Attorney Anand Swaminathan stands with Tony Robinson's mother, Andrea Irwin, as he explains details about the lawsuit settlement to reporters outside the Capitol.
Attorney Anand Swaminathan of the Chicago firm of Loevy & Loevy, which represents Robinson's estate, says that the settlement shows that their case against the city was strong.
Interview with writer Steven Potter heard on WORT 89.9 fm.
Mayor Paul Soglin stresses that the decision to settle the lawsuit was made by attorneys hired by the city's insurance carrier.
The mayor says the settlement leaves the public and "all local governments" still struggling to understand how police officers are to proceed in dangerous situations.
"Matt [Kenny] strongly believes that this lawsuit should have gone to trial, and he deeply regrets that he is being deprived of the opportunity to defend himself before a jury of his peers," Palmer says. "While we continue to extend our sympathies to Mr. Robinson's family, they have made some outrageous claims about Matt that will never be resolved. We would have preferred that they demand that the case go to trial, because rather than offer any amount of closure, this settlement only serves to further cast a pall over Matt's devotion and service to the community, and that of the dedicated men and women of the Madison Police Department."
Dan Frei, president of the Madison Professional Police Officers Association, also condemned the settlement in a statement: "The outrageous decision by the city's insurance company to settle this lawsuit over Matt's objection is tantamount to throwing him under the bus. Despite the fact that he and his conduct have been rigorously scrutinized by multiple agencies, the Robinson family has done their best to drag his name through the mud. Frankly, given how the plaintiffs in this case have publicly demanded justice, I'm actually shocked that they would settle it short of having their day in court."
This article has been updated to include statements from Jim Palmer, executive director of the Wisconsin Professional Police Association, Mayor Paul Soglin, and Dan Frei, president of the Madison Professional Police Officers Association.
if a man is attacked, hands, arms, legs, etc. are all considered weapons (this - by the FBI in their homicide statistics. robinson may not have had a gun - but he was armed. so - i don't understand how the officer wasn't defending himself.


text:
Home Funding GoEuro closes...
Germany-Startups
GoEuro closes Series A funding of $27 million led by New Enterprise Associates
Marko Srsan
Multi-mode travel platform GoEuro has announced yesterday that it has successfully raised $27 million in Series A funding, led by New Enterprise Associates (NEA), one of world's largest venture capital firms.
As it is known, GoEuro currently operates in seven countries, Germany, UK, Spain, Italy, Belgium, Netherlands and Luxembourg, providing coverage to 20,510 railway stations, 10,011 bus stations and 207 airports. The company is addressing the complex challenges of the multi-billion dollar European travel market by using technology to build a new platform that normalises massive amounts of rail, bus and air data, resulting in a solution that presents users with all travel options in a clear and concise manner. It gives travellers an option to plan routes from towns and villages, and then compare and combine air, rail and bus travel in a single search.
The new funding will enable GoEuro to significantly increase its rate of growth – with further key European markets to be opened later this year – and reach its goal of being the largest multi-mode travel platform across the Continent. As a further sign of confidence in GoEuro's current progress and future prospects, all three existing investors — Battery Ventures, Hasso Plattner Ventures and Lakestar —have increased their funding in the company.
Naren Shaam, founder and CEO of GoEuro, said: "We are thrilled to welcome NEA to GoEuro, they are a top tier investor with long-term thinking and deep roots in the technology sector. This is a very significant backing of the progress we have made since we launched just over a year ago. It's a clear signal that NEA understands the market opportunity for the entire multi-mode travel industry worldwide, and GoEuro's position as a recognized leader in the segment in Europe. We continue to see rapid increase in all our key metrics in the business and this funding will enable us to execute our growth strategy over the next few years."
On the other side, Forest Baskett, General Partner at NEA, said: "By delivering a beautifully simple solution to a highly complex technology problem, GoEuro is rapidly gaining traction in a market that is ripe for transformation," "Addressing the €200B-plus European travel market is an enormous opportunity, and NEA is thrilled to partner with GoEuro as the company expands its platform across Western Europe and to the international travel community."
Forest Baskett
GoEuro
Naren Shaam
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redpajama_set_name: RedPajamaCommonCrawl


February is all about love. A life without love will always be incomplete - and that starts with your own ability to love yourself.
It's three days before Christmas. No doubt you are making lists to gather the last of what you'll need for holiday gifts, parties, and family get-togethers. Did you forget anything? I thought I hadn't, then I received a JollyVoxBox from influenster with some free samples of goodies that help dot the i's and cross the t's off of your Jolly Holiday list. These products each give you one less thing to stress over as you prep for for family and friends, presents and merry-making.
When someone like Spy Girl throws down the gauntlet to wear purple you know good and well I'm going to take it. After all, it is my signature color.
And don't forget your high-heeled boots. Although these pictures were taken several weeks ago I wanted to use them today, post-Thanksgiving, to talk about some of the things I'm thankful for that may seem a bit trivial to some but mean the world to me.
Celebratiing International Pink Day with a thrift store spree and two trailblazing young women.
Next month I will be celebrating my 52nd birthday. I'm not depressed or upset by it, but I do feel a pressing sense that time is running out for me to accomplish everything I'm planning to achieve before my warranty expires. So, in that vein, I'm going to venture outside of my comfort zone and try a few things I've always been afraid would look ridiculous. There will be successes and there will be failures, and I will be the first one to point them both out.
I'm standing in front of Nashville's iconic Exit Inn night club. The wall behind me is actually a who's who of predominantly rock royalty that have graced the club with their performances. I've never been - they used to be inaccessible - but I plan on trying to check them out soon.
I'm wearing all white (terrifying), sleeveless (mortifying), and wearing florals to boot (mystifying). But I'm being brave about my leukophobia, anthrophobia, and cellulitephobia! Here I am in a white peplum top by Premise. I've always has big arms so I normally steer very clear of sleeveless tops and elasticized waistbands but this one is cut in such a way that it really works for my body type. Here are my white jeans from Chico's making their inaugural appearance. They are very comfy and fit just right. I do think they're still a tad too long and probably need to go back to Ann Rich to be hemmed even shorter. The floral scarf is from Shi. I was visiting my friend Matty there recently and I confessed that I was clueless as to how to wear a scarf. He draped it on me just so and said, "Like that". The beautiful silver multicolored cuff is from Stein Mart. The bandage on my other wrist is from Vanderbilt Orthopaedic Hand Clinic - more on that in the future. I'm reprising my metallic pewter skull clutch, also from Shi, as are the ZigiSoho metallic pewter spiked shoes from several seasons ago.
Linking up for Visible Monday, Monday Mingle and How I Wear My Metallics.


When Republican leaders tried to repeal health care in the spring and summer, many Americans raised the alarm and made a ruckus. We asked hard questions, looked at the independent analyses, held town halls, told our health care stories, and took to the streets.
Because of that overwhelming opposition, plans to slash health care to pay for corporate tax breaks failed. Republican leaders haven't given up. In fact, they've already begun voting on a scheme to slash taxes for corporations and multi-millionaires — paid for by cuts to health care.
Now the plan to raid our health care is buried in the GOP tax scheme and budget process. Here's how they're putting it into place.
On October 5, the House of Representatives passed a budget resolution that cuts $1.5 trillion from Medicaid and other health programs, capping and starving Medicaid.
On top of that, the budget also slices almost $500 billion from Medicare — and proposes turning it into a privatized voucher program and raising the eligibility age to 67.
The Senate budget proposal is just as bad. It would cut Medicaid, Medicare, and the financial assistance people get to buy insurance through the Affordable Care Act exchanges.
For the better part of 30 years, I've been organizing people and communities to win quality, affordable health care for all. These cuts will hurt all of us, especially people who need health care the most: seniors, people with disabilities, children.
The payoff? Our elected representatives get to dole out tax breaks to their big-money donors and corporate friends.
Don't expect House Speaker Paul Ryan, Senate Majority Leader Mitch McConnell, and the others behind this scheme to be honest about it. They're saying their tax cut plan for corporations and multi-millionaires will help ordinary people.
Their idea of who ordinary people are is pretty strange.
The richest 1 percent of Americans will reap 80 percent of the tax cuts, the non-partisan Tax Policy center calculates, while taxes for many moderate and low-income people would go up. The richest 5 percent of Americans will gain income from these tax cuts — but the rest of us will lose income once those cuts are paid for.
We'll wind up with what Republican leaders wanted from health care repeal: tens of millions of people thrown off their health care to clear the way for more than $1 trillion in tax breaks for the mega-rich.
All this comes at a time of soaring corporate profits — with prescription drug corporations continuing to rake in exorbitant profits by price-gouging patients on lifesaving medications.
Instead of requiring Medicare to negotiate lower prices with drug corporations — which would mean huge savings for the public — Republican leaders in Congress want us to pay for corporate tax cuts with our health care.
What part of "no" didn't they understand?


text:
using std::vector;
#include <algorithm>

#include <fst/mutable-fst.h>
#include <fst/rational.h>

namespace fst {

// Computes the concatenation (product) of two FSTs. If FST1
// transduces string x to y with weight a and FST2 transduces string w
// to v with weight b, then their concatenation transduces string xw
// to yv with Times(a, b).
//
// This version modifies its MutableFst argument (in first position).
//
// Complexity:
// - Time: O(V1 + V2 + E2)
// - Space: O(V1 + V2 + E2)
// where Vi = # of states and Ei = # of arcs of the ith FST.
//
template<class Arc>
void Concat(MutableFst<Arc> *fst1, const Fst<Arc> &fst2) {
  typedef typename Arc::StateId StateId;
  typedef typename Arc::Label Label;
  typedef typename Arc::Weight Weight;

