3Division of Cardiology, Department of Medicine, Duke University Medical Center, Durham, North Carolina, USA.
Address correspondence to: Christopher Kontos, Department of Medicine, Division of Cardiology, Department of Pharmacology and Cancer Biology, Duke University Medical Center, Durham, North Carolina 27710, USA. Phone: 919.684.2119; E-mail: cdkontos@duke.edu.
J Clin Invest. 2016;126(9):3188–3191. https://doi.org/10.1172/JCI89963.
Angiopoietin-1/Tie2 (ANG1/Tie2) signaling is well documented as regulating angiogenesis and vessel maturation. This pathway is complicated by involvement of the orphan receptor Tie1, which has been implicated as both a positive and negative regulator of ANG1/Tie2 signaling, and ANG2, which can serve as both a Tie2 agonist and antagonist, depending on the context. Two papers in this issue of the JCI provide new insight into this complicated pathway. Korhonen et al. reveal that Tie1 acts to modulate the effects of ANG1 and ANG2 on Tie2 in vitro and in vivo. Kim et al. demonstrate that ANG2 acts as a Tie2 agonist in non-pathological conditions, whereas in the setting of inflammation, ANG2 functions as a Tie2 antagonist and promotes vascular dysfunction. Both studies indicate that inflammation promotes cleavage of the ectodomain of Tie1 and that this cleavage event corresponds with the switch of ANG2 from a Tie2 agonist to an antagonist. The results of these studies lay the groundwork for future strategies to therapeutically exploit this pathway in diseases characterized by adverse vascular remodeling and increased permeability.
Tie receptors (tyrosine kinases with immunoglobulin and epidermal growth factor homology domains) are a family of receptor tyrosine kinases (RTKs) that are expressed primarily in endothelial cells (ECs) (1, 2). Both Tie1 (encoded by TIE1) and Tie2 (encoded by TEK) are required for appropriate developmental angiogenesis and vessel maturation in mice and are therefore attractive therapeutic targets for a variety of diseases characterized by vascular dysfunction (3–5).
Angiopoietin-1 (ANG1), the first Tie2 ligand identified, behaves as a canonical RTK agonist when bound to Tie2 (6). ANG1/Tie2 signaling promotes EC survival and vascular integrity, is associated with a stable, quiescent EC phenotype, and is likely the primary factor responsible for baseline Tie2 activation in resting adult tissues (7–10). ANG1 also plays a role in angiogenesis, serving as an EC chemoattractant, and acts as a weak EC mitogen to promote vascular enlargement (11–13). ANG2, which has homology to ANG1, was originally described as a competitive antagonist of ANG1/Tie2 signaling based on the fact that it binds Tie2 with nearly the same affinity as ANG1 (~3 nM) and blocks ANG1-induced Tie2 phosphorylation (14, 15). However, ANG2 was later shown to induce Tie2 activation in a variety of contexts, including at high concentrations, in stressed ECs, and in a fibrin matrix model of angiogenesis (16–19). Thus, ANG2 is now largely accepted as a context-dependent agonist/antagonist for Tie2.
Unlike Tie2, Tie1 is considered to be an orphan receptor, as it is unable to bind directly to any of the angiopoietins, and no other natural ligands have been identified. This lack of a natural ligand for Tie1 has significantly complicated investigation of its function (20). However, in the presence of Tie2, Tie1 activation by ANG1 or a modified recombinant oligomeric ANG1 protein (COMP-ANG1) has been shown to occur (21, 22). Prior in vitro studies suggested that Tie1 functions primarily as a negative regulator of Tie2 signaling (reviewed in ref. 23), but recent in vivo data show that Tie1 can both negatively and positively regulate Tie2 signaling during angiogenesis, depending on the cellular context. For example, Tie1 expression in stalk cells stabilizes Tie1-Tie2 heteromultimers at the cell membrane, perpetuating ANG1/Tie2 signaling, while Tie1 expression in tip cells negatively regulates Tie2 surface presentation (24).
In addition to vascular development, angiogenesis, and vessel stabilization, the angiopoietins and Tie receptors have been shown to play important roles in inflammation, both in vitro and in vivo. ANG1/Tie2 activity is generally associated with antiinflammatory signaling. For example, ANG1 reduces the adhesion and transendothelial migration of leukocytes, in part via decreased expression of inflammatory adhesion molecules and cytokines following recruitment of the NF-κB inhibitor ABIN-2 (7, 25, 26). Conversely, Tie1 has been associated with EC activation and inflammation. In vitro, Tie1 positively regulates the expression of proinflammatory genes (27, 28), while EC-specific deletion of Tie1 reduces expression of inflammatory adhesion molecules and results in a dose-dependent improvement of atherosclerosis in Apoe–/– mice (29).
The role of ANG2 in inflammation may also be context dependent. Based on the antagonistic effects of ANG2 on ANG1/Tie2 signaling, ANG2 may be considered a proinflammatory factor. Moreover, ANG2 release from Weibel-Palade bodies sensitizes ECs to the inflammatory effects of TNF-α (30), and ANG2 has also been shown to promote β2 integrin–dependent myeloid cell infiltration in vivo, even in the absence of inflammatory stimuli (31). Conversely, ANG2 has been shown to promote nitric oxide release and is considered to be atheroprotective in Apoe–/– mice, supporting an antiinflammatory function in this model (31).
A better understanding of how the angiopoietins and Tie receptors function in inflammation may have clinical implications. For example, targeting this system may be valuable in numerous disorders of dysregulated inflammation and vascular integrity. Indeed, data from several clinical studies indicate that elevated levels of ANG2 are associated with poor prognosis and increased mortality in sepsis, regardless of microbial etiology (32, 33), while elevated levels of ANG1 are associated with decreased mortality (34). Additionally, compared with their WT counterparts, mice heterozygous for Ang2 exhibit a substantial improvement in survival following both cecal ligation-perforation and LPS-endotoxemia models of sepsis (35).
While much has been learned about angiopoietin/Tie receptor signaling over the last 20 years, two critical questions have remained unanswered. First, what are the mechanisms that are responsible for the context-dependent effects of ANG2 in both inflammation and angiogenesis? Second, how does Tie1 regulate angiopoietin/Tie2 signaling? In this issue, two complementary studies provide critical insight into both of these questions. Specifically, Kim et al. address the issue of the context-dependent effects of ANG2 (36), while Korhonen et al. investigate the role of Tie1 in angiopoietin/Tie2 signaling (37). Together, these studies provide clinically relevant information by focusing on angiopoietin/Tie receptor signaling in the regulation of vascular remodeling in infection and/or inflammation.
Using inhibitory antibodies targeting ANG2 and Tie2 along with powerful mouse models, Kim et al. demonstrated that ANG2 antagonism of Tie2 promotes loss of vascular integrity during infection with Mycoplasma pulmonis (36). Moreover, compromised vascular integrity was further exacerbated by antibody-mediated inhibition of Tie2 activation. Consistent with previous studies of septic patients and preclinical sepsis models, Kim and colleagues report a reduction of vascular remodeling, including vascular leak during infection, by inhibitory ANG2-targeting antibodies or by administration of exogenous ANG1; however, this group also determined that ANG2 acts as a Tie2 agonist under pathogen-free conditions, whether it is expressed systemically via adenovirus or in the endothelium of transgenic mice. Moreover, inflammation (infection, TNF-α, or LPS) was shown to induce a critical contextual switch in the effects of ANG2, promoting the shift from agonist to antagonist. Kim et al. demonstrated that this change in function is mediated through a feedback loop that involves AKT inhibition, FOXO1 activation, increased ANG2 expression, and subsequent adverse vascular remodeling. But the question remains: how does inflammation allow ANG2 to become a Tie2 antagonist? Kim et al. partially addressed this question by capitalizing on the results of Korhonen et al. (37) and have shown that inflammation induces shedding of the Tie1 ectodomain, which renders ANG2 a Tie2 antagonist, although the precise mechanism or mechanisms of this process remain undetermined.
In a complementary paper, Korhonen et al. address the role of Tie1 in the regulation of Tie2 by both ANG1 and ANG2 (37). Using in vitro techniques, including fluorescence (Förster) resonance energy transfer (FRET) and fluorescence lifetime imaging microscopy (FLIM), these authors demonstrated that Tie1 and Tie2 interact in the absence of ligand, confirming previously published results (38). Both in cultured ECs and in conditional Tie1-knockout mice, endothelial expression of Tie1 was required for the agonist effects of both ANG1 and autocrine ANG2 on Tie2 activation and vascular remodeling. Furthermore, in support of the Kim et al. study (36), Korhonen and colleagues also have shown that endotoxemia-induced cleavage of the Tie1 ectodomain reduces ANG2-mediated Tie2 phosphorylation, decreases downstream AKT activation, and increases nuclear localization of FOXO1. Based on these findings, Korhonen and colleagues propose a model in which Tie1 interacts with Tie2 at baseline to promote angiopoietin signaling, but is cleaved in the setting of inflammation, resulting in loss of ANG2 agonist activity and thereby promoting adverse vascular remodeling and leakiness. Importantly, although Tie1 cleavage diminishes the agonist effects of ANG1, these effects are not completely lost.
These two groups of investigators used distinct approaches to show that inflammation-mediated loss of Tie1 expression is responsible for the context-dependent effects of ANG2. Furthermore, these studies provide an important step toward better understanding this complex ligand/receptor signaling pathway. It should be noted, however, that a limitation of both reports is that, although the loss of Tie1 was associated with ANG2's antagonist effects, a causal link between Tie1 and ANG2 was not demonstrated. Such a relationship could possibly be demonstrated by generating a noncleavable Tie1 mutant to determine whether sustained expression of Tie1 at the plasma membrane in the setting of inflammation would reverse the antagonistic effects of ANG2. Although the site of TNF-α–induced Tie1 cleavage has been identified (39), the specific protease that mediates this cleavage has not, and targeting receptor cleavage would likely prove challenging. The exact mechanism by which the presence of Tie1 alters ANG/Tie2 signaling is also unclear. For example, does the Tie1-Tie2 interaction alter ANG binding? Are ANG-mediated signals differentially transduced in the presence of Tie1? Or is ANG/Tie2 signaling altered by the Tie1 endodomain? There is some precedent for all of these possibilities. Structural data have shown that a specific domain within ANG1 confers agonist activity, although this was suggested to result in dissociation of Tie1 and Tie2 rather than association of the receptors (40). The Tie1 endodomain has been shown to associate with SHP2, although the functional significance of this interaction remains unclear (41). As noted by Korhonen et al., acute and chronic inflammation may have very different effects on Tie1 cleavage and ANG/Tie2 signaling (37), and understanding these differences may help explain how loss of Tie1 could have beneficial effects in the context of atherosclerosis and detrimental effects in acute infectious or inflammatory processes.
In light of these new findings, it is important to recognize that both Kim et al. and Korhonen et al. demonstrated agonist effects of ANG2 following overexpression of ANG2 in the absence of inflammation or infection (36, 37). Because ANG2 is typically expressed at high levels only in remodeling vessels or in pathological conditions, it seems unlikely to behave as an agonist under physiological conditions. The notion that ANG2 does not routinely serve as an agonist is further supported by the fact that ANG2 is upregulated by FOXO1 following inhibition of ANG1/Tie2/AKT signaling. Prior reports of ANG2 activating Tie2 have been primarily in cultured ECs rather than in vivo; thus, the context in which this ANG2 action on Tie2 has been shown previously may not be physiological. However, the results presented by Kim et al. and Korhonen et al. highlight an important therapeutic opportunity — the potential to convert ANG2 into an agonist in conditions such as sepsis — in which ANG2 concentrations are very high. Further studies to improve our understanding of the mechanisms by which inflammation cleaves Tie1 and alters the function of ANG2 as well as identification of downstream signaling pathways that regulate this switch could provide therapeutic targets for effectively converting high circulating levels of ANG2 into an ANG1 equivalent, thereby promoting vascular normalization, inhibiting vascular leak, and reducing morbidity and mortality in conditions such as sepsis. Korhonen et al. show that, although COMP-ANG1 is able to partially overcome the inhibitory effects of inflammation on ANG1-mediated Tie2 activation, these effects are nonetheless blunted (37), likely in part because of the high levels of antagonistic ANG2. Approaches to promote ANG2 agonist activity may therefore be more effective than delivery of ANG1 or COMP-ANG1 or may act synergistically with ANG1-based therapies.
Recent evidence provides support for the potential efficacy of pharmacological approaches for redirecting ANG2 toward vascular normalization. Because ANG2 fails to induce Tie2 phosphorylation, it has long been speculated that the antagonist effects of ANG2 might be linked to recruitment of a protein tyrosine phosphatase (PTP). One such candidate molecule is vascular endothelial–PTP (VE-PTP), which has been shown to associate with, dephosphorylate, and inhibit Tie2 (42). Although some evidence suggests that VE-PTP is not responsible for ANG2's antagonistic effects (43), pharmacological inhibition of VE-PTP potently activates Tie2, an effect that persists even in the presence of high ANG2 concentrations, such as in diabetic macular edema and cerebral ischemia (44, 45). VE-PTP inhibition has been advanced to clinical trials for eye disease (46, 47) and has shown promise in LPS-induced vascular leak (48). It is unknown whether VE-PTP's effects on Tie2 are influenced by the presence of Tie1 or its cleavage. Elucidation of the detailed molecular mechanisms responsible for ANG2's antagonist effects may result in additional pharmacological approaches to promote vascular normalization in disease states.
Further investigation of the complex and dynamic interactions among the Tie receptors, the angiopoietins, and their molecular regulators, including VE-PTP and other as-yet-unknown modulators, will be critical for maximizing the therapeutic potential of this signaling pathway. In this regard, the results of Kim et al. and Korhonen et al. provide critical new insights into the mechanisms regulating ANG2's context dependence and its regulation by Tie1.
This work was supported in part by NIH grant R01 HL124444 and the Duke Medical Scientist Training Program (T32 GM007171).
Reference information: J Clin Invest. 2016;126(9):3188–3191. doi:10.1172/JCI89963.
See the related articles at Tie1 controls angiopoietin function in vascular remodeling and inflammation and Opposing actions of angiopoietin-2 on Tie2 signaling and FOXO1 activation.
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Fiedler U, et al. Angiopoietin-1 and angiopoietin-2 share the same binding domains in the Tie-2 receptor involving the first Ig-like loop and the epidermal growth factor-like repeats. J Biol Chem. 2003;278(3):1721–1727.
Kim I, Kim JH, Moon SO, Kwak HJ, Kim NG, Koh GY. Angiopoietin-2 at high concentration can enhance endothelial cell survival through the phosphatidylinositol 3′-kinase/Akt signal transduction pathway. Oncogene. 2000;19(39):4549–4552.
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Daly C, et al. Angiopoietin-2 functions as an autocrine protective factor in stressed endothelial cells. Proc Natl Acad Sci U S A. 2006;103(42):15491–15496.
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Saharinen P, et al. Multiple angiopoietin recombinant proteins activate the Tie1 receptor tyrosine kinase and promote its interaction with Tie2. J Cell Biol. 2005;169(2):239–243.
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Singh H, Tahir TA, Alawo DO, Issa E, Brindle NP. Molecular control of angiopoietin signalling. Biochem Soc Trans. 2011;39(6):1592–1596.
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Chan B, Sukhatme VP. Suppression of Tie-1 in endothelial cells in vitro induces a change in the genome-wide expression profile reflecting an inflammatory function. FEBS Lett. 2009;583(6):1023–1028.
Chan B, Yuan HT, Ananth Karumanchi S, Sukhatme VP. Receptor tyrosine kinase Tie-1 overexpression in endothelial cells upregulates adhesion molecules. Biochem Biophys Res Commun. 2008;371(3):475–479.
Woo KV, et al. Tie1 attenuation reduces murine atherosclerosis in a dose-dependent and shear stress-specific manner. J Clin Invest. 2011;121(4):1624–1635.
Fiedler U, et al. Angiopoietin-2 sensitizes endothelial cells to TNF-α and has a crucial role in the induction of inflammation. Nat Med. 2006;12(2):235–239.
Scholz A, et al. Angiopoietin-2 promotes myeloid cell infiltration in a β2-integrin-dependent manner. Blood. 2011;118(18):5050–5059.
Fisher J, et al. Elevated plasma angiopoietin-2 levels are associated with fluid overload, organ dysfunction, mortality in human septic shock. [published online ahead of print July 15, 2016]. Crit Care Med.
Hahn WO, et al. Host derived biomarkers of inflammation, apoptosis, and endothelial activation are associated with clinical outcomes in patients with bacteremia and sepsis regardless of microbial etiology. Virulence. 2016;7(4):387–394.
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Campochiaro PA, et al. Enhanced benefit in diabetic macular edema from AKB-9778 Tie2 activation combined with vascular endothelial growth factor suppression. Ophthalmology. 2016;123(8):1722–1730.
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Get Scheumann Stadium essential facts below. View Videos or join the Scheumann Stadium discussion. Add Scheumann Stadium to your PopFlock.com topic list for future reference or share this resource on social media.
American football stadium on the Ball State University campus in Muncie, IN, US
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The stadium in 2019
Location in Indiana
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Location in the United States
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Former names
Ball State Stadium (1967-2005)
Tillotson Avenue, Muncie, Indiana 47306
40°12?58?N 85°25?0?W / 40.21611°N 85.41667°W / 40.21611; -85.41667Coordinates: 40°12?58?N 85°25?0?W / 40.21611°N 85.41667°W / 40.21611; -85.41667
16,319 (1967-1997)
22,500 (2007-present)
Grass (1967-1996, 2001-2006)
Astroturf (1997-2000)
FieldTurf (2007-present)
September 16, 1967 (1967-09-16)
Walter Scholer and Associates
Ball State Cardinals (NCAA) (1967-present)
The Scheumann Stadium, formerly known as the Ball State Stadium, is in Muncie, Indiana. It is primarily used for football, and it is the home field of the Ball State University Cardinals. The stadium opened in 1967, and it has a capacity of 22,500 for football games.
Anticipating rapid growth after transitioning from a teacher's college to a comprehensive college, the Ball State University Board of Trustees approved construction of a new athletic stadium one mile north of campus in 1965. The stadium was completed in 1967 with a capacity of 16,000 for football. It replaced the previous stadium closer to campus, on University Avenue across from Ball Memorial Hospital. The site is now used as a band practice field. A grandstand on the south end of the stadium was added in the 1990s, increasing the capacity to 22,500. In 2005, the stadium was renamed after Ball State alumni and benefactors John B. and June M. Scheumann.
In 2007, a $13 million renovation of the stadium was completed. Planned improvements included new lighting for night games, enclosing the north end zone with lawn seating to create a 22,500-seat horseshoe-shaped venue, new concession stands, a brick facade, a larger press box, and private luxury suites. On August 29, 2013, a new video board was installed.
For a time, notable Ball State alumnus David Letterman jokingly campaigned via his television program to have the stadium named after him. When the stadium was named after the Scheumanns for their donations to the university, Indianapolis mayor Bart Peterson joked that the city would rename the I-465 Beltway as the David Letterman Expressway. Indianapolis was Letterman's childhood home town.
Mean game attendance at the stadium was at the highest in school history in 2008, with an average of about 21,000 visitors per game. This can be attributed to the team's success, as well as recent renovations to the stadium. A school record 10,546 students attended the September 6, 2008 game versus the Navy Midshipmen. The game was televised nationally on ESPN, and Ball State won, 35-23.[1] The all-time record attendance for a game at Scheumann Stadium is 23,861, set on November 25, 2008 when Ball State completed an undefeated regular season by beating MAC rival Western Michigan. The Cardinals won handily, 45-22, pushing their record to 12-0, but they lost to Buffalo in the MAC championship game and to Tulsa in the GMAC Bowl.
List of NCAA Division I FBS football stadiums
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Scheumann Stadium home page at Ball State Athletics
Stadium renovation home page
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Words: Ashley Berry
On Friday, February 19, the Delta Scholars held their annual event, Food for the Soul, in celebration of Black History Month from 6 – 8pm. Students and families from around the county came to Centennial to enjoy a diner comprised of various "soul foods." Along with a dinner, 15 students performed various poems, songs, and dances. Some of the performers were a part of the Delta Scholars, but some were students at Centennial who wanted to take part in the event.
Last year, the Delta Scholars asked students from Mr. O'Brien's English classes to perform at the event as well. Unfortunately, because of all the snow and canceled school days, his students could not help this year. This gave more students in Delta Scholars, who would not have considered it otherwise, a chance to perform. According to Shalonda Holt, a advisor of the Delta Scholars, approximately 120 people came to Food for the Soul and the Delta Scholars raised around $700.
Ashley Berry
Delta Scholars
Shalonda Holt
"It will be okay": Ms. Jones' jobs
Galante's galavant through careers
Centennial's holiday wishlist
The path to success is not linear: A conversation with Carr
Is junior year the hardest year?: Fact or fiction
It's perfectly normal: Breaking the period stigma
What do runners think about when running?
What is ARL?


