Urban mortality was also much higher than in rural regions. In 1829, the average urban and rural Dutchman was 164 cm (5 ft 4+1⁄2 in). By 1856, the average rural Dutchman was 162 cm (5 ft 4 in) and urban Dutchman was 158.5 cm (5 ft 2+1⁄2 in).A 2004 report citing a 2003 UNICEF study on the effects of malnutrition in North Korea, due to "successive famines," found young adult males to be significantly shorter. In contrast South Koreans "feasting on an increasingly Western-influenced diet," without famine, were growing taller. The height difference is minimal for Koreans over forty years old, who grew up at a time when economic conditions in the North were roughly comparable to those in the South, while height disparities are most acute for Koreans who grew up in the mid-1990s – a demographic in which South Koreans are about 12 cm (4.7 in) taller than their North Korean counterparts – as this was a period during which the North was affected by a harsh famine where hundreds of thousands, if not millions, died of hunger. A study by South Korean anthropologists of North Korean children who had defected to China found that eighteen-year-old males were 13 centimetres (5 in) shorter than South Koreans their age due to malnutrition.The tallest living man is Sultan Kösen of Turkey, at 251 cm (8 ft 3 in).
do not produce disease symptoms, although some people may experience a mild form of the disease with symptoms such as fever, headaches, nausea and vomiting and only a very small number of these cases go on to develop MVE. In fact, serological surveys which measure the level of anti-MVEV antibodies within the population estimate that only one in 800–1000 of all infections result in clinical disease.The incubation period following exposure to the virus is around 1 to 4 weeks. Following infection, a person has lifelong immunity to the virus. When a patient appears to show MVE symptoms and has been in an MVE-endemic area during the wet season, when outbreaks usually occur, MVE infection must be confirmed by laboratory diagnosis, usually by detection of a significant rise of MVE-specific antibodies in the patients serum. Of those who contract MVE, one-quarter die from the disease. Clone
The scientific study of the genetics of MVEV has been facilitated by the construction and manipulation of an infectious cDNA clone of the virus. Mutations in the envelope gene have been linked to the attenuation of disease in mouse models of infection. References


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