Belgian Shepherd Dog - Complete blindness by eight weeks old. Cone degeneration
Alaskan Malamute - Temporary loss of vision in daylight (hemeralopia) at eight to ten weeks old. There is a purely rod cell retina by four years old. Cone-rod dystrophy
Glen of Imaal Terrier - CRD3 results in gradual blindness with onset around 4 years of age (often detectable as retinal thinning as early as 3 years of age). Caused by a mutation in gene ADAM9, the disease is analogous to CRD9 in humans. A genetic test is now available from Optigen, LLC, that will identify whether a dog is affected, a carrier (heterozygous), or clear. Progressive rod-cone degeneration (PRCD)
This is a disease with normal rod and cone cell development but late onset degeneration of the rod cells that progresses to the cone cells. It is inherited as an autosomal recessive trait and has been linked to the ninth canine chromosome. Poodle - Night blindness by three to five years old, blind by five to seven years old. English Cocker Spaniel - Occurs late in life, usually at four to eight years old. American Cocker Spaniel - Night blindness by three to five years old, blind one to two years later. Labrador Retriever - Night blindness by four to six years old, blind at six to eight years old. Portuguese Water Dog
Chesapeake Bay Retriever
Australian Cattle Dog
American Eskimo Dog
Nova Scotia Duck Tolling Retriever

X-linked PRA
This condition is linked to the X chromosome. Siberian Husky - Night blindness by two to four years old.
Samoyed - More severe disease than the Husky. Dominant PRA
Bullmastiff - Inherited as an autosomal dominant trait due to a mutation in the gene for rhodopsin. Feline PRA
Abyssinian - Two forms exist. One is inherited as an autosomal dominant trait and has an early age onset. The other is inherited as an autosomal recessive trait and has a middle age onset. Early onset PRA has also been reported in the domestic shorthaired cat and Persian. The Siamese also likely has a hereditary form of PRA. Despite belief among breeders to the contrary, there is apparently no link between coat color in Persians and the development of PRA. Central progressive retinal atrophy (CPRA)
CPRA is also known as retinal pigment epithelial dystrophy (RPED). The cause of this condition is the loss of the retinal pigment epitheliums ability to effectively process the photoreceptor outer segment (POS) and subsequent accumulation of POS material in the RPE and loss of function. The loss of function of the RPE leads to photoreceptor degeneration. Vitamin E deficiency may play a role in the development of CPRA. It is characterized by accumulation of pigment spots in the retina surrounded by retinal atrophy and a mottled appearance of the pigmented nontapetal fundus. The pigmented spots eventually coalesce and fade as the atrophy of the retina increases. It is an inherited condition (in the Labrador Retriever it is inherited as an autosomal dominant trait with variable penetrance). CPRA occurs in older dogs. Peripheral vision is retained for a long time.