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Home entertainment Another Unsuccessful Battle for Alfredo Adams: Fallen in Attempt to Expel Carlos...
Another Unsuccessful Battle for Alfredo Adams: Fallen in Attempt to Expel Carlos Trejo's Lawyer
Alfredo Adams starred in a new battle
press conference with Alfredo Adam As the protagonist, it became a new fight for the actor, although – again – the results were not the best for him.
Adam was talking to the media in a restaurant, when he was interrupted by a lawyer Carlos Trejo, the famous "ghost hunters" With whom the actor had a public confrontation for years.
The lawyer, surnamed Montalvo, accused Adam of being the person who did not attend the famous battle that was to take place in August 2019 and which was even called the "Battle of the Century".
As he will remember, Adame and Trejo agreed on a public showdown that caught the spotlight of all the entertainment press in Mexico.
In July 2019, Adame and Carlos Trejo fought a fight in the middle of a press conference (Cuartoscuro)
However, the meeting did not take place because in the press conference for that fight, Trigo threw a water bottle at Adam And that ended with a hit under the eye.
In the new discussion, Adam was harassed again, because although the videos currently show him angrily rising from his spot and resisting the lawyer, he did not hit him as expected.
Everything was unleashed because Adam assured the media that the fight didn't happen due to Trejo's demands.
"He asked for 3 million pesos, they won't give it to him because he doesn't deserve it. Then he wanted an advance of one million pesos, which would not be given to him for lack of confidence because he had already tried to steal." "He asked for his shoes, knives, leather belt and stinking jacket. He asked for all this nonsense so that it wouldn't be done."
Adame and Trejo have had problems for years (Photo: Infobae México)
However, Montalvo got up from his seat and shouted at the actor: "The Tracalero was you, the one who didn't attend the press conference is you."
See also "It is something we cannot forget": Pascal Obispo was distinguished by an overwhelming meeting ...
Adam got up from his seat and walked towards him despite the others trying to stop him. Looks like the actor tried to apply his style in "Bike Kick"toThe same thing he used in his famous street fight.
Adam ended up on Earth in his new fight (screenshot)
However, he was unable to contact the lawyer, tried to pass between the chairs in the restaurant, and ended up stumbling on the floor, forcing him to hire other people to get up.
Of course, the harassment and memes were immediately unleashed, although the "ghost hunters" had yet to comment on the new, failed battle of his enemy.
Trego and Adam could not stand the urge to confront each other and cross paths at the press conference before the duel that never happened (Photo: Andrea Murcia/Quartoscuro)
It is not known whether Adame and Trejo will at some point be able to finish the famous "battle of the century", because in 2019 – after attacking him at the conference – the actor said that he needs time to recover.
The actor even reported Trejo to the authorities and said the fight could be postponed. "I will not cancel it, I will postpone it to get better. "
The Ghostbusters didn't like the idea of postponing the fight. "Everyone made a fuss about a small 250ml bottle. Lawyers are ready to respond, if you don't show up you have to pay."
"It amazes me that he says it takes so many days, not even a woman who had a caesarean section," he added. "I don't care about the suit, I'll introduce myself that day, in the ring I'll see how cowardly he is, get ready, because August 2 is coming anyway. We'll wait for him, because I'm going to introduce myself that day, I'll be there."
Mexican actor Alfredo Adams starred in a street fight with a woman. (filming)
Despite Trigo's warning, the fight never took place, although the two's public confrontation did not end.
See also Recordings for the new version of 'Hasta que la plata nos separe' have begun, this is the cast
"Ghostbusters" does not miss an opportunity to criticize or ridicule the actor, especially after some of his scandals such as A fight he had two months ago against an entire family.
The best memes left by the fall of Alfredo Adams in his new fight
Laura Pozzo compared Alfredo Adam to an animal
The best memes of Alfredo Adam teaching karate at Venga La Alegria
Gustavo Adolfo Infante Alfredo Adam accused with CDMX prosecutor: 'He walks with a pistol and knives'
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In this paper we investigate the problem of integrating the complementary audio and visual modalities for speech separation. Rather than using independence criteria suggested in most blind source separation (BSS) systems, we use the visual feature from a video signal as additional information to optimize the unmixing matrix. We achieve this by using a statistical model characterizing the nonlinear coherence between audio and visual features as a separation criterion for both instantaneous and convolutive mixtures. We acquire the model by applying the Bayesian framework to the fused feature observations based on a training corporus. We point out several key exisiting challenges to the success of the system. Experimental results verify the proposed approach, which outperforms the audio only separation system in a noisy environment, and also provides a solution to the permutation problem.


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Estimating catch-at-age of fish
In order to set quotas for the catch of commercial fish species such as cod, complex population dynamics models have been developed. An essential input to these models is the age distribution of the fish caught every year. This is estimated from fish sampled from boats as they land their catch. The major problem with this data is that although the number of fish sampled is large, there is a very large within-boat correlation, and the number of boats sampled is relatively small. Also the age distribution changes in space and time, and with the type of gear used by the boats.
We analyse the data by developing a hierarchical model. We consider the numbers of fish at each age from an individual boat to have a multinomial distribution, the parameters of which are themselves random variables. These variables are correlated in space and time, and between different fishing gears. This analysis allows us not only to estimate the age distribution in areas where there are few or no samples, but also allows us to give a measure of the uncertainty in the estimation.
The model is applicable to several fish species. Improvements of the model includes simultaneous modelling of North-East Atlantic cod and coastal cod as separate stocks, handling of several sampling designs used in various European countries, and simultaneous modelling of landed and discarded fish. This is joint work with the Institute of Marine Research.
Hirst, D., Storvik, G., Rognebakke, H., Aldrin, M., Aanes, S. and Vølstad, J. H. (2012): A Bayesian modelling framework for the estimation of catch-at-age of commercially harvested fish species. Canadian Journal of Fisheries and Aquatic Sciences, Vol 69, p. 2064-2076. DOI: 10.1139/cjfas-2012-0075
Hirst, D., Storvik, G., Aldrin, M., Aanes, S. and Huseby, R.B. (2005): Estimating catch-at-age by combining data from different sources. Canadian Journal of Fisheries and Aquatic Sciences, Vol. 62, p. 1377-1385. DOI: 10.1139/F05-026. [pdf]
Hirst, D., Aanes, S., Storvik, G., Huseby R.B. and Tvete, I.F. (2004): Estimating catch at age from market sampling data by using a Bayesian hierarchical model. Applied Statistics, Vol. 53, pp. 1-14. DOI: 10.1111/j.1467-9876.2004.00422.x
c-code documentation
Magne Aldrin (eng)
Chief Research Scientist Magne Aldrin. Research Leader for Marine Resources and Health
Statistics for marine resources and health
The Institute of Marine Research
The Research Council of Norway

redpajama_set_name: RedPajamaCommonCrawl


Is there a cure for Bell's palsy? MRI scanning is painless and does not involve X-ray radiation. Anonymous March 31, Reply. When combined with increased blinking and the occasional faint smile, a seductive appearance and demeanor is achieved. Taping the eye shut overnight may also be suggested. Dryness of the eye or the mouth, alteration of taste on the affected side, or even excessive tearing or salivation can be seen as well. Reading Expressions on One's Face.
They are responsible for a variety of specialised functions such as smell, equilibrium balance , pupil contraction and hearing. Mostly cloudy, with a high near Migraine headaches, tension headaches, and cluster headaches are considered primary headaches. There is not a blood test to diagnose HFM. However, this kind of surgery is used rarely as it carries a high risk of nerve damage. Parents and other family members should have a thorough evaluation to help give more definite recurrence information.
His facial expression is one of nervousness when Basil asks for the true status of his soul. Anonymous March 31, Reply. Traits of a Habitual Liar. Generally, faces that are babyish think less pointy, with a high forehead are rated as more trustworthy. Researchers found people display the expressions on their faces as subconscious calls for help. Scientists believe we routinely pull 21 distinct faces - more than three times the long-accepted figure of six.
However, this kind of surgery is used rarely as it carries a high risk of nerve damage. In other words, a parent may unknowingly have a very mild sign of hemifacial microsomia, such as preauricular tags skin tags by the ear , but the child is more severely affected. Home Self-assesment Paper 1 Paper 2 Paper 3. Practicing and using the muscles are keys to regaining mobility. Post-stroke exercises can help you to overcome them.


