Jennifer Jarosik is a rising star in today's world of multimedia. In addition to being a published poet, a children's books author, and a documentary producer, Jarosik is noted as the host of the television show Star Spirit for Hulu in America, where she has candid interviews with noted celebrity guests regarding their outlook on life challenges, success and spirituality.
Jennifer Jarosik is a rising star in today's world of multimedia. In addition to being a published poet, a children's books author, and a documentary producer, Jarosik is noted as the host of the television show Star Spirit for Hulu in America, where she has candid interviews with noted celebrity guests regarding their outlook on life challenges, success and spirituality. Among recent guests on her show was legendary American talent manager, Hollywood film agent, and producer Shep Gordon, who was the main subject in the feature documentary Supermensch: The Legend of Shep Gordon directed by Mike Myers. As an author, Jarosik's writing is often inspirational and soul searching. Her first poetry book Amuse Yourself has been very well received, garnering glowing reviews from numerous readers. Following on its success, Jarosik's second poetry book Conversations with Silence is presently being prepared of publication.
My definition of women's empowerment is one of many facets. Yet, really only one truly defines this. To begin, I believe that women have been supressed for centuries. Growing up in America I know I am blessed in so many ways than other women around the country. I visited Agra, India in February to read my kid's book to the kids at Indian dreams foundation, it was brought to my attention the crucial issue of women's health and hygiene. I had no idea of what these girls go through and what lack of knowledge they have of why they even have a period. I sat with them and explained what a gift it is to be a woman and to be on our cycle. And how it is the time where we are most in our power, most intuitive, and preparing our body like nature for the rose to bloom. The rose is this essence of a woman, the divine feminine of love which is the healing force whose voice and body has been excluded and supressed for too long. My eyes and heart were opened and I cannot turn back. I am a global ambassador for Indian dreams foundation and I asked the founders that the men need also to be educated. Part of female empowerment is also for us to use our voice to make light of what's been in the dark. Men do not understand women, especially little boys with girls on their cycle. They have shunned them and made the feel like they are a disease. I say the cure is compassion, love and empowering women to see we are all alike and that is a wonderful blessing to be a woman. I also see women friends in other countries who are covered up, full bodies and not able to wear freely what they would choose. Or being controlled in a patriarchal society where men call the shots in governments, jobs, families. I say we empower the women to have freedom to speak how they choose to live their life and how their bodies are treated. That there be more of a gender egalitarianism not only society but in economically being paid same wages for same job which there is not one country in the world that has this. To answer the one thing that is what I believe women empowerment is , is that as one awakens to the truth of who one is and becomes self-empowered, then it is like a ripple effect, or a contagious disease which cause a fire to burn in our hearts to assist others to find their power and voice.
I was inspired to get involved with creating a change and to inspire other women by accident. I was sitting at home on the couch with my 5 year old son Dakota. I was reading a book called the goddess shift, about women leading for a change in the world. As single mother, depressed, and not feeling empowered or even with a dream my friend suggest we film it. He said we can interview these women and find out their journey. How did they get to where there are or be who they are and what challenges do they have? This will be part of my healing and empowerment. I said no way!! My ego wanted something else for my life, not to share what was going through. As I said yes a few. As I said yes a few months later, doors opened and I found myself travelling the world meeting amazing women healers, motivational speakers as Marianne Williamson, and even men as Russell Simmons and David lynch speaking on happiness and the goddess. The goddess is my docu drama on the story of discovering and embodying the goddess within by meeting 7 women in history (goddesses) who share universal messages for my healing and empowerment and assisting others in the same.
Some characteristics of an empowered person are I believe in the eye of the beholder, but again, most importantly in how you feel about yourself. When I was young I looked up to movie star women, and of recent women like Oprah who is doing soulful and heartful work. Yet I also saw that they too struggled behind the scenes of the camera image with the image of who they are, both internal and external. In my own struggles of trying to be someone other than who I was, or to accomplish something to be known or accepted, or being excluded by jealous people who hurt me deeply, including family, lovers, and colleagues I have discovered that the persons I truly wanted to be like was myself, the girl I once knew who was happy, creative, and spreading love and inspiration to people. And the sought out of being accepted sent me further from this girl, woman. The people I believe who have the most characteristics of empowerment are not the successful famous ones, or the wealthy ones necessarily though there are some who are, but the true quality of who they are remaining intact and this being the truth of inspiring others to also re discover this. So many people I have met who hone this trait are simple people who live simply so others can simply live. They are content with themselves, loving, giving souls living their highest life. Some are mothers, some are teachers, and some are famous like Oprah who I believe is doing the greatest service by sharing her struggles. This to me is one who embodies empowerment not just is given the word.
I believe that leaders or people interested in advocacy for empowerment can do several things to inspire others and make a difference. Again, it comes down to being the change you wish to see. So in order for one to portray this and assist in truth, one can only really only do this as one embodies the empowerment. Like peace, one can go to conferences, yoga, meditation etc. but if one is other than a peaceful person it will only last for so long. Same with one's life of empowerment. It doesn't have to be grandiose it can be as simple as taking personal responsibility for one's own happiness, one's own peace, and speaking one's truth, find the inner voice even if it upsets others or ones culture or religion. The next step is for those who wish to make a global change, start a blog, join women's conferences like heart talk, women's economic forum, all and use media to reach the masses.
My advice to those who want to give up due to lack of empowerment, thinking and feeling is to give up. You're probably wondering what I mean by this. Well, for years I have felt disempowered, from the thinking to feeling to believing this was who I am or my life. No matter what I did, how many healers, or friends I sought for advice, or what to do to change this feeling I could not get the other empowerment thoughts and feelings to stick. I tried so hard to escape my deep sadness and feelings of unworthiness, lack of love etc. I practiced yoga, I went on trips, I met amazing people, travelled the world and yet at the end of the day it was a feeling of who I was that haunted me. In time I have learned to surrender. As one awakens to truth of who one is, one is not the thinking or feeling so its having a perception that allows one to have a gap between this label one has given oneself , and especially one that others has given them. The emotions came and went. My thinking like rubbish in the wind went from positive to lowest low. And eventually got lower and lower until I stopped trying, I stopped fighting the truth. We are not that the emotions are not good to have. I say the emotions are necessary, the sadness, the grief, the lack, all of it is like a storm. If you face it will eventually pass. So I cried and I felt what I felt. Yet knowing it was a part of me based upon my experience that led me to this place. And I surrendered. I began to embrace me instead of feeling there was something wrong with me. Maybe being sensitive is not wrong. Maybe it was wrong to deny the truth of feeling and thinking. And from this place slowly I began to realize that exist beyond this, like the sun and the thoughts and feelings were simply clouds. Even happy thoughts and thinking can be deceiving. A peace began to occur within me, a love from the inside and a grace of god that gets me still to this day through the tough days. So my greatest advice is to give up trying to change what you are feeling or thinking, accept it, observe it and give a new perception to make changes from the inside out.


Please browse through our accommodation directory below and book online using a unique booking system that guarantees the best rates and free reservation service. Make sure you look at the customer reviews to see what other guests have to say about your accommodation choice! There's plenty to choose from with prices and styles to suit everyone.
Skycity Darwin is Darwin's only 5 star hotel and is situated close to Darwin's Central Business District and sits next to the beach. The rooms are comfortable and hotel offers guests free wireless internet access. There is a casino on site.
These contemporary apartments are located in this high rise hotel in the middle of Darwin. Stay at the Mantra Pandanas and be close to Darwin Harbour, shopping mall and other nearby attractions.
Argust Apartments offers guests a stylish and luxurious stay in the centre of Darwin City. Enjoy your very own view over the city and harbour from your modern apartment. Stay close to Darwin's famous beaches, restaurants and shops.
Located in the heart of Darwin and overlooking the harbour is the modern Holiday Inn Esplanade Darwin. Enjoy the tropical gardens and large pool area.
Book a self contained apartment and have all the comforts of home at Quest Darwin. Located in the city centre with a variety of features including outside pool, barbeque area, laundry and private parking.
Situated in the heart of the city on Darwin's thriving Esplanade the Novotel Atrium Darwin has great views of Darwin Harbour. It's also just a short stroll from the dining and entertainment area on Mitchell Street.
The Medina Grand Darwin is nestled amongst stylish waterfront residences, restaurants, clothing boutiques, the beach, boating lagoon and stunning parklands.
If you are just passing through Darwin, book yourself in at the Darwin Airport Resort which is just 350 metres from the airport. You'll have 4 star luxuries as well as free ainport transfers, Wi-Fi and parking.
The Darwin Central Hotel is a 4 star hotel with onsite restaurant, outdoor pool, tour desk and 24 hour room service and concierge.
Just 250 metres from Darwin Airport, the Darwin Airport Inn is great for those on early or late flights. There is a free shuttle bus to the airport, outside pool, meeting room and bistro.
The Crowne Plaza Darwin's rooms are comfortable and modern with seating areas, air conditioning and LCD televisions. The hotel is located in the heart of Darwin and is a very short stroll from the harbour and the Esplanade.
Located a short 5 minute drive from Darwin's CBD is the peaceful suburb of Parap. Here you will find the Alkira Darwin which offers guests a modern, boutique style stay.
Enjoy panoramic views from your room and apartment Mantra on the Esplanade. It is located in the heart of Darwin City and overlooks the Darwin Harbour.
Just 15 minutes south of Darwin is the Darwin FreeSpirit Resort in Holtze. This is a great location to base yourself as you jump off to explore the Top End.
The 4 star Travelodge Mirambeena Resort Darwin offers guests affordable rooms in the middle of Darwin.
The Botanic Gardens Apartments is set in a tranquil setting, situated on the border of Darwin's Botanical Gardens. There are two pools and Mindil Beach is within walking distance.
The Best Western Darwin Airport Gateway Motel is great for travellers as it located less than 5 minutes from the Airport and has free private parking.
The Vibe Hotel is just moments from the thriving Darwin Waterfront, shops and nightlife. The rooms are modern and come with free wireless internet, outside you will find a pleasant wave pool.
10 minutes out of Darwin CBD is the Hidden Valley Resort in Berrimah. Enjoy relaxing at the outdoor swimming pool.
The Comfort Inn Vitina offeres guests affordable motel style accommodation 1 kilometre from Darwin's city centre. Guests have free parking.
Close to the Mindil Beach Markets and Darwin Botanical Gardens is the self catering Altai Holiday Apartments.
Neslted in the heart of Darwin's vibant city centre in an oasis of tropical garden is the Palms City Resport. Relax at the resort and then stop out onto the Esplanade to find some entertainment.
The City Gardens Apartments offers guests self contained apartment accommodation, lagoon swimming pool surrounded by tropical gardens, great barbequre area and free off street parking - perfect for a family holiday.
This budget hotel is located on Mitchell Street, Darwin's entertainment and restaurant precinct, is the Value Inn. There is an outdoor swimming pool for relaxing by.
Just minute's from Darwin is Coconut Grove Holiday Apartments in the suburb of Coconut Grove. There are moern apartments with balconies set amongst tropical gardens.
These affordable, self catering apartments are located a 15 minute walk from Darwin's city centre. They are close to the Mindil Beach Markets and Botanic Gardens.
This award winning hostel is a great place to stay to meet other like minded travellers. There is a leisure deck, 2 swimming pools, jacuzzi and bar. The rooms are private with your choice of en suite or shared bathroom facilities.