  // TODO(riley): restore when voice actions issues fixed
  // Check that the symbol table are compatible
  if (!CompatSymbols(fst1->InputSymbols(), fst2.InputSymbols()) ||
      !CompatSymbols(fst1->OutputSymbols(), fst2.OutputSymbols())) {
    LOG(ERROR) << "Concat: input/output symbol tables of 1st argument "
               << "do not match input/output symbol tables of 2nd argument";
    // fst1->SetProperties(kError, kError);
    // return;
  }

  uint64 props1 = fst1->Properties(kFstProperties, false);
  uint64 props2 = fst2.Properties(kFstProperties, false);

  StateId start1 = fst1->Start();
  if (start1 == kNoStateId) {
    if (props2 & kError) fst1->SetProperties(kError, kError);
    return;
  }

  StateId numstates1 = fst1->NumStates();
  if (fst2.Properties(kExpanded, false))
    fst1->ReserveStates(numstates1 + CountStates(fst2));

  for (StateIterator< Fst<Arc> > siter2(fst2);
       !siter2.Done();
       siter2.Next()) {
    StateId s1 = fst1->AddState();
    StateId s2 = siter2.Value();
    fst1->SetFinal(s1, fst2.Final(s2));
    fst1->ReserveArcs(s1, fst2.NumArcs(s2));
    for (ArcIterator< Fst<Arc> > aiter(fst2, s2);
         !aiter.Done();
         aiter.Next()) {
      Arc arc = aiter.Value();
      arc.nextstate += numstates1;
      fst1->AddArc(s1, arc);
    }
  }

  StateId start2 = fst2.Start();
  for (StateId s1 = 0; s1 < numstates1; ++s1) {
    Weight final = fst1->Final(s1);
    if (final != Weight::Zero()) {
      fst1->SetFinal(s1, Weight::Zero());
      if (start2 != kNoStateId)
        fst1->AddArc(s1, Arc(0, 0, final, start2 + numstates1));
    }
  }
  if (start2 != kNoStateId)
    fst1->SetProperties(ConcatProperties(props1, props2), kFstProperties);
}

// Computes the concatentation of two FSTs.  This version modifies its
// MutableFst argument (in second position).
//
// Complexity:
// - Time: O(V1 + E1)
// - Space: O(V1 + E1)
// where Vi = # of states and Ei = # of arcs of the ith FST.
//
template<class Arc>
void Concat(const Fst<Arc> &fst1, MutableFst<Arc> *fst2) {
  typedef typename Arc::StateId StateId;
  typedef typename Arc::Label Label;
  typedef typename Arc::Weight Weight;

  // Check that the symbol table are compatible
  if (!CompatSymbols(fst1.InputSymbols(), fst2->InputSymbols()) ||
      !CompatSymbols(fst1.OutputSymbols(), fst2->OutputSymbols())) {
    LOG(ERROR) << "Concat: input/output symbol tables of 1st argument "
               << "do not match input/output symbol tables of 2nd argument";
    // fst2->SetProperties(kError, kError);
    // return;
  }

  uint64 props1 = fst1.Properties(kFstProperties, false);
  uint64 props2 = fst2->Properties(kFstProperties, false);

  StateId start2 = fst2->Start();
  if (start2 == kNoStateId) {
    if (props1 & kError) fst2->SetProperties(kError, kError);
    return;
  }

  StateId numstates2 = fst2->NumStates();
  if (fst1.Properties(kExpanded, false))
    fst2->ReserveStates(numstates2 + CountStates(fst1));

  for (StateIterator< Fst<Arc> > siter(fst1);
       !siter.Done();
       siter.Next()) {
    StateId s1 = siter.Value();
    StateId s2 = fst2->AddState();
    Weight final = fst1.Final(s1);
    fst2->ReserveArcs(s2, fst1.NumArcs(s1) + (final != Weight::Zero() ? 1 : 0));
    if (final != Weight::Zero())
      fst2->AddArc(s2, Arc(0, 0, final, start2));
    for (ArcIterator< Fst<Arc> > aiter(fst1, s1);
         !aiter.Done();
         aiter.Next()) {
      Arc arc = aiter.Value();
      arc.nextstate += numstates2;
      fst2->AddArc(s2, arc);
    }
  }
  StateId start1 = fst1.Start();
  fst2->SetStart(start1 == kNoStateId ? fst2->AddState() : start1 + numstates2);
  if (start1 != kNoStateId)
    fst2->SetProperties(ConcatProperties(props1, props2), kFstProperties);
}

// Computes the concatentation of two FSTs. This version modifies its
// RationalFst input (in first position).
template<class Arc>
void Concat(RationalFst<Arc> *fst1, const Fst<Arc> &fst2) {
  fst1->GetImpl()->AddConcat(fst2, true);
}

// Computes the concatentation of two FSTs. This version modifies its
// RationalFst input (in second position).
template<class Arc>
void Concat(const Fst<Arc> &fst1, RationalFst<Arc> *fst2) {
  fst2->GetImpl()->AddConcat(fst1, false);
}

typedef RationalFstOptions ConcatFstOptions;

// Computes the concatenation (product) of two FSTs; this version is a
// delayed Fst. If FST1 transduces string x to y with weight a and FST2
// transduces string w to v with weight b, then their concatenation
// transduces string xw to yv with Times(a, b).
//
// Complexity:
// - Time: O(v1 + e1 + v2 + e2),
// - Space: O(v1 + v2)
// where vi = # of states visited and ei = # of arcs visited of the
// ith FST. Constant time and space to visit an input state or arc is
// assumed and exclusive of caching.
template <class A>
class ConcatFst : public RationalFst<A> {
 public:
  using ImplToFst< RationalFstImpl<A> >::GetImpl;

  typedef A Arc;
  typedef typename A::Weight Weight;
  typedef typename A::StateId StateId;

  ConcatFst(const Fst<A> &fst1, const Fst<A> &fst2) {
    GetImpl()->InitConcat(fst1, fst2);
  }

  ConcatFst(const Fst<A> &fst1, const Fst<A> &fst2,
            const ConcatFstOptions &opts) : RationalFst<A>(opts) {
    GetImpl()->InitConcat(fst1, fst2);
  }

  // See Fst<>::Copy() for doc.
  ConcatFst(const ConcatFst<A> &fst, bool safe = false)
      : RationalFst<A>(fst, safe) {}

  // Get a copy of this ConcatFst. See Fst<>::Copy() for further doc.
  virtual ConcatFst<A> *Copy(bool safe = false) const {
    return new ConcatFst<A>(*this, safe);
  }
};

// Specialization for ConcatFst.
template <class A>
class StateIterator< ConcatFst<A> > : public StateIterator< RationalFst<A> > {
 public:
  explicit StateIterator(const ConcatFst<A> &fst)
      : StateIterator< RationalFst<A> >(fst) {}
};

// Specialization for ConcatFst.
template <class A>
class ArcIterator< ConcatFst<A> > : public ArcIterator< RationalFst<A> > {
 public:
  typedef typename A::StateId StateId;

  ArcIterator(const ConcatFst<A> &fst, StateId s)
      : ArcIterator< RationalFst<A> >(fst, s) {}
};

// Useful alias when using StdArc.
typedef ConcatFst<StdArc> StdConcatFst;

}  // namespace fst

#endif  // FST_LIB_CONCAT_H__

redpajama_set_name: RedPajamaGithub


text:
Joaquin Phoenix to Play Napoleon in New Ridley Scott Film Kitbag
Described as an "original and personal" film about Napoleon's story.
Joaquin Phoenix and Rooney Mara Name First Child After River Phoenix
The couple recently welcomed a baby boy to the world.
on September 28, 2020, 8:51am
Warner Bros. Offering Joaquin Phoenix $50 Million for Two Joker Sequels: Report
The Oscar-winning actor may return to Gotham City.
on September 15, 2020, 10:52pm
Joaquin Phoenix and Rooney Mara Expecting Their First Child Together
The pair originally met on the set of Spike Jonze's Her.
Joaquin Phoenix Rescues Cow and Calf from LA Slaughterhouse: Watch
The Joker actor named the bovines Liberty and Indigo, respectively.
Oscars 2020 Winners List: Parasite Conquers the Academy Awards
Bong Joon-ho's comedy thriller changes the game for South Korea and international filmmaking.
on February 09, 2020, 11:45pm
Oscars 2020: Joaquin Phoenix wins Best Actor for Joker
This is the second time Gotham's Clown Prince has won the gold.
2020 Oscar nominations revealed: See the full list
Joker, Once Upon a Time in Hollywood, 1917, and The Irishman lead this year's contenders.
on February 08, 2020, 11:15am
Oscars 2020 Predictions: Who Will Win, Who Should Win
A complete assessment of this year's slate of nominees.
by Dominick Suzanne-Mayer and Blake Goble
A History of the Sympathy Oscar (and How The Academy Can Stop Saying Sorry)
Every time someone wins one, someone else gets snubbed.
by Carrie Wittmer
Joaquin Phoenix criticizes the 2020 BAFTAs: We have to "truly understand systemic racism"
The actor addressed #BAFTAsSoWhite during his Best Actor acceptance speech.
Joaquin Phoenix honors Heath Ledger with SAG acceptance speech
Phoenix said, "I'm standing here on the shoulders of my favorite actor Heath Ledger."


Interesting. Personally, I have always felt the City Slicker as a whole was a low point for JY.
I really liked the City Slicker song and the entire album. Raised by Wolves was even better but that was more of a Styx sounding album.
yogi wrote: I really liked the City Slicker song and the entire album. Raised by Wolves was even better but that was more of a Styx sounding album.
For me, I thought the production was lacking. To me it sounded amateurish. Those awful drum machines, thank you Jan Hammer. Don't get me wrong, I liked his Miami Vice soundtrack stuff. But I just thought he was not the best choice for JY to collaborate with on his first solo outing. Remember JY likes to fancy himself as "cut from the cloth of Hendrix". I also thought that the majority of the songs were weak. If I remember correctly, some of them were songs rejected for Styx to do.


