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What is neurofibromatosis type 1 (NF1)? NF1 is one of the most common inherited neurological disorders, affecting about one in every 3,000 people.NF1 ranges from mild to severe, and can cause more symptoms in some people than in others
What is Neurofibromatosis Type 1 (NF1)? Johns Hopkins
Neurofibromatosis type I (NF-1) is a complex multi-system human disorder caused by the mutation of a gene on chromosome 17 that is responsible for production of a protein called neurofibromin which is needed for normal function in many human cell types Continued What Are the Symptoms of Neurofibromatosis? The following symptoms appear in people with NF1: Several (usually 6 or more) café au lait spot
Neurofibromatosis type I - Wikipedi
NF1 can manifest very differently from patient to patient, and no one person will have all of the possible symptoms of NF1. The severity of NF1 ranges from extremely mild cases to more severe cases, in which one or more serious manifestations may develop
Neurofibromatosis type 1 (NF1) is a genetic disorder that can affect multiple systems of the body. It is characterized by the presence of: Skin changes, such as café-au-lait spots (light brown patches on the skin), and freckles in the armpits or groin are
NF type 1 can affect many parts of the body. Most people with NF1 have some combination of these symptoms by age 10. Be aware that this list is not diagnostic of NF1
Neurofibromatosis is a term that encompasses at least two distinct, hereditary disorders caused by abnormality in a gene. Neurofibromatosis type 1 (NF1) is the more common disorder, affecting about 1 in 4,000 people throughout the world Mutations in the NF1 gene cause neurofibromatosis type 1.. The NF1 gene provides instructions for making a protein called neurofibromin. This protein is produced in many cells, including nerve cells and specialized cells surrounding nerves (oligodendrocytes and Schwann cells) This gene product appears to function as a negative regulator of the ras signal transduction pathway. Mutations in this gene have been linked to neurofibromatosis type 1, juvenile myelomonocytic leukemia and Watson syndrome Symptoms. There are three types of neurofibromatosis, each with different signs and symptoms. Neurofibromatosis 1. Neurofibromatosis 1 (NF1) usually appears in childhood Neurofibromatosis Type 1 (NF1) More common than cystic fibrosis and hereditary muscular dystrophy combined, Neurofibromatosis Type 1 (NF1) occurs in approximately one out of every 3,000 births
General Discussion. Summary. Neurofibromatosis 1 (NF1), also called von Recklinghausen's disease, is a genetic disorder characterized by the development of multiple noncancerous (benign) tumors of nerves and skin (neurofibromas) and areas of abnormal skin color (pigmentation) Neurofibromatosis (NF) is a term used to describe two completely separate genetic conditions - NF1 and NF2. These two types of neurofibromatosis are caused by different faulty genes, which may be inherited or may have spontaneously changed (mutated) at conception Neurofibromatosis type 1 (NF1) is a progressive condition characterized by skin discolorations, including large, café-au-lait-colored freckles (macules) and freckling in the armpit (axillary freckling) and groin (inguinal freckling)
Neurofibromatosis 1 and 2: Symptoms, Treatments, Cause
About Neurofibromatosis. Neurofibromatosis (NF) is a neurocutaneous syndrome that can affect many parts of the body, including the brain, spinal cord, nerves, skin, and other body systems
Neurofibromatosis (NF) is a genetic disorder that causes tumors to grow on nerves. Learn about the types, their symptoms, and how they are treated
What are the neurofibromatoses?What is NF1?What are the signs and symptoms of NF1?What other symptoms or conditions are associated with NF1?When do symptoms appear?What is the prognosis for someone with NF1?How is NF1 treated?What is NF2?What are the signs and symptoms of NF2
Neurofibromatosis type 1 (NF1) is a genetic condition characterized primarily by changes in skin color and the growth of benign (non-cancerous) tumors along the nerves of the skin, brain, and other parts of the body
NF1 Children's Tumor Foundatio
Neurofibromatosis (NF) is a genetic neurological disorder that can affect the brain, spinal cord, nerves and skin. Tumors, or neurofibromas, grow along the body's nerves or on or underneath the skin. Scientists have classified NF into two distinct types: neurofibromatosis type 1 (NF1) and NF2. NF1.
NF1. NF1 is the most common of the rare genetic conditions which come under the umbrella of neurofibromatosis. NF1 affects up to 1 in 2,500 people
1 (NF1) is a gene in humans that is located on chromosome 17. NF1 codes for neurofibro
Eye exam. An eye doctor can detect Lisch nodules and cataracts. Hearing and balance exams. A test that measures hearing (audiometry), a test that uses electrodes to record your eye movements (electronystagmography) and a test that measures the electrical messages that carry sound from the inner ear to the brain (brainstem auditory evoked response) can help assess hearing and balance problems.
Superior Performance With Less Power Consumption. The NF1 SSD delivers 4x faster performance* - up to 540K/50K IOPS and up to 3,100/2,000MB/s sequential read/write speeds - at 54 percent lower power consumption*, introducing a new caliber of performance per unit Neurofibromatosis - an easy to understand guide covering causes, diagnosis, symptoms, treatment and prevention plus additional in depth medical information Neurofibromatosis type 1 (NF1) is a genetic condition that causes tumours to grow along your nerves. The tumours are usually non-cancerous (benign) but may cause a range of symptoms. Neurofibromatosis type 2 (NF2) is much less common than NF1. It's covered separately as it has different symptoms and.
Neurofibromatosis type 1 (NF1) is an autosomal dominant, multisystem disorder affecting approximately 1 in 3500 people. Significant advances in the understanding of the pathophysiology of NF1 have been made in the last decade. While no medical therapies are currently available, trials are ongoing to. NF1 Clinical Trial of Selumetinib Most Exciting Says National Cancer Institute Director. Speaking before the House Energy and Commerce Committee of the U.S. House of Representatives on the implementation of the 21st Century Cures Act, Norman (Ned) Sharpless,.
The symptoms of neurofibromatosis type 1 (NF1) are often mild and cause no serious health problems. But some people will have severe symptoms. The symptoms of NF1 can affect many different areas of the body, but it's unlikely someone will develop all of them. The most common symptom of NF1 is the. Neurofibromatosis is a rare genetic disorder that causes typically benign tumors of the nerves and growths in other parts of the body. Some people with this disorder have barely noticeable neurological problems, while others are affected profoundly What is NF? Neurofibromatosis (NF) is a set of complex genetic disorders that affects almost every organ system, causing tumors to grow on nerves in the brain and throughout the body The NF1 gene is associated with autosomal dominant neurofibromatosis type 1 (NF1) (MedGen UID: 18013), neurofibromatosis-Noonan syndrome (NFNS) (MedGen UID: 419089), and Watson syndrome (MedGen UID: 107817) NF1, Legius Syndrome, and RASopathies TESTING OPTIONS: Blood-based. Next-Gen Sequencing and Deletion/Duplication Analysis of NF1 only (NF1-NG); Next-Gen Sequencing and Deletion/Duplication Analysis of NF1 and SPRED1 (NFSP-NG
Video: Neurofibromatosis Type 1 Children's Hospital of Philadelphi
Symptoms of Neurofibromatosis (NF) Type 1 Johns Hopkins
Neurofibromin 1. The NF1 gene is a tumor‐suppressor gene, mutation of which results in uncontrolled or poorly controlled cell growth. From: Pediatric Clinical Advisor (Second Edition), 200 Neurofibromatosis is an inherited disorder of the nervous system in which tumors develop on nerves, leading to a range of complications. Neurofibromas are usually non-cancerous and grow on the. NF1 Gene, Drug Resistance, Tissue Distribution, Mutation Distribution, Variants, NF1 Genome Browser, NF1 Reference
What is neurofibromatosis type 1? Neurofibromatosis type 1 (NF1) is a hereditary condition commonly associated with multiple café-au-lait spots on the skin Visit us on the web at bcnf.bc.ca.ca Fact Sheet: Neurofibromatosis Type 1 Neurofibromatosis Type 1 (NF1) is the most common single gene disorder to affect the human nervou
Neurofibromatosis. The neurofibromatoses primarily affect the development and growth of neural (nerve) cell tissues. These disorders cause tumors to grow on nerves and produce other abnormalities such as skin changes and bone deformities Parents Abby and Paul speak with an expert about their daughter Hannah's NF1. You will need QuickTime 5 to view the video resources in this site
