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Ethnicity: Spanish
13 matching books
Any Child 1
Cross Group 6
Incidental 1
Oppression & Resilience 3
Angolan 1
Australian 1
Bolivian 1
Brazilian 1
Cuban 2
Filipino 1
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ALA Pura Belpré Award 1
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Sydney Taylor Book Award Winner 1
Around the World 1
Central America 3
Latin America and the Caribbean 3
Northern Europe 1
Southern Europe 2
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Bi/multilingual 5
Economic Struggle 2
Nahua 3
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Yanomami 1
Direct 6
Negative 1
Non-Central 4
Resolving 5
Bi/Multiracial/Mixed Race 3
Brown-Skinned and/or Race Unspecified 2
Latinx/Hispanic/Latin American 5
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White/European American/Caucasian 7
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Christian 6
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by Blanca Gómez
"A grandmother and grandchild nurse an injured bird together in this touching story about caring for all creatures, the wonder of nature, and letting go On a snowy day, a grandmother and grandchild find an injured bird. They take it home and care for it until it can fly around the living room. It is fantastic—just like everything at Abuela's house! But a fantastic moment is also bittersweet, for the little bird's recovery means that it's time to let it fly free. Drawing inspiration from a formative childhood experience, Blanca Gómez crafts a deceptively simple story that is morally and emotionally resonant and is brimming with love, wonder, and a deep respect for the natural world." -- publisher
Any Child
by Valerie Bolling and Maine Diaz
"Tap, twirl, twist, spin! With musical, rhyming text, author Valerie Bolling shines a spotlight on dances from across the globe, while energetic art from Maine Diaz shows off all the moves and the diverse people who do them. From the cha cha of Cuba to the stepping of Ireland, kids will want to leap, dip, and zip along with the dances on the page!" -- publisher
The Kite of Dreams
by Pilar López Ávila, Paula Merlán and Concha Pasamar
"A kite trip that will take us around the world to meet the hopes and dreams of children around the world, from Bolivia to Nepal. If something is capable of bringing the children of the world together, that is the ability to dream and imagine a better world. Every single one of us has with them a kite of dreams, a flying kite sheltering our hopes and dreams. Through this kite trip, we will meet Mohesiwä, a boy who lives in the jungle, Amunet, who can't stop but smiling when imagining a better future, Anja and his brother Tuvo, who rummage through the garbage to find something of value… The Kite of Dreams is an invitation to discover the different cultures of the world and to dream of a better world. A world with unpolluted air, where every child has access to education, a world with clean and uncontaminated waters, where biodiversity is preserved and forests are conserved. And more importantly, a world in which each child can be happy and reach out to those who need it." -- publisher
Grandpa stops a war
by Susan Robeson and Rod Brown
""Daddy always said it takes a man of peace to stop a war." Based on the true story of Paul Robeson's visit to the front lines of the Spanish Civil War, comes this recollection of his bravery and activism by his granddaughter, Susan Robeson, with her debut book. When Susan was a child her father and grandfather told her family stories over and over. Grandpa Paul was a great man, a singer with a deep and rumbling voice, a man of peace and principle who worried about the safety of the children and families living in countries at war. His songs were always full of emotion, and evoking the African-American spirituals of his own father's childhood, he was able to communicate even with people who didn't speak the same language. Though it was dangerous, Robeson went to Spain and traveled to the front lines of the war (in a Buick!). There, he asked the soldiers to set up speakers facing the fighters on both sides of the battlefield. And then he sang.... With gorgeous illustrations from the fine artist Rod Brown, When Grandpa Stops A War celebrates the activism and achievements of the great Paul Robeson, and shows readers the power of art in times of discord and war."--Provided by publisher
Biography Cross Group Oppression & Resilience
Gifts of our Lady of Guadalupe
by Demi
In this beautiful retelling of the Marian story by award-winning author, Demi, find out how the astonishing miracles of the Virgin of Guadalupe persuaded the bishop in Mexico City to build a church dedicated to her; how ten million Aztecs converted to Catholicism within just eight years; and why the basilica dedicated to Our Lady of Guadalupe today receives 20 million pilgrims per year, making it the most popular Christian pilgrimage site in the world.--ONIX annotation
Beautiful Life Biography Cross Group
The flying girl
by Margarita Engle and Sara Palacios
"Six months before the famous Wright Brothers' first flight, Aída de Acosta became the first woman to fly a powered aircraft."--Provided by publisher
Biography Oppression & Resilience
Never say a mean word again
by Jacqueline Jules and Durga Bernhard
When Samuel's father, the grand vizier, hears Hamza call Samuel names and tells his son to make sure Hamza never speaks an unkind word to him again, Samuel knows he must obey but has a hard time finding the right means to do so. Includes information about Jewish poet Samuel Ha-Nagid and the legend which inspired the story
Cross Group
Parrots Over Puerto Rico
by Cindy Trumbore and Susan L. Roth
"A nonfiction picture book about the history of Puerto Rico and the Puerto Rican parrot, which was brought back from the brink of extinction. Above the treetops of Puerto Rico flies a flock of parrots as green as their island home. . . . These are Puerto Rican parrots. They lived on this island for millions of years, and then they nearly vanished from the earth forever. Puerto Rican parrots, once abundant, came perilously close to extinction in the 1960s due to centuries of foreign exploration and occupation, development, and habitat destruction. In this compelling book, Roth and Trumbore recount the efforts of the scientists of the Puerto Rican Parrot Recovery Program to save the parrots and ensure their future. Woven into the parrots' story is a brief history of Puerto Rico itself, from before the first human settlers to the present day. With striking collage illustrations, a unique format, and engaging storytelling, Parrots Over Puerto Rico invites readers to witness the amazing recovery efforts that have enabled Puerto Rican parrots to fly over their island once again. Above the treetops of Puerto Rico flies a flock of parrots as green as their island home. . . . These are Puerto Rican parrots. They lived on this island for millions of years, and then they nearly vanished from the earth forever. " -- publisher
Martín de Porres
by Gary D. Schmidt and David Diaz
The story of Saint Martín de Porres--an endearing tale of perseverance, faith, and triumph over racial and economic prejudice.
by Carmen T. Bernier-Grand and Tonya Engel
Juan Diego hears the voice of the Virgin Mary asking him to petition the bishop for a shrine to be built in her honor, but the bishop will not agree unless Juan can bring him a sign.


Snare Drum Ornament – Add some extra Christmas spirit to your tree with a playful, Snare Drum Ornament! Wonderfully decorated with colorful paint, this tree decoration is sure to complete any drummer's holiday. It is a red colored snare drum with black music notes, drum sticks, and accented with iridescent glitter around the edge. This music ornament is perfect for the X-mas holiday season. Made from resin. Approx. 3.0″ L.


There will be pressure on the fiscal situation, especially at a time when the monsoon can also disappoint. More populist expenditure is on cards if the mandate is a hung Parliament or a coalition government.
The Congress' flagship poll plank this time around is the minimum income guarantee scheme, NYAY (Nyuntam Aay Yojna).
The manifesto is structured with the aim of capitalising on popular discontent against the incumbent BJP on areas like farm distress, unemployment, anti-poor perception, and tax and regulatory challenges for small businesses.
"The NYAY scheme, along with proposals of universal farm loan waiver, increased healthcare and education outlay – are at odds with the INC's concurrent aim of fiscal deficit at 3 per cent of GDP (gross domestic product).
"Likely compromises will be in the momentum of public capex, or higher taxation; both of which could also run afoul of the manifesto's commitments," wrote Sonal Varma, managing director and chief India economist at Nomura in a recent co-authored report with Aurodeep Nandi.
"We expect the other parties to compete for the attention of the poor in the run-up to the elections. Besides the focus on economy, the marketing plank will be focussed on pro-poor schemes.
"The pressure on the fiscal situation is just justified, especially at a time when the monsoon can also disappoint. I think more populist expenditure is on cards if the mandate is a hung Parliament or a coalition government.
"It can happen at the Central level, State level, or both," says Jigar Shah, CEO, Maybank Kim Eng Securities.
"Both the BJP and Congress are seen indulging in competitive populism to woo the voters.
"Farmers have become an important vote bank, especially after the recent state elections where in their distress has impacted the outcome for the BJP.
"This is particularly true in the Hindi heartland, where many states have 70 – 80 per cent rural/agri constituencies.
"For the markets, competitive populism will cause concerns on the fiscal situation," said Krishna Kumar Karwa, managing director, Emkay Global Financial Services.
"The markets are not worried about the fiscal situation right now. The only thing on their mind is political stability and continuity of reforms and policies by the next government.
"If the monsoon, too, is below normal, inflationary pressures could increase," Chokkalingam says.


