The chief complaint for this 29-year-old woman was impaired vision in her left eye. At the initial visit for this this patient, a photograph of her left fundus showed white dots (Figure B) with hyperautofluorescence on FAF (Figure D), while OCT revealed a disrupted or irregular photoreceptor inner outer segment (IS/OS) junction (Figure F) indicated by the arrows. At 1 month after her initial visit, this patient complained of metamorphopsia in her right eye. FP showed that there were white dots in the posterior pole (Figure A), while FAF images obtained using FAF revealed hyperfluorescence in the area surrounding the optic disc (Figure C). OCT showed a disrupted or irregular photoreceptor IS/OS junction in both eyes (Figure E and F) indicated by the arrows. At 4 months after her initial visit, there was almost complete improvement of the metamorphopsia in her right and left eyes. In addition, OCT indicated there was ongoing restoration of the IS/OS line in both eyes (Figure A and B). Although the deviation map of ganglion cell analysis showed there was a decrease of the GCL + IPL thickness in both eyes at her initial visit, her right eye was not affected. During the 4 months of follow-up, the deviation map (Figure C~J) did not indicate any recovery of the decreased GCL + IPL thickness. Signal strengths of all the Cirrus OCT measurements were 10/10.
A 48-year-old female presented to us with blurring and distortion of vision in OS since 2 weeks. Her BCVA was 6/6; N6 in OD and 6/24;N10 in OS at presentation. Fundus examination showed an extrafoveal CNVM. Based on multimodal imaging including SD-OCT and FFA, patient was diagnosed to have type-2 CNVM with subfoveal choroidal thickness of 505 μm. She was also treated with antiVEGF monotherapy on pro re nata (PRN) basis and had multiple recurrences over 5 years. Interestingly, she developed FCE at the site of resolved CNVM scar, during 23 months of treatment, which progressively increased from 85 μm to 154 μm in depth over the subsequent follow-ups. We report this as a case of acquired FCE secondary to CNVM [].