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Monday Micro – Ebola outbreak in Guinea
By Siouxsie Wiles • 24/03/2014 • 36
The news is full of reports of an outbreak of Ebola in the west African country of Guinea. Ebola is one of a group of related viruses which cause viral haemorrhagic fever. There is no vaccine or treatment for these viruses. So far reports are that there have been at least 80 cases with 59 deaths.
Here's a little FAQ for those of you wanting to know more about these amazing and terrifying viruses.
1. What is Ebola?
Ebola is one of two members of the Filoviridae family of single-stranded RNA viruses. The other member is Marburg. There are five types of Ebola virus, named after where outbreaks first appeared, and which differ in their mortality rates:
(i) Zaire ebolavirus (ZEBOV) – most frequent cause of outbreaks (last in the Democratic Republic of Congo 2008) and highest mortality rate (47-100%)
(ii) Sudan ebolavirus (SEBOV) – mortality rate of 42-65%, last outbreak in 2012 in Uganda
(iii) Reston ebolavirus (REBOV) – Discovered in 1989 when there was an outbreak of disease in laboratory macaques in Reston, Virginia, USA. Has since been found in nonhuman primates in Pennsylvania, Texas and Italy. Did not cause disease in exposed human laboratory workers*
(iv) Côte d'Ivoire ebolavirus (CIEBOV) (or Tai ebolavirus) – killed chimpanzees in Tai national park in 1994, only human case was in one of the scientists performing the necropsies on the infected chimps but they recovered
(v) Bundibugyo ebolavirus – two outbreaks to date (2007 in Uganda and 2012 in the Democratic Republic of Congo), mortality rate of 34-50%
2. How is Ebola transmitted?
The primary mode of transmission of Ebola is unknown. Bats are thought to be the most likely reservoir (1). Some people have caught Ebola from contact with or by eating infected bushmeat. Secondary spread occurs through direct contact with infected patients, their body fluids or remains, so family members, hospital workers and those burying the dead are at greatest risk. The incubation period for infection ranges from 2 to 21 days (mean 4-9 days).
An interesting paper was published in the journal Scientific Reports in 2012 in which piglets were inoculated oronasally with Zaire ebolavirus (2). The piglets had a high temperature and rapid breathing for a few days but soon recovered. But they were infectious, spreading the virus through the air to macaques who developed symptoms of Ebola infection and had to be euthanised.
3. What's the largest recorded Ebola outbreak?
The biggest outbreak to date was in Uganda in 2000, when 425 people were infected with Sudan ebolavirus – 224 died. Ebola is a little like the great white shark of the virus world. Before this current outbreak, there had only been 2317 clinical cases and 1671 confirmed deaths in the last 50 years (3), a negligible number compared to many other infectious diseases.
4. Do people with viral haemmorrhagic fever really bleed out of every orifice before they die?
The name viral haemorrhagic fever is a bit of a misnomer. In less than 10% of Ebola cases, patients will experience bleeding from their mucous membranes, including those in the nose, gums, gastrointestinal tract and vagina. Death occurs by multiple organ failure. Most frequently infected people will have a fever with chills, malaise, joint and muscle pain. They also may have a rash. Other symptoms include nausea, diarrhoea, vomiting, stomach pains, throat and chest pain, a cough. From there patients experience severe headaches and confusion which can progress to delirium and coma.
5. Why is this such a big story?
With the exception of the dead chimps and the scientist who recovered in the Ivory Coast, Ebola has never been reported from west Africa; the majority of outbreaks have been in central Africa. What has alarmed many is that the cases have moved from forest communities to the capital city, Conakry. The capital has a port and airports and, with an estimated population of 2 million, will be difficult to quarantine should the need arise.
6. Can survivors continue to shed virus?
Yes. Survivors from an outbreak in 1995 in the Democratic Republic of the Congo which killed 244 of the 315 people infected, were followed for several months afterwards. Researchers tested their tears, sweat, faeces, urine, saliva, semen, and vaginal secretions. For semen, 4 of the 5 convalescents tested had at least 1 specimen that tested positive for Ebola genetic material. The latest semen sample was obtained over 3 months after the onset of infection (4). There were no cases of transmission from those individuals with positive semen samples.
* Go read Richard Preston's book The Hot Zone, which is all about the outbreak in Reston.
1. Leroy EM, Kumulungui B, Pourrut X, Rouquet P, Hassanin A, Yaba P, Délicat A, Paweska JT, Gonzalez JP, Swanepoel R (2005). Fruit bats as reservoirs of Ebola virus. Nature. 2005 Dec 1;438(7068):575-6.
2. Weingartl HM, Embury-Hyatt C, Nfon C, Leung A, Smith G, Kobinger G (2012). Transmission of Ebola virus from pigs to non-human primates. Sci Rep. 2012;2:811. doi: 10.1038/srep00811. Epub 2012 Nov 15.
3. Leroy EM, Gonzalez JP, Baize S (2011) Clin Microbiol Infect. 17(7):964-76. doi: 10.1111/j.1469-0691.2011.03535.x
4. Rowe A1, Bertolli J, Khan AS, Mukunu R, Muyembe-Tamfum JJ, Bressler D, Williams AJ, Peters CJ, Rodriguez L, Feldmann H, Nichol ST, Rollin PE, Ksiazek TG (1999). Clinical, virologic, and immunologic follow-up of convalescent Ebola hemorrhagic fever patients and their household contacts, Kikwit, Democratic Republic of the Congo. Commission de Lutte contre les Epidémies à Kikwit. J Infect Dis. 1999 Feb;179 Suppl 1:S28-35.
chimps
haemorrhagic fever
macaques
36 Responses to "Monday Micro – Ebola outbreak in Guinea"
Monday Micro – First documented cases of cat to human transmission of TB | Infectious Thoughts says:
[…] « Monday Micro – Ebola outbreak in Guinea […]
Sounds like the Ebola virus puts the body into an acute state of scurvy. Massive dosses of IV vitamin C should be given , at least to try to save these patients since modern medicine has no cure.
Siouxsie Wiles says:
April's Fool Jim?!
I don't joke about people losing their lives Siouxsie
Apologies, the idea of curing Ebola with high dose vit C was so absurd I thought you were joking. Now I'm just flabbergasted.
Grant Jacobs says:
This may be the same Jim who has touted vitamin C as a remedy for other illnesses on my blog.
Irrespective of that, the idea that vitamin C (or it's high-dose equivalent) is going to 'cure' Ebola is, to be polite, a bad idea.
Please tell me grant why would treating Ebola with vitamin c be a bad idea
I could ask you to first say why you think it's a good idea (you should answer this really, you're reversing the natural burden of proof as it were), but what you are suggesting is not much different to this homeopath who thinks that homeopathic remedies should be given to them. Everyone with a 'favourite' cure-all elixir rushes out and suggests it as the answer to the latest big medical story…
Hi Grant I suggest you google this study and read it
Phase I safety trial of intravenous ascorbic acid in patients with severe sepsis
Since patients die of multiple organ failure from the ebola virus this study shows that IV vitamin C not only is proof of safety but also helps prevent against organ failure.
Not to give IV vitamin C to help these patients leaves me flabbergasted. Please tell why doing so is a bad idea?
I replied earlier; my point there stands.
I haven't time to read others' papers as I have other things to do but it's worth remembering that phase I trials don't test for efficiacy (useful effect), but lack of obvious [short-term, direct] harm. (Which is likely why their conclusion re organ failure" reads "may", not "does".)
Interesting when you only read the conclusion to a trial as you miss the important bits
SOFA scores are robust indicators of mortality during critical illness. SOFA score increases during the first 48 hours of ICU care predict a mortality rate of at least 50% [26]. In this study, the extent of organ failure accompanying patients with severe sepsis was high with an average SOFA score for all patients equal to 11.4±3 confirming that multiple organ failure was present at enrollment. Given that the mean plasma ascorbic acid levels on admission were subnormal (17.9±2.4 μM), a mean initial SOFA score of 11.4±3 in patients with severe sepsis was not surprising. This study is in agreement with other studies which show that plasma ascorbic acid levels in severe sepsis correlate inversely with the incidence of multiple organ failure [13]. We showed that the addition of ascorbic acid to standard of care practice (i.e., fluid resuscitation, antibiotics, vasopressor medication) for patients with severe sepsis significantly reduced organ injury. Ascorbic acid treated patients exhibited prompt and sustained reductions in SOFA scores during the 4-day treatment regimen unlike placebo controls where SOFA scores slowly increased over time. SOFA score reduction was most remarkable in patients receiving the high dose ascorbic acid infusion (Figure 2).
C-reactive protein (CRP) [19] and procalcitonin (PCT) [20] levels are known to correlate with the overall extent of infection and higher levels of both have both been linked to higher incidences of organ injury and death in the critically ill. CRP in circulation has a short half-life of approximately 19 hours. Thus, the kinetics of CRP make it a useful monitor for tracking the inflammatory response produced by infection, and the response to antibiotic treatment. Lobo et al. reported that patients with CRP levels greater than 10 mg/dL at ICU admission exhibited significantly higher rates of multiple organ failure as well as higher mortality rates [34]. A decrease in CRP levels in Lobo's study after 48 hours was associated with a mortality rate of only 15.4%, while a persistently high CRP level was associated with a mortality rate of 60.9%. Both low and high dose ascorbic acid infusion in this trial promptly reduced serum CRP levels in septic patients (Figure 3A). Thus, the findings in this study support the findings of Lobo et al. with descending CRP levels being associated with lower mortality rate and reduced levels or organ failure.
Sorry about the copy an paste however as I have stated all along , these patients will have extremely low ascorbic levels, correcting that would lead to a more positive outcome than what medicine is doing for them at present. This in a nut shell is administering the essentials of life. Failure to do so in newzealand carries a seven year jail term from what I have been told.
Jim, as I tried to point out to you earlier, I other & better things to do; please direct your attention to others, thank you.
@Jim – you seemingly arbitrarily decide that ebola is scurvy, then cut & paste a small part of a paper that is about sepsis and wonder why people don't treat with vitamin C.
You can't draw a sensible conclusion without ensuring a sensible assumption at every single point in your argument.
Human beings don't like to be guinea pigs in experiments, you can't just guess at what sounds good for a treatment (especially from such a flimsy basis) and apply it. In the past people did run things like this, bad stuff happened and now we have rigorous medical regulations to ensure proper processes.
"I other" should read "I have other" – sorry.
Steve P says:
Just found this chain of posts after searching for the term Ebola and Vitamin C.
Very interesting topic and I find it strange that the comments against Vitamin C and possible treatment to improve the outcome of a high fatality disease such as Ebola is met with such resistance and scorn. On the basis of 'People do not like to be treated like Guinea Pigs' I am afraid lots of people would like the option of something that might work against doing nothing and dying a horrible death. It's interesting that Andrea uses the term Guinea Pig as they are also one of the 4 mammalian groupings that do not create Vitamin C in their Livers (Science Fact) the others are Apes, Fruit Bats and Humans (Science Fact). It is therefore a very strange coincidence that of the 3 carrier groups Fruit Bats are a confirmed reservoir for Ebola and the disease is transmitted through Apes and humans. All these groups do not produce Vitamin C themselves. It would be very interesting to see how Guinea Pigs fair when exposed to Ebola (this completes the 4 mammal set). Either you are ignorant of this fact or you choose to ignore it (ie in Science, Opinions don't count). In terms of the other points that Jim made yes I agree that the Sepsis treatment was not related to Ebola as a potential treatment, it just demonstrates that High Doses of Ascorbic acid (Vitamin C) can be consumed with no perceived detrimental effect to health. As you are aware no Healthcare Professional or Scientist can use 'does' as with 'cure' but have to include 'may' as the terminology as this outcome 'may' not be the case in the future or requires further scientific analysis. The response to this from Grant is that you don't have time to read research but back up your opinion with spurious evidence ie the reference to Homeopathy (non-science, this still doesn't mean it doesn't work) versus using Vitamin C as a possible treatment for Ebola. Vitamin C is a key constituent of Collagen making up skin and capillaries, veins and arteries (Science Fact). It also is the energy carrier to get energy to the mitochondria within the cell by its conversion to Coenzyme A and then plays an important part of the Krebs cycle (don't worry about Research just knowledge of basic Human Biochemistry will do). This reinforces the significant part Ascorbic Acid (Vitamin C) plays in the Human Body any reduction in Vitamin C ingestion will significantly affect processes in the body at a cellular level something like Ebola would and does wreak havoc.
So connecting the fact that Ebola presents haemorrhaging as a symptom which is basically blood seeping through the gaps in the collagen it would be entirely reasonable to relate the symptom to weakening or destruction of the capillaries transporting the blood. So let's theorise for a moment (an integral part of Science methodology and advancement) the Ebola virus is so virulent that the body reacts by first using all of the available vitamin c (the human body does not manufacture and store, therefore only can use what is ingested). The Ebola Virus or the body then starts to consume the ascorbic acid in the capillaries. This leads to blood seeping through the cracks in the collagen presenting haemorrhaging. A possible treatment to this presentation is high doses of Vitamin C which would absorb the virus growth and result in the time to recovery extending. I agree that this is a theory but it is based upon scientific fact about Vitamin C, not opinion. If it doesn't work what have you lost as you already have a disease with one of the highest mortality rates so whoever catches the disease is more likely to die and using a High Dose of Ascorbic Acid is the least of their worries. Given the option, in that position where all Western Medical Approaches have failed, would I try it, every time.
Please, Please send me links to research that refutes this I would like to see it. Papers, Academic research, images, trials etc etc as I always will review and compare my current knowledge against evidence. PS I bet this doesn't even posted.
NP says:
TROLLS!
Massive doses of vitamin C are a proven treatment for diverse viral illnesses. See the 1949 publication by Dr Fred R Klenner, a Duke University Medical School graduate. The fact that Dr Klenner's work has been ignored by mainstream medicine is tragic.
Steve P- I was hunting around on the internet to see if anyone else had wondered about Vitamin C and Ebola. I remembered reading how serious infection radically depletes the body of this vitamin, and about Linus Pauling's experiments, and about how intravenous vitamin C has saved people when suffering from an overwhelming infective condition. I also thought about how important vitamin C is in the maintenance of the vascular system, and it seemed likely to me that the bleeding caused by Ebola may indeed be caused by this vitamins depletion. As it's not a harmful treatment, someone who contracted Ebola may be grateful if someone gave it a go – the alternative isn't great.
"So let's theorise for a moment (an integral part of Science methodology and advancement) " – let's hypothesise – because you are simply floating an idea.
It seems to me that what's being argued for here is a very rapid onset of the symptoms of scurvy. For the hypothesis to be accepted, someone would need to present actual evidence that this is the case. Has such evidence been published?
Just for thought –
Those propounding "mega" vitamin C often mix or confuse consequence and cause.
Many things will depleted as a consequence of illness, vitamin C levels will be just one. (Notice how advocating "mega" vitamin C don't mention any others, cherry-picking and favouring their interest.)
Supplementing what is depleted might contribute to helping patients (I'm not medical), but a cure would want to tackle the unlying cause and, as Alison said you'd want evidence that it does that.
Zra My says:
Megadose intravenous Vitamin C might be helpful in fighting the Ebola virus at its onset. I have recommended it to the health authorities in my country following 1 confirmed case of the virus in the city of Lagos (there are 2 more unconfirmed cases). I hope they follow up on it as it might just help.
david austin says:
Grant Jacob's responses are reasonable and expected since no known results of even a test-tube study of IVC (Intravenous Vitamin C) vs. Ebola have ever been published, let alone has there been an RDBPC study.
Jacob's consideration that "Supplementing what is depleted might contribute to helping patients" is also spot on and deserves considerable esteem. Indeed he is "not medical", but a recently comprehensive review found only 1/200 patients with an adverse reaction to high-dose IVC (PMC 2898816, PMD 20628650).
And … ebola symptoms are identical with scurvy and there are a number of other reasons suggesting that patients are massively depleted of vitamin C – so mega-supplementation for QOL seems judicious.
Of all the peer-reviewed studies of high-dose IVC (40+ g/dose) NONE have ever indicated decreased patient quality of life regardless of the condition treated. In fact. rather the opposite has been observed in many published studies: decreased nausea, pain, fatigue, dizziness, bleeding, fever, and anxiety (see http://goo.gl/4odny8).
In a perfect world we would have an ebola cure, but in the meantime we should be strengthening the patient and improving their quality of life. I have long thought that the refusal to integrate IVC despite it's low cost, amazing safety record and unsurpassed ability to improve QOL should be astonishing to everyone.
IVC may not fight ebola itself, but let's not forget that happy, energized and confident patients fight disease better than do their miserable, exhausted, and despondent counterparts.
In 1995 a 27yr old woman was treated for ebola with vitamin c, papaverine and iv calcium. 2 days later a 400 cm^3 blood transfusion was also performed, and survived.
As posted in the journal of infectious deseases
http://m.jid.oxfordjournals.org/content/179/Supplement_1/S18.full
It's a case report of 8 patients, one of whom is the patient you describe. This patient was the only one given vitamin C although it is unclear whether she received intravenous high dose vitamin C. What she did receive was a transfusion of blood taken from people who had survived Ebola. So did the other seven people. Only one of the eight died. The doctors noted though that the patients all received good care which also included infusions of glucose and electrolytes, treatment with antibiotic and antimalaria drugs, and food supplementation.
To put that one patient's survival down to vitamin C is cherry-picking, to say the least.
Michael Edmonds says:
If I remember correctly large doses of vitamin C cause diarrhoea which would be the last thing a patient with Ebola needed. This could possibly only be with oral doses, but to assume large doses of vitamin C would have no negative effects is assuming a lot.
Any substance in high enough doses would be harmful
"Ebola symptoms are identical to scurvy"
Says who? Sounds like we are entering the territory of sympathic pseudo medicine.
Survey is a deficiency disease, Ebola is caused by a micro-organism. What you are erroneously suggesting is that Ebola could be dealt with by treating the symptoms rather than the cause – isn't that what alternative "medicine" proponents usually use as an argument to complain about "western" medicine?
Oh dear, that should be scurvy not survey. Stupid autocorrect.
In the study you listed regarding one patient being given vitamin C and surviving did you notice that some of the other patients who survived received other supplements/drugs/transfusions and also survived? So how do you decide a treatment with vitamin C is better than one with vitamin K or paracetamol or sodium salts?
To call it cherry picking is an understatement
Apologies, the previous comment relates to the link provided by DM not David Austin
Michael Edmonds 3 days ago
I think when you are dealing with a disease that has such a high fatality rate that the need for placebo's is unnecessary. If a treatment works, the results tend to be obvious
I take it then that IV vitamin C would apply here as well?
I said placebos are unnecessary, not that any substance can be used to try and treat Ebola.
It is not clear to me what the value IV vitamin C would be – there needs to be a more convincing argument than that Ebola and scurvy have some similar symptoms.
Mark Hanna says:
Jim, I a distinction needs to be drawn between not requiring a placebo control (as Michael was talking about in that comment) and not requiring a large sample size when testing a treatment.
For deadly diseases such as this, where there is no existing standard treatment, I can definitely see how it would be unethical to randomise patients to receive either placebo or a potentially lifesaving new treatment as part of a clinical trial. Testing a treatment would still need a decent sample size for us to be able to draw reliable conclusions, and its results would presumably be compared to the statistics for untreated patients.
Derek Syms says:
Possible E bola treatment
http://rt.com/usa/181900-ebola-doctor-discharged-recovery/
David Austin's reply gives empty praise (and his quote mines somewhat misrepresent, too!), seemingly in the hope of giving credence to his later suggestions. These suggestions are poor.
I didn't have time to reply at the time (was travelling) and still haven't much time, so very briefly:
'not medical' should read 'not a medic' – I was still grappling with the spell-checker on a new smartphone I'd just purchased. There's nothing lacking on my part in writing this, the reference is simply that I don't want to be offering a medical prescription. I would imagine this goes for everyone else writing here, including David Austin – i.e. no-one writing here is a medic. (Put another way, at least I have the decency to acknowledge it!)
"ebola symptoms are identical with scurvy"
Not correct, as a few minutes research would show. In any event they're quite different diseases as most people realise. I see Michael has tackled this. (Thanks.) As he noted, one is a deficiency and the other an infection.
"regardless of the condition treated"
Grand sweeping claims like this are invariably false and almost always hallmark of trying too hard to make something true.
(Off-topic: Why is it that people think that the apostrophe on Jacobs, should be rendered as Jacob's, not the correct Jacobs'? I've seen this a few times and am left wondering why these people don't realise their misplaced apostrophe has them changing my surname from Jacobs to Jacob, which it clearly isn't! It's really quite bizarre.)
What you and others don't seem to understand is that Ebola creates scurvy. A simple blood test would confirm this. Do you not think that by correcting this deficiency the patient may have a better chance of survival.
ZMapp is manufactured in the tobacco plant nicotiana in the bioproduction process known as "pharming" by Kentucky BioProcessing, a subsidiary of Reynolds American.Monoclonal antibodies (mAbs) were first created in mice by injecting them with antigens from Ebola, harvesting their spleens, and fusing mature B-cells producing monoclonal antibodies (mAbs) with cancer cell lines to create hybridomas. After the best antibody is selected, the gene encoding the antibody was extracted, and certain portions were replaced with portions encoding human proteins, in the process called humanization. To create a system to produce the humanized mAbs at commercial scale, Mapp used a process called "Rapid Antibody Manufacturing Platform" (RAMP), using magnICON (ICON Genetics) viral vectors. In a process called "magnifection," tobacco plants are infected with the viruses, using Agrobacterium cultures.Subsequently, antibodies are extracted and purified from the plants. Once the genes encoding the humanized mAbs are in hand, the entire tobacco production cycle is believed to take a few months.
Trying to dress others down really just suggests you wish to push your own interests irrespective of what evidence might show. (Anyone pointing out issues is dismissed out-of-hand—"just doesn't understand"—but dismissal out-of-hand doesn't make a case.)
You're welcome to point to scientific papers (from sound authors in reputable journals) that show ebola is a form of scurvy or induces scurvy.
If true, you'd expect there to be some scientific articles in reputable journals relating the Ebola and scurvy and for their to be some sort of consensus on this.
The latter (consensus) is why I have previously generally suggested that people try review papers, but unfortunately you (or I) can't get that far because…
A simple check of PubMed shows that while PubMed holds over 1800 papers on Ebola and over 2300 papers on scurvy, not a single paper in PubMed mentions both Ebola and scurvy.
Not one scientific paper.
On the face of it, this looks to be a very clear and simple illustration that the idea that Jim and others are suggesting is not supported by those that work on Ebola or scurvy.
Similarly, a quick google search suggests this belief that Ebola = scurvy, or variants of that, have arisen because someone has promoted the idea that scurvy is hemorrhagic fever amongst those that advocate 'high-dose vitamin C' as, essentially, a cure-all. It's pretty clear that they're trying too hard to string a line of dots between Ebola and their favourite treatment/remedy/supplement.
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Реферат / Рефераты /category/by/type/referaty Иностранные языки /category/by/type/referaty/inostrannye-yazyki The Crucible To Kill A Mockingbird Essay
The Crucible To Kill A Mockingbird Essay
The Crucible/ To Kill A Mockingbird Essay, Research Paper Often the title of a piece of literature will have significant implications in terms of the theme of that book. In Arthur Millers The Crucible and Harper Lees To Kill a Mockingbird the title had significant implications on the books theme. By definition a crucible is a trying, or testing situation. When approaching the theme of The Crucible it is painfully obvious that Miller wanted the reader to see a parallel between the title and the theme. The theme being that when the screws are tightened in a testing situation people often do things that they would not normally be apt to do. Whether it be doing any thing to save them selves or sacrificing their lives for the sake of a good name. Miller presents this theme
throughout the book. Early on the rumor of witches spread through Salem like wild fire; the court began interrogations with Abigail.When Paris asked Abigail if she was "conjuring up sprits last night. Abigai replied "Not I, sir- Tituba and Ruth."(Act1 Pg. 16). When Tituba was asked about the devil or if she had talked to him she said that it was Sara Good and Goody Osburn (Act1 Pg.47). The pressure changed hands and moved on to the next person. Like a hot potato being passed around no one wanted the pressure, they would just pass the buck to the next person. On the flip side thistesting situation brought out the redeeming qualities in several characters. In John Procter's case he found his name and the passing on of his name more important than life itself. Rather than
leaving a marred name to his children John Procter climbed the scaffold. Indeed this crucible brought out a righteous quality in John Procter. Another work in which the author chooses a title that deliberately conveys a message to the reader is To Kill a Mockingbird. The message conveyed is that to kill a mockingbird is an unforgivable crime. This is because mocking birds are innocent creatures are here for our benefit. Atticus said "It is a sin to kill a mocking bird."(Pg.94). They exist for the express purpose of enriching our lives. We can see the relation in the work. An innocent black man Tom Walker, is accused of raping Mayella Ewell. Tom is obviously innocent but the town people are rather daft ignorant lot. To kill Tom would be an abomination towards society. This is
because Tom is a loving innocent human being that is here, as all people are, to enrich the lives of others. He is a gift to society as all people are. Taking this gift the world would be a morbid and twisted resolution to Mayellas rape. Whether it is an obvious relationship as in The Crucible or a more subtle one a sin To Kill a Mockingbird. We can see that authors do in fact relate the titles of their works and the themes of their works.

