History & examination\nA 31-year-old African-American female with no significant medical history presented with a sudden onset of severe headaches that were highly suggestive of subarachnoid hemorrhage (SAH). The patient was initially evaluated in the emergency department where a neurological examination revealed no deficits.\nPathological findings\nA computed tomography (CT) scan revealed a SAH filling the pre-pontine, crural, ambient, and quadrigeminal cisterns more prominently on the left side of the brain, with mild hydrocephalus (Figure ). A CT-angiography (CTA) showed an enlarged vessel within the left ambient cistern. Digital subtraction angiography (DSA) also revealed the large and irregular appearance of the left SCA in the ambient cistern. No immediate intervention was undertaken.\nThe patient was admitted to the neurosurgical intensive care unit (ICU) and treatment was initiated with regular neurological examinations and daily transcranial-doppler (TCD) studies. The patient remained intact, with TCD values showing only moderate vasospasm. The patient did not require any treatment for the mild radiographic hydrocephalus. On Day 14, the patient underwent a repeat DSA which showed a persistent vasospasm in the distal basilar and bilateral posterior cerebral arteries. Three-dimensional (3-D) reformatting also demonstrated a dissecting fusiform SCA aneurysm in the lateral ponto-mesencephalic segment (Figure ). Due to the persistent vasospasm, no intervention was undertaken at that time as definitive treatment measures were being discussed in a multidisciplinary board meeting.\nEndovascular intervention\nOn Day 21, the patient underwent a successful placement of a low-profile visualized intraluminal support (LVIS Jr.) device (MicroVention, Tustin, CA) covering the fusiform aneurysm, which measured 3-mm wide and 18-mm in length. After premedication with aspirin and clopidogrel, a 7-Fr sheath was inserted into the common femoral artery and a 6-Fr guide catheter was inserted into the left vertebral artery. The left SCA was catheterized using the microcatheter and microguidewire. The LVIS Jr. device was then deployed successfully along the fusiform segment of the SCA without evidence of contrast extravasation. The treatment goal was to preserve the intraluminal flow with flow diversion from the aneurysm for reduction and eventual obliteration.\nPostoperative course\nAt the three-month follow-up, the patient had no complaint and was neurologically intact on examination. A CTA performed at that time showed the SCA patency proximal and distal to the stent without evidence of persistent dilatation or an aneurysm (Figure ). A formal catheter angiogram was deferred by the patient. At the 10-month follow-up, a DSA demonstrated the interval healing of the aneurysm with preserved vessel patency as well as mild distortion caused by the stent in the SCA (Figure ). The patient remained neurologically intact on examination with no complaints.
A 64-year-old man, who had a history of hypertension, presented with headache and vomiting. CT scan showed SAH in the superior cerebellar cisterns []. DSA done revealed a fusiform aneurysm of the distal SCA, predominantly involving the branch of the cortical segment [Figures and ]. Standard heparin protocol was followed and an activated clotting time greater than 250 s was maintained during the procedure. 5F Envoy (cordis) guiding catheter was inserted in the distal vertebral artery. The aneurysm was accessed with a combination of micro-catheter (echelon-10) and micro-wire (traxcess12). Meticulous pre-shaping of the micro-catheter according to the shape of the target aneurysm aided in proper positioning of the micro-catheter in the aneurysm which was subsequently packed densely with the platinum micro-coils. Post-procedure angiogram showed complete exclusion of the aneurysm along with the branch of the cortical segment and preservation of the flow in the superior cerebellar artery (SCA) []. There were no untoward events in the post-procedure hospital stay and patient was discharged after 1 week. No neurological deficit was seen during the clinical follow-up after 1 month. Control angiogram carried out after 1 year showed stable occlusion of the aneurysm with preservation of the ante-grade flow in the SCA [].