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Moving Harrisburg forward might be impossible without settling present
Updated Jan 05, 2019; Posted Sep 27, 2013
Econ Forum at Harrisburg University
Gallery: Econ Forum at Harrisburg University
By Nick Malawskey | nmalawskey@pennlive.com
The forum was supposed to focus on the future of Harrisburg. And to a large extent, it did. But the reality of city's present casts a long shadow over any discussion in the capital these days.
At some point in the next three months, it is likely the Harrisburg recovery plan, whether you support it or not, is going to go into effect, essentially eliminating the city's massive debts and putting its government back on the path to some form of solvency.
How much it is able to carry that past the next four years remains unknown. But the future of the city, whether it stagnates or begins a new renaissance, will largely be in its own hands, unencumbered from debt obligations.
At its core, argues developer Ralph Vartan, is a simple equation. Of the 58,000 people who work in the city, only about 10,000 live in the city. That is further unbalanced by the fact that over the last several decades the regional population has almost doubled, while Harrisburg's has stagnated.
Vartan, one of five panelists at a PennLive/Patriot-News forum on Harrisburg's future at Harrisburg University on Thursday night, said the city needs to address that imbalance. Not only do developers in the city need to invest in the city, by rehabilitating older residential units and building new ones, but the city needs to work to attract residents.
A chief part of that, Vartan said, is taxes. City property owners pay roughly three to four times in property taxes what a similarly assessed property owner would pay in surrounding townships.
That, in turn, drives up the cost of housing in the city, making it unaffordable, he said.
Brad Jones of Harristown Development also said the city needs to look at other tools, namely tax abatements, to entice property owners and developers to invest in the city. Jones said he and other members of the city's business community have looked at other cities that are growing, and the programs they have used. Tax abatements, which defer taxes on property improvements, work, he said.
But Penn State Harrisburg professor Beverly Cigler, warned of painting with too broad a brush. While tax abatements might work, more targeted programs have been more successful, she said, noting that abatement programs are "contentious."
"These are not simple things," she said.
Several audience members questioned how Harrisburg could attract new residents – even if the city's financial recovery plan works – given the poor condition of the public school system.
State Sen. Rob Teplitz, D-Dauphin County, said he doesn't think it's a coincidence that problems at the school and the city have come to a head at the same time.
The district, he said, faces a chicken-and-egg problem. People don't want to live in the city if the schools are bad, but the schools lose tax money when people move out, exacerbating its financial problems.
The district needs to be fixed, Teplitz said, if the city is to move forward.
Rogette Harris, a PennLive/Patriot-News columnist also on the panel, stressed the importance of the schools in the city's future. Without good schools, Harris argued, children from Harrisburg would not be able to get good jobs and rise above the cycle of poverty.
But even with the forum's emphasis on moving forward, the panel couldn't escape the city's ongoing financial plight or the plan on the table to move it forward.
Harrisburg mayoral-hopeful Nevin Mindlin several times pressed the panel on the city's recovery plan, especially regarding the portion of the plan that would lease the city's parking assets. Mindlin said the terms of that lease have not been made public and could be a long-term detriment to the city.
He also said he was concerned about other aspects of the plan, which, he argued, could infringe on the city's sovereignty by including non-Harrisburg residents in boards related to economic development or infrastructure.
"It's easy to pull out things we don't like [about the plan]," Teplitz said. "But no one has shown me a comprehensive alternative."
Teplitz later commented on calls that the city should have entered bankruptcy. He said he doesn't think the results would have been any different, except that bankruptcy would have cost more and taken more time.


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Amongst all the doughnuts and latkes on offer this year, the delights of Arab food is taking centre stage in the form of A-Sham, it's meaning referring to the geographic area famous for the cuisine, the Levant area.
Food on offer is being cooked by famous chefs including Atamna-Ismaeel, winner of the lasts year's season of Master Chef, very popular now in Israel too. Dishes include Sfiha and Manakish, Levantine pizza-type dishes with dough bases as well as the more well known dishes of Kibbeh, Fattoush and Baklava.
The Festival of Festivals is simply about bringing cultures together, and to my mind there is no better way than with the sharing of foods….
Next PostIsraeli restaurants named in 'La Liste'…..


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When it started in the late 1990s, this phone retailer had to expand at a breakneck speed to catch its rivals. It now has 440 high-street stores. Providence Equity Partners bought the company from John Caudwell for a reported £779m in 2006. In February, the firm, based in Newcastle-under-Lyme, bought a call centre and Dial-a-phone, which sells mobile phones over the internet. Sales reached £782m in 2006 under chief executive Tim Whiting. In May the company was named in an Ofcom investigation into alleged mis-selling.


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Gallery of Entries 2019
National Dog Portrait Awards (NDPA) – Competition Terms & Conditions
The promoter is Click Connection Corporation LTD (CCC) whose registered office is Unit F Penfold Trading Estate, Imperial Way, Watford, Hertfordshire, WD24 4YY.
The competition is open to residents of the United Kingdom aged 18 years or over except employees of CCC and their close relatives and anyone otherwise connected with the organisation or judging of the competition.
There is a £10 entry fee to enter this competition.
All profits from this competition will be donated to worthy dog charities in the UK.
The photographers who come 1st, 2nd and 3rd in the end of year competition will decide which charities receive a donation. The donation breakdown will be as follows: 1st place choice will receive 50% of profits, 2nd place choice will receive 30% of profits and 3rd place choice will receive 20%.
Route to entry for the competition and details of how to enter can be found at dogportraitawards.co.uk or on our Facebook page https://www.facebook.com/nationaldogportraitawards/
A photograph can only be entered once. Multiple entries of the same image will be disqualified.
A pet can only be entered once. Multiple entries of the same image will be disqualified.
A person can enter as many times as they choose (with images of different pets). The £10 entry fee is payable for every entry.
The competition will open on in March 2020.
Closing date for entry will be 12 months after the first monthly winner is announced. After this date the no further entries to the competition will be permitted.
By entering customers become entitled to a Pet Portrait Experience with a participating photographer* and a viewing session to select the image to be entered into the NDPA. The Pet Portrait Session may include family members but the image for the competition must only include one dog per entry. At the viewing session there will be the opportunity to purchase additional prints and products. This will be the only opportunity to purchase from the session.
The competition is open to portraits of dogs only taken by professional photographers who have signed up to be part of the competition
Customers must attend a professional photo shoot and select their favourite image to submit to the competition at the end of the session.
To enter the participating photographer must upload a photograph to the NPDA website, populate a form which will capture data including studio name, contact name, location and email address plus customer name, pet name and type and email address and pay the £10 entry fee.
Photographers will be required to tick a box which states that they are agreeing to the T&Cs of the competition on behalf of themselves and the customer.
Once entered, both the photographer and customer will receive email confirmation of entry.
Monthly winners: In the first week of each month the entrants from the previous month will be uploaded to a Facebook album. Entrants will also be encouraged to share with their friends and family (through the share function on Facebook or within the comments on Facebook posts). The top 5 photos with the most likes on the competition by the end of the week will become finalists. Winners will be chosen by an expert panel of judges.
Yearly winners: In May 2021 the portraits that were shortlisted in each monthly competition will be combined and uploaded to Facebook. The public will then vote for their favourites, to help us find a shortlist of 10. Once the votes are in, the shortlist will be judged by our expert judging panel, who will decide on the overall winner and the two runners up.
There will be monthly prizes and a yearly prize for both the winning entry (customer) and photographer who took the photograph.
The customer monthly prize is £100 and the photographer monthly prize is a £100 Click Props voucher.
The yearly prizes are as follows:
Prize Customer Studio
1st prize £1,500 plus a £500 portrait £750 plus £750 Click Props Voucher
2nd prize £1,000 plus a £500 portrait £500 plus £500 Click Props Voucher
3rd prize £500 plus a £500 portrait £250 plus £250 Click Props Voucher
The prizes are as stated and no other alternatives will be offered. The prizes are not transferable. Prizes are subject to availability and we reserve the right to substitute any prize with another of equivalent value without giving notice.
The winner will be notified by email and/or DM on Facebook and/or letter within 28 days of the closing date. If the winner cannot be contacted or does not claim the prize within 14 days of notification, we reserve the right to withdraw the prize from the winner and select a replacement winner.
The promoter will notify the winner when and where the prize can be collected / is delivered.
Winners may be asked to return to the studio to collect their prize and may be asked to have a photograph taken accepting their prize.
The competition and these terms and conditions will be governed by English law and any disputes will be subject to the exclusive jurisdiction of the courts of England.
The winner agrees to the use of his/her name and image in any publicity material, as well as their entry. Any personal data relating to the winner or any other entrants will be used solely in accordance with current UK data protection legislation and will not be disclosed to a third party without the entrant's prior consent.
Both the photographer and entrant irrevocably consent to CCC using the submitted photograph for any advertising purpose and hereby grant a license for CCC to use the submitted image for the marketing & future promotion of the National Dog Portrait Awards. Images may be cropped or inserted into artwork and frames.
We may transfer your personal data outside the European Economic Area to our affiliated companies. Where we do so it will only be with the observance of applicable laws after applying adequate security measures
This promotion is in no way sponsored, endorsed or administered by, or associated with, Facebook, Twitter or any other Social Network. You are providing your information to CCC and not to any other party.
These Terms and Conditions will be governed and construed in accordance with the laws of England and any dispute hereunder, shall be subject to the exclusive jurisdiction of the courts of London, England
*some studios charge a session fee.
Photographer Terms & Conditions
© 2021 National Dog Portrait Awards Built For The Future By B Technologies

