Clinical signs and symptoms
According to a group case study, the most common symptoms in mild eosinophilic meningitis tend to be headache (with 100% of people in the study suffering from this symptom), photophobia or visual disturbance (92%), neck stiffness (83%), fatigue (83%), hyperesthesias (75%), vomiting (67%), and paresthesias (50%). Incubation period is often 3 weeks, but can be 3–36 days and even 80 days.Possible clinical signs and symptoms of mild and severe eosinophilic meningitis are:

Fever is often minor or absent, but the presence of high fever suggests severe disease. Headaches are progressive and severe, a bitemporal character in the frontal or occipital lobe. Meningismus - neck stiffness
Photophobia - sensitivity to light
Muscle weakness and fatigue
Nausea with or without vomiting
Paresthesias - tingling, prickling, or numbing of skin, may last for several weeks or months
Hyperesthesia - severe sensitivity to touch; may last for several weeks or months
Radiculitis - pain irradiated along certain areas of skin
Bladder dysfunction with urinary retention
Constipation
Brudzińskis sign
Vertigo
Blindness
Paralysis localized to one area; e.g. paralysis of extraocular muscles and facial palsy
General paralysis (flaccid) often ascending in nature starting with the feet and progressing upwards to involve the entire body
Coma
Death

Treatment
The severity and clinical course of Angiostrongylus disease depends significantly on the ingested load of third-stage larvae, creating great variability from case to case, making clinical trials difficult to design, and effectiveness of treatments difficult to discern. Typical conservative medical management including analgesics and sedatives provide minimal relief for the headaches and hyperesthesias.
Removing cerebrospinal fluid at regular 3- to 7-day intervals is the only proven method of significantly reducing intracranial pressure and can be used for symptomatic treatment of headaches. This process may be repeated until improvement is shown. There is growing evidence of moderate quality that suggests corticosteroid therapy using prednisolone or dexamethasone has beneficial effect in treating the CNS symptoms related to A. cantonensis infections. Although early research did not show treatment with antihelminthic agents (parasite-killing drugs) such as thiobendazole or albendazole effective in improving the clinical course of the illness, a number of recent studies from Thailand and China show that the combination of glucocorticoids and antihelminthics is safe and decreases the duration of headaches and the number of patients who had significant headache. Although the addition of antihelminthic agents for management of A. cantonensis infection has a theoretic risk of precipitating a neurologic crisis by releasing an overwhelming load of antigens through simultaneous death of the larvae, no study has shown this to exist in the clinical setting. Additionally, the failure to kill parasites before they attempt to migrate out of the CNS increases the risk of mechanical damage by migrating larvae. Although combination therapy using albendazole and prednisolone has no significant advantage compared to treatment using prednisolone alone in mild cases, the treatment with antihelminthics is demonstrably safe and may have significant benefit for patients with high parasite loads at risk for permanent disability or death.