The assay aims to identify molecules that stimulate Gsα mutations, which cause McCune-Albright syndrome, in order to better understand the disease and potentially develop drugs to treat it. The mutations impair the intrinsic GTPase activity of Gsα, leading to ligand-independent activation of GPCR pathways and excess Gs/cAMP signaling in affected cells. The project focuses on finding compounds that can activate GPCR pathways without relying on ligand interaction. Is the molecule effective to this assay?