A 60-year-old woman who developed intermittent diplopia underwent MRI scan revealing a tentorial meningioma as well as aneurysms of the posterior communication artery, right vertebral artery, and peripheral superior cerebellar artery. The tentorial meningioma tumor was located in the posterior aspect of the cerebellopontine angle and causing compression of the brainstem along the course of the 4th cranial nerve, and the complex, partially thrombosed dissecting aneurysm of the SCA was situated just proximal to the entry of the 4th nerve into the free edge of the tentorium. Angiography revealed a partially thrombosed wide-necked aneurysm not amenable to endovascular intervention. Microsurgical clipping and simultaneous tumor removal were recommended.\nA combined subtemporal, presigmoidal approach was performed. A wide bone removal of the middle cranial fossa was necessary to reach the tentorial incisura allowing exposure of the tumor. Due to the associated brainstem compression from the tumor, the meningioma was debulked first, and then the aneurysm was addressed. Interestingly, on reaching the SCA there was obvious hemosiderin staining of the arachnoid surrounding the aneurysm dome, indicating a previous rupture. During microsurgical dissection, it was confirmed that this aneurysm was thick-walled and partially thrombosed [Figure and ]. The aneurysm was reconstructed with a single clip placed well up on the aneurysm dome to avoid compromise of the parent artery. Intra-operative angiogram demonstrated complete obliteration of the aneurysm with normal flow through the SCA and distal branches. Post-operatively, apart from transient hyponatremia and hyperglycemia, the patient did well without complication.
A 56-year-old male patient came to the emergency room due to a right-sided occipital headache and vertigo, which suddenly began three days before the visit, and a symptom of tilting to the right, which began one day before the visit. There was no history of an external injury within the past one week. The patient did not have any underlying diseases, history of migraines, or risk factors for stroke, except for smoking. He also did not have a history of taking antiplatelet agents. When the patient visited the emergency room, his blood pressure was 160/100 mmHg, the pulse rate was 62/min, breathing rate was 19/min, and body temperature was 36.5 °C. The results of a neurological examination showed lateropulsion to the right side, hypoesthesia on the right side of the face, ataxia in the right upper and lower limbs, and the NIH stroke scale was 2. The results of serological studies and electrocardiography were normal.\nThe diagnosis was determined to be an acute ischemic stroke because the diffusion weighted imaging (DWI) of MRI revealed a high signal intensity area in the right lateral medulla. CT angiography (CTA) was taken first at 3 days after the symptom occurred. HR vw-MRI was taken a day after (at the 4th-day). The right PICA was not observed in the first CTA, and partial spontaneous recanalization was observed on the time-of-flight MR angiography (TOF-MRA), which was taken one day after. Moreover, the sagittal 3D T1 weighted HR vw-MRI showed a T1 hyperintense intramural hematoma in the right proximal PICA (Fig. ). However, a T2 weighted 3D curved MPR image revealed the luminal dilation and intimal flap in the right proximal PICA (Fig. ). The patient was diagnosed with spontaneous isolated PICA dissection on the basis of the neurological symptoms and imaging. He was treated with antiplatelet agents and the neurological symptoms of the patient improved during course of admission.