PATERSON, NJ – As the City Council reviewed the budget of the Bunker Hill Industrial Park Special Improvement District during its workshop meeting Tuesday night, one official had a question. Councilman William McKoy wanted to know who the district's executive director was.
McKoy did a double-take. "The ex-mayor?'' he asked. Yes, the ex-mayor.
As it turns out, Jose "Joey" Torres has been the Bunker Hill district's chief executive for more than two years, taking the $36,000 part-time position months after he lost the top job at City Hall. But the disclosure of that fact caught McKoy and a few other folks in the Council Chambers Tuesday night by surprise.
"How's he the director when he's working all the way down in South Jersey?'' McKoy asked, referring to Torres' full-time $135,000 job as business administrator in Jackson Township in Ocean County.
"I work the hours, but they're not steady hours,'' Torres said in an interview with PatersonPress.com on Thursday. Once a month, Torres said he attends the Bunker Hill district's board meetings. Other than that, he said he works a makeshift schedule of an average of 20 hours per week for the board, administering and monitoring the various contracts issued by the improvement district for landscaping, security and other services.
The Bunker Hill district is a private group of about 100 businesses in the 4th Ward that assess themselves an extra tax they use to make improvements within their area. Under state law, the city council must approve the district's budget every year because it generates its revenue through the tax collections.
Paterson has one other Special Improvement District – the one that covers the downtown business area.
Bunker Hill district's previous executive director had been Jamie Dykes, the director of the Greater Paterson Chamber of Commerce. Dykes said he resigned from the position in 2010 because he no longer had the time to handle the Bunker Hill job along with his other duties. Dykes' final salary at Bunker Hill was similar to what Torres is being paid, officials said. Torres was picked for the position months after Dykes left, officials said.
Torres was the most qualified among several applicants for the position, according to David Hertz, co-chairman of the Bunker Hill board. So far, Torres has handled the job well and not been sidetracked by his full-time job in Jackson, Hertz said.
Torres said he is proud of his work at Bunker Hill, including the cleanup of the old NJ Transit rail station in the area. "I think if you drive through the district, you'll see what I mean,'' Torres said.
The council is scheduled to take a preliminary vote on the Bunker Hill district's $201,000 budget for 2013 at its meeting on January 22. After that, the council will hold a public hearing and final vote on the budget, neither of which has been scheduled yet.
Torres has said he is mulling another run for mayor in 2012. If he won, he said he would step down from the Bunker Hill position. "I couldn't do both,'' the former mayor said.


text:
<?php

namespace PHPagstractTest\Token\Tokens;

use PHPagstract\Token\Tokens\Comment;

class CommentTest extends \PHPUnit_Framework_TestCase
{
    public function setUp()
    {
        // clean-up: re-init default tokenizer setup
        new \PHPagstract\Token\MarkupTokenizer();;
    }

    /**
     * @dataProvider parseDataProvider
     */
    public function testParse($html, $expectedValue, $expectedRemainingHtml)
    {
        $comment = new Comment();
        $remainingHtml = $comment->parse($html);
        $this->assertEquals($expectedValue, $comment->getValue());
        $this->assertEquals($expectedRemainingHtml, $remainingHtml);
    }

    public function parseDataProvider()
    {
        return array(
            'simple' => array(
                '<!-- asdf -->',
                'asdf',
                ''
            ),
            'with element' => array(
                '<!-- asdf --><whoa />',
                'asdf',
                '<whoa />'
            ),
            'with whitespace' => array(
                '   <!--     asdf      -->    <whoa />',
                'asdf',
                '    <whoa />'
            ),
            'no whitespace' => array(
                '<!--asdf-->yo',
                'asdf',
                'yo'
            ),
            'two comments' => array(
                "<!-- asdf -->\n\n<!-- asdf -->",
                'asdf',
                "\n\n<!-- asdf -->"
            ),
            'parse error' => array(
                '<!-- asdf',
                null,
                ''
            )
        );
    }

    /**
     * @expectedException PHPagstract\Token\Exception\TokenizerException
     */
    public function testExceptionInParse()
    {
        $comment = new Comment(null, true);
        $comment->parse('<!-- asdf');
    }

    /**
     * @dataProvider toArrayDataProvider
     */
    public function testToArray($html, $expectedArray)
    {
        $comment = new Comment();
        $comment->parse($html);
        $this->assertEquals($expectedArray, $comment->toArray());
    }

    public function toArrayDataProvider()
    {
        return array(
            'simple' => array(
                '<!-- asdf -->',
                array(
                    'type' => 'comment',
                    'value' => 'asdf',
                    'line' => 0,
                    'position' => 0
                )
            )
        );
    }
}


If you are in doubt as the British automotive industry – here Evoque German production. Of course, we joke, no this is not the Evoque, and Volkswagen CrossBlue Coupe, which has all chances to become the prototype of a new generation of the Tiguan .
For the first time about the coupe Volkswagen CrossBlue we learned only yesterday, after the publication of design sketches of the model. Today, at our disposal we were already high-quality press photos, which depict the car, waiting for his imminent debut at the VW stand in Shanghai.
In general, the Shanghai Motor Show will be a real mecca for conceptual crossovers – the BMW the X4 , the Mercedes of GLA and the Citroen Wild Rubis the DS , as well as the future, "Tiguan" we will see tomorrow.
Judging by the fact that Volkswagen is not usually produces unpromising concept cars – CrossBlue Coupe will soon go into production and will be based on the MQB platform.
The prototype was a hybrid installation with a V6 TSI engine and two electric motors that will consume only 3 liters of fuel per 100 kilometers, and the trip distance on one electric will be 33 kilometers.
Exterior CrossBlue Coupe was more angular than we expected the VW, and the headlights are two large LED spotlights.
Look closely at the interior photos, because it may be that it will look like the next interior the Touareg .


Soaring rates of suspensions and expulsions are not the answer, says academic.
The number of students being suspended or expelled from Australian schools is "skyrocketing", according to news reports. These note a 10% increase in suspensions over two years at NSW primary schools and that students in south-western Sydney are being suspended more than four times as often as students in other parts of the city.
Suspension and expulsion is widely used in Australia, the UK and the US to respond to problematic behaviour. But evidence shows these tactics aren't effective in changing a student's conduct, and carry major long-term risks for their welfare. Students most affected tend to be those with higher and more complex needs, such as those with disabilities and mental health issues.
Instead of punishing young people, parents should be encouraged to work with schools to improve their child's behaviour, and governments should install programs to tackle teacher stress. In the case of some schools, this might mean honestly tackling the quality of teaching and leadership, as well as teacher attitudes to students with disabilities.
Suspension refers to when a student is sent home from school after a serious incident (such as physical assault on a teacher) while a decision is being made about how to respond. Exclusion (also called expulsion in NSW and Victoria) is either for a set period or permanently. It can mean a decision has been taken by the principal or school leadership that the student cannot attend the school as a result of their behaviour.
Providing education for children is the legal responsibility of state governments. A decision to exclude a student means the child either attends an alternative education provider or is home-schooled, depending on family circumstances and judgements of the professionals (typically the principal or senior staff team) involved.
A 2017 Victorian Ombudsman report noted school expulsion was an escalating educational issue. It concluded many schools were not equipped with the resources, expertise and assistance to provide support to children with higher needs.
Research shows students who are expelled have a higher future risk of engaging in criminal and anti-social behaviour, or consuming drugs. Excluded young people also have lower odds of a stable, happy and productive adult life.
Suspensions and exclusions also raise serious ethical questions about how our school system accommodates disadvantaged students. Vulnerable ("at risk") students, such as those with disabilities or mental health difficulties, are disproportionately represented in school exclusions and school suspensions.
A recent SA report, for example, explicitly recommended schools avoid using exclusion or suspension as a default behaviour management strategy for students with disabilities and challenging behaviours.
Many evidence-based strategies are available to help improve student (and teacher) behaviours in schools. These can be highly effective if teachers know how to use them and have the confidence to do so. Children with higher behavioural needs may need to receive more intensive, specialist intervention, focused on developing appropriate communication, social skills and emotional regulation.
Teachers can also be helped to gather and record data about when and where problem behaviours occur. This can then be used to gauge what may be driving the behaviour.
For example, George wants attention because the academic work set by the teacher is too difficult for him and his disruptive behaviour is a call for help. Annoying the teacher, and being excluded from class, allows a demotivated Carli to escape from the tedium of worksheets. Andy is irritable and prone to aggressive behaviour on Monday morning because he stays at his dad's on a Sunday night, which unsettles him emotionally and constantly reminds him of his parents' separation.
Insights from this structured process of data gathering can then enable teachers to help the student learn to modify their behaviour. This includes teaching appropriate behaviours to replace negative ones.
In some cases, teachers and schools, as well as students, need to change their attitudes. Several Australian studies have indicated that unhelpful attitudes by teachers, by parents and school leaders toward "at risk" students (students with disabilities, poor mental health or from disadvantaged backgrounds) can spur behaviours that lead to exclusion or self-exclusion.
My own research shows reducing educational exclusions is an opportunity for changing attitudes toward disability and mental health in schools. Changes in teacher attitudes towards at-risk children are fundamental for positive change in behaviours by teachers and in favour of inclusion.
Teaching is widely regarded as extremely stressful. While the reasons for this are complex, several studies suggest poor classroom behaviour is an important risk factor.
Emerging knowledge about the relationships between teacher stress and student behaviour suggests it goes two ways. It's known as a "burnout cascade", where unwell and poorly coping teachers resort to punitive and ineffective responses. These rapidly escalate incidents and trigger further feelings of inadequacy in a teacher.
Other research suggests stressed teachers are more disconnected from their students. They often fail to notice their needs, which can trigger poor student behaviours. And many schools, and their teachers, just don't have the skills or knowledge necessary to meet the unique learning and behavioural needs of students with disabilities.
So, future national initiatives designed to reduce teacher occupational stress are likely to reduce unnecessary suspensions and exclusions. The flow on benefits of such initiatives for vulnerable students and for our hard-working teachers could be enormous.
Previous A school trip to Canberra includes 23 cultural institutions and attractions.


