text:
Ishan Kacker
Mar 03, 2019 IST
West Indies are currently ranked at No.8 in the ICC ODI Team Rankings
With less than three months to go before the ICC Cricket World Cup 2019 kicks off in style in England and Wales, cricket pundits, all over the world, are busy picking their favorites for the tournament. For some, it may be Team India, and for others, the England Cricket team might be the front-runners.
But there seems to be some sort of a presumed consensus that the West Indies Cricket Team is quite down in the pecking order; maybe even below Afghanistan.
It is sincerely requested that one should not 'sleep' on the West Indies just yet. They are two-time World Cup Champions, to say the least. The Jason Holder-led Windies' will definitely put on a tough show for the fellow competing teams, as was evident from their performances in the series against India and England.
Chris Gayle will be retiring after the 2019 ICC Cricket World Cup
The return of familiar names to the squad coupled with the new, young talented individuals would definitely make Windies a formidable force. The English bowlers witnessed the first-hand wrath of the 'Gayle storm', who returned to form in the recently concluded ODI series, wherein Gayle amassed 424 runs in just 4 matches.
Jason Holder's 202 not-out was the third-highest score by a No.8 in Tests
Jason Holder, who has been a regular feature of the team, is leading from the front, contributing with both bat and ball. His recent form has pushed him atop the ICC Test All-Rounders rankings. He would want to continue his form well into the World Cup.
The inclusion of Shai Hope and Shimron Hetmyer in the batting line-up have strengthened the foundations of the team, which has lost several matches due to poor batting performances. Both Hope and Hetmyer put on incredible performances when the Windies toured India, showing complete dominance over both pacers and spinners.
The bowling department has also undergone a rejig of sorts and has come out looking a much more improved and balanced unit. Oshane Thomas and Sheldon Cottrell have infused new life into the pace department, reminiscent of the Windies of 'old', using sheer pace and bounce to render opposition batting-line ups obsolete.
Their performance in the recently concluded ODI series against England is a testimony to the fact that the West Indies are here to lay down their challenge for the upcoming World Cup. They were able to showcase their prowess against the No.1 ODI side in the world, and it seems that they have a lot left in them. Even when they lost, the West Indies went down fighting, as was evident in the 4th ODI of the series.
Given that this would be Chris Gayle's last World Cup before he hangs his boots, the Windies have one more reason to put on an incredible showing during the summer in England. The conditions would be ripe for their pace attack to cause destruction, and they would also want their batting department, featuring the likes of Chris Gayle, Darren Bravo, Shai Hope and Shimron Hetmyer to deliver the goods.
Cricket World Cup 2019 West Indies Cricket England Cricket Team Chris Gayle Jason Holder


text:
IN NUMBERS: Is it too soon for Italy to relax its coronavirus restrictions?
As Italy's government prepares to begin easing the strict coronavirus measures in place across the country, here's what the latest health data tells us.
Published: 19 April 2021 15:23 CEST
Updated: 20 April 2021 16:34 CEST
Photo: Filippo Monteforte/AFP
Italy last week unveiled plans to begin relaxing restrictions from the end of April. The news came as a relief to many – not least the business owners who've been protesting about the economic impact of the country's prolonged shutdown.
But many in the country have questioned whether it's really safe to do so yet amid a still-high infection rate and a sluggish vaccine rollout.
READ ALSO: Schools, restaurants, gyms, travel: Here's Italy's new timetable for reopening
The government insisted it was taking a "calculated risk" as it announced the reopening. However, some health experts warned on Monday that reopening too much too soon would risk triggering a new wave that could mean closures during the summer tourist season.
"This is a very delicate phase. If the curve starts rising again, we're risking the summer season," Dr Nino Cartabellotta, president of the Gimbe foundation for evidence-based medicine, told Italian Radio Cusano Campus on Monday.
He stressed that the government's plan relied heavily on the public following the rules, particularly on masks and social distancing.
And, with many older and more vulnerable people still not vaccinated in Italy, doctors have warned of the risks if infections were to rise again following a relaxation of the rules.
Medical workers' unions last week wrote to the government to urge caution, saying "any premature relaxation of restrictions could put the lives of Covid-19 patients at great risk".
"A slowdown of the restrictions will only be possible if daily infections remain below 5,000 cases, while maintaining a large capacity for testing, and resuming contact tracing to control the spread of the epidemic," their recommendation read.
The number of new cases nationwide is currently at around 15,000 daily.
"Hospitalizations would need to be far below the critical thresholds, and vaccinations complete at least for frail subjects and those over 60, the categories at the highest risk of hospitalization and mortality," the unions warned.
While the numbers are moving in the right direction, Italy has some way to go before those requirements are met.
"It is true that new cases are progressively reducing, but we have half a million positive cases, and that's an underestimated number," Cartabellotta said.
"Hospital admissions are decreasing, they have decreased by almost 20% in 11 days, but in the critical area and in intensive care the descent is slower, and we still have regions that are beyond the critical threshold," he said.
Photo: Marco Bertorello/AFP
"We need to be aware that reopening is happening on a tightrope."
The health situation in Italy varies considerably from region to region – which is why the restrictions currently vary across the country under a system of 'red zone' and 'orange zone' rules.
MAP: Which zone is your region in under Italy's coronavirus restrictions?
Most of the planned easing of restrictions from April 26th would only apply in areas that qualify as lower-risk 'yellow' zones – a classification which does not apply to any part of the country at the moment.
We won't know whether any regions actually qualify for 'yellow' zone status until Friday, April 23rd, when any changes will be announced based on the previous week's regional health data.
Meanwhile, Italy's vaccination programme appears to be speeding up, with a new daily record of almost 350,000 shots administered on Friday.
The latest tallies are welcome news after the country has faced a string of setbacks and missed targets in the vaccination rollout.
But Italy is still lagging behind the European Union average, with the number of people receiving one dose amounting to 16.9% of the population.
READ ALSO: How fast is Italy vaccinating its population compared to other European countries?
More than 10 million people in Italy have received at least one shot of a vaccine, according to official government figures.
Of those, 4.4 million have had both doses required for immunisation.
However, Italy is a long way from having all over-60s vaccinated.
Many of those in older age groups remain at risk, with only 3.4% of people aged 70-79 fully vaccinated so far.
IN CHARTS: Who is Italy vaccinating fastest?
The government is now aiming to get 80% of the population fully vaccinated "by autumn", which the prime minister said on Friday was an "achievable" target.
Despite recent improvements, concerns remain that the relatively slow pace of Italy's vaccination rollout will make it more difficult for the country to safely restart travel this summer.
With many of its own citizens and residents currently unable to access the vaccine, it's not yet known if Italy will allow vaccinated tourists to enter the country for summer holidays.
How much, and how soon, Italy is able to reopen businesses and restarts travel will depend on the health data in the coming weeks and months.
Can I travel to Italy if I've had both doses of the Covid vaccine?
OPINION: Bureaucratic barriers must not stop Italy vaccinating its foreign residents
The government has not yet confirmed any plans to relax the current travel restrictions. Nor has it said whether the country will definitely be taking part in the EU's Digital Green Certificates scheme, due to begin in June.
The proposed EU travel certificates will have information on whether a traveller has been vaccinated or not, if they have received a negative test result, or if they have recovered from Covid-19, allowing them to travel throughout the bloc more easily.
At the moment, the Italian government is discussing implementing a domestic travel "pass" that would allow holders to freely enter and leave Italy's higher-risk orange and red zones, however no details of the plan have yet been announced.
HEALTHSTATISTICSDATANEWSLETTERCOVID-19
Steve Minchington - 19 April 2021 at 20:27 CEST
There seems a strange logic to reopening the country here, its so scatter gun…. Less than a month ago Sardinia was a triumph…they went white a couple weeks later they were in red….My part of Tuscany was declared red weeks ago, yet surrounding areas in Tuscany stayed orange….and after weeks our numbers now still barely clear us from red zone!!!
We have to stop the talking and get vaccines in arms, of any type if they are approved. Not just for the sake of our populations health and mental wellbeing, but to save our tourist season…Many business lost easter business for a second year and they are now immanently looking at loosing a second summer…what are we doing here…Stop this procrastination…Look no further than the UK…hurts me to say it but they got there act together on this….they stumped up the money bought vaccines by the bucket load… and got it into people…over the last weeks they've are recording less deaths daily in the whole county than we have in just Tuscany daily…
Living in Italy: Five tips to help you survive the local pharmacy
How is Italy planning to tighten laws on smoking?
From ear piercings to flu jabs, Italian 'farmacie' are among the most useful stores in the country, but they're also very odd places. Here are our tips on getting through the pharmacy experience.
Italian pharmacies aren't just stores selling prescription or over-the-counter medicines.
As a customer, you'll find all sorts of natural remedies, basic health supplies and personal care items on their shelves.
You'll also be able to receive basic medical services (for instance, blood pressure checks, Covid tests and flu jabs) and some non-health-related ones (like getting your ears pierced!) in most branches.
READ ALSO: Reader question: Can I still get the flu vaccine in Italy?
But, while being extremely useful stores, Italian farmacie (pronunciation available here) are also peculiar places and their set of unwritten rules and solidified traditions may well throw off newcomers..
So here are five tips that might help you complete your first expeditions to your local pharmacy without making a fool of yourself.
1 – Decipher your doctor's scribbles before your trip
Much like some of their foreign colleagues, Italian GPs have a penchant for writing prescriptions that no one else is actually able to read.
We might never find out why doctors seem so intent on making ancient hieroglyphs fashionable again, but their calligraphic efforts will surely get in the way of you trying to buy whatever medicine you need to survive.
To avoid hiccups, make sure you know exactly what you need to get. If in doubt, reach out to your GP to confirm.
Don't rely on pharmacists being able to figure out your doctor's handwriting because they often have no clue either.
READ ALSO: EXPLAINED: How to make a doctor's appointment in Italy
In most small towns and rural areas local pharmacies have very 'thin' opening hours. Photo by Miguel MEDINA / AFP
2 – Double-check the pharmacy's opening times
If you're from the UK or the US, you might be used to pharmacies being open from 8am to 10pm on weekdays and having slightly reduced opening times over the weekend.
You can forget about that in Italy. In big cities, most pharmacies will shut no later than 8pm on weekdays and will be closed on either Saturdays or Sundays.
READ ALSO: Coughs, colds and flu: What to say and do if you fall sick in Italy
As for small towns or villages, opening times will have a nice Middle Ages vibe to them, with local stores remaining shut on weekends and keeping their doors open from 9am to 12.30pm and then from 3.30pm to 7.30pm on weekdays.
So always check your local pharmacy's hours before leaving home and, should their times not be available online, call them up. An awkward phone conversation with the pharmacist is still preferable to a wasted trip.
3 – Get the 'numerino'
Some Italian pharmacies have a ticket-dispensing machine with the aim of regulating the queue – a concept which is still foreign to many across the country.
All customers are expected to get a numbered paper ticket (the famed 'numerino') from the above machine and wait for their number to be called to walk up to the pharmacist's desk.
Now, the law of the land categorically prohibits customers from getting within a five-metre radius of the desk without a numerino.
Also, trying to break that rule may result in a number of disdainful sideways glances from local customers.
4 – You cannot escape the in-store conversations, so embrace them
Pharmacies aren't just stores. They're a cornerstone of Italian life and locals do a good deal of socialising on the premises.
After all, the waiting times are often a bit dispiriting, so how can you blame them for killing the time?
Pharmacies are an essential part of Italian life and culture. Photo by Marco SABADIN / AFP
You might think that locals won't want to talk to you because you're a foreigner or don't know the language too well, but you'll marvel at how chatty some are.
While chit-chat might not be your cup of tea, talking with locals might help you improve your Italian, so it's worth a shot.
5 – "Vuoi scaricarlo?"
The pharmacist finally gets you what you need and you're now thinking that your mission is over. Well, not yet.
Before charging you for the items in question, the pharmacist will ask you whether you'd like to 'scaricarli' (literally, 'offload them') or not, which, no matter how good your Italian is, will not make any sense to you.
What the pharmacist is actually asking you is whether you want to link the purchase to your codice fiscale (tax code).
READ ALSO: Codice fiscale: How to get your Italian tax code (and why you need one)
That's because Italy offers residents a 19-percent discount on some health-related expenses, which can be claimed through one's annual income declaration (dichiarazione dei redditi) by attaching the receipts of all the eligible payments.
Whether you want to scaricare or not, this is the last obstacle before you can make your way back home.
Giampietro Vianello [email protected]
'Absurd decision': Italy slams Ireland's plan to put health warnings on wine
OPINION: Italian healthcare is stuck in the past – but now it has a chance to modernise