text:
package models.id.ac.unpar.siamodels.matakuliah;

import models.id.ac.unpar.siamodels.Mahasiswa;
import models.id.ac.unpar.siamodels.MataKuliah;
import models.id.ac.unpar.siamodels.matakuliah.interfaces.HasPrasyarat;

import java.util.List;

@MataKuliah(kode = "AIF342", nama = "Administrasi Jaringan Komputer 2", sks = 3)
public class AIF342 implements HasPrasyarat {

	@Override
	public boolean checkPrasyarat(Mahasiswa mahasiswa, List<String> reasonsContainer) {
		if (!mahasiswa.hasLulusKuliah("AIF341")) {
			reasonsContainer.add("Tidak memenuhi prasyarat lulus AIF341");
			return false;
		}
		return true;
	}

}

redpajama_set_name: RedPajamaGithub


The next slice of the Ghost in the Shell called Ghost in the Shell: Arise is premiering on June 22nd, 2013. I don't think we've reviewed any of the GitS series of shows or the movies, but I personally have seen all of them. I enjoyed the shows a lot. The Stand Alone Complex series always had 2 split stories; one main story like the Laughing Man (the first Stand Alone Complex) or the Individual Eleven (Stand Alone Complex 2nd gig), and then a series of somewhat independent one or two episode stories.
Ghost in the Shell (the film) which sparked this all came out on October 18, 1995 which was an adaptation of a manga in the same name which started in May, 1989 (funnily enough the year I was born). And now the franchise lives on having made numerous movies and TV series.
Ghost in the Shell: Arise will start with the episode titled "border:1 Ghost Pain" and will be a double length episode of roughly 50 minutes and happens in the year A.D. 2027 and is presented by Production I.G. The show seems to be an alternate era with the same characters, much like how the original movies and the Stand Alone Complex are similar yet different. There will also be a pay-per-view streaming service for the show, so I would assume it won't be airing in the western hemisphere on Crunchyroll.
The trailer gives us a glimpse of what is to come, showing that Kusanagi Motoko is now heavily Cyborg and generally shows us scenes of what you would expect in a Ghost in the Shell series.
I myself am not super excited about the show right now (actually maybe I am a little bit), I feel like it is being milked for its success however that is my opinion. I plan to be reviewing it (unless Rasul catches up by watching all of the previous seasons) nevertheless.
I will keep everyone informed as we get more information on the upcoming Ghost in the Shell series.
Well, it's really more of a movie from my understanding. It will presented similar to the tradition of Kara no Kyoukai. It will be released in cinemas (limited theatre showing) and on pay-per-view streaming as you've said. The next month it will be released on optical media. You can call it an OVA but it's really a series of 4 films that are less that an hour long (approximately 50 minutes each).
Oh, just 4 films? Oh that's not bad, an interesting take on things. Not excited about waiting years for the movies to come out if that's the case.
I hope we don't have to wait years but quality is better than speed, in my opinion. Also, the Blu-Ray and DVD release of the first movie is coming out in July which isn't a long after the theatre release. I'm intrigued by the name of the first film "Ghost Pain". I wonder if it is about exploring some pain in the Major's past and how she came to terms with it.
I'm hoping that after these 4 films, another Ghost in the Shell project is announced. Since this series of films is basically a reboot they may as well continue the storyline.
Sounds interesting, I shall look forward to seeing these movies. I might have to refresh my mind by rewatching the SAC's.
It's good they are releasing the Blu-Rays pretty soon after, its unfortunate that the Theatre releases don't come to the UK or USA. I would definitely love to see them in the cinema!


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Home Best You Health
Are energy drinks dangerous to your health?
Caffeine has proven health benefits, but how much is too much?
Source: Best Health magazine, December 2012
Recently, the U.S. Food and Drug Administration (FDA) was reportedly investigating five deaths and one heart attack in the U.S. for their possible connection to the consumption of Monster Energy drink. A 24-ounce can of Monster Energy contains about 240 mg of caffeine, according to Monster Beverage Corp.
Energy drinks are not yet clearly labelled with nutritional information, including caffeine content. In October last year, our federal government announced new measures to provide Canadians with more information about energy drinks, which up until that point were classified as Natural Health Products (NHP), and were not required to put nutrition labels on their products. Under the new measures'scheduled to be in effect by late 2013′Health Canada also moved to limit the amount of caffeine in an energy drink to 180 mg in a single serving.
Caffeine is a naturally occurring substance that is found in coffee, cola, tea and chocolate. It's also added to certain medications, and to soft drinks other than cola, including energy drinks. About 15 minutes after it's consumed, it's absorbed into the bloodstream.
When drunk in moderation, caffeine has proven benefits. According to Massimo Marcone, an associate professor and food scientist at the University of Guelph, caffeine 'travels to the brain, where it acts as a stimulant.' You may feel wide awake and experience temporary improvement in memory, mood, reflexes and comprehension, he says.
Too much, however, can be harmful to your health: Consuming 600 mg or more a day can spark body shakes, insomnia, irritability, dehydration, dizzy spells, diarrhea and even convulsions. Health Canada states that adult Canadians should consume no more than 400 mg of caffeine per day'which is equal to approximately three 237 mL cups of coffee.
How much caffeine is in your drink?
' Brewed coffee (237 mL): 135 mg
' Black tea (237 mL): 43 mg
' Green tea (237 mL): 30 mg
' Cola (355 mL): 36-46 mg
This article was originally titled "The buzz on caffeine" in the December 2012 issue of Best Health. Subscribe today to get the full Best Health experience'and never miss an issue!
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A pair of locals have been arrested by Southington police after two shoplifting incidents at Stop & Shop at 505 North Main St.
Police were called to the store just after 8 p.m. on Tuesday, Feb. 16 after receiving a report of a shoplifter in custody. During the investigation, officers identified a second shoplifting incident earlier in the day involving the same suspect. Two people were found to have been working together in both incidents.
The investigation showed that, at approximately 2 p.m., Steven Galipeau, 32, of Southington, entered the store and selected merchandise from the shelves before leaving the store without paying. The items were allegedly sold in Hartford later in the day. Police identified Aimee Sycz, 28, of Southington, waiting for Galipeau in a vehicle in the parking lot.
The stolen items were identified as three 12-packs of Red Bull (valued at $21.59 each) and four 4-packs of Red Bull (valued at $8.19 each). The total value of items stolen was $97.53.
At approximately, 8:02 p.m., the couple returned. This time, Sycz entered Stop & Shop while Galipeau waited in the car. Sycz selected merchandise from the shelves and left the store without paying. At this point Sycz was stopped by Store Security, and the investigation was initiated.
During the theft at 8:02 pm incident, the following items were stolen: three 12-packs of Red Bull ($21.59 each), five 4-packs of Red Bull ($8.19 each), one Gatorade ($1.59), and one 25-ounce can of Bud Light ($2.54). The total value of items stolen was $109.85.
Sycz and Galipeau were each issued a misdemeanor summons, and both were charged with two counts of sixth degree larceny, and two counts of conspiracy to commit sixth degree larceny. They were released on $500 bonds and are scheduled to appear in Bristol Superior Court on Feb. 29, 2016.


Why Listening to Your Inner Voice Can Inspire Productivity!
There are many ways in which you can increase your productivity. Sometimes external shifts in your life can cause you to have time to do more work. By looking at your schedule, you can have a better idea of how you spend your time and where you can slim down to be more efficient. You can even cut back on certain commitments that you don't find enriching, so that you can spend more time creating things that are more important to your career trajectory! Other times the changes you make are internal, and can involve changing your schedule of working and sleeping so that you feel recharged. This could also mean that you spend more time reflecting so you have a centered focus on where you should aim your time and talents. Either way, science says that people who talk to themselves are more likely geniuses. If you are looking to become more productive, read this article to learn why quiet reflection leads to more productivity!
How Relaxation Can Inspire a Greater Productivity!
How to Creatively Inspire Productivity!
The Reason Why Checking Your To-Do Lists Feels Great!