Credit & big thanks for this review go to Donna Westcott, a long-time friend from church who lives in Mechanicsville. I only recently learned about the Habitat stores and can't wait to check them out!
Two of my favorite places to hunt for bargains other than clothing are the two stores run by Habitat for Humanity in this area. When you donate to or buy from Habitat, you are helping support an organization which truly helps people to help themselves achieve ownership of safe, decent housing. In addition to individual donations, contractors donate both materials they have torn out as well as their excess supplies, and hotel chains donate furniture they are phasing out.
This store is a real treasure hunt! It has some furniture out front, a mix of used items and new (scratch and dent) donated items. But the adventure begins when you enter the warehouse area, jam-packed with all manner of items yanked out of houses dating from the 1920s to the 1980s, to judge from the look of things. This includes many, many of the following type items: kitchen cabinets; counter tops with and without sinks; appliances; toilets; vanities with sinks; bathtubs; windows; doors, both interior and exterior; old radiators; light fixtures; electrical supplies; plumbing supplies and fixtures. There are buckets of nails, including those for nail guns; dozens, if not hundreds, of partial cans of paint, stain, adhesive; bags of cement; tubes of caulk. They often have those fiberglass columns and bases, and today there was one that had already been marbleized. I don't get to this one as much because the Mechanicsville store is only a few minutes from my house. But some fun things I have gotten there in the past are a dozen or two crystal cabinet knobs, and some old-school, army green desk files and rubber stamp holders.
My bargain picks of the day for today: a large mirror—about maybe 3×6, the kind you might just lean against a wall, with beveled edges and a wide plastic frame which really looks like dark would with gold beading. It was $65.
Also, a couple hundred packages of long, shiny, glow-in-the-dark false eyelashes in assorted colors. Only 25 cents a pair.
This is where I have spent more time and found more of my bargains, since it is only a few minutes from my house. This store is smaller and features much more furniture than the Richmond store, but not as much hardware. It does have appliances, doors and windows, kitchen cabinets, lighting, carpets, flooring, bathroom fixtures, and a few hardware and plumbing supplies. But it also always has a selection of sofas and chairs, dining room sets, bedroom dressers, other cabinets, sewing machines, and many practical and decorative household items.
From this store I have purchased enough of the really nice birch and melamine pull-out drawers for all of my kitchen cabinets for a few dollars a piece (we had to purchase the runners to install them); matching lamps for my living room; and—best of all—a complete set of under-the-bed drawers (not the kind you slide under the bed, but the kind that forms the base for your mattress) for $10! That case was an anomaly. I went by one morning and saw them. They were not priced, so I asked the nice lady how much they were. She said, "Oh, someone left it on the sidewalk last night, and I think there are missing parts, so you can have it for $10. Nothing was missing that we could tell, and the bed hasn't fallen in yet.
I could not find a brand name on the cabinets that the Homestore carries; apparently they are custom made. But the promotional material claims that they are all wood and cost 50% less than "big box store" prices. There are a couple of sets of cabinets set up in the store, and indeed they are all wood, nice and thick on the exterior pieces, lovely birch on the drawer sides, and very sturdy and smooth-working hardware. It got me thinking about my old 1958 kitchen.
Some new items I noticed today: quite a few brand new stainless steel kitchen sinks. One had a large and deep bowl on one side, and a smaller bowl with disposal mount on the other side- $150.
I saw a sign in the Homestore that announced that on the last Saturday of every month, there will be many items heavily discounted.


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Home Posts Tagged "Election 2019"
Op/Ed: Time for a new culture in the District Attorney's Office
Oct 29th, 2019 · 0 Comment
By Joseph W. Carroll, Former District Attorney, Chester County Joe Carroll (left) and Deb Ryan On November 5th Chester County voters will elect a District Attorney. I know from having held that office for 10 years the power and responsibility that goes with the position. As the chief law enforcement officer of the county, the DA can help keep our citizens safe and bring comfort to the victims of...
Setting the record straight on blame for the pipeline
Editor's Note: Corrected the source of the mailers, which is Conservation Voters of PA By Mike McGann, Editor, The Times A popular trope on the Internet is "vote blue, no matter who." As we've seen with the likes of Tulsi Gabbard (not to mention so many GOP examples it is impossible to list them all), blind party loyalty is a massive disservice to our country — and how too many elected...
Op/Ed: Preserve law and order in Chester County with Michael Noone
By Christopher P. Gerber, Esq., Special To The Times Christopher P. Gerber The election of Chester County's next District Attorney will be upon us in less than a month. Voters will have to decide which candidate is best qualified to lead the county's chief law enforcement agency that is charged with the vital responsibility of keeping the public safe from dangerous criminals and the proliferation...
Op/Ed: The importance of a good deed
Oct 1st, 2019 · 0 Comment
By Chris Pielli, Special to The Times Chris Pielli Why do people run for political office? Ideally, it should be to serve the public and put the public's interest above all other interests, including your own. I think of Sophocles' famous quote –"To be doing good deeds is man's most glorious task." I have always tried to keep this in mind. As a veteran, I have served my country....
D.C. pols may have big impact on local 2019 election
By Mike McGann, Editor, The Times Today at 4 p.m., in Washington, D.C., there will be a meeting that may well decide the fate of the county elections in Chester County — not to mention thousands of similar elections nationwide. The Democratic House Caucus will meet this afternoon to decide whether of not to move for a formal Impeachment Inquiry of Donald Trump. How that meeting goes will have a giant...
Ryan, Noone tout law enforcement endorsements
Sep 23rd, 2019 · 0 Comment
Deb Ryan While Democratic District Attorney Candidate Deb Ryan hailed a first-ever endorsement of a Democrat by Chester County Fraternal Order of Police, Lodge 11 late last week, the Republican in the race, Mike Noone, sought to do damage control by pointing out his own law enforcement endorsements from the likes of Chester County Police Chiefs Association, the Phoenixville Police Association, the...
Hogan withdraws from County DA race
Jul 3rd, 2019 · 0 Comment
Tom Hogan Tom Hogan withdrew from the race for Chester County District Attorney, Wednesday. Hogan, once seen as a potential gubernatorial or U.S. Attorney candidate in some political circles, cited family needs as the key factor behind his decision, after serving two terms in office after winning election in 2011. Hogan moved to endorse First Assistant District Attorney Michael Noone as his replacement...
Longo announces run for Register of Wills
Feb 14th, 2019 · 0 Comment
Lisa Longo PHOENIXVILLE — Lisa Longo, a former Phoenixville Board of Education President and current board member announced Tuesday that she will seek the Democratic nomination for Register of Wills/Clerk of the Orphans Court for Chester County. Longo said her decision to run stems from a desire to do even more for the community, and that she has strong reasons to run for the office. ...


Articles showcasing card motif products, card novelties, card supplies, and card accessories.
No bridge or card room is complete without a proper table. It's more than a functional piece – it's a functional centerpiece and sets the tone of the entire room around it. You've got to think about beauty, practicality, comfort and space all at the same time. Alex shares some of the coolest tables money can buy.