redpajama_set_name: RedPajamaCommonCrawl


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Justin Bieber 10-Part Docuseries Is Premiering on YouTube
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Unreasonable Consent Judgments; What Next?
Julius "Rick" Parker III
Partner | First-Party Coverage, Third-Party Coverage, Extra-Contractual
jparker@butler.legal
Overview | Publications/ Whitepapers | Julius "Rick" Parker III | Related	| Print | Share
This is one of a series of articles originally published in Mealey's Litigation Report: Insurance Bad Faith, Vol. 22, #14, page 28 (November 25, 2008). © 2008
[Editor's Note: Julius F. "Rick" Parker III is a senior associate with the law firm of Butler Weihmuller Katz Craig LLP with offices in Tampa, Tallahassee, Miami, Mobile, and Charlotte. He is an experienced trial lawyer in the firm's Third-Party Coverage and Extra-Contractual Departments. This commentary, other than the quoted material, is the author's opinion; not his law firm's, and not Mealey's Publications'. Copyright © 2008 by the author. Responses are welcome.]
The scene is all too familiar: an insured, disenchanted with its insurer's refusal to defend an action the insured believes is within coverage, decides to enter into a "consent judgment" with the plaintiff, in return for which, the plaintiff agrees only to pursue satisfaction of the "judgment" against the insurer. Some form of this type of judgment is recognized in almost every jurisdiction in the United States,1 and generally referred to by any number of monikers, generally based upon the "seminal" case in that jurisdiction. See, e.g., Coblentz v. Am. Surety Co. of New York, 416 F.2d 1059 (5th Cir. 1969). While the names may change, the agreements are unwaveringly uniform. The critical aspects of these judgments are: 1) entry into a judgment purporting to represent a reasonable settlement of the claim; 2) an agreement not to execute on the judgment against the insured; 3) an assignment of any and all claims the insured may have against the insurer under the insurance policy; and 4) an agreement to satisfy the judgment upon conclusion of the contemplated litigation against the insurer. In addition, every jurisdiction recognizes the inherent potential for fraud and/or collusion in such judgments and, accordingly requires that they be reasonable in amount and entered into in good faith.2 The question which seems to be left open in virtually every jurisdiction, however, is what happens if the judgment is found either to be unreasonable in amount or entered into in bad faith.
One of the best examples of how consent judgments are treated emanates from a Florida intermediate court. See Steil v. Fla. Physicians Ins. Reciprocal, 448 So. 2d 589 (Fla. 2d DCA 1984). Steil involved a medical malpractice case in which the insured physician entered into a consent judgment following a denial of defense by the insurer. The insurer defended the judgment on the ground that entering into the settlement violated the "no action" clause of the policy, prohibiting the insured from entering into settlements without the insurer's consent. The court held that, because the insurer wrongfully refused to defend, it could not rely on that provision of the policy. However, the court also held that the judgment was only enforceable against the insurer in the event it was entered into in good faith and was reasonable in amount.3
From that situation, the court crafted the procedure followed by virtually ever state in the nation:
… [I]n the instant case or one involving a consent judgment with a covenant not to execute, the settlement figure is more suspect. The conduct of an insured can hardly be characterized as fraudulent simply because he stipulates to a large settlement figure in order to obtain his release from liability. He has little or nothing to lose because he will never be obligated to pay. As a consequence, the settlement of liability and damages may have very little relationship to the strength of the plaintiff's claim. Due to this problem, the ordinary standard of collusion or fraud is inappropriate…. Thus, we hold that in a case such as this, a settlement may not be enforced against the carrier if it is unreasonable in amount or tainted by bad faith. Moreover, because the circumstances surrounding the settlement will be better known to the party seeking to enforce it, he should assume the burden of initially going forward with the production of evidence sufficient to make a prima facie showing of reasonableness and lack of bad faith, even though the ultimate burden of proof will rest upon the carrier.
Steil, 448 So. 2d at 592 (emphasis added). See also, Griggs v. Bertram, 443 A.2d 163 (N.J. 1982).
This procedure makes logical sense. Proving coverage is uniformly the burden of the insured. Thus, requiring the insured to come forward with evidence that the consent judgment was reasonable in amount and entered into in good faith properly belongs to the insured. However, since the insurer is usally seen to have abandoned its insured, shifting the ultimate burden of persuasion to the insurer to prove that the judgment was unreasonable in amount or entered into in bad faith also makes logical sense. This is, after all, an affirmative defense. But the opinions which address this question all simply state that such judgments can only be enforced against the insurer if they are reasonable in amount and entered into in good faith. Does this mean that the converse is also true, i.e., that consent judgments which are found to be unreasonable in amount or entered into in bad faith leave the plaintiff with no recovery whatsoever? It appears the answer to that question, at least in every state other than Iowa or Missouri, is, "Yes."
It is frankly surprising that the plaintiffs' bar has not mounted a better attack on the insurance industry in this area of the law. Considering the fact that an absolute prerequisite to prevailing on a consent judgment is that the claimant prove both that the policy covers the underlying claim and that the insurer wrongfully refused to defend its insured, a procedure which leaves a claimant with no recovery at all despite the existence of coverage seems harsh. But, as the courts have routinely recognized, the potential for collusion and fraud in such situations is so high that requiring good faith and reasonableness is the only way to keep such conduct at bay. However, courts in Iowa and Missouri have both created a third alternative, namely that the finder of fact can determine what a reasonable amount would have been and impose that figure on the non-defending insurer. A close examination of those opinions reveals the fallacy of allowing such an ex post determination to be made.
The first court to allow the fact finder to determine what amount should be enforced against the insurer was the Missouri supreme court. See Gulf Ins. Co. v. Noble Broadcast, 936 S.W.2d 810 (Mo. 1997). In Noble Broadcast, the insured, a radio station, was sued by a parade bystander who was injured when the station's van ran over her foot. After the station's insurer declined to defend the suit, the claimant and the station entered into a consent judgment in the amount of $1,000,000, complete with a covenant not to execute and an assignment of the station's rights as against the insurer. In a preemptive declaratory judgment action filed by the insurer, the trial court found the agreement unreasonable in amount and therefore refused to enforce it. On review by the supreme court, the court adopted the same test of reasonableness as the Florida court in Steil. However, for the first time, it addressed what effect a finding of unreasonableness should have:
Finally, this Court must determine an appropriate process for disposition of a case in which the settlement agreement is judged to be unreasonable. There are two possibilities. First, the court, after holding an agreement unenforceable, could release the insurer from any liability. Alternatively, the trial court, acting as the finder of fact, could determine a reasonable settlement amount for which the insurer should be held liable. This Court concludes that the second of the possibilities is the more fair. This requires that the case be remanded. The question of what constitutes a reasonable settlement in this case would have been necessarily addressed, at least in part, by implication in the determination that the settlement amount was unreasonable. The question may, however, require further argument by the parties. Whether such argument is helpful in this case is left to the sound discretion of the trial court to determine on remand before making a finding of a reasonable settlement amount for which the insurer should be held liable.
Noble Broadcast, 936 S.W.2d at 816—17.
The following day, the supreme court of Iowa addressed the same question, apparently without any knowledge that the Missouri supreme court had just addressed it. See Six v. American Family Mut. Ins. Co., 558 N.W.2d 205 (Iowa 1997). Six involved a claimant, injured in a motor vehicle accident, who took an assignment of the insured's rights as against its insurer in a typical consent judgment. The trial court submitted the issue of reasonableness to the jury, which found the judgment to be unreasonable in amount. On appeal, the claimant argued that the insurer should still be held liable for the amount of a reasonable settlement. The court sympathized with that position, explaining simply:
American Family suggests in its argument that the insurance company's liability is extinguished when a negative finding is made concerning whether a settlement is reasonable and prudent. We disagree with that contention. We are convinced that, if coverage exists, an insurer that declines to defend a claim continues to be liable to hold its insured harmless for that portion of the stipulated judgment that represents a reasonable and prudent settlement.
Six, 558 N.W.2d at 207. Thus, in the Six case, the appellate court remanded for the court to determine a reasonable settlement and enforce that amount against the insurer.
Noble Broadcast has been followed in Missouri. See, e.g., Auto Owners Ins. Co. v. Ennulat, 231 S.W.3d 297 (Ct. App. Mo. 2007). However, the Six opinion appears to be an island unto itself, which has not been followed by any subsequent court and has only been mentioned by one other reported decision. See B.M. Co. v. Avery, 2001 WL 1658197 (Guam. Terr. 2001). Due to the fact that the decisions were released within twenty-four hours of each other, their silence each as to the other is no mystery. Given the eleven years which have passed since the decisions were released, the likelihood that they might open the floodgates to collusive settlements, at least outside Missouri or Iowa, is low.
Perhaps the best explanation for why these decisions represent such a minority approach comes from the enigmatic logic on which they appear to be based. When an insurer denies coverage and refuses to defend its insured (whether based on a valid or invalid reason), the alignment of the parties changes. No longer is the defendant adversarial to the claimant. Rather, both the claimant and the defendant become adversarial to the insurer. Even the most honorable insured will be tempted to consent to a much larger number than he would if he were to remain responsible for satisfying the judgment. As the recent financial debacle has amply demonstrated, when one takes risks with other people's money, the level of care exercised tends to decrease. Just ask any shareholder in Enron, Worldcom, Tyco, Lehman Brothers or Washington Mutual.
The only other approach taken by courts to this problem is that adopted by the Court of Appeals of California in Pruyn v. Agricultural Ins. Co., 42 Cal. Reptr. 2d 295 (Ct. App. Cal. 1995). Pruyn crafted an elaborate procedure by which the claimant seeking to impose the consent judgment on the insurer must prove the amount of a settlement which the insured would have agreed to had he remained personally liable for the judgment. While that procedure is superficially appealing, it suffers from the same lack of true adversarial conflict which any consent judgment lacks. As the Texas supreme court explained when it critized the Pruyn decision:
The procedure required by Pruyn to enforce an agreed judgment against an insurer is, to be generous, complicated. Its goal is to determine what judgment would have been rendered against an insured, or what settlement he would have agreed to, had he remained personally liable to plaintiff. Put another way, the inquiry is what result would plaintiff and defendant have reached had they remained fully adversarial to the end. The validity of the holdings in Griggs, 443 A.2d at 163 (New Jersey), Red Giant, 528 N.W.2d at 524 (Iowa)4, and other cases that uphold prejudgment assignments of claims against insurers is based on the premise that this inquiry is answerable. We think it is not, and that Pruyn shows why. It is one thing to say that a defendant's liability must be determined as if he had not settled with the plaintiff; it is quite another thing to do it. We think Pruyn's listing of factors to be considered in the process of assessing a defendant's liability after he has settled shows that the undertaking is virtually impossible. Once the parties have changed positions, their views are altered, and it is very difficult to determine what might have been.
State Farm Fire & Cas. Co. v. Gandy, 925 S.W.2d 696 (Tex. 1996)(footnote added)(emphasis in original). Thus, in Texas, even if the insurer wrongfully refuses to defend its insured, the only liability which can be imposed on the insurer is that which is reached after a truly adversarial adjudication of liability. Given the recognition of the parties' altered motives after a denial of defense, this may be the only approach that makes true logical sense.
The Texas approach recognizes that, even if the parties litigate the case, in the event there is any agreement by the claimant not to seek satisfaction of the judgment from the insured, the insured is insulated from further liability and therefore has no incentive to defend the case vigorously. Thus, the possibility of a sham trial is as real as the possibility of a sham settlement. In short, it is not the fact that the parties have resolved the claim between themselves which raises the spectre of fraud and collusion; rather it is the fact that once the claimant agrees to pursue only the insurer, the insured loses all incentive to defend. In the event the liability proves to be catastrophic and the insurer has indeed wrongfully refused to defend, in most cases the judgment will be recoverable in full from the insurer under bad faith principles.
It is therefore clear that the Iowa and Missouri approaches to consent judgments cannot be justified logically. If the insured and the claimant know that the only consequence for colluding to create an inflated judgment to be recovered from the insurer is the possibility of a remittitur of the judgment to a "reasonable" sum, there will be no limit on the amount to which insureds will confess judgment. The risk of agreeing to too little would be far more costly than the risk of agreeing to too much. Thus, in every case, "settlement" of the case accomplishes nothing. It is merely one more step to recovering from the insurer, something which will require presentation of all the evidence necessary to prove the case in the first instance against the defendant. If the courts' goals were to encourage settlement (and they are notably silent as to the true rationale of their decisions), they failed miserably. As the old adage goes, the road to Hell is paved with good intentions.
The final logical misstep taken by those courts is the failure to recognize that a trial in which the insurer is the defendant rather than the insured runs afoul of the rule applicable in most states that the fact finder may not be apprised of the existence of insurance available to satisfy a judgment against the defendant. The logic behind that rule is obvious: it encourages the fact finder to increase the award to the plaintiff because it is not the defendant's assets which are at risk. True, an insurer which actually abandons its insured in the face of a clear duty to defend should arguably lose that protection, but the potential for an inflated recovery remains. If the fact-finder is purporting to decide what a reasonable judgment should have been, its knowledge that the insurer is paying the judgment will unquestionably affect its judgment whether consciously or sub-consciously.
The conclusion is clear: where a jury finds that the insured and a claimant conspired to enter into an inflated or unreasonable judgment to recover from the insurer, the judgment should not be enforced, period. Insurers have long been forced to exercise the utmost "good faith" toward their insureds. Imposing that reciprocal obligation upon insureds makes perfect sense. As Benjamin Franklin said over two hundred years ago, "There is no kind of dishonesty into which otherwise good people more easily and frequently fall than that of defrauding the government." Had he lived to see the proliferation of insurance, surely he would have said the same of that institution.
A small minority of jurisdictions refuse to recognize such agreements, instead requiring that the claimant and insured actually litigate the claim to judgment. See, e.g., State Farm Fire & Cas. Co. v. Gandy, 925 S.W.2d 696 (Tex. 1996), and cases cited therein.
This article does not address the issue of whether consent judgments which exceed the limits of the insurance policy may be enforced against the insurer as a consequence of its failure to defend. The approaches to that question are as varied as the number of jurisdictions considering it.
Many of the opinions use the phrases "without fraud or collusion" and "reasonable in amount and entered into in good faith" interchangeably. There does not appear to be any meaningful distinction made between the two in the decisions.
The Red Giant decision formed the basis for the Noble Broadcast opinion.