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Vicksburg resident dies following Claiborne County wreck
Published 11:58 am Wednesday, February 9, 2022
By Anna Guizerix
Vicksburg resident Chandler Roesch died Monday due to injuries sustained in a Jan. 11 wreck that occurred on Fisher Ferry Road, about 2 miles inside Claiborne County.
Roesch was a graduate of St. Aloysius High School and a student at Mississippi State University. He lettered in four sports in high school — soccer, cross country, track and field and baseball.
Vicksburg resident Caroline Simrall Hood, also a St. Aloysius graduate, was a passenger in the car and was declared dead at the scene.
Vicksburg Catholic School acknowledged the passing of Hood and Roesch in a social media post. Quoting Revelation 21:4, the school offered prayers for the Roesch and Hood families.
"We continue to pray for the families and friends of Chandler Roesch (Class 2018) and Caroline Simrall (Class of 2018) during this most difficult time," the statement read.
Immediately following the wreck, Roesch was taken by ambulance to Merit Health River Region. He was then flown to the University of Mississippi Medical Center in Jackson.
Arrangements for Roesch were announced Wednesday afternoon.
The visitation will take place from 5 to 8 p.m. Thursday at Fisher-Riles Funeral Home, 5000 Indiana Ave.
A funeral Mass will be held on Friday at 11 a.m. at St. Paul Catholic Church in Downtown Vicksburg.


