For the purposes of this article, then, a "fugue state" occurs while one is "acting out" a "dissociative fugue". The DSM-IV  defines "dissociative fugue" as:

sudden, unexpected travel away from home or ones customary place of work, with inability to recall ones past
confusion about personal identity, or the assumption of a new identity
significant distress or impairmentThe Merck Manual  defines "dissociative fugue" as:

One or more episodes of amnesia in which the inability to recall some or all of ones past and either the loss of ones identity or the formation of a new identity occur with sudden, unexpected, purposeful travel away from home.In support of this definition, the Merck Manual  further defines dissociative amnesia as:

An inability to recall important personal information, usually of a traumatic or stressful nature, that is too extensive to be explained by normal forgetfulness. Prognosis
The DSM-IV-TR states that the fugue may have a duration from days to months, and recovery is usually rapid. However, some cases may be refractory. An individual usually has only one episode. Cases
Shirley Ardell Mason (1923–1998), also known as "Sybil", would disappear and then reappear with no recollection of what happened during the time span. She recalled "being here and then not here" and having no identity of herself; it was claimed by her psychiatrist, Cornelia Wilbur, that she also had dissociative identity disorder. Wilburs diagnosis of DID was disputed by Wilburs contemporary Herbert Spiegel.
It is indicated for the treatment of inborn errors in primary bile-acid synthesis due to 3β-hydroxy-Δ5-C27-steroid oxidoreductase deficiency or Δ4-3-oxosteroid-5β-reductase deficiency in infants, children and adolescents aged one month to 18 years and adults.Cholic acid FGK (Kolbam) was approved for medical use in the European Union in November 2015. It is indicated for the treatment of inborn errors of primary bile acid synthesis, in infants from one month of age for continuous lifelong treatment through adulthood, encompassing the following single enzyme defects:
sterol 27-hydroxylase deficiency (presenting as cerebrotendinous xanthomatosis, CTX);
2- (or alpha-) methylacyl-CoA racemase (AMACR) deficiency;
cholesterol 7 alpha-hydroxylase (CYP7A1) deficiency.The most common side effects include peripheral neuropathy (nerve damage in the hands and feet), diarrhea, nausea (feeling sick), acid reflux (stomach acid flowing up into the mouth), esophagitis (inflammation of the food pipe), jaundice (yellowing of the skin and eyes), skin problems (lesions) and malaise (feeling unwell). Interactive pathway map
Click on genes, proteins and metabolites below to link to respective articles. References
External links
"Cholic acid". Drug Information Portal. U.S. National Library of Medicine.