Neurological disorders
Latent infection has been linked to Parkinsons disease and Alzheimers disease.Individuals with multiple sclerosis show infection rates around 15% lower than the general public. Traffic accidents
Latent T. gondii infection in humans has been associated with a higher risk of automobile accidents, potentially due to impaired psychomotor performance or enhanced risk-taking personality profiles. Climate change
Climate change has been reported to affect the occurrence, survival, distribution and transmission of T. gondii. T. gondii has been identified in the Canadian arctic, a location that was once too cold for its survival. Higher temperatures increase the survival time of T. gondii. More snowmelt and precipitation can increase the amount of T. gondii oocysts that are transported via river flow. Shifts in bird, rodent, and insect populations and migration patterns can impact the distribution of T. gondii due to their role as reservoir and vector. Urbanization and natural environmental degradation are also suggested to affect T. gondii transmission and increase risk of infection. See also
Toxoplasmic chorioretinitis
TORCH infection
Pyrimethamine

References
Parts of this article are taken from the public domain CDC factsheet: Toxoplasmosis

Bibliography
Weiss, L. M.; Kim, K. (28 April 2011). Toxoplasma gondii: The Model Apicomplexan. Perspectives and Methods. Academic Press. ISBN 978-0-08-047501-1. Retrieved 12 March 2013. Dubey, J. P. (2016). Toxoplasmosis of Animals and Humans (2nd ed.). Boca Raton: CRC Press. pp. xvii+313. ISBN 978-1-4200-9237-0. OCLC 423572366. ISBN 1-4200-9236-7 ISBN 9781420092363
Dubey JP, Lindsay DS, Speer CA (April 1998). "Structures of Toxoplasma gondii tachyzoites, bradyzoites, and sporozoites and biology and development of tissue cysts". Clinical Microbiology Reviews. 11 (2): 267–299. doi:10.1128/CMR.11.2.267. PMC 106833. PMID 9564564.
Antibiotics – doxycycline and tetracycline
Quinidine
Bisphosphonates – used to treat osteoporosis
Steroids
Potassium chloride

Chemical injury by alkaline or acid solutions
Physical injury resulting from nasogastric tubes. Alcohol use disorder – Can wear down the lining of the esophagus. Crohns disease – a type of IBD and an autoimmune disease that can cause esophagitis if it attacks the esophagus. Stress – Can cause higher levels of acid reflux
Radiation therapy - Can affect the immune system. Allergies (food, inhalants) – Allergies can stimulate eosinophilic esophagitis. Infection - People with an immunodeficiencies have a higher chance of developing esophagitis. Vitamins and supplements (iron, vitamin C, and potassium) – Supplements and minerals can be hard on the GI tract. Vomiting – Acid can irritate esophagus. Hernias – A hernia can poke through the diaphragm muscle and can inhibit the stomach acid and food from draining quickly. Surgery
Eosinophilic esophagitis, a more chronic condition with a theorized autoimmune component

Mechanism
The esophagus is a muscular tube made of both voluntary and involuntary muscles. It is responsible for peristalsis of food. It is about 8 inches long and passes through the diaphragm before entering the stomach. The esophagus is made up of three layers:  from the inside out, they are the mucosa, submucosa, muscularis externa. The mucosa, the inner most layer and lining of the esophagus, is composed of stratified squamous epithelium, lamina propria, and muscularis mucosae. At the end of the esophagus is the lower esophageal sphincter, which normally prevents stomach acid from entering the esophagus.