text:
Questions about example sentences with, and the definition and usage of "Reciprocal"
The meaning of "Reciprocal" in various phrases and sentences
Q: O que significa reciprocal?
A: As an adjective, the word most likely means something that is done in return, whereas in a mathematical perspective it's the multiplicative inverse of a number. Hope I'm right though.
A: The opposite. For example: The reciprocal of 1/3 is 3/1.
Q: O que significa reciprocal (like in the sentence "the rate of sedimentation is proportional to the reciprocal of f)?
A: Reciprocal is the opposite of something like it you had a fraction 6/7 the reciprocal would be 7/6
Q: O que significa reciprocal linking?
A: I guess, A links to B and B links to A.
"Reciprocal" means something goes both ways. For example, if someone sends you a card you can reciprocate by sending them one.
Example sentences using "Reciprocal"
Q: Mostra-me frases de exemplo com reciprocal.
A: A reciprocal is a mathmatical expression, a reciprocal of a fraction is basically to flip the fraction so the reciprocal of 1/2 would be 2/1
Q: Mostra-me frases de exemplo com is "reciprocal" used in the everyday language ? .
A: souvent, on utilise le mot "reciprocal" quand on parle des mathématiques, comme "l'inverse de 4 est 1/4"
"the reciprocal of 4 is 1/4"
is this what you mean?
A: Yes, she likes him but he doesn't like her in return - > it isn't reciprocal 👍
A: The reciprocal of 10 is 1 divided by 10, which is 0.1
Countries which enter in reciprocal relationships need to establish balanced responsibilities and rights if they want to achieve a long-lasting relationship.
Synonyms of "Reciprocal" and their differences
Q: Qual é a diferença entre reciprocal e mutual ?
A: They're very similar in meaning (and probably interchangeable, for the most part, I think... I haven't thought this through all the way).
But there's a slight difference:
"reciprocal" implies doing something in return / exchange for something else. For instance, if you do this for me, I'll do that for you.
"mutual" doesn't necessarily have any conditions -- it's more of a two-way relationship based on a shared feeling or understanding or benefit.
A: Reciprocal is taking turns. Mutual is something you have in common.
A conversation is reciprocal, because you take turns talking and listening.
You and other having the same interests are "mutual interests"
Q: Qual é a diferença entre a reciprocal visit e a mutal visit ?
A: A reciprocal visit would mean someone came to visit you, so you later went to visit them. A mutual visit isn't a very commonly used phrase, but mutual means you both agree to something. A mutual happiness would mean you're both happy. So, a mutual visit would mean you both agree to a visit.
Q: Qual é a diferença entre reciprocal e interactive ?
A: Generally speaking, "reciprocal" describes a relationship where both parties are benefiting. "I brought my new neighbor some cake and he reciprocated by giving me some flowers from his garden."
"Interactive" describes a system that can be affected in someway by a person. "The displays at the museum were interactive. You could see a 3D model of a dinosaur skeleton and rotate it around by moving your hands across the screen."
Translations of "Reciprocal"
Q: Como é que se diz isto em Inglês (EUA)? how do you say reciprocal in English
mutal
奴
取り扱う
ismay
済ませる
括る
うっとうしい
振る
立つ
はじく
reciped
recipi
recipriocal
reciprocally
Reciprocamente
reciprocate
reciprocated
reciprocates
Words that start with "R"
See more Q&A about Reciprocal


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Q: Add row to table inside a loop error I had a table inside a loop, and I want to add a row to the table by clicking a button "add row". Here is my code
HTML:

    <input type="button" value="Cancel" onclick="closePopup()">
    <input type="button" value="ADD" onclick="addRow(this)"/>

@for (int i = 0; i < 3; i++) {

                Items
                Quantity
                Price

               Apple
               5
               100

               Organe
               5
               200

               Total

<input type="button" class="addRow" onclick="addRow(this)" value="ADD ROW"/>

}

then in javascript i wrote function:
function addRow(table) {
    $(table).find('tbody tr:last').clone().appendTo(table);
}

but it does not add a row. If I use the function without parameter "table", the new row will be added to all tables inside a loop.
My desired outcome is: When the user clicks the button a new row will be added only in the one table, not to all.

A: You need to use closest to find its parent table first:
function addRow(table) {
    $(table).closest('table')//find parent table
      .find('tbody tr:last')//find last tr of the table
      .clone()//clone it
      .appendTo($(table).closest('table').find('tbody'));//append it to tbody
  //append to 'tbody' not this row
}

As per your updated question, here you go:
function addRow(table) {
    $(table).parent()//find parent div
      .next()//get the table
      .find('tbody tr:last')//find last tr of the table
      .clone()//clone it
      .appendTo($(table).parent().next().find('tbody'));//append it to tbody
  //append to 'tbody' not this row
}

A: onclick="addRow(this)" on a td. this will represent the surrent td. Not the table. You may pass the id or some selector of the table or find the parent table in with your script.
You can use parents() to find the parent table.
function addRow(td) {
    var table = $(td).parents('table');
    var cloned = table.find('tbody tr:last').clone();
    table.find('tbody').append(cloned);
}

And also if you want all the events to work then use clone(true). Else you have to bind the events to the tr.

redpajama_set_name: RedPajamaStackExchange


Android N is making headlines all over the web with its amazing additions that include new features, upgrades, and improvements to the previous Android M.
Developers from all over the Android niche have been moving in swiftly to try this new OS and as it seems, a lot has been a success. Google surprised many by rolling out this Android N preview earlier than usual, but things are paying off as more and more developers sign into the program.
As noted, the current version is still in beta and as such, it is only meant for developers. Usually, beta versions are associated with bugs and errors and generally, most components are unfinished. In other cases, the features that have been showcased in beta versions have failed to make it to the final version. It has happened with Android Lollipop, it happened with Android Marshmallow and as it seems, history is going to repeat itself when the final version of Android N is here.
According to Google, the Android N preview will be receiving monthly updates, something that could suggest this coming Google I/O 2016 event will reveal a more polished version of the OS.
The current version of Android N developer preview has lots of goodies on the table. However, lots of them are only but competing for a spot in the final version. With this in mind, expect to see some of these features present and others missing when the final version is released later this year.
Of course, there is no certainty in these predictions, so, better take them with a grain of salt.
With iOS users already enjoying a similar feature, the confidence level is high in the Android camp that split-screen multitasking will be on board Android N when the final version is released. This is going to focus on the tablet niche, but there is no surety on whether the feature will be availed on smartphones. But given that Samsung has been offering this functionality on its phones for quite some time now, there is no better time to introduce native support for Android devices.
The Quick Settings in Android N has also received some tweaks, with the menu adding some extra tricks. For instance, swiping down from the top of the screen reveals the options for Wi-Fi, Bluetooth and Airplane mode. You can easily turn on and off these options from this menu. It gets even better as users can customize this menu by sliding their most-used toggle in a preferred slot.
Expect this to reach the final version of Android N, with Google expected to make it even better with the stable release.
The App Drawer has been subject to change with respect to the latest LG G5. The device comes with the app drawer missing, meaning that users will have to get a different launcher in case they need one. LG is not the only Android OEM getting rid of the app drawer and even though this feature is still available in Android N preview, there are chances that it may be deactivated in the final version of the OS. Google might give users the option to choose on whether to enable or disable it.
It has somehow been very hard to move back to the previous apps you've been using with the current Marshmallow. Google is making this easier with Android N, ensuring that users can easily jump from the current app into recently used apps without hassles. All you need is to tap on the Recents button and you'll be taken to the app you previously visited.
Given that multitasking is solidly making its way to Android N, chances are high that this setting will be retained in the final version.
As mentioned earlier, this article is based on opinion and as such, take everything with a grain of salt. With the Google I/O 2016 event expected in mid-May, we'll definitely know more on what's is coming and what's not coming together with Android N.