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Life and death as a T lymphocyte: from immune protection to HIV pathogenesis
Nienke Vrisekoop1,
Judith N Mandl1 &
Ronald N Germain1
Detailed analysis of T cell dynamics in humans is challenging and mouse models can be important tools for characterizing T cell dynamic processes. In a paper just published in Journal of Biology, Marques et al. suggest that a mouse model with its activated CD4+ T cells are deleted has relevance for HIV infection.
See research article http://jbiol.com/content/8/10/93
T lymphocytes have a difficult existence. As mature cells, they are essential for immunity to infection, but in the early stages of their development in the thymus, more than 90% of them fail selection for the appropriate antigen receptors and die before export to the peripheral immune system. Those that achieve maturity spend weeks, months or even years circulating through the body, in constant search of a foreign antigen that their antigen-specific receptor can recognize, and needing continuously to compete for trophic signals necessary for their survival. Most fail to find an antigenic match and remain as small resting cells until death. A few encounter the right partner and undergo a transient bout of exponential clonal expansion, only for more than 90% of these progeny to be lost by apoptosis shortly after the antigen is cleared. The remaining 10% are maintained as memory cells (Figure 1), conferring lasting protection.
Schematic diagram of T cell dynamics in normal mice. The width of the arrows denotes the rate of death and division or of transit from one pool to another. Naïve T cells are T cells that have matured and left the thymus where they are generated, but have not yet encountered antigen. RTEs, recent thymic emigrants; Ag, antigen.
In normal individuals the T cell population shows excellent homeostatic control, with stable numbers for decades in adult humans, except for intermittent bursts of expansion during infection. Moreover, this homeostasis applies not only to the total number of T cells but to the proportions of these cells in the two major functional subsets of T lymphocytes - CD8+ T cells, whose principal function is to kill virus-infected cells, and CD4+ T cells, which are critical for activating other immune cells, including CD8+ T cells and the B cells that that secrete antibodies. Understanding what happens to the performance of the immune system when this balance is disturbed is of both fundamental interest and clinical relevance. In perhaps one of the most relevant examples, HIV-infected individuals lose their CD4+ T cells, a loss that results in acquired immunodeficiency syndrome (AIDS) and death from secondary infections if the original infection is untreated.
In this issue of Journal of Biology, Marques et al. [1] present a novel genetic approach to eliminating CD4+ T cells that the authors present as a mouse model of HIV-induced CD4+ T cell death. Marques et al. [1] marked activated CD4+ T cells for elimination in mice through genetic manipulation of a protein known as OX40 (TNFRSF4), which is expressed by almost all antigen-stimulated (and thus activated) CD4+ T cells [2]. Their strategy was to construct a mouse in which the Tnfrsf4 gene encoding OX40 drives expression of Cre recombinase. This enzyme, in turn, mediates the activation of a gene encoding diphtheria toxin A fragment (DTA), whose expression results in the death of the activated CD4+ T cell within 48 hours of induction of OX40 expression. In these mice (referred to from here on as OX40-DTA mice), about 55% of memory CD4+ T cells are always in the process of being deleted, whereas other immune cell populations, including naïve circulating CD4+ T cells, remain largely intact in overall number, although they show an increased rate of conversion to an activated state. The loss of activated CD4+ T cells is accompanied by partial immunodeficiency, which was seen following infection with Friend virus, influenza A virus and Pneumocystis murina. In these several respects, the model phenocopies some of the pathology of HIV infection, and it would be tempting, therefore, to conclude that these data show that direct cytopathicity of HIV for activated CD4+ T cells is the driving force in the T cell loss and resulting immunodeficiency in AIDS patients.
However, there is a complication. A subpopulation of CD4+ T cells called T regulatory cells (Tregs) that suppresses immune activation and is thought to be important in preventing autoimmunity is specifically targeted in the OX40-DTA mouse. (The different subsets of CD4+ cells are illustrated in Figure 2). Most Tregs constitutively express OX40 [3], and Marques et al. [1] find that over 80% of these T cells are also lost in the OX40-DTA mice. Their data suggest that the loss of these regulatory and/or activated CD4+ T cells results in substantial changes in T cell dynamics and induces chronic immune activation, which is a key characteristic of HIV infection. This raises interesting questions about both lymphocyte dynamics in the absence of antigen and the possible relevance of these results [1] to HIV infection.
The major subsets of CD4+ T cells that differentiate from naïve circulating cells. Naïve T cells differentiate into at least four functional subsets following stimulation by antigen presented by dendritic cells, which are specialized for driving the activation of T cells and are thought to help direct their differentiation by differential secretion of cytokines determining the different subsets. Three subsets - TH1, TH2 and TH17-activate other immune cells with distinct roles in immunity, including B cells, which secrete antibody, natural killer (NK) cells, which are important in defense against viruses, and inflammatory cells, such as neutrophils and macrophages (which also have non-inflammatory functions). The fourth subset shown here comprises regulatory T cells (Tregs), which suppress the activation of the other subsets, partly by acting on dendritic cells. Modified from Figure 5-22 in DeFranco AL, Locksley RM, Robertson M: Immunity: The Immune Response in Infectious and Inflammatory Disease. London: New Science Press; 2007.
CD4+T cell loss in HIV-infected individuals
Although it is known that HIV kills activated CD4+ T cells, it is still a major unresolved question why these cells progressively decline after infection. It is clear that in infected individuals, the rate of loss of CD4+ T cells is greater than the rate of production so that the CD4+ T cell pool is gradually eroded over time, but it remains to be determined how the balance between these processes is impaired. It is unlikely that direct killing of infected target cells by HIV is sufficient to cause CD4+ T cell depletion. Natural hosts for simian immunodeficiency virus (SIV), such as sooty mangabeys, do not progress to AIDS and maintain near-normal levels of peripheral CD4+ T cell numbers despite high rates of viral replication [4]. In fact, the level of immune activation is a better predictor of disease progression than viral load. Consistent with this, HIV infection in humans leads to chronic generalized immune activation characterized by an increased rate of exit of CD4+ and CD8+ T cells and of natural killer (NK) cells from the resting state, increased T and NK cell turnover and death, polyclonal B cell activation with increased levels of gamma globulins, and elevated production of pro-inflammatory cytokines. Conversely, chronic immune activation is not seen following SIV infection in natural hosts that do not show progression to AIDS [4].
Two general causal models have been proposed to account for the association between HIV infection and immune activation. The first is that T cell homeostasis is disrupted by the chronic activation of the innate immune system by HIV infection. The cells of the innate immune system recognize and are activated by conserved components of microorganisms, and include inflammatory cells and specialized cells known as dendritic cells that are critical in the activation of T lymphocytes (Figure 2). HIV stimulates innate immunity in two ways. It directly activates a subset of dendritic cells known as plasmacytoid dendritic cells, leading to production of large amounts of type I interferon, an antiviral cytokine that has been associated with disease progression [5, 6]. HIV can also indirectly cause innate immune stimulation because the infection results in damage to the integrity of mucosal surfaces and the translocation of pro-inflammatory microbial products from the intestinal lumen into the circulation [7]. The ensuing release of inflammatory cytokines by innate immune cells may result in generalized immune cell activation, increasing T cell division and promoting senescence, apoptotic death, or clearance by various mechanisms. Chronic immune activation may also result in bone marrow suppression, reduced thymic function and changes in lymphoid tissue architecture, all of which could reduce the capacity of the T cell pool to maintain itself.
The second possibility is the inverse - that HIV infection dysregulates CD4+ T cell homeostasis and that it is the decline in CD4+ T cell numbers that leads to chronic immune activation. However, HIV infection is associated with activation of multiple distinct branches of the immune system, and it is not obvious how this could reflect a simple homeostatic response to the loss of CD4+ T cells. It is also difficult to reconcile with the observation that there is a rapid decrease in levels of immune activation following antiretroviral therapy, despite persistent low CD4+ T cell numbers [8]. A variant of the CD4+ T depletion hypothesis more consistent with available data is that the loss of particular subsets of effector CD4+ T cells, presumably as a result of direct viral depletion, may contribute to generalized immune activation. For instance, the reduced level of TH17-type T helper cells (Figure 2) in the gastrointestinal tract has been proposed to have a key role in the loss of the integrity of the intestinal mucosa during HIV infection, enabling the translocation of microbial constituents from the intestinal tract into systemic circulation, as discussed above [9]. It has also been suggested that a reduction in Tregs contributes to the aberrant levels of immune activation, as concluded in the Marques et al. paper [1], although (as discussed in their paper and below) it is still unclear whether this population is indeed decreased during HIV infection [10].
Clearly, the two possible causal relationships between chronic immune activation and CD4+ T cell loss are not mutually exclusive. In fact, chronic immune activation and the loss of CD4+ T cells may amplify each other in a loop that makes it difficult to establish which process underlies and drives the other.
Given the complexity of T cell dynamics and the numerous as yet undefined perturbations to normal T cell homeostasis that HIV is likely to induce either directly or indirectly, together with the difficulties in following the dynamic changes in the T cell compartment in the whole body in humans or primates, there is a case to be made for investigating aspects of HIV pathogenesis in mouse models. Although any results have to be extrapolated to the human system with caution, the extensive array of tools available in experimental mouse models to track the rates of division and death of T cells, the rates of flux between naïve and memory pools, and the maintenance of T cell populations over time within lymphoid and peripheral tissues enables a more complete accounting of T cell numbers to be undertaken.
Marques et al. [1] suggest that the OX40-DTA mouse is one approach to this issue and that the findings in these mice provide important insight into the control of lymphocyte dynamics in infected humans. Indeed, in the absence of exogenous infection, OX40-DTA mice do show features consistent with generalized immune activation (Table 1), including an expansion of effector CD8+ T cell numbers that inverts the usual CD4+:CD8+ T cell ratio, and increased serum levels of inflammatory cytokines. This generalized activation cannot be attributed to the release of microbial components into the circulation from the gut, because deletion of activated CD4+ T cells does not in itself lead to a breach in the gut epithelium. Notably, Marques et al. [1] show that the expansion of effector CD8+ T cells and increases in serum levels of inflammatory cytokines can be reversed following reintroduction of Tregs from normal mice, suggesting that the increased immune activation in OX40-DTA mice can in part be ascribed to a Treg insufficiency, which they propose is a key event in HIV-infected individuals leading to CD4+ T cell depletion. However, the reality is a bit more complicated, as we discuss below.
Table 1 Cellular dynamics in OX40-DTA mouse model [1]
T cell dynamics in OX40-DTA mice and HIV-infected humans
Changes in the underlying dynamics of the CD4+ and CD8+ T cell compartments in the OX40-DTA mouse are summarized in Table 1 and Figure 3. Although there is no alteration in the size of the naïve and memory CD4+ T cell pools, there is an increase in the rate of entry of naïve CD4+ T cells into the memory compartment, and an increase in turnover in both. In contrast, the rate of entry of naïve CD8+ T cells into the memory compartment remains unchanged, but the size of the memory CD8+ T cell compartment is almost doubled (Figure 3).
Schematic diagram of dynamic changes in T cell compartments described in OX40-DTA mice [1]. The width of the arrows denotes the rate of death and division or of transit from one pool to another. RTEs, recent thymic emigrants.
