A-77-year old woman complained of blurred vision in her left eye. She had undergone cataract surgery with implantation of an IOL 13 years earlier without any complications. She had been diagnosed with presumed sarcoidosis about 15 years earlier, and the mild iritis had been controlled by steroid eyedrops.\nHer BCVA at presentation was 20/30 in the left eyes. The IOP was 15 mmHg and 14 mmHg in the right and left eyes, respectively. Slit-lamp examination revealed no cells in the anterior chamber, but a nasally dislocated in-the-bag IOL was detected (). Slit-lamp examination showed no pseudoexfoliation or vitreous opacities, and ophthalmoscopy showed pigmented chorioretinal atrophy in the periphery. The subfoveal EZ was disrupted in the OCT images.\nShe underwent 27-gauge PPV and the one piece in-the bag PMMA IOL was removed through a 7.5-mm corneal scleral incision without PFCL in September 2017. A CZ70BD IOL (Alcon, Texas, USA) was implanted and fixed with scleral sutures as in the other cases. Thereafter, the PC-IOL was stable (), and the BCVA has remained 20/30 for 2 years without obvious signs of iritis. The peripheral pigmented chorioretinal atrophy and mild macular degeneration could be clearly observed after the surgery (). The visual field was constricted due to the chorioretinal atrophy ().
A 71-year-old male presented with decreased visual acuity in the left eye for 2-month duration. On ocular examination, the best-corrected visual acuity in the right and left eyes was 20/20 (MR−, +0.50Ds -0.50Dc Ax90) and 20/60 (MR-error), respectively. On slit lamp examination, an empty capsular bag with anterior continuous curvilinear capsulorhexis (CCC) was found in the anterior chamber []. In addition, the slit lamp examination showed downward subluxation of a three-piece IOL and some vitreous strands in the anterior chamber in the mydriasis test []. The medical history of this patient was as follows. In 2009, he was diagnosed with a senile cataract (posterior subcapsular opacity type) in both eyes and underwent cataract surgery on both eyes in our clinic. There was no zonular weakness or pseudoexfoliation in the crystalline lens on the preoperative examination. The cataract surgery consisted of one-stitch, a 2.8-mm temporal clear corneal incision, phacoemulsification, and implantation of a three-piece hydrophobic acrylic IOL (TECNIS ZA9003, Abbott Medical Optics, Santa Ana, CA, USA) in the left eye and a one-piece hydrophobic acrylic IOL (TECNIS ZCB00, Abbott Medical Optics, Santa Ana, CA, USA) in the right eye. He denied accidental trauma before or after the cataract surgery. He also had no hereditary genetic diseases, including Marfan syndrome, homocystinuria, spherophakia, and retinitis pigmentosa. However, he stated that he had a habit of rubbing his eyes.\nThree years postoperatively, he presented with decreased visual acuity in the left eye and anterior dislocation of the capsular bag. The IOL was dislocated into the posterior chamber. We planned to reposition the IOL with scleral fixation of the existing lens. The dislocated capsular bag was extracted from the anterior chamber and spread on the corneal surface, and we observed a partial circumferential laceration along the equator of the capsular bag; that is, at the junction of the anterior and posterior capsular bags. The bag had an intact margin of anterior capsular curvilinear capsulorhexis [Fig. and ]. To reposition the IOL, two partial-thickness, limbal-based scleral flaps were created about 1.5 mm from the limbus, under which sclerotomies were performed. The IOL was repositioned through two 1-mm side punctures made using two 23-gauge end-gripping micro-capsulorhexis forceps (Micro Surgical Technology, Redmond, WA, USA), and both haptics were externalized under the scleral flap with Colibri forceps. The haptic ends were tucked into intralamellar scleral tunnels made with a 26-gauge needle. The scleral flaps and conjunctiva were closed with fibrin glue (Tisseel; Baxter, Deerfield, IL, USA; []). The surgery was completed with an anterior vitrectomy. This method, called a glued intrascleral fixation of the posterior chamber of the IOL, was introduced by Kumar et al.[] The uncorrected visual acuity recovered to 20/30 in the left eye [].