Les Pràctiques Externes corresponen a un treball de laboratori eminentment pràctic encaminat a consolidar, perfeccionar i completar la formació adquirida a través dels diferents mòduls teòrics del màster. Aquesta estada proporciona als estudiants l'oportunitat de guanyar experiència i aprendre a desenvolupar-se en l'entorn professional.
Les estades de pràctiques duren entre tres i sis mesos i es poden realitzar en un laboratori de recerca del VHIR o en una institució externa, com centres de recerca o empreses farmacèutiques i biotecnològiques. Durant aquest període, l'estudiant estarà supervisat per un tutor de pràctiques, que guiarà i avaluarà el treball diari de l'alumne.
El VHIR ofereix a tots els estudiants la possibilitat de realitzar les seves pràctiques en un dels laboratoris de l'Institut de Recerca. Els estudiants interessats han d'adreçar-se per correu electrònic a la persona de contacte indicada en cada oferta.
Una cop haguis estat admès per fer pràctiques en un grup de recerca o empresa, hauràs d'adreçar-te a la Secretaria Acadèmica del VHIR per tal de signar l'acord de col·laboració i fer tots els tràmits necessaris per iniciar l'estada.
Regulation of gene expression during cell differentiation and cancer.
Tutor: Ester Sánchez Tilló – PhD, Miguel Servet I principal investigator | Data d'obertura: 06/11/2018 | Centre en pràctiques: IDIBAPS – Department of Oncology and Hematology. Human and Experimental functional oncomorphology. CELLEX P1B, Facultat de Medicina, Universitat de Barcelona-Hospital Clínic i Provincial de Barcelona, c/Casanova 143, 9333275400, ext 3325, Barcelona.
The proteasome mediated system is involved and regulates the development, metastasis and chemoresistance of colorectal cancer. Colorectal cancer is one of the diseases with the greatest impact in our society, being the second and third cause of cancer death in women and men, respectively. The group investigates the regulation and functionality of the E3 ubiquitin ligase GRAIL in the different colorectal cancer subtypes harboring activating mutations of the KRAS, BRAF oncogenes and inactivating mutations of the tumor suppressor APC. For this purpose, we will retrospectively use human and transgenic mice samples of colorectal cancer to characterize the expression of GRAIL and its value as a biomarker for predicting the different clinical parameters associated with prognosis, diagnosis and treatment. By using cellular systems, we will determine whether GRAIL expression is dependent on TGFβ-Smads, EGF-MAPK/PI3K and Wnt-β-catenin pathways involved in tumorigenesis of colorectal cancer. Transcriptional regulation of GRAIL by EMT transcription factors and miRNAs induced by these pathways will also be studied. Research will aim at the study of GRAIL ubiquitination targets by protein arrays and the functions of these targets in the development and metastasis of colorectal cancer. For this purpose, organoid cultures from patient samples and cellular systems will be used in experiments ex vivo and in vivo. This translational study will assess the use of GRAIL as a potential target for the sensitivity or resistance to chemotherapy treatments used in the clinical practice of metastatic colorectal cancer.
The study of the HIV reservoirs that persist in patients treated with antiretroviral drugs and novel therapeutic strategies to target the persistent virus.
The Pediatric Neurology Research group is mainly involved in the study of genetic diseases of the developing nervous system. The main emphasis is on neuromuscular disorders, pediatric movement disorders and paroxysmal neurological disorders, such as epilepsy. A common theme across the different projects, besides the identification of the molecular basis of several of these rare disorders, is the investigation of molecules involved in their pathophysiological mechanisms and the effective translation of these findings into the fields of molecular diagnosis, genetic counselling and newly developed gene or drug therapies.
We are a research group located in SJD Barcelona Children's Hospital in the area of Developmental Cancer. Our final aim is to translate the basic knowledge acquired in the laboratory into the clinic.
We focus on Ewing Sarcoma and Rhabdomyosarcoma (RMS). The project on which the student will be involved is the identification of new therapeutic targets for each subtype of RMS and develop customised treatments in animal models generated in the laboratory that recapitulate the original tumours.
- Previous work experience with animal models will be positively evaluated.
Genetic predisposition to radiotherapy-induced toxicity: identification of cellular and molecular biomarkers as predictive test for late toxicity after radiotherapy. Assess the role of microRNAs and long non coding RNAs in the susceptibility to radiotherapy-induced clinical toxicity.
Extracellular vesicles, biomarkers and translational medicine: The knowledge of extracellular vesicles and their analysis have experienced an important boost in the past few years. Our group has been working for more than 10 years on its characterization, isolation and function, both from a basic research point of view (as tools to induce immunological tolerance and/or tissue repair) and translational (with special interest in the definition of biomarkers associated to pathological processes in renal and cardiovascular diseases, etc.).
The main objective of our research group is to deepen our knowledge on the molecular mechanisms underlying tumour pathology to identify new diagnostic, prognostic and therapeutic targets. We have chosen to focus on the mechanisms underlying female breast cancer, the most diagnosed malignant neoplasm and the second leading cause of cancer-related deaths. Our current research aims at understanding the molecular mechanisms of clonal cooperation within heterogeneous tumours.
We are looking for highly motivated candidates, interested in the field of oncology and aiming at pursuing a professional career in the field of Biomedicine, Molecular Biology or other related fields. Candidates must hold a B.S. in Biomedicine or a related field and average score should be at least 2 (scale 1-4). Previous experience in molecular and cell biology techniques, work with animal models as well as good English skills will be positively evaluated.
Lysosomal diseases are a group of rare diseases in which the defect of a lysosomal protein results in the accumulation in the lysosomes of non-metabolised products that derive in diverse pathologies. The current treatment of lysosomal diseases consists of enzyme replacement therapy in which the defective / mutant enzyme is administered parenterally. This therapy is applied with relative success in the diseases of Gaucher, Fabry, Pompe and mucopolysaccharidosis I, II, IV and VI. However, since recombinant proteins are not able to cross the blood-brain barrier. The treatment is not applicable in diseases with neuronal affectation (Sanfilippo, Niemann-Pick, Tay-Sachs, etc.).
In this scenario, the DDT/FVPR group attempts to improve enzyme replacement therapy in Fabry and cystinosis disease. To this end, specific models have been developed and various drug delivery systems have been tested from polymer capsules (Nazarenus et al, Particle & Particle System Charact 2015), chitosans (Gianotti et al, ACS Appl Mater Interfaces, 2016), liposomes (Cabrera et al. Adv Heahtc Mat 2016, Cabrera et al., Nano Letters, 2013) and alternative systems (pegylation, aggressives, etc.). Work is currently being done on optimizing nanoliposomal systems within the Smart4Fabry project (www.smart4fabry.eu) as well as studying the effect of protein corona in the biodistribution of different nano systems in a recently founded AES-FIS project (PI18/00871).
- High marks in undergraduate studies (>7.5 on a scale 1 to 10) is desirable. Please, send complete CV and academic records along with the application.
Alzheimer´s disease (AD) is the most frequent cause of dementia and manifests with progressive memory impairment, language disorders, apraxia and deficits in visual function. There is no viable therapy to slow or reverse AD progression. The key pathological changes observed in AD brains are amyloid-b (Ab) deposited extracellularly in plaques, and hyperphosphorylated tau (p-tau) accumulating intracellularly as neurofibrillary tangles (NFTs). Additional changes include reactive microgliosis and loss of neurons, white matter and synapses. Brain function depends on coordinated interactions between neurons and glial cells that include microglia, astrocytes, and oligodendrocytes. Role of astrocytes has gained much interest in recent years as a strategy to develop new treatments for the disease.
We aim to study the factors implicated in the communication between astrocytes and neurons as AD progresses. The role of astrocytes, as a significant player in neuronal signaling, may elucidate some potential targets for treatment. For this goal, we will study the interactions of astrocytes with neurons using in vitro models, such as immortalized human astrocytes and neuroblastoma cell lines. We will investigate the proteins secreted by activated astroctyes (with Abeta or proinflammatory cytokines, a situation mimicking the alteration observed in AD) and their effects on neurons (viability, death, differentiation, expression profile), with particular interest in alterations in signaling pathways and expression of antiapoptotic molecules.
1. Study of the role of lipid metabolism in the pathogenicity of PD associated to mutations in the GBA.
2. Development of new preclinical therapies for the restoration of GBA activity through i) enzymatic replacement therapy with recombinated GBA nanoconjugates (collaboration with ICN2-UAB) and ii) pharmacological chaperones (collaboration with Gain Therapeutics).
3. Cystic fibrosis from childhood to adulthood: risk factors for premature onset of adult comorbidities.
Exosomes and its biomedical application: We are currently running several research lines concerning exosomes for biomedical use, including cancer, tissue regeneration and rare disease (Fabry disease) . The offered position is to cover the master student position that will learn the basic techniques to isolate, to validate and to use exosomes in in vitro settings.
1) Investigation of the molecular mechanisms of colon cancer. Different techniques of cell and molecular biology will be used to study the molecular mechanisms involved in the initiation and progression of colorectal tumors using in vitro cellular systems, animal models and large collections of human primary tumors.
2) Identification and validation of new molecular markers of response to treatment for cancer. Here, high throughput techniques will be used to identify new biomarkers of response to chemotherapeutic treatment for colorectal cancer patients, that will subsequently be validated in large collections of patients with colorectal cancer that received chemotheapy.
Experience working in a research laboratory with techniques of cell and molecular biology/High marks in undergraduate studies (>7.5 on a scale 1 to 10) is desirable.
We are interested in how direct cell-to-cell communication via gap junctions is implicated in healthy and diseased tissue and in particular cancer. See our review: "Gap junctions and cancer: communicating for 50 years. Nat Rev Cancer. 2016 Dec;16(12):775-788.PMID: 27782134" LINK.
We study the gap junction proteins (connexins) as well as the channel protein Pannexin 1, promising prognostic markers and therapeutic targets. Techniques used in the lab includes cell communication assays, cloning, cell culture (overexpression and RNAi-knockdown using virus), CRISPR-technology, drug-response studies (cell viability, proliferation and apoptosis assays), protein analysis (Immunohistochemistry, western blot) etc.
