Aluminosis (also known as aluminium lung) is a restrictive lung disease caused by exposure to aluminum-bearing dust. Aluminosis is a form of pneumoconiosis that can lead to pulmonary fibrosis. First cases of lung damage from aluminium exposure were reported in 1930s in Germany. It can be detected by using high-resolution computed tomography. Findings may vary, showing nodular or slightly irregular opacities that may merge into more prominent forms, most frequently in the upper lung fields, sometimes in the lower lung fields, and less frequently a diffuse micro nodular pattern. In severe cases, pulmonary fibrosis with honeycombing was described.Workers exposed to aluminium dust are often involved in industries such as explosives manufacturing (where aluminium powder is involved), aluminium welding (and grinding), and bauxite smelting. References
External links

https://icdlist.com/icd-10/J63.0
References
Further reading
Likoff, Jessup Mariell, Chandler Sheryl L., Kay Harold R. (1987). "Clinical determinants of mortality in chronic congestive heart failure secondary to idiopathic dilated or to ischemic cardiomyopathy". The American Journal of Cardiology. 59 (6): 634–638. doi:10.1016/0002-9149(87)91183-0. PMID 3825904. {{cite journal}}:  CS1 maint: multiple names: authors list (link)
Carmeliet Peter;  et al. (1999). "Impaired myocardial angiogenesis and ischemic cardiomyopathy in mice lacking the vascular endothelial growth factor isoforms VEGF164 and VEGF188". Nature Medicine. 5 (5): 495–502. doi:10.1038/8379. PMID 10229225. S2CID 7536458. Menasché Philippe;  et al. (2008). "The myoblast autologous grafting in ischemic cardiomyopathy (MAGIC) trial first randomized Placebo-controlled study of myoblast transplantation". Circulation. 117 (9): 1189–1200. doi:10.1161/circulationaha.107.734103. PMID 18285565. Beltrami Carlo Alberto;  et al. (1994). "Structural basis of end-stage failure in ischemic cardiomyopathy in humans". Circulation. 89 (1): 151–163. doi:10.1161/01.cir.89.1.151. PMID 8281642. == External links ==