instruction:
Given an abstract of a paper, generate a title for this paper such that conveys the key focus of the paper.
question:
Abstract: The short-term effects of incremental doses of procainamide (7.5, 15, 22.5, and 30 mg/kg) on right ventricular effective refractory period, intraventricular conduction, and induction of ventricular tachycardia were determined in 31 patients who had a history of sustained, unimorphic ventricular tachycardia. QRS duration during incremental ventricular pacing was used as an index of rate-dependent changes in intraventricular conduction. The mean plasma procainamide concentrations corresponding to the incremental doses were 5.5 +/- 1.2 (+/- SD), 9.0 +/- 1.6, 12.6 +/- 2.2, and 16.3 +/- 3.2 mg/liter. Each incremental dose of procainamide up to a dose of 30 mg/kg resulted in a significant increment in right ventricular effective refractory period and each dose up to 22.5 mg/kg potentiated a rate-dependent prolongation of QRS duration. After the 7.5 mg/kg dose of procainamide, induction of ventricular tachycardia was suppressed in eight of 31 patients. After higher doses of procainamide, induction of ventricular tachycardia was suppressed in two additional patients. In three of 10 patients in whom the induction of ventricular tachycardia was suppressed by 7.5, 15, or 22.5 mg/kg of procainamide, sustained unimorphic ventricular tachycardia was again inducible after a higher dose of procainamide. In three of 31 patients, only nonsustained ventricular tachycardia was inducible after a 7.5 to 22.5 mg/kg dose of procainamide; however, in two of these three patients, sustained ventricular tachycardia was again inducible after administration of a higher dose of procainamide. In conclusion, during electropharmacologic testing with procainamide, it is worthwhile to test a dose of 7.5 mg/kg, because this dose is often effective in patients who respond to this drug. However, the results of this study indicate that procainamide may be effective in suppressing the induction of sustained ventricular tachycardia at a relatively low plasma concentration, but not at a higher plasma concentration. Therefore, during long-term therapy with procainamide it may be important to avoid plasma procainamide concentrations not only lower, but also higher than the concentration that results in the suppression of induction of tachycardia.
answer:
Effects of incremental doses of procainamide on ventricular refractoriness, intraventricular conduction, and induction of ventricular tachycardia.


question:
Abstract: To determine the relative contributions of the atrial and ventricular septa to total cardiac septation in patients with atrioventricular (AV) canal defect, two-dimensional echocardiography was used to measure the length of each portion of the septum in 14 patients with partial AV canal and 13 patients with the complete form of this lesion. Results were compared with similar data from 30 normal children, 10 patients with dilated cardiomyopathy and 11 patients with ostium secundum atrial septal defect. In patients with partial AV canal, the portion of total cardiac length occupied by the atrial septum did not differ from normal (24.8 +/- 9.9% versus 28.7 +/- 7.9%, p greater than 0.05), but there was a deficiency of both AV and ventricular septal tissue, the latter occupying only 48.2 +/- 7.0% of total cardiac length (versus 57.9 +/- 4.1% in normal subjects, p less than 0.05). Similarly, patients with complete AV canal had a normal amount of atrial septal tissue, a deficiency of AV septal tissue and a deficiency of ventricular septal tissue which was even greater than that of patients with a partial defect (39.4 +/- 5.0% versus 48.2 +/- 7.0%, p less than 0.05). Thus, although partial AV canal defect is often called ostium primum atrial septal defect, the amount of atrial tissue is usually normal, the defect being formed by a deficiency of AV and ventricular septal tissue. In the complete form of the lesion, the deficiency of ventricular septal tissue is greater.
answer:
Cardiac septation in atrioventricular canal defect.


question:
Abstract: A patient with severe chronic gastritis and duodenitis that clinically and endoscopically disappeared after an intestinal revascularization procedure is reported. Chronic intestinal ischemia appears to be a cause of gastritis as seen in this patient.
answer:

Chronic ischemic gastritis reversed after revascularization operation.



