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Worldwide, 40% Are Employed Full Time for an Employer
New measure of good jobs is more clearly related to GDP than unemployment
by Jon Clifton and Jenny Marlar
WASHINGTON, D.C. -- Forty percent of the global workforce was employed full time for an employer in 2009 and 2010, according to Gallup surveys from 129 countries and areas. Nineteen percent were underemployed, including 7% who were unemployed.
These are the first results Gallup has released from its newly developed global unemployment measures. Based on a series of employment questions Gallup asks respondents in surveys conducted at least once per year in most countries, Gallup quantifies the percentage of the global, regional, and country-level workforce that is employed full time for an employer, underemployed, and unemployed. Together, these metrics paint an unprecedented picture of the state of employment worldwide based on data that are easily comparable across countries. The results reported in this article represent an aggregate of data collected in 2009 and 2010.
Gallup's Employed Full Time for an Employer Index quantifies the percentage of workers in good jobs, rather than subsistence jobs that do little to raise individuals out of poverty or contribute to the country's formal economic output.
The Employed Full Time for an Employer Index has a strong, positive relationship with GDP per capita, meaning that countries with a higher percentage of workers employed full time for an employer tend to have higher GDP per capita. Gallup's Underemployment Index also has a strong negative relationship with GDP per capita. That is, countries with high underemployment tend to have lower GDP per capita. When examined across countries, Gallup research has found no relationship between unemployment rates and GDP per capita.
In many developing countries, unemployment is often relatively low, particularly compared with that in developed economies, because it takes into account people who are doing whatever work they can find to get by or are self-employed in subsistence jobs. Economically developed countries are more likely to have larger percentages of the workforce employed for an employer, but these percentages vary by country.
Regionally, sub-Saharan Africa has the smallest percentage of its workforce working full time for an employer, with nearly one in five reporting that they have a full-time job with an employer. Asia follows, with more than one-third of its workforce working for an employer. Countries in the former Soviet Union region have the largest percentage of the workforce working full time for an employer.
Underemployment is highest in sub-Saharan Africa. The relatively low number of individuals working for an employer and high number of underemployed underscores the economic woes in the region and that good jobs are needed.
Gallup's Underemployment Index captures traditional unemployment, as well as those who are employed part-time but seeking additional work. Traditional unemployment measures classify an individual as employed if he or she works for even a few hours a week. However, this does not tell the true story of employment status. The availability of part-time work may disguise an underlying lack of full-time jobs, which goes undetected with unemployment measures. Gallup's underemployment measure is a truer depiction of the percentage of the workforce that is not working at its desired capacity.
Global leaders today are making job creation a top priority. But until now, they did not have the measures they needed to determine whether they are creating good jobs.
Gallup's new Employed Full Time for an Employer Index measures the availability of quality jobs, and Gallup's Underemployment Index provides a true picture of the global workforce working at its desired capacity. Worldwide leaders can use these measures to track their progress toward increasing the number of quality jobs available, the percentage of workers employed in those jobs, and, in turn, their nation's GDP.
Results are based on telephone and face-to-face interviews with approximately 1,000 adults, aged 15 and older, per survey administration. Interviews were conducted in 129 countries throughout 2009 and 2010. In many countries, the data have been aggregated. For results based on the total sample of national adults, one can say with 95% confidence that the maximum margin of sampling error ranged from a low of ±1.4 in India to a high of ±4.7 in Latvia. The margin of error reflects the influence of data weighting. In addition to sampling error, question wording and practical difficulties in conducting surveys can introduce error or bias into the findings of survey data.
Multi-country All Gallup Headlines Credit Crisis Economy Global Employment Good Jobs Jobs Leadership Social and Policy Issues Underemployment Unemployment Workplace Africa Americas Asia Asia Pacific Europe Former USSR Latin America Muslim World Sub-Saharan Africa World
Gallup 2016 Global Great Jobs Report
Across most of the world, the percentage of adults with great jobs rarely tops 10%
Americans Want Congress, Obama to Tackle Economic Issues
Americans' top priorities for the president and Congress this year are domestic issues, particularly those related to economics -- the economy, unemployment, and the federal budget deficit. Other high public priorities are government corruption, education, healthcare, and terrorism.
In U.S., Jobs Are Top Problem, While New High Cite Deficit
Unemployment is the top-ranking issue when Americans are asked to name the most important problem facing the country today. Mentions of "the economy" in general have been trending downward, while those for the federal budget deficit have increased to 12%, the highest in Gallup trends over the past decade.
Gallup Finds Unemployment at 9.6% in December
Unemployment, as measured by Gallup without seasonal adjustment, increased to 9.6% at the end of December -- up from 9.3% in mid-December and 8.8% at the end of November. Broader U.S. underemployment worsened to 19.0% in December -- up sharply from 17.2% at the end of November.
by Jon Clifton
Nearly 150 million people -- or 4% of the world's adult population -- want to move to the U.S.
Gallup https://news.gallup.com/poll/145595/worldwide-employed-full-time-employer.aspx


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Online Betterhelp Contact – Mental Health Challenges
Finding Betterhelp Contact…
Remote, text-based counselling is growing in popularity in the UK. The physician app Babylon provides therapy to 150,000 active users, while PlusGuidance, an online counselling service, has 10,000 users. The US-based service BetterHelp likewise has actually 150,000 registered UK users (though not all are active). Talkspace, another online treatment platform, reports it has 500,000 signed up users worldwide, with the majority of in the US.
Online training advises therapists on everything from utilizing emojis to preventing misinterpretations. They likewise require to protect clients' individual information– an issue that has triggered controversy in the US, where huge online treatment platforms have come under the spotlight.
Buckley said clients must check services' personal privacy policies before signing up. "Not all online counselling websites utilize professionally trained therapists or abide by a principles policy, so ask your GP for a suggestion in the very first circumstances. Just like all sort of services and assistance, what works for a single person may not work for another person," he said.
Marc Bush, primary policy adviser at Young Minds, said that while online counselling services are important, "they shouldn't change in person therapy with a qualified professional. If a young person is having a hard time, we would encourage them to speak to their GP in the very first circumstances, or to call a recognized service like The Mix, Childline or the Samaritans.".
For Rackham, who has generalised stress and anxiety condition, online counselling wasn't the right fit. "I felt it was near difficult for the therapist to actually get a sense of the problems I was handling, as all they had to go from was my typed-out words. I believe I understood after that online session how essential interpersonal interaction was.
" I'm a big fan of using technology in all locations of my life as a solution to everyday issues. I have apps for everything, however when it concerns mental health, you have to choose how technology plays a role in your healing extremely carefully.". Betterhelp Contact
Rather, the app prides itself on having licensed therapists and mental health experts readily available to assist individuals through text, call or video chat. That's what lots of YouTubers who have actually accepted sponsorships from the business typically say in their own videos, where they speak on the tensions in their individual lives and feelings bordering on anxiety or depression. Bobby Burns, Elle Mills, Philip DeFranco, Heath Hussar, Boogie2988, Shane Dawson and ChandlerNWilson are all creators who have Betterhelp Contact sponsors now.
Many of these creators have spoken about mental health issues in the past, but as burnout ends up being a larger subject within the neighborhood– and mainstream world– sponsorships including BetterHelp have actually increased, in spite of the app not being precisely what the creators are touting.
Online Betterhelp Commercial Girl – Mental Health Challenges
Online Betterhelp Vs In Person Therapy – Mental Health Challenges

