I love gingerbread and Coco is getting rather a taste for it too.
There is a bakery right at the school gates and every now and again we buy gingerbread men for an after school snack. It's getting to the point where Henry will also request a gingerbread man every time we pass the bakery now so I thought it was about time I had a go at making some. Also, after our gingerbread from The Biscuiteers which was vastly superior to - but not quite as convenient as our normal supply I wondered if the homemade stuff would also be better.
I also needed an excuse to fill these beautiful cake tins which were kindly sent to me by Joules. I didn't even know they did a home range. They are going to be put to good use over the next few months I think with the onset of autumn and the baking season. The flowery one is truly massive, the perfect transportation for a victoria sponge... the smallest fox one (my favourite) looks like it's destined to become our permanent biscuit tin.
So anyway - the seemingly simple task of making some gingerbread somehow turned into an epic two stage process. That's what happens when you add Coco and Henry into the mix.
We followed this recipe from the BBC website.
Above is pretty much the moment that I wondered what I'd started... Henry was determined to press the buttons on the food mixer and Coco wanted to lick the syrup spoon... meanwhile I was just taking pictures. Hairy.
Of course Coco had to wear a princess dress throughout the whole process.
I'm not sure if it was because the dough got a little overworked... but our gingerbread spread a lot (and obviously I failed to didn't put the biscuits far enough apart) meaning we ended up with some interesting shapes.
The next day I tried to get prepared so that Coco could decorate a gingerbread man as her after school snack in the 20 minutes we have between getting back from picking her up from school and heading out to get Henry from nursery.
Mid - decorating. This went on for pushing on two hours if you include the 20 minute Henry collection interval.
Henry joined in when he got home too.
I feel like I want to put this batch down as my 'experimental' batch - it can only get better; I'm definitely up for some further practice batches, without the hindrance of children around possibly. The gingerbread tastes good and we have snacks for the foreseable future... but at some point in the process I decided that maybe I should make Coco a gingerbread house for her birthday in a months time. Can a gingerbread novice reach such dizzying heights? We'll see.
Thank you very much to Joules for the cake tins - now I just need an exercise plan to counteract all my baking plans.


In the first of a number of trial run rehearsals of her 10 minute roadshows, Mrs. McGoo will be offering round samples of her special melt in the moment homemade Scottish confectionery.
To book a table for the taste sensation moment, add a I:30pm booking in your Ocean View diary and after your dessert, you will hit the 2:30pm ten minute roadshow moment as a little extra to send you on your sweet way.
Think fudge, but think a much lighter flaky texture, think subtlety with flavours, and ask if you can sample the special oranges and lemons flavour samples.
And of course think Christmas special little gifts at the foot of the tree. with beautiful packaging as part of the show for the little ones, loved ones, family and friends. Orders will be taken on the day, delivery by hand in Pevensey Bay.
And did we mention that Mrs McGoo used to work for Thornton's? Think perhaps we should.
Is Mrs Goo headed for Dragon's Den or Harrod's?
The 10 minute roadshow rehearsal will be photographed at the Ocean Bakery, ready for the launch of the state of the art Mrs Magoo web platform nationwide on Friday 7 December.
More information about Mrs McGoo and her Scottish tablet confectionery here.


A court case brought in the NSW Land and Environment Court last week could threaten thousands of people who rent out their investment properties or holiday homes to short term renters.
What began as a neighbour dispute over noise in the central coast town of Terrigal has grown into a major test case for the right of investment property owners to rent out their homes for short periods such as a week or weekends.
The neighbour complained of loud and drunken parties next door when the six bedroom house was rented out for school holidays and weekends.
The neighbour took the case to the Land and Environment Court arguing the owner of the property should be prohibited from renting it out on a short term basis. He told the court the noise of drunken parties from next door was causing psychological damage to his family.
The case hinges on the definition of "dwelling house" as set out in the residential zoning rules of the local council. The neighbor argues "dwelling" means a degree of permanent occupancy, and argued if it is let out for parties on a regular basis it is in breach of the Environmental Planning Assessment Act and needs approval from the local council.
Michael McHugh of Stacks Business, the lawyer representing the owner of the property, said if noise was the problem then the neighbour should complain to police rather than seek to stop an owner letting out their property.
"We argue a property does not have to have permanent residents to be considered a dwelling house," Mr McHugh said. He said his client bought the property as an investment to let out to holidaymakers, and if she can't do that it would ruin her.
The owner didn't know it was being used for parties, and since the neighbour complained about noise she's asked renters to sign an agreement they won't use the house for noisy parties.
"This case will have enormous ramifications for thousands of property owners up and down the coast who rent out their investment properties or holiday homes for short term stays," said Mr McHugh.
The case continues and many investment and holiday home owners are watching closely.
In a strange twist, the judge told lawyers for both sides that she also owned a holiday home. The lawyers didn't object to her hearing the case.


