Are Superteams Good for Sports?
Sport produces two types of fans. The casual fan who will take in a game for leisure or simply the aesthetic of the sport. And then you have the die-hard. The die-hard is there week in and week out. They have jerseys, know the players' origin stories, and let them tell that they could run their organization better than management via their fantasy team. Super teams for both sets of fans have differing consequences. On the one hand, the casual fan who is only here to be entertained enjoys the dominance that the Golden State Warriors bring to the table. The die-hard outside of Oakland, of course, hates this scenario. Parity in sport has always been ideal to maximize interest and so profits. The more teams with a realistic chance to reach the pinnacle means a healthy league and in turn a profitable one. While the notion of multiple Hall of Famers joining forces is not new, there is certainly a trend.
The NBA and NFL as examples are billion-dollar industries with a monopoly on viewership. They also have very differing ways of getting there. In comparison, both sports have a redistribution of resources via free agency and the salary cap and both have the star power that carries the sport. In 2010, Lebron James left Cleveland via free agency and joined Chris Bosh and Dwyane Wade in Miami, creating the NBA's first major power move on behalf of one player since Shaquille O'Neal left Orlando for Los Angeles in the 90s. What that did for viewership was telling. After the 2009-10 season in which Lebron and Chris Bosh bolted to South Beach, NBA viewership across all providers saw a 36% increase followed by another 6% during the lockout season. Conclusion, excitement means growth which means dollars.
The narrative matters when it comes to super teams. In 2010, the perception of Lebron James was that he had exhausted all possibility in Cleveland. Arguably, the best player in the world at the time had found himself without a ring in the NBA's vast wasteland of Ohio with no sign of rescue. And so collectively we were okay with that. Fast forward to 2017, and Kevin Durant finds himself in a similar situation with a significantly different result. Reports indicate that viewership was down 15% in local markets during the 2016-17 season, local markets being the operative phrase. The Warriors disrupted an already stacked Western Conference with sheer dominance. Not only did they disrupt, but they also lived up to the expectation.
Super teams in sport exist under certain provisions. In football and basketball, the salary cap and free agency work to limit hegemony while also allowing player mobility. Thanks to players like Curt Flood, the modern athlete has more control over his or her destiny. The departure of Lebron to Miami in 2010 devastated the Cavalier franchise. However, the system through the draft lottery which serves as a safety net for organizations allowed them to build talent through the draft which may one day draw free agents to the franchise. In this case, it was Lebron James' return.
The problem with super teams is they create a competition gap for the league. In the NBA, realistically only a handful of teams have a shot at a title. As Sir Charles says, the better team always wins a seven-game series which in most cases is true. Eventually, talent wins out. You rarely see eight seeds dethrone regular season champs and when they do circumstances such as injury are usually an indicator. The NFL does not have this problem. In part, the nature of the sport, as well as the nature of the salary cap, is preventative of super teams. Often middle of the road teams reach the Super Bowl; see the Giants in both defeats of the Patriots. Point being, when competition becomes lopsided interest dwindles. With the new television money in the case of the NBA, they already got the bag. It is the fans that will suffer.
Take soccer as another example. Regardless of how lopsided the competition viewership has not dwindled. La Liga saw a 16% percent increase in viewership after last season due to the Spanish telecast on BEin Sports. That is despite Barcelona and Real Madrid winning all titles besides conceding one to Atletico since the 2004-2005 season. What does that say? We as fans will watch if the storylines are compelling or the top dog has a rival to contend.
Athletes make the storylines and thus control the narrative. The modern athlete has more resources and freedom than ever before to maximize their talents. Neymar's record transfer to PSG is yet another example. In futbol's top five leagues, the most dominant countries in international play fill the rosters of many of these clubs. Players find themselves playing with in some cases multiple players from their national team making the transition from club to club easier. Neymar is leaving Barcelona to play with Dani Alves, Lucas Moura, Thiago Silva, and Marquinhos. Almost half of Paris' XI will be represented by Brazil. These are all players with whom Neymar has come through the ranks. The same is true in basketball. AAU creates a network of talent from an early age. Players that come up in the AAU circuit communicate and often become peers as opposed to rivals. In the old NBA where rivalries existed more fervently, this was not necessarily the case. Since the institution of free agency in 1996 and the understanding that money and winning are more important than loyalty, players are more inclined to do what is best themselves and their future. In recent years that has meant teaming up with a fellow Olympian much like the 2012 Lakers attempted in the wake of the Miami Heat's acquisitions.
What does that mean for the fans? The fans are ultimately left with a decision to make. Ratings and revenue will determine whether super teams are good for sport or not and athletes have zero incentive given the current landscape not to look out for their interest. As fans, the decision to support depends on why you are here. Are you here for the league or are you here for your team? If the former, you should be excited for the chain reaction teams like the Warriors will bring. The need to adapt breeds innovation. In a league that is ever evolving, whoever is next will certainly push the envelope to dethrone the title holder. If the latter, on that same point, you may find yourself disappointed or moving to a new sport as players team up to contend with their new reality.
Fade in: Back at it again bringing you episode No. 2 of the weekly TV roundup! The theme of the week: THIS. JUST. IN! 'American Gods', 'Dear White People', & 'The Handmaid's Tale.' Let's get started!


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Dove will stop using virgin plastic in 2019
Mar 31 2019 by Victoria DiPlacido
Categories : Skincare
At a global media forum in London, England this week, Dove announced its commitment to ending the use of virgin plastics in all product packaging in North American and Europe, where technically feasible, by the end of 2019. Bottles will instead be made of 100% recycled plastic.
It's no secret that plastic waste is one of the biggest global environmental challenges of our time. According to one research article published in a 2017 issue Science Advances, most of the 8.3 billion tonnes of plastic humans have created has been discarded—and is now sitting in landfills and polluting the natural environment.
"In the beauty world, we need to think seriously about whether we are over-packaging; how much do we really need to make something look gorgeous?" says Gavin Warner, director of sustainable business at Unilever, which owns Dove, as well as beauty brands like TRESemmé, Simple, St. Ives, Pond's and Vaseline, among others.
In January 2017, Unilever announced all plastic packaging across all of its brands would be made fully reusable, recyclable or compostable by 2025. With Dove's new commitment, the personal care brand leaps significantly ahead of Unilever's global ambitions.
Today is Global Recycling Day! ⠀⠀⠀⠀⠀⠀⠀⠀⠀ ⠀⠀⠀⠀⠀⠀⠀⠀⠀ At Unilever we are working hard to tackle plastic packaging waste. ⠀⠀⠀⠀⠀⠀⠀⠀⠀ ⠀⠀⠀⠀⠀⠀⠀⠀⠀ We're focusing our efforts in three areas: designing our packaging so we use less plastic in the first place; looking for alternative materials so we move away from plastic; and, where we do use plastic, making sure it stands the best possible chance of being recycled. ⠀⠀⠀⠀⠀⠀⠀⠀⠀ ⠀⠀⠀⠀⠀⠀⠀⠀⠀ Click the link in our bio to read more about why and how we're moving to a circular economy for plastic. ⠀⠀⠀⠀⠀⠀⠀⠀⠀ ⠀⠀⠀⠀⠀⠀⠀⠀⠀ Photo taken by Carmen Heah, Unilever Category Revenue Manager, Malaysia⠀⠀⠀⠀⠀⠀⠀⠀⠀ -⠀⠀⠀⠀⠀⠀⠀⠀⠀ -⠀⠀⠀⠀⠀⠀⠀⠀⠀ -⠀⠀⠀⠀⠀⠀⠀⠀⠀ -⠀⠀⠀⠀⠀⠀⠀⠀⠀ -⠀⠀⠀⠀⠀⠀⠀⠀⠀ -⠀⠀⠀⠀⠀⠀⠀⠀⠀ #takeaction #circulareconomy #noplanetb #lineinthesand #sustliving
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It's a move that's not without risk, says Warner. "The material available to buy to make these products is not perfect," he explains. Dove bottles made from recycled plastic will have "imperfections" in them. On a prototype of a recycled plastic Dove Baby Shampoo bottle displayed at the event, dots and streaks of grey were only somewhat evident—still, it's a departure from the pristine all-white containers Dove is known for. But so what, asks Warner. "I'd rather have a recycled plastic bottle than one that contributed to perpetuating the use of fossil fuel and raw materials."
It's not a one-size-fits-all solution. For some products, like the colour-vignetted bottles in the brand's haircare range, no current technical solution exists to recreate the packaging with recycled plastic.
Another (misleadingly simple sounding) solution to the plastic problem: stop making things out of it. It's an avenue Dove is currently exploring. Together with Terracycle-owned Loop—a waste-free, direct-to-consumer shopping system—they are working to create a Dove deodorant stick packaged in reusable and refillable stainless steel, though no launch date has been set.
"Every player in the space needs to accept their responsibility," says Warner, of how plastic pollution must be combated at the individual, company and government levels. "The start is to say: 'I accept my part.' And fix the area you can control." For example, he says, brands like Unilever can control the design and material of packaging, government workers can implement systems that make it easy for people to know how to recycle, and individuals can be more conscious of what they buy and how they dispose of it.
"That's where the secret lies, in everybody accepting their part," says Warner. "And then finding a way to change the system."
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Why Family Who Hid Saugerties Girl Under Steps Wore Blue to Court
Boris Published: March 7, 2022
Saugerties PD
Family members accused of abducting 4-year-old Paislee Shultis and hiding her under their staircase all showed up in court dressed in blue.
The story sent shockwaves through the Hudson Valley. Last month, police investigated a Saugerties home and uncovered Paislee Shultis, a young girl who was reported missing from her home back in 2019. Police found the girl in a makeshift room hidden beneath a staircase.
Authorities arrested the young girl's biological parents, Kimberly and Kirk Shultis, as well as Paislee's grandfather, Kirk Shultis Sr. All three were charged with custodial interference and endangering the welfare of a child.
According to News 10, Kimberly Shultis appeared in court last week and joined her family and supporters in sending a secret message to the public with the clothes they wore to the courthouse. Kimberly, as well as the rest of her family, all dressed entirely in the color blue. Many were unaware of the reason for the wardrobe choice until the family's lawyer told News 10 that it was done on purpose to send a message.
Attorney Carol Morgan said the color choice was made because blue was a "calming" color. She indicated that it conveys feelings of peace and unity.
This family wants peace. All of the people that showed up with them, will continue to stand by them throughout this entire process. They know them. They support them. They love them. They are a strong community and there are many others like them.
While in court, Kimberly Shultis pleaded not guilty to custodial interference and endangering the welfare of a child. Her case was adjourned until April 27 when she and Paisley's biological father and grandfather appear in Saugerties Town Court.
Source: Why Family Who Hid Saugerties Girl Under Steps Wore Blue to Court
Popular Poughkeepsie Pizza Place Destroyed by Overnight Fire


An introspective journey through music, guided by Francesco & Sofia. Uroboro, brings the fusion of opposites. It also symbolizes the transcendence of duality. Melodies for an eternal return, celebrating the wholeness with music.
Servant Jazz Quarters is a boutique music venue and cocktail bar in the heart of Dalston. Experience London's thriving live music scene during the week and some of the capital's hottest DJs on the weekend.


