In certain situations, the cyst is entirely removed, while with cysts with low recurrence risk, younger patients, or which are in anatomically eloquent areas of the pelvis, they can be drained. Features that may indicate the need for surgery include:
Persistent complex ovarian cysts
Persistent cysts that are causing symptoms
Complex ovarian cysts larger than 5 cm
Simple ovarian cysts larger 10 cm or larger than 5 cm in postmenopausal patients
Women who are menopausal or perimenopausal

Frequency
Most women of reproductive age develop small cysts each month, and large cysts that cause problems occur in about 8% of women before menopause. Ovarian cysts are present in about 16% of women after menopause and if present are more likely to be cancer.Benign ovarian cysts are common in asymptomatic premenarchal girls and found in approximately 68% of ovaries of girls 2–12 years old and in 84% of ovaries of girls 0–2 years old. Most of them are smaller than 9 mm while about 10–20% are larger macrocysts. While the smaller cysts mostly disappear within 6 months the larger ones appear to be more persistent. References
Further reading
McBee, W. C; Escobar, P. F; Falcone, T. (1 February 2007). "Which ovarian masses need intervention?". Cleveland Clinic Journal of Medicine. 74 (2): 149–157. doi:10.3949/ccjm.74.2.149. PMID 17333642. S2CID 45443236. Simcock, B; Anderson, N (February 2005). "Diagnosis and management of simple ovarian cysts: An audit". Australasian Radiology. 49 (1): 27–31. doi:10.1111/j.1440-1673.2005.01389.x. PMID 15727606. Ross, Elisa K.; Kebria, Medhi (August 2013). "Incidental ovarian cysts: When to reassure, when to reassess, when to refer". Cleveland Clinic Journal of Medicine. 80 (8): 503–514.
Aniridia is a heterozygous disorder, meaning that only one of the two chromosome 11 copies is affected. When both copies are altered (homozygous condition), the result is a uniformly fatal condition with near complete failure of entire eye formation. In 2001, two cases of  homozygous aniridia patients were reported; the fetuses died prior to birth and had severe brain damage. In mice, homozygous small eye defect (mouse Pax-6) leads to loss of the eyes and nose and the murine fetuses sustain severe brain damage. Types
Aniridia may be broadly divided  into hereditary and sporadic forms. Hereditary aniridia is usually transmitted in an autosomal dominant manner (each offspring has a 50% chance of being affected), although rare autosomal recessive forms (such as Gillespie syndrome) have also been reported. Sporadic aniridia mutations may affect the WT1 region adjacent to the AN2 aniridia region, causing a kidney cancer called nephroblastoma (Wilms tumor). These patients often also have genitourinary abnormalities and intellectual disability (WAGR syndrome). Several different mutations may affect the PAX6 gene. Some mutations appear to inhibit gene function more than others, with subsequent variability in the severity of the disease. Thus, some aniridic individuals are only missing a relatively small amount of iris, do not have foveal hypoplasia, and retain relatively normal vision. Presumably, the genetic defect in these individuals causes less "heterozygous insufficiency," meaning they retain enough gene function to yield a milder phenotype.