Naïve and memory CD4+ T cell division and death rates (turnover) are increased in both HIV infection and the OX40-DTA mouse. However, whether the increased turnover of the CD4+ compartment in the OX40-DTA mouse is a result of Treg depletion or of the deletion of activated cells, or both, is not yet clear. If the increased recruitment of naïve CD4+ T cells in OX40-DTA mice is due to Treg depletion, why are naïve CD8+ T cells unaffected by the loss of these regulatory cells? Perhaps the more likely explanation for the increased turnover of the naïve and memory CD4+ T cell pool is that it results directly from the continuous depletion of activated CD4+ T cells, which provides empty niches and removes the competition for signals (cytokines and other molecules) required for transit into the activated/memory pool. This would explain why naïve CD4+ T cells but not naïve CD8+ T cells are being recruited to the memory pool in the OX40-DTA mouse.
It is still a matter of debate whether continuous recruitment of naïve T cells is required to maintain CD4+ T cell numbers during HIV infection, because memory cells themselves are self-renewing. On one hand, the increased naïve T cell turnover and decreased naïve T cell numbers during HIV infection [8, 11] have led to the hypothesis that progression to AIDS occurs because continuous recruitment of naïve T cells is required to maintain the memory pool and this eventually becomes unsustainable [12]. On the other hand, SIV-infected rhesus macaques can progress to AIDS without naïve CD4+ T cell depletion [13], arguing against a role for CD4+ naïve T cell depletion in disease progression. However, in SIV-infected rhesus macaques, in which disease progression is generally faster than it is in HIV-infected humans, there is a decrease in naïve CD4+ T cells in animals with slower disease progression [14]. This might imply that continuously recruiting naïve T cells can actually delay disease progression.
What do we learn from the OX40-DTA mouse? In these animals, even the ongoing depletion of nearly all activated CD4+ T cells does not result in the progressive erosion of naïve and memory CD4+ T cells seen during HIV infection [1]. Thus, artificially increasing the death of activated CD4+ T cells does not, on its own, seem to have any impact on CD4+ T cell compartment homeostasis. However, this may, at least in part, reflect a difference between humans and mice, in that thymic output in adult mice is relatively high compared with that of humans in mid-life [15] and could counteract the losses in the naïve CD4+ T cell pool in the OX40-DTA mouse. There are other important differences in lymphoid dynamics between the OX40-DTA mouse and HIV-infected humans: memory CD8+ T cell numbers are increased in both, yet although memory CD8+ turnover seems unaffected in OX40-DTA mice, it is increased during HIV infection [8]. Naïve CD8+ numbers and recruitment are unaltered in the OX40-DTA mouse model, whereas naïve CD8+ numbers are decreased and naïve CD8+ proliferation is increased during HIV infection [8, 11]. These considerations limit the extent to which data in the mouse can be directly extrapolated to HIV-infected humans.
Regulatory T cells in HIV infection
Is the role suggested by Marques et al. [1] for Tregs in immune activation in the OX40-DTA mouse consistent with what we know about HIV infection? This question is not straightforward to answer. HIV does not demonstrably infect Tregs at a higher frequency than non-Treg CD4+ T cells [16], so these cells would not be expected to be deleted preferentially as is the case in the OX40-DTA mouse. But neither the role nor the status of Tregs in HIV infection is clear. First, opposing roles have been hypothesized for Tregs [10, 17], each with some evidence to support it. On one hand, Tregs are thought to have a detrimental effect during HIV infection by suppressing anti-HIV immune responses. Indeed, a positive correlation has been described between the fraction of Tregs within the CD4+ T cell pool and viral load (reviewed in [10]). On the other hand, Tregs have been hypothesized to be beneficial by reducing chronic immune activation and limiting inflammatory responses. Evidence for this comes from studies showing a negative correlation between the percentage of Tregs within total T cells and both CD4+ and CD8+ T cell activation [17]. Second, the status of Tregs in HIV-infected individuals is murky, with reports suggesting both increased and decreased numbers, perhaps because of different assays used to measure the size of this compartment. Furthermore, in correlating disease progression with Tregs, the measure of Tregs that is used (for example, the Treg fraction within CD4+ T cells, within total T cells or within total hematopoietic cells, or the absolute numbers of Tregs) can greatly influence the outcome of the analysis. Part of the problem is that whereas in mice Tregs can be unambiguously identified by two markers - the transcription factor FoxP3, which is exclusively expressed by Tregs in this species, and high levels of CD25, a cytokine receptor expressed on activated T cells - in humans, CD25 levels on Tregs are only marginally higher than those on effector CD4+ T cells, and FoxP3 is not exclusively expressed by Tregs, so they are more difficult to quantify accurately.
The role of microbial translocation
During HIV infection, high systemic levels of the microbial product lipopolysaccharide, a potent inflammatory stimulus also known as endotoxin, have been shown to correlate with increased T-cell activation. Despite this correlation, it has not been clear whether endotoxemia directly causes chronic immune activation and concomitant CD4+ T-cell depletion, or whether it only reflects loss of CD4+ host protection or the amount of mucosal damage by existing levels of immune activation. Marques et al. [1] have addressed this important question by extending their studies to a second model mouse (the NEMO mouse [18]), which is engineered to allow systemic leakage of microbial components from the gut (known as microbial translocation), and they do not detect any striking effects on T cell numbers or incorporation of bromodeoxyuridine (BrdU) in these mice, suggesting that microbial translocation may not be the cause of general immune activation. However, further analysis is warranted before a role for microbial translocation on T cell dynamics can be excluded.
A reductionist approach to HIV pathogenesis
Where do all these twists and turns leave us, especially with respect to the insights that can be gleaned from the Marques et al. study [1]? Although mouse models are unlikely to entirely reproduce the complex etiology of AIDS, there is a clear need to shed more light on the dynamic processes underlying disease progression during HIV infection, and experimental models may provide important opportunities to do so. With the goal of investigating key processes in the absence of direct infection, rather than replicating HIV pathogenesis, it is likely that not only the similarities with HIV infection, but also the differences, will teach us something about the basic biology of the system. For instance, the study by Marques et al. [1] highlights the impact that the depletion of activated CD4+ T cells can have on the dynamics of the CD4+ T cell pool and shows that imbalances that result from this process can, by causing a deficiency of Tregs, affect other branches of the immune system. Interestingly, in this mouse model the depletion of activated CD4+ T cells does not progressively erode the CD4+ T cell pool, suggesting that even removing a large proportion of activated CD4+ T cells does not, on its own, impair the renewal capacity of the CD4+ T cell compartment. (However, the continuous recruitment of naïve CD4+ T cells was required to prevent the erosion of the CD4+ T cell pool, and as we pointed out earlier, this may be harder to sustain in humans because of loss of thymic function). In addition, systemic microbial translocation did not induce any immediate changes resembling the cellular dynamics seen during HIV infection.
Overall, insights gained from such reductionist approaches might inform studies more difficult to undertake in humans or primates. In addition, they may generate novel hypotheses as to how the balance of production and loss of CD4+ T cells can be therapeutically altered in the setting of HIV infection to prevent the decline of CD4+ T cells or restore their renewal capacity.
Marques R, Williams A, Eksmond U, Wullaert A, Killeen N, Pasparakis M, Kioussis D, Kassiotis G: Generalized immune activation as a direct result of activated CD4 T cell killing. J Biol. 2009, 8: 93-10.1186/jbiol194.
Croft M, So T, Duan W, Soroosh P: The significance of OX40 and OX40L to T-cell biology and immune disease. Immunol Rev. 2009, 229: 173-191. 10.1111/j.1600-065X.2009.00766.x.
Takeda I, Ine S, Killeen N, Ndhlovu LC, Murata K, Satomi S, Sugamura K, Ishii N: Distinct roles for the OX40-OX40 ligand interaction in regulatory and nonregulatory T cells. J Immunol. 2004, 172: 3580-3589.
Silvestri G, Sodora DL, Koup RA, Paiardini M, O'Neil SP, McClure HM, Staprans SI, Feinberg MB: Nonpathogenic SIV infection of sooty mangabeys is characterized by limited bystander immunopathology despite chronic high-level viremia. Immunity. 2003, 18: 441-452. 10.1016/S1074-7613(03)00060-8.
Mandl JN, Barry AP, Vanderford TH, Kozyr N, Chavan R, Klucking S, Barrat FJ, Coffman RL, Staprans S, Feinberg MB: Divergent TLR7/9 signaling and type I interferon production distinguish pathogenic and non-pathogenic SIV infections. Nat Med. 2008, 14: 1077-1087. 10.1038/nm.1871.
Meier A, Chang JJ, Chan ES, Pollard RB, Sidhu HK, Kulkarni S, Wen TF, Lindsay RJ, Orellana L, Mildvan D, Bazner S, Streeck H, Alter G, Lifson JD, Carrington M, Bosch RJ, Robbins GK, Altfeld M: Sex differences in the Toll-like receptor-mediated response of plasmacytoid dendritic cells to HIV-1. Nature Med. 2009, 15: 955-959. 10.1038/nm.2004.
Brenchley JM, Price DA, Schacker TW, Asher TE, Silvestri G, Rao S, Kazzaz Z, Bornstein E, Lambotte O, Altmann D, Blazar BR, Rodriguez B, Teixeira-Johnson L, Landay A, Martin JN, Hecht FM, Picker LJ, Lederman MM, Deeks SG, Douek DC: Microbial translocation is a cause of systemic immune activation in chronic HIV infection. Nat Med. 2006, 12: 1365-1371. 10.1038/nm1511.
Hazenberg MD, Stuart JW, Otto SA, Borleffs JC, Boucher CA, de Boer RJ, Miedema F, Hamann D: T-cell division in human immunodeficiency virus (HIV)-1 infection is mainly due to immune activation: a longitudinal analysis in patients before and during highly active antiretroviral therapy (HAART). Blood. 2000, 95: 249-255.
Brenchley JM, Paiardini M, Knox KS, Asher AI, Cervasi B, Asher TE, Scheinberg P, Price DA, Hage CA, Kholi LM, Khoruts A, Frank I, Else J, Schacker T, Silvestri G, Douek DC: Differential Th17 CD4 T cell depletion in pathogenic and nonpathogenic lentiviral infections. Blood. 2008, 112: 2826-2835. 10.1182/blood-2008-05-159301.
de St Groth BF, Landay AL: Regulatory T cells in HIV infection: pathogenic or protective participants in the immune response?. AIDS. 2008, 22: 671-683. 10.1097/QAD.0b013e3282f466da.
Roederer M, Dubs JG, Anderson MT, Raju PA, Herzenberg LA: CD8 naive T cell counts decrease progressively in HIV-infected adults. J Clin Invest. 1995, 95: 2061-2066. 10.1172/JCI117892.
Hazenberg MD, Hamann D, Schuitemaker H, Miedema F: T cell depletion in HIV-1 infection: how CD4+ T cells go out of stock. Nat Immunol. 2000, 1: 285-289. 10.1038/79724.
Okoye A, Meier-Schellersheim M, Brenchley JM, Hagen SI, Walker JM, Rohankhedkar M, Lum R, Edgar JB, Planer SL, Legasse A, Sylwester AW, Piatak M, Lifson JD, Maino VC, Sodora DL, Douek DC, Axthelm MK, Grossman Z, Picker LJ: Progressive CD4+ central memory T cell decline results in CD4+ effector memory insufficiency and overt disease in chronic SIV infection. J Exp Med. 2007, 204: 2171-2185. 10.1084/jem.20070567.
Brown CR, Czapiga M, Kabat J, Dang Q, Ourmanov I, Nishimura Y, Martin MA, Hirsch VM: Unique pathology in simian immunodeficiency virus-infected rapid progressor macaques is consistent with a pathogenesis distinct from that of classical AIDS. J Virol. 2007, 81: 5594-5606. 10.1128/JVI.00202-07.
Vrisekoop N, den Braber I, de Boer AB, Ruiter AF, Ackermans MT, Crabben van der SN, Schrijver EH, Spierenburg G, Sauerwein HP, Hazenberg MD, de Boer RJ, Miedema F, Borghans JA, Tesselaar K: Sparse production but preferential incorporation of recently produced naive T cells in the human peripheral pool. Proc Natl Acad Sci USA. 2008, 105: 6115-6120. 10.1073/pnas.0709713105.
Chase AJ, Yang HC, Zhang H, Blankson JN, Siliciano RF: Preservation of FoxP3+ regulatory T cells in the peripheral blood of human immunodeficiency virus type 1-infected elite suppressors correlates with low CD4+ T-cell activation. J Virol. 2008, 82: 8307-8315. 10.1128/JVI.00520-08.
Eggena MP, Barugahare B, Jones N, Okello M, Mutalya S, Kityo C, Mugyenyi P, Cao H: Depletion of regulatory T cells in HIV infection is associated with immune activation. J Immunol. 2005, 174: 4407-4414.
Nenci A, Becker C, Wullaert A, Gareus R, van Loo G, Danese S, Huth M, Nikolaev A, Neufert C, Madison B, Gumucio D, Neurath MF, Pasparakis M: Epithelial NEMO links innate immunity to chronic intestinal inflammation. Nature. 2007, 446: 557-561. 10.1038/nature05698.
The authors' work is supported by the Intramural Research Program of NIAID, NIH, by The Netherlands Organization for Scientific Research (NV) and by the NIH Office of AIDS Research (JNM). The opinions expressed in this article are those of the authors and do not necessarily reflect official views of NIAID or NIH.
Lymphocyte Biology Section, Laboratory of Immunology, National Institute of Allergy and Infectious Diseases, National Institutes of Health, 10 Center Dr MSC-1892, Bethesda, MD, USA
Nienke Vrisekoop
, Judith N Mandl
& Ronald N Germain
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Correspondence to Ronald N Germain.
Nienke Vrisekoop, Judith N Mandl contributed equally to this work.
Vrisekoop, N., Mandl, J.N. & Germain, R.N. Life and death as a T lymphocyte: from immune protection to HIV pathogenesis. J Biol 8, 91 (2009) doi:10.1186/jbiol198
Immune Activation
Simian Immunodeficiency Virus
Cell Pool
Simian Immunodeficiency Virus Infection
Microbial Translocation