The Louvre is, so they say, the most visited museum in the world, racking up 6 plus million each year. Tickets run about 10 Euro, so do the math. It's a business. As a business those who run it know what "sells" and at the entry are large notices, with Leonardo's pin-up girl blown big, pointing the way to Mona Lisa, the fabled mysterious lady. There, pushed away about 3 meters by a large wooden barrier, and encase behind thick glass, she looks out, bemused. It is not a very big painting, so between the distance and the glass casing, it is not really possible to look at it, qua "painting." What you see is Mona Lisa, spectacle, and the spectacle of Japanese and Korean girls posing, leaning against the barrier, smiling and/or with their ubiquitous V fingers, long ago having lost its meaning and now simply a mandatory reflex before a camera.
In my view this painting is not actually a very good painting, especially when compared to contemporaneous work. While Leonardo was clearly an intellectual giant of his times – polymath engineer, inventor, painter, poet, etc., he wasn't really that great a painter. Or so says humble me. (A similar opinion I hold for Van Gogh, whose museum I visited yesterday in Amsterdam.) But the matter of whether La Gioconda is really a great painting or not is irrelevant: it is a most famous painting, and that is what the Louvre is selling. Along with the name The Louvre, which is as its numbers testify, also famous. And hence the new Louvre Abu Dhabi, building designed French architectural star Jean Nouvel, begins to rise, the arts logo mania gone wild.
But, back in Paris, the old Louvre, formerly the absurdly vast home of French royalty, became a museum, and not so long ago, I.M. Pei, installed his pyramid, gracefully placed in the court-yard, an impeccable work of high-modernist style, nestled almost invisibly into the ornate classical French architecture which houses the museum. It was Pei's new entrance which kick-started the Louvre, shifting from fusty old museum to modern market economy logo champ.
Regardless of the revamping, the vast spaces of the Louvre, while housing unquestionable works of art of the highest order, is also the warehouse for great swath's of art of questionable taste and quality. In fact, from an artistic viewpoint, perhaps 80-90% could be skipped without any loss, unless one is a historian or has some other specialized reason to look at huge canvases of dubious artistic value, however much prized they were in their own time. Thus it becomes a kind of amusement to look at bedraggled tourists glancing at the little placards beside the paintings, and looking upward to vast battle scenes, historical tableaux or religious tripe, gazing at the meters of canvas, and thinking it is, because it is in the Louvre, great art. Small wonder they collapse on the benches, exhausted from taking in this vast collection of stuff, and wonder what it's all about.
Here's a little compendium of things I took shots of, not because I thought it great art, but because I found it of interest in one way or another – because it was odd, kitschy, or otherwise stuck out from the generalized dross which covers the walls of this massive museum.
Those are some of the paintings I took photographs of, for this reason or that. As it turned out the one painting I really wanted to see, and wanted to show to Marcella, Vermeer's tiny Lacemaker, just happened to be in a wing that was closed on Thursdays, the day we were there. So we didn't get to see it. I'd seen it before, when it was in a different place, in a narrow and tiny little alcove backed up against the huge room full of ghastly Rubens paintings. Alas.
Paris is a place of cafes and bistros, where the social life is a major element in daily life – to sit with friends and talk, share a coffee or a glass of wine, glance out the window and watch life pass by, while displaying oneself as if on a stage.
Since my last visit to L'Orangerie they did a major change: moved Monet's two oval Water Lilly rooms from the basement to the top floor, with a big architectural restructuring. The new space has filtered natural light from the ceiling, and seems cleaner in design, and more spacious (though I imagine that is just a trick of the light.) The other collection, which I find rather weak, is now banished to the basement.
I have spent some hours in these two rooms, some time ago with a DV camera, before they installed little barricades, dancing a few inches from the surface, shooting the wonderful painterly delirium which was Monet's world. Once I watched an older American woman brush her hand against the surface – which is quite textured and rather invites a tactile approach, though the woman was promptly reproached by the guard. I find myself wondering about most of those who come to this, as in a pilgrimage, an obligatory tourist matter, as they sit on the benches and gaze. Few come actually close to the surface of the work, but sit back at a distance, where the abstraction of painterly coloring forms a diffuse impressionist image of water, lillies, tree trunks, reflections. Only a few move close, where the scale of the work takes over and immerses one in a sea of painterly effect – strokes of the brush, the gentle build-up of color, the textures, taking in one's whole sense of vision. Monet was clearly here a precursor of the abstract expressionists, of color field artists like Rothko or Morris Louis, demanding that you immerse yourself into the totality of the painting, which in a push-pull manner reveals itself as precisely painting, and as a sublime spiritual experience of pure color and in a Heideggerian sense, "being". One goes into this kind of painting and experiences the hard reality, literal paint, and in doing so it makes a quantum leap to something else. My guess is those that sit on the bench and see a pond with water lillies do not get this.
Just what each of us sees, I am not at all sure. What I am sure of is that Monet made a work of art that will last as long as we humans care about such things.
Stepping out to the damp gray cold of January, into the central court of the place we're staying in – graciously offered by a friend's parents, we move along to the Metro for another day of museums. This day was the August Rodin museum, south of the Seine.
I'd been there before, many years ago, as well as seen a lovely exhibition at the Villa Medici in Rome still longer ago. And naturally seen the occasional piece here and there, in museums or otherwise. Like really good work, one can come back again and again, and be stunned by his capacity to capture the core of character and life in the hard materials of his art. His work, particularly the ensembles of multiple figures, is effectively cinematic, alive with tensions and interchanges between the figures, the spaces they fill. Truly wonderful work.
The museum was also showing a collection of Henry Moore pieces in the context of his studio work – macquettes, plaster versions in preparation, and the like. For a "muscular" sculptor like Moore the setting next to Rodin was frankly unfortunate, and made his work seem weak by comparison, as well as rather dated. While Rodin's work is clearly of its time, this comes across as a strength. In Moore's case it's the opposite.
Unfortunately we'll just be missing the installation at the Louvre Pyramid of a very large scale work by another British (though living for some time in Germany) sculptor, Tony Cragg. It opens in the next days, just as we are leaving.
Under a regime of what might be called "torture tourism," prompted by the constraints of limited time, we bought a four day ticket to an array of Paris' museums, for, if one crams things, a discount. Our first use was to visit the Concierge, formerly a prison for none other than that Marie, as well as others headed for a beheading in the Terror. It was crammed with a film-oriented exhibition of historical movies done in Paris. Yet another exhibition mounted by people who apparently also lost their heads, so absent was their "work" from anything compelling. Mainly we were biding time until the chapel adjacent, the Sainte Chapelle, opened. Curiously while living in Paris a year and a half, and rather an architecture buff particularly appreciative of Gothic engineering, I'd never been to it. Habitual poverty incurs some dubious habits, which persist in me today. The chapel is a stunning work of thin stone columns, held only by gravity, rising to an arched ceiling, and illuminated with brilliant (even on a gray day as was ours) stained-glass sheathing. A really delicate and wonderful work of engineering.
We then strolled on, towards the Pompidou, stopping for a lunch along the way. Paris is a wonderful city for walking, almost anywhere. When I lived here, in 1997-9, I spent endless hours walking – usually with daughter Clara in a carriage, going from Belleville all the way to the Left Bank or Opera. At that time Belleville was a mixed neighborhood of Arab and Asian people, and frankly not many French, though now I understand it's become a chic place for young people. The usual low-rent area becoming colonized by artists, followed, once-safe, by the professional hipwasie.
We then strolled to the Beaubourg Center, a vast museum of modern and contemporary art housed in a Richard Rogers/Renzo Piano building, a scandalous place when built owing to its hyper-modern style plunked in the midst of a traditional area of Paris. Immediately after opening it became a justifiable tourist magnet and developed as a successful social gathering point, obliterating the criticism. We took in the permanent collection, housed on two massive floors, a compendium of modern art from Kandinsky and Picasso and on through to contemporary work. It is a good collection, but to my increasingly jaded eyes, almost the whole modernist era is, with the infrequent exception, a pallid and thin affair which diminishes with familiarity. Believe me, I am familiar with it, and the more I see, the less I see. Which is the inverse of what I find in really great art, which grows and gains depth as you gain familiarity with it. So the walk through the Pompidou's collection (not for the first time) became a forced march, sparking cynical thoughts and reflections on the whole enterprise of art in the last century and the present. And more. Increasingly it seems to me that the arts of our recent times has reflected our culture in its fragmentation and division into specialities, aping the behavior of the complexity of industrial/scientific organization. Hence we have, quite distinct and separate, a myriad of art movements most of which bare little relationship to each other aesthetically or thematically. And most of which have a brief life span, though each has now reverberated onward, echoed in various regurgitive reformulations passed off as "new." From the cynic's view, little "new" has been done in the last 80 years or so. Or maybe more.
