text:
Maps & Cams
StormWrangler 12
Amateur Athlete
Take a Hike
Golf Tip of the Week
Oregon Institute of Technology
Medford Mustangs
Ron Brown's Best
Sparrow Clubs
FYI Southern Oregon
Community Chalkboard
Southern Oregon Strong
NewsWatch 12 Shows & Specials
Justice Network
Two Oregon men face federal hate crime charges after alleged beating of Black man in Washington
The FBI says that a total of four men from across the Pacific Northwest were indicated for beating a Black man at a bar in Lynwood, Washington.
Posted: Dec 18, 2020 2:14 PM
Posted By: Jamie Parfitt
SEATTLE, Wash. — Two Oregon men have been charged with a federal hate crime for their alleged involvement in the beating of a Black man at a bar in Lynwood, Washington during 2018, according to Federal Bureau of Investigation.
A total of four men from across the Pacific Northwest were indicated in federal court in connection with the December 8, 2018 attack. 44-year-old Jason Desimas of Tacoma, Washington, 43-year-old Jason Stanley of Boise, Idaho, 38-year-old Randy Smith of Eugene, and 24-year-old Daniel Dorson of Corvallis are accused of "aiding and abetting one another, as they punched and kicked a Black man and making derogatory comments about his actual and perceived race."
Two other men were also assaulted during the attack, the FBI said.
Both Smith and Dorson are in custody in Oregon, the former on unrelated charges. They'll be transferred to Washington state for arraignment, the FBI said. Dorson is set to appear at the U.S. District Court in Eugene on Friday.
"The U.S. Attorney's Office for the Western District of Washington has a long and distinguished history of prosecuting those who act on hate," said U.S. Attorney Brian Moran. "Whether it is 'The Order' in the 1980s, the 'Atomwaffen' of today, or this group accused of assaulting a Black man at a local business. These defendants will be held accountable for their criminal conduct."
In addition to the hate crime charges, each of the four men are accused of giving false statements to FBI investigators. Desimas told investigators that no-one used racial slurs during the assault. Stanley claimed that he was not in Washington state on the day of the attack. Smith allegedly lied about how his knuckles came to be bloody.
Dorson, one of the Oregon men, claimed that he had no plans to attend a white supremacist "Martyr's Day" observance in Washington, and said that he did not own a jacket associated with white supremacist groups. The FBI asserts that those statements were false.
White supremacists call December 8 "Martyr's Day," according to the Anti-Defamation League, marking the 1984 death of Robert Jay Mathews in a shootout with federal agents on Whidbey Island in Washington. Mathews was a neo-Nazi and leader of white supremacist terrorist group "The Order."
The federal hate crime charges carry a maximum penalty of ten years in prison. The false statements charge carries a maximum penalty of five years.
"The FBI is committed to investigating federal hate crimes and protecting civil rights," said FBI Seattle Acting Special Agent in Charge Earl Camp. "These violent acts motivated by bias are not only an attack on the victim, but also threaten and intimidate an entire community and are contrary to our values of equality for all Americans to live without fear."
An Oregon man faces hate crimes charges after allegedly beating a woman from India
The suspect in the New York Hanukkah stabbings now faces federal hate crime charges
The suspect in the El Paso Walmart shooting is now facing federal hate crime charges
Oregon State Student Convicted of Hate Crime
Oregon man convicted of hate crime pleads guilty to new charge
Effort Underway to Strengthen Oregon Hate-Crime Laws
AG: Oregon Senate passes bill tightening state hate crime laws
Oregon overhauls hate crime laws for first time in decades
Dense morning fog with afternoon sunshine
Weather Cam
Jackson County Public Health reports 30 new Covid-19 cases
Medford School District plans for COVID vaccine and reopening
New York Times: Trump allies have collected tens of thousands of dollars from pardon seekers
Multiple adult-in-custody deaths reported across two Oregon prisons
Baby giraffe dies at Nashville Zoo after being stepped on by mother
Biden to sign executive orders rejoining Paris climate accord and rescinding travel ban on first day
This Illinois dad surprised his family with a bachelor's degree from the same university as his daughter
Josephine County will hold three-day COVID-19 vaccine clinic at two locations
Everything you need to know about COVID-19 vaccinations in Oregon
Medford School District Plans for the Future of the COVID-19 Vaccine
Monday, January 18 morning weather
Oregon State falls to No. 11 Arizona on the road, 67-51
Weinermobile Story
Car thief returns to scold mother
MLK Day Virtual Event
Jackson County reports 30 new cases
Sunday, January 17th Evening Weather
Astronomers discover "Super Earth"
1090 Knutson Avenue
Medford, OR 97504 USA
FCC Public Inspection File - KDKF
FCC Public Inspection File - KDRV
Inside NewsWatch 12's Shows
All content © copyright KDRV. Copyright © 2021 Allen Media Broadcasting, LLC All Rights Reserved. Generated: 2020-12-31 11:00:02pm


text:
est un film allemand muet de Fred Sauer, sorti en 1929. Il est adapté du roman Mr Brown d'Agatha Christie et met en scène Pierre Lafitte et Lucienne Fereoni inspirés de Tommy et Tuppence Beresford. C'est la première adaptation cinématographique non britannique d'une œuvre d'Agatha Christie. Adaptation de Mr Brown.

Synopsis
Un jeune couple en quête d'aventure est impliqué dans le mystère d'une femme disparue et d'un réseau d'espionnage.

Fiche technique
 Titre original :
 Réalisation : Fred Sauer
 Scénario : Jane Bess, d'après le roman Mr Brown d'Agatha Christie
 Direction artistique : Leopold Blonder et Franz Schroedter
 Photographie : A.O. Weitzenberg
 Sociétés de production : Orplid-Film GmbH
 Société de distribution : Messtro-Film Verleih GmbH (Allemagne)
 Pays d'origine : Allemagne
 Langue originale : allemand
 Format : Noir et blanc — 35 mm — 1,33:1 — film muet
 Genre : Aventure, espionnage
 Durée : 76 minutes
 Dates de sortie :
  :

Distribution

Notes et références

Voir aussi

Articles connexes
 Tommy et Tuppence Beresford
 Mr Brown (roman)

Liens externes

Film allemand sorti en 1929
Film d'aventure allemand
Film d'espionnage allemand
Film tiré d'une œuvre d'Agatha Christie
Adaptation d'un roman britannique au cinéma

redpajama_set_name: RedPajamaWikipedia


Heh. Well, I seem to be having trouble uploading that freakin' video to the freakin' FTP. So i'm thinking, stuff that. Anyway, I'm an admin of Mido-Ban's new forum! You can check it out here. In other news, i've finished writing my second review, I just can't be bothered posting it up. And that's about it for today, lol.
The Tank is back and with a whole new sub-section! Check it out, Megiddo's comic section! I urge you guys to check it out. I've finished the FF8 video, and it's all a matter of just uploading it to my FTP..... Well, there won't be much going on tomorrow for me, but I will be watching the news for any er....... news on the virus thingymajig...... nothing much else to say *awkward silence*....... i'll go now.
Hey there! It's been very lazy lately. We have a new Mod on the Forums. WELCOME MIDOBAN! Anyway, There is a new link up, for my friend SpideyKonfusion's Megaman Comix. I will announce a new Video section will be posted up, and every week will see a new video posted! The first video will be Final Fantasy 8! Enjoy your stay!
To other news, i've added a guestbook to the site, so all you visitors out there drop me a line, why don't ya?
Well, it's not really Thursday 7th. But I'm almost ready for bed, so it might as well be lol. All the links now work and i've updated the Supersonic Comik. We've had a bit of a forum attack (I'm not surprised if it was master, or it could of been my Brother, cos he stayed at a friends house.) but that was taken care of.... i'm kinda impressed at the stats for the site though.... 150+ for the first 24 hours, with little advertising. Anyway, expect more sections to the side, and i've almost finished my review for Marvel vs. Capcom 2..... so enjoy the forums while your here!
Hey there and welcome to my site! After careful consideration and a bit of advice by miztik, i've finally decided to make my own website! Special thanks goes to Steve for letting me have this template. Although I had to link to his site, but that really ain't a problem for me. Now this site is only in it's infancy.... the only links that would work will be the Forums, the review and Steve's link :P. Anyway, soon i'll be posting my reviews on all the fighting games i've played so far! anyway, you guys can sign in on the forums and whatnot and enjoy that until I get the site up and running properly.


The nation on Wednesday paid rich tributes to Grandmaster Viswanathan Anand for retaining the World Championship title with Prime Minister Manmohan Singh and Congress President Sonia Gandhi congratulating the chess legend and calling him an "inspiration" to the youth.
Congratulating Anand, PM Manmohan Singh said in a message, "You are a great inspiration to the youth of our country, particularly the sports enthusiasts. You have made the nation proud of you with this monumental achievement." The Prime Minister wished Anand many more successes in the future.
Sonia Gandhi called Anand's feat "stupendous".
"Anand has made India proud. His feat of winning the title consecutively for the fourth time is indeed stupendous," Sonia said in her message.
The Indian Grandmaster showed immense resilience to beat challenger Boris Gelfand of Israel in a tense rapid chess tie-breaker to win the World Championship crown for the fifth time and fourth in a row in Moscow on Wednesday.
Speaking on Anand's win, Rajendra S Pawar, Chairman, NIIT said, "I heartily congratulate NIIT MindChampion Viswanathan Anand for retaining the World Chess Champion title. Anand's fifth World Chess Championship win is a new FIDE record and a milestone in the history of Chess."
"Anand's fighting spirit, perseverance and determination to overcome any challenge will surely inspire youngsters in India to embrace the game of chess," added Pawar. Anand is a brand ambassador for NIIT.


