Prokinetic agents reduce the amount of acid available by stimulating movement in the digestive tract.Patients who are prone to laryngospasm during illness can take measures to prevent irritation such as antacids to avoid acid reflux.For acute context, making an upright position of the upper part of the body has been shown to shorten the spasm episodes. Fixation  of  the arms  on  stabilization  of  the  body  and  slowing  of  breathing is also recommended. See also
Laryngotracheal stenosis

References
External links

Larson, Philip C., "Laryngospam -The Best Treatment", Anesthesiology, 11 1998, Vol.89, 1293–1294
Abfraction is a theoretical concept explaining a loss of tooth structure not caused by tooth decay (non-carious cervical lesions). It is suggested that these lesions are caused by forces placed on the teeth during biting, eating, chewing and grinding; the enamel, especially at the cementoenamel junction (CEJ), undergoes large amounts of stress, causing micro fractures and tooth tissue loss. Abfraction appears to be a modern condition, with examples of non-carious cervical lesions in the archaeological record typically caused by other factors. Definition
Abfraction is a form of non-carious tooth tissue loss that occurs along the gingival margin. In other words, abfraction is a mechanical loss of tooth structure that is not caused by tooth decay, located along the gum line. There is theoretical evidence to support the concept of abfraction, but little experimental evidence exists.The term abfraction was first published in 1991 in a journal article dedicated to distinguishing the lesion. The article was titled "Abfractions: A New Classification of Hard Tissue Lesions of Teeth"  by John O. Grippo. This article introduced the definition of abfraction as a "pathologic loss of hard tissue tooth substance caused by bio mechanical loading forces". This article was the first to establish abfraction as a  new form of lesion, differing from abrasion, attrition, and erosion.Tooth tissue is gradually weakened causing tissue loss through fracture and chipping or successively worn away leaving a non-carious lesion on the tooth surface. These lesions occur in both the dentine and enamel of the tooth.