Factor XIa (FXIa) is a serine protease enzyme that plays an important role in the coagulation (clotting) cascade of the blood. It is activated by its zymogen, Factor XI (FXI), in response to vascular injury or other triggers. Once activated, Factor XIa cleaves and activates Factor IX, leading to the formation of thrombin and ultimately a blood clot. Dysfunction or overactivity of FXIa can lead to bleeding disorders or excessive clotting, respectively. Because of its role in the coagulation cascade, FXIa is an important target for the development of anticoagulant therapies. Is this molecule inhibitor of FXIa? Factor XIa (FXIa) is a serine protease enzyme that plays an important role in the coagulation (clotting) cascade of the blood. It is activated by its zymogen, Factor XI (FXI), in response to vascular injury or other triggers. Once activated, Factor XIa cleaves and activates Factor IX, leading to the formation of thrombin and ultimately a blood clot. Dysfunction or overactivity of FXIa can lead to bleeding disorders or excessive clotting, respectively. Because of its role in the coagulation cascade, FXIa is an important target for the development of anticoagulant therapies. Can this molecule inhibite FXIa? Factor XIa (FXIa) is a serine protease enzyme that plays an important role in the coagulation (clotting) cascade of the blood. It is activated by its zymogen, Factor XI (FXI), in response to vascular injury or other triggers. Once activated, Factor XIa cleaves and activates Factor IX, leading to the formation of thrombin and ultimately a blood clot. Dysfunction or overactivity of FXIa can lead to bleeding disorders or excessive clotting, respectively. Because of its role in the coagulation cascade, FXIa is an important target for the development of anticoagulant therapies. Is this molecule effective to FXIa? Factor XIa (FXIa) is a serine protease enzyme that plays an important role in the coagulation (clotting) cascade of the blood. It is activated by its zymogen, Factor XI (FXI), in response to vascular injury or other triggers. Once activated, Factor XIa cleaves and activates Factor IX, leading to the formation of thrombin and ultimately a blood clot. Dysfunction or overactivity of FXIa can lead to bleeding disorders or excessive clotting, respectively. Because of its role in the coagulation cascade, FXIa is an important target for the development of anticoagulant therapies. Is this molecule effective to this assay?