redpajama_set_name: RedPajamaCommonCrawl


text:
Q: Need help to understand SQL query I have the following code that gets the months between two date ranges using a CTE
declare
    @date_start DateTime,
    @date_end DateTime

;WITH totalMonths AS
(
    SELECT
        DATEDIFF(MONTH, @date_start, @date_end) totalM
),
numbers AS
(
    SELECT 1 num

    UNION ALL

    SELECT n.num + 1 num
    FROM numbers n, totalMonths c
    WHERE n.num <= c.totalM
)
SELECT
    CONVERT(varchar(6), DATEADD(MONTH, numbers.num - 1, @date_start), 112)
FROM
    numbers
OPTION (MAXRECURSION 0);

This works, but I do not understand how it works
Especially this part
numbers AS
(
    SELECT 1 num

    UNION ALL

    SELECT n.num + 1 num
    FROM numbers n, totalMonths c
    WHERE n.num <= c.totalM
)

Thanks in advance, sorry for my English

A: This query is using two CTEs, one recursive, to generate a list of values from nothing (SQL isn't really good at doing this).
totalMonths AS (SELECT DATEDIFF(MONTH, @date_start, @date_end) totalM),

This is part is basically a convoluted way of binding the result of the DATEDIFF to the name totalM. This could've been implemented as just a variable if you can declare things:
DECLARE @totalM int = DATEDIFF(MONTH, @date_start, @date_end);

Then you would of course use @totalM to refer to the value.
numbers AS (
    SELECT 1 num
    UNION ALL
    SELECT n.num+1 num FROM numbers n, totalMonths c
    WHERE n.num<= c.totalM
)

This part is essentially a simple loop implemented using recursion to generate the numbers from 1 to totalMonths. The first SELECT specifies the first value (1) and the one after that specifies the next value, which is int greater than the previous one. Evaluating recursive CTEs has somewhat special semantics so it's a good idea to read up on them. Finally the WHERE specifies the stopping condition so that the recursion doesn't go on forever.
What all this does is generate an equivalent to a physical "numbers" table that just has one column the numbers from 1 onwards.
The SELECT at the very end uses the result of the numbers CTE to generate a bunch of dates.
Note that the OPTION (MAXRECURSION 0) at the end is also relevant to the recursive CTE. This disables the server-wide recursion depth limit so that the number generating query doesn't stop short if the range is very long, or a bothersome DBA set a very low default limit.

A: totalMonths query evaluates to a scalar result (single value) indicating the number of months that need to be generated. It probably makes more sense to just do this inline instead of using a named CTE.
numbers generates a sequence of rows with a column called num starting at 1 and ending at totalM + 1 which was computed in the previous step. It is able to reference this value by means of a cross join. Since there's only one row it essentially just appends that one column to the table horizontally. The query is recursive so each pass adds a new row to the result by adding 1 to the last added row (really just the one column) until the value of the previously added row exceeds totalM. The first half of the union is the starting value; the second half refers to itself via from numbers and incrementally builds the result in a sort of loop.
The output is derived from the numbers input. One is subtracted from each num giving a range from 0 to totalM and that value is treated as the number of months to add to the starting date. The date value is converted to a varchar of length six which means the final two characters containing the day are truncated.
Suppose that @date_start is January 31, 2016 and @date_end is March 1, 2016. There is never any comparison of the actual date values so it doesn't matter that March 31 is generated in the sequence but also falls later than the passed @date_end value. Any dates in the respective start and end months can be chosen to generate identical sequences.

