This gene mediates chloride and bicarbonate exchange and additionally transports sulfate and other anions at the apical membrane, part of the plasma membrane of enterocytes. It is also known that related phenotypes are  Increased shRNA abundance (Z-score > 2). Diagnosis
Tenonitis can be identified and diagnosed a few different ways. Tenons capsule may have inflammation from a disease called idiopathic orbital inflammation syndrome. There is no history of trauma or adjacent focus of infection with this disease. In order to diagnose tenonitis some type of imaging is needed, such as a CT or MRI. The imaging is used to capture the inflammation and to spot any possible chance of infection. For chronic diseases a biopsy may be needed to determine an underlying condition that may be causing the recurring inflammation. Treatment
The main course of treatment for this specific disease would be to surgically excise the capsule. There is no info that implies the inflammation of this capsule will go down over time so the known course of action would be to have it surgically removed in order to relieve discomfort and to prevent further irritation to this area. Prognosis
Although this may not be a lethal disease, it will progressively get worse over time. Depending on the severity of the disease, this may have months of progression. When going past 3 months of having issues, the main course of action would be to surgically relieve the pain or total excision of the affected area. Epidemiology
This disease has no specific population it targets more.
Thromboembolism (blood clots)
Embolism (foreign bodies in the circulation, e.g. amniotic fluid embolism)

Trauma
Traumatic injury to an extremity may produce partial or total occlusion of a vessel from compression, shearing, or laceration. Acute arterial occlusion may develop as a result of arterial dissection in the carotid artery or aorta or as a result of iatrogenic arterial injury (e.g., after angiography). Other
An inadequate flow of blood to a part of the body may be caused by any of the following:

Thoracic outlet syndrome (compression of the brachial plexus)
Atherosclerosis (lipid-laden plaques obstructing the lumen of arteries)
Hypoglycemia (lower than normal level of glucose)
Tachycardia (abnormally rapid beating of the heart)
Radiotherapy
Hypotension (low blood pressure, e.g. in septic shock, heart failure)
Outside compression of a blood vessel, e.g. by a tumor or in the case of superior mesenteric artery syndrome
Sickle cell disease (abnormally shaped red blood cells)
Induced g-forces which restrict the blood flow and force the blood to the extremities of the body, as in acrobatics and military flying
Localized extreme cold, such as by frostbite or improper cold compression therapy
Tourniquet application
An increased level of glutamate receptor stimulation 
Arteriovenous malformations and peripheral artery occlusive disease
rupture of significant blood vessels supplying a tissue or organ. Anemia vasoconstricts the periphery so that red blood cells can work internally on vital organs such as the heart, brain, etc., thus causing lack of oxygen to the periphery. Premature discontinuation of any oral anticoagulant.