Treatment
The treatment and management of radial neuropathy can be achieved via the following methods:
Physical therapy or occupational therapy
Surgery (depending on the specific area and extent of damage)Tendon transfer (the origin remains the same but insertion is moved)Splinting

Prognosis
Radial neuropathy is not necessarily permanent, though there could be partial loss of movement or sensation. Complications include deformity of the hand in some individuals. If the injury is axonal (the underlying nerve fiber itself is damaged), recovery may take months or years and full recovery may never occur. EMG and nerve conduction studies are typically performed to diagnose the extent and distribution of the damage, and to help with prognosis for recovery. Culture and society
There are a number of terms used to describe radial nerve injuries, which are dependent on the causation factor such as:

Honeymoon palsy from another individual sleeping on and compressing ones arm overnight. Saturday night palsy from falling asleep with ones arm hanging over the arm rest of a chair, compressing the radial nerve. Squash palsy, from traction forces associated with the sport squash, happens to squash players during periods between matches. See also
Crutch paralysis
Peripheral neuropathy

References
Further reading
Cartwright, Michael S.; Yoon, Joon Shik; Lee, Kyu Ho; Deal, Nicole; Walker, Francis O. (1 April 2011). "Diagnostic Ultrasound for Traumatic Radial Neuropathy". American Journal of Physical Medicine & Rehabilitation. 90 (4): 342–343. doi:10.1097/PHM.0b013e3181e29daa. ISSN 0894-9115. PMC 2964388. PMID 20531154. Tuncel, Umut; Turan, Aydin; Kostakoglu, Naci (1 January 2011). "Acute closed radial nerve injury". Asian Journal of Neurosurgery. 6 (2): 106–109. doi:10.4103/1793-5482.92175. ISSN 1793-5482. PMC 3277063. PMID 22347334.
In some cases, there has been reported psychiatric and medical conditions in first degree relatives such as schizophrenia, psychosis, alcoholism, suicide, affective disorders, obsessive compulsive disorder, anxiety, paranoia, neurosis, sociopathy, and epilepsy. Sometimes more than one family member had a noteworthy condition.Neuroimaging has been used to investigate ORS. Hexamethylpropyleneamine oxime single-photon emission computed tomography (HMPAO SPECT) demonstrated hypoperfusion of the frontotemporal lobe in one case. That is to say, part of the brain was receiving insufficient blood flow. In another, functional magnetic resonance imaging was carried out while the person with ORS listened to both neutral words and emotive words. Compared to an age and sex matched healthy control subject under the same conditions, the individual with ORS showed more activation areas in the brain when listening to emotionally loaded words. This difference was described as abnormal, but less pronounced as would be observed in the brain of a person with a psychotic disorder. Diagnosis
Classification
Although the existence of ORS is generally accepted, there is some controversy as to whether it is a distinct condition or merely a part or manifestation of other psychiatric conditions, mainly due to the overlapping similarities. Similarly, there is controversy with regards how the disorder should be classified. As ORS has obsessive and compulsive features, some consider it as a type of obsessive–compulsive spectrum disorder, while others consider it an anxiety disorder due to the strong anxiety component.