This type is called somatic or craniocervical tinnitus, since it is only head or neck movements that have an effect.There is a growing body of evidence suggesting that some tinnitus is a consequence of neuroplastic alterations in the central auditory pathway. These alterations are assumed to result from a disturbed sensory input, caused by hearing loss. Hearing loss could indeed cause a homeostatic response of neurons in the central auditory system, and therefore cause tinnitus. Hearing loss
The most common cause of tinnitus is hearing loss. Hearing loss may have many different causes, but among those with tinnitus, the major cause is cochlear injury.Ototoxic drugs also may cause subjective tinnitus, as they may cause hearing loss, or increase the damage done by exposure to loud noise. Those damages may occur even at doses that are not considered ototoxic. More than 260 medications have been reported to cause tinnitus as a side effect. In many cases, however, no underlying cause could be identified.Tinnitus can also occur due to the discontinuation of therapeutic doses of benzodiazepines. It can sometimes be a protracted symptom of benzodiazepine withdrawal and may persist for many months. Medications such as bupropion may also result in tinnitus. In many cases, however, no underlying cause can be identified. Associated factors
Factors associated with tinnitus include:
ear problems and hearing loss:
conductive hearing loss
acoustic shock
loud noise or music
middle ear effusion
otitis
otosclerosis
Eustachian tube dysfunction
sensorineural hearing loss
excessive or loud noise; e.g.
Retinal tear accounts for 11.4–44% of vitreous hemorrhage cases. Posterior vitreous detachment
As one gets older, pockets of fluid can develop in the vitreous. When these pockets develop near the back of the eye, the vitreous can pull away from the retina and possibly tear it. Posterior vitreous detachment accounts for 3.7–11.7% of vitreous hemorrhage cases. Other causes
Less common causes of vitreous hemorrhage make up 6.4–18% of cases, and include:

Proliferative sickle cell retinopathy
Macroaneurysms
Age-related macular degeneration
Terson syndrome
Retinal neovascularization as a result of branch or central retinal vein occlusion
Other

Diagnosis
Vitreous hemorrhage is diagnosed by identifying symptoms, examining the eye, and performing tests to identify the cause. Some common tests include:

Examination of the eye with a microscope
Pupil dilation and examination
An ultrasound examination may be used if the doctor does not have a clear view of the back of the eye
Blood tests to check for specific causes such as diabetes
A CT scan to check for injury around the eye
Referral to a retinal specialist

Complications
Ghost cell glaucoma: Secondary open angle glaucoma caused by degenerated red blood cells. Treatments
The treatment method used depends on the cause of the hemorrhage. In most cases, the patient is advised to rest with the head elevated 30–45°, and sometimes to put patches over the eyes to limit movement prior to treatment in order to allow the blood to settle. The patient is also advised to avoid taking medications that cause blood thinning (such as aspirin or similar medications). The goal of the treatment is to fix the cause of the hemorrhage as quickly as possible.