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Roh, Park, Lee, and Hwang: Thyrotoxic hypokalemic periodic paralysis due to Graves' disease in 2 adolescents
Annals of Pediatric Endocrinology & Metabolism 2019;24(2): 133-136.
Thyrotoxic hypokalemic periodic paralysis due to Graves' disease in 2 adolescents
Jung Gi Roh, MD, Kyu Jung Park, MD, Hae Sang Lee, MD, PhD , Jin Soon Hwang, MD, PhD
Department of Pediatrics, Ajou University Hospital, Ajou University School of Medicine, Suwon, Korea
Address for correspondence: Hae Sang Lee, MD, PhD Department of Pediatrics, Ajou University Hospital, Ajou University School of Medicine, 164 World cupro, Yeongtong-gu, Suwon 16499, Korea
Tel: +82-31-219-5166 Fax: +82-31-219-5169 E-mail: seaon98@naver.com
Received August 18, 2018 Revised September 9, 2018 Accepted October 30, 2018
© 2019 Annals of Pediatric Endocrinology & Metabolism
This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial License (http://creativecommons.org/licenses/by-nc/4.0/) which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited.
Thyrotoxic periodic paralysis (TPP) is a notable and potentially lethal complication of thyrotoxicosis, and Graves' disease is the most common cause of TPP. TPP is commonly reported in Asian males between 20–40 years of age, but it is rare in children and adolescents. We report 2 Korean adolescents (a 16-year-old male and a 14-year-old female) with episodes of TPP who were previously diagnosed with Graves' disease. These 2 patients presented with lower leg weakness in the morning after waking up. They were diagnosed with TPP-associated with thyrotoxicosis due to Graves' disease. After they were initially treated with potassium chloride and antithyroid drugs, muscle paralysis improved and an euthyroid state without muscle paralytic events was maintained during follow-up. Therefore, clinicians should consider TPP when patients have sudden paralysis and thyrotoxic symptoms such as goiter, tachycardia, and hypertension.
Keywords: Thyrotoxic periodic paralysis, Hypokalemia, Graves' disease
Thyrotoxic periodic paralysis (TPP) is an uncommon but serious complication of hyperthyroidism, and it is associated with hypokalemia [1]. TPP is characterized by recurrent muscle weakness and paralysis. Fatal complications, including ventricular arrhythmias, can occur in severe cases of TPP [2]. TPP mainly affects male patients of Asian descent, especially those 20–40 years of age, and the incidence has been reported at a rate of approximately 2% in hyperthyroidism patients [3]. The incidence of TPP in pediatric patients is very low compared to that in adult patients. Although it is rare in children and adolescents, some cases of TPP in adolescents have been reported from China, Korea, and in other ethnic groups [4]. Most recently, the reported cases of TPP have been attributable to Graves' disease, although autoimmune thyroiditis can be a very rare cause of TPP in children and adolescents. Here we report cases of 2 Korean adolescents who presented with TPP as clinical manifestation of Graves' disease along with a comprehensive literature review of pediatric patients in Korea.
1. Case 1
A 16-year-old Korean boy had one episode of lower leg paralysis and upper extremity weakness in the morning after waking up and without having any symptoms associated with thyrotoxicosis. Prior to showing symptoms, the patient had practiced Japanese fencing for one hour the night before.
The patient did not have any past or family history associated with paralysis symptoms. He had a height of 168.3 cm, a weight of 69.4 kg, and a body mass index of 24.5 kg/m2. He had a grade 1 goiter based on World Health Organization criteria. When he arrived, his vital signs were stable with normal neurological examination findings, with the exception of motor weakness. His upper and lower extremity motor grades were 4 for both arms and 3 for both legs. His sensory responses were intact and his initial electrocardiogram and echocardiography results were normal.
