Additional findings
Additional findings that may be present in HFGS according to the latest research are:
Limited metacarpophalangeal flexion of the thumb or limited ability to oppose the thumb and fifth finger
Hypoplastic thenar eminences
Medial deviation of the great toe (hallux varus), a useful diagnostic sign when present
Small great toenail
Fifth-finger clinodactyly, secondary to a shortened middle phalanx
Short feet
Altered dermatoglyphics of the hands; when present, primarily involving distal placement of the axial triradius, lack of thenar or hypothenar patterning, low arches on the thumbs, thin ulnar loops (deficiency of radial loops and whorls), and a greatly reduced ridge count on the fingersRadiographic findings 

Hypoplasia of the distal phalanx and first metacarpal of the thumbs and great toes
Pointed distal phalanges of the thumb
Lack of normal tufting of the distal phalanges of the great toes
Fusions of the cuneiform bones to other tarsal bones or trapezium-scaphoid fusion of the carpals
Short calcaneus
Occasional bony fusions of the middle and distal phalanges of the second, third, fourth, or fifth toes
Delayed carpal or tarsal maturation
Metacarpophalangeal profile reflecting shortening of the first metacarpal, the first and second phalanges, and the second phalanx of the second and fifth digitsUrogenital Defects
Females may have the following:

Vesicoureteral reflux secondary to ureteric incompetence
Ectopic ureteral orifices
Trigonal hypoplasia
Hypospadiac urethra
Subsymphyseal epispadias
Patulous urethra
Urinary incontinence (related to structural anomalies and weakness of the bladder sphincter muscle)
Small hymenal opening
Various degrees of incomplete Müllerian fusion with or without two cervices or a longitudinal vaginal septumMales may have the following:

Retrograde ejaculation (related to structural anomalies and weakness of the bladder sphincter muscle)

Treatment
References


== External links ==
The eponym Gee-Herter disease was sometimes used to acknowledge both contributions. Sidney V. Haas, an American paediatrician, reported positive effects of a diet of bananas in 1924. This diet remained in vogue until the actual cause of coeliac disease was determined.While a role for carbohydrates had been suspected, the link with wheat was not made until the 1940s by the Dutch paediatrician Dr Willem Karel Dicke. It is likely that clinical improvement of his patients during the Dutch famine of 1944 (during which flour was scarce) may have contributed to his discovery. Dicke noticed that the shortage of bread led to a significant drop in the death rate among children affected by coeliac disease from greater than 35% to essentially zero. He also reported that once wheat was again available after the conflict, the mortality rate soared to previous levels. The link with the gluten component of wheat was made in 1952 by a team from Birmingham, England. Villous atrophy was described by British physician John W. Paulley in 1954 on samples taken at surgery. This paved the way for biopsy samples taken by endoscopy.Throughout the 1960s, other features of coeliac disease were elucidated. Its hereditary character was recognised in 1965. In 1966, dermatitis herpetiformis was linked to gluten sensitivity. Social and culture
May has been designated as "Coeliac Awareness Month" by several coeliac organisations. Christian churches and the Eucharist
Speaking generally, the various denominations of Christians celebrate a Eucharist in which a wafer or small piece of sacramental bread from wheat bread is blessed and then eaten.