text:
Q: jQuery parse multidimensional-array I am making an ajax request to an external php file, then returning some json. Most of the json is straight forward but I can't seem to figure out how to parse the multidimensional part.

JSON:
{
    "success":"TRUE",
    "action":"JSON",
    "date":"06/29/12",
    "results":"true",
    "numResults":2,
    "0":[
        "id":1234,
        "name":"John Appleseed",
        "gender":"male",
        "average":2.5
    ],
    "1":[
        "id":5678,
        "name":"Jessica Smith",
        "gender":"female",
        "average":1.4
    ]
}

jQuery:
$.ajax({
url: "searchController.php",
data: searchData,
type: "GET",
success:function(q) {
    if (q) { // Results
        $('#search .container .body .ajax .returnedHTML .loadingScreen').hide();
        var json = $.parseJSON(q);
        console.log(json);
        if (json.success == "true") {
            var numResults = json.numResults;
            if (numResults == 1) {
            $('#search .container .body .ajax .returnedHTML .content').contents().remove();
            var htmlString = '

'+json.name+'

'+json.average+'

Average

'+json.numrates+'

Rates

'+json.followers+'

Followers

';
            $('#search .container .body .ajax .returnedHTML .content').append(htmlString);
            console.log(htmlString);
        }
    }
});

How do I parse the JSON so I can access the all the data and not just the data in the first dimension? I have looked around on this site and I can't find anything that is either useful and/or helpful. Thank you so much!

A: Your arrays are at json[0] and json[1]
 ...
for(var i in json) {
  if(!isNaN(i)) {
    ...
    refer to you variables as json[i].name, json[i].average, etc.
    ...
  }
}

A: $.ajax({
url: "searchController.php",
data: searchData,
type: "GET",
success:function(q) {
    if (q) { // Results
        $('#search .container .body .ajax .returnedHTML .loadingScreen').hide();
        var json = $.parseJSON(q);
        console.log(json);
        if (json.success.toLowerCase() == "true") {
            ...
        }
    }
});

for(var key in json){
    if(json.hasOwnProperty(key){
       console.log(key);
       console.log(json[key]);
    }
}

A: About your JSON you have to pars same again,
var jsonArr1 = $.parseJSON(json.0);
var jsonArr2 = $.parseJSON(json.1);

redpajama_set_name: RedPajamaStackExchange


August 26, 2008 Federal Financial Markets Service registered the Report on results of the issue of ordinary shares placed by OGK-4 via converting of the preferred JSC OGK-4 Holding shares into JSC OGK-4 ordinary shares.
The share placement took place at the same time as JSC OGK-4 Holding was spun off from RAO UES of Russia, which spin-off was accompanied by the merger of JSC OGK-4 Holding with and into OGK-4. OGK-4 actually placed 3,027,744 ordinary shares, each with a nominal value of 0.40 Russian rubles.
August 5, 2008 Federal Financial Markets Service registered the Report on results of the issue of 28,562,562 ordinary shares each with a nominal value of 0.40 Russian rubles, placed for the purposes of conversion of ordinary shares of JSC OGK-4 Holding into ordinary shares of JSC OGK-4.
As reported earlier, FFMS on April 29, 2008, registered two additional issues totaling 32,539,735 OGK-4 shares. The share placement was part of the final phase of the reorganization of RAO UES of Russia. The Company carried out the share placement on July 1, 2008, the day when the dissolution of JSC OGK-4 Holding according to its shareholder register as of that date was registered on the Uniform State Register of Legal Entities.
The open joint stock company Forth Generation Company of the Wholesale Electricity Market (OGK-4) operates five power generation plants with the total capacity of 8,630 MW, including Surgutskaya GRES-2 (4,800 MW, Khanty-Mansi Autonomous District), Berezovskaya GRES (1,500 MW, Krasnoyarsk territory), Shaturskaya GRES (1,100 MW, Moscow region), Smolenskaya GRES (630 MW, Smolensk region) and Yayvinskaya GRES (600 MW, Perm territory). 76% of OGK-4 shares belong to E.ON Russia Power GmbH – a 100% affiliate of E.ON AG, coordinating corporation's energy business in Russia.


One of our first days in Michigan was spent out on the pontoon with family...We just float out on the lake drinking lots of beer, eating lots of snacks and enjoying the sun.
We brought floats so we can just jump off the boat and bath for a bit.
Even my aunt's dog, Missy, enjoyed being out for the day.


With Viper4android You Will Get A Great Music Experience, But You Can Still Find Low Volume Just Because Samsung Has Chosen The Ceil Volume At Some Point. Typically, Hardware Capabilities Of Smart Phone Manufacturers Limited To An Optimum Level To Ensure The Life Of The Device. However, These Adjust The Level To A Certain Extent It Will Not Hurt Your Device Significantly (may Be Slightly), But You Will Feel Improvement In Performance. So, If You Are In Today, We Will See How To Use The Xda Developers Create The Department Of Defense To Increase The Volume Raw2000j Galaxy S6 And S6 Side Speaker.
Mod V3 Higher Volume Will Increase Speaker, Headphone Volume, Bluetooth 7 Level, Less Than Three Tones. Ministry Of Defence Is In Zip Format And File Erasable Mixer_paths.xml Modified Are Available, So You Can Choose Any Of Them. If You Choose The Simple Recovery Method, In Accordance With Our Installation Guide Twrp Galaxy S6.
MOD rewritable version of downloads, and transfer it to your device storage roots.
Turn off your device, wait for it to completely shut down.
Hold down the power + home + volume buttons together, when you see the Galaxy S6 logo on the screen as soon as they are released.
First, TWRP backup option to back up your current ROM and settings.
In the restoration, click Install, and select the volume MOD zip file and confirm flashes.
Restart the device blinks once completed.
Download and transport modify mixer_paths.xml file and transfer it to a storage space on your device.
Open Windows Explorer application, rename the original file mixer_paths.xml.bak mixer_paths.xml just have a backup of the original file.
Now copy the modified file and modify the permissions to 644 (RW-RR), and carefully check the permissions again.
After You Install This Mod, You Will Find A Significant Increase In The Volume Level Of Your Device. If You Feel Like A Return To The Original, The Replacement File Path Stock Mixer The Modified File. That's It.


text:
Улица Коњовићева једна је од старијих градских улица Сомбору. Протеже се правцем који повезује Видовдански трг и Супотички пут. Својим почетком заједно са крајем Венца војводе Живојина Мишића прави кружни ток око Видовданског трга.

Завршетак Коњовићеве улице јесте на спајању улице Светозара Милетића и Суботичког пута.

Име улице
Улица се некада звала Коњовачка.

Данас улица носи име по Коњовићима који су живели у Сомбору и чијих се пет кућа налазило у овој улици.

Коњовићи су патрицијска сомборска српска породица, чији су потомци скоро три века представљали духовну, културну, привредну, политичку и интелектуалну елиту Сомбора.

Неки од познатих Коњовића су: Давид Коњовић(1786-1849), сенатор и главни судија (градоначелник) слободног и краљевског града Сомбора, један од оснивача Српске читаонице 1845. године.

Млађи син Давида Коњовића, Јустин (1836-1896), један је од најутицајнијих српских политичких првака сомборских Срба током друге половине 19. века.

Јустинов син Давид Коњовић (1865-1925) био је сомборски посланик у угарском Државном сабору, члан Месног народног већа Срба и Буњеваца у новембру 1918. године и председник Српске читаонице, председник Окружног суда и краљевски јавни бележник у Сомбору.

Син Давида Ј. Коњовића, а унук Јустинов је знаменити сомборски, војвођански и српски сликар, академик Милан Коњовић (1898-1993).

