Creatine kinase test (checks the level of Creatine Kinase proteins in the blood. Creatine Kinase proteins are normally found inside of healthy muscle cells, but can be found in the blood when muscle cells are damaged.) Electromyography (shows that weakness is caused by destruction of muscle tissue rather than by damage to nerves.) Genetic testing (looks for deletion, duplication, or mutation of the dystrophin gene.) Treatment
There is no known cure for Becker muscular dystrophy yet. Treatment is aimed at control of symptoms to maximize the quality of life which can be measured by specific questionnaires. Activity is encouraged. Inactivity (such as bed rest) or sitting down for too long can worsen the muscle disease. Physical therapy may be helpful to maintain muscle strength. Orthopedic appliances such as braces and wheelchairs may improve mobility and self-care.Immunosuppressant steroids have been known to help slow the progression of Becker muscular dystrophy. The drug prednisone contributes to an increased production of the protein utrophin which closely resembles dystrophin, the protein that is defective in BMD.The cardiac problems that occur with EDMD and myotonic muscular dystrophy may require a pacemaker.The investigational drug Debio-025 is a known inhibitor of the protein cyclophilin D, which regulates the swelling of mitochondria in response to cellular injury. Researchers decided to test the drug in mice engineered to carry MD after earlier laboratory tests showed deleting a gene that encodes cycolphilin D reduced swelling and reversed or prevented the diseases muscle-damaging characteristics. According to a review by Bushby, et al.
Esophageal webs are seen associated in 50% of patients with this condition. Rarer forms of cervical esophageal diverticula are the Killians diverticulum and the Laimers diverticulum. Killians diverticulum is formed in the Killian-Jamiseon triangle (located inferior to the cricopharyngeus on both sides of this muscles insertion into the cricoid cartilage). Laimers diverticulum is formed in Laimers triangle (located inferior to the cricopharyngeus in the posterior midline above the confluence of the longitudinal layer of esophageal muscle). Laimers triangle is covered only by the circular layer of esophageal muscle. Diagnosis
A combination of the simple barium swallow and a  thorough endoscopy will normally confirm the diverticulum. Treatment
If small and asymptomatic, no treatment is necessary. Larger, symptomatic cases of Zenkers diverticulum have been traditionally treated by neck surgery to resect the diverticulum and incise the cricopharyngeus muscle. However, in recent times non-surgical endoscopic techniques have gained more importance (as they allow for much faster recovery), and the currently preferred treatment is endoscopic stapling (i.e. diverticulotomy with staples ). This may be performed through a diverticuloscope. Other methods include fibreoptic diverticular repair.Other non-surgical treatment modalities also exist, such as endoscopic laser, which recent evidence suggests is less effective than stapling. Epidemiology
Zenkers diverticulum mainly affects older adults. It has an incidence of 2 per 100,000 per year in the UK, but there is significant geographical variation around the world. References
External links

Zenkers diverticulum at eMedicine