A 70-year-old woman with hypertension, hyperlipidemia, and a history of smoking presented with a peripheral fusiform SCA aneurysm found incidentally during a routine MRI investigation due to “black out” symptoms. The aneurysm was located 2 cm from the SCA origin, and measured 7 mm in maximal diameter [Figure and ]. After a thorough discussion of treatment options, the patient elected to undergo a combined subtemporal, presigmoidal approach. On the lateral aspect of the brainstem, the aneurysm was exposed along with the parent artery and two major efferent branches. One of these branches, the largest one, was superiorly directed and a double, smaller branch, presented inferiorly around the aneurysm fundus. It was necessary during microsurgical dissection to detach the aneurysm dome from the tentorial undersurface as well as from the 4th cranial nerve. Multiple clips were used to reconstruct the aneurysm without intra-operative complication. Intra-operative angiogram confirmed reconstruction of the fusiform aneurysm with preservation of distal flow [Figure –]. The patient tolerated the procedure well and was extubated and returned to the intensive care unit in stable condition. She was discharged home on the fourth post-operative day without new deficit.
A 38-year-old male patient came to the emergency room due to vertigo and vomiting that had started suddenly one day before the visit. The patient complained of a left-sided occipital headache that began two days before the visit. He had no history of trauma within 1 week prior to admission. The patient did not have any underlying diseases, history of migraines, or risk factors for stroke except for smoking and alcohol consumption. His blood pressure was 170/100 mmHg, the pulse rate was 70/min, breathing rate was 20/min, and body temperature was 36.5 °C. A neurological examination showed ataxia in the left upper extremity and the NIH stroke scale was 1. The results of serological studies and electrocardiography were normal.\nThe patient was determined to have an acute ischemic stroke because the DWI of MRI showed a high signal intensity area in the left PICA territory. CTA was taken at 2 days after the onset of a headache and at 1 day after the onset of neurological symptoms. Moreover, HR vw-MRI was taken at 6 days after the onset of a headache and at 5 days after the onset of neurological symptoms. Although the left PICA was not observed on the TOF-MRA, other vessels including the left vertebral artery were normal. An intraluminal high signal intensity and mild dilation of the left proximal PICA was revealed on routine 3D HR vw-MRI, but pathognomic signs of dissection was not seen. Curved MPR image reconstructed from 3D T2 weighted HR vw-MRI showed dilation and an intimal flap in the left PICA origin and post dilation stenosis in the left proximal PICA (D). (Fig. ). The patient was diagnosed with spontaneous isolated PICA dissection based on the neurological symptoms and imaging. The patient was treated with antiplatelet agents and discharged after his neurological symptoms improved.