Знаменити српски композитор Петар Коњовић (1883-1970) и његов рођени брат Димитрије Коњовић (1888-1982), први Сомборац који је био пилот, а затим оснивач и власник фабрике авиона "Икарус", такође су потомци ове породице.

Сомборски адвокат и српски добровољац из Првог светског рата др Јован Јоца Коњовић, касније дугогодишњи председник Удружења ратника у Сомбору, заслужан за изградњу костурнице са спомеником за умрле заробљене руске и српске војнике у Сомбору.

Из ове породице потекао је и Никола Коњовић (1869-1950), један од оснивача Југословенског шаховског савеза.

Суседне улице
 Венац војводе Живојина Мишића
 Улица Милоша Обилића
 Улица Јосипа Козарца
 Улица Партизанска
 Улица Светозара Милетића
 Суботички пут

Коњовићевом улицом
Улица Коњовићева је улица у којој се налази неколико продајних објеката, продавница, угоститељских објеката али је пре свега насељена улица са једноспратним, двоспратним кућама како старијом тако и новијом градњом.

Видовдански трг

На раскршћу два сомборска венца (Радомира Путника и Живојина Мишића), те Коњовићеве улице и Улице Милоша Обилића налази се најмањи и најскромније уређен сомборски трг.

У другој половини 18. века на тргу је постављен камени православни крст.

Трг је у првој половини 19. века називан и Коњовића баштом због пет кућа породице Коњовић које су излазиле на овај трг и због којих је и Коњовићева улица добила свој назив.

Од 1907. године на тргу се налазио артески "Коњовачки бунар", један од најпрометнијих у Сомбору.

После Првог светског рата ту су приређиване велике видовданске свечаности, које су имале званичан градски и национални карактер.

Године 1953. на средини трга постављен је споменик палим борцима Народноослободилачког рата. Истовремено је уклоњен стари камени православни крст. Нови крст је постављен крајем деведесетих година 20. века, а старо дрвеће замењено је младим садницама током 2013. године, када су поправљене и стазе на тргу.

Нова реконструкција трга започета је 2017. године. ЈКП "Водоканал" Сомбор је, након реконструкције трга, поставило моделе некадашњих водарских кола и артеског бунара на месту где су се налазили и раније.

Током прве половине 2020. године реконструисан је коловоз око трга, који је постао класичан кружни ток за учеснике саобраћаја.

Атеље "Станишић"
На броју број 5 (некада кући Коњовића) налази се Атеље "Станишић" - пет генерација породичне традиције израде витража и мозаика.

Атеље "Станишић" основан 1908. године специјализован је за услуге израде нових и рестаурацију витража и мозаика. Уметничка дела која настају у Атељеу "Станишић" саставни су део архитектуре многих духовних и секуларних грађевина широм земље и света.

Вртић "Невен"
Од значајнијих објекта у улици ту се налази и Вртић "Невен" који је у саставу Предшколске установе "Вере Гуцуње" и он је на броу 24.

Галерија

Референце

Улице у Сомбору
Сомбор

redpajama_set_name: RedPajamaWikipedia


This investigation, if required, is performed at an alternative provider (not in-house).
A coronary computed tomography angiogram (CTA) uses advanced CT technology, along with intravenous (IV) contrast material (dye), to obtain high-resolution, three-dimensional pictures of the moving heart and great vessels.
CT coronary angiogram of the Right Coronary Artery.
This procedure is undertaken in one of out partner facilities. A CTA scan is performed with you going into a metal tube. You have to lie still to allow the pictures to be taken. A drug may need to be given to slow your heart rate down. The test involves the use of X-rays and X-ray dye, which is injected via a needle in the back of your hand. There has been great progress in taking pictures of the heart and brain blood vessels. The radiation dose of the CT scan is often equivalent the coronary angiogram. In addition, X-ray contrast dye still has to be used. However, it is less invasive than the angiogram and so will be the diagnostic tool of choice in some cases. You cannot do coronary angioplasty (PCI) guided by CT angiography alone.
Once the images have been processed and the specialist has formed the report your cardiologist will tell feedback the result to you along with any recommendations for treatment.


As you join the University of Rochester community, it is important that you feel prepared by understanding the requirements of your US immigration status and availability of campus services. The International Student Orientation Program (ISOP) is designed to provide essential information on maintaining your valid F-1 or J-1 nonimmigrant status while in the United States as well as other important details about life at the University and in Rochester.
Our office has developed a program called TIPS On-Demand: Tools for Immigration and Preparation Support to share relevant information specifically with incoming international students. This program serves as ISO's pre-arrival orientation and completing it is required of all new international students sponsored by ISO.
The details covered in this program directly align with the ISO's in-person orientation you will attend when you arrive in Rochester. You must review this information in advance of your in-person ISOP.
We hope these tools will be useful and look forward to welcoming you to campus soon!
Attendance is mandatory for new international students throughout all University divisions. ISO's in-person orientation is designed to supplement your online pre-arrival orientation by providing opportunity to think more critically about and understand key aspects of maintaining your status during your studies at the University of Rochester.
Please review the ISOP Summer/Fall 2019 schedule for details about your meeting, and be sure to complete pre-arrival online orientation in advance!
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This isn't the first time the 'Teen Mom' star has been facing charges.
Amber Portwood is being sued by her current landlord over claims she trashed her home. The Teen Mom OG star could face eviction because she allegedly failed to pay rent for months.
The landlord is asking for almost $3,000 in damages plus court costs and other forms of relief.


text:
Statement from Press Secretary Robert Gibbs on Prime Minister Netanyahu's speech
FOR IMMEDIATE RELEASE June 14, 2009
"The President welcomes the important step forward in Prime Minister Netanyahu's speech. The President is committed to two states, a Jewish state of Israel and an independent Palestine, in the historic homeland of both peoples. He believes this solution can and must ensure both Israel's security and the fulfillment of the Palestinians' legitimate aspirations for a viable state, and he welcomes Prime Minister Netanyahu's endorsement of that goal. The President will continue working with all parties - Israel, the Palestinian Authority, Arab states, and our Quartet partners - to see that they fulfill their obligations and responsibilities necessary to achieve a two-state solution to the Israeli-Palestinian conflict and a comprehensive regional peace."

redpajama_set_name: RedPajamaCommonCrawl


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text:
In the late 1980s, an epidemic of attacks by Pit Bull type dogs, and other related breeds, led to widespread bans. In 1991, the Parliament of the United Kingdom banned the ownership of Japanese Tosa Inus, Argentine Dogos, Fila Brasilieros and Pit Bulls, with many other countries following suit soon after. Even in areas where having such dogs is legal, it can be nearly impossible for homeowners to get liability insurance if they own one of the breeds below:
10. American Bulldog:
Banned in Denmark, Singapore and various municipalities, the American Bulldog's origins are in the deep south, where it was used as a farm dog.
9. Bandog:
The term 'bandog' has been in use since the Middle Ages and is used to describe a large dog that was let off its chain at night to guard its property. Bandogs are generally prohibited anywhere there are restrictions on its parent breeds.
8. Neapolitan Mastiff:
The Neapolitan Mastiff or Neo, comes from Italy, where once it was used as a gladiator dog in the bloody spectacles of the Coliseum. They were also used as war dogs by the Roman legion.
7. Wolfdog :
There are many established breeds of wolves and domestic dogs, including the Czechoslovakian Wolfdog (a mix of German Shepherd and Carpathian wolf) and the Saarloos Wolfhound (German Shepherd and Mackenzie Valley timber wolf). There have been many attacks on humans, most commonly on small children, which they may view as prey items. It is prohibited in Norway.
6. Boerboel:
The Boerboel comes from South Africa, and closely resembles a more athletic Bull Mastiff in appearance. They are banned in Denmark.
5. Dogo Argentino:
First bred in Argentina in 1928, the Dogo was taken from the now extinct Cordoba Fighting Dog, which was mixed with various other breeds, including the Great Dane, Dogue de Bordeaux and Irish Wolfhound. The Dogo is banned in at least 10 countries, including Australia, New Zealand and Portugal.
4. Presa Canario:
The Presa Canario is a massive fighting dog hailing from Spain's Canary Islands, generally weighing over a hundred pounds. Presa Canarios are banned in Australia and New Zealand.
3. Fila Brasiliero:
The Fila or Brazilian Mastiff, is a huge dog bred for hunting boar and jaguar, and was even used for tracking down runaway slaves. It has Mastiff, Bulldog and Bloodhound ancestry. It is illegal to own a Fila in the United Kingdom.
2. Japanese Tosa Inu:
The Tosa Inu can weigh anywhere from 80-200lbs. It is a mix of indigenous Japanese dogs and various Western breeds, such as the Mastiff and Bull Terrier. It is illegal to own in Denmark, Malta and Norway, amongst other countries.
1. American Pit Bull Terrier:
They are illegal to own in Miami-Dade County, Florida; Ontario, Canada; and many countries throughout the world.
Categories banned top breeds, dogs, top 10 illegal dogs


My wife, daughter and i had the pleasure of dining there last night-excellent cuisine -the atmosphere, food and service was par excellence. Very affordable and sooooo tasty. Second time that we have been there and will definitely return. thoroughly recommend.
Have to say one of my favourite restaurants in Hamilton, great atmosphere! Great place to take a lady on a date cause its small and quite intimate. The food is always lovely and fresh! The service is fabulous!


text:
B In The Mix: The Remixes é o primeiro álbum de remixes da cantora pop estado-unidense Britney Spears, lançado no dia 22 de Novembro de 2005 pela gravadora Jive Records. O álbum consiste em 11 faixas remixadas de álbuns anteriores da cantora, sendo sete destas singles e três do álbum In the Zone, por DJ's como Peter Rauhofer e Stuart Price. Os remixes têm influência em vários gêneros de música eletrônica, como trance e techno. B in the Mix: The Remixes também inclui uma canção nova, "And Then We Kiss".