A: SELECT 1 num

is your starting point of your recursive CTE and that is (numbers n) in the first teration.In the second iteration the out put of the first
SELECT n.num+1 num FROM numbers n, totalMonths c
WHERE n.num <= c.totalM

becomes numbers (n) and so on.

redpajama_set_name: RedPajamaStackExchange


text:
Home Social Media Instagram How to Lend More Punch to Your Email Marketing with Instagram
Instagram Social Media
How to Lend More Punch to Your Email Marketing with Instagram
Marketing is all about reaching out to customers with a credible and persuasive message. After the arrival of social media, many people were inclined to write off email as an effective mode of communication saying that it was too common and uninteresting to hold the attention of the Gen Y customers who are more used to the excitement and the interactivity provided by the social media.
However, studies have shown that email has consistently held on to its number one position in terms of ROI. However, the fact of email being effective does not in any way diminish the ability of social media to attract and engage with customers. The best marketers are now trying to stitch together marketing initiatives that try to combine the best of email marketing with that of social media.
Social Media – A Great Platform for Marketers
The prime reasons why marketers are drawn to the social media are its huge popularity, the frequency, and volume of use. Sharing original content or content from other sources is today a huge part of content management initiatives by marketers. While Facebook and LinkedIn had taken the first initiatives and remain relevant for many of their audiences even today, it's networks like Twitter and Instagram that have set the online marketing environment on fire with their huge popularity and level of interaction.
Those who may have looked upon Instagram's visual orientation as being irrelevant for serious marketing and brand building will find it surprising that today Instagram has over 800 million monthly active users who are young and look upon the network as a prime source of information about brands and businesses. With such a huge number of potential customers, no marketer worth its salt would today dare to ignore the potential of Instagram and thus are willing to buy real Instagram likes.
Instagram for Email Engagement
Instagram and email work so beautifully together because they are completely different. While Instagram is great for raising brand awareness and user engagement, it lacks the e-commerce potential of email simply because Instagram pictures are not clickable and you cannot initiate sales. The only way to access the website of the marketer is to click on the link allowed in the bio.
Since being able to achieve this consistently can be a challenge for most marketers, it is often a better technique to provoke the interest of the Instagram user with some outstanding visual content and the promise of something valuable for free when they sign up for an email subscription by visiting a specially designed landing page from the link in the bio section. Now the marketer is in a better position to directly communicate with the potential user with content that is customized to his profile and interests.
Using Instagram to raise the awareness level and obtain customer feedback is great but when you are able to get people who are interested in your business or brand to sign up for the email subscription, you are in a better position to get the conversions you want.
TagsE-commerceINSTAGRAMsocial media marketing
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Ski holiday and ski hire in Hafling with SNOWELL. The latest and top tuned models of skis you can rent in Hafling in South Tyrol - Trentino easily and comfortably online from your house and then start relaxed with your winter sports vacation in Hafling - Meran - Merano. But also the latest trends in snowboarding, freeriding and other winter sports you can rent online at our ski rental stations in Hafling in Italy - always centrally located and close to the ski resort Meran 2000.
Ski hire Hafling online - Just above Meran the two villages Hafling - Avelengo and Vöran are located. The accompanying resort, Meran 2000, belongs to the car-free zone and is highly recommended for families and beginners. It offers its visitors an absolutely stress-free zone during their winter holidays. The ski resort offers more than 40 km of slopes, which are marked mainly red and are very popular among snowboarders and skiers. The "Luckis Kinderland" welcomes its little guests and gives it utmost to make the stay as pleasant and fun as possible for the young skiers, without losing sight of the objective, namely, that they should learn to ski. The ski area in Hafling - Avelengo is really ideal for this. Freeriders will find deep snow fun on the non-groomed slopes. Furthermore, there is a cool snowpark for freestylers and snowboarders, which was recently opened. Night skiing in the ski resort Meran 2000 is certainly worth mentioning. It is a nice tradition and should not be missed. The beautiful work horses, the famous Haflinger horses come from this area and are being used for the wonderful horse sleigh rides. Near Hafling - Avelengo one can also do cross-country skiing. Besides natural toboggan runs, there is another attraction near Hafling - Avelengo. An Alpine Bob winds down over one kilometer into the valley and offers plenty of fun and thrills for young and old.
Using SNOWELL you can rent your skis and snowboards online in advance in Hafling and save money at the same time. At the time of your arrival you can pick up your booked rental equipment directly at the co-operating ski hire shop directly in Hafling. An extremely practical but also economical solution for your ski hire in Hafling.


text:
Assessing the feasibility and effectiveness of COMPASS programming to prevent violence against adolescent girls
Significant gaps persist in the evidence base on the most effective interventions to prevent violence against adolescent girls aged 10–19 years old in humanitarian crisis settings. The Creating Opportunities through Mentorship, Parental involvement, and Safe Spaces (COMPASS) program provides opportunities for girls to gain life skills and build assets to protect against and respond to gender-based violence (GBV) through mentorship, learning, and peer interaction in safe spaces, and is combined with capacity-building activities to improve the ability of service providers and caregivers to address the specific needs of young girls. The feasibility, acceptability, and impact of these activities on reducing violence experienced by young girls were assessed in Ethiopia, the DRC, and Pakistan.
Approximately 900 girls in the DRC and their caregivers, as well as nearly 1,000 girls in Ethiopia were interviewed as part of two independent cluster-randomized controlled trials. The primary outcome was sexual violence while secondary outcomes included family violence, early marriage, positive relationships, and gender norms. More than 200 girls participated in a pre-and post-test in Pakistan, which centered on feasibility and acceptability. Specific research questions included:
Ethiopia - What is the overall impact of the COMPASS program on girls' experiences of violence and well-being compared to a waitlist group?
DRC - What is the incremental impact of the parental component of the COMPASS program in addition to the girls' programming on girl's experiences of violence and well-being compared to girls' programming only?
Pakistan - What is the feasibility and acceptability of delivering COMPASS?
Findings from these studies combined indicate very high acceptability and feasibility of the COMPASS program. While no significant changes related to violence were observed, numerous secondary outcomes such as gender norms, aspirational attitudes, and increased social networks were improved. Results of this work have led to an adapted adolescent girl program, Girl Shine.
Research Brief - Democratic Republic of Congo
Research Brief - Ethiopia
Research Brief - Pakistan
COMPASS program: Multi-country study protocol to protect girls from violence in humanitarian settings
Implementation of Audio-computer assisted self-interview (ACASI) among adolescent girls in humanitarian settings: feasibility, acceptability and lessons learned
The effect of gender norms on the association between violence and hope among girls in the Democratic Republic of the Congo
Caregiver parenting and gender attitudes: associations with violence against girls in South Kivu, Democratic Republic of Congo;* (8) How narratives of fear shape girls' participation in community life in two conflict-affected populations
Disclosure bias for group versus individual reporting of violence amongst conflict-affected adolescent girls in DRC and Ethiopia;
Prevalence and associated risk factors of violence against conflict- affected female adolescents: a multi-country, cross-sectional study
Journal: How gender and violence related norms affect self-esteem among adolescent refugee girls living in Ethiopia.
Journal: The school participation effect: investigating violence and formal education among girls in the Democratic Republic of the Congo
Report: A Safe Place to Shine - Creating Opportunities and Raising Voices of Adolescent Girls in Humanitarian Settings


I've got an intel core i3 with 2gb of ram, and 500gb SATA harddisk space, will it work?
Which items should I click on the ktexts that appear before the installation process begin.
I've also another laptop with the same specifications but it has 4gb of RAM. Is that ok? Will it work for both of them? I'm sorry, I'm new to this.


