MR Spectroscopy Head There is no relevant literature to support the use of MR spectroscopy head in the initial evaluation of a child with chronic progressive ataxia. Altered metabolite ratios or presence of specific metabolites on MR spectroscopy may be useful in distinguishing posterior fossa tumor types and molecular subtypes. MRA Head and Neck There is no relevant literature to support the use of MRA head and neck in the initial evaluation of a child with chronic progressive ataxia. MRA Head There is no relevant literature to support the use of MRA head in the initial evaluation of a child with chronic progressive ataxia. MRA Neck There is no relevant literature to support the use of MRA neck in the initial evaluation of a child with chronic progressive ataxia. MRV Head and Neck There is no relevant literature to support the use of MRV head and neck in the initial evaluation of a child with chronic progressive ataxia. MRV Head There is no relevant literature to support the use of MRV head in the initial evaluation of a child with chronic progressive ataxia. Variant 5: Child. Chronic nonprogressive ataxia. Initial imaging. Chronic nonprogressive ataxias are comprised of a heterogenous group of cerebellar ataxias with early onset of cerebellar symptoms and no clinical change in severity during follow-up [1,4,25]. Commonly, symptom onset in these cases is within the first year of life, and this group of ataxias are referred to as chronic nonprogressive congenital ataxia [49]. Chronic nonprogressive ataxia in children may be due to syndromic, nonsyndromic, genetic, and acquired etiologies. In some syndromic causes such as Joubert syndrome and related disorders, ataxia is characteristic [50]. There are genetic causes of nonprogressive congenital ataxia with autosomal dominant, autosomal recessive, and x-linked inheritance patterns but with poor correlation between genotype and phenotype defined by imaging and clinical status [49].
Ataxia Child. MR Spectroscopy Head There is no relevant literature to support the use of MR spectroscopy head in the initial evaluation of a child with chronic progressive ataxia. Altered metabolite ratios or presence of specific metabolites on MR spectroscopy may be useful in distinguishing posterior fossa tumor types and molecular subtypes. MRA Head and Neck There is no relevant literature to support the use of MRA head and neck in the initial evaluation of a child with chronic progressive ataxia. MRA Head There is no relevant literature to support the use of MRA head in the initial evaluation of a child with chronic progressive ataxia. MRA Neck There is no relevant literature to support the use of MRA neck in the initial evaluation of a child with chronic progressive ataxia. MRV Head and Neck There is no relevant literature to support the use of MRV head and neck in the initial evaluation of a child with chronic progressive ataxia. MRV Head There is no relevant literature to support the use of MRV head in the initial evaluation of a child with chronic progressive ataxia. Variant 5: Child. Chronic nonprogressive ataxia. Initial imaging. Chronic nonprogressive ataxias are comprised of a heterogenous group of cerebellar ataxias with early onset of cerebellar symptoms and no clinical change in severity during follow-up [1,4,25]. Commonly, symptom onset in these cases is within the first year of life, and this group of ataxias are referred to as chronic nonprogressive congenital ataxia [49]. Chronic nonprogressive ataxia in children may be due to syndromic, nonsyndromic, genetic, and acquired etiologies. In some syndromic causes such as Joubert syndrome and related disorders, ataxia is characteristic [50]. There are genetic causes of nonprogressive congenital ataxia with autosomal dominant, autosomal recessive, and x-linked inheritance patterns but with poor correlation between genotype and phenotype defined by imaging and clinical status [49].