text:
Q: How to create a url path to an unknown rails engine polymorphic record? I created a rails (4) application that embeds 2 isolated engines. One of the engine defines a polymorphic association that targets models in the main application and the second engine.
module Engine1
  class Engine1Model < ActiveRecord::Base
    belongs_to :testable, polymorphic: true
  end
end

module Engine2
  class Engine2Model < ActiveRecord::Base
  end
end

class MainAppModel < ActiveRecord::Base
end

In the Engine1ModelsController views (show/index) I would like to create a link to the associated model. When you know from which domain come the record you can do that:
polymorphic_path([main_app, record1.testable])

or
polymorphic_path([engine2, record2.testable])

But here the issue is that I don't know the origin of the model. How I can create a valid url path?

A: I created this helper:
module Engine1
  module ApplicationHelper
    def external_polymorphic_path(record)
      engine_name = record.class.name.deconstantize.underscore
      engine_name = 'main_app' if engine_name.empty?
      send(engine_name).polymorphic_path(record))
    end
  end
end

redpajama_set_name: RedPajamaStackExchange


Tricare is the Department of Defense's worldwide health care program for active duty and retired uniformed services members and their families.
Tricare For Life, available for Medicare-eligible beneficiaries age 65 and over (effective October 1, 2001).
Tricare Prime offers less out-of-pocket costs than any other Tricare option. Active duty members and their families do not pay enrollment fees, annual deductibles or co-payments for care in the Tricare network. Retired service members pay an annual enrollment fee of $230 for an individual or $460 for a family, and minimal co-pays apply for care in the Tricare network. Although prime offers a "point-of-service" option for care received outside of the Tricare Prime network, receiving care from a nonparticipating provider is not encouraged.
Tricare Prime enrollees receive most of their care from military providers or from civilian providers who belong to the Tricare Prime network. Enrollees are assigned a primary care manager (PCM) who manages their care and provides referrals for specialty care. All referrals for specialty care must be arranged by the PCM to avoid point of service charges.
Tricare Extra and Tricare Standard are available for all Tricare-eligible beneficiaries who elect not to enroll in Tricare Prime. Active duty service members are not eligible for extra or standard. There is no enrollment required for Tricare Extra or Standard - no annual enrollment fees, no enrollment forms. Beneficiaries are responsible for annual deductibles and cost shares. Beneficiaries may see any Tricare authorized provider they choose, and the government will share the cost with the beneficiaries after deductibles.
Tricare Extra is a preferred provider option (PPO) in which beneficiaries choose a doctor, hospital or other medical provider within the Tricare provider network. Network providers can be located by calling your local Tricare service center or visiting our website.
For more information about any of the Tricare options, please contact your local Tricare service center or visit the Tricare Website.


Brodie Croft and Robbie Rochow have dropped out of the extended squad named on Tuesday.
Ryley Jacks has been named as the current replacement for Cameron Munster at five-eighth while Jahrome Hughes still remains in contention to step in having retained his place on the extended bench.
Craig Bellamy will finalise his squad an hour before kick-off tomorrow night.