Mother of Pearl and Garnet Bracelet from the discontinued Renaissance stock of the Antiquities Couture Collection by 1928.
Filigree links connect 8x10mm cabochon ovals of mother of pearl and garnet. The gold settings are nickel-free 24k gold electroplate. Bracelet measures 7" in length.
This listing is for the bracelet only! Matching MOP Garnet Necklace and Earrings (shown in last picture) are listed separately. As well as a matching MOP Garnet Cross Brooch.
NOTE: These vintage items have never been worn. They have just been in a display box in the shop or storage for over 20 years and it is time for them to go! Many are one of a kind so don't delay in getting the ones you like because they will not last long!


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Kim Kardashian's App Gets Tweeted By the EPA Because the Kardashians Run the Government
Pascal Le Segretain/Getty Images Entertainment/Getty Images
Kim Kardashian's video game has gone from being the most bizarre phrase anyone has ever had to say to a common staple of many people's lives. Our own Bustle writers are addicted and Kardashian stands to make $200 million off the game this year alone. As if it wasn't enough for the iPhone game to consume people's lives to the point where Kardashian had to issue an in-game apology in the form of free game stars when Kim Kardashian: Hollywood crashed last Friday, it appears that the app has also taken over the government. The Environmental Protection Agency tweeted about Kim Kardashian: Hollywood on Monday and the since-deleted tweet makes it clear now more than ever that the Kardashians are running the world.
"I'm now a C-List celebrity in Kim Kardashian: Hollywood," read the EPA tweet, which, according to US Weekly, was deleted after being up for three hours. "Come join me and become famous too by playing on iPhone!"
People didn't know quite how to respond to such a tweet coming from the account that is usually used to update the public on the government's efforts to protect our clean water and reduce pollution in the United States. Some people thought it was a hilarious joke or hack while some people were outright offended that the government had time to play Kim Kardashian's iPhone game when they should be, I don't know, doing government stuff like paperwork and more paperwork.
Listen, it doesn't really matter if the EPA was hacked or if an employee was playing a funny joke or, worse, didn't realize the mistake until it was too late to avoid those 3,000 retweets. What matters is that this is all the proof we need to confirm what we've all been silently thinking: The Kardashian-Jenner clan run the world. Sure, they may have confined their sphere of influence to television, fashion, and bikini selfies, but now they've made the leap into politics.
There's a reason no one can drag themselves away from the Kim Kardashian game and that's because it's controlling everyone's minds — including the EPA. I mean, honestly, if the EPA loses any respect over this wayward tweet, it's probably going to be because they're lowly C-List celebrities and who wants to associate with those?
Kim Kardashian might have turned mind control into the world's most mindlessly entertaining iPhone game, but that's not necessarily a bad thing. I'm going to give the EPA the benefit of the doubt and assume that they're still getting their jobs done around playing Kim Kardashian: Hollywood. Besides, anyone who claims there's not some degree of amusement in their confusion over the tweet is a liar.
Image: AnabiolicMinds (1)


A-MAY-ZING! The Dip was outstanding, the hoagie was perfectly toasted, provolone cheese melted, au just was perfectly seasoned, plenty of meat and grilled mushrooms, and the horseradish sauce gave it a very nice kick.
The only downside for me, it is going to be really difficult for me to try anything else because I love this sandwich so much! Please show this new addition to our community some love, it isn't easy starting a new business PLX welcomes you PLX Deli.


In a large saucepan over medium heat, cook garlic and onion in oil until onion is translucent. Stir in tomato paste and cook until the color is rusty. Pour in chicken broth, and stir in tomatoes, basil, marjoram, oregano, thyme, bay leaf and wine.


My darling daughter, Hannah, is turning five years old tomorrow and I wanted to dedicate this week's Curtain Call Color Challenge to her. I found these amazing Mini Birthday Cookies at Lori's Place etsy shop. The icing colors reminded me of the Stampin' Up! colors, Tempting Turquoise, Lavender Lace, So Saffron, Pumpkin Pie, and Whisper White. Go check out Lori's Place to see many more holiday and themed favor cookies.
To play along in the Curtain Call Color Challenge Act 5, simply create a papercrafted project using these Stampin' Up! colors (or similar shades from other companies), post it on your blog or other public gallery (Splitcoaststampers, Paper Craft Planet, etc.) using the keywords Curtain Call, link your posting back here, and then come back to enter your name and URL in the Mr. Linky box. You have until Sunday, October 11th at noon (CST) to enter your project.
I will once again be picking my FAVORITE(s) Curtain Call Color Challenge submission(s) that will be announced as the Curtain Call Show Stopper at the end of the week!
Those who are chosen as Show Stoppers will receive this award to post on their blog and share the exciting news! I will be picking more than one Show Stopper each week depending on the number of entries there are. There will be awards given out for every ten Curtain Call Color Challenge entries!
Here's the birthday card that I was inspired to make for my big five-year-old!
What FUN colors!! Can't wait to play! Happy Birthday, Hannah!!
Happy Birthday, Hannah! Stacey, I created a card this evening using this amazing color combo! Will photograph in the morning before work and post in the evening.
great colors to play with Stacy, a big happy birthday to your daughter Hannah!
This is a great color combo - thanks for the challenge!
Thanks for the challenge Stacey, fabulous colours. And happy birthday to Hannah - hope she had a wondeful birthday!!
I am very excited to participate!


Lingonberry sauce with dark red colour. Fresh and tangy, but a soft, a bit sugary taste.
Ingredients: Strained lingonberry (55%), water, sugar, pectin.
Golden yellow cloudberry sauce, with seeds removed. Sweet taste, aroma of cloudberry leaves.
Try with crème brûlée, crêpes.
Ingredients: Strained cloudberry (55%), water, sugar, pectin.
Sauce prepared from wild blueberries – bilberries. Intense midnight blue color. Sweet taste of fresh bilberries.
Ingredients: Strained bilberry (55%), water, sugar, pectin.
Cranberry sauce with a fresh and a slightly sour taste.
Ingredients: Strained cranberry (55%), water, sugar, pectin.
Ask us to inform you, when Savouring Bear arrives near you.


Our best sellers among all airlines these past few months!
We make for you a good selection among the best of the french cinema.
Moreover, our catalog can now propose some foreign movies from Italia, Arabic countries and South America.


The e-summon letter for the Union Public Service Commission (UPSC) Engineering Services Examination (ESE) 2016 personality test has been released. All those candidates who have qualified in the written examination are required to download the same from the official website, www.upsc.gov.in. The qualified candidates will now be appearing for the personality test.
"Candidates who have qualified for the personality test of Engineering Services Examination, 2016 are advised to download their e-summon letter and its attachments for the personality test. No summon letter is being sent by post."
The eligible candidates will have to clear the written examination, which comprises of both conventional and objective type tests. Once the candidate clears the same, he/she will then be called for an interview. The candidates will be required to produce the original certificates in support of their claims relating to age, educational qualifications, community, physical disability (where applicable) etc. at the time of the personality test.


or 4 fortnightly payments of $80.25 with Whats this?
HP Colour LaserJet printing supplies offer a professional print quality and consistent performance for affordable colour printing. Through a new toner formulation, cartridge architecture and smart printing feature HP's second generation spherical toner provides professional colour print quality, consistently accurate results and extra-sharp black text.
Has an estimated page yield of 6000 pages under standard conditions.