Laboratory findings showed normal complete blood cell counts and electrolytes with the exception of decreased potassium levels (K+, 2.5 mmol/L). A thyroid function test showed hyperthyroidism: serum free thyroxine (fT4), 2.46 ng/dL (range, 0.79–1.86 ng/dL), triiodothyronine (T3), 2.46 ng/mL (range, 0.76–1.90 ng/mL), and thyroid-stimulating hormone (TSH), < 0.008 IU/L (range, 0.15–5.0 IU/L). His serum thyroglobulin antibody (TG Ab) level was at 336 U/mL (range, 0–60 U/mL) and his thyroid peroxidase antibody (TPO Ab) level was 33 U/mL (range, 0–60 U/mL). However, his serum TSH receptor antibody level was 1.0 IU/L (range, 0–1.5 IU/L). Thyroid ultrasonography showed diffuse mild enlargement with heterogeneous echogenicity and prominent vascularity. A thyroid scan with technetium 99m pertechnetate showed that both lobes were normal in size with homogenous trapping. He was initially treated with potassium chloride (KCl), 1.1 mEq/kg/day intravenously for 4 hours, and oral potassium (K-contin), two 600-mg tablets once daily for 3 days, and 5 mg of methimazole once daily. We did not use a β-adrenergic blocker because he was asymptomatic for tachycardia based on the thyrotoxicosis symptoms. After 2 hours of loading KCl intravenously, his upper and lower extremity motor grades were 5 and 4, respectively. After 10 hours, his motor grades were all intact and his K+ level was normalized to 4.8 mmol/L. On hospital day 4, he was discharged with methimazole. At 1-year follow-up, his serum levels of TG Ab, TPO Ab, and TSH receptor antibodies were 482 U/mL, 134 U/mL, and 10.3 IU/L, respectively (Table 1). Based on these results, the patient was diagnosed with hypokalemic paralysis due to Graves' disease. Two years later, he is still taking methimazole 5 mg once a day, and maintaining an euthyroid status without muscle paralytic events.
A 14-year-old female patient was admitted to the hospital through an outpatient clinic for treatment of tachycardia, palpitation, and thyroid hypertrophy. On admission, she had no symptoms of limb paralysis. However, on the following day, she complained of lower leg weakness in the proximal area in the morning after waking up. Unlike the first case, she was in a bed rest status to prevent worsening of thyrotoxicosis. Thus, she had not performed any significant physical activity before the onset of paralytic symptoms.
She had no past or family history associated with paralysis symptoms. Her height was 166.1 cm with a weight of 45.9 kg. Her body mass index was 16.64 kg/m2. At our hospital, her blood pressure was 164/96 mmHg with a body temperature of 36.6℃ and a heart rate of 136 beats/min. Exophthalmos and grade 2B goiter were confirmed on physical examination. Her neurological examination findings were normal except for motor weakness.
When she had symptoms of weakness, her upper and lower extremity motor grades were 5 and 4, respectively. However, her sensory responses were intact. Laboratory findings showed a serum potassium level of 3.1 mmol/L. There were no abnormalities of blood gas or inflammatory factors, including erythrocyte sedimentation rate and C-reactive protein. A thyroid function test showed hyperthyroidism, with a fT4 level at 4.97 ng/dL (range, 0.79–1.86 ng/dL), a T3 level at 6.15 ng/mL (range, 0.76–1.90 ng/mL), and a TSH level at 0.06 IU/L (range, 0.15–5.0 IU/L). Her serum TG Ab level was at 114 U/mL (range, 0–60 U/mL) and her TPO Ab level was at 152 U/mL (range, 0–60 U/mL) with a serum TSH receptor stimulating antibody level at >40 IU/L (range, 0–1.5 IU/L) (Table 1). Her initial electrocardiogram showed a sinus rhythm with a first degree atrioventricular block while her echocardiography results were normal. Thyroid ultrasonography showed an enlarged thyroid size and hypervascularity. A thyroid scan with technetium 99m pertechnetate showed diffuse enlargement of both lobes with increased trapping. These results indicated hypokalemic paralysis due to Graves' disease.
She was initially treated with KCl (0.348 mEq/kg, oral [K-contin], two 600-mg tablets 3 times) for 2 days, an antithyroid drug (prophylthiouracil) 150 mg 3 times a day (10 mg/kg/day) and a β-adrenergic blocker (propranolol 1 mg/kg/day) 15 mg 3 times a day (Table 2). Because she showed high fT4 and T3 levels with tachycardia, hypertension, and diarrhea, we determined that her condition was an imminent thyroid storm. Therefore, we prescribed propylthiouracil which can help inhibit peripheral conversion [5]. After 8 hours, her sensory and motor grades were all intact and her serum potassium level was normalized to 4.7 mmol/L. On hospital day 8, she was discharged with a β-adrenergic blocker (propranolol 15 mg 3 times a day) and an antithyroid drug (propylthiouracil 150 mg 3 times a day). At her 6-month follow-up, she maintained an euthyroid state without muscle paralytic events. Follow-up serum TG Ab and TPO Ab levels were 131 U/mL and 58 U/mL (range, 0–60 U/mL), respectively. Three years later, she is still taking an antithyroid drug. Thyroid hormone levels and thyroid-associated antibodies are constantly maintained at normal levels.
