of follicle stimulating hormone (FSH) and luteinizing hormone (LH). FSH and LH then act on the ovaries to stimulate the production of estrogen and progesterone which, respectively, control the proliferative and secretary phases of the menstrual cycle.