The employer and employee must have a clear understanding (preferably in writing) that the employee will receive a fixed salary (apart from overtime premiums) that remains the same in both long and short workweeks.
The employer must pay a fixed amount per week for whatever hours the employee is called up on to work (i.e. no docking the salary for short weeks).
The amount must provide at least the legally-required minimum wage for each hour worked, including those weeks where the employee works the greatest number of hours.
The employer must pay overtime premiums at one-half the employee's regular rate of pay for hours worked over 40 in a workweek (based on the regular rate for that week, which will vary).
There are, with anything else, pros and cons to this method. Obviously, employers may be paying employees more in those weeks where they work fewer hours. Employers also must continuously and carefully monitor the employee's hours to ensure that they are not receiving less than minimum wage in long weeks. Even more, the method may not comport with state or local minimum wage and overtime laws, so employers must carefully review all applicable rules before implementing the FWW. Nonetheless, some employers prefer the consistency of fixed salaries in terms of more predictable wages and the potential for savings. Additional forms of compensation, such as bonuses or commissions, can further complicate the method, so employers should also seek legal input before adopting the FWW. See you next time for Letter G!


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Leary and Ramesh team up to take on state
By Alec Hamilton on Oct 23, 2018 at 1:27 p.m.
Leary and Ramesh pose together as young girls at a tennis tournament while playing for Fred Well Tennis and Education Center. They were friends and teammates long before playing together for the Irish. Photo courtesy of Sally Leary1 / 3
Sophomore Sonya Ramesh goes to the state tournament for the first time and should take over the No. 1 singles spot from Leary next season. Alec Hamilton / RiverTown Multimedia2 / 3
Senior Livia Leary will end her prep tennis career on a high note by playing at the state tournament Thursday. Alec Hamilton / RiverTown Multimedia3 / 3
Senior Livia Leary and sophomore Sonya Ramesh are state-bound, but their road to the state tournament was anything but easy. The two top singles players for the Rosemount Irish, Leary and Ramesh knew they would be playing doubles with each other in the postseason since the start of the year. What they did not know is that they would have to play six matches in two days, including the second day that saw them play until almost 11 p.m. But they overcame adversity and took second in the Section 3AA doubles tournament to qualify for the state tournament at the University of Minnesota Baseline Tennis Center on Thursday, Oct. 25.
Leary and Ramesh cruised through their first three matches on Oct. 12 at the Lifetime Fitness Center in Lakeville. They beat a team from Humboldt 6-0, 6-0, followed that up with a 6-0, 6-3 win over the pairing from Cretin-Derham Hall and then finished the day by beating the team from St. Paul Central 6-1, 6-1. Three days later, the team faced some stiff resistance as they attempted to seal their state birth. They went three sets against a tough team from Visitation, coming from behind to win 4-6, 7-6, 6-2. In the section championship match, Leary and Ramesh fell to the team from Henry Sibley 6-3, 6-2. However, from there they were forced to play one more match to preserve their second-place finish—a True Second match against the team from Eagan who won their third-place match and had not lost to the Rosemount or Henry Sibley teams—which Leary and Ramesh took 6-4, 7-6 and finished just shy of 11 p.m.
Leary said that reaching state was an unlikely goal for her senior year.
"My No. 1 goal was to play the No. 1 spot (singles) for sure, and another goal that I honestly never thought would happen is making it to state, which both ended up taking place this year and I couldn't be more proud," she said.
Making it to state was also a goal for Ramesh, who described what it felt like to win that True Second match and know she and Leary were headed to state.
"We were ecstatic it was over and we had pulled out a win!" she said. "It was an emotional time for me and Liv. We had advanced to the finals before that so when we won the semi-final match we were so happy and jumping for joy."
Friends and teammates
While Leary and Ramesh are new to playing doubles with each other (outside of one tournament when they were younger, which Leary said was "very unsuccessful"), they have been friends and teammates for years, long before playing for the Irish. The two first met as young girls, and actually played each other in Leary's first ever tennis match, before they both went on to play for the Fred Wells Tennis program at the Fred Wells Tennis and Education Center.
Leary and Ramesh were exposed to tennis at a young age. Leary's mom (Sally) played in high school and college and introduced the sport to her two daughters, Anna and Livia. Leary also played basketball and for a long time said that basketball was her favorite sport.
"But once I got older I found more of an interest in tennis and actually quit basketball so that I could focus on tennis," Leary said. "The thing that I enjoy the most about tennis is the competition and focus. Being a tennis player is very challenging mentally, especially for singles players, the match is all on you, you have to pump yourself up and keep your confidence going and it can be very hard to stay positive."
Ramesh also started at a young age and like Leary played numerous sports until her love for tennis grew.
"I enjoy how fun the sport is and how you can build so many connections with other people from it," she said.
They both also had the opportunity to play with their sisters during their Rosemount tennis careers—Anna Leary and Tanya Ramesh, who graduated last spring—which they said was a huge part of their love for the sport.
"Tennis has always been the thing that bonded me and Anna," Leary said. "Playing tournaments, and dealing with the stress of it was a way that we always seemed to connect. Having my sister on the tennis team with me was the greatest thing, she was always my biggest supporter and for me, she was my absolute inspiration."
"Playing with my sister was so much fun, we would always support each other and cheer each other on," Ramesh said. "Not having her on the team (this season) felt different, but I also had my other teammates supporting me."
With Anna and Tanya graduating last year, Livia and Sonya found themselves as the oldest/most experienced players on the team and in leadership positions. Head coach Matt Knutson praised both for the roles they played this season, especially Livia who he said has not only improved greatly on the court but has also been such a role model to her teammates off the court and in practices.
"I'd say we (Leary and Ramesh) have gained a lot of respect from the other players and our coach and that's a really awesome feeling," Leary said. "When we were the rookies on the team it took awhile for the older players to gain respect for us and we definitely had to work for it, but now playing the role as the upperclassmen it's a lot of fun and I love setting an example for the newer girls."
Preparing for state
The two knew that they would be playing together this whole season, so they had time to practice together throughout the fall before sections hit. However, both said as singles players it is a major adjustment to switch to playing doubles, though their games have melded well."
"The biggest adjustment is remembering where to stand when up at the net and honestly just playing at the net in general," Leary said. "Me and Sonya both aren't big with volleys so we had to practice a lot on them."
"We are very similar players in terms of how we play so I think we mesh really well together," Ramesh said. "We each have our own strong shots that correspond with one another as well."
Leary and Ramesh will play at 10 a.m. at the Baseline Tennis Center on the University of Minnesota's campus on Thursday, Oct. 25. They take on the No. 4 seeded team from Rochester Mayo, seniors Emily Norman and Amalin Sorajja. Should they win, they would go on to play the winner of teams from Mounds View and St. Michael-Albertville that same day at 2 p.m.
"I'm going to take it with as little stress and worry as possible," Leary said about playing at state. "Just play my best tennis and really live up and soak in the experience."
"My mindset is just to play my best and work really hard in each and every match," Ramesh said.
Race your car on a track at DCTC
Explore related topics:sportstennisRosemountMinnesotarosemount irishgirls' tennisLivia LearySonya Rameshsouth suburban conferenceMSHSL girls tennis state tournament
Alec Hamilton
Alec Hamilton is a RiverTown Multimedia sports reporter covering Hastings, Farmington and Rosemount athletics. He graduated from Drake University with a journalism degree in 2014.
ahamilton@rivertowns.net

redpajama_set_name: RedPajamaCommonCrawl


Somehow I ended up living in a tiny village in Galicia, North-West Spain.
Hi Bridget! Thanks for for following! I have been thinking for a long time to write about my village experiences and the stories I've found here, hence I decided to join the A-Z challenge…I'm following your blog too now!
Have really enjoyed reading your posts for the A-Z challenge and look forward to many more village stories. Would you like to do a guest post over on my blog – perhaps on your experience of blogging and what it has meant to you/ where it has lead you….
My name is Nikolai, i am helping out marketing agency to find bloggers suitable for their spanish campaigns, and your blog (chicaderock.wordpress.com) looks like a perfect fit for what they're looking for.
We quite enjoy your blog about living in Galicia, it is very interesting and informative. Would it be possible to interview you to further share some of your tips and feature some of your first hand experience as an Expat and your interview will be published on our Expat Interview section as a guide for our expat readers. The questions are mainly about the day to day lifestyle of an expat. If it would be possible, could you also send some photographs that we can use?
I'm a writer living in Barcelona and I've just found your blog while about to sign up for the A-Z challenge (hesitating – ot sure I can cope!) Excited to find someone blogging in Galicia so wanted to say hello.