text:
Where Leicester City are best in Europe as Marc Albrighton chases Ryan Giggs – moments missed
Moments you may have missed from Leicester City's 5-0 victory over Sheffield United, looking at Youri Tielemans' repeat trick, Ayoze Perez's scoring streak, and more
Jordan Blackwell
Marc Albrighton on the ball during Leicester City's win over Sheffield United (Image: Plumb Images/Leicester City FC via Getty Images)
Leicester City recorded their biggest win of the season on Sunday, showing off their ruthless side to thrash Sheffield United 5-0.
Here are moments that may have slipped under the radar.
Tielemans' repeat trick
Youri Tielemans had not just City fans in awe, but Premier League followers the world over when he slipped in Kelechi Iheanacho for the equaliser at Brighton while looking in a different direction.
And it seems the no-look pass is becoming his trademark, as he again pulled it off to help set up a City goal.
This time there wasn't the camera angle that showed it off in all its glory, but the Belgian was looking across to the right wing when he subtly played a ball forward to Ayoze Perez for the opener against Sheffield United.
Two more passes, from Perez to Jamie Vardy, and then Vardy to Kelechi Iheanacho, and City were well on their way.
Ndidi's disbelief
No player protests their innocence quite like Wilfred Ndidi, but his misfortune helped City at the weekend.
He stuck a leg in to try to stop George Baldock from advancing, but realising he could not get the ball, he pulled out of the tackle. Baldock went down anyway, without contact and referee Peter Bankes awarded a free-kick.
A despairing Ndidi then put his hands to his head, dropped to his knees and pounded the floor at the decision. Baldock gave him a knowing pat on the back.
But the smile quickly returned to Ndidi's face, as City countered from the Sheffield United free-kick, Marc Albrighton robbing Baldock of possession before setting up Perez.
Leicester City prospect signs new contract after first-team promotion
Brendan Rodgers' message to Leicester City's latest debutant as production line keeps rolling
Perez's Blades streak
Perez marked his return to the side with an impressive performance, capped with a goal that continued his odd scoring streak.
The Spaniard's last three Premier League goals have all been against Sheffield United, his latest effort following up a goal at Bramall Lane in December, and the opener in City's final win of last season in July.
In total, seven of his 11 City goals have come against teams who predominantly play in red-and-white stripes.
Albrighton chasing down Giggs
By setting up Perez's goal, Albrighton registered his 13th Premier League assist as a substitute.
While it may not sound that many, it's actually an impressive tally, given that he is now one off the lead in this particular stat. Ryan Giggs and Theo Walcott lead the way on 14.
Leicester City celebrate the third of their five goals against Sheffield United (Image: Alex Pantling/Getty Images)
Iheanacho's tears (and Vardy's impression)
The emotions of scoring a maiden Premier League hat-trick appeared to overwhelm Iheanacho, who after rifling in a third, went down onto one knee, and covered his face, perhaps shedding a tear.
That was certainly Vardy's grasp of the situation as when Iheanacho returned to the dressing room after the match and received a round of applause from his team-mates, the number nine did his best impression of the Nigerian, himself going down on one knee and imitating those tears.
Vardy's evergreen century
While Vardy didn't find the net, he did register a hat-trick of sorts in setting up three of City's five goals.
Those assists took him to 101 Premier League goal involvements since turning 30, one of just six players to hit a century in their thirties.
As you'd expect, there are other Premier League legends on the list, with Gianfranco Zola, Alan Shearer, Ian Wright, Frank Lampard ahead of him, and the archetypal evergreen forward Teddy Sheringham leading the way on 127.
Jamie Vardy prompts hope of scoring streak as he shows off 'overlooked' attribute
Why Leicester City's new system worked to secure win that could be worth four points
Where City are best in Europe
Spotting the opportunity to twist the knife and boost their goal difference, City kept pushing even after the victory was secured against the Blades.
And by scoring goals four and five inside the final 15 minutes of the game, City became the best in Europe at netting late on.
They were already the best in the Premier League, but their 18 goals after the 75th minute means they are now the leading late scorers when sides from the top flights in Spain, France, Germany, Italy, and Portugal are factored in too. PSG are second on 17.

redpajama_set_name: RedPajamaCommonCrawl


text:
Follow OBHC on
OBHC Site
Mission and Need
WHAT'S NEW AT OBHC?
Cabin Clips Newsletter, November, 2013
Cabin Clips Newsletter, June, 2014
Cabin Clips Newsletter, May, 2015
Cabin Clips, November, 2016
Cabin Clips, May, 2017 Newsletter
Cabin Clips, November 2017 Newsletter
Cabin Clips, June, 2018
Cabin Clips, May, 2019
Financial Reports >
IRS NON-PROFIT DETERMINATION
2011 FORM 990
Fish Tales, BH 2-Fly Tournament
Chili Cookoff, 2014
Healing Hike
Cabin Partners
Family Partners
In February of 2011, Pat and Jeff Baird of Custer, South Dakota, were inspired by a segment aired on the Oprah Winfrey show entitled "The Bravest Families in America". Well known journalist and author, Tom Brokaw, interviewed a wounded Afghanistan war veteran and his family regarding the day-to-day difficulties they faced. Oprah challenged her listeners with the question: "What can YOU do to help?" Because of their 41 combined years of military service and their true giving hearts, Jeff and Pat felt a special bond to these returning wounded veterans. "Since we live in paradise, what better place to provide a chance for these families to reconnect with each other and enjoy our area and all it has to offer?" thought Pat. Thus, Operation Black Hills Cabin was born.
Operation Black Hills Cabin is located in Custer, South Dakota, a close-knit, caring community of approximately 1,800 citizens. Custer is nestled in the middle of the Black Hills of South Dakota, and is a vacation and recreational paradise to many visitors each year. We are fortunate to have Mt .Rushmore National Monument within 20 miles; Jewel Cave National Monument, 12 miles; Wind Cave National Park, 20 miles; Custer State Park, 4 miles; and Crazy Horse Monument, 4 miles.
In addition to our many national parks and monuments, we are blessed with thousands of acres of Black Hills National Forest which offers endless recreational opportunities in many forms. Hiking, biking, hunting, fishing, spelunking, rock climbing, offroading, horseback riding and ATV riding are very popular.
The beauty of our parks and national forest is enhanced with the ever-present opportunity to view a variety of wildlife such as deer, elk, antelope, turkey, buffalo, big horn sheep, mountain goat, burros and prairie dogs.
Mt. Rushmore National Monument boasts close to three million visitors each year and the Black Hills of South Dakota have become a very popular vacation destination to many. Because of our close proximity, 40 miles, to Rapid City, the second largest city in South Dakota, our area has all of the cultural activities, sporting venues, dining and shopping opportunities and regional medical facilities close at hand.
Custer and the Black Hills are truly a family vacation destination with endless opportunities for family fun.
We operated in 2011 and 2012 due to the generosity of several private vacation homeowners and motels. We soon realized the need for our own cabin as it was oftentimes very difficult to coordinate the available weeks in the private vacation homes with the available weeks of the families. Our permanent cabin arrived on March 6, 2013. It was granted to Operation Black Hills Cabin by the State of South Dakota. In addition, the City of Custer has leased us a beautiful, wooded city lot very near to the hospital for 30 years at $1.00 per year. The support for our project has been overwhelming! The State, the City and many private citizens and business owners in Custer as well as the surrounding area have stepped on board to provide many meals, free admissions and recreational opportunities.
We are a 100% volunteer 501(c)(3) organization with no paid staff. We rely on the continued generosity of many donors, grants and fundraisers.
Our on-going expenses include maintenance of our cabin, utility costs, insurance and basic supplies, snacks and food that we provide to each family. Our out-of-pocket cost to accommodate an average family of five in our permanent cabin for seven days/six nights runs approximately $500.00 per family. From May through September, we are able to host 20 families each year.


text:
Q: Grid.MVC Single view for CRUD operations I am using Grid.MVC(https://gridmvc.codeplex.com/) and I need to render a grid and CRUD for the grid in the same view.
@model IEnumerable<Grid.Models.Catagory>
...
...
  @Html.Grid(Model).Columns(columns =>
        {
            columns.Add()
            .Encoded(false)
            .Sanitized(false)
            .Titled("CatagoryName").RenderValueAs(o => RenderCatagoryName("CatagoryName", o.CatagoryName));

            columns.Add()
            .Encoded(false)
            .Sanitized(false)
            .Titled("Color").RenderValueAs(o => RenderCatagoryName("Color", o.Color));

            columns.Add()
            .Encoded(false)
            .Sanitized(false)
            .Titled("Print Template").RenderValueAs(o => RenderCatagoryName("Print Template", o.PrintTemplate));
        }

My controller
    public ActionResult Index()
    {
        return View(clients);
    }

This works fine, but when I need to do an insert in that same view, I am unable to use the same model since it is an IEnumerable type.
 --Insert values--
 //couldn't use the model for insert directly since it is a list type
        @Html.LabelFor(m => m.)
        @Html.TextBoxFor(m => m., new { maxlength = 50 })

My ViewModel
public class Catagory
{

    public int CatagoryID { get; set; }
    public string CatagoryName { get; set; }
    public string Color { get; set; }
    public string PrintTemplate { get; set; }
    public string Language { get; set; }
}

Is there any way to use the same view for both the Grid.MVC and insert operations

A: make the list of Catagory (btw it's spelled 'Category') be a property of a new view model.
public class MyViewModel
{
     public Category CategoryToInsert { get; set; }
     public IEnumerable<Category> CategoriesToShow { get; set; }
}

View:
 @model MyViewModel
...
...

@Html.Grid(Model.Categories).Columns(columns =>
  ...

@* Insert form: *@
@Html.LabelFor(m => m.CategoryToInsert.CategoryName)
@Html.TextBoxFor(m => m.CategoryToInsert.CategoryName, new { maxlength = 50 })

