A 33-year-old female presented 3 months postpartum with a headache of moderate severity and progressive visual loss in both eyes. On examination, the patient's Glasgow coma scale (GCS) was 15/15. Visual field examination showed left homonymous incomplete hemianopia. Her visual acuity was 20/25 in the right eye and 20/30 in the left eye. Her discs and macula were healthy bilaterally. Extraocular movements were intact and pupils were reactive. The rest of her examination was unremarkable. Complete endocrine workup was normal.\nMagnetic resonance imaging (MRI) revealed a large heterogeneous, hyperintense, hemorrhagic right suprasellar extra-axial complex cystic structure measuring 31 × 30 × 90 mm on T1-weighted images. There was mass effect on the adjacent hypothalamus and third ventricle displacing them toward the left and superiorly in addition to the optic pathway. The pituitary stalk was displaced toward the left. The lesion encased the right posterior cerebral artery and displaced the right carotid artery laterally []. Computed tomography (CT) arteriography demonstrated a completely thrombosed center. The imaging findings were compatible with suprasellar CM.\nThe patient underwent right frontal craniotomy and gross total resection of her suprasellar intrachiasmatic large infiltrative hemorrhagic CM. Organizing blood clots with reactive fibrohistiocytic and inflammatory reaction admixed with some ectatic vascular channels suggestive of a vascular malformation were noted. There were small foci admixed with granulation tissue, showing some dilated cavernous spaces that would be compatible with a vascular malformation such as cavernous angioma. On immunohistochemistry, the lesion was CD163+, CD20 rare, CD3+, CD34+, CD31+, CD38+, CTK−, EMA plasma cells, GFAP−, S100 dendritic cells, SMA vascular smooth muscle.\nThe patient had an uneventful operative course. Her visual acuity improved to 20/20 in both eyes. Extraocular muscles showed mild limitation of both eyes in an upward gaze. Otherwise, she was stable with no neurological deficits. Follow-up MRI at 12 months revealed complete removal of the suprasellar hemorrhagic CM with no evidence of a residual lesion or recurrence [].
A 42-year-old woman presented with a 6-day history of severe headache and a 3-day history of blurred vision. She had visual field defect in the confrontation test with no other neurological deficits. Ophthalmologic evaluation revealed central scotoma on both eyes () with normal visual acuity. Funduscopic examination showed intact optic discs with no abnormalities. A computed tomography scan showed a 1.3 cm high density ovoid lesion in the right suprasellar area (). Magnetic resonance (MR) imaging showed that the lesion was hypointense in the T2-weighed and T2* gradient-echo (GRE) images and isointense in the T1-weighted images with no vascular abnormalities (). Transfemoral cerebral angiography did not reveal any vascular abnormalities. On the presumptive diagnosis of a hemorrhagic mass such as thrombosed aneurysm, meningioma or CM, craniotomy was performed via a right supraorbital keyhole approach to relieve the mass effect. Once the frontal lobes were retracted, a mulberry-shaped lesion was present at the junction of the right optic nerve and optic chiasm with no connection to the anterior cerebral artery (). The mass was completely removed with no damage to the optic apparatus Histologic analysis confirmed a diagnosis of CM (). The patient made an uneventful recovery after surgery. One year after surgery, the patient's visual symptoms remained unchanged.