In contrast to aspirin, dietary salicylates are not acetylated and therefore do not block cyclooxygenases and hence, there is no rationale why a low salicylate diet would be beneficial for AERD patients.A diet low in omega-6 oils (precursors of arachidonic acid), and high in omega-3 oils may also be of benefit. In a small study, aspirin-sensitive asthma patients taking 10 grams of fish oil daily reported relief of most symptoms after six weeks, however, symptoms returned if the supplement was stopped. In another study, a diet low in omega 6 fatty acids and high in omega 3 fatty acids significantly reduced sinus symptoms in AERD patients. Alternate and related names
Aspirin-induced asthma
Leukotriene associated hypersensitivity 
Samters triad
Acetylsalicylic acid triad 
Widals triad
Francis triad
Aspirin triad
Aspirin-exacerbated respiratory disease (AERD). NSAID-exacerbated respiratory disease (NERD)
Aspirin-induced asthma and rhinitis (AIAR) A person who has not yet experienced asthma or aspirin sensitivity might be diagnosed as having:

Non-allergic rhinitis
Non-allergic rhinitis with eosinophilia syndrome (NARES)

History
The syndrome is named for Max Samter.Initial reports on a linkage among asthma, aspirin, and nasal polyposis were made by Widal in 1922. Further studies were conducted by Samter and Beers in reports published in 1968. See also
Salicylate sensitivity
Drug intolerance
Alcohol-induced respiratory reactions

References


== External links ==
Tricyclic antidepressants such as amitriptyline, or carbamazepine may help stabbing pains and have central and peripheral anticholinergic and sedative effects. These agents have central effects on pain transmission and block the active reuptake of norepinephrine and serotonin.Anticonvulsant drugs like gabapentin or pregabalin block the active reuptake of norepinephrine and serotonin and have properties that relieve neuropathic pain. However, these medications take a few weeks to become effective and are rarely used in the treatment of acute pain.Topical analgesics like capsaicin may also relieve minor aches and pains of muscles and joints. Prognosis
Alcoholic polyneuropathy is not life-threatening but may significantly affect ones quality of life. Effects of the disease range from mild discomfort to severe disability.It is difficult to assess the prognosis of a patient because it is hard to convince chronic alcoholics to abstain from drinking alcohol completely. It has been shown that a good prognosis may be given for mild neuropathy if the alcoholic has abstained from drinking for 3–5 years. Early stage
During the early stages of the disease the damage appears reversible when people take adequate amounts of vitamins, such as thiamine. If the polyneuropathy is mild, the individual normally experiences a significant improvement and symptoms may be eliminated within weeks to months after proper nutrition is established. When those people diagnosed with alcohol polyneuropathy experience a recovery, it is presumed to result from regeneration and collateral sprouting of the damaged axons. Progressed disease
As the disease progresses, the damage may become permanent.