On the other hand, Tropical Cyclone Heat Potential is one of such non-conventional subsurface oceanographic parameters influencing the cyclone intensity. Wind shear must be low; when wind shear is high, the convection and circulation in the cyclone will be disrupted. Usually, an anticyclone in the upper layers of the troposphere above the storm must be present as well—for extremely low surface pressures to develop, air must be rising very rapidly in the eyewall of the storm, and an upper-level anticyclone helps channel this air away from the cyclone efficiently. However, some cyclones such as Hurricane Epsilon have rapidly intensified despite relatively unfavorable conditions. Dissipation
There are a number of ways a tropical cyclone can weaken, dissipate, or lose its tropical characteristics. These include making landfall, moving over cooler water, encountering dry air, or interacting with other weather systems; however, once a system has dissipated or lost its tropical characteristics, its remnants could regenerate a tropical cyclone if environmental conditions become favorable.A tropical cyclone can dissipate when it moves over waters significantly cooler than 26.5 °C (79.7 °F). This will deprive the storm of such tropical characteristics as a warm core with thunderstorms near the center, so that it becomes a remnant low-pressure area. Remnant systems may persist for several days before losing their identity. This dissipation mechanism is most common in the eastern North Pacific.
Satoyoshi syndrome, also known as Komura-Guerri syndrome, is a rare progressive disorder of presumed autoimmune cause, characterized by painful muscle spasms, alopecia, diarrhea, endocrinopathy with amenorrhoea and secondary skeletal abnormalities. The syndrome was first reported in 1967 by Eijiro Satoyoshi and Kaneo Yamada in Tokyo, Japan. To this date, fewer than 50 cases worldwide have been reported for the Satoyoshi syndrome.People with the syndrome typically develop symptoms of the illness at a young age, usually between the age of six and fifteen years old. The initial symptoms are muscle spasms in the legs and alopecia, also known as baldness. The spasms are painful and progressive and their frequency varies from 1 or 2 to 100 per day, each lasting a few minutes. It can be sufficiently severe to produce abnormal posturing of the affected limbs, particularly the thumbs. With progression the illness involves the pectoral girdle and trunk muscles and finally the masseters and temporal muscles. The spasms usually spare the facial muscles. Severe spasms can interfere with respiration and speech. During an attack-free period, non-stimulus-sensitive myoclonus can occur in the arms, legs and neck. Diarrhea occurs in the first 2–3 years with intolerance to carbohydrate  and high glucose diets. Endocrinopathy manifests as amenorrhea and hypoplasia of the uterus. Affected children fail to attain height after 10–12 years of age.The syndrome is not known to be a primary cause of mortality, but some patients have died as a result of secondary complications, such as respiratory failure and malnourishment.