O álbum recebeu críticas variadas. Alguns críticos consideraram B in the Mix: The Remixes como um bom álbum de remixes, enquanto outros consideraram o álbum fraco, assim como os vocais de Spears. B in the Mix: The Remixes teve ótimas vendas boas, entrando apenas na #19 posição nas paradas musicais da Bélgica, e em #34 nos Estados Unidos. "And Then We Kiss" foi lançada como o único single promocional do álbum.

Lançamento
Em 28 de Setembro de 2004, a Jive Records anunciou que Spears lançaria seu primeiro álbum de remixes, e que este se chamaria Remixed. Entretanto, no dia 8 de Novembro de 2005, a repórter Jennifer Vineyard da MTV anunciou que o álbum na verdade se chamaria B in the Mix: The Remixes, e que seria lançado em 22 de Novembro de 2005. O álbum inclui canções de seus outros álbuns remixadas por DJ's como Peter Rauhofer e Stuart Price. Price havia remixado "Breathe on Me" do álbum In the Zone anteriormente para a edição limitada da coletânea Greatest Hits: My Prerogative. B in the Mix: The Remixes também inclui uma canção inédita, chamada "And Then We Kiss"; a canção seria lançada anteriormente como parte do CD bônus do DVD do reality show Britney & Kevin: Chaotic, mas foi removida por motivos desconhecidos. B in the Mix: The Remixes possui duas capas diferentes. Na edição norte-americana, Spears não aparece na capa; foi utilizada uma borboleta ao invés da cantora. Na edição internacional, Spears aparece atrás da mesma borboleta.

Recepção da crítica

Kurt Kirton do site About.com deu destaque aos remixes de "Everytime" e "Don't Let Me Be the Last to Know", dizendo que o álbum seria melhor se tivesse mais faixas. Ele resumiu sua crítica dizendo que foi um álbum "decente que deve agradar todo fã de Britney e a maiora dos fanáticos por música de boates". Barry Walters da revista Rolling Stone era "mais redundante" do que Greatest Hits: My Prerogative, mas acrescentou que o remix de "Toxic" era a grande exceção do álbum, e disse que "praticamente todas as faixas deste álbum estão melhores do que as originais". Stephen Thomas Erlewine do Allmusic comentou que "B in the Mix não elimina o fato de que Spears não está de acordo com o que foi feito em sua carreira", e ainda acrescentou que o álbum é apenas um "pedaço de produto". Gregg Shapiro do Bay Area Reporter deu uma crítica extremamente negativa ao álbum, chamando-o de "desperdício". Também comentou que a voz de Spears parecia "forçada, fria e mecânica" quando removidos os instrumentais originais. Entretanto, ele deu destaque a duas faixas, dizendo que "Spears chegou perto de conseguir o status de diva do dance com o remix de Valentin da faixa 'Everytime', enquanto que o remix de Davidson Ospina da faixa '...Baby One More Time' transforma-a em um clássico".

Performance nos charts
Nos Estados Unidos, B in the Mix: The Remixes estreou na #34 posição da Billboard 200, vendendo 14 mil cópias na semana de lançamento. O álbum estreou também na quarta posição da parada Dance/Electronic Albums, também da Billboard, permanecendo um total de vinte e uma semanas nesta.  Desde Fevereiro de 2010, B in the Mix: The Remixes vendeu cerca de 500 mil cópias nos Estados Unidos, de acordo com a Nielsen SoundScan. O álbum estreou na #9 posição nas paradas musicais da Bélgica. Sua compilação B In The Mix: The Remixes (2005) é o 7º álbum de remixes mais vendido da história com 1.7 milhão do cópias.

Promoção
B in the Mix: The Remixes não foi tão promovido quanto os álbuns anteriores de Spears. Um disco de vinyl promocional intitulado Key Cuts from Remixed com cinco remixes do álbum foi lançado mais de duas semanas antes do lançamento oficial do álbum. Um concurso promovido pelo website oficial de Spears para os que comprassem o álbum com antecedência premiava o vencedor com uma cópia do DVD Britney & Kevin: Chaotic, um frasco de sua fragrância Fantasy, com um creme e um kit de maquiagem promocionais, e uma foto personalizada da cantora e autografada. Em 22 de Novembro de 2005, uma festa de lançamento do álbum realizada em Los Angeles, California, foi orgazinada pela equipe de Spears e o webmaster do fã-site WorldOfBritney.com. A festa foi um evento limitado a 500 pessoas, incluindo membros do fã-clube oficial da cantora. Spears comentou, "Eu só queria dizer que eu adoro a ideia de reunir todos os meus fãs para celebrar o lançamento de meu novo álbum. Eu fiquei muito feliz em ajudar! Espero que todos vocês tenham uma ótima noite e que dancem muito!". "And Then We Kiss" foi lançada como single promocional em CD e vinyl. Jennifer Vineyard da MTV comentou que a canção teria "o potencial para ser um grande hit - se a Jive estivesse promovendo o álbum ativamente". "And Then We Kiss" falhou ao entrar em grandes paradas musicais como Hot 100.

Lista de faixas

Charts

Créditos do álbum
Masterização: Chaz Harper
Gerenciamento: Dan Dymtrow
Design: Jackie Murphy
Fotografia: Anthony Carlucci

Referências

Ver também
 Álbuns remixados mais vendidos do mundo
 Someday (I Will Understand)
 And Then We Kiss
 Discografia de Britney Spears

Álbuns de Britney Spears
Álbuns de remisturas de 2005


text:
Adele Might Release a New Album the Same Day as Ariana Grande
In the past decade, more and more artists have moved over to releasing new music on Fridays to capitalize on sales and chart success for the following week. Because of this, there have been many instances where two big or similar artists have planned releases on the same day. Rumors are swirling that the next big "New Music Friday" conflict will be between Ariana Grande and Adele.
Adele | Kevork Djansezian/Getty Images
Ariana Grande announced her latest album and single
If there's anything fans know about Ariana Grande, it's that she usually doesn't take a break when it comes to creating music and releasing albums. Her last two LPs, Sweetener and thank u, next, came out within six months of each other.
Now, after over a year and a half, Grande is back in album mode, having spent months in lockdown writinga and recording music. She teased on Twitter earlier this month that she "can't wait" to give fans the album by the end of October 2020. She soon unveiled the project name as Positions.
The album's lead single, also named "Positions," is due for release on October 23; the album will come a week later on the 30th. Grande's website has a countdown clock for both.
Ariana Grande | Axelle/Bauer-Griffin/FilmMagic
RELATED: 'Saturday Night Live's Newest Cast Member is a First For the Show
Adele is hosting 'SNL' the week before Ariana's album comes out
Season 46 of Saturday Night Live is in full swing, coronavirus-style. The sketch comedy show announced on Twitter that Adele would be hosting the October 24 episode of the show, with fellow Grammy winner H.E.R. as the musical guest.
Adele expressed her excitement about the SNL announcement on Instagram. "Bloooooody hellllll I'm so excited about this!! And also absolutely terrified! My first ever hosting gig and for SNL of all things!!!!" she said. She goes on to reminisce on how it'll be "almost 12 years to the day" that she first appeared on the show — an appearance that made her a household name in the US — and says it's a "full circle" moment she "just couldn't possibly say no" to.
Ariana Grande performs at Lollapalooza | Kevin Mazur/Getty Images for AG
Adele is rumored to be releasing an album the same day as Ariana
Given that Adele has been relatively quiet in recent months since her divorce, Showbiz411 is reporting that the British songstress is getting ready to release an album of her own on the Friday following her SNL appearance — the same day Grande is releasing Positions.
Many times, artists appear on SNL when they have new music to promote. In his recent stint as musical guest, for example, Justin Bieber performed his new songs "Holy" and "Lonely," which are set to appear on his upcoming sixth album.
While Adele hasn't indicated she's gearing up to release any music, her SNL hosting gig is a sign that she's ready to enter into the public discourse again. Her Grammy-winning album 25 was released in the fall and capitalized on holiday sales, so Adele could very well be planning to repeat her success with her next release.