redpajama_set_name: RedPajamaStackExchange


text:
About Blog Since 1998 Elitefts.com Inc has published over 350,000 Q&As, 4,000 articles, 14,000 videos, 800 exercises, 35 pro training logs, the Iron Subculture Podcast and an unparallelled shopping cart geared for those serious about training. Elitefts's main purpose is to inform, educate and outfit powerlifters, athletes, strength coaches, personal trainers, gyms and training facilities around world.
In the trailer, Lisa is looking for someone to back her health start-up, and that's when Anne pops the question. — Chloe Foussianes, Town & Country, "Renée Zellweger's Netflix Series What / If Is Out Now on Netflix," 24 May 2019 The 53- year-old choreographer and reality star has been open about her painful health journey, sharing raw photos of her post-surgery and updating fans every step of the way. — Mackenzie Dunn, Woman's Day, "Abby Lee Miller Is Officially Cancer Free," 23 May 2019 As for the holistic approach, Pollak points out that the skin, being our largest organ, is inextricably tied to whole-body health. — Prue Lewington, Harper's BAZAAR, "Women Are Giving Up Botox for Facial Massage," 23 May 2019 Best of all, the Flatform will help raise money for the Tegan and Sara Foundation, the sisters' initiative that fights for health, political, and economic equality for young, LGBTQ+ girls and women. — Leah Prinzivalli, Allure, "Tegan and Sara Collaborated on Rainbow Teva Sandals for Pride Month, and Fans Are Losing It," 22 May 2019 The furry favorite has offered grooming services, training workshops, and plenty of toys for good boys for more than 50 years, and now, the brand's doubling down on its commitment to pet health with a new artificial-free policy. — Sara Rodrigues, House Beautiful, "How To Get Free Pet Food At Petco This Weekend," 17 May 2019 Those include Labor Department powers under the law that sets minimum standards for private-industry health plans and current hospital-payment rules under Medicare. ... — Stephanie Armour, WSJ, "White House Wants Patients to Know Health-Care Prices Up Front," 15 May 2019 Under the Affordable Care Act, most private health plans must cover all birth control, including EC, as prescribed. — Glamour, "All of Your Emergency-Contraception Q's, Answered," 22 Apr. 2019 In already fragile health ecosystems, these efforts to ban abortions could cost women their lives. — Lauren Rankin, Teen Vogue, "Alabama's Abortion Ban Is Dangerous, and it May Even Be Deadly," 16 May 2019
In 1990, professional wrestling promoter Vince McMahon announced that he was forming a new bodybuilding organization named the World Bodybuilding Federation (WBF). McMahon wanted to bring WWF-style showmanship and bigger prize money to the sport of bodybuilding. A number of IFBB stars were recruited but the roster was never very large and featured the same athletes competing; the most notable winner and first WBF champion was Gary Strydom. McMahon formally dissolved the WBF in July 1992. Reasons for this reportedly included lack of income from the pay-per-view broadcasts of the contests, slow sales of the WBF's magazine Bodybuilding Lifestyles (later WBF Magazine), and the expense of paying multiple six-figure contracts while producing two TV shows and a monthly magazine.
Drugs.com provides accurate and independent information on more than 24,000 prescription drugs, over-the-counter medicines and natural products. This material is provided for educational purposes only and is not intended for medical advice, diagnosis or treatment. Data sources include IBM Watson Micromedex (updated 3 June 2019), Cerner Multum™ (updated 4 June 2019), Wolters Kluwer™ (updated 31 May 2019) and others.


A Problem Like No Other: Science And Politics : 13.7: Cosmos And Culture The products of science define modern life. But existential danger lurks in the chasm between the relentless advance of science and the values of politics, says commentator Adam Frank.
We have a problem. It's a big one that raises big challenges for the human future and the ability of our most cherished political institution — democracy — to guide that future.
First, however, let's begin by acknowledging what we have already done. Over the past 200 years, we've used science to fundamentally change the meaning of human culture via revolutions in industry, transportation and communications. This globally connected society of instant information transfer and godlike powers of production and destruction would seem like a dream (or a nightmare) to 99 percent of the generations that preceded us.
Looking forward, science will soon be giving us even greater powers. Using techniques like genetic engineering, for example, we are poised to redefine what it even means to be human. On another front, brain-computer interfaces being developed now may soon have the power to redefine mind and consciousness.
So it's clear that science has given us — and will give us — great power. Now what about the great responsibility?
What group gets to tell us if (and how) we'll live with genetically modified foods?
In other words, the problem is not science — it's science and policy. Or, better yet, it's science and politics.
The critical and endlessly exasperating case of climate change is the urexample of the problem. The basic scientific case that the planet's climate is changing due to human activity has been settled for at least 25 years. What's more, policymakers have known about the dangers of climate change going back to the 1960s. But, as we are painfully aware, not only has nothing been done, but opposition is so prevalent that we can't even get to debating the real issue.
The political battle over the science of climate change makes it clear how vast the chasm is separating how science understands the world from how politics acts in the world. As Micheal Mann has written from personal experience and Andrew Revkin has covered for years, climate science and politics have made for an ugly stew.
What makes this gulf all the more frightening is that the climate issue is only one of the existential questions we face. Genetics, robotics, big data, artificial intelligence, brain-machine interfaces, energy modalities — each one has the capacity to drive radical shifts in our culture. All of these may — in fact — pose fundamental dangers to human culture. Or they may lift us all up. It depends on the choices we make.
And before we take another step, I want to be clear: The problem is not simply "let's pay more attention to scientific experts."
In some cases, such as climate change, it is clear that the science is being ignored or distorted by some in the arena of politics. Ignoring what scientists are telling us in this domain (or, worse, vilifying them) is an ultimately self-destructive act. But when it comes to telling us what we should do about climate change — a question that hinges on values as much as on data — scientists are not in a privileged position.
It's exactly the emphasis on values that makes the problem of science and politics so wicked. Politics is a domain ruled by values as well as facts. Science usually strives to step back from questions of what we value and asks only how the world behaves with or without us.
In addition, scientific training includes next to nothing in the way of exploring the societal function and consequences of science. In fact, scientists can go through their entire education with little training in anything outside their technical specialties.
Thus the reasons for the gulf between science and politics are many and are not universally the fault of politicians. Lets face it: Only a small fraction of the general population are actually scientists. That means very few people have direct contact with the day-to-day realities of how science reaches its conclusions and builds consensus. As a result, most people have a hard time telling the difference between firmly established laws of nature and the back and forth of unsettled questions.
It's this incomprehension about how science works that makes the gulf between science and politics so dangerous for democracies. Between the public's understandable lack of understanding and the ever-growing pervasiveness of science in our lives lies a fault line that can be exploited for all the wrong reasons. If people don't understand how science reaches its conclusions, it's all too easy for vested interests (and there will be many) to misconstrue, misinform and misdirect public debate.
Imagine, for a moment, if the typewriter industry in the early 1980s had managed to manipulate discussions of the burgeoning science of networked computers for its own purposes. Might it have succeeded in ensuring that the fruits of computer science never reached the masses?
When it comes to big data or genetics or energy, there will be many interests pushing to spin the relevant science. But how can we, as members of a democratic society, respond as informed citizens? How can we act with wisdom? And if we can't, who will we choose to be our proxies and on what basis will they act? Most importantly, if, in our incomprehension, the wrong choices are made and we suffer, what will be the consequences? Will we seek to empower a stronger hand to deal with the mess, abandoning democracy in favor of expediency?
No one expects politics to be pretty or bloodless. But when the issues at hand are existential — as with the genetic future of our species — a functioning democracy will only function when the place at the table for science is clearly understood.
There is no easy answer to the dilemma of science and politics. But in a world whose politics will be increasingly dominated by issues that exist only because of science, we can begin by recognizing the existence of the problem and its gravity. Our fate as a democratic society depends on, at least, taking that first step.


S 1/c William A. Norred, son of Mrs. Guy Norred, Graham. Graduate of Graham High. Entered Seabees in 1944, trained in San Diego, Camp Endicott, R. I. and Port Hueneme, Cal. Served in Okinawa. Awarded Victory, Amer. Theater and APO Ribbon with 1 Battle Star. Dscharged in 1946.


text:
Make a Referral | Join Our Team | 888-978-3801 | info@myuscare.com
Finding the light in life
Elaine's struggle with her weight has made life hard. People who can see past it—like U.S. Care's professionals—know her as a bright, smiling sweetheart, one of the most generous people they've met. Unfortunately, this generosity has been taken advantage of, and Elaine's depression has wrecked her efforts to get healthy time and time again. Thanks to U.S. Care, she now uses transportation resources to get to appointments. Her prescriptions are sorted out now and make sense, and her doctors can't stop marveling at the improvements in her health. When she sees her U.S. Care professional, her already-bright smile lights up the whole room. "I don't know what I'd do without you," she says. "You're like an angel sent from heaven."
Back to Success Stories
© 2019 U.S. Care, LLC | All rights reserved | Site developed by U.S. Care, LLC | Privacy Policy
SING UP STAY TUNED!
Be the 1st to know about


