Also in the 18th century, "Devonshire colic" was the name given to the symptoms experienced by people of Devon who drank cider made in presses that were lined with lead. Lead was added to cheap wine illegally in the 18th and early 19th centuries as a sweetener. The composer Beethoven, a heavy wine drinker, had elevated lead levels (as later detected in his hair) possibly due to this; the cause of his death is controversial, but lead poisoning is a contender as a factor.With the Industrial Revolution in the 19th century, lead poisoning became common in the work setting. The introduction of lead paint for residential use in the 19th century increased childhood exposure to lead; for millennia before this, most lead exposure had been occupational. William James Furnival (1853-1928), research ceramist of City & Guilds London Institute, appeared before Parliament in 1901 and presented a decades evidence to convince the nations leaders to remove lead completely from the British ceramic industry. His 852-page treatise, Leadless Decorative Tiles, Faience, and Mosaic of 1904 published that campaign and provided recipes to promote lead-free ceramics. At the request of the Illinois state government, Alice Hamilton (1869-1970) documented lead toxicity in Illinois industry and in 1911 presented results to the 23rd Annual Meeting of the American Economic Association. Dr. Hamilton was a founder of the field of occupational safety and health and published the first edition of her manual, Industrial Toxicology in 1934, yet in print in revised forms.
The following are some common early signs, though the presence of a listed symptom does not definitively mean that a child has spastic CP:Prior to 6 months

Legs crossing when a child is picked up, also called scissoring 
Stiffness
Head "lagging" when the child is picked up6–10 months

Fisting one hand
Difficulty rolling
Difficulty bringing hands togetherOlder than 12 months of age

Difficulty crawling
Difficulty standing even with support
Walking with an unsteady, uneven, or stiff gaitSpastic CP is distinguished from other forms of cerebral palsy by its prominent symptom of spasticity or stiff, tight movements and gait patterns such as the scissor gait. However, spasticity as a symptom is also seen in other conditions such as stroke and multiple sclerosis. Thus, the presence of spasticity alone does not warrant a conclusive diagnosis of spastic cerebral palsy. Changes in spasticity and corresponding postures may also occur with other brain activity, such as excitement, fear or anxiety, or even pain, which increase muscle tension.A person with spastic CP will commonly show, in addition to higher muscle tone, persistent primitive reflexes, greater stretch reflexes, plantar reflex, and ankle clonus.A third of people with cerebral palsy have seizures - this is most common in spastic CP. Audiovisual, cognitive compromise and behavioral disorders can occur. Cause
Spastic cerebral palsy is caused by malformation of or damage to the parts of the brain that control movement.