A 13-year-old male presented to our hospital after sustaining injuries from a motor vehicle accident (MVA). The patient had bee sitting unrestrained in the front passenger seat of a car when he met with the accident. He had been found unconscious inside the car. Upon arrival in the emergency room, he regained consciousness. As part of the trauma protocol, the patient underwent a full-body CT, which revealed moderate subgaleal hematoma, small hemorrhagic brain contusion, multiple lung contusion, and moderate-size pseudoaneurysm in the descending aorta just distal to the left subclavian artery (LSCA) with associated mediastinal hematoma (Figure ).\nThe CT scan also revealed hepatic, right renal, and splenic lacerations without any evidence of active bleeding. Multiple fractures in the ribs, sternum, right femur, and tibia were also observed. The patient was subsequently admitted to the pediatric intensive care unit (ICU). After a combined surgical and cardiac team discussion, and in the view of other stable lesions, the decision was made to proceed with treating the aortic pseudoaneurysm with impending rupture first. Because of the presence of intracerebral haemorrhage and visceral organ lacerations, the patient was deemed at high risk for systemic anticoagulation required for an operative aortic repair. Available percutaneous endograft sizes were deemed too large for his descending aortic diameter, and the associated potential risk of vessel injury from the large sheath required to implant the endograft removed this option from consideration. The decision was made to attempt percutaneous implantation of a balloon-expandable covered stent across the pseudoaneurysm in the cardiac catheterization laboratory. Under general anesthesia, the right femoral artery access was obtained, and an 11 French (Fr) sheath was inserted. A pigtail catheter was advanced to the transverse aorta, and an angiogram was performed (Figure ).\nThe angiogram revealed a diameter of 14 mm at the mid-aortic arch, descending aorta immediately after the pseudoaneurysm, and aorta at the level of the diaphragm. The length of the disrupted aortic segment was 22 mm, and it was 10 mm away from the origin of the LSCA. Based on the angiogram measurements, a 16x38 mm BeGraft aortic stent, which is a balloon-expandable covered stent, was used successfully. This was followed by post-stenting dilation with short non-compliant 17 x 30 mm Z-Med balloon (B. Braun, Melsungen, Germany). There was no residual aneurysmal filling, and comparison pressures demonstrated no significant gradient from ascending to descending aorta (Figure ).\nThe patient was transferred in stable condition to the pediatric ICU for further management. His post-catheterization course was uneventful. Subsequently, he underwent several orthopaedic procedures until he was discharged home in good condition.
A 67-year-old woman with hepatitis C was found to have a liver tumor located in segment 8 during follow-up computed tomography (CT) for pyogenic spondylitis. She also had a past medical history of cirrhosis due to hepatitis C infection and diabetes mellitus. Laboratory examination showed normal levels of carcinoembryonic antigen, carbohydrate antigen 19–9, and α-fetoprotein but elevated protein induced by vitamin K antagonist-II at a level of 106 mAU/mL. Other laboratory studies showed the following: total bilirubin 0.6 mg/dL, albumin 3.2 g/dL, prothrombin time 13.7 s, aspartate aminotransferase 28 IU/L, alanine aminotransferase 22 IU/L, blood urea nitrogen 16 mg/dL, and creatinine 0.54 mg/dL. The Child-Pugh score was grade A (6 points), and the indocyanine green retention rate at 15 min was 16.6%. A diagnosis of hepatocellular carcinoma (HCC) located in segment 8 was indicated by CT (Fig. ), magnetic resonance imaging (Fig. ), and ultrasonography. The tumor was attached to the MHV. CT scans also showed multiple varices (Fig. ). MHV resection was necessary to obtain sufficient surgical margins. Central bisectionectomy (S4, S5, and S8 resection) was suggested but could not be tolerated because of her poor liver function and low future liver remnant volume, which preoperatively was only 56% by CT volumetry. Therefore, we decided to perform partial hepatectomy with MHV resection. Preoperative CT scans revealed the existence of duplicated IVC (Fig. ), so we chose to use the left IVC as the graft for reconstructing the MHV.\nThe surgery started with an inverted L-shaped incision. Intraoperative ultrasonography confirmed a 3.5-cm tumor that was touching the MHV. After mobilization of the right lobe, the liver parenchyma was transected, preserving the right hepatic vein and right anterior glissonian pedicle. A 3-cm portion of the MHV was resected around the tumor to obtain sufficient surgical margins. Immediately before resecting the MHV, we harvested the left IVC for use as the graft. After incising the retroperitoneum beside the Treitz ligament, the LRV, which had ramified from the IVC, was detected. The left IVC, which had ramified 3 cm peripherally from the origin of the LRV, was then isolated and harvested for 5 cm from the confluence of the LRV. The cut ends of the left IVC were closed with 4–0 polypropylene running sutures. The venous graft was anastomosed first to the proximal end of the MHV and then to the distal end using 6–0 polypropylene (Fig. ). A good Doppler flow through the graft was confirmed after the reconstruction. The operation required 394 min, and the estimated blood loss was 1,894 mL.\nMacroscopic evaluation showed a 35-mm bile-stained tumor (Fig. ). Histologic examination revealed moderately differentiated HCC without direct invasion of the MHV and a tumor-free surgical margin (Fig. ). The tumor was classified as stage II (T2N0M0) according to the TNM staging system for HCC. The patient recovered uneventfully and was discharged on postoperative day 12.\nThe patency of the reconstructed MHV was confirmed postoperatively by ultrasonography and dynamic CT (Fig. ). Laboratory examination 2 years after surgery showed normal levels of total bilirubin (0.6 mg/dL), albumin (4.0 g/dL), aspartate aminotransferase (24 IU/L), alanine aminotransferase (24 IU/L), prothrombin time of 13.5 s, blood urea nitrogen (15 mg/dL), and creatinine (0.46 mg/dL).\nNew lesions were found in the left lobe of the liver 4 months after surgery and in the right lobe 26 months after surgery. Both lesions were treated using transabdominal catheter embolization.