From the hallowed halls of Oxford University came the inspiration to study early modern women's writing. Surrounded by the world's top Renaissance scholars, Professor Ros Smith began to explore the world of women's writing from the 16th and17th centuries.
"It's a strange world with some recognisable and some very unfamiliar elements - that's what encapsulates the oddness of studying the past. I find that tension very interesting. You think you understand something but then you dig deeper and find there's lots more to explore," Professor Smith said.
At that time, research into early modern women's writing was in its infancy. Today it's a burgeoning field with over 100 scholars working in the area. New appointments in 2019 means there is now a group of seven early modern literature specialists based at the University of Newcastle – the largest concentration in Australia. Professor Smith has led international teams of researchers to ARC DP success in 2012 and 2016, and currently holds an ARC Future Fellowship (2019-2022) on early modern women's marginalia.
As part of her Future Fellowship, Professor Smith will partner with the Centre for Early Modern Studies at the University of Oxford and the Folger Shakespeare library in Washington.
"When I started researching in this field, it was exciting and cutting-edge work because there was hardly anyone doing it. I wanted to understand, from a feminist perspective, what early modern women's writing can tell us about gender and culture in the Renaissance and across the ages."
"Women have always been half the population, but their contribution to the literature of the past was once thought to be negligible. My work, alongside that of others in the field, has discovered that women did write an enormous amount in the early modern period, in a variety of forms from private letters to major published romances."
"Through their writing, they also contributed to literary, religious and political cultural life in important ways. Understanding the active literary roles that women had in the past changes the way we think about women's literary agency and ability in the present. It also reconfigures the way we think about the early modern period – it is not just a period dominated by men but one in which women had vital roles in shaping the culture," Professor Smith said.
A career turning point came with the co-founding of the Early Modern Women's Research Network (EMWRN) at the University of Newcastle in 2008, with her colleague Professor Patricia Pender.
"Prior to the establishment of the network, scholars working in the field of early modern women's writing in Australia were quite isolated and disconnected," Professor Smith said.
"But we've been able to bring together a broad range of well-known scholars from across Australia, as well as international scholars who are at the top of their field and run similar networks internationally."
"This joining of the minds has allowed us to become a really effective network and we have built our reputation in the field to become one of its most dynamic and generative research groups nationally and internationally," Professor Smith said.
In addition to her ongoing research, Professor Smith will take up a senior leadership role as the Acting Pro Vice-Chancellor (Research and Innovation) for a six month period.
"This unique opportunity will extend my capacity to lead research, to understand research strategy across disciplines and faculties, as well as to mentor and train the next generation of researchers at my university" Professor Smith said.
Ros Smith examines the contribution of female writers to the culture of the early modern era.
My primary field of research is Renaissance literature, specialising in women's poetry, and the relationships between genre, politics and history in the period. My secondary field is in true crime writing in Australia, examining a neglected but increasingly central popular genre and its relationship to constructions of Australian nationalism.
My teaching expertise, developed at the Universities of Newcastle, Oxford and London, is in the fields of Renaissance literature and contemporary Australian literature, with a special interest in women's writing and true crime. I have taught at all levels in both fields, developing a number of new courses and majors.
I am also co-convenor of the Early Modern Women Research Network, and directed the Centre for 21st Century Humanities in 2018. I was a member of the ARC College of Experts, HCA panel (2015-17).
© 2018 Informa UK Limited, trading as Taylor & Francis Group. This essay examines sixteenth-century women¿s marginalia in devotional books as a mode of transmission, particularly in circumstances of where early modern women themselves were in circumstance of limited circulation, under house arrest or imprisoned. Recent work on prison literature has highlighted the importance of the prison as a crucible for writing in early modern England. However, it has focused less on the material cultures through which such texts were circulated, which for women writers in particular included marginal annotations to texts then circulated through domestic and coterie circles to a broader world. Anne Boleyn, Jane Dudley, Elizabeth Tudor and Mary Stuart all circulated writing as marginalia while under forms of imprisonment, providing a means of political engagement through lamentation, critique and protest. This essay uncovers the ways in which such texts constructed and disguised their political objectives, as well as the material means through which these prison poems were transmitted, showing the ways in which material and rhetorical cultures operated together to make meaning in this neglected group of texts.
p16CDKN2 specifically binds to and inhibits the cyclin-dependent kinases CDK4 and CDK6, which function as regulators of cell cycle progression in G1 by contributing to the phosphorylation of the retinoblastoma protein (pRB). Human cell lines lacking functional pRB contain high levels of p16 RNA and protein, suggesting a negative feedback loop by which pRB might regulate p16 expression in late G1. By a combination of nuclear run-on assays and promoter analyses in human fibroblasts expressing a temperature-sensitive simian virus 40 T antigen, we show that p16 transcription is affected by the status of pRB and define a region in the p16 promoter that is required for this response. However, the effect is not sufficient to account for the differences in p16 RNA levels between pRB-positive and -negative cells. Moreover, p16 RNA is extremely stable, and the levels do not change appreciably during the cell cycle. Primary human fibroblasts express very low levels of p16, but the RNA and protein accumulate in late-passage, senescent cells. The apparent overexpression of p16 in pRB-negative cell lines is therefore caused by at least two factors: loss of repression by pRB and an increase in the number of population doublings.
Sustained growth of solid tumors and their metastasis requires paracrine signals between the tumor cells and the normal surrounding host tissue. One crucial function of these signals is to recruit endothelial cells and thus new blood vessels for the nourishment of the expanding tumor mass. In addition, the newly recruited blood vessels serve as routes for metastasis of the primary tumor. The proliferation and migration of endothelial cells in the vicinity of progressing tumors contrasts with a low turn-over rate of endothelial cells in the healthy adult. The only physiologic exception to this rule is the female reproductive cycle. Based on the selective angiogenesis in tumors in contrast to normal tissues, a blockade of this process should be feasible with few adverse effects. We present data on angiogenesis factor gene expression in tumors and discuss different methods of targeting these gene products. Furthermore, we discuss the potential to combine conventional cytotoxic treatments with this new therapeutic approach. © 1995 Informa UK Ltd All rights reserved: reproduction in whole or part not permitted.
One in six primary human breast cancers has DNA amplification centered on the cyclin D1 gene (CCND1) on chromosome 11q13. This genetic abnormality is preferentially associated with estrogen-receptor positive tumors and may define a sub-class of patients with an adverse prognosis. Although CCND1 has the credentials of a cellular oncogene, being a target for chromosomal translocation and retroviral integration, the 11q13 amplicon encompasses several other markers and CCND1 is not the only candidate for the key gene on the amplified DNA. To assess their relative importance, we have constructed a physical map of the amplified DNA and compared the extent and frequency of amplification across the region. Since it is likely that the gene providing the selective force for amplification will be expressed at elevated levels, we have also examined expression of both RNA and protein. By these criteria, cyclin D1 remains the strongest candidate for the key oncogene on the amplicon and we are currently investigating the functional consequences of its over-expression. © 1995 Kluwer Academic Publishers.
In this paper we describe how research on the mouse mammary tumor virus model of breast cancer resulted in the identification of an amplified region of DNA on human chromosome 11 band q13. This amplification occurs in approximately 15% of primary breast cancers. Several candidate oncogenes map within the amplicon but by analysing expression of these genes a strong case can be made for a role for cyclin D1 in tumorigenesis. Immunohistochemical staining indicates that cyclin D1 is expressed at elevated levels in around 40% of breast cancers, including those with the 11q13 amplification. The potential function of cyclin D1 as a regulator of early cell division cycle events would be consistent with a role in neoplasia. © 1995.
To determine the genomic organization of the mouse cyclin D1 locus (Cyl-1), a series of cosmids and cDNAs were recovered by hybridization with a genomic probe representing the 5' end of the homologous human gene, CCND1. Primer extension indicated that transcripts originate from one of three adjacent nucleotides at a single start site. Two overlapping cDNA clones that essentially accounted for the complete sequence of the larger 4.0-kb Cyl-1 transcript were characterized. A combination of RNase protection and sequencing across intron-exon boundaries established that the gene is organized into five coding exons with a long 3' untranslated region. Repeated attempts to isolate clones corresponding to the minor 3.5-kb RNA were compromised by the presence of an internal poly(A) domain. However, hybridization with specific probes revealed that the minor transcript lacks approximately 800 nucleotides from the 3' end of the major transcript and may be generated by a novel mechanism or by RNA processing. © 1995 Academic Press, Inc.
