Acute radiodermatitis 
Chronic radiation keratosis
Chronic radiodermatitis
Eosinophilic, polymorphic, and pruritic eruption associated with radiotherapy
Fluoroscopy burn
Radiation acne
Radiation cancer
Radiation dermatitis (radiodermatitis)
Radiation recall reaction
Radiation-induced erythema multiforme
Radiation-induced hypertrophic scar
Radiation-induced keloid
Radiation-induced morphea

Urticaria and angioedema
Urticaria is a vascular reaction of the skin characterized by the appearance of wheals, which are firm, elevated swellings of the skin. Angioedema, which can occur alone or with
urticaria, is characterized by a well-defined, edematous swelling that involves subcutaneous tissues, abdominal organs, or upper airway. Acquired C1 esterase inhibitor deficiency
Acute urticaria
Adrenergic urticaria
Anaphylaxis
Aquagenic urticaria
Cholinergic urticaria
Chronic urticaria (ordinary urticaria)
Cold urticaria
Dermatographism (dermographism)
Episodic angioedema with eosinophilia (Gleichs syndrome)
Exercise urticaria (exercise-induced urticaria)
Galvanic urticaria
Heat urticaria
Hereditary angioedema (Quinckes edema)
Localized heat contact urticaria
Mast cell-independent urticaria
Physical urticaria
Primary cold contact urticaria
Pressure urticaria (delayed pressure urticaria)
Reflex cold urticaria
Schnitzler syndrome
Secondary cold contact urticaria
Solar urticaria
Systemic capillary leak syndrome
Urticarial allergic eruption
Urticaria-like follicular mucinosis
Vibratory angioedema

Vascular-related
Vascular-related cutaneous conditions result from dysfunction of the blood or blood vessels in the dermis, or lymphatics in the subcutaneous tissues.
Hyperalgesia ( or ; hyper from  Greek ὑπέρ (huper, “over”), -algesia from Greek  algos, ἄλγος (pain)) is an abnormally increased sensitivity to   pain, which may be caused by damage to nociceptors or peripheral nerves and can cause hypersensitivity to stimulus. Prostaglandins E and F are largely responsible for sensitizing the nociceptors. Temporary increased sensitivity to pain also occurs as part of sickness behavior, the evolved response to infection. Types
Hyperalgesia can be experienced in focal, discrete areas, or as a more diffuse, body-wide form. Conditioning studies have established that it is possible to experience a learned hyperalgesia of the latter, diffuse form. The focal form is typically associated with injury, and is divided into two subtypes:

Primary hyperalgesia describes pain sensitivity that occurs directly in the damaged tissues. Secondary hyperalgesia describes pain sensitivity that occurs in surrounding undamaged tissues.Opioid-induced hyperalgesia may develop as a result of long-term opioid use in the treatment of chronic pain. Various studies of humans and animals have demonstrated that primary or secondary hyperalgesia can develop in response to both chronic and acute exposure to opioids. This side effect can be severe enough to warrant discontinuation of opioid treatment. Causes
Hyperalgesia is induced by platelet-activating factor (PAF) which comes about in an inflammatory or an allergic response. This seems to occur via immune cells interacting with the peripheral nervous system and releasing pain-producing chemicals (cytokines and chemokines).One unusual cause of focal hyperalgesia is platypus venom.Long-term opioid (e.g.