instruction:
Given an abstract of a paper, generate a title for this paper such that conveys the key focus of the paper.
question:
Abstract: The short-term effects of incremental doses of procainamide (7.5, 15, 22.5, and 30 mg/kg) on right ventricular effective refractory period, intraventricular conduction, and induction of ventricular tachycardia were determined in 31 patients who had a history of sustained, unimorphic ventricular tachycardia. QRS duration during incremental ventricular pacing was used as an index of rate-dependent changes in intraventricular conduction. The mean plasma procainamide concentrations corresponding to the incremental doses were 5.5 +/- 1.2 (+/- SD), 9.0 +/- 1.6, 12.6 +/- 2.2, and 16.3 +/- 3.2 mg/liter. Each incremental dose of procainamide up to a dose of 30 mg/kg resulted in a significant increment in right ventricular effective refractory period and each dose up to 22.5 mg/kg potentiated a rate-dependent prolongation of QRS duration. After the 7.5 mg/kg dose of procainamide, induction of ventricular tachycardia was suppressed in eight of 31 patients. After higher doses of procainamide, induction of ventricular tachycardia was suppressed in two additional patients. In three of 10 patients in whom the induction of ventricular tachycardia was suppressed by 7.5, 15, or 22.5 mg/kg of procainamide, sustained unimorphic ventricular tachycardia was again inducible after a higher dose of procainamide. In three of 31 patients, only nonsustained ventricular tachycardia was inducible after a 7.5 to 22.5 mg/kg dose of procainamide; however, in two of these three patients, sustained ventricular tachycardia was again inducible after administration of a higher dose of procainamide. In conclusion, during electropharmacologic testing with procainamide, it is worthwhile to test a dose of 7.5 mg/kg, because this dose is often effective in patients who respond to this drug. However, the results of this study indicate that procainamide may be effective in suppressing the induction of sustained ventricular tachycardia at a relatively low plasma concentration, but not at a higher plasma concentration. Therefore, during long-term therapy with procainamide it may be important to avoid plasma procainamide concentrations not only lower, but also higher than the concentration that results in the suppression of induction of tachycardia.
answer:
Effects of incremental doses of procainamide on ventricular refractoriness, intraventricular conduction, and induction of ventricular tachycardia.


question:
Abstract: To determine the relative contributions of the atrial and ventricular septa to total cardiac septation in patients with atrioventricular (AV) canal defect, two-dimensional echocardiography was used to measure the length of each portion of the septum in 14 patients with partial AV canal and 13 patients with the complete form of this lesion. Results were compared with similar data from 30 normal children, 10 patients with dilated cardiomyopathy and 11 patients with ostium secundum atrial septal defect. In patients with partial AV canal, the portion of total cardiac length occupied by the atrial septum did not differ from normal (24.8 +/- 9.9% versus 28.7 +/- 7.9%, p greater than 0.05), but there was a deficiency of both AV and ventricular septal tissue, the latter occupying only 48.2 +/- 7.0% of total cardiac length (versus 57.9 +/- 4.1% in normal subjects, p less than 0.05). Similarly, patients with complete AV canal had a normal amount of atrial septal tissue, a deficiency of AV septal tissue and a deficiency of ventricular septal tissue which was even greater than that of patients with a partial defect (39.4 +/- 5.0% versus 48.2 +/- 7.0%, p less than 0.05). Thus, although partial AV canal defect is often called ostium primum atrial septal defect, the amount of atrial tissue is usually normal, the defect being formed by a deficiency of AV and ventricular septal tissue. In the complete form of the lesion, the deficiency of ventricular septal tissue is greater.
answer:
Cardiac septation in atrioventricular canal defect.


question:
Abstract: A patient with severe chronic gastritis and duodenitis that clinically and endoscopically disappeared after an intestinal revascularization procedure is reported. Chronic intestinal ischemia appears to be a cause of gastritis as seen in this patient.
answer:

user
Chronic ischemic gastritis reversed after revascularization operation.



