A particularly dangerous form of slow earthquake is the tsunami earthquake, observed where the relatively low felt intensities, caused by the slow propagation speed of some great earthquakes, fail to alert the population of the neighboring coast, as in the 1896 Sanriku earthquake. Co-seismic overpressuring and effect of pore pressure
During an earthquake, high temperatures can develop at the fault plane so increasing pore pressure consequently to vaporization of the ground water already contained within rock. In the coseismic phase, such increase can significantly affect slip evolution and speed and, furthermore, in the post-seismic phase it can control the Aftershock sequence  because, after the main event, pore pressure increase slowly propagates into the surrounding fracture network. From the point of view of the Mohr-Coulomb strength theory, an increase in fluid pressure reduces the normal stress acting on the fault plane that holds it in place, and fluids can exert a lubricating effect. As thermal overpressurization may provide positive feedback between slip and strength fall at the fault plane, a common opinion is that it may enhance the faulting process instability. After the mainshock, the pressure gradient between the fault plane and the neighboring rock causes a fluid flow which increases pore pressure in the surrounding fracture networks; such increase may trigger new faulting processes by reactivating adjacent faults, giving rise to aftershocks. Analogously, artificial pore pressure increase, by fluid injection in Earths crust, may induce seismicity. Tidal forces
Tides may induce some seismicity.
Light chain deposition disease (LCDD) is a rare blood cell disease which is characterized by deposition of fragments of infection-fighting immunoglobulins,  called light chains (LCs), in the body. LCs are normally cleared by the kidneys, but in LCDD, these light chain deposits damage organs and cause disease. The kidneys are almost always affected and this often leads to kidney failure. About half of people with light chain deposition disease also have a plasma cell dyscrasia, a spectrum of diseases that includes multiple myeloma, Waldenströms macroglobulinemia, and the monoclonal gammopathy of undetermined significance premalignant stages of these two diseases. Unlike in AL amyloidosis, in which light chains are laid down in characteristic amyloid deposits, in LCDD, light chains are deposited in non-amyloid granules. Signs and symptoms
The kidney is the organ most frequently affected. Proteinuria, the presence of protein in the urine, is characteristic. More than 90% of people with LCDD develop kidney failure, often with rapid progression of disease.Light chains may be deposited in many other organs and may or may not result in any symptoms. Other than the kidneys, liver and heart are the most commonly involved organs. Deposition of light chains in the liver may lead to hepatomegaly, an enlarged liver, or rarely portal hypertension or liver failure. The heart is affected in up to 80% of patients with LCDD, and may cause arrhythmias and congestive heart failure. Cause
Diagnosis
A number of laboratory tests are required in order to assist in diagnosing LCDD.