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Dorel Juvenile Group Recalls Eddie Bauer Rocking Wood Bassinets Due to Infant Suffocation Hazard
Picture of recalled rocking wood bassinet
Eddie Bauer Rocking Wood Bassinets
The bottom locking mechanism can fail to lock properly if a spring is not installed, allowing the bassinet to tip to one side and cause infants to roll to the side of the bassinet. This poses a suffocation hazard to infants.
About 97,000
Dorel; toll-free at (877) 416-0165, 8 a.m. to 5 p.m. ET Monday through Friday, or www.djgusa.com and click on Safety Notices for more information.
This recall involves Eddie Bauer Rocking Wood Bassinets. The bassinets have a dark brown wooden headboard and footboard and a cotton bassinet that comes in a variety of colors and patterns. "Eddie Bauer" is printed on a metal plate on the bassinet's footboard. These bassinets have a dial at the base of the footboard that locks and unlocks the rocking motion of the bassinet. Recalled bassinets have the following model numbers: 10632, 10639, 10832, 10835, 10839 and BT021. The model number is located under the mattress, on the top surface of the mattress support board or on the wash and care label.
Consumers should immediately stop using the recalled bassinets and contact Dorel to obtain a free repair kit, which includes a spring and new instructions for assembly.
Dorel has received 17 reports of incidents with the recalled bassinets involving infants primarily younger than three months old. In two of the incidents, infants were reported to have had breathing difficulties after they rolled into the side of their bassinets.
Target, Toys R Us and Sears stores nationwide and online at Ebay and other websites from December 2007 through January 2011 for about $150.
The recalled children's sleepwear garments fail to meet the United States flammability standards for children's sleepwear, posing a risk of burn injuries to children.
The base of the silicone nipple has a fine slit that can cause the nipple to detach from the plastic shield, posing a choking hazard.
Children's Robes Recalled by HulovoX Due to Violation of Federal Flammability Standards and Burn Hazard
The recalled robes fail to meet flammability standards for children's sleepwear, posing a risk of burn injuries to children.
Native Creation Recalls Sweaters Due to Violation of the Federal Lead Paint Ban
Paint on the zipper pull contains levels of lead that exceed the maximums allowed. Excessive levels of lead can be toxic or cause other health problems to young children if the paint is ingested.
Longwood Forest Recalls Angel Line Bunk Beds with Angled Ladders Due to Serious Entrapment and Strangulation Hazards; 2-Year-Old Child's Death Reported
The metal hook fastening the ladder to the top bunk bed frame can move away or detach from the bed frame when the ladder is lifted, causing the gap between the ladder step and bed frame to open wider than 3.5 inches, posing serious entrapment and strangulation hazards.
Karmas Far Recalls Infant Bath Seats Due to Drowning Hazard (Recall Alert)
The bath seats fail to meet the federal safety standard for infant bath seats, including requirements for stability and leg openings, and can tip over while in use, posing a drowning hazard to babies.