redpajama_set_name: RedPajamaCommonCrawl


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Final Fantasy 1 Walkthrough - Chapter 7
Before you come to this dungeon, make sure you prepare yourself by purchasing at least 20 Gold Needles, 40 Antidotes and one or two Cottages to save us some trips to town in the future. Navigating to the top of this tower is a piece of cake, 1F to the second floor all you have to do is run a few feet to get there.
2F to the Teleport Pad is a little less straight forward but still not that difficult. You only have to go out of your way if you want the Treasure Chests that are in the central portion of the tower on each floor. As per usual it's recommended you go into the dungeon and grab all the loot then leave and rest up only to return and finish the dungeon the second time.
Tower of Mirage Treasure Chests 1F: Healing Helm, Vorpal Sword, Aegis Shield, Tent and 24960 Gil
Tower of Mirage Treasure Chests 2F: Thor's Hammer, Cottage, Dragon Mail, Sunblade, 58,985 Gil
On the third floor of the Tower of Mirage you'll find the Teleport Pad which can only be accessed if you have the Warp Cube which I told you to get earlier in Chapter 6. Assuming you have the Warp Cube, step on the Teleporter to be taken to the Flying Fortress.
The Flying Fortress is the second to last dungeon in the game. It's also the dungeon with the hardest enemy in the entire game, Warmech. But he only spawns on the same floor as Tiamat so all in all not too much to worry about. For the first floor of the Flying Fortress, go North to proceed to Floor 2. Every other direction you'll find Treasure Chests.
Flying Fortress 1F Treasure Chests: Venom Blade, Potion, 21950 Gil, Protect Ring, Healing Helm, 11900 Gil
Second floor in The Flying Fortress is split into 8 different paths. The southern most path takes you to floor 3 while the northern most path will take you back to floor 1. Every other direction has Treasure Chests. Of course, I recommend going each direction and grabbing every Treasure Chest you can.
On the second floor the most important item for you to grab is the Adamantite which is a little crystal in the southwestern room. It's used to craft the second best sword in the game when you give it to the Smith in Mt. Duergar.
Flying Fortress 2F Treasure Chests: Diamond Gloves, Diamond Shield, Ribbon, Black Robe, White Robe, Cottage, Mythril Helm, Adamantite*
Flying Fortress 3F Treasure Chests: Protect Ring, Potion, 14450 Gil, Gold Needle, Sasuke, 7550 Gil, Gold Needle, Shirt, 24035 Gil and Protect Cloak
As for the Flying Fortress 4F, things can get a bit confusing since the area goes on and on without many clues as to where you are. There's a really good map on Gamefaq which you can find by following the link; incase they ever take that picture down or if you don't feel like clicking on it... From the Flying Fortress 4F portal, you go down twice and to the right twice. In other words, the portal to the Flying Fortress 5F is southeast from where you the Flying Fortress 3F portal is at.
Once you arrive on the Flying Fortress 5F, Tiamat is just ahead. Much like every other boss battle you'll want to cast any available buffs you have on your team. For Tiamat I recommend NulBolt since he uses Bolt to damage your entire party as well as Shld2 since his melee attacks can be pretty hard hitting. Tiamat is pretty resistant to Magical attacks so I'd recommend having your Black Mage use Haste/Steel on whomever they can rather than attack. All in all, Tiamat isn't too difficult.
After beating Tiamat, collect the Wind Crystal and leave the dungeon via the portal. As aforementioned it's time to head to Mt. Duergar which is where we'll have the Adamantite forged into the Excalibur. The smith that you need to talk to is in the northern portion of Mt. Duergar. If you forget where Mt. Duergar is, it's just a tad east of Melmond.
Once you've gotten the Excalibur in hand it's time to head to the final dungeon of the game, which is actually a place we've already been! Remember one of our very first dungeons, Temple of Chaos? That's where we're heading! Inside of this dungeon, where we fought the first boss Garland you'll be able to use the power of all four crystals and go back in time.
The dungeon you find yourself in is a giant dungeon with multiple floors and a few puzzles for you to figure out. There's some, but not much, treasure in this dungeon and my guide below covers all of it.
Temple of Chaos 1F Staircase down to 2F: In the southeastern most point of the room.
Temple of Chaos 2F Staircase down to 3F: Staircase is immediately to the right of where you come up.
Temple of Chaos 3F Ladder up to 2F: In the southern most central portion of the area there'll be a door leading to a room.
Inside of that room on the third floor you'll find two Treasure Chests with about 110,000 Gil and a stone tablet similar to what we saw in Terra Cave. Except this time we don't use the Earth Rod on it, we're going to use the Lute which we got way earlier in the game. This location is shown in the screen shot below.
After taking the ladder down you'll find yourself in the room on the 2F we passed earlier that was between the two staircases. We're going to be heading back up to 1F next so directions may be confusing; keep this in mind!
Temple of Chaos 2F Staircase down to 1F: Staircase is in the northwestern portion of this area.
Temple of Chaos 1F Staircase down to B1: West; linear path
Temple of Chaos B1 Staircase down to B2: Linear path but you have to go North, East then back South to find the staircase.
Temple of Chaos B1 Boss fight at Staircase down to B2 Lich (Weak to Fire & Dia)
Don't worry about how much MP you use while fighting the Lich, so long as you save enough MP for 1 Warp2 spell on your White Wizard. We're going to do this dungeon in waves since it's too darn long to tackle all at once. After you defeat the Lich take the staircase down.
You can see the Staircase down to the next floor right when you get to this one but you'll need to take a really convoluted way there. Go north, east then south to find your way here. Also before you head up the Staircase, which is guarded by another boss, there's Treasure Chests to the south.
Temple of Chaos B2 Treasure Chests: Protect Cloak, Sasuke and Protect Ring, 26,000 Gil
Marilith is just like she was before in Mt Gulg. She's weak to Melee damage first and foremost, Stun, Sleep and Darkness abilities next but almost all other magic is ineffective against her. Use Shld2 and NulFire on your party and let her have it with your melee attacks. She went down in a few attacks for me since I have some high damage dealing melee characters.
After Marilith is defeated, take the stairs down and you'll want to head to the southeast corner of this area to find the next staircase down... and another boss - Kraken!
Kraken is pretty easy, much like he was when you got the Water Crystal. Cast Shld2 on your party once (saving MP for the next fights) and wail away on the Kraken with your strongest Melee attacks. He'll go down easily in a few turns.
Take the stairs down and on this floor travel to the southeastern portion of the area to find the best 1h sword in the game, Masamune. At this point in time it's recommended that you Warp out of the dungeon, rest up, equip the Masamune and then return to the dungeon to complete it.
You'll need to battle every boss that we fought on the way to the Masamune again which if you want to avoid that, I recommend you just continue on to the end boss. He's really not all that difficult if you're at least level 32 on your team and save some high level spells MP for him.
Whatever your choice is, continue onto the next part of my guide to learn more about the final boss and how to defeat him. The next page in this guide also gives you a quick list of how to navigate each floor on the Temple of Chaos instead of going into a lot of detail like on this page. Check it out!
Continue to Final Fantasy 1 Walkthrough - Chapter 8 (Temple of Chaos final boss)
My party end of Chapter 7.


Slowing Global Growth concerns have been hitting some markets more than others...German bond yields, for instance, have been falling steadily since early October...they are now nearing their historic lows made in the Brexit summer of 2016...while US equity markets, crude oil, copper and the commodity currencies have rallied from their oversold Christmas lows on the "Fed is done tightening" idea.
My macro view for the next few months is that slowing global growth concerns become the BIG story across asset classes (overshadowing the "Fed is done tightening" idea) with market sentiment becoming increasingly risk adverse which produces 1) lower bond yields, 2) wider credit spreads, 3) lower share prices, 4) lower commodity prices, and, 5) a stronger USD. Gold made its low for the year in mid-August 2018...just as the USD was making a 14 month high...and has rallied ~$150 since. Gold often trades as the "mirror image" of the USD but I think it's also benefited from a "haven" bid just like the German bond.
My favorite trade the past couple of weeks has been long US 10 year treasuries as they rally on slowing global growth. The Eurozone bond rally helps "pull" US bonds higher and treasuries may also catch a bid if the stock market rolls over.
This week I bought S+P puts thinking that the rally from the Christmas lows may be rolling over.
I'm expressing my bearish view on commodities with short positions in WTI and copper. Both of these commodities (and the COMDOLS) have been strongly correlated with the bellwether US stock market since the turn of the year and may be rolling over.
I'm expressing my bullish view on the USD with short positions in CAD and NZD...they "rolled over" a day or two ahead of the S+P.
I trade futures and options and I'm quick to abandon my positions if they are not working. My 40+ years of experience trading these markets tells me that most of my trades will result in small profits or losses which more or less offset each other. I definitely want to avoid taking any big losses and hopefully I'll occasionally get a big winner.


