Acneiform eruptions
Acneiform eruptions are caused by changes in the pilosebaceous unit.Acne aestivalis (Mallorca acne)
Acne conglobata
Acne cosmetica (cosmetic acne)
Acne fulminans (acute febrile ulcerative acne)
Acne keloidalis nuchae (acne keloidalis, dermatitis papillaris capillitii, folliculitis keloidalis, folliculitis keloidis nuchae, nuchal keloid acne)
Acne mechanica
Acne medicamentosa
Acne miliaris necrotica (acne varioliformis)
Acne vulgaris (acne simplex)
Acne with facial edema (solid facial edema)
Blepharophyma
Erythrotelangiectatic rosacea (erythematotelangiectatic rosacea, vascular rosacea)
Excoriated acne (acne excoriée des jeunes filles, Pickers acne)
Glandular rosacea
Gnathophyma
Gram-negative rosacea
Granulomatous facial dermatitis
Granulomatous perioral dermatitis
Halogen acne
Hidradenitis suppurativa (acne inversa, pyoderma fistulans significa, Verneuils disease)
Idiopathic facial aseptic granuloma
Infantile acne
Lupoid rosacea (granulomatous rosacea, micropapular tuberculid, rosacea-like tuberculid of Lewandowsky)
Lupus miliaris disseminatus faciei
Metophyma
Neonatal acne (acne infantum, acne neonatorum, neonatal cephalic pustulosis)
Occupational acne
Oil acne
Ocular rosacea (ophthalmic rosacea, ophthalmorosacea)
Otophyma
Periorificial dermatitis
Persistent edema of rosacea (chronic upper facial erythematous edema, Morbihans disease, rosaceous lymphedema)
Phymatous rosacea
Pomade acne
Papulopustular rosacea (inflammatory rosacea)
Perifolliculitis capitis abscedens et suffodiens (dissecting cellulitis of the scalp, dissecting folliculitis, perifolliculitis capitis abscedens et suffodiens of Hoffman)
Perioral dermatitis
Periorbital dermatitis (periocular dermatitis)
Pyoderma faciale (rosacea fulminans)
Rhinophyma
Rosacea (acne rosacea)
Rosacea conglobata
Synovitis–acne–pustulosis–hyperostosis–osteomyelitis syndrome (SAPHO syndrome)
Steroid rosacea
Tar acne
Tropical acne

Autoinflammatory syndromes
Autoinflammatory syndromes are a group of inherited disorders characterized by bouts of inflammatory skin lesions and periodic fevers. Blau syndrome  
Chronic infantile neurologic cutaneous and articular syndrome
Familial cold urticaria (familial cold autoinflammatory syndrome)
Familial Mediterranean fever
Hyper-IgD syndrome
Majeed syndrome
Muckle–Wells syndrome
TNF receptor associated periodic syndrome (familial Hibernian fever, TRAPS, tumor necrosis factor receptor associated periodic syndrome)

Chronic blistering
Chronic blistering cutaneous conditions have a prolonged course and present with vesicles and bullae.
Apart from the O-methyl group at the reducing end of the molecule, the identity and sequence of the five monomeric sugar units contained in fondaparinux is identical to a sequence of five monomeric sugar units that can be isolated after either chemical or enzymatic cleavage of the polymeric glycosaminoglycans heparin and heparan sulfate (HS). Within heparin and heparan sulfate this monomeric sequence is thought to form the high-affinity binding site for the anti-coagulant factor antithrombin (AT). Binding of heparin or HS to AT has been shown to increase the anti-coagulant activity of antithrombin 1000 fold. In contrast to heparin, fondaparinux does not inhibit thrombin. Chemistry
Abbreviations
GlcNS6S = 2-deoxy-6-O-sulfo-2-(sulfoamino)-α-D-glucopyranoside
GlcA = β-D-glucopyranuronoside
GlcNS3,6S = 2-deoxy-3,6-di-O-sulfo-2-(sulfoamino)-α-D-glucopyranosyl
IdoA2S = 2-O-sulfo-α-L-idopyranuronoside
GlcNS6SOMe = methyl-O-2-deoxy-6-O-sulfo-2-(sulfoamino)-α-D-glucopyranosideFondaparinux is only accessible by chemical synthesis. Recently, Supriya Dey et al. reported an effective and scalable one-pot synthesis of Fondaparinux.The sequence of monosaccharides is D-GlcNS6S-α-(1,4)-D-GlcA-β-(1,4)-D-GlcNS3,6S-α-(1,4)-L-IdoA2S-α-(1,4)-D-GlcNS6S-OMe, as shown in the following structure:

References
External links
"Fondaparinux". Drug Information Portal. U.S. National Library of Medicine.