According to the US Department of State, anyone under the age of 16 is classified as a child and only qualifies to receive a passport book or card valid for five years. Under this system, assuming the parents start traveling with their child early, someone can hold three (or more, if lost or stolen) passports before he or she turns 16.
Every time a parent is applying for a passport for their child, whether it's the first time as a baby or a renewal at age 12, they must use Form DS-11, the same for adult passports. In addition to this, evidence of the child's US citizenship, proof of the relationship with the parents, the parents' IDs (passports are acceptable), photos (age progression in the case of a renewal), and fees must be provided with the application.
Where does Form DS-3053 fit in?
must be included as part of that application. Both parents or legal guardians must authorize the US Department of State to issue their child's passport by appearing in person with the child at a regional passport office.
However, because we live in a modern world, with children with only one parent, no parents (emancipated), one parent who may not be able to appear with the other, etc., the State Department allows for exceptions to be made provided the appropriate paperwork is included.
Form DS-3053 allows for one parent to appear in person with the child for their passport application, provided the other has completed the form giving their approval for a passport to be issue in their child's name in front of a notary public with their ID. Therefore this form isn't so much a necessity in applying for a child's passport as much as a statement of consent if one parent cannot appear.
This form may also be used if neither legal guardian is able to appear to apply for the child's passport. Both must fill out the form in front of a notary public with their IDs and authorize a third party – like Travel Visa Pro – to apply on behalf of their child. If the child has only one legal guardian who is unable to appear, proof of this must be submitted along with a copy of Form DS-3053.
If only one parent or legal guardian can appear, but he or she happens to have sole legal authority of the child, evidence of this must be presented with the passport application, e.g. a court order granting the parent sole legal custody of the child or one specifically permitting the individual to apply for the child's passport. Other documents may be submitted for cases including the death or legal incompetence of one parent.
1. Consider how old your child is. Both children under 16 and those between 16-18 may use Form DS-11 when applying for a new passport, but children under 16 must have the approval of both parents or legal guardians, barring the aforementioned exceptions (Form DS-3053).
2. Applications for children's passports can only be made in person at a regional passport office or a US embassy or consulate. Please make an appointment when necessary. Third party agencies like Travel Visa Pro can process your child's passport on your behalf with a copy of Form DS-3053 from both parents.
3. Bring the child, both parents, the parents' IDs and copies, Form DS-11, evidence of the child's US citizenship, proof of the relationship with the parents, and photos (age progression in the case of a renewal) to the consulate or passport office.
4. Payment varies. A $25 Execution Fee is required at regional passport offices in addition to the $80 processing fee. Expedited service is available for $60.
5. Wait until your passport is returned by mail, or ready to be picked up.
6. Start traveling with your child!