Immunohistochemical staining with a monoclonal antibody against human cyclin D1 can be used to identify breast cancers that have an amplification of the q13 region of chromosome 11. In general, the intensity of staining is directly proportional to the degree of DNA amplification. In two unusual tumors, in which the CCND1 locus is highly amplified but staining is relatively weak, it appears that the DNA has undergone rearrangement and that the amplified/rearranged CCND1 allele may have reduced transcriptional activity. More significantly, the immunohistochemical technique identifies additional tumors in which the cyclin D1 gene is overexpressed with only marginal or undetectable increases in copy number, implying that other mechanisms can lead to deregulated expression. These results suggest that the frequency of overexpression is much higher than previously concluded from DNA-based analyses and that more than one-third of human breast cancers may contain excessive levels of cyclin D1. The technique we describe should facilitate the detection of this abnormality in a clinical setting and clarify its prognostic significance. © 1994, American Association for Cancer Research. All rights reserved.
We have isolated cDNA and genomic clones for the murine FGF-7 gene and examined its expression throughout development. Transcripts were transiently detected in the developing myocardium, differentially regulated between the atrium and ventricle. The gene was also expressed in the myotomes of the somites, coincident with FGF-4 and FGF-5 transcripts, and was detected transiently in cleaved muscles. Regional expression was detected in the ventricular zone of the developing forebrain at 14.5 d.p.c. Later in development, FGF-7 RNA was detected in mesenchymal tissues suggesting a role in epithelial-mesenchymal interactions and in the dermis consistent with its proposed role as a keratinocyte mitogen. Our results suggest that FGF-7 is likely to have diverse roles during development. © 1994.
Amplification of markers centred on band q13 of human chromosome 11 is a consistent feature in a subset of oestrogen receptor positive breast cancers. Although the amplification was initially scored via FGF3/INT2, which has strong credentials as a mammary oncogene, current data suggest that some other gene on 11q13 provides the driving force for amplification. Here we have reviewed our understanding of the amplified DNA, the genes it encompasses and the evidence in favour of two candidate oncogenes, CCND1 and EMS1. As well as being among the most frequently amplified markers in the region, these genes are expressed at elevated levels as a consequence of amplification, and their predicted functions would be consistent with a role in tumorigenesis. Irrespective of the final conclusions regarding their biological relevance, the overexpression of CCND1 or EMS1 should provide a more amenable assay for the amplification and help to clarify its clinical significance.
By isolating genomic DNA clones that encompass the mouse Cyl-1 (cyclin D1) locus, we have identified a putative CpG island close to the 5' end of the gene. Pulsed-field gel electrophoresis with probes derived from either the 5' or 3' side of the CpG island established physical linkage to two independent markers on mouse chromosome 7, in a region that is syntenic with human chromosome 11q13. On the 3' side, Cyl-1 is approximately 75 kb from Hst-1 and Int-2, although there is an additional CpG island in the intervening DNA, while on the 5' side, Cyl-1 is less than 300 kb from Fis-1, an integration site for Friend murine leukaemia virus. As there is no intervening CpG island, proviral insertions at Fis-1 could influence the expression of Cyl-1 and we describe two virally induced tumours in which this appears to be the case. The data suggest that proviral insertions near Cyl-1 in mouse lymphomas are functionally equivalent to the BCL1 translocations that activate cyclin D1 expression in human B-cell malignancies.
We have developed transgenic mice in which expression of the mouse int-2/Fgf-3 gene is regulated by a single long terminal repeat from mouse mammary tumor virus. Such mice contain and transmit a replica of the activated int-2/Fgf-3 allele present in a spontaneous mammary tumor from a BR6 mouse. Although free of infectious mouse mammary tumor virus and with a different genetic background, the transgenic mice develop pregnancy-responsive mammary epithelial proliferations that are similar to the early stages of tumorigenesis in the BR6 strain. Histological examination revealed that most of these tumors showed pronounced tubular and acinar structures, features usually associated with morphological differentiation. In some cases, the tumors were locally invasive, causing disruption of the dermis which manifested itself as local hair loss. In situ hybridization showed that patterns of transgene expression in the abnormal glands were markedly nonuniform. In contrast, mouse mammary tumor virus-induced neoplasms showed more uniform expression of int-2/Fgf-3, as did the urogenital epithelial proliferations that occur among males of this transgenic line. These data suggest that mammary tumors in virally infected animals may depend primarily on autocrine stimulation by the int-2/Fgf-3 gene product, whereas both autocrine and paracrine mechanisms may contribute to tumors and hyperplasias found in transgenic animals.
The int-2 gene, which encodes a member of the fibroblast growth factor family, is expressed at specific sites and times during mouse development. In certain embryonal carcinoma cell lines, multiple int-2 transcripts accumulate when the cells are induced to differentiate with retinoic acid and dibutyryl cyclic AMP. Nuclear run-on analyses indicate that the apparent induction of int-2 expression results from an increase in the rate of transcription initiation. Six distinct types of RNA have been delineated, originating from three promoters and terminating at either of two polyadenylation sites. Since each transcript appears to encode the same protein, this complexity may reflect the need for lineage-specific or differentiation-dependent control of expression. By comparing the kinetics of induction and turnover of the different RNA species, we show that the choice of promoter or length of the 3'-untranslated region has no significant effect on the half-lives of the various mRNAs. To further evaluate control at the transcriptional level, we have shown that a 1.7-kilobase fragment of int-2 genomic DNA, when fused to the chloramphenicol acetyltransferase gene, can act as a regulated promoter(s) in differentiated versus undifferentiated embryonal carcinoma cells. This segment of DNA encompasses the three promoter regions previously delineated by RNase mapping plus about 900 base pairs of additional upstream sequences.
We examined the effects of mouse mammary tumor virus integration on the multiple RNA transcripts expressed from the int-2 proto-oncogene in virally induced breast tumors. Proviral insertion either upstream or downstream of the gene could simultaneously activate transcription from three dissimilar int-2 promoters. In some tumors, the activating provirus lies within the transcription unit and disrupts the structures of the various RNAs. Insertions in the 5' region of the gene had complex effects depending on the orientation and position of the provirus relative to the three promoters and intron-exon boundaries. RNase protection experiments identified transcripts initiated in the viral long terminal repeat, at normal and cryptic sites in the int-2 sequences, and from cryptic promoters in an inverted provirus. At the 3' end, insertions occurred within the untranslated trailer and provided alternative termination signals that substituted for one or both of the normal the poly(A) addition sites. However, in no instance, of the 20 tumors analyzed in detail, did a provirus perturb the presumed open reading frame of the gene. These data strongly implicate the normal product of the int-2 gene, which is related to the fibroblast growth factor family, as a contributory factor in virally induced mammary tumors.
We have analysed DNA from 183 primary breast cancers for amplification or rearrangement of a number of cellular proto-oncogenes, focusing primarily on a cluster of markers on the long arm of chromosome 11. Two of these oncogenes, INT2 and HST1, both of which encode members of the fibroblast growth factor family, are implicated in the generation of virally induced mammary tumours in mice. Here we confirm earlier reports that the q13 region of chromosome 11, in which these genes are tandemly linked, is modestly amplified in approximately 15% of primary human breast cancers. This amplification is confined, with one exception, to cases in which the oestrogen receptor (ER) levels are in excess of 20 fmol/mg protein (P = 0.001). However, DNA amplification does not usually result in detectable expression of either the INT2 or HST1 gene. The data imply that some other gene in the vicinity must contribute to the development of a subset of ER-positive tumours and that assessing the amplification of this region of DNA may be of value in defining a separate category of ER-positive tumour. © 1990.
The fibroblast growth factor-related protooncogenes, int-2 and hst/k-FGF, are within 17 kilobase pairs of one another on mouse chromosome 7 and are in the same transcriptional orientation. Approximately 70% of tumors induced in BR6 mice by mouse mammary tumor virus have proviral insertions adjacent to the int-2 gene. We find that the murine homolog of the hst/k-FGF gene can also be transcriptionally activated by the insertion of mouse mammary tumor virus DNA either upstream or downstream of the gene. In most tumors, only one of these adjacent genes is activated, but in some cases both genes are expressed. One of the hst-expressing tumors also has a virally activated int-3 gene. At least five distinct cellular genes (int-1, -2, -3, -4, and hst/k-FGF) can therefore contribute, either singly or in concert, to the development of histologically indistinguishable mammary tumors in mice infected by mouse mammary tumor virus.
We report the genomic organization and DNA sequence of the human homologue of int-2, a proto-oncogene implicated in virally induced mammary tumours in the mouse, and expressed at specific sites and times during embryogenesis. Direct comparisons with the coding domains of mouse int-2 allowed us to delineate the intron-exon boundaries of the human gene. These boundaries were subsequently confirmed by ribonuclease protection analyses of the single 1.7 kilobase (kb) int-2 transcript detectable in the human teratocarcinoma cell line, Tera-2. The data suggest that human int-2 may also function in embryonic lineages but that its transcription may be less complex than in the mouse. The predicted human protein comprises 239 amino acids and is 89% homologous to its murine counterpart, except at the carboxy terminus. This divergence occurs distal to the region of int-2 that shows homology to other members of the FGF family of growth modulators and oncogenes.
Mouse embryonal carcinoma cell lines that differ in their patterns of expression of the potential oncogene int-2 have been exploited in a structural analysis of the multiple RNA transcripts characteristic of this gene. Ribonuclease protection experiments indicate that four major classes of int-2 RNA initiate at heterogeneous cap sites within two distinct promoter regions, P1 and P2, spanning approximately 50 and 150 bp respectively. The more downstream promoter P2 is located in a region of the DNA that constitutes an intron in RNA transcripts that initiate at the upstream promoter P1. Otherwise, all four RNA structures share the same splice donor and acceptor sites that define the boundaries of the second and third exons. Further complexity arises through usage of two distinct polyadenylation signals, both variants of the normal consensus, that are separated by 1100 bp. Despite these structural variations, the results suggest that all four major classes of RNA encode the same protein product which shows significant homology to the family of heparin-binding proteins typified by basic fibroblast growth factor (FGF).