Posted on July 7, 2014 by kcg1974 •	This entry was posted in Awards and tagged Appreciation, Awards, Blogging, Gratefulness, Gratitude, Support. Bookmark the permalink.
Congratulations on the award and thank your for listing my blog.
Thank you so much for nominating me for the award. I am most flattered and touched. Thanks!
I am thrilled to to do so. Congratulations!
Thanks for nominating me for the Versatile Blogger Award, Kim!!
You definitely deserve this one!!
You are most welcome my friend (smile) Blessings!!
With sincere thanks to you, Greg.
Somehow I missed this comment until late last evening, Bobby. I accept with humble pride your nomination which stirs an abundance of emotion within me. Already an "award" in itself to think my words are inspirational. I thank you deeply.
Another silverware (but that seems to be gold!) in the showcase! Congratulations, Kim!
Sarvit, so nice to see you. Thank you very much. I've lost touch with you. Will have to 'find' you once again. Thanks so much!
Oh, you are so welcome, Sue. No rush, as I am the very worst at getting my acceptance post's out. So guilty I always feel! Thank you very much, Sue.
So pleased, Evelyn. 🙂 Congratulations.
Congratulations Kim, a well deserved award for an informative and enjoyable site.
Always a pleasure to read your blogs and visit your world.
Thank you very much for considering my site worthy of recommendation for the award.


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Volume 4 Issue 10 - May 15, 2006
Decision to eliminate key personnel in area of Disability; D.P.I. writes to World Bank President
D.N.I.S. News Network � In its letter to the World Bank President Paul Wolfowitz, Disabled People�s International (D.P.I.) has expressed dismay at changes the Bank will be making with regard to inclusion of disability in its development agenda. Whilst retaining the position of �Disability Adviser�, the Bank has decided not to renew the contract of the current Disability and Development Adviser, Judith Heumann, and other key staff positions within the wider Disability and Development Team.
D.P.I. has also mentioned that it has further understood that one of the regional disability focal point positions will be eliminated and that D.P.I. would query the criteria used in making this assessment, and also whether any stakeholders in the region, especially representative organisations of persons with disabilities, were consulted in reaching this determination.
Appreciating that specific personnel decisions are within the Bank's prerogative, D.P.I. has expressed concern that in addition to losing the excellent leadership of Heumann, it appeared that the proposed changes would substantially deprive the Bank of the institutional knowledge of disability and development built-up over the last four years - a knowledge base that would seem to be an essential component of any plan to further operationalise disability within the Bank�s activities.
D.P.I. also added that these developments signal a significant change in direction and commitment by the Bank, and a retreat from the leadership role that the Bank has played in the field of disability and development over the last several years. It strongly urged the World Bank President to reconsider the proposed changes that would substantially weaken the Bank's capacity to meet its commitment to the 400 million plus people with disabilities living in the developing world.
Social Security and Disabled People in India
"There should be minimum 5% budget allocation in all sectors for inclusion of disability":G.Syamala
Delhi High Court gives deadline to various airlines to file affidavits
Ten years after Disability Act less than 0.9 per cent disabled employees in private sector
I.A.S. results declared; Twelve disabled candidates qualify
C.I.I. reiterates its commitment to mainstream disability
Lease of houses without proper access can be cancelled: Allahabad High Court
H.C. orders I.D.B.I. bank to allow visually impaired person to open account
Special Olympics Bharat accorded priority status
The capital all set to get its first fully-accessible bus route
Students of world's only university for the deaf protest appointment of new President


You could say that Dean and Len grew up in the food business. Their father and grandfather had a retail food market at 9th and Susquahana since 1947. This is where the boys got their work ethic; work hard and treat people the right way. And always give the people what they want.
In 1973 they started Wilson Beef Company, a wholesale meat supply company that served the finest restaurants and institutions in the Philadelphia area. The business grew rapidly, and soon the boys had to move to a bigger facility.
In 1981, they opened their doors to the public on weekends. Through word of mouth, our business boomed. People were calling our warehouse placing orders and picking them up on the weekends. There was a big demand to be open more hours, but it was impossible to run the retail and the wholesale business at the same location.
The boys picked a location on Aramingo Avenue and in 1983 opened their first retail store, The New Wilson Meats. Their concept was a winner; sell restaurant quality meats and poultry to the public at the same reduced prices that the restaurants paid.
Business continued to grow and in 1994, the brothers decided to start a new company. They opened Wilson's Outlet II at Adams and Rising Sun Avenues. It wasn't quite the same concept; but it was the same great quality and low pricing.
Today, in an era of big box stores, Wilson's still gives the same personal service as they did over 27 years ago. We always have plenty of butchers on hand to prepare whatever you need, the way YOU want it. Our discount deli area, features only the finest products at unbeatable prices. The poultry we sell is only all natural and as always our meats are still restaurant quality.
We accept all major methods of payment including food stamps.


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Q: DELETE default ubuntu user I am new to this. Should I delete user ubuntu and Create new one. As everyone knows the username and they can try to login. What's say?
This a ec2 of AWS

A: As this is an EC2 question, the short answer is "No, you do not need to delete the default user".
There are two primary reasons for this:

*

*Port 22 should be limited via the security policy that the EC2 instance(s) are using. If SSH is open to 0.0.0.0 then something needs to be changed. Immediately.

*Even if people know the account name, they will need the .pem private key file that you have associated with the instance(s).

You certainly can change or remove accounts from EC2 instances, but you shouldn't need to.

redpajama_set_name: RedPajamaStackExchange


How Do You Solve A Problem Like Junior?
Oh, these kids! The mischief they get into! The grief they bring down on their parents!
That Donald Junior, what a little scoundrel!
Oh, whoops! That's Draco Malfoy.
Here's the one I meant. (I always get those two mixed up).
Anyway, that boy does not does not know how to behave himself, and he's so mean.
...other than to get his picture taken next to their bodies.
In fact, both the Trump boys like to kill animals.
...this is their heart's desire.
...and meet with powerful Russian government lawyer Natalia Veselnitskaya, who's like, "I didn't do anything!"
Except that the grown-ups found out, and now young Junior is in big trouble!
Of course, Daddy Trump will probably get Junior off the hook. Just yesterday Donald Sr. said that his son was "a high quality person." That's apparently Rich-Speak for "He's a good kid."
(Sigh). Still, if that was my kid I'd be praying, too.
...and making sure the picture got posted all over the internet.
The hunting pictures are disgusting testosterone displays. But the prayer picture with the hands of aging sycophants eagerly touching Trump is beyond appalling.
I know, right? And what happened to "But when you pray, go into your room and shut the door and pray to your Father who is in secret. And your Father who sees in secret will reward you"?