text:
On the heels
of a big break
Lauren Patel, the Bolton-based newcomer, talks about starring in the film version of hit musical Everyone's Talking About Jamie
27 Sep 2021 No comments
Home Features On the heels of a big break
As Lauren Patel procrastinated on her drama homework, she stumbled upon an opportunity that would change her life forever. Then an A-level student living at home with her parents in Horwich, Bolton, Patel, an acting newbie with no formal training, spotted a casting call for South Asian teenage girls to attend an audition 20 miles down the road in Manchester.
"I'm always going to love the North. And even if I'm further away, I'll still be here every weekend."
Patel auditioned for the role of Pritti Pasha in the film version of hit stage musical Everybody's Talking About Jamie for "experience", if nothing else. She never imagined she'd get the role.
Fast-forward two years and Patel has just returned from a tour across the United States promoting the film ahead of its launch on Amazon Prime on 17 September.
"It all happened really quickly," says Patel. "I think I sent my first tape in on 5 May [2019] and I found that I got the job three weeks later, and we started filming the following month. It was really fast."
For those who haven't yet heard of the Jamie everybody's talking about, the musical was inspired by the 2011 BBC Three documentary Jamie: Drag Queen At 16, which followed Jamie Campbell, then a year 11 boy who decided to wear a dress to his school prom, and the fall-out when his teachers tried to ban him.
The stage show premiered at Sheffield's Crucible Theatre in 2017, to critical acclaim, before transferring to the West End, opening up conversations about gender, identity, prejudice, acceptance and diversity to audiences across the country when the show went on tour.
This year those conversations, and the musical's catchy numbers, are set to reach a global audience with the release of the film adaptation.
The film follows Jamie New (Max Harwood), a teenager growing up in Parson Cross, a working-class suburb of Sheffield.
While his classmates mull over their futures, Jamie secretly dreams of bright lights, lipstick and a career as a drag queen, one day finding fame on the stages of San Francisco.
His best friend Pritti (Patel) and his loving mum (Sarah Lancashire) shower him with support, while drag legend Miss Loco Chanelle (Richard E Grant) mentors him towards his debut stage performance at a drag night.
But it's not all rainbows for Jamie as his unsupportive dad (Ralph Ineson), an uninspiring career adviser (Sharon Horgan) and school bullies try to rain on his dreams. In rousing and uplifting musical numbers, Jamie and his community inspire one another to be more accepting.
"Our director [Jonathan Butterell] always says that this is not a coming out story," says Patel. "Jamie is already out as a gay boy so the film isn't really about that.
"It's about how he has decided that he wants to be a drag queen, and he is in Parson Cross in Sheffield, where you don't get a lot of drag queens, and it's about him navigating that decision and how the people in his life react to him just wanting to live as himself."
When Jamie's mum Margaret buys him his first pair of red, sparkly sky-high stilettos for his 16th birthday – proudly telling the shop assistant they are for her son, not her daughter – Pritti, a studious, hijab-wearing Muslim who dreams of becoming a doctor, is the first person Jamie shows them to.
"I think their friendship is just so pure and so wholesome," says Patel. "Pritti is very much by the book, she's very sensible, she's got her head screwed on, and she knows exactly where she wants to be. She is absolutely a Hermione. She's terrified of getting into trouble, which I can definitely relate to.
"I love that she and Jamie are just polar opposites of each other. They're just from two completely different worlds, but they both love each other so much and they both try so hard to understand each other."
Patel hopes her character's non-judgmental questions about Jamie's desire to become a drag performer open him up to a range of audiences who may not have encountered a character like Jamie before.
"I'm sure there are audience members who don't understand Jamie and have never seen someone like Jamie or known someone like Jamie," she says. "So I think it's really lovely to have Pritti there to ask the questions and be asking them because she loves him and because she wants to know him properly."
For Patel, acting isn't something that runs in the family. She comes from a family of pharmacists and teachers, but she credits her initial interest in acting to her older brother, now a lighting technician, who often talked her into putting on shows with him when they were young.
Patel attended Carol Godby's Theatre Workshop in Bury, joining alumni Helen Flanagan, Georgia May Foote and Jennie McAlpine with her early acting success.
Patel was in the process of applying for drama schools when she was selected for the role of Pritti, but she's put those plans on hold for now.
"I decided not to go to drama school, because I'm just going to enjoy this time with the film coming out," she says. "I've got a wonderful agent and I'm auditioning for lots of fun stuff, so I'm just going to do this and see where it takes me. And if I want to go back to drama school, then I can go in the future."
Patel knows her acting career may eventually take her all over the country and beyond, but she insists her heart will always be in the North.
"I'm always going to be a northern gal," she says. "I'm always going to love the North. And even if I'm further away, I'll still be here every weekend."
Patel's ambition is to become a versatile actor who can do "everything", transforming with every role she takes on. But for now she is fully immersed in the world of Jamie and Pritti.
"I feel like Everybody's Talking About Jamie is just a celebration of LGBT people and of anybody who feels different or marginalised. And I feel like that's going to resonate with a lot of people."
Everybody's Talking About Jamie is available on Prime Video
A sweet and sour wind up
by Dan Whitehead
Film review: Brian and Charles
by Christian Lisseman
Film review: Hit the Road
Popcorn politics
Interact: Responses to On the heels of a big break