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Q: array.filter doesn't seem to be working at all? The HTML for the javascript that's going to follow
<button onclick="myFunction()">Click</button>

The javascript that's working is
var ages = [32, 33, 16, 40];

function checkAdult(age) {
  return age >= 18;
}

function myFunction() {
  document.getElementById("demo").innerText = ages.filter(checkAdult);
}

No problem with it. But this code below doesn't seem to work.
var ages = [32, 33, 16, 40];

function myFunction() {
  document.getElementById("demo").innerText = ages.filter(function(checkAdult){
    checkAdult > 18;
  });
}

I want to know why it doesn't work and how can I make it work?

A: You return nothing in the function. Write an arrow function or use return keyword:
let ages = [32, 33, 16, 40];

function myFunction() {
  document.getElementById("demo").innerText = ages.filter(checkAdult => checkAdult > 18);
}

A: You are missing return in callback function.
As to documentation on: Array.prototype.filter() the callback function should return a value that coerces to true to keep the element, or to false otherwise.
So your code should look like this
  document.getElementById("demo").innerText = ages.filter(function(checkAdult){
    return checkAdult > 18;
  });

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Purdue University is celebrating a record-breaking year for annual giving, research awards and patents and startups. The university says prospective student applications were also the highest in school history.Donations from over 76,000 benefactors totaled more than $343 million. Vice President for Development Amy Noah says $108 million in additional funding was...


Marble Archives - A1 Granite & Marble Ltd.
We stock an extensive and diverse selection of Marble from around the world. Marble is a natural stone that has been used for centuries as a building material, as well as a platform for some of the world's most famous sculptures, such as the Taj Mahal, Washington Monument, and the Bust of Artemis. Marble is known for its soft, beautiful look, which is unique to this product. Marble has a very unique color and movement that is not found in any other stone.
Many famous buildings known worldwide have been constructed of marble since ancient times. Marble is high in calcium carbonate, which means it is softer than granite. Marble is prone to acid etching of the surface, primarily caused by highly acidic drinks such as cola, fruit juices etc, but like similar to wood, marble can be refinished if necessary. Marble has a very unique beauty, one that is not found in any other material.


FOREST CITY, Mo. -- Many folks in the small town of Forest City spent the day on pins and needles.
"It just keeps creeping up all the time," a weary Doyal Acton said.
He lives just yards away from the Missouri River.
"If it would come all at once, you know, I could get trapped in my home where I couldn't get out," he said.
Acton and many of his neighbors have lived in Forest City for decades. They've never seen the Missouri River as high as it is right now.
"I don't understand it, but I guess there was a lot of recent snowmelt and stuff in between the two dams, and we had that big storm come across and all just hit us all at once," Acton said.
On Thursday, Acton and dozens of moms, dads, students, city officials and many others spent the day filling up scores of sandbags and stacking the bags all around the town's water plant and other businesses in the heart of downtown Forest City.
"My biggest fears are the water coming into Forest City possibly and, you know, everybody's livelihood being affected," said Dana Medsker, who works in Forest City.
Their livelihoods and more could be on the line if the rising river overtakes three major levees in the small town.
"It's frustrating, but there's really not much you can do about it. My four kids and my wife are all out here shoveling, bagging, whatever it takes. You got to serve your community and lend a helping hand," South Holt School District Principal Ted Quinlin said.
"I think it's nice that all the community is coming together. It's a life experience," eighth grader Zoey Schroeder said.
In the meantime, Acton didn't take any chances Thursday night. He packed up and spent the night at a motel.
"I'm just hoping that the river goes down to where it doesn't get into town," he said.


text:
Q: Complex Sumproduct formula Please could someone help me work out the sumproduct equation for something a little tricky.
Here is what I am trying to acheive.
Look in column B:B of sheet Postings, where it matches cell A3 = Tier 1, then   sumproduct Postings! Column K:K, M:M
I have tried this.
IF(SUMIF(Postings!B:B,Sheet1!A3)<>0,Postings!$K$3:$K$201,Postings!$M$3:$M$201)

A: Sounds like you want this:
=SUMPRODUCT((Postings!$B$3:$B$201=Sheet1!A3)+0,Postings!$K$3:$K$201,Postings!$M$3:$M$201)
That will multiply columns K and M only for rows where the column B criterion is met, and sum the results, is that what you need?


text:
Chelnik (Bulgarian: Челник) - a village in South-Eastern Bulgaria in the Yambol Province, in the Tundzha Municipality. According to the National Institute of Statistics, in the year of 2011, the village had 300 inhabitants.