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Дурга:
 Дурга — богиня в индуизме.
 Дурга — деревня в Кильмезском районе Кировской области.
 Дурга — деревня в Балтасинском районе Татарстана.

redpajama_set_name: RedPajamaWikipedia


Sonora Planning Commission, 5:30 p.m. Monday, City Hall, at 94 N. Washington St.
The new owners of the former bowling alley on South Stewart Street in downtown Sonora will seek permission from the city's Planning Commission on Monday to move forward with their plans for converting the long-vacant building into a self-storage business with about 100 units.
Joe Pluim, of Plum Construction, and his partners in PWM Properties, a limited-liability company that purchased the building for $375,000 in December, are seeking a conditional-use permit from the commission in hopes of beginning construction this summer.
While the partners previously talked about the possibility of housing other types of businesses in the building as well, Pluim said they are now moving forward with only self-storage units.
In February, the commission endorsed a change in the city's zoning laws to allow for self-storage businesses within commercial and limited manufacturing zoning districts. The City Council ratified the change in early March.
Pluim said he anticipates the meeting on Monday will go smoothly because they haven't received any negative feedback about the project.
The building was home to Sonora Family Bowl for 50 years before the business permanently closed in July 2010 due to slumping attendance. It has remained empty since that time.
Several groups and individuals have made a variety of proposals over the years about what to do with the unique space, ranging from a dance club to indoor farmers market, but parking requirements due to the size of the building have prevented those plans from moving forward.
The city charges an "in-lieu" fee of $1,500 for each parking space that's needed and can't otherwise be provided by the developer, with the required number of spaces determined by the maximum occupancy based on the size of building and type of use that's proposed.
The project proposed by PWM Properties would not be required to pay any in-lieu parking fees because the building has a credit for 75 spaces when it was a bowling alley and would only need five of those as an indoor self-storage facility, according to the city's analysis.
However, the owners are also planning to develop a gated parking lot with three spaces in the currently overgrown, vacant lot directly adjacent to the former bowling alley to provide side access into the self-storage facility.
Pluim said tenants would swipe a card to unlock the gate and enter the lot.
The partners in the project decided to turn the former bowling alley into self-storage units because of what they see as a growing demand for such facilities throughout the county, especially in the city where such businesses were previously not allowed and many rental units and homes do not have places for storage.
Businesses and residents would be able to rent the storage units, which Pluim said would vary in size and shape from roughly 25 to 150 square feet.
Pluim said he and his partners attended a self-storage conference in Las Vegas this week and learned that converting underutilized commercial buildings into self-storage units is a growing trend nationwide as the retail industry shrinks due to online shopping behemoths like Amazon.
"While retail is shrinking, the need for storage is increasing because people are still buying stuff and need a place to store things," he said, adding that they heard one man who converted a former roller rink into self-storage units.
The commission's decision on the conditional use permit will be final unless it's appealed to the City Council.


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Employment-Based Fifth Preference (EB-5) Program Set For Changes
In an effort to curb possible abuses, Congress held a hearing last year on "Citizenship for Sale: Oversight of the EB-5 Investor Visa Program." The EB-5 program has been around for almost 30 years and was created by Congress to stimulate the economy. Congress acknowledges that some steps might need to be taken to limit abuses.
According to the UCSIS, here are improvements that have already been made to the EB-5 program:
USCIS hired additional officers dedicated to conducting EB-5 site visits.
USCIS expanded security checks.
USCIS partnered with other federal agencies like the SEC, FBI and ICE to strengthen the EB-5 program.
USCIS began remotely interviewing foreign investors seeking to remove their conditional status as part of a pilot program.
USCIS published revised forms to enhance data collection.
The Investor Program Office (IPO) created a new Compliance Division to review annual certifications and conduct compliance reviews.
USCIS publishes regional center termination notices and makes available lists of currently designated and terminated regional centers.
USCIS has also published a Notice of Proposed Rulemaking for the EB-5 program. Late last month, the final regulation was sent to the Office of Management and Budget (OMB). Upon approval, the rule could be in effect by the Summer of 2019.
Here are the changes to expect:
The current minimum investment is anywhere from $500,000 to $1,000,000, which would be raised from $1 million to $1.8 million for standard direct investments and from $500,000 to $1 million for targeted employment areas.
Allowing certain EB-5 petitioners to retain older EB-5 priority dates; and
Changing the designation process for targeted employment areas.
We will continue to keep you posted on any developments, and it remains to be seen if the same number of wealthy overseas investors will attempt to participate in the program after changes are adopted.
March 5, 2019 News & Updates

redpajama_set_name: RedPajamaCommonCrawl


I sometimes find quotes I believe will help in my ministry or teaching. If it's worth the time for me to post it, it's worth your time to read it.
Thou movest us to delight in praising Thee; for Thou hast formed us for Thyself, and our hearts are restless till they find rest in Thee. — Saint Augustine, Confessions, I.i.
When the frontier between God and man, the last inexorable barrier and obstacle, is not closed, the barrier between what is normal and what is perverse is opened. — Karl Barth, The Epistle to the Romans, i.26-31.
A curse on him who begins in gentleness. He shall finish in insipidity and cowardice, and shall never step foot in the great liberating current of Christianity. — Andrè Trocmè, Huguenot pastor in World War II, quoted by Malcolm Gladwell, David and Goliath, p. 271.
Forgiveness does not mean ignoring what has been done or putting a false label on an evil act. It means, rather, that the evil act no longer remains as a barrier to the relationship. — Martin Luther King, Jr.
It is the duty of a prudent minister of God to hold his ministry in honor and to see to it that it is respected by those who are in his charge. Moreoever, it is the duty of a faithful minister not to exceed his powers and not to abuse his office in pride, but, rather, to administer it for the benefit of his subjects. — Martin Luther, Commentary on the Romans, I.i.
He who controls the past controls the future. He who controls the present controls the past. — George Orwell, 1984, 1947.