Understanding NF1 Identifying: What is NF1
Neurofibromatosis 2 (NF2) is a disorder that causes tumors to form on nerves in the brain and spinal cord. Learn about symptoms, diagnosis, and treatment
NF1 is an inherited disease. If either parent has NF1, each of their children has a 50% chance of having the disease. NF1 also appears in families with no history of the condition. In these cases, it is caused by a new gene change (mutation) in the sperm or egg. NF1 is caused by problems with a gene.
Tested at 24.83% THC 0.12% CBD 0.02% CBN. If you are a regular cannabis user, there is a very high probability that you have tried either Northern Lights or Chemdawg strain
Neurofibromatosis type 1 (NF1) is a multisystem genetic disorder that is characterized by cutaneous findings, most notably café-au-lait spots and axillary freckling (see the images below), by skeletal dysplasias, and by the growth of both benign and malignant nervous system tumors, most notably benign neurofibromas
Neurofibromatosis type 1 - Genetics Home Reference - NI
Researchers either at or funded by the National Institute of Neurological Disorders and Stroke (NINDS) are working to identify signaling pathways in the nervous system, with the hope of eventually developing drugs and techniques to help diagnose and treat NF. Understanding the natural history of. Overview Neurofibromatosis type 1 (NF1) is a common autosomal dominant neurocutaneous genetic disorder, first described in the medical literature in 1882 and previously known as von Recklinghausen disease
NF1 neurofibromin 1 [ (human)] - National Center for
Hi everyone I have a son who is 2 yrs and is awaiting results for NF1 as has cafe late spots varying in size and position. Lots in groin area under arms and one large area around knee cap
NF1. NF1 is the most common of the neurofibromatoses, affecting approximately 1 in 3000 individuals worldwide. The hallmark feature is multiple neurofibromas, benign tumors that arise from peripheral nerves, but in addition there are multiple other tumor and nontumor manifestations
Because NF1 is an unpredictable disorder, it varies widely in severity - even among two people in the same family. Some people with NF1 may be completely unaware that they are affected, experiencing only a few café-au-lait spots and bumps on the skin during their lifetime. Others may develop, at.
ant genetic syndromes. Autosomal do
Neurofibromatosis type 1, is a genetic disorder that can affect many areas of the body, including the skin, eyes, bones, blood vessels, nerves and central nervous system, and increases the risk for developing certain kinds of cancerous and non-cancerous tumors
Neurofibromatosis Type 1 (NF1) Neurofibromatosis Type 1 is the more common form of the disease, occurring in 1 in 3,000 to 4,000 births. Also known as von Recklinghausen disease, NF1 mostly affects nerves of the outer parts of the body (peripheral nervous system)
The Classic RASopathy Syndromes. Scroll down to find information about and resources for the following germline mutation syndromes: CFC syndrome (CFCS
A simple animated video about Neurofibromatosis Type 1 (NF1), a genetic condition that causes lumps to grow on the nerves. We explain why it occurs, the main signs, the complications to be aware. Neurofibromatosis (NF) is a group of three conditions in which tumors grow in the nervous system. The three types are neurofibromatosis type I (NF1), neurofibromatosis type II (NF2), and schwannomatosis A single gene. Our bodies hold 25,000 genes, but one is responsible for suppressing tumor growth. That gene is known as NF1. When that gene fails, it unleashes uncontrollable tumor growth Recent Examples on the Web. Then she was diagnosed at birth with neurofibromatosis, a genetic condition where tumors form on nerve tissue. — Fiona Tapp, SELF, What It's Like to Fill in the Gaps in Your Health History When You're Adopted, 18 Jan. 2019 He was born with a crack cocaine addiction and has neurofibromatosis — a genetic condition that causes tumors to grow along the nervous.
Neurofibromatosis - Symptoms and causes - Mayo Clini
How is NF1 diagnosed? Children can have different pathways to a diagnosis of NF1, but a typical diagnostic process might look like this: The pediatrician notices some café-au-lait spots on the child's skin (these may look to the parent just like birthmarks)
Neurofibromatosis type 1 (NF1) is a common genetic disorder of deregulated cell growth, affecting approximately 100,000 Americans. People with NF1 are at an increased risk of developing a variety of benign and malignant tumors
Complications. Complications of neurofibromatosis vary, even within the same family. Generally, complications result from tumor growth distorting nerve tissue or pressing on internal organs
Neurofibromatosis Type 1 (NF1) - Neurofibromatosis (NF
This is a phase II, single-arm, open-label, multicenter clinical trial evaluating the efficacy and safety of trametinib monotherapy in patients with advanced non-squamous NSCLC whose tumors non-synonymous NF1 mutations and are KRAS wildtype
d that any of these symptoms - even neurofibromas and plexiform neurofibromas - can also appear in people who don't have NF1.. It's also important to remember that two people in the same family can both have NF1 but experience different symptoms and levels of severity
This study, A Phase II Study of Cabozantinib (XL l84) for Plexiform Neurofibromas in Subjects with Neurofibromatosis Type I in Children and Adults diagnosed with Neurofibromatosis Type 1 (NF1) and have a type of tumor called a plexiform neurofibroma (PN). Neurofibromas are tumors that develop from.
Neurofibromatosis (NF) is a genetic disorder of the nervous system which causes tumors to form on the nerves anywhere in the body at any time. This progressive disorder affects all races, all ethnic groups and both sexes equally
Neurofibromatosis 1 - NORD (National Organization for Rare
ant disorder on the long arm of chromosome 17, characterized by predo
Neurofibromatosis Support. 8,690 likes. This page was set up to help people share stories and give support to others living with NF.There are links in..
Neurofibromatosis type 1 (NF1) is often diagnosed by birth or early childhood. The first signs may be light brown spots on the skin or freckles in the armpit. Later issues may include optic nerve tumors and peripheral nerve tumors
Neurofibromatosis - Better Health Channe
Preferred test to confirm a suspected diagnosis of neurofibromatosis type 1 (NF1) for individuals not meeting NIH clinical criteria It's no secret that we're dropping pants for a cause, but how much do you know about neurofibromatosis (NF)? A lot of our participants know that they're fundraising to find a cure, but what do we actually know about the disease I'm so over this NF1 and the pain that came with mine. I just keep pushing myself a hard as I can end up in more pain days later, but I refuse to give up or in everyone ask me you say your in pain and yet you do so much
Test Invitae Neurofibromatosis Type 1 Tes
Overview of Neurofibromatosis. Neurofibromatosis (NF) is a genetic neurological disorder that affects cell growth in nerve tissue. NF produces tumors of the skin, internal organs, and nerves that may become cancerous (malignant)
Neurofibromatosis is a genetic disorder characterized by tumors or neurofibromas that grow in the nervous system and under the skin. The disease occurs as two distinct types - neurofibromatosis 1 (NF1) and neurofibromatosis 2 (NF2) - with each type having a unique set of manifestations. NF1 occurs.
The Neurofibromatosis Network is the leading national organization advocating for federal funding for NF research and building and supporting NF communities
Oncogenic alterations in the RAS/RAF/MEK/ERK pathway drive the growth of a wide spectrum of cancers. While BRAF and MEK inhibitors are efficacious against BRAF V600E-driven cancers, effective.
Neurofibromatosis (NF) is a group of genetic disorders: NF1, NF2 and schwannomatosis (shwon-oh-ma-toe-sis).NF1 (also known as von Recklinghausen disease) is the most common type, affecting an estimated one in 3000 people in Australia
Samsung PM983 NF1 NVMe PCIe SSD NF1, the highest density form factor By building the NF1 form factor for SSDs in 1U servers, maximum system density can be achieved
Neurofibromatosis (for Parents) - KidsHealt
Diagnosis of Neurofibromatosis. Family history, physical examination for clinical signs of the disease (e.g., multiple cutaneous and subcutaneous lesions), and magnetic resonance imaging (MRI) are used to diagnose neurofibromatosis Symptoms of NF-1. NF-1 is more common and is characterized by multiple (more than 5) tan patches on the skin (caf-au-lait spots), freckles in the groin or armpit, nodules within the iris of the eyes, skin neurofibromas (small, rubbery skin lesions), bone defects, and visual disorders Researchers at UC San Diego Moores Cancer Center, with colleagues from Memorial Sloan Kettering Cancer Center and Fox Chase Cancer Center, have determined that a specific region of the small bowel, called the duodenal-jejunal flexure or DJF, shows a high frequency of gastrointestinal stromal tumors. Glioblastoma (GBM) is the most aggressive tumor of the brain. NF1, a tumor suppressor gene and RAS-GTPase, is one of the highly mutated genes in GBM.Dysregulated NF1 expression promotes cell.