TPP is the most frequent form of acquired muscle paralysis in young adults [6]. The ratio of cases in males to females ranges from 17:1 to 70:1 [7]. TPP is common in Asia. To date, genes that have been studied in relation to TPP include the KCNJ18 and KCNJ2 genes. The KCNJ18 gene is known to be more common in Western countries, and has been reported at rates of 33.3% in Caucasian populations, primarily among individuals with Brazilian heritage, 25.9% in Singaporeans, and 1.2% in people from Hong Kong [8]. In contrast, in recent studies, genetic variants of the KCNJ2 gene have been reported to affect the development of TPP in Korean and Chinese populations [9,10]. These ethnic genetic differences might explain the difference in the incidence of TPP. The most common cause of TPP is Graves' disease. However, TPP can occur in any type of thyrotoxicosis [6]. In general, autoimmune thyroid diseases such as Graves' disease are more common in pubertal girls than boys. However, the incidence of TPP is higher in boys than in girls [7]. Reported TPP cases in Korean adolescents have been primarily males (5 males and 2 females) and all patients had been diagnosed with Graves' disease (Table 2). Six patients had elevated serum thyroid hormone levels and they were treated with methimazole and/or a β-adrenergic blocker. Among previously reported cases, two patients had recurrent paralytic episodes during antithyroid drug treatment.
Hypokalemia in TPP develops by a rapid and massive shift of potassium from the extracellular to the intracellular compartment, mainly into muscles. Thyroid hormone can stimulate Na+–K+ ATPase in skeletal muscle by transcriptional up-regulation of the gene that encodes Na+–K+ ATPase and by enhancing the intrinsic activity or membrane insertion of the pump [11]. Thus, there is no pure potassium depletion in the body [12]. Insulin, a high carbohydrate meal, and exercise are other well-known activators of Na+–K+ ATPase [13]. Although serum potassium levels are usually decreased in TPP, as in our reported cases, in exceptionally rare circumstances normal serum potassium levels can lead to normokalemic TPP. Patients who initially showed normokalemic TPP also eventually progressed to hypokalemia after [14]. However, the pathogenesis of normokalemic TPP remains unclear.
Diagnosis of TPP is based on recurrent clinical features of TPP and blood test results suggestive of hyperthyroidism and hypokalemia. The severity of paralysis is correlated with the degree of hypokalemia, but not with clinical signs/symptoms of hyperthyroidism or thyroid hormone levels [7]. Previously reported cases with TPP had only mildly elevated serum thyroid hormone levels. Ko et al. [15] have reported that only 10% of patients have mild thyrotoxic symptoms. Therefore, TPP should be distinguished from other causes of acute paralysis such as familial hypokalemic periodic paralysis, Guillain-Barré syndrome, myasthenic crisis, and conditions that produce spinal cord compression [16]. TPP is very rare in children and adolescents and is even more unusual in girls. In addition, although there are aggravating factors, such as a high carbohydrate meal and exercise, that are associated with the occurrence of TPP, TPP can occur without such factors as was seen in our second case. Therefore, based on the two cases in this report, the occurrence of TPP should always be considered regardless of sex when treating patients with Graves' disease.
Treatment of TPP includes correction of hypokalemia and maintenance of an euthyroid status. Immediate supplement with KCl is needed to prevent major cardiopulmonary complications and to foster muscle paralysis recovery [12]. However, another study has reported that there is lack of correlation between recovery time of muscle paralysis and the dose of infused KCl [17]. Despite intravascular hypokalemia, the total body potassium level is normal. Thus, KCl infusion should be administered with caution. KCl infusion causes rebound hyperkalemia in up to 70% of cases, and as a result, fatal arrhythmias may occur. Rebound hyperkalemia can be prevented by lower doses of KCl and close cardiac monitoring is necessary [16]. A β-adrenergic blocker is an alternative treatment that can reduce paralysis without rebound hyperkalemia. Because TPP does not usually recur once an euthyroid state has been attained, adequate control of hyperthyroidism is important after the acute phase [7]. Currently, antithyroid drugs remain the mainstay treatment of hyperthyroidism.
In conclusion, TPP is a very rare muscle disorder in children and adolescents. Any etiology of hyperthyroidism can be associated with TPP, including Graves' disease, Hashimoto's thyroiditis, and toxic nodular adenoma. Thus, pediatricians should perform thyroid function tests when treating patients with sudden paralysis and thyrotoxic symptoms including goiter, tachycardia, and exophthalmos.
Ethical statement: This study was approved by the Institutional Review Board of the Ajou University Hospital (AJIRB-MED-EXP-18-250). The requirement for informed consent was waived due to the retrospective nature of this study.
Conflict of interest: No potential conflict of interest relevant to this article was reported.