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Educated Patient® Kidney Cancer Summit: April 9, 2022 - Episode 3
Educated Patient® Kidney Cancer Summit Diagnosis Panel: April 9, 2022
Conference | Educated Patient® Kidney Cancer Summit
Watch Dr. Vitaly Margulis, Dr. Rana Mckay and Kwame Garrett-Price answer questions about kidney cancer diagnoses during the CURE Educated Patient Kidney Cancer Summit.
This panel featured Dr. Thomas Hutson, Dr. Vitaly Margulis, Dr. Rana Mckay and Kwame Garrett-Price.
Hutson: Kwame can you tell us about your journey?
Garrett-Price: In February 2019, I was having the worst headache I ever had, didn't know what it was, didn't know what was going on. And I passed out completely and thankfully I was in the presence of family and was rushed to Baylor Scott & White in Dallas, Texas, where I had an emergency craniotomy and no one knew what it was. And the biopsy results came back. And turns out it was von Hippel Lindau (disease).
And from that point on, we put together a team, you know, endocrinologist, obviously, PCP neurosurgeon, urologist because of the RCC, and we also did more testing. And at that point, we found RCC, and that was October 2019. That same year also had a partial adrenalectomy performed at Baylor Scott & White. So, the most important part of it was figuring out what to do, where to look, and where to, you know, find the specialists, because these tumors manifest themselves throughout different organs in the body, brain and eyes, kidney, pancreas. And, yeah, that's been my journey with it thus far.
Hutson: Thank you for sharing that. And if you had one statement to make to others, our patients and families, what would that be? Would it be to find expert care? Or what did you find to be the most useful for you with the support groups like the VHL Alliance? What advice would you give?
Garrett-Price: Support groups help as well, the VHL alliance has been phenomenal. You know, they're the world's leading organization, when it comes to, you know, von Hippel Lindau, and clinical care, and research and education. But one thing that's helped me so much, I created this flow of information between myself and my physician. And that's really kept me alive. You know, that's how we got the CT that found the RCC. And that's what's given me the best care because my visits are high-yield now, because they're kept in the know, for instance, my endocrinologist, I send her my blood pressure readings every 45 to 60 days. And she's such a wizard, Dr. Raffia Valera, here in Dallas. And, you know, she takes this data, it's just the fact that she has it. You know, as I said, it allowed my visits to always be high yield. So when I go in, it's boom, boom, boom, we can get straight to the point, you know, what's going on, we don't have to spend the first five minutes catching up on everything. And, you know, over the past, however many months since our last visit, so I would say, you know, you have to advocate for yourself, you have to be ready, you have to be knowledgeable, and you can't come in clueless, don't be scared to have notes, don't be scared to be able to move forward in that way and speak up for yourself. Because, you know, I know I'm on a panel full of physicians, but depending on what's happening, you know, a physician can come in the room, and they're out before you know, and you don't know anything. So make sure you're ready. That's what I would say that's how you get the highest yield results in the highest yield care.
Hutson: Well, that's amazing. And thank you for sharing your story. We've got several questions that have come in here. So let me just start giving them out. So any of the people on the panel can certainly jump in, but this this first one is going to Vitaly's talk on TNM. And the question is what does TNM stand for?
Margulis: Tumor, node, metastases and so I remember when I discussed the three components of the system, the TNM system, one has to do with the extent of the tumor in a site of origin, ie the kidney, how advanced or how extensive the tumor is, the N stands for has the cancer spread to any other regional lymphatic, there enlarged lymph nodes around the tumor, and the M stands for metastases as the cancer has spread to the other parts of the body. So that's in a nutshell.
Hutson: Perfect. And then I guess a follow up in there was once diagnosed, when is stage and predicted model established?
Margulis: It's a good question. And there are two opportunities to do that. You know, the general scenario is that the patient, we see a patient and then there is a mass that's discovered in the kidney. And we can get clinical staging, ie, staging based on X rays, CAT scans, etc. We haven't removed the tumor, we haven't treated the tumor. You can get a clinical stage based on the tumor size. Do we see any signs of the tumor spread? Was there any signs of the spread to the regional lymph nodes etc so we can get clinical stage aside. And the second opportunity to stage, and it's probably more accurate staging, is when we actually remove the primary tumor with surgery. We can look at the tumor we can get an exact understanding, has the tumor spread beyond the confines of the kidney as its invaded into the surrounding structures looking down at what's called the pathologic stage, the stage based on the battle logic evaluation, it's probably a little bit more accurate. So yeah, so those will be the usually the two times when we get appropriate staging done.
Hutson: And I guess this next question could be really as a kind of a in the gray between Dr. McKay's and Dr. Margulis's presentations, but is there an imaging or blood test that can determine if someone still has, what the questioner is calling, cancer stem cells once the tumor has or has already disappeared? Is there any way to use cancer stem cells or blood test to follow?
McKay: We certainly talked about that. I think there are blood tests that are being developed to detect what we call minimal residual disease after somebody has had their cancer resected and we think that they're in remission. I tell my patients remission plus time equals cure for those patients with localized disease. But during that period of remission, there are blood tests that are being developed to detect MRD. None of them have been, you know, clinically available for patients with RCC. There are certain tests that are done in colorectal cancer that are commercially available. The application of those tests in RCC has been a little bit more challenging, really given the nature of the kind of tumor that RCC tends to be. So though there's a lot of research that's being done into the identification of MRD tests for kidney cancer, but none that are available in primetime.
And just to kind of step back, I think just to comment on that first question that was, you know, provided to Kwame, whenever I approach kidney cancer, I think the three key questions that are always critically important to try to get answered for any given patient is what is it? Where is it? What are we going to do about it? So the first question of what is it really refers to what's the histology? What does the biopsy show? Is it a clear cell? Is it a non-clear cell? What are the genetics of the tumor? The second question is, where is it? And that gets to be your stage. What stage of cancer do you have? Is it localized or has spread? You know, where is it? Do I need additional imaging to figure out where the tumor is? Have I gotten all the right imaging? The last question is, what do we do about it? We can't really answer the last question until we know the answers to the first two questions. We get to the last question. I usually break it down into, okay, I see the tumor, do I need to do something to things that I see what are the focal or local therapy options? And then the second part of the what do I do question is do I need anything in my system? Do I need systemic therapy, something that would be taken orally or IV from the tumor? And I think if you can put that in a framework so that when you're coming in to see your doctor or somebody has a new diagnosis, those are always the three questions that I asked at any treatment decision change. That's okay. What is it? Where is it? What are we going to do about do we know what it is and reconfirmed it's kidney cancer? Where is it? I need imaging? What do I do?
Hutson: Excellent. And then, I guess following up on this, this would be for Kwame. You know, what ties into the VHL disease? You know what you mentioned Rana was that one of the big, scientific advances from the 1990s. I remember a science publication from 1993, talking about the discovery of the VHL gene, which you said was on the short arm of chromosome three, and then the tie into the development of the VHL syndrome, and then, which was more of a germline hereditary kind of syndrome. But the question here is, well, if that's the case, then how can this happen somatically? Why is VHL an issue?
McKay: No, very good question. I mean, there's a lot of things that can cause alterations in genes that can predispose somebody to cancer as we talked about, like, somebody could just be born with it. They're born with a genetic alteration, or environmental factors, various things play in sometimes we don't really know the cause most of the time, we don't know the cause. That can cause a mutation in the gene.