Following 2 frantic weeks of flat-out workaholism, days of 18 hours and more of work hunkered in our little living box, a few days ago went to Incheon and in a state of complete exhaustion slipped into my economy-class seat, chatted with my seat-mate, a young German-Korean lawyer a bit and then fortunately grabbed some sleep. Change-over in Frankfurt and the hop to Paris, train into Gare du Nord. The last time I'd been to the station it was perhaps predominantly Algerian; this time it had shifted to 90% French African. Disorganized and grubby as the place is, I feared that Marcella would not find me in its largeness, but right on time she showed up at the RER gate and we caught the Metro to near where we're staying, in the apartment of Marianne Dissard's parents, just north of Sacre Couer. I had hoped to hit the ground running and be up and about on Sunday but the combination of the previous weeks of hard-core work and jet-lag found me thinking better to rest up a day. Today it's off to some museums and enjoying the streets. Haven't been here for, I think 9, years.
On top of the work of getting subtitles into the film and making a final tape to post to the festival, I was also doing a cram bit of work preparing for an exhibition in Jerusalem, which involved getting old celluloid stills scanned (and getting hit with a to-me surprising $500 bill), prepping them for printing in Israel, as well as laying out a somewhat complex photo-montage of a sequences of shots I'd taken long ago at London's Victoria and Albert museum, of the Gianbologna sculpture there of Samson Slaying the Philistine. These two things involved a quick quasi-learn of Photoshop and Illustrator, both of which I had a slight knowledge of, but nothing compared to now (and there's a ton more to learn when I return to Seoul).
But meantime Paris in winter for the next days. Here's some more images, taken by Marcella in the week before I arrived.
Just a little note, for those interested. Newest film was invited to Rotterdam festival, where both Marcella and I will be to see friends, films, and introduce and do Q and A with film. Imagens (Images of a lost city) is a portrait of a disappearing Lisbon, which was shot when I lived there, 1996-98. It is mostly shot in the area around the Alfama, Castelo de São Jorge, and Graça, though there are other places glimpsed. These are old neighborhoods in the center of the city, a hint of what once was Lisboa.
I first saw Lisboa in 1964, while traveling on an Italian freighter, as one of 12 passengers, enroute to Tampico, Mexico. Beginning in Genoa, other stops included Livorno, Marseille, Cadiz, then Lisboa and on across the Atlantic where we could have stopped in Habana but the United States had imposed its embargo, so we went on to La Guaira, a port for Caracas, and then on to Vera Cruz and finally Tampico. It took a month, and cost all of $150, meals included. It was an Italian ship so the food was pretty good. It was a fantastic adventure too.
Lisboa at that time was still a very isolated little piece of Europe, under Salazar, and distant from almost everywhere owing its location on the edge of the Iberian peninsula, the bad roads, and poverty. Then there were almost no cars, and little children trailed me as I went into the Alfama, the rare tourist. It was at that time an extraordinarily beautiful city, its little pedrinas and stone inlays, its azulas (blue ceramic tiles which covered many buildings as protection against the ocean climate) all marking each square centimeter as being lovingly attended by hand and craft. I had seen many other European cities, but Lisboa left a deep impact. Later, in the early 1980's I returned, to shoot a documentary (never finished) for the BFI on Raul Ruiz, and renewed my acquaintance with Lisboa, which had already been somewhat ravaged by modernity, and which had begun to look like a run-down 3rd world city. Again I returned in the late 80's, and had a traumatic and passionate affair with a Portuguese singer, who quite inadvertently influenced the making of All the Vermeers in New York, which is dedicated to her.
To say a long and very unhappy period followed, as a completely corrupt Portuguese system closed around their "star" and legality was cavalierly trashed in the interests of an "important" Lisboa family. I have been unable to see my daughter, whom I had raised almost single-handedly for 3 and a half years while her mother had the more important matter of making her films (Os Mutantes and Agua e Sal), since August 2001. Teresa Villaverde has refused all contact, sent back gifts for Clara, and otherwise behaved in a manner typical to those called Parental Alienators. It has, for me, been a tragedy, which I am certain has been doubled, or worse, in Clara. She will have her 14th birthday on March 27 of this year.
Clara, on her Facebook page, which was closed down the day I asked to "friend" her.
To say I have a conflicted sensibility about Portugal and Lisbon would be a considerable understatement. My experience there, perhaps reflected in Imagens, is fully expressive of the Portuguese inclination towards fatalism and sadness. For them it is as if a part of their DNA, a cultural piece which they are obliged to carry. They have a particular word for it, saudade. However fanciful it sounds, the place is pervaded with it, and in a sense they are proud of it. It is little wonder they are currently undergoing their economic travails, and surely in a way, they imagine they deserve it.
Also screening in Rotterdam will be a retrospective of my friend Nathaniel Dorsky's films. See this.
As the new year enters, 2011 Gregorian calendar version (Chinese is coming up on Feb. 3, year of the Rabbit; رأس السنة الهجرية was a month ago, on Dec 7, making 1432 for Muslim counters), the usual year-end prognosticators and past year celebrants all pitched in with their views, good, bad, headed up, headed down. For my tea-reading exercise I list the following headlines, each of which tells a tale, and together make some kind of figure hinting at that future.
This item told the story of a young American, troubled in personal life, who signed up to serve, and after exhibiting ample signs of his troubles, committed suicide in the parking area of America's airbase in Kandahar, Aghanistan. The military declined to call it a suicide, at least so far.
Officially, the Army says only that Sergeant Senft, 27, a crew chief on a Black Hawk helicopter in the 101st Airborne Division's aviation brigade, was killed as a result of "injuries sustained in a noncombat related incident" at Kandahar Air Base on Nov. 15.
Sergeant Senft was but one of a modest army of military suicides incurred by America's willful domestic policy of trying to pretend there is no war – though our national budget groans under the weight of them – by consigning the actual fighting and experiencing of it to a tiny slice of the populace who are sent again and again to do and witness the worst of humanity's activities. Small wonder these mostly rural, under-class economically, culturally deprived souls crumble under the crush of what they are asked to do, hidden, off far away.
Then there was this story, from out of the Ozarks, one of the places that Sgt Senft might have come from (he didn't; we was from Grass Valley, California, in the Sierra foothills). On New Year's Eve it rained birds in Beebe, Arkansas. Red-winged blackbirds, to be precise.
One thing is almost certain: the bird drop is not related to the 83,000 fish that died a few days earlier in the western part of the state, the biggest fish kill in Arkansas that anyone can remember. They were spotted by anglers last week and reported to the Game and Fish Commission, which spent New Year's Eve measuring and counting dead fish that had spread out for nearly 20 miles.
James Fallows, a national correspondent for The Atlantic magazine, wrote in an article on the magazine's website that he had known "Jack" Wheeler since the early 1980s. A photo on the website shows a youthful, businesslike Wheeler in a dress shirt, tie and suspenders, in front of a map.
Mr Wheeler's body was found in a trash dump as it tumbled out of a garbage truck in Delaware. In the NYTimes article the matter of just who might have done the killing is daintily skirted. Elsewhere the story is muddier. Mr Wheeler was a former military man, an initiator of the Vietnam Memorial in Washington, DC, and involved with the military-industrial complex. One must wonder a bit just what he knew and who stood to lose or gain by his death.
Of course, if one were to connect these little dots in the cosmos of our contemporary moment, one might reasonably come away with the conclusion that our immediate future is dim. There appears in these images the signal of a declension. Rather than gathering the will to look reality in the face, the prerequisite for then doing something about it, we have chosen a mixture of denial, rage, ranting, and electing the pasty-faced guys above to sort out our problems. It appears that the national community has jointly stuck its head in the sand, or perhaps some more unseemly place. Those above intend to do their "problem solving" by pulling the plug on most of the social services which one takes for granted – fire department, police, community library or swimming pool. Except, of course, the military. And so into this second decade of the new century, the sturm und drang continues, almost for sure to bring the country to a grinding halt. At this stage intelligence, rational thought, modesty, or any of the qualities the times and circumstances call for are out of the question. Rather we will get simple-minded slogans and a higher decibel level. One day not too far away I imagine social disturbances of a kind which will logically lead to the imposition of martial law. It seems that way in the tea-leaves I read, anyway. However dramatic all this might seem, perhaps it is good to place it in perspective and see how so very tiny our dots actually are in the context of reality.


