Lambrecht, B.N., and Hammad, H.: Allergens and the airway epithelium response: gateway to allergic sensitization. J. Allergy Clin. Immunol. 2014, 134:499–507.
Nordlee, J.A., Taylor, S.L., Townsend, J.A., Thomas, L.A., and Bush, R.K.: Identification of a Brazil-nut allergen in transgenic soybeans. N. Engl. J. Med. 1996, 334:688–692.
Papzian, D., Wagtmann, V.R., Hansen, S., and Wurtzen, P.A.: Direct contact between dendritic cells and bronchial epithelial cells inhibits T cell recall responses towards mite and pollen allergen extracts in vitro. Clin. Exp. Immunol. 2015, 181:207–218.
Sehgal, N., Custovic, A., and Woodcock, A.: Potential roles in rhinitis for protease and other enzymatic activities of allergens. Curr. Allergy Asthma Rep. 2005, 5:221–226.
Sprecher, E., Tesfaye-Kedjela, A., Ratajczak, P., Bergman, R., and Richard, G.: Deleterious mutations in SPINK5 in a patient with congenital ichthyosiform erythroderma: molecular testing as a helpful diagnostic tool for Netherton syndrome. Clin. Exp. Dermatol. 2004, 29:513–517.
Wan, H., Winton, H.L., Soeller, C., Tovey, E.R., Gruenert, D.C., Thompson, P.J., Stewart, G.A., Taylor, G.W., Garrod, D.R., Cannell, M.B., et al.: Der p 1 facilitates transepithelial allergen delivery by disruption of tight junctions. J. Clin. Invest. 1999, 104:123–133.
14-3 Genetic factors contribute to the development of IgE-mediated allergic disease.
Cookson, W.: The immunogenetics of asthma and eczema: a new focus on the epithelium. Nat. Rev. Immunol. 2004, 4:978–988.
Illing, P.R., Vivian, J.P., Purcell, A.W., Rossjohn, J., and McCluskey J.: Human leukocyte antigen-associated drug hypersensitivity. Curr. Opin. Immunol. 2013, 25:81–89.
Li, Z., Hawkins, G.A., Ampleford, E.J., Moore, W.C., Li, H., Hastie, A.T., Howard, T.D., Boushey H.A., Busse, W.W., Calhoun, W.J., et al.: Genome-wide association study identifies TH1 pathway genes associated with lung function in asthmatic patients. J. Allergy Clin. Immunol. 2013, 132:313–320.
Peiser, M.: Role of TH17 cells in skin inflammation of allergic contact dermatitis. Clin. Dev. Immunol. 2013, doi:10.1155/2013/261037.
Thyssen, J.P., Carlsen, B.C., Menné, T., Linneberg, A., Nielsen, N.H., Meldgaard, M., Szecsi, P.B., Stender, S., and Johansen, J.D.: Filaggrin null-mutations increase the risk and persistence of hand eczema in subjects with atopic dermatitis: results from a general population study. Br. J. Dermatol. 2010, 163:115–120.