VHL is very important because this VHL regulates a certain protein called HIF. And VHL helps get rid of HIF when it's not supposed to be there and helps let it be there when it's supposed to be there. But if VHL helps kind of degrade this, it's not necessarily a bad protein, but it can. When things are normal, it really shouldn't be hanging around. And so HIF helps get rid of it. But when when VHL is gone, this protein just builds up in the cell and can cause all this badness and actually studying VHL syndromes from families of individuals that have this disease that has really catapulted us in developing all of these drugs for kidney cancer, the reason that we have (Sutent [sunitinib]), pazopanib, (Welireg [belzutifan]), all of these drugs that actually ended up winning a Nobel Prize in Medicine in 2019, is because of studying and understanding the pathogenesis of kidney cancer in patients with VHL syndrome.
Hutson: Absolutely, I often will tell my patients, sometimes we just don't know it's a cosmic ray. There's certainly risk factors and things smoking and obesity. Now there's stuff but we really don't know. And I remember back during the development of one of the drugs from the original group, it was pazopanib, we looked at our original phase 2 study of that drug. And we tried to determine could we detect these types of mutations and somatically. And using modern technology of the time, we could find some type of loss of function heterozygosity of the gene function or something that from the VHL gene that was that was in 97% or so of patients. And so I suspect that that some type of abnormality and function of VHL is the requirement to get clear cell RCC at some level and whether we can identify 99.999% of the null but we can certainly identify 97% of them using the technology of 2008. So, yes, it happens for reasons we don't know. So thank you.
And then we've got one another one that maybe anyone could take but I think Rana may be for you is I have several family members who have melanoma and I have RCC. I read that there might be a genetic similarity between the two cancers is this true?
McKay: Not necessarily a direct link, but I would probably say for people who have a strong family history. So when I think about, you know, a lot of melanoma in the family, you always have to factor in hereditary factors, but also lifestyle factors. So, you know, fair skin, where do y'all live? Like, you know, so there's a lot of things that come into play, because a lot of times the things that link us on a hereditary level, there's things that link us at an environmental level, like you're in the same environment, eating the same foods doing the same thing. But nonetheless, I think there are clear guidelines for who with RCC needs to get genetic testing. I think if there's any concern, definitely talk to your clinician, your provider about that. It's actually very easy to do genetic testing, we do it in our clinic all the time. We talk about it, we understand the results and the implications. And it's a simple blood test or a simple saliva swab. And then you get the test results back in, you know, two weeks, and I think the field is really moving into integrating this more into clinical practice. So I would definitely say talk to your clinician.
Hutson: And as a follow up to that. Another question was my family has been identified by the NIH as susceptible to kidney cancer (mother, mother's brothers and sister) but not because of any identified genetic mutation. One family member has a VUS. What should our surveillance plan be for the family members?
McKay: So, like I said, generally you don't need to do anything about them. For most it is not the reason. Their VUS is more because we just don't know. But based off of all the data that we have, thus far, they have not been identified to be pathogenic, that pathogenic but we just haven't investigated that specific mutation in that specific gene. So we really don't 100% know. So there's this kind of gray line of saying, okay, 95% of these are going to get reclassified as benign, but 5% may in the future, like five to 10 years from now we learn more about these conditions and they may get reclassified. Generally, there is no surveillance monitoring for somebody that has a VUS or no reason to do genetic testing for family members for somebody with VUS but talk to your provider, talk to your clinician and talk to a genetic counselor about it.
Hutson: That sounds great. And I remember what I use as just as a rule of thumb is that patients you know, younger than age 50 with kidney cancer should be set for genetic testing, for sure. And sometimes that is an older person, but they you knew that they had the tumor for quite a while. The median age of diagnosis of this cancer is 63 to 65. So that certainly seems appropriate. But anytime someone requests for genetic testing, I certainly will send them.
This is a good question for Vitaly. But I'm sure Rana knows the answer to but how long can a cell sample be stored, and still be able to provide pertinent genetic information?
Margulis: If the tumor is frozen appropriately, it can be essentially frozen indefinitely. So for germline testing, that tissue can be for somatic testing, that tissue can be essentially refrozen and assayed, especially for DNA, which is stable, you know, years and years afterwards. So it's all about proper storage and proper tissue handling, by the by the clinicians.
McKay: Yeah, I would agree. I mean, most of the time, most of these tests can actually be done on what we call the paraffin embedded block. So after Dr. Mercola does his surgery, a lot of times they'll put the specimen on wax and store it in pathology for a pretty long time. And we can actually do a lot with that paraffin embedded block that's basically sitting on wax.
Hutson: Excellent. And if we go back, Vitaly to your first slide, you had some statistics, I guess outcome survival statistics or whatnot in, in the question opposed is, is that before or after immunotherapy became a common treatment approach?
Margulis: I think my slide was very general. And I just talked a little bit about the incidence or number of new cases and a number of cases that progress after treatment. So when we take the tumor out, when we take the kidney out roughly, about 30% of the patients, if you take all comers will progress, or will cancer come back, that does not factor the latest sort of emergence of immunotherapy that's given an adjuvant or preventative space. So we'll have to see a little bit how that will, you know, affect the recurrence rates, especially in more advanced tumors. But roughly the rule of thumb, you know, out of 100 patients that I treat for kidney cancer, assuming some have more advanced disease, some less advanced disease, about 30%, at some point at five years, will have some degree of recurrence.
Hutson: Great, thank you. And then, Kwame, do you have any advice on how to have the talk with your family about a genetic cancer?
Garret-Price: Yes, so within my family, within four months, of me doing my own genetic testing, half of my immediate family had done it, and the other half had a little anxiety about it. But I find that if you can reach the matriarch of the family, oftentimes, the drivers or the decisions for everything, they're setting the appointments for everybody. So if she understands the importance of it, then I think that's a win for getting genetic test, and then you make it a family thing, making it a bit, you know, make sure everyone can do it together, or, you know, around the same time, so those who are anxious, you know, they're a little bit less apprehensive, because, you know, understanding your health profile is a huge advantage. You know, there I say competitive advantage, and just navigating this world and being healthy and being able to function day to day. And to be quite frank, it was one of the coolest things I've ever done sitting down with my genetic counselor, and she's going through everything and, and just, I don't know, it's an awesome thing. So I think communicating that as well, the same way you do a family tree, you know, in another way for another reason, you know, you're doing a similar thing. You know, with this, we jump up, we do family trees, instantly. And, you know, with this, I feel, if you can just break down, that it's a great thing. It's a cool thing. And you can reach the key decision maker, you're good and makes the conversation a lot easier to have.
Hutson: Thank you for that. That's very insightful. The next couple of questions have to do again with kind of genetics and prognosis. So there's a question: Is there any research around prognosis and or therapies for NF2 mutation in non-clear cell unclassified?
McKay: I think there is research that is being done. There's nothing that's sort of close to entering into the limelight. I mean, we don't have a target, a drug that can specifically target that gene. I think with regards to understanding its prognosis and RCC, it hasn't historically stood out as sort of a big hitter, with regards to really swaying how people do one way or another. But certainly, I think we need to learn and understand these genes more and how to better target them when we think that they could be drivers of the cancer.
