A 48-year-old female presented to us with blurring and distortion of vision in OS since 2 weeks. Her BCVA was 6/6; N6 in OD and 6/24;N10 in OS at presentation. Fundus examination showed an extrafoveal CNVM. Based on multimodal imaging including SD-OCT and FFA, patient was diagnosed to have type-2 CNVM with subfoveal choroidal thickness of 505 μm. She was also treated with antiVEGF monotherapy on pro re nata (PRN) basis and had multiple recurrences over 5 years. Interestingly, she developed FCE at the site of resolved CNVM scar, during 23 months of treatment, which progressively increased from 85 μm to 154 μm in depth over the subsequent follow-ups. We report this as a case of acquired FCE secondary to CNVM [].
A 46-year-old male patient presented to us with blurring of vision OS since 2 months. His BCVA was 6/6, N6 in OD and 6/12, N8 in OS at presentation. Fundus examination of OS showed an irregular elevated, yellowish-white choroidal lesion with welldefined geographic borders involving the inferior macula with areas of subretinal hemorrhage at and above the fovea suggestive associated choroidal neovascular membrane. SD-OCT showed a dense hyper-reflective choroidal mass with significant widening of the choroidal layers. B-scan ultrasonography revealed high-spike echoes through the lesion suggesting choroidal osteoma. FFA in that area showed staining conforming to the diagnosis of choroidal osteoma with a secondary CNVM. Patient underwent treatment with multiple antiVEGF injections and standard fluence photodynamic therapy (PDT). The patient developed an acquired FCE after 17 months of treatment [].