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Periodontal Disease and Systemic Disease
The Relationship Between Periodontal Disease and Systemic Disease
Abdulrahman B Al-Awadhi – Ministry of Defense*
Hamad Al-Yaseen – Ministry of Interior*
Abdulaziz Al-Duwaisan (dr.bozeeez@gmail.com) – Ministry of Interior*
Ali E Al-Kandari – National Guard of Kuwait*
*Postgraduate student, Department of Prosthodontics & Medical materials, University Hospital Tuebingen, Tuebingen, Germany.
Dental News December Issue 2019
For many years, medical and dental researchers have been focusing on their own fields of study, conducting researches in medicine related to the general well-being and the health of the oral cavity, respectively. However, the strong relationship between oral disease and systemic conditions has been increasingly studied and proved through the past few decades. Recent studies have provided obvious proof that oral hygiene and systemic well-being are directly related.
The recent findings are strongly suggestive of the presence of an association between periodontal disease and systemic diseases such as osteoporosis, cardiac disease, low birth weight, poor control of diabetes mellitus, and pregnancy complications. The numerous efforts of the researchers from both fields have brought about many breakthroughs in our understanding of the etiological and patho-physiological mechanisms underlying the link between the chronic inflammation present in dental disease and other systemic conditions.
Consequently, there is hope that the important findings from these
researches may help dentists to greatly improve the current guidelines in treatment of periodontal disease, to prevent the negative consequences of oral disease on systemic health. Therefore, researchers must continue to not only establish the link between oral health and systemic conditions but also to recognize the positive effects of eliminating periodontal infections on the success of the treatment of systemic disease.
Key words: Periodontitis, Systemic disease, Osteoporosis, Diabetes Mellitus, Cardiac disease, Pregnancy complications.
Chronic Periodontitis
Periodontal disease is the condition resulting from accumulation of bacteria in the tissues around the teeth which trigger a chronic inflammatory response from the body. The chronic invasion of the gums by bacteria results in bone destruction and weak attachment between the teeth and the gum. There are several types of periodontal disease, from gingivitis which is the mildest form to periodontitis which is severe and difficult to treat.
Several risk factors predispose to the development of periodontitis, such as stress, alcohol consumption, smoking, the normal process of aging and conditions that affect the immune system response such as diabetes and (HIV) infection. 1
Gingivitis is the most common form which affects 75 percent of adults in the United States. It involves red inflamed gums with frequent bleeding. There are more severe types that affect about 40 percent of the population in the United States. In these types, there are more accumulations of pathogens which lead to stronger inflammatory process and more severe symptoms.
The diagnosis of periodontal disease depends on probing depth as an accurate measurement of the grade of inflammation. A depth less than 2 mm of the periodontal pocket is not considered periodontitis.
If the depth of periodontal pockets is 4 mm or more, periodontitis is confirmed. Periodontal pocket depths of 6 mm or more refers to severe periodontitis. 2
The treatment of periodontal disease is usually effective. Such treatments are aimed at eliminating the invading bacteria from the body, ameliorating the inflammatory process, and removing the predisposing factors to prevent further recurrences of the same condition in the future. 3
The main line of treatment of periodontitis is antibiotics that are usually broad spectrum to target most types of bacteria. Antibiotic therapy together with maintaining good oral hygiene, is usually successful in eliminating the infection. However, If the periodontal pockets are not removed, then surgical treatment is indicated to protect against further bone destruction and teeth loss. 4
Bone loss occurs in both osteoporosis and periodontitis. A decrease in bone mass occurs because of the increased resorption of bone compared to bone formation, leading to demineralization of bone and finally osteoporosis. 5
Similarly, in addition to weakened attachment of the soft tissue to the teeth, resorption of bone is a feature of periodontal disease: specifically, the alveolar bone.
Because of this common pathological mechanism that is shared between periodontal disease and osteoporosis, researchers have tried to establish the links between the two diseases to recognize the possible effect of having one as a predisposition to the other.
The underlying pathology in the case of increased resorption of bone may be explained by increased local and systemic osteoclastic activity, or by the local action of inflammatory cytokine on the bone tissue. 6 Increased resorption by osteoclasts is characteristic to chronic inflammations as in periodontitis.
In chronic inflammation, the T-lymphocytes express Receptor Activator of Nuclear Factor-kB Ligand and hence it is possible that direct contact between T-lymphocytes and precursor cells to monocytes is involved in the formation of osteoclasts. 7
Osteoprotegerin (OPG) released by stromal cells and osteoblasts inhibits the activation of mature osteoclasts. B-lymphocytes also have a role in formation of osteoclast, either by working as osteoclast progenitor cells or by expressing RANKL. 8
Nagasawa et al., 9 stated that «OPG mRNA was stimulated by P. gingivalis and A. actinomyecetemcomitans, indicating that OPG released by the action of LPS might have a role in the process of osteoclast formation in periodontal disease. The findings linking OPG and periodontitis is supported by other studies involving gram negative bacteria. 10 Because the infection due to these pathogens is usually transient, exposure to periodontal infection may stimulate the activation of RANKL and the activity of osteoclasts, leading to development of osteoporosis in patients with chronic periodontitis.
Another major mechanism of osteoporosis in women is oestrogen deficiency. 11 Oestrogen, whether directly or indirectly, has a modulatory action on cytokines that work as regulators of bone metabolism and also modulators of the host inflammatory reaction, such as IL-1 alpha and beta, TNF-alpha, and macrophage colony-stimulating factor (M-CSF). Therefore, oestrogen deficiency leads to an increased number of osteoclasts, because of the same cytokines which inhibit formation of osteoblasts. The disturbed balance of bone metabolism will result in decreased bone mineral density (BMD).12
Chronic periodontitis also triggers the host inflammatory reaction, thereby recruiting cytokines and leading to formation of osteoclasts and therefore stimulating absorption of bone. High levels of some of these cytokines, such as IL-1 beta, TNF-alpha, and IL-8, were found in the inflamed gingival tissue in patients with periodontal disease, with concentrations that can cause absorption of alveolar bone. 13
Therefore, numerous studies have found a significant positive link between chronic periodontitis and oestrogen deficiency.
Another research study in Japan focused on the link between oral health and bone mineral density; these findings proved that chronic periodontitis and teeth loss after menopause are directly related to decreased BMD in post-menopausal females, and the deficiency in oestrogen is the suggested underlying mechanism. 14
In a related study, Wactawski-Wende et al., 15 stated that "a strong and consistent association between alveolar crestal height (ACH) and osteoporosis was found through measurements of bone density and ACH in postmenopausal women, with icreasing age as an important modifier of that association".
Despite the big achievements made in understanding the relationship between periodontal disease and osteoporosis, more efforts are required to clarify the link.
The recognition of this link might provide useful warnings for the risk of osteoporosis, as well as important applications in treatment.
Chronic periodontitis and diabetes mellitus
The relationship between periodontal disease and
diabetes mellitus is bidirectional. As it was proved that patients with diabetes are more liable to develop one or more types of periodontal disease, recent data
suggests that periodontal disease can lead to the increased
probability of suffering from endocrinal disturbance,
including diabetes mellitus. 29
Chronic periodontitis could lead to bacterial spread to blood and various body organs, and thereby activate cells of the immune system. The stimulated cells trigger inflammatory active substances (cytokines) which have a catabolic action all over body systems. 30
Moreover, the chronically elevated levels of inflammatory cytokines in the body can lead to damage of the beta cells of the pancreas, leading to a decrease in the secretion of insulin. This process can lead to development of diabetes in patients with no family history, obesity, hypercholesterolemia or other risk factors. 31
Due to the possible effect of periodontal disease on the development of insulin resistance and glucose intolerance and finally overt diabetes mellitus, studies have suggested that preventive dental and diabetes measures, in addition to regular dental and diabetes evaluation and follow up, are necessary to break this link between the two conditions. 32
Chronic periodontitis and malignancy
Many research studies have indicated that chronic periodontitis plays a key role as an independent factor for the development of head and neck squamous cell carcinoma (HNSCC), with specific characteristic site in the oral cavity. Also, it was found that periodontitis predisposes to carcinoma of the oropharynx and the larynx. 33
Michaud et al., 34 found a strong relationship between the chronic presence of periodontal disease and high risk of being diagnosed with lung, renal, pancreatic, and haematological tumours. These relations are the results of a prospective cohort study and they were confirmed in various studies with modification of risk factors, including cigarette smoking and patients' socioeconomic status. 34
However, the strongest evidence was found in the research studies of the relationship between oral and oesophageal cancers and chronic periodontal disease.
A relationship was proven between gastric and pancreatic cancers and periodontal disease in vast majority of the reviewed papers, although tumours of the lungs and the blood and other tumours were less proven or there were insufficient studies to establish a strong connection. 35
Moreover, Tezal et al. 36 concluded that having periodontal disease can affect the pathologic type of cancers. The study found that patients with periodontal disease are more liable to have poorly-differentiated types of squamous cell carcinoma of the oral cavity than patients without chronic periodontitis.
These results were said to have applications in clinical and preventive measures, diagnosis, and treatment of head and neck squamous cell carcinoma. The possible connection between chronic periodontitis and malignancy is not obvious, but chronic infection exposure is being studied. 37
Chronic periodontitis and pregnancy
Numerous research studies have focused on the possible relationship between periodontal disease and poor pregnancy outcomes, pregnancy complications and defective health status of the new-born. Recently, many studies have confirmed the presence of a link between low birth weight and preterm babies and chronic periodontal disease regardless of the paternal health status, race, and the age of the mother. 38
There is also evidence that periodontal disease may be an independent factor for increased susceptibility to pregnancy complications. 39 Primary evidence indicates that treatment of periodontal infection might lead to a decrease in pregnancy-related complications. 40 The underlying mechanism is not clear yet. However, some studies suggest that the presence of bacterial infection leads to stimulation of cell-mediated immunity and then the production of various cytokines including interleukins, prostaglandins, which is important during the process of delivery, and tumour necrosis factor. 41