References

Villages in Yambol Province

redpajama_set_name: RedPajamaWikipedia


British Gas parent firm Centrica has signed a seven-and-a-half year HR business process outsourcing contract covering its 30,000 UK staff.
The deal, with HR services firm Hewitt Associates, includes workforce administration, payroll, reward management, performance management, succession planning, customer services and related HR information services.
The value of the deal is undisclosed, but Hewitt said it was a "large-scale" agreement.
Anne Minto, group HR director at Centrica, said: "Our HR transformation programme is already well underway and our new partnership with Hewitt will both accelerate and support the delivery of enabling technologies for our line managers.
"This will provide them with clearer accountability in managing their people whilst reducing our cost to serve.


Lucha Underground is killing it right now. They are putting on incredible shows each and every week and we will have a full post in the future detailing all the awesomeness. In the meantime enjoy this match from the opening of the Trios Tournament. Crazy spots, good storytelling, it's great.


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L/S of a Comet IV B jet in a huge aircraft hangar at London Airport; M/S of the plane as a man works on one of the wings; M/Ss of this BEA inspector working on the Comet wing, then he turns and walks down the ladder and out of shot.
High angle M/S as he walks past a man working on an Avro 504K Bomber, then M/S as he walks round to work on the ski of the plane; C/Us of him tightening bolts there. M/S of the Avro showing the inspector working there and another man working on the cockpit; high and low angle M/Ss of this man who is fixing glass to a compass. Commentator says "Volunteers from all walks of life are spending their spare time restoring these veterans of flight, painstakingly ensuring that every detail of design and colour is accurate".
M/S of two men working on a Bleriot Type 27; C/U as one of the men tightens the stays; M/S and C/U as the other man cleans the stays. M/S of the Bleriot with both men working on her; one of them, Mr Ash-Hughes, walks to the centre of the plane and starts to move the joystick; C/Us of him doing this, and the mechanism moving about. M/S and C/U showing the tail-fins moving.
M/Ss of a Fokker D.VII, a German fighter from World War One, as three men drape a canvas fabric over the framework and attach it underneath the plane; there is a skull and crossbones on the side; commentator says, "As you can imagine, the canvas in those days wasn't exactly bullet proof", and we can see small tears and bullet holes in the fabric in a C/U of the black cross printed on the side; C/U of the skull and crossbones.
M/S of the SE5A British fighter as two men wheel it into position; C/U of the wheel as a man places a chock beneath it; M/Ss as the other man enters shot and looks into the cockpit; C/U of the control panel in the cockpit. M/S of the aeroplane as a man walks up carrying a Vickers Machine Gun that will be mounted above the engine cowling; the two men examine it; C/Us as they examine the gun and trigger mechanism.
M/S of two men looking at part of the tail of a Sopwith Camel; man on the left holding a pad of drawings or blueprints is RJ Ashfield, "the original designer who first sketched it on his drawing board 44 years ago, and who is still active in aircraft design today", says the commentator; C/U of RJ Ashfield. C/U of one of the men tightening a stay on the plane.
Commentator says "In this case the enthusiasts are faced with the problem of rebuilding a Bleriot type XI from what is almost a heap of scrap" as we see in M/S Mr Lumsden examining part of the tailpiece of the plane where the fabric has worn away; high angle M/S of Mr L as he walks round what is left of the undercarriage of the plane. C/U as he picks up part of it, a metal bar attached to a wheel, and examines it; C/U of the wheel.
Note: on file are extensive notes on the planes in the film and a letter from ASC Lumsden, Assistant Secretary of the Royal Aeronautical Society. Nice shots of the veteran planes being restored and repaired.
"M/S of the SE5A British fighter" - it's known as a Royal Aircraft Factory S.E.5A.
Romantic but educational travelogue around Italy seen through the lens of a schoolgirl's camera.
A look at the history of armour.


text:
Santo contra los Zombies (also known as Santo vs. the Zombies) is a 1961 Mexican film directed by Benito Alazraki. It depicts the popular character El Santo in a pulp-style film.

Plot
After the police are unable to stop a crime wave by zombies, they turn to Santo for help.

Cast

 Santo as  Santo / the Saint
 Armando Silvestre as  Lt. Sanmartin / Lt. Savage
 Jaime Fernández as  Det. Rodriguez
 Dagoberto Rodríguez as  Det. Chief Almada
 Irma Serrano as  Det. Isabel
 Carlos Agostí as  Genaro / Uncle Herbert
 Ramón Bugarini as  Rogelio / Roger, the male nurse
 Fernando Osés as  Dorrell, zombified wrestler
 Eduardo Bonada
 Eduardo Silvestre
 Julián de Meriche
 Alejandro Cruz as  Black Shadow, a wrestler (as Black Shadow)
 Gory Guerrero as  Wrestler (as Gori Guerrero)
 Sugi Sito as  Wrestler
 El Gladiador as  Wrestler

Release
This was one of the earliest Santo films and the first to be distributed in the US.

Reception
Writing in The Zombie Movie Encyclopedia, academic Peter Dendle said, "The adventure movie with its comic-book plot, hero, and villain is fun to watch in an MST-3K spirit".

References

External links

1961 films
1960s science fiction horror films
1961 horror films
Mexican science fiction horror films
1960s Spanish-language films
Mexican black-and-white films
Lucha libre films
Mexican zombie films
Films directed by Benito Alazraki
Cultural depictions of El Santo
1960s Mexican films


text:
// This file was generated by Mendix Modeler.
//
// WARNING: Code you write here will be lost the next time you deploy the project.

package excelimporter.proxies;

/**
 * Besides the basic settings in the Template this entity contains all advanced settings.
 *
 * This entity can not exist without a template
 */
public class AdditionalProperties
{
	private final com.mendix.systemwideinterfaces.core.IMendixObject additionalPropertiesMendixObject;

	private final com.mendix.systemwideinterfaces.core.IContext context;

	/**
	 * Internal name of this entity
	 */
	public static final java.lang.String entityName = "ExcelImporter.AdditionalProperties";

	/**
	 * Enum describing members of this entity
	 */
	public enum MemberNames
	{
		PrintStatisticsMessages("PrintStatisticsMessages"),
		PrintNotFoundMessages_MainObject("PrintNotFoundMessages_MainObject"),
		IgnoreEmptyKeys("IgnoreEmptyKeys"),
		CommitUnchangedObjects_MainObject("CommitUnchangedObjects_MainObject"),
		RemoveUnsyncedObjects("RemoveUnsyncedObjects"),
		ResetEmptyAssociations("ResetEmptyAssociations"),
		Template_AdditionalProperties("ExcelImporter.Template_AdditionalProperties"),
		AdditionalProperties_MxObjectMember_RemoveIndicator("ExcelImporter.AdditionalProperties_MxObjectMember_RemoveIndicator");

		private java.lang.String metaName;

		MemberNames(java.lang.String s)
		{
			metaName = s;
		}

		@Override
		public java.lang.String toString()
		{
			return metaName;
		}
	}

	public AdditionalProperties(com.mendix.systemwideinterfaces.core.IContext context)
	{
		this(context, com.mendix.core.Core.instantiate(context, "ExcelImporter.AdditionalProperties"));
	}

	protected AdditionalProperties(com.mendix.systemwideinterfaces.core.IContext context, com.mendix.systemwideinterfaces.core.IMendixObject additionalPropertiesMendixObject)
	{
		if (additionalPropertiesMendixObject == null)
			throw new java.lang.IllegalArgumentException("The given object cannot be null.");
		if (!com.mendix.core.Core.isSubClassOf("ExcelImporter.AdditionalProperties", additionalPropertiesMendixObject.getType()))
			throw new java.lang.IllegalArgumentException("The given object is not a ExcelImporter.AdditionalProperties");

		this.additionalPropertiesMendixObject = additionalPropertiesMendixObject;
		this.context = context;
	}

	/**
	 * @deprecated Use 'AdditionalProperties.load(IContext, IMendixIdentifier)' instead.
	 */
	@Deprecated
	public static excelimporter.proxies.AdditionalProperties initialize(com.mendix.systemwideinterfaces.core.IContext context, com.mendix.systemwideinterfaces.core.IMendixIdentifier mendixIdentifier) throws com.mendix.core.CoreException
	{
		return excelimporter.proxies.AdditionalProperties.load(context, mendixIdentifier);
	}

	/**
	 * Initialize a proxy using context (recommended). This context will be used for security checking when the get- and set-methods without context parameters are called.
	 * The get- and set-methods with context parameter should be used when for instance sudo access is necessary (IContext.createSudoClone() can be used to obtain sudo access).
	 */
	public static excelimporter.proxies.AdditionalProperties initialize(com.mendix.systemwideinterfaces.core.IContext context, com.mendix.systemwideinterfaces.core.IMendixObject mendixObject)
	{
		return new excelimporter.proxies.AdditionalProperties(context, mendixObject);
	}

	public static excelimporter.proxies.AdditionalProperties load(com.mendix.systemwideinterfaces.core.IContext context, com.mendix.systemwideinterfaces.core.IMendixIdentifier mendixIdentifier) throws com.mendix.core.CoreException
	{
		com.mendix.systemwideinterfaces.core.IMendixObject mendixObject = com.mendix.core.Core.retrieveId(context, mendixIdentifier);
		return excelimporter.proxies.AdditionalProperties.initialize(context, mendixObject);
	}

	public static java.util.List<excelimporter.proxies.AdditionalProperties> load(com.mendix.systemwideinterfaces.core.IContext context, java.lang.String xpathConstraint) throws com.mendix.core.CoreException
	{
		java.util.List<excelimporter.proxies.AdditionalProperties> result = new java.util.ArrayList<excelimporter.proxies.AdditionalProperties>();
		for (com.mendix.systemwideinterfaces.core.IMendixObject obj : com.mendix.core.Core.retrieveXPathQuery(context, "//ExcelImporter.AdditionalProperties" + xpathConstraint))
			result.add(excelimporter.proxies.AdditionalProperties.initialize(context, obj));
		return result;
	}

	/**
	 * Commit the changes made on this proxy object.
	 */
	public final void commit() throws com.mendix.core.CoreException
	{
		com.mendix.core.Core.commit(context, getMendixObject());
	}