text:
Associations of genetic variants of endothelin with cardiovascular complications in patients with renal failure
Chih-Chin Kao1,2,
Shih-Ying Cheng3,4,
Mei-Yi Wu5,6,
Shu-Chen Chien3,4,7,
Hsing-Fang Lu4,8,
Yu-Wen Hsu4,9,
Yan-Feng Zhang10,
Mai-Szu Wu2,5,6 &
Wei-Chiao Chang4,11
BMC Nephrology volume 18, Article number: 291 (2017) Cite this article
Cardiovascular (CV) complications are the main cause of death in end-stage renal disease (ESRD) patients. The high CV risks are attributable to the additive effects of multiple factors. Endothelin (EDN) is a potent vasoconstrictor and plays a role in regulating vascular homeostasis. However, whether variants of the EDN gene are associated with risks of CV events is not known. We conducted a study to investigate associations of variants of the EDN gene with CV events in ESRD patients.
A cohort of 190 ESRD patients was recruited, and 19 tagged single-nucleotide polymorphisms within the EDN gene family were selected for genotyping through a TaqMan assay. Data on clinical characteristics and hospitalizations for CV events were collected. Associations of genetic variants of the EDN gene with CV events were analyzed.
In this cohort, 62% (n = 118) of patients were hospitalized for a CV event. The EDN1 rs4714384 (CC/TC vs. TT) polymorphism was associated with an increased risk of a CV event after multiple testing (p < 0.001). Further functional exploration showed that it was a quantitative trait locus which may significantly alter gene expression in the tibial artery.
EDN1 rs4714384 is very likely an important biomarker of CV events in ESRD patients.
End-stage renal disease (ESRD) patients have a high risk of mortality, and 50% of these deaths are from cardiovascular (CV) complications [1]. Sudden cardiac death is observed in half of those cases, which is much more than coronary artery disease (CAD) [2]. Left ventricular hypertrophy (LVH) is considered to be one of the culprit pathophysiological expressions. It may contribute to the excess risk of sudden cardiac death and indicates poor survival in ESRD patients [3]. Besides LVH, vascular disorders, including atherosclerosis and arteriosclerosis, also account for the high risk of CV complications [4]. The exact mechanisms responsible for the excess CV risk in ESRD patients are not well understood.
Traditional and non-traditional risks factors, such as hypertension, diabetes, dyslipidemia, anemia, uremia, chronic inflammation, oxidative stress, calcium-phosphate vascular calcification, and autonomic dysfunction, contribute to a proportion of the excess risk [5, 6]. However, combining these risk factors does not fully explain the excess risk in these patients [5, 7]. Imbalances in some humoral factors and regulatory systems may also account for the excess CV risk. Activation of the renin-angiotensin-aldosterone system, and imbalanced endothelin (EDN) and nitric oxide levels were reported [8]. Demuth et al. [9] reported that an increased plasma endothelin level was associated with LVH and arterial intima-media thickening, suggesting this humoral factor may be important in cardiovascular remodeling. Whether genetic variants of candidate genes are related to the risk of CV disease (CVD) in ESRD patients is not known.
The EDN peptide family includes three isoforms (ET1, ET2, and ET3), which are coded by different genes, located on chromosomes 6p24.1 (EDN1), 1p34 (EDN2), and 20q13.2 ~ 13.3 (EDN3) [10]. Of these isoforms, ET1 is predominant. ET1 was reported to be strongly correlated with CVD, since it has a predominant vasoconstriction effect and hypertensive effect [10]. It has myocardial hypertrophic effects, [11] and its expression in endothelial tissues may be associated with instability of atherosclerotic plaque [12]. In addition, Minami et al. [13] reported that the plasma ET1 level was correlated with asymptomatic lacunar infarct and carotid plaques. ET2 was reported to be necessary for normal ovulation [14] and is associated with breast tumor invasion [15]. ET3 was identified as being abundant in the intestines and pituitary/brain tissues, which indicates it may have functions in nervous and endocrine systems [16]. Associations of genetic variants of the EDN genes with CV complications in ESRD patients are not well known. Thus, we investigated genetic variants of the EDN genes and CV events. We hypothesized that common variants of the EDN genes are associated with increased risks of hospitalizations for CV events in ESRD patients.
This study recruited adult patients older than 18 years, who had received chronic dialysis for at least 3 months at Taipei Medical University Hospital. 90% (n = 171) of patients received hemodialysis and the remaining received peritoneal dialysis. Demographics and clinical data of all patients were collected, including dialysis vintage, smoking, the erythropoietin resistance index (ERI), hemoglobin, albumin, iron profiles, adequacy of dialysis (Kt/V), pre-existing CV comorbidities, and cause of ESRD. ERI was calculated by the average weekly erythropoietin dose per kg body weight per average hemoglobin, which indicated a patient's response to erythropoietin. After enrollment, patients were followed up until the development of hospitalization for a CV event. The length of time from study enrollment to the development of CV event was collected. We defined hospitalized "CV events" as including CAD, congestive heart failure (CHF), arrhythmia, aortic aneurysm, stroke, and peripheral arterial occlusive disease (PAOD). These outcomes were prespecified in our protocol. CAD was defined as patients who received coronary angiography and ≥75% stenosis of a major coronary artery was noted. Congestive heart failure was documented according to the clinical diagnostic criteria by cardiologist, either by an episode of pulmonary edema, systolic dysfunction by cardiac sonography or cardiomegaly. Arrhythmia was documented as newly onset of irregular heart beat by electrocardiogram. Aortic aneurysm was confirmed by aortic imaging via radiographic studies. Stroke was defined as focal neurologic symptoms with image evidences. Peripheral arterial occlusive disease was documented by symptoms of ischemic muscle pain and radiographic studies. This study was approved by the Institutional Review Board of Taipei Medical University (no. 201309026). Written informed consent was obtained from all patients.
These tagged SNPs were determined to have a minimum allele frequency of >1% in a Beijing Han Chinese population (https://www.ncbi.nlm.nih.gov/variation/news/NCBI_retiring_HapMap/). Genotyping was done using the TaqMan Allelic Discrimination Assay (Applied Biosystems, Foster City, CA). Polymerase chain reaction (PCR) was carried out with an ABI StepOnePlus Thermal Cycler (Applied Biosystems). The fluorescence from different probes was detected and analyzed via the System SDS software version 2.2.2 (Applied Biosystems).
R 3.2.0 was used for the statistical analyses. The Chi-squared test and Student's t-test were used for comparing demographic characteristics between groups as indicated. A multivariable logistic regression model was performed to control for possible confounding factors, including age, gender, smoking, diabetes, hypertension, pre-existing CV comorbidities, hemoglobin, albumin, ferritin, and the ERI. We analyzed the magnitude of the association between the different genotypes and hospitalization for a CV event through a likelihood ratio test. Odds ratios (OR) with 95% confidence intervals were determined. Multiple testing correction was carried out using the false discovery rate (FDR), and q-values of <0.05 were determined to indicate statistical significance.
SNP functional annotation
In order to evaluate the relationship between the SNPs and gene expression profiles, we queried the GTEx Portal (https://www.gtexportal.org/home/), which contain expression quantitative trait loci (eQTLs) across multiple tissues. The SNP function prediction web site (https://snpinfo.niehs.nih.gov/snpinfo/snpfunc.html), which provides a variety of possible downstream influence of variants, was also applied to identify potential impact of candidate SNPs.
In total, 19 tSNPs of the EDN family (EDN1: rs5370, rs2070699, rs2248580, rs4714384, and rs3087459; EDN2: rs2759257, rs11210278, rs11572340, and rs11572377; and EDN3: rs742650, rs260740, rs260741, rs6064764, rs197173, rs197174, rs882345, rs926632, rs3026575, and rs11570352) were genotyped (Fig. 1a-c ). The minor allele frequencies of these tSNPs were close to those of the reference population from the Taiwan Biobank database and HapMap project (Table 1).
a. Graphic view of the genotyped human EDN-1 gene. b. Graphic view of the genotyped human EDN-2 gene. c. Graphic view of the genotyped human EDN-3 gene
Table 1 Minor allele frequencies of selected tagged single-nucleotide polymorphisms (SNPs) of the endothelin (EDN) gene family
In total, 190 patients were recruited for this study. The length of follow-up for the entire cohort was 22.9 +/− 10.6 months. The mean age was 64 years, and 54% of them were male. Pre-existing CV events were found in 43 patients with more than 83% due to CHF (n = 16, 37%) and CAD (n = 20, 47%). After enrollment, there were 118 patients (62.1%) who experienced hospitalization for a CV event with a mean duration of 17.2 ± 8.8 months. Of these CV events, CAD and CHF remained the most commonly observed (Additional file 1: Table S1). We divided patients into study and control groups according to the development of CV events. Patients in the study group included more males, were older, and had more diabetes. Lower albumin, serum iPTH, and Kt/V levels were observed in study group patients (Table 2).
Table 2 Baseline characteristics of study patients according to cardiovascular disease (CVD)
After multivariable adjustment, we found that patients carrying EDN1 rs2248580 (AA/CA vs. CC), rs2070699 (GT/TT vs. GG), rs4714384 (CC/TC vs. TT) (Table 3), EDN2 rs11210278 (TC/CC vs. TT) (Table 4) were associated with an increased risk of being hospitalized for a CV event in the recessive models. All of the EDN1 variants remained statistically significant after multiple testing correction (q values of 0.013, 0.003, and <0.001). However, none of EDN3 SNPs showed significant association with the susceptibility of developing cardiovascular disease (Table 5). In addition, we conducted functional annotation for these SNPs via several publicly available databases. The results showed that EDN1 rs4714384 is an eQTL in the tibial artery tissue (p = 6.3*10−9) (Table 6). We further analyzed the association of EDN1 rs4714384 with each CV comorbidity, which showed non-significant findings (Additional file 1: Table S2).
Table 3 Association analysis of genetic polymorphisms of the EDN1 gene and cardiovascular disease susceptibility in end-stage renal disease patients
Table 6 Endothelin (EDN) gene family-related expression quantitative trait loci (eQTLs)
In this study, we systemically performed genotyping of the EDN gene family, and three variants in the EDN1 gene [rs2248580 (AA/CA vs. CC), rs2070699 (GT/TT vs., GG), and rs4714384 (CC/TC vs. TT)] were associated with an increased risk of a CV event. These tSNPs were not located in the exon region, and therefore were not correlated with protein-coding functions. These genetic variants may alter disease phenotypes through other pathways, such as non-coding RNA, transcriptional regulation, or alterations in splicing [17]. Although there is no eQTL evidence between rs4714384 and EDN1 being observed in the current database due to limited available tissue specific profile, we found that rs4714384 has impact on RN7SKP293 expression in the tibial artery. The RN7SKP293 is a pseudogene, belonging to the 7SK RNA class. 7SK RNA is found in a small nuclear ribonucleoprotein (snRNP) complex, which regulates the activity of positive transcription elongation factor b (P-TEFb) [18]. P-TEFb is a kind of cyclin-dependent kinase (Cdk) which controls the elongation phase of transcription by RNA polymerase II [19]. Cdk is a cell-cycle check point regulator, and one study showed Cdk9 may have transcriptional roles in cardiac hypertrophy and mitochondrial dysfunction [20]. Another study showed overexpression of Cdk2 may promote smaller, less differentiated cardiomyocytes which have increased response to pressure overload [21]. Cdk dysregulation may be related to LVH, which may result in future CHF. Endothelial cells are the main origin of ET-1 production. Endothelial dysfunction is an important pathophysiology in ESRD patients, and is strongly associated with a risk of atherosclerosis and consequent CV events [22]. Its manifestations represent a systemic pathogenic condition, which implies an inflammatory state, prothrombotic state, and impaired vasomotor and cellular proliferation in the vascular wall [23]. Imbalances of humoral factors, including nitric oxide, oxidative stress, chemokines, angiotensin II, and EDN-1, on vascular homeostasis may contribute to this condition [22]. In addition, Ganz et al. [24] reported that the peripheral artery endothelial function has rather better prognostic predictions of CV events than coronary artery endothelial function. Our findings suggest that genetic variants of EDN1 may alter the balance of the homeostasis of peripheral vascular regulation, and affect the susceptibility to CV comorbidities.
We further investigated EDN gene expressions among different tissues through GTEx. However, low expression levels of both EDN2 and EDN3 were noted in the heart and major vessel tissues [25]. By querying the SNP function prediction web site, we found that rs11210278 (caTAATCgag) is a potential binding site for GATA6, which is a transcription factor involved in hypertrophic cardiomyopathy (Additional file 1: Table S3). This finding may be correlated with our finding, since the C allele of the rs11210278 is a predominant binding site for GATA6; therefore, patients carrying this allele may confer an increased binding affinity of the GATA6 transcription factor and consequently increased risk of hypertrophic cardiomyopathy. Cardiomyopathy is an important pathophysiology of CV events in ESRD patients [5]. Although this allele was not statistically significant after multiple testing correction, further enlarging sample sizes and functional validation studies are warranted for confirmation.