Hutson: Yeah, absolutely. That's an area ripe for ongoing research here. This last question, I think could be maybe handled by all three panelists. And there's really two parts to it. One is if it's if suspected genetic and they put in 5%. RCC, is it wise to be with a teaching academic hospital? It's one important question. And then how do you determine the appropriateness of your doctors and team? How are you able to assess whether you're at the right place?
McKay: I think maybe we're biased. I mean, I think, for me, for somebody who has a cancer, I think, wherever you are, trying to find an NCI-designated cancer center is probably a good thing. If for the minimum of just getting a second opinion, to make sure that you're on the right path, and you're getting, you know, the right treatment. And hopefully, there is, you know, I think, consistency in messaging across all your doctors regarding what's the right strategy, I think second opinions can be helpful in doing that. I think for people with hereditary syndromes that have a complex disease, honestly, they are best managed and best treated at an NCI designated cancer center or center where they truly have a multidisciplinary team that can assess all of the different components for that given syndrome. You know, it's not just medical oncology, it's not just urology, it's actually the whole gamut neurosurgery, ophthalmology. So pulmonary, depending on where the disease is neurology, so making sure that there's a system of specialists and that those, it's not just having the specialists, it's that the specialists actually talk to one another is an important thing.
Garret-Price: I found the research institutions to be the most thorough, the most comprehensive. I enjoy my visits at UT Southwestern, and no patient says they enjoy their visits. But, you know, I do find it to be very thorough, like a little symposium, you know, during the visit. So I, there's a balance in the way I found my care team. Luckily, I have some family members in health care. So I kind of I was fortunate to be able to put aside insurance in there work out in network, and I just wanted to find the best care. So we did extensive research, reputation, quality of care, different things like that and put together such a great care team.
Hutson: And then from the surgical perspective, what do you say, Dr. Margulis?
Margulis: Yeah, I would say that the key here is going to a place where there is number one, multidisciplinary team. The word there are people that understand and know this disease or any genetic disease, I mean, these conditions are highly nuanced. And, you know, you want to go to or you want to ask your doctor, you know, , how many VHL patients do you see is am I the first one and my second one? Having said that, I don't think that that necessarily has to happen in an academic center. To give you an example, we, you know, for example, there's a ophthalmologist that's in private practice with a very, very large number of VHL patients that he treats for retinal issues. And he's fantastic, that's his passion. And he's outstanding at that. So when we actually see patients from him because he's the first one to diagnose VHL. So, I echo what's been said, I do agree that all things being equal, you know, probably academic center or cancer center will be a place to start. But these are highly nuanced, you know, conditions with highly nuanced treatments. And I think that you really want to have a high-level team built around this
Hutson: Absolutely. Well, as we conclude this first part, which has been awesome, and thank you to everyone who's participated, can we just get some final thoughts as we close this first part of our meeting today, and then we're going to have a little 10-minute break and come back by 12:15. But I want to get an opportunity for Kwame, Dr. Margulis and Dr. McKay to give their final thoughts.
Margulis: Well, I started this, so I give my two cents. And, you know, I was tasked to talk about TNM staging system, which is fantastic, which has been the cornerstone of, you know, tumor assessment, treatment management, etc. But I think, you know, as other panelists, as Rana has pointed out, I think we're moving away from the crude histologic assessments into molecular tumor assessment. And I think, you know, the MRD space and minimal residual space where we can determine Is there any circulating DNA in patients' blood or urine? This is how we will monitor the patients in the future. And this is how we will make treatment decisions. I think it's just a matter of time.
McKay: I think there's been a lot of advancements in the field of oncology over the last really actually five years with catapulting a lot of genetic testing kind of, you know, right there at the point of care in the clinic. I think, finding a physician and care team where your physician should be your biggest advocate, your group of physicians should be your biggest advocates is going to be like, instrumental in just optimizing treatment and optimizing a care plan for any given patient. And, you know, making sure to find that and strive for that, I think is key.
Garret-Price: Yeah, just wanted to reemphasize how to get a high yield visit. You know, Dr. McKay touched on three things that she looks for are three questions she would like answered, you know, before you walk into a room, and that's very important, it changes the course of your care. And there are so many things you can do on your own preventative measures, you can take diet, lifestyle changes, before you even walk into the office. So just want to make sure we know those are things that we have to proactively do for yourself, as a patient, you know, before you even reach the physician.
Hutson: Thank you. And I'll just end by saying from my own experience, as a patient, you need to advocate for your own care, also. And so don't feel hesitant about questioning the choices, your doctor and your care team should have a rationale for why they're choosing a therapy or a pathway, especially as you'll hear later throughout the day that, we don't have the answers to everything. And there's subjectiveness. But getting second opinions is key and at least among the key leaders in the field, you know, everyone communicates. Well, I mean, this is the nice thing about our the RCC community, whether it be from the advocacy side, or the physician researcher, or the care teams is that we all are. We all get along well, and we all share information. And certainly no one should be offended. No doctor or care team should be offended by a patient requesting a second opinion or choosing another care team. Because they just align better with what their values are. So just you know, the biggest thing is don't be reluctant or hesitate to do that.
McKay: I just want to say, if you're feeling pressure right now of Oh, my God, I need to go find this and do this and do a, like, take a deep breath. And you have your team of doctors, you've got social workers, you got nurses, relay these thoughts to them, have them help guide you. Like there's advocacy groups, there's people to help you don't feel like you're leaving this with like with a homework assignment of all these 20 things you gotta go do. There are people that are there to help you through the process. There's advocacy groups, and lots of, you know, resources there. And like I said, Your doctor should be your biggest advocate to help you through this process. So if you feel this pressure, like relay that to your care team, so they can help you, you know.
Raw transcriptions have been lightly edited for clarity.
EP. 1: Educated Patient® Kidney Cancer Summit Staging Presentation: April 9, 2022
EP. 2: Educated Patient® Kidney Cancer Summit Genetic Testing in RCC Presentation: April 9, 2022
EP. 3: Educated Patient® Kidney Cancer Summit Diagnosis Panel: April 9, 2022
EP. 4: Educated Patient® Kidney Cancer Summit Surgical Options Presentation: April 9, 2022
EP. 5: Educated Patient® Kidney Cancer Summit Adjuvant Therapy Options Presentation: April 9, 2022
EP. 6: Educated Patient® Kidney Cancer Summit Frontline Therapy Panel: April 9, 2022
EP. 7: Educated Patient® Kidney Cancer Summit Panel on Immunotherapy/TKI Combo Treatment: April 9, 2022
EP. 8: Educated Patient® Kidney Cancer Summit Panel on Side Effects from Immunotherapy/TKI Combo Treatment: April 9, 2022
EP. 9: Educated Patient® Kidney Cancer Summit Panel on Communicating With Providers About Immunotherapy/TKI Combo Treatment: April 9, 2022
EP. 10: Educated Patient® Kidney Cancer Summit IO/IO Therapies Presentation: April 9, 2022
EP. 11: Educated Patient® Kidney Cancer Summit IO/TKI Therapies Presentation: April 9, 2022
EP. 12: Educated Patient® Kidney Cancer Summit IO/TKI-Related Side Effects Presentation: April 9, 2022
EP. 13: Educated Patient® Kidney Cancer Summit Combination Therapy Panel: April 9, 2022
Educated Patient® Kidney Cancer Summit: April 9, 2022Educated Patient® Kidney Cancer SummitKidney CancerSummits On DemandConference
SBRT Reduces Side Effects, Treatment Visits in Pancreatic Cancer
Better Quality of Life Reported with Minimally Invasive Esophageal Cancer Treatment