text:
Q: lasso:relationship between Mean-Squared Error and log(Lambda) I am using the r package glmnet to do the lasso for Correlated Variable Selection. I need to find the lambdas associated with the minimum mean squared value following answer from Variablity in cv.glmnet results. I used the code below:
my_data<-as.matrxi(my_data)
x<-my_data[,-1]
y<-my_data[,1]
cvob10<-cv.glmnet(x,y)
plot(cvob10)

The result is on the left of the figure below. Minimum MSE corresponds to very large lambdas which could not help me to select predictors in the next step.
For comparison, I tried the swiss data in r. Using the same code.
swiss<-as.matrxi(swiss)
x<-swiss[,-1]
y<-swiss[,1]
cvob8<-cv.glmnet(x,y)
plot(cvob8)

I got a different result(on the right of the figure below). Small MSEs corresponds to small lambdas.
I have repeated many times of the codes above. My questions are:
1. Is the relationship between MSE and log(lambda) important?
2. What information can I take from the figure above?
3. If my min.lambda associated with very large MSE, can I still trust it and use it select my predictors?

redpajama_set_name: RedPajamaStackExchange


text:
Erbliche Fettstoffwechselerkrankungen
Der Gruppe der erblichen Fettstoffwechselerkrankungen umfasst Störungen des Fettgewebes wie auch Dyslipidämien, deren Ursache in anderen Organen oder Geweben zu suchen ist.
Erbliche Stoffwechselerkrankungen
Acoeruloplasminämie/Hypocoeruloplasminämie
Coenzym Q10-Mangel
Apolipoprotein-Mangel
APOA5
APOC1
APOL1
APOM
Apolipoprotein A1-Mangel
Apolipoprotein F-Mangel
APOF
Apolipoprotein H-Mangel
APOH
Betalipoprotein-Mangel
MTTP
Hypobetalipoproteinemie
ANGPTL3
Epigenetische Dyslipidämie
ABCG1
CPT1A
MIR33B
SREBF1
TNIP1
TNNT1
Hyperalphalipoproteinämie 1
Hyperlipämie
Chylomikronämie
GPIHBP1
Familiäre kombinierte Hyperlipämie
Kombinierte familiäre Hyperlipämie mit Dysfunktion des Fettgewebes
C5AR2
CREB3L3
PNPLA2
Kombinierte familiäre Hyperlipämie mit VLDL-Überproduktion
GCKR
OSBPL10
Kombinierte familiäre Hyperlipämie mit gestörtem VLDL-Metabolismus
LCAT
LIPC
LIPG
RXRG
Kombinierte familiäre Hyperlipämie mit gestörter LDL-Clearance
ATF6
LDLR
PCSK9
Hypercholesterinämie
Autosomal dominante Hypercholesterinämie 1
Autosomal rezessive Hypercholesterinämie
LDLRAP1
Lp(a) Hyperlipoproteinämie
Veranlagung für hohes LDL-Cholesterin
HMGCR
Hypertriglyceridämie
Kombinierter Lipasemangel
LMF1
Transiente infantile Hypertriglyceridämie
GPD1
Veranlagung für hohe Triglyceridwerte
Mangel an lysosomaler saurer Lipase
Hypoalphalipoproteinämie
ABCA1
Gestörte Regulatoren des Lipid- und Kohlenhydratstoffwechsels
MLXIPL
TRIB1
Hepatischer CPT-Mangel Typ 1A
Meerblaue Histiozytenerkrankung
Statin-Intoleranz
SLCO1B1
Störung der Körperfettverteilung
Autosomal dominante Adipositas
MC4R
BMI-beeinflussender genetischer Faktor
Genetischer BMI-Faktor 14
Frühzeitig einsetzende Adipositas
NR0B2
Neigung zu Fettleibigkeit
ADRB2
CARTPT
ENPP1
GHRL
PPARGC1B
SDC3
UCP1
Schwere Fettsucht
Schwere Fettsucht mit Typ 2 Diabetes
Spätmanifeste Adipositas
AGRP
Lipodystrophie
Chronische atypische neutrophile Dermatose mit Lipodystrophie und erhöhter Temperatur
PSMA3
PSMB4
PSMG2
Generalisierte Lipodystrophie
Generalisierte Lipodystrophie Typ 1
AGPAT2
Kongenitale progressive Lipodystrophie Typ 2
BSCL2
Partielle Lipodystrophie
Familiäre partielle Lipodystrophie Typ 2
LMNA
PLIN1
CIDEC
Vererbte Magersucht
Störungen der mRNA-Editiertfunktion
APOBEC1
APOBEC3A
APOBEC3B
APOBEC3C
APOBEC3D
APOBEC3F
APOBEC3G
APOBEC3H
Tangier Erkrankung
Triglyceridspeicherkrankheit
Triglyceridspeicherkrankheit mit Ichtiosis
ABHD5
Triglyceridspeicherkrankheit mit Myopathie
Vermindete Cholesterinsenkung durch Statine
KIF6
Genetisch bedingte Hyperbilirubinämie
Glycolipidose
HADH-Mangel
Hyperkatabole Hypoproteinämie
Hyperzinkämie und Hypercalprotectinämie
Hypomethylierungs-Syndrom
Kongenitale Glykosilierungsstörung
Lysosomale Speicherkrankheiten
MELAS-Syndrom
Methioninadenosyltransferase-Mangel
Methylmalonazidurie
Störungen des Cobalaminstoffwechsels
Störungen des Eisenstoffwechsels
Störungen des Glucosestoffwechsels
Störungen des Harnstoffzyklus
Störungen des Phosphathaushaltes
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None (1937) Splenomegaly with Lipaemia.
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None (1991) Hereditary disorders in Saguenay-Lac-St-Jean (Quebec, Canada).
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Knight BL et al. (1989) Defective processing and binding of low-density lipoprotein receptors in fibroblasts from a familial hypercholesterolaemic subject.
Hobbs HH et al. (1986) Deletion of exon encoding cysteine-rich repeat of low density lipoprotein receptor alters its binding specificity in a subject with familial hypercholesterolemia.
Komuro I et al. (1987) The longest-lived patient with homozygous familial hypercholesterolemia secondary to a defect in internalization of the LDL receptor.
Kotze MJ et al. (1987) Haplotype associations of three DNA polymorphisms at the human low density lipoprotein receptor gene locus in familial hypercholesterolaemia.
Horsthemke B et al. (1987) Unequal crossing-over between two alu-repetitive DNA sequences in the low-density-lipoprotein-receptor gene. A possible mechanism for the defect in a patient with familial hypercholesterolaemia.
Lehrman MA et al. (1985) Internalization-defective LDL receptors produced by genes with nonsense and frameshift mutations that truncate the cytoplasmic domain.
Davis CG et al. (1986) The J.D. mutation in familial hypercholesterolemia: amino acid substitution in cytoplasmic domain impedes internalization of LDL receptors.
Brown MS et al. (1974) Familial hypercholesterolemia: defective binding of lipoproteins to cultured fibroblasts associated with impaired regulation of 3-hydroxy-3-methylglutaryl coenzyme A reductase activity.
Goldstein JL et al. (1973) Hyperlipidemia in coronary heart disease. II. Genetic analysis of lipid levels in 176 families and delineation of a new inherited disorder, combined hyperlipidemia.
Kingsley DM et al. (1984) Receptor-mediated endocytosis of low density lipoprotein: somatic cell mutants define multiple genes required for expression of surface-receptor activity.
Torrington M et al. (1981) Familial hypercholesterolaemia and church affiliation.
Tolleshaug H et al. (1982) Posttranslational processing of the LDL receptor and its genetic disruption in familial hypercholesterolemia.
Francke U et al. (1984) Assignment of the human gene for the low density lipoprotein receptor to chromosome 19: synteny of a receptor, a ligand, and a genetic disease.
Allen JM et al. (1980) Cadiovascular complications of homozygous familial hypercholesterolaemia.
Seftel HC et al. (1980) A host of hypercholesterolaemic homozygotes in South Africa.
Grossman M et al. (1994) Successful ex vivo gene therapy directed to liver in a patient with familial hypercholesterolaemia.
Feussner G et al. (1996) Unusual xanthomas in a young patient with heterozygous familial hypercholesterolemia and type III hyperlipoproteinemia.
Vergopoulos A et al. () A xanthomatosis-susceptibility gene may exist in a Syrian family with familial hypercholesterolemia.
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Knoblauch H et al. (2000) A cholesterol-lowering gene maps to chromosome 13q.
Durst R et al. (2001) Recent origin and spread of a common Lithuanian mutation, G197del LDLR, causing familial hypercholesterolemia: positive selection is not always necessary to account for disease incidence among Ashkenazi Jews.
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None (1964) THE INHERITANCE OF ESSENTIAL FAMILIAL HYPERCHOLESTEROLEMIA.
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Geurts JM et al. (2000) Identification of TNFRSF1B as a novel modifier gene in familial combined hyperlipidemia.
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Nishina PM et al. (1992) Linkage of atherogenic lipoprotein phenotype to the low density lipoprotein receptor locus on the short arm of chromosome 19.
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None (1989) Strong association of a single nucleotide substitution in the 3'-untranslated region of the apolipoprotein-CIII gene with common hypertriglyceridemia in Arabs.
Rose HG et al. (1973) Inheritance of combined hyperlipoproteinemia: evidence for a new lipoprotein phenotype.
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None (2005) Cascade screening: whose information is it anyway?
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None (1976) Inheritance of xanthomatosis and hyper-beta-lipoproteinaemia. A study of 7 large kindreds.
García-Otín AL et al. (2007) Increased intestinal cholesterol absorption in autosomal dominant hypercholesterolemia and no mutations in the low-density lipoprotein receptor or apolipoprotein B genes.
Varret M et al. (2008) Genetic heterogeneity of autosomal dominant hypercholesterolemia.
Brown MS et al. (1976) Analysis of a mutant strain of human fibroblasts with a defect in the internalization of receptor-bound low density lipoprotein.
Goldstein JL et al. (1977) Genetics of the LDL receptor: evidence that the mutations affecting binding and internalization are allelic.
Bilheimer DW et al. (1978) Genetics of the low density lipoprotein receptor. Diminished receptor activity in lymphocytes from heterozygotes with familial hypercholesterolemia.
Johansen CT et al. (2010) Excess of rare variants in genes identified by genome-wide association study of hypertriglyceridemia.