Vargas-Alarcon et al. [26] reported that an EDN1 rs3087459 polymorphism (AA allele) was associated with an increased risk of developing acute coronary syndrome. However, two other studies did not find such an association of EDN1 rs3087459 with the risk of myocardial infarction or ventricular hypertrophy [27, 28]. In our study, we also did not find a statistically significant association of higher risk of CV events with the SNP rs3087459. Rankinen et al. [29] reported that an EDN1 (Glu106Glu) polymorphism had a risk of HTN in a Caucasian population. Another study showed that genetic polymorphisms of the EDN1 rs5370 T allele and rs2070699 G allele were associated with an increased risk of ischemic stroke, [30] which is contrary to our findings. We found that rs3087459 may alter the gene expression in transformed fibroblasts according to the results of eQTL database; however, the association of fibroblasts with CV events remained elusive. Comparing the above studies to ours, different outcome measurements were noted. The small sample size may also limit our observations.
Previous studies showed dialysis vintage is a risk factor for coronary artery calcification, which may reflect the major risk of CV events [31, 32]. Therefore, the increased risk of CV events occurred with a longer duration of dialysis, which may account for the increased events during our observation period. According to the USRDS 2012 annual data report, [33] the incidence of hospitalization for CV morbidity is 4.5–5 times per 100-patient-months. Our cohort showed a relative fewer hospitalization of CV events, as compared to US database. We have a better 5-year overall survival in dialysis patients. Compared to other countries, the CV-related morbidity and mortality are much lower in Taiwan [34]. It may reflect the different epidemiology of CV events among populations. In our study, being male, being elderly, having hypoalbuminemia, and having underlying diabetes mellitus were risk factors for CV events. Being male and elderly are well known risk factors for CVDs [35, 36]. Malnutrition-inflammation complex syndrome is frequently observed in dialysis patients and is associated with an increased risk of CVD. Protein energy wasting and low albumin levels are poor outcome indicators [37]. Diabetes patients exhibit increased insulin resistance and an inflammatory status [38]. A previous study by Chang et al. [39] reported that diabetes and ESRD synergistically contribute to an increased risk of CV events.
Lower PTH and Kt/V level were associated with increased risk of CV events. Some studies showed hyperparathyroidism was associated with increased risk of CV mortality and all-cause mortality in dialysis patients [40,41,42]. However, a meta-analysis showed no significant association between PTH level and non-fatal cardiovascular events or CV mortality [43]. The PTH level is pulsatile in character and highly sensitive to change in ionized calcium and calcitriol levels [44]. There may be selection bias in our small sample size cohort. Therefore the association between low PTH and risk of CV events may need further investigation. In renal failure patients, there are a lot of uremic toxins leading to inflammation and subsequently CV events. Better Kt/V is associated with less uremic toxins retention, which may be related to lower risk of CV events. Đurić PS et al. [45] reported longer dialysis duration was associated with lower CV comorbidities. The FHN (Frequent Hemodialysis Network) trial group [46] also found better composite outcomes of death or change in left ventricular mass in frequent hemodialysis group as compared to conventional hemodialysis. In this view, the patients who received better dialysis clearance may have lower risk of CV events.
As we know, there were several identified risk factors contributing to cardiovascular comorbidities in renal failure patients. However, the prevalence and severity of CV events in ESRD patients is disproportionate to the identified risk profiles [47]. In the current study, we found potential genetic risk alleles and this may further improve the prediction model for CV comorbidities in these patients.
Several limitations of this study should be noted. First, the sample size was not large, and thus the statistical power may be limited. Second, we did not check the plasma ET-1 level to prove associations of genetic variants with its expression, as it was reported to be associated with several phenotypes in previous studies [13]. Third, we did not find the association between the SNPs of EDN and pre-existing CV events. It may be due to different composition of pre-existing CV events and different dialysis stage of these patients.
We found three genetic variants of the EDN1 gene to be associated with increased risk of hospitalization for a CV event, and rs4714384 was responsible as an eQTL in the peripheral artery. It may influence the EDN1 gene expression and alter vascular homeostasis in the peripheral artery. A further validation study is required to confirm the roles of these polymorphisms in the risk of CV events.
Cdk:
Cyclin-dependent kinase
CHF:
EDN:
Endothelin
eQTL:
expression quantitative trait locus
ERI:
Erythropoietin resistance index
ESRD:
End stage renal disease
ET1:
Endothelin 1
FDR:
False discovery rate
GTEx:
Genotype-Tissue Expression
Kt/V:
Adequacy of dialysis
LVH:
Odds ratios
PAOD:
Peripheral arterial occlusive disease
P-TEFb:
Positive transcription elongation factor b
snRNP:
small nuclear ribonucleoprotein
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Liao HS, Kang PM, Nagashima H, Yamasaki N, Usheva A, Ding B, et al. Cardiac-specific overexpression of cyclin-dependent kinase 2 increases smaller mononuclear cardiomyocytes. Circ Res. 2001;88(4):443–50. Epub 2001/03/07
Dhaun N, Goddard J, Webb DJ. The endothelin system and its antagonism in chronic kidney disease. J Am Soc Nephrol. 2006;17(4):943–55. Epub 2006/03/17
Widlansky ME, Gokce N, Keaney JF Jr, Vita JA. The clinical implications of endothelial dysfunction. J Am Coll Cardiol. 2003;42(7):1149–60. Epub 2003/10/03
Ganz P, Vita JA. Testing endothelial vasomotor function: nitric oxide, a multipotent molecule. Circulation. 2003;108(17):2049–53. Epub 2003/10/29
Lonsdale J, Thomas J, Salvatore M, Phillips R, Lo E, Shad S, et al. The Genotype-Tissue Expression (GTEx) project. Nat Genet. 2013;45(6):580–5. Epub 2013/05/30
Vargas-Alarcon G, Vallejo M, Posadas-Romero C, Juarez-Rojas JG, Martinez-Rios MA, Pena-Duque MA, et al. The -974C>A (rs3087459) gene polymorphism in the endothelin gene (EDN1) is associated with risk of developing acute coronary syndrome in Mexican patients. Gene. 2014;542(2):258–62. Epub 2013/09/17
Castro MG, Rodriguez-Pascual F, Magan-Marchal N, Reguero JR, Alonso-Montes C, Moris C, et al. Screening of the endothelin1 gene (EDN1) in a cohort of patients with essential left ventricular hypertrophy. Ann Hum Genet. 2007;71(Pt 5):601–10. Epub 2007/03/06
Palacin M, Rodriguez-Pascual F, Reguero JR, Rodriguez I, Avanzas P, Lozano I, et al. Lack of association between endothelin-1 gene variants and myocardial infarction. J Atheroscler Thromb. 2009;16(4):388–95. Epub 2009/08/13
Rankinen T, Church T, Rice T, Markward N, Leon AS, Rao DC, et al. Effect of endothelin 1 genotype on blood pressure is dependent on physical activity or fitness levels. Hypertension. 2007;50(6):1120–5. Epub 2007/10/17
Zhang L, Sui R. Effect of SNP polymorphisms of EDN1, EDNRA, and EDNRB gene on ischemic stroke. Cell Biochem Biophys. 2014;70(1):233–9. Epub 2014/03/19
McCullough PA, Sandberg KR, Dumler F, Yanez JE. Determinants of coronary vascular calcification in patients with chronic kidney disease and end-stage renal disease: a systematic review. Journal of nephrology. 2004;17(2):205–15. Epub 2004/08/06
Rosas SE, Mensah K, Weinstein RB, Bellamy SL, Rader DJ. Coronary artery calcification in renal transplant recipients. Am J Transplant. 2005;5(8):1942–7. Epub 2005/07/06
2012 USRDS, annual data report (ADR). https://www.usrds.org/atlas12.aspx
Yang WC, Hwang SJ. Incidence, prevalence and mortality trends of dialysis end-stage renal disease in Taiwan from 1990 to 2001: the impact of national health insurance. Nephrol Dial Transplant. 2008;23(12):3977–82. Epub 2008/07/17
D'Agostino RB Sr, Grundy S, Sullivan LM, Wilson P. Validation of the Framingham coronary heart disease prediction scores: results of a multiple ethnic groups investigation. JAMA. 2001;286(2):180–7. Epub 2001/07/13
Wilson PW, D'Agostino RB, Levy D, Belanger AM, Silbershatz H, Kannel WB. Prediction of coronary heart disease using risk factor categories. Circulation. 1998;97(18):1837–47. Epub 1998/05/29
Kalantar-Zadeh K, Ikizler TA, Block G, Avram MM, Kopple JD. Malnutrition-inflammation complex syndrome in dialysis patients: causes and consequences. Am J Kidney Dis. 2003;42(5):864–81. Epub 2003/10/29
Cefalu WT. Inflammation, insulin resistance, and type 2 diabetes: back to the future? Diabetes. 2009;58(2):307–8. Epub 2009/01/28
Chang YT, Wu JL, Hsu CC, Wang JD, Sung JM. Diabetes and end-stage renal disease synergistically contribute to increased incidence of cardiovascular events: a nationwide follow-up study during 1998-2009. Diabetes Care. 2014;37(1):277–85. Epub 2013/08/08
Slinin Y, Foley RN, Collins AJ. Calcium, phosphorus, parathyroid hormone, and cardiovascular disease in hemodialysis patients: the USRDS waves 1, 3, and 4 study. J Am Soc Nephrol. 2005;16(6):1788–93. Epub 2005/04/09
Tentori F, Blayney MJ, Albert JM, Gillespie BW, Kerr PG, Bommer J, et al. Mortality risk for dialysis patients with different levels of serum calcium, phosphorus, and PTH: the Dialysis Outcomes and Practice Patterns Study (DOPPS). Am J Kidney Dis. 2008;52(3):519–30. Epub 2008/06/03
Kimata N, Albert JM, Akiba T, Yamazaki S, Kawaguchi T, Fukuhara S, et al. Association of mineral metabolism factors with all-cause and cardiovascular mortality in hemodialysis patients: the Japan dialysis outcomes and practice patterns study. Hemodial Int. 2007;11(3):340–8. Epub 2007/06/20
Palmer SC, Hayen A, Macaskill P, Pellegrini F, Craig JC, Elder GJ, et al. Serum levels of phosphorus, parathyroid hormone, and calcium and risks of death and cardiovascular disease in individuals with chronic kidney disease: a systematic review and meta-analysis. JAMA. 2011;305(11):1119–27. Epub 2011/03/17
Chiavistelli S, Giustina A, Mazziotti G. Parathyroid hormone pulsatility: physiological and clinical aspects. Bone research. 2015;3:14049. Epub 2015/08/15
Duric PS, Duric Z, Jankovic A, Tosic J, Popovic J, Dimkovic N. Influence of hemodialysis duration per week on parameters of dialysis adequacy and cardiovascular morbidity. Med Pregl. 2014;67(11–12):385–91. Epub 2015/02/14
Chertow GM, Levin NW, Beck GJ, Depner TA, Eggers PW, Gassman JJ, et al. In-center hemodialysis six times per week versus three times per week. N Engl J Med. 2010;363(24):2287–300. Epub 2010/11/26
Zoccali C, Mallamaci F, Tripepi G. Novel cardiovascular risk factors in end-stage renal disease. J Am Soc Nephrol. 2004;15(Suppl 1):S77–80. Epub 2003/12/20
C.C.K. is the guarantor of this work and, as such, had full access to all the data in the study and takes responsibility for the integrity of the data and the accuracy of the data analysis. All the authors of this work have read and approved the final version of the manuscript.
The study was supported by grants from the Ministry of Health and Welfare (MOHW104-TDU-B-212-113,001) and Taipei Medical University Hospital (103TMUH-NE-04).
All data related to this article are shown in the manuscript or available upon request from the corresponding authors.
Division of Nephrology, Department of Internal Medicine, Taipei Medical University Hospital, Taipei, Taiwan
Chih-Chin Kao
Graduate Institute of Clinical Medicine, College of Medicine, Taipei Medical University, Taipei, Taiwan
Chih-Chin Kao & Mai-Szu Wu
Department of Pharmacy, Taipei Medical University Hospital, Taipei, Taiwan
Shih-Ying Cheng & Shu-Chen Chien
Department of Clinical Pharmacy, School of Pharmacy, Taipei Medical University, Taipei, Taiwan
Shih-Ying Cheng, Shu-Chen Chien, Hsing-Fang Lu, Yu-Wen Hsu & Wei-Chiao Chang
Division of Nephrology, Department of Internal Medicine, Shuang-Ho Hospital, Taipei Medical University, New Taipei City, Taiwan
Mei-Yi Wu & Mai-Szu Wu
Department of Internal Medicine, School of Medicine, Taipei Medical University, Taipei, Taiwan
Clinical Research Center, Taipei Medical University Hospital, Taipei, Taiwan
Shu-Chen Chien
Department of Pharmacy, Taipei Medical University-Shuang Ho Hospital, Taipei, Taiwan
Hsing-Fang Lu
The Ph.D. Program for Translational Medicine, College of Medical Science and Technology, Taipei Medical University and Academia Sinica, Taipei, Taiwan
Yu-Wen Hsu
HudsonAlpha Institute for Biotechnology, Huntsville, AL, USA
Yan-Feng Zhang
Master Program for Clinical Pharmacogenomics and Pharmacoproteomics, School of Pharmacy, Taipei Medical University, Taipei, Taiwan
Wei-Chiao Chang
Shih-Ying Cheng
Mei-Yi Wu
Mai-Szu Wu
CCK: study concept and design; researched data; analysis and interpretation of data; drafting of the manuscript. SYC: the molecular genetic studies; researched data. MYW: researched data; analysis and interpretation of data. SCC: study concept and design; researched data. HFL: statistical analysis and interpretation of data. YWH: analysis and interpretation of data. YFZ: statistical analysis and interpretation of data. MSW: study concept and design; final approval of the version to be published. WCC: study concept and design; final approval of the version to be published.
Corresponding authors
Correspondence to Mai-Szu Wu or Wei-Chiao Chang.
This study was approved by the Institutional Review Board of Taipei Medical University (no. 201309026). Written informed consent was obtained from all patients.
Demographic characteristic of the study patients. Table S2. Association of EDN1_rs4714384 and cardiovascular disease in ESRD patients. Table S3. Potential function prediction of SNP in transcription factor binding site in EDN gene family. (DOCX 21 kb)
Kao, CC., Cheng, SY., Wu, MY. et al. Associations of genetic variants of endothelin with cardiovascular complications in patients with renal failure. BMC Nephrol 18, 291 (2017). https://doi.org/10.1186/s12882-017-0707-2
Renal failure

redpajama_set_name: RedPajamaCommonCrawl


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In addition to Euro Sports Brasil, 195 other teams from 39 different countries played in Boys 15. They were divided into 48 different groups, whereof Euro Sports Brasil could be found in Group 22 together with IF VP Uppsala, JFV Verden/Brunsbrock, Kulladals FF 1 and Prep Schools District Bravo.
Euro Sports Brasil continued to Play off B after reaching 5:th place in Group 22. In the playoff they made it to 1/64 Final, but lost it against Vänersborgs IF with 0-1. In the Final, FC Chambly won over Glumslövs FF and became the winner of Play off B in Boys 15.


Celebrity Website Design. Actress Ellen Gerstein Website Design by Adchix. We design and develop websites for all sorts of businesses. From local mom and pop to global organizations.
The Challenge: Create a responsive website for comedienne, actress, writer, and director Ellen Gerstein while maintaining the look of the original website as much as possible.
The Solution: We loved working on this WordPress site. It's not our design. I took a theme that wasn't responsive and duplicated it (somewhat) so the site would be modern and responsive and be able to stay updated with the latest software. AND I actually did this for another designer.
If you have a client with almost impossible design needs, we consider that a challenge.we love problem solving and yes – we're available for subcontracting work!
Visit Ellen Gerstein's live website.


Psytrance- many refer to it as the last pure genre of EDM culture. But what is it really? Vice's Noisy reporter Clive Martin recently took an in-depth look at the genre at a rave somewhere in the woods of the U.K.
7. It Makes You Feel Like "fuuuhhhhhh..."
Peaked your interest? Take view of Clive Martin's full report on Vice's "Big Night Out" below. All jokes aside, Clive sees the psytrance scene as "a breath of fresh air" in an often mundane club culture.
EDM Culture: Attention Everyone! We Finally Found Molly!


text:
Alexander Cummings Downey (February 6, 1889 – July 10, 1949) was an outfielder in Major League Baseball who played for the Brooklyn Superbas in its 1909 season. He attended Georgetown University.

External links

1889 births
1949 deaths
Major League Baseball outfielders
Baseball players from Indiana
Brooklyn Superbas players
Minor league baseball managers
Oklahoma City Mets players
Webb City Webfeet players
Oklahoma City Indians players
Austin Senators players
Beaumont Oilers players
Georgetown University alumni

redpajama_set_name: RedPajamaWikipedia


Because the improvements in both audio software and pc hardware plus the fact there were several factual errors in the original article I have taken some time and rewritten the entire article.
1.. I have a computer with a soundcard, can I record my songs on it?
2. I have a multi track recorder, how can I use it and my PC together?
3. What should I look for in a multi track audio card?
4. I have a standalone Hard Drive recorder and a boatload of effects (compressor, reverb, etc.) what would be gained by going to the computer and using a program such as Sound Forge over the effects and compression I have available to me at present?
5. Does the computer and/or software program take the place of my mixer and processors?
6. What hardware do I need?
8. How can I get demos of the programs you are talking about?
9. Who are you and how do you know this stuff?
1. I have a computer with a soundcard, can I record my songs on it?
Sure. How well depends upon several things. First, how fast is your computer? I recommend a Pentium class machine running at 450 mhz or better. Of course, faster is better. Also, you will need a large hard drive, wave files take a lot of space (10 meg for 1 minute of stereo). To connect your sound card with your sound source (mixer, recorder, etc.) you will need an adapter. Most consumer sound cards have 2 inputs, line in and mic in* and use stereo mini plugs. Normally, you will have RCA plugs from your sound source. Whirlwind, Radio Shack and others sell the adapters you need (1/4" mini plug to dual RCA jacks). Next, you will need a software program to use for recording. If you have Windows 95 you already have one, Sound Recorder, but I don't recommend it. You may also have a program that was included with your sound card. They can be very useful to start with, but most have basic or limited features. Instead I would download a program such as Goldwave or Cool Edit 96. Both are good programs, not just for recording but editing your sound files as well. An excellent retail program is Sound Forge by Sonic Foundry. There are many others, demo as many as you can and pick the one you feel the most comfortable with. Realize of course, that any recording is only as good as the weakest link in the chain. In this case it is probably the sound card. The most dramatic improvement in PC recording will come with a quality sound card (see Recommended Hardware Setups) .
*The mic in is essentially useless. Always use the line in when recording from an outside source.
If you want to use the PC to mix with you will need some more stuff. First you will need a multi track software package. One that I recommend is Quartz Studio. You may also want to look at Pro Tools which is available on our freebies page. Next, you will need to download your tracks to the computer. Using a stereo sound card transferring your tracks can be a problem, especially if you have a tape multitracker (cassette or reel to reel). If you have a digital deck it is a lot simpler, but you will still have synchronization problems. Trust me on this one ~grin~. The basic idea is to first record tracks 1 & 2, then rewind and do tracks 3 & 4. If you punch play on the tape deck and record on the computer at the same time you can get very close. A better solution is a multi track sound card. There are several good ones, the one I have used is the Wave/4 card by Gadget Labs. Unfortunately they have gone out of business. Another card I considered when I purchased the Wave/4 was the Dman 2044 by Midiman. Which also is not made anymore. It has been replaced by the Audiophile 2496 which looks like a very good card at a very good price. Compare features and price and get one that will fit your needs and pocketbook. And read the answer to the next question.
3. What should I look for in a stereo or multi track audio card?
The higher the better. A 16 bit resolution is the minimum you should consider, 24 is better. Also, Any card you buy should have at the minimum a 44.1 sampling rate. The newer cards have 96khz. For comparison the sampling rate for a cd is 16 bits at 44.1khz. Usually you will see this listed as resolution/sampling rate, look for the highest numbers.
Also reffered to as dynamic range it is a measure of how quiet the card is. Anything over 90db is good.
If a card does not have full duplex capability you will not be able to listen and record at the same time. That can make overdubs really hard. Some cards will offer 16 bit recording but playback at 8 bit while monitoring. This will work and will be less of a strain on a lower powered pc but the sound quality of the monitored sound won't be very good.
Will it work with my pc and software?
Perhaps the most important. It also depends upon the order you buy things. If you get the card first then you need to make sure you get compatible software. Almost all audio multitrack cards have a list of minimum requirements and the better ones have a list of audio editing programs the card has been tested with. And vice versa, if you get the software first you need to get a compatible sound card.
Inputs being the more important. For instance if you are recording drums you will need a mimimum of four inputs - kick, snare and two overheads. More inputs can also reduce the amount of cable switching you need to do. Outputs are less important, unless you wish to transfer tracks back to your stand alone multi track recorder. Digital ins and outs can also be handy but not required. Think of how you record and/or how you want to record. That should give you a good idea of what you need.
Not a requirement but a goodie. Unless you enjoy reaching around behind your pc and trying to find where to plug in everything.
An extra bit of polish. It's kind of like the difference between taking your car to a carwash or to a detail shop. The carwash will get the car shiny and clean but the detail shop adds that last little bit of sparkle. There are sound cards that will transfer the data from your Hard Drive recorder to the computer digitally. Once in the computer you can manipulate the file in ways you can't on the HD recorder. For instance, say the singer has some serious popping p's going on. You can zoom in on the offending phrase and massage the waveform until it sounds perfectly pleasant.
Yes and no. You will still need your mixer, but the computer and software programs can handle many of the processing chores. There is some debate as to whether PC based effects are as high a quality as stand alone units. You can use both and decide which you prefer. Also, you can of course use both. For instance if you are recording a drum you can use your current compressor to compress the kick while you record it to the pc. Then use a software compressor on the entire kit.
To use your PC as a mix down deck.
Recommended Minimum: Computer Pentium class 450 or better. Memory - 64 meg. Hard Drive - 20 GIG Sound Card - Sound Blaster Live, Turtle Beach Montego or equivalent. Backup - Tape, Jazz or Zip drive. Optimally a CD burner. This will get you started and the quality will be much better than a cassette. But to realize the full potential of your PC you will want to upgrade things. If you find yourself runnning out of hard drive space get a second hard drive, as fast as or faster than the one you have now. You may also want to get a better audio card, possibly a multi track card. And of course, you can never have enough RAM.
To use your PC as a multitrack recording and mixing station.
Recommended Minimum: Computer - Pentium class 450 or better Memory - 128 meg Hard Drives (2 recommended for serious audio work) - 15 gig for data and 20 Gig for audio Sound Card - Midiman Audiophile 2496* Backup - Tape, Jazz, Zip drive. Optimally a CD burner. Mix down - What else, a CD burner. *See the section on What card should I buy?
There is a boat load of stuff out there for multitrack recording on your PC. There are several complete packages, such as the Paris system from Ensonic. Be forewarned, you can spend some major dollars. I prefer to build things myself. I started with a generic 166 with 16 meg of ram, a 2.2 gig hard drive and a Sound Blaster Pro. I have since upgraded (piece by piece) to a 500 with 128 meg of ram, a 5.7 gig hard drive, a 15 gig hard drive, a Gadget Labs Wave/4 and a Mitsumi CD burner. The best thing about PC recording is the ability to upgrade. It's easier to convince the spousal unit you need $300 for a sound card than $3000+ for a new system .
7. Should I get a IDE or SCSI hard drive and or CD burner?
Yes. Just kidding. You can record very well with the speed of today's UDMA hard drives. I would recommend either (SCSI or IDE). What I wouldn't recommend is to mix and match. While you can do it, it adds unnecessary complications. Whichever you go with stick with it. If you have an IDE hard drive I would get an IDE CD burner or extra hard drive. And vice versa.
Right now your best buy is the IDE drives. The newer IDE drives with a 7200 spindle speed are more than adequate. Add a large buffer or cache and you will be able to record more tracks without stuttering. Also look at the Sustained Data Transfer Rate (SDTR), the higher the number the better.
for more information on Quartz check out our freebies page.
I guess I'm a computer geek. I don't know how it happened for sure. Way back in 1983 I was a normal guy driving a fork truck in a local factory. Then one day the factory closed and I needed a job in the middle of a recession. I took a test at the local job service and they said, "hey you would do good in computers". They sent me to school to learn COBOL programming and I was hooked.
Since then I have worked with computers in various ways, programming, teaching, administration. I even installed an IBM system 36 (which still gives me nightmares). And I am constantly tinkering with my home pc, both the software and hardware.
I am still learning recording. In my youth I was in several different bands, rock and folk. The first recorder I bought was a Radio Shack cassette. Then I got an Akai reel to reel which right now is sitting on a shelf about 2 feet to my left. When my kids were born I quit my low down ways (playing in bars till the wee hours) and concentrated on raising a family. I must have done something right cuz about 5 years ago my son started a band of his own. Of course they wanted to be recorded and old dad could do it, right! So I called an old band mate and he brought over a Tascam PortaStudio and I was hooked. I bought a Yamaha MiniDisc (the word 'digital' hooked me). Unhappy with the mixes I was getting I tried importing tracks into the computer and manipulating them with the sound editor that came with my sound card.
My obsession has progressed from there. I am constantly reading and tinkering. I don't think my computer has had a cover on in months. If it is true you learn by your mistakes I must be a genius . I may not have all the answers, but usually know where to look for them. Also, I have learned a bunch just by hanging out here at The Recording Website and from Devin Devore, the owner of Trinity Sound Co. Like everyone else I am still learning. Maybe someday I will know it all (like my kids).
(c) 1999, Dan E. Monk, All rights reserved.


text:
U.S. Amateur: Here's why you should know the name Cohen Trolio
by Julie Williams of AmateurGolf.com
see also: View results for U.S. Amateur, Oakmont Country Club
Cohen Trolio (USGA photo)
PINEHURST, N.C. – If Cohen Trolio holes a putt, the fairways at Pinehurst No. 2 are going to sing. There's a large Mississippi-based crowd following the 17-year-old this week at the U.S. Amateur, and they have a trademark phrase when Trolio jars one: "Putting arc!"
Dad and caddie V.J. Trolio explains that's the training aid he trademarked "a million years ago," way back in 2002. Trolio is the teaching professional at Old Waverly Golf Club, site of last week's U.S. Women's Amateur. It's also where the Mississippi State teams practice.
When Cohen dropped his clinching par putt against Austin Squires at No. 17 on Friday, he looked first at his father V.J., noticeably turning his head mid fist-pump. Theirs is a calm and close on-course vibe, which can be hard ground to find in a father-son golf relationship.
"Golf is kind of like our hunting," said V.J., who also has a younger son Collins.
V.J. may be toting the clubs at Pinehurst, but Cohen is running the show. It's a quick show, too. Squires, who recently graduated from the University of Cincinnati, is another quick player and the two put holes between themselves and the next group very quickly during the quarterfinal match.
The week is built around "Big C's" expectations, the caddie says dutifully. What does that mean?
"I wanted to win the golf tournament," Cohen said. "I didn't think any less of that. I knew that if I stuck to my plan, then I'm good enough to beat just about anybody here."
Squires spent the day driving it past Trolio by as much as 50 or 60 yards. Still, Trolio played his own game. If there was an advantage in all of that, it's that Trolio generally got to hit his approach first. That can eliminate some nerves.
"You kind of start thinking, 'Oh, I've got to hit it harder, oh, I've got to hit it harder,'" Trolio said. "It got kind of hard after I saw him bomb it about 50 past me I guess on No. 4, but I still knew that what I had was good enough. So I just stuck to it.
Trolio's game is the product of a lot of things, not the least of which is the caliber of players he grew up around. Central Mississippi is a hot bed of good golf. He has spent considerable time playing with Jim Gallagher Jr., a five-time PGA Tour winner, but also played frequently with former World No. 1 amateur Braden Thornberry. Mississippi-based professionals Jonathan Randolph, Chad Ramey and Matt Fast have also been sparring partners.
When asked to name the best player he's ever beaten, the edges of Trolio's lips curl up.
"I don't even want to throw his name out there," Trolio said in his slow southern drawl. "I've gotten a little bit of Jim Gallagher. I've gotten a little bit of him. Now, he still can put on a fight, but I got him once or twice."
The families are so close that the Gallagher's youngest daughter Kathleen, who graduated recently from LSU and played on the golf team, often goes by Kathleen Trolio.
125 years of golf in America. The USGA highlights golf in Mississippi! https://t.co/9N2tj4P6XX
— MS Golf Association (@MissGolfAssn) August 8, 2019
Gallagher's wife Cissye, herself an 11-time Mississippi Women's Amateur champion and former LPGA player, was part of a Southern mob that tossed Trolio around in one embrace after another when he secured his place in the semifinals. Earlier in the week, when Trolio looked to make a deep run here, Cissye threw a suitcase together and started driving east. She and a few other friends drove until midnight on Thursday, stopped in Atlanta, woke up at 5 a.m. and started going again.
She wanted to get to Pinehurst mid-morning so she could "hug his neck" before the start of the quarterfinals. Her husband had to be in Orlando this week because of previous work obligations as an analyst at the Golf Channel, but he's giving the live scoring page a workout.
"Cohen has laid it out there," Cissye said. "He's given up a lot of things like these kids do, just stayed in the process, just doing the things that his dad and the programs he created for him to do. Developed a great golf swing and he loves it."
The U.S. Amateur is Trolio's first real amateur start. It's been mostly junior golf up until now. As such, Trolio doesn't even appear in the World Amateur Golf Rankings. He was the only quarterfinalist without a number beside his name.
Then again, the high schooler just turned 17 last week. That he's played so well here tells V.J. that the preparation he's had at home is working.
"We put some Jesus in there, and then some golf," V.J. Trolio says.
Cohen Trolio has been one cool customer through this run, but in his mind, why wouldn't he be?
"I do this every day," Cohen said. "I mean, I play golf every day."
It's a freeing way to look at things.
Results: U.S. Amateur
Win Andy Ogletree Little Rock, MS 2000
Runner-up John Augenstein Owensboro, KY 1500
Semifinals Cohen Trolio West Point, MS 1000
Semifinals William Holcomb Crockett, TX 1000
Quarterfinals Austin Squires Union, KY 700
View full results for U.S. Amateur
ABOUT THE U.S. Amateur
The U.S. Amateur, the oldest USGA championship, was first played in 1895 at Newport Golf Club in Rhode Island. The event, which has no age restriction, is open to those with a Handicap Index of 2.4 or lower. It is one of 14 national championships conducted annually by the USGA, 10 of which are strictly for amateurs. It is the pre-eminent amateur competition in the world. Applications are typically placed online in the spring at www.usga.org.


text:
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Tag: Resident Evil 4 Archives | Broken Joysticks
Home / Tag: Resident Evil 4
Resident Evil Sale Causes Deals To Rise From The Grave On PSN
News of a Resident Evil 6 re-release on PlayStation 4 isn't the only reason to resurrect your interest in this series starring the undead. Capcom has discounted nearly every RE title available on PlayStation 3, 4 and Vita including the […]
Rae Michelle Richards No comments PSN Sale, Resident Evil 4, Resident Evil 5, Resident Evil 6, Resident Evil Revelations 2, Resident Evil Zero, Resident Evil: Operation Raccoon City
Resident Evil 4, 5 and 6 Get PS4 / Xbox One Ports
Capcom will bring 2012's Resident Evil 6 to 8th generation consoles alongside 2009's Resident Evil 5 and 2005's critically acclaimed Resident Evil 4 the publisher announced this morning. The release window for each title will be spaced out by a […]
Rae Michelle Richards No comments Capcom, PS4, Resident Evil 4, Resident Evil 5, Resident Evil 6, Xbox One


text:
BOT_EMAIL=wdaher@gmail.com
API_KEY=xxxxxxxxxxxxxxxxxxxxxxxxxxxxxxxx
STREAMS="event1 hongkong etc"
COUNT=50

mkdir -p output
while true; do

if ./show-last-messages --api-key="$API_KEY" --user="$BOT_EMAIL" --streams="$STREAMS" --count="$COUNT"; then
    echo "[`date`] Success";
    mv output-candidate.html output/zulip.html
    touch output/zulip.html
else
    echo "[`date`] FAILURE";
fi

sleep 30;

done

redpajama_set_name: RedPajamaGithub


What, casual work aprons aren't a thing?
Rifle Paper Co. may finally convince a generation of people to wear aprons. Because, let's be real: If you don't work in a restaurant or at a Home Depot, how often are you tying one on? Every time you preheat the oven to 350? Yeah, didn't think so.
Aprons just become one more thing to wash, even if it means sacrificing a few shirts to the occasional oil splatter or exploding batch of brownie batter. At least, that was my perspective — even amid spending three years at Delish, often destroying every shirt I owned while filming recipe videos in the test kitchen, common sense be damned — until I saw Rifle Paper Co.'s new collaboration with Hedley & Bennett.
Hedley & Bennett's known for making sleek, cool aprons that consider everything you'll actually need while cooking — including pockets at your waist, a towel loop (because you're always mopping something up), and a pen pocket near your chest. This collab's no different.
H&B launched two designs with the Florida-based illustrator: one featuring tiny greens and flowers against a black background, known as "Herb Garden"; and a half-denim, half-pink-and-khaki floral number called "Rosa." Oh, and there's a child-size design, so your mini-me can match you, too.
They also have a cell phone-sized chest pocket, perfect for keeping the 2018 cookbook close at hand (because honestly, what is paper?). But the best part, hands down, is the sturdy canvas material they're made out of. It's somehow soft, not itchy or bulky, yet thick enough to withstand your baking — or gardening — worst.
If $99 is a little too rich for your blood (I get it), you can always snag the design on a smaller scale, like this cell phone case or on a scented candle.


Once we receive your order, we'll personally email you an order confirmation and provide an invoice for payment.
*All orders must be paid in full within 1-hour of receiving the invoice. Any delay in payment will make delivery the following day.
3. We Craft and Deliver!
Once we receive the order payment, we get straight to work on crafting your custom floral arrangement for delivery!
Once the item is hand delivered, we send a Delivery Notification email directly to you for confirmation.
Copyright © 2018 Vincent Design Co. - All Rights Reserved.


For this session we will have the pleasure to have with us Ms Sarah Camilleri, Senior Manager - Organisational Development at GO plc who will present us with the Masterclass session.
We will also be joined by Ms Pearl Vella, People Business Partner at Casumo Gaming and Mr David Vella, Director Human Resources at Premier Capital who will present us with Case Studies on the subject.
The focus of this series is to create a strong community of HR Leaders and Practitioners that will redefine and reshape HR in Malta, through the collaboration and sharing of best practices. Throughout the series, we will be featuring leading local and foreign HR Directors and business leaders from different companies and sectors.
The event will be hosted by Analise Germani, Project Leader at salariesinmalta.com.


Only the Best Quality Food and Coffee on the Go!
Fed up with the morning traffic? Need that coffee to get you going on your way? Stop in on the way passed and get your coffee fix. Maybe even a muffin too.
Our experienced staff are very friendly baristas, who will make your coffee just the way you like it. Each custom request is a challenge - let them know how they go!
Using freshly roasted beans by Melbourne's own Atomica, 15 years barista experience combined with world class machinery, your coffee is sure to taste delicious every time.
Keep up with all the latest news and specials posted on Facebook.


As much as I would like to say that I have no favorites when it comes to my Pokemon crew because I love them all equally, I have to admit that there are a few who hold an extra special place in my heart.
My very first Pokemon - Chikorita - whom I already talked about in my last entry is one of them. Another one who just so happens to be the second Pokemon I added to my team is my Sentret-turned-Furret.


the perfect family holiday house waits for you !
Wow factor that extends beyond the front door - this vast, beautifully newly built Villa with traditional thatched roof and wonderful airy living area inside and out - you can breathe, relax you are on HOLIDAYS !
Villa Le Couchant 1 is part of a duplex and built on a big family land with 3 more houses. Le Couchant 2, the other part of the duplex, is rented by a couple on long term - every Villas has it's perfect privacy.
The entrance is a little piece of art itself - a wooden carving door which opens to the generous and airy living area.
The ground floor consists in an open American kitchen, a TV corner with a huuuuge flat screen TV, a big dining area and a lounge area which opens to a spectacular sea view. Le Morne Mountain (UNESCO world heritage site) can be seen from the terrace.
The ground floor offers one bedroom en-suite, a very convenient set up for people that cannot easily use stairs – the whole family will love it for sure !
The master bedroom en-suite with its own balcony with a breath taking sea view.
Two single bed bedroom sharing one bathroom.
But the most important ingredient for your holiday comes now - a grand wooden deck with a salt water pool (7m x 3,5m), the kids can splash and jump in and out the pool, whereas the "lazy" ones can chill and sun bath on the pool deck or right on the beach under shady pine trees. The sandy beach in front has enough space for everyone. Take a walk on the beach to the famous beach of La Preuneuse and don't forget your camera, the blue of the sea is just stunning !
The west coast of Mauritius is known for its beautiful mountains and the many activities offered within a 10-30 minutes car drive away.
But make sure you get back to the decked pool in time for the superb west-coast sunsets - and a nice drink with your dear ones !
You cannot stand still and want action ? Be active with lots of offered activities in the area - hiking in the Black River Gorges National Park, Casela Safari, Chamarel and the nearby LE MORNE Cultural Landscape - UNESCO World Heritage Site ... kite surfing, catamaran excursion, dolphin watching and lots, lots more.


HOLA GENTE !!! YA TENEMOS LOS NUEVOS CATALOGOS DE MARTINA PARA LAS CAMPAÑAS 5 Y 6 DE INVIERNO.
TENEMOS MUCHISIMOS LANZAMIENTOS DE COLECCIÓN.
CUALQUIER CONSULTA A LAS ORDENES !!!
PODES VER EL CATALOGO EN LA SOLAPA "CATALOGO" DE NUESTRA WEB.
SUMATE A NUESTRO EQUIPO, TE ESTAMOS ESPERANDO, NO TE VAS A ARREPENTIR.
PRESENTA UNA AMIGA AL EQUIPO Y GANA !!!
4) PODRAS ADQUIRIR PRENDAS DEL CATALOGO A PRECIOS ESPECIALES.
PERO ADEMAS, TENEMOS PREMIOS SOLO PARA LAS INTEGRANTES DE NUESTRA ZONA.
YA PENSASTE EN ALGUNA AMIGA, CONOCIDA, COMPAÑERA, PARIENTA?
DALE !!! QUE ESTAS ESPERANDO ? TODOS ESTOS PREMIOS SON PARA TI, NO TE LOS PIERDAS.
PASAME SU NOMBRE Y TELEFONO QUE DEL RESTO ME ENCARGO YO.
APROVECHANDO EL COMIENZO DE UN NUEVO MES TRABAJANDO TODAS JUNTAS CODO A CODO, LE QUIERO DAR LA MEJOR DE LAS BIENVENIDAS A TODAS LAS CHICAS QUE SE SUMARON A NUESTRO GRAN EQUIPO Y TAMBIEN A QUIENES VOLVIERON A SUMARSE DESPUES DE UN TIEMPITO DE AUSENCIA.
A TODAS LAS INTEGRANTES DE LA ZONA 2047 DE MARTINA, QUE ESTAN CUMPLIENDO AÑOS EN ESTE MES DE ABRIL, LES DESEO MUCHISIMAS FELICIDADES Y QUE SIEMPRE ESTEN RODEADAS POR SUS SERES QUERIDOS.


Links to articles on web design. Here we explain what responsive design is, we present its evolution and we focus on responsive design.
Web design is about creating web interfaces and not just about the graphical design of the user interface. Web design requires technical skills (programming), visual and graphic creativity.
Web design is no longer limited to creating a simple HTML/CSS Web site.
Web design takes more orientation towards web programming and more and more sites contain Web applications (to register members or customers, to charge, etc.).
But the birth of mobile phones has complicated Web design because their browsers are not able to correctly display the pages of websites designed so far for desktop computers. Web sites were often designed for 960-pixel screen widths.
To adapt websites to mobiles, some created a mobile version of their site and detected the features of the device to redirect to the corresponding page.
New problems occurred with the introduction of tablets and devices with small screens.
Several techniques appeared: the adaptive design then the responsive design.
The responsive design that uses the CSS media queries to change certain properties of objects according to the size of the browser window seems to have taken over.
Responsive design is also recommended by major search engines.
There are currently many CSS frameworks based on this principle including Bootstrap and Zurb.
The major disadvantage of responsive design is the difficulty of creating style sheets with the media queries since we must modify the properties of certain objects according to the different screen resolutions. This encourages working with a CSS framework to avoid recreating all common objects each time. Note that CSS frameworks are always quite heavy because they try to support a maximum of browser versions and some features of the CSS language may not be supported by some versions.
Responsive Design is a technique that attempts to adapt the same HTML page to all screen sizes by primarily using CSS, media queries, and defining almost all object sizes as percentages of the parent. To facilitate the adaptation of the content to the width of the screen, we use a grid whose column widths are expressed in variable units (percentages, em, vw, etc.).
As the width of the screen decreases and a media queries event is triggered, the width of some columns is automatically increased, causing the next column or columns to be pushed down.


text:
Areas of StudySocial SciencesDiversity StudiesCourse Descriptions
WOMEN 200: Introduction to Women's Studies:CD
Introduction to interdisciplinary methods and concepts related to women's studies. This course explores political, historical, and cultural constructions of gender, race, class, and sexuality. Topics include women's histories, intersections of identity, family, work, body politics, health, violence and protection, oppression, empowerment, and resistance. Prerequisite(s): Placement in ENGL& 101 or instructor permission. Crosslisted as: DIVST 200.
Course Level Objectives
Apply key concepts and theories from the field of women's studies to a broad spectrum of historical, political, international, and social issues.
Reason and think critically about gender relations and women's positions from a wide variety of theoretical perspectives.
Analyze and explore relationships between sociopolitical institutions and individual experience.
Explore overlapping meanings and constructions of race, class, gender, and sexuality.
Compare and contextualize the histories, stories, and current situations of different groups of women, by race, ethnicity, sexuality, class, and physical ability.
Examine the ways that intersections of race, class, sexuality, and gender shape life experience and identity formation.
Conduct participant observation and service-learning projects with local community partners, and reflect on the value of active, engaged learning.
Identify and assess empirical research findings to critically analyze issues related to women's positions and experiences.
Apply knowledge, awareness, and/or skills to identify and analyze issues related to diversity.
WOMEN 220: Women of the World:CD
Explores global diversity of ways that women create, influence, and challenge conventions and representations of gender, sexuality, race, class, and nationality. Comparative analysis of women's lives from class to culture, from work to marriage patterns, health issues, violence and protection, and grassroots organizing. (Dual listed as ANTH 220.) Prerequisite(s): Placement in ENGL& 101 or instructor permission. Crosslisted as: ANTH 220.
Define anthropology and culture.
Define women's studies and feminism.
Conduct participant observation and service-learning projects with local community partners.
Explain the value of participant observation and service learning as research tools in anthropology and women's studies.
Describe and discuss the significance and impact of women in cultural systems.
Describe the impact of women's lives on the cultural landscapes they inhabit.
Reflect orally and in writing on the value of service as a means of making a difference in a community.
Explain how societies are interrelated and the impact of globalization on women's lives and cultures.
Use a culturally relative approach to the exploration of women's lives in various cultures.
Diversity Studies


The CCCS is a non-profit organization. They can search for non-profit credit counseling agencies. The Delray Credit Counseling Corp. is a non-profit educational service dedicated to providing financial education and resources to consumers.
Occasionally I will see a prospective customer who is not a good prospect for a petition for bankruptcy. Thus, for example, the prospective customer could not pass the "neediness test" pursuant to 707 of the German Insolvency Act. Alternatively, a petition for insolvency will not resolve the main issue of the prospective customer.
Thus, for example, most of the debts of the prospective customer may be due to a students loans or a non-deductible income taxes. Alternatively, the vast majority of a prospective customer's debts could be a guaranteed one - that is, a liability for which a home or car is a security. Which possibilities are there for the prospective customer who is not a good bankrupt?
According to 523(a)(1) of the Insolvency Statute, only a restricted kind of liability is deductible in the context of an insolvency application. In order to be substantially taxable, an initial statement of revenue must be made. Furthermore, more than three years after the submission of the declaration, insolvency must be submitted for the relevant basic duty.
If an application is submitted belatedly, the income will not be tax-deductible unless insolvency is submitted more than two years after the date on which the applications are made. Lastly, in order for the deduction to be effective, insolvency must be submitted 240 working days after the date on which a levy is imposed.
"Fiduciary fund" taxation is never deductible in a chapter seven collapse. Alternatively, if insolvency is not an optional solution, the customer could consider an instalment arrangement with the IRS; a compromise bid; or trying to persuade the IRS that the IRS should be placed in "currently uncollectible" state.
Instalment payment agreements. They are approved by § 6159 of the Internal Revenue Code. IRS may conclude a payment instalment arrangement in writing which allows the customer to fulfil its obligations over a specified timeframe. A instalment payment arrangement is permissible if a tax payer has no wealth to meet a duty, but has a constant stream of available earnings.
The IRS considers the taxpayer's total personal earnings less expenditure and other allowances, inclusive of maintenance and children's allowance, when calculating the amount of the periodicity. To conclude an installment contract, the customer would have to draft a 433-D installment contract for the customer. Furthermore, the customer would probably have to fill in a 433-A sheet titled "Collection information card for employees and self-employed persons", 433-B, "Collection information card for companies" or 433-F, "Collection information card" (short sheet).
One of the advantages of an instalment agreement is that the IRS does not take any further steps during the term of the instalment agreement (e.g. seizure of wages). Punishments and interest remain due during the instalment agreement. Payment made under an instalment payment agreement shall be deducted from the final contractual fine or interest before being added to the outstanding amount.
And while the IRS may not be allowed to collect on any of a taxpayer's property while an installment payment arrangement is in force, the IRS may nevertheless submit a notification of the federal lien to safeguard the government's interests. Bids in compromise are approved by § 7122 of the Internal Revenue Code. A compromise proposal is a letter of understanding between the IRS and a payer that allows the payer to make a smaller payment to fully satisfy the payer's total income withholding.
IRS considers the taxpayer's working capital, ongoing earnings, prospective revenues and eligible expenditures. If, for example, it seemed that a prospective customer would never be able to settle the debt, but if a relation who owed nothing to the IRS was able to raise funds to settle the IRS (and therefore paying under the bid was in compromise cash that the IRS otherwise could not get), then the IRS would consider a compromise bid.
I once had a customer who was handicapped; who was not eligible for Worker's Compensation and whose dad (who owed nothing to the IRS) volunteered to settle a small portion of the son's IRS bill if the IRS accepted the compromise bid. For the IRS this is called a "Code 53.
" As an example, I had a customer who was 63 years old, had serious health problems, and it was clear that the customer could never even make a cut in his or her taxation. Note, however, that when the next declaration is submitted by a taxable person, if the declared revenue is higher than the amount encoded, the collection services bank is reissued.
According to 523(a)(8)(B) of the German Insolvency Act, study credits can only be declared bankrupt if the borrower can prove that the borrower would be "subject to unreasonable hardship". Whilst the term "unreasonable hardship" is not included in insolvency law, most jurisdictions have adopted the "Brunner Test" on a case titled Brunner v. New York State Higher Education Services Corp.
The Brunner test would require a debtor attempting to redeem a student loan to demonstrate that (1) due to ongoing revenues and expenditure, the debtor is unable to sustain a "minimum" level of subsistence for himself and his family if compelled to pay back the credit; (2) there are extra factors suggesting that this condition is likely to continue for a substantial part of the redemption term of the loan; and (3) the debtor has made a good-faith effort to reimburse the credit.
Under certain conditions, up to and beyond employment, a prospective customer with mainly study credits could aim for a postponement. As an alternative, the customer can see whether he or she would profit from the income-based redemption plan. The authorization for the income-dependent redemption plan is defined by the borrower. Finally, a customer could try to solidify students' mortgages in order to reduce his/her periodic repayments by adding several mortgages to one package deal and prolonging the payback time.
In the event that a students credit is already in arrears, the authorities have extensive enforcement authority, including: seizure of the client's return; seizure of a part of the client's salary; use of a part of certain state pension services, such as social security; or legal action against the customer in the courts. Lawsuits for the recovery of German study credits are not subject to a limitation period.
One customer could try to "rehabilitate" the cancelled students loans, which would involve concluding an arrangement and making at least nine qualified, punctual students credit repayments. When the customer's debt is primarily mortgages and the customer is not qualified for insolvency, the customer should ascertain whether he is qualified for a change in mortgages, either through the Home Affordable Refinance Program (HARP), the Home Owner Affordable Amendment Program (HAMP) or a personal change.
Customer should consult its creditor to check whether it is eligible for a HAMP change. An uncovered sale means a purchase of immovable assets where the sales revenue is less than the amount of the mortgage or other pledge on the immovable, but the holders of the pledge agreement have agreed to relinquish their pledge on the immovable and to pay less than the amount due.
Prospective customers who are not able to declare themselves bankrupt can take advantage of credit advice from a non-profit organisation such as a consumer credit bureau. In general, the customer will meet with a consumer credit advisor, develop a household plan, and then the customer will contact the lenders (usually a credit bureau ) to try to reduce the debts, make making money more accessible, lower the interest rates on the debts, or a mix of the three.
Firstly, the prospective customer must ensure that the services are a genuine non-profit organisation (e.g. a United Way agency). Customer is asked not to do anything. A number of customers came to my offices after I made an agreement with a rough "service" only to find that they had been taken to court by the credit cards group.
North Carolina has only one real "consumer credit advisory service" that can "customize" the customer's level of indebtedness before the customer is included in a corporate or legal loan agreement. Pardon the forgiveness of servicing the indebtedness. That is another thought when dealing with any kind of indebtedness.
If a creditor chooses to waive all or part of a liability and accepts less than what is due, the amount awarded is regarded as revenue for the debtor and subject to tax. It is the exemption established by the Mortgage Debt Relief Act of 2007 and is applicable to most home owners.
Under the Mortgage Forgiveness Debt Relief Act, debts waived between 2007 and 2012 are covered. Until 2 million dollars of waived debts are possible for this exemption. Insolvency - Debts settled by insolvency are not deemed taxpayable assets. If the tax payer is unable to pay at the time of remission, part or all of the terminated liability may not be subject to tax.
Specific agricultural debts - If the tax payer has accumulated the debts directly on the holding of an agricultural holding and more than half of the taxpayer's previous three years' revenue comes from agriculture and the credit was due to a regular creditor or credit institution, the waived debts are generally not regarded as taxpayable revenue.
Irrecourse credit - A non-recourse credit is a credit where, in the event of delay, the creditor only remedies the situation by taking back the object that has been funded or used as security. Under a non-recourse credit, the creditor cannot take the debtor to court in person in the event of delay. The waiver of a non-recourse credit resulting from a levy of execution does not lead to a waiver of proceeds from debts.
For the purpose of complying with certain IRS rules relating to taxation consulting, we hereby notify you that, unless otherwise specifically indicated, all taxation consulting services included in this blogs are not designed or written and cannot be used (i) to avoid Internal Revenue Code fines; or (ii) to promote, market or recommend to any other person any transactions or matters referred to herein.
According to the Bundeskonkursgesetz this is a statement that we are a credit rating institution. Helping those who go bankrupt under the German Insolvency Act. G. Martin Hunter is Charlotte, NC insolvency lawyer with expert knowledge in debtor/creditor issues. Provides insolvency counseling in Charlotte and throughout western North Carolina.
G. Martin Hunter is accredited as a specialist in commercial and consumer insolvency law.


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This charming industrial wire basket sporting a fantastic look due to its age and the type of setting it's been salvaged from. The vintage wire bin would make a fantastic laundry basket in your home.
there are signs of wear including small dents and scratches, the odd spots of rust. This does nothing to detract, however, and is, in fact, what gives the cabinet its character.


The holder of intellectual property rights and industrial property and responsibility of the data file and its treatment collected through this website is: Great Plastic, S.L.
www.greatplastic.com is the website of Great Plastic, S.L. through which it offers Internet users information about the company and products.
Great Plastic, S.L. not responsible for the misuse by the user of the content of this website, in turn , retains the legal action to recover any data or damage caused as a result of improper or illegal use of it.
Great Plastic, S.L. reserves the right to deny access discretion and at any time to any user of this site.


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6 NC counties are under alert. Details
Coast Guard, fishing crew rescue 4 following 'in-air mishap' off NC coast
Posted May 26, 2016 12:16 p.m. EDT
Updated May 26, 2016 6:12 p.m. EDT
Nags Head, N.C. — The U.S. Coast Guard and a Good Samaritan fishing vessel crew rescued four survivors Thursday morning following an "in-air mishap" involving two F/A-18F Super Hornets off the coast of North Carolina.
Officials said the incident happened about 25 miles east of the Oregon Inlet, in Nags Head, at about 10:30 a.m. The crew of the commercial fishing vessel Tammy help to recover the four survivors.
An HH-60 Jayhawk helicopter crew from Coast Guard Air Station in Elizabeth City took the Navy pilots to a hospital in Norfolk. All four of the pilots suffered minor injuries.
The jets were from Strike Fighter Squadron (VFA)211 at Naval Air Station Oceana.
The cause of the incident is under investigation.
No additional information was immediately available.
IMAGES: Coast Guard, fishing crew rescue 4 following 'in-air mishap' off NC coast
Two Navy fighter jets involved in 'mishap' off NC coast
A spokesperson speaks to the media about 'in-air mishap'
Bryan Mims
Keith Baker
Natalie Matthews
Copyright 2018 NBC News/WRAL. All rights reserved. This material may not be published, broadcast, rewritten or redistributed.
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