The newly-suggested mechanism of labour states that the levels of these cytokines increase significantly within the intra-amniotic through all phases of pregnancy, until a certain high level is reached which induces the process of labour. 41 Therefore, it is quite reasonable to suppose that the presence of any type of infection, for example periodontitis, could lead to increased levels of the normal mediators of delivery leading to preterm birth and possibly low birth weight. 42
Researchers also suppose that overt infections such as periodontitis can lead to preterm birth and low birth weight through the invading microorganisms or the byproducts and toxins of the bacteria including lipopolysaccharide (LPS), which invade the uterus through blood stream, stimulating cytokine production in the uterus and placenta or in the amniotic fluid, leading to elevated prostaglandin, which stimulates the contraction of the uterine muscles. 41, 42
Moreover, it was indicated that pregnant women who suffer from some degree of periodontal disease are more liable to gestational diabetes mellitus than pregnant women who are free of periodontitis. 43 However, more research studies are needed in the coming years to have a strong statistically significant correlation between periodontal disease and endocrinal disorders including diabetes, as well as the link with poor pregnancy outcomes.
The relationship between periodontal disease and the development of various systemic diseases, such as diabetes mellitus (DM), osteoporosis, pregnancy complications, and coronary vascular disease, is the topic of many research studies.
Although most studies have provided evidence suggesting the presence of a positive relationship between periodontal disease and various systemic diseases, many physicians and dentists greatly advocate the need for more studies to confirm these conclusions. In general, more controlled clinical trials and more randomized samples are needed to confirm the relationship between periodontal disease and these systemic diseases.
Studies on each of the systemic conditions have their own defects that prompt further research.
Current studies of the relationship between periodontal disease and coronary vascular disease should provide more data to differentiate between the stimulation of inflammatory cytokines by periodontitis as opposed to the possibility of a direct infection of the endothelium
(or both).
Furthermore, anti-inflammatory and antibiotic therapy as a treatment strategy for periodontal disease should be incorporated in the studies to assess their role in preventing the deterioration of cardiovascular disease.
Regarding diabetes mellitus, the current notion that chronic inflammation associated with periodontal disease may result in the metabolic disturbances associated with DM needs further evidence to clarify the underlying mechanisms. Also, elucidation of the mechanism underlying the relationship between periodontal infection and coronary vascular disease may provide a hypothesis that unifies both diseases, which may aid in the prevention of complications associated with both diseases.
Regarding the relationship between periodontal disease and osteoporosis, more information about the molecular mechanisms involved in the activation of RANKL/RANK system could help in the development of new pharmacological methods for the inhibition of excessive bone resorption. Also, larger studies are needed to assess the value of osteocalcin, calcitonin, and parathyroid hormones as laboratory indicators of periodontal disease activity.
Furthermore, more studies are needed to clarify the mechanisms by which periodontal disease may lead to malignancy, low birth weight, and pregnancy complications. It is of specific importance to differentiate between the presence of a causative relationship and a simple association.
These findings will be of importance to dentists as well as clinicians, as periodontal infection could become a modifiable risk factor for the development of several systemic diseases, and this may result in advances in the prevention of those conditions.
Lastly, oral health has direct and/or indirect effects on the individual's general health. Both dentists and physicians should recognize the increasing significance of this relationship in modern health care. Dentists should increase their awareness and their clinical exposure to relevant systemic diseases to be able to interact meaningfully about these issues with their medical colleagues. Team approach is strongly advocated for the management of severe periodontal disease associated with relevant systemic conditions.
1. Newman MG. Classification and epidemiology of periodontal diseases. In: Newman MG, Takei H, Carranza FA, editors. Carranza's Clinical Periodontology. 10th ed. Philadelphia: WB Saunders Company; 2007. pp. 100–29.
2. Elter JR, Champagne CME, Offenbacher S, Beck JD. Relationship of periodontal disease and tooth loss to prevalence of coronary heart disease. J Periodontol. 2004;75:782–90. [PubMed]
3. Ebersole JL, Capelli D, Steffen MJ. Longitudinal dynamics of infection and serum antibody in A. actinomycetemcomitans periodontitis. Oral Dis. 1995;1:129–38.[PubMed]
4. Hayes C, Antczak-Bouckoms A, Burdick E. Quality assessment and meta-analysis of systemic tetracycline use in chronic adult periodontitis. J Clin Periodontol. 1992; 19: 164–8. [PubMed]
5. Wactawski-Wende J, Grossi SG, Trevisan M, Genco RJ, Tezal M, Dunford RG, Ho AW, Hausmann E, Hreshchyshyn MM. The role of osteopenia in oral bone loss and periodontal disease. J Periodontol. 1996; 67(10 Suppl): 1076–84. [PubMed]
6. Amar S, Han X. The impact of periodontal infection on systemic diseases. Med Sci Monit. 2003; 9: RA291–9. [PubMed]
7. Lerner UH. Osteoclast formation and resorption. Matrix Biol. 2000; 19: 107–20.[PubMed]
8. Manabe H, Kawaguchi H, Chikuda H, Miyaura C, Inada M, Nagai R, et al. Connection between B lymphocyte and ostoclast differentiation pathways. J Immunol. 2001; 167: 2625–31. [PubMed]
9. Nagasawa T, Kobayashi H, Kiji M, Aramaki M, Mahanoda R, Kojima T, et al. LPS-stimulated human gingival fibroblast inhibits the differentiation of monocytes into osteoclasts through the production of osteoprotegerin. Clin Exp Immunol. 2002; 130: 338–44. [PubMed]
10. Jiang Y, Mehta CK, Hsu TY, Alsulaimani FFH. Bacteria induce osteoclastogenesis via an osteoblast-independent pathway. Infect Immun. 2002; 70: 3143–8. [PubMed]
11. Jacobs R, Ghyselen J, Konincks P, van Steeberghe D. Long-term bone mass evaluation of mandible and lumbar spine in a group of women receiving hormone replacement therapy. Eur J Oral Sci. 1996; 104: 10–6. [PubMed]
12. Pacifici R. Cytokines, estrogen, and postmenopausal osteoporosis – the second decade. Endocrinology. 1998; 139: 2659–61. [PubMed]
13. Gemmell E, Marshall RI, Seymour GJ. Cytokines and prostaglandins in immune homeostasis and tissue destruction in periodontal disease. J Periodontol. 2000; 1997: 14:112–43.[PubMed]
14. Inagaki K, Kurosu Y, Yoshinari N, Noguchi T, Krall EA, Garcia RI. Efficacy of periodontal disease and tooth loss to screen for low bone mineral density in Japanese women. Calcif Tissue Int. 2005; 77 :9–14. Epub 2005 Jul 14. [PubMed]
15. Wactawski-Wende J, Hausmann E, Hovey K, Trevisan M, Grossi S, Genco RJ. The association between osteoporosis and alveolar crestal height in postmenopausal women. J Periodontol. 2005; 76: 2116–24. [PubMed]
16. Amar S, Han X. The impact of periodontal infection on systemic diseases. Med Sci Monit. 2003; 9: RA291–9. [PubMed]
17. Lusis AJ. Atherosclerosis. Nature. 2000; 407: 233–41. [PubMed]
18. Beck J, Garcia R, Heiss G, Vokonas PS, Offenbacher S. Periodontal disease and cardiovascular disease. J Periodontol. 1996; 67: 1123–37. [PubMed]
19. Geerts SO, Legrand V, Charpentier J, Albert A, Rompen EH. Further evidence of the association between periodontal conditions and coronary artery disease. J Periodontol. 2004; 75: 1274–80.[PubMed]
20. Haraszthy VI, Zambon JJ, Trevisan M, Zeid M, Genco RJ. Identification of periodontal pathogens in atheromatous plaques. J Periodontol. 2000; 71: 1554–60. [PubMed]
21. Cairo F, Gaeta C, Dorigo W, Oggioni MR, Pratesi C, Pini Prato GP, Pozzi G. Periodontal pathogens in atheromatous plaques. A controlled clinical and laboratory trial. J Periodontal Res. 2004; 39: 442–6. [PubMed]
22. Amar S, Gokce N, Morgan S, Loukideli M, Van Dyke TE, Vita JA. Periodontal disease is associated with brachial artery endothelial dysfunction and systemic inflammation. Arterioscler Thromb Vasc Biol. 2003; 23: 1245–9. [PubMed]
23. Chun YH, Chun KR, Olguin D, Wang HL. Biological foundation for periodontitis as a potential risk factor for atherosclerosis. J Periodontal Res. 2005; 40: 87–95. [PubMed]
24. Sinisalo J, Paronen J, Mattila KJ, Syrjala M, Alfthan G, Palosuo T, Nieminen MS, Vaarala O. Relation of inflammation to vascular function in patients with coronary heart disease. Atherosclerosis. 2000; 149: 403–11. [PubMed]
25. Wang CH, Li SH, et al. C-reactive protein upregulates the angiotensin type 1 receptors in vascular smooth muscle. Circulation. 2003; 107: 1783–90. [PubMed]
26. Fuster V, Badimon L, Badimon JJ, et al. The pathogenesis of coronary artery disease and the acute coronary syndromes. N Engl J Med. 1992; 326: 242–50. [PubMed]
27. Fichtlscherer S, Breuer S, Schachinger V, Dimmeler S, Zeiher AM. C-reactive protein levels determine systemic nitric oxide bioavailability in patients with coronary artery disease. Eur Heart J. 2004; 25: 1412–8. [PubMed]
28. Hung HC, Merchant A, Joshipura KJ. Oral health and peripheral arterial disease. Circulation. 2003:1152–7. [PubMed]
29. Iacopino AM. Bethesda: American Academy of Periodontology; c2009. Chronic periodontal disease may contribute to diabetes.
30. Loos BG. The systemic markers of inflammation in chronic periodontitis. J Periodontol. 2005; 76: 2106–15. [PubMed]
31. Mealey BL. Diabetes mellitus and inflammatory periodontal diseases. Curr Opin Endocrinol Diabetes Obes. 2008; 15: 135–41. [PubMed]
32. Dunning T. Periodontal disease- the overlooked diabetes complication. Nephrol Nurs J. 2009; 36: 489–95. [PubMed]
33. Rosenquist K. Risk factors in oral and oropharyngeal squamous cell carcinoma: A population-based case-control study in southern Sweden. Swed Dent J Suppl. 2005; 179: 1–66. [PubMed]
34. Meyer MS, Michandi DS. Periodontal disease and cancer. Cancer causes control. 2008; 19: 895–07. [PubMed]
35. Fitzpatrick SG. The association between periodontal disease and cancer: A review of the literature. J Dent. 2010; 38: 83–5. [PubMed]
36. Tezal M, Sullivian MA, Hyland A, Marshall JR, Stoler D, Reid MA, et al. Chronic periodontitis and the incidence of head and neck squamous cell carcinoma. Cancer Epidemiol Biomarkers Prev. 2009; 18: 2409–12. [PubMed]
37. Yu-Kang T, Gilthorpe MS. Commentary: Is tooth loss good or bad for general health? Int J Epidemiol. 2005; 34: 475–6. [PubMed]
38. Sánchez AR, Sheridan PJ. Maternal chronic infection as a risk factor in preterm low birth weight infants: The link with periodontal infection. J Int Acad Periodontol. 2004; 6: 89–4. [PubMed]
39. Pitiphat W, Joshipura K, Gillman MW, Douglass CW. Maternal periodontitis and adverse pregnancy outcomes. Community Dent Oral Epidemiol. 2008; 36: 3–11. [PubMed]
40. Scannapieco FA, Bush RB, Paju S. Periodontal disease as a risk factor for adverse pregnancy outcome: A systematic review. Ann Periodontol. 2003; 8: 70–8. [PubMed
41. Rai B, Kaur J, Kharb S. Pregnancy gingivitis and periodontitis and its systemic effect. The Internet Journal of Dental Science 2009
42. Gibbs RS. The relationship between infections and adverse pregnancy outcomes: An overview. Ann Periodontol. 2001; 6: 153–63. [PubMed]
43. Periodontal disease may lead to gestational diabetes mellitus. J Am Dent Assoc. 2008; 139: 541. [PubMed]
Cardiac disease , Diabetes Mellitus , Oral Pathology , Osteoporosis , Periodontal Disease , Periodontitis , Pregnancy complications , Prosthodontics , Systemic disease
1 LOCATOR R-Tx – Improved & Advanced Removable Attachment System
2 The Relation Between Periodontal Disease and Vitamin D
3 Causes of Complete Dentures Renewal
4 Periodontal Links to Systemic Disease
5 The Pharmacological Management of Dental Caries
6 Unusual Large Submandibular Gland Calculus
7 Studying Porcelain Laminate Veneers & All-Ceramic Crowns
8 Pain Relief of Aphthous Ulcers by Lasers: Literature Review
9 Adhesion to mild fluorosed enamel: A comparative study of two etching protocols
10 A Simplified Impression Technique for Distal Extension Removable Partial Dentures