Sunday, June 7, 2009, somewhere in Bibb County, Ala. Joe Guy eats his breakfast in a field along Highway 82. He'll spend the next several hours, the hottest part of the day, here. He broke (started) "Bon Jovi," his horse, only a week ago and doesn't want to put him to the limit.


I have been looking for a photograph of the exterior of Town School for several years, so far without success. Recently, however, in my surfing of the internet, I accidentally came across one. I recognised it instantly, but you can imagine my surprise when the title of the photograph was "Trosnant Board School".
I thought there couldn't possibly be another school that looked so exactly like Town School in every detail so I contacted Brian Jenkins, the author of "Mill Road" who lived in Trosnant when he was young, and sent him a copy of the photograph. He confirmed that it was quite definitely Town School. He was not aware of there ever being a school in Trosnant.
The photograph was black and white and slightly blurred so I enhanced it on my computer and also added some colour and a few figures to liven it up a bit. The door into boys' cloakroom was closed as was the iron gate which closed off the entry into the girls' cloakroom. I therefore opened the door and took away the gate.
The small plaque just above the shoulder of the man about to cross the road bore the date of the opening of the school: 1838. The narrow alleyway just behind the girl in the yellow dress was the entrance to the girls' cloakroom. Immediately at the end of the alleyway was a step and to the left a door into the classroom of Mr Rees.
If you carried on through the girls' cloakroom you passed into the alleyway between the back of the school and the high retaining wall on your right. At the end of the alleyway to the right was a flight of steps leading up to the top class playground. On the far left of the playground was "The Bungalow" where Mr Petty taught the 11+ class. Just before the entrance to this building to the left was a wide wooden gate which led into a lane. If you turned right you walked out into Bridge Street.
The green door in the photograph, to the left of the young boy, was the main entrance to the school. It led into the boys' cloakroom. The left hand wall had a row of wash basins; the other three walls were covered with pegs for hanging up our coats etc. At the far end of this room on the right were a few steps which led up past Miss Brooks' room along a corridor with a door on the left into the playground. On the right were two steps leading into the main corridor. The headmaster's office was on the right with the school bell tower, visible almost top centre in the photograph, and bell (hardly ever used) above.
In a recess to the left was the door into Mr Rees's room. The other classes ran off the corridor to the right. At the far end of this corridor on the left was the entrance to the Infants' Department.
The only other photograph of the school I have is in the Centenary Booklet and is of the playground. It appears elsewhere on this blog.
If anyone has any more photographs of the school I'd be pleased to include them here.


text:
Q: What is the *.def file at c++ project? I compile old c++ project with visual studio 2013.
I get error:
LNK114:cannot open file 'Banana.def' at my Banana project.

What is it? And how to fix it?
P.S. i am new at c++ on visual studio.
Yes, i building DLL.

A: It's a module definition file, probably specifying the exports of a DLL library.
You're missing the file, or it's not accessible.

A: The .def file is normally not used for building an exe. Extract from LINK documentation :
A .def file is most useful when building a DLL. Because there are linker options that can be used instead of module-definition statements, .def files are generally not necessary. You can also use __declspec(dllexport) as a way to specify exported functions ... If you are building an .exe file that has no exports, using a .def file will make your output file larger and slower loading.
Unless you are building a DLL, banana.def should not be used.


The unknown knight would seem to belong in the realm of fiction - witness Gareth in Le Morte d'Arthur and Wilfred in Ivanhoe, among others - but incognito knights fighting in jousts or melees were not uncommon in reality.
In January 1510, Henry VIII, together his Groom of the Stool and courtier William Compton, entered a joust as 'Stranger' knights with their identity hidden. This was to circumvent a ban on the teenage king taking part in jousts, a ban which had been imposed by his father Henry VII for the obvious reason that it was too dangerous a sport for the then heir to take part in.
Although his father was now dead, the new king was too young to openly flout the ban which the senior officials of the realm supported; his wife, Catherine of Aragon, was pregnant but Henry had no heir. The King was forced to reveal himself when Compton sustained a serious injury; one of the few people who knew who the stranger knights really were panicked, thinking that it was Henry who injured.
Are there any other cases of a King, identity unknown at the time to those he was fighting and to the general audience, taking part in a joust or a melee?
As this question seems to be quite tough, I would also accept an answer for an heir to the throne or, failing any such examples, the legitimate son of a reigning monarch.
Yes. King Edward III took part in more than 50 tournaments, sometimes incognito. His son and heir, the Black Prince, also jousted incognito, as did many knights during Edward III's reign.
The knights were in uniform costumes of green tunics and mantles with red hoods....To conceal the king's identity, they were all masked.
The easily accessible article Tournaments in the Fourteenth Century also mentions this.
In 1334 at a major tournament at Dunstable the king appeared under the arms of the Arthurian knight Sir Lionel. On several later occasions he appeared wearing the arms of members of his household entourage. Participating incognito in the manner of Arthurian heroes was a way of linking the fantasy world of romance to the practical world of preparation for war.
Edward III appeared several times at tournaments as Sir Lionel, though, so presumably someone eventually figured out who 'Sir Lionel' was.
Edward III was a great patron of tournaments, in which he himself was often an eager participant....often taking part incognito. There was a playacting element, as when Edward and some knights dressed as the Pope and some cardinals at a tournament held at Smithfield in 1343.
Meetings of the Garter were often accompanied by splendid tournaments, many of which were staged in true Arthurian style with knights fighting incognito or wearing fantastic costumes. Both Edward III and the Black Prince are known to have participated wearing disguises or costumes.
Remarkably perhaps, even the donning of disguises in tourneying served a practical purpose. It made for equality between competitors by ensuring that a lesser knight did not defer to the superior blood of his rival.
This question went unanswered for 3 months, and I'd given up looking myself. I came across this answer completely by chance but, perhaps with hindsight, Edward III is as obvious a candidate as anyone.
If anyone can find any other clear examples of kings or heirs jousting incognito, I'll accept that answer ahead of my own. For those interested, Richard II is an unlikely candidate (only one joust apparently), Henry V didn't like jousting (though this doesn't necessarily rule him out), while Edward I took part in many tournaments but doing it incognito doesn't seem to have been common then - still, maybe worth investigating. Also, this question is not restricted to English kings.
Not quite an heir to the throne, but while second-in-line to the throne of England, Henry II's son Geoffrey was killed in a joust in Paris, according to the contemporary chronicler Roger of Hoveden. I don't know whether he would have been jousting as an "unknown", and another contemporary chronicler, Rigord, describes Geoffrey as having died suddenly after an onset of chest pain.
Not the answer you're looking for? Browse other questions tagged middle-ages monarchy early-modern sports or ask your own question.
Why did the Australian IOC deny that Peter Norman was excluded from the 1972 Olympics for supporting racial equality?
Henry I. (the Fowler): What kind of contact to hostages was seen as appropriate?
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text:
Search underway for two murder suspects who escaped from California jail
MONTEREY, Calif. - Officials are trying to find out how two men charged with murder were able to escape from a central California jail.
The Monterey County Sheriff's Office warned that the fugitives should be considered dangerous. Authorities gave no other details on how they broke out of jail in the city of Salinas early Sunday.
Authorities say 21-year-old Santos Fonseca and 20-year-old Jonathan Salazar had been behind bars since 2018 and were awaiting trial on murder counts and "numerous other felony charges" in separate cases.
The Californian newspaper in Salinas reports that Fonseca is accused of killing two men in two different shootings in June 2018.
The newspaper says Salazar was arrested in the shooting death of man who was driving with his girlfriend and young son in October 2017.


