Society and culture
Generic names
Testosterone cypionate is the generic name of the drug and its USP. The drug does not have an INN, USAN, or BAN. It has also been referred to as testosterone cipionate, as well as testosterone cyclopentylpropionate or testosterone cyclopentanepropionate. Brand names
Testosterone cypionate is or has been marketed under a variety of brand names, including:
Andro Cyp
Andronaq LA
Andronate
Dep Andro
Dep Test
Deposteron
Depostomead
Depotest
Depo-Testosterone
Depovirin
Durandro
Duratest
Jectatest
Malogen CYP
Pertestis
Testa-C
Testadiate Depo
Testex Elmu Prolongatum
Testoject LA
Virilon

Availability
Testosterone cypionate is marketed in the United States. It is not widely available outside of the United States, though it has been marketed in Canada, Australia, Spain, Brazil, and South Africa. Legal status
Testosterone cypionate, along with other AAS, is a schedule III controlled substance in the United States under the Controlled Substances Act and a schedule IV controlled substance in Canada under the Controlled Drugs and Substances Act. == References ==
Risk Factors
Infection with certain strains of Neisseria gonorrhoeae
Prolonged infection
Female sex (due to asymptomatic infection, and therefore prolonged infection)
Sexual promiscuity
Immune system deficiencies
Infection during menstruation, pregnancy, or in the puerperium period

Symptoms
Fever
Migratory arthralgias
Hemorrhagic pustules
Tenosynovitis
Rarely headache, neck stiffness, and visual changes (associated with meningitis)

Treatment
Treatment typically consists of cephalosporin and fluoroquinolone antibiotics. Gonococcemia is typically treated with intravenous or intramuscular cephalosporin antibiotics. Approximately 10-30% of gonorrheal infections present with a co-infection of chlamydia, so it is common to add a one-time dose of oral azithromycin or doxycycline for coverage of Chlamydia trachomatis. Bacterial resistance to antibiotics is increasingly common in Neisseria gonorrhoeae, so it is often advised to check susceptibility of the bacterial culture and then adjust the antibiotic therapy as needed. Pathogenesis
Neisseria gonorrhoeae is transmitted during sexual contact with an infected individual. The bacteria invade the non-ciliated columnar epithelium of the urogenital tract, oral mucosa, or anal mucosa following exposure. Invasion of the host cells is made possible due to virulence factors such as Pili, LOS, Opa, and others. Similarly, these virulence factors can be used for avoiding the host immune system, which may explain prolonged infection, bacterial resistance, and gonococcemia. See also
Primary gonococcal dermatitis
List of cutaneous conditions


== References ==