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Home > Bach Books >
The Medical Discoveries of Edward Bach Physician by Nora Weeks
The Medical Discoveries of Edward Bach
The Medical Discoveries of Edward Bach Physician by Nora Weeks, tells how Dr. Bach's work developed, from his childhood hopes and dreams of a healthy society, to the discovery of a complete system of 38 remedies which address all aspects of human nature, emotional outlook and personality.
In 1912 Edward Bach qualified as a medical doctor and embarked upon a career which not only had a profound effect on medical science, but brought to ordinary people the world over, a simple, safe and effective system of healing.
Dr. Bach made a detailed study of bacteriology, immunology and homoeopathy and found a clear connection between chronic disease and negative mental attitudes. It was the relationship between mind and body which formed the basis of his further research and as the became increasingly convinced that emotional harmony was the key to good health, he was determined to find a simple, non-invasive and harmless method of healing which could be used safely by everyone.
The Medical Discoveries of Edward Bach tells how Dr. Bach's work developed, from his childhood hopes and dreams of a healthy society, to the discovery of a complete system of 38 remedies which address all aspects of human nature, emotional outlook and personality. Edward Bach was an eminent physician whose remarkable contribution to medicine, healing and humanity has yet to be fully realised.
- From the book's back cover
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Bach Flower Remedies Illustrations and Preparations by Nora Weeks & Victor Bullen Heal Yourself by Christopher Hoyt Bach Flower Remedies for Cats by Martin J. Scott & Gael Mariani Bach Flower Remedies for Beginners: 38 Essences that Heal from Deep Within by David F. Vennells
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Trojan Journalism
School placed on modified lockdown to insure safety
Katie Quick, Staff member
The modified lockdown which occurred on October 5th was caused by three separate fights.
Assistant Principal Mr. Roger Lee said, "There was a modified lockdown due to several conflicts between students. We had three fights and a couple verbal altercations that didn't get physical.
One of the main issues in the commons was that students were not dispersing when they were told to." Lee went on to point out that this "created a safety risk" for all of the staff and students and didn't allow "the staff to do their jobs and work with the kids that were having the issues."
The situation required the school to call for backup from the Auburn Police Station which responded quickly, and the school was placed in a modified lockdown. The lockdown was needed to "limit student movement." said Lee, "because when students move in a situation like that things happen. So we wanted to restrict that for everybody's safety."
The administration met during that afternoon and decided what would be done for the following day. It was decided to lift the modified lockdown, but place restrictions on student movement during class time. Part of the restriction was a reminder to all teachers to follow the 10/10 Rule, which has been in place the last several years, requiring teachers to not allow any movement outside of the classroom during the first and last ten minutes of every class period.
Lee said that the "response by students has been amazing," and wanted students to know that they "have done[their] job in helping us make this a safer place."
Assistant Principal Mrs. Stacy Jordison agreed with Lee that the lack of student dispersal during the incident did not allow the administration to deal with the situation "effectively."
Mr. Lee also wanted to clear up several misconceptions about the incident. There were no weapons involved and the police who were in the school were "regular police officers" who were a great help in resolving the situation.
Jordison added that, "safety is our top priority, and when crowds don't disperse it makes it more difficult." She added, " in the future I would hope the students would disperse when asked. It takes all of us to make sure the school is safe, staff, students and administration."
Katie Quick, Managing Editor
Katie Quick is a sophomore student in Auburn High school and has been a member of the newspaper staff for over a year. Katie was born in Tacoma and is...
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The online student news site of Auburn High School


Then there are days like yesterday when I can set myself down on a large boulder in the cold, crazy rain in the middle of a glen and have them wander by. At those times they are awesome and it's an experience that's good for the soul.
This entry was posted in Landscape and tagged Deer, Glen Etive, Nature, Scotland, Wildlife. Bookmark the permalink.


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Brežuljkasti ili kolinski pojas dio je Eurosibirsko-sjevernoameričke regije u fitogeografskoj raspodjeli Hrvatske koji se nastavlja na nizinski pojas u rasponu nadmorskih visina od 150 do 500 metara.

Prirodno su, zbog povoljnih klimatskih i ekoloških prilika, šume ovog pojasa bujne i bogate vrstama, no kako je to područje izuzetno povoljno i za ljudski život i djelatnosti, one su od Srednjeg vijeka nadalje pretvarane u antropogene tipove vegetacije, kao što su pašnjaci, livade, oranice, vinogradi, živice, te naselja. Ovom pojasu pripadaju brežuljci i donji dijelovi panonskog gorja (Medvednica, Ivanščica, Kalnik, slavonsko gorje). Brežuljkasti pojas razvijen je također i južnije od Karlovca, i to u smjeru Severina na Kupi, u smjeru Josipdola, te na rubu ličke visoravni i u Istri.

Prosječna godišnja temperatura ovog područja iznosi oko 10oC, a prosječna godišnja količina oborina oko 1 200 mm u sjeverozapadnom dijelu Hrvatske, odnosno oko 800 mm u istočnom dijelu panonskoga gorja.

Najznačajnija drvenasta vrsta je hrast kitnjak (Quercus petraea), koji čini više tipova šumskih zajednica. Od ostalih drvenastih vrsta značajne su obični grab (Carpinus betulus), pitomi kesten (Castanea sativa), obična breza (Betula pendula), hrast cer (Quercus cerris), hrast medunac (Quercus pubescens), klen  (Acer campestre), divlja trešnja (Prunus avium), te bukva (Fagus sylvatica).

Ovisno o ekološkim prilikama, u ovom pojasu razlikujemo tri skupine šumskih zajednica:
Šume na neutralnim do slabo bazičnim tlima, na vapnenačkoj ili dolomitnoj geološkoj podlozi. Glavne drvenaste vrste su hrast kitnjak, obični grab, trešnja i klen.
Acidofilne šume razvijene na kiselim tlima, odnosno silikatnoj geološkoj podlozi. Glavne drvenaste vrste su hrast kitnjak, pitomi kesten i obična breza.
Termofilne šume razvijene na južnim i jugozapadnim ocjeditim obroncima, na toplim prozračnim tlima, također iznad vapnenačke ili, češće, dolomitne podloge. Glavne drvenaste vrste su hrast kitnjak, hrast medunac, crni grab, crni jasen i druge submediteranske, termofilne vrste.

Šume hrasta kitnjaka i običnoga graba

Ovaj tip šume predstavlja široko rasprostranjenu klimatogenu (klimazonalnu) vegetaciju nižeg kontinentalnog područja. Raste izvan dosega visokih podzemnih voda na brdskim terenima, nižim gorjima i podnožjima većih masiva, i to na pseudoglejnim, umjereno podzoliranim i smeđim šumskim tlima. Ne dolazi na plitkim dolomitnim tlima, na jako zakiseljenim, te poplavnim tlima. Optimalno je razvijen na neutralnim, te slabo bazičnim do slabo kiselim tlima.

Istočnu granicu svog rasprostranjenja dosiže na obroncima Dilja kod Đakova. Dobro je razvijen oko gorja sjeverozapadne Hrvatske, te u središnjoj Hrvatskoj južnije od Karlovca prema obroncima Dinarida i rubovima krških polja. Velike površine ovih šuma su iskrčene i pretvorene u različite tipove antropogene vegetacije, a rasprostiru se u rasponu nadmorskih visina od 150-450 m. U odnosu na srodne srednjoeuropske šume, ove se odlikuju znatno bogatijim florističkim sastavom i specifičnim ilirskim vrstama, pa ih stoga nazivamo i "ilirskim šumama hrasta kitnjaka i običnog graba".

Neke od tih ilirskih vrsta su mišje uho (Omphalodes verna), kukurjeci (Helleborus dumetorum, H. niger, H. artrorubens), žućkasta grahorica (Vicia oroboides), režuhe (Cardamine enneaphyllos, C. polyphylla, C. trifolia, C. waldsteiniana), biskupska kapica (Epimedium alpinum), volovsko oko (Hacquetia epipactis).

Od drvenastih vrsta najvažnije su hrast kitnjak (Quercus petraea) i obični grab (Carpinus betulus). Ako se tlo zakiseljuje, počinje izostajati grab, tako da šuma konačno prelazi u čisti hrastik, a sastojine na vlažnijim staništima, u jarcima, klancima, te uz rubove potoka često su čisti grabrici. Od ostalih drvenastih vrsta česti su klen (Acer campestre), divlja trešnja (Prunus avium), zatim brijest (Ulmus glabra), javori (Acer pseudoplatanus i A. platanoides), te pitomi kesten (Castanea sativa) i bukva (Fagus sylvatica).