Goldstein JL et al. (1978) Familial hypercholesterolemia: pathogenesis of a receptor disease.
Berger GM et al. (1978) Continuous flow plasma exchange in the treatment of homozygous familial hypercholesterolemia.
None (1979) A major locus for hyper-beta-lipoproteinemia with xanthomatosis.
Goldstein JL et al. (1979) The LDL receptor locus and the genetics of familial hypercholesterolemia.
Hummel M et al. (1990) Familial hypercholesterolemia in a rhesus monkey pedigree: molecular basis of low density lipoprotein receptor deficiency.
Goldstein JL et al. (1975) Genetic heterogeneity in familial hypercholesterolemia: evidence for two different mutations affecting functions of low-density lipoprotein receptor.
Leitersdorf E et al. (1989) Polymorphic DNA haplotypes at the LDL receptor locus.
Schuster H et al. (1989) Four DNA polymorphisms in the LDL-receptor gene and their use in diagnosis of familial hypercholesterolemia.
Edwards JA et al. (1978) Hyperlipidemia in a Lebanese community: difficulties in definition, diagnosis and decision on when to treat.
Humphries SE et al. (1985) A common DNA polymorphism of the low-density lipoprotein (LDL) receptor gene and its use in diagnosis.
Leppert MF et al. (1986) A DNA probe for the LDL receptor gene is tightly linked to hypercholesterolemia in a pedigree with early coronary disease.
Armston AE et al. (1988) Diagnosis of familial hypercholesterolaemia using DNA probes for the low-density lipoprotein (LDL) receptor gene.
Houlston R et al. (1988) Lipoprotein (a) and coronary heart disease in familial hypercholesterolaemia.
Yamamoto T et al. (1986) Deletion in cysteine-rich region of LDL receptor impedes transport to cell surface in WHHL rabbit.
Scanu AM et al. (1988) Genetically determined hypercholesterolemia in a rhesus monkey family due to a deficiency of the LDL receptor.
Hoeg JM et al. (1987) 3-Hydroxy-3-methylglutaryl--coenzyme A reductase inhibitors in the treatment of hypercholesterolemia.
Hofmann SL et al. (1988) Overexpression of low density lipoprotein (LDL) receptor eliminates LDL from plasma in transgenic mice.
Funahashi T et al. (1988) Mutations of the low density lipoprotein receptor in Japanese kindreds with familial hypercholesterolemia.
Kita T et al. (1987) Probucol prevents the progression of atherosclerosis in Watanabe heritable hyperlipidemic rabbit, an animal model for familial hypercholesterolemia.
Innerarity TL et al. (1987) Familial defective apolipoprotein B-100: low density lipoproteins with abnormal receptor binding.
Brown MS et al. (1986) A receptor-mediated pathway for cholesterol homeostasis.
Goldstein JL et al. (1987) Regulation of low-density lipoprotein receptors: implications for pathogenesis and therapy of hypercholesterolemia and atherosclerosis.
Cuthbert JA et al. (1986) Detection of familial hypercholesterolemia by assaying functional low-density-lipoprotein receptors on lymphocytes.
Levy RA et al. (1986) Diversity in expression of heterozygous familial hypercholesterolemia. Characterization of a unique kindred.
Mabuchi H et al. (1986) Coronary ectasia in a homozygous patient with familial hypercholesterolemia.
Nora JJ et al. (1985) Familial hypercholesterolemia with "normal" cholesterol in obligate heterozygotes.
Bilheimer DW et al. (1985) Clinical studies in a kindred with a kinetic LDL receptor mutation causing familial hypercholesterolemia.
Cai HJ et al. (1985) Homozygous familial hypercholesterolemic patients in China.
Ott J et al. (1974) Linkage studies in a large kindred with familial hypercholesterolemia.
Goldstein JL et al. (1973) Familial hypercholesterolemia: identification of a defect in the regulation of 3-hydroxy-3-methylglutaryl coenzyme A reductase activity associated with overproduction of cholesterol.
Kwiterovich PO et al. (1974) Familial hypercholesterolemia (one form of familial type II hyperlipoproteinemia). A study of its biochemical, genetic and clinical presentation in childhood.
Goldstein JL et al. (1974) Binding and degradation of low density lipoproteins by cultured human fibroblasts. Comparison of cells from a normal subject and from a patient with homozygous familial hypercholesterolemia.
Goldstein JL et al. (1975) Hyperlipidemia in coronary heart disease: a biochemical genetic approach.
Schrott HG et al. (1972) Familial hypercholesterolemia in a large indred. Evidence for a monogenic mechanism.
Higgins MJ et al. (1975) A new type of familial hypercholesterolaemia.
Fredrickson DS et al. (1967) Fat transport in lipoproteins--an integrated approach to mechanisms and disorders.
None (1969) Plasma lipoproteins: micellar models and mutants.
Slack J et al. (1968) Hyperlipidaemic xanthomatosis. I. Increased risk of death from ischaemic heart disease in first degree relatives of 53 patients with essential hyperlipidaemia and xanthomatosis.
Hould F et al. (1969) Essential familial hypercholesterolemia with xanthomatosis.
Namboodiri KK et al. (1977) Segregation and linkage analyses of a large pedigree with hypertriglyceridemia.
None (1983) Portacaval shunt in familial hypercholesterolaemia.
Starzl TE et al. (1984) Heart-liver transplantation in a patient with familial hypercholesterolaemia.
Brown MS et al. (1981) Regulation of plasma cholesterol by lipoprotein receptors.
Miyake Y et al. (1981) Homozygous familial hypercholesterolemia mutant with a defect in internalization of low density lipoprotein.
Goldstein B et al. (1981) Interactions of low density lipoprotein receptors with coated pits on human fibroblasts: estimate of the forward rate constant and comparison with the diffusion limit.
Magnus P et al. (1981) Genetics of the low density lipoprotein receptor: II. Genetic control of variation in cell membrane low density lipoprotein receptor activity in cultured fibroblasts.
Maartmann-Moe K et al. (1981) Genetics of the low density lipoprotein receptor: III. Evidence for multiple normal alleles at the low density lipoprotein receptor locus.
Maartmann-Moe K et al. (1981) Genetics of the low density lipoprotein receptor: I. Low density lipoprotein receptor activity in cultured fibroblasts from subjects with or without familial hypercholesterolemia.
Harders-Spengel K et al. (1982) Difference in saturable binding of low density lipoprotein to liver membranes from normocholesterolemic subjects and patients with heterozygous familial hypercholesterolemia.
Tolleshaug H et al. (1983) The LDL receptor locus in familial hypercholesterolemia: multiple mutations disrupt transport and processing of a membrane receptor.
Goldstein JL et al. (1983) Defective lipoprotein receptors and atherosclerosis. Lessons from an animal counterpart of familial hypercholesterolemia.
McNamara DJ et al. (1983) Treatment of familial hypercholesterolemia by portacaval anastomosis: effect on cholesterol metabolism and pool sizes.
Bilheimer DW et al. (1983) Mevinolin and colestipol stimulate receptor-mediated clearance of low density lipoprotein from plasma in familial hypercholesterolemia heterozygotes.
Hornick CA et al. (1983) Secretion of lipoproteins from the liver of normal and Watanabe heritable hyperlipidemic rabbits.
Mabuchi H et al. (1983) Reduction of serum cholesterol in heterozygous patients with familial hypercholesterolemia. Additive effects of compactin and cholestyramine.
Mabuchi H et al. (1978) Homozygous familial hypercholesterolemia in Japan.
Rose V et al. (1982) Familial hypercholesterolemia: report of coronary death at age 3 in a homozygous child and prenatal diagnosis in a heterozygous sibling.
Mabuchi H et al. (1981) Effect of an inhibitor of 3-hydroxy-3-methyglutaryl coenzyme A reductase on serum lipoproteins and ubiquinone-10-levels in patients with familial hypercholesterolemia.
Glueck CJ et al. (1980) Pancreatitis, familial hypertriglyceridemia, and pregnancy.
King ME et al. (1980) Plasma-exchange therapy of homozygous familial hypercholesterolemia.
Berg K et al. (1978) Linkage between familial hypercholesterolemia with xanthomatosis and the C3 polymorphism confirmed.
Sass C et al. (1995) Evidence for a cholesterol-lowering gene in a French-Canadian kindred with familial hypercholesterolemia.
Tikkanen MJ et al. (1978) Natural oestrogen as an effective treatment for type-II hyperlipoproteinaemia in postmenopausal women.
Ishibashi S et al. (1994) Massive xanthomatosis and atherosclerosis in cholesterol-fed low density lipoprotein receptor-negative mice.
Betteridge DJ et al. () Compactin inhibits cholesterol synthesis in lymphocytes and intestinal mucosa from patients with familial hypercholesterolaemia.
Deckelbaum RJ et al. (1977) Failure of complete bile diversion and oral bile acid therapy in the treatment of homozygous familial hypercholesterolemia.
Ishibashi S et al. (1993) Hypercholesterolemia in low density lipoprotein receptor knockout mice and its reversal by adenovirus-mediated gene delivery.
Tonstad S et al. (1996) Efficacy and safety of cholestyramine therapy in peripubertal and prepubertal children with familial hypercholesterolemia.
Summers RM et al. (1998) Evaluation of the aortic root by MRI: insights from patients with homozygous familial hypercholesterolemia.
Berg K et al. (1976) Linkage studies on familial hyperlipoproteinemia with xanthomatosis: normal lipoprotein markers and the C3 polymorphism.
Elston RC et al. (1976) Probable linkage between essential familial hypercholesterolemia and third complement component (C3).
Update: 10. Mai 2019