text:
G-Dragon: Preparing for new album was mentally, physically demanding
SEOUL, June 10 (Yonhap) — G-Dragon, the leader of the popular boy band BIGBANG, hit an unexpected snag when he returned as a soloist after four years: his band mate T.O.P's recent drug scandal.
"It was mentally and physically hard to prepare for the album because many things happened in the process," the singer said during his individual concert at Seoul World Cup Stadium in western Seoul on Saturday, the first leg of his concert tour of 19 cities around the world.
G-Dragon, however, did not directly mention the drug scandal involving T.O.P.
T.O.P was hospitalized Tuesday from an apparent overdose of benzodiazepine, a day after he was indicted without detention on charges of smoking marijuana. He is suspected of smoking the banned substance four times with a 21-year-old female trainee singer at his home in Seoul in October last year.
This file photo shows G-Dragon. (Yonhap)
"Kwon Ji Yong," the fourth solo album by G-Dragon, conquered iTunes album charts in 39 countries upon its release on Thursday, despite the scandal. Its title track "Untitled" also swept the number one spots on major domestic online music charts and all five category charts on China's QQ Music.
"I felt great upon hearing the good news," the 29-year-old rapper-singer said after performing three of his hit songs "Heartbreaker," "Breathe" and "A Boy" on stage.
"I came to open this concert without any troubles thanks to you. So, I thank you once again."
Kwon Ji-yong is G-Dragon's real name. As the title suggests, the album has a strong autobiographical aspect to it, with lyrics focused on G-Dragon's personal character and growing pains as he is about to enter his 30s.
"Personally, it was a time to look back on me pondering over what is the true nature of myself and who this guy Kwon Ji-yong really is after many years of life under the stage name G-Dragon. It was a precious time that I realized what I did not know of myself and what I had forgotten."
After Seoul, G-Dragon is scheduled to perform in three Asian, eight North American, four Oceanian and three Japanese cities in the world tour titled "Act III, M.O.T.T.E."
Exhibition in L.A. to shed light on Korean alphabet
Voting begins in S. Korea's presidential election
Scandal-ridden Park Yu-chun opens up about marriage
Girls' Generation to release sixth full-length album next month
Lee Min-ho's DMZ wild life documentary to start next week
Kang Ho-dong reunited with Lee Soo-geun for tvN's new variety show
BTS album 'Love Yourself: Her' sells over 1.2 mln copies: data

redpajama_set_name: RedPajamaCommonCrawl


text:
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We're always looking for talented, high-energy individuals to join our team. If you want to help drive our "sweet" future success then we'd love to hear from you!
Thank you for your interest in employment at Manny's Sweet Treats! Please complete the application form below.
Are you seeking permanent position?


text:
RYCJ
Do You Look Like A Writer?
Read more by RYCJ
On the outskirts of things, it really doesn't, and shouldn't, matter what you look like. But then there's that other thing, like work put into designing 'eye-catching' book covers because ...like it or not... many people do buy books based on the first thing they see... THE COVER.
At any rate, this thought came to me while watching a TV program about Janis Joplin... and after receiving a nudge by a casual observer pointing out something I hadn't paid much attention to before. During the telling of Janis Joplin's story, one of the narrators talked about how Janis changed her look... to look more like the rock stars of that time. This observation was similar to the nudge I got. Once upon a time I used to wear the bad heels and black suits. Today I rock hats, twists and attire often fashioned from the first piece of fabric I touch in the morning. While I can't speak for Janis, I can speak for myself. I'm not trying to dress or look like an artist...or writer.
And no, none of this is a big deal. I happen to be a staunch believer that content rules. It's like the time a professor got to telling his class how, for a period of time, after getting fed up with life he used to sleep on park benches. Let me tell you, it changed my perception of anyone I saw holding signs and sleeping on streets ever since.
As writers, and aspiring authors, we really must stay focused on helping potential audiences get beyond whether or not we look like writers, or even judging books by the cover. Now, I won't go as far as to promote stripping covers from books, or perhaps stripping authors down to taking portraits in our birthday suits, but do raise the question to keep focused on what really matters.
Rhonda Y.C. Johnson
RYCJ/OEBooks
Karen McManus on Poking Holes & Finding Your Writing...