text:
package com.be.android.library.worker.interfaces;

import com.be.android.library.worker.models.JobParams;

import java.util.Collection;
import java.util.Map;

public interface ParamsBuilder {
    ParamsBuilder group(int groupId);

    ParamsBuilder priority(int priority);

    ParamsBuilder payload(Object payload);

    ParamsBuilder tags(String... tags);

    ParamsBuilder tags(Collection<String> tags);

    ParamsBuilder addTag(String tag);

    ParamsBuilder removeTag(String tag);

    ParamsBuilder addExtra(String key, Object value);

    <T extends Map<String, Object>> ParamsBuilder addExtras(T extras);

    ParamsBuilder removeExtra(String key);

    ParamsBuilder flag(String flag, boolean value);

    ParamsBuilder flag(String flag);

    ParamsBuilder flags(String... flags);

    /**
     * Set job class name
     * <br>
     * Similar to {@link #addExtra(String, Object)} with {@link JobParams#EXTRA_JOB_TYPE}
     * as first argument
     * @param jobClazz Job class
     * @return this object
     */
    ParamsBuilder jobClass(Class<? extends Job> jobClazz);

    JobParams build();
}

redpajama_set_name: RedPajamaGithub


Home Days Out It's Onwards and Upwards for Thomas Land's™ newest addition!
Comments Off on It's Onwards and Upwards for Thomas Land's™ newest addition!
Europe's only Thomas Land™ attraction is welcoming a brand new ride this season to mark further expansion to the thriving attraction at Drayton Manor Park, Staffordshire.
James and the Red Balloon, set to delight visitors from Saturday 1st April 2017, is based on the beloved Thomas and Friends™ character James and his naughty antics with a red hot air balloon. James will be joining the 20 other rides and attractions based on the popular series. Families can experience the magic of Sodor like never before as they fly up, up and away in their very own balloon.
The £250,000 ride, made by Italian manufacturer Zamperla, takes visitors on an adventure onwards and upwards to the lofty heights of 1.5 metres with a maximum speed of 5.5 RPM, a perfect ride for the whole family to enjoy. Families will get to enjoy extensive views of the park from the ride's highest points, the perfect opportunity to plan which ride is next!
The successful multi-million pound Thomas Land was launched in 2008 and recently underwent a £2.5 million expansion, cementing its status in the hearts of UK families. The well themed attraction ensures visitors of any age are immersed in Thomas and Friends fun from the moment they step in the gates.
For those who want to plan a Thomas filled staycation, the Drayton Manor Park Hotel also extends the fun from Thomas Land into its halls with special themed rooms and a 'Stay and Play' package, perfect for those young and old locomotive enthusiasts!
William Bryan, Managing Director of Drayton Manor Park said: "This is another exciting step in Thomas Land's progression. We are always looking for new ways to excite and entertain our visitors and James and his Red Balloon seemed the perfect addition to our family of rides.
To book tickets, or for more information about the park, visit Drayton Manor or call 0844 472 1950. Reduced prices are available for tickets bought in advance. For regular updates and news about Drayton Manor Park join it on Twitter and Facebook.
Today, Drayton Manor Park, still family owned and run by second and third generation family members, covers 286 acres of beautiful woodland and countryside and features some of the wildest and scariest rides around. Rides include: Rollercoasters – Shockwave, Ben 10: Ultimate Mission and G-force. Major rides: Maelstrom, Apocalypse and Storm Force 10, our wettest ride. It is home to the hugely popular Thomas Land™ featuring many named Thomas & Friends™ character rides for the children and adults alike. Thomas Land™ at Drayton Manor is the only one of its kind in Europe. The park's 152 seat 4D Cinema, over 50 rides and attractions makes for a truly unforgettable day.
The popular Grill Inn Restaurant and bar is adjacent to the hotel, seats up to 150 guests and is open all year round.
Thomas & Friends has scooped prestigious accolades including a Parent's Choice Award, a Webby and a Licensing Award for Best Classic Licensed Property. The famous and cheeky No.1 blue engine and his friends invite children to enter a world of imagination through the tracks of a train. Together, the children and engines embark on timeless adventures while experiencing valuable yet fun life lessons of discovery, friendship and cooperation. Thomas & Friends™ is viewable on more than 30 world-class broadcasters including PBS KIDS® in the US, Super RTL in Germany, and on Five's Milkshake! and Nick Jr. in the UK.
Downloadable episodes are available through iTunes and on YouTube You Tube Thomas and Friends .
For more information about the wonderful world of Thomas & Friends, please visit Thomas and Friends, Facebook and Twitter.


text:
Exploitable Flaw in NPM Private IP App Lurks Everywhere, Anywhere
November 24, 2020 22:31 by Paul Roberts
A serious security flaw in a commonly used, but overlooked open source security module may be undermining the integrity of hundreds of thousands or even millions of private and public applications, putting untold numbers of organizations and data at risk.
A team of independent security researchers that includes application security professionals at Shutterstock and Squarespace identified the flaw in private-ip, a npm module first published in 2016 that enables applications to block request forgery attacks by filtering out attempts to access private IP4 addresses and other restricted IP4 address ranges, as defined by ARIN
The SSRF Blocker That Didn't
The researchers identified a so-called Server Side Request Forgery (SSRF) vulnerability in commonly used versions of private-ip. The flaw, CVE-2020-28360, allows malicious attackers to carry out SSRF attacks against a population of applications that may number in the hundreds of thousands or millions globally. It is just the latest incident to raise questions about the security of the "software supply chain," as more and more organizations shift from monolithic to modular software application development built on a foundation of free and open source code.
Report: Cybercriminals target difficult-to-secure ERP systems with new attacks
According to an account by researcher John Jackson of Shutterstock, flaws in the private-ip code meant that the filtering allegedly carried out by the code was faulty. Specifically, independent security researchers reported being able to bypass protections and carry out Server-Side Request Forgeries against top tier applications. Further investigation uncovered a common explanation for those successful attacks: private-ip, an open source security module used by the compromised applications.
SSRF attacks allow malicious actors to abuse functionality on a server: reading data from internal resources or modifying the code running on the server. Private-ip was created to help application developers spot and block such attacks. SSRF is one of the most common forms of attack on web applications according to OWASP.
Black Box Device Research reveals Pitiful State of Internet of Things Security
The problem: private-ip didn't do its job very well.
"The code logic was using a simple Regular Expression matching," Jackson (@johnjhacking) told The Security Ledger. Jackson, working with other researchers, found that private-ip was blind to a wide number of variations of localhost, and other private-ip ranges as well as simple tricks that hackers use to obfuscate IP addresses in attacks. For example, researchers found they could send successful requests for localhost resources by obscuring those addresses using hexadecimal equivalents of private IP addresses or with simple substitutions like using four zeros for each octet of the IP address instead of one (so: 0000.0000.0000.0000 instead of 0.0.0.0). The result: a wide range of private and restricted IP addresses registered as public IP addresses and slipped past private-ip.
Private-IP: small program, BIG footprint
The scope of the private-ip flaws are difficult to grasp. However, available data suggests the component is very widely used. Jackson said that hundreds of thousands, if not millions of applications likely incorporate private-ip code in some fashion. Many of those applications are not publicly addressable from the Internet, but may still be vulnerable to attack by an adversary with access to their local environment or by remote attackers.
Private-ip is the creation of developer Damir Mustafin (aka "frenchbread"), a developer based in the Balkan country of Montenegro, according to his GitHub profile, which contains close to 60 projects of different scopes. Despite its popularity and widespread use, private-ip was not a frequent focus of Mr. Mustafin's attention. After first being published in August 2016, the application had only been updated once, in April 2017, prior to the most recent update to address the SSRF flaw.
A Low Key, High Distribution App
The lack of steady attention didn't dissuade other developers from downloading and using the npm private-ip package, however. It has an average of 14,000 downloads weekly, according to data from GitHub. And direct downloads of private-ip are just one measure of its use. Fully 355 publicly identified npm modules are dependents of private-ip v1.0.5, which contains the SSRF flaws. An additional 73 GitHub projects have dependencies on private-ip. All told, that accounts for 153,374 combined weekly downloads of private-ip and its dependents. One of the most widely used applications that relies on private-ip is libp2p, an open source network stack that is used in a wide range of decentralized peer-to-peer applications, according to Jackson.
While the flaw was discovered by so-called "white hat" vulnerability researchers, Jackson said that it is almost certain that malicious actors knew about and exploited it -either directly or inadvertently. Other security researchers have almost certainly stumbled upon it before as well, perhaps discovering a single address that slipped through private-ip and enabled a SSRF attack, while failing to grasp private-ip's role or the bigger flaws in the module.
In fact, private-ip may be the common source of a long list of SSRF vulnerabilities that have been independently discovered and reported in the last five years, Jackson said."This may be why a lot of enterprises have struggled with SSRF and block list bypasses," he said.
After identifying the problem, Jackson and his team contacted the developer, Damir Mustafin (aka "frenchbread"), looking for a fix. However, it quickly became clear that they would need to enlist additional development talent to forge a patch that was comprehensive. Jackson tapped two developers: Nick Sahler of the website hosting provider Square Space and the independent developer known as Sick Codes (@sickcodes) to come up with a comprehensive fix for private-ip. The two implemented the netmask utility and update private-ip to correctly filter private IP ranges and translate all submitted IP addresses at the byte level to catch efforts to slip encoded addresses past the filter.
Common Mode Failures and Software Supply Chain
Even though it is fixed, the private-ip flaw raises larger and deeply troubling questions about the security of software applications on which our homes, businesses and economy are increasingly dependent.
The so-called Heartbleed vulnerability in OpenSSL, which was first disclosed in 2014, helped raise alarms about the fragility of the open source supply chain. In the wake of that flaw, Google and other wealthy technology firms devoted money, resources and staff to help shore up the security of critical open source projects like OpenSSL.
But the headlong growth of the Internet, an ever-greater reliance on open source components and the shift to agile development and modular applications works against such efforts. The broad reach of an obscure tool like private-ip suggests that there are thousands – if not tens- of thousands of secret OpenSSLs out there: critical, widely used, but overlooked.
Security experts say the increasingly byzantine ecosystems of open source software accessible via sites like npm, with their poorly understood 'dependencies' are ripe for "common source" failures, in which the failure of a discrete, but common component causes hard-to-predict ripple effects that lead to the collapse of larger and more complex systems.
Sites like npm are a critical part of that ecosystem -and part of the problem. Created in 2008, npm is a package manager for the JavaScript programming language that was acquired by GitHub in March. It acts as a public registry of packages of open source code that can be downloaded and incorporated into web and mobile applications as well as a wide range of hardware from broadband routers to robots. But vetting of the modules uploaded to npm and other platforms is often cursory. Scores have been called out as malicious and an even greater number are quietly dropped from the site every day after being discovered to be malicious in nature.
Less scrutinized is low quality code and applications that may quickly be adopted and woven into scores or hundreds or thousands of other applications and components.
"The problem with (software) dependencies is once you identify a problem with a dependency, everything downstream is f**ked," the developer known as Sick Codes told The Security Ledger. "It's a house of cards."
Patch Now
In the short term, organizations that know they are using private-ip version 1.0.5 or earlier as a means of preventing SSRF or related vulnerabilities should upgrade to the latest version immediately, Jackson said. Static application security testing tools can help identify whether private-ip is in use within your organization.
The bigger fix is for application developers to pay more attention to what they're putting into their creations. "My recommendations is that when software engineers use packages in general or third party code, they need to evaluate what they're using and where its coming from," Jackson said.
Tags: software bug, vulnerabilities
Author: Paul RobertsI'm an experienced writer, reporter and industry analyst with a decade of experience covering IT security, cyber security and hacking, and a fascination with the fast-emerging "Internet of Things."
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$50k fine for passengers not wearing masks at...
Joyce hints Qantas could buy Project Sunrise A350s...
Qantas adds virtual reality option for in-flight entertainment
Qantas will provide select passengers on A380 flights and in its first class lounges a Samsung Gear VR virtual reality headset.
Some Qantas passengers will have the chance to experience the wonders of virtual reality at 38,000 feet when the airline commences a three-month trial of the new Samsung Gear VR headset in February.
The headsets are fitted with a Samsung smartphone pre-loaded with some promotional Qantas material such as an on-the-ground perspective of an Airbus A380 taking off and a tour of its Los Angeles lounge.
The airline has also teamed with Northern Territory tourism to produce a Kakadu-themed clip showcasing the best of the Top End in a 360-degree format. Motion picture films are also on the cards.
The smartphone that attaches to the headset is pre-loaded with virtual reality content.
Qantas group executive for brand, marketing and corporate affairs Olivia Wirth says the virtual reality technology is expected to be popular with travellers.
"It is a truly amazing experience and we believe that our customers, during this trial period, will absolutely love," Wirth told reporters at the trial's launch in Sydney on Thursday.
"Not only will it allow us to connect better with our customers and offer them a unique experience, it also allows us to work with tourism partners to showcase fantastic destinations both in Australia and overseas.
"We will also have the opportunity to showcase some Hollywood blockbusters on board this fantastic equipment."
The headsets will be available at Qantas's first class lounges in Sydney and Melbourne, as well in the first class cabin of A380-operated flights from Melbourne and Sydney to Los Angeles, the airline said in a statement.
A potential scene on board a Qantas flight. (Qantas)
VH-OJA finds a retirement home
Last Virgin Blue aircraft enters paintshop
adammudhen January 30, 2015 - 2:04 pm
But they refuse to put wi-fi on board. Go figure.


