To some extent, this "heritability" may not be specific – for example, studies have found that if a parent has any kind of anxiety disorder or clinical depression, then a child is somewhat more likely to develop an anxiety disorder or social phobia. Studies suggest that parents of those with social anxiety disorder tend to be more socially isolated themselves (Bruch and Heimberg, 1994; Caster et al., 1999), and shyness in adoptive parents is significantly correlated with shyness in adopted children (Daniels and Plomin, 1985). Growing up with overprotective and hypercritical parents has also been associated with social anxiety disorder. Adolescents who were rated as having an insecure (anxious-ambivalent) attachment with their mother as infants were twice as likely to develop anxiety disorders by late adolescence, including social phobia.A related line of research has investigated behavioural inhibition in infants – early signs of an inhibited and introspective or fearful nature. Studies have shown that around 10–15 percent of individuals show this early temperament, which appears to be partly due to genetics. Some continue to show this trait into adolescence and adulthood and appear to be more likely to develop a social anxiety disorder. Social experiences
A previous negative social experience can be a trigger to social phobia, perhaps particularly for individuals high in "interpersonal sensitivity".
Tethering the underlying ventricular wall is the most common for the posterior and septal leaflets, and sail-like anterior leaflets may be tethered to the RV free wall also. About 50% of individuals with Ebsteins anomaly have an associated shunt between the right and left atria, either an atrial septal defect or a patent foramen ovale. Electrophysiologic abnormalities
About 50% of individuals with Ebsteins anomaly have an accessory pathway with evidence of Wolff-Parkinson-White syndrome, secondary to the atrialized right ventricular tissue. This can lead to abnormal heart rhythms including atrioventricular re-entrant tachycardia. Other abnormalities that can be seen on the ECG include:

signs of right atrial enlargement or tall and broad Himalayan P waves
first degree atrioventricular block manifesting as a prolonged PR-interval
low amplitude QRS complexes in the right precordial leads
atypical right bundle branch block
T wave inversion in V1-V4 and Q waves in V1-V4 and II, III and aVF. Risk factors
An enlargement of the aorta may occur; an increased risk of abnormality is seen in babies of women taking lithium during the first trimester of pregnancy (though some have questioned this) and in those with Wolff-Parkinson-White syndrome. Diagnosis
Diagnosis is mainly depends on Echocardiography
Chest x will shows cardiomegaly

Treatment
Medication
Ebsteins cardio physiology typically presents as an (antidromic) AV reentrant tachycardia with associated pre-excitation. In this setting, the preferred medication treatment agent is procainamide. Since AV-blockade may promote conduction over the accessory pathway, drugs such as beta blockers, calcium channel blockers, and digoxin are contraindicated.