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Moses Farrow Defends Woody Allen, and His Family Pushes Back
Woody Allen and Mia Farrow in the mid-1980s with four of her children, Fletcher, Dylan (in her arms), Moses and Soon-Yi, at right.
Moses Farrow, the adopted son of Woody Allen and Mia Farrow, posted a 4,600-word essay on Wednesday defending his father against sexual molestation allegations and claiming his mother was abusive.
In the blog post, titled "A Son Speaks Out," Mr. Farrow, 40, described repeated spankings by his mother and other abuse, and what he called "a deep and persistent darkness within the Farrow family."
He recalled, too, his version of the events on Aug. 4, 1992, when his sister, Dylan Farrow, then 7, claimed Mr. Allen, one of the most prolific directors in modern cinema, sexually molested her at their home in Bridgewater, Conn. Moses was 14.
On Thursday night, Mia Farrow said that her estranged son's claims were "completely made up."
And Dylan Farrow, now 32, responded to the essay in a tweet, writing that her brother sought to "impugn my mother who has only ever been supportive of me and my siblings."
She called his comments hurtful and an attempt to discredit her. "I'm so sorry he's doing this," she wrote.
The journalist Ronan Farrow, 30, also defended his mother and dismissed his older brother's essay, saying Thursday on Twitter, "I believe my sister."
This is not the first time Moses Farrow has supported his father in an acrimonious family battle that has pitted mother, father, brothers and sisters against one another in a very public scrum. In his essay, he said he decided to speak out again now, hoping people will be more receptive to his story.
[A timeline: Woody Allen, Mia Farrow and Dylan Farrow]
His mother acted in more than a dozen of Mr. Allen's films while they were together from 1979 to 1992, although they lived in separate Manhattan apartments on opposite sides of Central Park.
They split after she discovered nude photographs Mr. Allen had taken of Soon-Yi Previn, then 22, the daughter she had adopted while married to André Previn, the pianist and conductor.
Mr. Allen and Ms. Previn married in 1997 and have two adopted children.
In "Start to Finish: Woody Allen and the Art of Moviemaking," a book published last year by the author Eric Lax, Moses Farrow said his mother was abusive and had coached his sister to accuse Mr. Allen of sexual abuse.
In his essay, Mr. Farrow, who is a family therapist, claimed that his mother spanked and slapped him, and sought to brainwash him and his siblings to keep them under her control. In all, Ms. Farrow oversaw a brood of 14 children. Ten were adopted. (Three of her adopted children are deceased.)
"In short, it was not a happy home — or a healthy one," he wrote.
The comments about his sister, Dylan, are the most provocative. In the essay, he questioned his sister's account of sexual molestation, writing that her memory was faulty. For example, he wrote that an electric train set she described seeing was not kept in the attic, where she said she was assaulted, but in a brother's room. Further, he wrote, there were too many people around not to notice something was amiss.
In 2014, Dylan wrote a letter detailing her allegations against her father. (It was published on the blog of Nicholas Kristof, a Farrow family friend and columnist for The Times, who also wrote about it on the Op-Ed page.)
Also in recent years, her brother Ronan, who was 4 at the time of the allegations, has spoken out about her case, while reporting on sexual misconduct by Harvey Weinstein and other powerful men for The New Yorker, work that shared a Pulitzer Prize with The Times this year. With greater attention on harassment and sexual misconduct from the #MeToo movement, many in Hollywood have said they would not work with Mr. Allen again.
In his statement on Twitter, the journalist said his brother's essay was part of a "repeated campaign to discredit my sister, often by attacking our mother."
He continued, "My mother did an extraordinary job raising us, and none of my siblings with whom I've spoken ever witnessed anything but love and care from a single mom who went through hell to keep her kids safe."
In a brief interview over direct message on Twitter, Mia Farrow said Thursday that Moses had cut off all ties with the family years ago.
"I guess I should be used to these attacks after 20 years," she said. "But still, using my son as a weapon is new and excruciating. Also, it's still flabbergasting that he can tell so many lies when the facts are readily available." She said they are clearly listed in court documents from the original investigation.
That 1993 investigation into Dylan Farrow's allegations took several turns. She was interviewed nine times, and the doctor who headed the investigation, John M. Leventhal, said there were inconsistencies in her account. That same year, Ms. Farrow was awarded custody of her three children with Mr. Allen. Months later, a Connecticut state attorney declined to prosecute Mr. Allen despite "probable cause" on charges of sexual molestation.
The decision was made to spare Dylan Farrow additional trauma, the prosecutor said, with agreement from her mother.
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WHAT DO SENIOR CITIZENS IN MALAYSIA WANT?
Our recent trip to Singapore for the 50plus Expo at Suntec left us more than a tad envious of Singaporean seniors. This year marks the silver jubilee of Singapore's independence in 1965. Singapore citizens, including permanent residents can look forward to receiving plenty of goodies from the government.
As a tribute to the pioneer generation for their contribution to nation-building, the government has put together a SG50 Senior Package that will benefit more than 700,000 older Singaporeans. From transport vouchers to discounts for dining, as well as free admission to some of the country's top tourist attractions, there is plenty in the package to put Singapore seniors in a celebratory mood.
The package comes with a 50-page booklet, with a complete list of the benefits. Most of the offers are valid from June to August this year.
And that's not all. In her speech at the launch of 50plus Expo on 16 May, Senior Minister Dr Amy Khor announced that Singapore seniors can sign up for 400 free courses ranging from literature and dance to business and culinary skills. The Ministry of Health aims to encourage lifelong learning with this generous perk.
For seniors that surely must be the icing on the cake to top off the year-long silver jubilee celebrations. No wonder the Singapore cheer turned into a deafening Singapore roar at Dr Khor's announcement.
So, that begs the question, what can our government offer us older Malaysians to make us happy and looking forward to our retirement years? We have given 30-40 of our prime years in the service of the country. Surely that must merit some recognition and appreciation?
We want an outdoor gym for seniors in every housing estate and public park where we can exercise
We don't want awards and titles, for they benefit only a few selected individuals, not the community. (By the way, Singaporeans from the PM to the man-in-the-street are addressed as Mr, and for the ladies, a plain Ms/Mdm.)
We don't want cash handouts which offer only short-term benefits to the needy. We want senior discounts on groceries, health supplements, fuel and meals, and other essentials.
Above all, we want to be accorded respect and dignity, not ignored or seen as unproductive and a burden to society
We certainly don't want empty pre-election promises of what the government can do, will do for us. We also don't want general references to what it plans to do for seniors. We want details, specifics and deadlines. And if the government doesn't deliver, we have the right to protest, to hold it accountable if it reneges on its word, and to withhold our vote if any of our under-performing ministers should stand for re-election. That's not a threat or a warning, but a straightforward case of cause and result.
So, what do we want?
Here's a short checklist to begin with for the relevant ministeries to take note of. In no particular order.
more elder-friendly facilities in public places
a public transport system that takes into account the physical limitations of the elderly
a senior privilege card with genuine discounts that covers items seniors regularly spend on.
senior community centres in every housing area or constituency
more opportunties for re-training and re-employment of seniors
more affordable nursing homes and assisted living facilities
lifelong learning programmes to be extended to more areas
respect and dignity, and no age discrimination, please
An impossible dream, or a soon-to-be reality?
If you click on any of the links above, you will find that SeniorsAloud have been making these proposals as far back as 2009. We will continue to voice our concerns till we are heard.
To give credit where credit is due, we appreciate the government's efforts in making public healthcare accessible and affordable to seniors. We welcome the discounts for seniors travelling on trains and buses. We also acknowledge the financial assistance given for funeral expenses (!) under the Mesra Usia Emas Scheme and other schemes.
With seniors making up 8.8% of the population and growing steadily, Malaysia is on its way to becoming an ageing nation in the next 10-15 years. UN defines a country as ageing when 7% of its population is aged 65 and above.
But these provisions are either limited, too slow in implementation, or if already available are not efficiently maintained or managed.
Each year when the budget is announced, we scan the papers looking for some tiny morsels of good news for seniors, only to be disappointed yet again. Last week, the PM announced details of the 11th Malaysia Plan for 2016-20. To date, we still have no clue what 'supporting active ageing for the elderly' involves, or how 'the government will address the needs of...the elderly'. (The Star 22/5/15)
How long do seniors in Malaysia have to wait before they hear these words from our ministers? Words are cheap, unless they are backed by prompt and effective action.
Our ministers are seniors themselves. Like us, they have elderly parents. One day they too will be elderly. Isn't it time they gave more attention to what the seniors and the elderly want?
Labels: dignity and respect for seniors, elder-friendly faciltiies
WHAT THE COMMUNITY CAN DO TO PROVIDE CARE FOR THE ...
50PLUS EXPO REVISITED 2015
SEX IN YOUR 60s - EASIER SAID THAN DONE