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Mixed Numbers Average Calculator
Percentage Pythagorean Theorem Divisibility Test GCF - Greatest Common Factor LCM - Least Common Multiple LCD - Least Common Denominator Fraction Arithmetic & Conversion Ratio & Proportion Logarithm & Anti-Logarithm Prime Number Prime Factors Arithmetic Progression Geometric Progression Billion - Million - Crores - Lakhs Conversion Binary to Decimal, Hex & Octal Decimal to Binary, Hex & Octal Hex to Decimal, Binary & Octal Octal to Decimal, Binary & Hex Weighted Average Grade Square & Square Root of a Number Cube & Cube Root of a Number Sum of Squares or Cubes Exponential Power e Raised to Power x Number to Radical Number Number to word Number to Roman Numeral Ascending & Descending Order
Fraction Addition Fraction Subtraction Fraction Multiplication Fraction Division Fraction Average Fraction Comparision Multiple Fractions Addition Mixed Numbers Addition Mixed Number Subtraction Mixed Numbers Multiplication Mixed Number Division Mixed Numbers Average Fraction to Decimal Decimal to Fraction Mixed Number to Fraction Improper Fraction to Mixed Number Fraction Simplification Equivalent Fraction
Mixed Fraction A
Mixed Fraction B
Average = `19/4`
Decimal Form = `4.75`
Average of Mixed Numbers - work with steps
Mixed Fraction A = `7 1/6`
Mixed Fraction B = `2 1/3`
Find the average of two given mixed numbers?
Formula :
Average = `(A + B)/2`
`7 1/6\divide7 1/6 = ?`
Convert mixed numbers into equivalent frations
`7 1/6 = ((7\times6)+1)/6` = `(42 + 1)/6` ` = 43/6`
`2 1/3 = ((2\times3)+1)/3` = `(6 + 1)/3` ` = 7/3`
denominator of the two fraction is different. Therefore, find lcm for two denominators (6, 3) = 6
Multiply lcm with both numerator & denominator
`43/6 + 7/3 = (43\times6)/(6\times6) + (7\times6)/(3\times6)`
= `(43\times1)/(6) + (7\times2)/(6)`
= `(43)/(6) + (14)/(6)`
Add two numerator of the fraction
`(43)/(6) + (14)/(6) = (43 + 14)/6 = 57/6`
Average = `((57/6))/2`
Average = `57/12`
Simplify above fraction `57/12`
Common divisor of (57, 12) is 3
Divide both numerator & denominator by gcd value 3
`57/12 = frac{57\divide3}{12\divide3} = 19/4`
Mixed numbers average calculator uses two mixed numbers, i.e. two numbers in terms of a whole numbers and proper fractions, $A\frac{a}{b}$ and $B\frac{c}{d}$ for positive integers $a,b,c$ and $d$, and calculates the halfway, mean or average of two mixed numbers by adding them and dividing by $2$. It is an online algebra tool for finding the average in the simplest form of two mixed numbers.
Enter two mixed numbers $A\frac{a}{b}$ and $B\frac{c}{d}$ in the box. These numbers must be in terms of whole numbers and proper fractions. The numerators and denominators in the proper fractions must be positive integers.
Press the "GENERATE WORK" button to make the computation;
Average of mixed fractions calculator will give the average of two numbers represented as mixed numbers.
Input: Two mixed numbers;
Output: A fraction in the simplest form.
Conversion of Mixed Number to Improper Fraction Rule: The mixed number $A\frac{a}{b}$ for $a,b>0$ can be rewritten as improper fraction by the following formula $$A\frac{a}{b}=\frac{A\times b+a}{b},\quad \mbox{for}\;a,b>0$$ Mixed Numbers Average Rule: The average of two numbers $A\frac{a}{b}$ and $B\frac{c}{d}$ is determined by the following formula
If proper fractions of mixed numbers are like, $b=d$:
$$\mbox{Average}=\frac{A\frac{a}{b}+B\frac{c}{b}}{2}=\frac{\frac{A\times b+a}{b}+\frac{B\times b+c}{b}}{2}=\frac{A+B}{2}+\frac{a+c}{2\times b},\quad \mbox{for}\;b\ne0$$
If proper fractions of mixed numbers are unlike, $b\ne d$:
$$\mbox{Average}=\frac{A\frac{a}{b}+B\frac{c}{d}}{2}=\frac{\frac{A\times b+a}{b}+\frac{B\times d+c}{d}}{2}=\frac{(A\times b+a)\times d+(B\times d+c)\times b}{2\times b\times d},\quad \mbox{for}\;b,d\ne0$$ or equivalently, $$\mbox{Average}=\frac{A\frac{a}{b}+B\frac{c}{d}}{2}=\frac{(A\times b+a)\times \frac{LCM(b,d)}{b}+(B\times d+c)\times \frac{LCM(b,d)}{d}}{2\times LCM(b,d)},\quad \mbox{for}\;b,d\ne0$$ where $LCM(b,d)$ is the least common multiple of $b$ and $d$.
How to Find Average of Two Mixed Numbers?
To find the average of a set of numbers, find the sum of the numbers and then divide by the number in the set. Average of one number is the number itself. In other words, a mixed numbers average is the midpoint or balanced point of these numbers. Since a mixed number can be converted to a proper fraction by the corresponding rule, then the average of mixed numbers is the same as the average of corresponding improper fractions.
When we deal with mixed numbers, there are two formulas for finding the mixed numbers average:
When proper fractions of mixed numbers are like
When proper fractions of mixed numbers are like, $b=d$, then the average of two mixed numbers can be expressed by the following formula: $$\mbox{Average}=\frac{A\frac{a}{b}+B\frac{c}{b}}{2}=\frac{\frac{A\times b+a}{b}+\frac{B\times b+c}{b}}{2}=\frac{A+B}{2}+\frac{a+c}{2\times b},\quad \mbox{for}\;b\ne0$$
When proper fractions of mixed numbers are unlike
When proper fractions of mixed numbers are unlike, $b\ne d$, to find the mixed numbers average of two or more numbers, it is necessary to follow the next steps:
Convert mixed numbers to corresponding improper fractions;
Find the LCM of denominators of derived improper fractions;
Rewrite these fractions over the LCM;
Add new numerators;
Put the sum of numerators over the LCM;
Divide this fraction by the number of fractions, $n$;
Simplify the result if needed.
This method can be expressed algebraically: $$\mbox{Average}=\frac{A\frac{a}{b}+B\frac{c}{d}}{2}=\frac{(A\times b+a)\times \frac{LCM(b,d)}{b}+(B\times d+c)\times \frac{LCM(b,d)}{d}}{2\times LCM(b,d)},\quad \mbox{for}\;b,d\ne0$$ If $LCM(b,d)=b\times d$, then the previous formula becomes $$\mbox{Average}=\frac{A\frac{a}{b}+B\frac{c}{d}}{2}=\frac{\frac{A\times b+a}{b}+\frac{B\times d+c}{d}}{2}=\frac{(A\times b+a)\times d+(B\times d+c)\times b}{2\times b\times d},\quad \mbox{for}\;b,d\ne0$$ For example, let us find the average of mixed numbers $7\frac 16$ and $2\frac 13$. After converting these numbers to improper fractions, we obtain $$7\frac 16=\frac{7\times 6+1}6=\frac{43}6\quad\mbox{and}\quad 2\frac13=\frac{2\times 3+1}{3}=\frac 73$$ Since $LCM(6,3)=6$, then $$\mbox{Average}=\frac {43\times 1+7\times 2}{2\times 6}=\frac {57}{12}$$ To write the mixed numbers average in simplest form, find the GCF of the numerator and denominator of the result. Because $GCF(57,12)=3$, the average of mixed numbers $7\frac 16$ and $2\frac 13$ is $$\frac{57\div 3}{12\div 3}=\frac{19}4$$ The Average of Mixed Numbers work with steps shows the complete step-by-step calculation for finding the mixed numbers average for $7\frac 16$ and $2\frac 13$ using the mixed numbers average rule. For any other mixed numbers, just supply two mixed numbers in terms of a whole number and proper fraction and click on the "GENERATE WORK" button. The grade school students may use this mixed numbers average calculator to generate the work, understand the methods of finding averages, or do their homework problems efficiently.
Real World Problems using Average of Mixed Numbers
Average describes a number which represents the general characteristics of a larger group of unequal objects.The average is a common measure of central tendency among a set of numbers. The arithmetic mean can be visualized as a balancing point on a scale.
There are three main types of averages: arithmetic mean, weighted average and average speed. The weighted arithmetic mean is similar to an arithmetic mean, except that instead of each of the number contributing equally to the final average, some numbers contribute more than others. In other words, we multiply each number by its weighting factor. The average speed of a body in time is the distance traveled by the body divided by the duration of the interval. In statistics, there are several types of "averages". For example, mean, median and mode. In algebra, here are four kinds of means of numbers: arithmetic mean, root mean, harmonic mean and root mean. We use the arithmetic mean when numbers add, geometric mean when numbers multiply, harmonic mean when the reciprocals of the numbers add, quadratic mean when the squares of the numbers add. The inequality between these means of a set of positive real numbers $x_1,\ldots,x_n$ says: $$ \sqrt{\frac{x_1^2+\ldots+x_n^2}{n}}\geq\frac{x_1+\ldots+x_n}{n}\geq\sqrt[n]{x_1\cdot x_2\ldots\cdot x_n}\geq \frac{n}{\frac{1}{x_1}+\ldots+\frac{1}{x_n}}$$
Mixed Numbers Average Practice Problems
Find the arithmetic mean of $9\frac 18$ and $3\frac {3}{7}$.
Ann washed a car for $4$ hours and earned $\$6\frac12$ per hour. Then she washed windows for $2$ hours and earned $\$5\frac 13$ per hour. Find Ann's average earnings per hour for this $6$ hours?
The mixed numbers average calculator, formula, example calculation (work with steps), real world problems and practice problems would be very useful for grade school students (K-12 education) to understand the arithmetic mean of two or more numbers represented as mixed numbers. Using this concept they can be able to find the mean of a data set in statistics and extend their knowledge developed in theory of numbers.
Fraction Comparison Calculator
Multiple Fractions Addition Calculator
Mixed Numbers Addition Calculator
Mixed Number Subtraction Calculator
Mixed Numbers Multiplication Calculator
Mixed Number Division Calculator
Fraction to Decimal Converter
Decimal to Fraction Converter
Mixed Number to Fraction Converter
Improper Fraction to Mixed Number Converter
Simplify Fraction Calculator
Equivalent Fractions Calculator


