Leucomatous corneal opacity (leucoma simplex)
Leucomatous corneal opacity is a dense white opacity which results due to scarring of more than half of the stroma. A number of different presentations of leucomatous corneal opacity exist:

Adherent leucoma: results when healing occurs after perforation of cornea with incarceration of iris. The iris is adherent to the back of a leucomatous cornea. One of the major complication of adherent leucoma is Secondary glaucoma
Corneoiridic scar: if iris tissue is incarcerated and incorporated within the scar tissue, as occurs in healing of a large sloughed corneal ulcer, it is called a corneoiridic scar. Corneal facet: corneal surface depressed at the site of healing (due to less fibrous tissue); such a scar is called facet. Kerectasia: In this condition, corneal curvature is increased at the site of opacity (bulge due to weak scar). Presentation
Signs and symptoms include the following:

Loss of vision or blindness (when dense opacity covers the pupillary area)
Blurred vision (due to astigmatic effect and light scattering)
Glare

Complications
Congenital corneal opacity that affecting vision will cause amblyopia. That type of amblyopia is known as form-deprivation amblyopia (or amblyopia ex anopsia). Secondary changes in corneal opacity
Secondary changes may be seen in long-standing cases include: hyaline degeneration, calcareous degeneration, pigmentation and atheromatous ulceration. Causes
Congenital opacities may occur as developmental anomalies or following birth trauma. Causes of congenital corneal opacities include sclerocornea, trauma, ulcer, mucopolysaccharidosis, Peter’s anomaly, congenital hereditary endothelial dystrophy. Ocular trauma 
Corneal ulceration 
Xerophthalmia, caused by Vitamin A deficiency
Trachoma
Onchocerciasis
Mucous membrane pemphigoid: Ocular form of mucous membrane pemphigoid may cause corneal opacity and loss of vision.
Transient neonatal diabetes mellitus (TNDM) is a form of neonatal diabetes presenting at birth that is not permanent. This disease is considered to be a type of maturity onset diabetes of the young (MODY). Types
Cause
This condition has to do with genetics and is often associated with having an added Chromosome 7 gene (mostly from the paternal side).The form on chromosome 6 can involve imprinting. Diagnosis
Management
See also
Permanent neonatal diabetes mellitus

References
Further reading
GeneReview/NIH/UW entry on 6q24-Related Transient Neonatal Diabetes Mellitus


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