question: My dad, 90, passed on 3.15pm last Sunday in the hospital s Intermediate Care Area after being admitted late Saturday morning following paleness and abdominal discomfort ealier that morning at home. Heamoglobin level was dangerously low at 4 and blood transfusion was carried out with 40mg Furosemide initially by late evening, and subsequently Epoitine and the usual heart medications he has been on for years since heart bypass. He was previously diagnosed as having stage 4 renal impairment, suspectedly caused by Voltaren treatment following a fall in Nov 20102 which resulted in mild L2 fracture. His Creatinine level improved with Kestosterone and Epoitin (twice weekly) which was subsequently reduced to once weekly. I had stopped him from taking Furosemide, prescribed after his Edema event in March this year, because of adverse side effects and his condition improved with a diet of oats, multivatimins, kidney milk (Nepro for breakfast), fruit and vegetable juice (lunch) and soya milk (night/supper). Was death precipitated by iron insufficiency, leading to heart failure hence Edema and shutdown of kidneys after 40-80-80 doses of Furosemide did not release no more than 750ml of urine (on Saturday only)?