In a trading day as strange as 'tis bogus, the GOLD PRICE scraped off $14.60 to close comex at $1,397.1 while the SILVER PRICE lost 31.5 cents to end at 2240c. In the late aftermarket gold has traded up to $1,400.15 and silver to 2258c.
I say "bogus" because this is very odd trading. Usually markets either make it or break it. If they fail, they follow through downward; if they make a new high, they climb. What do you say about gold? Thursday it had a great up day, bursting through $1,395 resistant to end around $1,415 — then Friday giving nearly all back? And Monday bouncing right back, but Tuesday having its pockets picket again?
Here's a guess about these last four days. That $1,491-$1,495 level was resistance, but gold shot through it. When it cam back Friday, it fell not through, but stopped at that resistance-now-become-support, $1,392.50. Yesterday it shot back to the top of the range, $1,415, then today fell back, but ratcheted higher with a $1,393.10 close.
Clearly, many happy souls are delighted by the GOLD PRICE at $1,392, while many unhappy souls are delighted to sell $1,415. A tense disagreement exists, and somebody's gonna lose and get a black eye.
Battleground leans back and forth across the 20 DMA ($1,405.55). If the gold price can cross that, it should shoot up to the 50 DMA ($1,461). However, if the Unhappy Ones can drive gold below $1,390, it might break.
No point in pretending to clarity where none is. Momentum indicators are pointing up, gold resiliently keeps popping back, but gold must first prove itself with a jump over $1,425. I am buying modestly but gladly, but can't quite bring myself yet to make a big commitment. I feel the same way a pig, a chicken, and a cow feel about breakfast. The chicken and the cow are involved, but the pig is committed.
Daily I search the news for pleasant or encouraging reports to share with y'all, but daily I only come away with acute hogwash poisoning. I notice that IMF apparatchik Christine Lagarde is warning of "somber trends" developing the world economy. I reckon Christine wants the world's central banks to print up more money, on the theory that although it hasn't worked already, it might work if they do a lot more of it. Akkk! Akkk! I can feel the hogwash poisoning climbing up my gullet!
Stocks were not pleased with anything today. Dow backed off 76.87 (0.5%) to 15,177.16. S&P500, resplendent with GM, which, coming out of bankruptcy or tankruptcy or the government's financial hospital has been re-admitted in the place of ketchup cook Heinz, cheapened by 9.04 or 0.55%.
Grief for stocks will begin when they break their 50 day moving averages (14,904 and 1,603), which roughly coincide with their trading channel bottom lines. Just to give y'all a notion how far away that lies, that's about 273 points for the Dow and about 28 points for the S&P500. Both fell almost that many points on 31 May. And downside points come easier than upside points.
Dow in Gold and Dow in Silver both rose a leetle today, but continue to roll over toward the center of the earth. Dow in gold rose 0.54% to 10.863 oz (G$224.56 gold dollars), not quite breaching the 20 DMA in its close but still in a steady downtrend.
Dow in silver closed 677.55, 6.01 oz or 0.9% higher than yesterday. It also rolleth over. 20 DMA at 667.38.
That US dollar index really smashed its chart yesterday. Closed through the uptrend line AND the 50 DMA, and looks sick as a yellow cat eating bad meat. Oh, today it scrabbled around and climbed 7.2 basis points, which you could almost see with a 10 power microscope. Ended at 82.751. A close below 82.50 will accelerate its weight-loss program.
I ought to be ashamed of myself. Making fun of the scrofulous, no-good, unbacked and spineless fiat currencies. Smacks too much of shooting fish in a rain barrel — with a 12 gauge shotgun. 'Tain't no hard target to hit.
Anyhow, yesterday's touch on the 50 DMA scairt that yen plumb to death. Today it shrank 0.55% to 99.98 cents/Y100. Euro edged up a microscopic 0.7% to $1.3085. Important to note there is that close lies above and outside the downtrend line, so promises more upside.
By the way, don't forget that the US bond market has broken down, forecasting a lower dollar and higher interest rates. I'm so sorry, Ben.