	/**
	 * Commit the changes made on this proxy object using the specified context.
	 */
	public final void commit(com.mendix.systemwideinterfaces.core.IContext context) throws com.mendix.core.CoreException
	{
		com.mendix.core.Core.commit(context, getMendixObject());
	}

	/**
	 * Delete the object.
	 */
	public final void delete()
	{
		com.mendix.core.Core.delete(context, getMendixObject());
	}

	/**
	 * Delete the object using the specified context.
	 */
	public final void delete(com.mendix.systemwideinterfaces.core.IContext context)
	{
		com.mendix.core.Core.delete(context, getMendixObject());
	}
	/**
	 * Set value of PrintStatisticsMessages
	 * @param printstatisticsmessages
	 */
	public final excelimporter.proxies.StatisticsLevel getPrintStatisticsMessages()
	{
		return getPrintStatisticsMessages(getContext());
	}

	/**
	 * @param context
	 * @return value of PrintStatisticsMessages
	 */
	public final excelimporter.proxies.StatisticsLevel getPrintStatisticsMessages(com.mendix.systemwideinterfaces.core.IContext context)
	{
		Object obj = getMendixObject().getValue(context, MemberNames.PrintStatisticsMessages.toString());
		if (obj == null)
			return null;

		return excelimporter.proxies.StatisticsLevel.valueOf((java.lang.String) obj);
	}

	/**
	 * Set value of PrintStatisticsMessages
	 * @param printstatisticsmessages
	 */
	public final void setPrintStatisticsMessages(excelimporter.proxies.StatisticsLevel printstatisticsmessages)
	{
		setPrintStatisticsMessages(getContext(), printstatisticsmessages);
	}

	/**
	 * Set value of PrintStatisticsMessages
	 * @param context
	 * @param printstatisticsmessages
	 */
	public final void setPrintStatisticsMessages(com.mendix.systemwideinterfaces.core.IContext context, excelimporter.proxies.StatisticsLevel printstatisticsmessages)
	{
		if (printstatisticsmessages != null)
			getMendixObject().setValue(context, MemberNames.PrintStatisticsMessages.toString(), printstatisticsmessages.toString());
		else
			getMendixObject().setValue(context, MemberNames.PrintStatisticsMessages.toString(), null);
	}

	/**
	 * @return value of PrintNotFoundMessages_MainObject
	 */
	public final java.lang.Boolean getPrintNotFoundMessages_MainObject()
	{
		return getPrintNotFoundMessages_MainObject(getContext());
	}

	/**
	 * @param context
	 * @return value of PrintNotFoundMessages_MainObject
	 */
	public final java.lang.Boolean getPrintNotFoundMessages_MainObject(com.mendix.systemwideinterfaces.core.IContext context)
	{
		return (java.lang.Boolean) getMendixObject().getValue(context, MemberNames.PrintNotFoundMessages_MainObject.toString());
	}

	/**
	 * Set value of PrintNotFoundMessages_MainObject
	 * @param printnotfoundmessages_mainobject
	 */
	public final void setPrintNotFoundMessages_MainObject(java.lang.Boolean printnotfoundmessages_mainobject)
	{
		setPrintNotFoundMessages_MainObject(getContext(), printnotfoundmessages_mainobject);
	}

	/**
	 * Set value of PrintNotFoundMessages_MainObject
	 * @param context
	 * @param printnotfoundmessages_mainobject
	 */
	public final void setPrintNotFoundMessages_MainObject(com.mendix.systemwideinterfaces.core.IContext context, java.lang.Boolean printnotfoundmessages_mainobject)
	{
		getMendixObject().setValue(context, MemberNames.PrintNotFoundMessages_MainObject.toString(), printnotfoundmessages_mainobject);
	}

	/**
	 * @return value of IgnoreEmptyKeys
	 */
	public final java.lang.Boolean getIgnoreEmptyKeys()
	{
		return getIgnoreEmptyKeys(getContext());
	}

	/**
	 * @param context
	 * @return value of IgnoreEmptyKeys
	 */
	public final java.lang.Boolean getIgnoreEmptyKeys(com.mendix.systemwideinterfaces.core.IContext context)
	{
		return (java.lang.Boolean) getMendixObject().getValue(context, MemberNames.IgnoreEmptyKeys.toString());
	}

	/**
	 * Set value of IgnoreEmptyKeys
	 * @param ignoreemptykeys
	 */
	public final void setIgnoreEmptyKeys(java.lang.Boolean ignoreemptykeys)
	{
		setIgnoreEmptyKeys(getContext(), ignoreemptykeys);
	}

	/**
	 * Set value of IgnoreEmptyKeys
	 * @param context
	 * @param ignoreemptykeys
	 */
	public final void setIgnoreEmptyKeys(com.mendix.systemwideinterfaces.core.IContext context, java.lang.Boolean ignoreemptykeys)
	{
		getMendixObject().setValue(context, MemberNames.IgnoreEmptyKeys.toString(), ignoreemptykeys);
	}

	/**
	 * @return value of CommitUnchangedObjects_MainObject
	 */
	public final java.lang.Boolean getCommitUnchangedObjects_MainObject()
	{
		return getCommitUnchangedObjects_MainObject(getContext());
	}

	/**
	 * @param context
	 * @return value of CommitUnchangedObjects_MainObject
	 */
	public final java.lang.Boolean getCommitUnchangedObjects_MainObject(com.mendix.systemwideinterfaces.core.IContext context)
	{
		return (java.lang.Boolean) getMendixObject().getValue(context, MemberNames.CommitUnchangedObjects_MainObject.toString());
	}

	/**
	 * Set value of CommitUnchangedObjects_MainObject
	 * @param commitunchangedobjects_mainobject
	 */
	public final void setCommitUnchangedObjects_MainObject(java.lang.Boolean commitunchangedobjects_mainobject)
	{
		setCommitUnchangedObjects_MainObject(getContext(), commitunchangedobjects_mainobject);
	}

	/**
	 * Set value of CommitUnchangedObjects_MainObject
	 * @param context
	 * @param commitunchangedobjects_mainobject
	 */
	public final void setCommitUnchangedObjects_MainObject(com.mendix.systemwideinterfaces.core.IContext context, java.lang.Boolean commitunchangedobjects_mainobject)
	{
		getMendixObject().setValue(context, MemberNames.CommitUnchangedObjects_MainObject.toString(), commitunchangedobjects_mainobject);
	}

	/**
	 * Set value of RemoveUnsyncedObjects
	 * @param removeunsyncedobjects
	 */
	public final excelimporter.proxies.RemoveIndicator getRemoveUnsyncedObjects()
	{
		return getRemoveUnsyncedObjects(getContext());
	}

	/**
	 * @param context
	 * @return value of RemoveUnsyncedObjects
	 */
	public final excelimporter.proxies.RemoveIndicator getRemoveUnsyncedObjects(com.mendix.systemwideinterfaces.core.IContext context)
	{
		Object obj = getMendixObject().getValue(context, MemberNames.RemoveUnsyncedObjects.toString());
		if (obj == null)
			return null;

		return excelimporter.proxies.RemoveIndicator.valueOf((java.lang.String) obj);
	}

	/**
	 * Set value of RemoveUnsyncedObjects
	 * @param removeunsyncedobjects
	 */
	public final void setRemoveUnsyncedObjects(excelimporter.proxies.RemoveIndicator removeunsyncedobjects)
	{
		setRemoveUnsyncedObjects(getContext(), removeunsyncedobjects);
	}

	/**
	 * Set value of RemoveUnsyncedObjects
	 * @param context
	 * @param removeunsyncedobjects
	 */
	public final void setRemoveUnsyncedObjects(com.mendix.systemwideinterfaces.core.IContext context, excelimporter.proxies.RemoveIndicator removeunsyncedobjects)
	{
		if (removeunsyncedobjects != null)
			getMendixObject().setValue(context, MemberNames.RemoveUnsyncedObjects.toString(), removeunsyncedobjects.toString());
		else
			getMendixObject().setValue(context, MemberNames.RemoveUnsyncedObjects.toString(), null);
	}

	/**
	 * @return value of ResetEmptyAssociations
	 */
	public final java.lang.Boolean getResetEmptyAssociations()
	{
		return getResetEmptyAssociations(getContext());
	}

	/**
	 * @param context
	 * @return value of ResetEmptyAssociations
	 */
	public final java.lang.Boolean getResetEmptyAssociations(com.mendix.systemwideinterfaces.core.IContext context)
	{
		return (java.lang.Boolean) getMendixObject().getValue(context, MemberNames.ResetEmptyAssociations.toString());
	}

	/**
	 * Set value of ResetEmptyAssociations
	 * @param resetemptyassociations
	 */
	public final void setResetEmptyAssociations(java.lang.Boolean resetemptyassociations)
	{
		setResetEmptyAssociations(getContext(), resetemptyassociations);
	}

	/**
	 * Set value of ResetEmptyAssociations
	 * @param context
	 * @param resetemptyassociations
	 */
	public final void setResetEmptyAssociations(com.mendix.systemwideinterfaces.core.IContext context, java.lang.Boolean resetemptyassociations)
	{
		getMendixObject().setValue(context, MemberNames.ResetEmptyAssociations.toString(), resetemptyassociations);
	}

	/**
	 * @return value of Template_AdditionalProperties
	 */
	public final excelimporter.proxies.Template getTemplate_AdditionalProperties() throws com.mendix.core.CoreException
	{
		return getTemplate_AdditionalProperties(getContext());
	}

	/**
	 * @param context
	 * @return value of Template_AdditionalProperties
	 */
	public final excelimporter.proxies.Template getTemplate_AdditionalProperties(com.mendix.systemwideinterfaces.core.IContext context) throws com.mendix.core.CoreException
	{
		excelimporter.proxies.Template result = null;
		com.mendix.systemwideinterfaces.core.IMendixIdentifier identifier = getMendixObject().getValue(context, MemberNames.Template_AdditionalProperties.toString());
		if (identifier != null)
			result = excelimporter.proxies.Template.load(context, identifier);
		return result;
	}

	/**
	 * Set value of Template_AdditionalProperties
	 * @param template_additionalproperties
	 */
	public final void setTemplate_AdditionalProperties(excelimporter.proxies.Template template_additionalproperties)
	{
		setTemplate_AdditionalProperties(getContext(), template_additionalproperties);
	}