It was over 13 years ago that many of you gathered together in a stake fast as I prepared for my double lung transplant. It is a blessing to have a platform to thank you these many years later for all that you have done for me! I am so humbled and honored to speak to you today about the lessons I have learned in my life.
Like all of you, I have experienced quite a few storms. Yet, I have learned that no matter the winds and the tempests that rage, I am eternally filled with peace and hope because of the great plan of salvation – knowing where I came from, who I am, why I am here and where I am going. This knowledge is my lighthouse. It is my beacon of hope and peace in this dark world. It illuminates the path home and it brightens my sometimes weary and heavy heart as it reminds me that because of the atonement of Jesus Christ all of our sorrow, sickness and pain will not follow us in the next life. If we follow the Savior, His light will guide us back to our loving father in heaven where we all came from and where we all ultimately belong.
I have learned this life is a time of proving our faithfulness to our Father in Heaven.
Before this earthly life we participated in the grand council in heaven where the plan of salvation was presented to us. We learn from Job 38: 7 that when this plan was presented, that we shouted for joy! We were excited at the chance to obtain a physical body, walk by faith, learn obedience to God's commandments and try to become more like Christ. Still, we weren't naive to life's difficulties.
We knew this life would be hard. We knew some of us would be born with health problems, a challenging family life, poverty, or any other difficult situation. I believe we each knew of our personal circumstances and we all—every single one of us—still shouted for joy to come to earth!
We knew each of us would have the capacity to "bear our challenges in life because of who we are, who God is, and who we are together." We had the eternal perspective to know that proving ourselves true and faithful on earth, no matter what, was all going to be worth it in the end.
Now that we are here on earth, the trials and disappointments of life can discourage us, break our hearts and even fill us with fear.
This life is a test of our faith and a test of our obedience.
Learning your child has a chronic disease is probably one of the most difficult situations a person can encounter. Instead of becoming embittered, and asking the often unanswerable and always destructive questions such as, "Why us?" "Why me?" My parents chose to draw strength from their eternal perspective.
Even though we can't control all of our circumstances, we can control our faith. All the waves of anger, heartache, guilt, doubt, despair, and loneliness cannot drown us unless we let them.
We are always free to choose faith, trust and hope in Jesus Christ and the glorious plan of salvation.
The same is true for us. Heavenly Father knew us before we were formed in the belly and he provided a plan to help bring us back to Him.
We are divine beings having a mortal experience.
In addition to knowing where we came from, I have learned it is important to remember who we really are.
I believe we will never be happy or feel peace…and we will definitely never live up to who we are as women of God until we overcome our "mortal identity" crisis by understanding who we really are.
Understanding who I really am –that I am a daughter of a King, my Heavenly Father, He who controls this universe – and how loved I truly am – has blessed me with peace, comfort and hope beyond measure.
At age thirteen my full, deep breaths had been replaced with the wheezing of tightened, obstructed airways. Over the years as my health involved more and more challenges, I was increasingly brought to my knees in prayer, seeking both strength and comfort. Many nights I would feel reassured simply after confiding in my Father in Heaven, knowing He was there, listening, caring. But on a handful of occasions as I called on my Heavenly Father with my shallow breath, He answered me with His deep, infinite and tangible love. There was no room to doubt what I already knew: God is real. God loves me. I had nothing to fear. I had every reason to hope.
True to Sheri Dew's statement, the knowledge of who I really am – and how loved I really am – has shaped my entire life. It has allowed me to trust my Heavenly Father and his will perfectly because I know He loves me perfectly.
I know God's arms of fatherly love are waiting to embrace every single one of us. We are all His literal spirit daughters with infinite worth and glorious potential!
Unfortunately, Satan, the master of deceit, will do everything he can to keep us from comprehending the majesty of who we are which leads to feelings of inadequacy, discouragement and defeat instead of confidence, hope and happiness.
But the truth is: We cannot buy happiness. We cannot buy peace. We cannot buy hope.
Sheri Dew continues: "Satan, however would have us see ourselves as the world sees us, not as the Lord see's us. The world would tell us that our roles as women and nurturers are not enough. That we have to do more, be more, have more, juggle more. That we have to be smarter, thinner, prettier just to feel a little bit good about ourselves. If we live by the world's standards, we will NEVER be enough.
Family is eternal. Careers, cars, clothes and cosmetics are not.
I believe that if we could catch a glimpse of the divinity of our roles, we would be so proud of ourselves and we would be kicking ourselves for being preoccupied with things that don't matter and never will! Family is what matters. Family is eternal.
God who knows us perfectly – both our weaknesses and our strengths- reserved us to come now, when the opposition would be strongest – When he would need fearless woman who would fulfill their divine callings as woman and mothers.
We need to trust, be obedient and be content in the knowledge that we have been placed where we can have the most impact and simply do the best we can, standing steadfast and immovable against all the lies and temptations Satan would challenge us with.
I will forever by grateful for my own mother who persevered every single day in hope and kindness as she fulfilled the divine role as our mother. She knew that as long as she did her best everything would work out – whether in this life, or the next.
At the time, these seemingly monotonous, mundane acts must have not seemed like much. But there is no way to count how many months, weeks or years my parents added to the lives of their children.
Just as my mom's daily ministering and nurturing must have seemed monotonous at times, it is our small and seemingly inconsequential actions and our steadfast examples that will have profound effects upon generations to come.
I pray that we won't forget who we really are and the divinity of our roles.
Like the whale, If we mistakenly look to the world to feed our identity, we will spiritually starve. We will never be enough. In contrast, when we feast upon the teachings of Christ, we will come to know Jesus Christ is our personal Savior and God is our Father in Heaven who love us unconditionally. The knowledge that we are beloved daughters of God with infinite worth and glorious potential allows us to love ourselves as ourselves. We will not only have the desire but also the determination to live up to our potential, full of hope and confidence instead of defeat and discouragement.
When we understand where we came from, who really are and why we are here, we have every reason to hope for a glorious conclusion of our mortal test when God will reward the faithful in a perfect way.
As Justin's health grew worse, he required oxygen while he slept. Still, he continued to his senior year in his typical optimistic fashion.
As always, although this time quite literally, Justin was determined to swim as opposed to sink.
The name of the "U.S.S. Slacker" embodies Justin's sense of humor – the paddling of the U.S.S. slacker embodies Justin's courage, determination and zeal for life. He was determined to keep paddling despite whatever waves came his way and despite his half sinking ship.
At age 18, Justin was a young man who knew his situation was precarious. He could have spent all his energy indulging in self-pity, worry or resentment, counting every breath as his body slowly decayed. Instead he spent his days uplifting others….
…playing volleyball with one hand to make a friend feel more comfortable whose arm was in a cast, joking that not everyone has both an "innie and and outie" when he had to have a gastro intestinal feeding tube inserted above his belly button or joking with his friend just three days before he died about how much fun they would have together "cruising the hospital halls looking for good-looking nurses"….never once mentioning the gravity of his health.
Justin was a model of quiet dignity. In all our talks, Justin never uttered a negative word about having cystic fibrosis. How often we pity ourselves and even parade our pity over matters that aren't life or death.
Justin's attitude was born from his testimony of His Savior.
Justin's hope not only encouraged him to endure all with dignity and a sense of humor, but allowed him to do so.
One of the hardest lessons to accept is that despite our prayerful pleadings, not all of our struggles will be resolved soon, or in the way we want, or even in this lifetime. But we can have peace and hope that they will be resolved.
EVERYTHING will be made permanently right through Christ if we are faithful.
He is making of us something more than we would choose to make of ourselves. We might be content to merely be "a nice person." He wants us to be like Him and accept and qualify for our inheritance.
Struggles are part of the test we willingly and enthusiastically shouted for joy for. They give us opportunities to prove ourselves and become who we were sent here to become.
As struggles threaten to topple us, our peace and hope largely depends on if we choose to continue to exercise even greater faith and trust in our Heavenly Father's love, wisdom and plan for us – or if we choose to fall victim to Satan's feelings of anger, bitterness, discouragement and helplessness.
Because of Christ, we have nothing to fear now and every reason to hope for the future.
We must strive to see our challenges with an eternal perspective and remember that our struggles and heartache will not continue in the next life. Instead we can have faith and rejoice that because of the Father's merciful plan of providing a Savior for us, all will be made right through Christ if we are faithful.
We were all there. Looking down upon that humble stable and shouting for joy again. That someone would love us enough to come to earth to save us all.
I love this hymn because it so clearly defines the challenges of this life: "I believe in Christ; so come what may," or as Joseph B. Wirthlin expressed, "Come what may, and love it." Because of Christ, we have nothing to fear now and every reason to hope for the future.
As I mourned Justin's passing, I often felt helpless. I chose to turn to my Savior, He who knew what I was experiencing firsthand. There is no physical pain, mental anguish, heartache, weakness, or infirmity that we experience that the Savior has not already experienced.
Because Christ suffered for us individually, He understands us completely, loves us unconditionally, and knows how to succor us perfectly.
Instead, if we come unto Him and let Him ease our agonizing and grieving, I know we will be encircled in the arms of His love as He cares, comforts and carries us. I have a testimony this is true as Jesus Christ my Savior, has become Jesus Christ my Savior as I have fallen at his feet and wept again and again and again. I could not have made it through my challenges without Him; and now I feel I can make it through anything because of Him.
A year after my brother's death, my sophomore year of high school, the end stages of lung destruction brought on by cystic fibrosis put me in a downward spiral. I was on constant oxygen, constant IV antibiotics, throwing up constantly and barely had the energy to brush my teeth in bed.
I know the utterence of "My Heavenly Father" can have a profound effect upon our lives.
He really does hear our prayers – no matter how big OR how small.
Sometimes when life gets hard, we feel paralyzed, we want to give up. But it is only by staying the course, exercising even greater faith, repenting and crying unto our Savior that we will learn that He is truly mighty to save.
As I struggled for breath and as I struggled for life, it was confirmed to me again and again that God was my companion.
His spirit and this knowledge filled with sweet and exquisite joy.
I cannot comprehend a greater blessing in life – to be filled with peace, hope and joy, every step of the way. To have nothing to fear, even death!
I was too ill to wait for a cadaveric lung transplant and so a living donor lung transplant was pursued. Everyone has five lobes – three on the right side and 2 on the side of a heart. In a living lobar lung transplant, all 5 lobes are taken out of the patient and replaced with the bottom lobe from two donors. When all of my family members and three friends who were tested were disqualified, 2 family friends –Graham Bullick and Jim Davies – stepped forward to offer me lobes of their lungs. I am breathing today, alive today, and forever grateful today because of their tremendous sacrifice.
After I was taken off the ventilator after my transplant, the weight of my donor's sacrifice became even more real as I sat inhaling and exhaling with lobes from their lungs, all the while not feeling like I was breathing at all.
They have given me a greater appreciation for the atonement. They exemplify one of the reason we are here on earth: to become like Christ.
Most of us won't have the chance to physically save someone's life. But we do have the opportunity, whether we know it or not, to save individual's emotionally and spiritually everyday simply through our small acts of kindness.
We pass people everyday who are literally and metaphorically hungered, naked, and homeless. I believe Satan wants desperately for us get caught up in own selves – either or our own problems, pride or insecurities – that we fail to see these individuals as the sons and daughters of God that they are -and that they are in need of our help. Or he tries to discourage us—that no matter how hard we try, we'll never make much of a difference – because Satan knows the beautiful truth that we all have the potential to change each other's lives through service and love.
I know my life has been forever changed by love. From loving parents who dedicated their waking hours to preserving my health, to selfless home teachers who have given me countess blessings, to friends who have brought me meals, to you, my stake who fasted for me, to my donors who offered up the ultimate act of love by risking their own lives to save mine. To the Ottesens who delivered a life size cardboard cutout of Stone Cold Austin to my ICU room after surgery to other loving sentiments such as letters, posters, stuffed bears and emails.
It is truly profound the healing, both physically and emotionally, that small acts of kindness bring.
Last Sunday, my husband saw someone who looked familiar sitting in a bench at church. He said, "Jen, is that the maintenance guy for the park?" Finally, he realized who it was: surfing royalty: big wave surfing legend Jeff Clark. My husband was starstruck. He said, "I wish I would have said "Hi" when I first saw him.
Would we be more willing to "Say Hi" if we could view everyone as Christ does – as the "royalty" that they truly are– divine sons and daughters of God?
Every single person is in need of our love. Feeling loved and appreciated is probably the greatest human need after food and sometimes before it.
No one knew the paralyzing pain I was in for months and months after Justin's death. No one knew the hours I would spend every morning before high school doing home IVs, breathing treatments, and fighting fevers. When I would arrive late to school after third period, no one know how hurtful their comments such as "you are so lucky," "its so not fair," and "I hate you." were.
None of us should have illusions about our right to evaluate others or about our time here on earth. We are all going to die someday….or as I once heard it said, "none of us are going to make it out alive." It is so important to be loving while we are alive!
It is the simple gestures of kindness that make a difference.
Christ loved and served all those who came in his path. He never withheld his love for ANY reason.
Too often we are guilty of withholding our love because we have judged the individual or their circumstances as not worthy of all of our love. As Mother Teresa said, "If you judge people, you have no time to love them." But, like Christ, we should never withhold our love for ANY reason.
The Savior loved and served everyone unconditionally, from the leper to the sinner, from the blind to the believer. Even as the crown of plated thorns pierced his head, even as his flesh was aching and tearing, Christ said of his murderers and mockers, "Father, forgive them; for they know not what they do." (Luke 23:34) He was crucified for the sins of the world. Yours, mine, theirs. All he asks of us is to follow him, trying in our own small way, to love as he loved. This includes forgiving.
Some months ago after I had been ill, someone at church commented to my husband in front of me, "You need to feed your wife more." Months later, after he learned of my health situation, he came up to my husband with tears in his eyes and apologized for his statement.
How we treat the weakest of us is how we are to be judged in the end. Let us avoid the shame of standing before our Maker, knowing that we have either demeaned, refused to offer a kind word or helping hand, or refused to forgive the sons and daughters of our King: those for whom He gave his very life to save. Instead, with Christ like charity, we can do as Christ, recognizing every interaction we have as an opportunity to minster and nurture, compliment and encourage: leaving others better off than when we first met them.
When love becomes the guiding principle in our care for others, our service to them becomes the gospel in action and not just scriptures we read.
As we strive to become like Christ, we become closer to Christ, and our faith and hope in his promises will become sure.
I have learned firsthand that faith and trust in the Lord is power and peace and the fastest way to increase faith is by personal righteousness. I know as I strive to follow the commandments, I am filled with His spirit and a perfect hope that all will be made right – in this life – or the next – and when we have a perfect hope – the timing of resolution really doesn't matter because WE KNOW everything will be fine. We know we are doing all we can and the rest is in God's omnipotent, loving hands.
It my absolute confidence and trust in the Savior and my great hope of what is to come that leads me to follow his commandments.
If we wish to spiritually live – to be filled with peace and hope, we must choose to follow the Savior every single day. As we sail through life – and sometimes doggy paddle – following the commandments is our life vest. They keep us from sinking and give us peace and protection.
When we were paddle boarding last week, our dog, Kiwi fell in…she can't swim. Thankfully she had a life vest that kept her afloat and kept us from panicking.
Now, imagine if you were in a ship and a storm were to come and you didn't have a life jacket, vs. the peace of being secured in a life preserver and knowing you would float.
The live vest of following the commandments keeps the consequences of sin from drowning our hope and from drowning us – from drowning our opportunities and our blessings. It is when we lose our eternal perspective and give into Satan's ensnaring traps of gossip lying, envying etc. that we are dragged down to suffocating depths of dark water where we cannot see clearly, think clearly or feel the warm rays of hope.
For quite simply, we cannot do wrong and feel right.
The commandments keep us afloat so we can hear the captains voice beckoning to us, encouraging us, guiding us and giving us direction. As we follow the commandments and seek to follow our savior, our lives will not be perfect, but I know we will be better able to traverse any storm that comes our way with faith, peace and a perfect brightness of hope.
Finally, I have learned we can only be as happy as we are grateful.
It can be so easy to focus on what's wrong or on what we don't have, that we no longer appreciate what we do have. This leads us to get discouraged and feel helpless and even hopeless. The ironic truth is that happiness does not come from the accumulation of our wants. It comes from wanting what we have. Until we choose to be grateful for everything we have, accepting there will always be a gap between our real circumstances and our "ideal" circumstances, we will never be content and therefore we will never be happy.
Even when it seems we have lost everything, when everything is going wrong, we still have all that matters. We have an infinitely loving and merciful Savior who suffered for our sins so that we can repent and be made clean again, worthy to live forever with our families in the kingdom of our Father.
Always remembering and being grateful for this exquisite gift makes superficial wants fade away and eternal wants made right when viewed with an eternal perspective.
We always have Christ to be grateful for, we always have the reason to hope.
As we exercise the power of the atonement in our lives, and strive to follow Christ, I know we will be endowed with peace, power and hope. We will have no reason to fear any storms in our path. We will be assured that we are safe in our savior's loving arms and come to know that with the Lord at our side, we will never feel alone or confused, helpless or hopeless, weak or overwhelmed.
Your talents and spirituality are shown everyday on your blog. And I am so thankful to be able to take from it. Thank you for sharing a piece of yourself with all of us. You are beautiful!!
You are such a sweetheart! Thank you so much for your kind words and for actually taking the time to share them with me – made my day! That is so cool you babysat us, I had no idea – I hope we were good for you 🙂 Thank you also SO MUCH for fasting and praying for me for my transplant surgery – I am so humbled and honored by your faith on my behalf – I know it makes a difference!
I hope you and your family are well and continue to enjoy beef and broccoli – glad you like it!
First time visitor to your site – a friend recommended and raved about your one-pot lemon tortellini soup! Looking forward to making soon.
Delving further, I found your story inspirational. My take-away for action is to be exceptionally kind in any circumstance possible. Thank you.
Hi Helen, welcome to my site and thank you so much for reading my story! I love your "take awey for action!" I am sure you are a kind light to all around you!
Jen, thank you for feeding my soul today! What a beautiful talk! Love you dear friend!
Thank you so much Sharlie! Love you too!!!
Thank you for coming to the Escondido Stake this past weekend and delivery your talk on the Perfect Brightness of Hope. I was truly uplifted by your positive spirit and testimony of the Gospel of Jesus Christ in your life! You have set a wonderful example for me and how to handle obstacles and challenges in my own life by having the atonement real.
I was in a terrible car accident in July 19th 2007. I had to have a three level neck fusion and left me now in horrible headaches every day ever since. I have been through all kinds of doctors, Physical therapy, massages, acupuncture and medications and nothing has relieved them. I can't drive more than 20 minutes because of my fused neck and pain. In addition to this, I have a rare disease call endometriosis for 35 years that has caused infertility and damaged to my reproductive organs. Consequently, I had a total hysterectomy and others surgeries because of this. I am facing having another surgery that only one doctor in the United States is qualified to do. The disease has spread to the nerves for my left leg. Atrophy of the calf muscles have set in with shrinking now 1 inch different than the right leg. I can't walk a lot and be on my feet all day. I am scared to do this surgery to remove endometriosis from the nerves. The surgeon is across the United States and it's very expensive to travel and do this surgery. He is not contracted with any health insurance and the risks are high. Thank you for helping me be prayerful in my decisions ahead and handling chronic pain in a different attitude than before. I just think you were my angel for the day in rescuing me from heartache.
I found your blog as a result of your father. He is the new area authority for our Stake here in Antioch, California. In his talk this past weekend, he told the story about your double lung transplant and kidney transplant too. I was just in ahh, of him and the gift that he has of telling stories and relating them to the gospel. So I wanted to know more about him, and found your blog!
What a treasure!!! Not only does it fill my soul with food for thought, but I love to cook and the recipes and the photographs are just outstanding! Thanks again for sharing yourself so amazingly. My life has been touched by your faith and your hope.
Jen- thank you so much for posting this! As I read it I felt like I was receiving answers to some of the questions I have been asking in prayer. Thank you.
My daughter has a serious congenital heart defect. Sometimes I feel strong and full of faith in my challenges – and then sometimes I feel physically and spiritually exhausted – especially as new challenges come and seem to add weight to the current ones that haven't resolved yet (and may not). Like you, I know that these physical challenges will only be for this life. If I just cleave to my Savior, my covenants, and my family then it will all be ok. Somehow, some way, it will all be ok – if not in this life then the next. How thankful I am for our Savior.
I wrote a few of your words on some sticky notes for my fridge ("We are always free to choose faith, trust, and hope in Jesus Christ and in the glorious plan of salvation." As well as, "…We have the power to choose our thoughts, and therefore the power to choose our hope and happiness."). My husband might wonder who the famous "Jen" is when he sees the quotes on the fridge. 🙂 Ha! But I need the reminders – so thank you!
P.S. I love seeing the strength of the relationship between you and your husband. It is beautiful. Thank you.
Ok, apparently I am a little long-winded. But I also just had to say "thank you" for the stories of your family members too. Your parents sound like amazing people, and so does Justin! Reading about how they have faced their challenges with faith has helped to renew my resolve to face challenges with faith and learn to be resilient, smile and laugh more.
And I totally get the giving your kids medications (and feedings) during the night thing. And I laughed out loud at Justin's optimism with his "innie and outie." Ha! 🙂 I hope my toddler can grow up to be so accepting, at peace, and content with her challenges.
I have been asked to speak at my Stake's YW camp on the topic "Perfect Brightness of Hope". I came across this post in my research and felt like it was an answer to prayer. I can empathize with a lot of what you expressed because I have a son who was born missing a portion of his brain and we have had a long road to travel these past 2 years and will continue to travel it for the rest of his mortal life. Some of what you expressed hit so very close to home and has helped me to clarify what I would like to express in my workshop at the camp. I will be quoting you and siting this blog post because I definitely think it is something that everyone can relate to on some level. Thank you for sharing.
Hi Cari, I am so glad you stumbled upon my blog – isn't it amazing how the spirit works?! You sound like an incredibly strong person to have care for such a special soul. I hope you continue to be inspired with your remarks – I have no doubht you will touch the hearts of everyone who hears you! I wish God's choicest blessings upon you and your sweet family! HUGS!
You are amazing. Everyone should read about your journey in life and with Christ. Thank you for being such an inspiration and for giving all the glory to God. It is so true, "In the eye of the storm, He remains in control. God is good all the time, all the time God is good. What a beautiful young woman you are. God Bless.
Hi Jen– I'm a long-time subsciber and was looking through recipes then felt compelled to read some of your personal posts once again. They are so uplifting! Thank you for sharing with the rest of us!