We sequenced two recombinant DNA clones constituting a single provirus of the milk-transmitted mouse mammary tumor virus characteristic of BR6 mice. The complete provirus is 9,901 base pairs long, flanked by 6 base-pair duplications of cellular DNA at the site of integration. Five extensive blocks of open reading frame corresponding to the gag gene, the presumed protease, the pol and env genes, and the open reading frame orf within the long terminal repeat of the provirus were readily discernible. Translation of gag, protease, and pol involved three different translational reading frames to produce the three overlapping polyprotein precursors Pr77, Pr110, and Pr160 found in virus-infected cells. Synthesis of the reverse transcriptase and endonuclease therefore required two separate frameshifts to suppress the termination codons at the ends of the Pr77 and Pr110 domains. Direct evidence is presented for translational readthrough of both stop codons in an in vitro protein synthesis system.
The third (4-cell) and fourth (8-cell) cell cycles of early mouse development have been analysed in populations of blastomeres synchronized to the preceding cleavage division. DNA content was measured microdensitometrically. The entry of blastomeres into these cell cycles showed considerable heterogeneity both within and between individual embryos. This heterogeneity was greater in the fourth than in the third cell cycle. The component phases of the third cell cycle were estimated as G 1 = 1h, S = 7h, and G 2 + M = 2-5h, and those of the fourth cell cycle as G 1 = 2h, S = 7h, and G 2 + M = 1-3h.
In the course of analyzing sites of proviral integration in tumors induced by mouse mammary tumor virus (MMTV), we have isolated recombinant DNA clones corresponding to the 5' and 3' ends of four endogenous MMTV proviruses present in BALB/c and BR6 mice. This has permitted the structural characterization of each locus by detailed restriction mapping and the preparation of DNA probes specific for the cellular sequences flanking each provirus. These probes have been used to trace the segregation patterns of the proviruses, designated Mtv-8, Mtv-9, Mtv-17, and Mtv-21, in a panel of inbred strains of laboratory mice and to map Mtv-17 and Mtv-21 to mouse chromosomes 4 and 8, respectively. The unambiguous resolution of these four proviruses on Southern blots has greatly facilitated the analysis of other endogenous MMTV proviruses in these inbred mice.
The effects of aphidicolin, a reversible inhibitor of DNA polymerase alpha, both on replication and on development of the mouse embryo from the 2- and 4-cell stages to the compacted late 8-cell stage have been assessed. The continuous presence of aphidicolin from G 1 of the 4-cell stage resulted in inhibition of DNA replication and prevention of division from 4 to 8 cells, but was without effect on the timing or incidence of cell flattening, surface polarization and cytoplasmic polarization. The continuous presence of aphidicolin from G 1 of the 2-cell stage resulted in inhibition of DNA replication, division, and polarization. Some slight intercellular flattening in a few embryos did occur. If addition of aphidicolin was delayed by 10 h to early in G 2 of the 2-cell stage, further rounds of replication were blocked and some embryos failed to cleave to 4-cells. Nevertheless, almost all embryos showed evidence of flattening and polarization regardless of cell number. In contrast, if aphidicolin was added in G 1 of th 2-cell stage and removed after 10h, the cells showed delayed DNA replication, little evidence of division, and no cell flattening or polarization. We conclude that DNA replication at the 2-cell stage may be essential for the components of compaction studied, but that DNA replication at the 4- and 8-cell stages is not.
Approximately 50% of tumors induced by mouse mammary tumor virus (MMTV) contain an acquired provirus within a limited region of chromosomal DNA, termed int-2. We have extended our previous characterization of this locus and have mapped provirus integration sites in 21 independent tumors. Although integration occurs at multiple sites, proviruses within int-2 are distributed into two oppositely oriented groups whose transcription is directed away from a central domain. Provirus insertion in int-2 is accompanied by expression of RNA derived, at least in part, from this central domain. Since the RNA is not detected in normal mammary tissue, we conclude that MMTV integration activates the expression of a cellular gene within int-2 and that this event may contribute to tumorigenesis. © 1984.
We have prepared specific probes for unique-sequence cellular DNA adjacent to each of the newly integrated proviruses in tumors induced by mouse mammary tumor virus (MMTV). The use of such probes to screen a large number of independent mammary tumors in the BR6 strain of mouse has indicated that in at least 17 out of the 40 tumors examined so far, an MMTV provirus has integrated into a common chromosomal domain. A 10 kb Eco RI fragment of single copy DNA from this region has been isolated and partially characterized by restriction enzyme mapping. Of the proviruses located within this fragment in different tumors, all but one are complete, in the same orientation, and clustered within about 3 kb of cellular DNA. These findings are consistent with an insertional mutagenesis model for tumorigenesis by MMTV, in which the integration of a provirus in a particular region of cellular DNA may activate a neighboring oncogene. The region we describe here appears to be different from that reported for mammary tumors in the C3H strain of mouse. © 1983.
In vitro protein synthesis and DNA sequence analysis indicate that mouse mammary tumor virus differs from other well-characterized retroviruses in that the long terminal repeat region of the provirus has the capacity to encode proteins. Different exogenously transmitted mouse mammary tumor virus strains and endogenous proviral units conserved this open reading frame feature in the long terminal repeat despite a variation in nucleotide sequence. The proteins encoded by the different long terminal repeats were clearly related, but showed minor variations in size and tryptic peptide maps. In each case, the largest in vitro product had a molecular weight of about 36,000 to 37,000, suggesting that the open reading frame sequences must extend for approximately 1,000 nucleotides beginning at the extreme 5' end of the long terminal repeat. The fact that the reading frame was conserved among these viruses argues in favor of an in vivo function for the open reading frame protein.
The integrated proviral DNA of RNA tumour viruses is bounded by long terminal repeat (LTR) segments of several hundred base pairs which result from duplication of sequences from both the 5' and 3' ends of the viral genome RNA1-3. Recently, DNA sequence analysis of the LTRs of several retroviruses has indicated that this region of the provirus may contain regulatory functions for viral RNA transcription 4-8. In this connection, mouse mammary tumour virus (MuMTV) is of particular interest in that transcription of the viral RNA can be specifically modulated by glucocorticoid hormones, suggesting that the site of steroid action may be contained within the LTR9. Moreover, the LTR of MuMTV is unusually long, extending for about 1,300 base pairs. We now present data which suggest that, in contrast with other retroviruses, the MuMTV LTR includes approximately 1,000 nucleotides in an open reading frame, capable of encoding a series of polypeptides which are quite distinct from any of the known structural components of MuMTV virions. These proteins can be expressed in vitro from cloned, recombinant DNA in which the MuMTV LTR has been inserted into the ampicillin-resistance gene of a bacterial plasmid. Tryptic peptide mapping has confirmed that these products are identical to the analogous series of proteins identified by in vitro translation of polyadenylated fragments of MuMTV genome RNA10. © 1981 Nature Publishing Group.
int-2 was discovered as a proto-oncogene transcriptionally activated by MMTV proviral insertion during mammary tumorigenesis in the mouse. Sequence analysis showed int-2 to be a member of the fibroblast growth factor family of genes. In normal breast and most other adult mouse tissues, int-2 expression was not detected except for low levels in brain and testis. However, using in situ hybridization, expression was found at a number of sites during embryonic development, from day 7 until birth. An analysis of the int-2 transcripts found in embryonal carcinoma cells revealed six major classes of RNA initiating at three promoters and terminating at either of two polyadenylation sites. Despite the transcriptional complexities, all size classes of RNA encompass the same open reading frame. Using an SV40 early promoter to drive transcription of an int-2 cDNA in COS-1 cells, several proteins were observed. These were shown to be generated by initiation from either of two codons: One, a CUG, leads to a product which localizes extensively to the cell nucleus and partially to the secretory pathway. In contrast, initiation at a downstream AUG codon results in quantitative translocation across the endoplasmic reticulum and the accumulation of products ranging in size from 27.5 x 103M(r) to 31.5 x 103M(r) in organelles of the secretory pathway. These proteins represented glycosylated and non-glycosylated forms of the same primary product with or without the signal peptide removed. These findings suggest the potential for a dual role of int-2; an autocrine function acting at the cell nucleus, and a possible paracrine action through a secreted product.
This grant investigates early modern women's participation in the mode of complaint, culminating in a symposium held in the final year of funding at VUW. It is a companion project to the ARC DP headed by A/Prof Rosalind Smith, Early Modern Women and the Poetry of Complaint, 1550-1640.
Prof Hugh Craig; Prof Lisa Adkins; A/Prof Ros Smith; Prof Roland Boer; Prof Philip Dwyer; Dr Bill Palmer; A/Prof Mark Harvey; Prof Victoria Haskins; Prof Lyndall Ryan;; Dr Trisha Pender.
This news story details a startup competition being held by the Centre for 21st Century Humanities.
Pitchfest 2018 was held by the Centre for 21st Century Humanities in August and invited industry to partner with the centre.
This page contains a news story about the Blacksmith Repair Day supported by the Centre for 21st Century Humanities.
Deputy Director of the Centre for 21st Century Humanities and the Early Modern Women Research Network, Professor Ros Smith is leading the development of a new Centre bringing together Chinese and Australian early modern studies academics.
What does the poetry of the disadvantaged people of the Renaissance tell us about the culture of that time?
Think Mary, Queen of Scots and a few key facts probably come to mind: she was Catholic, she was imprisoned and she had her head chopped off.