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Got $25,000 saved? That might be enough for a Kiwibuild home
Just how affordable is a KiwiBuild house? Credits: Newshub.
ASB has thrown a lifeline to all the would-be first-home buyers out there with $25,000 handy.
They're going to lend up to 95 percent of the purchase price for KiwiBuild homes.
"Our history shows we're committed to supporting Kiwis into their first home by empowering them to confidently take steps towards realising their dream," said acting executive general manager retail banking Claire McKinnon.
On top of that, the bank is offering $2500 cold, hard cash once their loan is approved.
"For example, it can be used towards the costs of purchasing their new KiwiBuild home, such as a property valuation or legal fees."
Can you afford a KiwiBuild home?
First KiwiBuild homes completed
The cheapest one-bedroom KiwiBuild homes in Auckland will be $500,000, requiring at least a $25,000 deposit. Not even the Government's own bank - Kiwibank - is matching that. Earlier this month Kiwibank made headlines when it said it would lend 90 percent of the purchase price, and give buyers $2000 cash.
"The Government's efforts to restore the Kiwi dream of home ownership is an aspiration Kiwibank can support," said Kiwibank group marketing manager Mark Wilkshire.
One of the first Kiwibuild homes. Photo credit: Newshub.
Around 40,000 people have applied for a KiwiBuild home, and with only a few hundred expected in the first year it could be a long time before buyers get a chance to take advantage of the offers, if they're still available.
KiwiBuild income caps announced
First-home buyers shouldn't rely on KiwiBuild - expert
Presently banks can't have more than 15 percent of their residential mortgage lending go to owner-occupiers with less than a 20 percent deposit.
But lending to KiwiBuild buyers is exempt, as the Reserve Bank's restrictions don't apply to new builds.
ASB's deposit minimums
$500,000 home - $25,000
$600,000 two-bedroom home - $30,000
$650,000 three-bedroom home - $32,500


Which of the following statements best describes the relative bond dissociation energies of the sigma and pi bonds present in the carbon-carbon double bond of an alkene?
Carbon-carbon single bonds tend to be ________ and ________ than carbon-carbon double bonds.
Which of the following best approximates the CCC bond angle of propene?
What two atomic orbitals or hybrid atomic orbitals overlap to form the CH bond in ethylene?
What two atomic orbitals or hybrid atomic orbitals overlap to form the CC σ bond in ethylene?
What two atomic orbitals or hybrid atomic orbitals overlap to form the CC π bond in ethylene?
The compound produced when 3-methylpent-2-ene undergoes hydrogenation in the presence of a platinum catalyst is ________.
Using Zaitsev's rule, choose the most stable alkene among the following.
D) They are all of equal stability according to Zaitsev's rule.
Consider the constitutional isomers 2-methylbut-1-ene, 2-methylbut-2-ene, and 3-methylbut-1-ene. When each of these alkenes is subjected to catalytic hydrogenation (H2, Pt), a single product results. Which of the following best describes the structural relationship among these products?
A) The products are cis-trans isomers.
B) The products are identical.
C) The products are constitutional isomers.
D) The products are enantiomers.
E) The products are diastereomers.
Which of the following alkenes has the largest molar heat of hydrogenation (ie, releases the most heat upon hydrogenation)?
Which of the following alkenes has the smallest molar heat of hydrogenation (ie, releases the least heat upon hydrogenation)?
The trans isomers of cycloalkenes with rings containing fewer than ________ atoms are unstable at room temperature.
Which of the alkyl chlorides listed below undergoes dehydrohalogenation in the presence of a strong base to give pent-2-ene as the only alkene product?
Dehydrohalogenation of 2-bromobutane in the presence of a strong base proceeds via which of the following mechanistic pathways?
Dehydrohalogenation of an alkyl halide by treating it with a strong base to yield an alkene product typically occurs by what reaction mechanism?
88) Which of the following alkenes is the major product when 2-bromo-2-methylpentane is treated with sodium ethoxide in ethanol?
89) Which of the following alkenes is the major product when 2-bromo-2-methylpentane is treated with potassium tert-butoxide in tert-butanol?
Which of the following statements applies to the E2 mechanism?
A) It occurs with inversion of stereochemistry.
B) It occurs with racemization of stereochemistry.
C) It proceeds through the more stable carbocation intermediate.
D) The C-H and C-X bonds that break must be anti.
E) Use of a bulky base gives the more highly substituted alkene product.
Provide the name of the major alkene product that results when (2S,3S)-2-bromo-3-methylpentane is treated with sodium methoxide in methanol.
Provide the name of the major alkene product that results when (2S,3R)-2-bromo-3-methylpentane is treated with sodium methoxide in methanol.
Provide the name of the major alkene product that results when (2R,3R)-2,3-dibromopentane is treated with zinc in acetic acid.
Provide the name of the major alkene product that results when (2S,3R)-2,3-dibromopentane is treated with zinc in acetic acid.
By what mechanism does cyclohexanol react when treated in sulfuric acid and what compound results?
In the group shown below, which of the following alcohols is (are) likely to yield a product where skeletal rearrangement has occurred when treated with sulfuric acid?
E) None of these three alcohols is prone to rearrangement.
Which of the following alkenes can show geometric isomerism: 2,3-dichloro-2-pentene, 4-chloro-3-ethyl-3-hexene, 3-chloro-2-methyl-2-butene, and 3-chloro-2-methyl-1-butene?
Which has the smaller heat of hydrogenation, (E)-2-pentene or (Z)-2-pentene? What is the structural origin of this difference?
E)-2-pentene has the smaller heat of hydrogenation. There is less steric strain between the alkyl substituents in the (E)-isomer than in the (Z)-isomer.
Which has the higher boiling point, cis- or trans-hex-3-ene? Briefly explain your choice.
Cis-Hex-3-ene has the higher boiling point since it has a net molecular dipole moment while the trans-isomer does not. The presence of a permanent molecular dipole allows for stronger intermolecular attractions.


text:
Dow Jones recorded its strongest quarterly growth since 2013
by jmcgovern | Mar 30, 2019 | Finance News, News
Share This On Social
The main Wall Street indexes ended the last session of the first quarter with growth amid the returning optimism for progress in Sino-American trade talks.
The blue-chip index Dow Jones Industrial Average finished the trading session with an increase by 211.22 points and reached a level of 25,928.68 points, with Boeing, UnitedHealth and Caterpillar performed best. The broader S&P 500 advanced by 0.67% to 2,834.40 points, supported by the industry and the health sector. The technology index Nasdaq Composite rose by 0.78% to 7,729.32 points.
The US officials said that China has made proposals on a number of issues, including the forced technology transfer. The Finance Minister Steven Mnuchin said on Friday that he had a "productive working dinner" with Chinese official trade representatives. The two countries are restarting their talks in the hope of ending their trade dispute.
The growth on Friday extends the strong performance of the Wall Street indexes in the first quarter. The S&P 500 rose by 13.1%, which is the highest quarterly increase since the third quarter of 2009. The benchmark also marked its best first quarter of 1998.
Dow Jones achieved a growth of 11.2% in Q1 2019, which is its best start of the year since 2013. Meanwhile, Nasdaq Composite marks its biggest quarterly increase in the first quarter since 2012, reporting growth of 61.5%.
The rally in the first three months of the year is due to a strong leap against the bottom of Christmas Eve last year, rising optimism in US-China trade talks and a sharp change in the Federal Reserve's monetary policy.
The concerns about a possible slowdown in the economy, however, hit the rally amid the worsening economic data. The growth in corporate earnings in the first quarter is also expected to be weak. FactSet data show a drop in profits of S&P 500 companies by 3.7% on an annual basis. However, data from The Earnings Scout show that analysts expect earnings growth to recover in the coming quarters of the year.
In the bond markets, yields on 10-year and 30-year US government bonds rose to 2.414% and 2.82%, respectively.
Corporate stocks performance
CarMax was the best-performing company in the S&P 500, with its shares rising by 9.5% thanks to its strong reporting. Moods were also raised by the shared travel company Lyft, whose stocks added more than 8% of their value on their first trading day.
The shares of technology giants FAANS performed strongly during the trading session. The stocks of Facebook rose by 0.7%, while Amazon and Apple appreciated by 0.4% and 0.6%, respectively. Meanwhile, Netflix and Google stocks appreciated by 0.4%, both.
Boeing rose by 1.9% boosting the industrials sector. Micron Technology rose 4.6% as makers of semiconductors saw solid gains.
Caterpillar and Intel ran hottest among Dow Jones stocks. DowDupont dropped 1.7% after lowering its first-quarter guidance late Thursday.
Shares of the industrial giant General Electric have gained 32% so far in 2019, making it one of the best-performing stocks in the S&P 500. The stocks of the company appreciated by 1% on Friday, being among the best performers in S&P 500. Other top performers in Q1 2019 include Chipotle, up 63 percent; Xilinx, up 50 percent; Ulta Beauty, up 42 percent; and Best Buy, up 34 percent.
The materials provided on this Website are for informational and educational purposes only and are not intended to provide tax, legal, or investment advice.
Nothing contained on the Website shall be considered a recommendation, solicitation, or offer to buy or sell a security to any person in any jurisdiction.
© FinanceandMarkets.com


