Headache is present in 90% of patients with IIH and is commonly the presenting symptom. The diagnostic criteria as per the ICHD-3 includes a documentation of intracranial pressure exceeding 250 mm H2O (or 280 mm H2O in obese children). A lumbar puncture to document opening pressure remains a required diagnostic tool for the diagnosis of IIH. Physical examination findings such as papilledema is a cardinal feature of IIH. Recognition of IIH and treatment is predominantly aimed preserving vision. The role of neuroimaging is mostly to exclude secondary causes of elevated intracranial pressure and to aid in the diagnosis of IIH [23,35,36]. It can also identify venous outflow problems, which can contribute to elevated intracranial pressure in both primary and secondary intracranial hypertension. Arteriography Cervicocerebral There is no relevant literature to support the use of cervicocerebral arteriography in the initial imaging evaluation of headache with features of intracranial hypertension. CT Head With IV Contrast There is no relevant literature to support the use of CT head with IV contrast in the initial imaging evaluation of headache with features of intracranial hypertension. CT Head Without and With IV Contrast There is no relevant literature to support the use of CT head without and with IV contrast in the initial imaging evaluation of headache with features of intracranial hypertension. CTA Head With IV Contrast There is no relevant literature to support the use of CTA head with IV contrast in the initial imaging evaluation of headache with features of intracranial hypertension. CTV Head With IV Contrast Theories regarding the pathophysiology of IIH include venous outflow obstruction leading to increased intracranial pressure, which can compress and narrow the distal transverse sinuses. Several studies have demonstrated a finding
Headache PCAs. Headache is present in 90% of patients with IIH and is commonly the presenting symptom. The diagnostic criteria as per the ICHD-3 includes a documentation of intracranial pressure exceeding 250 mm H2O (or 280 mm H2O in obese children). A lumbar puncture to document opening pressure remains a required diagnostic tool for the diagnosis of IIH. Physical examination findings such as papilledema is a cardinal feature of IIH. Recognition of IIH and treatment is predominantly aimed preserving vision. The role of neuroimaging is mostly to exclude secondary causes of elevated intracranial pressure and to aid in the diagnosis of IIH [23,35,36]. It can also identify venous outflow problems, which can contribute to elevated intracranial pressure in both primary and secondary intracranial hypertension. Arteriography Cervicocerebral There is no relevant literature to support the use of cervicocerebral arteriography in the initial imaging evaluation of headache with features of intracranial hypertension. CT Head With IV Contrast There is no relevant literature to support the use of CT head with IV contrast in the initial imaging evaluation of headache with features of intracranial hypertension. CT Head Without and With IV Contrast There is no relevant literature to support the use of CT head without and with IV contrast in the initial imaging evaluation of headache with features of intracranial hypertension. CTA Head With IV Contrast There is no relevant literature to support the use of CTA head with IV contrast in the initial imaging evaluation of headache with features of intracranial hypertension. CTV Head With IV Contrast Theories regarding the pathophysiology of IIH include venous outflow obstruction leading to increased intracranial pressure, which can compress and narrow the distal transverse sinuses. Several studies have demonstrated a finding