Hormone shifts such as those that precede menstruation and those during menopause
Life events such as fear, stress, anxiety, emotional or mental tension, work problems, financial stress, birth of a child, and bereavement
Gastrointestinal issues such as heartburn or constipation
Mental, neurobehavioral, or neurodevelopmental disorders such as bipolar disorder, clinical depression, generalized anxiety disorder, post traumatic stress disorder, schizophrenia, obsessive compulsive disorder, autism, dementia,: 326  ADHD, Asperger syndrome, and FASD
Disturbances of the circadian rhythm, such as shift work and jet lag, can cause an inability to sleep at some times of the day and excessive sleepiness at other times of the day. Chronic circadian rhythm disorders are characterized by similar symptoms. Certain neurological disorders such as brain lesions, or a history of traumatic brain injury
Medical conditions such as hyperthyroidism
Abuse of over-the-counter or prescription sleep aids (sedative or depressant drugs) can produce rebound insomnia
Poor sleep hygiene, e.g., noise or over-consumption of caffeine
A rare genetic condition can cause a prion-based, permanent and eventually fatal form of insomnia called fatal familial insomnia
Physical exercise: exercise-induced insomnia is common in athletes in the form of prolonged sleep onset latency
Increased exposure to the blue light from artificial sources, such as phones or computers
Chronic pain
Lower back pain
AsthmaSleep studies using polysomnography have suggested that people who have sleep disruption have elevated nighttime levels of circulating cortisol and adrenocorticotropic hormone. They also have an elevated metabolic rate, which does not occur in people who do not have insomnia but whose sleep is intentionally disrupted during a sleep study.
GSD I is typically treated with frequent small meals of carbohydrates and cornstarch, called modified cornstarch therapy, to prevent low blood sugar, while other treatments may include allopurinol and human granulocyte colony stimulating factor. Epidemiology
Overall, according to a study in British Columbia, approximately 2.3 children per 100,000 births (1 in 43,000) have some form of glycogen storage disease. In the United States, they are estimated to occur in 1 per 20,000–25,000 births. Dutch incidence rate is estimated to be 1 per 40,000 births. While a Mexican incidence showed 6.78:1000 male newborns. References


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