It is speculated that children will experience developmental linguistic delays at some point due to their muscle and motor plans not working as quickly as their brains. While they are learning speech, they are also developmentally mastering other skills (walking, potty training, motor skills, etc.) The origin of stuttering is not yet fully understood but parents/adults can mitigate the risk of developmental dysfluency by reducing the conversational demands on their child. Modeling slow, smooth speech and acknowledging the demanding and complex nature of learning language can help. Background
Speech is a complicated achievement that involves a series of cognitive, linguistic, sensorimotor and auditory processes that generate an in-depth understanding of language and speech. As children grow up their language and vocabulary grows with them. However, as this happens it is possible that the child might begin to demonstrate forms of disfluencies in their speech as they struggle to get words out when they are engaged in conversation or speaking in general. Preschool children usually go through a period of dysfluency as they attempt to learn linguistic and speech skills. About 10% of these children will experience a speech or language delay that is serious enough to benefit from early referral and assessment by a speech language pathologist. Normal disfluency begins during a childs intensive language learning years and resolves on its own as the child undergoes growth and development. This is considered the normal phase of language development.
Gastroenteritis. Gastritis. Chronic viral hepatitis. Hepatic schistosomiasis, or intestinal schistosomiasis. This is caused by the parasite Schistosoma mansoni. Iatrogenic injury (invasive procedure such as endoscopy or transesophageal echocardiography)
Atrio-oesophageal fistula. Yellow fever. Strongyloidiasis. Certain drugs. Plague, most commonly from septicemic plague. Mechanism
The source of vomited blood is usually from the upper gastrointestinal tract. This can include the esophagus, stomach, and parts of the small intestine. This may be the suspensory muscle of duodenum. Hematemesis tends to occur only after significant blood loss. Evaluation
Hematemesis may be investigated with endoscopy of the upper gastrointestinal tract. Barium meal may also be used. Management
Hematemesis is treated as a medical emergency. The most vital distinction is whether there is blood loss sufficient to cause shock. Correct management is required in such conditions. It is required to perform all tests such as endoscopy before medication. A platelet test is also an important test in such conditions. Medicines such as painkillers or antibiotics, e.g. ciprofloxacin, could decrease platelet count which can lead to thrombocytopenia (when the body does not have sufficient platelets in the blood and cannot form clots). In such conditions wrong medication or management could be deadly. Blood transfusion is required in such conditions if the body loses more than 20 percent of body blood volume. Severe loss makes it impossible for the heart to pump a sufficient amount of blood to the body. In such conditions unmaintained blood volume could lead to hypovolemic shock (hypovolemic shock could lead to damage of body organs e.g.