A number sign (#) is used with this entry because of evidence that some cases of neurofibromatosis-Noonan syndrome are caused by heterozygous mutation in the neurofibromin gene (NF1; 613113) on chromosome 17q11 The investigational drug selumetinib shrank tumors in some children with a genetic syndrome called neurofibromatosis type 1 (NF1). As this Cancer Currents post explains, the drug also improved symptoms related to tumors known as neurofibromas, which can cause pain, difficulty breathing or walking, and disfigurement What is Neurofibromatosis Type 1? NF1 is a common genetic condition that causes nerve tumours to grow where they shouldn't. The spelling mistake in the gene is found on chromosome 17 and occurs in 1 in 2,500 of the population At NF Midwest we are frequently asked what the awareness color is for neurofibromatosis for use in ribbons, etc. So we're going to attempt to set the record straight
Neurofibromatosis NF MedlinePlu
Samsung this week demonstrated progress in its new NF1 form-factor (formerly known as NGSFF and sometimes called M.3), unveiled last August. At the OCP Summit this week, the company exhibited its. Neurofibromatosis type 1 (NF1), also known as von Recklinghausen disease, is a multisystem neurocutaneous disorder, the most common phakomatosis, and a RASopathy Children's researchers work hard to treat and diagnose children with neurofibromatosis and pediatric learning disabilities The NF Research Initiative is a multi-institutional, international collaborative leveraging genomic data from NF1-related MPNSTs to develop clinical applications for improved care of our patients
Neurofibromatosis 1 (NF1) is a common neurocutaneous condition with an autosomal dominant pattern of inheritance. The complications are diverse and disease expression varies, even within families Category Number And Operations—Fractions Sub-Category Use Equivalent Fractions As A Strategy To Add And Subtract Fractions. State Standard: Add and subtract fractions with unlike denominators (including mixed numbers) by replacing given fractions with equivalent fractions in such a way as to produce an equivalent sum or difference of fractions with like denominators Non-optic gliomas occur in 5% of children with NF1, but little is known about these tumours in adults. We aimed to investigate progression, spontaneous regression and the natural history of non-optic gliomas in adults and compare these findings to the results found in children Neurofibromatosis type 1. Article Translations: () () What is neurofibromatosis type 1? Neurofibromatosis (nur-oh-fie-broe-mah- toe-sis) type 1 (also called NF1) is a condition that causes skin changes as well as tumors along the nerves in the body
Neurofibromatosis Fact Sheet National Institute of
What We Do. The Neurofibromatosis Therapeutic Acceleration Program (NTAP) is focused exclusively on improving treatment options for people living with Neurofibromatosis type 1-related plexiform neurofibromas Neurofibromatosis type 1 (NF1) is a skin condition that causes growths on nerves. Find out what NF1 genetic testing is, what the test results mean and why to consider testing for it e485 J Clin Exp Dent. 2011;3(5):e483-6. Neurofibromatosis Type I - oral manifestations Neurofibromatosis (NF) is a genetic disease that causes tumors to develop in the nervous system. There are three types of neurofibromatosis that are each associated with unique signs and symptoms
A number sign (#) is used with this entry because neurofibromatosis type I (NF1) is caused by heterozygous mutation in the neurofibromin gene (NF1; 613113) on chromosome 17q11
Neurofibromatosis type 1 Genetic and Rare Diseases
NF1 is a life-long condition usually diagnosed early in life, often within the first year. NF1 is diagnosed based on specific skin, ocular, and other physical findings, and genetic blood testing in selected cases The typical age of presentation for sporadic OPGs typi-cally is less than 8 years (6,11). Similar to NF1-associated OPGs, however, onset can occur in the second decade Awareness, Education, & Support. Consider making a financial contribution - donations of all sizes can serve to get new community groups launched, cover expenses associated with global communications or build a fund to provide grants to the medical and research communities
Neurofibromatosis genetic testing by Ambry. This test can detect ~65% of described mutations in the gen - Multiple café-au-lait spots in a patient with NF1 - Neurofibromata and cafe-au-lait macules - Axillary freckling in a patient with NF1 - Lisch nodules - Plexiform neurofibroma in a child with NF1 Neurofibromatosis Midwest is dedicated to the increasing number of men, women and children challenged by neurofibromatosis. Help raise NF support Key points. There are two ways your child's doctor can diagnose NF1: clinical diagnosis and genetic testing. Clinical diagnosis means the doctor looks for symptoms of NF1 on the child's body
ICD-9-CM 237.71 is a billable medical code that can be used to indicate a diagnosis on a reimbursement claim, however, 237.71 should only be used for claims with a date of service on or before September 30, 2015 Neurofibromatosis (NF) is a genetic disease in which multiple soft tumors (neurofibromas) develop under the skin and throughout the nervous system Neurofibromatosis type 1 (NF1), was referred to in the past as von Recklinghausen disease or sometimes as peripheral neurofibromatosis. The term NF1 is now preferred Presentation [6, 8]. The major NF1-associated tumour is the neurofibroma. In addition, clinical manifestations include bone dysplasia, learning disabilities, and an increased risk of malignancy a gene associated with neurofibromatosis. The gene is normally part of a family that helps regulate the timing of cell divisions. It may become defective, leading to neurofibromatosis expression, when an itinerant sequence of a deoxyribonucleic acid molecule becomes wedged in the NF1 gene
Neurofibromatosis type 1 (NF1) is a clinically heterogeneous, neurocutaneous, genetic disorder characterized by café-au-lait spots, iris Lisch nodules, axillary and inguinal freckling, and multiple neurofibromas The Massachusetts General Hospital Neurofibromatosis Clinic provides comprehensive care for adults and children who have been diagnosed with, or are at risk for, various forms of neurofibromatosis (NF), including NF1, NF2 and schwannomatosis Neurofibromatosis (NF) is a genetic abnormality that affects the cell growth of neural tissue, leading to tumor growths that impact the skin, nervous system, eyes and other organs. NF is divided into two primary subgroups: neurofibromatosis type 1 (NF1), also known as von Recklinghausen or. • Neurofibromatosis 1 (NF1) is a common autosomal dominant neurocutaneous disorder caused by germline mutations in the NF1 gene at chromosome 17 that encodes the protein neurofibromin; penetrance is complete, but expression is highly variable and age-dependent See more images of neurofibromatosis.... Other features of NFI. The severity of cutaneous involvement in NF1 is not an indicator of the extent of the disease as internal manifestations are common and are often more serious
NIH Diagnostic Criteria for NF1. Clinical diagnosis based on presence of two of the following: 1. Six or more café-au-lait macules over 5 mm in diameter in prepubertal individuals and over 15mm in greatest diameter in postpubertal individuals The NF1 gene, located on chromosome 17, encodes neurofibromin, a protein needed for normal function in many human cell types. Loss-of-function mutations in the NF1 gene can lead to neurofibromatosis type 1 (NF-1), one of the most common dominantly inherited genetic disorders with a birth incidence of 1 in 2,000-3,000 Neurofibromatosis type 1. This is the most common form of neurofibromatosis. Its symptoms include: Six or more brown spots (know as cafe-au-lait spots) on the skin measuring more than 5 millimeters in children and more than 15 millimeters in adolescent NeuroForce1 (NF1) is a progressive neuroscience & medical-grade diagnostic testing company that emphasizes the importance of training and reprogramming the nervous systems (enteric, autonomic & central; the brain & gut) in conjunction with the physical body to optimize human athletic performance, overall health & longevity
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text:
Melenciuc este un nume de familie. Se poate referi la una din următoarele persoane:

 Emelian Melenciuc (1871–d. sec. al XX-lea), politician țarist, deputat în Duma de Stat din partea Basarabiei.
 Alexandru Melenciuc (n. 1979), fotbalist moldovean.

redpajama_set_name: RedPajamaWikipedia


text:
Q: Ubuntu installation error on Windows 10 I am getting the following error when installing Ubuntu (version?)

WslRegisterDistribution failed with error: 0x80370102

Please enable the Virtual Machine Platform Windows feature and ensure virtualization is enabled in the BIOS.

For information please visit https://aka.ms/enablevirtualization

Press any key to continue...

I already enabled it in bios, I have hyper v, and I followed the steps in this link about installing wsl. https://answers.microsoft.com/en-us/windows/forum/all/encountering-wslregisterdistribution-error/dc9508ac-7ece-4d07-a695-24c7b0a592c1
I have a custom build PC model z370 A Pro
Bios version 2.40, Intel Core I7 8700K is this is needed information.

redpajama_set_name: RedPajamaStackExchange


Fort Macon offers access to the beach, the sound and the Beaufort inlet. It is also a historic landmark for this area. It is located at the east end of Bogue Banks and has water on 3 sides, the ocean, the sound and the inlet. This is a perfect place to explore or to go to the park that is associated with it. The park has beach access with a board walk and shelter, a bath house, concession stand, picnic areas and life guards.
Fort Macon is the home of a Civil War fort. It has five sides and is built out of bricks and stones with walls that are 4 ½ feet thick. There are 26 vaulted rooms. In the 18th & 19th centuries, Beaufort and the surrounding area were vulnerable to attacks. There were pirates that came through the Beaufort Inlet, like Blackbeard, and there were the wars with Spain, France and Great Britain which provided threats. Beaufort was actually captured by the Spanish in 1747 and in 1782 it was captured by the British. After that the people realized the need for protection and started building forts. The fort was built where it is because of the access of the inlet. The first fort was named Fort Dobbs but it was never finished. So in the early 1800's the US government decided to build coastal forts and a fort known as Fort Hampton was built. In 1825, a hurricane swept this fort right into the ocean.
The war of 1812 proved the lack of coastal defenses, so the US government built what is now Fort Macon. Construction of the fort started in 1826 and took 8 years to complete. Its purpose was to guard Beaufort and Beaufort Harbor since it was a deep water port.
Fort Macon was a federal prison in 1867 until 1876. It was garrisoned in the Spanish/American War and then closed in 1903. After that, Congress sold the fort to the state and it was made a state park. It was restored in 1934-1935. It was garrisoned for the final time during WWII. The cost of building the fort was $463,790.
When the Civil War began, only two days went by before NC military forces from Beaufort seized the fort for NC and the confederacy. They occupied the fort for a year and prepared it for battle adding 54 cannons.
Confederates in the fort that refused to surrender. The Union forces bombarded the fort from land and sea. They hit the fort 560 times with rifled cannons. The damage was so bad that the Confederates were forced to surrender and they were taken as prisoners. The Union held Fort Macon for the rest of the war and Beaufort Harbor was a coaling and repair station for its navy.
The US Army occupied Fort Macon during the Reconstruction Era. There were no state or federal penitentiaries in the military district of NC or SC so this fort was used for that purpose for 11 years. The fort was deactivated in 1877 only to be garrisoned again in 1898 for the Spanish-American war. The US army completely abandoned the fort in 1903. Fort Macon was offered for sale as surplus military property in 1923, but in 1924, a Congressional Act sold the fort for a sum of $1 to the state of NC to be used as a park. In 1934 & 1935 the Civilian Conservation Corps restored the fort and on May 1, 1936, NC's first state park opened. When WWII started the US Army leased the park from the state and it was manned with Coast Artillery troops from December 1941 to November 1944. On October 1, 1946 the Army returned the fort and the park back to the state.


So you went onto babywisp.com, filled up your cart with a bunch of your favourite hair accessories, paid, waited for delivery and were so excited when they finally arrived! But then, you try to put these clips in your baby girl's hair and she takes them right out! That's ok, you have come to the right place- we CAN help you.
Getting your baby girl to keep a hair clip in her wispy fine hair- if she isn't used to the idea- can really be a challenge. Over the past couple of weeks we have been very inspired by the methods our Baby Wisp mamas use to keep these clips in.
First and foremost, you must try our MINI LATCH CLIPS!!
Thank you so much for sharing your methods- some of you are so lucky to have girls who just naturally adjust to having a clip on their head! If your daughter is not as comfortable don't panic- just try some of these suggestions and she will be wearing bows in no time!
Use a toy ( or a doll) as a distraction. Some times babies just need theirattention to be switched and they will totally forget that the clip was put in!
You have your self a happy and well accessorized baby girl!
A couple of weeks ago we saw this video from Youtube Mommy and Hair Guru Whitney from Naptural85. If you skip ahead in this video to 3:48 Whitney shows you how she puts 2 bows in her daughters hair side by side so that she will pull out the second one - and completely ignore the one you actually want to stay in! We talked to some Baby Wisp mamas at our Spring Session and they also found that this trick has worked for them!
Do you have any other tips and tricks that you use to keep your baby girl from pulling out her hair bows? Share your ideas on Facebook, Instagram or Twitter using the hashtag #babywisp- we would love to hear your unique ideas!
Our shop will re-open in late August when you can place your order and try out our products!


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Johnston Press survives — but at a human cost
Johnston Press has escaped its debts — but it leaves behind angry investors, and hits employees right in the pensions.
The Final Days of Johnston Press
The clock is counting down to the end of Johnston Press, one of the great regional newspaper companies in the UK. The company is doomed - but can local news be saved?
Adam Tinworth 22 Oct 2018 • 3 min read
The digital transition of local news
How are the traditional local publishers managing the transition to digital? The panelists would like us to think it's going well, but the audience is skeptical.
Buzzwording your way to irrelevance
Johnston Press has clearly mastered the art of buzzwording more than it has the art of social media: "Twitter users are much more engaged with news brands," he said, so the network's place in Johnston Press' social strategy is "to get the virality

redpajama_set_name: RedPajamaCommonCrawl


Real talk for real kids (and parents too!). Join our discussion of temptations and trolls, boundaries and bullies, and just knowing when something isn't right, online or in person.
If you're transitioning or if you'd like to start a conversation with your kids, or if you aren't sure of your best parenting strategy--this workshop is for you!
Join FSYF and Laurie Tasharski from the International Center for Missing and Exploited Children for this informative and fun workshop.
Registration is required in advance of the program.


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Date of Birth November 04, 1920
Date of Passing January 18, 2016
Vivian Routten Wilson
from Hampton, Virginia, United States
HAMPTON - Vivian Routten Wilson, 95, a lifelong resident of Hampton, passed away at Sentara CarePlex on Tuesday, October 18th. Vivian grew up in Fox Hill and graduated from Hampton High School in 1938. She worked as a secretary for the Cummings Law Firm until the birth of her first son. Vivian was the widow of Kenneth (Kenny) F. Wilson, Sr. She was also preceded in death by her parents, Bessie and Edward Routten, sister Betty Karasick, and brother, Edward Routten. She is survived by her two sons, both of Hampton, Kenneth F. Wilson, Jr. (Gwen) and Curl S. Wilson; two grandchildren, Kathryn Claeys (Ed) of Columbus, OH and Kenneth F. Wilson, II (Christina) of Hampton; and two great grandchildren, Mackenzie and Zachary Claeys. Vivian was an avid bridge player and was a member of the ladies bridge group where she played at the Hampton Yacht Club for many years. Vivian most of all loved her family. After moving to the Devonshire, she always looked forward to family night on Tuesdays and to Wednesday lunch at the Yacht Club where she was known by the staff and was greeted with a glass of wine and a few jokes. Vivian had been in excellent health until this recent illness. She was proud that she was able to get around without a cane at 95. She was just shy of 96, being born on November 4th, 1920. Vivian will be missed by her family and a void has been created that only time can fill. A graveside service will be held at 11 am, Monday, October 24, 2016 at Parklawn Memorial Park. The family will receive friends at R. Hayden Smith Funeral Home, 245 S. Armistead Avenue, Hampton, VA 23669 on Sunday from 6-8 PM. Following the burial there will be a reception, details to follow.
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Somali President Addressed the Session of the Parliament
Mogadishu, Somalia (tf.sf ) Somali President Sharif Sheikh Ahmed addressed a session of the Somali Parliament on Wednesday. In a wide ranging speech, the President urged MPs to herald a change in the government as it moves to tackle key challenges.The president began his address by paying tribute to the fallen MPs and the injured, as he also saluted the Parliamentarians for their fortitude in this trying time.The President informed the Parliament that the collective responsibilities the leaders and TFG institutions share, necessitates timely gathering and serious consultations and reflection. The President said that "there has been number of reshuffles to improve the political and security situation and to reinvigorate the government, but the security situation has gotten worse."On the controversial proposed constitution, the President also informed the Parliament that half of the Independent Federal Constitutional Committee were not approved by the Parliament and without this whatever they are working on will not be legal unless and until they are approved by the Parliament. "It is my responsibility to uphold the law. We need the drafting process to go through the proper legal channels." The President added. Emphasising the need to get the constitution making process right, the President said, "Constitution it is the most important document fashioned for a nation; it is what shapes their destiny, it reflects their history and it is the foundation of their government … the constitution embodies the soul of the Somali people …we cannot play fast and loose with this … we will support its making provided it is done properly."Speaking of the transitional tasks and the end of the TFG mandate the President said, "We should not be preoccupied with what we will be doing after 2011; we should be occupied with what we should be doing before 2011. We have security problem and that is our foremost priority.""What's left of the transitional period is short and long, if we produce tangible results, we have lots of time but if we fail to produce we won't have much time. Let us aim and intensify our efforts to produce results; that we can." The President remarked.Speaking of the alleged infighting among TFG leadership, the President said,"There is not infighting among Somali leaders. There is no conflict between myself and the PM or myself and the Speaker and there is no problem between myself and the Chief Justice.""You elected me to discharge duties and among my duties are appointing a Prime Minister who shall form a government and I am responsible for his supervision. If he falls short of our expectations, it is my duty to come back to you and inform you this is not working and I should ask you where shall we go from here, because I cannot be salient on the squandering of the future of Somalia…Reform is possible. Let us march forth for positive change…" The President said.
Three hundred-plus MPs attended the session.
Contact: media@presidency.gov.so