In the philosophy of Martin Heidegger, Being-in-itself is contrasted with the being of persons, which he terms Dasein. (Heidegger 1962, p. H.27) "Dasein means: care of the Being of beings as such that is ecstatically disclosed in care, not only of human Being...Dasein is itself by virtue of its essential relation to Being in general." (Heidegger 2000, p. H.31) Heidegger recognized the dangers inherent to talking about Being in general and particular beings, and thus devoted space in Being and Time and the Introduction to Metaphysics to an explication of the differences; often noted by translators who distinguish Being (Sein), from a being (das Seiende). His attention to the complication is helpful for those who are looking for detailed explanation, but rarely clears the air of confusions.
↑ "Existentialism Is a Humanism", Lecture by J.P. Sartre, 1946.


Okay, I'll be honest, when JP first presented the idea of a "Chili Cookoff" to me, I was less than excited.
Exhibit A) Everyone's in school! People don't want to take the time to make chili!
Exhibit B) I don't even know how to make chili.
Exhibit C) We hosted a BBQ just 3 weeks ago! I don't want to dominate people's social calendar.
But luckily JP just ignored these hollow protestations and eventually I got on board and voilá! We had the most fabulous chili cookoff I have ever attended (also, the only chili cookoff I have ever attended. But the bar is set preeeeetty high now).
Everyone brought amazing chili's, we got to spend quality time with friends we love, play the most hilarious game ('Head's Up' -- look it up on the app store!), and eat JP's signature pie again! So I would call the night a success.


text: Mujuonly Git Page

redpajama_set_name: RedPajamaGithub


All of Gene Watson's Peers, who were contacted by The Gene Watson Site, during 2008, were most gracious with their time and words.
It is here, within this special part of The Gene Watson Fan Site, that you have an opportunity to read a quote from Darrell McCall, which he submitted to this site on Saturday 4 October 2008.
Sean Brady would like to take this opportunity to say 'thank you' to Darrell McCall who made a special contribution to this unique part of this online 'celebration of a Lone Star Hero'.
Sean Brady would also like to express a heartfelt 'thank you' to Tracy Pitcox at Heart of Texas Records in Brady, Texas without whose help this Gene Watson Peer's Quote from Darrell McCall would not have been possible.
Tracy Pitcox is also a disc jockey at KNEL in Brady, Texas which broadcasts on 95.3 FM & 1490 AM.
This quote was submitted on Saturday 4 October 2008.
'Gene Watson has one of country music's greatest voices.
He can turn any song into his own and I admire his talent and his ability as a great entertainer'.
Thank you, Darrell McCall, for your support of Gene Watson.
Darrell McCall is a native of New Jasper Township, Greene County, Ohio where he was born on 30 April 1940.
When he was fifteen years old, Darrell McCall landed a job as a disc jockey at a local Ohio radio show on Saturday mornings. During this time, Darrell McCall also performed as a musician at dances and other events.
After graduating from high school, Darrell McCall joined the military and was stationed in Kentucky.
After his tour of duty was completed, Darrell McCall and his childhood friend Johnny Paycheck (Tuesday 31 May 1938 - Wednesday 19 February 2003) moved to Nashville in 1958.
Darrell McCall and Johnny Paycheck (Tuesday 31 May 1938 - Wednesday 19 February 2003) attempted to record as a duo, but they were unsuccessful.
Darrell McCall then became a studio harmony vocalist, singing on records by Faron Young (Thursday 25 February 1932 - Tuesday 10 December 1996), George Jones (Saturday 12 September 1931 - Friday 26 April 2013) and Ray Price (Tuesday 12 January 1926 - Monday 16 December 2013).
In a short time, Darrell McCall's studio work led to road work as he played bass and sang harmony for the touring bands of Faron Young (Thursday 25 February 1932 - Tuesday 10 December 1996), Ray Price (Tuesday 12 January 1926 - Monday 16 December 2013) and Hank Williams Junior.
It was during a recording session in 1959 when Darrell McCall met Buddy Killen (Sunday 13 November 1932 - Wednesday 1 November 2006), a renowned Nashville producer and publisher.
Buddy Killen (Sunday 13 November 1932 - Wednesday 1 November 2006) was sufficiently impressed with Darrell McCall's abilities and asked him to join a group he was putting together called The Little Dippers, a group which included Hurshel Wayne Wiginton (Saturday 29 January 1938 - Monday 6 March 2017), Delores Dinning and Emily Gilmore.
The Little Dippers enjoyed one major Billboard pop music hit single in 1960, a Top 10 single called 'Forever'.
In 1961, Darrell McCall signed a solo recording contract with Capitol Records and saw the release of two pop music singles, 'My Kind of Loving' and 'Call The Zoo', both of which did not chart.
In 1962, Darrell McCall returned to the country music genre when he signed a recording contract with Philips.
In January 1963, a single, Darrell McCall saw the release of 'A Stranger Was Here', which was backed by The Milestones; the track, which reached No.17, spent eight weeks on the Billboard country music singles chart.
It was also in 1963 when Darrell McCall sang the theme song for the Martin Ritt-directed movie 'Hud', which starred Paul Newman (Monday 26 January 1925 - Friday 26 September 2008) in the lead role; the theme song was released as a single, but it did not chart on the Billboard country music singles chart.
In 1965, Darrell McCall decided to begin an acting career and appeared in numerous films, including 'Nashville Rebel', 'Road To Nashville' and 'What Am I Bid'. During his acting career, Darrell McCall also traveled to the American Southwest to perform in rodeos as a cowboy.
In 1968, Darrell McCall decided to return to recording and joined the roster of Wayside Records, an independent record label; between 1968 and 1970, Darrell McCall achieved four Billboard country music hit singles for the label; 'I'd Love To Live With You Again', which was written by Dicky Overbey (Monday 11 May 1942 - Saturday 4 October 2014), 'Wall of Pictures', 'Hurry Up' and 'The Arms of My Weakness'.
Gene Watson recorded 'I'd Love To Live With You Again', which was written by Dicky Overbey (Monday 11 May 1942 - Saturday 4 October 2014), and included the track on 'Beautiful Country' (Capitol Records, 1977).
On Monday 26 September 2005, England's Hux Records released 'Beautiful Country' (Capitol Records, 1977), along with 'Because You Believed in Me' (Capitol Records, 1976), as a special 2-for-1 CD set (HUX 072).
The Nashville Edition, including Joe Babcock, Dolores Edgin, June Page and Hurshel Wayne Wiginton (Saturday 29 January 1938 - Monday 6 March 2017) (vocals).
In 1970, Darrell McCall saw the release of 'The Arms of My Weakness', a non-album single which reached No.62 on the Billboard country music singles chart.
In 1971, Darrell McCall's contract with Wayside Records expired, but Darrell did not sign another recording contract immediately.
Hank Williams Junior recorded Darrell McCall's 'Eleven Roses' (co-written with Lamar Morris) and included the track on 'Eleven Roses' (MGM Records, 1972); the track was No.1 on the Billboard country music singles chart for two weeks in July 1972.
As a consequence of Hank Williams Junior's recording of 'Eleven Roses', Darrell McCall was signed by Tree International as a professional songwriter.
Faron Young (Thursday 25 February 1932 - Tuesday 10 December 1996) recorded Darrell McCall's 'Eleven Roses' (co-written with Lamar Morris) and included the track on 'This Time The Hurtin's On Me' (Mercury Records, 1973).
In 1974, Darrell McCall reactivated his recording career when he signed a recording contract with Atlantic Records; his debut single for the label, 'There's Still a Lot of Love in San Antone', reached No.48 on the Billboard country music singles chart in 1974.
In 1975, Darrell McCall left Atlantic Records and joined the roster at Columbia Records, where his first single was 'Pins & Needles (in my heart)', which reached No.52 on the Billboard country music singles chart in 1976.
Darrell McCall's 'Lily Dale' (Columbia Records, 1977) was subsequently re-issued, on CD, by Brady, Texas-based Heart of Texas Records in 2008.
In 2005, Gene Watson recorded the Bob Wills' classic 'Lily Dale' (written by T. Moore and Billy Jack Wills); the track was included on the various artists compilation album, 'Bob Wills: Tribute to Bob's 100th Birthday' (Common Ground Records, 2005).
Gene Watson recorded 'Dreams of a Dreamer', which was written by David Hugh 'Jug' Brown (Tuesday 28 July 1936 - Tuesday 7 May 2013), and included the track on 'Back in the Fire' (Warner Bros. Records, 1988).
On Friday 11 November 2016, Morello Records released Gene Watson's 'Back in the Fire' (Warner Bros. Records, 1988) and 'At Last' (Warner Bros. Records, 1991) as a special '2-for-1' CD set (WMRLL 66).
In 1979, Gene Watson recorded 'Sad Songs & Waltzes' (written by Willie Nelson); the track was intended to be included on Ernest Tubb's 'The Legend & The Legacy' (First Generation Records, 1979), but it was not included.
First Generation Records, and this tribute album to Ernest Tubb (Monday 9 February 1914 - Thursday 6 September 1984), was the brainchild of famed session steel guitar player Pete Drake (Saturday 8 October 1932 - Friday 29 July 1988).
Gene Watson's recording of 'Sad Songs & Waltzes' (written by Willie Nelson) did not see the light of day until Step One Records released their version of this Ernest Tubb set in 1989 as 'Ernest Tubb Collection' (Step One Records, 1989).
The Ernest Tubb album was subsequently released as 'The Legendary Ernest Tubb & Friends' (Laserlight Records, 1992) and 'Ernest Tubb & Friends' (Prism Leisure, 2004); both releases included Gene Watson's two 'duets' with Ernest Tubb, 'Sad Songs & Waltzes' (written by Willie Nelson), along with 'Yesterday's Winner is a Loser Today'.
Darrell McCall later switched record labels to RCA Records, where he recorded the single 'Long Line of Empties', which was produced by Ronnie Milsap, and which reached No.43 on the Billboard country music singles chart in 1980.
In 1984, Darrell McCall saw the release, on Indigo Records, of 'Memphis in May', a non-album single, which reached No.79 on the Billboard country music singles chart.
In the 1990s, Darrell McCall signed with Artap Records and saw the release of three successful album projects; 'All She Did Was Fall in Love' (Artap Records, 1992), 'A Way To Survive' (Artap Records, 1995) and 'Pictures Can't Talk Back' (Artap Records, 1997).
On Monday 1 June 2009, Brady, Texas-based Heart of Texas Records released Darrell McCall's 'A Way To Survive' (Artap Records, 1995), along with 'All She Did Was Fall in Love' (Artap Records, 1992), as a special '2-for-1' CD set.
In 1997, Terry Awards honoured Darrell McCall with its prestigious 'Living Legend Award' in Fort Worth, Texas.
In 1998, Darrell McCall was inducted into The Texas Country Music Hall of Fame in Austin.
In late 2004, Darrell McCall joined the roster of Heart of Texas Records, a record label based in Brady, Texas and saw the release of 'Old Memories & Wine' (Heart of Texas Records, 2004), which was produced by Justin Trevino, and afforded Darrell McCall, for the first time in his career, the opportunity to select both the material and the musicians.
At the time of the acquisition of this Gene Watson 'Peer's Quote', in October 2008, Darrell McCall, along with his wife, Mona McCall, an accomplished vocalist in her own right, continued to tour and perform on a regular basis.
Mona McCall is also part of Darrell McCall's touring band, where she contributes guitar and vocals - Darrell & Mona McCall reside close to Brady in Texas.
In 2008, Darrell McCall saw the re-issue, on CD, by Brady, Texas-based Heart of Texas Records, of 'Lily Dale' (Heart of Texas Records, 2008), which was originally released by Columbia Records in 1977.