According to the National Institute on Drug Abuse, there has been a 4.3-fold increase in overdose deaths from benzodiazepine abuse in the last 15 years. Benzodiazepines are being abused at higher rates than ever, and we're seeing a national trend of more and more people admitting to addiction treatment centers after their abuse. Because tolerance builds quickly, physical dependence is created easily, and the detoxification process can be deadly, benzodiazepine addiction is dangerous and can be lethal. As with many other prescription medications, people believe they are safe because a doctor prescribed them. However, prolonged use and misuse can lead to a strong addictive cycle that may be hard to break.
Benzodiazepines belong to a class of medication known as tranquilizers, often colloquially called benzos. Benzodiazepines are prescribed to help treat a variety of conditions including anxiety disorders, depression, insomnia, alcohol withdrawal, muscle spasms, and more. You can read more about the prescription information for benzos here. Taken as prescribed, these drugs can be extremely useful. They act on the central nervous system producing a sedated state, relaxed muscles, and decreased anxiety. Because of the effects and side effects produced by these medications, they are highly susceptible to abuse.
There are dozens of different kinds of benzos that are available by prescription. Whenever we talk about psychoactive medication there is both a generic name and a brand name. Here is a list of common benzodiazepines as well as what they are usually described for. Usually, people are more familiar with the brand name rather than the generic drug name so we have included both here for convenience.
No longer legally available in the U.S.
Call us today at (844) 883-3869 to get help!
Benzo medications can be helpful for many people. However, there are a number of side effects associated with the use of this type of medication. When discussing adverse reactions there are generally three types of effects we talk about.
The first is allergic reactions. Like any medication it is possible to have an allergic reaction from benzo medication. They can be as mild as a rash or skin irritation or as severe as anaphylactic shock. Allergic reactions like anaphylactic shock can be extremely dangerous and might lead to death. The second type of adverse reaction is often called "dose related". These are the most common types of side effects. They are known by the drug company and often printed on the prescription insert. Dose related reactions happen when someone is prescribed too high a dose of the medication.
The final type of adverse reactions are drug interactions. Drug interactions occur when the medication you are taking interacts with another medication or drug. When benzos are combined with alcohol, tranquilizers, barbiturates, or narcotics they can cause excessive sedation or memory loss. Drugs that slow the liver down also might interact with benzos. When medications slow down the liver it takes longer for benzos to leave the system. This can lead to high concentrations of benzos in the blood which can increase the risk of other side effects.
Benzodiazepines are intended for short term use. Research indicates that the longer these medications are used for, the higher the risk for dependence and adverse effects. It is important to talk to your doctor if you are taking benzo medication and experiencing any of these symptoms.
Benzodiazepines affect the GABA receptors, like alcohol does. Because of this, withdrawal symptoms may be fatal. The most common benzo withdrawal symptoms are insomnia, anxiety, irritability, sweating, and difficulty concentrating. It's important to know that with benzodiazepines, you can develop serious withdrawal symptoms even if you were taking the medication exactly as prescribed. Withdrawal symptoms have been found to arise in patients taking benzos for only three months. In more extreme cases usually related to abuse, withdrawal may cause hallucinations, seizures, psychosis, mania, delirium tremens, and suicidal or homicidal ideation.
Because these symptoms can potentially be fatal, it's important to find benzo withdrawal help. If you are coming off benzos, it is necessary to do so with doctor approval and medical oversight. It can be necessary to come off benzos in a safe environment like a detox facility.
The benzo withdrawal timeline depends on the drug you were taking. Some benzos are short-acting (like Xanax®), while some are long-acting (like Valium®). Generally speaking, withdrawal effects begin to arise within a few hours after your last dose, with the worst symptoms coming 2-4 days later. The short-acting benzos will cause harsher and quicker withdrawals, while the long-acting ones will often have longer withdrawal periods.
Although the worst is generally over within a week, symptoms of coming off benzos can last for a couple weeks or months. Depending on your body's metabolism, your medical care, and how long you were taking your medication for, withdrawal will vary. Because everyone is different, it's important to seek professional help to stay safe.
When someone is detoxing from benzos they might need to take medication to ease withdrawal symptoms like the ones listed above. One medication that can be used to ease benzo withdrawal is Clonidine, sometimes known by the brand names Catapres® or Kapvay®. Clonidine is not approved by the FDA to treat benzo withdrawal. However, medications can be prescribed by psychiatrists for off-label uses. When a medication is given for an off-label use it means that is is not approved for that use but clinical evidence suggests that it can help.
Clonidine works by reducing blood pressure. A reduction in blood pressure allows the blood vessels to open and the heart to beat more slowly. This decrease in heart rate can help people feel less anxious. Often anxiety is associated with a faster heart rate. So, slowing it down can help reduce anxiety.
Benzo detox must be taken seriously. In order to stay safe during detox from benzos, professional medical care is highly advised. As the symptoms of withdrawal can potentially be fatal, it's imperative that you come off benzodiazepines the right way. Wanting to rid your body of drugs like Xanax®, Valium®, or Klonopin® is a wonderful intention, but it's not worth the risk to attempt the process by yourself at home.
Here at Changing Tides, we have years of experience working with those going through the withdrawal process. We will make sure you are provided with the best care in your detox process, starting you off with a strong and healthy foundation for a lasting recovery. If you wish to get off your benzodiazepines, why not start by detoxing the right way? Don't try to detox at home, or do some do-it-yourself method. Benzo detox can be lethal, and it is not something to be taken lightly. We provide a medical detox that also incorporates a holistic approach so that every part of you will be taken care of.
If you or a loved one is struggling with benzodiazepine dependence or abuse, give us a call today for help! We are standing by and ready to take your call.