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South Bay Table Tennis
About South Bay Table Tennis
South bay table tennis is a full-time table tennis sports club also involved in table tennis equipment and accessories sales.
It began full operation in 2009. It somewhat started as an online community of table tennis enthusiasts, players and professionals.
Since then, its operation has advanced to accommodate many more services and clientele over the years.
The table tennis sports club was established to advance the advocacy for all year-long sport, regardless of the weather and other conditions.
Today, South Bay table tennis is a full-fledged table tennis sports club, table tennis facility, and retailer of ping pong accessories and equipment. Together with those, they have a table tennis community and offer private lessons and other tutoring services related to ping pong.
South Bay table tennis is a home for all things related to ping pong.
The tennis club offers table tennis players a standard playing facility filled with plenty of table tennis tables, paddles, and ping pong balls to play ping pong. They also offer players a proper showroom filled with all sorts of table tennis equipment and accessories such as paddles, ball machines, balls and other ping pong gears.
In total, South Bay table tennis offers a friendly and knowledgeable staff to help players of all levels develop properly.
South Bay table tennis is at 540 Maple Avenue, Torrance, adjacent to the L.A. Galaxy Soccer Center. The sports club is keeping up the area's legacy for sports.
https://southbaytt.com/
Monday Thursday 12am – 8:30pm
Friday 12pm – 7:30pm
Follow @southbaytt
https://www.youtube.com/channel/UCCiUjGb-pZZ5MypUFY5Dycw
https://www.instagram.com/sobaytt/


2 Oct Indological Books, 'Valmiki Ramayan II – Gita Press '. 1 Mar Hindi Book-Valmiki-Ramayan-Part-I by Gita Press. Identifier HindiBookValmikiRamayanPartIByGitaPress. Identifier-ark ark://tbq1x . Gita Press Gorakhpur, India. Founded in to propagate the Gita, over the years, the institution has made available more than million copies of the Gita, .
Published 15 days ago. If you are a seller for this product, would you like to suggest updates through seller support? Ramcharitramanas fromgeeta press is gita press ramayan ultimate truth!! Shopbop Designer Fashion Brands.
Write a product review. These items are dispatched from and sold by different sellers. Why I Am a Hindu. Published 3 months ago.
Amazon Prime Music Stream millions of gita press ramayan, ad-free. This version of Srimad Ramacharitamanas contains more than pages. Enter your mobile number or email address below and we'll send you a link to download the free Kindle App. This book is great in every way… printing, colour, cover, pages but only problem is it is not complete as Gita press ramayan Kand is missing, otherwise all 7 kands excellent…!!!
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Connect with Us Facebook Twitter Instagram. Everything is in it. See all free Kindle reading apps. Book quality is good. Don't have a Kindle? Page 1 gita press ramayan 1 Start over Page 1 of 1.
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Every Indian must have this at home and must read it. The Hindi translation is good. Quality of the book represents that only!!
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Visit our Help Pages. Geeta Press Language: Sponsored Products are advertisements for products sold by merchants on Amazon. Published 1 month ago.


text:
Książę Asystent Tronu Papieskiego – najwyższy dziedziczny świecki urząd na dworze papieskim (obecnie w Domu Papieskim) przysługująca dwóm głowom określonych włoskich rodzin arystokratycznych.

Niegdyś do obowiązków księcia należało pełnienie honorowej warty przy tronie papieskim podczas wszelkich uroczystości liturgicznych oraz innych uroczystości z udziałem papieża. Obowiązki te zostały zniesione przez papieża Pawła VI w ramach reform ceremoniału papieskiego po Soborze watykańskim II. Dziś jest to urząd wyłącznie reprezentacyjny i honorowy. Do jednego z nadal istniejących obowiązków Księcia Asystenta jest asysta podczas oficjalnych wizyt głów państw w Watykanie.

Uważa się, że urząd został ustanowiony przez papieża Juliusza II w 1511. Papież  Benedykt XIII postawił, że urząd będzie obsadzany naprzemiennie pomiędzy głowami dwóch rodzin, co było spowodowane wrogością obu rodzin: Colonna i Orsini. Urząd przysługuje głowie rodu Colonna od 1710 roku. Dawniej urząd przysługiwał głowie rodu Orsini. W 1958 roku zastąpił go senior rodu Torlonia, gdyż dotychczasowy Książę Asystent Filippo Orsini został pozbawiony tej funkcji przez papieża Piusa XII w wyniku ujawnienia romansu księcia z aktorką Belindą Lee.

Obecnie tytuł noszą:
 Książę Marco Torlonia, książę Torlonia, Fucino, Canino i Musignano
 Książę Marcantonio Colonna, książę Paliano

Zobacz też
 Asystent Tronu Papieskiego

Bibliografia
 T. Kowalewski, Liturgika, czyli Wykład Obrzędów Kościoła Katolickiego, Płock 1920.

Linki zewnętrzne
 Obecnie istniejące świecki urzędy w Domu Papieskim.

Urzędy i tytuły kościelne

redpajama_set_name: RedPajamaWikipedia


Every year we are amazed and inspired by how people are using the Story Massage programme to share the benefits of positive touch with children and adults. We are delighted to announce our new Best Practice Winners who have worked with such passion and commitment that we feel they deserve special recognition.
Becky is a teacher who uses Story Massage with her class of 15 learners with PMLD (Year 1 – 6) at Springside Primary School. She particularly enjoys using Story Massage as uninterrupted one-to-one time with the pupils. Becky also find Story Massage very helpful at home with her own two children aged 4 and 7 years.
Astrid uses Story Massage within her work as Life Skills Lecturer at Hertford Regional College, helping young people with severe learning difficulties to gain personal confidence and social skills, independent living skills and skills for life and work. Astrid is so passionate about Story Massage that she organised a group training for her colleagues at the college.
Serena has a varied working life as a Relax Kids and Mindfulness Coach. Serena runs regular Story Massage sessions in primary schools as well as community classes for children and adults. She was funded to run 15 Story Massage sessions in prison visiting rooms and felt really touched on one occasion when she watched a family of Mum, Dad and four boys all sitting in a row massaging each other.
Tracey says that Story Massage works really well as a follow on to her five week Baby Reflexology Classes. Tracey recently retired from her job within the community nursing team at a Special School where she worked for 13 years and decided to take the online training course. She feels inspired by the opportunities offered by Story Massage and has lots of plans for the future!
Hazel is a Relax Kids Coach and independent nurture practitioner based in a school campus in Glasgow which encompasses two primary schools, a nursery and a Learning and Communication Unit. Hazel will be leading a workshop with Vicky Mulholland on Writing Scripts for Difficult Situations at our Story Massage Conference in May 2019.
Diane uses Story Massage with her class at Millwood School to bring a sense of calmness and relaxation to her class of 7 learners (Year 4 – 6) with PMLD in a busy environment. Diana says that both children and staff benefit from sharing a peaceful time together.
Christine is an Early Level Practitioner working with children aged 3-5 years who attend Shotts Family Learning Centre. Christine has run workshops for parents and carers with excellent feedback about how Story Massage has helped with bedtime routines and sleeping patterns. The benefits for the children and families using the centre have been so noticeable that all the staff recently attended an INSET training day with colleagues from Dykehead Primary School.


We are a well-known name in the industry for supplying and exporting Inconel Pipe fittings; we are offering manufactured pipe fittings in numerous stipulations and specifications. The use of best grade material, latest manufacturing techniques and International standard quality gives us the leading edge products. The main features of our Pipefittings are that they are highly efficient, accurate and sturdy. The grades used are premium quality Inconel Alloys of Inconel 800, 825, 600, 625, 601, etc. Owning to the rich variety Jain Steels Corporation has been able to provide their valuable customers with the best pipe fittings at competitive price.
Range ½"NB to 24″NB in Sch 10s, 40s, 80s, 160s, XXS.
We have exported our material to numerous countries in the world including but not limited to Oman, South Africa, Qatar, Nigeria, Turkey, Netherlands, Singapore, Yemen, Italy, Australia, Mexico, New Zealand, UAE, USA, UK, Venezuela, Indonesia, Denmark., Bahrain, Srilanka, Germany, Iran, Malaysia, Poland, Brazil, Jordan, Saudi Arabia, Madagascar, Kuwait, Canada, Iraq, Spain, Kuwait, Serbia, Peru, Lebanon, Angola, Egypt.


The ability of the Rotary Club of Haddonfield to support local community events and initiatives, award meaningful scholarships to deserving high school students, and grants to service organizations with important missions depends entirely upon donations, sponsorships and grants. We welcome contributions at any time, and in several ways.
If you want to make a tax deductible donation that supports local, regional and international nonprofit organizations that serve humanity -- as determined through a Grants Committee selection process each year-- please make your contribution to the Rotary Club of Haddonfield Foundation. This can be done by mailing a check made payable to the Rotary Club of Haddonfield Foundation through our Club Administrator, Vin Parker, at the address below.
Major contributions in the form of unrestricted gifts, estate and other planned gifts are encouraged through the Rotary Club of Haddonfield Foundation. Those wishing to discuss such contributions or bequests should contact Karen Hagerman, Chair of the Club's Foundation, at 856-428-1999.
If you want to support local civic and community activities and do not need a tax deduction, donate to the Rotary Club of Haddonfield. Donations to the Club (rather than the Club's Foundation) support summer concerts along Kings Highway in Haddonfield, leadership programs for local high school students, holiday gifts for children in need, 4th of July fireworks, New Year's Eve First Night event, among others. This can be done by clicking the link on the website home page, or by mailing a check to the Club Administrator, at the address below.
Fundraisers are held routinely, which are not only great fun for everyone, but provide critical revenue to support the grants, scholarships and community activities we support. Check back to our website routinely to see what's planned, and make sure you're on our mailing list. Send us an email from the Contact Us page of this website to request to be added to our list.
Click below to make a contribution by credit card through Pay Pal.