Acquired generalized lipodystrophy (Lawrence syndrome, Lawrence–Seip syndrome)
Adiposis dolorosa (Dercums disease)
Alpha-1 antitrypsin deficiency panniculitis (alpha1-protease deficiency panniculitis, alpha1-proteinase deficiency panniculitis)
Atrophic connective tissue panniculitis
Barraquer–Simons syndrome (acquired partial lipodystrophy, cephalothoracic lipodystrophy, progressive lipodystrophy)
Benign symmetric lipomatosis (benign symmetric lipomatosis of Launois–Bensaude, Madelungs disease)
Centrifugal abdominal lipodystrophy (centrifugal lipodystrophy, lipodystrophia centrifugalis abdominalis infantalis)
Chronic erythema nodosum (erythema nodosum migrans, subacute migratory panniculitis of Vilanova and Piñol, subacute nodular migratory panniculitis) 
Cold panniculitis (popsicle panniculitis)
Congenital generalized lipodystrophy (Berardinelli–Seip syndrome)
Cytophagic histiocytic panniculitis
Drug-induced lipodystrophy
Factitial panniculitis
Familial partial lipodystrophy (Köbberling–Dunnigan syndrome)
Gouty panniculitis
Hemihyperplasia–multiple lipomatosis syndrome
HIV-associated lipodystrophy
Involutional lipoatrophy
Lipoatrophia annularis (Ferreira–Marques lipoatrophia)
Lipoatrophia semicircularis (semicircular lipoatrophy)
Lipodermatosclerosis (chronic panniculitis with lipomembranous changes, hypodermitis sclerodermiformis, sclerosing panniculitis, stasis panniculitis)
Lipohypertrophy
Localized lipodystrophy
Neutrophilic lobular panniculitis
Nodular vasculitis
Non-progressive late-onset linear hemifacial lipoatrophy
Pancreatic panniculitis (enzymatic panniculitis, pancreatic fat necrosis, subcutaneous fat necrosis)
Polands syndrome
Post-steroid panniculitis
Sclerema neonatorum
Sclerosing lipogranuloma (paraffinoma)
Septal panniculitis
Subcutaneous fat necrosis of the newborn
Traumatic panniculitis
Tumor lysis syndrome
Weber–Christian disease (relapsing febrile nonsuppurative panniculitis)

Congenital anomalies
Cutaneous congenital anomalies are a diverse group of disorders that result from faulty morphogenesis, the biological process that forms the shape of a human body.
Smith–Magenis Syndrome (SMS), also known as 17p- syndrome, is a microdeletion syndrome characterized by an abnormality in the short (p) arm of chromosome 17. It has features including intellectual disability, facial abnormalities, difficulty sleeping, and numerous behavioral problems such as self-harm. Smith–Magenis syndrome affects an estimated between 1 in 15,000 to 1 in 25,000 individuals. Signs and symptoms
Facial features of children with Smith–Magenis syndrome include a broad and square face, deep-set eyes, large cheeks, and a prominent jaw, as well as a flat nose bridge (in the young child; as the child ages it becomes more ski-jump shaped). Eyes tend to be deep-set, close together and upwards-slanted. Eyebrows are heavy with lateral extension. The mouth is the most noticeable feature; both upper and lower lips are full, and the mouth is wide. The mouth curves downwards and the upper lip curves outwards, due to a fleshy philtrum. These facial features become more noticeable as the individual ages, as Mandible growth outstrips that of the maxilla leading to a clear midface hypoplasia. There is also a mild brachycephaly.Disrupted sleep patterns are characteristic of Smith–Magenis syndrome, typically beginning early in life. Affected individuals may be very sleepy during the day, but have trouble falling asleep and awaken several times each night, due to an inverted circadian rhythm of melatonin.People with Smith–Magenis syndrome have engaging personalities, but all also have a lot of behavioral problems. These behavioral problems include frequent temper tantrums, meltdowns and outbursts, aggression, anger, fidgeting, compulsive behavior, anxiety, impulsiveness, and difficulty paying attention.