Sloj grmlja dobro je razvijen i grade ga mnogobrojne vrste. Neke od njih su poljska ruža (Rosa arvensis), lijeska (Corylus avellana), divlja kruška (Pyrus pyraster), likovac (Daphne mezereum), glogovi (Crataegus monogyna i C. oxyacantha). Sloj prizemnog rašća također je izuzetno dobro razvijen, a uz već spomenute ilirske vrste, karakterizira ga i velik broj vrsta proljetnica koje te šume čine izuzetno šarenim u proljeće prije prolistavanja drvenastih vrsta. S obzirom na vrstu tla na kojoj je razvijena, ova šumska zajednica može se podijeliti na nekoliko podtipova.

Šume hrasta kitnjaka i pitomog kestena

Ove šume razvijaju se na kiselim tlima iznad silikata i pješčenjaka, na nešto toplijim staništima, na platoima, sedlima i blažim padinama gdje se mogla skupiti veća količina tla, i to u rasponu nadmorskih visina od 250 do 550 m. Najveći kompleksi šuma kestena i hrasta kitnjaka razvijeni su na Zrinskoj i Petrovoj gori, Medvednici, te na ostalom gorju sjeverozapadne Hrvatske. Uz pitomi kesten (Castanea sativa) i hrast kitnjak, u sloju drveća mogu rasti i obični grab i bukva. Zadnjih desetljeća, zbog napada parazitske gljivice, mnoge sastojine pitomog kestena su teško oštećene, tako da su te šume danas u fazi znatnih promjena i predstavljaju vrlo ranjiv ekosustav. Na nekim staništima odumrla stabla kestena počinju zamjenjivati bukva i grab.

Sloj grmlja dobro je razvijen, a grade ga acidofilne vrste kao što su borovnica (Vaccinium myrtillus), obična žutilovka (Genista tinctoria), žučica (Chamaecytisus supinus), crnkasta žučica (Lembotropis nigricans) i dr. Sloj niskog raslinja također čine acidofilne vrste runjika (Hieracium sylvaticum, H. racemosum, H. rotundatum), urodica (Melampyrum pratense), raznolisna vlasnjača (Festuca heterophylla), bjelkasta bekica (Luzula albida) i dr. Karakterističan je i relativno velik broj vrsta mahovina od kojih su najčešći rodovi Polytrichum i Leucobryum.

Na područjima Like, Korduna, Gorskog kotara i dijela Žumberka, na mjestima gdje su iskrčene prije svega šume pitomog kestena i hrasta kitnjaka, nastaje specifičan oblik vegetacije nazvan vrištine ili bujadnice. Razvijaju se na zakiseljenim, sivim, ilovastim tlima. Služe kao pašnjaci, a ako se prekine s ispašom, zarastaju u šikare i brezike. Karakteristične vrste vriština su vrijes (Calluna vulgaris), žučice (Genista pilosa, G. germanica, Chamaespartium sagittale) i bujad (Pteridium aquilinum). Ako se kose, daju sijeno niske kvalitete koje se najčešće koristi kao stelja. Vrištine su zanimljive pčelarima zbog vrijesa, koji predstavlja medonosnu vrstu.

Termofilne šume s crnim grabom i hrastom meduncem

Ove šume razvijaju se na plitkim karbonatnim crnicama iznad dolomitne i vapnenačke podloge, na nagnutim ocjeditim terenima južnih i jugozapadnih ekspozicija. Takve prilike uvjetuju termofilni karakter ovih šuma, pa one predstavljaju ekstrazonalno razvijenu submediteransku vegetaciju. Rasprostranjene su na istočnim i zapadnim obroncima Medvednice, Žumberačkom i Samoborskom gorju, Ivanščici, Cesargradskoj gori, Kalniku, ali mjestimice i u Gorskom kotaru (npr. Skradska gora), na Kleku i u Lici. Često su razvijene u obliku prorijeđenih šikara.

Od drvenastih vrsta, uz hrast medunac (Quercus pubescens) i crni grab (Ostrya carpinifolia), česte su i hrast kitnjak (Quercus petraea), hrast cer (Quercus cerris), crni jasen (Fraxinus ornus) i mukinja (Sorbus aria).

U sloju grmlja rastu drijen (Cornus mas), kalina (Ligustrum vulgare), crna hudika (Viburnum lantana), žutika (Berberis vulgaris), merala (Amelanchier ovalis) i obična borovica (Juniperus communis).

U sloju niskog raslinja rastu mnoge termofilne submediteranske vrste. Česte su npr. pčelinja ljubica (Mellitis melissophyllum), vrapčje sjeme (Lythospermum purpurocaeruleum), crni grahor (Lathyrus niger), crvena djetelina (Trifolium rubens), travolisna perunika (Iris graminea), hrvatska perunika (Iris croatica), lastavičnjak (Cynanchum vincetoxicum), velecvjetni naprstak (Digitalis grandiflora), sjajnolisna zvončika (Campanula persicifolia) i mnoge druge.
Šume u Hrvatskoj

redpajama_set_name: RedPajamaWikipedia


If you have a dull and boring outdoor space, it is time that you add some life to it. By creating a fun and welcoming outdoor area, you, your friends, and guests can sit around and enjoy each other's company while enjoying nature that surrounds you. Below, we will go over some outdoor decorating ideas that can turn any boring lawn area into a fun and happening place to be.
If you have an outdoor patio, do not be afraid to plant flowers around your sitting area. You should choose flowers that are easy to maintain and are tough when it comes to the weather elements. If you want to be able to smell the flowers as you relax outdoors, choose fragrant varieties such as magnolias, hydrangeas, or even jasmine trees.
In addition, there are flowers that attract birds and butterflies, so if you want nature to really surround you, consider planting some of these types of flowers too.
If you live in an area where the sun beats down on you all of the time, consider having a cover over top of your patio area or outdoor garden area. You can have a gazebo installed or you can opt in for a metal canopy that sits over your social gathering area.
If you do have a cover option, consider hanging lights throughout the space to provide you with an ambient and relaxing place even at night.
You do not have to take your patterns and colors too seriously when you decorate your outdoor space, which means that you can be playful with your choices. Many people choose to mix and match patterns to create a whimsical and entertaining look to their outdoor area.
If you are looking for ideas, consider having bright, neon colors in the spring and summer as this will really add a nice touch to the area.
If you are interested in redecorating your outdoor space, consider creating an outdoor entertainment room for you and your guests to enjoy. In addition, you could have an outdoor kitchen installed and host all of your BBQs right from your backyard.