text:
All For Mary
Articles & Reviews
In a Swiss Alpine resort shortly after the War an army officer and upper-class Humpy Miller both set their sights on Mary, the landlord's daughter. When the two come down with chicken pox they are put in the charge of fellow guest Miss Cartwright, who turns out to be Humpy's old nanny. The two Englishmen unite not only against her tyranny but against a dense Greek who is also after Mary.
Wendy Toye
Nigel Patrick
Clive Morton
Nanny Cartwright
David Tomlinson
"Humpy" Miller
David Hurst
Monsieur Victor
J Arthur Rank Organization
Gaston Nikopopoulos
Nicholas Phipps
Joan Young
Mrs Hackenfleuger
Maitre D'Hotel
Neil Hallet
Arthur Alcott
Production Controller (Pinewood)
Kay Bannerman
Play As Source Material ("All For Mary")
James Bawden
Peter Blackmore
Harold Brocke
Maurice Carter
Song Performer ("Far Away From Everybody")
Milton Delugg
Song ("Far Away From Everybody")
John S Dennis
Joan Ellacott
Robert Farnon
Song ("All For Mary")
Music; Music Director
Bob Hilliard
Archie Ludski
T. S. Lyndon-haynes
Gordon K. Mccallum
Alan Melville
Additional Dialogue
Norman Newell
Adrian Pryce-jones
Geoffrey Rodway
Paul Soskin
Frederick Wilson
Reginald Wyer
TCM Remembers - Leo McKern
TCM REMEMBERS LEO MCKERN, 1920-2002
The recent death of Leo McKern, 82, marked the passing of one of Britain's finest and most respected character actors. He was suffering from ill health in recent years and was moved to a nursing home a few weeks before his death on July 23 2002 in Bath, England. An actor of commanding presence with a deep-throated voice, the portly, bulbous-nosed McKern had a long, distinguished career spanning more than half a century, earning numerous plaudits along the way in all major mediums: theatre, film and television.
Born Reginald McKern on March 16, 1920 in Sydney, Australia; he served with the Australian Army during World War II and worked in regional theatre in his native Sydney before immigrating to England in 1946. It was a slow start, but after a three-year apprenticeship of painting scenery, stage-managing and acting, McKern eventually joined the celebrated Old Vic theatrical company in 1949 and proved one of the more versatile actors in the troupe tackling diverse roles in comedy, the classics and serious contemporary parts.
His film debut came in Murder in the Cathedral (1952) but it took a few years before he made his mark in cinema. Some of his best film work included roles as Peter Sellers' comic henchman in the classic satire The Mouse That Roared (1959); a bungling train robber in the charming Disney film The Horse Without a Head (1963); a nefarious professor who kills off his colleagues for amusement in the brilliant black comedy A Jolly Bad Fellow (1964); Clang, a cartoonish villain in the Beatles' pop film Help! (1965); Cromwell, the persecutor of Sir Thomas More in A Man for All Seasons (1966) and as Thomas Ryan in the David Lean drama, Ryan's Daughter (1970).
Yet despite all the accolades McKern earned in theatre and films, it was television where he foundinternational fame as the wily, irascible barrister Horace P. Rumpole in John Mortimer's Rumpole of the Bailey in 1975. Infusing the character with beguiling skill and energy, McKern made the acerbic, wine swilling, Tennyson-quoting Rumpole a much loved figure that was adored by critics, audiences and even its creator Mortimer. Perhaps Mortimer offered the most fitting tribute when he once referred to McKern - "His acting exists where I always hope my writing will be: about two feet above the ground, a little larger than life, but always taking off from reality." Enough said.
By Michael T. Toole
KATY JURADO, 1924 - 2002
Katy Jurado, an Oscar nominee and major actress in Westerns, died July 5th at the age of 78. She was born in Guadalajara, Mexico on January 16th 1924 as Maria Cristina Estella Marcela Jurado Garcia, daughter of a cattle rancher and an opera singer. Jurado started to appear in Mexican films in 1943. After 15 films in her native country, director Budd Boetticher saw Jurado attending a bullfight (Jurado wrote about the subject for Mexican newspapers) and cast her in his Bullfighter and the Lady (1952), her Hollywood debut. For much of her career Jurado alternated between the two film industries. In the US, she was memorable for the sensual energy she brought to roles in High Noon (1952), One-Eyed Jacks (1961) which was directed by Marlon Brando, Sam Peckinpah's Pat Garrett and Billy the Kid (1973) and John Huston's Under the Volcano (1984). She was nominated for an Oscar as Best Supporting Actress for Broken Lance (1954). Jurado's Mexican films were in a broader range of genres and included Luis Bunuel's El Bruto (1952), Ismael Rodriguez's We the Poor and Miguel Littin's The Widow Montiel (1979). She won three Ariel Awards (Mexican equivalent to the Oscars) and one special award. She was married to Ernest Borgnine from the end of 1959 to summer 1963. One of her final films was The Hi-Lo Country (1998), a contemporary Western directed by Stephen Frears and co-starring Woody Harrelson, Billy Crudup and Penelope Cruz.
by Lang Thompson
DOLORES GRAY, 1924 - 2002
Broadway and nightclub star Dolores Gray died June 26th at the age of 78. Her movie career was brief but consisted of high-profile MGM musicals which guaranteed her a place in film history. Gray was born in Chicago on June 7th, 1924 (and where, according to a common story, she was accidentally shot by a gangster as a child and had a bullet in her lung her entire life). As a teenager she began singing in California until Rudy Vallee featured her on his radio show. Gray moved to Broadway in 1944 and then to the London stage in 1947, solidifying her reputation as a singer/actress while constantly giving the gossip columnists plenty to write about. She had two small singing roles in Lady for a Night (1941) and Mr. Skeffington (1944) but didn't really light up the big screen until It's Always Fair Weather (1955) even though Gray reportedly didn't much care for the role. Her rendition of "Thanks a Lot, But No Thanks," which has her gunning down a slew of male dancers on-stage and kicking them through trap doors, is a genuine showstopper. Three more unforgettable musical roles quickly followed: Kismet (1955), The Opposite Sex (1956, which Gray turned down Funny Face to do) and Designing Women (1957). That was it for Gray's film career. She kept busy with TV appearances (mostly singing though she did one 1988 episode of the cult show Dr. Who) and a busy recording and nightclub schedule. In 1987, she appeared in a British production of Follies at Stephen Sondheim's request.
TCM REMEMBERS LEO MCKERN, 1920-2002 The recent death of Leo McKern, 82, marked the passing of one of Britain's finest and most respected character actors. He was suffering from ill health in recent years and was moved to a nursing home a few weeks before his death on July 23 2002 in Bath, England. An actor of commanding presence with a deep-throated voice, the portly, bulbous-nosed McKern had a long, distinguished career spanning more than half a century, earning numerous plaudits along the way in all major mediums: theatre, film and television. Born Reginald McKern on March 16, 1920 in Sydney, Australia; he served with the Australian Army during World War II and worked in regional theatre in his native Sydney before immigrating to England in 1946. It was a slow start, but after a three-year apprenticeship of painting scenery, stage-managing and acting, McKern eventually joined the celebrated Old Vic theatrical company in 1949 and proved one of the more versatile actors in the troupe tackling diverse roles in comedy, the classics and serious contemporary parts. His film debut came in Murder in the Cathedral (1952) but it took a few years before he made his mark in cinema. Some of his best film work included roles as Peter Sellers' comic henchman in the classic satire The Mouse That Roared (1959); a bungling train robber in the charming Disney film The Horse Without a Head (1963); a nefarious professor who kills off his colleagues for amusement in the brilliant black comedy A Jolly Bad Fellow (1964); Clang, a cartoonish villain in the Beatles' pop film Help! (1965); Cromwell, the persecutor of Sir Thomas More in A Man for All Seasons (1966) and as Thomas Ryan in the David Lean drama, Ryan's Daughter (1970). Yet despite all the accolades McKern earned in theatre and films, it was television where he foundinternational fame as the wily, irascible barrister Horace P. Rumpole in John Mortimer's Rumpole of the Bailey in 1975. Infusing the character with beguiling skill and energy, McKern made the acerbic, wine swilling, Tennyson-quoting Rumpole a much loved figure that was adored by critics, audiences and even its creator Mortimer. Perhaps Mortimer offered the most fitting tribute when he once referred to McKern - "His acting exists where I always hope my writing will be: about two feet above the ground, a little larger than life, but always taking off from reality." Enough said. By Michael T. Toole