text:
The Van Maren Show Episode 142: Will Roe v. Wade be overturned next year?
Posted on November 24, 2021 by Jonathon Van Maren
LISTEN TO THIS PODCAST ON OTHER PLATFORMS
This entry was posted in Blog Post and tagged Eric Scheidler, Jonathon Van Maren, Pro-Life Action League, The Van Maren Show, Van Maren Show.
It is really possible that, by this time next year, Roe v. Wade will be no more
Justin Trudeau's crusade against crisis pregnancy centres (and other stories)
5 thoughts on "The Van Maren Show Episode 142: Will Roe v. Wade be overturned next year?"
joe a martin says:
If Trump was still our president, I believe Ros vs Wade would be overturned.
Jonathon Van Maren says:
The president isn't relevant to how the Court rules.
Navi says:
President Biden is a man that repeatedly sold out his pro-life values to get more and more political power. Perhaps most notable was orchestrating a successful smear campaign against an anti-Roe Supreme Court nominee, which he credits for saving abortion on demand. Roe v. Wade being overturned while he's at the height of his power and completely unable to do anything about it would be delicious. It would be even better if Ted Kennedy was still around to see it, but sadly that wasn't meant to be.
Very interesting discussion. While it's an article of faith among Democratic pundits (and some Republicans, no doubt), I think the "sleeping giant" theory of Roe v. Wade is wrong. A national backlash sweeping Democrats into power would be disastrous (they would nuke the filibuster, codify abortion until birth into federal law, pack the Supreme Court, and take other actions to increase their political power such as DC statehood), and we can't decisively rule it out until after the election, but it's a lot less likely than the conventional wisdom suggests. After the Texas ban went into effect, many of the so-called experts said the GOP had finally "caught the car" and that it would reset the race for control of congress in 2022. But in fact, the Texas law had zero effect on Joe Biden's approval rating or on the generic congressional ballot poll. Biden's approval continued to decline, and Republicans are now ahead in the generic ballot (they were behind in August). Everyone went about their lives after the dust cleared. The Dave Chappelle saga was a bigger deal and lasted longer than the outrage over the Texas law. Which, it must be pointed out, banned 90% of abortions in the second-biggest state.
https://trends.google.com/trends/explore?geo=US&q=dave%20chappelle,texas%20abortion%20law
The real data (election returns) tell a similar story. Democratic pundits Paul Waldman and Greg Sargent called the Virginia gubernatorial election the "first big test of the new politics of abortion":
https://www.washingtonpost.com/opinions/2021/09/02/get-ready-our-first-big-test-new-politics-abortion/
Indeed, Virginia is a battleground state that's become increasingly tricky for Republicans over the course of the Obama-Trump era. This resulted in abortion extremists winning total control of the state government and rolling back pro-life policies. Terry McAuliffe heavily campaigned on abortion, even campaigning inside an abortion clinic. But all his fearmongering, combined with two months of media hysteria, fell completely flat. Pro-life Republicans won the elections for governor, lt. governor, attorney general, and control of the house. Meanwhile, in Texas itself, Republicans picked up a house seat in a district that went heavily for Joe Biden. These results shouldn't be a surprise. Some previous "sleeping giant" moments:
– George W. Bush signed the Partial-birth Abortion Ban into federal law, the first such federal abortion bill without a health exception, in 2004. Abortion advocates were outraged, staging the March for Women's Lives in Washington DC. Hollywood speakers and high profile Democrats like John Kerry and Hillary Clinton flocked in. Abortion advocates argued he was only one justice away from overturning Roe but Bush was re-elected later that year, going on to appoint two Supreme Court justices.
– In 2013 Texas tried to pass a law banning late-term abortions and making earlier ones rarer and safer for the woman. Wendy Davis staged an 11 hour filibuster, complete with her bright pink runners, and abortion advocates held a massive protest in the legislature. She successfully delayed passage of the bill until a second special session. She then ran for governor, with the national media declaring her as the Great White Hope for turning Texas blue and this generation's equivalent of Governor Ann Richards. Davis went on to lose so badly that the liberal Texas Monthly magazine named her Bum Steer of the Year.
http://www.texasmonthly.com/wp-content/uploads/2014/12/texas_monthly_bum_Steer_wendy_davis_texas_democrats_0.jpg
– In early 2016, Justice Antonin Scalia suddenly passed away. Senate Republicans, led by Mitch McConnell, insisted on leaving the seat vacant until after the election. Several other justices were very elderly at the time, and Hillary Clinton (much like her crony McAuliffe) campaigned heavily on the prospect of Roe v. Wade being overturned. Her opponent promised that the justices he appointed would "automatically" overturn Roe. After a very long campaign, Donald Trump won the election and Republicans held both chambers of Congress. As Planned Parenthood president Cecile Richards said after the election, Roe v. Wade was on the ballot – and Roe v. Wade lost.
– In 2020 Justice Ruth Bader Ginsburg, a cultural icon for liberal feminists and abortion advocates, passed away less than two months before the election. Early polls showed a solid majority of Americans against replacing her. But Mitch McConnell and his caucus believed they had a great nominee that Americans would support once they got to know her, and that it was the right thing for the country anyway. So they worked to confirm Amy Coney Barrett (who is openly pro-life). Polls showed McConnell was right, with support for confirmation growing to a majority as she became better known. By the time the final presidential debate rolled around, the Supreme Court and abortion were not even an issue. Although President Trump was defeated, Democrats lost seats in the House and could only get to 50-50 in the Senate (handing the balance of power to the pro-life moderate Joe Manchin). Appointing the would-be fifth justice didn't hurt Republicans at all.
It's very much possible that Roe v. Wade will have blown over long before the election next November, or that the backlash will end up helping Republicans. Democrats will inevitably try (a second time) to codify abortion up to birth into federal law, which would go against public opinion and make for good attack ads. The prospect of street violence raised by Eric is an interesting one as well, though hopefully it will not come to be. If it does happen though, looting and rioting is a very bad issue for Democrats even when it's supposedly for a popular cause (protesting the murder of George Floyd).
There's another path the Supreme Court could take, which would be upholding the Mississippi law without affirming or overruling the "central holding" in Roe and Casey. This would allow the courts to consider subsequent abortion restrictions (such as heartbeat laws) without having to overrule Dobbs. Some discussion about the likelihood and ramifications of this possibility would be interesting.
This is a very good analysis.
Leave a Reply to Navi Cancel reply


Iron bar magnets Fill a bowl or pan with some water. Find something that will float on the water. Lay the magnet on top of this object. The magnet will twirl in the water until it comes to rest pointing exactly north and south due to the magnetic force of the North Pole. Carefully take the magnet off the water and lay it on a hard (preferably wooden) surface in the exact same north-south position. Strike one end of the magnet hard with a hammer repeatedly. The force may loosen the tiny magnetic domains in the bar so they can point north like they are supposed to [source: Newton].
Magnets that have lost their strength Sometimes you can recharge a magnet that has lost some of its original charge. If you can find a very strong magnet, repeatedly rub it across your weakened magnet. The strong magnet will realign the magnetic domains inside the weakened magnet [source: Luminaltech].
Magnet stacking One way to make weak magnets stronger is by stacking more of them together. There's a big problem with this, though. Magnets attract each other in opposite directions, which will lower their overall strength. If you can find a way to hold or clamp them together, so they're aligned in the same direction, they will in fact produce a stronger magnetic field [source: Balbach].