text:
Q: Oculus basic Makefile I am trying to create a makefile for compiling an Oculus Rift based program. I have the object files of the Oculus Rift library in a directory and the header files in another directory. Here's my makefile:
#####
# MAIN PROGRAM
#####

CXX = g++
CXXFLAGS =

DEPS_OVR = ($wildcard LibOVR/Src/*.h) ($wildcard LibOVR/Src/CAPI/*.h) ($wildcard LibOVR/Src/Util/*.h)
DEPS =

OBJ_OVR = ($wildcard LibOVR/Obj/Linux/Release/x86_64/*.o)
OBJ = main.o
EXE = BenchOculus

LIBS =

#####
# RULES
#####

all: $(EXE)

%.o: %.cpp $(DEPS_OVR) $(DEPS)
    $(CXX) -Wall -c -o $@ $< $(CXXFLAGS) $(LIBS)

$(EXE): $(OBJ) $(OBJ_OVR)
    $(CXX) -o $@ $(OBJ) $(OBJ_OVR) $(CXXFLAGS) $(LIBS)
    chmod +x $@

clean:
    rm -f $(OBJ)
    rm -f $(EXE)

When I run make, I have the following error:
make: *** No rule to make target `(ildcard', needed by `BenchOculus'. Stop.

I think the problem may reside in DEPS_OVR and OBJ_OVR but I can't figure how to fix this.

A: Look at the error message, and ask yourself why it's complaining about a target (ildcard, when what you wrote was ($wildcard ....  That tells you, to within a character or so, where your error is.  Then go back and check the documentation for the wildcard function in GNU Make.

redpajama_set_name: RedPajamaStackExchange


Saab earned the reputation as having the highest levels of owner loyalty in the US auto market. Because of this, Saab owners were especially disappointed when the brand withdrew from American dealerships. As its relationship with GM ended in 2010, Saabs are now only available overseas. Still, owners of these premium Swiss cars will keep driving their Saabs for many more years, and will need affordably, high quality replacement parts. LKQ Online offers the nation's largest inventory of reclaimed and recycled Saab parts and accessories, most including a warranty.