text:
The two faces of transforming growth factor β in carcinogenesis
Anita B. Roberts and Lalage M. Wakefield
PNAS July 22, 2003 100 (15) 8621-8623; https://doi.org/10.1073/pnas.1633291100
Anita B. Roberts
Lalage M. Wakefield
Transforming growth factor β signaling impairs Neu-induced mammary tumorigenesis while promoting pulmonary metastasis
Hero or villain? Trustworthy guardian of normal homeostasis or double agent? Over the past two decades, the perceived role of transforming growth factor β (TGF-β) in carcinogenesis has undergone more plot twists than an Agatha Christie mystery. The initial experiments leading to the discovery of TGF-β and its naming as a "transforming" growth factor were based on its ability to induce malignant behavior of normal fibroblasts, leading to the notion that TGF-β might be a key factor in uncoupling a cell from normal growth control in such a way that it could become tumorigenic (1). However, at the time, this presumed function of the protein was difficult to reconcile with its ubiquitous pattern of expression in normal tissues, including its prevalence in human platelets. The next twist in the story came several years later, when it emerged that TGF-β has profound growth-suppressive effects on many cells, including epithelial cells and lymphoid cells, which form the basis of the majority of human cancers. At this point TGF-β began to be given serious consideration as a candidate tumor suppressor gene (2). Indeed, data from both experimental model systems and studies of human cancers clearly show that not only the ligand itself but also its downstream elements, including its receptors, and its primary cytoplasmic signal transducers, the Smad proteins, are important for suppressing primary tumorigenesis in many organs (3, 4).
While solid credentials were being established for the role of TGF-β as a good citizen in the battle to maintain cellular order, a darker side was emerging. It is now appreciated that metastasis of many different types of tumor cells actually requires TGF-β activity and that, in the context of advanced disease, it actually has prooncogenic effects (3, 5). To date, understanding of this complex, dual role of TGF-β in carcinogenesis has come principally from inference based on the synthesis of studies of the behavior of many different tumor cell lines and many different animal model systems (Fig. 1). But in a recent issue of PNAS Siegel et al. (6) convincingly demonstrated this duality in a single model of mammary cancer that is metastatic to lung, by using bitransgenic mice expressing forms of the Neu oncogene driven by the mouse mammary virus long-terminal repeat (MMTV) and either constitutively activated or dominant negative forms of the TGF-β receptors similarly directed to the mammary gland. This model system is highly relevant to human breast cancer as amplification of erbb2/her2/neu, encoding an epidermal growth factor receptor family tyrosine kinase, is found in >30% of human breast cancers (7) and changes in expression of both the TGF-β ligand and response system occur during cancer progression (8–10). The results of Siegel et al. (6) show that activation of TGF-β signaling delays the appearance of primary mammary tumors, whereas tumors appear earlier in mice in which TGF-β signaling has been compromised, consistent with a tumor suppressor role of the pathway in the earlier stages of tumorigenesis. In contrast, these same manipulations of TGF-β signaling have opposite effects on formation of spontaneous lung metastases in the mice. Mice expressing an activated TGF-β receptor exhibit an increased percentage of metastatic foci that have extravastated from the vasculature, consistent with a prooncogenic effect for TGF-β in late-stage disease.
TGF-β switches from tumor suppressor in the premalignant stages of tumorigenesis to prooncogene at later stages of disease leading to metastasis. Progression to metastatic disease is generally accompanied by decreased or altered TGF-β responsiveness and increased expression or activation of the TGF-β ligand. These perturbations, along with other changes in genetic or epigenetic context of the tumor cell and its stromal environment, combine to alter the spectrum of biological responses to TGF-β.
This dual- or multifunctionality of TGF-β has plagued and confused researchers for years. Indeed, it seems that for every presumed function of TGF-β, an example can be found in which it displays exactly the opposite activity (11). In part, this stems from the fact that, unlike many peptide growth factors that act on a restricted set of target cells, TGF-β is produced by and can act on nearly every cell type, with the consequence that the output of its signaling pathway is both cell type- and context-dependent. Recent advances in understanding molecular aspects of the TGF-β receptor serine/threonine kinases and their interacting partners, as well as the signal transduction pathways and effects on the transcriptome, now offer the potential for understanding the molecular mechanisms contributing to these superficially opposite effects of TGF-β, including especially its transition from tumor suppressor to oncogene during tumorigenesis (3, 12).
The accepted model of the tumor suppressor role of TGF-β in carcinogenesis is that it is critical for maintaining homeostatic control of growth not only in premalignant cells but also in cells progressing through the early stages of carcinogenesis. Central to its growth inhibitory effects, at least in vitro, are its abilities to suppress expression of c-Myc and to enhance expression of the cyclin-dependent kinase inhibitors p15INK4b and p21CIP1 (13). Effects of TGF-β on genomic stability, replicative senescence, and apoptosis also come into play in particular cell types (14, 15). Although less well understood molecularly, effects of TGF-β on tissue architecture and tumor stroma are also important in tumorigenesis (16), and likely contribute to the outcome in human disease and in animal models such as the MMTV-Neu model used by Siegel et al. (6). Although these researchers did show reduced proliferation in primary mammary tumors in which TGF-β signaling had been activated, the contribution of other TGF-β-dependent mechanisms, such as induction of apoptosis, was not explored.
Many cancer researchers are now focused on attempts to understand and control metastasis, because metastases are responsible for most patient deaths. The idea that TGF-β exerts an opposite, prooncogenic role in metastatic disease, and possibly is even required for metastasis in some cells, is not a new one. Analysis of immunohistochemical staining of TGF-β1 in breast cancers showed enhanced expression in lymph node metastases with preferential expression at the advancing edges of the tumors (10). Consistent with this observation, treatment of metastatic breast cancer cell lines with TGF-β in vitro enhanced metastases (17), whereas suppression of TGF-β receptor function markedly reduced their ability to metastasize (18). Indeed, TGF-β may even play a specific role in directing metastatic cells to particular organ sites such as bone, which is a common site of metastatic foci of breast and prostate cancer. For example, studies have shown that TGF-β/Smad and p38 signaling pathways cooperate to promote metastasis of human breast cancer cells to bone by inducing expression of the osteolytic factor, PTH-related protein (PTHrP) (19, 20). Similar to the approach that Siegel et al. (6) have taken to analyze effects of TGF-β on tumorigenesis in MMTV-Neu mice, these researchers showed that expression of a constitutively active TGF-β type I receptor in the breast cancer cells enhanced expression of PTHrP and occurrence of osteolytic bone metastases, whereas expression of a dominant negative TGF-β type II receptor had the opposite effects (19, 20). Together, these studies clearly point to tumor cell autonomous oncogenic effects of TGF-β and its gene targets on metastases.
The mechanisms whereby metastatic tumor cells break off from their primary site and invade the vasculature, travel through the blood stream to a remote site, and eventually colonize and grow in that organ are still being debated (21). Indeed, there is some suggestion that the mechanisms may differ between organ sites or tumor cell types. One theory put forth by Chambers and colleagues (21, 22) suggests that extravasation into the target organ is an efficient step for most cells and that the rate-limiting steps for metastasis are the successful breaking of dormancy to establish micrometastases in the new environment, and the subsequent transition to vascularized macrometastases. In an alternative view, Al-Mehdi, Muschel, and colleagues (23, 24) suggest that for some tumor cells, hematogenous metastases to lung originate from adherence of tumor cells to the vasculature of the target tissue and that the cells proliferate initially within the vessel, before and without the need for extravasation. In this model, the rate-limiting step is escape from apoptosis and, eventually, extravasation after intravascular expansion. Of interest here are the observations of Siegel et al. (6), which show that whereas the total number of lung metastases in bigenic MMTV-Neu-activated TβRI mice did not differ from that found in monogenic MMTV-Neu mice, activation of TGF-β signaling in the tumor cells enhanced the proportion of extravasated metastases. Surprisingly, tumor cells in which signaling was suppressed by expression of a dominant negative receptor showed no difference in either the number of metastatic foci or the proportion of metastases that had extravasated. In contrast, systemic expression of a soluble TGF-β antagonist has been shown to decrease the incidence of metastatic disease in the MMTV-Neu model (25). Together, the two approaches suggest that although the extravasation step may require cell-autonomous action of TGF-β on the tumor cell, as implied by earlier studies (17), there are likely to be additional rate-limiting steps that involve other aspects of metastasis contributed by host stromal cells (Fig. 1).
TGF-β may play a role in directing metastatic cells to particular organ sites.
To take these observations beyond the phenomenological and enable development of new therapeutic approaches, we must now address the mechanisms whereby the TGF-β signaling pathway and/or its gene targets change as cells acquire the capacity to invade and metastasize. It is known that as cells progress toward fully malignant tumor cells, they undergo changes that result in reduced expression of TGF-β receptors, increased expression of TGF-β ligands, and resistance to inhibition of growth by TGF-β (3). Use of small molecule inhibitors of the TGF-β type I receptor kinase has shown that inhibition of growth requires a sustained signal of at least 12–14 h, but that induction of certain immediate early target genes of TGF-β requires only a transient signal (26, 27). Thus, we may find that as signaling flux is reduced in more malignant cells, the set of gene targets of TGF-β will become restricted to those that require a lower signaling intensity or a signal of shorter duration. This can now be approached in systems such as that described by Siegel et al. (6) by gene profiling and analysis of mechanistic correlates.
Another question to be addressed is whether TGF-β utilizes the same or different signaling pathways to mediate its tumor suppressor and oncogenic effects. The predominant pathway for signal transduction from the TGF-β receptor serine/threonine kinases is mediated by cytoplasmic proteins called Smads. In this pathway, Smad2 and Smad3 are phosphorylated directly by the TGF-β type I receptor kinase and after partnering with the common mediator, Smad4, translocate to the nucleus, where they regulate transcription of target genes (12). Although tumor suppressor activity has been attributed to all of the components of this pathway, including TGF-β itself, only in rather rare instances do the genes follow the classic Knudsonian paradigm for a tumor suppressor. For example, the TGF-β ligand shows haplo-insufficiency as a tumor suppressor and the second allele is not deleted in tumorigenesis, but rather is overexpressed as cells acquire more malignant properties (28). Receptors are often transcriptionally repressed in such a way that some function still remains (12, 29). As evidence of the dual role of the TGF-β receptor in tumorigenesis, the prognosis of patients with hereditary nonpolyposis colorectal cancer tumors, where receptors are biallelically lost by microsatellite instability, is more favorable that that of patients with colon cancers, where receptor expression is merely repressed (30). This pattern is reiterated at the level of the Smad proteins, which are mutationally inactivated in only a small subset of cancers, but for which there exists a large variety of epigenetic mechanisms to suppress their activity in carcinogenesis (4). Therefore, the possibility that these mechanisms have evolved to permit the tumor cell to reactivate this pathway as needed in metastasis must be considered. TGF-β also activates mitogen-activated protein kinase pathways, and it has been suggested that a possible imbalance between signaling through these pathways and the Smad pathway or cooperation between these pathways might contribute to the prooncogenic role of TGF-β (3, 31, 32).
We have recently developed a system to examine the dual roles of TGF-β as tumor suppressor and oncogene by using a series of breast cancer cell lines each derived from Ha-ras transformed MCF10A cells, and thus sharing a common origin, but exhibiting distinct features ranging from premalignant to fully invasive and metastatic (33). By manipulating either TGF-β receptor function (B. Tang, M. Vu, T. Booker, S. J. Santner, F. R. Miller, M. R. Anver, and L.M.W., unpublished data) or the activity of the Smad2 and/or Smad3 pathway in these cells (F. Tian, S. Byfield, W. T. Parks, S. Yoo, A. Felici, B., Tang, E. Piek, L.M.W., and A.B.R., unpublished data), we have been able to show that either reduction in TGF-β receptor function or inhibition of the Smad2/3 pathway, as might be found in a tumor cell, enhance the tumorigenesis of xenografted premalignant and well differentiated carcinoma cell lines, but substantially reduce the formation of metastatic foci in the lung after tail vein injection of a metastatic cell line of common origin. Thus, our results demonstrating the dual tumor suppressor/oncogene role of TGF-β in this genetically and biochemically tractable system parallel those of Siegel et al. (6). Moreover, by using this approach, we have been able to demonstrate that the same signaling pathway mediated by Smad2 and/or Smad3 is responsible for both of these activities of TGF-β.
Ultimately, it is hoped that the use of either a mouse model system, such as that described by Siegel et al. (6), or a cell-based system, such as that described above, will provide information about the specific pathways and gene targets that effect this unique switch from tumor suppressor to oncogene. This information is critical for design of new approaches to prevent metastasis as well as for understanding effects of inhibitors of this pathway that might be used to treat other chronic diseases, including fibrotic diseases (26). It is very promising in this regard that two recent studies have shown that an anti-ligand approach based on use of a soluble TGF-β-receptor selectively inhibits metastatic disease in the absence of effects either on normal physiology or on tumorigenesis at the primary site (25, 34). This exciting result adds yet another level of complexity to this TGF-β system by suggesting that the form or accessibility of the ligand is different in primary and metastatic sites. Although TGF-β still remains elusive in terms of our understanding of its multifunctional modes of action, we are moving ever closer to unraveling this mystery at a molecular level and to design of new therapeutic approaches directed toward modulating its activities.
↵* To whom correspondence should be addressed. E-mail: robertsa{at}dce41.nci.nih.gov.
See companion article on page 8430 in issue 14 of volume 100.
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You are going to email the following The two faces of transforming growth factor β in carcinogenesis
Anita B. Roberts, Lalage M. Wakefield
Proceedings of the National Academy of Sciences Jul 2003, 100 (15) 8621-8623; DOI: 10.1073/pnas.1633291100