Use a tool to encrypt your connection, like CyberGhost VPN or Tor (which routes your search through servers all over the globe, the way criminals in the movies route their calls to avoid having... There has been so much buzz about the Top 10 Free Movies Download Websites Without Registration where you don�t have to pay money to get quality entertainment.
Helmed by Kai Ryssdal, our flagship program examines what the day in money delivered, through stories, conversations, newsworthy numbers and more. how to add a music bot to discord 2017 Helmed by Kai Ryssdal, our flagship program examines what the day in money delivered, through stories, conversations, newsworthy numbers and more.
Despite the privacy precautions you take, there is someone who can see everything you do online: your Internet Service Provider (ISP). When it comes to online privacy, there are a lot of steps you can take to clean up your browsing history and prevent sites from tracking you.
This is the reason why some viewers are looking for ways to download YouTube full movies from YouTube. Now this article will give you some solutions for you to watch your favorite movies even without internet connection.


text:
package ru.spoddubnyak.start;

import org.junit.Test;
import ru.spoddubnyak.models.Item;

import static org.hamcrest.core.Is.is;
import static org.junit.Assert.assertThat;

/**
 * Tests class Tracker.
 *
 * @author Sergei Poddubnyak (forvvard09@gmail.com)
 * @version 1.0
 * @since 26.12.2016
 */
public class TrackerTest {
    /**  first element array Item of Tracker.  */
    private final int firstElement = 0;

    /**
     * Test method add class Tracker Add two new items in storage Tracker.
     */
    @Test
    public void whenAddTwoNewItemsThenGetSameResultInTracker() {
        Tracker tracker = new Tracker();
        Item[] items = new Item[2];
        final Long timeOne = 10L;
        final Long timeTwo = 20L;
        items[0] = new Item("name0", "desk0", timeOne);
        items[1] = new Item("name1", "desk1", timeTwo);
        tracker.add(items[0]);
        tracker.add(items[1]);
        assertThat(items, is(tracker.findAll()));
    }

    /**
     * Test method add class Tracker Add one new items in storage Tracker.
     */
    @Test
    public void whenAddOneNewItemThenGetSameResultInTracker() {
        Tracker tracker = new Tracker();
        final Long timeOne = 10L;
        Item item = new Item("name0", "desk0", timeOne);
        tracker.add(item);
        assertThat(item, is(tracker.findAll()[firstElement]));
    }

    /**
     * Test method update class Tracker Update item of itemUpdate storage Tracker.
     */
    @Test
    public void whenUpdateItemThenGetSameResultInTracker() {
        Tracker tracker = new Tracker();
        final Long timeOne = 10L;
        final Long timeTwo = 20L;
        final Long createUpdate = 99L;
        Item itemOne = new Item("name0", "desk0", timeOne);
        Item itemTwo = new Item("name1", "desk1", timeTwo);
        tracker.add(itemOne);
        tracker.add(itemTwo);
        Item itemUpdate = new Item("nameUpdate", "descreptionUpdate", createUpdate);
        itemUpdate.setId(itemOne.getId());
        tracker.update(itemUpdate);
        assertThat(itemUpdate, is(tracker.findAll()[firstElement]));
    }

    /**
     * Test method delete class Tracker delete item of itemDelete storage Tracker.
     */
    @Test
    public void whenDeleteItemThenGetSameResultInTracker() {
        Tracker tracker = new Tracker();
        final Long timeOne = 10L;
        final Long timeTwo = 20L;
        Item itemOne = new Item("name0", "desk0", timeOne);
        Item itemTwo = new Item("name1", "desk1", timeTwo);
        tracker.add(itemOne);
        tracker.add(itemTwo);
        tracker.delete(itemOne);
        assertThat(itemTwo, is(tracker.findAll()[firstElement]));
    }
    /**
     * Test method findByID class Tracker find Item by id in storage Tracker.
     */
    @Test
    public void whenFindByIdThenGetItembyId() {
        Tracker tracker = new Tracker();
        final Long timeOne = 10L;
        final Long timeTwo = 20L;
        Item itemOne = new Item("name0", "desk0", timeOne);
        Item itemTwo = new Item("name1", "desk1", timeTwo);
        tracker.add(itemOne);
        int idItemOne = itemOne.getId();
        tracker.add(itemTwo);
        assertThat(itemOne, is(tracker.findById(idItemOne)));
    }

    /**
     * Test method findByName class Tracker find items by Name item in storage Tracker.
     */
    @Test
    public void whenFindByNameThenGetItembyName() {
        Tracker tracker = new Tracker();
        Item[] testItems = new Item[2];
        final Long timeOne = 11L;
        final Long timeTwo = 22L;
        final Long timeThree = 33L;
        testItems[0] = new Item("name01", "descr01", timeOne);
        testItems[1] = new Item("natt02", "descr02", timeTwo);
        Item itemThree = new Item("test", "test", timeThree);
        tracker.add(testItems[0]);
        tracker.add(testItems[1]);
        tracker.add(itemThree);
        assertThat(testItems, is(tracker.findByName("na")));
    }
}

redpajama_set_name: RedPajamaGithub


text:
Seth Meyers Calls Out Trump For Stealing From Chevy Chase
"I'm the president and you're not" seems like a very familiar Chevy Chase joke...
By Alex Firer
March 24, 2017 1:30 pm PT
Source: www.youtube.com / Via: www.youtube.com
The fraternity of Weekend Update hosts is a small but close one. When Jimmy Fallon falls, Tina Fey is there to pick him up. When Amy Poehler falters, Jane Curtin is there to prop her up. So right now, when Seth Meyers is hosting a late night show that delves into politics, how can he not take apart Trump's latest proclamation to Time Magazine.
When asked if he thinks he's doing something wrong, the weirdo responded, "Well, I'm president and you're not". Seth Meyers is quick to point out — my friend. Is a play on a Chevy Chase line. A classic Chevy Chase line which goes as such — "I'm Chevy Chase. And you're not".
Source: www.hulu.com / Via: www.hulu.com
Mr. Trump, have you no shame? (I mean, I guess he 100% doesn't but whatever.) Stealing from beloved comedian and noted weirdo Chevy Chase!? As Seth Meyers points out, it wouldn't be the first time he's stolen from SNL, and plays a clip of Norm McDonald shouting "Fake news!".
Oh Trump. You're just one "Seriously" away from getting a cease and desist from Frozen Caveman Lawyer! What other references can I throw in… I dunno, Kate MacKinnon's Russian character helped you hack? There are options.
What do you think of the video? Let us know in the comments or on Twitter at @WhatsTrending.


Raise a glass to the burgeoning wine region of Niagara-on-the-Lake during this 6-hour wine tour of the area's growing collection of winemakers. Explore four award-winning wineries in the heart of what's now known as Canada's 'Wine Belt,' savoring free tastings at each stop and plenty of opportunities to purchase your favorite bottles. You'll also have time to stop to purchase lunch at local eatery, tour and taste samples at a chocolate factory. A driver-guide, shuttle bus or touring van, and admission fees are all included.
Journey deep into Niagara wine country on this private 5-hour tour that takes you to nine wineries, small and large, located on the Lake and the Escarpment. With just your family and friends (six people max), savor delicious wine offerings and indulge in artisanal chocolate. Experience a variety of sites, including a winery that will make you feel as you've just stepped into Tuscany. Hotel transport by upscale private vehicle from Niagara Falls, Niagara-on-the-Lake, Canada or Buffalo, Niagara Falls, New York is included.
Let a guide navigate for you to the best pastry shops on Queen Street West. Enjoy 4 treats over the course of the tour as you listen to the history of each shop and its specialties. Next, stop for a coffee and take a stroll through Trinity Bellwoods Park, weather permitting. Your guide leaves you to explore the independent shops, bars, and cafes that make up Ossington Avenue.
See a whole new side of Toronto on a local chocolate tasting tour through the city's hippest downtown neighborhoods. Your guide brings you to the area's best local businesses, including a bean-to-bar chocolate maker. Going with a guide gives you the chance to chat with store-owners about their trade, as well as watch an award-winning chocolatier make their signature truffles.


Tea Tree is a two storeyed apartment offering luxury features in a versatile holiday solution for up to five people.
Tea Tree is suitable for couples wanting more space or a "children-free" holiday zone; couples travelling together, or a small family.
Paperbark is a studio apartment equipped to the same standard as its larger neighbour but in a smaller, romantic space. For differences, see the facilities page (new window).
Paperbark is suitable for couples only.


This new model, with two engines totaling 300 HP of propulsion, promises a wide variety of pleasure such as fishing, all sorts of water sports and cruising. The perfect day boat for large families.
2014 Power Boat of The Year elected by Vela e Motor in Italy.
Fresh water (l.): 0 Lt.