We value the contribution of each Master student highly, and will give them plenty of freedom to develop their own project, with full support from the rest of the team members. Possible student projects will be tailored to the latest results and the experience of the student. We will provide daily interaction, training, feedback, discussions and will be eager to search for funding for the possibility for a PhD position.
WHERE TO APPLY: trond.aasen@vhir.org (attachment of CV, support letters and/or previous mini-projects, will be highly appreciated if available). Informal visits and questions are welcomed.
TITLE: DONOR-DERIVED CELL-FREE DNA AS A NON-INVASIVE BIOMARKER AFTER LUNG TRANSPLANTATION.
Graft dysfunction is one of the main causes of death after lung transplantation. The aim of this study is to identify the role of donor-derived cell-free DNA (ddcfDNA) levels in the lung transplant recipient as a non-invasive biomarker of primary graft dysfunction (PGD), infection and rejection. Besides, the association of this biomarker with the development of chronic lung allograft dysfunction (CLAD) would be studied. To do so, pre-transplant genotyping will be performed with RT-qPCR for the detection of an informative INDEL polymorphism for each donor/recipient. Subsequently, with the aim of determining the levels of ddcfDNA, the informative INDEL will be quantified by digital PCR in the plasma of the recipient at different time points after transplantation. The association of these measurements with the onset of PGD, infection, rejection and the development of CLAD will be analyzed.
New therapeutic targets and factors predictors of resistance in metastatic prostate cancer/The role in the resistance to therapy of newly discovered differential factors will be studied in metastatic prostate cancer cells by molecular and cellular biology techniques/ Function of the oncogenic protein PTOV1 in the resistance to drugs in cancer cells. Identification of specific inhibitors.
Candidates should have good academic records (>2, scale 0 to 4), good English level, and be highly motivated.
Relapsed or metastatic tumors acquire multi-drug resistance, raising the need for alternative treatments. Owing to the diverse mechanisms that are responsible of tumor chemoresistance, we aimed to target epigenetic factors that control multiple pathways to bypass therapy resistance. The modulation of chromatin structure or function is a promising approach to targeting gene expression programs that sustain the proliferation and viability of tumor cells. The combination of different epigenetic elements is considered to be tissue-specific; therefore, identifying and understanding cancer-specific dependencies on chromatin regulatory activities pose a major challenge. We perform functional screenings to identify those epigenetic regulators for each tumor type that contribute to the proliferation and viability of the tumor cells and provide new targets for therapeutic intervention. We also test new epigenetic drugs on models of pediatric tumors of the nervous system such as Neuroblastoma or Ependymoma.
The impaired B-amyloid (AB) clearance and a deficient efflux across the blood-brain barrier (BBB) are key points involverin Alzheimer's Disease (AD) and cerebral Amyloid Angiopathy (CAA). Due to the evident involvement of certain Apolipop ApoE isoforms, these lipid.carriers appear to be excellent candidates to mediate the AB traffic througth thr BBB. In the deeply investigate the association of different variants of apolipoproteins and AB, regarding BBB-crossing efficiency and using in vivo in vitro models of AD/CAA.
willing to be enrolled or enrolled in the course of experimental animals and have a record equal or higher than 2-2.5.
The main research lines of our laboratory focus at understanding the molecular mechanisms that control energy homeostasis in the organism and how alterations in such mechanisms are linked to the development of metabolic diseases, including obesity, type 2 diabetes or metabolic syndrome.
Iron deficiency is one of the most prevalent nutritional deficiencies in the world population. However, it is unknown whether this deficiency modifies myocardial tolerance to ischemia or the progression to post-infarction adverse ventricular remodeling.
The student will participate in a project that addresses the effect of iron deficiency induced by diet on the tolerance to myocardial ischemia and the occurrence of post-infarction adverse ventricular remodeling in an in vivo mouse model with transient coronary occlusion. The student will be directly involved in the analysis of the mechanisms associated to oxidative/nitrosative stress and eNOS/sGC/PKG pathway inhibition in correlation with the development of myocardial fibrosis induced by iron deficiency.
Motivated students with interest in basic and clinical research and good academic marks.
The project will be focused on increase our knowledge on endometrial carcinogenesis to improve the clinical management of the disease. We work in the areas of early diagnosis, prediction of recurrent patient and identification and validation of therapies.
We propose to perform "in vitro" experimental studies to assess the molecular changes when RAS blockade is combined with an SGLT2 inhibition in the renal cells mainly involved in podocytes, SGLT2 has demonstrated a protective effect in diabetic kidney disease. Its effect is mainly related to the glucosuria and natriuresis for its direct action in the proximal tubular cells. However, other mechanism seems to be involved. We propose to study the effect of SGLT2 and RAS blockade on the podocyte in diabetic conditions (with modifications of glucose in culture media). We will study fibrotic and inflammatory genes within the podocyte.
We are a young group at VHIO devoted to sarcoma translational research, with an emphasis on biological understanding of sarcomas oriented to drug development. Particularly, our group deepens on mechanisms of oncogenic addiction to oncogenic KIT signaling in GIST models with clinically relevant primary and resistance mutations. The final goal is to model cutting-edge therapeutic strategies to target tumor heterogeneity.
Immunosenescence in multiple sclerosis. Innate immunity as potential therapeutic target in multiple sclerosis. The general objective of the study is to investigate the impact of aging in adaptive and innate immune responses, both in the periphery and in the central nervous system in the context of multiple sclerosis (MS). For that, we propose the following specific objectives: i) To study the clinical course and the histopathological features of experimental autoimmune encephalomyelitis (EAE) in young and in old mice; ii) To study the immunological changes that occur during EAE in young and in old mice.
The study will be conducted in the experimental model of MS, EAE. In vivo studies will be carried out to characterize the clinical evolution of animals of different ages. Histopathological and ex vivo and in vitro immunological studies will also be carried out to fully characterize how aging influences the immune system in the periphery and in the CNS in MS.
The number of elderly people is exponentially growing worldwide. Advanced age increases the risk of myocardial infarction (AMI) and aggravates its clinical outcome, due to reduced tolerance to ischemiareperfusion (IR) of senescent hearts. Because advanced glycation end-products (AGEs) accumulate with age and are associated with oxidative stress, we hypothesize that increased production/accumulation of AGEs are determinants of the deleterious effects of aging on cardiac function and susceptibility to IR. The purpose of this study is to investigate the determinants of AGE production/accumulation using myocardial samples, isolated cardiomyocytes, cultured cell lines and in vitro mitochondria obtained from the heart of aged mice, and to determine the functional consequences of glycative damage of certain key proteins involved in mitochondrial function and cell survival (i.e., mitochondrial ATP synthase). The obtained results will be corroborated in mitochondria and myocardial human samples obtained from patients undergoing cardiac surgery.
Ischemic heart disease is the leading cause of death and disability worldwide. The social impact of ischemic heart disease is enormous, not only due to its mortality, but also because of its high morbidity, the loss of quality of life and its high economic costs. Cardiomyocyte death is the most important factor that determines mortality and morbidity associated with ischemic heart disease. Therefore, the treatment of choice in patients with acute myocardial infarction is to reperfuse the ischemic tissue as quickly as possible (through angioplasty or thrombolytic treatments). However, despite the improvements made in recent years to shorten the time of application of reperfusion, many patients end up with extensive areas of infarction, which will eventually affect their quality of life. For this reason, research in the field is now focused to develop adjuvant therapies added during reperfusion, with the aim to reduce reperfusion injury, that is, to increase the number of surviving cardiomyocytes, or to reduce the subsequent adverse remodeling (which includes collagen deposition and hypertrophy of surviving cardiomyocytes). Currently, we are investigating treatments aimed at reducing the size of the myocardial infarction by inhibiting mitochondrial succinate dehydrogenase, or to attenuate adverse remodeling by modulating the expression of connexin 43 (Cx43), a protein that forms channels in the plasma membrane of cardiomyocytes, and that is essential for the propagation of the cardiac electrical impulse. The candidate would join one of these two lines (depending on the degree of progress of each one obtained up to the date of incorporation). In the first line, therefore, we intend to determine the utility of the selective inhibition of succinate dehydrogenase in reperfused myocardium by intracoronary administration of malonate to prevent reperfusion damage after transient coronary occlusion. We will use a porcine in situ heart model of ischemia-reperfusion, together with atrial and plasma samples from animals and patients. Techniques to be used include nuclear magnetic resonance, Western Blot, or immunofluorescence. The second line aims to analyze the role of Cx43 in myocardial scarring, adverse left ventricular remodeling and heart failure in an in vivo model of transient coronary occlusion in mice. This line includes the use of transgenic animals, deficient for Cx43. Techniques such as Western Blot, immunofluorescence, or confocal microscopy will be used.
Good knowledge of english. High degree qualifications will be a value.
Colorectal cancer (CRC) is a leading cause of death worldwide, mostly because acquisition of resistance to current treatments. Our team has discovered that hyperactivation of the Wnt oncogenic pathway is involved in such resistance. Hypermutant CRC tumors affect 15-20% of all CRC patients and are characterized by presenting mutations in key components of the DNA mismatch repair (MMR) machinery, resulting in amplifications in repetitive sequences in the genome known as microsatellites (MS).
The hosting group recently described that frameshift mutations in upstream components of the Wnt signaling, RNF43 or ZNRF3 (R/Z), that confer Wnt addiction, are present in 70% of MMR-deficient CRC tumors, and these mutations preferentially occur in a codifying MS. Recently, pharmaceutical companies have developed inhibitors that revert Wnt addiction and have shown clinical potential in tumors with mutations in R/Z.
In this project we propose to identify molecular alterations driving the response to Wnt inhibitors and gene expression signatures to predict drug response. We also aim to evaluate the efficacy of Wnt inhibitors on hypermutant human organoids as well as in transgenic mouse models with sporadic MMR deficient intestinal tumors. Finally, we propose to correlate immune infiltration with Wnt signaling activation in human CRC tumors and determine the synergistic effect of Wnt inhibitors and checkpoint inhibitors in mouse models. By achieving our main expectations, we believe that not patients with hypermutant CRC tumors but also those with any other cancer type presenting Wnt activation could benefit from these combined treatments. Our collaboration with the Oncology Service and major pharmaceutical companies will accelerate the translation of our findings into the clinical practice.
Hemophagocytic lymphohistiocytosis (HLH) is a life-threatening syndrome characterized by persistent macrophages/lymphocytes activation and a multisystem hyperinflammatory syndrome. HLH can be classified into familial or primary (FHL) and acquired or secondary. FHL is caused by homozygous mutations in genes encoding proteins of the cytotoxic pathway. Patients developing a clinical picture of HLH but carrying a single mutated allele, hypomorphic variants or defects in two different HLH-related genes have been recently reported. Monoallelic mutations in HLH-related genes in patients with HLH is a challenge in the diagnosis, and our objective is study the functional and molecular impact of monoallelic mutations involved in Hemophagocytic lymphohistiocytosis in order to determine whether they act as dominant negatives.
Study of the role of specific immune cells (mast cells, eosinophils and B cells) as major players in the control of mucosal barrier mechanisms in health and disease. Our approach to translational research in digestive diseases involves the development of preclinical and clinical studies aimed at identifying biomarkers of gastrointestinal dysfunction and gut health.
We are a young, newly formed research group at the Vall Hebron Research Institute (VHIR), within the group of Biomedical Research in Gynecology. The primary interest of the group is to understand the mechanisms that give rise to tumorigenesis, with the main focus in ovarian cancer. From a broad perspective, we study mechanisms contributing to aneuploidy with the aim of being able to translate the potential knowledge that we will acquire into the clinics. We are interested in the role of key mitotic kinases and other cell cycle regulators focusing on cancer progression and resistance to chemotherapy, but making it also extensible to other topics such as the role of miRNAs in ovarian cancer progression or genes involved in tumor dissemination. We also cover a transversal line studying the role of mitotic kinases and downstream effectors in Prostate cancer.
We seek for a motivated student. The ideal candidate should be an organized person, happy to work as part of a team and with good communication skills and a positive attitude. Fluid English and previous experience will be advantageous.
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Double Amputee Veitnam Veteran Becomes Sponsored Golfer
Post by: amputee, disables, golf, golfer, handicap, Kyra Phillips, mike reeder, st. andrews, tennessee, veteran
Filed under: Anchors • Kyra Phillips • Military Matters
Real Help For Real Warriors
Today in the CNN NewsRoom we're talking with Staff Sgt. Megan Krause her about her battle with PTSD.
She's featured in a military campaign to encourage service members who need help to get it.
To see Sgt. Krause's PSA or to find out where military members can go for help click here.
Filed under: Kyra Phillips • Military Matters
Buckets of Gratitude for U.S. Vets
[cnn-photo-caption image=http://i2.cdn.turner.com/cnn/2010/images/01/08/hfh.logo.png caption=""]A New Hampshire man's stepped up to the charity stripe, to raise money for our wounded troops.
David Cummings hopes to sink 1,000,000 free throws by Veterans Day 2011. Almost 82,000 in (as of this writing) - he's netted nearly $3,000 in donations.
If you'd like to notch an assist, you can make a pledge on Cummings' website.
Army Veteran Sues McDonald's
Filed under: Clips from CNN Newsroom • Kyra Phillips • Military Matters
We'd planned to tell you about these troops in our second hour Friday; unfortunately, the rundown kept changing, and we weren't able to air the Fallen Heroes segment.
Here's the original write-up:
These four soldiers from the Fourth Engineer Battalion trained together... deployed together... and died together.
On October 15, an IED blew up their vehicle in Kandahar Province, Afghanistan.
Spc. Jesús Flores Jr. joined the Army in December 2003. His family had moved to the U.S. from the Philippines when he was young. Friends there - and here - knew him as "Jun Jun" and "Lil' Flo".
Spc. Daniel Lawson joined the army in 2008, and shipped out about a year later. He had a degree in computer programming, but wanted to provide more for his family: wife Latoria, and their three kids - ages 10, eight, and five.
Pfc. Brandon Styer was just 19. He'd joined up his senior year of high school, where he was a student-athlete - and a big-time Phillies and Redskins fan. Private Styer's other passion: customizing his prized Mazda MX8.
At 34, Staff Sgt. Glen Stivison Jr. was a paternal figure to a lot of his troops, who called him "Stivey" or "Papa Sti". He loved pranks, karaoke, and fishing. But most of all, he loved his wife Eryn, and their boys - ages six and eight.
We honor the sacrifices of these troops - and the nearly 5,300 other Americans lost in Operation Enduring Freedom and Operation Iraqi Freedom.
[cnn-photo-caption image=http://i2.cdn.turner.com/cnn/2009/images/11/30/lawson.daniel.jpg caption="Spc. Daniel Lawson"]
[cnn-photo-caption image=http://i2.cdn.turner.com/cnn/2009/images/11/30/flores.jesus.jpg caption="Spc. Jesús Flores Jr."]
[cnn-photo-caption image=http://i2.cdn.turner.com/cnn/2009/images/11/30/stivison.glen.jpg caption="Staff Sgt. Glen Stivison Jr."]
[cnn-photo-caption image=http://i2.cdn.turner.com/cnn/2009/images/11/30/styer.brandon.jpg caption="Pfc. Brandon Styer"]
A Four-Star Friendship
It sounds too far-fetched for even a war movie:
Top general visits wounded soldier in the hospital.
Soldier snaps out of a coma… at the general's command.
But in fact, that's pretty much what happened last year.
The place: Walter Reed Army Medical Center.
The protagonists: General David Petraeus, and 1st Lieutenant Brian Brennan.
Today - Veteran's Day - both gentlemen joined Kyra live, to talk about it.
She also got a chance to ask the general about his White House 'war cabinet' meeting, scheduled for later that same hour.
A flight of honor for our "Greatest Generation"
It's 0600 hours in Fayetteville, Georgia and I'm up with troops preparing for a very special mission. I start making rounds, talking to many of the nearly 80 World War II Veterans about to take a trip of a lifetime.
They are heading to Washington DC to see the memorial built in their honor. For many of these veterans, it will be the first time they've ever seen it. The trip was provided by Honor Flight…an organization with one mission: to honor veterans for their sacrifice by sending them to their memorial in Washington DC for free!
As I speak to the vets, their excitement is easy to see. Many tell me this was a trip they never thought they'd be able to make. They are happy to talk about their appreciation towards Honor Flight, but when it comes to the war… most don't tell me much.
Soon, we loaded the buses and headed towards the airport with an escort by local law enforcement and patriot guard riders. On the plane, some veterans start opening up to me and photojournalist Rich Brooks. But still, the war stories were few and far between.
Two hour later we arrived in Washington, DC and the sight brought tears to my eyes. A heroes welcome in the terminal. A brass band played old war tunes. A massive crowd waved flags, cheered and thanked each and every veteran for their service as they stepped off the plane. Watching their expressions, their reaction to this was truly one of the best things I've experienced. I stood at the end of the reception line. So many stopped and told me this was more elaborate then the reception they received when they returned home from war and one of the first times they've ever been thanked for their service.
At their memorial, even more thanks. Most gasped to see how big it was. They slowly made their way down into the memorial. Facing the reflection pool and Lincoln memorial is a wall of stars. Each represents 100 service members who lost their life in WWII. There are 4,000 stars on that wall! I felt a tap on my arm and saw one of the veterans standing beside me. He told me about his experience storming the beaches of the Pacific, the men they lost and how lucky he was to be witnessing this moment when so many of his comrades didn't make it. He told me that was the first time he'd ever told anyone his story.
It was amazing! The same veterans who didn't want to speak about the war just hours before were now willingly sharing their stories. Luckily, Rich was around to record a lot of it on tape! The stories were unbelievable. Stories of the D-Day invasion, pilots being shot down by enemy fire and chilling tales from inside POW camps. I felt so privileged to be there at that moment.
From the memorial we took a quick trip to Arlington National Cemetery to see the changing of the guard at the Tomb of the Unknowns.
By the time we got back to Atlanta it had been a 20 hour trip! The elderly warriors seem renewed rather than exhausted by their whirlwind trip.
Peharps because this was meant to honor these WWII Veterans who never asked for thanks. They went off to save the world and when they got home, continued on with their lives. They never asked for honors. The real honor, for me, was to spend the day with this great generation of heroes. It is a trip I am grateful to have the opportunity to take and one that I will never forget!
Today, if you see a veteran of any war, take the time to thank them! I can tell you from experience, that small thanks means more to them then you'll ever know!
Click here to read more on this trip.
Or if you want to learn more about Honor Flight and their mission, click here.
Filed under: Anchors • Heidi Collins • Impact Your World • Josh Levs • Kyra Phillips • Military Matters • Tony Harris
If you're a regular visitor to the Team Kyra blog, you probably know she's a big supporter of TAPS - a group that helps families of fallen U.S. troops.
Well, a little while back, Kyra heard about a song called "Soldier On"; the people behind it are donating half the song's proceeds to TAPS.
You can find more info on "Soldier On" right here.
Filed under: Kyra Phillips • Military Matters • On TV
AWOL Soldier Points to PTSD
We closed out our first hour today with a segment on mental health issues and the military.
First up: Gary Tuchman's piece on an AWOL soldier - who couldn't take Army life, but couldn't take life on the lam either. This soldier says he had to leave, for the safety of his fellow troops. That he wasn't getting enough help for his PTSD, and felt like a ticking time-bomb.
When he turned himself in yesterday to face a desertion charge, Gary was right there.
Pentagon officials wouldn't go into the specifics of Spc. Gartin's case - but they say there's plenty of mental health treatment available to troops.
We discussed all of this with Tom Tarantino, from Iraq and Afghanistan Veterans of America.

redpajama_set_name: RedPajamaCommonCrawl


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Синьтя́нь () — уезд городского округа Юнчжоу провинции Хунань (КНР).

История
Во времена империи Тан в 767 году был создан уезд Дали (大历县), в территорию которого входила в том числе и значительная часть территории современного уезда Синьтянь, но в 965 году он был присоединён к уезду Нинъюань.

Во времена империи Мин в 1639 году на стыке уездов Нинъюань и Гуйян был создан новый уезд, получивший название по находящемуся на его территории Синьтяньскому укреплению (新田堡).

После образования КНР в 1949 году был создан Специальный район Юнчжоу (永州专区), и уезд Синьтянь вошёл в его состав. В мае 1950 года Специальный район Юнчжоу был переименован в Специальный район Линлин (零陵专区).

В октябре 1952 года Специальный район Линлин был расформирован, и уезд перешёл в состав новой структуры —Сяннаньского административного района (湘南行政区). В 1954 году Сяннаньский административный район был упразднён, и уезд вошёл в состав Специального района Чэньсянь (郴县专区).

22 марта 1959 года уезд Синьтянь был присоединён к уезду Гуйян.

29 августа 1960 года Специальный район Чэньсянь был опять переименован в Специальный район Чэньчжоу.

9 июля 1961 года уезд Синьтянь был вновь выделен из уезда Гуйян.

30 октября 1962 года уезд Синьтянь был передан в состав Специального района Линлин (零陵专区).

В 1968 году Специальный район Линлин был переименован в Округ Линлин (零陵地区).

Постановлением Госсовета КНР от 21 ноября 1995 года округ Линлин был преобразован в городской округ Юнчжоу.

Административное деление
Уезд делится на 11 посёлков и 1 национальную волость.

Ссылки

Уезды Юнчжоу

redpajama_set_name: RedPajamaWikipedia


The Pump Advanced Training Course covers theory and design for non-standard pumps: viscosity, impurities and erosion, motor selection, variable speed and its interaction with pumps and systems. The Pump Advanced Course also includes a plant tour of the manufacturing facility in Montecchio, Italy or Strzelin, Poland. Each attendee will learn how to select and size the proper pump for the application. In addition, attendees will receive valuable experience in selecting, sizing and installing all the pumps available in the Xylem Portfolio.
***Prerequisite: Minimum three years' experience with pumps and drives. Pre-approval is required.
The Pump Advanced session covers all special working conditions and installation of clean water pumps. Attendees receive information on how to select and size the right system in different situations, like heating systems (how the NPSHR changes, which material is more suitable, etc), multipump operations, the different behaviors of viscous liquid, and how to install a pump or system properly in particular conditions. Examples, exercises and practical tips will be delivered.
A complete overview of the Xylem portfolio for the region, including pumps, controls, Variable Speed Drives (VSD) and booster sets.
A session dedicated to water supply system design for building. Attendees will learn how to calculate the flow and head of the system, together with standard design methods. Additionally how to select and size the proper booster sets, how to choose the right VSD system and how to prevent problems and issues during installation will be covered. A hands-on session is included in the lesson.
A lesson totally dedicated to heating and cooling systems and pumps. During the morning session the different approaches to system design and how to calculate and size the proper system will be covered – starting from basic up to the latest system design. In the afternoon attendees will learn how to select and size the right pump for the heating system together with suitable electronic systems and equipment in the Xylem portfolio. Finally attendees will have the opportunity to experiment with a working unit in order to understand the different possible situations they may find in the market.
The last day of the training is dedicated to a site visit, where the attendees will have the chance to see a real working system, depending on site availability and time of year.


Member for Mulgoa, Tanya Davies MP today announced local road safety in Mulgoa could receive a welcome boost with applications to open shortly for the third round of the NSW Government's Community Road Safety Grants program.
"The Community Road Safety Grants program supports community organisations to work with their local communities to improve road safety in their areas," Tanya Davies said.
"I encourage all community groups to apply for these grants. Local knowledge is vitally important in identifying and addressing local road safety issues.
"We all have a role to play in making our roads safer. Last year 384 people lost their lives on our roads and every year more than 12,000 people are seriously injured.
"These are not just numbers, they are real people lost to their families, friends and communities forever.
The Government will award up to $1.6 million in grants over four years through the program using a competitive process.
Applications open for this latest round of grants on Tuesday 13 June and community groups are invited to submit their local road safety projects by 5pm on Sunday 23 July.
Two categories of grants are available: one for smaller projects with up to $5,000 available for each idea and another for more substantial projects, worth up to $30,000.
Community-based organisations can get more information about the grants, and submit their application, by visiting: http://roadsafety.transport.nsw.gov.au/aboutthecentre/communitygrants/.


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We stayed at a great place called Sun Club. We met so many nice people from all over the world. We went on the cruise "Bora Bora". We went snorkeling, and hiked to a waterfall. On a different day, we went Marlin fishing, but only caught a Yellow Tail. The restaurant at the resort prepared our catch, and we ate it for dinner. We LOVE our timeshare!
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Thank you for a great vacation. This was our first RCI exchange, and a dream come true. A blue week in the Bahamas, such a deal. Had a great time night shark fishing.


Or the hamster cage smells, it is the very best resolution to absorb the smell produced carbon supply pet outlets often is the time of purchase. Guinea pigs do not likely get so much for its size as 18-inch partitions shouldhigh enough to keep them protected in a cage.
If you want to find this animal hamster adventure dome may normally be found in pet Zhu Zhu. Usually discover a much wider vary of online and cages in the space of wildlife (especially when you reside in a small town). Choosing the proper bedding in your pet gerbil, he or she will dwell an extended life and happier.


text:
De Granaattoren is een van de twee zevenhoekige torens behorende bij het Kasteel van Breda. Deze torens, de Duiventoren en de Granaattoren, liggen aan het Spanjaardsgat, dicht bij de haven in het centrum van Breda en zijn de enige resten van de versterkingen die in 1530 door Hendrik III van Nassau aan de reeds bestaande vestingwerken werden toegevoegd.

De torens hebben hun militaire functie al lang verloren. In 1903 - 1910 zijn ze gerestaureerd, waarbij ook het bolletje op de torens is geplaatst.

Het Kasteel van Breda is sinds 1826 in gebruik door de Koninklijke Militaire Academie (KMA). In de Granaattoren is op de eerste verdieping een bar voor het personeel van de KMA gehuisvest. Op de begane grond is een rooms-katholieke kapel gevestigd.

Externe link
 Granaattoren

Kasteel van Breda
Toren in Noord-Brabant


Updated 4/4/17 3:32 PM: Six more companies have pulled advertising from The O'Reilly Factor. See full list at the end of this article.
Updated 4/4/17, 12:20 PM: BMW became the third company to pull advertising from The O'Reilly Factor.
Is the ice beginning to crack beneath Bill O'Reilly? Mercedes-Benz has "reassigned" its advertising away from Fox News' ratings juggernaut The O'Reilly Factor after the New York Times published details over the weekend of five settlements that O'Reilly or Fox News has made following allegations of sexual harassment or other inappropriate behavior by the host.
These settlements have cost a total of $13 million. O'Reilly has denied all allegations.
"The allegations are disturbing and, given the importance of women in every aspect of our business, we don't feel this is a good environment in which to advertise our products right now," a spokesperson for Mercedes-Benz told Forbes.
Till now, Fox has stood steadfast by O'Reilly given his record as the ringmaster of cable's most-watched news program for the past 68 quarters. In the first quarter of 2017, it enjoyed the highest-rated quarter for any program in cable news history, with about 4 million viewers each night.
These ratings have meant big money for the network. Last year, The O'Reilly Factor garnered an estimated $110.8 million in ad revenue, according to iSpot.tv. That 's over five times the $20.7 million in advertising brought in last year by MSNBC's biggest star, Rachel Maddow, whose show airs an hour later. Fox News makes up about 10% of its parent company 21st Century Fox's revenue and about 25% of its operating income.
Mercedes-Benz purchased an estimated $266,477 worth of spots on The O'Reilly Factor over the past 30 days, making it the shows tenth biggest advertiser, and the company spent $1.3 million in 2016 for commercials during the show, according to iSpot.tv. Hyundai bought $102,902 worth of commercials over the last 30 days and $913,445 worth in 2016.
The question now is whether more advertisers besides Mercedes will pull their ads to avoid being associated with the contentious host.
"I expect some advertisers will make adjustments to their buys and others won't," says media buyer Louis Roloff. "These are serious accusations, and I've seen C-suites stop and drop everything to decide whether to keep ads on air."
If more advertisers follow, it could mean the end for the tolerance that Fox News has shown O'Reilly, despite a string of legal settlements and controversies. The New York Times revealed on Saturday that O'Reilly has been involved in at least five cases in which he or the network settled with women claiming sexual harassment or other inappropriate behavior, three more than had previously been reported. In 2004, O'Reilly paid his former producer Andrea Mackris an estimated $9 million to settle a sexual harassment suit. In return, she reportedly had to keep quiet about her claims, including phone conversations in which O'Reilly allegedly told her about his sexual fantasies about her.
He's been accused of racist remarks (just last week he mocked Rep. Maxine Waters' hair, calling it a "James Brown wig"), and of falsely claiming he reported on conflicts in El Salvador and Northern Ireland.


text:
Az amuri füzike (Phylloscopus tenellipes) a verébalakúak (Passeriformes) rendjébe, ezen belül a füzikefélék (Phylloscopidae) családjába és a Phylloscopus nembe tartozó faj. 10-11 centiméter hosszú. Költési területe északkelet-Kína, Észak-Korea és délkelet-Oroszország erdős vidékei, telelni Délkelet-Ázsiába vonul. Többnyire rovarokkal táplálkozik. Májustól júliusig költ.

Fordítás

Források
 Phylloscopus tenellipes - hbw.com
 Phylloscopus tenellipes - birdlife.org
 Phylloscopus tenellipes - canto.org

Madárfajok
Füzikefélék
Phylloscopus
Ázsia madarai
Észak-Korea madarai
Kambodzsa madarai
Kína madarai
Laosz madarai
Malajzia madarai
Mianmar madarai
Oroszország madarai
Vietnám madarai
Thaiföld madarai


text:
Is It Harder to Drive a Pickup Truck?
Pickup trucks have become increasingly popular in recent years and have developed a reputation for being powerful, reliable, and convenient. But is it harder to drive a pickup truck than other vehicles?
The answer is yes and no. While they may be more difficult to maneuver due to their size and weight, driving a pickup truck can be easier in certain situations.
Pickup trucks are designed for hauling large loads and as such, they are often more powerful than other vehicles. This means that they can accelerate faster and are less likely to become bogged down when driving on rough terrain.
Additionally, many pickups feature four-wheel drive which provides better traction on slippery roads. They also offer increased visibility from the elevated cabin which can make it easier to spot potential hazards.
On the other hand, pickup trucks are much larger than other vehicles which can make them harder to maneuver in tight spaces. They require more space to turn and have longer stopping distances due to their weight. Pickup trucks also tend to be noisier than other vehicles due to their large engines so they may not be suitable for quiet neighborhoods or streets.
Overall, driving a pickup truck is not necessarily harder than other vehicles but there are certain drawbacks that should be considered before making the switch. They offer advantages such as power, traction, and visibility but require more space for maneuvering in tight areas. Ultimately, the decision of whether or not a pickup truck is right for you will depend on your individual needs and preferences.
Is It Hard to Drive a Pickup Truck?
Pickup trucks are a popular vehicle choice for those looking for a reliable, dependable ride, but they can be tricky to drive. Drivers need to weigh the pros and cons before deciding if a pickup truck is the right vehicle for them. For starters, pickup trucks are more difficult to maneuver than smaller cars and SUVs because of their size and weight.
Is It Hard to Drive Pickup Truck?
Pickup trucks are one of the most desirable vehicles on the market, offering a combination of power, utility, and style. While some people might be intimidated by the size and power of a pickup truck, driving one is actually quite easy. Pickup trucks can be more powerful than regular cars due to their larger engines and superior towing capabilities.
Is It Difficult to Drive a Pickup Truck?
Pickup trucks are becoming increasingly popular vehicles for people who need a reliable and spacious vehicle. But, it can be a bit tricky to drive a pickup truck, especially if you're not used to them. Here are some tips to help make driving a pickup truck smoother and easier.
Is It Hard to Drive a Big Pickup Truck?
Driving a big pickup truck can be intimidating for some people. The size of the vehicle can make it difficult to maneuver and park in smaller spaces, and the increased weight can take some getting used to. However, driving a big pickup truck can be easier than people expect once the basics are understood.
Is It Hard to Get Used to Driving a Pickup Truck?
Driving a pickup truck can be intimidating at first, but with a bit of practice it can become second nature. The size of the truck and its higher ground clearance can be a challenge to maneuver, particularly if you're used to driving smaller cars. But with patience and some basic tips and tricks, you can learn how to handle a pickup truck like a pro.
How Hard Is It to Drive a Pickup Truck?
Driving a pickup truck can be surprisingly challenging for those who are accustomed to driving a regular car. It usually takes some practice to get used to the size and weight of a pickup truck, as well as its added capabilities. Due to the extra power and torque it commands, it is important for drivers to understand how the vehicle will respond when accelerating, braking, and cornering.
Is Driving a Pickup Truck Difficult?
Driving a pickup truck is no harder than driving any other type of vehicle, but there are some tips that can be helpful when driving a pickup truck. For starters, it's important to remember that pickups are larger than many other vehicles on the road, so they may require more space to maneuver. It's also important to check your blind spots often as pickups have larger blind spots than other vehicles.
Is a Pickup Truck Difficult to Drive?
A pickup truck is a vehicle that is both versatile and reliable. It can be used for work, play and anything in between. But when it comes to driving one, many drivers may feel intimidated due to its size and power.
How Hard Is Driving a Pickup Truck?
Driving a pickup truck can be an intimidating experience, especially for those who are new to the idea of driving a large, powerful vehicle. Pickup trucks are significantly larger than other types of vehicles and require more skill to drive safely. They also have a higher center of gravity which can make them more difficult to maneuver in tight spaces and on curvy roads.
How Difficult Is It to Drive a Pickup Truck?
Driving a pickup truck is often seen as an intimidating prospect. While it may not be as difficult as some people think, it does require skill and attention to detail. For those new to driving pickups, the size and weight of the vehicle can be daunting.


text:
Q: Get keyboard code of a keypress in Python I'm trying to get the keyboard code of a character pressed in python.  For this, I need to see if a keypad number is pressed.
This is not what I'm looking for:
import tty, sys

tty.setcbreak(sys.stdin)

def main():
    tty.setcbreak(sys.stdin)
    while True:
        c = ord(sys.stdin.read(1))
        if c == ord('q'):
            break
    if c:
        print c

which outputs the ascii code of the character.  this means, i get the same ord for a keypad 1 as as a normal 1.
I've also tried a similar setup using the curses library and raw, with the same results.
I'm trying to get the raw keyboard code.  How does one do this?

A: As synthesizerpatel said, I need to go to a lower level.
Using pyusb:
import usb.core, usb.util, usb.control

dev = usb.core.find(idVendor=0x045e, idProduct=0x0780)

try:
    if dev is None:
        raise ValueError('device not found')

    cfg = dev.get_active_configuration()

    interface_number = cfg[(0,0)].bInterfaceNumber
    intf = usb.util.find_descriptor(
        cfg, bInterfaceNumber=interface_number)

    dev.is_kernel_driver_active(intf):
        dev.detach_kernel_driver(intf)

    ep = usb.util.find_descriptor(
        intf,
        custom_match=lambda e: usb.util.endpoint_direction(e.bEndpointAddress) == usb.util.ENDPOINT_IN)

    while True:
        try:
            # lsusb -v : find wMaxPacketSize (8 in my case)
            a = ep.read(8, timeout=2000)
        except usb.core.USBError:
            pass
        print a
except:
    raise

This gives you an output: array('B', [0, 0, 0, 0, 0, 0, 0, 0])
array pos:
    0: AND of modifier keys (1 - control, 2 - shift, 4 - meta, 8 - super)
    1: No idea
    2 -7: key code of keys pushed.
so:
[3, 0 , 89, 90, 91, 92, 93, 94]

is:
ctrl+shift+numpad1+numpad2+numpad3+numpad4+numpad5+numpad6

If anyone knows what the second index stores, that would be awesome.

A: To get raw keyboard input from Python you need to snoop at a lower level than reading stdin.
For OSX check this answer:
OS X - Python Keylogger - letters in double
For Windows, this might work:
http://www.daniweb.com/software-development/python/threads/229564/python-keylogger
monitor keyboard events with python in windows 7

A: if you have opencv check this simple code:
import cv2, numpy as np
img = np.ones((100,100))*100
while True:
    cv2.imshow('tracking',img)
    keyboard = cv2.waitKey(1)   & 0xFF
    if keyboard !=255:
        print keyboard
    if keyboard==27:
        break
cv2.destroyAllWindows()

now when the blank window opens press any keyboard key. ESC breaks the loop

redpajama_set_name: RedPajamaStackExchange


text:
Q: Meu logo em bootstrap está demasiado grande e os restantes menus têm um grande espaço vazio
Alguém consegue ajudar me a concertar isto? Já utilizei o CSS, mas mesmo assim não consigo fazer com que os menus fiquem "colados" à imagem.

O meu HTML é o seguinte, e embaixo estará também o css

A: Precisamos do seu código para ter certeza. Acredito que nos seus links você está chamando a classe

nav navbar-nav navbar-right

Basta retirar a classe navbar-right e o conteúdo virá para o lado da logo de imediato.


Originally the end of the Celtic year, Halloween now celebrates Eve of All Saint's Day in most countries. The Day of the Dead has been celebrated in Mexico as a 100% indigenous observance, as an Aztec holiday. However, the Aztecs celebrated their tribute to the dead in August. When Mexico begin embracing holidays from other countries, like Valentines Day, which is called the Day of Love and Friendship in Mexico, they decided to move the traditional August holiday to October 31, to coincide with Halloween. But in Cuernavaca, as in all of Mexico, Halloween is celebrated over several days.
For those who have not been in Cuernavaca during Halloween, you are in for a surprise! While Day of the Dead has been celebrated in Mexico for decades, the wearing of costumes and especially the Trick or Treat part of the holiday only started in the late 1960s and is still not as widely accepted as in other countries. Today, the celebration mimics that other countries, such as the wearing of costumes by both children and adults. Don't be surprised if you go to one of your favorite restaurants to find the wait staff dressed in gore.
October 27 brings the start of Feast of the Holy Souls (Fiesta de las Santas Animas), when people will visit the graves of relatives where they clean the graves and adorn them with pine needles and pungent marigold (cempasuchil) flowers, which have long been associated with death, assemble a temporary alter (ofrendas) and place candles to guide the souls. They will bring the favorite food of past relatives and friends, which yes, sometimes means alcohol, depending on the choices of the departed. There will be photos and other memorabilia.
In some locations, celebrants wear shells on their clothing, so that when they dance, the noise will wake up the dead; some will also dress up as the deceased.
Along with the offerings there will be a picnic, which may last all day and into the night. It is a day of remembrance, happiness and celebration of past lives. In fact, the holiday may last as long as it takes to visit all of the graves of the dearly departed.
On October 31, Halloween is celebrated in Mexico and it is gaining in popularity. However, in the beginning most Mexicans were hesitant to embrace. At the time, most Halloween costumes and candy came from the U.S. and Mexicans saw it as a plot by U.S. businesses as a way make money. Some detractors claimed that Halloween and the Day of the Dead have nothing to do with one another.
Decades ago, when students attempted to hold parties at Halloween at the public schools, The SEP (the Mexican Education Department) banned them in attempt to carry on the tradition of the Day of the Dead.
However, the sales have shifted to the Mexican businesses that make a considerable amount of money from the celebration. Today, Mexican businesses manufacturer and sell costumes, candy, makeup and more. It has officially became a Mexican festival.
However, Mexico celebrates Halloween with a slightly different twist to Halloween in other countries. In Mexico, for a few days before and after this date, children will visit neighboring homes in search of treats, but with a difference in that the well behaved children of Cuernavaca do not play tricks on those who do not provide them with candy or other treats. When the reach your door, they will ring the bell and shout "¡Me da mi calaverita!", which translates to "Give me my candy skull" Calaveritas, are little sugar candy skulls that are given out on this holiday.
The calavera skull candy comes from a humorously morbid poem which is traditionally address to a friend or foe. The poetry has it's origin in Cervantes' Don Quixote, in early 17th century Spain.
You will find organized Halloween themed parties, especially with the teenagers, but adults are not beyond throwing their own parties, which do not necessarily fall on the day of Halloween. So you may see a devil driving past you one night.
November 1 is All Saints' Day in Cuernavaca, coincides with the first day of the Day of the Dead (Dia de los Muertos) celebration. It is also known as Day of the Innocents (Día de los Inocentes), as it honors deceased children and infants (angelitos), who were less than 18 years of age and unmarried, with candles, food and flower offerings.
In Mexico All Saints' Day is devoted to Los Angelitos – that is, all the dead children, and is a prelude to November 2's Dia de los Muertos, Day of the Dead, a national holiday on which all the grown-up ghosts will be arriving in full force. The smaller ghosts get a head start.
To help the departed find their way back to the homes where they once lived, parents and still-living family members often shoot off firecrackers. The children may run through the streets with lanterns and ask for coins. People light bonfires, set off firecrackers, and hang lanterns on trees to guide the souls of the dead home. In some parts of the country on this night they strew a path of flower petals from the graveyard to the front porch.
November 2, brings All Souls Day (Día de los Fieles Difuntos), which honors dead relatives and/or friends (who were more than 18 years of age or married) with candles, food and flower offerings, altars, and pre-Hispanic and Christian rituals. It is not a state holiday.


text:
18 December Fifa World Cup Table Predictions
18 December 2022 Fifa World Cup Table
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text:
Q: Random commas appearing on html page on inspect when not in code I have a simple HTML page and I am mapping an array to populate a div with cards. The card alignment was all wrong, so when I inspected it, I saw that there are commas after every card except the last one, which is ruining the alignment. I haven't added these commas in the code. My code:
    itemsdiv.innerHTML = items.map(el => {
        return (`

            ${el.name}

                ${el.color}

        ` )
    })

itemsdiv is an empty div to populate, and items is an array that looks like this:
    items = [
        {
            name: 'T-Shirt',
            src: 'Images/Shirt.png',
            color: 'blue',
        },
        {
            name: 'Coffee Cup',
            src: 'Images/Coffee.jpg',
            color: 'red',
        }
    ]

When I inspect it looks something like this:

   ","

   ","

A: After .map try tagging on .join(" ")
Solution based on @VLAZ's comment:

When you convert an array to a string, it automatically joins the elements with commas between them. Assigning an array to innerHTML converts it to a string.


Few incidents are as scary or life-altering as a serious car accident. One moment you're fine, the next moment you're physically and emotionally battered. And while you won't soon forget about your accident, you can place emphasis on reaching a full recovery.
Every situation is unique, but the more you focus on recovery – and the less you dwell on the unfortunate circumstances that led to your accident – the faster you'll heal. Keeping this in mind, let's explore some of the top tips for reaching a full recovery.
When you've been injured in a car accident, you don't have the luxury of waiting around and hoping things will get better. The longer you let issues go without receiving medical care, the less likely it is that you'll reach a full recovery. Quickly seek out the best possible care, and everything else will fall into place.
There are many aspects of healing and recovery, but rest almost always plays a role in the process. If you push yourself too hard, you're going to make the situation worse. But that doesn't mean you have to lay in bed all day.
While you're focused on healing and recovering, insurance companies are busy trying to protect their bottom lines. You'll hear from them personally within just the first few days. Make sure you're prepared.
To make sure your best interests are protected, hire an attorney to help you navigate these conversations and proceedings. A good lawyer will significantly increase the amount of money you receive.
While you have to listen to your body during recovery, it's also necessary to heed your doctor's advice and recommendations. They know what's best and will help you maintain a sustainable pace of recovery.
One aspect of the recovery process that most people aren't prepared for is the emotional trauma of trying to get back in a vehicle for the first time. Many people experience symptoms of PTSD, which can last longer than physical injuries.
There are some coping strategies, so listen to yourself and implement the ones that help. You'll likely have to ease back into things and avoid triggering circumstances for a while (such as the accident scene). However, you'll find that even the emotional wounds heal with time.
Rushing your recovery is not the answer to reaching a full recovery. The key is to take things slow and listen to the advice your doctors, lawyers, and close friends give you. By taking it one step at a time, you can avoid allowing emotionally charged choices to compromise your situation and instead make pragmatic decisions that positively impact your future and well being.


Rare and hard-to-find vinyl record. We are sorry, but we have no track listings and/or artwork available for this particular item. Product may be marked with manufacturer's corner cuts or stickers, but is a factory-new (not used) item. Limited quantities available, no backorders.


As mentioned before in my previous post, I have decided to make another game for the s9 to side along with Flight S9 – a tower defense for the s9! The game is still very far from completion in fact, I'm far from it. However, I just wanted to give a little update on the Tower Defense game currently in the works.
There were a few things I had to note before I started making this game.
The s9 is not a super computer. The s9 can not handle the FPS our computers can handle when creating more than 5+ objects running around the screen. This was a serious problem for the game as I tried to add more and more towers and as the "mobs" began to trod on the route. As a result, towers may lose their rotating barrels and the number of towers and mobs may be limited for better performance.
The screen size limitation. Since the s9 has a relatively small form factor, it was difficult in designating a field and a description box for a game like tower defense which requires a relatively large space.
Nevertheless, the game is on the works despite these limitations. So far, it seems to be ok, but I will be assigning a few alpha testers in the near future to test out the gameplay.
oooo. ahhh. 😀 yum yum. i am definitely waiting regardless of those limitations.


The Service Director is responsible for closely managing the day to day operations of a select group of global clients.
This client base typically includes firms that are large multi-geography fund managers, global insurance companies, global banks and other like businesses. In this capacity the incumbent will closely monitor daily operational and system workflows to ensure proper service levels are maintained or exceeded.
They will work with the Relationship Executive and Business Executive to ensure deployment of new business opportunities and will look for ongoing ways to continuously improve the relationship. The service director is responsible to traverse a broad base of global service teams to ensure clients are served exceptionally.
They are expected to have a deep understanding of all products and services provided to a client. They participate in critical decisions about servicing, highlight opportunities for improvement and recommend enhancements to the operating model.
SD's must work through multiple client issues at varying levels of complexity. Examples of the type of issues they may manage include: billing inquiries, contract negotiations, and other documentation-related matters. They are fully engaged in all matters related to the client, from routine service issue escalation to new product roll-outs. In order to be effective in the execution of such a broad range of responsibilities, the SD must be integrated with all servicing teams providing products and services to the clients. In this manner, the SD monitor that all of the global service teams are performing their daily activities effectively and that we are meeting our service commitments.
Client and internal-facing role charged with ensuring overall service quality, client satisfaction, and a consistent client experience working hand in hand with the RE and global service delivery. Demonstrates a deep understanding of all products and services provided to a client.
Determines critical decisions about servicing, and assists in developing the strategic direction for the client relationship. Highlights opportunities for improvement and recommends enhancements to the operating model. Works with Service Executive on client issues of significant nature.
Works through multiple operational and administrative client issues at varying levels of complexity. Examples include billing inquiries, BAC documentation, assisting in contract negotiations, and other documentation-related matters.
Monitors Service Delivery performance to ensure we are meeting service commitments (SLAs, Dashboards, etc.) and ensures extensive oversight of legal/compliance and risk responsibilities are accounted for.
Partners with the sales organization at the onset of a potential or new relationship to understand the unique needs and requirements of the prospective client. Leads the client through the on-boarding process. Partners with the Implementation teams to develop a highly customized process designed to successfully launch a multiple product business relationship.
Maintains full understanding of client business profile, including monitoring changes in structure, personnel, products and services, etc. to ensure client is in compliance with acceptable risk profile and supports the RE and BE in designing and proposing solutions to facilitate new business development efforts.
Business Expertise - Must demonstrate a broad and deep knowledge of the banks products within Asset Servicing, as well as extensive industry/market knowledge. Should deeply understand the full cycle of global service delivery, and the value proposition and differentiation.
Problem Solving - Must have the ability to work in a highly matrixed global organization and drive service accountability. Needs to be able to effectively communicate client requirements and mobilize teams to resolve service/operational issues expeditiously.
Nature & Area of Impact - This position has a significant impact on the overall relationship with the complex, global clients. SDs are accountable for ensuring overall service quality, client satisfaction, and a consistent client experience working hand in hand with the RE and BE.
Interaction and Inter personnel skills - SDs, in collaboration with the REs need to establish and maintain highly effective partnerships with global service delivery constituents to influence and drive excellent performance.


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Бальдассаре Донато або Донаті () (між 1525 та 1530 — червень 1603) — італійський композитор пізнього Відродження, представник Венеціанської школи, кантор. Був капельмейстером головного храму Венеції Собору святого Марка. У кінці XVI ст. був однією з ключових фігур у розвитку італійської світської музики, особливо віланели.

Біографія
Про першу частину життя Бальдассаре Донато не збереглося певних відомостей; не знане місце його народження. Перша згадка його імені знаходиться у книгах Базиліки св. Марка 1550 року, де він був кантором, а пізніше, у 1562 році, — інструктором співу дитячого хору. Коли у 1565 році Джозеффо Царліно прийняв від Чіпріано де Роре посаду капельмейстера, Донато був розжалуваний до простого півчого. Певно, конфлікт між ними був зумовлений незгодами у капелі св. Марка і досяг кульмінації у публічній сварці у 1569 році під час проведення урочистостей свята євангеліста Марка, викликавши скандал серед народу.

У 1577 році Донато обійняв високу посаду в школі іншого важливого храму Венеції, Церкви св. Рокко, зі значною музичною традицією та дуже солідною музичною капелою.Однак у 1580 році він залишив це місце. У 1588 році, коли Царліно був ще живий, став помічником капельмейстера Собору Св. Марка (причини примирення невідомі) і у 1590 році заступив свого колишнього суперника на посаді капельмейстера Базиліки св. Марка (однієї з найвпливовіших музичних посад Італії).

Музика та впливи
Донато є представником новаторської течії у складі Венеціанської школи,, яка сама репрезентувала прогресивну течію порівняно з іншими музичними школами і зокрема з Римською. Окрім Донато, до цієї течії належать Джованні Кроче, Андреа та Джованні Ґабріелі; до консервативної — Джозеффо Царліно. Чіпріано де Роре та Клаудіо Меруло, які притримувались настанов франко-фламандської школи, що домінувала у європейській музиці до другої половини століття.

Духовна музика Донато є найконсервативнішою частиною його творчості; вона пов'язана з використанням поліфонії у стилі Палестріни, але з додаванням поліхоральних ефектів обох Ґабріелі. Незважаючи на свою очевидну зневагу до консерватизму Царліно, він увібрав частину його стилю та настанов, як це можна помітити з використання ним контрапункту та Дисонансів, принаймні коли (зумисно) писав у стилі франко-фламандської школи.

Певне, його найбільший внесок в історію музики полягає у розвитку віланели та легкої форми мадригалу в світській музиці. Деякі з цих творів були написані для танцю у народному стилі. Вони були схожі на французькі шансони, мали мелодії, що легко запам'ятовувалися, базувалися на перехресних ритмах, а не на поліфонії та хроматизмі, властивих мадригалу середини століття. Донато писав також мадригали традиційніші, а окрім того псалми, мотети та церемоніальну музику.

Бібліографія
  Стаття «Baldassare Donato», in The New Grove Dictionary of Music and Musicians, ed. Stanley Sadie. 20 vol. London, Macmillan Publishers Ltd., 1980. ISBN 1-56159-174-2
  Gustave Reese, Music in the Renaissance. New York, W.W. Norton & Co., 1954. ISBN 0-393-09530-4
  Eleanor Selfridge-Field, Venetian Instrumental Music, from Gabrieli to Vivaldi. New York, Dover Publications, 1994. ISBN 0-486-28151-5 (Notas: ця книга не має в собі нічого стосовного Донато, але детально розповідає про венеціанську школу та діяльність капели св. Марка.)

Посилання
 Baldassare Donato — Chi la gagliarda, donne, vo' imparare — Ensemble Daedalus, Roberto Festa — YouTube
 Ноти на Choral Public Domain Library (ChoralWiki)
 Історія Базиліки Сан Марко

Італійські композитори
Композитори венеціанської школи
Композитори епохи Відродження
Композитори XVI століття
Народились 1525


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Home XBox 360
A playable early Left 4 Dead prototype has appeared online
in XBox 360
An early playable prototype for Valve's zombie shooter Left 4 Dead has appeared online.
Zombie City, which is a Counter-Strike: Source mod, is now playable, with dozens of videos of the mod appearing online.
The prototype is very rudimentary, featuring no zombies, instead, players face off against AI terrorists that can only attack the player using melee attacks.
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The mod, which was made playable thanks to modder WolfCl0ck is the latest in a wider spread leak of Counter-Strike maps. As PC Gamer points out, Turtle Rock founder Michael Booth discussed the mod in the Left 4 Dead commentary track.
"While we were developing bots for Counter-Strike: Source we discovered that a few of us armed to the teeth with automatic weaponry against 30 knife-wielding enemy bots was a lot of fun. After shipping Counter-Strike: Source in late 2004 we started experimenting with new game prototypes. That basic kernel of 'small team of friends against hordes of clawing enemies' was something we kept coming back to and we soon realized that the 'co-op vs the horde game' had a ton of potential."
Released in 2008, the original Left 4 Dead was developed by Turtle Rock Studios, which was known as Valve South at the time. Release on the Xbox 360 and PC, the game saw players progress through campaigns in groups of four, taking down hundreds of zombies.
Following strong reviews, a sequel, Left 4 Dead 2 was released in 2009. The series has been largely dormant since then.
The team behind the game, Turtle Rock Studios, would go on to develop Evolve, a multiplayer game in which four players played as hunters against one human-controlled monster. The game was met with disappointing reviews and player reception.
Turtle Rock's latest game, Back for Blood, was released in 2021.
VGC's review of the game, which saw the studio return to its Left 4 Dead roots, read "It seems backhanded to say that a game is only worth playing if you can find people to distract you from the actual game, but if your group wants something mindless to wind down after an intense night on Warzone or Rainbow Six: Siege, Back 4 Blood is ideal."
A bunch of games will be taken down from the Xbox 360 Marketplace soon
Deals with Gold: Xbox Sonderangebote KW05/2023
Xbox Series X|S vs Xbox 360 Sales Comparison in Japan
GoldenEye 007 Dev Responds To Lack Of 60FPS And Online Multiplayer In Xbox Version
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UK fishing fleet may become 'unviable' without switch to net-zero, says report