This week's featured room is truly fabulous. I was absolutely thrilled when longtime reader, Tiffany Richey e-mailed me to share some photos of her newly-completed office, headquarters for Tiffany Richey Design, thinking I would like it. Well, it was certainly right up my alley! Tiffany's office is glamorous with an eclectic spin. I'm sure that your eye, like mine, is initially drawn in by the spectacular Tanzania wallpaper by Thibaut. How could you not feel completely inspired and upbeat in a space swathed in this daring wallcovering? Naturally, I love that Tiffany paired the black and white wallpaper with black furniture and gold accents. I was just telling my friend over lunch the other day that you may as well call me King Midas because I am addicted to all things gold! The gorgeous etageres, along with the high-gloss desk, and classic chair are all from World Away. The Matisse-esque print behind the desk and the table lamp are from IKEA, believe it or not!
After a fabulous week in New York, I am happy to be home and am ready for a low-key weekend. I will have a recap of my adventures in New York next week, so stay tuned! Have a great weekend!
We have that hicks pendent over our kitchen island-love it!
Enter The 30 Day Cleanse Challenge!
Gorgeous room! I absolutely love the black, white and gold.
Stunning who wouldn't want to work in a room like that job well done!
Beautiful! What an amazing work space!
The wallpaper the wallpaper! Too good! Also the Prada art reminds me of the VanderWoodsen loft on Gossip Girl. Amazing space!
I am in love with this office! What a happy, inspiring place to start your day. That wallpaper is so crazy good! Love it and love your blog.
Love all the cool elements in this room!
Small bedroom design is a matter of making every inch of floorspace count. Make good decisions and you'll have a small bedroom that feels light and spacious, get it wrong and everything will seem cramped and awkwardly positioned.
Gorgeous! Love the wallpaper and the desk.
I love the wall covering in the room!! I love the organic feel of the varying size and shape of the dots, making it look like Dalmatian spots! Such a fun black, white, and gold color arrangement. Such a great post!
Brilliant! Definitely a bright, inspiring space to work & be happy. I'm lusting after those book cases and desk chairs.
Oh my gosh, I'm DYING over those walls. I don't just want them, I neeed them!! (dang it, I hate renting!) I absolutely love this room!
A lovely tribute to Meredith Heron's office design which was featured in Chloe Magazine in April 2012 http://chloemagazine.ca/2012/04/canadas-top-interior-designer-meredith-heron/ Amazingly, Meredith even has the same chairs, though not seen in the Chloe feature!
This room was "heavily inspired" by Canadian Designer Meredith Heron.
Looks gorgeous!!! I absolutely love the room she designed. Bravo.
Troppo.... troppo tutto.... preso singolarmente sarebbe tutto carino, ma insieme: TROPPO!
That office is so glamorous! A truly inspiring and relaxing place to work in. Thanks for this post.
Love it or Leave it: Stripes, Stripes, Stripes!