Twelve years ago, Conrad Grebel University College planted a black walnut tree and erected a sign marking the 200th anniversary of the arrival in 1805 of the first Mennonite settlers from Pennsylvania and the establishment of the "German Company Tract." But time has a way of altering understandings of events and history. On Sept. 22, 2017, a new sign was erected beside the old one, acknowledging the larger and longer history of the land.
Led by Marlene Epp, Grebel's dean, and Reina Neufeldt, professor of peace and conflict studies, the ceremony began with Indigenous smudging and a Mohawk welcoming song performed by the Waterloo Aboriginal Education Centre at St. Paul's University College at the University of Waterloo.
The unveiling ended with the singing of "Come Walk with Me, the Journey is Long" and the sharing of fry-bread from the education centre, apples and popcorn.


Arguing the Indiana Supreme Court "asserted a novel public right to access the entire beach" of Lake Michigan, private lakeshore landowners Friday asked the Supreme Court of the United States to rule that the public was entitled to use no part of the beach above the water itself.
The landowners' petition for writ of certiorari urges the justices to take an appeal the landowners say poses this unsettled question: "whether — in conflict with the rule on the seashore — the newly-admitted states took title to the entire beach surrounding the Great Lakes" at statehood.
The Indiana Supreme Court opinion challenged in this petition did not actually rule as such, instead holding that the state's public-trust rights extend to the ordinary high water mark. The Indiana justices' ruling preserved the public's right to walk along the shore of Lake Michigan, leaving any expansion of public rights to the Indiana General Assembly.
The cert petition in Gunderson v. Indiana is the culmination of years of state court litigation initiated by private landowners in the LaPorte County town of Long Beach, just south of the Michigan state line. Attorneys from Faegre Baker Daniels in Minneapolis and Chicago arguing on behalf of the private landowners wrote in the cert petition that the outcome of the Indiana case could extend well beyond the 45 miles of Indiana Great Lakes shoreline.
But landowners have lost that argument at every level of state courts, which variously ruled for the public interest in attempting to draw a line in the sand between public and private property.
Landowners Don and Bobbie Gunderson's names remain on the petition as the sole plaintiffs, even though property records show the couple has not owned the property at issue for years. The petition records the transfer of ownership from the Gundersons in a footnote, but it does not identify who the current plaintiffs are, noting that Federal Rule of Civil Procedure 25(c) does not require a transfer of interest.
"The Indiana courts did not order substitution, so under both state and federal Rule 25 the Gundersons remain proper plaintiffs," the petition says.
Meanwhile, the case in state court attracted numerous friends of the court aligning to argue for both public and private property rights. Those interests are certain to line up at the U.S. Supreme Court, should justices grant the cert petition.
After the petition is docketed, the state and intervenors will have 30 days to file a brief in opposition or request an extension.


Usually, your dad only yells at you to mow your own family's lawn. Ben Schmitz's father had to do a bit more.
Schmitz, a sophomore in the College of Business Administration, owns and operates Benjamin Lawn and Landscape, a mowing company in Kansas City, Kan. Schmitz has 40 clients, two part-time employees and a vision to make a lot of dough out of the hated chore.
Schmitz said he worked almost 60 hours per week this summer dealing with clients, advertising, filling out paperwork and actually mowing the lawns. Next summer, Schmitz hopes to take a strictly administrative role in his business, and plans to hire two full-time employees to mow the lawns.
Schmitz plans on expanding his business through the Internet, potentially creating his own website. Kansas City is a great place for a lawn mowing business because there is more green space than in most areas of the country, he said.
He has made mowing lawns his line of work from the age of 10, when he mowed his neighbor's lawn for the summer.
Scott Long was the neighbor Schmitz first started working for when he was 10 years old. Long said Schmitz learned to cut lawns under pretty tough conditions from his dad, Mike Schmitz.
"Ben's dad is a pretty massive guy, maybe 6'3 or 6'4," Long said. "And (Mike) would walk behind Ben while he was mowing my lawn making sure the rows were straight.
After a couple years of mowing just a few of his neighbors lawns, Mike Schmitz offered to loan his seventh grade son the money to buy a commercial mower to expand his business.
However, it was not an easy expansion.
Ben and his father had a lot of trouble operating the complicated commercial lawn mower.
Mike Schmitz has been an integral part of Benjamin Lawn and Landscape from the beginning, whether it was mowing the lawns while his son was at track practice in high school or passing out flyers to potential customers in the neighborhood.
According to the elder Schmitz, however, his involvement in the company has gone down since Ben has gotten older despite the huge surge in workload. Mike Schmitz has been making Ben pay taxes for the last few years.
While it may seem that Ben Schmitz may be the richest kid at Marquette, lawn mowing is not an expenses-free profession. Schmitz estimates he has spent about $20,000 on equipment in the last seven years or so, and Schmitz is paying for a hefty portion of his education with the money he is making.


text:
Q: message.member.displayAvatarURL(); is not a function Im trying to add a command to my bot, said command will display the avatar of whoever is mentioned. For example
>pfp @user1 would display user1's avatar.
However my code is returning an error of  message.member.displayAvatarURL(); is not a function
let member = message.mentions.members.first();

    if(member){
        message.member.displayAvatarURL();
    }

What would be the appropriate syntax to execute this?

A: displayAvatarURL is a function on User, not GuildMember.
You can either get the user from the member like this:
message.member.user.displayAvatarURL();

or just directly use the author property:
message.author.displayAvatarURL();

redpajama_set_name: RedPajamaStackExchange


text:
Q: Rails: How to reference a path from one Model in another Models Controller? This is an easy question, but I am new to Rails. Simply put, I want to use a route from another model (Score) in my Prediction controller. Once a new prediction is made, I want to redirect to the new_score_path but I keep getting an error -
 undefined local variable or method `new_scores_path' for #<PredictionsController:

I guess I need to somehow reference the Score model in the Predictions Controller but I am not sure how to do this. Here is my create method
  def create
    @prediction = Prediction.new(prediction_params)
    respond_to do |format|
      if @prediction.save
       user = User.find(@current_user.id)
       user.lastcase = user.lastcase + 1
       user.save
       @user = user
       @patient = Patient.find(@current_user.lastcase)
       format.html { redirect_to url: new_scores_path, notice: 'Prediction WAS successfully created.' }
       format.json { render :new, status: :created, location: @prediction }
      else
       format.html { render :new, status: :unprocessable_entity }
       format.json { render json: @prediction.errors, status: :unprocessable_entity }
      end
   end
  end

Many thanks for reading.
EDIT
I tried
  new_score_path

and it simply routed me to predictions (i.e. in the index.html.erb file). This was in the address
bar
   http://localhost:3000/predictions?notice=Prediction+WAS+successfully+created.&url=%2Fscores%2Fnew

A:
This is an easy question, but I am new to Rails. Simply put, I want to
use a route from another model (Score) in my Prediction controller.

You're thinking about it wrong. The controller doesn't belong to a model. Nor does the route. Models are just an internal implementation detail of the application (or at least they should be in the best of worlds).
All the routing helpers generated from routes.rb are available into all your controllers and views no matter what they are named.

I guess I need to somehow reference the Score model in the Predictions Controller but I am not sure how to do this.

Provided you're following the conventions and have declared the route with resources :scores you just need to call the correct helper method new_score_path. The new action route helper is always new_singular_path.
The only actual link between routes and models is that Rails provides helpers that can guess what route helper to use based on the name of a model and the Rails conventions.

A: After reviewing the documentation, I explicitly gave the path
 redirect_to "/scores/new"

This solved the problem; I am unsure if this is the correct/good way to do this, but it worked. Happy to be schooled on better solutions if there are any.

A: The problem is how the redirect_to method is invoked. Instead of:
redirect_to url: new_scores_path, notice: 'Prediction WAS successfully created.'

Try:
redirect_to new_score_path, notice: 'Prediction WAS successfully created.'

NOTE The correct route helper is new_score_path (in singular).


The new Sony G series XQD cards achieve a maximum read 440 MB/s and write 350 MB/s speeds by using advanced PCIe Gen2 interface. The increased write speed of G series will enhance the capability of high speed burst shooting with DSLR camera or when using 4K video. Transfer times are greatly reduced even for large quantities of RAW images or data-intensive 4K video files.