I am The Body Temple Luminary, lighting the path for women who feel lost and out of ease in their body. I guide my clients back to themselves by combining the scientific and clinical approach of Functional Medicine with the art of Spiritual Healing.
My work is designed for women who are looking to uncover and heal the true root cause of their dis-ease, find more peace and calm in their mind and connect more deeply to their self expression and purpose.
Via my signature program, The Body Temple Method you get to end the merry-go-round of symptom chasing and begin healing from the inside out.
The Body Temple Method is a deeply private journey back home, back to yourBody Temple. It is the healing of both the physical, emotional, mental and spiritual; connecting you closer to who you really are, allowing you to fully step into all of your gifts and be who you are here to be.
Whether you've been suffering from Fibromyalgia, Lupus, Thyroid dysfunction or depression, chronic fatigue, severe anxiety or anything in between, healing all parts of you – body, mind and spirit – is The Body Temple Method. This is your path to true, long-lasting, easy, health, happiness and wealth.


Everybody has a computer, a laptop, an iPad,... Computers are ubiquitous. Do you sell or repair computers? Do you give computer lessons? Are you a programmer? Do you develop software. Do you write for a computer magazine?
Then .computer is the ideal extension for you!


Background: Anaemia is a common disease in children resulting in many problems including impaired cognitive performance, behavioural and language development, and scholastic achievements. The use of simple investigations to make an accurate diagnosis is of paramount importance. Material and Methods: The present study is a prospective study done on 100 children in the school-going age group of five years to fifteen years. The automated haematology analyser was used to calculate various haematology parameters and the iron profile was used to confirm the diagnosis of iron deficiency anaemia. Results: Mild iron deficiency anaemia is the commonest type of microcytic anaemia in school age children. The differentiation between iron deficiency anaemia and thalassaemia trait is possible with the RBC indices provided by the automated analysers. The significant tests in differentiation between iron deficiency anaemia and thalassaemia trait were found to be mean corpuscular volume (MCV), mean corpuscular haemoglobin (MCH) and red blood cell (RBC) count. In iron deficiency anaemia, MCV and MCH are decreased proportionately with decrease in haemoglobin value. The RBC count is slightly decreased, normal or in few cases slightly increased. Peripheral smear examination is also helpful to corroborate the findings. Conclusion: Use of basic haematological parameters is useful for the diagnosis of anaemia in the Indian children where expensive tests cannot be afforded by many.
Anaemia is a common problem and is considered as one of the silent killer diseases as it causes a variety of problems over a period of time. Some of the effects of anaemia may not be reversed if it is not treated in time, especially if the anaemia is in childhood. The various medical conditions that lead to anaemia encompass nearly the full spectrum of human disease. Anaemia results in impaired cognitive performance, behavioural and language development, and scholastic achievements in children. Therefore, detection of even mild degree of anaemia may be of great significance.
About one third of the world's population is found to be anaemic and the prevalence of anaemia worldwide among the 6-12 years age group is about 36%, and 7% in developing countries.[1, 2] India is one of the countries with very high prevalence of anaemia in the world. Nutritional anaemia is a major public health problem in India and is primarily due to iron deficiency. Microcytic hypochromic is seen in iron deficiency anaemia (IDA), thalassaemia trait (TT), lead poisoning, and anaemia of chronic disease (ACD). Various haematological indices have been proposed as simple and inexpensive tools to diagnose IDA to reduce the investigative costs.
This study was done to evaluate the routine haemocytometric parameter profile in the diagnosis of iron deficiency anaemia as accurately as possible.
The present study is a prospective study done on 100 children in the school-age group of five years to fifteen years, who came to the hospital. A detailed history was elicited, and a thorough clinical examination was undertaken.