text:
The Pretenders. Not Pretenders.
Friday, January 27, 2023 - 08:00 AM
Posted by: Karl
THE WEEKEND STARTS HERE:
....wtih THE PRETENDERS! Circa 1981, via one of Pate's two fab drummers, Jon Hahn. Your setlist includes "The Wait," "The Adultress," "Message of Love/Louie Louie," "Talk of the Town," "English Roses," "Birds of Paradise," "Kid," "Stop Your Sobbing," "Private Life," "Day After Day," "Jealous Dogs, " Up the Neck," "Tattooed Love Boys," "Bad Boys Get Spanked," "Precious," and "Brass In Pocket/MysteryAchievement."
ALL SONGS CONSIDERED features boygenius, The National, and more.
CURTIS MAYFIELD left an indelible mark on Chicago.
THE BOO RADLEYS share "Seeker."
MICK JAGGER announced plans to release his own limited-edition line of harmonicas.
THE BEACH BOYS are getting an all-star tribute concert after the Grammys.
WEDNESDAY (the band), profiled by Pitchfork.
ROD STEWART called a phone-in segment on Sky News to offer to pay for people to have hospital scans, amid the rising number of people on NHS waiting lists.
SEX PISTOLS bassist Glen Matlock has reflected on his time touring with Iggy Pop, admitting that he got "got a bit fed up" of seeing the singer's penis.
RICK ASTLEY is suing rapper Yung Gravy for imitating his "Never Gonna Give You Up" vocal on the single "Betty (Get Money)."
DEAN DAUGHTRY, keyboardist and co-founder of Southern soft-rock act Atlanta Rhythm Section, has died at 76.
PAM ANDERSON says she saw Jack Nicholson in a threesome at the Playboy mansion.
SHAZAM! FURY OF THE GODS has a second trailer online.
TOBEY MAGUIRE & ANDREW GARFIELD on making Spider-Man: No Way Home.
MARVEL's THUNDERBOLTS adds Ayo Edebiri to a cast led by Florence Pugh, Sebastian Stan, and David Harbour.
BRITNEY SPEARS feels "gaslit and bullied" by fans who called the cops after she deactivated her Instagram account.
MIA GOTH, on the Oscars avoiding horror.
THE OSCARS: Could a sequel win Best Picture this year?
THE RAZZIES: Sucking more than ever.
AN ARMADILLO plays peek-a-boo.
A DOGGO enjoys a roof-clearing.
STOWAWAY GECKO travels 3000 miles from Egypt to England.
Someone needs to work on that 10-day forecast.
Thursday, January 26, 2023 - 08:00 AM
999: "Homicide." You better believe it.
ALEJANDRO ESCOVEDO visits KUTX. Scott McCaughey is there too.
MUDHONEY shares "Almost Everything."
DEPECHE MODE's David Gahan covers The Raveonettes' "Chains."
PATRICK WOLF returns with "Nowhere Game."
PAUL McCARTNEY has announced a new photography book containing unseen pictures from Beatlemania.
THE OLD 97s are anything but jagged afer 30 years.
ANTHONY "TOP" TOPHAM, the founding guitarist of The Yardbirds, has died at 75.
THE NUMBER ONES looks at Ne-Yo's sad R&B jam "So Sick."
JEREMY RENNER was trying to stop his snow-removal tractor from sliding and hitting his grown nephew when he was pulled under the vehicle and crushed on New Year's Day.
ALEC BALDWIN helped talk himself into homicide charges.
RICK & MORTY co-creator Justin Roiland is also being dumped by Hulu after accusations of felony domestic violence.
GHOSTBUSTERS: Bill Murray, Sigourney Weaver and Dan Ackroyd are confirmed for the upcoming Afterlife sequel.
TITANS & DOOM PATROL are the latest DC properties ending under the new DCU regime.
BARBARIAN filmmaker Zach Cregger's new project is a hot item.
DAISY JONES & THE SIX has a teaser trailer online.
LANCE KERWIN, a former child star known for television projects James at 15, Salem's Lot and The Loneliest Runner, has died. He was 62.
A CLEVER PUP makes his escape.
CATS & DOGS, living together...
THE FLOOR is lava.
On such a winter's day.
Wednesday, January 25, 2023 - 08:00 AM
THE MAMAS & PAPAS lip-synced "California Dreaming" really well on Hullaballoo, but not showing you the go-go dancers, who are worth the wait.
SYLVAN ESSO & JAN WASNER play CBS This Morning.
TOM WAITS on David Letterman, 1983-2015.
GRUFF RHYS shares "Layer Upon Layer" and "Orea."
NICKEL CREEK returns with "Strangers."
NICK CAVE, on joy, optimism and the state of the world.
KING TUFF: The Aquarium Drunkard Interview.
JUSTIN BIEBER sold his catalog to Hipgnosis for 200MM.
PANIC! AT THE DISCO is dunzo.
THE OSCARS NOMINATIONS went to these folks. Congrats. As usual, there were snubs and surprises.
RICK & MORTY dumped co-creator Justin Roiland after he was charged with felony domestic abuse.
FRANCIS FORD COPPOLA is adding Giancarlo Esposito to the cast of Megalopolis in mid-production.
SEAN PENN & ROBIN WRIGHT: Spotted together, but not together together.
MARVEL: Kevin Fiege doesn't think audiences will tire of comic book movies.
JAMES GUNN may cast MCU actors in his DCU.
ANTONIO BANDERAS said his 2017 heart attack brought him clarity.
MADONNA's self-directed biopic is dunzo.
GUY PEARCE threw shade all over Cate Blanchett, then backtracked.
YOU GOBBLE, we gobble back.
A DOGGO, window shopping.
BABY RAMS, practicing.
Vague recollections of a strange glowing object in the sky.
Tuesday, January 24, 2023 - 08:00 AM
RICHARD THOMPSON: The culmination of 1000 Years Of Popular Music.
MARGO PRICE, Live at Rockwood Music Hall.
DOS SANTOS plays World Cafe.
IAN HUNTER shares "Bed of Roses."
BOYGENIUS: The book club.
DAVID CROSBY, essayed at Vulture and Pitchfork.
TICKETMASTER & SEATGEEK EXECS go to Washington.
THE NUMBER ONES looks at James Blunt's caterwauling broken-heart ballad "You're Beautiful."
ALEC BALDWIN will not be charged with the shooting of Rust director Joel Souza.
ZACK SNYDER's Rebel Moon will be two movies, because Zack Snyder.
PAM ANDERSON accuses TIM ALLEN of flashing her, which he denies.
MAYA RUDOLPH is becoming the spox for M & Ms in an elaborate Superbowl PR stunt.
ANNE HATHAWAY is thrilled about Princess Diaries 3 but has not signed on to the project.
DUNGEONS & DRAGONS: HONOR AMONG THIEVES has a trailer online.
GERARD BUTLER Knows Exactly Who He Is.
A CHEETAH & A TORTOISE are best friends.
KANGAROOS: Fighting and swimming.
SORRY KIDS, didn't mean to do that.
Shakespeare in a bad Scottish accent.
Monday, January 23, 2023 - 08:00 AM
DOBIE GRAY is in with "The In Crowd."
NEW RELEASES: Paste will point you to Guided by Voices, John Cale, Mac DeMarco, and more.
BILL'S INDIE BASEMENT has the Bad Ends, the Murder Capital, and more.
BRAINIAC: The band's surviving members release the lost The Predator Nominate EP.
DOLLY PARTON, Belinda Carlisle, Cyndi Lauper, Gloria Estefan, and Debbie Harry team up on "Gonna Be You." It's about Tom Brady?
LUCIUS performs "The Man I'll Never Find" on "The Kelly Clarkson Show."
DAVID CRSBY: See video from his last shows.
NEIL YOUNG paid tribute to David Crosby.So did the Byrds' Chris Hillman.
DOC POMUS, profiled at Paste.
STEVE ALBINI takes listeners on atour of his studio.
JOURNEY: Still more legal drama.
BLACK EYED PEAS is suing a pooping unicorn.
THE NUMBER ONES looks at "Check On It," the tossed-off between-albums number that Beyonce recorded with Slim Thug, Bun B, and Swizz Beatz.
WEEKEND BOX OFFICE: Avatar 2's staying power is evident with another week atop the chart, with 19.7MM on a 40 percent drop.It's approaching 600MM dmoestic and 2B worildwide. Almost as impressive is Puss in Boots 2 placing with 11.5MM on a 21 percent drop in its fifth frame, almost as good as its debut. And Missing shows with a 9.3MM debut against a 7MM budget.
JEREMY RENNER broke over 30 bones in that snowplow accident.
JASON MOMOA remains Aquaman.
TIME'S UP: The now-embattled anti-harassment organization founded with fanfare during the early days of the MeToo reckoning against sexual misconduct — is ceasing operations, at least in its current form.
RILEY KEOUGH and her husband, Ben Smith-Petersen, have welcomed a daughter.
JEAN SMART shares the soundtrack of her life.
NIKKI FINKE: The Last Days of Hollywood's Most Reviled Reporter.
BUZZ ALDRIN celebrated his 93rd birthday by marrying his longtime girlfriend Anca Faur.
SMOL DOG, big ball.
LIONS fight while eating a water buffalo, then it walks off.
TOADZILLA: A Giant cane toad is found in Australia.
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PROTECT-A-BED® FABRIC SANITIZER is a safe and effective way to kill bacteria and protect surfaces for up to 30 days. It eliminates odour-causing bacteria and inhibits mould and mildew. Spraying PROTECT-A-BED® FABRIC SANITIZER will destroy bacteria on the treated surface. Allowing the spray to dry will also coat the surface with a microscopic protective layer for continuous protection. It is safe for use around children, pets and plants.


text:
Q: Parsing XML using JQuery and PhoneGap I know my question might be asked previously couple times. I tried many solution but it didn't work for me.
I am trying to build a mobile app using JQuery and PhoneGap that get data from XML file .
This is my XML file:
<book>
<chapter id="1">
<title>title 1</title>
<question id="1.1"> Question 1 </question>
<answer id="1.1.1"> answer for q1 </answer>
</chapter>

<chapter id="2">
<title>title 2</title>
<question id="2.1"> Question 2 </question>
<answer id="1.2.1"> answer for q2 </answer>
</chapter>
</book>

and this is my JavaScript:
$(document).bind("pageshow", function() {

$.ajax({
        type: 'GET',
        url:'Book.xml',
        datatype:'xml',
        success:function(xml){
            alert("success");
        }
    });
});

If I have a function the success alert never shown, however if the alert is shown in this code
   $(document).bind("pageshow", function() {
$.ajax({
        type: 'GET',
        url:'Book.xml',
        datatype:'xml',
        success: alert("success");

    });
});

I am stuck here as I can't add any further code.