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Publisher: Includes bibliographies and indexes.Vol. 2: Catalogue of the collection / Bruce Davis..
Includes bibliographies and indexes.Vol. 2: Catalogue of the collection / Bruce Davis.
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to depart in their trading ships?
but they perished on your cliffs.
and seaweed fastened to the rocks.
than the salt stretch of your beach.
none venture to that spot.
from the splendour of your ragged coast.
the heel is cut deep.
I have lost the third in the packed earth.
rain has furrowed the roots.
the root snapped under your weight.
And you climbed yet further!
give life—I am almost lost.
in the larch-cones and the underbrush.
pallid under the cracked pitch.
you must rise to refute.
broken by the rasp and after-rasp.
blue violets, moss, cliff, river-violets.
of your own white surf.
and circle the jagged rocks.
gather and pour weight upon the beach.
will be witness of your track.
the lintel of wet sand with froth.
and drift laurel-wood from hot coasts!
we will answer with a shout.
and cherish and shelter us.


text:
Lo scudo fiscale è una tipologia di regolarizzazione in materia tributaria e penale simile ad un condono perché inibisce l'azione penale e di accertamento tributario nel caso di alcuni illeciti tributari e penali. Esso sana alcuni eventuali comportamenti illeciti o irregolari effettuati dal contribuente riguardo alla produzione e detenzione di capitali detenuti all'estero derivanti da redditi non denunciati e presumibilmente imponibili e dall'acquisto di immobili con i suddetti capitali, tramite il pagamento di una imposta forfettaria, una tantum, di valore inferiore alle normali aliquote tributarie.

Lo scudo fiscale nel diritto internazionale
Nei condoni fiscali avvenuti in passato in Francia, Regno Unito e Stati Uniti, non era mai stata garantita la riservatezza degli evasori, come principio legato al concetto di perdono. Agli evasori è sempre stato richiesto di dichiarare la propria identità, l'ammontare della propria evasione, e di pagare le tasse arretrate con gli interessi, ricevendo in cambio la decadenza delle conseguenze penali ed uno sconto nelle sanzioni. In questo modo tali governi hanno utilizzato gli scudi fiscali per comprendere tutti i meccanismi di evasione utilizzati dai propri concittadini.

Lo scudo fiscale in Italia

Lo scudo 2009
Il governo Berlusconi IV con la pubblicazione nella Gazzetta ufficiale italiana del D.L. 194/2009, cosiddetto "Mille proroghe", ha riaperto i termini per poter usufruire dello scudo fiscale (cd. scudo fiscale Ter), per favorire il rimpatrio o la regolarizzazione delle attività finanziarie e patrimoniali illegalmente detenute all'estero fino al 31 dicembre 2008, a fronte del pagamento di una somma del 5%, a titolo di imposte, interessi e sanzioni.
Inoltre viene previsto il pagamento delle imposte sui redditi relativi alle attività scudate prodotti nel periodo dal 1º gennaio 2009 alla presentazione della dichiarazione riservata, da farsi entro il 15 dicembre 2009. Il rimpatrio è obbligatorio per tutti i paesi del mondo esclusi quelli della Comunità Europea e la Norvegia.

Tale provvedimento consente in tal modo il pagamento del minimo della sanzione prevista in caso di scoperta di violazione delle norme sul monitoraggio dei capitali (dal 5% al 25%), e non intacca il rendimento fruttato dai capitali all'estero nel periodo in cui non vi sono state pagate imposte dovute in Italia. Lo scudo non prevede la sanatoria totale per tutti i reati e per tutte le irregolarità relativa all'IVA.

Il governo stimò un rientro di capitali per un totale di circa 300 miliardi di euro, il Ministero dell'Economia previde un gettito fiscale una tantum di 3-5 miliardi di euro.

A metà febbraio 2010 secondo le stime governative sono rientrati grazie allo scudo fiscale 80 miliardi di euro, di cui circa 60 miliardi dalla Svizzera, 4 miliardi circa rispettivamente dal Lussemburgo e dal Principato di Monaco ed i restanti 12 miliardi da altri paesi.

Precedenti dichiarazioni di Silvio Berlusconi
Il 31 marzo 2008, in campagna elettorale per le elezioni politiche del 2008, Silvio Berlusconi aveva dichiarato:

L'iter parlamentare
Il provvedimento sullo scudo fiscale è contenuto in un emendamento del 15 luglio 2009, proposto da Chiara Moroni e Maurizio Fugatti, al decreto legge "anticrisi" , votato dal Parlamento il 24 luglio 2009 con una questione di fiducia, e causa di aspre contestazioni dell'opposizione e di Avvenire, che lo bollò come «una soluzione premia-furbi». (vedi l'intero iter parlamentare)

A seguito di un emendamento del Senato, che ha introdotto molte delle norme più contestate del provvedimento (tra cui l'estensione dell'estinzione dei profili penali dell'evasione), la Camera ha approvato una mozione di fiducia il 30 settembre 2009; in tale occasione il presidente della Camera Gianfranco Fini ha parlato di "oggettive anomalie procedurali nella complessiva vicenda dell'iter del decreto, trasmesso dal Senato a dieci giorni dalla sua scadenza", mentre l'opposizione ha denunciato il "riciclaggio di stato". Il voto finale alla Camera sulla conversione del decreto-legge è avvenuto il 2 ottobre; la maggioranza è riuscita a far passare la norma con 20 voti di scarto, a fronte di 56 parlamentari del PdL e 30 dell'opposizione assenti (23 del PD, 6 dell'UDC, 1 dell'IdV), scatenando le polemiche sull'assenteismo in aula.

Effetti fiscali e penali

Riservatezza delle dichiarazioni di emersione
Un ulteriore profilo di preoccupazione è stato espresso a proposito della provenienza dei capitali espatriati, generalmente considerati frutto di evasione fiscale, e come tali in teoria soggetti, in caso di accertamento da parte del fisco, al pagamento delle imposte evase (Irpef, Irap, Iva, la cui evasione non era comunque coperta dallo scudo, interessi e sanzioni) per un totale facilmente superiore al 50% del capitale. Ciò viene evitato, nel provvedimento di scudo fiscale 2009, dalla forma riservata delle dichiarazioni di emersione, "coperte per legge da un elevato grado di segretezza", che non possono perciò essere utilizzate a sfavore del contribuente in sede amministrativa né giudiziaria.

Se il denaro rimpatriato viene utilizzato per l'acquisto di capitali azionari, esso mantiene il carattere di riservatezza. Se invece esso viene reinvestito in attività imprenditoriale, ciò comporta la rinuncia alla riservatezza. Una tale norma penalizza l'utilizzo a fini imprenditoriali ed economici, mentre favorisce l'uso dei capitali scudati per accrescere le rendite.

Non è invece possibile il ricorso allo scudo per i contribuenti già soggetti ad accertamento fiscale o a procedimenti penali già avviati. Il termine della sanatoria è fissato al 15 dicembre 2009.

Tale aspetto di riservatezza delle dichiarazioni di emersione distingue nettamente lo scudo fiscale italiano del 2009 dai provvedimenti concernenti il rientro dei capitali in Gran Bretagna e negli Stati Uniti, paesi nei quali chi vuole regolarizzare i capitali esportati deve pagare in toto le imposte evase negli anni precedenti.
Nella maggioranza dei casi, lo scudo fiscale italiano assume i connotati di un vero e proprio condono.

La non punibilità dei reati
Lo scudo fiscale prevede anche la non punibilità di reati tributari, che prevederebbero pene fino a 6 anni di reclusione:
 omessa e infedele dichiarazione dei redditi;
 dichiarazione fraudolenta mediante utilizzo di fatture per operazioni inesistenti;
 falsa rappresentazione di scritture contabili obbligatorie;
 occultamento o distruzione di documenti;
 false comunicazioni sociali (falso in bilancio).
La non punibilità è estesa ad altri reati, commessi per eseguire o nascondere i reati precedenti, tra cui:
 falsità materiale,
 falsità ideologica in atto pubblico,
 falsità nelle scritture private,
 soppressione e occultamento di atti
 false comunicazioni sociali.

Il provvedimento riguarda in tal modo anche quelle società controllate estere, spesso situate in paradisi fiscali, verso cui confluiscono i capitali sommersi.
Condizione necessaria per l'esclusione della punibilità è l'assenza di un procedimento penale già in corso alla data di presentazione della dichiarazione riservata.