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Finding the root cause of ERP implementation failures
The root cause of ERP implementation failures may be delays in decision-making. Delays add up, which wastes time and imposes extra costs on a deployment.
Patrick Thibodeau, Senior News Writer
ERP implementation failures, caused more often than not by decision-making delays, are a major risk. Delays in decision-making are cumulative, they slow progress and gradually derail the ERP project.
That's part of the argument made in a paper, partially titled "Why Large Projects Fail," by Jim Johnson and Hans Mulder, who are both on the faculty of the University of Antwerp Management School. Johnson is also chair of the Standish Group, a research organization that studies software projects.
The paper looks at a major software project involving the Number Portability Administration Center, an example that sets the stage for a larger discussion. Very large software projects, costing over $10 million, face a failure rate as high as 41%, according to the paper's data. Only 4% can claim success; the rest are "challenged," meaning they are running overbudget or overtime.
Johnson further discussed project delays in this Q&A. His responses were edited for clarity and length.
Are ERP implementation failures common, and if so, why?
Jim Johnson: Most projects don't die from starvation. They die from indigestion. What ERP, especially, is trying to change is the whole landscape. It is trying to force-feed all these features, functions and changes on the organization down people's throats. That's where the real challenge is.
If you have a $1 million project, you have a thousand decisions to make. If you have a $100 million project, you have 100,000 decisions to make. Those are hard to swallow. They become bottlenecks. At some point, the decision latency becomes so long and convoluted you just choke on it.
What is decision latency?
Johnson: It's the time between when an issue is raised and when the decision is made -- that's called the interval. The longer the interval, the longer the decision latency. You measure latency by the length of the interval.
If decisions are delayed, what happens?
Johnson: People go off and do other things. They get demoralized.
Aren't some of these decisions leading to ERP implementation failures relatively minor? For instance, a configuration decision? Is that the kind of thing that can slow down a project?
Johnson: Yes -- you need to distribute the decision-making to the right level. We see this, especially, in government and highly regulated and controlled organizations. They don't give power to the people that actually know what they are doing, so those decisions get moved up [to people] that don't have the time to make the decisions, or they don't have enough information, or they're not equipped to make that decision. They need to move that decision down to people that can make a good decision and let them have the freedom to make those decisions.
How do you best avoid decision latency?
Johnson: The first thing you do is you become aware of it. We call it the 'root cause of all project performance problems.' It causes things to get screwed up. Secondly, you need to distribute decisions down to people that can make decisions and not be punitive if they make a mistake. The interval is more valuable than the quality of your decision. If you make a mistake, you reverse it; you fix it. You want to benchmark decision latency -- let's look at how long it takes for us to make a decision.
Is it costlier to delay a decision than it is to make a mistake in implementation?
Johnson: Yes.
What's the best way to manage a project?
Johnson: You want to have a competent executive sponsor. Committees are useless. The executive sponsor should have complete authority -- unfettered responsibility. He or she is totally responsible for the success of the project.
One of the things your paper discusses is downward trajectory of projects. Is that easy to recognize, and what typically is the response?
Working code is the real test of progress. Everything else is just window dressing.
Jim JohnsonChairman, Standish Group
Johnson: Usually, the response is the opposite of what should happen. You see projects that are green and green and green, and suddenly, they are red. No one wants to stand up and say the project is going south. It's an emotional thing. What you need to see is evidence of delivery. Working code is the real test of progress. Everything else is just window dressing.
The initial response is to cover up project delays?
Johnson: In the more Agile-type delivery, they don't. They are actually empowered to say things are going wrong.
You need to have a competent team that you can trust, that's talented but also has good emotional maturity. The relationship of the team is so important. When we see high-performance teams, we see people that like to work together. They have a good working rapport.
A lot of companies bring on systems integrators. How does that work in this model?
Johnson: The problem -- and this is my own opinion -- is the integrator is not working for the best interests of the client. They want to extend the time. They want to set high decision latency because that's billable hours.
How can you bring in a systems integrator and have a successful project?
Johnson: [One thing to require] is a firm deliverable every two weeks so there is no hiding. You want to give them freedom to deliver, but you want them to deliver.
By: Linda Rosencrance
By: Andrew Froehlich
coded orthogonal frequency-division multiplexing (COFDM)
By: Rahul Awati
Pandemic will delay, cancel ERP deployments and test vendors
By: Patrick Thibodeau
4 Things You Need to Know Now About Edge Computing –HPE
'One Size Fits All' IT Will Strangle Digital Businesses with a Lack of Choices –Citrix
One of the Keys to Digital Transformation Success: Enhancing the Customer and ... –Dell Technologies
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Santa Fe's ERP implementation project fix stirs debate – ERP
For one company, an ERP project failure is now a ... – ERP
Spatial analysis a portal to deeper insights from data
When combined with other types of data, location information can add important context that leads to the well-informed insights ...


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Eternal Soldier Starter Campaign
The Free Prologue Becomes the Eternal Soldier Campaign.
Raiders of the Broken Planet is getting a huge change in preparation for the future releases of the first season of content. What was once the Prologue has now become the Eternal Soldier free Starter Campaign featuring two missions: the single-player tutorial "First Blood" and the 4 vs 1 multiplayer mission "Hanging by a Thread".
With full access to the games' features, players who download the free Eternal Soldier Starter Campaign will be able to use gold, faction points and character points to unlock new characters, build weapons from blueprints, unlock Faction and Character Cards and unveil pieces of lore from the world of Raiders of the Broken Planet. Eternal Soldier includes a total of seven playable characters and sets the stage for the incredible story to follow in Alien Myths and the other parallel stories of the next three campaigns of the first season.
The business model for Raiders of the Broken Planet is unique in its release of a free to play base game, supplemented by individual story campaigns. The naming of the Prologue has caused some confusion among gamers. With the re-naming of the Prologue to the Eternal Soldier Starter Campaign, MercurySteam want to re-emphasize the importance of this campaign, serving as the base game from which the additional campaigns build off from.
The re-introduction of the free campaign in concert with the opening of the economy for players, lays the groundwork for the future of Raiders of the Broken Planet. Any Xbox One, PS4 or PC player will now be able to experience the thrilling and unique 4 vs 1 action of Raiders of the Broken Planet and enjoy the awesome customisation options as they earn loot and of course get to grips with the incredibly diverse set characters, who all bring their own unique flavour to the battlefield.
Players who want to experience the story of Raiders of the Broken Planet can purchase additional campaigns, each of which contains 4 intense missions. These missions occur in parallel, focusing on different characters and factions. The first campaign, Alien Myths is available now, while the second, Wardog Fury, will launch before the end of the year.
Watch the amazing new Eternal Soldier Campaign trailer here!
Tweet to @mercurysteam
Ginebra Now Available for All Alien Myths Players.
Reward System Hot-Fix Update
Inside Raiders #02
Killfest Event!
Animated Comics


MEET Millie Innes, the cute as a button young Scottish actress who is the star of a brand new eponymous BBC television show.
The 13-year-old from Cardonald, Glasgow, has been cast in the lead role in CBBC comedy series, Millie Inbetween, which starts on Wednesday. It charts the growing pains of blended family life for the fictional Millie and her older sister Lauren after their parents split up.
Ms Innes, who has previously had roles in dramas Case Histories and Single Father, landed her own show after first impressing BBC bosses when she auditioned for a part in the Tracy Beaker spin-off series Dani's House three years ago, beating 20,000 youngsters to the role of Maisy.
Millie Inbetween, which was filmed over the summer at the River City studios in Dumbarton, looks set to make the teenager a household name, but Ms Innes is keeping both feet planted firmly on the ground.
"A few of my close friends know about the show but I don't tend to talk about it in school," she says. "Some of my teachers suggested we should do something to celebrate, but I told them I'd prefer not to. People will find out about it in their own time. I don't want to be seen to make a fuss, brag or show-off.
"We don't make a big thing of it as a family," she adds. "It's such a normal thing for me now and just like any other person going out to get a weekend job in Sainsbury's."
Ms Innes, who is in third year at Ross Hall Academy and attends the Glasgow Academy of Musical Theatre Arts, lives with her parents, Julia, 41 and Campbell, 50, and younger brother Murray, five.
By the time she was two, such was her burgeoning passion for acting and dancing that her father built a small, wooden stage in her bedroom. While still at primary school, Ms Innes won parts in cBeebies shows Nina and the Neurons and Me Too! before landing her breakthrough role in Single Father opposite David Tennant when she was nine. Most recently she played the daughter of Jason Isaacs in popular crime drama Case Histories.
She may share a name with her on-screen alter ego in Millie Inbetween, but Ms Innes is keen to emphasise that it is a character she plays. "I was anxious about Millie being perceived as me and the real Millie," she says. "I was able to have a say in what she is like, though. I would describe her as quirky and the boss of the house. When things go wrong, everyone looks to her."
Ms Innes admits to rarely watching her own acting performances afterwards, saying that as perfectionist she would only pick them apart. "Millie Inbetween was a bit different," she says. "We had a screening for it, so I was forced to sit through an hour and a half of my face on a big cinema screen. I don't watch myself usually because I'm my worst critic and always think: 'I could have done that a bit better' or 'why did they choose that take?'"
Away from acting, she enjoys going to concerts and having sleepovers with her friends. Her favourite subject at school is English, one which Ms Innes hopes to study at Oxford University when she is older. Pursuing an acting career, however, remains her main ambition. "My dream job would be a gritty Hollywood movie similar to what Natalie Portman did in Leon when she was around my age," she says.
Her mother Julia confirms that from an early age the signs were there that her daughter was destined for a thespian's life. "When Millie was six or seven, she loved the Tracy Beaker show," says Mrs Innes. "It used to drive myself and Campbell insane, hearing the title music over and over as she watched all the repeats. One day we were sitting on the sofa with Millie sandwiched between us, wearing her tutu and high heels, and she said: 'I'm going in a show like that one day, I promise you.'"