The automated haematology analyser Sysmex XS-1000i was used to calculate various haematology parameters. The analyser is a 5-part differential which uses Fluorescent Flow Cytometry principle or WBC-Diff, Direct Current–Sheath Flow principle for RBC, haematocrit, platelet count and Noncyanide, Sodium Lauryl Sulphate (SLS) for haemoglobin estimation.
The children with haemoglobin value of less than 12 grams per decilitre (gm/dl) in this age group were selected and the children with MCV less than normal (microcytic hypochromic anaemia) were included. All other haematology parameters in these patients were recorded. This was followed by a detailed peripheral smear examination. Peripheral smear examination was done on slides stained with Leishman's stain.
The iron profile included serum iron, serum ferritin and total iron binding capacity (TIBC). Erba Manheim Chem 7, an automated clinical chemistry analyser, was used for ferritin measurement. It is based on immunoturbidimetric quantitative assay in which antigen antibody reaction by endpoint method is used. For serum iron and TIBC, VITROS 5,1 FS Chemistry System analyser was used.
Data obtained were compiled, tabulated, and analyzed. Descriptive statistics were applied. Means were compared by using one-way ANOVA. Chi-square tests were applied to find out the association between two attributes. A p-value of < 0.05 was statistically significant. SPSS version 20 was used to analyze the data.
Out of the children coming to the Paediatric outpatient department, we selected 100 children in the school going age group (5-15 years) who had anaemia. The children were divided into two age groups, 10 and under and over 10 years (adolescents). Out of the 100 children, 67% were 10 years or below and 33% were over 10 years of age. The mean age of the children was 9 years. The female to male ratio in the present study was found to be 1.2:1.
mild, moderate, and severe anaemia having haemoglobin values of 10.5-11.5g/dl, 7-10.4g/dl and <7g/dl respectively. In this study, 71% (69% below 10 years of age) had mild anaemia, 24% (70.8% below 10 years of age) cases had moderate and only 5% (80% over 10 years of age) had severe anaemia. There was no statistical significance found between age and the severity of anaemia as determined by Pearson chi-square test (p=0.77).
In the present study, 38 out of 54 girls had mild anaemia, 12 had moderate and 4 had severe anaemia. Out of 46 boys, 33 had mild anaemia, 12 had moderate and only 1 boy had severe anaemia. No significant correlation was found between the severity of anaemia and gender as determined by Pearson chi-square test (p=0.468).
The MCV, MCH, MCHC, packed cell volume (PCV), RBC count and red blood cell distribution width (RDW) were analysed and correlated with the grades of anaemia. The details are shown in Table 1. A statistically significant relation was found between severity of anaemia and MCV (p = 0.001), with MCH (p = <0.001), with PCV (p = <0.001) and RBC count (p = 0.003). MCV, MCH, and PCV were mild, moderate and severe anaemia, while RBC statistically significant which differed between count was significant only when compared between mild and moderate cases. A statistically non-significant relation was found between RDW and severity of anaemia (p = 0.537).
Iron profile and haemoglobin electrophoresis were done to corroborate the diagnosis. Out of the 78 cases of iron deficiency anaemia in this study, 57 were mild, 17 were moderate and only 4 cases were severe grade.
When Bonferroni Correction was applied as an adjustment for all pair-wise comparisons of means, there was a significant association between the MCV, MCH, and RBC counts and the three types of microcytic anaemia (IDA versus TT and ACD versus TT). MCV, MCH and RBC count were the most significant indices for comparison between IDA and TT as per the results of this study. For differentiating between IDA and ACD, no single index was found significant statistically.
Anaemia is functionally defined as an insufficient RBC mass to adequately deliver oxygen to peripheral tissues, thereby causing tissue hypoxia. Although red cell mass is the gold standard in the assessment of anaemia, its measurement is cumbersome and rarely performed outside of clinical research. Anaemia can also be defined as a reduction in haemoglobin (Hb) concentration, haematocrit or number of RBCs per cubic millimetre. As per the World Health Organization (WHO), the haemoglobin threshold for children aged 5.00 to 11.99 years is 11.5 g/dl and for children aged 12.00-14.99 years is 12.0 g/dl.