	/**
	 * Set value of Template_AdditionalProperties
	 * @param context
	 * @param template_additionalproperties
	 */
	public final void setTemplate_AdditionalProperties(com.mendix.systemwideinterfaces.core.IContext context, excelimporter.proxies.Template template_additionalproperties)
	{
		if (template_additionalproperties == null)
			getMendixObject().setValue(context, MemberNames.Template_AdditionalProperties.toString(), null);
		else
			getMendixObject().setValue(context, MemberNames.Template_AdditionalProperties.toString(), template_additionalproperties.getMendixObject().getId());
	}

	/**
	 * @return value of AdditionalProperties_MxObjectMember_RemoveIndicator
	 */
	public final mxmodelreflection.proxies.MxObjectMember getAdditionalProperties_MxObjectMember_RemoveIndicator() throws com.mendix.core.CoreException
	{
		return getAdditionalProperties_MxObjectMember_RemoveIndicator(getContext());
	}

	/**
	 * @param context
	 * @return value of AdditionalProperties_MxObjectMember_RemoveIndicator
	 */
	public final mxmodelreflection.proxies.MxObjectMember getAdditionalProperties_MxObjectMember_RemoveIndicator(com.mendix.systemwideinterfaces.core.IContext context) throws com.mendix.core.CoreException
	{
		mxmodelreflection.proxies.MxObjectMember result = null;
		com.mendix.systemwideinterfaces.core.IMendixIdentifier identifier = getMendixObject().getValue(context, MemberNames.AdditionalProperties_MxObjectMember_RemoveIndicator.toString());
		if (identifier != null)
			result = mxmodelreflection.proxies.MxObjectMember.load(context, identifier);
		return result;
	}

	/**
	 * Set value of AdditionalProperties_MxObjectMember_RemoveIndicator
	 * @param additionalproperties_mxobjectmember_removeindicator
	 */
	public final void setAdditionalProperties_MxObjectMember_RemoveIndicator(mxmodelreflection.proxies.MxObjectMember additionalproperties_mxobjectmember_removeindicator)
	{
		setAdditionalProperties_MxObjectMember_RemoveIndicator(getContext(), additionalproperties_mxobjectmember_removeindicator);
	}

	/**
	 * Set value of AdditionalProperties_MxObjectMember_RemoveIndicator
	 * @param context
	 * @param additionalproperties_mxobjectmember_removeindicator
	 */
	public final void setAdditionalProperties_MxObjectMember_RemoveIndicator(com.mendix.systemwideinterfaces.core.IContext context, mxmodelreflection.proxies.MxObjectMember additionalproperties_mxobjectmember_removeindicator)
	{
		if (additionalproperties_mxobjectmember_removeindicator == null)
			getMendixObject().setValue(context, MemberNames.AdditionalProperties_MxObjectMember_RemoveIndicator.toString(), null);
		else
			getMendixObject().setValue(context, MemberNames.AdditionalProperties_MxObjectMember_RemoveIndicator.toString(), additionalproperties_mxobjectmember_removeindicator.getMendixObject().getId());
	}

	/**
	 * @return the IMendixObject instance of this proxy for use in the Core interface.
	 */
	public final com.mendix.systemwideinterfaces.core.IMendixObject getMendixObject()
	{
		return additionalPropertiesMendixObject;
	}

	/**
	 * @return the IContext instance of this proxy, or null if no IContext instance was specified at initialization.
	 */
	public final com.mendix.systemwideinterfaces.core.IContext getContext()
	{
		return context;
	}

	@Override
	public boolean equals(Object obj)
	{
		if (obj == this)
			return true;

		if (obj != null && getClass().equals(obj.getClass()))
		{
			final excelimporter.proxies.AdditionalProperties that = (excelimporter.proxies.AdditionalProperties) obj;
			return getMendixObject().equals(that.getMendixObject());
		}
		return false;
	}

	@Override
	public int hashCode()
	{
		return getMendixObject().hashCode();
	}

	/**
	 * @return String name of this class
	 */
	public static java.lang.String getType()
	{
		return "ExcelImporter.AdditionalProperties";
	}

	/**
	 * @return String GUID from this object, format: ID_0000000000
	 * @deprecated Use getMendixObject().getId().toLong() to get a unique identifier for this object.
	 */
	@Deprecated
	public java.lang.String getGUID()
	{
		return "ID_" + getMendixObject().getId().toLong();
	}
}

redpajama_set_name: RedPajamaGithub


text:
Posted on February 28, 2015 March 6, 2015 by Ed McNulty
Timbuktu (2014)
Review of: Timbuktu (2014)
Abderrahmane Sissako
Reviewed by: Ed McNulty
Last modified:March 6, 2015
The horror of daily life under sharia law is shown in this African film set in the city of Timbuktu, one family especially suffering from its application.
Rated PG-13. Running time: 1 hour 37 min.
Our content ratings (1-10): Violence 5; Language 1; Sex/Nudity 2.
Our star rating (1-5): 5
Again I saw all the oppressions that are practiced under the sun. Look, the tears of the oppressed—with no one to comfort them! On the side of their oppressors there was power—with no one to comfort them.
Kidane and Satima relaxing with their daughter. (c) 2014 Cohen Media Group
A visual tract against religious intolerance, Mauritanian director Abderrahmane Sissako's masterful film is set in the early days of the jihadist takeover of northern Mali in 2012. In the city of Timbuktu the local imam (Adel Mahmoud Cherif) orders the jihadists to leave when they enter his mosque with their weapons. Although they do out of respect for his position, outside they issue the orders rather than follow those of others. The film opens with them using confiscated traditional wooden statues for target practice. (This serves as a stand-in for their destruction of the city's many artistic monuments.) They insist that women vendors cover not only their hair, but their hands as well. When a vendor tells her she cannot work wearing gloves, he ignores her protest. They are ready to move in on any group of youth that dare to play soccer. They patrol the streets and rooftops at night trying to catch anyone playing or listening to music, or, horrors, dancing. They careen through the streets in pickup trucks, black flags fluttering and AK-47 guns at ready.
The focus of the film is the herdsman/musician Kidane (Ibrahim Ahmed) who lives out in the desert with his beautiful wife Satima (Toulou Kiki) and their 12 year-old daughter Layla Walet Mohamed (Toulou Kiki). Also with them is Issan (Mehdi A. G. Mohamed), an orphan whom they have taken under their care. They are proud of the way that he takes care of their eight cows. Satima would prefer to move away from the area and join their former neighbors who have fled from the Jihadists' oppressive rule, but Kidane is convinced that he can keep them safe in their somewhat isolated location. Not so isolated, however, as to be within driving range for one of the Jihadists, Abdelkrim (Abel Jafri), who, when her husband is away, rides out to tell Satima to cover her head.
Nor can Kidane escape Sharia law when tragedy results from his dispute with a fisherman over one of his cows becoming entangled in the man's net. We also witness one of the musicians punished by thirty lashes for playing his music. More horrible is the fate of an unmarried couple found together in a room, buried up to their necks in sand and then stoned to death by their reluctant neighbors. Be glad that this is not shown with Mel Gibsonish details. The filmmaker has said that this scene was inspired by a video that had been posted by jihadists.
The citizens resist as best they can. One delightful sequence is a soccer game which the youth play despite their ball having been confiscated by the alien occupiers. They rush, dodge, kick, and maneuver their bodies so imaginatively that you can virtually see the ball. When warned of jihadists approaching in their pickup trucks, the boys immediately drop down and practice calisthenics.
This is a simple but powerful depiction of the horror of daily life under Sharia law. The brutal ugliness of the latter is underline by the beauty of the cinemaphotography. Now when I hear someone criticizing Muslims for not speaking out strongly enough against the Fundamentalists in their ranks, I can refer them to Abderrahmane Sissako's well-crafted film. Here is a Muslim artist
showing us the ugly barbarism and misuse of religion in unequivocal terms, but with words and visuals of great beauty. I began by labeling this marvelous film as "a visual tract against religious intolerance," but by the end we see it is far more than this: it is also the filmmaker's tribute to the power of ordinary people to endure, even to resist—and hopefully outlast—oppressors who would attempt to shape and force them into their narrow mold.
CategoriesMovie Reviews TagsAfrica, Islam, jihad, jihadi, Muslim, Oscar nomintated, sharia, Timbuktu
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Cox's Orange Pippin apples, after storage in 2% O2 at 3 deg C for 31/2 months, produced on ripening at 20 deg much smaller amounts of volatile esters than they did after harvest or after storage in air. This was correlated with a marked loss of flavour. The inhibition of volatile production could be relieved considerably if the apples were first kept in air at 5-15 deg after storage, before transfer to 20 deg.


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"Cookies" are small bits of information which allow Ilunion Hotels to recognise its clients and avoid the Ilunionclients having to identify themselves. The cookies do not contain any type of personal information and the majority of internet browsers automatically accept cookies. In any event, if the user does not wish to be recognised it is possible to configure the internet browser not to accept cookies without the same implying any impediment to access our web-site.


Have an innovative idea and you want to develop it? Want to meet a team of people who have similar interests? Come to Columbia University's WE Pitch and meet our women mentors who can help bring your idea to fruition.
Join a supportive community of innovative women, where you can collaborate and receive mentorship for your idea. Attend workshops to learn more about your role as a woman in entrepreneurship. Experience not required! Pitches at every stage of development encouraged.
Attend WE Pitch for a weekend of prizes and pitch development!


text:
28 July 2015 @ 9:00am - 7:00pm
Sponsored by the Arab Studies Institute, Middle East Studies Program (GMU) & The Issam Faris Institute for Public Policy & International Affairs (AUB)
The meteoric expansion of the ISIS movement in Iraq, Syria and beyond during the past year has led to numerous, often conflicting interpretations of this phenomenon and its seeming success, as well as divergent perspectives on its prospects and future.
The workshop, "Understanding the ISIS Phenomenon", jointly convened by the Arab Studies Institute and the Issam Faris Center of the American University of Beirut on 28 July 2015, sought to promote and provide a multi-dimensional, inter-disciplinary examination of its subject.
The purpose of this workshop was to achieve a comprehensive understanding of the Islamic State phenomenon, both in terms of the contextual factors that have produced it; its territorial, organizational, political, socio-economic, and religious-theological features; the extent of continuity and change with respect to other Islamist and particularly other radical Jihadi movements; and informed discussion on its further trajectory.