Sono stati sollevati dubbi di costituzionalità, circa la necessità di approvare la legge con le maggioranze previste per le leggi di amnistia e indulto (due terzi del Parlamento). L'eventualità è tuttavia esclusa, anche sulla base del recente orientamento della Corte Costituzionale secondo cui «La previsione di ipotesi di non punibilità subordinata a condotte dirette ad ottenere la sanatoria di precedenti comportamenti, non è ritenuta qualificabile come amnistia, da ultimo con ordinanza 9 aprile 2009, n. 109».

Le limitazioni al potere di accertamento tributario

La presentazione della dichiarazione riservata determina l'estinzione delle sanzioni tributarie previste per le violazioni commesse, salvo che il procedimento amministrativo sia già avviato al momento della presentazione della dichiarazione.

Inoltre, nel limite dell'importo indicato nella dichiarazione riservata, il contribuente può, presentando copia della stessa, opporre la sanatoria ad eventuali accertamenti futuri dell'Amministrazione per periodi anteriori al 31 dicembre 2008. Ad esempio, qualora siano contestati maggiori redditi per  euro, il contribuente può opporre di aver sanato  euro, evitando il recupero dell'imposta e le sanzioni. Se l'importo sanato è inferiore (ad esempio  euro), può essere accertata la differenza.

In ogni caso l'adesione allo scudo fiscale non può essere utilizzata come elemento a sfavore del contribuente (ad esempio per presumere maggiori redditi partendo dall'esistenza di patrimoni esteri non dichiarati). Per inciso, data la riservatezza delle dichiarazioni, gli uffici non dispongono di informazioni sui contribuenti aderenti, salvo che siano questi ultimi a fornirle ad esempio per opporsi ad accertamenti. Il segreto vige anche in caso di richiesta di accesso ai conti bancari.

Deroghe alla normativa comunitaria antiriciclaggio
In contrasto con la normativa comunitaria vigente, lo scudo fiscale consente ai soggetti sottoposti alla legge antiriciclaggio di non effettuare le segnalazioni di operazioni sospette di riciclaggio (limitatamente ai reati fiscali "coperti" dallo Scudo). Secondo il testo di legge, "il rimpatrio del denaro non è da considerarsi di per sé operazione sospetta".

Secondo Roberto de Vita, docente dell'Accademia della Guardia di Finanza, "è difficile immaginare in concreto quali possano essere gli indici rivelatori di una anomalia degna di necessitare una segnalazione di operazione sospetta".

Mario Draghi, governatore della Banca d'Italia, nella sua relazione del 29 ottobre 2009 in occasione della giornata mondiale del risparmio, sottolineava come "è opportuno un intervento che nell'ambito dello scudo fiscale ribadisca la regolare applicazione della normativa antiriciclaggio".

Il presidente del GAFI, il Gruppo di Azione Finanziaria Antiriciclaggio dell'OCSE, secondo Der Spiegel avrebbe scritto al ministro Giulio Tremonti segnalando i potenziali effetti negativi dello scudo fiscale italiano.

Già nell'ambito dei due scudi fiscali precedenti (2001 e 2003), le segnalazioni sospette delle banche erano state solo 94 in tutto, su un totale di 78 milioni di euro incassati.

Con circolare del 16 febbraio 2010 il Ministero dell'Economia e Finanze ha precisato che "le Banche e gli altri intermediari sono tenuti a dare applicazione agli obblighi di adeguata verifica della clientela ai sensi degli articoli 15 e 16 del D.lgs 231/2007 (antiriciclaggio) e in particolare a:

- identificare e verificare l'identità del cliente compreso l'eventuale titolare effettivo. Una particolare cura dovrà essere messa nell'evitare l'utilizzo di prestanome o di soggetti interposti;

- raccogliere informazioni dettagliate sull'attività del cliente e sulle sue capacità economiche, sulla natura e sullo scopo/destinazione del rapporto continuativo connesso all'operazione scudata"

Quindi le Banche e gli altri intermediari dovranno inoltrare segnalazioni di operazioni sospette ai sensi della normativa antiriciclaggio, qualora riscontrino anomalie che possono nascere da clienti che non sembrano avere o non avevano mai dichiarato le disponibilità economiche, il tenore di vita, il giro d'affari compatibile con l'entità delle somme rimpatriate.Sempre la stessa circolare del MEF stabilisce che le operazioni di scudo fiscale possono rientrare tra quelle per le quali è necessaria l'Adeguata Verifica Rafforzata (ai sensi dell'art. 28 del D.Lgs 231/07) per cui le Banche e gli altri intermediari sono tenuti ad acquisire informazioni ed eventuali riscontri documentali sulla costituzione dei capitali oggetto del rientro dall'estero, atti a ricostruire l'origine degli stessi.

Inoltre le Banche dovranno svolgere un controllo costante nel corso del rapporto continuativo, anche successivamente all'operazione di emersione, al fine di rilevare eventuali elementi che possano condurre a individuare profili di anomalia meritevoli di approfondimento anche ai fini della segnalazione di operazioni sospette.

L'effettività dello scudo in tema di Iva
Vi sono dubbi sulla certezza degli effetti fiscali dello scudo in tema di Iva. La sentenza della Corte di cassazione n. 20068 del 18 settembre 2009 infatti ha stabilito il principio secondo cui non è ammissibile per l'amministrazione finanziaria rinunciare all'accertamento tributario dell'Iva. Tale sentenza è il risultato della condanna dell'Italia, da parte della Corte di Giustizia Europea (causa C-132/06), adita a suo tempo dalla Commissione europea, e relativo annullamento del condono Iva 1997-2002 (L. 289/2002 art. 8 e 9), per violazione della sesta direttiva europea in materia di Iva.

Le obiezioni e i rischi dello scudo

Il rischio di utilizzo dei capitali da parte della criminalità organizzata
Diversi osservatori hanno segnalato il rischio che la criminalità organizzata faccia uso dello scudo fiscale per riciclare i proventi dei propri traffici illeciti (spaccio di droga, prostituzione, traffici d'armi, finanziamento del terrorismo), frequentemente detenuti all'estero.

Tale rischio viene segnalato a causa di vari provvedimenti contenuti nello scudo fiscale:
 la riservatezza garantita alle dichiarazioni di emersione;
 l'estensione dello scudo alle società controllate e collegate estere;
 il mancato obbligo di segnalazione di operazioni sospette in materia di riciclaggio di denaro da parte di chi riceve le dichiarazioni anonime di emersione.

In tal modo, le organizzazioni criminali potrebbero disporre di denaro pulito fresco, in grado di consentir loro di acquisire le imprese in difficoltà a causa della crisi.

La ricezione del provvedimento da parte delle banche
Le banche hanno generalmente messo in atto procedure per prepararsi a fare da tramite per l'emersione dei capitali esportati. Alcune banche di minori dimensioni, fra le quali Banca Etica, hanno dichiarato che non intendono o non sono in grado di assistere la loro clientela in queste operazioni.

Il giudizio dell'OCSE
Secondo l'OCSE, «lo scudo fiscale dovrà essere visto dai contribuenti come una misura straordinaria, nell'ambito dell'impegno generale alla trasparenza sugli scambi di informazioni sulle tasse recentemente concordato a livello internazionale, altrimenti i contribuenti potrebbero arrivare alla conclusione che sono probabili altri condoni fiscali».

Gettito effettivo ed utilizzo delle risorse
La bozza di legge finanziaria 2010 prevede un gettito da scudo fiscale di 3,7 miliardi, così ripartiti:
 500 milioni per il rifinanziamento delle missioni internazionali,
 400 milioni al 5 per mille,
 400 milioni all'autotrasporto,
 350 milioni al fondo per l'università,
 250 milioni per il fondo di solidarietà dell'agricoltura,
 130 milioni ai libri di testo scolastici,
 130 milioni alle scuole private,
 130 milioni per l'adempimento degli impegni dello Stato derivati dalla partecipazione a banche e fondi internazionali,
 100 milioni alle popolazioni colpite da calamità naturali.