The purpose of this article was to demonstrate that companies which compete within dynamic industries do not only adopt a single strategy. Instead, they pursue a number of strategies focusing on the value net rather than on the value chain. We carried out a survey with 47 event-planning firms based in the City of Sao Paulo and we have discovered five distinct groups of firms pursuing different strategies. Our findings support our hypothesis for the searched companies and also demonstrate how the Delta Model by Hax and Wilde II (2001) is useful to explain companies' strategic configuration. Particularly within the event industry, we have observed that companies are migrating to strategies based upon the value net. This study contributes to managing approaches concerning the challenges faced by event firms when learning how to adopt different strategic positions based upon both the value network and the value chain, in order to obtain and sustain competitive advantages in dynamic markets.
Value net; Delta model; Strategic management.


text:
Mrs. Janie Macoy,
"Fairholme",
396 Loose Road,
Kent ME15 9TX
Mr David May,
3 Meadow View
Kingsmead Park
Allhallows,
Kent, ME3 9TE
Miss Diana Welburn,
61 Barberry Ave,
Chatham,
Kent.
ME5 9TA
Mr. Chris Saville,
Sparrow Court,
Northbourne Road,
Great Mongeham, Deal,
Kent CT14 0LB.
Minutes of KBBA A.G.M. 2008
at the Janie Macoy's, Maidstone on 9th September 2007.
The Chairman welcomed everyone and opened the meeting at 2010hrs.
Present; President Janie Macoy (Tunbridge Wells)
Chairman David May (Medway)
Secretary Chris Saville (D & D)
Maidstone; Not currently affiliated
Tunbridge Wells; Marion Wraight, Brian Stevens, Brian Mason, Jean Mason
Medway; Jackie Peckham, Gerry Peckham.
Dover & Deal; Ann Keen.
The Secretary advised the meeting of the voting procedure of three votes per league.
Apologies For Absence;
Di Welburn (Treasurer), Peter Sainsbury, Martin Russell, Vinnie Mitchell, Gerry Fitzjohn (Delhi Belly), Garry Speller (Vice Chair), Craig Carlson
Minutes of Previous A.G.M. 9th September 2007 at Janie Macoy's, Maidstone.
The minutes had been circulated in advance but the secretary furnished a copy to those people who required one. After a suitable pause it was proposed by Jackie Peckham and seconded by Gerry Peckham that they be accepted. – carried unanimously.
Matters Arising;
There were none
Correspondence;
There was none,
President's Address;
Janie Macoy thanked everyone for coming. She thanked Chris Saville for his continuing hard work as secretary. She also added that it was very disappointing to hear that the Chairman and Secretary were both stepping down after many years, thanking them for their efforts. She hoped that officers could be found to continue the association.. She wished Dave May a happy and successful time in his move to Romania.
Secretary's Report (as dictated);
Good evening this is the Secretary's report for 2007-08.
Nationally, once again, all of this is almost a year old now due to the current playing AEBBA format. Open Intercounties we were relegated from division 1. No one excelled on the day, the best anyone winning was two out of four. We needed to beat Hampshire 5 – 2 in the last to survive, but could only manage 4 – 3. The 'B' team similarly came bottom of division 3, with Curt Diver winning four out of six. The ladies consolidated their promotion last year with third place in division one, winning two out of three matches, Janie Macoy and Ann Keen winning two out of three matches. Once again, in the open, it was a question of getting those players who were willing to give their time for Kent rather than those who were top of the rankings.
Individuals were a blank year. AEBBA open singles, Mark Brewster was second in his group to Kevin Tunstall who he lost narrowly to in his final group match.. Jackie represented KBBA in her group, but did not qualify. The Tunbirge Wells team (Greyhound??) did not qualify in the NBBA Pub Team. In the opens Curt Driver made the final at Portsmouth and JJ Adley won the plate at Bucks, a bleak year again.
The 'A' interleague was won by Dover and Deal,. Medway continued their good run of late by winning the 'B' and the Ladies Interleague. Curt Driver played superbly in the 'B' with a lowest score of almost 9k The knock outs day was poorly supported this year, I've just realised I have not done a write-up on it and will need to find the sheets. The loss caused by this event meant that the interleagues lost money for the first time.
The KBBA web page has continued to be the principal source for info on all matters and entry forms, saving me a massive amount of work posting forms, most of which are a waste of printing. Once again, because of this, there are no Secretary's expenses this year.
I gave notice of my intention to cease as secretary last year. I have devoted an enormous amount of time running bar billiards in this county and locally for a number of years. The office needs someone with fresh ideas and fresh enthusiasm. I hope that person is to be found and I will assist and advise where required for the first year.
The trailer and tables project has been a bitter disappointment to me. The tables were widely acknowledged as being better than the national set. The two hires per year they I have been restricted to due to the stance of the other consortium have not covered costs this last two years. I would have had to increase charges to close on the Daniels hire rates to cover the money I've been losing. Doing that would mean little point in keeping eight tables at my place all year. I was taking a long hard look at the future of the set when the news broke that Bar Billiards Ltd were ceasing trading. That news meant that Dover & Deal had to dispose of the trailer which they owned to finance table purchase. That would have meant me spending another £1k for no return to buy the trailer. Sadly I had but one choice.
Dover & Deal and Tunbridge Wells now operate their own tables. D&D expect to be financially independent in two years. After that there will be a healthy surplus in our budget and we could look once again at the table situation if a joint project were considered.
The Betteshanger Club has proved to be a fine venue for the interleagues and was free of charge. The venue would still be available next year, possibly free, if the interleagues continue.
Finally, the association has major decisions, both constitutionally and financially, to make on its future tonight and I wish you all well.
Treasurers Report;
The Secretary reported that work commitments had prevented the Treasurer from attending this evening and had furnished the secretary with a copy of the accounts. The Secretary informed the meeting that the present total balance was approximately £310 a fall of £80 on the previous balance, largely caused the £53 loss on the interleagues. We could survive another year on this budgeting, but what have to re-examine finances after if losses continued. It was proposed that the accounts be accepted, seconded by Ann Keen an passed unanimously.
Proposals;
There were none.
Election of Officers;
The Chairman and secretary confirmed they were standing down. All other officers had offered to stand.
Secretary, the secretary had circulated a job description, though the TW reps present had not seen it. TW nominated Martin Leach though he was concerned about the workload, Medway said Gerry Fitzjohn had volunteered, provided Chris Saville was prepared to help in the first year. It was proposed by Brian Stevens that Gerry Fitzjohn take over, this was seconded by Jackie Peckham and passed unanimously.
Chairman, the Chairman confirmed his resignation. The meeting then considered who would be suitable to take over as a responsible and sober Chairman, but being devoid of ideas came up with Vinnie Mitchell who had volunteered. It was proposed by Brian Stevens and seconded by Gerry Peckham that he be elected. This was passed unanimously.
It was proposed by Brian Stevens and seconded by Janie Macoy that the rest of the officers be voted in en-bloc. It was passed unanimously that Garry Speller continue as vice chair and Di Welburn as treasurer.
Chris Saville would act as secretary for the remainder of the meeting and Dave May would act as Chairman.
Secretary's Honorarium;
The Secretary did not withdraw, pointing out that he was happy that the current honorarium should not be increased due to finances, but expressed concern that it should not be reduced this year for an incoming secretary. The chairman agreed with this assessment pointing out that it was retrospective, so no change was made, proposed Gerry Peckham and seconded Janie Macoy, passed unanimously,.
Life Vice Presidents;
The secretary informed the meeting of the current LVP's and said that this honorary award was given to people who had made an outstanding contribution to KBBA or had left office recently. He suggested that this situation applied to the Chairman. He proposed that he be accorded the rank and privileges of Life Vice Presidents of the association,. This was seconded by Brian Stevens and passed unanimously.
AEBBA Affiliation;
It was proposed by Jackie Peckham and seconded by Brian Stevens that we remain affiliated, subject to there not being a large increase, this was carried unanimously. It appeared that the affiliation fees had not been paid by KBBA the previous year. Chris Saville said that it was likely he had paid them himself and forgotten to claim them. he would check this at Reading. Janie Macoy raised the question on KBBA paying entry fees at Reading, she had paid some last year, Chris Saville said he had also, but had left it. The meeting agreed that the association could no longer afford this gesture and that players should pay their own entry fee in future.
AEBBA Inter County Selection.
1) Open, Chris Saville reported that once again he was having trouble selecting the teams. Mark Brewster, Chris Saville, Doug Phillips, Peter Sainsbury and Derek Latham had accepted as the five high ranking players. Curt Driver and Gerry FitzJohn had been elected as the two best wildcards with John Addley as reserve.
2) Ladies, the secretary said that the three automatic places had gone to the first three on the list, Jackie Peckham, Janie Macoy and Jean Baker. Ann Keen and Karen Keen had been elected as the best wildcards
3) Open 'B' Side; Chris Saville reported that he had failed to get votes from TW or Medway for team selection for over three weeks, as such the team had not been finalised. The leagues would let Chris know ASAP.
KBBA Competitions.
Interleagues;
Chris Saville outlined the financial situation with a bill for £420 being faced for table hire instead of £250, now that KBBA would have to hire tables. We would also have to take what was available on weekends and that the normal Spring Bank Holiday weekend was already booked. Any league hosting would also have to consider hall costs, currently free at Deal. Inteleague fees would have to be radically increased. The Kent Knock-Outs Day could not be hosted unless a Bank Holiday weekend was used. This day had lost substantially last year due to poor attendance. If the event was moved from Deal he thought it unlikely Dover and Deal would get more than one team in any competition any other league would need to budget for this.
Much discussion followed.
Brian Stevens suggested going to a home / away format. There was general agreement that this would not work over commitments and difficulties that caused this format to be dropped in the old interleague 'League' competition (AKA Mickey Mouse colloquially).
Tunbridge Wells had also suggested running it instead of the Tunbridge Wells Masters in the day before the classic, this would involve ten tables. Costing this option with hall charges was little cheaper than using Betteshanger Club.
Other ideas were outlined but none were financially viable.
It was then agreed that the only option was the Betteshanger Club. Chris Saville said that he would play there again to get the resultant free access to the hall if that helped the association.
Chris Saville said that interleague fees would have to be radically increased to £40 for an open/'B' team and £30 for a Ladies. Leagues agreed that they would try and get their own players to contribute.He would do what he could to get the same number of teams from Dover and Deal
It was proposed by Jackie Peckham and seconded by Brian Stevens that the interleagues be held at the Betteshanger Club on 21st ('A' and Ladies)/22nd ('B') March 2009. The Kent Knock-Outs Day will be dropped. This was passed unanimously.
Tunbridge Wells would continue to run the Classic whenever they decided.
Any Other Business;
Marion Wraight asked Ann Keen if she had the Lady's runner up trophy, Ann said she would investigate.
Brian Stevens gave his thanks to Dave May and Chris Saville for their work.
Chris Saville said that affiliation fees were normally set at this point. It was proposed by Marion Wraight and seconded by Janie Macoy that they be raised to £3 per team, this was passed unanimously.
Date and Venue of Next Meeting;
Next A.G.M or committee meeting TBA if circumstances necessitating one arose
The Acting Chairman closed the meeting at 2225hrs.


Life In The Wild Water Bottle Labels make a fun addition to your event.
Add Life In The Wild Personalized Water Bottle Labels to your wild adventure. Each water bottle label features a giraffe and an elephant along with your custom text.


text:
← Initiatives
Human Rights and the U.S. Immigration System
UUSC believes that migrants in the United States should have the right to seek asylum, be free from arbitrary detention and deportation, and that families should stay together out of detention.
We are supporting our partners in their work to document abuses in immigration detention centers and at the U.S.-Mexico border, to provide legal assistance to seek asylum, and to establish and maintain a national pro bono hotline for immigrants in detention to receive legal support, connect with family, and advocate for themselves.
We collaborate on original research and documentation, such as our reports on mental health assessment in detention and the Central American Minors In-Country Refugee Processing (CAM) refugee program.
We also campaign for more just and humane U.S. immigration policies that will keep families together in our communities, and do not deport them to danger. Tearing our communities apart makes no one safer.
Defending Dreamers
The Deferred Action for Childhood Arrivals (DACA) program provided protections from deportations and the ability to work and attend school for more than 800,000 young immigrants. In September 2017, the Trump administration cancelled this program, removing DACA recipients' (commonly referred to as "Dreamers") legal status and leaving them vulnerable to deportation.
UUSC and our partners and allies continue to advocate for passage of a clean Dream Act, which would create a pathway to citizenship for more than 2.1 million undocumented young immigrants who came to the United States as children, without additional anti-immigrant provisions that harm our communities, such as billions of dollars to expand the border wall, more immigration detention centers, or restrictions to diversity visas and family-based migration.
Temporary Protected Status (TPS) is designation that the U.S. government can offer non- citizens who are unable to safely return to their countries of origin due to political instability and violence or as a countries recovers from a natural disaster. As of October 2017, 437,000 people had TPS in the United States from 10 countries—El Salvador, Haiti, Honduras, Nepal, Nicaragua, Somalia, Sudan, South Sudan, Syria, and Yemen.
Defend Immigrant Rights by Protecting TPS
Sign and send a message to your legislator urging them to protect TPS holders and allow them to continue to build their lives here.
TPS is one way the U.S. government can honor its moral and humanitarian obligation to provide safe haven to people who may be in danger. However, under the Trump administration, TPS has been drastically curtailed. Since August 2017, the Department of Homeland Security (DHS) has cancelled TPS for multiple countries, hundreds of thousands of people at risk of losing their livelihoods and being separated from their families.
Love Resists
In the wake of the 2016 election and the emergence of President Trump's anti-immigrant agenda, UUSC joined together with the Unitarian Universalist Association (UUA) to ensure our communities were protected. This partnership developed into Love Resists, a joint campaign that activates people of faith and conscience to resist the criminalization of our neighbors and communities, and create a safer, more just, welcoming, and sustainable world. Learn more about the campaign and join us in this work at loveresists.org.
Freedom for Immigrants
Georgia Latino Alliance for Human Rights
Rural Community Workers Alliance
UndocuBlack Network
Love Resists Deportation on the Capitol Steps
Criminalization: Legalized Discrimination


The English Pale Ale has been a long-lasting and inspirational style for brewers; it gave birth to the IPA that is loved by many. But with everything, the pale ale has evolved over the years. The idea of a pale ale was changed in 1979 when Ken Grossman launched Sierra Nevada Brewing Company and redefined what a pale ale was, using American Cascade hop to create the iconic Sierra Nevada Pale. Since that day, Sierra Nevada Pale has been a principle example of the American pale for brewers. This style continues to evolve with new American brewing and hop technics.
pFriem's Extra Pale Ale is a stronger version of the classic pale ale style. This beer has more alcohol, hop flavor, and hop aroma then a pale ale, but not as much as an American IPA. New American hop varietals make this beer very fruit forward, juicy, and round.
This strong pale ale is bright golden with an amber hue. Fruity aromas of melon, pineapple, berry, and currant. Fresh and juicy flavors of strawberry and gooseberry. Finishes piney and slightly tangy.
Mexican food, sweet and sour flavors found in thai and vietnamese cuisine. White fish such as halibut, rock cod, and dungeness crab. Fruit dishes such as chutney can play off the fruitiness of the beer.


text:
Q: Finding by id in Cassandra does not work I'm having a weird behavior with Cassandra. Whenever I try finding some rows by id, it returns me null (either by cqlsh console or clients), but the register exists if I find by another field.
For example:

My table schema:
CREATE TABLE IF NOT EXISTS user_aggregation_new.user_aggregation_new (
    userid timeuuid PRIMARY KEY,
    fullname text,
    birthdate text,
    password text,
    mobile text,
    address frozen <address>,
    thumbs map<text, text>,
    driverlicense frozen <driverlicense>,
    cpf text,
    facebookid text,
    membershipsince timestamp,
    rating double,
    email text,
    status text,
    facebookemail boolean,
    bankaccount list<frozen <bankaccount>>,
    dateupdated timestamp,
    version bigint
);

Cassandra version: 3.7
Why is this happening  ?

A: You have two options :

*

*Run nodetool repair on each node to improve data consistency

*Increase your client consistency level. In cqlsh, run the command CONSISTENCY with value QUORUM or ALL
Then retry and get back to us if it is still not working.