text:
PET/CT with Ga-68 tracer better for cancer patients
November 2, 2020 -- By attaching gallium-68 (Ga-68) to a fibroblast activation protein molecule inhibitor, German researchers have achieved greater accuracy with PET/CT in staging patients with pancreatic ductal adenocarcinoma, according to a study published online October 23 in the Journal of Nuclear Medicine. Discuss
FES-PET could predict endometrial cancer outcomes
October 7, 2020 -- PET imaging with the radiotracer F-18 fluoroestradiol (FES) can help determine how well patients will fare after a diagnosis of endometrial cancer, according to a study published October 2 in the Journal of Nuclear Medicine. Discuss
Researchers offer a guide to optimum PET lung imaging
September 25, 2020 -- Can you name the best strategies to optimize FDG-PET/CT to assess lung disease? If not, researchers from imaging centers in the U.S. and the U.K. have provided their consensus recommendations on how best to proceed in a study published online September 18 in the Journal of Nuclear Medicine. Discuss
FET-PET offers more info on treatment of brain metastases
September 14, 2020 -- Using PET with the radiolabeled amino acid F-18 fluoroethyl tyrosine (F-18 FET) can significantly help determine the efficacy of radiotherapy in certain cancer patients who develop brain metastases, according to a study published online September 4 in the Journal of Nuclear Medicine. Discuss
How running an MRI service is like captaining a ship
August 14, 2020 -- Running an MRI service is like being the captain of a ship on the ocean, according to a Friday presentation at the International Society for Magnetic Resonance in Medicine virtual conference. Smooth navigation depends on effective collaboration with physicists, technologists, and other staff. Discuss
Thermal burns top FDA list of MRI adverse event reports
August 13, 2020 -- Ten years of MRI adverse event reports show burns from coils and electrocardiography leads during scans are among the most common -- and preventable -- causes for patient injuries, according to a study presented Thursday at the International Society for Magnetic Resonance in Medicine meeting. Discuss
FDG levels show link between Alzheimer's, Down syndrome
July 29, 2020 -- PET imaging has revealed reduced FDG uptake in key brain regions associated with early- and late-stage Alzheimer's disease among people with Down syndrome. The findings could indicate the degree of brain atrophy in individuals, as well as one reason why this patient population is susceptible to dementia. Discuss
SNMMI 2020: Image of the Year scans reveal 'super-agers'
July 14, 2020 -- A group of German researchers has received the Image of the Year award from the Society of Nuclear Medicine and Molecular Imaging (SNMMI) for a series of PET images that show that older adults who maintained peak cognitive skills exhibited greater resistance to buildup of tau and amyloid proteins. Discuss
SNMMI 2020: How nuclear medicine handled COVID-19
July 14, 2020 -- Implementing a proactive, multipronged strategy to combat the COVID-19 pandemic helped Mount Sinai Hospital safeguard its patients and staff as much as humanly possible, according to a Monday presentation at the Society of Nuclear Medicine and Molecular Imaging (SNMMI) 2020 virtual annual meeting. Discuss
SNMMI 2020: Medical students key to the specialty's future
July 13, 2020 -- The early recruitment of medical students into nuclear medicine and molecular imaging is the key to the specialty's future, said Dr. Vasken Dilsizian during his president's address on Sunday at the Society of Nuclear Medicine and Molecular Imaging (SNMMI) virtual annual meeting. Discuss


Becky McFadden, moved from Odessa, TX to Kerrville in 1986 with her husband Bruce (Sul Ross State University) and their two sons who were ages four and two at the time. Kerrville has been a wonderful place to raise a family, make friends, work, and enjoy life in the beautiful Texas Hill Country.
Becky grew up in Pleasanton, Texas and after 8th grade moved to Portland, Texas where she graduated from Gregory-Portland High School. She attended Southwest Texas State University in San Marcos, worked for the University of Texas Medical and Dental Application Center in Austin, married in 1974 and has lived in Alpine, Beaumont, Waco and Odessa.
Two years after moving to Kerrville, Becky started working in real estate for Schreiner Bancshares Main Properties. Her sons both graduated from Tivy High School and from Texas Tech University. Becky cherishes her wonderful Father who worked for Exxon for 40 years. He lives in Kerrville. Becky's parents were married 71 years before her Mother passed away. Her siblings include an older brother (Texas A&M) & older sister (Tarleton), as well as one younger brother (Texas A&M) who all live in Texas. Becky feels blessed to have a wonderful family whom she loves dearly.
Becky equally feels blessed to have an expansive family of friends whom she's met and cultivated rewarding relationships with over her lifetime. Selling real estate has been the perfect avenue to meet and help so many terrific people.


I'm very disappointed with Rooms To Go lack of customer service.
(three weeks later) they delivered the furniture but they delivered the wrong poles (two right poles for the foot board instead of a right and a left), we lost 4 hrs of work because they didn't want to call us an hour ahead to let us know the delivery time.
The delivery person told us that Rooms to go will call us to reschedule the delivery of the poles. On 16 April I received a call at work (No one call me to set up a delivery date or time) and told me that the delivery crew was at my house. I asked If they could wait 30 min so I could get at home from work and I was told that they couldn't do it.
Made up the story. I will never buy anything fro rooms to go and I will make sure everyone knows about my nightmare.


The item Doctor Maisy, Lucy Cousins represents a specific, individual, material embodiment of a distinct intellectual or artistic creation found in Dryden Township Library.
"It's Doctor Maisy and Nurse Tallulah, and they're playing hospital. Poor Panda is sick, but Maisy takes good care of him. Then it's Maisy's turn to need the help--and Nurse Tallulah comes to the rescue!"
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EQIP is a voluntary conservation program which addresses soil, water, and related natural resources through 2 -10 year contracts. You can receive technical, financial, and educational assistance to apply conservation solutions to your natural resource concerns. You must be engaged in livestock or agricultural production and meet conservation compliance conditions outlined by the 2008 Farm Bill. Each area has different ranking criteria, application periods, eligible practices, and cost share rates. You may apply at your local USDA Service Centers, NRCS, or Conservation District Offices.


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Finance Committee Approves $12.3 Million Settlement With Police Torture Victims Ronald Kitchen and Marvin Reeves
Posted on September 6, 2013 by Kris Clutter
CHICAGO – The Finance Committee of the City of Chicago on Friday approved payment of a $12.3 million settlement to be divided between Ronald Kitchen, a former Death Row prisoner who spent 21 years behind bars, and his co-defendant Marvin Reeves. The criminal convictions of Kitchen and Reeves were overturned in 2009 with the agreement of Illinois Attorney General Lisa Madigan, and the men later received certificates of innocence from the Cook County Courts. Kitchen was convicted based on his false confession extracted through torture by former Chicago Police Commander Jon Burge, detective Michael Kill and two other detectives, and the false testimony of a jailhouse snitch.
Kitchen's lawsuit, filed in 2010, alleged that he was arrested on a false tip, deprived of food and sleep and repeatedly tortured by Burge and Kill and their associates who beat him with their fists, a nightstick and a telephone, inflicting serious injury to his genitals.
During discovery in the lawsuit, Kitchen subpoenaed former Mayor Richard M. Daley, who, while State's Attorney, had approved the seeking of the death penalty in Kitchen's case, to testify about his knowledge of the torture scandal. Daley refused to appear, and a motion to compel his testimony was pending before the court at the time the settlement was reached this summer.
People's Law Office lawyers G. Flint Taylor, Joey Mogul and Ben Elson and MacArthur Justice Center lawyers Locke Bowman and Alexa Van Brunt represented Kitchen in the lawsuit and negotiated the settlement with the City of Chicago. The portion of the lawsuit naming the Cook County State's Attorney's Office and Assistant State's Attorney Mark Lukanich as defendants has not been settled and is still pending.
"We regret that former Mayor Richard M. Daley may once again escape testifying about his involvement in the Burge torture scandal," said G. Flint Taylor of the People's Law Office. "We hope that he will be compelled to do so in Mr. Kitchen's continuing case against Cook County and in several other cases that are pending in the state and federal courts. We also call on Mayor Emanuel to apologize to the survivors of police torture and to the African American community for the damage done by the Burge torture scandal and to create a fund, in an amount equal to the $20,000,000 paid in 'pinstripe patronage' to the lawyers who have defended Burge, Daley, and their confederates, to compensate those torture survivors who have been prevented from bringing lawsuits by the City's decades long cover-up."
"More than 100 African American men were victims of torture, wrongful prosecution and false imprisonment in Cook County between 1973 and 1991," said Locke Bowman, Director of the MacArthur Justice Center. "The abusive police actions spanning more than two decades were made possible by other authority figures, who helped keep the torture secret, thwarted attempts to discipline police officers, and refused to act on calls for investigations and prosecutions of Burge and others responsible for the systemic pattern of torture. Ronald Kitchen was tortured and spent much of his life in prison because so many of the people we entrusted to uphold justice opted instead to destroy it."


