See also
Cutaneous T-cell lymphoma
Pagetoid reticulosis
Premycotic phase
Sézarys disease
Secondary cutaneous CD30+ large cell lymphoma
Angiocentric lymphoma
List of cutaneous conditions

References
Further reading


== External links ==
Neonatal encephalopathy (NE), previously known as neonatal hypoxic-ischemic encephalopathy (neonatal HIE or NHIE), is defined as a encephalopathy syndrome with signs and symptoms of abnormal neurological function, in the first few days of life in an infant born after 35 weeks of gestation. In this condition there is difficulty initiating and maintaining respirations, a subnormal level of consciousness, and associated depression of tone, reflexes, and possibly seizures. Encephalopathy is a nonspecific response of the brain to injury which may occur via multiple methods, but is commonly caused by birth asphyxia, leading to cerebral hypoxia. Signs and symptoms
In neonates born at or beyond 35 weeks, neonatal encephalopathy may present itself as the following symptoms:
Reduced level of consciousness
Seizures (which peak at 48 hours)
Difficulty initiating and maintaining respiration
Depression of tone and reflexes

Diagnosis
Cord blood gas analysis can be used to determine if there is perinatal hypoxia/asphyxia, which are potential causes of hypoxic-ischemic encephalopathy or cerebral palsy, and give insight into causes of intrapartum fetal distress. Cord blood gas analysis is indicated for high-risk pregnancies, in cases where C-sections occurred due to fetal compromise, if there were abnormal fetal heart rate patterns, Apgar scores of 3 or lower, intrapartum fever, or multifetal gestation.Evidence of brain injury related to the hypoxic-ischemic events that cause neonatal encephalopathy can be seen with brain MRIs, CTs, magnetic resonance spectroscopy imaging or ultrasounds. Neonatal encephalopathy may be assessed using Sarnat staging. Brain MRI is usually performed within eight days of life.