Formulations
Fenofibrate is available in several formulations and is sold under several brand names, including:

Tricor by AbbVie
Lipofen by Kowa Pharmaceuticals America Inc
Lofibra by Teva
Lipanthyl, Lipidil, Lipantil micro and Supralip by Abbott Laboratories
Fenocor-67 by Ordain Health Care
Fibractiv 105/35 by Cogentrix Pharma (India)
Fenogal by SMB Laboratories
Antara by Oscient Pharmaceuticals
Tricheck by Zydus (CND)
Atorva TG by Zydus Medica
Golip by GolgiUSA
Stanlip by Ranbaxy (India)The formulations may differ in terms of pharmacokinetic properties, particularly bioavailability; some must be taken with meals, whereas others may be taken without regard to food.The choline salt of fenofibrate is available in the United States, sold as Trilipix, and may be taken without regard to meals. Environmental presence
Fenofibric acid was one of the 12 compounds identified in sludge samples taken from 12 wastewater treatment plants in California that were associated with estrogenic activity in in vitro. History
Fenofibrate was first synthesized in 1974, as a derivative of clofibrate, and was initially offered in France. It was initially known as procetofen, and was later renamed fenofibrate to comply with World Health Organization International Nonproprietary Name guidelines.Fenofibrate was developed by Groupe Fournier SA of France. In the United States, Tricor was reformulated in 2005. This reformulation was controversial, seen as an attempt to stifle competition from generic equivalents, and was the subject of antitrust litigation by Teva. References
External links
"Fenofibrate". Drug Information Portal. U.S. National Library of Medicine.
The defense mechanisms of the body present as cold and flu-like symptoms that occur in individuals who experience either acute or chronic reactions.The mold spores are inhaled and provoke the creation of IgE antibodies that circulate in the bloodstream, these types of immune response are most often initiated by exposure to thermophilic actinomycetes (most commonly Saccharopolyspora rectivirgula), which generate IgG-type antibodies. Following a subsequent exposure, IgG antibodies combined with the inhaled allergen to form immune complexes in the walls of the alveoli in the lungs. This causes fluid, protein, and cells to accumulate in the alveolar wall which slows blood-gas interchange and compromises the function of the lung. After multiple exposures, it takes less and less of the antigens to set off the reaction in the lung. Prevention
Farmers lung disease (FLD) is permanent and cannot be reversed, therefore in order to prevent the onset of further stages, farmers should inform their doctor of their occupation and if they have mold in their work environment. Prevention of this respiratory illness can be facilitated through the ventilation of work areas, drying of materials, and the use of a mask when working in confined areas with moldy hay or crops. Diagnosis
Diagnoses of Farmers lung is difficult due to its similarity to cold and flu-like symptoms. Doctors diagnose patients with Farmers lung under the following conditions:

A clinical history of symptoms such as cough, fever, and labored breathing when exposed to mold in work environment. The presence of diffuse lung disease in chronic cases.