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Vernacular Terms In Architecture Pdf
Metabetchouan–Lac-a-la-Croix
Friday, January 17, 2020 2:52:20 PM
Some Vernacular Design Principles (& their application today)
Archeoz Vernacular Term and Construction Term. Study Philippine Architecture - Vernacular Terms flashcards from Philip Reytiran's class online, or in Brainscape's iPhone or Android app. Learn faster with spaced repetition., In terms of architecture, the vernacular defines the built environment as structures created by untrained individuals. This does not mean that the designs are aimless or uninformed. To the contrary, it means that the structure is suited for the particular needs of the individual who conceived of and built the structure. Vernacular Malaysian housing provides us with a highly localized example.
What does Vernacular architecture mean? Definitions.net
FROM TRADITIONAL RESIDENTIAL ARCHITECTURE TO THE. Vernacular architecture Architecture that makes use of common regional forms and materials at a particular place and time; usually modest and unpretentious, and often a mixture of traditional and modern styles, or a hybrid of several styles. vernacular architecture Architecture that makes use of common regional forms and materials at a, Vernacular architecture: A term of recent popularity, this age old phenomenon, indicated by analogy with language, refers to buildings in indigenous styles constructed from locally available materials, following the traditional.
1 Introduction: The Study of Vernacular Architecture Every weekend, thousands of people, mostly from the towns and suburbs of England, engage in the most innocent of pursuits – a visit to the countryside. PDF The quest for a deeper approach of contemporary architecture to nature sometimes conceals the achievements of the past. Vernacular architecture is, by its definition, aim and structure, the
In terms of architecture, the vernacular defines the built environment as structures created by untrained individuals. This does not mean that the designs are aimless or uninformed. To the contrary, it means that the structure is suited for the particular needs of the individual who conceived of and built the structure. Vernacular Malaysian housing provides us with a highly localized example Vernacular architecture Architecture that makes use of common regional forms and materials at a particular place and time; usually modest and unpretentious, and often a mixture of traditional and modern styles, or a hybrid of several styles. vernacular architecture Architecture that makes use of common regional forms and materials at a
Vernacular Architecture is now included in the Arts and Humanities Citation Index. Vernacular architecture utilizes materials that are available locally. Using materials that are local and building with local labor is environmentally friendly; it cuts the distance materials need to travel, cutting down on the pollution created during transportation.
The architecture of Africa, like other aspects of the culture of Africa, is exceptionally diverse. Throughout the history of Africa , Africans have had their own architectural traditions. In some cases, broader styles can be identified, such as the Sudano-Sahelian architecture of West Africa . designs and its knowledge in the form of vernacular architecture. This traditional wisdom of building human habitats is known to This traditional wisdom of building human habitats is known to be tested by time for its sustainability in various settlements.
in terms of physical form and spatial configuration in the case study of vernacular housing. Keywords Urban Morphology, Spatial Configuration, Persian Vernacular Housing, Spatial Patterns, "vernacular architecture, given the insights it gives into issue of environmental adaptation, will be necessary in the future to ensure sustainability in both cultural and economic terms
PDF The first challenge to encounter, when addressing a comprehensive understanding of the vernacular architecture problematic, is to try to define the … PDF The quest for a deeper approach of contemporary architecture to nature sometimes conceals the achievements of the past. Vernacular architecture is, by its definition, aim and structure, the
Vernacular architecture Architecture that makes use of common regional forms and materials at a particular place and time; usually modest and unpretentious, and often a mixture of traditional and modern styles, or a hybrid of several styles. vernacular architecture Architecture that makes use of common regional forms and materials at a Vernacular term in architecture in the Philippines is used locally by Filipino who worked in site. It's very useful for professionals such as architects & engineers to …
Title: Vernacular Architecture Author: JSBC/JaGBC Created Date: 11/12/2008 7:55:59 PM Vernacular architecture Architecture that makes use of common regional forms and materials at a particular place and time; usually modest and unpretentious, and often a mixture of traditional and modern styles, or a hybrid of several styles. vernacular architecture Architecture that makes use of common regional forms and materials at a
HISTORY OF ARCHITECTURE 4.0 PHILIPPINE Architecture of the Philippines The front entrance of Fuerza de Santiago towering 40 metres high. The architecture of the Philippines is a reflection of the history and heritage of the country. The most prominent historic constructions in the archipelago are from the Spanish Colonial period, though much Philippine architecture is also influenced by CULTURE AND ARCHITECTURE OF MADHYA PRADESH. The tribal and folk traditions of Madhya Pradesh are reflected in the vernacular architecture. The state is known for its visual and performing arts, these art forms are closely associated with their beliefs,
Paul Oliver Encyclopedia of Vernacular Architecture
Learning from Vernacular Architecture Sustainability and. prises Philippine vernacular architecture, there is in terms of plan compactness and simplicity, in-clude the shotgun cottage and railroad houses. Meanwhile, dwelling units above garage spaces are commonly seen in tuck-under houses, and, for smaller dwellings, in carriage houses and mews. Live/works and courtyard houses are also popu-lar house types in many traditional neighborhood, pdf. Vernacular Terms for Architecture (Philippines) 2 Pages. Vernacular Terms for Architecture (Philippines) Uploaded by. Катрина Облефиас. Download with Google Download with Facebook or download with email. Vernacular Terms for Architecture (Philippines) Download. Vernacular Terms for Architecture (Philippines) Uploaded by. Катрина Облефиас. ADOBE ANCHOR.
Philippine Architecture Vernacular Terms Flashcards by
INSTITUTIONALISED ARCHITECTURE VERNACULAR ARCHITECTURE. The vernacular architecture of Iran has a reasonable relationship with climate conditions. Nowadays for some reasons, such as fast growing population, rising land and building price and the changes in life style densely layout and tall buildings are constructed in all cities and towns CHAPTER 3 VERNACULAR ARCHITECTURE OF THANJAVUR REGION 3.1 INTRODUCTION Thanjavur is a traditional town which dates back to the 3rd century AD, and is located in south- east part of India at longitude with an elevation of 88m MSL. It is well known for its strong and long existing vernacular settlements. The integrated district of Thanjavur consisted of Thanjavur, Nagapattinam, ….
8/12/2016В В· An Inventory of Vernacular Architecture taken in terms of legislation, pro-active planning policies, research, and raising of awareness through promotion of positive conservation models. vernacular building exhibition showcasing the vernacu- 8/12/2016В В· An Inventory of Vernacular Architecture taken in terms of legislation, pro-active planning policies, research, and raising of awareness through promotion of positive conservation models. vernacular building exhibition showcasing the vernacu-
terms of language, vernacular refers to a time, place or group [1]. In terms of architecture, it In terms of architecture, it refers to the style which is indigenous to a specific place or time. in terms of physical form and spatial configuration in the case study of vernacular housing. Keywords Urban Morphology, Spatial Configuration, Persian Vernacular Housing, Spatial Patterns,
1 Introduction: The Study of Vernacular Architecture Every weekend, thousands of people, mostly from the towns and suburbs of England, engage in the most innocent of pursuits – a visit to the countryside. Vernacular architecture is composed of local materials and derived from local customs and techniques that have been passed down from generation to generation. But vernacular architecture in most (if not all) African countries is disappearing, being abandoned for Western materials and techniques.
Vernacular term in architecture in the Philippines is used locally by Filipino who worked in site. It's very useful for professionals such as architects & engineers to … vernacular architecture and traditional settlements. The course is designed to provide the The course is designed to provide the students with knowledge about ethnic architecture, with …
In this study, vernacular architectural culture in the Eastern Black Sea Region is examined in terms of sustainable architectural principles. The goal of the paper is to investigate various ecological lessons terms of language, vernacular refers to a time, place or group [1]. In terms of architecture, it In terms of architecture, it refers to the style which is indigenous to a specific place or time.
Start studying ARCHITECTURE VERNACULAR TERMS. Learn vocabulary, terms, and more with flashcards, games, and other study tools. The Vernacular Architecture Of Rajasthan Both in India and abroad, several concepts dealing with architectural spaces havetheir roots in some abstract notions. Spaces oscillate between 'nothingness' and something universal.
Vernacular Architecture Architecture created from mostly local materials, by and for the use of local people. Vernacular architecture responds to local methods of building construction, local climates, and local living needs and traditions. Vernacular architecture is a category of architecture based on local needs and construction materials, and reflecting local traditions. The 'vernacular', in India, denotes low cost, traditional village and small town settlements, where construction is carried out without the help of architects and professionals, where building activity is regulated by a long tradition that stretches back
in terms of physical form and spatial configuration in the case study of vernacular housing. Keywords Urban Morphology, Spatial Configuration, Persian Vernacular Housing, Spatial Patterns, Vernacular architecture is a category of architecture based on local needs and construction materials, and reflecting local traditions. The 'vernacular', in India, denotes low cost, traditional village and small town settlements, where construction is carried out without the help of architects and professionals, where building activity is regulated by a long tradition that stretches back
The Vernacular Architecture Of Rajasthan Both in India and abroad, several concepts dealing with architectural spaces havetheir roots in some abstract notions. Spaces oscillate between 'nothingness' and something universal. SOME EXAMPLES OF VERNACULAR ARCHITEC- TURE IN HOT-ARID REGIONS OF TURKEY Fatma Nur DEMiRBtLEK The Scientific and Technical Research Council of Turkey BUILDING RESEARCH INSTITUTE, Ankara, TURKEY The study supports the of working on Vcr- nacular architecture and examines the of some or the vernacular dwellirv that were formed in hot-arid zones …
Start studying ARCHITECTURE VERNACULAR TERMS. Learn vocabulary, terms, and more with flashcards, games, and other study tools. Vernacular Architecture is now included in the Arts and Humanities Citation Index.
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The Naughtiest Girl: Naughtiest Girl Marches On : Book 10 Paperback / softback
by Anne Digby
Part of the The Naughtiest Girl series
In Enid Blyton's bestselling school series Elizabeth Allen is sent away to boarding school and makes up her mind to be the naughtiest pupil there's ever been. In book ten, Elizabeth is overjoyed to be appointed monitor again. But one of the second form boys is turning all the other boys against her, starting with a nasty note in her desk and ending with a false tip-off about a midnight pillow fight in the boys' dormitory.
Can she find out who has a grudge against her - and why?Between 1940 and 1952, Enid Blyton wrote four novels about Naughtiest Girl, Elizabeth Allen.
Books 5-10 are authorised sequels of the series written by Anne Digby in 1999.
Bonus material: A rare, complete serial story about a very special school. An interview with Enid Blyton about her school days. Enid Blyton's experiences as a teacher. A timeline of the author's life. Photos from Enid Blyton's younger days.
Publisher:Hachette Children's Group
Also by Anne Digby | View all
The Naughtiest Girl: Naughtiest Girl...
The Naughtiest Girl: Well Done, The...
Also in the The Naughtiest Girl series | View all


Establishes failure to provide certain specific protections from weather and during emergencies for domestic companion animals as animal cruelty.
An Act concerning exposure of domestic companion animals to potentially harmful weather and supplementing Title 4 of the Revised Statutes.
1. a. It shall be unlawful to leave outdoors unattended a cat, dog, or other domestic companion animal when the outside ambient temperature is 32 degrees Fahrenheit or less or 90 degrees Fahrenheit or more, unless the cat, dog, or other domestic companion animal is provided readily available access to shelter, in which the ambient temperature is properly and safely maintained above 32 degrees Fahrenheit and below 90 degrees Fahrenheit in accordance with any applicable local code, ordinance, or regulation. If the cat, dog, or other domestic companion animal is relocated, or provided readily available access, to a basement or garage, the basement or garage shall be properly ventilated in accordance with the "State Uniform Construction Code Act," P.L.1975, c.217 (C.52:27D-119 et seq.).
(2) secured in an indoor area constructed to be as protective of the animal as possible under the circumstances. If the cat, dog, or other domestic companion animal is secured in a basement or garage, the basement or garage shall be properly ventilated, in accordance with the "State Uniform Construction Code Act," P.L.1975, c.217 (C.52:27D-119 et seq.), and reasonably safe from flooding.
c. A violation of subsection a. or b. of this section shall constitute failure to provide necessary care pursuant to R.S.4:22-17 and R.S.4:22-26.
d. The requirements of subsections a. and b. of this section shall not apply to any cat living outside with no apparent owner, commonly referred to as, or considered to be, a feral cat.
e. As used in this section, "domestic companion animal" means any animal commonly referred to as a pet that was bought, bred, raised or otherwise acquired, in accordance with local ordinances and State and federal law, for the primary purpose of providing companionship to the owner, rather than for business or agricultural purposes. "Domestic companion animal" does not include livestock as defined in N.J.A.C.2:2-1.1.
2) leave a cat, dog, or other domestic companion animal outdoors when evacuation of the area has been recommended by State or local law enforcement due to weather or other emergency conditions.
2) secured in an indoor area constructed to be as protective of the animal as possible under the circumstances.
If, for the purposes of complying with the bill's requirements, the cat, dog, or other domestic companion animal is relocated to, or provided readily available access to, a basement or a garage, the bill requires the basement or garage to be properly ventilated in accordance with the State Uniform Construction Code, and during weather or other emergency conditions, also reasonably safe form flooding.
The bill defines "domestic companion animal" means any animal commonly referred to as a pet or one that has been bought, bred, raised or otherwise acquired, in accordance with local ordinances and State and federal law for the primary purpose of providing companionship to the owner, rather than for business or agricultural purposes. It does not include livestock.
Furthermore, the bill establishes that violations of any of the bill's provisions would constitute failure to provide "necessary care" pursuant to R.S.4:22-17 and R.S.4:22-26.
Current law defines "necessary care" as care sufficient to preserve the health and well-being of an animal, and includes, but is not limited to: food of sufficient quantity and quality to allow for normal growth or maintenance of body weight; adequate access to water in sufficient quantity and quality to satisfy the animal's needs; access to adequate protection from the weather; and veterinary care to alleviate suffering and maintain health.
Under R.S.4:22-26, a violator may be liable for civil penalties of $500 to $2,000 for failure to provide necessary care, depending on the consequences of failure to do so. Under R.S.4:22-17, failure to provide necessary care would be a disorderly persons offense, a crime of the fourth degree, or crime of the third degree, depending on the consequences of failure to do so.


