An 11-year-old male, with a diagnosis of pathological demand avoidance (PDA) and migraine, presented to a community optometrist with a 2-day history of a black spot in the central vision of his right eye. The optometrist reported that the best corrected visual acuity (BCVA) in the right eye was reduced to 6/10 Snellen having been normal at a prior visit. Left was 6/5 Snellen. New pigmentary changes at both macula were observed by the optometrist and referral was made to the hospital eye service (HES). He was taking pizotifen prescribed for migraine. There was no relevant past ocular, medical or family history. Six weeks later, in the HES review, he described a persistent ‘blur’ in the central vision of the right eye. Unaided VAs were 6/9 right and 6/5 left. Pigmentary changes were noted at the central macula in both eyes. SD-OCT and imaging revealed bilateral outer lamellar layer defects (Fig. ). Electrodiagnostic tests were normal but with limited co-operation. His mother accompanied him for all HES visits. In due course and following direct questioning he admitted to constructing a device made from Lego™ consisting of a laser pointer with a condensing lens used just prior to the onset of visual symptoms. The patient’s mother revealed that she had purchased the laser pointer online for him. During follow-up he reported symptomatic improvement and the VA remained stable. Centre involving structural defects at both macula persisted on clinical examination and OCT imaging to most recent follow-up 24 months later. (Supplementary Image ). The mother confirmed the laser had been purchased from a well-known UK online retailer and was still available for online purchase a year following the incident.
A 13-year-old male with attention deficit disorder (ADD) presented to the Emergency Medicine Department accompanied by his mother complaining of visual disturbance after staring into the beam from a toy laser for a few hours earlier that day. The patient stated that the toy laser belonged to a friend but the injuries were self-inflicted. The BCVA was 6/60 in the right eye improving to 6/36 with pinhole, and 6/12 in the left eye. SD-OCT images on presentation showed full-thickness hyper-reflective damage involving both fovea (Fig. ). The patient was on methylphenidate 57 mg daily treatment for ADD and was known to Child and Adolescent Mental Health Services (CAMHS). He attended mainstream school with additional classroom support but was not classified as having special educational needs, with no statement of educational needs undertaken previously. Six weeks later, his BCVA had improved to 6/12 right and 6/9 left. An improvement in SD-OCT images was observed, notably an improvement in inner retinal layers. However, the centre involving outer lamellar layer defects on OCT and fundus changes persisted throughout 24 months of follow-up but decreased.