It reaches the cerebellum partly through the brachium conjunctivum (superior cerebellar peduncle) and partly through the restiform body (inferior cerebellar peduncle). Pathway for dorsal and spinocuneocerebellar tracts
The sensory neurons synapse in an area known as Clarkes nucleus or "Clarkes column". This is a column of relay neuron cell bodies within the medial gray matter within the spinal cord in layer VII (just beneath the dorsal horn), specifically between T1-L3. These neurons then send axons up the spinal cord, and project ipsilaterally to medial zones of the cerebellum through the inferior cerebellar peduncle. Below L3, relevant neurons pass into the fasciculus gracilis (usually associated with the dorsal column-medial lemniscal system) until L3 where they synapse with Clarkes nucleus (leading to considerable caudal enlargement). The neurons in the accessory cuneate nucleus have axons leading to the ipsilateral cerebellum via the inferior cerebellar peduncle. Pathway for ventral and rostral spinocerebellar tracts
Some neurons of the ventral spinocerebellar tract instead form synapses with neurons in layer VII of L4-S3. Most of these fibers cross over to the contralateral lateral funiculus via the anterior white commissure and through the superior cerebellar peduncle. The fibers then often cross over again within the cerebellum to end on the ipsilateral side. For this reason the tract is sometimes termed the "double-crosser." The Rostral Tract synapses at the dorsal horn lamina (intermediate gray zone) of the spinal cord and ascends ipsilaterally to the cerebellum through the inferior cerebellar peduncle

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References
Further reading
OSCARSSON, O.; UDDENBERG, N. (1 May 1965).
While genetic testing may be useful in the diagnosis of FEVR, a negative genetic test does not rule out the disease.Diagnosis

Diagnosis of FEVR is often made through direct visualization of the retina and fluorescein angiography, along with personal and family medical history. Hallmark characteristics of FEVR include lack of blood vessels in the peripheral retina. Other findings may include vessel and macular dragging, sub-retinal exudates, neovascularization, retinal folds, and retinal detachments. FEVR must be differentiated from other diseases involving incomplete vascularization of the retina including retinopathy of prematurity (ROP), Norrie disease, Coats disease, and others. Severity of disease is highly variable and can range from mild visual impairment to complete vision loss. Based on the severity of the disease, FEVR is diagnosed based on a clinical staging scale from 1 to 5. Since FEVR often runs in families, immediate relatives of someone diagnosed with FEVR should be examined by an ophthalmologist because the disease can have no symptoms before complications arise including retinal detachments.Treatment
Treatment is largely aimed at reducing the amount of new blood vessel growth and preventing complications that may arise as a result, including retinal tears and detachments. Using a laser, an ophthalmologist burns the portions of the retina that are not supported by blood vessels, a technique known as laser photocoagulation. By doing so, this tissue will no longer release molecules that stimulate blood vessel growth. If a retinal detachment occurs, laser therapy or surgery may be required to repair the retina.