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Q: Excel programmatically swap activex image I want to, in code, swap an image for a different one. Here's what I have now:
Private Sub CommandButton3_Click()
    Dim Pic As Picture
    Me.Shapes(5).Delete
    Set Pic = Me.Pictures.Insert("C:\Downloads\cards\2h.gif")
End Sub

It's working for the most part, except that it keeps deleting the wrong thing. How can I know and control what is in the Shapes array? In other words, when I add a new picture, how do I know it's index in the Shapes array?

A: FYI, that is not an ActiveX image.
You can name the picture after you add it:
Private Sub CommandButton3_Click()
    Dim Pic As Picture
    On Error Resume Next
    Me.Pictures("MyPic").Delete
    On Error Goto 0
    Me.Pictures.Insert("C:\Downloads\cards\2h.gif").Name = "MyPic"
End Sub

redpajama_set_name: RedPajamaStackExchange


text:
Examples include olaparib, which has been approved by the FDA for Personal computer with BRCA mutations; rucaparib, which has been authorized for androgen-receptor-guided therapy and paclitaxel-based chemotherapy for individuals with mCRPC and harmful mutations (germline and somatic); and two additional PARP inhibitors, veliparib and talazoparib, which are under investigation in clinical tests
PKA November 20, 2022 nostradamus2018
Examples include olaparib, which has been approved by the FDA for Personal computer with BRCA mutations; rucaparib, which has been authorized for androgen-receptor-guided therapy and paclitaxel-based chemotherapy for individuals with mCRPC and harmful mutations (germline and somatic); and two additional PARP inhibitors, veliparib and talazoparib, which are under investigation in clinical tests. benefit from treatment. It is necessary to study the combination of PARPis and additional therapeutic agents such as anti-hormone medicines, USP7 inhibitors, BET inhibitors, and immunotherapy. This short article reviews the mechanism of PARP inhibition in the treatment of PC, the progress of clinical study, the mechanisms of drug resistance, and the strategies of combination treatments. [4,5,6,7]. Genomic DNA is definitely continually confronted with a large number of DNA lesions, which are generated by intrinsic (e.g., reactive oxygen varieties) and extrinsic sources (e.g., by ionizing radiation, ultraviolet radiation). In addition to DNA damage-induced lesions, the cells have to deal with spontaneous lesions, such as AP (apurinic/apyrimidinic) sites or the deamination of bases [8]. If remaining unrepaired or repaired incorrectly, DNA damage may lead to cell death, genomic instability, and mutagenesis. To keep genome stable and secure cellular homeostasis, it is essential for the cells to counteract DNA damage by activating the DNA damage response (DDR), which finally coordinates cell fate decision making [9]. The activation of DNA restoration (orchestrated by set of DNA damage response proteins, including BRCA1, NBS1, as well as restoration proteins Ku70/80 as well as others) and cell-cycle checkpoints (regulated by MDC1, 53BP1 and checkpoint kinases Chk1/Chk2) act as an immediate response to DNA damage to provide safety and recovery of hurt cells, whereas activation of cell death happens much later on and seeks to remove the irreversibly damaged cell. Depending on the type and the severe nature of stimulus and mobile context, DNA harm can stimulate cell-cycle arrest, senescence or different cell loss of life programs, such as for example mitotic catastrophe, apoptosis, necrosis and autophagy [10,11]. Activation of p53 was reported to become essential for the initiation and development of senescence and apoptosis pursuing DNA harm generally in most cell types [10]. Activated p53 regulates some its apoptotic focus on genes additional, such as for example cyclin reliant kinase inhibitor 1A (p21), p53-upregulated modulator of apoptosis (PUMA), p53AIP1, BCL2 linked X (BAX), aswell as many types of miRNAs including miR-34a, resulting in cell apoptosis [10]. A great many other types of substances and regulators including Caspase-2, Bcl-2, Nur77, TSC2/mTORC1 signaling JNK and pathway signaling pathways get excited about the legislation of cell loss of life pursuing DNA harm [10,12]. DNA double-strand breaks (DSBs) will be the most cytotoxic DNA lesions, which might trigger disruption of chromatin framework, including chromosomal deletions, insertions, duplications, and translocations, which additional cause cell loss of life [13]. Some DNA harm repair pathways provides advanced in cells to correct various kinds of harm, including homologous recombination fix (HRR), non-homologous end-joining (NHEJ), bottom excision fix (BER), nucleotide excision fix (NER), and mismatch fix (MMR) [14]. HRR and NHEJ are in charge of DNA double-stranded breaks (DSBs) that are due to ionizing rays, DNA replication tension or chemotherapeutic agencies. BER is principally in charge of the fix of DNA single-stranded breaks (SSBs), that Z-FL-COCHO are due to alkylating reactive or agent oxygen species. NER plays an integral role of mending SSBs that are due to ultraviolet light or chemotherapeutic medications such as for example cisplatin. MMR corrects the DNA dual helix mismatch of bottom pairs. Genomic instability due to flaws in the DDR can be an essential basis of cancers development and initiation [15], therefore, concentrating on the DDR pathway is certainly an extremely feasible technique for cancers treatment. Small-molecule inhibitors centered on the DDR pathway can focus on abnormally expressed protein in cancers cells and also have yielded appealing therapeutic effects. Many small-molecule inhibitors concentrating on DNA harm check factors (ATM inhibitors [16,17], ATR inhibitors [18], CHEK1/2 inhibitors [19,20], and poly (adenosine diphosphate-ribose) polymerase inhibitors [21] (PARPis) or DNA fix pathway protein (RAD51 inhibitors [22,23], and FEN1 inhibitors [24]) have already been designed and produced under preclinical exams or clinical studies [25]. PARPis possess garnered worldwide interest for their exceptional curative effect. Many PARPis have already been employed for treatment of many types of malignancies medically, including breasts ovarian and cancer cancer. In 2005, two groupings described the artificial lethality (SL) relationship between ITGA3 PARP inhibition and or mutation, recommending a novel technique for dealing with sufferers with mutations, this proclaimed the first scientific acceptance from the feasibility of PARP1 as an anti-tumor focus on as well as the SL theory. Another PARPi, rucaparib, received accelerated acceptance in america in Dec 2016 for advanced ovarian cancers with gene mutations and several chemotherapy remedies. In March 2017, niraparib was accepted by the FDA for maintenance therapy in sufferers with repeated epithelial ovarian, fallopian pipe, and principal peritoneal cancers. Based on the successful.These mechanisms are summarized in Figure 2. Open in a separate window Figure 2 Mechanism of resistance. In ovarian or breast cancer, olaparib resistance is associated with HRR recovery, including the reversal of mutations. PARPis have shown great efficacy, their widespread use is restricted by various factors, including drug resistance and the limited population who benefit from treatment. It is necessary to study the combination of PARPis and other therapeutic agents such as anti-hormone drugs, USP7 inhibitors, BET inhibitors, and immunotherapy. This article reviews the mechanism of PARP inhibition in the treatment of PC, the progress of clinical Z-FL-COCHO research, the mechanisms of drug resistance, and the strategies of combination treatments. [4,5,6,7]. Genomic DNA is continuously confronted with a large number of DNA lesions, which are generated by intrinsic (e.g., reactive oxygen species) and extrinsic sources (e.g., by ionizing radiation, ultraviolet radiation). In addition to DNA damage-induced lesions, the cells have to deal with spontaneous lesions, such as AP (apurinic/apyrimidinic) sites or the deamination of bases [8]. If left unrepaired or repaired incorrectly, DNA damage may lead to cell death, genomic instability, and mutagenesis. To keep genome stable and secure cellular homeostasis, it is essential for the cells to counteract DNA damage by activating the DNA damage response (DDR), which finally coordinates cell fate decision making [9]. The activation of DNA repair (orchestrated by set of DNA damage response proteins, including BRCA1, NBS1, as well as repair proteins Ku70/80 and others) and cell-cycle checkpoints (regulated by MDC1, 53BP1 and checkpoint kinases Chk1/Chk2) act as an immediate response to DNA damage to provide protection and recovery of injured cells, whereas activation of cell death occurs much later and aims to eliminate the irreversibly damaged cell. Depending on the type and the severity of stimulus and cellular context, DNA damage can induce cell-cycle arrest, senescence or different cell death programs, such as mitotic catastrophe, apoptosis, autophagy and necrosis [10,11]. Activation of p53 was reported to be crucial for the initiation and progression of senescence and apoptosis following DNA damage in most cell types [10]. Activated p53 further regulates a series of its apoptotic target genes, such as cyclin dependent kinase inhibitor 1A (p21), p53-upregulated modulator of apoptosis (PUMA), p53AIP1, BCL2 associated X (BAX), as well as several kinds of miRNAs including miR-34a, leading to cell apoptosis [10]. Many other kinds of regulators and molecules including Caspase-2, Bcl-2, Nur77, TSC2/mTORC1 signaling pathway and JNK signaling pathways are involved in the regulation of cell death following DNA damage [10,12]. DNA double-strand breaks (DSBs) are the most cytotoxic DNA lesions, which may cause disruption of chromatin structure, including chromosomal deletions, insertions, duplications, and translocations, which further cause cell death [13]. A series of DNA damage repair pathways has evolved in cells to repair different types of damage, including homologous recombination repair (HRR), nonhomologous end-joining (NHEJ), base excision repair (BER), nucleotide excision repair (NER), and mismatch repair (MMR) [14]. HRR and NHEJ are responsible for DNA double-stranded breaks (DSBs) which are caused by ionizing radiation, DNA replication stress or chemotherapeutic agents. BER is mainly responsible for the repair of DNA single-stranded breaks (SSBs), which are caused by alkylating agent or reactive oxygen species. NER plays a key role of repairing SSBs that are caused by ultraviolet light or chemotherapeutic drugs such as cisplatin. MMR corrects the DNA double helix mismatch of base pairs. Genomic instability caused by defects in the DDR is an important basis of cancer initiation and progression [15], therefore, targeting the DDR pathway is a very feasible strategy for cancer treatment. Small-molecule inhibitors focused on the DDR pathway can target abnormally expressed proteins in cancer cells and have yielded promising therapeutic effects. Several small-molecule inhibitors targeting DNA damage check points (ATM inhibitors [16,17], ATR inhibitors [18], CHEK1/2 inhibitors [19,20], and poly (adenosine diphosphate-ribose) polymerase inhibitors [21] (PARPis) or DNA repair pathway proteins (RAD51 inhibitors [22,23], and FEN1 inhibitors [24]) have been designed and generated under preclinical tests or clinical tests [25]. PARPis have garnered worldwide attention for their superb curative effect. Several PARPis have been clinically utilized for treatment of several kinds of cancers, including breast tumor and ovarian malignancy. In 2005, two organizations described the synthetic lethality (SL) connection between PARP inhibition and or mutation, suggesting.Clinical Development of PARPi Resistance in PC Although PARPi can cause a response in patients with mutations, some patients may develop resistance [71]. of Personal computer, the progress of clinical study, the mechanisms of drug resistance, and the strategies of combination treatments. [4,5,6,7]. Genomic DNA is definitely continuously confronted with a large number of DNA lesions, which are generated by intrinsic (e.g., reactive oxygen varieties) and extrinsic sources (e.g., by ionizing radiation, ultraviolet radiation). In addition to DNA damage-induced lesions, the cells have to deal with spontaneous lesions, such as AP (apurinic/apyrimidinic) sites or the deamination of bases [8]. If remaining unrepaired or repaired incorrectly, DNA damage may lead to cell death, genomic instability, and mutagenesis. To keep genome stable and secure cellular homeostasis, it is essential for the cells to counteract DNA damage by activating the DNA damage response (DDR), which finally