A cat named Frankenlouie was a famous example noted by the Guinness Book of World Records in 2012 for being the longest surviving Janus cat. Treatment
Few individuals survive until birth. For those who do, the only treatment available is to surgically remove the parasitic twin. Of the two documented attempts, however, one child died within hours and neither reached their second birthday. The problem with surgical intervention is that the arterial supplies of the head are so intertwined that it is very hard to control the bleeding, but it has been suggested that cutting off the parasitic twins arterial supply might improve the odds of the developed twins survival. Prevalence
Only ten cases of craniopagus parasiticus have been documented, out of a total of at least eighty known cases of craniopagus in general.Only four cases have been documented by modern medicine to have survived birth:

An earlier case was the so-called "Two-Headed Boy of Bengal", who was born in 1783 and died of a cobra bite in 1787. His skull remains in the collection of the Hunterian Museum of the Royal College of Surgeons of London. On December 10, 2003, Rebeca Martínez was born in the Dominican Republic with this rare condition. She was the first baby born with the condition to undergo an operation to remove the second head. She died on February 7, 2004, after an 11-hour operation. On March 30, 2004, Manar Maged was born with the rare condition. On February 19, 2005, 10-month-old Manar underwent a successful 13-hour surgery in Egypt.
PANDAS can present with chorea but more typically there are tics, stereotypies with a psychological component (e.g., OCD). Differential diagnosis
Other disorders that may be accompanied by chorea include benign hereditary chorea, bilateral striatal necrosis, abetalipoproteinemia, ataxia–telangiectasia, biotin-thiamine-responsive basal ganglia disease, Fahr disease, familial dyskinesia–facial myokymia (Bird–Raskind syndrome) due to an ADCY5 gene mutation, glutaric aciduria, Lesch–Nyhan syndrome, mitochondrial disorders, Huntingtons disease, Wilson disease, hyperthyroidism, lupus erythematosus, pregnancy (chorea gravidarum), drug intoxication and side effects of certain anticonvulsants (e.g. phenytoin) or psychotropic agents. Although some of these can similarly present in an acute way, there will typically be other neurological signs (such as ataxia or cognitive impairment), or other disease manifestations, or positive family history, which will help distinguish between them. Pathology
One of the important manifestations of acute rheumatic fever, Sydenhams chorea is similarly caused by an autoimmune response following infection by group A β-hemolytic streptococci.Two cross reactive streptococcal antigens have been identified, the M protein and N-acetyl-beta-D-glucosamine, whereby infection leads to autoantibodies being produced against host tissues (molecular mimicry) causing a variety of streptococcal related diseases including Sydenhams chorea but also rheumatic heart disease and nephritic syndrome. Autoantibodies against basal ganglia proteins have been found in Sydenhams chorea but these are non-specific. Dopamine receptor autoantibodies have been reported to correlate with clinical symptoms. Whether these antibodies represent an epi-phenomenon or are pathogenic, remains to be proven. Epidemiology
As with rheumatic fever, Sydenhams chorea is seen more often in less affluent communities, whether in the developing world or in aboriginal communities in the global North.