Official or state terrorism – "referring to nations whose rule is based upon fear and oppression that reach similar to terrorism or such proportions". It may be referred to as Structural Terrorism defined broadly as terrorist acts carried out by governments in pursuit of political objectives, often as part of their foreign policy.Other sources have defined the typology of terrorism in different ways, for example, broadly classifying it into domestic terrorism and international terrorism, or using categories such as vigilante terrorism or insurgent terrorism. One way the typology of terrorism may be defined:
Political terrorism
Sub-state terrorism
Social revolutionary terrorism
Nationalist-separatist terrorism
Religious extremist terrorism
Religious fundamentalist Terrorism
New religions terrorism
Right-wing terrorism
Left-wing terrorism
Communist terrorism
State-sponsored terrorism
Regime or state terrorism
Criminal terrorism
Pathological terrorism

Causes and motivations
Choice of terrorism as a tactic
Individuals and groups choose terrorism as a tactic because it can:

Act as a form of asymmetric warfare in order to directly force a government to agree to demands
Intimidate a group of people into capitulating to the demands in order to avoid future injury
Get attention and thus political support for a cause
Directly inspire more people to the cause (such as revolutionary acts) – propaganda of the deed
Indirectly inspire more people to the cause by provoking a hostile response or over-reaction from enemies to the causeAttacks on "collaborators" are used to intimidate people from cooperating with the state in order to undermine state control.
Small vessel vasculitis
There are several vasculitides that affect small vessels. Pauci-immune
Granulomatosis with polyangiitis. Systemic vasculitis of medium and small arteries, including venules and arterioles. Produces granulomatous inflammation of the respiratory tracts and necrotizing, pauci-immune glomerulonephritis. Most common cause of saddle nose deformity in USA (nose flattened due to destruction of nasal septum by granulomatous inflammation). Almost all patients with granulomatosis with polyangiitis have c-antineutrophil cytoplasmic antibody, but not vice versa. Current treatment of choice is cyclophosphamide. At least two out of four criteria yields sensitivity and specificity of 88 and 92%. Nasal or oral inflammation (oral ulcers or purulent/bloody nasal discharge, may be painful)
Abnormal chest X-ray with showing nodules, infiltrates, or cavities
Microscopic hematuria or red blood cell casts
Vessel biopsy shows granulomatous inflammation
Peak incidence: ages 40–60, males > femalesEosinophilic granulomatosis with polyangiitis (formerly known as Churg–Strauss syndrome). Affects medium and small vessels with vascular and extravascular granulomatosis. Classically involves arteries of lungs and skin, but may be generalized. At least four criteria yields sensitivity and specificity of 85 and 99.7%. Asthma (history of wheezing or presently wheezing)
Eosinophilia > 10% on complete blood count
Mononeuropathy or polyneuropathy
Migratory or transient pulmonary opacities on chest x-ray
Paranasal sinus abnormalities
Vessel biopsy showing eosinophils in extravascular areasMicroscopic polyarteritis/polyangiitis. Affects capillaries, venules, or arterioles. Thought to be part of a group that includes granulomatosis with polyangiitis since both are associated with antineutrophil cytoplasmic antibody and similar extrapulmonary manifestations. Patients do not usually have symptomatic or histologic respiratory involvement. Immune complex
Hypersensitivity vasculitis (allergic vasculitis). Usually due to a hypersensitivity reaction to a known drug.