Are you an experienced family law attorney interested in a contract job opportunity in Vancouver, WA? The ideal candidate will have strong written and verbal communication skills and excellent multitasking abilities. This role offers a very comparative salary and benefits package.
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Organic shaped floating homes will be unveiled by New Living on Water at Cityscape Global, 6-8 September, DWTC.
Dubai, UAE, 15 August 2016: Dubai has earned a global reputation for launching unique and spectacular real estate projects, and a Dutch developer hopes the emirate will now provide a successful springboard for stunning floating homes designed to take their owners back to nature.
"A lot of people nowadays live in very densely populated areas, and for most of them this is no problem in everyday life," said NLW's managing director and co-owner, Menno de Roos. "But the need for some privacy and the inner need of being connected to nature has always been with us, and that's what we're offering.


text:
New CEO joins eMed
Mark Smith is the new CEO and chairman of the board at eMed, a developer of PACS and Web-based systems. Smith has held positions as chairman and CEO of several growing companies, most recently the Potomac Group, a holding company for healthcare software
Mark Smith is the new CEO and chairman of the board at eMed, a developer of PACS and Web-based systems. Smith has held positions as chairman and CEO of several growing companies, most recently the Potomac Group, a holding company for healthcare software and services.


text:
Poliovirus
Poliovirus, the causative agent of polio (also known as poliomyelitis), is a member virus of Enterovirus C, in the family of Picornaviridae.[1]
Poliovirus 1
TEM micrograph of poliovirus virions. Scale bar, 50 nm.
A type 3 poliovirus capsid coloured by chains
Virus classification
(unranked): Virus
Realm: Riboviria
(unranked): incertae sedis
Order: Picornavirales
Family: Picornaviridae
Genus: Enterovirus
Species: Enterovirus C
Virus:
Poliovirus is composed of an RNA genome and a protein capsid. The genome is a single-stranded positive-sense RNA genome that is about 7500 nucleotides long.[2] The viral particle is about 30 nm in diameter with icosahedral symmetry. Because of its short genome and its simple composition—only RNA and a nonenveloped icosahedral protein coat that encapsulates it, poliovirus is widely regarded as the simplest significant virus.[3]
Poliovirus was first isolated in 1909 by Karl Landsteiner and Erwin Popper.[4] In 1981, the poliovirus genome was published by two different teams of researchers: by Vincent Racaniello and David Baltimore at MIT[5] and by Naomi Kitamura and Eckard Wimmer at Stony Brook University.[6] Poliovirus is one of the most well-characterized viruses, and has become a useful model system for understanding the biology of RNA viruses.
Replication cycleEdit
The replication cycle of poliovirus is initiated (1) by binding to the cell surface receptor CD155. The virion is taken up via endocytosis, and the viral RNA is released (2). Translation of the viral RNA occurs by an IRES-mediated mechanism (3). The polyprotein is cleaved, yielding mature viral proteins (4). The positive-sense RNA serves as template for complementary negative-strand synthesis, producing double-stranded replicative form (RF) RNA(5). Many positive strand RNA copies are produced from the single negative strand (6). The newly synthesized positive-sense RNA molecules can serve as templates for translation of more viral proteins (7) or can be enclosed in a capsid (8), which ultimately generates progeny virions. Lysis of the infected cell results in release of infectious progeny virions (9).[7]
Poliovirus infects human cells by binding to an immunoglobulin-like receptor, CD155 (also known as the poliovirus receptor or PVR)[8][9] on the cell surface.[10] Interaction of poliovirus and CD155 facilitates an irreversible conformational change of the viral particle necessary for viral entry.[11][12] Attached to the host cell membrane, entry of the viral nucleic acid was thought to occur one of two ways: via the formation of a pore in the plasma membrane through which the RNA is then "injected" into the host cell cytoplasm, or that the virus is taken up by receptor-mediated endocytosis.[13] Recent experimental evidence supports the latter hypothesis and suggests that poliovirus binds to CD155 and is taken up by endocytosis. Immediately after internalization of the particle, the viral RNA is released.[14]
Poliovirus is a positive-stranded RNA virus. Thus, the genome enclosed within the viral particle can be used as messenger RNA and immediately translated by the host cell. On entry, the virus hijacks the cell's translation machinery, causing inhibition of cellular protein synthesis in favor of virus-specific protein production.[15] Unlike the host cell's mRNAs, the 5' end of poliovirus RNA is extremely long—over 700 nucleotides—and highly structured. This region of the viral genome is called internal ribosome entry site (IRES), and it directs translation of the viral RNA. Genetic mutations in this region prevent viral protein production.[16][17][18] The first IRES to be discovered was found in poliovirus RNA.[19]
Poliovirus mRNA is translated as one long polypeptide. This polypeptide is then autocleaved by internal proteases into about 10 individual viral proteins. Not all cleavages occur with the same efficiency. Therefore, the amounts of proteins produced by the polypeptide cleavage vary: for example, smaller amounts of 3Dpol are produced than those of capsid proteins, VP1–4.[20][21] These individual viral proteins are:[3][22]
The genomic structure of poliovirus type 1[7]
3Dpol, an RNA dependent RNA polymerase whose function is to make multiple copies of the viral RNA genome
2Apro and 3Cpro/3CDpro, proteases which cleave the viral polypeptide
VPg (3B), a small protein that binds viral RNA and is necessary for synthesis of viral positive and negative strand RNA
2BC, 2B, 2C (an ATPase)[23], 3AB, 3A, 3B proteins which comprise the protein complex needed for virus replication.
VP0, which is further cleaved into VP2 and VP4, VP1 and VP3, proteins of the viral capsid
After translation, transcription and genome replication which involve a single process (synthesis of (+) RNA) is realized. For the infecting (+) RNA to be replicated, multiple copies of (−) RNA must be transcribed and then used as templates for (+) RNA synthesis. Replicative intermediates (RIs) which are an association of RNA molecules consisting of a template RNA and several growing RNAs of varying length, are seen in both the replication complexes for (−) RNAs and (+) RNAs. The primer for both (+) and (−) strand synthesis is the small protein VPg, which is uridylylated at the hydroxyl group of a tyrosine residue by the poliovirus RNA polymerase at a cis-acting replication element located in a stem-loop in the virus genome. Some of the (+) RNA molecules are used as templates for further (−) RNA synthesis, some function as mRNA, and some are destined to be the genomes of progeny virions.[20]
In the assembly of new virus particles (i.e. the packaging of progeny genome into a procapsid which can survive outside the host cell), including, respectively:[24]
Five copies each of VP0, VP3, and VP1 which its N termini and VP4 form interior surface of capsid, assemble into a 'pentamer' and 12 pentamers form a procapsid. (The outer surface of capsid is consisting of VP1, VP2, VP3; C termini of VP1 and VP3 form the canyons which around each of the vertices; around this time, the 60 copies of VP0 are cleaved into VP4 and VP2.)
Each procapsid acquires a copy of the virus genome, with VPg still attached at the 5' end.
Fully assembled poliovirus leaves the confines of its host cell by lysis[25] 4 to 6 hours following initiation of infection in cultured mammalian cells.[26] The mechanism of viral release from the cell is unclear,[2] but each dying cell can release up to 10,000 polio virions.[26]
Drake demonstrated that poliovirus is able to undergo multiplicity reactivation.[27] That is, when polioviruses were irradiated with UV light and allowed to undergo multiple infections of host cells, viable progeny could be formed even at UV doses that inactivated the virus in single infections.
Origin and serotypesEdit
Poliovirus is structurally similar to other human enteroviruses (coxsackieviruses, echoviruses, and rhinoviruses), which also use immunoglobulin-like molecules to recognize and enter host cells.[9] Phylogenetic analysis of the RNA and protein sequences of poliovirus suggests that it may have evolved from a C-cluster Coxsackie A virus ancestor, that arose through a mutation within the capsid.[28] The distinct speciation of poliovirus probably occurred as a result of change in cellular receptor specificity from intercellular adhesion molecule-1 (ICAM-1), used by C-cluster Coxsackie A viruses, to CD155; leading to a change in pathogenicity, and allowing the virus to infect nervous tissue.
The mutation rate in the virus is relatively high even for an RNA virus with a synonymous substitution rate of 1.0 x 10−2 substitutions/site/year and non synonymous substitution rate of 3.0 x 10−4 substitutions/site/year.[29] Base distribution within the genome is not random with adenosine being less common than expected at the 5' end and higher at the 3' end.[30] Codon use is not random with codons ending in adenosine being favoured and those ending in cytosine or guanine being avoided. Codon use differs between the three genotypes and appears to be driven by mutation rather than selection.[31]
The three serotypes of poliovirus, PV1, PV2, and PV3, each have a slightly different capsid protein. Capsid proteins define cellular receptor specificity and virus antigenicity. PV1 is the most common form encountered in nature, but all three forms are extremely infectious.[4] As of November 2015, wild PV1 is highly localized to regions in Pakistan and Afghanistan. Wild PV2 was declared eradicated in September 2015 after last being detected in October 1999 in Uttar Pradesh, India.[32] As of November 2015, wild PV3 has not been seen since its 2012 detection in parts of Nigeria and Pakistan.[33]
Specific strains of each serotype are used to prepare vaccines against polio. Inactive polio vaccine is prepared by formalin inactivation of three wild, virulent reference strains, Mahoney or Brunenders (PV1), MEF-1/Lansing (PV2), and Saukett/Leon (PV3). Oral polio vaccine contains live attenuated (weakened) strains of the three serotypes of poliovirus. Passaging the virus strains in monkey kidney epithelial cells introduces mutations in the viral IRES, and hinders (or attenuates) the ability of the virus to infect nervous tissue.[26]
Polioviruses were formerly classified as a distinct species belonging to the genus Enterovirus in the family Picornaviridae. In 2008, the Poliovirus species was eliminated and the three serotypes were assigned to the species Human enterovirus C (later renamed Enterovirus C), in the genus Enterovirus in the family Picornaviridae. The type species of the genus Enterovirus was changed from Poliovirus to (Human) Enterovirus C.[34]
PathogenesisEdit
Main article: Polio
Electron micrograph of poliovirus
The primary determinant of infection for any virus is its ability to enter a cell and produce additional infectious particles. The presence of CD155 is thought to define the animals and tissues that can be infected by poliovirus. CD155 is found (outside of laboratories) only on the cells of humans, higher primates, and Old World monkeys. Poliovirus is, however, strictly a human pathogen, and does not naturally infect any other species (although chimpanzees and Old World monkeys can be experimentally infected).[35]
The CD155 gene appears to have been subject to positive selection.[36] The protein has several domains of which domain D1 contains the polio virus binding site. Within this domain, 37 amino acids are responsible for binding the virus.
Poliovirus is an enterovirus. Infection occurs via the fecal–oral route, meaning that one ingests the virus and viral replication occurs in the alimentary tract.[37] Virus is shed in the feces of infected individuals. In 95% of cases only a primary, transient presence of viremia (virus in the bloodstream) occurs, and the poliovirus infection is asymptomatic. In about 5% of cases, the virus spreads and replicates in other sites such as brown fat, reticuloendothelial tissue, and muscle. The sustained viral replication causes secondary viremia and leads to the development of minor symptoms such as fever, headache, and sore throat.[38] Paralytic poliomyelitis occurs in less than 1% of poliovirus infections. Paralytic disease occurs when the virus enters the central nervous system (CNS) and replicates in motor neurons within the spinal cord, brain stem, or motor cortex, resulting in the selective destruction of motor neurons leading to temporary or permanent paralysis. In rare cases, paralytic poliomyelitis leads to respiratory arrest and death. In cases of paralytic disease, muscle pain and spasms are frequently observed prior to onset of weakness and paralysis. Paralysis typically persists from days to weeks prior to recovery.[39]
In many respects, the neurological phase of infection is thought to be an accidental diversion of the normal gastrointestinal infection.[13] The mechanisms by which poliovirus enters the CNS are poorly understood. Three nonmutually exclusive hypotheses have been suggested to explain its entry. All theories require primary viremia. The first hypothesis predicts that virions pass directly from the blood into the central nervous system by crossing the blood–brain barrier independent of CD155.[40] A second hypothesis suggests that the virions are transported from peripheral tissues that have been bathed in the viremic blood, for example muscle tissue, to the spinal cord through nerve pathways via retrograde axonal transport.[41][42][43] A third hypothesis is that the virus is imported into the CNS via infected monocytes or macrophages.[7]
Poliomyelitis is a disease of the central nervous system. However, CD155 is believed to be present on the surface of most or all human cells. Therefore, receptor expression does not explain why poliovirus preferentially infects certain tissues. This suggests that tissue tropism is determined after cellular infection. Recent work has suggested that the type I interferon response (specifically that of interferon alpha and beta) is an important factor that defines which types of cells support poliovirus replication.[44] In mice expressing CD155 (through genetic engineering) but lacking the type I interferon receptor, poliovirus not only replicates in an expanded repertoire of tissue types, but these mice are also able to be infected orally with the virus.[45]
Immune system avoidanceEdit
CD155 molecules complexed with a poliovirus particle. Reconstructed image from cryo-electron microscopy.
Poliovirus uses two key mechanisms to evade the immune system. First, it is capable of surviving the highly acidic conditions of the stomach, allowing the virus to infect the host and spread throughout the body via the lymphatic system.[3] Second, because it can replicate very quickly, the virus overwhelms the host organs before an immune response can be mounted.[46] If detail is given at the attachment phase; poliovirus with canyons on the virion surface have virus attachment sites located in pockets at the canyon bases. The canyons are too narrow for access by antibodies, so the virus attachment sites are protected from the host's immune surveillance, while the remainder of the virion surface can mutate to avoid the host's immune response.[47]
Individuals who are exposed to poliovirus, either through infection or by immunization with polio vaccine, develop immunity. In immune individuals, antibodies against poliovirus are present in the tonsils and gastrointestinal tract (specifically IgA antibodies) and are able to block poliovirus replication; IgG and IgM antibodies against poliovirus can prevent the spread of the virus to motor neurons of the central nervous system.[26] Infection with one serotype of poliovirus does not provide immunity against the other serotypes, however second attacks within the same individual are extremely rare[citation needed] .
PVR transgenic mouseEdit
Although humans are the only known natural hosts of poliovirus, monkeys can be experimentally infected and they have long been used to study poliovirus. In 1990–91, a small animal model of poliomyelitis was developed by two laboratories. Mice were engineered to express a human receptor to poliovirus (hPVR).[48][49]
Unlike normal mice, transgenic poliovirus receptor (TgPVR) mice are susceptible to poliovirus injected intravenously or intramuscularly, and when injected directly into the spinal cord or the brain.[50] Upon infection, TgPVR mice show signs of paralysis that resemble those of poliomyelitis in humans and monkeys, and the central nervous systems of paralyzed mice are histocytochemically similar to those of humans and monkeys. This mouse model of human poliovirus infection has proven to be an invaluable tool in understanding poliovirus biology and pathogenicity.[51]
Three distinct types of TgPVR mice have been well studied:[52]
In TgPVR1 mice, the transgene encoding the human PVR was incorporated into mouse chromosome 4. These mice express the highest levels of the transgene and the highest sensitivity to poliovirus. TgPVR1 mice are susceptible to poliovirus through the intraspinal, intracerebral, intramuscular, and intravenous pathways, but not through the oral route.
TgPVR21 mice have incorporated the human PVR at chromosome 13. These mice are less susceptible to poliovirus infection through the intracerebral route, possibly because they express decreased levels of hPVR. TgPVR21 mice have been shown to be susceptible to poliovirus infection through intranasal inoculation, and may be useful as a mucosal infection model.[53]
In TgPVR5 mice, the human transgene is located on chromosome 12. These mice exhibit the lowest levels of hPVR expression and are the least susceptible to poliovirus infection.
Recently, a fourth TgPVR mouse model was developed. These "cPVR" mice carry hPVR cDNA, driven by a β-actin promoter, and have proven susceptible to poliovirus through intracerebral, intramuscular, and intranasal routes. In addition, these mice are capable of developing the bulbar form of polio after intranasal inoculation.[53]
The development of the TgPVR mouse has had a profound effect on oral poliovirus vaccine (OPV) production. Previously, monitoring the safety of OPV had to be performed using monkeys, because only primates are susceptible to the virus. In 1999, the World Health Organization approved the use of the TgPVR mouse as an alternative method of assessing the effectiveness of the vaccine against poliovirus type-3. In 2000, the mouse model was approved for tests of vaccines against type-1 and type-2 poliovirus.[54]
Cloning and synthesisEdit
Model of poliovirus-binding CD155 (shown in purple)
In 1981, Racaniello and Baltimore used recombinant DNA technology to generate the first infectious clone of an animal RNA virus, poliovirus. DNA encoding the RNA genome of poliovirus was introduced into cultured mammalian cells and infectious poliovirus was produced.[55] Creation of the infectious clone propelled understanding of poliovirus biology, and has become a standard technology used to study many other viruses.
In 2002, Eckard Wimmer's group at Stony Brook University succeeded in synthesizing poliovirus from its chemical code, producing the world's first synthetic virus.[56] Scientists first converted poliovirus's published RNA sequence, 7741 bases long, into a DNA sequence, as DNA was easier to synthesize. Short fragments of this DNA sequence were obtained by mail-order, and assembled. The complete viral genome was then assembled by a gene synthesis company. This whole painstaking process took two years. Nineteen markers were incorporated into the synthesized DNA, so that it could be distinguished from natural poliovirus. Enzymes were used to convert the DNA back into RNA, its natural state. Other enzymes were then used to translate the RNA into a polypeptide, producing functional viral particle. The newly minted synthetic virus was injected into PVR transgenic mice, to determine if the synthetic version was able to cause disease. The synthetic virus was able to replicate, infect, and cause paralysis or death in mice. However, the synthetic version was between 1/1,000th and 1/10,000th as lethal as the original virus.[57]
Modification for therapiesEdit
A modification of the poliovirus, called PVSRIPO, was tested in early clinical trials as a possible treatment for cancer.[58][needs update]
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^ Acute Poliomyelitis at eMedicine
Pediatric Poliomyelitis at eMedicine
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Medicine portal
Viruses portal
ICTVdb virus classification 2006
Home of Picornaviruses (latest updates of species, serotypes, & proposed changes)
Goodsell, David. "Poliovirus and Rhinovirus". August 2001 Molecule of the Month.
3D macromolecular structures of the Poliovirus archived in the EM Data Bank(EMDB)
"Human poliovirus 1". NCBI Taxonomy Browser. 12080.
Retrieved from "https://en.wikipedia.org/w/index.php?title=Poliovirus&oldid=903052307"


This is an e-book copy of the beautiful, hard to find Le Bon Ton and Le Moniteur de la Mode sewing pattern magazine. It was printed in English, in the U.S. by S.T. Taylor to advertise their sewing patterns and pattern drafting system.
The fashion illustrations within are absolute perfection and this particular one has all of its full-sized fashion plates included. These are usually taken out and sold separately and are very hard to find still included with the magazine! Two interior and two exterior pages are in color.
This edition is January/ Winter of 1909, Vol. 58, No. 1.
Each page measures a stunning 11 x 16".
Digital Reproduction - Copyright Mrs. Depew Vintage LLC, 2017.


Self-catering Villa with 5 bedrooms and 4 bathrooms. Sleeps 12. Saturday changeover.
Self-catering Villa with 5 bedrooms and 4 bathrooms. Sleeps 10. Saturday changeover.
Self-catering Villa with 3 bedrooms and 4 bathrooms. Sleeps 8. Saturday changeover.
Self-catering Villa with 5 bedrooms and 0 bathrooms. Sleeps 20. Non-applicable changeover.
Self-catering Villa with 5 bedrooms and 5 bathrooms. Sleeps 10. Non-applicable changeover.


Dr. Oz – Just 10 lbs.
This past week, I once again has the pleasure of meeting with Dr. Oz and the Fox Chicago "Ozzer's." Dr. Oz wanted to talk to the Chicago group about their weight loss challenges and successes since starting his weight loss challenge in January. Dr. Oz offered everyone an opportunity to ask questions. Many of the questions were poignant and probably ones you might have wanted to ask as well. "How do I stay motivated when my family isn't behind me?" " I am 46 years old and exercise regularly, yet I still find myself gaining weight?" "I've only lost 35 pounds in a year, and I'm frustrated, it should be coming off faster." Dr. Oz, in his kind way, responded to these questions as I had hoped, with practical, insightful and reasonable responses.
Dr. Oz, like me, believes that weight loss and finding a healthy weight is different for everyone. In other words, not everyone is meant to be 5'8″ tall and 130 pounds, not the way we were designed. He believes that any amount of weight loss triggers positive changes in the body and results in improved health. Dr. Oz also noted that anything extreme or crazy is always going to be temporary and his goal, like me, is for people to find a healthy lifestyle that is sustainable.
There is so much misinformation about weight loss. So often, clients come in for the first time with their head spinning. With the profile that Dr. Oz has, it's so nice to see that he is spreading the word that small changes can add up to better health. I've been touting that for years and finally, I have a respected voice that echoes that same sentiment.
So what can losing simply 10 pounds do for you?
Well, Dr. Oz stresses that even losing 10 pounds can make a huge difference in your health.
"The benefits of losing just 10 pounds are enormous for your heart, liver, knees, pancreas and blood pressure. The incidence of heart disease, diabetes and arthritis can be cut in half," said Dr. Oz.
So there you have it, just 10 pounds can make a positive difference in your health and quality of life. If you're local, hopefully you can join me either on October 13th or October 20th at 7:00 p.m. as I share my thoughts and insight into why weight loss alludes us and how you can begin and sustain a healthy, manageable weight and why just 10 pounds can be the start to a longer, healthier life!
Here's to your healthy living success!
BTW, it's not too late to join the Fox Chicago "Oz" group.
If you look at the most common reason people fall away from weight loss efforts, it's motivation. The excitement and enthusiasm felt when starting a weight loss plan is like the first day of school, the possibilities are endless and everything is new and fresh. But into the third or fourth week, the newness wears off and the reality of hard work sets in, no so fun anymore. But unlike weight loss programs, school is a must (well, I'd like to think it is) and the motivation to stick with it is far greater. But with weight loss, if you quit, you quit. You walk away quietly and no one knows, except of course you, and very often that doesn't feel very good.
Last week, the Fox Chicago News Dr. Oz group came to my studio in hopes of finding that lost motivation. I wrote a column about it last week. Losing motivation is not uncommon, and getting it back is always a challenge. Just remember, realistic expectations combined with a realistic exercise and nutrition program is the best way to stay on track and realize a healthier you!
Avoid the "on" or "off" lifestyle.
I met with Anthony this week and we talked about his challenges and successes. Like anyone trying to lose weight, Anthony wants desperately for the weight to be off! However, when you're doing it right, weight does not drop off quickly and changes need to be made for the long term, not just until a wedding, reunion, etc. What I'm talking about here is getting away from the "On a diet" or "Off a diet" mentality.
In my humble opinion, (and you know I have many of them), I really believe that being "on" a diet puts the mind in a very negative place. Being "on" a diet tells the brain, "You must suffer. You can not have anything you want ever again. You need to be hungry all the time and eat food you really don't enjoy." Hmmm, I don't know about you, but with that kind of mental input, there is no way one can possibly beat the weight loss game.
The healthy living mentality is your best defense against a negative experience as you strive to change unhealthy habits to healthy ones. Try, just for this week (as I encouraged Anthony to do) to think "lifestyle" not diet. To think about the long-term and choices that seem appropriate vs. so painfully out of character for you you'll never stick with them. Acknowledge the positive changes you make, even if they're small.
Healthy living is supposed to encourage a healthier you, both mentally and physically. The best way to achieve that is to find your center and avoid the "on" or "off" when it comes to lifestyle change!
Most of my adult life I have been driven to help others develop and maintain a dedication to healthy living. Trust me, it hasn't been easy. Unfortunately, enthusiasm alone can not make others successful, people have to be ready and willing to step in to the world of change, period end of statement.
I have always been a follower of Dr. Oz and bought his original book, You on a Diet, when it came out a couple of years ago. He recently revised the book and has been touring the country to promote it. Little did I k now, the book that I so loved and encouraged others to read, would come back to me directly through Dr. Oz himself.
This past week, I had the pleasure and honor of meeting Dr. Oz as part of a Weight Loss Challenge by Fox News here in Chicago. Mark Saxenmeyer of Fox News, put the challenge together and included not only some of his colleagues to participate in the weight loss challenge, but his viewers as well. On Wednesday, I received an email from Mark, letting me know that I would be part of a select group of people that would be able to meet Dr. Oz, talk to him, ask questions and be part of his Weight Loss Challenge using his new book. What I didn't know is that I would also have the opportunity to meet and train a very special young man, Anthony Marzullo.
Anthony wrote to Mark at Fox News when he heard about Dr. Oz's Weight Loss Challenge and shared his story about his life long struggle with weight. He has done every diet, tried every weight loss program out there, yet at 31 years old, he continues to struggle with his 350 pound frame.
"I did it to myself," he said, "I put the fork in my mouth. I take total responsibility. But now it's time to take responsibility in a positive direction." So, I have the privilege of working with Anthony over the next year, using Dr. Oz's book and philosophy which is exactly like mine (thank God, or I'd never do it!). Mark will check in with us during the course of the year to see how we're doing.
I met with Anthony for the first time and reviewed where he's been, what he wants to do and what is going to be different this time. I told him, "Anthony, your entire life you have 'fought' weight, it's time to stop fighting. To be successful, you'll need to change how you think and instead of focusing on weight loss, you need to focus on health gains. Every choice you make from here on out needs to be about your health. Up until now, it's been all about the weight. So let's try to redirect your efforts. Remember, a byproduct of healthy living is a healthy weight!" Anthony agreed and we set a few goals and are ready to forge ahead.
I hope that you will join us on our journey. I'll be blogging about it and hopefully I can get Anthony to do the same! Until then, join us on Facebook and share your thoughts and encouragement for Anthony and the millions of others that strive for a shot at a healthy body every single day.


text:
These prototypes are a proof-of-concept for a potential game's User Interface. Some are redesigns of an existing game's UI, and some are for game concepts to test my skills and creativity.
For example, the GIF to the right displays an idea for a game's menu to be displayed on playing cards that would have its intractable elements on the face of each card.
UI Redesigns
In an attempt to better the design of UI in video games, I research for some truly troubling Menus and Screens and rebuild them from the ground up. I do my best to preserve the original design choices and update them into better and more usable ones.
In reading order we have:
Super Smash Bros. for WiiU - by Nintendo (2012)
Mario Party 8 - by Nintendo (2010)
#ReUI
@Nintennerd
This game was described as a retro-inspired dungeon crawler and action-adventure game. Player's would search through mysterious caverns and temples battling monsters and solving puzzles.
The player would have found several items by at this point, each of which granting new abilities that can be assigned to different buttons.
I decided to make the icons match the pixelated art of the screenshot. The layout would also appear simple as if emulating a classic game. It was meant to avoid distracting the player's eyes away from the gameplay, and a large focus was put on health and what items are equipped to the character.
Credit to the original screenshots goes to Greg Beaton.
Animal Heist
*Image by Allison Dubé
In addition to designing the game's mechanics, puzzles and levels, I also worked on what the layout of controls would be for both of our play methods. (Xbox gamepad, and Keyboard & Mouse)
© 2019-2020 Kevin Van Doormaal,
Double O Double A Media,


text:
Judge: Murder-by-arson sentence wrong
By Michael RubinkamAssociated Press
The arsonist who killed Ji Yun Lee was especially cruel and calculating, dousing her small cabin in Pennsylvania's Pocono Mountains with more than 60 gallons of gasoline and heating fuel and setting at least eight fires, ending at the front door to block any chance of escape. Then he watched calmly as the cabin turned into an inferno.
That was the prosecution's case against the victim's father, Han Tak Lee, and it persuaded a jury to convict the South Korean immigrant of first-degree murder. He's serving life without parole.
But the arson science underlying his conviction turned out to be all wrong.
This month — nearly 25 years after the blaze — a federal magistrate recommended that Lee should either be given a new trial or released from prison outright. A federal judge must approve the recommendation, and prosecutors are expected to file papers this week arguing that Lee's conviction should stand.
His case is one of dozens around the country to come under scrutiny because of entrenched but now-discredited beliefs about how arson can be detected. The Arson Research Project at the Monterey College of Law in California has highlighted at least 31 convictions based at least partly on debunked fire investigations, including that of a Texas man executed in 2004, and experts believe there are many more.
"There was just no science behind" the old assumptions about arson, said Paul Cates of The Innocence Project, a group that works to overturn wrongful convictions, primarily through the use of DNA. "A lot of this was just guesswork and voodoo."
Lee, now 79, has consistently maintained his innocence.
A clothing store owner in New York City, Lee had taken his volatile, mentally ill 20-year-old daughter to a northeastern Pennsylvania religious retreat at the suggestion of the family pastor. Early on July 29, 1989, the cabin they shared became engulfed in flames. Lee escaped, but his daughter's body was found in the ashes, curled in the fetal position.
When firefighters showed up, they found Lee sitting stoically on a bench outside the cabin. Inside the wreckage were clues that led authorities to suspect foul play.
At the time, investigators were taught that unusually hot and intense fires indicated the use of an accelerant and that arson could be confirmed by the presence of deep charring or shiny blistering of wood as well as "crazed glass," tiny fractures in windows.
Research conducted in the 1980s debunked these and other notions about arson. By 1992, the National Fire Protection Association had published new standards to guide fire investigations.
But acceptance did not come right away.
"Most arson investigators' heads exploded, and they just went nuts for the next seven or eight years trying to discredit that document," said John Lentini, one of the nation's leading experts in fire analysis and a defense consultant for Lee.
At Lee's trial, a fire marshal and other prosecution experts said the physical evidence overwhelmingly pointed to arson. In addition to the telltale burn patterns, they said, testing revealed accelerants on Lee's shirt, pants and a jug found in the cabin.
Lee's attorney didn't challenge the arson finding, arguing instead the fire was set by Lee's troubled daughter to commit suicide. The jury didn't buy it.
A quarter-century later, U.S. Magistrate Judge Martin Carlson said scientific progress had invalidated the conviction.
"Over the past two decades, there has been a revolution in fire science," he wrote June 13. "It is a revolution that has toppled old orthodoxies, and cast into doubt longstanding assumptions regarding fire scene analysis."
Monroe County prosecutors concede the point but say other evidence points to Lee's guilt, including the accelerants found on his clothing.
But that evidence, too, has been "substantially undermined" by new testing, Carlson said. And Lee's tranquil demeanor at the fire scene — a powerful bit of circumstantial evidence that helped sway the jury — might have stemmed from a cultural taboo against showing emotion in public, the magistrate said, not from a lack of feeling.
He said justice dictates throwing out Lee's conviction and sentence.
"Sometimes, with the benefit of insight gained over time, we learn that what was once regarded as truth is myth, and what was once accepted as science is superstition," Carlson wrote. "So it is with this case."
Kyung Sohn of New York City, who heads the National Committee to Free Han Tak Lee, said he got a letter from his old friend Thursday, nearly a week after Carlson's decision. Sohn said Lee, who has been in poor health, hopes to soon reclaim his reputation as well as his freedom.
"He mentioned that 'God is with me, that I am innocent,' " Sohn said. "In my thinking, 99.99 percent he will be free. We pray every day."