In flooding low lands, elevation changes such as tidal fluctuations are significant determinants of coastal and estuarine flooding. Less predictable events like tsunamis and storm surges may also cause elevation changes in large bodies of water. Elevation of flowing water is controlled by the geometry of the flow channel and, especially, by depth of channel, speed of flow and amount of sediments in it  Flow channel restrictions like bridges and canyons tend to control water elevation above the restriction. The actual control point for any given reach of the drainage may change with changing water elevation, so a closer point may control for lower water levels until a more distant point controls at higher water levels. Effective flood channel geometry may be changed by growth of vegetation, accumulation of ice or debris, or construction of bridges, buildings, or levees within the flood channel. Coincidence
Extreme flood events often result from coincidence such as unusually intense, warm rainfall melting heavy snow pack, producing channel obstructions from floating ice, and releasing small impoundments like beaver dams. Coincident events may cause extensive flooding to be more frequent than anticipated from simplistic statistical prediction models considering only precipitation runoff flowing within unobstructed drainage channels. Debris modification of channel geometry is common when heavy flows move uprooted woody vegetation and flood-damaged structures and vehicles, including boats and railway equipment.
Atrioventricular reentrant tachycardia (AVRT), or atrioventricular reciprocating tachycardia, is a type of abnormal fast heart rhythm and is classified as a type of supraventricular tachycardia (SVT). AVRT is most commonly associated with Wolff–Parkinson–White syndrome, but is also seen in permanent junctional reentrant tachycardia (PJRT). In AVRT, an accessory pathway allows electrical signals from the hearts ventricles to enter the atria and cause earlier than normal contraction, which leads to repeated stimulation of the atrioventricular node. Signs and symptoms
An episode of SVT may present with palpitations, dizziness, shortness of breath, or losing consciousness (fainting). The electrocardiogram (ECG) would appear as a narrow-complex SVT. Between episodes of tachycardia the affected person is likely to be asymptomatic; however, the ECG would demonstrate the classic delta wave in Wolff–Parkinson–White syndrome. Pathophysiology
Two distinct pathways are involved: the normal atrioventricular conduction system, and an accessory pathway. During AVRT, the electrical signal passes in the normal manner from the AV node into the ventricles. Then, the electrical impulse pathologically passes back into the atria via the accessory pathway, causing atrial contraction, and returns to the AV node to complete the reentrant circuit (see figure). Once initiated, the cycle may continue causing the heart to beat faster than usual.Initiation of AVRT may be through a premature impulse of atrial, junctional, or ventricular origin. Treatment
Acute management is as for SVT in general. The aim is to interrupt the circuit. In the shocked patient, DC cardioversion may be necessary. In the absence of shock, inhibition at the AV node is attempted.