Chronic fatigue
Orange discoloration of the skin, particularly the feet (Carotenosis)
Infertility
Halitosis (from vomiting or starvation-induced ketosis)
Hypotension or orthostatic hypotension
Having severe muscle tension, aches and pains
Insomnia
Abdominal distension

Cognitive symptoms
An obsession with counting calories and monitoring fat contents of food. Preoccupation with food, recipes, or cooking; may cook elaborate dinners for others, but not eat the food themselves or consume a very small portion. Admiration of thinner people. Thoughts of being fat or not thin enough
An altered mental representation of ones body
Impaired theory of mind, exacerbated by lower BMI and depression
Difficulty in abstract thinking and problem solving
Rigid and inflexible thinking
Poor self-esteem
Hypercriticism and perfectionism

Affective symptoms
Depression
Ashamed of oneself or ones body
Anxiety disorders
Rapid mood swings
Emotional dysregulation
Alexithymia

Behavioral symptoms
Food restrictions despite being underweight or at a healthy weight. Food rituals, such as cutting food into tiny pieces, refusing to eat around others, and hiding or discarding of food. Purging (only in the anorexia purging subtype) with laxatives, diet pills, ipecac syrup, or diuretics to flush food out of their system after eating or engage in self-induced vomiting. Excessive exercise, including micro-exercising, for example making small persistent movements of fingers or toes. Self harming or self-loathing. Solitude: may avoid friends and family and become more withdrawn and secretive. Perceptual symptoms
Perception of self as overweight, in contradiction to an underweight reality (namely "body image disturbance")
Intolerance to cold and frequent complaints of being cold; body temperature may lower (hypothermia) in an effort to conserve energy due to malnutrition. Altered body schema (i.e.
IGF-1 is necessary for bone formation, and decreased levels in anorexia nervosa contribute to a loss of bone density and potentially contribute to osteopenia or osteoporosis. Anorexia nervosa can also result in reduction of peak bone mass. Buildup of bone is greatest during adolescence, and if onset of anorexia nervosa occurs during this time and stalls puberty, low bone mass may be permanent.Hepatic steatosis, or fatty infiltration of the liver, can also occur, and is an indicator of malnutrition in children. Neurological disorders that may occur as complications include seizures and tremors. Wernicke encephalopathy, which results from vitamin B1 deficiency, has been reported in patients who are extremely malnourished; symptoms include confusion, problems with the muscles responsible for eye movements and abnormalities in walking gait. The most common gastrointestinal complications of anorexia nervosa are delayed stomach emptying and constipation, but also include elevated liver function tests, diarrhea, acute pancreatitis, heartburn, difficulty swallowing, and, rarely, superior mesenteric artery syndrome. Delayed stomach emptying, or gastroparesis, often develops following food restriction and weight loss; the most common symptom is bloating with gas and abdominal distension, and often occurs after eating. Other symptoms of gastroparesis include early satiety, fullness, nausea, and vomiting. The symptoms may inhibit efforts at eating and recovery, but can be managed by limiting high-fiber foods, using liquid nutritional supplements, or using metoclopramide to increase emptying of food from the stomach. Gastroparesis generally resolves when weight is regained. Cardiac complications
Anorexia nervosa increases the risk of sudden cardiac death, though the precise cause is unknown.