coordinates cell fate decision making [9]. The activation of DNA restoration (orchestrated by set of DNA damage response proteins, including BRCA1, NBS1, as well as restoration proteins Ku70/80 while others) and cell-cycle checkpoints (regulated by MDC1, 53BP1 and checkpoint kinases Chk1/Chk2) act as an immediate response to DNA damage to provide safety and recovery of hurt cells, whereas activation of cell death occurs much later on and aims to remove the irreversibly damaged cell. Depending on the type and the severity of stimulus and cellular context, DNA damage can induce cell-cycle arrest, senescence or different cell death programs, such as mitotic catastrophe, apoptosis, autophagy and necrosis [10,11]. Activation of p53 was reported to be important for the initiation and progression of senescence and apoptosis following DNA damage in most cell types [10]. Activated p53 further regulates a series of its apoptotic target genes, such as cyclin dependent kinase inhibitor 1A (p21), p53-upregulated modulator of apoptosis (PUMA), p53AIP1, BCL2 connected X (BAX), as well as several kinds of miRNAs including miR-34a, leading to cell apoptosis [10]. Many other kinds of regulators and molecules including Caspase-2, Bcl-2, Nur77, TSC2/mTORC1 signaling pathway and JNK signaling pathways are involved in the rules of cell death following DNA damage [10,12]. DNA double-strand breaks (DSBs) are the most cytotoxic DNA lesions, which may cause disruption of chromatin structure, including chromosomal deletions, insertions, duplications, and translocations, which further cause cell death [13]. A series of DNA damage repair pathways offers developed in cells to repair different types of damage, including homologous recombination restoration (HRR), nonhomologous end-joining (NHEJ), foundation excision restoration (BER), nucleotide excision restoration (NER), and mismatch restoration (MMR) [14]. HRR and NHEJ are responsible for DNA double-stranded breaks (DSBs) which are caused by ionizing radiation, DNA replication stress or chemotherapeutic brokers. BER is mainly responsible for the repair of DNA single-stranded breaks (SSBs), which are caused by alkylating agent or reactive oxygen species. NER plays a key role of fixing SSBs that are caused by ultraviolet light or chemotherapeutic drugs such as cisplatin. MMR corrects the DNA double helix mismatch of base pairs. Genomic instability caused by defects in the DDR is an important basis of malignancy initiation and progression [15], therefore, targeting the DDR pathway is usually a very feasible strategy for malignancy treatment. Small-molecule inhibitors focused on the DDR pathway can target abnormally expressed proteins in malignancy cells and have yielded encouraging therapeutic effects. Several small-molecule inhibitors targeting DNA damage check points (ATM inhibitors [16,17], ATR inhibitors [18], CHEK1/2 inhibitors [19,20], and poly (adenosine diphosphate-ribose) polymerase inhibitors [21] (PARPis) or DNA repair pathway proteins (RAD51 inhibitors [22,23], and FEN1 inhibitors [24]) have been designed and generated under preclinical assessments or clinical trials.The results showed that this pharmacological inhibition of USP7 could lead to downregulation of the CCDC6 protein and DNA repair of gene defects, making CRPC cells sensitive to PARPis, either alone or in combination with standard radiotherapy. Clarke et al. research, the mechanisms of drug resistance, and the strategies of combination treatments. [4,5,6,7]. Genomic DNA is usually continuously confronted with a large number of DNA lesions, which are generated by intrinsic (e.g., reactive oxygen species) and extrinsic sources (e.g., by ionizing radiation, ultraviolet radiation). In addition to DNA damage-induced lesions, the cells have to deal with spontaneous lesions, such as AP (apurinic/apyrimidinic) sites or the deamination of bases [8]. If left unrepaired or repaired incorrectly, DNA damage may lead to cell death, genomic instability, and mutagenesis. To keep genome stable and secure cellular homeostasis, it is essential for the cells to counteract DNA damage by activating the DNA damage response (DDR), which finally coordinates cell fate decision making [9]. The activation of DNA repair (orchestrated by set of DNA damage response proteins, including BRCA1, NBS1, as well as repair proteins Ku70/80 as well as others) and cell-cycle checkpoints (regulated by MDC1, 53BP1 and checkpoint kinases Chk1/Chk2) act as an immediate response to DNA damage to provide protection and recovery of hurt cells, whereas activation of cell death occurs much later and aims to eliminate the irreversibly damaged cell. Depending on the type and the severity of stimulus and cellular context, DNA damage can induce cell-cycle arrest, senescence or different cell death programs, such as mitotic catastrophe, apoptosis, autophagy and necrosis [10,11]. Activation of p53 was reported to be crucial for the initiation and progression of senescence and apoptosis following DNA damage in most cell types [10]. Activated p53 further regulates a series of its apoptotic target genes, such as cyclin dependent kinase inhibitor 1A (p21), p53-upregulated modulator of apoptosis (PUMA), p53AIP1, BCL2 associated X (BAX), as well as several kinds of miRNAs including miR-34a, leading to cell apoptosis [10]. Many other kinds of regulators and molecules including Caspase-2, Bcl-2, Nur77, TSC2/mTORC1 signaling pathway and JNK signaling pathways are involved in the regulation of cell death following DNA damage [10,12]. DNA double-strand breaks (DSBs) are the most cytotoxic DNA lesions, which may cause disruption of chromatin structure, including chromosomal deletions, insertions, duplications, and translocations, which further cause cell death [13]. A series of DNA damage repair pathways has developed in cells to repair different types of damage, including homologous recombination repair (HRR), nonhomologous end-joining (NHEJ), base excision repair (BER), nucleotide excision repair (NER), and mismatch repair (MMR) [14]. HRR and NHEJ are responsible for DNA double-stranded breaks (DSBs) Z-FL-COCHO which are caused by ionizing radiation, DNA replication stress or chemotherapeutic brokers. BER is mainly responsible for the repair of DNA single-stranded breaks (SSBs), which are caused by alkylating agent or reactive oxygen species. NER plays a key role of fixing SSBs that are caused by ultraviolet light or chemotherapeutic drugs such as cisplatin. MMR corrects the DNA double helix mismatch of base pairs. Genomic instability caused by defects in the DDR can be an essential basis of tumor initiation and development [15], therefore, concentrating on the DDR pathway is certainly an extremely feasible technique for tumor treatment. Small-molecule inhibitors centered on the DDR pathway can focus on abnormally expressed protein in tumor cells and also have yielded guaranteeing therapeutic effects. Many small-molecule inhibitors concentrating on DNA harm check factors (ATM inhibitors [16,17], ATR inhibitors [18], CHEK1/2 inhibitors [19,20], and poly (adenosine diphosphate-ribose) polymerase inhibitors [21] (PARPis) or DNA fix pathway protein (RAD51 inhibitors [22,23], and FEN1 inhibitors [24]) have already been designed and produced under preclinical exams or clinical studies [25]. PARPis possess garnered worldwide interest for their exceptional curative effect. Many PARPis have already been clinically useful for treatment of many kinds of malignancies, including breast cancers and ovarian tumor. In 2005, two groupings described the artificial lethality (SL) relationship between PARP inhibition and or mutation, recommending a novel technique for dealing with sufferers with mutations, this proclaimed the first scientific acceptance from the feasibility of PARP1 as an anti-tumor focus on as well as the SL theory. Another PARPi, rucaparib,.This study investigated if the pharmacological inhibition of USP7 could impair the AR-dependent proliferation of PC cells by damaging the stability of AR. lesions, that are generated by intrinsic (e.g., reactive air types) and extrinsic resources (e.g., by ionizing rays, ultraviolet rays). Furthermore to DNA damage-induced lesions, the cells suffer from spontaneous lesions, such as for example AP (apurinic/apyrimidinic) sites or the deamination of bases [8]. If still left unrepaired or fixed incorrectly, DNA harm can Z-FL-COCHO lead to cell loss of life, genomic instability, and mutagenesis. To maintain genome steady and secure mobile homeostasis, it is vital for the cells to counteract DNA harm by activating the DNA harm response (DDR), which finally coordinates cell destiny decision producing [9]. The activation of DNA fix (orchestrated by group of DNA harm response proteins, including BRCA1, NBS1, aswell as fix proteins Ku70/80 yet others) and cell-cycle checkpoints (controlled by MDC1, 53BP1 and checkpoint kinases Chk1/Chk2) become an instantaneous response to DNA harm to offer security and recovery of wounded cells, whereas activation of cell loss of life occurs much afterwards and aims to get rid of the irreversibly broken cell. With regards to the type and the severe nature of stimulus and mobile context, DNA harm can stimulate cell-cycle arrest, senescence or different cell loss of life programs, such as for example mitotic catastrophe, apoptosis, autophagy and necrosis [10,11]. Activation of p53 was reported to become essential for the initiation and development of senescence and apoptosis pursuing DNA harm generally in most cell types [10]. Activated p53 additional regulates some its apoptotic focus on genes, such as for example cyclin reliant kinase inhibitor 1A (p21), p53-upregulated modulator of apoptosis (PUMA), p53AIP1, BCL2 linked X (BAX), aswell as many types of miRNAs including miR-34a, resulting in cell apoptosis [10]. A great many other types of regulators and substances including Caspase-2, Bcl-2, Nur77, TSC2/mTORC1 signaling pathway and JNK signaling pathways get excited about the legislation of cell loss of life following DNA harm [10,12]. DNA double-strand breaks (DSBs) will be the most cytotoxic DNA lesions, which might trigger disruption of chromatin framework, including chromosomal deletions, insertions, duplications, and translocations, which further cause cell death [13]. A series of DNA damage repair pathways has evolved in cells to repair different types of damage, including homologous recombination repair (HRR), nonhomologous end-joining (NHEJ), base excision repair (BER), nucleotide excision repair (NER), and mismatch repair (MMR) [14]. HRR and NHEJ are responsible for DNA double-stranded breaks (DSBs) which are caused by ionizing radiation, DNA replication stress or chemotherapeutic agents. BER is mainly responsible for the repair of DNA single-stranded breaks (SSBs), which are caused by alkylating agent or reactive oxygen species. NER plays a key role of repairing SSBs that are caused by ultraviolet light or chemotherapeutic drugs such as cisplatin. MMR corrects the DNA double helix mismatch of base pairs. Genomic instability caused by defects in the DDR is an important basis of cancer initiation and progression [15], therefore, targeting the DDR pathway is a very feasible strategy for cancer treatment. Small-molecule inhibitors focused on the DDR pathway can target abnormally expressed proteins in cancer cells and have yielded promising therapeutic effects. Several small-molecule inhibitors targeting DNA damage check points (ATM inhibitors [16,17], ATR inhibitors [18], CHEK1/2 inhibitors [19,20], and poly (adenosine diphosphate-ribose) polymerase inhibitors [21] (PARPis) or DNA repair pathway proteins (RAD51 inhibitors [22,23], and FEN1 inhibitors [24]) have been designed and generated under preclinical tests or clinical trials [25]. PARPis have garnered worldwide attention for their excellent curative effect. Several PARPis have been clinically used for treatment of several kinds of cancers, including breast cancer and ovarian cancer. In 2005, two groups described the synthetic lethality (SL) interaction between PARP.
This work was supported with the Japan Society for the Promotion of Science (Kakenhi grant no
A
Hair around the dorsal surface of mice was removed by a hair-removing cream (Nair, Church and Dwight Company, Princeton, NJ) with a moisturized cotton stick
Next Unlabelled the crystals was ready at a 20?mM share ahead of make use of in a remedy filled with 20 simply?mM dibasic potassium phosphate and 20?mM potassium hydroxide
Previous In addition, these results fit well with the observation that and resting hair follicles contain a similar number of resident stem cells that deficiently respond to a proliferative stimulus in haploinsufficiency conditions