text:
Prakash Kumar1,
Shashi Ranjan Prasad1,
Anushil Anand2,
Rajneesh Kumar1 &
Sajalendu Ghosh2
Familial hypercholesterolemia (FH) is an under-diagnosed autosomal co-dominant genetic disorder characterized by very high plasma levels of low-density lipoprotein cholesterol (LDL-C), premature coronary artery disease (CAD) with arcus cornealis, and xanthomas. Among patients with CAD, the frequency of FH is significantly higher than that of the general population, but little data are available in India in this regard. This study aimed to assess the prevalence of FH in patients with premature coronary artery disease for the first time in the Jharkhand population.
The study was conducted on 200 premature CAD patients at RIMS hospital, Ranchi, from January 2020 to June 2021 with CAG-confirmed acute coronary syndrome. The study, without taking the aid of genetic profiling of the patients and using the Dutch Lipid Clinic Network Criteria, revealed quite a high (23.5%) prevalence of potential FH in patients with premature CAD apart from the conventional risk factors. Mean LDL-C levels among patients with definite, probable, possible, and no FH were recorded as 250.39, 184.32, 136.11, and 108.09 mg/dl, respectively. Arcus cornealis was seen in 55.31% of patients with potential FH, 90% in definite FH, and 44.40% with probable FH. Patients with potential FH were more likely to be younger (age < 40 years) males, having a history of CAD and a family history of premature CAD as compared to patients without FH.
There was no previous report of large studies on FH or its epidemiology and its natural history from India. The present study is the first one to show a high prevalence of potential FH in premature CAD (about 23.5%). This preliminary study revealed that the prevalence of FH in patients with premature CAD who came to the tertiary care hospital of Ranchi, Jharkhand, was high, apart from the conventional risk factors.
Familial hypercholesterolemia (FH) is an under-diagnosed autosomal co-dominant genetic disorder characterized by very high plasma levels of low-density lipoprotein cholesterol (LDL-C), xanthomas, arcus cornealis (AC), and premature coronary artery disease (CAD). Tendon xanthomata (TX) are widely regarded as a specific physical sign, but are often absent and may be difficult to assess. FH increases the risk of early CAD by up to 20-fold [1, 2]. It is the most common monogenic disorder leading to premature CAD and cardiac death.
Among patients with CAD or other atherosclerotic diseases, the frequency of FH is significantly higher than that of the general population showing that these patients are at particularly elevated risk of recurrent events [3]. In different ethnic groups across the USA, the prevalence of FH varies [4] and less than 10% of individuals with FH were diagnosed [5, 6]. The estimated prevalence of heterozygous FH (HeFH) is widely variable from 1/200 to 1/500 which contributes to its diagnostic difficulty [7, 8], and homozygous FH (HoFH) is 1/106 in the general population in the west [1]. There are very little data in India about the prevalence of FH. FH is primarily caused by mutations in the gene encoding the low-density lipoprotein receptor (LDLR). Less frequent mutations in the APOB and PCSK9 genes have similar functional consequences [9].
The Dutch Lipid Clinic Network criterion has been used extensively to identify the HeFH phenotype, which is based on LDL-C level, family history of FH, presence of tendon xanthoma, and arcus cornealis. Diagnosis of FH is a key factor in the prevention as this would lead to early recognition of FH in a family member of the index case and in preventing the morbidity and mortality due to CAD by lifestyle modification and judicial usage of hypolipidemic drugs.
The most efficient method of cascade testing for FH is to look for genotyping to relatives of index patients with an identified mutation, but it is in its infancy at the present situation in India. This is better than using family cholesterol testing alone as it is nonspecific [10].
The underdiagnosis of FH in the general population has been recognized as an important issue. For many patients, unaware of the disease, the first clinical manifestation may be acute coronary syndrome (ACS). The number of large-scale studies of FH or its epidemiology and natural history reported from India is close to nil. Although many studies have been conducted in India to study the lipid profile in acute coronary syndrome (ACS), there has been little mention of FH in these publications. There are huge gaps among Asian countries about the knowledge, frequency of occurrence, and care of FH. To fill this gap, it was aimed to assess the prevalence of FH in patients with premature coronary artery disease.
This study was conducted on 200 patients of premature CAD, who were admitted to the Ward/CCU of the Department of Cardiology RIMS, Ranchi, Jharkhand, from January 2020 to June 2021 with CAG-confirmed acute coronary syndrome (ACS). ACS is defined as severe chest pain or equivalent ischemic discomfort (dyspnea and epigastric discomfort) and has at least one of three features: (1) It occurs at rest (or with minimal exertion) lasting for more than 10 min. (2) It is of recent onset (i.e., within the prior 2 weeks), and/or (3) it occurs with a crescendo (upsurge) pattern.
Sample size calculation
Based on clinical experience and study of the literature, the prevalence of the FH in premature CAD was expected to be about 15%. The sample size was calculated with a 5% margin of error and a 5% level of significance. The required minimum sample size was found to be 180 as per the following formula.
Formula used:
$$n = Z^{2}_{\alpha /2} \,pq/d^{2}$$
p is the expected prevalence of Familial hypercholesterolemia in patients of premature coronary artery disease and:
$$q = 1 - p$$
d is the margin of error.
Zα/2 is the ordinate of standard normal distribution at α/2% level of significance.
Patients of premature CAD with age 45 years or less for males and age less than 55 years for females based on the study by van Loon et al. [11].
Patients with advanced liver/kidney disease.
Pregnant and lactating women.
Patients with hypothyroidism.
Patients with cancer
Patients fulfilling the inclusion criteria were enrolled in the study.
The following data were collected:
Demographics: name, age, gender, address, etc.
Clinical history and risk factors for coronary artery diseases.
A family history of premature coronary artery diseases, high LDL-C, and tendon xanthomas.
A physical examination of the patient was carried out to look for clinical features of FH such as hypercholesterolemia, tendon xanthomas, arcus cornealis, coronary artery disease, peripheral artery disease, and cerebrovascular artery disease.
Blood sample for serum lipid profile was collected at the time of admission either in fasting or nonfasting state, and blood sample was sent for biochemical analysis in the department of Biochemistry of RIMS hospital, Ranchi, Jharkhand. The LDL cholesterol was estimated according to the clearance method followed by the enzymatic method (cholesterol oxidase peroxidase method). Total cholesterol, VLDL, HDL cholesterol, and triglycerides were also estimated.
Patients were diagnosed as clinical FH according to the Dutch Lipid Clinic Network criteria (Table 1) which incorporate clinical history, physical examination, family history, and LDL-C level.
Table 1 The Dutch Lipid Clinic Network criteria
Method of study
The patients with confirmed CAD on coronary angiography were enrolled. Demographic details, clinical history, family history, and treatment history were taken. Physical stigmata of familial hypercholesterolemia like arcus cornealis and tendon xanthoma were examined. Lipid profile was estimated and for a patient on regular cholesterol-lowering medication in whom a pre-treatment LDL-C was not available, and an estimate of untreated LDL-C was obtained by multiplying the measured LDL-C by the validated LDL-C correction factor. Then according to DLCN criteria (Table 1), patients were divided into definite FH, probable FH, possible FH, and no FH groups. This was followed by an analysis of data among the different groups.
Descriptive statistics were analyzed with SPSS version 17.0 software. Continuous variables were presented as mean +/− SD. Categorical variables were expressed as frequencies and percentages. Association between two categorical variables was done using Chi-squared or Fisher's exact test.
A total of 200 premature CAD patients were included from the OPD/wards/CCU of the Cardiology department of RIMS hospital at Ranchi, Jharkhand. Among the premature CAD patients, 77% were male and the rest were female. In the present study, genetic testing was not carried out by DNA sequencing of candidate genes (LDLR, APOB, PCSK9) and thus premature CAD patients were classified as definite, probable, possible, or no FH (familial hypercholesterolemia) patients only using the most effective DLCN (Dutch Lipid Clinic Network) criteria. The prevalence of different categories present in this study calculated by DLCN criteria points can be summarized in the following pie chart (Fig. 1).
Pie diagram shows the relative percentage of occurrence of different types of familial hypercholesterolemia as classified using the most effective DLCN (Dutch Lipid Clinic Network) criteria because genetic testing was not done by DNA sequencing of candidate genes (LDLR, APOB, PCSK9) in this study
Definite familial hypercholesterolemia in patients with premature CAD was 5.5%, while the prevalence of potential FH [combined definite + probable FH (18%) was 23.5% and the prevalence of possible FH was 26.0% (Table 2).
Table 2 Prevalence of potential, possible, and no FH
The mean age of patients was 40.78 years at the time of enrollment in the hospital, but for patients with definite FH, the same was 35.06 ± 5.64 years, which was younger and statistically significant than other FH categories (Table 3).
Table 3 Mean age of FH patients
Relatively nonspecific diagnostic sign of AC (Arcus cornealis) was present only in 13% (26) of patients with premature CAD, but among potential FH patients, the incidence of AC was fairly high about 55.31% (26/47) (p value < 0.00001) and within definite FH group it was almost 90%.Untreated LDL-C levels from 155 to 189 mg/dL in 18.21% of patients and 190–249 mg/dL in 10% of patients were detected. Coronary angiography showed ~ 36% and 34% of patients having a single (SVD)- and double-vessel disease (DVD), respectively, while TVD was reported in less number of patients (~ 28%). Almost 49% were having a positive family history of premature CAD and 58 (29.0%) patients were smokers. A comorbidity symptom like diabetes was high (42%) as compared to hypertension (28%) in the premature CAD patients. The correlation of these two comorbidities cannot be correlated with the FH-related premature CAD incidence (Tables 4, 5).
Table 4 Frequency of diabetes in FH with CAD patients
Table 5 Frequency of hypertension in FH patients
A positive correlation of FH can be drawn with patients having a positive family history of premature CAD (Table 6).
Table 6 Frequency of patients having a positive family history of premature CAD
Details of all the baseline characteristics of the patients are grouped in Table 7.
Table 7 Baseline characteristics
The majority of the definite FH group were male patients (10 out of 11), and the comparative age group distribution of different FH groups showed that 81.81% of definite FH belong to the less than 40 years of age cluster (Fig. 2).
The relative percentage of premature CAD patients that were classified by using the most effective DLCN (Dutch Lipid Clinic Network) criteria grouped into two age categories. The less than 40 years of age group showed more prevalence in definite and possible FH types
A statistically significant positive correlation of FH can also be drawn with patients with different levels of LDL-C concentration. Mean concentrations of LDL-C among patients with definite FH, probable FH, possible FH, and no FH group was 250.39 ± 39.88, 184.32 ± 23.41, 136.11 ± 79.72, and 108.09 ± 12.72.72, respectively (Fig. 3).
The relation of the presence of mean LDL-C concentration in blood plasma with different types of FH patient groups. The maximum concentration was found in definite FH, and the minimum concentration was found who had no FH detected by DLCN criteria. This finding very well relates the increase in LDL-C directly related to the FH occurrence in premature CAD patients
Coronary angiography findings: Among patients with premature CAD with FH, single-vessel disease (SVD) was more common in probable FH and possible FH groups than in definite FH. Multi-vessel disease (DVD/TVD) was more frequent in the potential FH group (72.34%) than in the possible FH group (51.92%) (p < 0.00001) (Fig. 4).
Multi-colored bar diagram showing more occurrences of DVD in definite FH and TVD in probable FH patients, while patients with no FH had an almost equal probability of developing SVD, DVD, and TVD
This is the first study to estimate the prevalence of FH in premature coronary artery disease in Jharkhand and may be the first study to address the issue of a large number of patients in India. The diagnosis of FH in patients with premature CAD has been under-recognized throughout India. The present study shows that FH is common in patients with premature coronary artery disease, and it is relatively easily diagnosed clinically without the aid of a genetic screening process.
Out of 200 premature CAD patients attended either in the OPD or wards or CCU of the Department of Cardiology, RIMS, Ranchi, females and males were 46 (23%) and 154 (77.0%) in number, respectively. The mean age of patients was 40.78 ± 6.95 years; the mean age of female and male patients was 47.48 years and 38.64 years, respectively. This ratio showed that the onset age of CAD occurs earlier in male patients than in female patients in Jharkhand. Different types of FH as classified by clinical observations as directed by DLCN criteria were shown to have different percentages of the previous history of CAD in patients in the population of Jharkhand, i.e., 18.18%, 16.70%, 11.30%, and 5.36% patients had a history of the previous CAD in patients diagnosed with definite FH, probable FH, possible FH, and no FH, respectively. It was also observed that 8.5% of the total 200 early CAD patients had a history of dyslipidemia.
The present study showed a high prevalence of FH in premature CAD as compared to previous studies in different parts of the world, based on clinical scoring systems according to DLCN criteria. The prevalence of definite familial hypercholesterolemia was 5.5%, and probable FH was 18.0% in patients with premature coronary artery disease. The prevalence of possible FH was 26.0%, which was fairly high compared to the theoretically estimated percentage of prevalence of heterozygous FH (1/500). The present study showed that the prevalence of potential FH (combining both definite and probable FH) is 23.5% which was even higher than 12.3% as reported by Dorsch et al. [12] among 404 patients of ACS surveyed.
A similar trend of lesser FH was also reported from Singapore by Yudi et al. [13] that included 210 premature CAD patients in the Gold Coast Hospital for 12 months and got 0% definite FH, 1% (3) probable FH, 24% (50) possible FH patients, and 60 (29%) had unlikely FH. The prevalence of FH was very low as compared to the present study. Gaudet et al. [14] reported a genetically confirmed prevalence of 16.4% FH including LPL-C deficient patients among 412 French Canadian men of less than 60 years of age. We report here a prevalence of 23.5% potential FH in premature CAD patients among the population of Ranchi, Jharkhand. Faggiano et al. [15] from Australia studied 175 patients admitted to a coronary care unit with CAD at age < 60 years. Based on modified phenotypic DLCN criteria, the estimated prevalence of probable/definite FH in early onset CAD was 14.3% (95% confidence interval).
In the present study, angiography revealed FH patients with premature CAD exhibited more SVD in the probable (33.33%) and possible (48.07%) FH group than in the definite FH (9%) group. Among multi-vessel diseases, DVD and TVD were more frequent in the potential FH group (23.70% and 31.57%) than in the possible FH group (20.58% and 22.80).
Our present study has several limitations. Firstly, the lipid measurements were performed in different laboratories. An inter-laboratory variation could have conditioned the prevalence of potential FH. Secondly, there were patients in whom the "untreated LDL-C levels" were not measured but estimated using a validated correction formula, and lastly, genetic analysis was not performed in potential FH patients to confirm the diagnosis.
The present study is the first one to show a high prevalence of FH in premature CAD (about 23.5% for potential FH). To the best of our knowledge, there were no reported large studies of FH or its epidemiology and natural history from Indian patients. Many studies in India have been carried out to study the lipid profile of acute coronary syndrome in Indian patients, but there has been little mention of FH in these publications. Only one previous study, which described clinical profiles and treatment patterns of 997 patients with premature CAD, just briefly mentioned that there was a 1.3% prevalence of possible FH in the study population. This preliminary study revealed that the prevalence of FH in patients with premature CAD who came to the tertiary care hospital of Ranchi, Jharkhand, was high, apart from the conventional risk factors.
All the raw data and materials mentioned in this manuscript were kept unaffectedly to be produced before the publishing authority, if required.
FH:
Double-vessel disease
TVD:
Triple-vessel disease
SVD:
Single-vessel disease
DLCN:
Dutch Lipid Clinic Network
ACS:
OPD:
CCU:
LDL:
Low-density lipoprotein
VLDL:
Very low-density lipoprotein
HDL:
High-density lipoprotein
Arcus cornealis
RIMS:
Rajendra Institute of Medical Science
CAG:
Coronary artery angiography
HeFH:
Heterozygous familial hypercholesterolemia
HoFH:
Homozygous familial hypercholesterolemia
APOB:
Apolipoprotein B
PCSK9:
Proprotein convertase subtilisin/kexin 9
Nordestgaard BG, Chapman MJ, Humphries SE, Ginsberg HN et al (2013) Familial hypercholesterolemia is underdiagnosed and undertreated in the general population: guidance for clinicians to prevent coronary heart disease: consensus statement of the European Atherosclerosis Society. Eur Heart J 34:3478–3490. https://doi.org/10.1093/eurheartj/eht273
Goldberg AC, Hopkins PN, Toth PP, Ballantyne CM, Rader DJ, Robinson JG et al (2011) Familial hypercholesterolemia: screening, diagnosis, and management of pediatric and adult patients: clinical guidance from the national lipid association expert panel on familial hypercholesterolemia. J Clin Lipidol 5:1–8. https://doi.org/10.1016/j.jacl.2011.04.003
Backer GD, Besseling J, Chapman J, Hovingh GK, Kastelein JJ, Kotseva K et al (2015) Prevalence and management of familial hypercholesterolemia in coronary patients: an analysis of EUROASPIRE IV, a study of the European Society of Cardiology. Atherosclerosis 241:169–175. https://doi.org/10.1016/j.atherosclerosis.2015.04.809
Shah NP, Ahmed HM, Wilson Tang WH (2020) Familial hypercholesterolemia: detect, treat, and ask about family. Cleve Clin J Med 87(2):109–120. https://doi.org/10.3949/ccjm.87a.19021
Mundal L, Retterstøl K (2016) A systematic review of current studies in patients with familial hypercholesterolemia by use of national familial hypercholesterolemia registries. Curr Opin Lipidol 27(4):388–397. https://doi.org/10.1097/MOL.0000000000000300
Myers KD, Knowles JW, Staszak D et al (2019) Precision screening for familial hypercholesterolemia: a machine learning study applied to electronic health encounter data. Lancet Digit Health 1(8):e393–e402. https://doi.org/10.1016/S2589-7500(19)30150-5
Lui DTW, Lee ACH, Tan KCB (2021) Management of familial hypercholesterolemia: current status and future perspectives. J Endocr Soc 5(1):bvaa122. https://doi.org/10.1210/jendso/bvaa122
Khera AV, Hegele RA (2020) What is familial hypercholesterolemia, and why does it matter? Circulation 141(22):1760–1763. https://doi.org/10.1161/CIRCULATIONAHA.120.046961
Austin MA, Hutter CM, Zimmern RL, Humphries SE (2004) Genetic causes of monogenic heterozygous familial hypercholesterolemia: a HuGE prevalence review. Am J Epidemiol 160:407–420. https://doi.org/10.1093/aje/kwh236
Datta BN, McDowell IF, Rees A (2010) Integrating provision of specialist lipid services with cascade testing for familial hypercholesterolemia. Curr Opin Lipidol 21:366–371. https://doi.org/10.1097/MOL.0b013e32833c14e2
van Loon JE, de Maat MP, Deckers JW, van Domburg RT, Leebeek FW (2012) Prognostic markers in young patients with premature coronary heart disease. Atherosclerosis 224(1):213–217. https://doi.org/10.1016/j.atherosclerosis.2012.06.067
Dorsch MF, Lawrence RA, Durham NP, Hall AS (2001) Familial hypercholesterolemia is under-diagnosed after acute myocardial infarction. BMJ 322:111–114
Yudi M, Omera L, McCubbery N, Dick S, Jayasinghe R, Hamilton CI (2012) Suboptimal consideration and management of potential familial hypercholesterolemia in patients with suspected premature coronary artery disease. Singapore Med J 53:174–178
Gaudet D, Vohl MC, Julien P, Tremblay G, Perron P, Gagne C et al (1998) Relative contribution of low-density lipoprotein receptor and lipoprotein lipase gene mutations to angiographically assessed coronary artery disease among French Canadians. J Cardiol 1998(82):299–305. https://doi.org/10.1016/s0002-9149(98)00328-2
Faggiano P, Pirillo A, Griffo R, Ambrosetti M, Pedretti R, Scorcu G et al (2018) Prevalence and management of familial hypercholesterolemia in patients with coronary artery disease: the heredity survey. Int J Cardiol Heart Vasc 252:193–198. https://doi.org/10.1016/j.ijcard.2017.10.105
Authors are thankful to the Department of Cardiology, RIMS Hospital, Ranchi, Jharkhand, India, for providing with the participants and diagnosis facilities for this research work.
The present work was not supported by any departmental research grant or any Government organizations.
Department of Cardiology, RIMS Hospital, Ranchi, Jharkhand, India
Prakash Kumar, Shashi Ranjan Prasad & Rajneesh Kumar
Department of Zoology, Dr. Shyama Prasad Mukherjee University, Ranchi, Jharkhand, India
Anushil Anand & Sajalendu Ghosh
Prakash Kumar
Shashi Ranjan Prasad
Anushil Anand
Rajneesh Kumar
Sajalendu Ghosh
SR, RK, and PK analyzed and interpreted the patient data. AA took the data and consent letter of the patients, and SG was a key contributor in writing the manuscript. All authors read and approved the final manuscript.
Correspondence to Prakash Kumar.
The work was conducted within the department of cardiology, Rajendra Institute of Medical Science, Ranchi, Jharkhand, India, and the ethical committee report was taken from the same Hospital's Ethical Committee. Filled up consent form with signature of patients was taken.
Written consent for publication of the data from each of the study participants was also taken.
Kumar, P., Prasad, S.R., Anand, A. et al. Prevalence of familial hypercholesterolemia in patients with confirmed premature coronary artery disease in Ranchi, Jharkhand. Egypt Heart J 74, 83 (2022). https://doi.org/10.1186/s43044-022-00320-7
Xanthomas