There's a new supplement on the market for those who are suffering from memory loss, whether that is caused by old age, chemo or dementia. Cognitive impairment is an issue for nearly 13 percent of Americans studies, according to the Center for Disease Control. This supplement is called apoaequorin, and it is a calcium binding protein that comes from jellyfish. The protein was first discovered in the Aequorea Victoria jellyfish in 1962 and allows the jellyfish to produce flashes of light by releasing calcium. Apoaequorin is the main ingredient in Prevagen, which was the number one selling brain support supplement in 2016, according to Nielsen.
There is no real science to prove that this protein actually does anything to help with memory in humans. The only study that reported helping with memory problems was sponsored by Quincy Bioscience, the company that produces Prevagen. Participants underwent testing using a computerized program at four points during, and reported improvements and executive functioning and delayed recall. However, the methods used are nonstandardized test methods, and other parts of the study had methodological issues. Its assessment questions were nonstandardized, it had an open-label design, and it had a very small sample size. Since the testing methods were not standard, the study is basically useless to compare this supplement to others that try to do the same thing, such as gingko biloba or vitamin B-12.
While there is some validity to the idea that, as a protein, may support more healthy and functional cells, there is absolutely no proof that apoaequorin specifically is helpful. There's technically no proof that it is not helpful, either, but if you are worried about your mental state and your memory loss, you should take something that you know is going to help.
If you want a supplement that will actually work in the same way that apoaequorin claims to, try something like AgelessBrilliance, which contains 100 percent of your daily requirement for folic acid and 1000 percent of your needed B-12 as well as a solid dose of gingko biloba, making sure that you have more than enough of the vitamins that help your brain function.
For more scientifically proven ways to help with your memory, make sure that you are staying active in both mind and body as well as eating fatty fish like salmon and tuna regularly for the omega three fatty acids, and sticking to a Mediterranean diet.
There might be science in the future that proves a connection between brain function, memory, and this jellyfish protein, but it's not worth it without knowing any of the other potential side effects of apoaequorin in the long term. It's been proven safe for consumption, but it's never been properly tested on humans in the long term. Other methods are safer and more tested to do exactly what they say they do. They make much better options than apoaequorin.


We're going to use Halloween as an excuse to gross you out with this viral zit-popping. Believe it or not, media outlets are eating this up -- but maybe that's the wrong choice of words.
It begins with a two-finger squeeze on what looks like a creature from science-fiction. Without getting too much into detail, the violent excavation of pus and blood from the pimple leaves a crater that one observer in the video describes as a "bullet hole." "It looks like you got shot," he says. And it keeps going from there. "This is YouTube-f--king worthy," a woman can be heard saying.
We'll give you one more chance to turn back -- we won't let anyone know you were here. But if you continue forward, hey, it's your lunch to lose.


text:
The Easterner
Sports Showcase
Bloomsday brings out EWU track and field members
Thousands make their way through downtown Spokane on Sunday as a part of the 43rd annual Bloomsday.
Mckenzie Ford
By Drew Lawson, Reporter
Bloomsday is one of the vanguards of Spokane's culture. Over the past 43 years the 12 kilometer road run has become one of the largest in the world, with over 40,000 yearly participants.
Many of those 40,000 include Spokane County natives, and as long time runners, members of the EWU track team are no stranger to Bloomsday.
25 years of Bloomsday
Men's head coach Stan Kerr, a graduate of EWU, said he has done Bloomsday roughly 25 times. He didn't run this year due to prior family commitments, but said he loves participating in the event. He and his wife used to run even when their two sons were young.
"We'd initially take our kids when they were under five (years) old," Kerr said. "We'd watch the elite athletes come through. If the weather's good it's just a tremendous event."
Kerr said that when his son was still young, he ran Bloomsday while pushing his son in a three-wheeled stroller.
"That was very unusual … because the carriages (start) at the back of the pack," Kerr said. "You look up and there's 50,000 people starting in front of you."
While his time in the race decreases every year, Kerr said that participating is what matters to him. Most years, he ran for enjoyment and to spend time with family. But in 2016, he ran in memory of his friend Richard Runyon.
Runyon, was killed when piloting a plane that crashed into the Spokane River near Felts Field in 2015. Kerr said he and his wife ran to support his wife, Karen Runyon.
"It was more of an awareness for (her)," Kerr said. " I still have the shirt that she made up for all of us that wanted to go with her in that run. It was a very positive event, very uplifting."
Running since fourth grade
Many current EWU distance runners have run Bloomsday, with some having run multiple times. Freshman Jacob Stirpe, a Ferris High School graduate, ran Bloomsday with his mom in the fourth grade. A picture of them was published in The Spokesman-Review on April 30, 2009, as Stirpe's mother Jody had been interviewed about the newly debuted Bloomsday Facebook page.
Jacob Stirpe has run Bloomsday a total of seven times. He preferred to run alone, but was willing to run with others "if they were setting a good pace." Stirpe said that his best time is 43 minutes, coming in his sophomore year of high school.
"I love all the little things people are doing as you run," Stirpe said. "The bands, (people) handing out water and lemonade. There's a lot of things you get to see when you run. .. it's the one time where a lot of people come together for a big race. It's cool, not a lot of places do that."
Redshirt freshman and Spokane Valley High alum Jack Sloan ran Bloomsday for the first time last year. He said that since he was in shape, he figured should run the event since he lives in Spokane and had never done Bloomsday before. He estimates his time was around 44 minutes. Sloan said he appreciates that the race is open to anyone.
"People walk it, people jog it, people crawl it," Sloan said. "People do everything."
For the love of running
Senior Gracie Ledwith, a Lewis and Clark High School graduate, has run Bloomsday four times. Her personal best was 50 minutes. Ledwith has ran another city's road race, the Butte to Butte, a 5K competitive in Eugene, Oregon.
"I just ran for fun, because I love running," Ledwith said.
Ledwith said that the layout of Bloomsday contributes to its popularity within the Spokane community.
"Running such a fun course makes it really fun," Ledwith said. "It's a challenging course for sure … it brings the running community together."
Elite speed
Redshirt freshman Jacob Christner, a North Central High School graduate, estimates he's ran Bloomsday seven or eight times. His fastest completion of 48 minutes was in eighth grade, while running with his middle school track team, which was the last time he participated.
Christner said running has become a staple of Spokane, which heavily contributes to Bloomsday's popularity.
"It just keeps getting bigger and bigger every year," Christner said. "Because of that, they're able to bring elite runners in. That really hypes me up and gets me excited to run Bloomsday. I'm sure it does the same for other people."
bloomsday spokane
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Drew Lawson, Sports Editor
Mckenzie Ford, Photographer
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text:
{% extends "_layouts/base.html" %}
{% load crispy_forms_tags %}

{% block title %}
    Login | {{ block.super }}
{% endblock title %}

{% block headline %}

Login to xGunicorn

{% endblock headline %}

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{% crispy form %}
{% endblock content %}


Or feel free to call us at 512-878-7283. If we don't answer we are out tending to our farm, please leave a message and we will call you back!
When you call please speak very clearly when leaving your contact information so that we can respond to you. If you do not hear back from us within a day or two please call again!!! You may have cut out or we couldn't hear your full phone number, etc. Thanks! We look forward to speaking with you.
To reach us by mail: H and H Feed, LLC PO Box 582 Blanco TX 78606.
We do not sell your information and will only share it with our distributor in your area, if appropriate.
Thank you for considering H and H Feed in your search for the best chicken, duck, gamebird, swine, rabbit, cattle, horse, sheep and goat feed available.


text:
Q: sum,count,distinct count fields with awk I have a text file like this:
A   B   C   D   E
----------------------
x   x   e   2   10
y   y   g   1   8
z   o   e   2   9
o   o   q   1   10
p   z   e   3   22
x   x   e   1   11
z   o   a   1   24
y   z   b   1   25

I want to use awk do the same thing as this SQL does:
select A,
       B,
       count(distinct C),
       sum(D),
       sum(case when E>20 then E else 0 END)
  from test
 group by A,B

output:
A   B  count(distinct C)    sum(D)  sum(case when E>20 then E else 0 END)
-------------------------------------------------------
o   o   1       1       0
p   z   1       3       22
x   x   1       3       0
y   y   1       1       0
y   z   1       1       25
z   o   2       3       24

Here is my solution but the distinct part is not completed:
awk '
{
    idx4[$1"|"$2]=idx4[$1"|"$2]+$4;
    idx5[$1"|"$2]=$5>20?idx5[$1"|"$2]+$5:idx5[$1"|"$2]
}
END {
    for (i in idx4) print i, idx4[i], idx5[i]
}' OFS="\t" test

=============================================================================
I have completed this by hours, here is my code:
    {
        if (idx3[$1"|"$2, $3] == 0) {
            idx3[$1"|"$2, $3]+=1;
        }
        idx4[$1"|"$2]=idx4[$1"|"$2]+$4;
        idx5[$1"|"$2]=$5>20?idx5[$1"|"$2]+$5:idx5[$1"|"$2]
    }
    END {
        for (j in idx3) {
            split(j, idx, SUBSEP)
            count[idx[1]]++
        }
        for (i in idx4) {
            print i, count[i], idx4[i], idx5[i]
        }
    } OFS="\t"

@Scrutinizer has given a more readable code below, i think that is better.

A: Try this (similar to your own solution):
awk '
  NR<3{
    next
  }

  {
    i=$1 OFS $2
    D[i]+=$4
  }

  !A[i,$3]++{
    C[i]++
  }

  $5>20{
    E[i]+=$5
  }

  END{
    for(i in D)print i, C[i], D[i], E[i]+0
  }
' OFS='\t' infile

NR<3 is used to skip the two header lines. If they are not present in the input file you can leave that section out.