assistant
instruction:
Given an abstract of a paper, generate a title for this paper such that conveys the key focus of the paper.
question:
Abstract: The short-term effects of incremental doses of procainamide (7.5, 15, 22.5, and 30 mg/kg) on right ventricular effective refractory period, intraventricular conduction, and induction of ventricular tachycardia were determined in 31 patients who had a history of sustained, unimorphic ventricular tachycardia. QRS duration during incremental ventricular pacing was used as an index of rate-dependent changes in intraventricular conduction. The mean plasma procainamide concentrations corresponding to the incremental doses were 5.5 +/- 1.2 (+/- SD), 9.0 +/- 1.6, 12.6 +/- 2.2, and 16.3 +/- 3.2 mg/liter. Each incremental dose of procainamide up to a dose of 30 mg/kg resulted in a significant increment in right ventricular effective refractory period and each dose up to 22.5 mg/kg potentiated a rate-dependent prolongation of QRS duration. After the 7.5 mg/kg dose of procainamide, induction of ventricular tachycardia was suppressed in eight of 31 patients. After higher doses of procainamide, induction of ventricular tachycardia was suppressed in two additional patients. In three of 10 patients in whom the induction of ventricular tachycardia was suppressed by 7.5, 15, or 22.5 mg/kg of procainamide, sustained unimorphic ventricular tachycardia was again inducible after a higher dose of procainamide. In three of 31 patients, only nonsustained ventricular tachycardia was inducible after a 7.5 to 22.5 mg/kg dose of procainamide; however, in two of these three patients, sustained ventricular tachycardia was again inducible after administration of a higher dose of procainamide. In conclusion, during electropharmacologic testing with procainamide, it is worthwhile to test a dose of 7.5 mg/kg, because this dose is often effective in patients who respond to this drug. However, the results of this study indicate that procainamide may be effective in suppressing the induction of sustained ventricular tachycardia at a relatively low plasma concentration, but not at a higher plasma concentration. Therefore, during long-term therapy with procainamide it may be important to avoid plasma procainamide concentrations not only lower, but also higher than the concentration that results in the suppression of induction of tachycardia.
answer:
Effects of incremental doses of procainamide on ventricular refractoriness, intraventricular conduction, and induction of ventricular tachycardia.


question:
Abstract: To determine the relative contributions of the atrial and ventricular septa to total cardiac septation in patients with atrioventricular (AV) canal defect, two-dimensional echocardiography was used to measure the length of each portion of the septum in 14 patients with partial AV canal and 13 patients with the complete form of this lesion. Results were compared with similar data from 30 normal children, 10 patients with dilated cardiomyopathy and 11 patients with ostium secundum atrial septal defect. In patients with partial AV canal, the portion of total cardiac length occupied by the atrial septum did not differ from normal (24.8 +/- 9.9% versus 28.7 +/- 7.9%, p greater than 0.05), but there was a deficiency of both AV and ventricular septal tissue, the latter occupying only 48.2 +/- 7.0% of total cardiac length (versus 57.9 +/- 4.1% in normal subjects, p less than 0.05). Similarly, patients with complete AV canal had a normal amount of atrial septal tissue, a deficiency of AV septal tissue and a deficiency of ventricular septal tissue which was even greater than that of patients with a partial defect (39.4 +/- 5.0% versus 48.2 +/- 7.0%, p less than 0.05). Thus, although partial AV canal defect is often called ostium primum atrial septal defect, the amount of atrial tissue is usually normal, the defect being formed by a deficiency of AV and ventricular septal tissue. In the complete form of the lesion, the deficiency of ventricular septal tissue is greater.
answer:
Cardiac septation in atrioventricular canal defect.


question:
Abstract: A patient with severe chronic gastritis and duodenitis that clinically and endoscopically disappeared after an intestinal revascularization procedure is reported. Chronic intestinal ischemia appears to be a cause of gastritis as seen in this patient.
answer:

Chronic ischemic gastritis reversed after revascularization operation.