Lucasfilm just released a trailer for the next set of Force of Destiny episodes airing in October.
Disney Channel will two half-hour "Star Wars Forces of Destiny" television specials in October. The first half-hour special debuts SUNDAY, OCTOBER 1 (8:00 p.m. ET/PT) followed by the second on SUNDAY, OCTOBER 29 (8:00 p.m. ET/PT) on Disney Channel and the Disney Channel App.
In addition, these new shorts will also launch on Disney's YouTube channel following the airing of the specials on Disney Channel.
These specials will feature a total of eight never-before-seen installments, which showcase compilations of the popular animated shorts featuring Rey, Jyn Erso, Sabine Wren, Princess Leia, Ahsoka Tano and Padmé Amidala, as well as new episodes including Han Solo, Hera Syndulla, Finn, Maz Kanata and many others.
In addition, Han Solo makes multiple appearances in these episodes. Wonder if this character is voiced by Harrison Ford himself or a sound alike?
Han with Leia on Endor and Rey, Finn, Chewbacca and BB-8 on the Millennium Falcon!
I am so looking forward to these upcoming Forces of Destiny episodes. Are you?


Samsung Galaxy series can be the flagship for Android smartphone. With the perfect outlook and high performance, it has been extremely popular among Android users. In this article, we will focus on how to root Samsung Galaxy A8 SM A8000 in 3 different ways.
iRoot claims to root nearly all models of Android phone and tablet in only one click, no damage caused on them. It includes 2 versions of rooting tool: iRoot for Android and iRoot for PC. You can choose the corresponding one according to your requirement.
If you choose the PC version, remember to enable USB debugging mode on your Samsung Galaxy A8 SM A8000 at first. And then connect your phone to the PC. Run iRoot. Click the "Root" button on the interface. Just leave your device to be rooted now.
On the other hand, if you choose iRoot for Android version, check "Unknown sources" on your phone. This is to ensure the installation of iRoot app on Samsung A8000. Open iRoot on your phone, just tab on "Get Root access" to start the rooting process.
One Click Root can be effective to root your Samsung A8000 with computer. As I know, it's free to use long time ago. But now you need to donate some to use it. Anyway, it does a great job on rooting devices on Android 2.2 to 4.4.
First, check the USB Debugging mode on your phone. Enable it and charge your phone into 85% battery life. Download One Click Root on your computer and prepare to root Samsung Galaxy A8 SM A8000.
Just check the usage: install and open Framaroot -> select one: Install SuperSU, Install Superuser, or Unroot -> Select the two former options and begin rooting process.
Whichever you choose among these rooting ways above, you can successfully root your Samsung A8 SM A8000 with and without computer. If you need toroot other model of Samsung Galaxy, click here.


WATCH: Jesse the Dog Performs House Chores!
Jesse the Jack Russell Terrier is not your average pup! He is proof that some dogs LIVE to work. Throughout the day, Jesse finds pleasure in taking out the trash, dusting the house, fetching household items for his master – and even polishing shoes. His talent for being helpful has landed him a number of staring roles in commercials, television and film.
Enjoy Jesse's You Tube blockbuster video "Useful Dog Tricks 3" for ideas on how you can train your canine for greatness!


Welcome to ENERGY AND FUELS web-site — your comprehensive Energy and fuels resource. Our mission is to give you the facts you need about Energy, tips on energy savings, fuels — fast — so you can get on the road to taking action right away`.
Our website provides a ton of information about anything concerning renewable energy, energy savings and fuels. In addition, you will find extensive information to help you on what you're searching.
Please have a look at our energy and fuels articles, products, resources, and additional information located throughout migrans.net/ENERGY.
We strive to provide only quality articles, so if there is a specific topic related to energy and fuels that you would like us to cover, please contact us at any time.
And again, thank you to those contributing daily to our ENERGY website.
We want to provide homeowners with tips for saving energy and money at home and on the road. By following just a few of the simple tips found on our Web site, you can make your home more comfortable and easier to heat and cool—while you save money. We bring you the latest information on energy-saving, efficient technologies. We even give tips for using clean, renewable energy to power your home.
We help you beat the high cost of fuel, with driving and car maintenance tips to save you money on the road.
We encourage you to check out the Web site and make improvements today and this year that will contribute to your energy bottom line and make our planet healthier and cleaner!