A: The second version is simply calling alert and assigning the return value (undefined) to the success member. That is why you see the alert message box.
The first version could be failing for any number of reasons. First you should confirm that you are in fact receiving data back (e.g. by using the developer tools in the browser)


text:
10th World Summit on Immunology & Immunotherapy August 30-31, 2022 London, UK
Organizing Committee Submit Abstract Register Now
Recommended Global Medical Conferences
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HORMONES 2023, Netherlands
Blockchain-2023, Netherlands
Theme: "Breakthrough insights of Immunology and Immunotherapy"
World Immunology 2022
We invite participants from all across the globe to attend "10th World Summit on Immunology & Immunotherapy" during August 30-31, 2022 webinar, which includes prompt keynote presentations, poster presentations and oral talks.
The main theme of the conference is "Breakthrough insights of Immunology and Immunotherapy". This conference plays a Global platform for biomedical companies, start-ups, clinical research divisions, professionals, specialists, consultants, doctors, scholars and students to frame new relationship and strengthen the Knowledge. This is a great opportunity for all the delegates from Universities and Institutes to interact with the top-grade world-class Scientists. The particular participants can confirm their participation through Registrations. To explore More Regarding the Recent Researches in the field of Immunology attend World Immunology 2022.
Why to attend this conference?
Be the first to showcase your research, innovations and the brand to attain competitive advantages. Meet your target audience and explore your knowledge about the research work.
Build your professional network.
Know about the latest research.
Improve your presentation and communication skills.
Get response on an early version of your latest research work.
Acquire knowledge beyond your field or interest.
Get opportunity to meet people.
Track 1: Clinical Immunology and Allergy:
Clinical immunology/allergy specialists in Australia and New Zealand diagnose, treat and manage patients with allergy, primary immune deficiencies and other system disorders. If they need trained in Immuno-pathology, they're going to even be fellows of the Royal College of Pathologists of Australasia (FRCPA). Clinical immunology/allergy specialists are specifically trained to diagnose, treat and manage patients with allergy and other immune diseases. They add hospitals and personal practice, as listed on the ASCIA and a referral from a GP (GP) is required. The body's system involves a posh network of organs, cells and proteins located throughout the body. The system defends against infections from germs (such as bacteria, viruses, fungi, parasites), and other invaders (such as cancer cells), whilst protecting the body's own cells.
Track 2: Immune Disorders:
Immune system disorders cause abnormally low activity or over activity of the system. Treatment for autoimmune diseases generally focuses on reducing system activity. The blood cells within the body's system help protect against harmful substances. These substances contain antigens. The system produces antibodies against these antigens that enable it to destroy these harmful substances. once you have an auto-immunedisease, your system doesn't distinguish between healthy tissue and potentially harmful antigens. As a result, the body triggers a reaction that destroys normal tissues. The exact explanation for autoimmune disorders is unknown. One theory is that some microorganisms (such as bacteria or viruses) or drugs may trigger changes that confuse the system. this might happen more often in folks that have genes that make them more vulnerable to autoimmune disorders.
Track 3: Infection and Allergy:
Allergens cause the assembly of immunoglobulin E (IgE), an antibody that everyone folks have in small amounts. Allergic persons, however, produce IgE in abnormally quantities. Normally, this antibody is vital in protecting us from parasites, but not from other allergens. These chemicals, in turn, cause inflammation and thus the standard allergic symptoms. this is often how the system becomes misguided and primed to cause an allergy when stimulated by an allergen. Hey Fever is that the commonest of the allergic diseases and refers to seasonal nasal symptoms that are thanks to pollens. Year round or perennial rhinitis is usually because of indoor allergens, like dust mites or molds. Symptoms result from the inflammation of the tissues that line the within of the nose (mucus lining or membranes) after allergens are inhaled. Adjacent areas, just like the ears, sinuses, and throat can also be involved.
Track 4: Microbiology and Cellular Immunology:
Microbiology is that the study of microscopic organisms, those being unicellular (single cell), multicellular (cell colony), or a cellular (lacking cells). As an application of microbiology, medical microbiology is typically introduced with medical principles of immunology as microbiology and immunology. The system has been divided into a more primitive innate system and, in vertebrates, an acquired or adaptive system. The system has the potential of self and non-self-recognition. An antigen could also be a substance that ignites the immune response. The cells involved in recognizing the antigen are Lymphocytes. Once they recognize, they secrete antibodies. Antibodies are proteins that neutralize the disease-causing microorganisms. Antibodies don't directly kill pathogens, but instead identify antigens as targets for destruction by other immune cells like phagocytes or NK cells.
Track 5: Autoimmunity and Inflammation:
Treatment for autoimmune diseases generally focuses on reducing system activity. Inflammation may be a process by which the body's white blood cells and substances they produce protect us from infection with foreign organisms, like bacteria and viruses. However, in some diseases, like arthritis, the body's defense system -- the system -- triggers an inflammatory response when there are not any foreign invaders to repel. In these diseases, called autoimmune diseases, the body's normally protective system causes damage to its own tissues. Inflammations are often classified as either acute or chronic. Prolonged inflammation, mentioned as chronic inflammation, results in a progressive shift within the sort of cells present at things of inflammation, like mononuclear cells. Conversely, there's pathology where microbial invasion doesn't cause the classic inflammatory response – as an example, parasitosis or eosinophilia. Inflammation isn't a synonym for infection. Inflammation on the other hand describes purely the body's immunovascular response, no matter the cause could even be. But thanks to how often the two are correlated, words ending within the suffix -itis (which refers to inflammation) are sometimes informally described as concerning infection.
Track 6: Immunology and Diseases:
Immunodeficiency's are often caused during a number of the way, including age, obesity, and alcoholism. In developing countries, malnutrition may be a common cause. AIDS is an example of an acquired immunodeficiency. In some cases, immunodeficiency's are often inherited, as an example, in chronic granulomatous disease where phagocytes don't function properly. The system must be able to tell self from non-self. An antigen is any substance which can spark an immune response. In many cases, an antigen could also be a bacterium, fungus, virus, toxin, or foreign body. But it can also be one of our own cells that's faulty or dead. Initially, a spread of cell types works together to acknowledge the antigen as an invader. As infection and immune reaction are so intricately intertwined, this book is effective reading to anyone interested by infectious diseases in humans. Maybe within the future prions will need to be included as a replacement sort of infective agent whose rise we are just witnessing. (Information on prion pathology remains hotly debated, and data on routes of transmission and system reactions are still scarce.)
Track 7: Immunogenetics:
Immunity is that the power of a personal to acknowledge the "self" molecules that structure one's own body and to differentiate them from such "nonself" molecules as those found in infectious microorganisms and toxins. This process features a prominent genetic component. Knowledge of the genetic and molecular basis of the mammalian system has increased in parallel with the explosive advances made in vegetative cell and genetics. There are two major components of the system, both originating from an equivalent precursor "stem" cell. The bursa component provides B lymphocytes, a category of white blood cells that, when appropriately stimulated, differentiate into plasma cells. These latter cells produce circulating soluble proteins called antibodies or immunoglobulins. Antibodies are produced in response to substances called antigens, most of which are foreign proteins or polysaccharides. An antibody molecule can recognize a selected antigen, combine with it, and initiate its destruction. This so-called humoral immunity is accomplished through a sophisticated series of interactions with other molecules and cells; a number of these interactions are mediated by another group of lymphocytes, the T lymphocytes, which are derived from the thymus.
Track 8: Cancer Immunology and Immunotherapy:
Cellular immunity depends mainly on the power of DCs to require up and process antigens within the peripheral blood and tissues. In cancer, this process isn't robust enough to supply meaningful antitumor responses. Monoclonal antibodies are proteins produced by B-cells that bind to a selected antigen. they're currently one among the foremost successful sorts of cancer immunotherapy. we should always differentiate between monoclonal antibody-based cancer therapy and monoclonal antibody-based immunotherapy of cancer. this will be illustrated by the differences in their mechanisms of action. Anti-neoplastic agents like bevacizumab block ligand–receptor interaction, thereby affecting growth or survival pathways, where monoclonal antibodies like rituximab cause antibody-dependent cellular cytotoxicity and complement-mediated cytotoxicity. Antibodies also are getting used to reinforce the strength of the immune reaction. As mentioned earlier, activated T-cells and tumour cells express CTLA4 receptors as an immune-suppressive mechanism to guard against autoimmunity and to flee elimination. Antagonism of those receptors has shown antitumor benefit. for instance, ipilimumab inhibits the receptor CTLA4, permitting persistent T-cell activation. Similarly, PD1 is expressed on activated T- and B-cells and when sure to by PD1 ligand they become deactivated (10-12). Tumour cells tend to overexpress PD1 ligands, allowing them to deactivate this response.
Track 9: Neonatal and Pediatric Immunology:
Track 10: Veterinary Allergology:
Allergic diseases are frequently observed in veterinary practice. With increasing standards in veterinary care, intradermal testing and allergen immunotherapy were introduced to small animal practice within the mid‐nineteen hundred; later, serum testing for allergen‐specific IgE was developed for dogs, cats and horses. Although atopic asthma is rare within the dog and not much is known with regard to rhinitis, atopic eczema may be a frequently encountered disease in small animal practice and attention of research in veterinary dermatology. Horses develop skin and respiratory disorders that are attributed to allergy. While recurrent airway obstruction, previously called 'heaves', has many similarities to human asthma, the simplest understood allergic disease in horses is insect‐bite hypersensitivity. an efficient treatment for this disease still remains elusive. The dog, cat and horse, although data on the main allergens relevant for dogs, cats and horses are limited. In contrast to environmental allergens, studies evaluating food allergens in medicine are rare. Food rechallenges after elimination diets are notoriously difficult and not performed during a double‐blinded fashion.
Track 11: Asthma and COPD:
Asthma could also be a chronic inflammatory disorder of the airways. The chronic inflammation is said to airway hyper responsiveness that leads to recurrent episodes of wheezing, breathlessness, chest tightness and coughing, particularly in the dark or in early morning. These episodes are usually associated with widespread, but variable, airflow obstruction within lung that's often reversible either spontaneously or with the treatment. Asthma could also be a significant global unhealthiness with an estimated 300 million affected individuals. People of all ages are suffering from this illness that, when uncontrolled, can place severe limits on lifestyle and is usually fatal. Clinical manifestations of asthma are often controlled with appropriate treatment. COPD is one among the main causes of chronic morbidity and mortality worldwide. many of us suffer from this disease for years and die prematurely from its complications. COPD is that the fourth leading explanation for death within the planet, and further increases in its prevalence and mortality are often predicted within the approaching decades. COPD may be a pulmonary disease with some significant extra pulmonary effects which will contribute to the severity in individual patient. Its pulmonary component is characterized by airflow limitation that's not fully reversible. Cigarette smoking is that the most ordinarily encountered risk factor for COPD, although in many countries, pollution resulting from the burning of wood and other biomasses fuels has also been identified as a COPD risk factor.
Track 12: Food Allergies and Infections:
An allergy is an abnormal immune response to food. The symptoms of the allergy may range from mild to severe. they'll include itchiness, swelling of the tongue, vomiting, diarrhoea, hives, trouble breathing, or low sign. This typically occurs within minutes to several hours of exposure. When the symptoms are severe, it's mentioned as anaphylaxis. A food intolerance and gastrointestinal disorder are separate conditions, undue to an immune reaction. The common allergies vary counting on the country. Risk factors include a case history of allergies, vitamin D deficiency, obesity, and high levels of cleanliness. Allergies occur when immunoglobulin E (IgE), a neighborhood of the body's system, binds to food molecules. A protein within the food is usually the matter. This triggers the discharge of inflammatory chemicals like histamine. Diagnosis is usually supported a medical history, elimination diet, skin prick test, blood tests for food-specific IgE antibodies, or oral food challenge. allergy is an abnormal response to a food triggered by your body's system. In adults, the foods that the bulk often trigger allergies include fish, shellfish, peanuts, and tree nuts, like walnuts. Problem foods for kids can include eggs, milk, peanuts, tree nuts, soy, and wheat. The allergy could also be mild. In rare cases it can cause a severe reaction called anaphylaxis.
Track 13: Vaccines and Immunization:
Vaccines are wont to boost your system and stop serious, life-threatening diseases. Vaccines help protect against many diseases that want to be far more common. Many of these infections can cause serious or life-threatening illnesses and will cause life-long health problems. due to vaccines, many of these illnesses are now rare. this is often a natural thanks to affect infectious diseases. Live virus vaccines use the weakened (attenuated) sort of the virus. The measles, mumps, and rubella (MMR) vaccine and thus the varicella (chickenpox) vaccine are examples. Killed (inactivated) vaccines are made from a protein or other small pieces taken from a plague or bacteria. The pertussis (pertussis) vaccine is an example. Toxoid vaccines contain a toxin or chemical made by the bacteria or virus. they create you immune to the harmful effects of the infection, instead of to the infection itself. Examples are the diphtheria and tetanus vaccines. Biosynthetic vaccines contain manmade substances that are very almost like pieces of the virus or bacteria. The hepatitis B vaccine is an example.
Track 14: Diagnostic Immunology:
Immunology is that the study of molecules, cells, and organs that structure the system. The function of the system is to acknowledge self-antigens from non-self-antigens and defend the body against non-self (foreign) agents. Through specific and non-specific defence mechanisms, the body's system is during a position to react to microbial pathogens and protect against disease. If the inflammation remains aggravated, antibody-mediated immune reaction is activated and differing kinds of immune cells are engaged to resolve the disease. The system consists of cellular and humoral elements. to assist within the diagnosis of disease caused by infectious microorganisms, immunoassays are developed. Serum is then isolated and thus the concentration of antibodies is measured through various methods. Most assays believe the formation of giant immune complexes when an antibody binds to a specific antigen which can be detected in solution or in gels. Recent methods employ pure antibodies or antigens that are immobilized on a platform which are often measured using an indicator molecule. These methods provide high sensitivity and specificity and have become standard techniques in diagnostic immunology.
Track 15: Gastrointestinal immunology and allergy:
Immune dysfunction in allergic diseases like asthma and atrophy seems to be associated with differences within the function and composition of the gut micro biome. The gut micro biome constitutes a highly complex ecosystem which incorporates eukaryotic fungi, viruses, and a few Achaea, although bacteria are the foremost prominent components. Its composition is usually formed during the primary 3 years of life; however, recent work has suggested that its colonization may begin in utero , contrary to the widely held dogma of the foetus as a sterile environment. Despite its early formation, its composition is very dynamic and hooked in to host-associated factors like age, diet, and environmental conditions with the main phyla being Actinobacteria, Bacteroidetes, Firmicutes, and Proteobacteria. The gut micro biome isn't homogeneous throughout the GIT, showing higher diversity within the mouth and intestine, and lower diversity within the stomach, mainly due to the acid environment. Symptoms caused by immediate sensitivity within the alimentary canal typically develop within minutes to 2 hours of ingesting the offending food. Symptoms can include lip, tongue and palatal pruritus and swelling, laryngeal oedema, nausea, abdominal cramping, vomiting and diarrhoea. Severe reactions may result in most or all symptoms related to anaphylaxis.
Track 16: Cancer and Tumour Immunology:
However, the system could even be able to mount an attack against the few tumour cells that are spared by the chemotherapeutic agent. there's an increased incidence of malignancies in immuno-deficient patients like AIDS patients who are susceptible to Kaposi sarcoma and transplant patients who are susceptible to Epstein Barr virus (EBV)-induced lymphoma. Tumor-specific antibodies and T lymphocytes. Hosts are often specifically immunized against various sorts of tumors demonstrating tumour antigens can elicit an immune reaction . A tumour that grows in an animal strain also will grow in another animal belonging to the same inbred strain obtained by repeated brother-sister mating's. These animals express the same MHC molecule and are mentioned as syngeneic. However, most conventional animal populations are allogeneic and have various MHC haplotypes. Thus, a tumour transferred from one animal to a special animal belonging to an outbred strain is rejected thanks to the allo-MHC rather than the TSTA. A tumour transferred from an animal belonging to a minimum of one species a special">to a special animal belonging to a special species is rapidly rejected because the animals are xenogeneic.
Track 17: Parasite Immunology:
Parasite Immunology may be a world journal dedicated to research on all aspects of parasite immunology in human and animal hosts. Emphasis has been placed on how hosts control parasites, and thus the immunopathological reactions which happen within the course of parasitic infections. Extensive research shows that parasitic worms have the facility to deactivate certain system cells, leading to a gentler immune response. Often, such a response is beneficial to both parasite and host, according to Professor of Medical Microbiology Graham Rook of University College London. This immune "relaxation" is incorporated throughout the system, decreasing immune responses against harmless allergens, gut flora, and thus the body itself. In their Parasite Immunology article on worms and viral infections, Dr. Kamal et al. explain why some parasitic worms aggravate the immune response. Because parasitic worms often induce Th2 cells and cause suppressed Th1 cells, problems arise when Th1 cells are needed. within the past, helminthic were thought to easily suppress T-helper Type 1 (Th1) cells while inducing T-helper Type 2 (Th2) cells. However, helminthic also regulate Th2-caused diseases, like allergy and asthma.
Track 18: Immune Tolerance:
Immune tolerances are often defined as a state during which a T -cell cannot answer antigen. The T cell "tolerates" the antigen. A state of unresponsiveness to a specific antigen or group of antigens to which a private is usually responsive. Immune tolerance is achieved under conditions that suppress the immune response and is not just the absence of an immune reaction. it's the prevention of an immune reaction against a specific antigen. as an example, the system is usually tolerant of self-antigens, so it doesn't usually attack the body's own cells, tissues, and organs. However, when tolerance is lost, disorders like autoimmune disease or allergy may occur. Encompasses the range of physiological mechanisms by which the body reduces or eliminates an immune response to particular agents. Typically, a change within the host, not the antigen, is implied. Though some pathogens can evolve to subside virulent in host-pathogen coevolution, tolerance doesn't ask the change within the pathogen, but are often wont to describe the changes in host physiology. Nor does it ask other kinds of non-reactivity like immunological paralysis.
Immunologists
Pharmacologists
Young research fellows
Residents, Fellows & Post Docs
Business Executives & Directors
Associate and Assistant Professors in Immunology
Association heads and Professors
Immunology laboratory heads
CEOs, COOs, Directors, Vice Presidents, Co-directors, Managing Directors,
Students and other affiliates related to the area of Immunology and Immunotherapy.
Immunotherapy Drugs Market – Forecasts (2022 - 2026):
Top-down and bottom-up methods were used to estimate and validate the size of the global cancer immunotherapy market and to evaluate the size of various other dependent submarkets. The Immunotherapy Drugs Market was valued at USD 164.68 billion in 2020, and it is predicted to reach USD 276.51 billion by 2026, registering a CAGR of 8.41% during the forecast period. The major factors accrediting to the growth of the immunotherapy drugs market are the rising adoption of targeted therapy over traditional therapy, emergence of biosimilars, rising prevalence of chronic diseases and lifestyle disorders, and rising demand for monoclonal antibodies.
The influence of COVID-19 on the immunotherapy drugs market is constructive because many companies are currently focused on developing immunotherapy-based drugs or vaccines for COVID-19. Currently, BioNTech SE, in collaboration with Pfizer Inc., is conducting a phase III clinical trial to assess the safety, tolerability, and immunogenicity of BNT162b2 against COVID-19 in healthy participants.
The growing prevalence of cancer and increasing investment in R&D activities for the development of cancer cell therapies is also stimulating market growth. The global Cancer Immunotherapy Market size is expected to reach USD 168.48 billion in 2028, registering a CAGR of 10.1% over the forecast period, according to a new report by Reports and Data. Key factors driving cancer immunotherapy market revenue growth are increasing adoption of immunotherapy drugs over conventional treatments, increasing prevalence of cancer, increasing investments for development of biosimilars and monoclonal antibodies, along with a favorable drug approval scenario.
Related Societies and Associations:
American Academy of Allergy, Asthma and Immunology (AAAAI)
American Board of Allergy and Immunology (ABAI)
Association of Allergy Asthma and Immunology "Buenos Aires" (AAIBA)
Association of Universities and Colleges of Canada (AUCC)
Canadian Society of Allergy and Clinical Immunology (CSACI)
Canadian Society for Immunology (CSI)
Macedonian Society for Basic Clinical Immunology & Allergology (MSBCIA)
Norwegian Society for Immunology (NSI)
Australasian Society for Immunology (ASI)
Association of Medical Laboratory Immunologists (AMLI)
International Cytokine Society (ICS)
Society for Leukocyte Biology (SLB)
Society for Immunology and Immunopathology (SIIP)
Federation of Clinical Immunology Societies (FOCIS)
Universities Related to Immunology
Conference Date August 30-31, 2022
Journal of Clinical and Cellular Immunology


