Researchers from the University of California have found that a peptide that forms deposits in the human brain and is thought to be responsible for the onset of Alzheimer’s disease, behaves in ways that are very similar to the way prions behave when propagating through mammalian neurological tissue. In their paper published in the Proceedings of the National Academy of Sciences describing their research into the ways amyloid-ß (Aß) peptides form deposits, the team found that they propagate across brain tissue in much the say way prions do when causing ailments such as Creutzfeldt-Jakob disease (CJD).