Acute cutaneous lupus erythematosus
Atrophoderma of Pasini and Pierini (dyschromic and atrophic variation of scleroderma, morphea plana atrophica, sclérodermie atrophique demblée)
Calcinosis–Raynaud phenomenon–esophageal dysmotility–sclerodactyly–telangiectasia syndrome (CREST syndrome)
Chilblain lupus erythematosus (chilblain lupus erythematosus of Hutchinson)
Childhood dermatomyositis
Childhood discoid lupus erythematosus
Childhood systemic lupus erythematosus
Complement deficiency syndromes
Dermatomyositis
Ehlers–Danlos syndrome
Eosinophilia–myalgia syndrome
Frontal linear scleroderma (en coup de sabre, morphea en coup de sabre) 
Generalized discoid lupus erythematosus
Generalized morphea
Interstitial granulomatous dermatitis
Juvenile rheumatoid arthritis (juvenile idiopathic arthritis, Stills disease)
Keloid morphea
Linear atrophoderma of Moulin (Moulin atrophoderma linearis)
Linear scleroderma
Localized discoid lupus erythematosus
Localized morphea 
Lupus erythematosus panniculitis (lupus erythematosus profundus, lupus panniculitis, lupus profundus, subcutaneous lupus erythematosus)
Lupus erythematosus–lichen planus overlap syndrome (lichen planus–lupus erythematosus overlap syndrome)
Methotrexate-induced papular eruption
Mixed connective tissue disease (Sharps syndrome, undifferentiated connective tissue disease)
Morphea profunda
Morphea–lichen sclerosus et atrophicus overlap
Mouth and genital ulcers with inflamed cartilage syndrome (MAGIC syndrome)
Neonatal lupus erythematosus
Nephrogenic systemic fibrosis (nephrogenic fibrosing dermopathy)
Nicolau–Balus syndrome
Nodulosis–arthropathy–osteolysis syndrome
Normophosphatemic familial tumoral calcinosis
Palisaded neutrophilic and granulomatous dermatitis
Pansclerotic morphea
Parry–Romberg syndrome (progressive hemifacial atrophy)
Progressive systemic sclerosis
Relapsing polychondritis (atrophic polychondritis, systemic chondromalacia)
Rheumatoid arthritis
Rheumatoid nodulosis (accelerated rheumatoid nodulosis)
Rheumatoid vasculitis
Rowells syndrome
Scleredema adultorum (Bushke disease, scleredema diabeticorum, scleredema adultorum of Buschke, scleredema of Buschke)
Silicosis
Sjögrens syndrome (Mikulicz disease, Sicca syndrome)
Subacute cutaneous lupus erythematosus
Systemic lupus erythematosus
Toxic oil syndrome
Tumid lupus erythematosus (lupus erythematosus tumidus)
Tuzun syndrome
Verrucous lupus erythematosus (hypertrophic lupus erythematosus)
Winchester syndrome

Abnormalities of dermal fibrous and elastic tissue
Abnormalities of dermal fibrous and elastic tissue are caused by problems in the regulation of collagen synthesis or degradation.
Eosinophilic pneumonia is a disease in which an eosinophil, a type of white blood cell, accumulates in the lungs. These cells cause disruption of the normal air spaces (alveoli) where oxygen is extracted from the atmosphere. Several different kinds of eosinophilic pneumonia exist and can occur in any age group. The most common symptoms include cough, fever, difficulty breathing, and sweating at night. Eosinophilic pneumonia is diagnosed by a combination of characteristic symptoms, findings on a physical examination by a health provider, and the results of blood tests and X-rays. Prognosis is excellent once most eosinophilic pneumonia is recognized and treatment with corticosteroids is begun. Classification
Eosinophilic pneumonia is divided into different categories depending upon whether its cause can be determined or not. Known causes include certain medications or environmental triggers, parasitic infections, and cancer. Eosinophilic pneumonia can also occur when the immune system attacks the lungs, a disease called eosinophilic granulomatosis with polyangiitis. When a cause cannot be found, the eosinophilic pneumonia is termed "idiopathic". Idiopathic eosinophilic pneumonia can also be divided into acute and chronic forms, depending on the symptoms a person is experiencing. Signs and symptoms
Most types of eosinophilic pneumonia have similar signs and symptoms. Prominent and nearly universal signs and symptoms include cough, fever, difficulty breathing, and night sweats. Acute eosinophilic pneumonia typically follows a rapid course. Fever and cough may develop only one or two weeks before breathing difficulties progress to the point of respiratory failure requiring mechanical ventilation. Chronic eosinophilic pneumonia usually follows a slower course.