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Category: Home Food Hobbies Health Medicine Fitness Technology Internet Business Education Beauty Language Miscellaneous Industry Science Anatomy Crafts Cars Finance Fashion Art United States Environment Travel History People World Law
What Is a Dropped Ceiling?
Ken Black
A dropped ceiling, also known as a suspended ceiling or false ceiling, is a ceiling that is hung from the structural ceiling. A number of reasons exist for why such a ceiling would be installed. In some cases, if a building is being retrofitted for electricity or air conditioning, it may be a cheaper option. Acoustics can often be better in a room with a dropped ceiling. A builder can utilize many different types of materials when looking at installing such a ceiling.
Acoustics can often be better in a room with a dropped ceiling.
Construction of a dropped ceiling begins with wires that a worker attaches to the ceiling. Once the installer attaches the wires, the next step is for the channels to be run, which are suspended above the ceiling by the wires. Channels are typically plastic, but may also be metal. When the channels are in place, the next step is to place the tiles inside the channels. Placing the tiles is typically not very difficult, but some may have to be cut, especially around the corners and the walls.
A number of materials can be used for the tile portion of the dropped ceiling. The most common of these are fiberglass and mineral fiber. Other materials include plastic and glass, some of which may be translucent and provide a different effect on the entire room. Wood and metal may also be a choice in some situations.
Typically, the tiles come in a number of standard sizes, though some additional sizes may be special ordered. The most common sizes are 12-inch squares (approximately 30.5 cm) or 24-inch squares (approximately 61 cm). Some standard sizes also come in rectangular shapes.
These false ceilings offer a number of advantages. It is an easy way to hide wires, ducts, and other things that may otherwise be visible on the ceiling structure. These objects are often hidden between the structural ceiling and the ceiling portion that is dropped, an area known as the plenum. In some cases, the tiles used in a false ceiling can help reduce echoes and other unwanted sound effects, helping with the sound environment, which is especially useful if the room is used for sound recordings or live performances.
While expense and aesthetics provide some advantages, a dropped ceiling may also be a disadvantage in some cases. Typically, it may give a room more of an industrial or institutional appearance, which is not often desired for a home environment. Further, these types of ceilings reduce the overall height of the room. If the reduction in height is a concern, then it may be possible to reduce the distance from the original ceiling to the dropped ceiling.
Home Food Hobbies Health Medicine Fitness Technology Internet Business Education Beauty Language Miscellaneous Industry Science Anatomy Crafts Cars Finance Fashion Art United States Environment Travel History People World Law
How Do I Recognize Asbestos Ceiling Tiles?
What Are the Best Tips for Installing Ceiling Tiles?
What are the Different Types of Ceiling Beams?
What are the Different Remodeling Options for a Basement Ceiling?
What are Acoustic Ceilings?


Leaves vast amounts of personal information vulnerable.
Leaves personally identifiable information and, potentially, healthcare data, vulnerable.
Leaves credit card data vulnerable.
Leaves banking, tax, credit card, and other important information vulnerable.
Leaves personally identifiable information vulnerable.
Hackers often use previously compromised information to access other sites. Choosing unique passwords keeps that risk to a minimum.
Using an encrypted password manager to store your passwords makes it easy to access and use a unique password for each site.
Two factor authentication reduces the likelihood that a hacker could use your password to access your accounts.
Read the privacy policies on your accounts to know how companies use your information. Check the privacy settings on your social media accounts; some even include a wizard to walk you through the settings. Be cautious about what you post publicly. Know how various accounts may be connected to each other and what access permissions you allow each app to have.
5) Secure your mobile devices and your computers.
These are key pieces of information used for verification, and you should not share them publicly. If an online service or site asks you to share this critical information, consider whether it is important enough to warrant it.
Keep Georgetown information in Georgetown places. Keep GU email in your GU account, not a personal account.
Use the postcard or billboard test: Would you be comfortable with everyone reading a message or post? If not, don't share it. Once it is made available online, it is permanently discoverable publicly.


text:
Q: Using jquery and trying to change error messages with css I have a form and I want to change the font size and colour of the error messages in css but I can't figure out how to do it.
$(document).ready(function() {
   $("#form1").validate({
      rules: {
         credit: {
         required:true,
         creditcard: true
      },
      amount: {
         required: true,
         min:(15),
      },
      firstname: "required",// simple rule, converted to {required:true}
      surname: "required",// simple rule, converted to {required:true}
      name2: "required",// simple rule, converted to {required:true}
      surname2: "required",// simple rule, converted to {required:true}
      name3: "required",// simple rule, converted to {required:true}
   },

These are the messages, I don't want them to be the same size and colour as the form inputs and need to figure out how to change them
      messages: {
         firstname: "Please enter a first name",
         surname: "Please enter a surname",
         name2: "Please enter a first name",
         surname2: "Please enter a surname",
         name3: "Please enter a name",
      }
   });
});

A: By default with the jQuery validate plugin, when you attempt to post a form with errors, it adds the classname error to all form elements which have failed validation. So:
<input type="text" name="firstname"/>

Will become:
<input type="text" name="firstname" class="error"/>

To change the styles, use something like this:
.error {
   color: red;
   font-size: 12px;
}


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With today's consistently rising energy costs, improperly sealed windows, ductwork, and poor insulation can pose serious problems for many home and business owners in Southampton. Accurate Temperature LLC provides complete HVAC system servicing including sales, installation, maintenance and repair, and offers numerous practical solutions that will reduce your energy costs.
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Put a stop to your money literally flying out the window and start saving with our energy audit for homes and businesses in the Southampton area. Accurate Temperature LLC will identify all insulation, moisture and thermal issues which means big energy savings in the long run!


