Neurological disorders
Latent infection has been linked to Parkinsons disease and Alzheimers disease.Individuals with multiple sclerosis show infection rates around 15% lower than the general public. Traffic accidents
Latent T. gondii infection in humans has been associated with a higher risk of automobile accidents, potentially due to impaired psychomotor performance or enhanced risk-taking personality profiles. Climate change
Climate change has been reported to affect the occurrence, survival, distribution and transmission of T. gondii. T. gondii has been identified in the Canadian arctic, a location that was once too cold for its survival. Higher temperatures increase the survival time of T. gondii. More snowmelt and precipitation can increase the amount of T. gondii oocysts that are transported via river flow. Shifts in bird, rodent, and insect populations and migration patterns can impact the distribution of T. gondii due to their role as reservoir and vector. Urbanization and natural environmental degradation are also suggested to affect T. gondii transmission and increase risk of infection. See also
Toxoplasmic chorioretinitis
TORCH infection
Pyrimethamine

References
Parts of this article are taken from the public domain CDC factsheet: Toxoplasmosis

Bibliography
Weiss, L. M.; Kim, K. (28 April 2011). Toxoplasma gondii: The Model Apicomplexan. Perspectives and Methods. Academic Press. ISBN 978-0-08-047501-1. Retrieved 12 March 2013. Dubey, J. P. (2016). Toxoplasmosis of Animals and Humans (2nd ed.). Boca Raton: CRC Press. pp. xvii+313. ISBN 978-1-4200-9237-0. OCLC 423572366. ISBN 1-4200-9236-7 ISBN 9781420092363
Dubey JP, Lindsay DS, Speer CA (April 1998). "Structures of Toxoplasma gondii tachyzoites, bradyzoites, and sporozoites and biology and development of tissue cysts". Clinical Microbiology Reviews. 11 (2): 267–299. doi:10.1128/CMR.11.2.267. PMC 106833. PMID 9564564.
Toxoplasma gondii is considered the second leading cause of foodborne-related deaths and the fourth leading cause of foodborne-related hospitalizations in the United States.The protist responsible for toxoplasmosis is T. gondii. There are three major types of T. gondii responsible for the patterns of toxoplasmosis throughout the world. There are types I, II, and III. These three types of T. gondii have differing effects on certain hosts, mainly mice and humans due to their variation in genotypes. Type I: virulent in mice and humans, seen in people with AIDS. Type II: non-virulent in mice, virulent in humans (mostly Europe and North America), seen in people with AIDS. Type III: non-virulent in mice, virulent mainly in animals but seen to a lesser degree in humans as well.Current serotyping techniques can only separate type I or III from type II parasites.Because the parasite poses a particular threat to fetuses when it is contracted during pregnancy, much of the global epidemiological data regarding T. gondii comes from seropositivity tests in women of childbearing age. Seropositivity tests look for the presence of antibodies against T. gondii in blood, so while seropositivity guarantees one has been exposed to the parasite, it does not necessarily guarantee one is chronically infected. History
Toxoplasma gondii was first described in 1908 by Nicolle and Manceaux in Tunisia, and independently by Splendore in Brazil. Splendore reported the protozoan in a rabbit, while Nicolle and Manceaux identified it in a North African rodent, the gundi (Ctenodactylus gundi). In 1909 Nicolle and Manceaux differentiated the protozoan from Leishmania.