The former Veritas of the Flame, and forger of Purple Lightning. He loves food, and eats like a food fighter. Or rather, since he spends more time eating than doing anything else during the day, he's exceeded the level of food fighter. Although he incessantly shoves food into his mouth, it seems he is actually quite particular about taste.
Remove all status effects from one enemy ?
Full Steam Ahead Increase ATK (100%) for 3 turns to caster ?
Physical damage (0.3x) as MP drain (10%) to one enemy ?
Increase LB gauge (5) to one ally ?
Increase LB gauge (3) to caster ?
Chance to add fire element to physical attacks (50%) for 5 turns to all allies ?
Increase chance of being targeted (100%) for 3 turns to caster ?
Increase fire resistance (100%) for 5 turns to all allies ?
Decrease fire resistance (75%) for 3 turns to one enemy ?
Double Stamp Use Stamp skills twice in one turn ?
Twofold Forge Use Forge skills or Forging Friendships twice in one turn ?
Critical Stamp Physical damage (5x) with critical hit to one enemy ?
Add fire element to physical attacks for 2 turns to caster ?
Increase LB gauge (10) to all allies ?
Auto-heal (1800 HP, 1x) per turn ?
MP +20% Increase MP (20%) ?
ATK +30% Increase ATK (30%) ?
DEF +30% Increase DEF (30%) ?
Flame Axe Wielder Increase fire resistance (50%) when equipped with an axe ?
Increase resistance to blind, paralyze, and petrify (100%) ?
HP +30% Increase HP (30%) ?
Axe Mastery Increase ATK (50%) when equipped with an axe ?
Warrior's Forte Increase HP/MP/ATK (20%) ?
Auto-cast Strike While the Iron's Hot! at the start of a battle ?
Auto-cast Full Steam Ahead at the start of a battle ?
Smith of Flames Strike While the Iron's Hot! Enable skill for 1 turn to caster: Strike While the Iron's Hot!
Strike While the Iron's Hot! Strike While the Iron's Hot!
Firaga Fire magic damage (1.8x) to all enemies ?
Firaja Fire magic damage (2x) with consecutive increase (4 times, 1x each, 6x max) to all enemies ?
The man in Raegen's team in charge of cooking. Generally not very detail-oriented, he is surprisingly skilled at cooking dishes with subtle flavors. To say nothing of Sieghard, his cooking ability even surpasses that of Citra and Folka. His specialty is curry and fried chicken with rice, and is constantly on the lookout for ingredients both in town and on the road.
He was quite popular with women during his time as one of the Sworn Eight of Paladia. His valorous personality also had a serious side, and he always returned favors, which made him a hit among the ladies. However, Ignacio prioritized spending time with his friends over any woman that came along.
Strike while the iron's hot. You can strike me too, but don't be surprised when I strike back!
As long as you maintain a good core, there's no limit to how strong you can get. You'll have bulging muscles in no time!
Eat well. Train well. That's how you get strong. Better yet, eat while you train.
Glad I removed that uncomfortable armor!
This power is yours to wield! You have my trust and I will swing my axe at your command.
Gimme someone tough to fight! Otherwise, what's the point of working out?
Our bond is like a fine blade—unbreakable, untarnished.
Está disponible como unidad invitada en el Evento de Historia, Herro de las Llamas.
Los jugadores puedes probar sus habilidades como unidad invitada en Misión de introducción de unidad desde 2/15 - 2/28 PST.
Para su versión Veritas, visita Veritas of the Flame.
Ignacio has no spoken dialogue when performing his CG-animated Limit Burst. He instead grunts and groans while executing his Limit Burst, befitting his personality as a formidable warrior.


text:
Q: How write non blocking code in nodejs and what I am doing wrong? I have a route named pdf and if I go to the route it waits 10 sec because I create a pdf and other things. But if I go to other screen and the pdf route is not finish yet then my other response waiting and I can not use the site until pdf route is finish. How can I not block the response ?
PDFCreate file
var pdf = require("pdf-creator-node");
const path = require('path');
import fs from 'fs-extra';

export default async function PDFCreate() {
  try {
    var options = {
      format: "A3",
      orientation: "portrait",
      border: "10mm",
      header: {
          height: "0mm",
      },
      footer: {
          height: "28mm",
          contents: {
              first: 'Cover page',
              2: 'Second page', // Any page number is working. 1-based index
              default: '{{page}}/{{pages}}', // fallback value
              last: 'Last Page'
          }
      }
  };
    console.log(path.join(__dirname));
    const html = fs.readFileSync('app/data/file.html', 'utf-8');
    const bitmap = fs.readFileSync('public/images/lg.jpeg');
    const logo = bitmap.toString('base64');
    const filename = Math.random() + '_doc' + '.pdf';

    const document = {
      html: html,
      data: {
        logo
      },
      path: './docs/' + filename
  }

    pdf.create(document, options)
    .then(res => {
        console.log(res);
    }).catch(error => {
        console.log(error);
    });

    const filepath = 'http://localhost:3000/public/images/' + filename;

    return filepath;
  } catch(e) {
    console.log(e);
    throw e;
  }
}

/PDF route (I use fullstack framework so the loader is equivalent with this: route.get('/s', (res, req)'))
import PDFCreate from '~/data/createPDF.server'

export default async function loader() {
  try {
    await PDFCreate();
    return null;
  } catch(e) {
    console.log(e);
  }
}

/home
export default function Home() {
  return (
    <>

test

    </>
  )
};

So when I call /pdf and its loading and if I go to home route then my home route loads until /pdf is finish... how can I not block this?

A: You have part of the code with await PDFCreate();, which makes your whole code stop and wait for PDFCreate() to finish.
And that is one-threaded, but there is a fix for it, and you can read about it on this question, you could use node-worker, as they suggested, or you could try the other's answer solution.

A: You can try to make use of abort the PDFCreate() execution on unmounting of loader component using AbortController.
For Example -

*

*Firstly wrap the function code to promise and register callback for abort event.
PDFCreate file
  var pdf = require("pdf-creator-node");
      const path = require("path");
      import fs from "fs-extra";

          export default async function PDFCreate(abortSignal) {
              return new Promise(function (resolve, reject) {
                  // Wrap to promise

                  // register abort event
                  abortSignal.addEventListener("abort", () => {
                      // 6
                      const error = new DOMException(
                  "Calculation aborted by the user",
                  "AbortError"
              );
              reject(error); // 8
          });

          try {
              var options = {
                  format: "A3",
                  orientation: "portrait",
                  border: "10mm",
                  header: {
                      height: "0mm",
                  },
                  footer: {
                      height: "28mm",
                      contents: {
                          first: "Cover page",
                          2: "Second page", // Any page number is working. 1-based index
                          default:
                              '{{page}}/{{pages}}', // fallback value
                          last: "Last Page",
                      },
                  },
              };
              console.log(path.join(__dirname));
              const html = fs.readFileSync("app/data/file.html", "utf-8");
              const bitmap = fs.readFileSync("public/images/lg.jpeg");
              const logo = bitmap.toString("base64");
              const filename = Math.random() + "_doc" + ".pdf";

              const document = {
                  html: html,
                  data: {
                      logo,
                  },
                  path: "./docs/" + filename,
              };

              pdf
                  .create(document, options)
                  .then((res) => {
                      console.log(res);
                      resolve(); // Resolve on success
                  })
                  .catch((error) => {
                      console.log(error);
                  });

              const filepath = "http://localhost:3000/public/images/" + filename;
              return filepath;
          } catch (e) {
              console.log(e);
              throw e;
          }
      });
    }

*Now initiate abortController in loader component and pass the abortController while calling PDFCreate() function.
Call abortController.abort() on unmounting of loader component, maybe here you can use framework-specific component life-cycle method that.
/PDF route
import PDFCreate from "~/data/createPDF.server";
export default async function loader() {
    let abortController = null;

    // Call abortController.abort when loader() component unmounts
    if (abortController) abortController.abort();

    try {
        abortController = new AbortController();
        await PDFCreate(abortController);
        return null;
    } catch (e) {
        abortController = null;
        console.log(e);
    }
  }