Note

Voci correlate
 Fisco
 Economia sommersa
 Condono
 Politica economica
 Gettito fiscale

Collegamenti esterni
 Rossella Galli, Mini guida allo scudo fiscale, Soldionline.it, 4 settembre 2009

Diritto tributario
Strumenti di ambito tributario

redpajama_set_name: RedPajamaWikipedia


Okay, I know what some of you are thinking...You...speeding? Hahaha. And for the most part you are correct in your assessment of my driving record. I have been driving for 28 years and have only gotten one ticket...Thank you Officer Barns for ruining a perfect record. But after I saw this photo I had to reassess my driving habits, especially in New Jersey.
At long last, my first work of literature has finally been released. "Spell Checked" is now available on Amazon, Barns & Noble and Smashwords. Printed copies will also be available on Amazon as soon as I get the proof in the mail and okay printing.
What a journey that has been. Now the fun part....Marketing! Okay, I'm being sarcastic here, I am not a sales person, but now it is time to become one. You see, what is the point in writing a book, if you are not willing to push it one everyone you know and meet? I just need a game plan, one that I can execute on my own or for little cost, as this little venture into writing has already cost about $700, before it was even published. I have a lot of books to sell to make up that cost.
I would love to know. If you have published anything before and what worked for you as far as marketing? I don't want to become that person on Facebook or Twitter who does nothing put plug their product. I don't like it when people do that to me and I'm sure others don't like it either. I like to talk to people, get to know them and not so I can push my book on them. I will honestly say, this is the part I have the hardest time with. Writing, editing, formatting and making a book cover were far easier in comparison.
I will now leave you with an excerpt of the book. The first 30% is free on Smashwords if you would like to read more before you decide to buy. Just click the Smashwords link in this post or under the book cover on the side, it will take you there.
Meredith and Gemma both got up and put their arms around her in a group hug. Mae hated being such a whiner, it was not a role she was used to playing and this whole pity party deal was not working for her. She just wanted to sleep through it for now and make another attempt to get over it at some later date.
"I will put you to sleep. I hope you don't mind if Gemma and I stay up and talk?" Meredith led the way down the hall and back to Mae's room.
"I don't mind. In fact I am glad you have become friends because I don't know what I would do without either one of you. Ironic how the one man in this house, that we all love to hate, has brought us girls together." Mae crawled back into bed and tried to relax as Meredith worked her magic. Her mind went completely blank as she drifted to sleep.
Gemma watched Meredith work her spell. "How long will your spell work?" Gemma asked.
"I hope the entire night, but with Mae you can't tell, she is very different than the rest of us Witches." Meredith walked around the side of the bed and turned off the lamp before she and Gemma went back to Gemma's room to finish their conversation about Aidan.
"I don't know how you have managed to date him no less than ten times and for three years straight!" Gemma squealed while sitting with her legs crossed on the bed.
Meredith was walking around the room when she confessed. "Were not for Helen, I think I would have gone after Beck instead." She stopped cold in her tracts, realizing the error of what she just said.
Gemma gave her a puzzled look. "Who is Helen?" She hugged her pillow waiting for the response.
"The story of Beck and Helen goes back to the days just prior to the Trojan War. His name had been Paris in those days. The Veshtu had become the Gods of Greece and Rome at the time and spent much of their time meddling in the business of their half breed children. It was at the wedding banquet of a friend that most of his troubles began. One of the Veshtu named Eris was left off the wedding list as she had a tendency to cause strife. She was insulted by this omission and plotted her revenge. During the banquet Eris threw the Apple of Discord engraved with the word Kallisti- "for the most beautiful one" in front of the three most prominent Female Veshtu….Hara – Wife to their leader Zeus, Athena – Tactician and strategist of the Veshtu, and Aphrodite – Scientist in charge of the breeding program and the most beautiful of them all. Not being ones to suffer from humility, their vanity took over and an argument ensued as to who the apple was meant for. Zeus was asked to settle the argument for them, but he feared that it would cause chaos, if he, their leader choose a winner. So he delegated the task to Paris, the most intelligent and handsome among the half breeds.
They all attempted to bribe poor Paris; Hara offered him ownership of Europe and Asia. Paris knew that keeping ownership would be the problem with this offer so he passed. Next was the bribe of Wisdom and skill in battle offered by Athena. Paris was already more intelligent than most and as a herdsman had no need for skill in battle, so he would also pass on Athena's offer. The last bribe was from Aphrodite, she offered him the love of the most beautiful human on earth…Helen of Sparta. This seemed like an odd bribe to Paris since Vampires are not one's to dabble with love. Love was not logical and went against their nature, but Paris had once allowed his human side to rule his life and could remember what it was like to be loved. He also knew the pain and emptiness love could leave at the hands of death. So a deal was struck with Aphrodite, he would accept the offer of Helen's love and proclaim her the winner, but in return Aphrodite had to promise that Helen's consciences would be reborn to a different body upon her death. Paris never wanted to feel the pain of loss again. The one thing Aphrodite failed to mention was that Helen was married to the king of Sparta and Paris would have to steal her thus starting the Trojan War." Meredith paused for a second before she would explain that Helen had returned in Mae's body.
Gemma furled her brow and shuttered. "You mean Mae could eventually become Helen? Is that why Beck keeps her close? If it is, I don't like this situation at all." Gemma got up and started to pace the room.
Meredith watched Gemma walking circles around the room. "When Beck went to meet Mae for the first time he had assumed that Helen had already taken over which is why he left the bar that night in a hurry. But it did not take him long to fall in love with Mae and not because of Helen. He battled his conscience over this very issue to the point of almost killing himself. That is when he decided it was Mae that he wanted and not Helen." Meredith was fidgeting as she tried to explain what Beck had confided to her.
"He is afraid she would leave him. You saw what kind of chaos that created today. Look Gemma, Beck is a good man and wouldn't do anything to hurt Mae, you have to understand what he is giving up to be with her…He and Helen have been together for thirty-five hundred years and it was not an easy choice for him to let her go, not to mention the fact that he has been off the market since the Trojan War and hasn't a clue how to date." She laughed stretching her legs across the bed next to Gemma.
"I just don't want anything to happen to Mae. I hate to add to her confusion but she needs to know about Helen. This is her life we are talking about." Gemma threw herself back on the bed and lay down.
"I have told him the same thing and he said he would when he thought her ready for the truth," Meredith sighed.
I know I just threw you into the middle of the book, but I like my bent up way of telling the Trojan War. The Veshtu are an Alien race that became the gods when they would visit. I tell their history and the history of another race in Chapter 2, which is part of the free download on Smashwords.
Please leave me a comment. I would love to hear from everyone that has read this blog.


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today with two events. First, take a guided nature hike on the Balconies Trail. The 1.5 round-trip hike ends with a bring-your-own picnic dinner at the Chaparral parking area. Call 831-389-4486 x265 for more information. Then see some intense star action in the Chapparal parking lot. The Star Party, from 7:30 to 10pm, includes a sky top and many peeks through a telescope. Call 831-389-4486 x243 for more information. Enter Pinnacles via the West Entrance for both events. Time flies when you're relaxing! It's been a decade since The Spa at Bernardus Lodge in Carmel Valley first opened. They celebrate their 10 year anniversary tonight with a reception that includes mini hand and feet treatments, massages, facials, skincare evaluation, spatinis, hors d'oeuvres, and naturally some newly released Bernardus wines. The event begins at 6:30pm at 415 West Carmel Valley Road. $25 per person; call 831-658-3560 to reserve. Scope out the stars tonight with "Astronomical Adventures" at Garland Ranch Regional Park. As visitors look at the stars with binoculars, telescopes and the unaided eye, docent Jim Eagle will explain exactly what they're seeing. Garland Ranch Regional Park Visitors Center, 7:30-9:30pm. $11 per person or $36 for a family of 6, discounts available for Monterey Peninsula district residents. Reserve online under "Star Gazing". The Next Blues Band has been playing the blues in Monterey County since the late 1980s, and have appeared at the county's top blues events Monterey Bay Blues Festival and Seaside's Blues in the Park. Catch them up close tonight at Bullwackers at 653 Cannery Row. 8:30pm-midnight, no cover. If Ladytron teamed up with My Bloody Valentine for an album...well, let's face it, with MBV's track record the album wouldn't come out until 2030. But if it did, it would probably sound like Los Angeles band Pioneer Ships. They team up with fourth wave ska band Fishbear, who tweak the ska sound with a dash of klezmer, and local artist Michael Glines. 638 Wave Street Monterey, 9pm, $5. Saturday Kids and their parents can learn all about the Gabilan Watershed today at Watershed Day at the National Steinbeck Center in Salinas. Learn about the ecology of Salinas, Prunedale and Castroville with hands-on art and science activities. 10am-4pm, One Main Street, Salinas. Activities included in the admission fee. Monterey's new The Inn at Del Monte Beach becomes party central today for the fundraiser "Patrick's Day." The party, on the Inn at Del Monte Beach's rooftop overlooking Monterey Bay, includes six DJs, appetizers, and lots more. All proceeds from the event will help the DuRant family with medical and other bills relating to Patrick DuRant's terminal testicular cancer. 110 Del Monte Avenue, Monterey. Doors open at 3:30pm; the party starts at 4 and runs until midnight. $50 per ticket; reserve online. Learn about owls with "Owls of Central California" at Garland Ranch Regional Park Museum. During daylight, you'll get a full education in all things owl. Once it gets dark, participants will explore the area listening for the noises that all eight species make. 4pm-9pm, ages 13+, $22 per person. Reserve online under "Teen to Adult Explorations." Enjoy British food, British beer and British style at the PacRep Theatre Company's "British Invasion" fundraiser at the Monterey Marriott. 350 Calle Principal, 6-10pm. Tickets are $100 each and can be booked online. Taste outstanding wines from Silver Oak and Twomey wineries tonight at the La Playa Hotel's Wine Dinner. The wines will be paired with a five-course meal created by Executive Chef Bunyan Fortune. Nora Comee of Silver Oak will speak. Starts at 6:30pm at Camino Real and Eighth Avenue in Carmel-by-the-Sea. $125 per person plus tax and gratuity; call 831-624-6476 to reserve. The award for most interesting show of the evening goes to Jose's in Monterey. Moody acoustic locals 1 < 12 (don't call them 11) play alongside Seaside singer/songwriter Bernadette Conant and Dakota Belle Witt's alter ego, mermaid spoken word artist and ukulelest (no, really) Sirene Von Calypso. The surrealism begins at 9pm at 638 Wave Street in Monterey. $5 Upbeat jam band 7 Come 11's unique sound is centered on the Hammond organ, with funky beats and a bit of a roots twist. They play Fernwood on Highway One in Big Sur tonight. 9pm, no cover.
Categories: Special Events
Author: Nicole Gustas