A: please try this script, I had test it can oupt the result as you expect.
awk '
{
    if( NR<3) {next}

    idx4[$1"|"$2]=idx4[$1"|"$2]+$4;
    idx5[$1"|"$2]=$5>20?idx5[$1"|"$2]+$5:idx5[$1"|"$2]

    if( index(TR[$1"|"$2],$3)==0 )
    {
         TR[$1"|"$2] = TR[$1"|"$2]"|"$3;
         TRD[$1"|"$2] +=1;
    }
}
END {
    for (i in idx4) print i, TRD[i], idx4[i], idx5[i]+0
}' OFS="\t" test

A: $ gawk '{ a[$1,$2]["C"][$3]; a[$1,$2]["D"]+=$4; a[$1,$2]["E"]+=($5>20 ? $5:0) }
END {
    PROCINFO["sorted_in"] = "@ind_str_asc"
    for ( i in a) {
        split(i, b, SUBSEP)
        print b[1], b[2], length(a[i]["C"]), a[i]["D"], a[i]["E"]
    }
}' OFS='\t' file
o       o       1       1       0
p       z       1       3       22
x       x       1       3       0
y       y       1       1       0
y       z       1       1       25
z       o       2       3       24


I am a big fan of inspirational books and "Who moved my cheese?", written by Dr. Spencer Johnson, is one of my personal favourites.
"Who moved my cheese?" is a small book that anyone can finish within a couple of hours. It is perfect for the beginners to get started and build their interest in books. The book captures the reader's attention with its beautiful and eloquent pictures which successfully help to visualise the setting.
The idea of being able to cope up with the unexpected changes of life is well illustrated by the author in a very endearing and amusing way.
The story revolves around 2 humans, named Hem and Haw, and 2 mice, named Sniff and Scurry, who used to get enough "Cheese" to eat at a particular station of a maze. They were not bothered about life's uncertainty. So one day their world came to a sudden pause when they did not find it at the usual place where they used to get it. The story will take you to an interesting tour when each of them took individual decisions for their survival.
The cheese being the metaphor for what we want in life, whether it is love, job, health, spiritual space or even an activity like jogging or sports. "Maze" represents the space where you spend your life in looking for what you want. It can be the organisation you work in, society you live in or the relationship you share.
It is a good book often recommended to professionals to find excellence and happiness at workplace. I was recommended too as well and no wonder it is a permanent member of my book shelf now.


As a food group, dark green leafy vegetables are by far the most important and yet hardly anyone consumes sufficient quantities. We want to share with you our love for greens and discuss ways to prepare them, including green smoothies. Wild edible greens will also be discussed along with a field trip around the property to identify them.


text:
His son Thomas, relationship with Françoise Hardy, his glasses, Corsica … Find out all
The sacred monster of the French song, Jacques Dutronc, who celebrated a 60-year career, was quickly removed from the stage where he interpreted hits that became cults on film sets. Not far from Françoise Hardy, with whom he formed a mythological couple, and Thomas Dutronc with whom he turned into a more complex father-son duo, Jacques Dutronc lived peaceful days in Corsica with his new partner.
Sacred monster of French song, Jacques Dutronc still at 79 this cheeky and elegant rocker hides his subtle appearance behind his timeless Ray-Bans. Once the idol of the yéyés, this Havana-smoking rock dandy who is now celebrating a 60-year career can easily disappear from the stage where he interprets hits that have become cults on film sets. Not too far from Françoise Hardy, whom he embodied in a mythical couple, and Thomas Dutronc who he formed a more complex father-son duo on the stage as in life, Jacques Dutronc found the his refuge in Corsica with his new partner.
The latest news on Jacques Dutronc
What are his biggest hits?
The first guitarist then assistant artistic director, Jacques Dutronc saw his career as a singer disappear thanks to a decisive encounter. At the end of the 1960s, the path of the young singer crossed that of the lyricist Jacques Lanzmann. Both Jacques will sign the most famous pipes of Jacques Dutronc as"And me and me and me"," "It was five o'clock, Paris woke up"," "The opportunist"where"Cacti". Songs that are almost part of the French heritage.
True to friendship, Jacques Dutronc counts his lifelong friends Johnny Hallyday and Eddy Mitchell in his happy band. The three old rascals, who had met each other as children in the Square de la Trinité, then met at legendary Golf Drouot. A fictional concert hall in the yé-yé years, the Golf Drouot was once one of the most famous radio-hook venues in Paris where Johnny Hallyday, Eddy Mitchell and the Chaussettes Noires and Jacques Dutronc made their first appearances. step on the stage.
Jacques Dutronc married Françoise Hardy, how long was their relationship?
Jacques Dutronc and Françoise Hardy were one of the most legendary French couples of the 1960s. The two yé-yé stars who started dating in 1967, waited until 1981 to finally say yes! Together they have a son, Thomas Dutronc, born on June 16, 1973. But only one year after the birth of Thomas that the singer moved with Françoise Hardy. Separated since 1987, the two artists never divorced and even remained very close.
Why did Jacques Dutronc have the reputation of being a seducer?
or Jacques Dutronc and Francoise Hady know great moments of happiness, their couple also suffered from many infidelities of the singer. In the set of The important thing is to love Jacques Dutronc lived a passionate love story Romy Schneider. But the playboy did not stop there, he is also the father of a girl named Aliyawas born in 1970 from his relationship with a Tahitian named Tera.
Who is Sylvie Duval, his partner?
In 1997, on the set of Place of Vendôme, a film by Nicole Garcia in which he played alongside Catherine Deneuve, Jacques Dutronc met Sylvie Duval who was a make-up artist on the set. Since then, the lovebirds have not left each other.
Where does Jacques Dutronc live?
Built over the years on the island of Beauty, Jacques Dutronc found refuge in Monticello, a small village located above L'Ile-Rousse. It is in this property far from the world, built by Françoise Hardy more than fifty years ago, that Jacques Dutronc lived happy days with his partner. Sylvie Duval and the many cats she adopted.
Why does Jacques Dutronc never take off his sunglasses?
If Jacques Dutronc is rarely seen without his cigar and his black leather jacket, it's almost impossible to see him without his famous sunglasses. When the Cactus singer hides his blue eyes behind his Ray-bans, it is said to protect his light-sensitive eyes.
What is his relationship with his son Thomas?
While father and son Dutronc recently recorded their first album together, the highly complex duo is currently on stage for a tour of the streets of France, Switzerland and Belgium. Now united by their common love of music, father and son don't always have a simple relationship.. The 49-year-old musician especially evokes his youth at a distance with his father "We never talk (…) We don't talk about feelings. We do little together, but we live together, we communicate a lot through unspoken words and silence." he confessed to his visit to the set Discard the arts in April 2022.
When to worry about dog vomiting?
the unique relationship between Africa and science fiction


OTTAWA, ONTARIO--(Marketwire - March 2, 2008) - We are very saddened to learn that Canadian Trooper Michael Yuki Hayakaze, of the Lord Strathcona's Horse (Royal Canadians), based out of Edmonton, Alberta, died today near Kandahar while taking part in a re-supply operation.
His team is in shock at having lost such a highly regarded member. Indeed, this loss is deeply felt by all of his comrades in the Afghan mission, as well as by all Canadians.
My husband, Jean-Daniel Lafond, joins me in honouring his memory, his valour and his determination to contribute generously, and to the best of his abilities, to restoring peace, stability and decent living conditions in Afghanistan.
On behalf of all Canadians, I would like to convey our heartfelt condolences to his family and friends. May they know that we stand with them in this moment of tragedy.


The BAOC comes in all black chrome frame with a green shock ring. The three foot aluminum oxide guides have a new high strength aluminum oxide ring. The new ring reduces weight and increases durability. High performance with a low price. The AOCC features a TiChrome frame with no shock ring.
The HLC/HLCC series of Halide ring guides are available in chrom and black chrome.
The new titanium nitride guides feature a zirconia inner ring coated with a gold titanium nitride for a super high hardness factor. The high gloss titanium plated S.S. frame looks more like jewelry than a rod component. We added black chrome and hard chrome frames for even more selection. The new braided lines necessitated this new technology.
The SCUE guides come with our super hard titanium nitride coating over zirconia ring and stainless steel frame.
The BCCB/BCUE/BCCE guides feature our blue titanium coating over a zirconia ring.


The PI3DBS16213 is a 6 to 2 differential channel multiplexer/de-multiplexer switch. This solution can switch multiple signal types up to data rate of 20Gbps. Using a unique design technique, Diodes Incorporated has been able to minimize the impedance of the switch such that the attenuation observed through the switch is minimal. The unique design technique also offers a layout targeted for USB3.0, USB3.1, 10GE, PCIe4, Thunderbolt 3, and SAS3.0 signals, which minimizes the channel to channel skew as well as channel to channel crosstalk as required by high speed signals.


Wistaria Youth Hostel is a low cost option for backpackers and students located in Xizha Scenic Area. Surroundings are beautiful and peaceful. Yida Silk Store and Shitian Square are located in front of the hostel, and the famous Water Stage is right behind. A clear winding river at the front gate and wistaria covered cloisters within are distinctive features. The hostel's buildings are of antique simplicity, and its rooms are clean and tidy. Hostel facilities include a public kitchen, showers, air conditioning, and hot water. Bunk beds in the guest rooms resemble those in a student dormitory, making a stay here reminiscent of your school days.
The hostel is equipped with a public kitchen, showers, air conditioning, and 24-hour hot water. Facilities in the room include lockers, bedside lamps, wash basins, and slippers. Towels are not provided. There is a book bar and a leisure bar on the first floor where the guests can watch TV and enjoy wireless broadband internet access service.
Each bunk in the youth hostel can accommodate only one guest.
The hostel is located in Xizha Scenic Area, and guests must purchase an admission ticket to enter. Guests may purchase tickets to Dongzha and Xizha Scenic Areas for 200 yuan/person, or an admission ticket to Xizha Scenic Area for 150 yuan/person.
Guests are required to present valid identification (government issued identification card, passport, driver's license, etc.) during check-in, and are required to use rooms according to the standard accommodation capacity for the length of their stay.


A major new initiative is being introduced at the 2014 Concours of Elegance, set to be held at the stunning Hampton Court Palace from 5 to 7 September.
For the very first time, a Club Trophy will be presented to the very best car to have won its own car club concours events earlier in 2014, in recognition of the spirit and dedication of Car Club owners, and their support of the grass roots concours movement in the UK.
Each of the major prestige car clubs in the UK has been invited to send the winning cars from their own car clubs' concours events along to Hampton Court Palace in September, to be then judged for the coveted Club Trophy on Saturday 6 September. Fittingly, the Royal Automobile Club will be coordinating the judging for this new prestigious prize.
Ultimately the overall 'Best of the Best' Club Trophy winner will be named the British 'Historic Car of the Year.' In addition to being awarded the coveted Club Trophy, the overall winner will also automatically be one of the 60 cars to be entered into the line-up for the 2015 Concours of Elegance event, to be held at an as-yet unspecified British Royal Palace.
More than 350 top cars from each of the major British car clubs will be displayed within the picturesque grounds of Hampton Court Palace on Saturday 6 and Sunday 7 September, including a number of each club's concours-winning vehicles. These cars will be displayed within a dedicated area, adjacent to the Concours.