Iron deficiency is the most common nutritional deficiency in the world; responsible for a staggering amount of ill health, lost productivity, and premature death. Anaemia resulting from iron deficiency affects approximately 2 billion people or 34% of the world population. It accounts for 2.4 per cent of the total Disability Adjusted Life Years (DALYs) worldwide. In India, the NFHS-2 data indicates a fairly extensive prevalence of iron deficiency among the children between age 6-35 months portending a difficult future for children.
Children with iron deficiency may present with no signs or symptoms and come to medical attention only because of abnormalities in laboratory tests; with the features of the underlying disorder responsible for the development of iron deficiency; with the manifestations common to all anaemias like lethargy and dyspnoea; or one or more of the few signs and symptoms considered highly specific for iron deficiency, namely, pagophagia, koilonychia, and blue sclerae.
Inherited disorders of iron metabolism include congenital atransferrinemia (congenital absence of transferrin is an autosomal recessive condition), congenital microcytic hypochromic anaemia with iron overload and microcytic anaemia due to an isolated abnormality of malabsorption and defective utilization of iron.
In our study, the mean age of the children was similar to a study by Muthayya and Sudhagandhi et al. The female to male ratio in the present study was found to be 1.2:1 similar to Sudhagandhi's study. However, in a study by Sabale et al, the prevalence of anaemia in female and male children was found to be almost equal. Verma et al in their study in Punjab found that anaemia was higher in girls over the age of 12 years. Thus, most of the studies conducted in India on the prevalence of anaemia in children showed a higher prevalence among girls compared to boys which could be explained by gender preference (better food intake in boys) and menorrhagia at menarche.
In our study the mean MCV, MCH and RBC counts in ACD, iron deficiency and thalassemia trait were similar to some studies[3, 39, 40] It was found that Milunsky in his text mentioned that MCV less than 60 fL and MCH less than 20 picograms (pg)/cell are considered indicators of a low probability of having IDA and a higher probability of thalassemia minor, which co-relate with our study. In a study by Madan et al, even values as MCV below 80 fL and MCH value below 27 pg were found to be very sensitive markers in the detection of beta thalassaemia.
As per Batebi, the RBC count is a valuable index for differentiation between IDA and TT with a high sensitivity but a low specificity. RBC count is increased in severe chronic cases of iron deficiency and in the initial response to treatment. RBC count and RDWI appear to be reliable and useful index for the differentiation of TT and IDA, as both indices have more than 80% sensitivity and specificity in the differentiation of IDA and beta-TT.[43, 44] RDWI was not done in this study.
It was observed in some studies,[45, 46] that there was an inverse relationship between RDW and the haemoglobin value, in iron deficiency anaemia. The sensitivity was 92.1% and specificity was 90.9% of RDW in detecting iron deficiency. However, RDW completely failed to differentiate between IDA and beta-TT. In our study also, RDW was not useful in differentiation between any of the microcytic anaemias.
In our study we concluded that haemoglobin and PCV does not differ significantly but MCV, MCH, and RBC count differ significantly, between thalassaemia trait and ACD and iron deficiency. Aslam et al. in their study had similar results. As per a study by Fahim and Ahmed, the most striking and consistent finding in typical heterozygous beta thalassaemia is the combination of a relatively high RBC count (> 5×1012/l), with a slightly low to normal haemoglobin and a disproportionate reduction of MCV and MCH in relation to the haemoglobin.
The findings of our study were comparable with most of the studies used for discrimination between IDA and TT. Low MCV, low MCH out of proportion to haemoglobin value and a high RBC count go largely in favour of TT. These indices combined with a thorough peripheral smear examination could diagnose TT cases efficiently.