Enjoy the specialties of the house like the Villa's Ravioli and accompany them with the traditional Juan Perez Jolote drink prepared from a regional spirit of the State of Chiapas.
During your pleasant stay enjoy our five-course tasting menu with typical dishes of Chiapas Cuisine, pairing two wines.
Hours from 7:00 A.M. to 11:00 A.M. and 6:00 P.M. to 11:00 P.M.
Room service available from 7:30 A.M. to 10:30 P.M.


Method 2: Open the address http://www.chinajob.com , click the "Evaluation System for Foreign Experts" on the bottom of the page.
4. Appear "User Register- Password" page. Fill in the contents: "Password"→"Password Confirm"→"Password Question"→"Answer"→Click "Next" button.
5. Appear "User Register - Basic Information of Foreign Language Expert" page. Please fill in the basic information. The items marked with ** must be filled. Then click the "Complete" button.
6. After finishing the above steps, the registration is completed.
1. Fill in your user ID and password on the home page, then click "OK" button.
2. Appear "About this System" page. Please finish the "Background Information", "Psychological Test", "Elementary Test", "Writing Examination" one by one.
a) Appear the "Basic Information" page. Fill the form→click "Submit this form" button.
b) Appear "Job Objectives" page. Fill the form→click "Submit this form" button.
c) Appear "Education Background" page. Click "Adding…"→ Appear "Education Background" window → Finish the form → Click "Save" → Click "Next step" button.
e) Appear "Other Working Experience" page. Click "Adding…"→ Appear "Other Working Experience" window → Finish the form → Click "Save" → Click "Next step" button.
f) Appear "Training" page. Click "Adding…"→ Appear "Training" window → Finish the form → Click "Save" → Click "Next step" button.
g) Appear "Volunteer Experience" page, Click "Adding…"→ Appear "Volunteer Experience" window → Finish the form → Click "Save" → Click "Next step" button.
h) Appear "Awarding" page. Finish the form → Click "Submit this form" → Click "Next step" button.
i) Appear "Certifications" page. Click "Browser" to select a file → Click "Upload" to upload → Click "Back to home".
Notes: In every step above, if there is nothing to fill in, please click "Next step".
a) Appear "Psychological Test" page. READ "NOTICE" AND UNDERSTAND THE REQUIREMENTS FOR THIS TEST.. Click the "Start a new Psychological Test" button to start.
b) Appear "Psychological Test" window. Test begin and timing.
c) After finishing the test, click "Submit" button.
a) Appear "Knowledge Test" page.READ "NOTICE" AND UNDERSTAND THE REQUIREMENTS FOR THIS TEST. Read the "Notice" before doing the test. Click the "Start a new Elementary Test" button to start.
a) Appear "Writing Test" page. READ "NOTICE" AND UNDERSTAND THE REQUIREMENTS FOR THIS TEST.Click the "Start a new Writing Test" button, a topic for the writing will be appeared. Please write an essay for about 800 words.
After you finish all the steps above, the Evaluation is completed. You can check the result by clicking "My Evaluation Report" on the left of the page.


