Quitting isn't easy, anyone who has tried can tell you that. Given that you're here, you probably already know from experience just how hard it can be.
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You're looking at headaches, nausea, and other digestive and sleep issues. And that's for all NRTs. They also each have their own specific side effects.
The gum can increase gastric acid and salivation, not to mention provide you with a case of the hiccups. These are just the most popular options.
Less common choices, such as the inhalers and the sprays have their own list of symptoms.
And all this time, there's still nicotine being delivered to your system. On top of all those nasty side effects, the NRT hasn't actually done anything to curb the nicotine addiction. According to WebMD, "Nicotine from medicines increases the levels of dopamine and norepinephrine just like nicotine from cigarettes does." You're getting the same fix that a cigarette provides, just in a different form.
Perhaps we can overlook all this. They wouldn't be so popular if they didn't work, right?
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ANSC 3305: Reproductive Biology in Health and Disease
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The audacity of questioning the root cause of canc...
The audacity of questioning the root cause of cancer.
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ricwally
Gilbert K Chesterton once referred to a paradox as "truth standing on her head to get attention". A prim example to illustrate this is with our solar system. Prior to Nicolaus Copernicus (1473- 1543), everyone in the world held the belief that the earth was in the centre of the universe. It was presumed that the sun, moon, planets and stars all orbited around the earth. The bulk of the observed evidence tended to support this first model of the world. But there was this one perplexing abnormality that persisted throughout the ages. The truth standing on her head was the fact that some of the planets appeared to move backwards during part of their orbit. This was the anomaly that caused Copernicus to first question the validity of the earth-centred model. He went on to design a new model for explaining the workings of the solar system, which placed the sun at the centre of the universe. This new model did account for the anomalies that had existed with the Earth-Centred model, yet nearly a century passed before this idea was taken seriously, and was not accepted until after Galileo (1564-1642) started observing the night sky with a telescope, which had just become available, and could for the first time see moons orbiting other planets. The 'truth' it appears, does not care if we believe it or not. The earth circled the sun even when everyone on it thought otherwise.
The word 'anomaly' is a prelude for us that points out that the current explanation is lacking or incomplete and inevitably a superior explanation will one day come along to explain/account for the shortcomings that the current beliefs do not address.
In the field of cancer, there remain a number of anomalies with the present DNA model. Why was it relatively scarce prior to the industrial revolution? Why is it less prevalent in developing (third world) countries? Why is it not contagious? Why can we not derive a vaccination for it, or 'shared immunity' from someone who does not develop cancer , yet has been exposed to all the same antigens as the cancer patient? Why do some treatments work well for some patients, and not at all for others? Why does the list of childhood cancers differ from that of adults, when our DNA does not change? And of primary concern; why does our immune system not respond to it? Just as in the case with our solar system model, it is possible that each paradox and each anomaly can be understood if we adopt an alternate framework for viewing cancer.
No one has ever questioned the premise that cancer is caused from a fault in the cell's DNA. There has never been a challenge brought forward or an opposing hypothesis suggested. It has always been taken as a 'given' that cancer was caused from a defect in our DNA. This premise would appear to be the central point from which all studies into the disease stem from. But what if this is wrong? What if we made a mistake right out of the gate? The last 125 years have been spent looking exclusively at our DNA as the culprit for this unwanted tissue growth, with questionable progress and frustrating statistics. Yet there are two distinct methods from which a living cell can be generated. Since the procedures that are capable of creating a living cell are limited to just two methods, then a flaw in the generation of unwanted cells should therefore be limited to one of these two methods. That is to say that cancer, which is defined as the growth of unwanted tissues, must either be the result of a defect in our DNA, or a defect in the repair process of our immune system ( the only other way in which a cell can come into being). A third scenario does not exist that is capable of creating a living cell, therefore a third scenario should not exist that could be a possible explanation for when something goes wrong. Thus far only one of the two methods of cell production has been considered as the culprit for when something goes wrong with the orderly generation of cells. Only the DNA method (the much studied process in which the cell's DNA instructs the cell to divide as outlined in the internal code of the cell) has been considered as the root cause of cancer.
Because the DNA method of cell regeneration has never been challenged or called into question, it has avoided the need of a label. For the purposes of this dissertation, I will be using the term 'DNA model' to refer to a sporadic mutation flaw which is currently believed to be the root cause of cancer. Cancer has only been considered as a defect at the cellular level and as a result, a solution to the problem of cancer has only been looked at from the cellular level. One cell generating method has been studied ad nauseam, and one cell generating method has been overlooked. Perhaps now it will be possible to turn the page and look at our immune system, the only remaining viable explanation that is capable of generating living tissue cells and therefore could also account for this unwanted cell activity.
This 'second method' (and only other process that exists) that is capable of generating a living cell will also require a label. I will refer to this as the scar tissue model, a much less studied, and less understood method whereby the body's own immune system is sent to a region immediately following some form of trauma; initially to stimulate the neighbouring cells into rapidly reproducing themselves in an endeavour to seal over a wound to hinder or prohibit any blood loss, as well as prohibiting the entrance into the body of foreign contaminants, and then the mending process of repairing the wound that set the activity in motion. We refer to this process as the formation of scar tissue, and it is a function of our immune system. The immune system is complex and much remains unknown about it. A similar situation exists now with our understanding of the immune system as it did with our DNA before the double helix sequencing chromosome model was first proposed in 1953. It could be observed and was understood that genetic traits were being passed down from generation to generation, however it was not known how that this information was being passed down After 1953 we were in a better position to understand some of the intricacies with which the system of information sharing actually worked. Similarly we presently live in a time where it is understood that the immune system is being sent to accomplish the tasks it was designed for, however we just don't yet know the intricacies which set the process in motion. Within our existing heading of the 'immune system' there are actually three distinct systems with three distinct functions. We don't yet have names for these divisions of the immune system so presently they are lumped together and collectively referred to as our immune system. For the purposes of this dissertation it will be necessary to divide the immune system into its three components. I will label the 3 divisions of the immune system simply as identify, destroy, and repair which are all completely different functions and require completely different sets of tools. First of all within our immune system there exists a complex system tasked with the responsibility of identifying foreign antigens that are deemed to be enemies of the body. There are instances when this system fails to properly identify or distinguish between foreign antigens and the bodies own cells and the classification of autoimmune diseases is the result. In addition to identifying these enemies , the immune system also has the task of setting up a strategy to destroy any and all identified threats. The immune system has control of an arsenal of specialized cells for the purpose of carrying out these missions, and a complete lymphatic system is at its disposal to deliver these specialized cells to the targeted areas. And on top of all that, and of principal interest to this alternate approach to cancer, the immune system has the responsibility of repairing any damaged cells that may have occurred during this onslaught. This new approach to understanding the root cause of cancer will be specifically concerned with this third branch of the immune system, namely the repair function.
Without fully understanding the process we can acknowledge that there is indeed a start code that sets everything in motion. Clearly once this process has been set in motion, there needs to be a corresponding mechanism in place to inform the body of when the healing process has been completed. That is to say, the body must be made to know when to start this healing process, and then when to stop this rapid formation of scar tissue, so that the immune system may end this elevated activity, and restore itself to the level of activity that existed prior to the trauma that set the event in motion. It doesn't require too much imagination to realize that the inability to shut off this 'repair process' would result in a situation indistinguishable from what we presently call 'cancer'.
Re: The audacity of questioning the root cause of cancer.
We generally tend to think of scar tissue as principally associated with our skin tissue. However any cell in the body is capable of being stimulated by the immune system into generating scar tissue. Broken bones or torn cartilage are repaired with the controlled formation of scar tissue. In fact there are over 200 different types of cells in the body, and not by coincidence there are now over 200 types of identified cancers. A defect in the 'start mechanism' would set the immune system into motion as if it had experienced some form of trauma, and the body would begin doing repairs to tissues without first being given a need too. Similarly, a defect in the 'stop mechanism' would result in the inability for this repair process once it had been started, to know when to shut off this process. Either one of these two scenarios would result in the manufacturing of unwanted tissues. Instead of viewing cancer as a defect in the p53 tumor suppressor gene, we could view it as a defect in our immune system which is carrying out repairs on tissues that do not first need repairing, and/or repairing cells and then not receiving the signal to stop. There must be a stop code.
All of what we presently know about cancer can be viewed from, and accounted for using this model for cancer. When further examined, all of the anomalies that currently surround cancer can be explained away. All of which has a tendency to add support to this new framework for understanding the disease, and simultaneously call into question the original premise of the DNA model.
The cancer cell is distinguishable from the normal cell because it was manufactured by a different process than normal cell replacement. But the DNA method of cell regeneration is not different and not distinguishable from the original. No explanation has been put forward to offer as to why this underlying characteristic would change just because the controlled order of cell replacement went astray. If the DNA model were to be true, then tumors should be clusters of cells indistinguishable from the host cells around them. A defect in the P53 gene should result in the formation of a wart, or a small polyp that could only grow to a size that could be supported by the existing blood supply. But we are still faced with the fact that the cancerous tumors are distinguishable from the surrounding tissue, and often accompanied by their own modified blood supply.
A close examination of tumor tissues reveals that there are similarities between the formation of scar tissue (with its accompanying inflammation that is necessary to support and maintain the existence of this newly generated cell) and cancerous activity. This relationship is most easily observed by comparing skin surface scars with skin cancer. Because scar tissue was manufactured rapidly, and by a different process from normal tissue replacement (normal cell division as outlined in the cell DNA), it has different characteristics. Scar tissue made from skin cells has a distinct appearance with a smoother surface, firmer density, (described as a waxy appearance) and a different pigment from that of the surrounding tissue. The following quote can be found at the final report on Grant GR/K71394 Mathematical Model of Scar Tissue
"Scar tissue formation is a ubiquitous feature of adult wound healing, with the resulting repair both functionally and cosmetically inferior to normal skin. At microscopic level, the main difference between scar and normal tissue is in the alignment pattern of the collagen fibers of which they are composed."
'Functionally and cosmetically inferior' are characteristics shared by cells thought to be manufactured by cancer cells, and cells known to be manufactured by our immune system. And yet 'functionally and cosmetically inferior' characteristics are not attributed to cells known to be manufactured by the normal DNA method in cases other than cancer.
If cancer was a disease of the cell losing the ability to replicate itself in a controlled manor, then we could expect to see uniformity between the cancer tumor and the parent cell that had lost this ability to replicate itself in a controlled manner. However; there would be no reason to expect to see uniformity between the various cancers themselves (if this uniformity did not first exist between the parenting cells). But the Nobel laureate Otto Warburg, while studying the metabolism of tumors, noted that "cancers of various species and tissue origins reveal a high uniformity from tumor to tumor." Warburg, O.: Stoffwechsel d. Tumore, Springer, Berlin, 1926. Engl. edn., The Metabolism of Tumors, tr. F. Dickens, London, 1930.
In fact there have been numerous studies all of which point to a number of parallelisms between cancer tumors of all types. There are a series of 'common denominators' that are shared between all cancerous tissues that do not have this shared characteristic with the host cells, which becomes an anomaly under the present DNA model for explaining cancer. The following 4 quotes with references point this relationship out.
i) "Correlatively, the Coris find the lactic acid and sugar content of the various exhibitions of cancer to be highly uniform. Williams and his co-workers report a pronounced degree of uniformity in the concentration of eight B vitamins in a great variety of animal and human tumours, regardless of the tissue of origin or the manner of their induction." Cori, C.F., and Cori, C.J.:J. Biol. Chem., 64:11, 1925
ii) "Shack describes an almost complete uniformity in cytochrome oxidase content in a number of mouse tumors." Shack, J.: J. Natl. Cancer Inst. 3:389, 1943
iii) "Maver and Barrett describe substantial evidence for an immunological uniformity among malignant tumors. Greenstein reports an impressive degree of uniformity in enzyme concentration among malignant tissues, regardless of their means of induction, tissue of origin or species of origin." Greenstein, J.P.: Symposium on Cancer, A.A.A.S. Research Conference on Cancer, ed. F.R.Moulton, Am. Assoc. Advancement of Science, Washington, D.C., 1945, p. 192
iv) "The uniformity of various exhibitions of cancer in respiratory properties, lactic acid production, vitamin content, enzyme content, action on a given substrate, effect on liver catalase, cytochrome oxidase content immunological properties, and many other characteristics is correlative to an uniformity of malignant tumors in the ability to metastasize, in their amenability to heterotransplantability, and in their autonomy, invasiveness and erosiveness. Indeed, there is no known basic property unique to any single exhibition of cancer---the only variation being a morphological one partially conditioned by admixed benign or somatic components." Cancer and the Immune System The Vital Connection
After considering all the above quotations, a fair question to be asked would be; why is there such uniformity between cancer tissues from tumor to tumor? If a fault in the DNA is causing this tissue growth, why then is the daughter cell even distinguishable from the normal cell? If we grant that a flaw in the p52 gene is allowing for the orderly manufacture of cells to going astray, this by itself does not give licence to allow for the cells that are spontaneously being generated to be any different from the host cell. All of this uniformity seems to point to the possibility that there is one common theme in all cancers, which implies a single source of manufacture. It is impossible for the present DNA model to account for this anomaly of uniformity when such shared characteristics were not first present in the host/parent cells. Yet 'uniformity' is an obvious inference if the cancers were all being formed from a single source (i.e. the 'repair' arm of our immune system). A pattern of uniformity would be necessary if the immune system were responsible for the manufacture of all of these tissues and a pattern of uniformity is what is being observed. This uniformity then becomes a truth standing on her head to get attention. To account for this uniformity under the present definition of the DNA model of cancer, it is said that the original cancer has metastasized to another part of the body. The word 'metastasized' imparts the belief that the new cancer has similar characteristics to the original cancer. The patient is never considered to have come down with two bouts of cancer, but rather one which has managed to have cells 'break away' and commence their havoc at a new site. The model for explaining how cancer can 'spread' like this under the DNA framework is troubling. The belief is that some cancer cells have broken away from the original site, and migrate to a new location and began work there (metastasized). But the original cancer is thought to have been caused by some antigen entering the body and attacking the DNA gene that is responsible for the orderly generation of cell replacement. The secondary site is never thought to be caused from a new antigen that has managed to enter this same patient, and started a new defect in the patients DNA. This 'metastasize' model allows for the similarities in the various cancer tumors to be understood and accounted for within the dogmatic framework of the current DNA model. The new cancer is described as a continuation of the original cancer at a new location. But the DNA model requires a host of special abilities being granted to these 'cancer cells' in order for them to be able to pull this off. They are given the ability to recruit allies, and 'cloak' themselves in the endeavour to explain how they can travel undetected and unharmed by the immune system as they journey to this new site.
Since there are only two ways in which a cell can be manufactured, and only one of the two methods can account for this 'uniformity', and in conjunction, account for why the new cell is distinguishable from the parent cell, and in turn account for why the cells can travel undetected in the body, then it would be prudent to entertain the possibility that the 'repair' aspect of our immune system (the only other way in which a living cell can come into existence) could be responsible for this non requested cell growth we refer to as 'cancer'.
There still remain many mysteries surrounding the immune system, and much is remaining to be learned. At present, the term 'immune system' is used to describe a complex series of body functions that more accurately could be described as three distinct systems with three distinct responsibilities. The term 'immune system' is currently used to describe all three of these functions; it is the responsibility of the immune system to
i) Identify foreign antigens that are deemed to be enemies of the body.
ii) Destroy these enemies of the body; and
iii) Repair any damage that may have occurred during this onslaught.
( Enveloped within the repair aspect, is the immune system's ability to 'inflame' the site with increased blood flow, a natural and vital component necessary to sustain the life of these newly generated cells by supplying oxygen and nutrients, as well as waste removal. The DNA model can account for the generation of new tissues, but not the accompanying modifications to the blood supply. An entirely distinct explanation must be employed that is thought to be happening simultaneously to account for the accompanying blood supply necessary to maintain the existence of these newly generated cells from the framework of the DNA model).
SarcomaSlayer
I've tried to have this exact conversation with people but unable to put the words together this well
May I forward this to a few friends ?
It's public so you don't need my permission but it's nice of you to ask. I was cutting and pasting this into sections that were small enough to fit into the 20,000 character limit and was cut off before I finished. I thought there may have been a 3 post limit per day. If that is the case I will resume pasting sections again today if I have 2 left. If you pm me your e-mail I could send the essay to you whole so you don't have to wait.
Alexanderwallen1@gmail.com
It has been observed and acknowledged that there is a corresponding activity in the lymphatic system in episodes of cancer. Often it is observed that the cancer has spread to the adjacent lymph nodes, and a study of the individual's lymph nodes is used to determine if the cancer has spread (metastasized). Yet the purpose of the lymph nodes is to serve as the center for production of phagocytes, which engulf bacteria and poisonous substances. Lymph nodes are a vital component of the immune system, and are always associated with immune system activity. In other words, with every non-cancerous situation, the enlarged lymph nodes indicates that the immune system is active and fulfilling its function. However; we are told in episodes of cancer, although it is acknowledged that the lymph nodes are active, the immune system is thought to remain inactive, and they are not fulfilling their function. Under the DNA model, it is not yet understood why the immune system would sit idle while events that it is designed to avert, takes place. No explanation is provided to account for how or even why the cancer employs the lymph nodes in the spread of the disease. From the framework of the scar tissue model, the lymph nodes would obviously be enlarged as the immune system was carrying out this work. But the present definition of cancer is thought to be having this activity take place without arousing an immune system response. If the cancer is to be defined as a defect at the cellular level as outlined in the DNA model, it is a necessary maxim to hold that the immune system is permitting the existence of these newly generated cells because it is unaware of their existence. From the framework of the DNA model the evidence that supports this observation is the fact that the immune system does not make any attempt to prevent this cancerous activity from taking place. The explanation for how these cancer cells do this requires that the cancer cells are bestowed a number of special abilities that are unique only to the cancer cells, all of which exceeds my level of gullibility. These special abilities are attributed to the cancer cells to help explain away why this event is being observed. This anomaly has never been adequately addressed and remains as a major conundrum of the present DNA model of cancer. It defies reason to accept that the immune system is doing nothing, and simultaneously accept that the lymph nodes are enlarged, but for a reason other than an immune system activity. A more credible explanation for this phenomenon is that the immune system is doing everything. If cancer were found to be a function of a defective immune system then this conundrum becomes a logical inference. The immune system will not attack the cancer because the immune system sent the cancer. The term 'cancer' should refer to a maverick arm of the immune system, i.e. the patient has a defective 'repair arm' of his or her immune system which has lost the ability to know when not to commence work on damaged tissues, and/or lacks the ability to know when to shut off this work once it has commenced.
All of the anomalies that surround this disease would suddenly vanish if cancer were to be viewed as a product of a defective immune system. We would not have to address how the cancer causing antigens enter the bodies of some individuals, and not others. These 'antigens' are the genesis point and mark the birth of cancer from the DNA model yet still remains to be unobserved at the cancer site. We would not have to address how cancer can move undetected throughout the body and take up residence at a new location without being detected, or encountering any form of resistance along the way. The study of cancer would then become an analysis of the way in which various cultures treat their immune systems, which then paves the way for this malfunction to take hold. Cancer becomes much less mysterious if we simply take the point of view that the immune system is causing the lawless proliferation of growth, (since it is its job to do so,) and the immune system is also supplying the essential blood supply to support this newly generated growth, by way of inflammation (again, because it is its job to do so). To read the present accounting of how the cancer cells manage to build the infrastructure of a blood supply system to support the existence of these newly generated cells requires an unrealistic level of trust. The cancer cells are attributed with a host of special abilities unique to them alone, all of which are necessary to account for this event taking place within the confines of the DNA model. Yet at the same time, the philosophy in treating cancer patients with radiation and chemotherapy is from the belief that these are the weakest cells and will be the first ones to die. Why would these cancer cells which are deemed invincible, immortal, perpetual cells be expected to be the first ones to die?
Mark Twain is quoted as having said "What gets us into trouble is not what we don't know… It's what we know for sure, that just ain't so."
Cancer should be viewed as the fulfillment of a two part equation. First, the individual must be in possession of a faulty immune system that is capable of generating cells without there being a need, and/or generating cells and not receiving the stop code. Secondly, this individual must then have their defective immune system directed toward and perform this non requested work on a specific group of tissues.
Statistical connections have been difficult to extract using the DNA framework because the scientific community is only looking at half the equation, i.e. why the cancer cells are attacking a specific tissue. Currently, scientists are only looking at the hierarchy of cell types to come under attack, which produces only 'links' to lifestyle choices or environmental exposures, etc.. If we factor into this how an individual treats their immune system, then perhaps some concrete relationships could be observed, and the global mapping of cancers would have significance. From the framework of this new model for cancer, the global mappings can be understood, and they do tend to have significance.
Immunosuppressant medications were developed to intentionally decrease the effect of the immune system in organ transplant patents, for the purpose of preventing the body's defence mechanism from attacking (rejecting) the foreign tissues of the transplant operation. If the patient survives the operation, and overcomes the rejection, they will live longer lives then they would have, had they not had the operation. Unfortunately, the statistical evidence shows that the transplant patient will ultimately succumb to a bout with cancer. This phenomenon has scientists struggling for an explanation:
"Scientists believe transplant recipients were already at risk
for cancer because their weakened immune system could not
keep healthy cells from becoming malignant".
"The use of immunosuppressants(cyclosporine) increases the
chance cancer cells will divide and invade surrounding tissue.
However it is not clear if cyclosporine can change normal cells
into cancer cells researchers say"
web search for 'organ transplants'
Organ Transplant Drug Increases Cancer Risk
Friday, Feb.12, 1999\
By using this new model for explaining cancer, we would predict that by creating a defective immune system, we increase the likelihood that some form of cancer will result. We would then have satisfied the first part of the two part equation. We have an individual with a weakened immune system that is capable of generating non requested tissues. All the other 'links' and 'markers' merely help to ascertain which of the numerous types of cancer the patient is apt to acquire. That is to say, the numerous lifestyle links, dietary links, and environmental links (physical carcinogens, chemical carcinogens and biological carcinogens.), all have a tendency to either promote any given tissue in the body towards cancerous activity, or to demote specific tissues away from being the likely candidate for cancerous activity. If the immune system is the root cause of cancer, then obviously it will be difficult to discover a cure so long as our efforts are focused on employing the immune system to attack itself. The immune system is designed to recognize and not attack itself. Perhaps this helps to explain why there are presently only treatments for cancer, and not yet any cures.
If a weakened immune system has been shown to causes cancer, as in the cases of patients taking immunosuppressants, would it not therefore follow that a strengthened immune system, should overcome, or at least prevent cancer? There is a disturbing pattern with immunosuppressant medications which clearly establishes that there is a cause-effect relationship between cancer and a weakened immune system. It would be anticipated that a concrete cause/effect relationship between cancer and a substance would be the Holy Grail in the cancer research world. This is the one thing that I would expect everyone would have been searching for. But no one seems to be able to recognise this because it doesn't fit with the DNA model. The DNA model is focused exclusively on the inner workings of the cell for the answer as to why it is reproducing itself relentlessly. When the malfunctioning immune system generates this relentless cell production from outside the cell itself, it is dismissed as an unexplained anomaly because it cannot be accounted for within the confines of the dogmatic view that cancer is happening at the cellular level.
Cold-Hot; Inactive-active; benign-malignant. These are the differences between non life threatening benign tumours, and life threatening malignant tumours, specifically one is active (cancerous) and one is benign (scar tissue). It may turn out that the fundamental difference between a benign tumor and a cancerous tumor is in the timing of when they are being observed. If you discover a benign tumor (or perhaps we could call it a tumor ''after-the-fact''), the body has stopped, and there is a mass of fibrous scar tissue that is currently not undergoing any development. If however, you were to stumble upon this very same tumor as it was being manufactured, it would be deemed to be a cancerous tumor. If your body is capable of producing a benign tumor, it is capable of producing a cancerous tumor. In the benign tumor, the immune system began a repair process that may or may not have been required, but evidently it did in due course receive the 'stop code'. In a cancerous tumor, either the cells do not receive the 'stop code', or it is being observed before it has received the 'stop code'. I have never heard of an Oncologist saying to a patient "You've got some sort of tumor being produced, but let's leave it be, and see if it doesn't stop and become benign on its own". If that same tissue were to be observed when it was inactive, it would simply be dismissed as a benign tumor that had previously been produced at some time in the past. It is dismissed as scarring, and is of no immediate concern, because it poses no danger to the patient. Everyone freely accepts that the inactive scar tissue was manufactured by the repair arm of the immune system. It should therefore be an easy inference to accept that cancer, or active scar tissue, or perhaps 'runaway scar tissue', is currently being manufactured by this same arm of the immune system, though be it a defective one.
When medical professionals discover an active tumor being produced, they may opt to surgically remove the tumor and the offending cancer cells that made it (excision biopsy). As this radical surgery has not yielded the desired success rates, the medical profession has expanded the scope of the surgery to include the surrounding tissues (margin), believing that these tissues might also contain some of the stray cancer cells. They then close up the wound and hope that they have managed to remove all of the cancerous tissue. Now they must wait until the immune system has had time to heal up the surgical wound before testing the area, because the activity of the inflammatory nature of the healing process will read as 'hot'. We then have the defective immune system, which may turn out to have caused the tumor to begin with, being invited back to the site, and being expected to heal up this surgical cut. Healing is what the immune system does. Therefore, this is an exercise for it. Often, the immune system heals over the surgery and then stops. The surgery was a success. Sometimes, however; the immune system doesn't stop. The immune system continues to produce scar tissue, and rapidly divide the adjoining tissues without receiving the message that the task has been completed. The poor surgeon is mystified that he or she could have missed some of the cancer cells, and now they appear to have merely taken up where they left off. If the DNA model were to be true, that some carcinogen ventured to the site and caused a defect in the cell DNA, then this patient, now rid of the offending tissues, should mathematically be given the same bill of health as a non patient (i.e. someone who has never had cancer). But the statistics do not support this optimistic expectation. Quite often, the cancer patients who undergo surgery have recurrences at the original site. If the cancer returns but at another location, then the surgery would be statistically labelled as a success. Even with this clemency being granted, the statistics for the surgery are not too favourable. The apparent failure of the surgery has given birth to the suspicions that exposing the cancerous tissue to the air helps it to spread. Or exposing the cancer to the light of the Operating Room, perhaps, is what causes it to flourish. Exposing the cancer to the light and air is merely a necessary by-product of the fact that these cells have been operated on, and as a result, the immune system is re-invited back to the region to repair the surgical wound. The suppositions that the light or air has anything to do with any re-occurrence can be dismissed because surgeries that are preformed on patients, who have not been diagnosed with cancer, are not subject to similar incidences of tumors, despite also being subjected to the light and air. These patients do not have the first prerequisite, namely the faulty immune system that is incapable of generating the "stop code". Even the supporters of the DNA model, acknowledge that cancer cells are in all of us (because the 'spontaneous existence of matter' is a hard concept to 'sell' and an absurd proposition). If we were to attribute this reaction to the light and/or air as yet another mystical feature enjoyed only by cancer cells, we would still need to account for why every surgery was not subject to the same level of re-occurrence. From the point of view of this new model, this anomaly would be addressed as follows; the non cancerous patient has a properly functioning immune system which still has the ability of knowing when to stop the healing process. In cancer patients, the immune system has already shown to be defective, therefore it should not be surprising to find out that sometimes it does turn out to be relentlessly continuing the healing process and in so doing, inflict the area with a new cluster of cancerous activity, despite how diligent and careful the surgeon had preformed.
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The library and archive collections are available for study to qualified researchers and scholars, by appointment only. The Library is open weekdays (except Mondays and holidays) from 10:00 a.m. to 2:00 p.m.
Appointments are arranged by the Museum's Research Manager who can be contacted by telephone at 817-509-8665, by email at info@cowgirl.net or by mail at 1720 Gendy Street, Fort Worth, Texas 76107. Appointments must be made in advance and will be granted depending on availability of staff and materials requested. Successful applicants must submit a user form describing their research project, read and sign a compliance form indicating that they understand the Library's handling policies and procedures, show photo identification with current address, and sign the daily visitor's register.
Library and archive materials are in closed stacks and are non-circulating. Materials must be consulted in the area in which they are housed. The may not be removed from that area or from the premises.
Smoking, eating, and drinking are not allowed.
Extraordinary care must be exercised when using materials in the library. Many items are fragile, extremely valuable, and/or irreplaceable.
Only items considered essential research tools (e.g., pencil, paper, notebooks, laptop computers, etc.) are allowed in the Library. Scanners and cameras are not permitted. Backpacks, bags (including purses), briefcases, coats, computer bags, and other such items must be stored at the Security Station. Security personnel reserve the right to inspect all belongings when anyone enters or leaves the premises.
law. The Library may decline a request to photocopy materials it considers too fragile or outside the "fair use" standard. Photocopies will be charged at $.20 per page.
It is the responsibility of the researcher to request and obtain permission to publish materials from both the Museum (as owner of the original) and the copyright owner, if the Museum does not own the copyright.
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The researcher agrees to indemnify and hold harmless the National Cowgirl Museum and Hall of Fame, its officers and employees, from and against all claims and actions arising out of the researcher's use of the Library's collections.
If you can not visit the Library, a staff member will conduct research on your behalf at a minimum fee of $25.00 per hour. Research costs will also include photocopies at $.20 per copy and shipping. Copies of the Research Services Agreement form can be obtained by calling the Museum or on the Museum's web site.


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FREEPHONE OR EMAIL US
www.mosquesecurity.com
Mosque Security Advisory Panel
Mosque Health & Safety and Security Assessment Service
Mosque Security Awareness Training
Imam and Alimah Personal Safety and Security Training
Mosque Security Guard Training
Places of Worship Security Scheme
Concern over mosque safety: British Muslims call for increase to govt security grant
Norway Mosque Gunman Gives Nazi Salute in Court
Far-Right Terrorism Is the Fastest Growing Threat to the U.K.
The fastest growing terrorist threat to the U.K. comes from the far-right, Britain's top counter-terrorism police officer said Thursday.
Seven of the 22 terrorist plots to cause widespread loss of life in the U.K. since March 2017 came from people with extreme right-wing ideologies, said Neil Basu, the assistant commissioner to the Metropolitan Police in charge of counterterrorism.
Basu told journalists his unit had seen "significant increases" in far-right terrorism. "When nearly a third of the plots foiled by police and security services since 2017 relate to right-wing ideology, it lays bare why we are taking this threat so seriously," he said.
"Together, we are bringing the full might of the U.K. [counterterrorism] machine to bear against those extremists of any ideology who wish to do us harm or incite violence," he added.
Unlike in the U.S., where guns are far more widely available, far right terrorists in the U.K. have not yet succeeded in carrying out a mass-casualty attack. But in 2016, the Labour Party lawmaker Jo Cox was attacked and killed by a rightwing extremist who shouted "Britain first" after shooting and stabbing her on the street in the run-up to the Brexit referendum. And in June 2017, a far-right attacker drove a van into a crowd of people outside a mosque in London, resulting in one death.
But the response of U.K. law enforcement has been more robust. After the El Paso attack in August, when a man opened fire in a Walmart in the border city killing 22 people, U.S. officials complained to TIME that the White House had ignored FBI warnings of a rise in right-wing terrorism, and that attempts to direct more federal spending toward the issue had been thwarted. President Donald Trump has been criticized for comments seen as emboldening white nationalist extremists, and for not doing enough to make tackling white nationalism a priority.
Far-right and white nationalist extremism is on the rise globally. In March, a gunman live-streamed an attack on two mosques in Christchurch, New Zealand, leaving 51 people dead.
The gunman posted a hate-filled "manifesto" online justifying his actions, and experts say social media has encouraged a copycat effect. The El Paso attacker posted a similar "manifesto" online, crediting the Christchurch shooter with his inspiration.
"This rise in far right extremism and terrorism is not unique to the U.K.," a spokesperson for the U.K.'s counter-terror police told TIME. "We have seen groups and individuals in other countries communicating with individuals here."
In the U.K. in May, a neo-Nazi who planned to assassinate an MP was jailed for life, after he admitted planning to "replicate" the murder of Jo Cox. And on Sept. 10, a 50 year-old white supremacist who stabbed a Bulgarian teenager the day after the Christchurch mosque attack was jailed for more than 18 years for carrying out a "terrorist act."
SOURCE – https://time.com/5681199/uk-far-right-terrorism/?fbclid=IwAR1FaQwXVs91mJe2XDqlcOaBBDFewH89NOb1T_mLh97DLjzX24VdMVgDNiw
Congregational Survey at Green Lane Mosque Highlights Need For Security Measures
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New to Jiu Jitsu. Been trying the truck. Been able to get to the position (probably not perfectly), but once I get there, 5 out of the 7 times, my partner has been able to sit up and I end up losing control of it.
Something that works for me: grab your leg that is hooked to your opponent with your same-side hand.
For example, if you are in the truck with your left leg hooked to your opponent's left leg, grab the inside of your own left thigh with your left hand. This allows you to keep your connection to your opponent's hips while they sit up.
Once they sit up, keep that hand-to-thigh connection and use the momentum of your opponent to roll with them. As you take that ride, roll over your left shoulder and you should end up back in the truck.
1) u have to make sure that you are beneath their hips. Meaning their hips are not too far away from you. If it is, then will gotta readjust the position.
Not sure the meaning of sitting up though.
Hope this helps! Gonna be following this thread for any tips that'll help improve truck game too!!
Last edited by Jason Wu; 01-30-2019 at 06:35 AM.
Usually, I'm hugging the free leg with my left arm for a crotch ripper setup. Not sure I'm getting the picture - while hugging his right leg, do I extend my left arm to touch his hips?
Ya Jason, when I'm under their hips, they kinda slip their lower back to the mat and then situp. Is it usually a battle to make sure you are under their hips?
Not sure which MTS episode explains the truck.
Maybe have a look at the YouTube link above of Master Eddie explaining the truck. It helped me previously.
Pay extra attention to how deep under the training partners hips goes so it's kinda elevated, how the legs are hooked and free legs are controlled.
Thank you, Jason! Yes, I will keep drilling it and go deeper under him, locking leg and free leg.


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Grace Notes Weekly E-News
The Tower Newsletter
Welcome Our New Priest
OUR NEW PRIEST…
My heart is filled with joy and gratitude for the honor, and trust given to me through the call to serve Grace as Priest in Charge. Acclyne and I are excited about this season of ministry and service at Grace and cannot wait to see what the Lord is doing in the Blue Water Region. We prayed for a church that is genuinely seeking after God, and we found that at Grace. If you are looking for a church home or church family, come and join us on this journey of exploration as we seek after God.
Paul says to the Corinthians, "…no one can lay any foundation other than the one already laid, which is Jesus Christ. If anyone builds on this foundation using gold, silver, costly stones, wood, hay, or straw, their work will be shown for what it is, because the Day will bring it to light. It will be revealed with fire, and the fire will test the quality of each person's work. If what has been built survives, the builder will receive a reward. If it is burned up, the builder will suffer loss but yet will be saved—even though only as one escaping through the flames." (1 Corinthians 3:11-14).
How are you building? May I invite you to join us as we build on this foundation? We might not have the answers, but we certainly will love the opportunity to know you and to listen to your story.
Rev. Shadrack Owuor, Priest in Charge
P: (810) 985- 9539
Email: info@graceporthuron.net
1213 Sixth Street
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Many adults are reluctant to pick up Young Adult books. Reasons vary, from being afraid to look silly to thinking YA books are pure fluff. Many think that YA is not worth their time. To try to remedy this, here are five YA books to get you started if you're reluctant to pick up a YA book or want to convert someone to the wonders that are Young Adult books.


The most popular of my pictures has been chosen as a bookcover for Jonathan Kellerman's new novel "Vina". It is the Czech translation of the popular novel "Guilt".
The #1 New York Times bestselling author Jonathan Kellerman's "psychology skills and dark imagination are a potent literary mix" (Los Angeles Times), and this intensely thrilling blend has never been so powerful as in the acclaimed author's new novel of murder and madness among the beautiful dreamers, seductive predators, and doomed innocents adrift in the glare of Southern California's eternal sunshine.
I am very happy to announce that one of my pictures has been chosen to illustrate an article in the Mountain Record Magazine. I am very thankful to participate in every kind of publication about wisdom because it reflects the essence of what I would like to express with my work. The article can be found here.
I have been invited to show some pictures at the photo exhibition from Collectif Image. The decline of the iron and steel industry in this region is in the minds and in the newspapers of everybody. I will present some pictures that show the beauty of an industry in decay. Sadness? Beauty? Melancholy? Or simply an evidence that life is an illusion and a dream where the objects we perceive appear, are animated and disappear like the figures created by the mist early in the morning when the sun comes out?
A new bookcover has been released for the novel "El viaje americano" from Ignacio Martínez de Pisón.
The cover can be previewed in the "Press and Public" Section.
I have the great pleasure to announce that a feature has been published in the Fotoritim Almanac 2012. It includes several double trucks and is 14 pages long. You may preview the magazine at issuu. The link is provided in the corresponding article unter Press and Public.
As you may see, the Internet Site has been completely redesigned in HTML 5 to be up to date with the latests standards and to allow people with mobile devices to surf easier through the pages. I wish you a good reading and don't hesitate to leave me comments or suggestions for this new layout.


Congratulations to Jade and Julie Work on their closing escrow with the purchase of this property. Their plans to replace the course with vineyards, implement a winery, plus redo the clubhouse and restaurant, are outlined in this article in the Village News.
Situated in beautiful Gird Valley, Fallbrook Golf Club offers a challenge to golfers of all skill levels.
Our regulation-length, Par-72 golf course is accented by mature trees, including native Live Oaks, Sycamores and Eucalyptus. Well manicured, strategically placed greens, sand, and water on many holes add not to the enjoyment of your round but also the beauty of the course.
After your round stop by AJ's Taphouse, relax and enjoy a drink. Try one of our daily food specials on our outdoor patio, with a deck that overlooks the putting and driving range, and fire pit for those cooler nights.
Whether you are looking to plan a tournament, banquet, wedding, or other special event, or just for a great round of golf, Fallbrook Golf Club is North County's best kept secret. Come visit and discover us for yourself.
Copyright © 2019 Fallbrook Golf Club All Rights Reserved.


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Pearl Harbor: Survivor Reports
USS Tennessee - Report by Survivor of Pearl Harbor Attack
Pacific Typhoon, 18 December 1944
Admiral Nimitz's Pacific Fleet Confidential Letter on Lessons of Damage in Typhoon
List of Commands and Ships Involved
Personnel Casualties Suffered by Third Fleet, 17-18 December 1944, Compiled from Official Sources
Aircraft Losses Suffered by Third Fleet, 17-18 December 1944, Compiled From Official Sources
Extracts Relating to the Typhoon from Commander in Chief, Pacific Fleet Report
Pacific Typhoon, June 1945 - Reports
Extracts from Commander Carrier Division Six Report
Extract from Report of Task Group Thirty-eight point one
Extract from Third Fleet Report
Pacific Typhoon October 1945 - Okinawa
Peacekeeping and Related Stability Operations: Issues of U.S. Military Involvement
The Pearl Harbor Attack, 7 December 1941 - Overview
Pearl Harbor Navy Medical Activities
"Pearl Harbor Revisited: USN Communications Intelligence"
Part-1
Notes on Sources
Pearl Harbor Salvage Report 1944
Pearl Harbor Submarine Base 1918-1945
USS Arizona - Reports by Survivors of Pearl Harbor Attack
USS California- Reports by Survivors of Pearl Harbor Attack
USS Maryland - Reports by Survivors of Pearl Harbor Attack
USS Oklahoma - Reports by Survivors of Pearl Harbor Attack
USS West Virginia - Reports by Survivors of Pearl Harbor Attack
Pearl Harbor: Why, How, Fleet Salvage and Final Appraisal
Pentagon 9/11
Personal Identification Tags or "Dog Tags"
BUREAU OF NAVIGATION CIRCULAR LETTER NO. 57-41
BUREAU OF NAVIGATION CIRCULAR LETTER NO. 152-41
Perspectives on Enhanced Interrogation Techniques
Philadelphia Experiment: ONR Information Sheet
Phonetic Alphabet and Signal Flags
Piracy Off the Horn of Africa
The Pivot Upon Which Everything Turned
Plea in Favor of Maintaining Flogging in the Navy
Pocket Guide to Hawaii
Pocket Guide to Japan
Pocket Guide to Netherlands East Indies
Pocket Guide to New Guinea and the Solomons
Port Chicago, CA, Explosion
William S. Parsons, Memorandum on Port Chicago Disaster
Station Order 10-45
War Diary
Court of Inquiry
Wartime History
Wartime History cont.
Press and Radio Release
Posse Comitatus Act and Related Matters: A Sketch
Post Mortem CIC [Combat Information Center] Notes
Potato Famine of 1847
Precisely Appropriate for the Purpose
Preserving an Honored Past
Priceless Advantage by FD Parker
Propaganda Foreign Military Studies 1952
Public Law 333, 79th Congress
Rescue, Rescue and Survivors, Pearl Harbor
Wallin, Homer N. Pearl Harbor: Why, How, Fleet Salvage and Final Appraisal. (Washington DC: Government Printing Office, 1968): 297-327.
Theater of Operations--Pacific
World War II 1939-1945
USS Tennessee - Reports by Survivors of Pearl Harbor Attack
USS Tennessee
The Executive Officer of USS Tennessee, Commander Colin Campbell, wrote as follows:
At 0800 Sunday morning, December 7, 1941, I was at my residence in Waikiki, Honolulu, T. H., on authorized liberty. At about that time I heard what appeared to be gun fire, and which I first thought to be target practice of some kind. Shortly after, word came over the radio that Pearl Harbor was being attacked by Japanese planes, and all service personnel should proceed at once to their stations. I started immediately by automobile to Pearl Harbor, but the traffic congestion was such that I did not arrive until about 0915. I went to the Officer's Club Landing. Bombing planes were still attacking. I was finally able to commandeer a boat. The Tennessee was moored inboard of the West Virginia at berth F-6. The West Virginia had been sunk and was on fire. The Arizona, about 75 feet astern of the Tennessee had been sunk and was on fire, and oil was burning on the water. I landed on Ford Island and about 0940 was able to get aboard over a pipe line. I went to the signal bridge and assumed command until the arrival of the Captain about 1000. Lieutenant Commander J. W. Adams, Jr., who had the head of Department duty, had been in command and was on the signal bridge. The stern of the Tennessee was on fire, and fires were raging on the Arizona and West Virginia, threatening destruction of this ship. The officers on the bridge of the West Virginia informed me that her after magazines had been flooded, but that efforts had been made to flood the forward magazines, but as the second deck was under water they were not sure that they had succeeded. I told them that their magazines must be flooded at all costs, as this ship was relatively undamaged and must be saved. When the Captain came aboard be directed me to go aft and take charge on the quarterdeck, where I remained practically continuously supervising the firefighting on this ship and against the oil fires on the water coming from the Arizona, until about sundown Tuesday the 9th, by which time the oil fires on the Arizona had been extinguished by this ship and yard tugs. The fires aft on this ship were under control by about 1030 Sunday morning, but continued to break out sporadically for the next couple of days due to the intense beat from the Arizona oil fires. During this time our main engines were run ahead and the wash from the propeller very successfully helped wash the burning oil astern, assisted by hoses from this ship. The Tennessee was wedged between the sunken West Virginia and the forward quay, preventing any movement ahead. As long as the intense fires raged on the Arizona, the Tennesseewas constantly in danger.
For me to mention the especially distinguished conduct of any particular individual would detract from the bravery, calmness, and efficiency of all officers and men. The conduct of all hands was superb, and I am proud of every one of them. I cannot help, however, mentioning at this time the distinguished conduct of Lieutenant Commander J. W. Adams, Jr., the gunnery officer; and that of Chief Boatswain I. W. Adkins, who had charge of the repair party fighting the fires aft, and whose leadership and heroic conduct helped to save the ship by keeping the fires under control.
Published: Wed Jul 22 12:00:24 EDT 2015


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« Wilton Candy Kitchen's Owner George Nopoulos Leaves a Legacy at 95 | Main | Gilbert's Highway 6 Coast to Coast Run for Kids Hits Iowa July 22 »
"River to River: Iowa's Forgotten Highway 6" to Premiere on Putnam Giant Screen in September
River to River: Iowa's Forgotten Highway 6 includes the Route 6 Retro Road Tour.
River to River: Iowa's Forgotten Highway 6 by Emmy® nominated documentary filmmakers Kelly and Tammy Rundle will premiere Thursday evening, September 17, 2015, 7:00 p.m. on the Putnam Giant Screen in Davenport, Iowa.
The event will kick off the Route 6 Tourist Association-Iowa's annual "River to River Retro Road Trip" which hits the pavement September 17-20.
River to River: Iowa's Forgotten Highway 6 guides viewers on a nostalgic classic car journey through yesterday's soda shops, filling stations, general stores, drive-ins, historic sites and roadside attractions that line Iowa's U.S. Highway 6.
River to River director Kelly Rundle "on the road."
"With Interstate 80's near universal use by travelers, Route 6 and the memories that define it have faded from the public's consciousness," said director Kelly Rundle of Fourth Wall Films. "The film is a celebration of a journey."
Producer Tammy Rundle with Dexter Museum curator Doris Feller following an interview about notorious outlaws Bonnie and Clyde whose crime spree landed them in the Iowa Route 6 town of Dexter.
"Most travelers are unaware of the many colorful stories from the past around every turn in the road," said producer Tammy Rundle. "Iowa's portion of the transcontinental Route 6 has a past that includes Bonnie and Clyde, Jesse James, Nancy Drew, Jack Kerouac, and others."
The project was suggested to the Rundles by Dave Darby, Executive Director of the Iowa Division of the Route 6 Tourist Association.
Dave Darby, Executive Director of the Iowa Division of Route 6 Tourist Association.
"Historic U.S. 6 is the longest highway ever created in the United States," said Darby. "It once stretched 3,652 miles from Provincetown, Massachusetts to Long Beach, California, right through the heart of Iowa." The Route 6 Tourist Association is responsible for an ongoing campaign to designate Historic Route 6 with special signs from Davenport to Council Bluffs.
The documentary will continue screening at film festivals and in art theaters and other venues, and will be released nationally on DVD in the fall. Broadcasts on Midwestern PBS stations will follow in 2016.
River to River: Iowa's Forgotten Highway 6 was funded in part by grants from Humanities Iowa, Silos and Smokestacks National Heritage Area and the National Endowment for the Humanities through the documentary's fiscal sponsor, The Iowa U.S. Route 6 Tourist Association.
The Rundles' Fourth Wall Films is an independent film and video production company. They produced the regional Emmy® nominated documentary Country School: One Room – One Nation; the award-winning films Movie Star: The Secret Lives of Jean Seberg (co-produced with Garry McGee), Letters Home to Hero Street (co-produced with WQPT), Any Kid Anywhere: Sex Trafficking Survivor Stories (co-produced with Braking Traffik), Lost Nation: The Ioway 1, 2 & 3, and Villisca: Living with a Mystery.
For more information, visit FourthWallFilms.com.
Posted at 01:34 PM in Highway 6 Events | Permalink


Enjoy our Youtube Virtual Training Videos.
If you are new to Jolt the first thing you will want to do is watch all of the Jolt Quickstart videos. These training videos will help you learn to use and set up People, Roles, Lists, iPads, Communication, and much more.


We offer places for babies from 3 months of age. We appreciate the importance of individual routines at this age and work closely with parents to ensure a personal approach for each child. We have a separate sleep room and nappy change facilities. At this age, play and learning is very exploratory and discovery based with all five senses involved.
Treasure baskets - encouraging children/s natural curiosity investigating "real life" items.
Messy play - paint, gloop, water, play dough.
Exploring the outside world - watching trees sway in the wind, listening to birds singing.
Physical play - tummy time, developing balance and control.
Cuddles and quality one-to-one time with a key person.
Our Toddler room caters to children from 18 months of age depending on their development.
Our toddler room has been carefully designed promote curiosity, wonder and excitement. It is a warm, cosy and homely place where each child is valued.
Active learning and the desire to be involved in new experiences is what we foster and encourage amongst our toddler children. We provide children with open ended learning experiences and enhance activities to follow their interests.
Children usually move up to the preschool room before their third birthday.
Our day is flexible yet brings elements of structure and challenge. Practitioners plan activities using current interests taken from observations and conversations with the children - therefore our children gain new knowledge through the planned experiences linked to events, seasons and celebrations. We recently enjoyed a visit from Joanna, a paramedic who came and introduced the children to the important role she plays in helping people.
Children learn so much from outdoor play. At Fledglings Day Nursery children have access to outdoor play daily as this is highly emphasised in the EYFS. Children have the opportunity to go outside no matter the weather.
As children run around outside they are learning about the effect physical exercise has on their bodies Being outdoors helps so many areas of children's learning and development, they learn to build confidence in themselves, they learn about problem-solving and socialising with other children. When outdoors children learn about risk taking, practitioners feel that children learn vital skills when risk taking.
Our back garden is specially designed for our younger children aged 0-3. Our 3-5 year olds have their own outdoor area with more challenging resources and materials. In both our outdoor areas we have made use of natural and recycled materials. These open ended resources encourage children to use their imaginations and problem solve.


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The Gay Marriage Debate
CP Current Page: Opinion | Sunday, September 04, 2011 | Coronavirus →
By Chuck Colson, Christian Post Guest Columnist Follow | Sunday, September 04, 2011
In a debate, you are judged in two main areas: substance and style. In the debate about so-called gay "marriage," we believers are winning on the substance. But we're not doing very well on style.
Study after study has shown that gay "marriage" undermines the institution of marriage. In those societies where homosexual "marriage" has been tried, traditional marriage is increasingly discarded. Furthermore, the research is nearly unanimous that children do best when they grow up with a mother and a father - which gay "marriage," by definition, denies them.
Based on all this, you might think that we are winning the debate. And yet, the tide of public opinion appears to be turning against us. According to columnist Michael Barone, "In 1996, Gallup found that Americans opposed it by a 68 percent to 27 percent margin. Last May, Gallup found Americans in favor by 53 percent to 45 percent. That's a huge change in 15 years." And a new Pew Research poll confirms this, finding those in favor are in a dead heat with those who oppose it.
Much of that new support is coming from young people, who increasingly see marriage in terms of civil rights and fairness. That's a demographic factor that makes winning the debate in the coming decades that much harder.
That's why, given current sensitivities, we've got to be very careful about how we frame the debate. Take the case of social studies teacher Jerry Buell in Lake County, Florida. Mr. Buell was named "teacher of the year" in 2010. He has a spotless classroom record. And he was suspended and placed under investigation for criticizing gay "marriage" … on his Facebook page!
Outrageous, you say? A violation of his right to free speech? Absolutely. However, listen to part of what Mr. Buell posted on his Facebook page: "I'm watching the news, eating dinner when the story about New York okaying same-sex unions came on and I almost threw up. … If they want to call it a union," he wrote, "go ahead. But don't insult a man and woman's marriage by throwing it in the same cesspool… God will not be mocked."
However much we sympathize with the substance of Mr. Buell's comments, I hope you'll agree that he earned a zero on style points. Of course it's ridiculous - and alarming - that Mr. Buell's livelihood and religious freedom came under threat. But that's nothing new with the gay-rights movement, which frequently attacks one person's rights to gain someone else's. That, I'm afraid, is the world we live in.
As frustrating as this debate can be, prudence dictates that we Christians be especially mindful of our responsibility to remain civil. First of all, it's the right thing to do. Secondly, the supporters of gay rights are waiting for any opportunity to pounce on their opponents if we give them any opening.
But there's another point I want to make here, one you'll hear me to return to again and again. We Christians are very good at saying "No." We've got to better saying "Yes": showing how God's plan for humanity is a blessing: That His ways - including faithful, live-giving marriage between one man and one woman - lead to human flourishing, physically, emotionally, and spiritually.
And that's a message I'd be happy to post on Facebook or anywhere else.
From BreakPoint. Reprinted with the permission of Prison Fellowship Ministries. All rights reserved. May not be reproduced or distributed without the express written permission of Prison Fellowship Ministries. "BreakPoint®" and "Prison Fellowship Ministries®" are registered trademarks of Prison Fellowship
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Naria est un genre de mollusques gastéropodes marins appartenant à la famille des Cypraeidae (les « porcelaines »). L'espèce-type est Naria irrorata.

Liste d'espèces
Ce genre, créé en 1837, a fait l'objet d'un vaste remaniement à partir de 2017, et a intégré de nombreuses espèces jusque là classées comme Cypraea ou surtout Erosaria.

Références taxinomiques

Notes et références

Genre de gastéropodes (nom scientifique)
Cypraeidae

redpajama_set_name: RedPajamaWikipedia


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Home/Schengen Visa News/COVID-19 Recovery Certificates for Entering Greece Are Now Valid for Only 90 Days
COVID-19 Recovery Certificates for Entering Greece Are Now Valid for Only 90 Days
Schengen VisaDecember 11, 2021
Travelers eligible to enter Greece, carrying proof of recovery from COVID-19, should now be aware that the validity of these certificates has been halved by the Greek authorities.
The Greek Civil Aviation Authority (CAA) announced on Friday, December 9, that COVID-19 recovery certificates will now be valid for entry into Greece for a shorter period of 90 days instead of 180 as they were previously.
"Travelers to Greece must bring a certificate of illness issued thirty (30) days after the first positive test, and its validity will continue for up to ninety (90) days thereafter," the Civil Aviation Authority notes in a press release.
So far, travelers who entered Greece on the basis of a recovery certificate could do so if they had evidence that they had contracted the disease from COVID-19 between the past 180 days, but not in the last 30 days. This means that the certificates that remain valid are those issued after July 15 but no later than November 15.
The decision will affect travelers from 43 countries in the world who have not been vaccinated but are yet eligible to enter with evidence of having previously had the disease, particularly those who contracted COVID 19 between April 15 and July 15 this year.
These 43 countries are: Australia, Albania, Andorra, Armenia, Azerbaijan, Argentina, Bahrain, Belarus, Bosnia and Herzegovina, Brunei, Canada, China, Chile, Chinese Taipei, Israel, India, Japan, Jordan, Kosovo, Kuwait, Lebanon, Moldova, Monaco, Montenegro, Mexico, New Zealand, North Macedonia, Oman, Qatar, Russia, San Marino, Saudi Arabia, Serbia, South Korea, Singapore, Turkey, Ukraine, United Arab Emirates, United Kingdom, United States America, Uruguay, Vatican .
However, the same can enter Greece if they underwent a PCR test for COVID-19 in the past 72 hours before arriving in Greece or a rapid test in the previous 48 hours.
Residents of these 43 countries who can no longer use their recovery certificate, and decide to get vaccinated against COVID-19, can use proof of vaccination to enter Greece without restrictions as well.
However, they need to "complete at least fourteen (14) days prior to being vaccinated against COVID-19 and present a certificate of vaccination, in Greek, English, French, German, Italian, Spanish or Russian, which has been issued by a public authority."
Apart from the residents of the third countries mentioned above, entry to Greece is allowed from other countries of the world, namely the European Union and Schengen agreement citizens, their spouses (or the person with whom they are cohabiting) as well as their minor children.
At the same time, the Civil Aviation Authority has extended the entry ban to South African countries where the Omicron variant was first detected, until December 17. These countries are South Africa, Botswana, Namibia, Lesotho, Zimbabwe, Mozambique, Eswatini, Zambia and Malawi.
Foreign nationals whose flights depart from these countries must obtain permission from the Greek authorities or from the local embassies, consulates and consular authorities of other countries, who represent Greece in the absence of a Greek embassy in their country.
>>Greece allows entry to travelers from Mexico, extends restrictions to other countries
Greece Refuses to Impose Travel Restrictions on US Residents
France Officially Extends Health Pass Requirement Until Mid-2022
Spain Adds Poland, Regions of Czechia & Hungary to List of High-Risk Areas
Denmark Imposes Testing Requirement for Unvaccinated Travellers From 6 More EU Countries
Malta's Embassy in Libya to Start Processing Travel Visas at the End of August


David Snow received his musical training at the Eastman School of Music, the Yale School of Music, and Brandeis University. A recipient of awards from BMI, ASCAP, Meet the Composer, the Maryland State Arts Council, and the National Endowment for the Arts, Snow has been a fellow at Yaddo in Saratoga Springs and has participated in the annual Composer-Choreographer Workshop at the American Dance Festival. His music has been performed in concert at the Kennedy Center, Carnegie Hall, the Aspen and Tanglewood Music Festivals, and across the United States. Snow's recording of "Larry, the Stooge in the Middle" earned him Musician magazine's "Best Unsigned Band" award in 1992. His composition "Dance Movements" is featured on the American Brass Quintet's New American Brass compact disc on the Summit label.
"In his monumental Tractatus Logico-Philosphicus, Austrian philosopher Ludwig Wittgenstein attempted to arrive at a comprehensive definition of knowledge based upon an analysis of language. Wittgenstein's application of logical methodologies to this task proved influential in the development of modern theories of linguistics, computation, and machine intelligence. Given that science, philosophy and art evolve in parallel, it is surely no coincidence that the rigorous formalism of Wittgenstein's thought found a counterpart in the development of contemporary music theory. As one noted academic succinctly observed, 'The ubiquitous preoccupation with polyphony (i.e. with fundamental voices contributing ordered subsets to form larger, more comprehensive sets which may themselves be subject to transformation as intrinsic voices in a musical dimension) should be viewed as an attempt to achieve profound levels of integration in the hierarchy of structures generated by the partition of fundamental lines relative to appropriate criteria of relatedness vis-a-vis the total set of structures.'"


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Matthew Messinger
Mr. Messinger has been the President and CEO of the Company since October 2013 and has served as a director of the Company since March 9, 2016. Prior to joining the Company, Mr. Messinger served as the Executive Vice President and Director of Investment Management at Forest City Ratner Companies ("FCRC"), a wholly owned subsidiary of Forest City Enterprises ("FCE"), where he served for more than 18 years. In this role, Mr. Messinger led the New York Investment Committee of FCRC and served on the Investment Committee and Executive Management Committee of FCE. Mr. Messinger brings extensive development, asset management, finance, strategic planning and tax credit structuring experience across a wide range of asset classes including retail, hotel, residential, office, arena and professional sports teams. Mr. Messinger is a graduate of Wesleyan University in Connecticut. He currently serves as Co-Chair of the board and on the real estate committee of the Children's Museum of Manhattan, and he is a member of the International Council of Shopping Centers (ICSC), Urban Land Institute (ULI), the Real Estate Board of New York (REBNY), the Low Income Housing Tax Credit Coalition, the New Markets Tax Credit Coalition, and the New York Hospitality Council.
Steven Kahn
Mr. Kahn joined the company in September of 2015 as its Chief Financial Officer. He has over 20 years of real estate accounting and financial experience including matters related to SEC compliance, tax compliance, development and acquisitions. Mr. Kahn previously served as CFO of United Realty Trust Inc., Senior Vice President Finance at SL Green and has held positions with both PricewaterhouseCoopers and Deloite & Touche. As CFO Mr. Kahn focuses on budgeting and expense control, shareholder and lender relations, and ensuring compliance with accounting, tax and SEC regulations. He is responsible for supporting and helping to develop the Company's strategic plan and maintaining certain key financial and accounting relationships.
Richard G. Pyontek
Chief Accounting Officer
Mr. Pyontek served as the Chief Financial Officer of the Company from October 10, 2012 until Mr. Kahn's appointment as Chief Financial Officer on September 21, 2015. Mr. Pyontek was instrumental in ensuring the Company complied with all aspects of the Chapter 11 Plan of Reorganization, including the payments of all approved claims and he oversaw the change back to going concern accounting. Mr. Pyontek now serves as the Company's Chief Accounting Officer. Mr. Pyontek also served as Director of Accounting and Reporting for the Company until his election as Chief Financial Officer. Before joining Syms Corp., Mr. Pyontek served as Director of Accounting and Reporting at Ashley Stewart, Inc., as Controller at The Vitamin Shoppe and as Director of Finance at Party City Corporation. Mr. Pyontek is a Certified Public Accountant.
Miriam G. Harris
Ms. Harris joined the Company in February 2014. Prior to joining, she served as Executive Vice President and co-headed the Real Estate Transaction Group at the New York City Economic Development Corporation ("NYCEDC"). In that capacity, she led a team of real estate professionals who executed transformative public-private redevelopment projects on behalf of the City of New York such as the Redevelopment of Seward Park on the Lower East Side of Manhattan and the Cornell Technion Campus on Roosevelt Island. Prior to joining NYCEDC, Ms. Harris spent over a decade at Forest City Ratner Companies where she served as the development manager for the 1.6 million square foot New York Times Building in Times Square and worked on the company's hotel developments.
Charles Gans
Mr. Gans joined the Company in October 2017. Prior to joining the Company, he worked at the New York City Economic Development Corporation ("NYCEDC") in the Real Estate Transactions department. NYCEDC's Real Estate Transactions department executes transformative public-private redevelopment projects on behalf of the City of New York. Mr. Gans held a progression of roles over his seven years at NYCEDC. He began his tenure at NYCEDC managing projects such as Essex Crossing and Cornell Tech, and rapidly rose to the title of Executive Vice President and co-head of the Real Estate Transactions department. As Executive Vice President, Mr. Gans led redevelopment projects such as the Spofford Live Work Campus in Hunts Point, the mixed-use waterfront project on 44th Drive in Long Island City, and Caton Flats in Flatbush. In total, Mr. Gans closed over $3 billion of transactions at NYCEDC. Prior to joining NYCEDC, Mr. Gans spent six years at Lend Lease where he served as construction manager for over 1.25 million square feet of ground-up, high-rise residential development projects and performed preconstruction work across a variety of commercial office, hospitality, senior living, and multifamily projects. Mr. Gans received his MBA from the Wharton School at the University of Pennsylvania and a BS in Civil Engineering from Northwestern University.
Jeffrey Travia
Vice President of Acquisitions and Development
Mr. Travia joined the company in September of 2015 as its Vice President of Acquisitions and Development. He has over ten years of experience in the real estate industry, most recently raising nearly $1 billion of capital for clients at Ackman-Ziff . Previously, Mr. Travia was the Deputy Director of the Long Island City Business Improvement District as well as having served as an associate in the real estate development departments of Tishman Speyer and the Related Companies. Mr. Travia focuses on the development and repositioning of Trinity's existing assets as well as pursuing new business opportunities. He holds an MBA from The Wharton School, University of Pennsylvania and a BA in Urban Studies from The University of Pennsylvania.


The associations at Excelia Group are fully active throughout the entire year. There are some 40 associations and clubs to choose from and getting involved will help your development on both a personal and a professional level.
With more than 800 active members generating an economic activity of some 700 000 euros, our Group's student associations and clubs are particularly dynamic.
"Since the beginnings of Excelia Group, student associations and clubs have always played an important role. Today, involvement in them has become an integral part of our student programmes," states Christelle Egreteau, Head of Associations, Excelia Group.
As part of your programme, you will take part in a team project within one of these associations, a sports club or even one of the Group's communication or promotional activities. Known as SCAP (Sport, Communications, Associations and Promotions), the principle aim of your involvement is to ensure you have a fully-rounded experience throughout your studies.
The associations at Excelia Group are responsible for the coordination of a large number of projects of differing scales and themes.
"The Group is fully committed to student involvement in associations and clubs as proven through the work of some of its key organisations such as Le Bureau Des Etudiants (The Student Office), Le Bureau Des Sports (Student Sports Society), Sup de Co Voile (the School's Sailing Club), or even Sup'Citoyenneté (a club organising community-based projects)," adds Christelle Egreteau.
By honouring the values of team spirit, solidarity, commitment, responsibility, honesty, integrity, respect and tolerance, your personality can only evolve accordingly.
For students choosing the Entrepreneurship Track (Master in Management Grande Ecole programme Year 2) or those choosing the Association Entrepreneurship Track (Bachelor in Business Year 2), these associations provide a perfect environment in which to acquire managerial skills.
Students take on a role of responsibility for a certain time period, in which they manage the association as well as its projects and team members. In addition, they benefit from extra management training and personal coaching. It is a unique opportunity to be responsible for a team, a budget and one or more projects.
"Supported by business professionals and senior personnel from within the School, the 60 or so students who we train every year to become association managers and heads of project, gain a real-life experience of leadership, project management and team management," notes Christelle Egreteau.
In short, these roles provide excellent grounding for students' professional lives and are considered valuable experience in the eyes of recruiters.


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Pandemic Increases Support for Weed Legalization Among New Jersey Voters, Survey Finds
Garden State residents will vote on legalizing cannabis in November, and 21 percent of former weed naysayers say COVID has changed their mind on the matter — for the better.
Image via
After suffering through multiple terms of Republican Gov. Chris Christie, a lying sack of weed-hate and repulsive Trump suck-up, New Jersey elected pro-pot Governor Phil Murphy in 2018, a Democrat who recently stated that legalizing weed would be "incredibly smart."
New Jersey residents are set to vote in November on Public Question 1, an initiative that will fully legalize ganja in the Garden State if approved. A poll conducted after the measure was announced found that 68 percent of respondents said they planned to vote in favor of the change.
Now, a new survey conducted by Brach Eichler's Cannabis Law Practice indicates that overall support for legal weed stands at about 65 percent. But, the results show that the coronavirus pandemic has inspired many participants who initially thought they'd vote against Public Question 1 to change their minds.
When asked about the pandemic, 79 percent of respondents said it had not shifted their position on cannabis, but 21 percent said it had. Of the latter, 13.5 percent said they switched to now support legalization — and most of those identified themselves as Republicans or conservatives.
The poll also found that support for legal weed in New Jersey crossed party lines to create a bipartisan majority. 72 percent of Democrats said they presently support legalization, followed by 65 percent of Independents and 56 percent of Republicans.
"I was somewhat surprised that [the pandemic] did influence that many people," Charles Gormally, co-chair of the firm's Cannabis Law Practice, told NJ.com. "I really think the reason it influenced people is a natural derivative of beginning to accept the concept that cannabis isn't harmful, and may in fact have a positive impact in many circumstances."
Ignoring their constituents' loud and clear mandate, the Republican-led New Jersey Senate killed a legalization bill last year, which ultimately led to the question landing on this November's ballot.
New Jersey's pro-marijuana momentum has been building into an undeniable force, though. Medical weed is legal, and Gov. Murphy has called for huge expansions to the program. In addition, New Jersey police officers no longer train drug dogs to sniff out pot. All that's left is to vote "yes" on Public Question 1.
Mike McPadden
Mike McPadden is the author of "Heavy Metal Movies" and the upcoming "Last American Virgins." He writes about movies, music, and crime in Chicago. Twitter @mcbeardo
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A trail is worth a thousand thoughts
In Vannevar Bush's 1945 Atlantic Monthly article titled As We May Think
(see www.theatlantic.com/unbound/flashbks/computer/bushf.htm ) he suggested that instruments are at hand (such as the Memex machine described in the article) which, if properly developed, will give man access to – and command over – the inherited knowledge of the ages.
I would like to explore how close we are to this vision some 45 years later. First, I draw your attention to Section 7 where one of the most useful functions of the Memex is described – the ability to develop associative links between two previously unrelated documents.
He describes the construction of a "named trail" which I like to call "Vannevar Trails" or "VTs." A VT is separate from the documents referenced. It consists of one or more items, with each item forming a link between a pair of documents via some unique code assigned to each document, and having some code word or word assigned to each item.
If a document is referenced, its VT also becomes visible, and the reader may reference the other linked document via the VT. All items in the VT can be reviewed, hence retracing the thinking of the original researcher or author who put this information together.
This scheme cannot be implemented well using HTML. A page developed by an author may be linked to any number of other pages, via the URL scheme. The pages referenced may represent the entire document (as in the Atlantic Monthly link above), or may represent some portion of the document. See www.isg.sfu.ca/~duchier/misc/vbush/vbush7.shtml ). This is a different copy of the same article that allows reference to section 7 directly. In this example, the original document was split into multiple HTML pages, each with a unique URL and hence could be referenced via the code .
Alternately, using HTML we could have added labels to the various sections of the original document and referenced the section via this label. For example if section 7 were modified with the label Section 7 , then Section 7 would take you directly to Section 7 of that document.
However, note that to accomplish this we have to modify the original document. Typically this is not achievable unless you are the author – and even then any document can be referenced in whole or in part by an arbitrarily large number of documents, rendering making a reference into a document via HTML impractical. As well, a page has no knowledge whatsoever of references being made to that page.
In SGML, a product called Panorama Pro from Softquad allows the construction of a separate file to reference any SGML document without modifying the original document. The reference, or collection of references, exists as a separate document encoded, according to the HyTime standard that provided explicit navigation into the structured document. For example, you might reference the 22nd to 43rd characters of the fourth paragraph, in the second section of Chapter 4. This particular application allows links between multiple annotation files and a source document without modifying the original document.
An interesting application called Thirdvoice (see www.thirdvoice.com) implements something similar. This Web-based application allows any user to annotate any Web page, start up dialogues around information on that page and make every word on the page "live" in the sense that if there is information about anything on the page, it can be made visible to the browser. Note that the referenced page viewed with Thirdvoice now appears to have knowledge about what pages are linked to it, all without modifying the original document. I have created a discussion group around the Atlantic Monthly article into which readers may contribute.
In XML, the Xlink and Xpointer standards (currently in Working Draft stage) offers a way to establish two way links between pages. Unlike the ThirdVoice solution, the Xlink solution would be non-proprietary. Xlink allows the construction of complex structured links between two or more "resources" associated with the same link item. These links may be in-line links (like the HTML lining mechanism) or links existing in an external file. Given these links may be structured and quite complex, we start to see technology that will allow the implementation of these "Vannevar Trails."
W. Hugh Chatfield, ISP, is president of CyberSpace Industries 2000 Inc. in Ottawa. He is engaged in XML/SGML consulting and teaching and can be reached at [email protected].
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Big Dunc's verdict – Jacob's Cracker and Blackburn (a)
By Duncan Edwards First day of the season, away from home; recent history suggested this would be a long old poke that would end up with a point at best. However, this was a strange day. Firstly, the weather was glorious in the traditionally grim North. The allocated away supporters pub, the Golden Cup, was […]
By Duncan Edwards
First day of the season, away from home; recent history suggested this would be a long old poke that would end up with a point at best.
However, this was a strange day. Firstly, the weather was glorious in the traditionally grim North. The allocated away supporters pub, the Golden Cup, was plenty hospitable enough if a little unprepared for the influx of yellow and green; "We weren't expecting this many.." said a bewildered landlady. Secondly, there was an air of confidence about the fans. Despite misgivings due to our horrendous opening day record, I'd gone as far as to predict a 3-1 victory and a "statement performance".
Ok, I got the score wrong but I was bang on with regard to the performance.
This wasn't a gulf in class, this was a "Grand Canyon and a half" sized chasm. City were simply exemplary at times. Ok, the inclusion of Jacob Murphy seemed a bit leftfield while we were sat in the pub but when he displayed the strength to hold off his man, get the ball out of his feet and let fly with an absolute snorter to give us the lead, leftfield it seemed no more.
It's early to make assumptions but in that one moment Jacob has perhaps helped answer the question of what we gain from the loan system. That season at Coventry in League One has helped him grow as a player and we reaped some immediate benefit yesterday. Interesting that he also got the nod over Josh. Before their season long loans, I'd have said Josh was definitely ahead, perhaps that's not the case now. Of course, I'm probably falling into the trap of treating them as identical players because they're twins.
I'm sure they have very different qualities, another day Josh might have been the chosen option. The positive is that it's clear Alex Neil isn't scared to play them.
Wes and Naismith were untouchable, Blackburn completely unable to cope with the little movements and fast intricate passing. They both scored too. It's hard to fathom that Wes could actually be improving as he approaches his testimonial but he's currently scoring for fun. If he's finally found the last piece of his enigmatic jigsaw, there won't be a more valuable player in the Championship this season.
There were other positives too. Cameron Jerome ran the Rovers defence ragged, scored his goal and showed what a good player is at this level.
Bennett got the nod over Bassong and made one excellent clearing header, the niggling worry remains that we'll retain that brittleness at the back though. Rovers' goal did little to quell that as Whittaker was beaten all too easily, we were caught beneath the cross and then there was the suspicion that Ruddy could have done better too.
Still, winning 4-1 is what counts and on the face of it, it looks like we have thrown down a statement of intent.
Let's not get carried away though, Blackburn are a poor, poor side. Yes, we still have to make them look that bad but while this is a great result, it shouldn't fool us into thinking it's straightforward from here.
The hard truth is that we still need a striker, probably two, to provide support, competition and at times replace Cameron Jerome.
We're told the money is there, we need to make it count. January is a long way off and the way things sit right now, we're one injury from seeing our season scuppered.
If the board were waiting for the team to show them that they warrant that investment, they couldn't have done more than they did at Ewood Park yesterday.
OTBC
Follow Duncan on Twitter @duncanedwards8
Introducing Alex Pritchard
By Jon Punt For years there's been an elephant in the room. For years we've wondered. For years it's seemed almost impossible. Yes, you've guessed it, for years we've never really had a natural successor for the god like Wes Hoolahan. So in time honoured tradition, you wait ages for a replacement, only for the […]
Delia out, toys out, pram empty
By Andrew Lawn So 1 week on from the end of the summer transfer window and frustration that we didn't sign a second striker has mostly dissipated into a realisation that while another striker would have been nice, our squad is still chocked full of talent at this level. Mostly. In some bedrooms across the […]