Mild iron deficiency anaemia is the commonest type of microcytic anaemia in school-age children. The differentiation between iron deficiency anaemia and thalassaemia trait which is very important to prevent iron overload is possible with the RBC indices provided by the automated analyzers. The differentiation can further be aided by a detailed peripheral smear examination. These findings are useful in the Indian population where expensive tests cannot be afforded by many. The necessity to treat iron deficiency because of its effects on the growth and development of children must be emphasized.
Sethi V, Goindi G, Kapil U. Prevalence of anaemia amongst primary school children (6-11 years) in national capital territory of Delhi. Indian J Pediatr. 2003; 70: 519-20.
Gomber S, Bhawna, Madan N, Lal A, Kela K. Prevalence and etiology of nutritional anaemia among school children of urban slums. Indian J Med Res. 2003; 118: 167-71.
Urrechaga E, Borque L, Escanero JF. The role of automated measurement of RBC subpopulation in differential diagnosis of microcytic anaemia and beta thalassaemia screening. Am J Clin Pathol. 2011; 135: 374-9.
Ghys T, Malafait R, Van J. Performance evaluation of the Sysmex XS-1000i automated haematology analyser. Int J Lab Hem. 2008: 1-7.
McKenzie SB, Otto CN. Introduction to Anaemia. In: McKenzie SB, Williams JL, Zeibig E. (Editors) Clinical laboratory Hematology. 2nd Edition. Prentice Hall; 2010; 146-73.
Bridges KR, Pearson HA. Anaemia and other red cell disorders. McGraw Hill companies; 2008.
Lanzkowsky P. Classification and diagnosis of anaemia during childhood. In: Manual of Pediatric Hematology and Oncology, 4th Edition. Elsevier Inc; 2005 1-11.
World Health Organization/UNICEF/UNU. Iron Deficiency Anaemia: Assessment, Prevention, and Control. A guide for programme managers. Document WHO/NHD/01.3. Geneva, Switzerland: World Health Organization; 2001.
Wu AC, Lesperance L, Bernstein H. Screening for Iron Deficiency. Pediatrics in Review. 2002; 23:171-7.
More S, Shivkumar VB, Gangane N, Shende S. Effects of Iron Deficiency on Cognitive Function in School Going Adolescent Females in Rural Area of Central India. Anaemia.2013. http://dx.doi.org/10.1155/ 2013/819136.
Guidelines for Control of Iron Deficiency Anaemia. NRHM handbook. Adolescent Division. Ministry of Health and Family Welfare. Government of India, 2013.
International Institute for Population Sciences (IIPS) and ORC Macro (2000). National Family Health Survey (NFHS-2), 1998-99. Mumbai: IIPS.
Miller JL. Iron Deficiency Anaemia: A Common and Curable Disease. Cold Spring Harb Perspect Med. 2013; 3. http://perspectivesinmedicine.cship.org.
Goodnough LT, Nemeth E. Iron deficiency and related disorders. In: Greer JP, Rodgers GM, Paraskevas F, Glader B, List AF, Arber DA, et al., editors. Wintrobe's Clinical Hematology 13th Edition. Philadelphia: Lippincott Williams & Wilkins; 2014: 617-42.
Hoffbrand AV, Hershko C, Camaschella C. Iron metabolism, iron deficiency and disorders of haem synthesis. In: Hoffbrand AV, Catovsky D, Tuddenham EG, Green AR. Editors. Postgraduate Haematology. 6th Edition. Wiley Blackwell; 2011; 26-45.
Adamson JW. Iron deficiency and other hypoproliferative anaemias. In: Longo DL, Kasper DL, Jameson JL, Fauci AS, Hauser SL, Loscalzo J, et al. Editors. Harrison's Hematology and Oncology. 17th Edition. USA: The McGraw-Hill Companies; 2010; 70-80.
Will AM. Disorders of iron metabolism: iron deficiency, iron overload and sideroblastic anaemias. In: Arceci RJ, Hann IM, Smith OP. Editors. Pediatric Hematology. 3rd Edition. Massachusetts: Blackwell Publishing; 2006; 79-99.
Lerner NB, Sills R. Iron deficiency anaemia. In: Kliegman RM, Behrman RE, Schor NF, Stanton BF, St. Geme JW, editors. Nelson's Textbook of Pediatrics. 19th Ed. Philadelphia: Saunders, Elsevier. 2011; 1655-8.
Brittenham GM. Disorders of iron metabolism: iron deficiency and overload. In: Hoffman R, Benz EJ, Shattil SJ, Furie B, Cohen HJ, Silberstein LE et al (eds). Hematology: Basic Principles and Practice. 4th Edition. Philadelphia: Churchill Livingstone; 2005.
Perkins S. Diagnosis of Anaemia. In: Kjeldsberg CR. Editor. Practical diagnosis of hematologic disorders. 4th Edition. Chicago: ASCP Press. 2006; 1-16.
Firkin F, Chesterman C, Penington D, Rush B. Editors. Hypochromic anaemia: Iron deficiency and sideroblastic anaemia. In: DeGruchy's Clinical Hematology in Medical Practice. 5th Edition. New Delhi; Wiley India; 2011; 37-61.
Bain BJ, Lewis SM, Bates I. Basic haematological techniques. In: Bain BJ, Lewis SM, Bates I, editors. Dacie and Lewis Practical Haematology. 10th Edition. Philadelphia; Churchill Livingstone: 2006; 26-54.
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