See also
Adelbert Ames, Jr. (Dartmouth Eye Institute, research in the 1930s and 1940s on aniseikonia)
Anisometropia
Macropsia
Micropsia

References
Further reading
Bannon, Robert E.; Neumueller, Julius; Boeder, Paul; Burian, Hermann M. (June 1970), "Aniseikonia and space perception: After 50 years", American Journal of Optometry & Archives of American Academy of Optometry, 47 (6): 423–441, doi:10.1097/00006324-197006000-00001, PMID 4912937, S2CID 37288776
Bisno, David C. (1994), Eyes in the Storm—President Hopkins Dilemma: The Dartmouth Eye Institute, Norwich, Vermont: Norwich Book Press, p. 288

External links
Aniseikonia Calculator, Chadwick Optical (U.S.A.) — a magnification calculator for iseikonic prescriptions or aniseikonia.
Metamorphopsia is a type of distorted vision in which a grid of straight lines appears wavy and parts of the grid may appear blank. People can first notice they suffer with the condition when looking at mini-blinds in their home. For example, straight lines might be wavy or bendy. Things may appear closer or further than they are. Initially characterized in the 1800s, metamorphopsia was described as one of the primary and most notable indications of myopic and senile maculopathies. Metamorphopsia can present itself as unbalanced vision, resulting from small unintentional movements of the eye as it tries to stabilize the field of vision. Metamorphopsia can also lead to the misrepresentation of an object’s size or shape.It is mainly associated with macular degeneration, particularly age-related macular degeneration with choroidal neovascularization. Other conditions that can present with complaints of metamorphopsia include: pathological myopia, presumed ocular histoplasmosis syndrome, choroidal rupture and multifocal choroiditis. Pathology
The mechanisms that result in the development of metamorphopsia involve structural changes in the retina of the eye (retinal mechanism) as well as processing changes in the cerebral cortex of the brain (cortical mechanism). The retinal mechanism involves the displacement of retinal layers which results in the mislocation of light on the retina. The cortical mechanism, which was discovered after the retinal mechanism, is affected by perceptual “filling-in” and visual crowding effects. The cortical mechanism was found to work in combination with the retinal mechanism to contribute to metamorphopsia in long-standing maculopathy or after the treatment of macular disorders.