Angiolymphoid hyperplasia with eosinophilia (epithelioid hemangioma, histiocytoid hemangioma, inflammatory angiomatous nodule, inflammatory arteriovenous hemangioma, intravenous atypical vascular proliferation, papular angioplasia, pseudopyogenic granuloma)
Annular erythema of infancy
Arthropod assault
Eosinophilic cellulitis (Wells syndrome)
Eosinophilic fasciitis (Shulmans syndrome)
Eosinophilic granuloma
Eosinophilic granulomatosis with polyangiitis
Eosinophilic pustular folliculitis of infancy (eosinophilic pustular folliculitis in infancy, infantile eosinophilic pustular folliculitis, neonatal eosinophilic pustular folliculitis)
Eosinophilic ulcer of the oral mucosa (eosinophilic ulcer of the tongue, Riga–Fede disease, traumatic eosinophilic granuloma) 
Eosinophilic vasculitis
Erythema toxicum neonatorum (erythema toxicum, toxic erythema of the newborn)
Granuloma faciale
Hypereosinophilia
Hypereosinophilic syndrome
Incontinentia pigmenti (Bloch–Siemens syndrome, Bloch–Sulzberger disease, Bloch–Sulzberger syndrome)
Itchy red bump disease (papular dermatitis)
Juvenile xanthogranuloma
Kimuras disease
Nodules–eosinophilia–rheumatism–dermatitis–swelling syndrome
Pachydermatous eosinophilic dermatitis
Papular eruption of blacks
Papuloerythroderma of Ofuji
Pruritic papular eruption of HIV disease

Epidermal nevi, neoplasms, and cysts
Epidermal nevi, neoplasms, and cysts are skin lesions that develop from the epidermal layer of the skin.
Causes
The cause of Todds paresis has been attributed to the affected cortex being ‘exhausted’ or silenced due to increased inhibition, but these conjectures are not supported. It has been observed that the impairments that follow seizures are similar to those that follow strokes, where for a period of time blood flow to certain areas of the brain is restricted and these areas are starved of oxygen. Diagnosis
The most significant issue regarding the Todds paresis is its differentiation from a stroke. The issue is further complicated by the fact that some strokes trigger a focal seizure during the acute phase. A Todds paresis in this context may overestimate the extent of neurological deficit due to the vascular process itself resulting in erroneous decisions with regards to acute stroke therapy  such as thrombolysis. For this reason a seizure during an acute stroke is generally accepted to be a relative contraindication to thrombolytic therapy, especially in the absence of documented cerebrovascular occlusion using vascular imaging techniques.An infant with Todds paresis does not necessarily preclude the diagnosis of a febrile convulsion. This view is as a result of a recent study that showed the incidence of Todds paresis to be in 0.4% of infants that have been diagnosed with a febrile convulsion. Treatment
There is no treatment for Todds paralysis. Individuals must rest as comfortably as possible until the paralysis disappears. Prognosis
An occurrence of Todds paralysis indicates that a seizure has occurred. The prognosis for the patient depends upon the effects of the seizure, not the occurrence of the paralysis.