The human immunodeficiency viruses (HIV) are a type of retrovirus, which induce acquired immune deficiency syndrome (AIDs).  Now there are six main classes of antiretroviral drugs to treating AIDs patients approved by FDA, which are the nucleoside reverse transcriptase inhibitors (NRTIs), the non-nucleoside reverse transcriptase inhibitors (NNRTIs), the protease inhibitors, the integrase inhibitor, the fusion inhibitor, and the chemokine receptor CCR5 antagonist. Due to the missing 3’hydroxyl group, NRTIs prevent the formation of a 3’-5’-phosphodiester bond in growing DNA chains. The hydroxyl group of the inhibitor interacts with the carboxyl group of the protease active site residues, Asp 25 and Asp 25′, by hydrogen bonds. The inhibitor-contacting residues of HIV protease are relatively conserved, including Gly 27, Asp 29, Asp 30, and Gly 48. Is this molecule effective to this assay? Human immunodeficiency viruses (HIV) are a type of retrovirus, which induces acquired immune deficiency syndrome (AIDs).  Now there are six main classes of antiretroviral drugs for treating AIDs patients approved by FDA, which are the nucleoside reverse transcriptase inhibitors (NRTIs), the non-nucleoside reverse transcriptase inhibitors (NNRTIs), the protease inhibitors, the integrase inhibitor, the fusion inhibitor, and the chemokine receptor CCR5 antagonist. Due to the missing 3’hydroxyl group, NRTIs prevent the formation of a 3’-5’-phosphodiester bond in growing DNA chains. The hydroxyl group of the inhibitor interacts with the carboxyl group of the protease active site residues, Asp 25 and Asp 25′, by hydrogen bonds. The inhibitor-contacting residues of HIV protease are relatively conserved, including Gly 27, Asp 29, Asp 30, and Gly 48. Is this molecule effective to this assay? Human immunodeficiency viruses (HIV) are a type of retrovirus, which induces acquired immune deficiency syndrome (AIDs).  Now there are six main classes of antiretroviral drugs for treating AIDs patients approved by FDA, which are the nucleoside reverse transcriptase inhibitors (NRTIs), the non-nucleoside reverse transcriptase inhibitors (NNRTIs), the protease inhibitors, the integrase inhibitor, the fusion inhibitor, and the chemokine receptor CCR5 antagonist.  Is this molecule effective to this assay? Human immunodeficiency viruses (HIV) are a type of retrovirus, which induces acquired immune deficiency syndrome (AIDs).  Is this molecule effective to this assay? The human immunodeficiency viruses (HIV) are a type of retrovirus, which induce acquired immune deficiency syndrome (AIDs).  Now there are six main classes of antiretroviral drugs to treating AIDs patients approved by FDA, which are the nucleoside reverse transcriptase inhibitors (NRTIs), the non-nucleoside reverse transcriptase inhibitors (NNRTIs), the protease inhibitors, the integrase inhibitor, the fusion inhibitor, and the chemokine receptor CCR5 antagonist. Due to the missing 3’hydroxyl group, NRTIs prevent the formation of a 3’-5’-phosphodiester bond in growing DNA chains. The hydroxyl group of the inhibitor interacts with the carboxyl group of the protease active site residues, Asp 25 and Asp 25′, by hydrogen bonds. The inhibitor-contacting residues of HIV protease are relatively conserved, including Gly 27, Asp 29, Asp 30, and Gly 48. Can this molecule inhibit HIV replication? Human immunodeficiency viruses (HIV) are a type of retrovirus, which induces acquired immune deficiency syndrome (AIDs).  Now there are six main classes of antiretroviral drugs for treating AIDs patients approved by FDA, which are the nucleoside reverse transcriptase inhibitors (NRTIs), the non-nucleoside reverse transcriptase inhibitors (NNRTIs), the protease inhibitors, the integrase inhibitor, the fusion inhibitor, and the chemokine receptor CCR5 antagonist. Due to the missing 3’hydroxyl group, NRTIs prevent the formation of a 3’-5’-phosphodiester bond in growing DNA chains. The hydroxyl group of the inhibitor interacts with the carboxyl group of the protease active site residues, Asp 25 and Asp 25′, by hydrogen bonds. The inhibitor-contacting residues of HIV protease are relatively conserved, including Gly 27, Asp 29, Asp 30, and Gly 48. Is this molecule effective for inhibiting Human immunodeficiency viruses (HIV) replication? Human immunodeficiency viruses (HIV) are a type of retrovirus, which induces acquired immune deficiency syndrome (AIDs).  Now there are six main classes of antiretroviral drugs for treating AIDs patients approved by FDA, which are the nucleoside reverse transcriptase inhibitors (NRTIs), the non-nucleoside reverse transcriptase inhibitors (NNRTIs), the protease inhibitors, the integrase inhibitor, the fusion inhibitor, and the chemokine receptor CCR5 antagonist.  Is this molecule effective for inhibiting Human immunodeficiency viruses (HIV) replication? Human immunodeficiency viruses (HIV) are a type of retrovirus, which induces acquired immune deficiency syndrome (AIDs).  Is this molecule effective for inhibiting Human immunodeficiency viruses (HIV) replication?