Thank you so much for setting up my first photo shoot. I was such a wonderful model for Mrs. Hope. I barely even cried & did everything that she asked me to do. I even got inside my Duke diaper bag & smiled real big. I looked so cute that Mrs. Hope would not stop saying, "he's so cute, he's just so cute!". I hope you liked these pictures, I tried really hard to put on my best smile for you. I love you!
These are wonderful pictures!!! Go Neita and Vicco and ELI!!! What a beautiful and blessed FAMILY!!!
Ariel - The pictures look AHHH-MAZING! besides that pukey DUKEy! bag you got him in 😉 Let me know when yall want a date night, Ill come over and babysitt!
Erica - The pictures are just Beautiful!
Praise be to God. You all are a beautiful family. Take care of each other always. We are so thankful for you and your gorgeous bundle of "Joy". He is so beautiful. We relly look forward to seeing you all sometime in the new year.


text:
Convicted axe murderer Henri van Breda told girlfriend 'everything'
By Liz Little • 60 Minutes Digital Producer
12:58pm Jul 29, 2018
Melbourne university student Henri van Breda made headlines around the world after his family was brutally murdered in their home in South Africa.
For three years, van Breda pleaded his innocence in the horrific crime, claiming a "tall, black man wearing a balaclava, gloves and dark clothes" broke into the house and began attacking his loved ones.
Liz Hayes' world exclusive story will be broadcast on 60 Minutes tonight on Channel 9, following Australian Ninja Warrior. For more on 60 Minutes, head to the official website.
But last month a South African court sentenced van Breda to life behind bars for the murder of his mother, father and brother, and the attempted murder of his sister.
Judge Siraj Desai described the attacks as "savage and continuous" – but the world was left asking: why would a 20-year-old man want to attack his family and in such a brutal way?
Tonight in a world exclusive, 60 Minutes reporter Liz Hayes is given unprecedented access to the Henri van Breda murder case.
For the first time, van Breda's girlfriend Danielle Janse van Rensburg breaks her silence, explaining to Hayes why she refuses to believe Henri is guilty.
"He told me everything," van Rensburg said.
"He was very open. He was very honest."
Van Breda is also backed by his aunt, Leenta Nell, who tells Hayes she has vowed to support her nephew – regardless of the guilty verdict.
"The Henri that I know – nothing in the 19 years that I knew him led me to think that there's a danger in Henri," she told Hayes.
Detective Sergeant Marlon Appollis, a policeman for 14 years, also speaks to Hayes in tonight's exclusive, revealing the van Breda case was one of the most confronting of his career.
"The moment you walk upstairs you see the blood spatter and stuff. Then you think to yourself, 'What happened here, what went on?'"
Police video footage taken inside the van Breda family home reveals a devastating crime scene which Detective Appollis describes as particularly vicious.
Tonight for the first time, cameras will take viewers inside the crime scene to reveal the extraordinary trail of evidence that ultimately exposed van Breda's guilt.
Despite the overwhelming evidence, van Breda told the court he was able to fight off the axe-wielding murderer and survived almost completely unscathed.
"He was a very lucky man with this axe-wielding attacker," Cape Town prosecutor Susan Galloway said. It was Ms Galloway's job to test the story as told by van Breda.
"One would like to think that there is something wrong with a person who does something like this," Galloway said.
"We want to know that there's something wrong, because otherwise the possibility is there that each one of us are capable of something like this."
This Sunday, in a world exclusive story, 60 Minutes reporter Liz Hayes is given unprecedented access to the Henri van Breda murder file. Picture: AP (AP)
His girlfriend reveals why she believes he is innocent. Picture: 60 Minutes (60 Minutes)
For the first time, van Breda's girlfriend Danielle Janse van Rensburg breaks her silence, explaining to Hayes why she refuses to believe Henri is guilty. Picture: 60 Minutes (60 Minutes)
"He told me everything," van Rensburg said. Picture: 60 Minutes (60 Minutes)
The extraordinary case of Melbourne university student Henri van Breda made headlines around the world after his family was brutally murdered in their home in South Africa. Picture: 60 Minutes (60 Minutes)
Van Breda is also backed by his aunt, Leenta Nell, who tells Hayes she has vowed to support her nephew – regardless of the guilty verdict. Picture: 60 Minutes (60 Minutes)
"We want to know that there's something wrong, because otherwise the possibility is there that each one of us are capable of something like this." Picture: 60 Minutes (60 Minutes)
Liz Hayes' world exclusive story will be broadcast on 60 Minutes this Sunday on Channel 9, following Australian Ninja Warrior. Picture: AP (AP)
Last month a South African court found van Breda guilty of murdering his mother, father and brother, as well as the attempted murder of his sister. Picture: AP (AP)
This video is available on 60 Minutes 9Now and YouTube.
Follow and join the conversation on 60 Minutes Facebook, Twitter and Instagram.
Find additional content on the 9Now 60 Minutes content hub.