ASAP Drain Guys & Plumbing is a full-service commercial and residential plumbing company available 24 hours a day, 7 days a week. We provide the utmost quality workmanship by highly trained, uniformed plumbers who are the best in the business. Unlike other plumbing companies, ASAP Drain Guys & Plumbing is experienced in all types of plumbing, sewer, septic and gas line work. We understand that properly functioning plumbing is vital to your home or business so we work around the clock. ASAP Drain Guys & Plumbing specializes in sewer and drain cleaning as well as all aspects of commercial, residential and industrial plumbing.
ASAP Drain Guys & Plumbing is experienced in all types of plumbing, sewer, septic and gas line work. We understand that properly functioning plumbing is vital to your home or business so we work around the clock.
ASAP Drain Guys & Plumbing has top of the line sewer cameras to video inspect sewer lines to see if there are any obstructions, roots or even cracks and broken portions. Cameras also help to quickly identify the problem's location thus saving time and money. You can see your results on a private YouTube link!
We install traditional and tankless water heaters and also highly recommend annual maintenance to be done to flush and drain your water heater to remove sediments that collect inside. This will make your water heater more efficient and extend the life of it.
Timely, professional, friendly service. Reasonable rates with honest assessment of your plumbing needs and the experience to do the job right.
Paul from ASAP was at my house quickly. I called at 6:05pm with my problem, He was there by 6:40pm. He was able to explain the problem by using a camera to scope the drain while I watched on an Ipad. He also went through the house and showed me where the lines ran through the house and how they all tied in. He is a great teacher of the plumbing system. I am very appreciative that he came promptly, was extremely nice, and was gracious enough to share his knowledge! Thank you so much ASAP-I will keep your card in a safe place for the next time I need it!
Mark W.Polite, professional & good value.
Some of the best customer service I've had in a while. They were flexible and had techs to my house very quickly. The techs were polite, helpful and did the job right. I definitely recommend ASAP Drain Guys.
Tanner and Christian did an excellent job repairing deteriorated drains in my bathroom. They arrived on time, worked long hours, were courteous and considerate, and kept my house clean. I highly recommend them.
Ramon C is great! My wife and I have used ASAP Drain Guys & Plumbing for our rental property and our personal residence. They are very professional and personal. Their prices are fair. Ramon recently came out for a noisy drain pipe when we flushed our toilet. He took the time to explain his approach to our problem and used a cost effective solution. I had other individuals say we would have to cut the drywall. As it turned out, the insulation around the vent pipe in the attic above our bathroom had fallen off causing a boombox affect to our walls. Ramon found the simple resolve without cutting any drywall saving us hundreds of dollars on unnecessary work. This company always approaches our plumbing problem as if they owned our home. Give Ramon C a call, you will always get a solution to your plumbing needs. Thank you for your great service (again)!!


text:
Poblenou é um bairro atual e histórico da cidade de Barcelona, no distrito de Sant Martí, no nordeste da cidade.

Sua origem está em um dos núcleos habitados de Sant Martí de Provençals que cresceu pronto na indústria e habitantes, habitualmente imigrantes. Chegou a ser, nos finais do , a área com a maior concentração industrial da Catalunha, e entre as maiores da Espanha, pelo que a sempre, San Martín ou Povoado Novo receberam o apelativo de «a Manchester Catalã». Desde 2006, o bairro está limitado entre as ruas Jaume Vicens Vives, Avinguda d'Icària, Ávila, Pere IV, la Llacuna, a Avenida Diagonal, Bac de Roda, o passeio do Taulat, e a Rua Bilbao até o espigão da Mar Bella. Seu eixo central e cívico mais popular é a Avenida do Poblenou que atravessa o centro de seu centro histórico de mar a montanha.

Tradicionalmente a extensão era maior e formavam parte deste território as zonas dos bairros adjacentes que, após uma revisão dos movimentos vizinhais sobre o projeto de divisão administrativa em bairros de Barcelona (2006), incluíram o topônimo «Poblenou» nesses bairros: El Parc i la Llacuna del Poblenou, La Vila Olímpica del Poblenou, Diagonal Mar i el Front Marítim del Poblenou e Provençals del Poblenou.

Educação, cultura e trabalho

No final de 2008, o bairro disponha de cinco centros de educação infantil, cinco de educação primária, quatro de secundária, uma escola de línguas, duas de informática e uma escola universitária (Bau - Escola Superior de Disseny)

Abriga também três teatros (de entidades associativas), sendo que o de maior relevância é o Teatro Cassino Aliança del Poblenou.

Forma parte do bairro, a praia do Bogatell e o Parc del Poblenou, na frente marítima. Em 2008 foi inaugurado o Parc del Centre del Poblenou, um parque desenhado pelo arquiteto francês Jean Nouvel, situado no histórico conjunto fabril de Can Ricart.

Outras instalações e serviços

Neste bairro encontram-se um mercado municipal, com o mesmo nome, e oito centros de culto: quatro igrejas católicas, três evangélicas, um salão do reino. Para além disto, também tem um Posto de saúde de serviço para a zona. O Cemitério de Poblenou é um cemitério antigo e de considerável valor artístico, próximo ao litoral, e foi construído pela Câmara Municipal de Barcelona em território de Sant Martí de Provençals quando era independiente, inicialmente aliado de zonas habitadas.

Arquitetura

O passado industrial do bairro tornou-se em numerosas fábricas que foram abandonadas e posteriormente recuperadas para diferentes usos, como lofts, estúdios e como sede de diferentes entidades. Neste local, também se encontram lojas construídas durante o  e XX, centradas no núcleo tradicional do bairro (ao redor da rambla com o mesmo nome).

Ao longo dos últimos anos, tem havido uma remodelação intensa, com a construção de edifícios modernos e a reabilitação de outros. Grande parte destas alterações têm sido promovidas pelo projeto Distrito 22@.

Transportes

O bairro conecta-se com o resto da cidade através da Estação Poblenou, da rede de metro de Barcelona. O setor é limitado pela diagonal permite o acesso a rede de elétricoa do Trambesòs mediante as estações Pere IV e Fluvià. Além disso, há 7 estações do sistema de transporte Bicing.

Ligações externas
 Ajuntamento de Barcelona, El Poblenou
 Web da Associação de Vizinhos de Poblenou
 Arxiu Històric de Poblenou
 ElPoblenou.com

Bairros de Barcelona


Justin Sun the owner of Tron Structure has actually welcomed and recognized the support to Tron [TRX] offered by Journal Blue hardware budget. Justin Sunlight has actually recognised this assistance from Ledger Blue on the prominent social networks website, Twitter. It has currently been few months that Tron has actually received aid in some of the Journal hardware purse variations.
Nevertheless, in the recent times, this particular version of hardware has obtained support. Tron is created with a democratic vision having a blockchain method that is open source as well as decentralised in nature. Networks of various types are sustained by Tron Network.
For the objective of developing apps with the help of software, it has Virtual Equipment for using the programmers. It has lately struck the crypto headings with Tron's effective initiative to obtain BitTorrent, the Super Agent election and also the launch of Online Maker.
The current situation suggests that the Journal works as the key manufacturer of the hardware budgets, worldwide. The incredibly popular models belong to this company and also they started offering their item much befire various other competitors began doing. This has actually aided the firm to have a top edge in market space. In 2014, the business has actually been developed in France with Éric Larchevêque as the CEO.
Just recently, Binance, one of one of the most prominent and fastest expanding cryptocurrency exchanges will be conducting Tron [TRX] trading competitors. The competitors will certainly be getting one million TRX symbols distributed by the exchange. The competitors has actually currently commenced from 6th September, 2018 and also would be continuing till 13th September, 2018.
For the function of creating apps with the help of software program, it has Virtual Maker for making use of the programmers. It has recently struck the crypto headings with Tron's effective initiative to obtain BitTorrent, the Super Rep political election as well as the launch of Virtual Device.


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Tennis player Elliot Benchetrit told off by umpire for asking ball girl to peel a banana for him
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Norma Hunt is going to her 54th Super Bowl. This time, her team, the Kansas City Chiefs, will join her
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If you don't ask, you don't get. So a reporter asked Serena Williams about her good friend Meghan Markle. Guess what? They didn't get. To recap, Markle and her husband Prince Harry, the Duke of Sussex, have decided to give up their royal titles in Britain and will split their time with between the United…


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Ostre koło – popularna nazwa bezpośredniego przeniesienia napędu stosowanego w pierwszych rowerach lub roweru z takim rodzajem napędu. Ostre koło jest stosowane w rowerach torowych i akrobacyjnych. Ze względu na niską wagę i niezawodność rozwiązanie jest popularne również wśród kurierów rowerowych.