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Mieczysław Pańkowski (ur. 6 października 1865 w Dublanach, zm. 9 listopada 1940 w Krakowie) – polski zootechnik, profesor. Syn Kazimierza.

Życiorys
Syn Kazimierza Pańkowskiego i Honoraty Komarnickiej. Brat Stanisława Pańkowskiego i Heleny Maciołowskiej – właścicielki Mokrzan koło Sambora. Żonaty z Elżbietą Czarnowską (1867–1941). Miał dwie córki Annę Pańkowską po mężu Dzierżanowską i Marię Pańkowską, po mężu Sopocińską.

Uczeń Gimnazjum Franciszka Józefa I we Lwowie. W latach 1884–1888 student Uniwersytetu we Lwowie. Następnie praktykował jako rolnik i studiował w Wyższej Szkole Rolniczej w Dublanach (1890–1893). W latach 1893–1894 studiował w Berlinie, a w latach 1894–1895 na Uniwersytecie w Lipsku. W 1895 uzyskał tytuł doktora filozofii. W celach naukowych przebywał w Holandii, Niemczech i Szwajcarii. W latach 1895–1903 związany ze Szkołą w Dublanach. Następnie w latach 1903–1919 inspektor hodowli bydła w Krakowie. W latach 1921–1931 profesor w Bydgoszczy, a w 1929 roku profesor na Uniwersytecie w Poznaniu.

Napisał m.in. książki: Zwierzęta domowe w zdrowiu i chorobie (1910), Budowa krów mlecznych i oznaki mleczności (1913), Postępy hodowli czerwonego bydła polskiego w Galicji Zachodniej i jej metody współczesne, Kilka uwag i spostrzeżeń nad owcą fryzyjską (Rocznik. Nauk Rolniczych Uniwersytet Poznański t. XIII. Poznań 1925, s. 150–157).

Brał udział w zjeździe Towarzystwa Lekarzy i Przyrodników Polskich w 1911 roku i został wybrany członkiem komitetu gospodarczego Towarzystwa.

2 maja 1923 został odznaczony Krzyżem Oficerskim Orderu Odrodzenia Polski.

Pochowany w Krakowie na Cmentarzu Rakowickim w grobowcu rodzinnym Czarnowskich i Sumińskich (kwatera X(iks), płn.-zach.).

Przypisy

Bibliografia
 Księga adresowa Polski 1929 (Mokrzany)
 Rycerstwo Polskie Podkarpacia – Władysław Pulharowicz – Przemyśl – 1937
 www.sejm-wielki.pl
 1858–1908 Księga pamiątkowa półwiekowego jubileuszu Gimnazyjum im. Franciszka Józefa I we Lwowie – Lwów 1909.
 Polski Słownik Biograficzny 21639: t. 25, s. 155, PAŃKOWSKI Mieczysław (1865-1940) zootechnik, profesor
 Polski Słownik Biograficzny t. 25, s. 154: psb.21638.4
 Polski Słownik Biograficzny t. 25, s. 155: psb.21639.1
 Ziemianie Polscy XX w. t. 6 – zi.6.16.r3a
 Kronika Uniwersytetu Poznańskiego za rok szkolny 1924/25
 Chemik Polski – nr 10 – 1 października 1911, s. 453

Linki zewnętrzne
 Dzieła Mieczysława Pańkowskiego w bibliotece Polona

Polscy zootechnicy
Absolwenci Uniwersytetu Lwowskiego
Absolwenci Akademii Rolniczej w Dublanach
Wykładowcy Akademii Rolniczej w Dublanach
Odznaczeni Krzyżem Oficerskim Orderu Odrodzenia Polski (II Rzeczpospolita)
Pochowani na Cmentarzu Rakowickim w Krakowie
Urodzeni w 1865
Zmarli w 1940

redpajama_set_name: RedPajamaWikipedia


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St Matthew's Church, Keasden
Local support for you during coronavirus
Churches Weekly Newsletter - No. 43, 17 January 2021, The Second Sunday of Epiphany
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Keasden Lancaster
Church of England Diocese of Leeds Clapham-with-Keasden
Keasden is situated three miles from Clapham, and the ecclesiastical district it covers is remote and scattered; covering a wide area measuring six miles by ten.
Church Services in Keasden were originally held in a bobbin mill but, in order that people might have the advantage of a more suitable place of worship, James Farrer of Clapham "came forward in the more liberal manner, not only providing a piece of land about 2 roods and 28 perches in extent, but also had erected a neat looking and substantial small Church".
The building, which can accommodate about 150 people, was built in a fairly simple character architecturally. The windows are single lancets with three windows grouped together to form an east window, and all are glazed with tinted rolled glass.
The Church and adjacent burial ground were consecrated by the Lord Bishop of Ripon in September, 1873.