text:
NORTH ROYALTON,OHIO
Son arrested for OVI after mother's car used in hit skip accident: North Royalton Police Blotter
Published: Aug. 26, 2013, 11:00 a.m.
John Benson, special to cleveland.com
OVI, RIDGE ROAD: At 7:30 p.m. Aug. 16 police were notified of a hit skip accident that happened 10 minutes prior in parking lot for Natural Pet on Ridge Road. Police went looking for the 83-year-old female owner of the car, who lived on Lynn Drive. Upon arriving at her residence, police knocked on the door and were met by a highly intoxicated 52-year-old male, who was the son of suspected driver. He said they were at Firehouse Bar and his mother struck a truck but it was no big deal. The mother was sleeping but son opened garage to show off car, which had scratches and dents over entire vehicle. The son appeared nervous and asked why the officer was asking so many questions. Eventually, he admitted he was driving his mother's car and that his license was suspended. That's why he left scene. The mother woke up and said she hadn't been driving for months due to medical condition. Also, she said she was not with him at bar. The son failed a sobriety test and was arrested. He was cited for OVI and hit skip.
CAR DAMAGE, ALBION ROAD: Around 8 a.m. Aug. 14 police were called to an Albion Road house after its owner reported three cars received brick damage during the night. A Chevy pickup had its window scratched, a Chrysler minivan had its window scratched and a PT Cruiser had a window scratched and door damaged. The owners had no idea who did this.
BURGLARY, BENNETT ROAD: A Bennett Road resident arrived home from work on the evening of Aug. 17 to find all three of his main doors open. Upon inspecting his home, he discovered cupboard doors were open in the kitchen and a desk drawer was open but nothing seemed to be missing. Two days later he noticed his laptop was gone and he reported it at the police department. There are no leads.
UNDERAGE POSSESSION AND CONSUMPTION, CASTLE DRIVE: A police officer at 11:32 p.m. Aug. 16 noticed a silver Mazda swerving on State Road. The car was pulled over on Castle Drive. It belonged to a 19-year-old Madison Lane resident who appeared drunk and said he was out looking for baby formula. At first he stressed he hadn't had anything to drink but during a sobriety test admitted to having "one beer." He was arrested for underage consumption. During a vehicle search, several pieces of a green leafy vegetable substance were discovered inside of a magazine, as well as two marijuana joints. He blew a .012 BAC. He was charged with underage possession and consumption, drug paraphernalia, marijuana possession, driving in weaving course and license plate.
OVI, STATE ROAD: At 9:29 p.m. Aug. 16 a Chevy Cobalt was pulled over for speeding and weaving on State Road. The dazed driver said he was coming from the Sip N Post and driving to his Parma home. He admitted to having five or six beers. "I'm not going to lie, I had a few," he said. During the sobriety test he fell twice against his car. He was arrested for OVI. He blew a .218 BAC. He was cited for OVI, BAC, Speed, weaving course and seat belt.
TRESPASSING, DISORDERLY CONDUCT, ROYALTON ROAD: At 10:44 p.m. Aug. 19 police were dispatched to Park Tavern for a report of a disorderly patron. The owner told the customer he wasn't going to be served any more alcohol. The customer got verbal and said he wouldn't leave. The police escorted the male to the parking lot where he got "lippy" and threw his hands in the air. Other patrons in the bar barked to get him out of there. The 31-year-old North Royalton resident smelled of alcohol. His girlfriend tried to reason with him. The police offered to take him home. Instead, he said he wanted to walk. The police said he was too intoxicated to walk. After arguing, the man was taken into custody. He was charged with criminal trespass and disorderly conduct.
THEFT, TURNPIKE CONSTRUCTION SITE: Police were dispatched to a Turnpike Bridge construction site near Akins Road and Bennett Road at 2:17 p.m. Aug. 15 when a superintendent noticed one of his aluminum platforms was missing. The 32-foot platform was there when worked ended on Aug. 14. The missing item is worth $1,000. There are no leads.
DRUG PARAPHERNALIA, MARIJUANA POSSESSION, MEMORIAL PARK: At 5:49 p.m. Aug. 19 police were dispatched to Memorial Park after receiving a call about five juveniles in a green Ford smoking pot. Officers arrived to find two males. While they didn't appear to be smoking, the police asked if they had been and the 26-year-old driver said there was a glass marijuana pipe in the center console. Upon a search, the Cleveland-resident driver had a grinder containing marijuana. The males were working for an upcoming carnival. The driver was cited for possession of drug paraphernalia and drug abuse.
OVI, WOODCROFT TRACE: At 2:50 a.m. Aug. 19 police noticed a Dodge Neon turning out of Taco Bell. The car had severe damage to its back bumper and the license plate light wasn't working. The car was also drifting between lanes. The driver said he was coming from a buddy's house and headed home to Brookpark. When asked whether he had anything to drink, the driver said he hadn't been drinking. "I passed all of your tests," he said. After being told he didn't indeed pass, the driver admitted to having a couple of beers. He blew a .135 BAC. He later admitted to having five or six beers. Police said he was very cooperative. The driver was cited with OVI, BAC, left of center and license plate.


text:
Georgetown law means the Supreme Court can overturn the Heller decision, but will it?
A Georgetown law professor is calling for a "major constitutional overhaul" of the nation's gun laws.
In an op-ed in The Washington Post on Monday, professor Richard Levin, who is the George Washington University Law School's director of constitutional studies, said he was "stunned" by Heller, which upheld the Second Amendment and established the right to bear arms.
"The Heller decision is the most significant and consequential constitutional amendment to the United States since the Second Amendments were ratified in 1791," Levin wrote.
"It's a seminal decision that has changed the way we govern the United Nations, the constitution of the United Kingdom, and our legal systems.
We need to redo it."
In an interview with The Washington Examiner last week, Levin said he would have voted for the Heller ruling if he had the opportunity to do so.
But Levin is a constitutional scholar, not a lawyer, so he says he can't comment on how a Heller ruling could be overturned.
And if the Supreme Council of Justice, which advises the court, is willing to overturn the decision, he said he could see a court overturning the decision.
If the Supreme Supreme Court overturns Heller, it will open up the door for future Heller cases, Levin wrote, and "the Heller decision itself could be overruled, as it did in 2013 in the landmark Citizens United case."
In a letter to the Supreme Judicial Court on Monday evening, the law professors who authored the opinion in Heller said that if the court overturned Heller, "there would be no reason for the Court to take any more steps to regulate gun ownership."
"It would be a constitutional tragedy," they wrote.
Law professors who wrote the Heller opinion in the case, however, have said that there are still a number of ways the court could overturn the case.
The law professors wrote that the court would need to first decide whether Heller is a Second Amendment right.
If so, the court must then decide whether it is a "liberty interest," a word that is used by the court to define a right.
They added that if Heller is not a Second the right, the Court cannot consider the case for review.
"As the Court is already well aware, the Heller case was not a case about whether the Second amendment applied to handgun possession, or about the Second right to keep and bear arms," the law faculty wrote.
"It was about whether Congress had the power to ban handguns as well as to ban firearms."
Levin said that, as a matter of fact, he thinks Heller would be ruled a Second amendment right if it were decided in the Supreme court.
"If the Court decides that the Second is a right, I think it would be right, because it would go to the heart of what the Constitution says, and that is, Congress cannot constitutionally ban firearms," he said.
The Heller case, he continued, is a case "about the limits of Congress' power over interstate commerce."
"This case is about the limits on Congress' authority over the interstate commerce of arms," he wrote.
Levin argued that the Court could overturn Heller if it decided that Congress has the power over the entire Second Amendment.
"But that would make it impossible for the Supreme Courts of the States to take away the Second," he continued.
"I don't see that happening.
It is a Constitutional issue."
Lester's opinion also noted that if Congress did not have the power, the Supreme courts could overturn other Supreme Court decisions that have upheld the right of states to regulate guns.
"A majority of the Court has expressed its concern that the Heller court could reverse a number, but not all, of the decisions of the lower courts on this issue," he concluded.
"We are in a position in which the Court would need the consent of the other three Justices, including Chief Justice Roberts, to make a ruling on Heller."
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Tagged newtons first law, nj gun laws
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text:
Q: WillSet/DidSet not being invoked on realm property I have a problem because willSet and didSet are not being called with dynamic realm object.
Code sample:
try! realm.write {
    sut = Backup()
    realm.add(sut) // here willSet and didSet are invoked with nil object
}

XCTAssertFalse(sut.didEditPatient) // ok
try! realm.write {
    print("CHECKING: will add the patient")
    let patient = Patient()
    realm.add(patient)
    sut.patient = patient // nothing gets printed here!
    print("CHECKING: added the patient")
}

XCTAssertTrue(sut.didEditPatient) // fails
XCTAssertNotNil(sut.patient) // ok

Where Backup class is defined this way:
final class Backup: Object {
    @objc dynamic var patient: Patient? {
        willSet {
            print("CHECKING: willSet: \(String(describing: newValue))")
            if newValue != patient {
                didEditPatient = true
            }
        }
        didSet { print("CHECKING: didSet: \(String(describing: patient))") }
    }
    @objc dynamic var didEditPatient: Bool = false

Output in the console is:

*

*CHECKING: willSet: nil

*CHECKING: didSet: nil

*CHECKING: will add the patient

*CHECKING: added the patient

While I'd rather expect that between will add the patient and added the patient I should get willSet and didSet with patient object. Obviously, patient is not nil.

A: There is one issue described about this in realm repo:

I would recommend using a private persisted property which has no logic, along with a non-persisted computed property which has the willSet/didSet functionality:
class Model : RLMObject {
    private dynamic var backingProp = 0

    var prop : Int {
        get {
            return backingProp
        }
        set(newValue) {
            // do willSet stuff
            backingProp = newValue
            // do didSet stuff
        }
    }

    override class func ignoredProperties() -> [AnyObject]! {
        return ["prop"]
    }
}

This is a bit verbose, but gives you identical behavior for objects in a realm and standalone objects.

Source: https://github.com/realm/realm-cocoa/issues/870#issuecomment-54543539

A: This is easy to fix. Just update the objects properties before writing to Realm. willSet and didSet are Swift constructs, not Objc.
let p = Patient()
try! realm.write {
    print("CHECKING: will add the patient")
    sut.patient = p // moved before add to realm.
    realm.add(p)
    print("CHECKING: added the patient")
}

Will print
CHECKING: will add the patient
willSet
didSet
CHECKING: added the patient

Which is the order you want.