Of the 1.7 percent, 1.5 percentage points (88% of those considered "nondimorphic sexual development" in this figure) consist of individuals with late onset congenital adrenal hyperplasia (LOCAH) which may be asymptomatic but can present after puberty and cause infertility.In response to Fausto-Sterling, Leonard Sax estimated that the prevalence of intersex was about 0.018% of the worlds population, after discounting several conditions including LOCAH, Klinefelter syndrome (47,XXY), Turner syndrome (45,X), the chromosomal variants of 47,XYY and 47,XXX, and vaginal agenesis. Sax reasons that in these conditions chromosomal sex is consistent with phenotypic sex and phenotype is classifiable as either male or female.In a 2003 letter to the editor, political scientist Carrie Hull analyzed the data used by Fausto-Sterling and said the estimated intersex rate should instead have been 0.37%, due to many errors. In a response letter published simultaneously, Fausto-Sterling welcomed the additional analysis and said "I am not invested in a particular final estimate, only that there BE an estimate". A 2018 review reported that the number of births with ambiguous genitals is in the range of 0.02% to 0.05%.The figure of 1.7% is still maintained by Intersex Human Rights Australia "despite its flaws", stating both that the estimate "encapsulates the entire population of people who are stigmatized – or risk stigmatization – due to innate sex characteristics," and that Saxs definitions exclude individuals who experience such stigma and who have helped to establish the intersex movement.The following summarizes prevalences of traits that have been called intersex:

Prevalences of specific conditions can vary across regions.
The mainstay diagnosis of the presenting DVT symptom is an ultrasound with venous Doppler. Ultrasound with venous Doppler rarely identifies the inferior vena cava anomalies present from birth. Difficulties in identifying the congenital stenosis of the vena cava makes diagnosis uncommon. Diagnosis of congenital stenosis of the inferior vena cava can be rarely discovered through abdominal CT examinations, and may present with an array of clinical findings dependent on the drainage patterns of the individuals. Further diagnostic tools such as a transthoracic echocardiogram can reveal the narrowing of the vena cava showing a low velocity of flow. Congenital stenosis of the vena cava can also present as acute respiratory failure and effusion of the right lung on chest X-rays. Treatments
Treatments may not be necessary for the congenital heart defect. If required, treatments can include medication, catheter procedures, corrective surgery and heart transplants dependent on the severity of the defect, age of the patient and patients general health. Symptomatic relief of venous hypertension, associated with the congenital heart defect, can be achieved through surgery as it reduces the likelihood of hepatic vein thrombosis. Intravascular stents are a treatment method for Congenital Stenosis of the Vena Cava. Expandable metallic stents have been used as part of a procedure to dilate and maintain stenotic vena cava. Experiments carried out on mongrel dogs resulted in some failures for dilation and early migration, and occlusion complications in patients. Immediate relief of persisting symptoms occurred in two patients. Double balloon dilation is another technique used to treat stenosis of the vena cava.