Pulmonary embolismAcute lung injury may also cause pulmonary edema directly through injury to the vasculature and parenchyma of the lung. It includes acute lung injury and acute respiratory distress syndrome. (ALI-ARDS) cover many of these causes, but they may also include:

Inhalation of hot or toxic gases
Pulmonary contusion, i.e., high-energy trauma (e.g. vehicle accidents)
Aspiration, e.g., gastric fluid
Reexpansion, i.e. post large volume thoracocentesis, resolution of pneumothorax, post decortication, removal of endobronchial obstruction, effectively a form of negative pressure pulmonary oedema. Reperfusion injury, i.e., postpulmonary thromboendartectomy or lung transplantation
Swimming induced pulmonary edema also known as immersion pulmonary edema
Transfusion Associated Circulatory Overload occurs when multiple blood transfusions or blood-products (plasma, platelets, etc.) are transfused over a short period of time. Transfusion associated Acute Lung Injury is a specific type of blood-product transfusion injury that occurs when the donors plasma contained antibodies against the recipient, such as anti-HLA or anti-neutrophil antibodies. Severe infection or inflammation which may be local or systemic. This is the classical form of acute lung injury-adult respiratory distress syndrome.Some causes of pulmonary edema are less well characterised and arguably represent specific instances of the broader classifications above. Arteriovenous malformation
Hantavirus pulmonary syndrome
High altitude pulmonary edema (HAPE)
Envenomation, such as with the venom of Atrax robustus

Signs and symptoms
The most common symptom of pulmonary edema is difficulty breathing (dyspnea), but may include other symptoms such as coughing up blood (classically seen as pink or red, frothy sputum), excessive sweating, anxiety, and pale skin.
Triphalangeal thumb (TPT) is a congenital malformation where the thumb has three phalanges instead of two. The extra phalangeal bone can vary in size from that of a small pebble to a size comparable to the phalanges in non-thumb digits. The true incidence of the condition is unknown, but is estimated at 1:25,000 live births. In about two-thirds of the patients with triphalangeal thumbs, there is a hereditary component. Besides the three phalanges, there can also be other malformations. It was first described by Columbi in 1559. Signs and symptoms
The triphalangeal thumb has a different appearance than normal thumbs. The appearance can differ widely; the thumb can be a longer thumb, it can be deviated in the radio-ulnar plane (clinodactyly), or thumb strength can be diminished. In the case of a five fingered-hand it has a finger-like appearance, with the position in the plane of the four fingers, thenar muscle deficiency, and additional length. There is often a combination with radial polydactyly. Complications
Generally, triphalangeal thumbs are non-opposable. In contrast to most people with opposable thumbs, a person suffering from TPT cannot easily place his or her thumb opposite the other four digits of the same hand. The opposable thumbs ability to effortlessly utilize fingers in a "pinch" formation is critical in precision gripping. For the thumb to adequately grip, certain thumb criteria must be met (e.g. suitable position and length, stable joints and good thenar muscle strength).