Q. 11. Br - ions form close packed structure. If the radius of Br - ion is 195 pm, calculate the radius of the cation that just fits in the tetrahedral hole.
Q. 19. What is adsorption? How does adsorption of a gas on a solid surface vary with (a) temperature and (b) pressure?
Q. 28. An aqueous solution containing 1.248 g of barium chloride (molar mass = 208.34 mol -1 ) in 100 g of water boils at 100.08320;C. Calculate the degree of dissociation of barium chloride.
Write the reaction and the conditions in each case.
(a) Tendency to show -2 oxidation state diminishes from sulphur to polonium in Group 16.
(b) Boron forms electron deficient compounds.
(c) PbCI4 is less stable than SnCl 4 .
Q. 33. (a) Describe the preparation of acetic acid from acetlyene.
(c) In what way can acetic acid be distinguished from acetone?
Q. 34. (a) What is the basic difference between the electronic configurations of the transition and inner transition elements?


Socially minded theEword and Juice Digital teamed up this week for 'Social Media. Sorted'; an event which saw several of the region's leading social media thinkers present a series of talks to the Twitterati of Manchester.
The evening, which was held at a sell-out Hive, featured talks from theEword's director Al Mackin, Steve Downes, the managing director of Juice Digital, Mel McGuirk from Pannone and Nick Massey, chief executive of Forever Manchester.
"Social Media. Sorted was a unique opportunity to discover how social media marketing can deliver incredible results. We were thrilled that Manchester's leading social media experts agreed to share their knowledge about this exciting medium."
For a full report on the Manchester social media event, you can read Richard Frost's blog on the evening at theEword blog. Alternatively, you can find out what others had to say by searching for the #socialsorted hashtag on Twitter.
"Yahoo! Japan announced a deal to switch to Google's search engine technology from its own technology. As per the deal, Yahoo! Japan will use Google's online advertising and distribution system, in return Google will be provided access to data on online shopping trends."
"Yahoo Japan made this decision as an independent and separate publicly traded company, in which Yahoo! holds a 35% equity interest. We amended our agreement with Yahoo! Japan as a result of this decision, and we do not anticipate that this amendment will have a material financial impact on our revenues."
Mobile monsters Google's dominance of the mobile search market was revealed this week after research suggested that the company had obtained over 98 per cent of the global mobile search audience.
The statistics from the data hounds at ComScore discovered the search engine giant had pocketed 98.29 per cent of mobile searchers. In contrast, Yahoo and Bing both scored 0.81 and 0.46 per cent of the market.
"We operate with the assumption people will carry with them a mobile device at all times, and that there are applications we can build/people will build on our platform that will allow people to be more productive, more fun."


"The service was efficient, flexible and gave me confidence."
Thomas is a Partner and Head of the Employment and Immigration department at Eric Robinson Solicitors. He qualified in 2009 and joined the firm in 2013.
Thomas acts for employers and employees on matters arising from the workplace. This covers all types of discrimination issues, TUPE regulations, termination issues and Settlement Agreements, bringing/defending Employment Tribunal claims and contractual disputes.
He also advises on non-contentious matters including disciplinary and grievance procedures, drafting agreements and policies, advising on changes to terms and conditions and redundancies.
From an employer perspective, Thomas is experienced in advising and assisting a wide range of commercial clients from FT100 companies to small businesses. He provides pragmatic advice suited to the particular situation and seeks to understand quickly the requirements and needs of his clients to achieve the best possible outcome.
Thomas undertakes seminars on a variety of employment topics and now also incorporates Immigration Law into his practice.
Terms and Conditions - How important are they?


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Council pressed on the future of Victoria Bridge
Bath and North East Somerset Council is to be pressed on what action it is taking to restore Bath's historic Victoria Bridge and bring it back into use for pedestrians and cyclists.
The bridge was closed last year after engineers found defects in the structure. It is one of the earliest examples of a wrought iron suspension bridge in Britain and provides an important link for pedestrians and cyclist across the River Avon, including to the Western Riverside development.
Conservative Councillors, who before the local elections pledged to fully restore the bridge to its former glory, are pressing the Liberal Democrat Cabinet on when the Council will start work on reopening the bridge. In a question tabled for next week's public Cabinet meeting, Conservatives have also asked if plans for a temporary 'Bailey Bridge' alongside Victoria Bridge whilst the bridge is closed may still go ahead.
Councillor Patrick Anketell-Jones (Lansdown), the Conservative Shadow Cabinet Member for Sustainable Development, said:
"Bath's historic Victoria Bridge has now been closed for almost a year, causing continued disruption to walkers and cyclists who have previously relied upon the bridge on a daily basis. With construction underway on the first phase of new homes at Western Riverside, it is more important than ever for the Council to get to work on bringing this bridge back into use.
"Fully restoring the bridge may take some time, but it's increasingly worrying that the Lib Dem Cabinet has still not published any plans or timescale for bringing the bridge back into use, or progress the possibility of erecting a temporary bridge whilst repair work takes place. This is something the Council should look into urgently, and I hope we will be reassured that action will be taken soon."


