MRI scans can also confirm a diagnosis; abnormal low intensities can indicate iron accumulation in the brain. Prevention
Children of affected individuals are obligate carriers for aceruloplasminemia. If the CP mutations has been identified in a related individual, prenatal testing is recommended. Siblings of those affected by the disease are at a 25% of aceruloplasminemia. In asymptomatic siblings, serum concentrations of hemoglobin and hemoglobin A1c should be monitored.To prevent the progression of symptoms of the disease, annual glucose tolerance tests beginning in early teen years to evaluate the onset of diabetes mellitus. Those at risk should avoid taking iron supplements. Treatment
Treatment includes the use of iron chelating agents (such as desferrioxamine) to lower brain and liver iron stores, and to prevent progression of neurologic symptoms. This, combined with fresh-frozen human plasma (FFP) works effectively in decreasing liver iron content. Repetitive use of FFP can even improve neurologic symptoms. Antioxidants such as vitamin E can be used simultaneously to prevent tissue damage to the liver and pancreas. See also
Human iron metabolism
Iron overload disorder

References
External links

GeneReviews/NCBI/NIH/UW entry on Aceruloplasminemia
OMIM entries on Aceruloplasminemia
Acanthamoeba infection
Amebiasis cutis
Ant sting
Arachnidism
Bakers itch
Balamuthia infection
Bedbug infestation (bedbug bite, cimicosis)
Bee and wasp stings
Blister beetle dermatitis
Bombardier beetle burn
Bristleworm sting 
Centipede bite
Cheyletiella dermatitis
Chigger bite
Coolie itch
Copra itch
Coral dermatitis
Creeping eruption (cutaneous larva migrans)
Cutaneous leishmaniasis (Aleppo boil, Baghdad boil, bay sore, Biskra button, Chiclero ulcer, Delhi boil, Kandahar sore, Lahore sore, leishmaniasis tropica, oriental sore, pian bois, uta)
Cysticercosis cutis
Demodex folliculitis, usually caused by the Demodex folliculorum mite
Dogger Bank itch
Dracunculiasis (dracontiasis, guinea worm disease, Medina worm)
Echinococcosis (hydatid disease)
Elephantiasis tropica (elephantiasis arabum)
Elephant skin
Enterobiasis (oxyuriasis, pinworm infection, seatworm infection)
Erisipela de la costa
Feather pillow dermatitis
Funnel web spider bite
Gamasoidosis
Gnathostomiasis (larva migrans profundus)
Grain itch (barley itch, mattress itch, prairie itch, straw itch)
Grocers itch
Head lice infestation (cooties, pediculosis capitis)
Hookworm disease (ancylostomiasis, ground itch, necatoriasis, uncinariasis)
Human trypanosomiasis
Hydroid dermatitis
Irukandji syndrome
Jellyfish dermatitis
Ked itch
Larva currens
Latrodectism (widow spider bite)
Leech bite
Leopard skin
Lepidopterism (Caripito itch, caterpillar dermatitis, moth dermatitis)
Lizard skin
Loaiasis (Calabar swelling, fugitive swelling, loa loa, tropical swelling)
Loxoscelism (brown recluse spider bite, necrotic cutaneous loxoscelism)
Mal morando
Millipede burn
Mosquito bite
Mucocutaneous leishmaniasis (espundia, leishmaniasis americana)
Myiasis
Nairobi fly dermatitis (Kenya fly dermatitis, Nairobi eye)
Nematode dermatitis
Norwegian scabies (crusted scabies) 
Onchocerciasis
Ophthalmia nodosa
Paederus dermatitis
Pediculosis corporis (pediculosis vestimenti, Vagabonds disease)
Pediculosis pubis (crabs, phthirus pubis, phthirus pubis, pubic lice)
Pneumocystosis (often classified as fungal)
Portuguese man-of-war dermatitis
Post-kala-azar dermal leishmaniasis (post-kala-azar dermatosis)
Protothecosis
Pulicosis (flea bites)
Reduviid bite
Scabies (itch mite infestation, seven-year itch)
Scorpion sting
Sea anemone dermatitis
Seabathers eruption (sea lice)
Sea urchin injury
Seaweed dermatitis
Snake bite
Sowda
Sparganosis
Spider bite
Stingray injury
Swimmers itch (cercarial dermatitis, schistosome cercarial dermatitis) 
Tarantula bite
Tick bite
Toxoplasmosis
Trichinosis
Trichomoniasis
Tungiasis (bicho de pie, chigoe flea bite, jigger bite, nigua, pique)
Visceral leishmaniasis (dumdum fever, kala-azar)
Visceral schistosomiasis (bilharziasis)
Viscerotropic leishmaniasis
Wheat warehouse itch

Virus-related
Virus-related cutaneous conditions are caused by two main groups of viruses–DNA and RNA types–both of which are obligatory intracellular parasites.