One of the perks of working in the Logistics industry is that you can take advantage of its skillsets at any time! At R+L Carriers, we believe in investing into a variety of candidates, such as high school, college, and trade school students, many of whom are at different stages in their life.
The Hometown Showcase hauler traveled all the way to Missouri to talk to the students of Crowder College and McDonald County High School on Sept. 28 and 29. With two of our Service Centers stationed in Missouri, we wanted to capitalize on the opportunity to introduce these students to an important industry that provides a variety of options for their future right in their backyard.
On Sept. 28, the Hometown Showcase team set up the hauler in front of Crowder College in Neosho, MO for students to interact with and get hands-on experience. From looking at the engine of the hauler to interacting with different portions inside the trailer like the Bose Seating simulation, students had a great time learning about the different positions that R+L Carriers has to offer. After this visit, many applications were filled out.
Since Crowder College is a community college, students consist of both college and high school students. The next day, the hauler drove down the street to hang out and talk logistics with the high school students of McDonald County High in Anderson, MO.
At R+L Carriers, we strive to provide students with the knowledge of a career that has many options.
We understand that some students in high school have not selected a career path yet. At R+L Carriers, we strive to provide those students with the knowledge of a career that has many options. The logistics industry is vital to the function of our everyday life. The industry also shows no signs of a decrease in growth. There are plenty of jobs available for those with a variety of different skillets.
During the school day, The Hometown Showcase team gave a presentation to each class about logistics and its impact on our everyday life. The students also had a chance to interact with the hauler and see the exciting parts of the company with hands-on experience. Students also applied for R+L Carriers' positions at the event. Later that night, The Hometown Showcase hauler parked at the football game, where the McDonald County Mustangs beat the Monett Cubs 21-18 in a close and exciting game. The fans at the game got to enjoy the game and interact with the trailer.
Bucky H., was the winner of the Hometown Achievement Award, received before the game.
It wasn't only the win that made this evening so great, but The Hometown Showcase team also got a chance to donate $1,000 to McDonald County High School and present a $1,000 scholarship to a student of the school's choice. Bucky H., the winner of the Hometown Achievement Award, received the award before the game. Congratulations, Bucky!
Our visits to Crowder College and McDonald County High School were full of fun and logistics. We always enjoy educating students about the industry and what it has to offer. Are you interested in the Hometown Showcase coming to your school or next community event? We are actively scheduling future events. Contact us today for more information!


text:
2017 Oklahoma Sooners Football Countdown To Kickoff | 11 Days!
Dede Westbrook gave Oklahoma and college football fans quite a show in 2016.
By Seth Oliveras@SethOliveras Aug 22, 2017, 8:53am CDT
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Photo by Brett Deering/Getty Images
Happy Tuesday, OU fans! We are now only 11 days away from the return of Oklahoma Sooners football! In today's edition of our countdown to kickoff we will highlight a player known for his highlights. We present to you the 2016 Biletnikoff Award winner and Heisman Trophy finalist, Dede Westbrook.
At 6'0" tall and 176 pounds, Westbrook has had to heavily rely on his primary advantage — speed. Unfortunately for the defensive backs he lines up against, his amount of speed is simply unfair.
Westbrook arrived in Norman from Blinn Community College in 2015. While he was regarded as one of the better junior college prospects in his class, the Oklahoma receiver that received the most attention at the time was Sterling Shepard, and justifiably so.
Westbrook had a solid first season and complimented Shepard well, recording 743 yards on 46 receptions with four touchdowns. Shepard would move on to the NFL after the season, and people were left wondering who would step up and become Baker Mayfield's go-to pass catcher.
In 2016, something amazing unfolded. After dealing with a hamstring injury during the first three games of the season, Dede exploded onto the scene. Game after game after game, Westbrook was re-writing the record books with 100-yard games, 200-yard games and multiple touchdowns per contest. Perhaps the most incredible part of his run was that aside from his sheer consistency, the plays he was making were all so consistently spectacular.
On October 8th, in the Red River Shootout, Westbrook had a game that would turn out to be his best, statistically, of the season. With 10 receptions, Dede totaled 232 yards and scored three touchdowns. His first score was a 71-yard bomb courtesy of Baker Mayfield, the next a display of phenomenal route running for a 42-yard TD, and his third was from 47-yards out in which he turned on the jets and zoomed right past seemingly the entire UT defense.
Overall, Dede Westbrook finished the season with 80 catches for 1,574 yards and 17 touchdowns, leading all Power 5 receivers in receiving yards and receiving touchdowns. His historic season made him a unanimous First Team All-American, and he became OU's first ever Biletnikoff Award winner, which is given to the nation's top receiver. Westbrook was also a finalist for the Heisman Trophy, along with his teammate Baker Mayfield, finishing 4th in the voting.
Westbrook has since turned the page on his Sooner career. He entered the 2017 NFL draft and was selected by the Jacksonville Jaguars in the 4th round. Westbrook has been making a lot of noise in the preseason, and in his first preseason game, he did this:
Dede is speedy.
The @OU_Football alum, @DedeTHEGreat11 gets his first NFL touchdown of his career.#WinToday #JAXvsNE pic.twitter.com/8ip1cwzinY
— Jacksonville Jaguars (@Jaguars) August 11, 2017
In his second preseason game against the Tampa Bay Buccaneers, Westbrook recorded an eye-popping 131 yards on six receptions. If there's one thing us Oklahoma fans know about Dede, it's that he has a knack for big plays, and by all accounts, he looks primed and ready to showcase his extraordinary ability in Jacksonville, too.
Follow Crimson & Cream Machine on Twitter!


Located in the heart of Jakarta City that is Sudirman Central Business District, Menara Mandiri offers the best location with the best access and would be the right choice for your Wedding Ceremony.
With its easy access, your beloved ones could easily attend the wedding and share the happiness with you on the happiest day of your life. Menara Mandiri offers two exclusive ballrooms that could accommodate guests from 600 people to 1700 people. Through our 1.035 m2 ballroom and 1.360 m2, we offer you high ceilings ballrooms that could be perfect for both traditional and international weddings, as well as national weddings. Menara Mandiri works together with so many well-known vendors in Jakarta, with high quality products and high quality services.


Four years ago this month, archeologists monitoring the excavation of the former World Trade Center site uncovered a ghostly surprise: the bones of an ancient sailing ship. Tree-ring scientists at Columbia University's Lamont-Doherty Earth Observatory were among those asked to analyze its remains for clues about its age and origins. In a study now out in the journal Tree Ring Research, the scientists say that an old growth forest in the Philadelphia area supplied the white oak used in the ship's frame, and that the trees were probably cut in 1773 or so—a few years before the bloody war that established America's independence from Britain.
Key to the analysis was wood sampled from Philadelphia's Independence Hall two decades earlier by Lamont tree-ring scientist Ed Cook. It turns out that growth rings still visible in the building's timbers matched those from the World Trade Center ship, suggesting that the wood used in both structures came from the same region. As trees grow, they record the climate in which they lived, putting on tighter rings in dry years and wider rings in wet years. In the process, a record of the region's climate is created, allowing scientists to see how Philadelphia's climate differed hundreds of years ago from say, New York's Hudson Valley. The climate fingerprint also serves as a kind of birth certificate, telling scientists where pieces of wood originated.
The ship itself has been tentatively identified as a Hudson River Sloop, designed by the Dutch to carry passengers and cargo over shallow, rocky water. It was likely built in Philadelphia, a center for ship-building in Colonial times. After 20 to 30 years of service, it is thought to have sailed to its final resting place in lower Manhattan, a block west of Greenwich Street. As trade in New York harbor and the young country flourished, Manhattan's western shoreline inched westward until the ship was eventually buried by trash and other landfill. By 1818, the ship would have vanished from view completely until the terrorist attacks on Sept. 11, 2001 set in motion the events leading to the World Trade Center's excavation and rebirth. The ship's removal from Ground Zero and the analysis of its timbers and some of the artifacts uncovered are described in the photo essay above.
All images courtesy of Lower Manhattan Development Corporation unless otherwise stated.


Love is a great medicine.
That was darn nice of them .
Seriously cool! Happy Saint Valentine's Day... patron saint of happy marriages, engaged couples.
I was at a nursing home this week where the staff wasn't doing as well, so I am very glad to see this. It's so good to know these people are out there!
BTW, the staff at the other facility was putting out donated funeral flowers (which is fine), but in the original fireside baskets and obviously funeral arrangements. I had to object. One should NEVER put funeral arrangements out by dying patients. Never. They need to be transferred to vases and arranged.
I've encountered it before, and I think that sometimes people just don't stop to think the way the nurses in the above report did. I am SO glad the nurses in this report are there.
That's a lovely story! And it's a sign of a good hospital too that they had the time and opportunity to do so.


An ESOP participant was not entitled to statutory penalties, despite an ESOP trustee's failure under ERISA §104(b)(4) to provide the participant with bank statements associated with the sale of company stock, the U.S. Court of Appeals in San Francisco (CA-9) has ruled.
An ESOP trustee, acting on advice given to him by a TPA, declined to provide a participant with copies of official bank statements related to the sale of all company stock to a new owner. The participant filed suit seeking (1) statutory penalties for the failure to provide the documents and (2) an accounting, in part because the trustee allegedly received $100,000 in compensation for the sale of stock privately owned by him. A magistrate judge denied the motions.
The participant failed in his initial complaint to properly plead his request for penalties and never availed himself of the opportunity to amend his complaint afforded him by the magistrate at a case management conference. Further, the appellate court held, the magistrate did not abuse his discretion when he ruled that, even if the request for penalties had been properly pled, he would have declined to impose penalties. The trustee acted not in bad faith but upon professional advice, and the participant demonstrated no harm from the delay (the trustee had responded promptly to an interim order from the magistrate to produce the bank statements).
The appellate court also upheld the magistrate's denial of the participant's request for an accounting. No evidence suggested the trustee breached any fiduciary duty by receiving payment for the sale of his privately owned stock shares. Common stock transactions that resulted in the ESOP owning less than 100% of the stock occurred before formation of the ESOP and are not relevant to ERISA claims.
Source: Kronzer v. Hintz (CA-9).


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Betting could be entertaining when had fun with the right attitude and one may win a game with the aid of betting advice given by unbiased players.