Calcium chloride can act as an irritant by desiccating moist skin. Solid calcium chloride dissolves exothermically, and burns can result in the mouth and esophagus if it is ingested. Ingestion of concentrated solutions or solid products may cause gastrointestinal irritation or ulceration.Consumption of calcium chloride can lead to hypercalcemia. Properties
Calcium chloride dissolves in water, producing chloride and the aquo complex [Ca(H2O)6]2+. In this way, these solutions are sources of "free" calcium and free chloride ions. This description is illustrated by the fact that these solutions react with phosphate sources to give a solid precipitate of calcium phosphate:

3 CaCl2 + 2 PO3−4 → Ca3(PO4)2 + 6 Cl−Calcium chloride has a very high enthalpy change of solution, indicated by considerable temperature rise accompanying dissolution of the anhydrous salt in water. This property is the basis for its largest-scale application. Molten calcium chloride can be electrolysed to give calcium metal and chlorine gas:

CaCl2 → Ca + Cl2

Preparation
In much of the world, calcium chloride is derived from limestone as a by-product of the Solvay process, which follows the net reaction below:
2 NaCl + CaCO3 → Na2CO3 + CaCl2North American consumption in 2002 was 1,529,000 tonnes (3.37 billion pounds).In the US, most of calcium chloride is obtained by purification from brine.As with most bulk commodity salt products, trace amounts of other cations from the alkali metals and alkaline earth metals (groups 1 and 2) and other anions from the halogens (group 17) typically occur, but the concentrations are trifling.
These might include:
Shortness of breath
Syncope (fainting)
Angina, but only in the presence of ischemic heart diseaseA person who has dilated cardiomyopathy may have an enlarged heart, with pulmonary edema and an elevated jugular venous pressure and a low pulse pressure. Signs of mitral and tricuspid regurgitation may be present. Causes
Although in many cases no cause is apparent, dilated cardiomyopathy is probably the result of damage to the myocardium produced by a variety of toxic, metabolic, or infectious agents. In many cases the cause remains unclear. It may be due to fibrous change of the myocardium from a previous myocardial infarction. Or, it may be the late sequelae of acute viral myocarditis, such as with Coxsackie B virus and other enteroviruses possibly mediated through an immunologic mechanism. Specific autoantibodies are detectable in some cases.Other causes include:

Chagas disease, due to Trypanosoma cruzi. This is the most common infectious cause of dilated cardiomyopathy in Latin America
Pregnancy: Dilated cardiomyopathy occurs late in gestation or several weeks to months postpartum as a peripartum cardiomyopathy. It is reversible in half of cases. Alcohol use disorder (alcoholic cardiomyopathy)
Non-alcoholic toxic insults include administration of certain chemotherapeutic agents, in particular doxorubicin (Adriamycin), and cobalt. Thyroid disease
Inflammatory diseases such as sarcoidosis and connective tissue diseases
Tachycardia-induced cardiomyopathy
Muscular dystrophy
Tuberculosis: 1 to 2% of TB cases. Autoimmune mechanisms
Thiamine deficiencyRecent studies have shown that those subjects with an extremely high occurrence (several thousands a day) of premature ventricular contractions (extrasystole) can develop dilated cardiomyopathy.