Every inch of this home is cleaned, painted or updated, ready to move in! 3 bedrooms, 3 full baths, upstairs loft/den 4th bedroom, private pool with large screen enclosed patio, Jacuzzi bath etc.
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text:
Mandell Douglas & Bennett's Principles & Practice of Infecti
Bennett John
Publisher Churchill Livingstone
Year 01/10/2004
Version hardback
Readership level Professional and scholarly
Categories Infectious & contagious diseases
$414.58 (with VAT)
1 843.00 PLN / €395.14 / £343.02
Delivery to United States
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Widely acknowledged as the definitive reference source of its kind, "Principles and Practice of Infectious Diseases" is back - in a new 6th Edition! Authored by a "who's who" of experts from around the world, it encompasses all that is currently known about the epidemiology, aetiology, pathology, microbiology, immunology, and treatment of infectious agents. Completely revised and updated - and with a bold, new 4-colour format - it is a complete, essential compendium of knowledge on this vast and complex subject. The 2-volume set also includes a bound-in CD-ROM with all of the images from the book available for PowerPoint download.
VOLUME 1 Part I. Basic Principles in the Diagnosis and Management of Infectious Diseases SECTION A. MICROBIAL VIRULENCE FACTORS 1. A Molecular Perspective of Microbial Pathogenicity 2. Microbial Adherence 3. Toxins SECTION B. HOST DEFENSE MECHANISMS 4. Innate (General or Nonspecific) Host Defense Mechanisms 5. Human Genetics and Infection 6. Antibodies 7. Complement 8. Granulocytic Phagocytes 9. Cell-Mediated Defense against Infection 10. Nutrition, Immunity, and Infection 11. Evaluation of the Patient with Suspected Immunodeficiency SECTION C. EPIDEMIOLOGY OF INFECTIOUS DISEASES 12. Epidemiologic Principles 13. Emerging and Re-emerging Infectious Disease Threats 14. Hospital Preparedness for Emerging and Highly Contagious Infections Diseases: Getting Ready for SARS or Whatever Comes Next SECTION D. 15. The Clinician and the Microbiology Laboratory SECTION E. ANTI-INFECTIVE THERAPY 16. Principles of Anti-infective Therapy 17. Molecular Mechanisms of Antibiotic Resistance in Bacteria 18. Pharmacokinetics and Pharmacodynamics of Anti-infective Agents 19. Penicillins 20. Cephalosporins 21. Other b-Lactam Antibiotics 22. b-Lactam Allergy 23. Fusidic Acid 24. Aminoglycosides 25. Tetracyclines and Chloramphenicol 26. Rifamycins 27. Metronidazole 28. Macrolides, Clindamycin, and Ketolides 29. Glycopeptides (Vancomycin and Teicoplanin), Streptogramins (Quinupristin-Dalfopristin), and Lipopeptides (Daptomycin) 30. Polymyxins (Polymixin B and Colistin) 31. Oxazolidinones 32. Sulfonamides and Trimethoprim 33. Quinolones 34. Urinary Tract Agents: Nitrofurantoin and Methenamine 35. Topical Antibacterials 36. Antimycobacterial Agents 37. Systemic Antifungal Agents 38. Antiviral Drugs (Other Than Antiretrovirals) 39. Immunomodulators 40. Hyperbaric Oxygen 41. Agents Active against Parasites and Pneumocystis 42. Complementary and Alternative Medicines for Infectious Diseases 43. Antimicrobial Management and Cost Containment 44. Interpretating the Results of Clinical Trials on Antimicrobial Agents 45. Outpatient Intravenous Antibiotic Therapy 46. Tables of Antimicrobial Agent Pharmacology PART II. Major Clinical Syndromes SECTION A. FEVER 47. Temperature Regulation and the Pathogenesis of Fever 48. Fever of Unknown Origin 49. The Acutely Ill Patient with Fever and Rash SECTION B. UPPER RESPIRATORY TRACT INFECTIONS 50. The Common Cold 51. Pharyngitis 52. Acute Laryngitis 53. Acute Laryngotracheobronchitis (Croup) 54. Otitis Externa, Otitis Media, and Mastoiditis 55. Sinusitis 56. Epiglottitis 57. Infections of the Oral Cavity, Neck, and Head SECTION C. PLEUROPULMONARY AND BRONCHIAL INFECTIONS 58. Acute Bronchitis 59. Chronic Obstructive Pulmonary Disease, Chronic Bronchitis, and Acute Exacerbations 60. Bronchiolitis 61. Acute Pneumonia 62. Pleural Effusion and Empyema 63. Lung Abscess 64. Chronic Pneumonia 65. Cystic Fibrosis SECTION D. URINARY TRACT INFECTION 66. Urinary Tract Infections SECTION E. SEPSIS 67. Sepsis, Severe Sepsis, and Septic Shock SECTION F. INTERABDOMINAL INFECTION 68. Peritonitis and Intraperitoneal Abscesses 69. Infections of the Liver and Biliary System 70. Pancreatic Infections 71. Splenic Abscess 72. Appendicitis 73. Diverticulitis and Typhlitis SECTION G. CARDIOVASCULAR INFECTIONS 74. Endocarditis and Intravascular Infections 75. Infections of Prosthetic Valves and Other Cardiovascular Devices 76. Prophylaxis of Infective Endocarditis 77. Myocarditis and Pericarditis 78. Mediastinitis SECTION H. CENTRAL NERVOUS SYSTEM INFECTIONS 79. Approach to the Patient with Central Nervous System Infection 80. Acute Meningitis 81. Cerebrospinal Fluid Shunt Infections 82. Chronic Meningitis 83. Encephalitis, Myelitis, and Neuritis 84. Brain Abscess 85. Subdural Empyema, Epidural Abscess, and Suppurative Intracranial Thrombophlebitis SECTION I. SKIN AND SOFT TISSUE INFECTIONS 86. Cellulitis and Subcutaneous Tissue Infections 87. Myositis 88. Lymphadenitis and Lymphangitis SECTION J. GASTROINTESTINAL INFECTIONS AND FOOD POISONING 89. Principles and Syndromes of Enteric Infection 90. Esophagitis 91. Nausea, Vomiting, and Noninflammatory Diarrhea 92. Antibiotic-Associated Colitis 93. Inflammatory Enteritides 94. Enteric Fever and Other Causes of Abdominal Symptoms with Fever 95. Foodborne Disease 96. Tropical Sprue/Enteropathy 97. Whipple's Disease SECTION K. BONE AND JOINT INFECTIONS 98. Infectious Arthritis of Native Joints 99. Osteomyelitis 100. Infections with Prostheses in Bones and Joints SECTION L. DISEASES OF THE REPRODUCTIVE ORGANS AND SEXUALLY TRANSMITTED DISEASES 101. Genital Skin and Mucous Membrane Lesions 102. Urethritis 103. Vulvovaginitis and Cervicitis 104. Infections of the Female Pelvis 105. Prostatitis, Epididymitis, and Orchitis SECTION M. EYE INFECTIONS 105. Microbial Conjunctivitis 107. Microbial Keratitis 108. Endophthalmitis 109. Infectious Causes of Uveitis 110. Periocular Infections SECTION N. HEPATITIS 111. Acute Viral Hepatitis 112. Chronic Viral Hepatitis SECTION O. ACQUIRED IMMUNODEFICIENCY SYNDROME 113. Global Perspectives on Human Immunodeficiency Virus Infection and Acquired Immunodeficiency Syndrome 114. Epidemiology and Prevention of Acquired Immunodeficiency Syndrome and Human Immunodeficiency Virus Infection 115. Diagnosis of Human Immunodeficiency Virus Infection 116. The Immunology of Human Immunodeficiency Virus Infection 117. General Clinical Manifestations of Human Immunodeficiency Virus Infection (Including the Acute Retroviral Syndrome and Oral, Cutaneous, Renal, Ocular, and Cardiac Diseases) 118. Pulmonary Manifestations of Human Immunodeficiency Virus Infection 119. Gastrointestinal and Hepatobiliary Manifestations of Human Immunodeficiency Virus Infection 120. Neurologic Diseases Caused by Human Immunodeficiency Virus-1 and Opportunistic Infections 121. Malignancies in Human Immunodeficiency Virus Infection 122. Human Immunodeficiency Virus Infection in Women 123. Pediatric Human Immunodeficiency Virus Infection 124. Antiretroviral Therapy for Human Immunodeficiency Virus Infection 125. Management of Opportunistic Infections Associated with Human Immunodeficiency Virus Infection 126. Vaccines for Human Immunodeficiency Virus-1 Infection SECTION P. MISCELLANEOUS SYNDROMES 127. Chronic Fatigue Syndrome Index, i VOLUME 2 PART III. Infectious Diseases and Their Etiologic Agents SECTION A. VIRAL DISEASES 128. Introduction to Viruses and Viral Diseases 129. Orthopoxviruses: Vaccinia (Smallpox Vaccine), Variola (Smallpox), Monkeypox, and Cowpox 130. Other Poxviruses That Infect Humans: Parapoxviruses, Molluscum Contagiosum, and Yatapox 131. Introduction to Herpesviridae 132. Herpes Simplex Virus 133. Varicella-Zoster Virus 134. Cytomegalovirus 135. Epstein-Barr Virus (Infectious Mononucleosis) 136. Human Herpesvirus Types 6 and 7 137. Kaposi's Sarcoma Associated Herpesvirus (Human Herpesvirus Type 8) 138. Herpes B Virus 139. Adenovirus 140. Papillomaviruses 141. JC, BK, and Other Polyomaviruses; Progressive Multifocal Leukoencephalopathy 142. Hepatitis B Virus and Hepatitis Delta Virus 143. Parvovirus B19 144. Orthoreoviruses and Orbiviruses 145. Coltiviruses and Seadornaviruses (Colorado Tick Fever) 146. Rotaviruses 147. Alphaviruses 148. Rubella Virus (German Measles) 149. Flaviviruses (Yellow Fever, Dengue, Dengue Hemorrhagic Fever, Japanese Encephalitis, West Nile Encephalitis, St. Louis Encephalitis, Tick-Borne Encephalitis) 150. Hepatitis C 151. Hepatitis G Virus and TT Virus 152. Coronaviruses, Including Severe Acute Respiratory Syndrome (SARS)-Associated Coronavirus 153. Parainfluenza Viruses 154. Mumps Virus 155. Respiratory Syncytial Virus 156. Human Metapneumovirus 157. Measles Virus (Rubeola) 158. Zoonotic Paramyxoviruses: Hendra, Nipah, and Menangle Viruses 159. Vesicular Stomatitis and Related Viruses 160. Rabdoviruses 161. Marburg and Ebola Virus Hemorrhagic Fevers 162. Influenza Virus 163. California Encephalitis, Hantavirus Pulmonary Syndrome, and Bunyavirid Hemorrhagic Fevers 164. Lymphocytic Choriomeningitis Virus, Lassa Virus, and the South American Hemorrhagic Fevers 165. Human T-Cell Lymphotropic Virus Types I and II 166. Human Immunodeficiency Viruses 167. Introduction to the Enteroviruses 168. Poliovirus 169. Coxsackieviruses, Echoviruses, and Newer Enteroviruses 170. Hepatitis A Virus 171. Rhinovirus 172. Noroviruses and Other Caliciviruses 173. Astroviruses and Picobirnaviruses 174. Hepatitis E Virus SECTION B. PRION DISEASES 175. Prions and Prion Diseases of the Central Nervous System (Transmissible Neurodegenerative Diseases) SECTION C. CHLAMYDIAL DISEASES 176. Introduction to Chlamydial Diseases 177. Chlamydia trachomatis (Trachoma, Perinatal Infections, Lymphogranuloma Venereum, and Other Genital Infections) 178. Chlamydophila (Chlamydia) psittaci (Psittacosis) 179. Chlamydophila (Chlamydia) pneumoniae SECTION D. MYCOPLASMA DISEASES 180. Introduction to Mycoplasma Diseases 181. Mycoplasma pneumoniae and Atypical Pneumonia 182. Genital Mycoplasmas: Mycoplasma genitalium, Mycoplasma hominis, and Ureaplasma urealyticum SECTION E. RICKETTSIOSES AND EHRLICHIOSES 183. Introduction to Rickettsioses and Ehrlichioses 184. Rickettsia rickettsii and Other Spotted Fever Group Rickettsiae (Rocky Mountain Spotted Fever and Other Spotted Fevers) 185. Rickettsia akari (Rickettsialpox) 186. Coxiella burnetii (Q Fever) 187. Rickettsia prowazekii (Epidemic or Louse-Borne Typhus) 188. Rickettsia typhi (Murine Typhus) 189. Scrub Typhus 190. Ehrlichia chaffeensis (Human Monocytotropic Ehrlichiosis), Anaplasma phagocytophila (Human Granulocytotropic Anaplasmosis), and Other Ehrlichieae SECTION F. BACTERIAL DISEASES 191. Introduction to Bacteria and Bacterial Diseases 192. Staphylococcus aureus (Including Staphylococcal Toxic Shock) 193. Staphylococcus epidermidis and Other Coagulase-Negative Staphylococci 194. Classification of Streptococci 195. Streptococcus pyogenes 196. Nonsuppurative Poststreptococcal Sequelae: Rheumatic Fever and Glomerulonephritis 197. Streptococcus pneumoniae 198. Enterococcus Species, Streptococcus bovis, and Leuconostoc Species 199. Streptococcus agalactiae (Group B Streptococcus) 200. Viridans Streptococci, Groups C and G Streptococci, and Gemella morbillorum 201. Streptococcus anginosus Group (or milleri Group or intermedius Group) 202. Corynebacterium diphtheriae 203. Other Coryneform Bacteria and Rhodococcus 204. Listeria monocytogenes 205. Bacillus anthracis (Anthrax) 206. Bacillus Species and Related Genera Other Than Bacillus anthracis 207. Erysipelothrix rhusiopathiae 208. Neisseria meningitidis 209. Neisseria gonorrhoeae 210. Moraxella (Branhamella) catarrhalis and Other Gram-Negative Cocci 211. Vibrio cholerae 212. Other Pathogenic Vibrios 213. Campylobacter jejuni and Related Species 214. Helicobacter pylori and Other Gastric Helicobacter Species 215. Enterobacteriaceae 216. Pseudomonas aeruginosa 217. Stenotrophomonas maltophilia and Burkholderia cepacia 218. Burkholderia pseudomallei and Burkholderia mallei: Melioidosis and Glanders 219. Acinetobacter Species 220. Salmonella Species, Including Salmonella typhi 221. Shigella Species (Bacillary Dysentery) 222. Haemophilus Infections 223. Brucella Species 224. Francisella tularensis (Tularemia) 225. Pasteurella Species 226. Yersinia Infections, Including Plague 227. Bordetella Species 228. Streptobacillus moniliformis (Rat-Bite Fever) 229. Legionnaires' Disease and Pontiac Fever 230. Other Legionella Species 231. Capnocytophaga 232. Infections Due to Bartonella Species, Including Cat-Scratch Disease 233. Calymmatobacterium granulomatis (Donovanosis, Granuloma Inguinale) 234. Other Gram-Negative Bacilli 235. Treponema pallidum (Syphilis) 236. Endemic Treponematoses 237. Leptospirosis 238. Borrelia Species (Relapsing Fever) 239. Borrelia burgdorferi (Lyme Disease, Lyme Borreliosis) 240. Spirillum minus (Rat-Bite Fever) 241. Anaerobic Infections: General Concepts 242. Clostridium tetani (Tetanus) 243. Clostridium botulinum (Botulism) 244. Gas Gangrene and Other Clostridium-Associated Diseases 245. Bacteroides, Prevotella, Porphyromonas, and Fusobacterium Species (and Other Medically Important Anaerobic Gram-Negative Bacilli) 246. Anaerobic Cocci 247. Anaerobic Gram-Positive Nonsporulating Bacilli 248. Mycobacterium tuberculosis 249. Mycobacterium leprae (Leprosy, Hansen's Disease) 250. Mycobacterium avium Complex 251. Infections Due to Nontuberculous Mycobacteria 252. Nocardia Species 253. Agents of Actinomycosis SECTION G. MYCOSES 254. Introduction to Mycoses 255. Candida Species 256. Aspergillus Species 257. Agents of Mucormycosis and Related Species 258. Sporothrix schenckii 259. Agents of Chromoblastomycosis 260. Agents of Mycetoma 261. Cryptococcus neoformans 262. Histoplasma capsulatum 263. Blastomyces dermatitidis 264. Coccidioides Species 265. Dermatophytosis and Other Superficial Mycoses 266. Paracoccidioides brasiliensis 267. Uncommon Fungi 268. Pneumocystis Species SECTION H. PROTOZOAL DISEASES 269. Introduction to Protozoal Diseases 270. Entamoeba histolytica (Amebiasis) 271. Free-Living Amebas 272. Plasmodium Species (Malaria) 273. Leishmania Species: Visceral (Kala-Azar), Cutaneous, and Mucocutaneous Leishmaniasis 274. Trypanosoma Species (American Trypanosomiasis, Chagas' Disease): Biology of Trypanosomes 275. Agents of African Trypanosomiasis (Sleeping Sickness) 276. Toxoplasma gondii 277. Giardia lamblia 278. Trichomonas vaginalis 279. Babesia Species 280. Cryptosporidiosis (Cryptosporidium hominis, Cryptosporidium parvum, and Other Species) 281. Cyclospora cayetanensis, Isospora belli, Sarcocystis Species, Balantidium coli, and Blastocystis hominis 282. Microsporidiosis SECTION I. DISEASES DUE TO TOXIC ALGAE 283. Human Illness Associated with Harmful Algal Blooms SECTION J. DISEASES DUE TO HELMINTHS 284. Introduction to Helminth Infections 285. Intestinal Nematodes (Roundworms) 286. Tissue Nematodes. Including Trichinosis, Dracunculiasis, and the Filariases 287. Trematodes (Schistosomes and Other Flukes) 288. Cestodes (Tapeworms) 289. Visceral Larva Migrans and Other Unusual Helminth Infections SECTION K. ECTOPARASITIC DISEASES 290. Introduction to Ectoparasitic Diseases 291. Lice (Pediculosis) 292. Scabies 293. Myiasis and Tungiasis 294. Mites (Including Chiggers) 295. Ticks (Including Tick Paralysis) SECTION L: DISEASES OF UNKNOWN ETIOLOGY 296. Kawasaki Syndrome PART IV. Special Problems 304. Human Immunodeficiency Virus in Health Care Settings 305. Nosocomial Herpesvirus Infections SECTION B. INFECTIONS IN SPECIAL HOSTS 306. Infections in the Immunocompromised Host: General Principles 307. Infections in Patients with Hematologic Malignancies 308. Prophylaxis and Empirical Therapy for Infection in Cancer Patients 309. Infections in Injection Drug Users 310. Risk Factors and Approaches to Infections in Transplant Recipients 311. Infections in Recipients of Hematopoietic Stem Cell Transplantation 312. Infections in Solid Organ Transplant Recipients 313. Infections in Patients with Spinal Cord Injury 314. Infections in the Elderly 315. Infections in Asplenic Patients SECTION C. SURGICAL- AND TRAUMA-RELATED INFECTIONS 316. Postoperative Infections and Antimicrobial Prophylaxis 317. Burns 318. Bites SECTION D. IMMUNIZATION 319. Immunization SECTION E. BIODEFENSE 320. Bioterrorism: An Overview 321. Plague as an Agent of Bioterrorism 322. Francisella tularensis (Tularemia) as an Agent of Bioterrorism 323. Smallpox and Bioterrorism 324. Anthrax 325. Botulinum Toxin as a Biological Weapon 326. Bioterrorism: Viral Hemorrhagic Fevers SECTION F. ZOONOSES 327. Zoonoses SECTION G. PROTECTION OF TRAVELERS 328. Protection of Travelers 329. Infections in Returning Travelers SECTION H. THE INTERNET 330. The Infectious Diseases Physician and Digital Resources Index
Malaria and the DDT Story
Richard Tren
New Cure for Tuberculosis by Its Own..
J Compton Burnett
Antibiotic Resistance: Causes & Risk..
Adriel R Bonilla
Salmonella Infections: New Research
Lorenzo S Giordano


text:
Hecate Enthroned - "Virulent Rapture" (CD)
"Virulent Rapture" track listing:
1. Thrones of Shadow	(4:46)
2. Unchained (4:37)
3. Abyssal March (4:08)
4. Plagued by Black Death (7:59)
5. Euphoria (4:43)
6. Virulent Rapture (5:30)
7. Life (5:19)
8. To Wield the Hand of Perdition (3:05)
9. Of Witchery and the Blood Moon (5:33)
10. Immateria (3:05)
11. Paths of Silence (6:43)
Reviewed by xFiruath on December 9, 2013
"To be honest, "Virulent Rapture" sounds nothing like the band's early days, but the new sound is pretty killer, and it caps and furthers the earlier material admirably."
"Upon Promethean Shores (Unscriptured Waters)" was one of my early forays into black metal, and that debut EP from U.K. outfit Hecate Enthroned remains a personal favorite to this day. Primarily active from the late '90s to early '00s, the band was around for the tail end of European black metal's heyday, last releasing an album back in 2004. Nine years later the group is back with "Virulent Rapture," and while the sound has changed, the band continues to show why it was considered a powerhouse long ago.
Hecate Enthroned has been through several vocalists at this point, and this time around Elliot Beaver offers the growls and screams. His higher shrieks are incredibly hoarse, and then there are the more guttural growls complimenting the band's death edge that keeps it from being straight kvlt black metal. To be honest, "Virulent Rapture" sounds nothing like the early days, but the new sound is pretty killer, and it caps and furthers the earlier material admirably.
While not a complete rehash of the '90s sound, these tracks still give off that old school vibe, but they are beefed up with bigger, more booming sounds. In proper U.K. black metal style, there are atmospheric and melodic parts interwoven throughout, and the symphonic elements even bring to mind the epic hybrid bands like Fleshgod Apocalypse. An instance where the mixing of melodic and extreme works particularly well is the piano intro of "Plagued By Black Death," which then continues unabated throughout the song in the background of the furious black metal.
With a varied lineup of songs – there are three minute tracks and eight minute tracks, symphonic parts and brutal parts – "Virulent Rapture" is an all-around solid disc. The mix of black with death and classic with modern creates a satisfyingly frozen experience to cap off the year in metal that will appeal to old fans or new converts.
Highs: Black meets death, symphonic meets extreme, classic meets modern
Lows: The band essentially sounds nothing like it used to, and the acoustic guitar interlude track is a bit out of place
Bottom line: Hecate Enthroned caps off the year with an impressive mixing of black with death, modern with classic, and extreme with symphonic.
Get more info including news, reviews, interviews, links, etc. on our Hecate Enthroned band page.


