Benedikt syndrome, also called Benedikts syndrome or paramedian midbrain syndrome, is a rare type of posterior circulation stroke of the brain, with a range of neurological symptoms affecting the midbrain, cerebellum and other related structures. Signs and symptoms
It is characterized by the presence of an  oculomotor nerve (CN III) palsy and cerebellar ataxia including tremor and involuntary choreoathetotic movements. Neuroanatomical structures affected include the oculomotor nucleus, red nucleus, corticospinal tracts and superior cerebellar peduncle decussation. It has a similar cause, morphology, signs and symptoms to Webers syndrome; the main difference between the two being that Webers is more associated with hemiplegia (i.e. paralysis), and Benedikts with hemiataxia (i.e. disturbed coordination of movements). It is also similar to Claudes syndrome, but is distinguishable in that Benedikts has more predominant tremor and choreoathetotic movements while Claudes is more marked by the ataxia. Causes
Benedikt syndrome is caused by a lesion (infarction, hemorrhage, tumor, or tuberculosis) in the tegmentum of the midbrain and cerebellum. Specifically, the median zone is impaired. It can result from occlusion of the posterior cerebral artery or paramedian penetrating branches of the basilar artery. Diagnosis
Oculomotor nerve palsy: eyeball gazing downward and outward position, diplopia, miosis, mydriasis, and loss of accommodation reflex. Contralateral loss of proprioception and vibration sensations. Cerebellar ataxia: involuntary movements. Treatment
Deep brain stimulation may provide relief from some symptoms of Benedikt syndrome, particularly the tremors associated with the disorder. See also
Claudes syndrome
Wallenberg syndrome

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An old cesarean scar may undergo dehiscence; with further labor the woman may experience abdominal pain and vaginal bleeding, though these signs are difficult to distinguish from normal labor. Often a deterioration of the fetal heart rate is a leading sign, but the cardinal sign of uterine rupture is loss of fetal station on manual vaginal exam. Intra-abdominal bleeding can lead to hypovolemic shock and death. Although the associated maternal mortality is now less than one percent, the fetal mortality rate is between two and six percent when rupture occurs in the hospital. In pregnancy uterine rupture may cause a viable abdominal pregnancy. This is what accounts for most abdominal pregnancy births. Abdominal pain and tenderness. The pain may not be severe; it may occur suddenly at the peak of a contraction. The woman may describe a feeling that something "gave way" or "ripped." Chest pain, pain between the scapulae, or pain on inspiration—Pain occurs because of the irritation of blood below the womans diaphragm
Hypovolemic shock caused by bleeding, evidenced by falling blood pressure, tachycardia, tachypnea, pallor, cool and clammy skin, and anxiety. The fall in blood pressure is often a late sign of haemorrhage
Signs associated with fetal oxygenation, such as late deceleration, reduced variability, tachycardia, and bradycardia
Absent fetal heart sounds with a large disruption of the placenta; absent fetal heart activity by ultrasound examination
Cessation of uterine contractions
Palpation of the fetus outside the uterus (usually occurs only with a large, complete rupture). The fetus is likely to be dead at this point.