Shred your lazy habits & jumpstart your fitness with this 6 week group training session. Workout with a certified personal trainer in a small group setting. Free fit book included. Limited to 8 participants.


text:
July 10, 2009: Alcatraz was a prison almost from the very beginning. In 1859, 11 soliders scheduled for confinement in the sally port basement arrived with the Fort's first permanent garrison. During the Civil War era, soliders convicted of desertion, theft, assault, rape, and murder; citizens accused of treason; and the crew of a Confederate ship were imprisoned here. The army also used Alcatraz as a place of incarceration for Hopi, Apache, and Modoc Indians captured during the various Indian wars of the mid- to late nineteenth century and for military convicts during the Spanish-American War (1898).
During the Great Depression of the 1930s, the newly created Bureau of Prisons became interested in the island as a place for a high-profile, maximum-security facility. Transferred from the War Department to the Department of Justice, Alcatraz reopened in 1934 as a federal penitentiary. Of the 1,545 men who did time on Alcatraz, only a handful were notorious - among them, Al "Scarface" Capone, "Doc" Barker, Alvin "Creepy" Karpis, George "Machine Gun" Kelly, Floyd Hamilton, and Robert Stroud, the "Birdman of Alcatraz" (who actually conducted his famous bird studies when he was imprisoned at Leavenworth.) Most of the inmates were men who had proved to be problems in other prison populations - escape risks and troublemakers.
Of the 14 attempted federal prison-era escapes, the best known occurred in June 1962, when Frank Morris and brothers John and Clarence Anglin slipped into the water. They used raincoats as flotation devices and were presumably bound for San Francisco. Although their bodies were never found, they are assumed to have drowned. In total, 36 prisoners tried to escape the Rock; all but five were recaptured or otherwise accounted for.
Alcatraz - Gardens [D] Alcatraz was barren rock until the late 1860s when the first residents planted gardens in pockets of imported soil. Sent to live on this cold, gray island, Army families took refuge in Victorian-style flower gardens around the Citadel. Prisoners also gardened. Army prison crews, and later, a few closely watched penitentiary inmates, created manicured landscape that bloomed until the prison closed in 1963. The flowering terraces, rose beds, and lawns then grew wild for 40 years. Luckily, hundreds of garden species brought to the island by a century of gardeners managed to survive and even thrive. In 2003, the Garden Conservancy, the Golden Gate National Parks Conservancy, and the National Park Service began a joint effort to preserve and restore the historic gardens.
Alcatraz - Gardens [D]


text:
A Metropol (2008-ig Metro) egy ingyenes, hétköznapokon megjelenő, színes, fővárosi napilap Magyarországon. 2016. június 14-ig az újság a Metro International tagja volt, ezután 4 évre megszűnt.

2020. július 20-án bejelentették, hogy a Közép-Európai Sajtó és Média Alapítványhoz tartozó Mediaworks Hungary Zrt. újraindítja a Metropolt, ami az év szeptember 7-én jelent meg először.

Története

Metro

A lap eredeti neve Metro. Először  1998. szeptember 7-én jelent meg Budapesten. Az első napon 160 000 példány ingyenes napilap került kiosztásra, majd fogyott el órák alatt. A Metro 2001-ben vált országos napilappá, immáron 18 megyeszékhelyen.

Névváltoztatás Metropolra

2003-ban a Metro International DL AG (a Metro áruház tulajdonosa) jelezte, hogy aggályosnak tartja a névegyezőséget, és védjegyoltalmi pert kezdeményezett az újság nevének megváltoztatásáért. Öt év pereskedést követően a lap nevét 2008. augusztus 7-től Metropol névre változtatták. A kiadó nem jelzett gyökeres változást, mivel a lap karaktere nem változott. A lap egyébként nem csak idehaza, más országokban is eltérő névvel jelenik meg: Chilében Publimetro, Dániában Metro Express, Görögországban Metrorama az újság neve.

A magyar kiadás jogát 2011-ben szerezte meg a Simicska Lajos üzlettársának, Fonyó Károlynak vagyonkezelő cége, a Megapolis Zrt. 2012 áprilisában 90 településen csaknem 400 ezer példányt osztottak ki naponta. Megszűnéséig a Metropol Magyarország legnagyobb példányszámú ingyenes napilapja volt. A Népszabadság szerint Fonyó Károly évtizedek óta jó viszonyt ápol a Simicska Lajossal, aki 2015-ös interjúsorozata során összekülönbözött az akkori miniszterelnökkel, Orbán Viktorral. Ezt követően visszaestek a lap reklámbevételei, 2015-ben felénél is kevesebb állami hirdetés jelent meg a lapban, mint 2014-ben, és a Budapesti Közlekedési Központ (BKK) sem hosszabbította meg a lap terjesztési bérleti szerződését. A 2015-ös adózás utáni eredmény 233 milliós veszteség volt.

2016. június 13-án jelentették be, hogy a 14-i (keddi) szám lesz az utolsó megjelenő Metropol. A Népszabadság információi szerint Fonyó Károly üzleti szempontok miatt döntött az újság megszüntetése mellett, mert 2015-ben 233 millió forintos veszteséggel zártak és a 2016-ra készített üzleti tervek egyike sem jósolt nyereséges működést. A terjesztési helyét pályáztatás nélkül a Habony Árpád és Győri Tibor cége, a Modern Media Group (MMG) által kiadott szintén ingyenes Lokál újság kapta meg a BKK-tól.

Újraindulás
2020. július 20-án a KESMA égisze alá tartozó Mediaworks közleményt adott ki, melyben bejelentette, hogy az év szeptemberétől négy év szünet után újraindítják a napilapot. A kiadvány továbbra is ingyenes lesz, a főszerkesztője Simon László, aki 18 éven át dolgozott az eredeti lapnál, azóta pedig a Ripost bulvárlapnál töltött be rovatvezetői pozíciót.

A Válasz Online már 2019-ben úgy értesült, hogy a KESMA fontolgatja a Metropol újraindítását, mert utódját, az erősen kormánypárti Lokált túl silánynak tartják. A Mediaworks akkor még közleményben tagadta ennek valóságtartalmát. Egyes vélemények szerint a Metropolt a közelmúltban indult szintén ingyenes Pesti Hírlap vetélytársának szánják.

Az új lap végül 2020. szeptember 7-én jelent meg először, ami egyben a Lokál végét jelentette. A lokal.hu domain innentől kezdve a metropol.hu-ra irányít, valamint ennek megfelelően a Lokál Facebook-oldala is átalakult.

Metro International

A Metro International ma a világ legnagyobb globális napilapja, amely több mint 10 milliós példányszámával bekerült a Guinness Rekordok Könyvébe. A Metrónak hétköznap 73 napi kiadása, 23 országban, 15 nyelven jelenik meg Európa, Észak- és Dél-Amerika, valamint Ázsia több mint 125 nagyvárosában különböző neveken. Naponta több mint 18 millióan olvassák világszerte. A Metro újság célcsoportját elsősorban a fiatal, tanuló, dolgozó, aktív városi lakosság képviseli. A lap hasonló formátumban megjelenik Brazíliában, Chilében, Csehországban, Dániában, Dél-Koreában, Finnországban, Franciaországban, Görögországban, Hollandiában, Hongkongban, Kanadában, Mexikóban, Olaszországban, Oroszországban, Portugáliában, Svédországban, Guatemalában, Puerto Ricóban, Kolumbiában, Ecuadorban, Peruban és az Egyesült Államokban. (Ugyanakkor a Metro lengyelországi és horvátországi kiadása már megszűnt).

Formátum, célcsoport
Az újság nagysága és terjedelme a tömegközlekedési eszközökön való olvasásra van tervezve. Saját bevallásuk szerint célcsoportjuk a fiatal dolgozó, tanuló, aktív városlakók, akik igényeihez próbálták igazítani a lapot.

Az újraindított lap a közlemény szerint minden hétköznap 24 oldalon megjelenő ingyenes újság továbbra is a városlakók, a közlekedő tömeg lapja lesz, de a szokásos utcai terjesztésen túl az eddigieknél nagyobb elérést biztosítva bevásárlóközpontokban és egyéb hálózatokban is hozzáférhető lesz. A lapot 178 osztóponton terjesztik a fővárosban, saját magukat az "utca hangjaként" definiálják.

Terjesztés
A Metropol korábban országosan több mint 1000 terjesztési ponton volt elérhető, összesen 340-390 ezer példányban. A Metropol Magyarország 37 városában – köztük Budapesten – egyedi terjesztési hálózaton, kihelyezett zöld, illetve fehér tartókban és terjesztőkön keresztül jutott el az olvasókhoz.

A Metropol új verziója csak a fővárosban érhető el.

Jegyzetek

További információk

 A Metro hivatalos weboldala
 A magyarországi Metropol hivatalos weboldala
 A Metro PDF-ben online olvasható kiadásai 23 országból

Ingyenes sajtótermékek
Magyarországi napilapok
Magyar nyelvű sajtótermékek
Napilapok

redpajama_set_name: RedPajamaWikipedia


Welcome to the M&StL Freight Cars Page. Here we will present a collection of images of freight cars of the Minneapolis & St. Louis Railroad. These images will be presented in numerical order of the road number. The purpose is to present information helpful the freight car modeler or historian. Submissions are always welcome.


This recipe is apparently unique to Northern Ireland and uses soup/herb celery which is hard to find outside the area, but regular table celery can be substituted (including the leaves).
Place the beef in a large stock pot, cover with water and stew for 1 - 2 hours.
Allow the stock to cool, skim off any fat.
Add the soup mix to the stock, bring to the boil and simmer for 1 hour.
Roughly chop the leeks (green and white parts), onion, carrots, celery and parsley. Add to the soup. Season well and simmer until the vegetables are soft.
Whole boiled potatoes are traditionally placed in the soup at time of serving.