To all of the fathers out there, enjoy your special day! There is no greater love than the one a daughter can get from her father and a son needs the example of what a good man is! Keep up the good work.
Would You Live in a Van for a Couple Years to Pay Off All of Your Student Loans?


text:
#import "NSObject.h"

@class CUIPSDImageRef;

@interface CUIPSDLayerBaseRef : NSObject
{
    CUIPSDImageRef *_imageRef;
    unsigned int _layerIndex;
}

- (struct CGImage *)_createMaskFromAlphaChannel:(long long)arg1;
- (id)vectorMaskRef;
- (id)layerMaskRef;
@property(readonly) _Bool hasVectorMask;
@property(readonly) _Bool hasLayerMask;
@property(readonly) int blendMode;
@property(readonly) double opacity;
@property(readonly) _Bool visibility;
@property(readonly) struct CGRect bounds;
- (_Bool)isLayerGroup;
- (id)name;
- (id)_psdImageRef;
- (struct CPSDLayerRecord *)_psdLayerRecord;

@end

redpajama_set_name: RedPajamaGithub


text:
Q: Should I worry about DDMS console log messages "Can't bind to local nnnn for debugger"? I'm new to Android programming (and Eclipse IDE and Android emulator).  I've got Hello World and some of Notepad working, but I'm still constantly getting quite a few DDMS console log messages (shown below) about not being able to bind locals for debugger.
[2010-05-29 21:03:16 - ddms] Can't bind to local 8601 for debugger
[2010-05-29 21:05:26 - Device] Failed to delete temporary package: device (emulator-5556) request rejected: device not found
[2010-05-29 21:06:47 - ddms] Can't bind to local 8600 for debugger
[2010-05-29 21:07:05 - ddms] Can't bind to local 8601 for debugger
[2010-05-29 21:07:05 - ddms] Can't bind to local 8602 for debugger

And so on. Is this a problem?  Can I get rid of these messages somehow?

A: You can get rid of the the messages by adding following to your hosts file:
127.0.0.1 localhost

ps. hosts file can be found from c:\windows\system32\drivers\etc\hosts
    or if you are on linux /etc/hosts

A: In Eclipse, goto
1) Windows->Preference
2) Expand Android menu in the side of the Preference Window.
3) Now select DDMS from it.
4) Then Set the Base local debugger port to "8601" and enable "Use ADBHOST" checkbox and the ABDHOST value should be "127.0.0.1".
5) Click apply and ok.
6) Now you have to restart your ADB, for this you have to login as root user(Assuming linux user) and navigate to Platfrom tools folder of your Android SDK. Then execute,
 ./adb kill-server
 ./adb start-server

You will be able to see the message "Daemon started Successfully" . If not repeat step 6 once again until you see the success message.

A: In my situation the problem has been solved by a uninstalling all Java 7. The debugger is now working again!