human
The task is to decide the Hallmarks of Cancer (HOC) taxonomy of the article based on its abstract. The input is an abstract text. There are 10 topics you will need to decide whether the article is related to. Topics: sustaining proliferative signaling, evading growth suppressors, resisting cell death, enabling replicative immortality, inducing angiogenesis, activating invasion and metastasis, genomic instability and mutation, tumor promoting inflammation, cellular energetics, avoiding immune destruction. The output should be topics from the above 10 topics, that are related to the input article. Please note one article can be related to multiple topics. Output format: provide a semicolon-separated list of relevant topics.
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    INPUT: Cellular senescence can be a functional barrier to carcinogenesis . We hypothesized that inflammation modulates carcinogenesis through senescence and DNA damage response ( DDR ) . We examined the association between senescence and DDR with macrophage levels in inflammatory bowel disease ( IBD ) . In vitro experiments tested the ability of macrophages to induce senescence in primary cells . Inflammation modulating microRNAs were identified in senescence colon tissue for further investigation .
    OUTPUT:

agent
enabling replicative immortality
The task is to decide the Hallmarks of Cancer (HOC) taxonomy of the article based on its abstract. The input is an abstract text. There are 10 topics you will need to decide whether the article is related to. Topics: sustaining proliferative signaling, evading growth suppressors, resisting cell death, enabling replicative immortality, inducing angiogenesis, activating invasion and metastasis, genomic instability and mutation, tumor promoting inflammation, cellular energetics, avoiding immune destruction. The output should be topics from the above 10 topics, that are related to the input article. Please note one article can be related to multiple topics. Output format: provide a semicolon-separated list of relevant topics.
    ###
    INPUT: Cellular senescence can be a functional barrier to carcinogenesis . We hypothesized that inflammation modulates carcinogenesis through senescence and DNA damage response ( DDR ) . We examined the association between senescence and DDR with macrophage levels in inflammatory bowel disease ( IBD ) . In vitro experiments tested the ability of macrophages to induce senescence in primary cells . Inflammation modulating microRNAs were identified in senescence colon tissue for further investigation .
    OUTPUT:
