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Damarwulan
This article is about the legend of Damarwulan. For the film, see Damarwulan (film).
Depiction of the hero Damarwulan in a wayang kulit puppet.
Damarwulan is a Javanese cultural hero who appears in a cycle of stories used in the performance of wayang klitik, as well as Langendriya (female dance-opera) and ketoprak (popular theater). These stories tell of the struggles between the Majapahit and Blambangan kingdoms, in which Damarwulan gains honor. The stories are especially popular in East Java.
3 Synopsis[2]
The Damarwulan legend is associated with the Majapahit court at the time of the queen Suhita, at which time there was a war with Blambangan. However, the names of the characters Damar Wulan ("radiance of the moon") and Menak Jingga ("red knight") suggest that it may incorporate elements of an older sun-moon myth. It is uncertain when the story was first recorded and by whom.[1]
Prabu Kenya, the maiden queen of Majapahit
Patih Logender, her prime minister
Layang Seta and Layang Kumitir, the prime minister's sons
Dewi Anjasmara, the prime minister's daughter
Damarwulan, the prime minister's nephew, raised away from court by his grandfather
Menak Jingga, the King of Blambangan, a vassal of Majapahit
Dewi Wahita and Dewi Puyengan, princesses held captive by Menak Jingga
Sabdapalon and Nayagenggong, Damarwulan's servants
Synopsis[2]
Depiction of the villain Menak Jingga in a wayang kulit puppet.
Damarwulan is a prince by birth, a nephew of the prime minister, Patih Logender, but was raised in the hermitage of his grandfather. Following his grandfather's advice, he goes to the Majapahit court seeking employment. His cousins, the prime minister's sons, mistreat him when he arrives. Patih Logender, not wanting him to compete with his own sons, assigns him as grass-cutter and stableboy. Though he is stripped of his fine garments, he still has his striking beauty. Rumors of this beauty eventually reach Anjasmara, the prime minister's daughter. She seeks him out secretly and they fall in love and are clandestinely married. One night, Anjasmara's brothers overhear voices in her chamber. They break in and try to kill Damarwulan, but he is able to overcome them. They flee to their father, who orders that Damarwulan be executed. Anjasmara pleas for her lover, and he doesn't execute Damarwulan, but imprisons the pair.
Meanwhile, Menak Jingga has wrote a letter to the queen asking for her hand. When the queen rejects him, he declares war on the Majapahit kingdom. He is successful in dispatching Majapahit's allies, and finally the kingdom is threatened by his forces directly. In distress, the queen announces that whoever kills Menak Jingga and brings her his head can have her hand. Worried when no saviors present themselves, she has a divine revelation that a young knight named Damarwulan can overcome him. She orders Patih Logender to release him from jail and send him forth on his mission.
Damarwulan, accompanied by his servants, makes his way to Blambangan. Arriving at night, he steals into the gardens and manages to overhear a conversation at the pavilion between two resentful captive princesses. Damarwulan enters the pavilion and confides in them, and, enraptured by his beauty, they become devoted to him. At this time, Menak Jingga decides to visit the princesses, and discovers Damarwulan. They fight, but Damarwulan is unable to hurt Menak Jingga, and, severely wounded, appears to die. Menak Jingga leaves, ordering his servants to guard the body. However, they fall asleep, and the two princesses carry him away, revive him, and explain to the secret of Menak Jingga's magic invulnerability, a club of yellow iron kept behind his headrest. If the king is hit on his left temple with this club, he will die. Risking their lives for the sake of their lover, the princesses manage to steal the club while the king is asleep. A second battle between Menak Jingga and Damarwulan follows, in which Damarwulan manages to behead the king. Successful, he returns to Majapahit, but the prime minister's sons ambush him outside the palace, killing him and presenting Menak Jingga's head to the queen. However, a hermit revives Damarwulan, and the queen learns what happened. In a final battle, Damarwulan defeats his cousins, is crowned King of Majapahit, and is permitted to retain Anjasmara as his other wife.
^ Holt, 276.
^ Holt, 276-277.
Claire Holt. Art in Indonesia: Continuities and Change. Ithaca: Cornell UP, 1967.
Yoe Djin Lim. The story of Damar-Wulan the most popular legend of Indonesia Semarang: Lim Yoe Siang. OCLC: 24973772
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Damarwulan — Damarwulan, Wayang Klitik aus Ostjava. Das Damarwulan ist eine rein indonesische Erzählung aus dem ostjavanischen Raum. Sie wird dort mit Wayang Klitik Figuren erzählt und gespielt. Es zählt also zu den Schattenspiel Geschichten. Die… … Deutsch Wikipedia
Wayang — is an Indonesian and Malay word for theatre. When the term is used to refer to kinds of puppet theater, sometimes the puppet itself is referred to as wayang. Bayang , the Javanese word for shadow or imagination, also connotes spirit. Performances … Wikipedia
Wajang — Wayang Kulit Figur (Bali, frühes 20. Jahrhundert) Als Wayang werden in Indonesien Figuren bezeichnet, mit denen religiöse, traditionelle Puppenspiele durchgeführt werden. Die Art der Figuren und der Darstellung hat sich seit dem Vordringen des… … Deutsch Wikipedia
Wayang Klitik — Wayang Kulit Figur (Bali, frühes 20. Jahrhundert) Als Wayang werden in Indonesien Figuren bezeichnet, mit denen religiöse, traditionelle Puppenspiele durchgeführt werden. Die Art der Figuren und der Darstellung hat sich seit dem Vordringen des… … Deutsch Wikipedia
Wayang kulit — Figur (Bali, frühes 20. Jahrhundert) Als Wayang werden in Indonesien Figuren bezeichnet, mit denen religiöse, traditionelle Puppenspiele durchgeführt werden. Die Art der Figuren und der Darstellung hat sich seit dem Vordringen des Hinduismus auf… … Deutsch Wikipedia
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Suhita — was a queen of the Majapahit Empire, ruling from 1429 to 1447. She was the daughter of Wikramawardhana, her predecessor, by a concubine who was the daughter of Wirabhumi, who was killed in the civil war with Wikramawardhana. She was succeeded by… … Wikipedia
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Banyuwangi — La ville de Banyuwangi est située dans la province de Java orientale, à l extrémité orientale de l île de Java en Indonésie. 8°11′60″S 114°20′60″E / … Wikipédia en Français


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1911 Encyclopædia Britannica/Quintus Smyrnaeus
From Wikisource
< 1911 Encyclopædia Britannica
Quintilian
1911 Encyclopædia Britannica, Volume 22
Quintus Smyrnaeus
Quipus
sister projects: Wikidata item.
See also Quintus Smyrnaeus on Wikipedia; and our 1911 Encyclopædia Britannica disclaimer.
21818601911 Encyclopædia Britannica, Volume 22 — Quintus Smyrnaeus
QUINTUS SMYRNAEUS, Greek epic poet, probably flourished in the latter part of the 4th century A.D. He is sometimes called Quintus Calaber, because the only MS. of his poem was discovered at Otranto in Calabria by Cardinal Bessarion in 140. According to his own account (xii. 310), he tried his hand at poetry in his early youth, while tending sheep at Smyrna. His epic in fourteen books, known as Ta µe6' "Oµrjpov or Posthomerica, takes up the tale of Troy at the point where Homer's Iliad breaks off (the death of Hector), and carries it down to the capture of the city by the Greeks. The first five books, which cover the same ground as the Aethiopis of Arctinus of Miletus, describe the doughty deeds and deaths of Penthesileia the Amazon, of Memnon, son of the Morning, and of Achilles; the funeral games in honour of Achilles, the contest for the arms of Achilles and the death of Ajax. The remaining books relate the exploits of Neoptolemus, Eurypylus and Deiphobus, the deaths of Paris and Oenone, the capture of Troy by means of the wooden horse, the sacrifice of Polyxena at the grave of Achilles, the departure of the Greeks, and their dispersal by the storm. The poet has no originality; in conception and style his work is closely modelled on Homer. His materials are borrowed from the cyclic poems from which Virgil (with whose works he was probably acquainted) also drew, in particular the Aethiopis of Arctinus and the Little Iliad of Lesches.
Editio princeps by Aldus Manutius (1504); Kochly (ed. major with elaborate prolegomena, 1850; ed. minor, 1853); Z. Zimmermann (author of other valuable articles on the poet), (1891); see also Kehinptzov, De Quinti Smyrnaei Fontibus ac Mythopoiia (1889); C. A. Sainte-Beuve, Etude sur . . . Quinte de Smyrne (1857) ; F. A. Paley, Quintus Smyrnaeus and the " Homer " of the tragic Poets (1879) ; G. W. Paschal, A Study of Quintus Smyrnaeus (Chicago, 1904).
Retrieved from "https://en.wikisource.org/w/index.php?title=1911_Encyclopædia_Britannica/Quintus_Smyrnaeus&oldid=6553638"
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STATEN ISLAND, N.Y. -- At least five people were injured in a residential fire in Port Richmond on Thursday morning.
A civilian was critically injured and four firefighters suffered minor injuries in the blaze that was called at 5:49 a.m. for a multi-family dwelling at 170 Hatfield Place, according to a spokeswoman for the FDNY and the FDNY Twitter feed.
All of the injured were taken to Richmond University Medical Center in West Brighton, according to the spokeswoman.
A person who appeared to be severely injured was spotted being treated at the scene.
At least 60 firefighters and EMS personnel and 12 units brought the "all-hands" fire under control at about 7:02 a.m., the spokeswoman said.


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Q: search cs using for auto populate in textbox <%@ WebHandler Language="C#" Class="Search_CS" %>

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I have a file ashx.. through which i want to populate suggestion in my text box using autocompleate plugin jquery...
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My favorite type of exercising is probably weightlifting.
I like to plot out my routine. And every week try to figure out how to lift more and get stronger, and better, and have a better form at it.
Yeah, it's kind of cool to be able to feel strong.
And then when you get to do things like carry all of your groceries in two hands, all the way up your three flights of stairs to your apartment. That's pretty cool, because you were able to figure that out by doing weightlifting.


text:
Are Tax Cuts Trickling Down Yet?
Surprise bonus payouts spur re-examination of cuts' benefits.
By Matt Townsend & Elena Popina, Bloomberg | Updated on January 30, 2018
Just how much of this Trump tax cut will really trickle down?
Since Republican leaders pushed through their once-in-a-generation tax overhaul, more than 70 major U.S. companies—including Wal-Mart Stores Inc., Apple Inc., and JPMorgan Chase & Co.—have announced either pay raises or bonuses for rank-and-file employees or more hiring and investment.
Coming Soon: A Nationwide Ban on Non-compete Agreements?
Randi Winter | January 23, 2023
The FTC recently announced proposed regulations that, if adopted, will ban the use of non-compete agreements for most workers nationwide.
Argentina's $1 Billion Bond Buyback Plan Has Investors Stumped
Scott Squires | January 20, 2023
Investors point out that the government doesn't have much room in central bank coffers to fund such a lavish buyback.


text:
Wood products company Setra has reported an operating profit of SEK 231 million (600) for the third quarter of 2022. Net sales totalled SEK 1,343...
Setra's CEO Katarina Levin leaves her position at Setra's Annual General Meeting in May 2023.
Setra has been commissioned to supply locally produced CLT and glulam for the first phase of Greenhouse Sthlm, a climate-smart development by...
Wood products company Setra has reported an operating profit of SEK 564 million (442) for the second quarter of 2022. Net sales totalled SEK 1,792...
The timber industry company Setra is the first business in Sweden to offer a new moisture protection for frames made from cross-laminated timber. T...
Wood products company Setra has reported an operating profit of SEK 480 million (177) for the first quarter of 2022. Net sales totalled SEK 1,834...
Wood products company Setra is reporting operating profit of SEK 1,588 million (117) for 2021. Net sales totalled SEK 5,841 million (4,131). The...


Get Guest Appearance essential facts below. View Videos or join the Guest Appearance discussion. Add Guest Appearance to your PopFlock.com topic list for future reference or share this resource on social media.


You are invited to share your research at the upcoming Abaco Science Alliance Conference in January 2018!
3. Stimulate further research in The Bahamas.
Researchers are invited to submit contributed papers and posters on research findings in natural history and environmental science of Abaco and The Bahamas. Papers and posters will be accepted based on their technical merit and contribution to our knowledge of species, populations, communities, ecological processes, management practices, conservation initiatives, education models or policy issues.
Contact Us to find out how you can be involved.


Three CP is back on his own label, Lenjam Records, with a three-track deep and dark-progressive EP called 'Make You Mine'.
Three CP is not new to the blog; we have already featured him thanks to his Pryda-style tune 'Léa's Midnight Drive' delivered earlier this year in June. He's now back, this time with a three-track EP which follows the style of the previous release but adding a more deep and dark touch to the records.
Lenjam Records head honcho Three CP just released a brand new deep and dark progressive EP called 'Make You Mine'.
Kicking off with 'Make You Mine', a super underground record which features a wonderful top vocal, perfect for the sunrise set at 6 am in the morning. The track introduces an electrified synth in the last drop giving to the tune more character. 'Inox Is Painting' is the second record of the EP and it features more energetic percussions loop and harder vibes. A wide and heavy bassline rise with its epic vibes delivering another outstanding record. Last but not least 'Angels' close the EP. As the name suggests this record features more angelic and relaxing vibes compared to the previews production. An amazing top melody synth complete the explosion of sounds o the drop.
The EP is an amazing journey through the more deep and underground vibes keeping alive the main elements of the progressive house. As for the previous release, Three CP has done an amazing job delivering, once again, an AAA EP.


New Angling, outdoor gear shop opening where East Port garage was, recruiting staff, if anyone's interested.
What a fantastic addition to the town!
Is this a fishing trip?
Hope they set the hook when they get a nibble. A dotnet discount would be nice.
But I'd just make a sprat of masel so I won't bother.
Dunfermline has been flounder-ing around waiting with baited breath for a plaice like this.
Think ill just sit on ma perch and ride this one out.


The bigger the dog, the more there is to love.
That is certainly the case with Presley, but for some reason, the 3-year-old pup is still missing the most important love of all — that of a forever family.
In 2017, Presley caught the eye of Paula Joyner, a lead volunteer for East Mississippi Animal Rescue (EMAR), at a local animal control facility.
Volunteers nicknamed the 103-pound American bulldog/Lab mix "Big Mama," for her size and loving nature. But two years later, the sweet dog is still hopping from foster home to foster home.
Though she may look overweight, the big-boned pup has so much to give as a playful family pet. Her foster dad, Dylan Lugibihl, explained that a lot of her unusual size actually comes from her breed background.
"The size comes from the bulldog side, but she has plenty of energy and her weight hasn't seemed to impact her health or lifestyle in any noticeable way," he explained. "She frequently does long runs at the lake and explores for an average of three hours without stopping."
Presley also loves to get clean, whether or not it's her bathtime. She will eagerly jump into a running shower or full tub with her humans and constantly begs to be vacuumed.
But bathtubs aside, Presley is generally quite respectful of her surroundings.
The sweet pup is so trusting that she will even let her foster dad brush her teeth — though she's still not a big fan of the activity.
If you are interested in adopting Presley, you can fill out an application on East Mississippi Animal Rescue's website.


Excellently situated in the centre of the Golden City, the luxury Kempinski Hotel Hybernská Prague occupies an attractive 17th century residential palace. Major attractions such as Wenceslas Square, Old Town Square with the Astronomical Clock, the Powder Tower or Charles Bridge can be reached within a short stroll. A metro station is just steps away, while the main train station is at a distance of about 1 km. |Once guests step into the hotel, they are surrounded by an ambience of luxury and timeless elegance, complemented by an art gallery with works of renowned Czech artists. The rooms are luxuriously appointed and combine historical architecture with contemporary interior design. Guests will appreciate a wide variety of world-class services and amenities including complimentary WIFI, luxury limousine transfer, a picturesque interior courtyard and fine dining. A truly wonderful place for an unforgettable Prague experience.


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Вежі-близнюки () — дві (рідше більше) однакових будівлі (збігається висота, поверховість, зовнішній вигляд та інші параметри), що стоять поруч одна з одною. Деякі відомі вежі-близнюки:

 Всесвітній торговий центр, Нью-Йорк, США (1973 - 11 вересня 2001)
 , Коломбо, Шрі-Ланка (1997)
 Вежі Петронас, Куала-Лумпур, Малайзія (1999)
 Центр Тайм-Ворнер, Нью-Йорк, США (2003)
  — «Вежі-близнюки Банку Німеччини», Франкфурт-на-Майні, Німеччина (1984)
 , у місті Санта-Крус-де-Тенерифе, Канарські острови, Іспанія
  (), Мадрид, Іспанія (1994)
 Deira Twin Towers, Дубай, Об'єднані Арабські Емірати (1998)
 Аль-Казім Тауерс, Дубай, Об'єднані Арабські Емірати (2008)
 , Дубай, Об'єднані Арабські Емірати (2013)
 , Манама, Бахрейн (2008)
 , Шанхай, КНР (2005)
 , Рамат-Ґан, Ізраїль (1994)
 , Мумбаї, Індія (2010)
 Житловий комплекс «Башти», Дніпропетровськ, Україна (2005)

к найвищих веж-близнюків у світі||en|List of tallest twin buildings and structures in the world}}
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redpajama_set_name: RedPajamaWikipedia


As momentum for basic education has grown in the U.S. and globally, our focus has shifted beyond school enrollment to learning. Ensuring that the world's poorest and most vulnerable children can learn to read, write, do basic math and acquire critical life skills remains one of the best investments the donor community can make toward eliminating global poverty.
BEC members have long recognized the power of community engagement to improve children's learning. In places such as Pakistan, Ethiopia, Peru, and Guatemala, USAID programs are helping parents and community members to engage in children's learning opportunities.
There is immense long-term potential for greater accountability at all levels and it is exciting that the Bank is looking at how to engage citizens more actively in their students' learning. The Basic Education Coalition looks forward to working with the World Bank, to ensure that the focus remains on students' learning and that citizens are contributing to learning goals in the most empowering and effective ways.
Watch the discussion on "Accountability in Education: The Role of Citizens in Accelerating Learning for All." Stoner's remarks start at 13:30, BEC ECD Co-Chair Katherine Merseth directs the panel to issues of early childhood education at 1:15:19.


A 5-year-old suffered "significant injuries" after falling from the third floor of the famed Mall of America in Minnesota and may have been thrown or pushed, according to police, who cited witnesses. The child was treated at the scene and transported to the hospital.
He said police responded to the southeast court at the mall around 10:17 a.m.
A woman who works at a nearby store and who did not want to be identified said she heard loud screams, went outside the store and saw the boy hit the ground. "Oh my God, my baby, someone threw him over the edge, '" Brian Johnson told WCCO.
He pleaded guilty to a gross misdemeanor charge of interfering with a peace officer in connection with a July 4, 2015, incident where he was seen throwing objects from the upper level off the mall. He also refused to give his name to police and resisted arrest. But three months later he was accused of returning and throwing a glass at a woman in a mall restaurant. The complaint says Aranda approached a woman who was waiting for the restaurant to open and asked her to buy him something. He had two previous trespass notices ordering him to stay away from the Mall of America, according to the Star Tribune newspaper in Minneapolis. Police said they are unaware of a possible motive.
Police said that the incident appears to be isolated. "But as to why he did that, is still something we're looking into and trying to figure out".
Get the day's breaking stories, weather forecast, and more sent straight to your inbox every morning. Attractions include Nickelodeon Universe indoor amusement park and Sea Life Minnesota Aquarium.
In 2015, the al-Shabab extremist group called for an attack on the Mall of America and other shopping centres in a video.


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You will be met at the San Jose Airport upon arrival and transferred to your hotel. Overnight at the Park Inn, daily breakfast included - 2 nights. Conveniently located near the National Museum, Jade Museum and Costa Rican Art Museum, Park Inn boasts an ideal location in San José. All rooms includes free wireless Internet access, a mini bar and a coffee maker. Hotel facilities include gym, business center, and outdoor swimming pool, RBG Bar and Grill.
Today you will visit Irazú Volcano National Park, the Orosi Valley and Lankaster Gardens. Head southeast of San José, past Cartago and up the fertile mountain-side to the top of Irazú Volcano. Continue to the lovely Lankaster Gardens, where you will see acres of cultivated orchids, anthuriums, ferns and the birds they all attract - a truly spectacular tropical garden. Continue your journey through the beautiful Orosi Valley, where you will see: large coffee farms, the forest clad Talamanca Mountains, the Basilica of Ujarras (one of the oldest churches in Central America), the Cachi hydroelectric dam, and the ancient monastery of San José de Orosi, which was built in the 1700's and one of the few colonial churches still in use.
You will be met at your San Jose hotel and transferred through Braulio Carrillo National Park to the docks in Guapiles, stopping for breakfast en route. From Guapiles board a boat for the trip up the Tortuguero Canals through 30 miles of tropical rainforest to your Tortuguero lodge - Laguna Lodge. Three buffet meals are included daily and served in the open-sided restaurant. Lodge facilities also include swimming pools with pool bars.
Included during your stay is a tour of Tortuguero National Park (with National Park entrnce fee) and a visit to Tortuguero Village. Optional tours (at additional cost) include: beach walks to witness sea turtle nesting and birthing.
Today you will take a two to three-hour boat ride touring side canals of Tortuguero National Park with a local guide to observe the spectacular flora and fauna. Using a system of natural and man-made canals you will travel in a lowland tropical rainforest with arching palms and massive trees with giant buttresses. With its abundant wildlife Tortuguero is one of the best areas in Costa Rica for viewing wild animals. You will have the chance to see everything from a variety of bird species, howling, white-faced, and spider monkeys, sloths, crocodiles; iguanas, basilisk lizards and more from the comfort of your boat. In the afternoon, your guide will take you on a tour of the colorful village of Tortuguero. During July to September, green sea turtles come ashore at night to nest on the beach.
This morning you will fly to San Jose. From the airport you will be transferred to Arenal.
En route stop at the La Paz Waterfall Gardens. Here there are over 2 miles of walking and hiking trails, 5 waterfalls, cloud forest and rain forest, an aviary with 40 species of birds, a toucan feeding station, marmoset monkeys, an insect exhibit, serpentarium, orchid exhibits, butterfly observatory and butterfly gardens. The area is home to two-toed sloths, Capuchin (white-faced) monkeys, black-handed spider monkeys, 26 documented species of hummingbird, ocelots, jaguarundi, margays and even oceltos and pumas. Lunch is included.
You will also visit Poas Volcano National Park. The drive to the 9,000-foot peak of Poas Volcano is a scenic journey through coffee plantations, strawberry fields and lush dairy farms. You can look into the crater and see the hot steam vents and gray-green crater lake. In contrast to the constant activity in the main crater, a ½-mile trail through a gnarled dwarf forest will bring you to an overlook above the extinct crater of Lake Botos which is surrounded by dense forest along the circular crater rim. Entrance fee included.
Overnight at the Arenal Springs Resort, daily breakfast included. Hotel facilities include restaurant, sushi bar, Spa, 2 swimming pools and a Thermal river.
Pick-up at your hotel for an shuttle transfer to Manuel Antonio on the Pacific coast. On the way, enjoy a guided boat tour of the Tarcoles River/Mangrove Adventure Tour. Board a comfortable boat for the ride along the Tarcoles River and Guacalillo Estuary. The Tarcoles River borders Carara National Park. Along the banks of the river you will have the opportunity to see one of the largest colonies of crocodiles in Costa Rica and a chance to see over 250 species of birds such as scarlet macaws, tanagers, trogons, orioles, ospreys, cormorants, pelicans and roseate spoonbills. After the tour continue with your drive to Manuel Antonio National Park. Lunch is included.
Overnight at the Costa Verde Hotel, located on a coastal rainforest bluff nestled between Manuel Antonio National Park and the Pacific Ocean. The hotel is just three hundred yards from the beach, and about 500 ft in elevation overlooking world famous Manuel Antonio National Park. Daily breakfast included - 3 nights.
Day at leisure. Optional activities include: horseback riding, canopy zipline tour, coastal catamaran tour, mangrove estuary tour, Marino Ballena National Park day trip, surfing, fishing, paddleboarding, whitewater rafting, dolphin watching. sea kayaking.
You will be met at your hotel and transferred overland to San Jose. Optional short flight from Quepos directly to the San Jose Int'l Airport.
How many national parks are there in Costa Rica?
To date there are 27 national parks.
How many national parks will I visit on this trip?
You will visit or travel through 6. On the Tarcoles River excursion you will be on the border with a 7th - Carara National Park.
Can I visit more than 6 parks?
Of course. With enough time you can visit all of them. On this itinerary you can add a 7th with a day trip to Marino Balleno National Park from Manuel Antonio. The Marino Ballena National Park is named after the Humpback whales that migrate here to mate or give birth to their calves. They are often seen between the months of July and October and again between December and April.
Certainly. This itinerary is only a suggestion. We work with a wide selection of hotels and offer a menu of optional excursions in each city. Your trip can be organized around your travel dates. The number of days and nights can be extended. With additional days we can arrange trips to other national parks, such as Corcovado, Rincon de la Vieja, Cahuita, Palo Verde, Guanacaste, and more.
The dry season is December-April. During our summer - called the 'Green Season' in Costa Rica - there is more rain but the landscape is very lush. Summers are hot and humid, but less crowded.


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Q: How to dynamically change bookmarklet parameters using Angular? I have an angular single page app. I'm trying to create a bookmarklet but want the bookmarklet to reference a different JS file depending on if I'm in prod or local development environment. Here's roughly the raw bookmarklet:
My Bookmarklet

This works just fine and will load up the bookmarklet.js. However in Angular, I'm setting a scope variable that is the root to either the prod domain or local host like this in the controller:
$scope.rootUrl = prod ? 'http://mydomain.com/' : 'localhost:3000/';

Then on the angular page I'm doing:
My Bookmarklet

For some reason, when the angular variable is added to the bookmarklet, chrome prepends an 'unsafe:' like this when it renders and makes the bookmarklet useless.:
My Bookmarklet

I've searched around and can't seem to find a solution or understand why this is happening. Any help towards solving this would be greatly appreciated!

A: Safety, organization, and testability are three core aims of the angular framework. As such, it will NOT allow you to set javascript as the href of an  tag by default -- it is generally considered sloppy and unsafe, can be easily exploitable, and is very difficult to test.
The framework provides the ng-click directive to execute javascript in response to a click event, which replaces the need for  in ALMOST all cases, and to enforce safety, the angular compiler will by default ONLY allow mailto:, ftp:, http:, and https: in an href.
This means that if you need an unsafe javascript href that can be dragged to the bookmarks toolbar, you must configure the angular compiler in a config block like so:
angular.module('moduleName').config(['$compileProvider', function($compileProvider) {
  $compileProvider.aHrefSanitizationWhitelist(/^\s*(https?|ftp|mailto|javascript):/);
}]);

(Note: Older versions of angular (< 1.2) use $compileProvider.urlSanitizationWhitelist)


Thousands of bloggers, perhaps tens of thousands, are raving mad because a major source of affiliate revenue has just been removed from them.
In an email to its 'Power Pinners' late last week, Pinterest stated all affiliate links, as well as all redirects and trackers, would be removed from the site.
Particularly hard hit are affiliates with links from two popular affiliate networks, RewardStyle and Hello Society, which are being specifically targeted.
According to Pinterest, no pins will be deleted. The past Pins will still show up and be clickable, but the tracking links will be stripped.
So, those who were using tracking links from bitly.com just for tracking will have their links stripped.
Why am I NOT surprised Pinterest banned affiliate links?
Ask yourself WHY as a Super affiliate marketer of 17+ years (15 years full-time) experience – did I NOT devote time working Pinterest, or add a section to my coaching programs about 'how to monetize Pinterest'?
Because the writing was always on the wall.
I've only come across one blogger who was smart enough to mention the 'additional step' affiliate bloggers need to do now in light of this turn of events at Pinterest — that they'll have to link back to their own sites — and they actually lamented that fact.
Ah, pity, they (affiliate bloggers) are SO HARD done by… yet that's what they should have been doing to make a secure full-time income as affiliate marketers … basic online business stuff that I've been teaching since 2003.
For those of you who remember what happened to affiliate marketers who played the "Google Cash" game – direct linking to merchants from Google Adwords (and Google was PAID for those links) – Pinterest was bound to follow suit sooner or later.
Pinterest has disallowed Amazon affiliate links for years, yet some affiliate blogger / marketers put their heart, soul and all of their effort into monetizing Pinterest through direct linking to their merchants, despite the obvious sign of impending doom.
Do I feel for them?
The message is and always will be – work to build a business (emphasis on WORK).
i just started using Pinterest to promote my niche site and Etsy selling platforms. I had heard that there was a problem using Pinterest and Amazon, that they both stripped out the affiliate links in Pinterest boards. As a workaround, you suggest original blog posts on your own sites. These I have. The question I have is should the product description in Pinterest match my blog post copy? I use a lot of product category tables that I found easy to add copy to, since several of the products in my hot sauce niche usually have very little. With the tables, I used 50-100 words of copy along with an image and a link. That worked pretty well. I was thinking of a using the description copy in the pin with a link back to my blog post and changing my table into providing only the product image and link to wherever it's selling. if I kept both descriptions the same, would I incur a Google penalty for duplicate content?
P.S. My current fave is Tapatio.
Against my better judgement, I actually tried affiliate marketing using Pinterest and it didn't even work. I think I felt too guilty and it affected the mojo. It always seems like a bad idea to link from a social site directly to an affiliate page.
I have used Pinterest for fun (and profit/traffic) and link graphics directly back to my website.
Excellent points have been made: don't be dependent on third-party websites, send traffic to your own website, build your list, develop relationships, and presell the affiliate product.
I have had the experience (more than once) where a major company has decided to discontinue its affiliate program (without telling anyone). If you had direct linked to their program, your traffic would be wasted. On the other hand, If you had linked to your own site, you could recommend an alternative product.
You are SO right. Over the past 17 years, I've had the experience of discovering that an affiliate program was closed, by accident, or more often through Broken Link Checker. What's worse is when they redirect the affiliate links and the plugin didn't pick up the problem.
Am I on Pinterest? Yes. But like you say, link back to your own blog or site. This just makes more sense to me anyway. It seems obvious that one would want the traffic to their own site for a couple reasons. One, they just may get someone to sign up to their list. Thus another opportunity to sell to them again. Two, the visitor just may come back to your site for more and bring their friends.
Excellent advice as always, and very timely as I'm a late-comer to Pinterest and just getting started. I noticed you linked to one of your previous posts about BANS. I know BANS has been around for several years now and wondered if it is still relevant?
Thanks for sharing and caring – I'm glad to be back too!
I would consider BANS still relevant provided you accompany their builder with your own commentary and recommendations.
Good to be here. I think it's the first time I here.and certainly this is no good news for those marketers who invested into drawing traffic from Pinterest.
Though I wasn't an active user, I can definitely tick right what you said here. Linking directly to merchant's site is old schooled and now, we are understanding that redirecting links is taking us to a huge wall.
The best as you said is to link back to your personal blog where you can presell a product and increase your chances to generate sales.
Thanks kindly for stopping by and sharing your thoughts with us. We look forward to hearing more from you!
I had a hard time getting to know, understand and Like Pinterest. And so I decided to devote my time through other Social Medias like Facebook and Twitter, and erased my account with Pinterest. Perfect timing.
Pinterest has also banned CJ redirect links for quite some time.
Content is still KING. Because of that I've always treated Pinterest the same way as I treat banners ads. It's just one way to get people to my site.
Once I get them to my site then it's up to me to help them decide if the item is really the best for them.
I just started to get into Pinterest so this article stands out to me. Thanks for opening my eyes.
My pleasure and you're welcome!
I knew there was a good reason I did not put much effort into Pinterest. It looks like everything is going back to the affiliates and their own sites to provide the value to readers and then monetize their own sites.
Directing folks to your own site first has always been the best way to do business as an affiliate. I don't see that changing at any point.
miss this update and good sound advice.
I'm glad you remembered what I said… too bad she didn't take your advice.
Those who fall for the hack-of-the-month trickery will always suffer. WHy do they never learn? What's the big deal about posting a useful article about the product to your blog first, then pinning/linking to that? Good solid principles – build your business and relationships first, and you won't go wrong.
Someone said that Pinterest uses its system that scans its network for affiliate links, and then change those links to its affiliate ID. So Pinterest makes more profit.
So create a landing page contain the aff link or using trustable and paid cloaking service. Don't directly put an aff link into a pin.


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No roof in the rain is worse than hunger, says Somali IDP mother in Gedo
Saturday November 14, 2020
File Photo/Ergo
(ERGO) –Ibrahim Barrow Samow and his 12-member family are sleeping exposed to the elements after torrential rains swept away their makeshift house in southern Somalia's Gedo region in early October.
The family, along with hundreds of other internally displaced or IDP families, were forced out by the deyr seasonal rains that flooded Jazira and other camps in Luq district, where they have been living for the past three to four years.
"The hut collapsed on us at night, so I fled with the kids to a neighbour's place. You can imagine two families sharing a small abode with nothing," he said.
Their host could only offer them a covered corner of his house that does not protect them from the cold as its sides are open. But at least they are faring better than hundreds of other families living out in the open without anything over their heads.
These families came to Luq years ago after being dispossessed by drought that wiped out their livestock and dried up their farms. Aid agencies have been assisting them with food and basic necessities, while some have been working as casual labourers earning menial wages from casual jobs.
Ibrahim's family came to Luq from Ufurow, Bay region, after losing their herd of 60 goats in the drought. He has no skills that would allow him to earn a living, so the family has been dependent on his wife's occasional earnings as a house help outside the camp.
"She rarely finds work these days, but on a good day she would normally earn 30,000 Somali shillings, sometimes 40,000, the highest being 60,000 a day," he said.
Kuulay Ali Mohamed, a mother of nine, is enduring a similar fate. Her family came to Luq three years ago after the drought turned their farm in Wajid district of Bakool region barren.
The destructive torrential rains left them without even a sleeping mat. They are camping out under a tree.
"Not having a roof over your head is sometimes worse than being hungry. We do need food, but at this moment what we need the most is shelter. We don't have a place for the kids even for a night and we don't have covers to protect them from the cold," Kuulay told Radio Ergo.
The head of social services in Luq district, Abdiaziz Sugow Salad, told Radio Ergo that the worst affected were the families who had moved three months ago to a new location after Jazira camp became too congested. They were living in flimsy huts in a low-lying area.
"The rains have affected about 500 families, and the downpours are still continuing. They need shelter the most, so we invite anyone who can assist them with their needs," he said.
The local authority says it does not have capacity to assist the IDP families and has been asking the host community to take in the destitute families in as a stop gap measure until aid comes.
Somali parliament forms COVID-19 committee as Ministry warns of 'second wave' - HOL
UN in Somalia mourns death of ex-Ghana president - HOL
Outside Mogadishu, locusts turn farmland into desert - Reuters
New U.S. Secretary of Defense initiated a diplomatic gambit to negotiate with Al-Shabaab - HOL
Ksh2B Plane at JKIA to be Auctioned at Throwaway Price - Kenyans
'I've joined the Tories', says the former mayor who quit Labour - Islington Tribune
Explosions reported in two cities in Ethiopia's Amhara state - Reuters
Al Qaeda's No. 2, Accused in U.S. Embassy Attacks, Was Killed in Iran - New York Times
Refugee exodus to Sudan swells as war crimes feared in Ethiopia's Tigray - Reuters
Sudan to host Russian military base - Defence News


Discussion in 'Introduce Yourself' started by Rubricnigel, Oct 24, 2018.
Just want to say hello to all.
Welcome Rubric. I too am a catechumen, and I have been enjoying my journey into Orthodoxy.
Prayers for you regarding your baptism.
A most hearty welcome to you and may God bless you very much!
To everyone else, thank you for your kind words.
Welcome to CF! Glad you joined us.
Hi and welcome to C F.
Glad to hear about your upcoming baptism.
May the Lord bless you .


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Q: Why is the essential numerical range defined as $W_e(T) = \bigcap_{K\in \mathcal K(H)} \overline{W(T+K)}$? I have been introduced to the following definition of the essential numerical range of a bounded, linear operator on a separable, infinite-dimensional Hilbert space:
$$W_e(T) := \bigcap_{K\in \mathcal K(H)} \overline{W(T+K)}$$
i.e., the essential numerical range $W_e(T)$ of $T$ is the intersection of the closures of numerical ranges of all compact perturbations of $T$.

*

*Why is the essential numerical range defined in this way, i.e., what motivates this definition and how is it useful? Even in my wildest dreams, I would have no reason to consider compact perturbations of $T$ in order to define $W_e(T)$.

I have noticed that definitions in operator theory on infinite-dimensional Hilbert spaces are often motivated by finite-dimensional analogs, but I'm not able to draw any connections here. Thanks for any insights!

Note: The numerical range of a (bounded, linear) operator $T\in \mathcal B(\mathcal H)$ is defined as
$$W(T) := \{\langle Tx,x\rangle: x\in \mathcal H, \|x\| = 1 \}$$

A: First of all, I am not an expert on this topic, but let me try and provide you with some insight.
One of the application of the numerical range is a rough approximation of spectrum. In particular, for $T\in \mathcal{B}({\mathcal{H})}$ closure of the numerical range is smallest convex set (i.e. convex hull) that contains the spectrum of $T$
$$\operatorname{conv}(\sigma(T))= \overline{W(T)}. \quad (*)$$
Now, for a self-adjoint operator there is a notion of essential spectrum:
$$\sigma_e(T)=\{\lambda\in\mathbb{C}: T-\lambda I \text{ is not  Fredholm }\}.$$
For $\mathcal{H}$ finite-dimensional the spectrum is always discrete and essential spectrum is empty, so in a way essential spectrum captures the infinite-dimensional part of the spectrum or the part of the spectrum which is not discrete (i.e. not finite dimensional).
There are several equivalent characterizations of essential spectrum for a self-adjoint case, each of which provides inequivalent definitions of essential spectrum for general bounded linear operator $T\in\mathcal{B}(\mathcal{H})$. Let's focus on two (here I use Wikipedia's notation):
$$\sigma_{e,4}(T)=\bigcap_{K\in\mathcal{K}(H)}\sigma(A+K),\\ \sigma_{e,3}(T)=\{\lambda\in\mathbb{C}: T-\lambda I \text{ is not  Fredholm}\}.$$
For self-adjoint operator two sets coincide and for general $T\in\mathcal{B}(\mathcal{H})$
$$\sigma_{e,4}=\sigma_{e,3}\cup \{\lambda\in\mathbb{C}: T-\lambda I \text{ is Fredholm and } \operatorname{ind}(T-\lambda I)\neq 0\}. \quad (**)$$
It should be noted that $\sigma_{e,4}(T)$ is the largest subset of spectrum $T$ invariant under any compact perturbations and $\sigma_{e,3}$ is the complement of the set for which $ T-\lambda I$ is invertible modulo compact operator (Atkinson's theorem). Both of the properties hold for the spectrum of self-adjoint operator and are quite natural for the intuitive definition of essential spectrum as "non-finite-dimensional part".
Lastly, using $(**)$ and continuity of index it can be shown that
$$\operatorname{conv}(\sigma_{e,4})=\operatorname{conv}(\sigma_{e,3}).$$
So what would be the appropriate definition of essential spectrum that would approximate essential (i.e. non-finite) part of the spectrum in the sense of $(*)$?
Atkinson's theorem gives us another characterization of $\sigma_{e,3}(T)$:
$$\sigma_{e,3}(T) = \sigma([T]),$$
where $[T]$ is equivalence class of $T$ in the Calkin algebra $\mathcal{B}(\mathcal{H})/\mathcal{K}(\mathcal{H})$. There's a notion of a numerical range for a unital Banach algebra $\mathcal{A}$, which generalizes the usual numerical range:
$$W_0(a)=\{f(a): f \text{ is a state of }\mathcal{A} \}.$$
Thus we can define a numerical range for an element of Calkin algebra $[T]$, which was originally called essential numerical range:
$$W_e(T):=W_0([T]).$$
Note that since Calkin algebra is a $C^*$-algebra, it can be thought of as a $C^*$-subalgebra of $\mathcal{B}(\tilde{\mathcal{H}})$ for some Hilbert space $\mathcal{H}$ with the usual definition of numerical range, which coincides with $W_0$. Thus
$$\overline{W_e(T)}=\operatorname{conv}(\sigma([T]) = \operatorname{conv}(\sigma_{e,3}(T))=\operatorname{conv}(\sigma_{e,4}(T)).$$
Moreover, it can be shown that
$$W_e(T)=\bigcap_{K\in\mathcal{K}(\mathcal{H})}\overline{W(T+K)}$$
and this formula can be used as a definition which is quite natural in that sense and for essential numerical range of bounded linear operator there's only one natural definition, unlike the case of essential spectrum.
Summing up, essential numerical range is specifically about infiniteness and finite-dimensional analogs can't be found. Essential numerical range was introduced in GROWTH CONDITIONS AND THE NUMERICAL RANGE IN A BANACH ALGEBRA by J. G. STAMPFLI and J. P. WILLIAMS. This answer is based on a paper On the essential numerical range, the essential spectrum, and a problem of Halmos by P. A. FILLMORE, J. G. STAMPFLI, and J. P. WILLIAMS which is a good survey on the subject. It contains the proofs for all of the statements above  as well as other equivalent characterizations of essential numerical range which can be used to generalize it to the case of unbounded linear operators:
$$W_e(T)=\{\lambda \in \mathbb{C}: \exists\ \{x_n\} \text{ s.t. } \|x_n\|=1, x_n\overset{w}{\to} 0, (Tx_n,x_n)\to\lambda\}\\= \{\lambda\in \mathbb{C}: (Te_n,e_n)\to\lambda \text{ for some orhtonormal sequence}\} \\ =\{\lambda \in \mathbb{C}: PTP-\lambda P \in \mathcal{K}(\mathcal{H}) \text{ for some infinite-rank projection } P\}.$$


We rented our Vintage booth to Beth and Chris last year for their Grass Valley, CA wedding reception. We all had a great time!
Read Beth's blog review of her booth HERE.
This week, we had all three of our fabulous Vintage Photo Booth rented out- in Rocklin, Placerville, and Sacramento. Our Sacramento booth was at the beautiful LeRivage hotel on the Sacramento River.
One of my favorite photobooth shots of the week from Audrey & Andy's wedding at the beautiful Newcastle Wedding Gardens. This photo also demonstrates the outstanding quality of each of our photo booth photos.
Grand Island Mansion Bridal Show Models!
We had a great time showing off our photo booth at the Grand Island Mansion bridal show, part of the Delta Wedding Fair, also featuring the Ryde Hotel. We also had our professional videography on display, including our fabulous wedding demo featuring the Mansion. We offer a $200 booth discount for wedding couples that book both our Photo Booth and wedding videography.


The keto diet was originally designed to help children and some adults manage epileptic seizures but in recent years it has morphed into a popular weight loss plan even a lifestyle. There have also been some claims that it will cure what ails you.
I have been quietly observing the keto diet craze. I see so many people who have been successful achieving their healthy weight goals and I want to be supportive and encouraging. On the other hand, with all those college courses in biochemistry and physiology some of the hype does not make a lot of sense on how this can be a permanent lifestyle solution.
I recently spoke with my friend who has been successfully moving towards her healthy weight goals (not on the keto diet by the way) and she told me that she would love for me to write a blog post on the less positive side of the keto diet and that she was tired of hearing about it. And I started considering it (I am not one for controversy and my Midwestern upbringing doesn't make it easy) then I ran into another dear friend who told me that she was moving towards her healthy weight goasl and I asked her "Are you doing the keto diet?" And she said, "Absolutely not! I would never go on a diet that eliminates healthy foods like fruits and vegetables". And last night, I had a friend text me and ask "What do you think about the keto diet? I want a registered dietitian's point of view". She told me that she and her spouse followed it for two weeks and felt awful.
So I decided it is time to share my thoughts and feelings on the keto diet while encouraging everyone who struggles achieving their own personal healthy weight journey that there are individual ways to do it.
All foods can fit within reason to provide a variety when it comes to our health! Dig in!
Adding more fat to someone's meal plan may help them feel more satisfied and there are lots of nutrition benefits to healthy fats like avocados, nuts and seeds that provide essential fatty acids. Here is a list of healthy fats.
Eating less calories than you need and exercising more can help someone move closer to healthy weight goals. Recent Study on eating less calories.
Eating a little more protein also helps someone feel full longer, I would buy that also. Here are a some health facts on high protein diet.
Most people probably never achieve ketosis, check out this recent article from Popular Science. Most of the weight loss by going on a very high fat diet is probably because of the lack of calories not necessarily the lack of carbs and going into ketosis. And the symptoms of ketoacidosis are not very desirable and most of the time you do feel bad when you are having them. In addition, not many people realize that high protein foods can actually be broken down into carbohydrates. As my college professor used to say "You are buying steak but getting baked potato". Meaning, if your body needs carbs it's preferred form of energy and it will find ways to get them so it will break down that filet mignon right into carbohydrates.
But it's the recent research study that made me take a double take. It looked at 25,000 Americans and found that the ones who ate the least amount of carbohydrates had a higher risk of dying in the next 6 years from heart disease, stroke and cancer. It might be okay in the short run to eat these low carb amounts to achieve weight but as soon as someone achieves their health goal, it's important to switch over to a more balanced food meal plan.
High fiber foods provide the body a level of fullness. For many people, high fiber is much more effective than a low carb, high fat and high protein diet plus it provides more of the nutrients that you need. So why do we not hear more about it? It just doesn't sound as much of a quick fix and it may be a little slower and more permanent. Here is a great article on some of the facts on fiber and a healthy weight.
One of the tools that I love to share with clients is the ability to eat more, feel full but get less calories. I don't know why a concept from a nutrition professor that has been around for years hasn't taken off like the keto. It seems to me that everyone would want a full, colorful and flavorful plate. Check out this review from the Center for Disease Control on the Volumetrics and let me know if you love the concept!
If you are insist on trying keto or stick with it, keep in mind that it probably is not a permanent way of eating but a temporary tool and long use may cause health issues.
If you want an alternative, healthy way to eat, I just want to remind you that we are all individuals and we all have personal ways of eating that work for us. And I want to leave you with a quote from my friend who had tried keto and felt terrible doing it so she stopped "I like the good old fashioned way of eating. Clean, whole foods. More fruits & vegetables. Less processed foods. Lifestyle change." That sums it up nicely! Thank you dear friend for sharing your wisdom and the other friends who inspired me to write this post!
I hope that this post sharing some of the latest research on keto is helpful on how it may or may not be for you. As a registered dietitian nutritionist living in Asheville, NC, I love sharing recipe ideas in my kitchen that help others reach a permanent healthy way of eating. I am so glad that you stopped by! Check out my recipe page, other blog posts and newsletter for more tasty ways to eat that include high fiber foods where you can often fill your plate full with fruits and vegetables and still reach your health goals!
A great recipe starts out with great ingredients and these fruits and veggies are as beautiful as they are delicious!


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USAJOBS is the federal government's official one-stop source for federal jobs and employment information. We are required to post all current job postings and receive applications through USAJOBS.
Apply on USAJOBS
The best way for you to join one of the Best Places to Work in the Federal Government® is through USAJOBS.
Here's a quick way to view all current job openings at the USPTO.
Here's a user-friendly resource to help guide you through the application process.
We hire veterans
At the USPTO, veteran employees have a proven track record of success after transitioning from the military into Federal Service. Our veterans contribute to its mission in the areas of science and engineering, information technology, contracts, procurement, finance, administration, project and program management, and customer support. We are strongly committed to hiring our nation's veterans and has been ranked among the top employers of choice for veterans.
For consideration, you must meet the following criteria:
If you served on active duty in the United States military and were separated under honorable conditions, you may be eligible for veteran's preference. To receive preference if your service began after October 15, 1976, you must have a Campaign Badge, Expeditionary Medal, or a service-connected disability. For further details about Federal employment and veteran's preferences, view OPM's veteran program offices website.
Veteran's preference is not a factor for Senior Executive Service jobs or when competition is limited to status candidates (current or former Federal career or career-conditional employees).
To claim 5-point veteran's preference, attach a copy of your DD-214, Certificate of Release or Discharge from Active Duty, or other proof of eligibility.
To claim 10 point veteran's preference, attach an SF-15, Application for 10-Point Veteran's Preference, plus the proof required by that form.
Find out more about our veterans hiring program. For questions, contact Hirevets@uspto.gov.
We recruit and hire people with disabilities
USPTO depends on thousands of talented and highly skilled individuals to help us protect intellectual property and promote innovation in the United States. Disabled individuals are an important talent pool to help further our goals.
We are committed to using special hiring authorities, including Schedule A, to recruit individuals with disabilities.
Find out more about our disability hiring program.
This page is owned by USPTO Jobs. Published on: Jun 2, 2016 03:24 PM EDT	Last Modified: Aug 1, 2017 09:18 AM EDT


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February 2019 Funded Micro and IT Grants
2936 - Designing a universal genome editing strategy to remove PLP1 duplications in Pelizaeus Merzbacher Disease patient cells.
Pelizaeus-Merzbacher Disease (PMD) is a rare pediatric disorder that affects around 1:100,000 boys at birth. The disease is caused by mutations of the PLP1 gene, encoding Proteolipid Protein 1, a key structural protein of the myelin, which is an insulating layer that wraps around nerve fibers to facilitate the flow of information in the nervous system. Mutations in PLP1 result in abnormal myelination, which ultimately leads to severe locomotor and cognitive impairment, and limited life expectancy. In 70% of cases, PMD patients carry duplication mutations of various sizes of the X chromosome region containing the PLP1 gene. Currently, there is no cure for PMD, with treatment options limited to symptom management. In a previous study, we successfully tested a novel CRISPR/Cas9 genome editing strategy that removed the Plp1 duplication mutation and that improved the disease's manifestations in a PMD mouse model. This project aims at translating the genome editing strategy to individual affected by PMD caused by duplication mutations. Our goal is to test a universal CRISPR/Cas9 strategy to precisely correct the duplication mutations in cells derived from 3 PMD patients having PLP1 duplications. We will assess the nature of the duplications through Whole Genome Sequencing, then we will design and test a universal approach that could be applied to all PLP1 duplication mutations. Successful completion of this project will lay the foundation to support the development of novel therapies for the majority of PMD patients and open novel potential avenues for the treatment of genetic disorders caused by genomic duplications.
2903- Towards a Non-Invasive Gene-Therapy Delivery System for the Blindness Aniridia
Aniridia is a rare eye disease that reduces vision from birth and causes a lifetime of visual impairment. Aniridia is caused by defects in the paired box 6 (PAX6) gene, which is important for the development and function of the eye. Carrying defective PAX6 results in the malformation or absence of the iris (the colored portion of the eye), clouding and blood vessel growth in the cornea (the clear surface of the eye), and other severe eye problems (1). The poor vision in children progresses to blindness by their mid-20s to 30s. Hence, before vision deteriorates, there is an opportunity to intervene, and prevent blindness. Current treatments are unable to preserve vision long term, and thus new treatment strategies are needed. One possible approach for treating aniridia is gene therapy, where DNA is introduced into cells to alter their behavior, with the aim of preventing blindness (2). Common gene therapies for the eye are delivered in a safe virus, but by injections with needles (3). In contrast, topical application would be a non-invasive and thus more patient-friendly administration route (4). However, before such treatments are ready to help patients, we need to show that viruses can be delivered effectively in an eye drop, by doing research in cells and animals. Fortunately, mice with a defective Pax6 gene have an eye disease that resembles aniridia. We hypothesize, an eye drop formulation can be discovered that will allow viruses to survive and transfer their therapy to the corneal cells in such mice. We will use a fluorescent marker for assessing the success of our topical delivery in aniridic and wild type control mice. Although beyond the scope of this application, once an optimized topical route is found, we will deliver PAX6 by gene therapy in eye drops to increase the amount of PAX6 in the aniridic mouse cornea, and thereby improve vision. The experiments proposed here are important first steps towards developing a new non-invasive treatment for aniridia.
2932 - An efficient CRISPR/Cas9 knock-in system to restore full-length dystrophin expression in a Duchenne muscular dystrophy deletion background
Duchenne muscular dystrophy (DMD) is the most prevalent X-linked neuromuscular disease, affecting 1 in 5000 boys3. DMD arises from mutations in the DMD gene that renders dystrophin non-functinoal4. This protein is essential to muscle integrity, and without it, progressive muscle wasting occurs, causing loss of independent mobility and cardiorespiratory failure early in life1. Medical interventions can only extend life expectancy by 15 years at best8. There is a dire need for a cure, as current and proposed treatments are not feasible, ineffective, or produce the milder variant, Becker Muscular Dystrophy6,10. Around 68% of DMD mutations are the result of DNA deletions3. Recovery of dystrophin could be achieved if the lost DNA is added back (or knocked-in) into the muscle cell genome. I aim to cure DMD at its genetic source by knocking-in the deleted DNA to restore fully functional dystrophin. This will be accomplished using the gene editing tool CRISPR/Cas9 and a new DNA repair pathway, homology-mediated end joining (HMEJ)12. Previously, knock-ins on non-dividing cells like muscle and neurons were difficult, with efficiencies less than 4%12. Efficiencies greater than 50% were achieved in the neurons of live mice when CRISPR/Cas9 was paired with HMEJ 12. HMEJ may be a viable knock-in method for treating DMD deletions if this can be achieved in muscle. Our lab has a DMD mouse model with deletions in DMD exons 52-54. I will first knock-in exons 52-54 into the muscle of our live DMD mouse using CRISPR/Cas9 and HMEJ delivered on locally injected plasmids. Following this, the same system will be tested across the entire body of DMD deletion mice by delivering the required components with injected adeno-associated viruses. If a suitable recovery of dystrophin is achieved in these mice, we may open the door to a gene therapy cure for DMD deletion patients and a knock-in system for other rare disease deletion mutations.
2919 - Next generation antisense oligonucleotides to treat cardiomyopathy in Duchenne muscular dystrophy
Duchenne muscular dystrophy (DMD) is a devastating genetic disease affecting approximately 1 in 5000 boys worldwide. An exciting new approach to treating DMD is DNA-like molecule-mediated molecular therapy called exon skipping. These molecules act like a stitch or Band-Aid to skip over the region in the genetic code that blocks the normal process of protein synthesis and mitigate the effects of mutations. Our group previously demonstrated that these molecules effectively restore protein synthesis and improve muscle symptoms in various cell and animal models. Although the first drug of this class, called eteplirsen, was conditionally approved by the FDA in late 2016, it is highly controversial as exon skipping faces major challenges including the limited efficacy, especially in the heart.
We will address this issue using two approaches using new types of DNA-like molecules. First, we will employ new cell membrane-penetrating molecules attached to DNA-like molecules called antisense oligonucleotides (AONs). We developed these new molecules in collaboration with Dr. Hong Moulton (Oregon State University). These molecules are designed to penetrate the cell membrane in skeletal and heart muscles. Second, we will employ muscle/heart-targeting antibodies attached to AONs. We developed these molecules in collaboration with a biotech company Avidity Biosciences. We will test the effects of these new AONs in a DMD mouse model. Our study will facilitate the identification of a next-generation drug for DMD therapy, providing the potential for developing a cure for this devastating genetic disorder in the future.
2928 - Identifying the Genetic cause for Familial Abdominal Aortic Aneurysm
An abdominal aortic aneurysm (AAA) is an enlarged area in the lower part of the aorta. AAAs are often without symptoms and difficult to detect. The aneurysms grow slowly and eventually rupture, a serious complication and often fatal. AAAs are mostly sporadic caused by genetic and environmental risk factors. Some families suffer from the inherited form of AAA. The precise genetic cause of inherited AAAs is still unknown, making it hard to predict the risk and detect the aneurysm prior to the aortic rupture. In this study we aim to identify the causative gene in a large family affected by this disorder. This will be done by analyzing the entire genetic code for affected individuals. If the responsible genetic change is identified, other members of the family would be screened to determine their risk. Such information is crucial to implement preventative measures in individuals at risk and to elevate the enormous stress and uncertainty in members who do not carry the genetic defect. We intend to investigate how such a mutation produces this condition. Understanding the mechanism will help us to explore possible treatment options.
2892- Analyzing the UK Biobank to provide a comprehensive review on genetic signatures for rare diseases
Rare disease projects are often limited in sample size and comprehensiveness, due to funding or organizational challenges, and as a result many rare diseases are underrepresented in research. Over the past five years, large biobanks are being established, which instead of focusing on a specific disease, invest vast effort to collect detailed clinical and molecular information from the general population. Although rare diseases are defined has affecting fewer than 1 in 2,000 people (RDF, 2018), there are >7,000 rare diseases (CORD, 2015) and they affect ~3 million Canadians (1 in 12 people), as well as over 350 million people worldwide (Global Genes, 2015), so population-level biobanks can significantly extend the sample size for rare conditions, especially those which are underrepresented. Furthermore, the diverse range of information provided by biobanks is useful for understanding the overall burden of rare diseases. We will analyze data available from the UK Biobank (~500,000 samples) and present our results online for patients, clinicians and researchers to utilize. Patients in the UK Biobank have a wide range of rare diseases (including at least 49 with Stevens-Johnson syndrome and 148 with myasthenia gravis); the sample size for seven of these diseases is shown in the attached figure and we will consider many other rare diseases as well. Our aim is to provide detailed population characteristics for rare diseases in the UK Biobank, along with information about newly identified environmental factors and genetic risk variants.
2916 - Engineering protein-protein interactions to probe and rewire the Fanconi anemia pathway
Fanconi anemia (FA) is a rare inherited disease affecting 1 in 130,000 births. Mutations in any of the 21 genes in the FA signalling pathway lead to FA or FA-like syndrome, including bone marrow failure and increased likelihood of cancer. During cancer treatment, FA patients are extremely sensitive to radiotherapy or chemotherapeutic agents that cause breaks in the DNA as the FA pathway is essential to repair these DNA lesions in both healthy and cancer cells. Therefore, targeted therapeutic strategies that are less toxic to healthy cells and precisely target cancer cells are in urgent need. To tackle this problem, we plan to exploit the abnormally regulated nature of cancer cells by inducing "synthetic lethality", a natural off-switch used by cells to prompt cell death when two highly important genes are dysfunctional. This approach has been recently successfully employed at the clinic in the treatment of triple negative breast cancer and ovarian cancer. Rather than targeting the DNA like wide-spectrum chemotherapeutic agents, we plan to leverage synthetic lethality by utilizing our recently developed protein engineering and synthetic biology platform. Our platform develops potent and selective inhibitors for selected target proteins by modulating protein-protein interactions (PPI) that modern drugs have been unable to target. We propose targeting an enzyme (RFWD3) crucial for the protein modification and function of the cell replication protein RPA, important for cell survival. We will model RFWD3 and RPA structure and characterize their interaction to understand the mechanism behind DNA repair and further improve our inhibitors to target the interaction between RFWD3 and RPA at unprecedented precision. Our approach should impact cancer cells without causing lasting DNA damage observed with current treatments. We hope our work will shed light into more effective and less toxic treatment routes for FA individuals diagnosed with cancer.
2918 - Defining the Pathogenesis of a Novel CARD11 Mutation Causing Profound Combined Immunodeficiency and Inflammatory Gastrointestinal Disease
We have recently identified an infant and 17-year-old boy with severe recurrent respiratory tract infections, lung disease, and inflammatory gastrointestinal disease requiring hospitalization and removal of the colon. Using sophisticated next-generation sequencing, we discovered some of the first patients (4th and 5th) in the world to have damaging alterations in a gene called CARD11 causing profound combined immunodeficiency. This gene is crucial for normal immune system function and development. Without it, certain immune cells develop abnormally and cannot respond efficiently to microbes, leaving patients more susceptible to infections. However, the specific mechanisms by which genetic changes in CARD11 impair these immune cells and cause disease is not fully understood. In the proposed research, we aim to carefully characterize the impact of this CARD11 alteration on immune cell activation and development. By focusing on these aspects of patient disease, this study will directly inform the treatment strategy of the current patients as well as future patients with similar CARD11 genetic changes.
2930 -Evaluation of high resolution peripheral quantitative computed tomography for bone health characterization and evaluation of pediatric patients with growth hormone deficiency
Growth hormone deficiency (GHD) is a rare disorder that results in people having a shorter height and overall poor growth. Research has shown that those with GHD also have a low amount of bone (i.e., bone quantity), as well as a higher risk of having a broken bone. Another important part of bone health is bone quality; some studies in animals have found that the quality of the bone is worse with GHD, but this has not yet been shown in humans. HR-pQCT is a non-invasive and safe way to image bone that can tell us about bone quantity and quality. HR-pQCT provides us with a detailed picture of bone health, and this information might allow us to better understand changes to the bone over time in a children/adolescent with GHD. There are currently no research studies that look at bone quality in children/adolescents with GHD. We will use HR-pQCT to measure bone quantity and quality in the lower leg and lower arm in 20 children/adolescents with GHD, who have not yet started growth hormone treatment. One year after beginning treatment, we will use HR-pQCT to measure bone health at the lower leg and arm again, to see if there are any changes to bone quantity and/or quality. We believe that the use of HR-pQCT will tell us more about bone health in children/adolescents with GHD, and will allow us to better monitor changes to the bone in response to treatment. This is a small pilot study that will be used to support a future larger study looking at how HR-pQCT technology can diagnose and assess changes to bone health (both in the short-term and long-term) in pediatric GHD patients.
2905 - International registry for congenital pseudarthrosis of the tibia
Congenital Pseudarthrosis of tibia (CPT) is a rare orthopaedic condition in children. It can present as a fracture of the tibia which may be present at the time of the birth or during the first decade of life. CPT is considered to be one of the most challenging conditions to treat in orthopaedics due to the difficulty in achieving bone healing. The fracture does not unite with standard methods of treatment adopted for fractures in children and the tibia can re-fracture even after satisfactory union of fracture There is a high risk of post-treatment complications such as re-fracture, nerve compression, limb shortening and even limb amputation. Currently, there is no standard surgical treatment and management protocol in orthopaedics that can successfully treat CPT. The reasons for this include the rarity of the condition, the lack of high quality research studies and the paucity of studies with an adequate follow-up till skeletal maturity. In order to plan meaningful studies it is important that multicenter studies are designed to recruit adequate numbers of children for statistical analysis. Hence we propose the establishment of a multicentre registry for congenital pseudarthrosis of the tibia. With a previous rare disease grant (#2290), our team at BC Children's Hospital has developed the international database for CPT in REDCap. We are requesting for funds to start recruiting international sites. Several international investigators have shown keen interests to join this registry.
2922 - What makes a peer mentoring relationship powerful? A qualitative exploration of the iPeer2Peer Program for youth with chronic pain
Chronic pain, or pain lasting more than 3 months, is a symptom of various rare diseases (Chronic Regional Pain Syndrome (CRPS) affecting 1/3,800 people; Ehlers-Danlos syndrome (EDS) affecting 1/10,000-1/25,000 people). It is critical that youth with rare diseases learn how to manage chronic pain symptoms to avoid long-term negative effects on their wellbeing and development. Connecting people with shared lived experiences is both a unique and high-priority source of meaningful social support that can improve illness management and quality of life. The iPeer2Peer Program is a Skype-based peer mentoring program that offers social support and disease self-management skills by connecting a young adult (mentor) who is successfully taking care of their chronic pain with a newly diagnosed younger person (mentee). Each mentor-mentee pair have 10 weekly-bi-weekly Skype calls to talk about disease and life topics they want advice on. Exploring the content of calls revealed that pairs talk about the impact of chronic pain on daily life as well as managing pain and other non-pain related issues. Adolescents who took part in iPeer2Peer reported better and more efficient self-management of pain symptoms. It is therefore crucial to explore mentoring relationships across pairs to understand what makes a relationship most likely to motivate change. This project will evaluate 8 mentor-mentee pairs with a total 10 calls each. The selected pairs include 3 different mentors (18-25) and 8 mentees (13-17 years) with a range of diagnoses (e.g., CRPS, EDS, and neuropathic pain). All mentoring calls will be transcribed to conduct a deep analysis of recurring content and emotional bond formed by each pair and how this content and bond develops over the course of 10 mentoring calls. A better understanding for the development of mentor-mentee relationships will allow us to provide better, more efficient individualized care through improved pairing strategies and mentor training.
2913- Do Patient Specific 3D Heart Models Improve Outcomes for Children Undergoing Interventional Cardiac Catheterization?
The aorta is the main blood vessel that carries oxygen rich blood from the heart to the rest of the body. Coarctation of the Aorta (CoA) is a type of heart disease that people are born with, where a part of the aorta is much narrower than normal. This narrowing prevents blood from flowing properly to the rest of the body and causes the heart to pump much harder (1). CoA is a rare condition, it is estimated that it affects about 1 in every 2500 live births (2). CoA can be treated with a procedure called cardiac catheterization. During cardiac catheterization, a cardiologist threads a long, thin tube, called a catheter, through the person's blood vessel to their heart. Using the catheter, the cardiologist can place an expandable metal tube, called a stent, into the person's narrowed aorta to hold it open. Cardiac catheterization is highly effective and is less invasive than surgery. However, cardiac catheterization utilizes radiation to be able to see the location of the catheter and it is known that greater exposure to radiation is associated with increased lifetime risk of cancer. Cardiac catheterization can also be technically challenging because the size and shape of each patient's heart is slightly different (3,4). 3D-printing is the process of making a 3D model from a digital file. Using 3D printing, it is possible to make an exact replica of a person's heart using images from a CT scan (5). In this study, we want to determine if simulating the catheterization procedure using 3D models of patients' hearts will improve cardiac catheterization. For half of the patients in our study, we will make exact an 3D model of the patient's heart and use this model in a simulation of the procedure, before the actual procedure. The other half of the patients will be treated according to standard of care. We will compare to see if the group of patients who had 3D models printed, had a lower radiation dose and better blood flow after the procedure.
2911 ITG - Investigator-initiated Trial of MPAN 1-H9 in patients with mitochondrial membrane protein-associated neurodegeneration (MPAN)
MPAN is a very rare autosomal recessive progressive neurological disorder with iron deposition in the brain. It belongs to the group of syndromes with neurodegeneration with brain iron accumulation (NBIA). Patients develop early-onset motor dysfunction, movement disorders, cognitive decline, psychiatric symptoms, and visual disturbance. Brain imaging shows the eponymous brain iron accumulation in the basal ganglia, a central region of the brain. On brain pathology features related to Parkinson's disease and dementia have been observed. MPAN is an inherited disorder caused by mutations in C19orf12. The exact pathophysiology is unknown. Recently , a drug screening study in MPAN flies demonstrated positive effects of the drug MPAN1-H9, which is approved for other indications. So far there are no controlled data of its use in human MPAN patients. We thus propose a pilot trial to investigate the tolerability of MPAN1-H9 and its positive effects on symptoms and disease course. Aim is to repurpose MPAN1-H9 for its new use in MPAN. In our database more than 60 MPAN patients are registered. The study will be performed at the Neurology Clinic of the University Hospital in Munich, Germany. We plan to treat 15 MPAN patients with a genetically -confirmed diagnosis in a monocenter openlabel single arm pilot trial over a period of 6 months (3 visits, baseline plus at month 2 and 6) and compare the data with natural history data pre treatment. Clinical outcome measures will include clinical rating scales of disease severity (such as the Unified Parkinson`s Disease Rating Scale, Dystonia rating scales, Quality of life measures, functional independence measure, Beck Depression Inventory), functional tests (gait studies) and imaging parameters to assess cerebral atrophy and MRI-connectivity.
2914 ITG - Does strength-training program can slow muscle weakness of women with DM1
This study aims to determine if muscle strengthening can diminish muscle weakness experienced by women with myotonic dystrophy type 1 (DM1). Loss of skeletal muscle mass (also known as muscle atrophy) leads to muscle weakness. Individuals with DM1 frequently report muscle weakness as a major symptom. Muscle weakness and fatigue are two major causes of disrupted social participation in DM1. To help these people, rehabilitation interventions must be developed to target muscle weakness and fatigue. Muscle strengthening is safe in DM1 population. However, only few studies have addressed its efficiency to increasing maximal muscle strength and physical performance. Moreover, to this date, no studies have been specifically conducted in women with DM1. While there are no significant differences in the responses to exercise between men and women in healthy subjects, this question remains unknown in DM1. We recently developed a 12-week training program that has led to spectacular improvement in men with DM1. Indeed, lower limb muscle strength and power as well as walking speed were improved while fatigue and apathy were decreased during and after the training program. Participants have also reported to feel in better physical shape, have a better mood and more physical capacities, and be able to walk more and to climb stair more easily. All those changes can have a positive impact on patient's lives by attenuating restrictions to social participation and improving patient's autonomy. This study would be the first one to evaluate women with DM1 exclusively. This is of great importance if we want to develop rehabilitation interventions that are suitable for both men and women. From a broader perspective, the difference between men and women to muscle strengthening remains unknown in many rare diseases. This study could constitute a starting point to evaluate this effect among them.
2933 ITG - Harnessing Exercise in the Diagnosis and Treatment of Catecholaminergic Polymorphic VT: A Case Cross-Over Design Trial
The tragic, unexpected death of a young person can be caused by undetected heart rhythm disorders. Finding and treating rhythm problems prior to unexpected death is our focus. One dangerous and rare heart problem is catecholaminergic polymorphic ventricular tachycardia (CPVT), which is caused by dysfunction of heart cells during exercise. Thus, it is missed when tests occur at rest in the clinic. To find CPVT, an exercise stress test (EST) is needed, which involves monitoring the patient while they jog on a treadmill. The treadmill setting used is called the Bruce protocol (ie. Bruce-EST), which is an endurance program designed 70 years ago to detect heart artery blockages in older people, not CPVT in the young. We now think that CPVT happens mainly during sudden sprints. Therefore, the Bruce-EST may miss CPVT, even if the condition is present. Thus, treatment is not started, leaving a patient at risk of a dangerous rhythm. Our first goal is to design a new "Burst-EST" to see if sprinting on a treadmill is more likely to find CPVT. Our second aim is to make sports safer in CPVT. Because rhythm problems happen during exercise, doctors advise against athletics. This is unhealthy and reduces quality of life. New research suggests that most CPVT patients who take medicine (called a beta-blocker), can safely go back to sports. A newer medication option also exists called flecainide. Right now, flecainide is only used when dangerous rhythms occur despite using a beta-blocker, and the extra pills must be taken 2-3 times per day for life. In our study, we also want to determine if CPVT patients could exercise safely if they take a flecainide pill before planned exercise, but not regularly. To study this, we will ask patients to do our new Burst-EST on a beta-blocker, and then do another Burst-EST after taking flecainide. We will compare the results between the two tests to see if there were fewer dangerous heart rhythms on the second Burst-EST compared to the first.


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Find Trails > Kansas > Arkansas River Bike Path
Arkansas River Bike Path
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Arkansas River Bike Path Facts
States: Kansas
Counties: Sedgwick
Length: 10 miles
Trail end points: 21st Street N. and Galena Street
Trail surfaces: Asphalt
Trail category: Greenway/Non-RT
Trail activities: Bike, Inline Skating, Fishing, Wheelchair Accessible, Walking
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Arkansas River Bike Path Description
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The 10-mile Arkansas River Path is Wichita's longest and most popular multi-use trail, running mainly along the southwest bank of the Big Arkansas River between 21st Street (near I-235) and Galena Street at S. Washington Street. There are also two disjointed paths along the west bank of the river; one parallels McLean Boulevard between Seneca Street and 13th Street. Here you can link in Sims Park via a pedestrian bridge (near Central and McLean Boulevard). The other loop is between Kellogg Street and Central Avenue.
The Arkansas River Path provides access to parks and city attractions, including museums, the stadium, ice rink and the zoo.
Parking and Trail Access
Parking for and access to the Arkansas River Bike Path is available in many places, including off 21st Street (at trail's end), at the Westside Athletic Fields, Exploration Place, Cowtown Museum, along Mclean Blvd. south of Harry Street, Watson Park and at the southern terminus at Galena and Washington streets.
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Arkansas River Bike Path Reviews
nice with few stops
June, 2017 by reverendstark
You can go along way on this path. You can start pretty far south(I have started at the dog park at hydraulic and i135) and go all the way to Sedgwick county park. This might be several paths but most of it is Arkansas river path. Lots of path. Few stops.
Some parts flooded
May, 2017 by robindeawaugh
Enjoyed the well maintained trail from Fairfield Inn to the Keeper of the Plains statue. After that the trail deteriorated - several dead birds on the trail and approximately 1 mile from the statue the trail was flooded!
Trail Surface Needs Maintenance
April, 2016 by adsaxs
Since we were staying at the KS State Fairgrounds RV Park, we cycled to the start of the trail at Rice Park off of 23rd Avenue. We cycled to and around Carey park. We took the surface roads back to the fairgrounds.
While the trail is a very nice ride along the river and through some of Hutchinson's city parks, the trail surface needs maintenance. In some sections there were large cracks in the pavement. Other sections had tree roots uprooting the path. It was a kthunk-kthunk-kthunk ride on our hybrids. Luckily, these large cracks were marked with paint and were visible but the ride could have been more comfortable. There weren't any benches to sit along the path until we reached a park like Rice or Carey.
Overall, we enjoyed the ride. Carey Park was quite an impressive park and a nice place to stop and have lunch. So, if you are visiting the city, as we were, I would recommend a ride along this trail.
Best ride in Wichita
September, 2014 by fcolonmp
Very pleasant ride and a very popular weekend trail. The section North of US 400 (Kellogg Avenue) has the better views. I would start my ride from the northwestern area (parking off 21st Street) and head SW. That way you have the wind on your back during the later part of the ride, when you are a little tired!
My home town gem
October, 2013 by mrfootbag69
I ride this trail daily. It has some great scenery paralleling the Arkansas river, on the west side it winds past the Keeper of the Plains statue and through Exploration Place and on down past Lawrence Dumont Stadium. On the east side of the river you can see the Veterans Memorial pass by Century II, and the water wall at the Hyatt. This path connects to other Wichita trails and parks. You can ride many miles without ever crossing a street. If you do ride these trails please remember to be courteous to pedestrians and Bicyclist as well by announcing your self when passing. Happy riding.
Arkansas River Bike Path Review
July, 2011 by ksstathead
If you only have time for one bike path ride in Wichita, this one should be it.
It has decent to excellent quality surface, mile after mile without traffic interruptions, very good scenery along the river, and connecting paths or routes to most parts of the city.
The path crosses only one street, 15th (at McLean Blvd), and it is a small street.
There is a closure at an overpass south of Kellogg (maybe Harry?) that is being worked on. I was able to pass by the closed signs, but that could change. In that event, you would just spur up to the street level and cross with a crosswalk, then spur back down to the river.
The 'big' climbs are just up and down the river bank. Other than that it is a gentle up grade with the river as you go north and west.
There is a water fountain and restrooms at a small park at the very south end of the path (Galena at Washington on the east side of the river, close to Hydraulic and MacArthur). There were at least two other water fountains along the path, though the one at 13th was not working for me. But this is in a populous area and one could find water re-fills about anywhere during business hours.
From the south end, the path follows the east bank to Broadway where the bridge has bike lanes well-separated from traffic on both sides of the bridge as you cross to the west bank. This southern-most section is a little rougher than the rest, but still ok even on a road bike. From this point the path stays on the west bank to the north end at 21st street. In addition, another segment on the east bank runs from just north of Kellogg up to 13th street. There are a handful of east-west connection opportunities along this stretch of dual path.
This path network connects to the Zoo/Park path network via either the Zoo Blvd bike path or the bike lanes on both sides of Westdale Drive. There is a playground north of the Zoo along the bike path with water fountains.
Connections to the east side of town are mainly via the bike routes/lanes along 1st and 2nd street. Coming from the east, find the path via the Central path or the 1st street bridge, among other chances. The sidewalks are legal to bike in the downtown area, though I tend to use the streets unless in rush hour.
Remember the city buses all have dual bike racks on the front at no extra charge if you need a lift toward home. Limited evening and saturday service, however.
One can reach most of the city's main attractions and events right along this path network without traffic.
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Q: Confusion in Notation Recently I am doing a project on KATRIN experiment, which motive is to find the neutrino mass. So in a paper the result shows $(m_\nu)^2 =(-1.0)^{+0.9}_{-1.1}$ $eV^2 $. I do not get the meaning of writing numbers in super and subscripts. Does it specify some kind of error?

A: Yes, it means that the experimental uncertainty is asymmetric.  In elementary contexts, one often writes uncertainties in the form $\overline x \pm \delta $, but this implies that the true value is just as likely to be $\overline x+\delta $ as it is to be $\overline x-\delta $.  In other words, the probability distribution associated to the measurement is symmetric about $\overline x$.
In more careful treatments, one often finds this not to be true, and that a more accurate description of the experimental uncertainty would capture the fact that the uncertainty is greater in one direction than the other. To reflect this, one writes $\big(\overline x\big)^{+\delta_1}_{-\delta_2}$.
For more information, you may find this PSE Q&A useful.