Western Producer Prairie Books, Saskatoon: 1982. Goddard, Ives (1969). "Owls and Cannibals: Two Algonquian Etymologies". Paper Presented at the Second Algonquian Conference, St. Johns, Newfoundland. Johnston, Basil (1990) [1976]. Ojibway Heritage. Lincoln: University of Nebraska Press. Johnston, Basil (2001) [1995]. The Manitous. St. Paul: Minnesota Historical Society Press. Marano, Lou (1982). "Windigo Psychosis: The Anatomy of an Emic-Etic Confusion". Current Anthropology. 23: 385–412. doi:10.1086/202868. S2CID 147398948. Parker, Seymour (1960). "The Wiitiko Psychosis in the Context of Ojibwa Personality and Culture". American Anthropologist. 62 (4): 603–623. doi:10.1525/aa.1960.62.4.02a00050. Smallman, Shawn (2014). Dangerous Spirits: The Windigo in Myth and History. Victoria, BC: Heritage House Publishing Company. ISBN 9781772030334. Teicher, Morton I. (1961). "Windigo Psychosis: A Study of Relationship between Belief and Behaviour among the Indians of Northeastern Canada." In Proceedings of the 1960 Annual Spring Meeting of the American Ethnological Society, ed. Verne P. Ray. Seattle: University of Washington Press. External links

Seeing Wetiko: on Capitalism, Mind Viruses, and Antidotes for a World in Transition
"Windigo: The Flesh-Eating Monster of Native American Legend", Monstrum documentary short series from PBS Digital Studios
Osteochondromas are the most common benign tumors of the bones. The tumors take the form of cartilage-capped bony projections or outgrowth on the surface of bones exostoses. It is characterized as a type of overgrowth that can occur in any bone where cartilage forms bone. Tumors most commonly affect long bones about the knee and in the forearm. Additionally, flat bones such as the pelvis and scapula (shoulder blade) may be affected. Hereditary multiple exostoses usually present during childhood. Yet, the vast majority of affected individuals become clinically manifest by the time they reach adolescence. Osteochondromas occur in 3% of the general population and represent 35% of all benign tumors and 8% of all bone tumors. The majority of these tumors are solitary non-hereditary lesions and approximately 15% of osteochondromas occur as hereditary multiple exostoses preferably known as hereditary multiple osteochondromas (HMOs). Osteochondromas do not result from injury and the exact cause remains unknown. Recent research has indicated that multiple osteochondromas is an autosomal dominant inherited disease. Germ line mutations in EXT1 and EXT2 genes located on chromosomes 8 and 11 have been associated with the cause of the disease. The treatment choice for osteochondroma is surgical removal of solitary lesion or partial excision of the outgrowth, when symptoms cause motion limitations or nerve and blood vessel impingements. In hereditary multiple exostoses the indications of surgery are based upon multiple factors that are taken collectively, namely: patients age, tumor location and number, accompanying symptomatology, esthetic concerns, family history and underlying gene mutation.