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Square Enix's PS3 Action RPG Revealed
By Siliconera Staff March 30, 2010
Square Enix just put a "new" (note the quotes) action RPG on Japan's PlayStation Network. It's not what you're thinking. No, it's not that either.
The game Square Enix was teasing on Monday is Trine, renamed Trine: The Lost Relic in Japan. Nobilis brought the game to the West and Square Enix has the Japanese publishing rights. While Square Enix localized Western WiiWare games such as Lit and LostWinds for Japan, Trine: The Lost Relic is their first PlayStation Network pickup. You can get Trine for 2,400 yen ($26) in Japan or a Jackson, if you use a North American region account.
FrozenByte


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About Inspired
ICRC Innovates
Understanding the science of behavior change – an innovation unit lecture series
2 October 2019 , Behavior change / New technologies
Nudging. Influencers. Socio-Economic Models. Human-centered design. This is the field of behavior change. How it happens, why it happens, and to whom it happens is of almost universal interest – from where the healthiest food is stocked in the supermarket to how your phone's newsfeed delivers personalized information.
Our days consist of endless information management. It can be difficult to know which parts of our daily lives are truly impacted by subtle yet incessant nudges. Understanding how specifically engineered influence campaigns really affect the way we think, believe and behave can change how we perceive the information itself. And understanding more about the neuroscience of behavior change and influence can increase the efficiency and efficacy of our own project design process.
The ICRC is committed to continually expanding institutional knowledge on the field of behavior change because influencing the behavior of parties to conflict is one of our primary goals. Protection and prevention activities are inherently people-centered; our proximity to affected populations is what allows us to carve out neutral spaces, prevent conflict escalation, and mitigate the impact of violence on civilians.
To explore the world of behavior change and influence, the Innovation Unit invited five experts to a one-day gathering in Geneva to pick their brains for insights on such topics as left versus right brain theory, community engagement, global behavioral economics research and medical virtual reality.
Our Panel of Experts
Dr. Gregor Hasler is a Swiss psychiatry researcher, psychiatrist and psychotherapist at the University of Fribourg whose work covers, among many other things, stress and resilience. The presentation he provided for the audience gave an in-depth and fascinating look into biomarkers and the intricate functionality and minutia of the human brain.
Rocky Sanchez Tirona traveled from Manila to showcase the amazing work she contributes to as Vice-President of the NGO, Rare. This organization has accomplished sustainable behavior change projects at community level throughout the Philippines. Her perspective on humanitarian work as a former advertising executive brought an impactful and meaningful exchange about the relevance of human-centered design to encourage ownership and sustainability.
Dr. Ana Maria Munoz Boudet is a behavioral scientist with The World Bank's Washington, D.C.-based Mind, Behavior and Development Unit (eMBeD). Their team of experts travel the world to diagnose, design, and evaluate behaviorally informed interventions. The team examines a wide set of influences, paying attention to the social, psychological, and economic factors that affect what people think and do.
Dr. Albert Rizzo is the Director for Medical Virtual Reality at the Institute for Creative Technologies for the University of Southern California in Los Angeles. A psychologist by training, he works with US soldiers suffering from PTSD using virtual reality as a treatment mechanism. His insights into the efficacy and opportunities of virtual environments to influence and measure stress responses shed a unique light on how technology interacts with the brain.
Finally, we were grateful to have Dr. Jürg Kesselring contribute opening remarks for our behavior change symposium. Dr. Kesselring is a highly distinguished neurologist with a career spanning several decades and multiple countries and he is also a member of the Assembly of the ICRC, which provided professional insights extremely central to our work. His welcome speech demonstrated the shared values between medical and humanitarian work and explored the beauty and complexity of the human mind.
The Symposium
The day's events originated with the ICRC's Influence Platform, a transversal group that consolidates ICRC's own knowledge and experience on influence to be transformed into widespread operational application. For participants, the day provided an opportunity to learn new methodologies and uses for behavioral science; many left with a deeper appreciation for the complexities of the brain and new ideas on how to incorporate behavior change techniques into their activities.
"Our goal for the behavior change symposium was to bring together decision-makers who are involved in influence activities," says Nan Buzard, Head of Innovation. "The discussion around the table after each lecture was amazing because the speakers had questions on everything from mental health to the future of humanitarian work."
ICRC's Director-General was able to provide the day's closing remarks.
"I am deeply convinced that having behavioral change put at the core of what we do – reflecting and being open about it – is central not just to the ICRC but to humanitarian work," said Yves Daccord. "I am struck by what is happening in Hong Kong, in Sudan, in France with the leaderless influence through social media. Regarding behavioral change and how we mobilize ourselves today, we must understand what works, what doesn't and why."
A podcast of the symposium will be available in the coming days.
The Innovation Unit has already developed research into the field of Extended Reality and how behavior change is measured and affected in Virtual Reality. As we continue to develop our knowledge of how technology and behavior change intertwine, ICRC's Innovation Unit is also working with internal partners like the Centre for Operational Research & Experience (CORE), International Law & Policy (DP), Relations with Arms Carriers (FAS) and the Virtual Reality Unit (VRU) to create a study that measures memory retention of IHL in armed forces through virtual environments. We hope to produce data that allows IHL training to be more measurable and impactful.
Innovation is also in the process of creating an Extended Reality User Manual for the ICRC that will guide users through the opportunities and risks of virtual environments, when XR can be most effective, and how context and technology can be best utilized together.
Read our Extended Reality Report – determining needs, expectations and the future of XR for the ICRC
Read our Article Review about virtual reality & behavior change
Read more about influencing behavior on InspiRED blog
Data and digitalization
New financing models
Restoring family links
Water and habitat
Tweets by ICRC_Innovation


Antonine started on the front foot with Fraser James having a shot saved. The same player then hit the post as Antonine piled on the pressure before Lewis Smith saw a couple of efforts saved by the Port keeper.
After 20 minutes, a Danny Hamilton corner was not cleared and Chris Donaghy fired home to make it 1-0.10 minutes later a Steven MacIver cross was headed goalwards by Smith but it was blocked on the line by a defender.
Two minutes before the break a long ball found the Port striker, who had beaten the offside trap and he made it 1-1 against the run of play.
Shortly after the restart, a Donaghy cross was flicked on by Dom Cairney and Hamilton smashed home a tremendous volley to give Antonine a deserved lead.
Three minutes later, a Port Glasgow striker broke free of the defence and headed over Andy Carroll to make it 2-2.
On the hour mark, Smith was pulled to the ground and a penalty was awarded which Brendan Kelly calmly fired home to regain the lead.
Antonine had not finished there. A quick Scott Hutton free kick found Hamilton at the corner of the box who cut in and fired home an unstoppable shot to complete the scoring.
It was a good professional performance by Antonine in game they controlled from start to finish.