P. gingivalis in Alzheimer's disease brains
Here’s some related material:"It turns out that [amyloid-beta proteins] form [plaques] because they're trying to help us. They're trying to fight an infection," said Robert Moir, an assistant professor of neurobiology at Harvard Medical School and the Massachusetts General Hospital."The Alzheimer's disease dementia that comes after years of this battle, are something like collateral damage from their actions. But it's not that it's intrinsically bad what they're doing. It's just that if you keep doing it for long enough the amyloid that is generated in this battle starts to become a problem in itself."If this theory, which Moir outlines in a review article published in the December issue of the journal Alzheimer's & Dementia, proves true, it could mean researchers' current strategies to combat the disease — fighting the plaques — is misguided.https://www.cbc.ca/radio/quirks/dec-8-2018-why-are-users-tak...The antimicrobial protection hypothesis of Alzheimer's disease:https://www.sciencedirect.com/science/article/pii/S155252601...