A new step in cancer immunotherapy: researchers from the Netherlands Cancer Institute and University of Oslo/Oslo University Hospital show that even if one's own immune cells cannot recognize and fight their tumors, someone else's immune cells might. Their proof of principle study is published in the journal Science on May 19th.
The study shows that adding mutated DNA from cancer cells into immune stimulating cells from healthy donors create an immune response in the healthy immune cells. Inserting the targeted components from the donor immune cells back into the immune cells of the cancer patients, the researchers were able to make cancer patients' own immune cells recognize cancer cells.
The extremely rapidly developing field of cancer immunotherapy aims to create technologies that help the body's own immune system to fight cancer. There are a number of possible causes that can prevent the immune system from controlling cancer cells. First, the activity of immune cells is controlled by many 'brakes' that can interfere with their function, and therapies that inactivate these brakes are now being tested in many human cancers. As a second reason, in some patients the immune system may not recognize the cancer cells as aberrant in the first place. As such, helping the immune system to better recognize cancer cells is one of the main focuses in cancer immunotherapy.
Ton Schumacher of the Netherlands Cancer Institute and Johanna Olweus of the University of Oslo and Oslo University Hospital decided to test whether a 'borrowed immune system' could "see" the cancer cells of the patient as aberrant. The recognition of aberrant cells is carried out by immune cells called T cells. All T cells in our body scan the surface of other cells, including cancer cells, to check whether they display any protein fragments on their surface that should not be there. Upon recognition of such foreign protein fragments, T cells kill the aberrant cells. As cancer cells harbor faulty proteins, they can also display foreign protein fragments - also known as neoantigens - on their surface, much in the way virus-infected cells express fragments of viral proteins.
To address whether the T cells of a patient react to all the foreign protein fragments on cancer cells, the research teams first mapped all possible neoantigens on the surface of melanoma cells from three different patients. In all 3 patients, the cancer cells seemed to display a large number of different neoantigens. But when the researchers tried to match these to the T cells derived from within the patient's tumors, most of these aberrant protein fragments on the tumor cells went unnoticed.
Next, they tested whether the same neoantigens could be seen by T-cells derived from healthy volunteers. Strikingly, these donor-derived T cells could detect a significant number of neoantigens that had not been seen by the patients' T cells.
"In a way, our findings show that the immune response in cancer patients can be strengthened; there is more on the cancer cells that makes them foreign that we can exploit. One way we consider doing this is finding the right donor T cells to match these neoantigens.", says Ton Schumacher. "The receptor that is used by these donor T-cells can then be used to genetically modify the patient's own T cells so these will be able to detect the cancer cells".
"Our study shows that the principle of outsourcing cancer immunity to a donor is sound. However, more work needs to be done before patients can benefit from this discovery. Thus, we need to find ways to enhance the throughput. We are currently exploring high-throughput methods to identify the neoantigens that the T cells can "see" on the cancer and isolate the responding cells. But the results showing that we can obtain cancer-specific immunity from the blood of healthy individuals are already very promising", says Johanna Olweus.


text:
The Ten Best Guest Features on the 'Black Panther: The Album' Project
Photo Credit: Ural Garrett for Okayplayer.
Black Panther: The Album is the best movie soundtrack in years
The movie soundtrack isn't dead. Yet.
On Friday the soundtrack for Marvel's Black Panther was released. The album was curated by Kendrick Lamar and his TDE crew.
HEAR: Stream 'Black Panther: The Album,' Presented By TDE
So what we have here is basically a TDE compilation album. Kendrick is almost on every track, and there are appearances from the likes of Jay Rock, ScHoolboy Q, and SZA.
However, TDE also shows love to non-affiliated acts like 2 Chainz, Jorja Smith, and The Weeknd, as well as up-and-coming rappers like SOB X RBE, Mozzy, and Reason.
After giving the album a couple of close listens, we determined who showed out the most on the soundtrack.
Here are the ten best features off the Black Panther: The Album project:
10. Reason
When the tracklist for Black Panther: The Album was released, there was confusion around track 12, "Seasons," which features Mozzy, Sjava, and Reason. Most assumed Reason was the South African rapper. It wasn't. The Reason featured on Black Panther The Album is an unheralded rapper from Carson, California, who, honestly, we never heard of before this. He turns out a passionate, star-making performance on the track, ending his verse by telling the tale of a tragic shooting:
Like when that car bent the block, brock, brock, brock
Hit the homie but we made it, he already saved us
Thinkin' that he could've died nigga
We was praying' to the sky and his mother cried rivers
Healed up, but tough luck, he tried to get revenge
Had to drop, bullets popped and they killed him
Worst part is that it sound so familiar
9. James Blake/Anderson .Paak
Blake and .Paak is an odd combination. On paper. But on their first collaboration, "Bloody Waters," the two find a way to make their contrasting styles — Anderson: nostalgic, warm; James is just frigid — work over Sounwave's sparse production.
8. Khalid/Swae Lee
"The Ways" is an infectious duet from Khalid and Swae Lee that sounds like could have been on Khalid's sweet debut American Teen. So it's a win for us.
7. Jay Rock
"King's Dead" is an odd song, in the sense that there is very little rapping when you consider how many actual rappers there are on the song (three). Jay Rock is the only person with an extended verse. (We're really not sure what to call Future's feature here.) And he makes the most of his time on the mic. At times, Jay Rock can be a bit meat and potatoes…but sometimes that's a good meal.
6. 2 Chainz
Is there a guest appearance that 2 Chainz doesn't kill? Don't answer that, the question is rhetorical. The rapper is a much need spark on the otherwise middling "X," which features ScHoolboy Q and Saudi.
5. Mozzy
When Kendrick Lamar won Best Rap Album at the Grammys last month, he shouted out Mozzy: one of the most underrated (and violent) lyricists in hip-hop right now.Mozzy appears on the heaviest song on the album, "Seasons." The first line alone tells you how deep Mozzy can get:
I cried when my lil' brotha died, got high and watched the sun rise
4. Babes Wodumo
"Redemption" is a collaboration between "LOVE." star Zacari and South Africa artist Babes Wodumo. The song is an absolute jam, and it's mostly because of the infectious flows Wodumo uses. (Sidenote: it's really cool to see so many South African artists part of this project.)
3. Kendrick Lamar
It was a pleasant surprise to see how involved Kendrick Lamar was with this project. He's almost on every song, in some capacity. Despite the fact that there are over a dozen guests, the album is cohesive (a trademark of Kendrick albums.) And, at times, Kendrick is spectacular, especially on the album's intro and on the Travis Scott collabo "Big Shot." The only reason why Kendrick isn't higher on this list is because his collabo with SZA, "All the Stars," and his song with The Weeknd, "Pray for me," both fall flat.
2. Ab Soul
When he wants to, Ab-Soul could hold his own with any rapper out there (including Kendrick.) On "Bloody Waters" he spits two verses that contain all of this amazing descriptive imagery. Just check the beginning of verse number two:
Low lows pulled outside of the Ralph's
After a car show at Dominguez
They had a disagreement, they had to air out
Just another day in Del Amo
1. SOB X RBE
SOB X RBE is a group of four rappers — Slimmy B, DaBoii, Lul G, and Yhung T.O. — from Vallejo, California. They first started making national noise with a street hit called "Anti." The Black Panther soundtrack is their biggest look so far. And they completely show out. On their track "Paramedic!" they rap with this great frantic, aggressive, sometimes off-beat energy that Kendrick can't keep up with. (Which is why he's only on the hook on this.)
Dimas Sanfiorenzo
Dimas Sanfiorenzo is the Managing Editor for Okayplayer. He specializes in coverage centered around music, pop culture, and politics. Email: Dimas@okayplayer.com
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After a long break I picked up the Erlang book again and my appetite for writing some erlang code was soon kindled.
A small Python component I produced at work seemed like a good candidate for my (sequential) erlang exercises. It is a fairly simple component that removes user/password data embedded in URLs.
I have played with erlang a little bit before (ring benchmark, XML parsing) and liked it in general although IMHO it lacks severely when it comes to the availability and quality of standard library components.
The initial implementation of the URL filter in erlang used its regexp library.
18 mktemppath(Prefix, N) -> Prefix ++ "/" ++ genname(N, ).
Please note how I had to implement functionality absent from the standard library above.
20 %% Removes passwords embedded in URLs from a log file.
22     %% make a temporary directory if it does not exist yet.
29     %% Move the original file out of the way.
36     %% Now open it for reading.
38     %% Open the original path for writing.
41     %% Call the function that will scrub the lines.
44     %% Close the file handles and return the path to the original file.
The code that scrubs the URLs is below, the scrub_lines() function is tail recursive.
50 %% the scrubbing function is invoked for lines that contain URLs.
56             %% Does the line contain URLs?
65             %% Continue with next line.
74     %% A single argument (the name of the file to be scrubbed) is expected.
79     %% in the place of the original file. Where was the latter moved to?
The cursory benchmarks performed (on log files of varying size) using the python and the erlang code confirmed other people's experiences with erlang's regex performance (but see also this interesting "rebuttal").
Curious to learn whether the performance can be improved by abstaining from regular expressions I came up with an alternative implementation that does not use regexp.
As you can see below the do-it-yourself construction is indeed performing slightly better at the expense of being very specialized and requiring 60% more code.
Every couple of months or so I develop a euphoria towards erlang which is consistently dampened by using the language to tackle problems for which the language admittedly was not designed in first place.
I guess most people start using a language for simple programming exercises first as opposed to building something like a Jabber/XMPP instant messaging server straightaway.
I hate to repeat myself but improving the standard library (by adding common functionality and making sure it performs decently) would do a lot to attract fresh talent to the erlang community and I hear that a certain rate of influx of "fresh blood" is a necessary prerequisite for success.
This is my second stab at Erlang (see the ring benchmark article for my first take on it). This time around I wanted to get a sense of how well Erlang and its libraries support more mundane tasks like e.g. XML parsing.
I am not a big fan of XML but it is the lingua franca of the web and any language that aspires to become "mainstream" has to support it in an efficient manner.
In order to get a feeling for how well Erlang is doing in this respect I am going to repeat my recent XML processing experiments with Groovy but this time using Erlang.
Depending on the publisher the format of the RSS file may vary slightly. The publication date is e.g. sometimes buried in a <dc:date> tag.
Likewise, the MP3 URL is sometimes not contained in a <link> tag but in the url attribute of the <enclosure> tag.
Unfortunately, the documentation of Erlang's XML parsing library is pretty scant and — apart from the xmerl User's Guide — I could not find any tutorials on xmerl on the web.
Again, a language aspiring to widespread adoption should have more material covering these kinds of basics.
using xmerl_scan with or without customization functions.
using xmerl_scan together with xmerl_xpath (an XPath 1.0 implementation).
I chose the first approach because it was better documented. Only after finishing the first cut of the parsing code did I find xmerl_xpath usage examples on a mailing list and played with it.
8     [ parse(F) || F <- FL ].
Erlang's filelib:fold_files() function is cool and a good example of how easy things should be.
beauty is in the eye of the beholder as they say and the promise of being able to write clean and attractive code is a major reason to pick up a new programming language. Again, maybe it's just me but I did not find the resulting Erlang code to be particularly attractive.
The XPath implementation appears to be incomplete: I could not use the | operator in an XPath expression to select several paths for example.
Anyway, that's just my $ 0.02 on XML parsing with Erlang. I am not an expert by any stretch of the imagination so feel free to point to anything I may have missed.
have come to realize that the conclusions I drew are skewed.
really exercise any of these strengths.
deployments (ejabberd, CouchDb and RabbitMQ just to mention a few).
erlang system and improving it continuously.
for what appears to be a more even-handed comparison.
I obtained a copy of Joe Armstrong's Erlang book recently. After reading through the first half I have found it to be a very commendable work, making a strong case for message passing concurrency — a different approach to building large scale and highly concurrent systems.
Write a similar program in some other programming language you are familiar with. Compare the results. Write a blog, and publish the results on the internet!
So, here we are, that's precisely what this article is about.
The "other programming language" is of course Python, in this particular case Stackless Python featuring tasklets and channels to counter Erlang's concurrency machinery.
This is my very first Erlang program, so please bear with me. If there's anything that could be done better, feel free to comment on it (in a polite way).
1 % sets up a ring of N processes, a message is passed M times around the ring.
3 -export([run_benchmark/2, loop/2, mloop/1, main/1]).
4 % builds a ring of N processes and send a message around the ring M times.
The function above sets up the ring. The lists:foldl construct is equivalent to Python's built-in reduce() function.
The process spawned on line 10 sends the M messages around the ring. The messages are just integers taken from the [M-1 .. 0] sequence.
The main process acts as process number N and invokes the function mloop() thus entering the appropriate receive loop on line 11.
The loop() function is executed by the first N-1 processes in the ring. These know their position in the ring (S) as well as their immediate neighbour (NextP) to which they forward all messages received.
The messages (R) are just integers. Values above zero keep the ring going, a zero value (the last message) results in the termination of the ring.
The mloop() function acts as the receive loop of the process number N. It "terminates" the messages that go around the ring. Its main purpose is to produce diagnostic output.
2 >> Erlang R11B-5 here (N=4, M=3)!
4 : Proc: <0.29.0>, Seq#: 1, Msg#: 2 ..
5 : Proc: <0.28.0>, Seq#: 2, Msg#: 2 ..
6 : Proc: <0.27.0>, Seq#: 3, Msg#: 2 ..
7 : Proc: <0.29.0>, Seq#: 1, Msg#: 1 ..
8 : Proc: <0.28.0>, Seq#: 2, Msg#: 1 ..
9 > Proc:  <0.1.0>, Seq#: 4, Msg#: 2 ..
10 : Proc: <0.27.0>, Seq#: 3, Msg#: 1 ..
11 * Proc: <0.29.0>, Seq#: 1, Msg#: terminate!
12 * Proc: <0.28.0>, Seq#: 2, Msg#: terminate!
13 > Proc:  <0.1.0>, Seq#: 4, Msg#: 1 ..
14 * Proc: <0.27.0>, Seq#: 3, Msg#: terminate!
15 @ Proc:  <0.1.0>, Seq#: 4, ring terminated.
Lines 4-6,9 are generated by the first message, lines 7,8,10 and 13 show the second message going around the ring — lines 11,12,14 and 15 are the result of the zero-valued (terminate ring!) message.
I have made an attempt to keep the python code as similar as possible to its Erlang counterpart. The run_benchmark() function below builds the ring of N tasklets (lines 9-18) and sends the M messages around it (lines 19-21).
One minor difference to note: the main process/tasklet here is not the last process in the ring but sends the messages (after spawning the last process on lines 16-18).
The loop() function is executed by the first N-1 processes in the ring as in the Erlang code. Apart from relaying the messages received, it mainly produces diagnostic output.
Each tasklet knows its position in the ring (S) and is given an input channel from which to read its messages (cin) as well as an output channel to which to write all the messages received (cout).
The last tasklet runs the mloop() function which mainly produces diagnostic output.
2 >> Python 2.5.1, stackless 3.1b3 here (N=4, M=3)!
4 > Proc: <6b870>, Seq#: 4, Msg#: 2 ..
5 : Proc: <6b730>, Seq#: 1, Msg#: 2 ..
6 : Proc: <6b770>, Seq#: 2, Msg#: 2 ..
7 : Proc: <6b7f0>, Seq#: 3, Msg#: 2 ..
8 > Proc: <6b870>, Seq#: 4, Msg#: 1 ..
9 : Proc: <6b730>, Seq#: 1, Msg#: 1 ..
10 : Proc: <6b770>, Seq#: 2, Msg#: 1 ..
11 : Proc: <6b7f0>, Seq#: 3, Msg#: 1 ..
12 @ Proc: <6b870>, Seq#: 4, ring terminated.
13 * Proc: <6b730>, Seq#: 1, Msg#: terminate!
14 * Proc: <6b770>, Seq#: 2, Msg#: terminate!
15 * Proc: <6b7f0>, Seq#: 3, Msg#: terminate!
Line 4-7 show the first message going around, lines 8-11 stem from the second message and lines 12-15 make the last (terminating) message visible (I guess the funny "order of execution" above arises from the fact that the Stackless python scheduler by default prefers the message receiver tasklets).
Model Name:	MacBook Pro 15"
In both cases the resulting total number of messages sent was between 1 and 9 million.
I expected that Erlang would clearly outperform Stackless python because this was the former's "sweet spot" scenario after all.
I was pleasantly surprised by python not only holding up to the challenge but winning it!
As can be seen from the diagrams depicting the results of the two benchmarks, Stackless Python performed much better than Erlang!
Eventually I came to believe that Erlang's I/O library is very slow and that that might be the reason for its sub-optimal performance in the benchmarks above.
I hence modified both sources (Erlang here, Stackless python here) and commented out all of the output.
Following that, I reran the benchmarks with the following results.
Now Erlang performed much more like a system that was built with message passing concurrency in mind BUT with Stackless python still close on its heels.
Even I as a Python aficionado am sometimes baffled in light of the gems that can be found in the Python treasure trove.
The Python community should stop worrying about the language falling out of fashion and/or being overtaken by RoR/Ruby or some other contender of the day.
Python's potential is so huge, all it takes is attracting notice to it e.g. to the fact that Python is ready for Our Manycore Future.
Last but not least, I find it's quite a shame that Stackless python seems to be treated like the "unloved stepchild" by the community.
Highly concurrent systems are the future of software and stackless facilitates the construction of such systems today.
Isn't such a capability worthy of being maintained, improved and expanded upon as opposed to building yet another web framework or whatever ..?