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Turcotte-Piper Mortuary
Send Flowers for Edward
Edward Lee Mitman
Edward Lee Mitman , 89, passed away on January 1, 2020 in Kingsville, Texas.
He was born on December 30, 1930 in Dayton, Ohio to Vernon and Edna Mitman . He was a graduate of Miamisburg high school class of 1948. Edward was a veteran of the US Navy. He was also a lifetime member of the NRA, and a former member of Mensa.
Ed was an avid outdoorsman most of his life that enjoyed fishing, hunting and competitive shooting sports. He attained master and expert class marksman at an early age and was lettered on the University of Dayton's shooting team in the early 50's. He began a career in tool and die and special machine building in the early 50's and retired in 1998 due to health reasons. Over the span of his career he built programs for G.M., Chrysler, Ford and other large automotive and truck production companies.
In the early 80's he began investing in real estate and built a healthy retirement for himself and his wife Judith whom he married in 1960. They spent 39 wonderful years together until her death in 1999. He also enjoyed woodworking and was actively building furniture and other items from trees that he harvested and milled start to finish from the property he so loved on Clyo road in Centerville, Ohio.
In his later years he spent his time with family, traveling and playing the stock market . Unfortunately Alzheimer' s began to take hold and in 2011 he moved to Texas to be closer to his son Thaddeus and get the assistance that he required. During this time he was able to enjoy time fishing and spending time with friends and family.
He is preceded in death by his wife, Judith Mitman, and both his parents.
Edward is survived by his son, Thaddeus Mitman (Heather); his brother Robert J. Mitman (Barbara); nieces, Samantha Palmer (Stephen), Marcia Davis (Ronnie), and Diane Schwarzman; and his grandchildren Anna Marie Mitman, Lily Jacoel, and Ella Jacoel.
A memorial service will be held on January 18, 2020 at Turcotte -Piper Mortuary at 2:00 p.m. with Reverend James Ebersole presiding. In lieu of flowers, donations can be made to the Alzheimer's Association.
Services are entrusted to Turcotte -Piper Mortuary, 205 General Cavazos Blvd. Kingsville, Texas 78363.
To send flowers to Edward's family, please visit our floral section.
Gathering of Family and Friends
Turcotte - Piper Mortuary (Kingsville])
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