Rower z ostrym kołem nie ma wolnobiegu, piasta tylnego koła jest połączona na sztywno z kołem łańcuchowym, a dalej poprzez łańcuch z korbami. Rower na ostrym kole ma zwykle tylko jedno przełożenie (jedno koło łańcuchowe na korbie, jedno koło łańcuchowe na tylnej piaście). W rezultacie gdy obraca się tylne koło, ma to bezpośrednie przełożenie na obrót pedałów, nie jest możliwa jazda bez pedałowania np. z górki, tak jak na rowerach z wolnobiegiem, ale można jechać do tyłu albo stać bez podparcia nogą (tzw. stójka).

Ostre koło można uzyskać poprzez nakręcenie koła łańcuchowego na tylną piastę na wolnobieg lub piastę torową która ma dodatkową kontrę na lewy gwint zapobiegającą odkręceniu koła łańcuchowego.

Zastosowanie
Ostre koło jest stosowane, oprócz kolarstwa torowego, do zimowego treningu kolarzy szosowych w celu wyrobienia nawyku ciągłego pedałowania oraz przyzwyczajenia do jazdy na ciągłym obciążeniu (brak możliwości redukcji biegu).

Akrobaci wykorzystują także ostre koło do różnych ewolucji, m.in. stójki, jazdy do tyłu, kręcenia się na tylnym kole.

Także w kulturze kurierów rowerowych ten typ pojazdu jest popularny (zwłaszcza zimą), ze względu na jego lekkość, niezawodność, względnie niski koszt.

Układ przeniesienia napędu
Rowery


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Cricket Buzz – It is becoming difficult for Rohit Sharma to lead the side in the absence of Kohli
by admin/ December 12, 2017/ news/ No Comments/
SPORTSLIBRO CRICKET – CRICKET BUZZ – LATEST CRICKET NEWS
1 SPORTSLIBRO CRICKET – CRICKET BUZZ – LATEST CRICKET NEWS
1.2 As per the Latest Cricket News & Cricket Buzz Rohit Sharma the frontline batsman of the India cricket team said that the team is playing as a unit across formats and the performance that have put in is very much up to the mark but the last Odi against Sri Lanka didn't seemed fruitful for the side
As per the Latest Cricket News & Cricket Buzz Rohit Sharma the frontline batsman of the India cricket team said that the team is playing as a unit across formats and the performance that have put in is very much up to the mark but the last Odi against Sri Lanka didn't seemed fruitful for the side
Rohit as of now is leading the team and is playing very well in the formats he is been a given chance to perform and the way team is playing need to be supervised to gain confidence
"The way we are playing, credit goes to everyone. Because irrespective of whether you play for a city or country, you have to win a match and it is not an easy task. We have won six back-to-back series because we have played together as a unit," Rohit said during a promotional event.
In the limited overs format, India defeated West Indies 3-1 in their backyard, they whitewashed Sri Lanka 5-0 in the island nation and decimated Australia 4-1 and this is not the end of what they have conquered
The right-handed batsman was speaking to reporters after the launch of Adidas Uprising 3.0 here. Unless a team performs collectively, it is difficult to sustain performances.
"If you (the team) want to win a tournament, it not that that one or two players will perform and you will win. It is essential that everybody contributes as this is a team sport. We are going towards a goal and that I think is good for team.
"The way the team is thinking that is in one direction, which is helping the side and everyone is contributing, not just one or two players," Rohit added.
Asked whether the goal was 2019 World Cup, Rohit said, "See, there is one and half year for the World Cup. Now, it will be too early for me to comment on it. Yes, our aim is the 2019 World Cup and the preparations are on."
"Like our selectors, coach and captain have said the way our team is playing and the combination of the team has been made keeping the World Cup in mind. It is clear that what the team (management) wants from the players."
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Fahrenheit 451 Scavanger Hunt
In Fahrenheit 451
heit Fahrenheit 451 Scavenger Hunt What is Sci-fi? Sci-fi is a kind that deals primarily with the effect of actual or thought of science upon society or people. If science issues itself with discovery, then science fiction concerns itself with the repercussions of discovery. It is a testimony to the visionary nature of the kind that science fiction authors forecasted the development of atomic weapons and sentient devices. It endures worth though remains in its capacity to ask probing concerns of each new clinical advance, to perform a dialogue with development that decodes its real meaning and exposes it to us.
Isaac Asimov defines science fiction for us. He says, "Modern science fiction is the only kind of literature that consistently considers the nature of the modifications that face us, the possible repercussions, and the possible services. That branch of literature which is interested in the effect of clinical advance upon humans." It is likewise essential that science fiction must to some level be in accordance with science, it must not be entirely incredible, since it then ventures into the category fantasy.
The border line does get fuzzy with some strange examples of combined magic and technology, but there is still a basic line separating sci-fi and fantasy: whether it is plausible or not. Science fiction was made possible by the rise of contemporary science itself, especially the revolution in astronomy and physics. Early sci-fi authors Mary Shelley who wrote Frankenstein( 1818 ), Robert Louis Stevenson Who composed Strange Case of Dr. Jekyll and Mr. Hyde (1886 ), and Jonathon Swift Who composed Gulliver's Travels( 1726 ). The Foundation for Fahrenheit 451
Define Communism (use meaning 2): a system of social organization in which all economic and social activity is managed by a totalitarian state controlled by a single and self-perpetuating political celebration. Answer the following questions about McCarthyism. 1. Throughout the 1940's and 1950's, what was a main concern for the United States? The Cold War/Red Scare 2. Who implicated more than 200 federal government officials of being Communists? Joseph McCarthy 3. Besides government officials, what other professions were implicated of being Communists and forced to give up working?
Directors, authors, the Hollywood Ten 4. How many artists were blacklisted in all? 320 Answer the following concerns about the Cold War. 5. When did the Cold War start? 1947 6. Who was the war between? United States and the Soviet Union 7. What is the distinction in between a Cold War and a Hot War? A hot war is a physical war, where individuals are physically battling each other using weapons. A cold war is indirect battling … no weapons involved. It's more political. 8. Why were the US and Soviet Union the only 2 superpowers following WWII?
The United States was the only nation with nuclear weapons and a strong sufficient military, so it was the only superpower. However, the Soviet Union had spies reveal the area of the United States nuclear weapons, therefore the quarrels began in between the 2 nations. 9. Explain the three groups in the Cold War. The United States led the West. This group had countries who were democratic. The Soviet Union led the East. This group had countries who supported communisms. The non-aligned group consisted of nations that did not want to be connected to either the West or the East. 0. Why did the Communist party gain popularity throughout Western Europe during WWII? Having bandwagon by setting up posters and marketing that they transcended. 11. Explain the Truman Teaching. Developed in the US by president Harry Truman and it said the US would provide support such as military political and economic to any nations under risk from the authoritarian forces. Address the following questions about the 1950's. 12. What production did President Truman authorize in 1950? Development of H-bomb 13.
Recognize the technology that was mass produced in 1950. Cars 14. What was popular in the United States in 1951? Tv 15. What kind of gowns was produced in 1951? Short evening dress with empire bodice and built-out skirt 16. Who were Julius and Ethel Rosenberg? They were executed for passing nuclear weapon tricks to the Soviets. 17. In 1953, what was found? DNA (double-helix structure and four nucleotides) 18. What was released in 1954? The first nuclear powered submarine 19. What became unlawful in the United States in 1954?
Partition 20. What historical event took place in 1955? Rosa Parks arrest starts Civil liberty Movement, US military intervention in Iran, and Warsaw Pact offers integration of military, financial, and cultural policy in between eight Communist countries 21. What crucial gadget did Robert Noyce and Jack Kilby invent in 1956? Microchip 22. In 1957 what did Britain do? Where did it happen? What are a few of the ramifications of it today? Great Britain introduced a hydrogen bomb over Christmas Island in the Pacific.
United States and Britain resumed collaborating on nuclear weapons, which likewise permitted Britain access to United States nuclear weapon styles. 23. What did the Journal of the American Medical Association determine as a problem for Americans in 1957? Alcohol addiction 24. What was designed for the US Navy in 1958? Navy launches Vanguard 1 into orbit 25. What ended up being a popular hangout for teenagers in 1958? Movies, drive-ins, and skating rinks (where they held "sock-hop" dances). Censorship likewise played a huge role in the structure of Fahrenheit 451. We will go over censorship in class.
451 Degrees Fahrenheit Is the Temperature at Which Book-Paper Catches Fire, and Burns.
Fahrenheit 451: Major characters and symbols


At Community Bank & Trust, we've developed a powerful corporate culture, one that's evident in every facet of our organization. It's based on the common goal of providing exceptional, personal service and built on the core values of teamwork, mutual respect, and honesty. There are no greater assets than our employees and it shows in our work environment, community focus, employee programs, and benefits.
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Each year, Community Bank & Trust hosts a number of employee events. The only objectives are fun and fellowship. In addition, employees gather annually for a company-wide annual retreat. The day away from the office promotes discussion on many topics and features fun team-building exercises.