text:
Hudya is a Nordic fintech company. The company has developed a digital platform simplifying the purchase of traditional  consumer services such as electricity, telecom, insurance and banking services. Headquartered in Oslo, Norway, Hudya has 32 subsidiaries in Norway, Sweden and Denmark, offering their services to customers in all Scandinavian countries. Hudya's first product, Hudya Power, was launched in Norway in March 2017.

Hudya and Norwegian fintech company Payr announced their intention to merge in July 2019. In January 2020 Hudya announced their intention to conduct an initial public offering at Nasdaq First North.

The service
Hudya offers a Scandinavian-wide digital platform for consumer services, including electricity, telecom, insurance and consumer loans. The company provides customers with the services all available on the same digital platform, making it easy to keep track of the various service agreements for the customer.
In June 2017 Hudya Power was launched in Sweden in partnership with Energi Försäljning Sverige.

In December 2017 Hudya launched Hudya Power in Denmark, in partnership with Energi Denmark.

Hudya Insurance was launched in Norway in November 2018, as a partner for Frende Forsikring.

As of January 2020 Hudya has 60.000 users in the Nordics.

History
Hudya was established in 2016 by Børge Leknes, Morten Kvam and Pål Lauvrak. In an equity investment round in June 2017 Hudya raised NOK 14 million. The fund was raised from various local private investors, including Adolfsen Group. Hudya has also received NOK 3 million from Norwegian Research Council.

In January 2020 Hudya announced their intention to conduct an initial public offering (IPO) on the Stockholm-based stock exchange Nasdaq First North. The IPO valued Hudya at SEK 400 million.

Acquisitions and merger with Payr
Hudya Group follows a systematic growth strategy that includes strategic acquisitions to gather consumer-oriented services and products on their platform. The acquisitions include:
 In August 2017 Hudya acquired Swedish sales call centre Gradén Mattsson(221 employees).
 Acquisition of Norwegian credit broker Aconto AS and telecommunications operator Telipol in July 2018.  With the acquisition of Telipol Hudya got access to their 15.000 customers.
 Acquisition of Swedish Conteo AB in April 2019 as a first step to launch refinancing in Sweden.
 The acquisition of fintech company SEQR Group in Sweden in June 2019 gave Hudya access to SEQR Groups fintech back-end infrastructure.

In July 2019 Hudya Group merged with Norwegian fintech startup Payr, with Payr CEO Espen Einn becoming Chief Payments Officer in Hudya. This gained attention from several Norwegian media outlets, who reported on their ambition to build a Nordic fintech giant.

Net worth
As of July 2017, the net worth of Hudya is NOK 140 million.
In February 2020 the net worth of Hudya is reported to be SEK 400 million.

References

Financial services companies established in 2016
Privately held companies of Norway
Companies based in Oslo
2016 establishments in Norway
Insurance companies of Norway
Telecommunications companies established in 2016
Electric power distribution network operators
Energy companies established in 2016
Electric power companies of Norway
Security companies of Norway

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