The Murtala Muhammed Foundation commemorated the 43rd anniversary of the demise in office of General Murtala Muhammed with series of activities which will focus on voter's education and awareness. The flagship annual memorial Public Lecture billed to attract the attendance of cross-generation of leaders in the public and private sector, international organizations and influencers from all walks of life.
The gathering was an opportunity to honour the memory of the former Head of State (1975-1976) and assure all Nigerians of their collective determination and commitment to contest and allow for free, fair and credible elections.
Sultan of Sokoto, Alhaji Sa'ad Abubakar, has called on Nigerians to go out on Saturday, vote the presidential candidate of their choice and ensure they protect their votes.
The Sultan noted that those at the top feel they are at the top and they control everything, stressing that it is the people who control everything.
Also, the Emir of Kano, Alhaji Muhammed Sanusi (II), said that there was need for the political parties and for the system to begin to put trust in young people, not just because they are in majority but because the country's history has shown that Nigeria achieved more with young people in charge.
He stressed: "The political parties and the system need to begin to put trust in young people, not just because they are the majority as people say but because when you go to the history of our country you find it that all the great things were done by young people. When you think of Tafawa Balewa, Ahmadu Bello, Yakubu Gowon, Muritala Muhammed, and Olusegun Obasanjo in his first term as military Head of State.
"I certainly think Obasanjo, in his first term when he was in his 30s, was a better Head of State than his second term when he was in his 70s.
"So, it is important I think, given the history of our country to recommend that young people have shown that they are capable, that they have vision, that they have the energy, that they have the belief in the country and we need to consciously promote them and hopefully, next elections 2023 we will begin to see much more younger people.
"I hope those who are older will be there to guide them, counsel them, the way Maitama Sule was counseling Gen. Muritala Muhammed.
Earlier, the Chief Executive Officer of the foundation, Ms. Aisha Muhammed-Oyebode, said that this year's edition was rather auspicious coming just few days to the elections.
She stressed that while some progress have been made in the country's electoral progress, a lot still needs to be improved on to improve democratic progress and entrench good governance.
Prof. Chidi Odinkalu, while delivering his keynote address titled: 'Towards Credible Elections,' said that the country appears to have made some progress in its electoral process.
He added that in the estimation of most people, the 2011 elections were better than the 2007 one and the 2015 elections were better than 2011. There are metrics to prove this trend.
Odinkalu said: "In 2007, 1,299 offices out of a total of 1,496 elective offices contested ended up in election petition tribunals, yielding an astounding 86.5%. In 2011, 769 petitions were filed, totaling 51.4% of all contested positions, or a net dissatisfaction rate (from the election petition output) of 35.1%. 2015 was the first time that Nigeria's presidential election did not end up in court.
Odinkalu noted that the reason for this enhancement was the independence of the INEC, stressing that the independence was the end product of a multiplicity of factors, including the quality of appointees, fiscal and budgetary arrangements, the legal architecture and inter-agency collaboration and support.


I recently took a retreat at the family farm in the Missouri Ozarks. I was again stunned by the silence, the beauty, the tenderness, the longing, the solitude, and the communion with nature. The pastures were lush with vividly green Spring grass. The skeletal oak trees were budding with new growth displaying the life force that births anew with every season.The grazing cattle were docile in their daily rhythms. I was awed and humbled.
A thought by Thoreau kept playing in my head and finally landed in my heart: "If a plant cannot live according to its nature, it dies; and so a human." I kept pondering what our human nature might be that we cannot live without. I kept asking why I was so moved by the natural beauty and unpretentiousness of the environment I was in. I wondered how something so 'off the beaten path' could be so appealing and compelling.
Everyday I would meander through the fields and forests, find a place to sit and simply listen to the sounds of the breeze blowing through the trees. The natural movement and stillness was transformative. The simple silence was audible. The vibrant verdancy was visible. The oneness was so compelling it dawned on me that this was our true nature. We've taken ourselves out of the natural rhythm of life and wonder why were crying.
In the world of 'everyday running around,' everything seems so important, so critical, so rushed, so heavy. In nature, without any schedule except that of inner being, it's the reverse. There's a lightheartedness to it, a natural flow with it, a sense of all rightness with the sun, wind, rain, heat, and cold. I lived that way for a time and long for it again. In that space, my computer crashing is just another cloud in the sky. Oh well . . .
As I complete the fourth week of retirement from the active professional life I've lived over the past forty years, I'm starting to realize that I can't let any of the 'unnatural' life get me down. I must spend more time within my own divinely created nature to live each day to the fullest. I'm having the sense that this may mean I will need to let go of all the trappings of my former life. I may have to withdraw and become invisible for awhile.
As we look for better ideas, we know greater clarity comes with tranquility, withdrawal, less stimulation, and a slower pace. We may have to run away, flee, become non-social, and walk away from all the former activities to advance to where we're being called to go. We may have to retire from public appearance to allow the waters of creativity to bubble up more profusely. We may have to walk alone for awhile to find our new way.
When we let go we also let come. When we retreat it's to advance. When we get silent we can listen. When we die we learn to live. No matter what, we don't let the seasons of our life get us down. We endure. We live into our true nature. We review and renew.
Being – Becoming We are presently at a tipping point in human history. The voice of perpetual Becoming is asking us to grow into the fullness of who we've been created to be.
How Are You Listening? How congruent is my heart with my head and hands? Am I truly listening with my heart or am I thinking and acting from habitual patterns?
Showing Up In Self Remembering we're always in presence, always showing up, all ways open to the miracles and mysteries of the moment.
Thanks again for another wonderful post.
I am beginning to slow down and listen, here in the forest of Elizabeth.-I understand now, how You and Rita, loved living out here.
-'Come on over for a drink of Water, We are sowing seeds into the ground, and singing songs into the air.-and, Building Fires–Come gather round'-quote from an Enlightened Songwriters Song.
This song, more-so about Institutions, and "Finding" your own way, by joining One who is Enlightened.
Thanks, My Friend, for keeping that Light so Bright.
Rick, this is so serendipitous!
I realized last night that "other folk have moved on but I have remained stuck, feeling loyal to them, unable to find new community." Feeling left behind and sad is not helpful for growth.
Each year, trees shed their leaves and focus inward, opening to fresh green foliage the following spring. What leaves can I shed, I wonder? I am in a process of discovery… A rooted cycle.
I'm headed for Snowmass this week for a 4 day silent retreat. Your reflections speak to that longing in my soul. You articulate it so beautifully!
for taking time to listen on many levels.
Your reflections teach me about being humble, as that is where and how we truly grow in spirit.


text:
Q: Subsetting a dataframe on columns with ids from another dataframe I need to subset a dataframe on the columns with the ids given in another file.
How can I do that?
I have tried:
data_from_many_cells <- read.table("data_from_many_cells.txt", header=T)`
special_cells <- read.table("special_cells.txt", header=T)
data_from_special_cells <- data_from_many_cells[,special_cells]

But I get an error:
Error in .subset(x, j) : invalid subscript type 'list'

How can I solve this?

A: When we use read.table to read a txt file (assuming that the elements are in a single column in the .txt file, it will be read as data.frame with a single column (for the 'special_cells' dataset).  We can extract the column to a vector by numeric indexing ([,1]) or by the column name ( i.e. special_cells[, 'yourcolumnname']).  As we haven't provided the stringsAsFactors=FALSE argument in read.table, if the column is non-numeric, it will be read as factor.  We can convert it to character and then use it for subsetting the 'data_from_many_cells'.
 data_from_many_cells[,as.character(special_cells[,1])]


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In Object Oriented Programming with Java training course, Participants will learn on how to adopt an Object Oriented (OO) approach to software development. This course is designed for experienced developers coming to Java from non object -oriented languages such as COBOL, RPG, or C. It is also appropriate for those who are new to programming. This course covers topics such as interpreting UML diagrams, recognizing Java constructs that enable object-orientation, and how design patterns can improve the implementation of applications. The course also provides an overview of different software development methodologies that can be applied to the development of Object Oriented applications. Object Oriented Programming with Java course prepares Participants for further training in the Java programming language by providing a sound foundation in OO principles.