Thyroid function tests in patients
Case 1 (male, 16 years old)
Case 2 (female, 14 years old)
At diagnosis
Free T4 (ng/dL) 2.46 1.56 1.23 4.97 1.72 1.58
T3 (ng/dL) 246.2 140 78 615 188 110
TSH (μIU/mL) <0.008 0.10 0.56 0.06 0.04 0.11
TPO Ab (U/mL) 33 79 115 152 58 71
TG Ab (U/mL) 336 488 387 114 131 108
TRS Ab (IU/L) 1.0 1.3 13.3 >40 10.9 3.7
Reference range: serum free thyroxine (free T4), 0.79–1.86 ng/dL; triiodothyronine (T3), 0.76–1.90 ng/mL; and thyroid-stimulating hormone (TSH), 0.15–5.0 IU/L; thyroid peroxidase antibodies (TPO Ab) and antithyroglobulin antibodies (TG Ab), 0–60 U/mL; TSH receptor-stimulating Ab (TRS Ab), 0–1.5 IU/L.
Summary of 7 patients with thyrotoxic periodic paralysis reported in Korean adolescents
Age (yr)
Free T4 (ng/dL)
T3 (ng/mL)
TSH (IU/L)
K+ (mmol/L)
Oh et al. [6] 2011 F 14 3.80 1.83 <0.03 2.3 Graves' disease β-adrenergic blocker, antithyroid drug
Ahn et al. [18] 2012 M 16 1.87 1.80 0.09 2.5 Graves' disease Antithyroid drug Three recurrent episodes
Previously diagnosed with Graves' disease before paralytic events
Wi et al. [19] 2012 M 16 NA NA NA 1.8 Graves' disease NA
Cho et al. [20] 2013 M 17 3.44 2.85 <0.01 2.2 Graves' disease β-adrenergic blocker, Two recurrent episodes antithyroid drug Two recurrent episodes
Jung et al. [4] 2014 M 16 2.1 2.95 <0.03 2.7 Graves' disease β-adrenergic blocker, antithyroid drug
Current case 1 2015 M 16 2.46 2.46 <0.008 2.5 Graves' disease Antithyroid drug
Current case 2 2015 F 14 4.97 6.15 0.06 3.1 Graves' disease β-adrenergic blocker, antithyroid drug
Free T4, serum free thyroxine; T3, triiodothyronine; TSH, thyroid-stimulating hormone; K+, potassium; NA, not applicable.
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Effects of short-term potassium iodide treatment for thyrotoxicosis due to Graves disease in children and adolescents2014 December;19(4)
A case of thyrotoxic periodic paralysis as initial manifestation of Graves' disease in a 16-year-old Korean adolescent2014 September;19(3)
The treatment of Graves' disease in children and adolescents2014 September;19(3)
The Clinical Course and Prognostic Factors to Medical Treatment of Graves' Disease in Children and Adolescents.2012 March;17(1)
A Case of Thyrotoxic Periodic Paralysis in a 16-Year-Old Adolescent.2012 June;17(2)


Increasing crop productivity to meet burgeoning human food demand is challenging under changing environmental conditions. Since industrial revolution atmospheric CO(2) levels have linearly increased. Developing crop varieties with increased utilization of CO(2) for photosynthesis is an urgent requirement to cope with the irreversible rise of atmospheric CO(2) and achieve higher food production. The primary effects of elevated CO(2) levels in most crop plants, particularly C(3) plants, include increased biomass accumulation, although initial stimulation of net photosynthesis rate is only temporal and plants fail to sustain the maximal stimulation, a phenomenon known as photosynthesis acclimation. Despite this acclimation, grain yield is known to marginally increase under elevated CO(2). The yield potential of C(3) crops is limited by their capacity to exploit sufficient carbon. The "C fertilization" through elevated CO(2) levels could potentially be used for substantial yield increase. Rubisco is the rate-limiting enzyme in photosynthesis and its activity is largely affected by atmospheric CO(2) and nitrogen availability. In addition, maintenance of the C/N ratio is pivotal for various growth and development processes in plants governing yield and seed quality. For maximizing the benefits of elevated CO(2), raising plant nitrogen pools will be necessary as part of maintaining an optimal C/N balance. In this review, we discuss potential causes for the stagnation in yield increases under elevated CO(2) levels and explore possibilities to overcome this limitation by improved photosynthetic capacity and enhanced nitrogen use efficiency. Opportunities of engineering nitrogen uptake, assimilatory, and responsive genes are also discussed that could ensure optimal nitrogen allocation toward expanding source and sink tissues. This might avert photosynthetic acclimation partially or completely and drive for improved crop production under elevated CO(2) levels.


Once I had connected all the ESC's and mounted all the arms into place, I began the process of sorting out the wiring mess in the middle. I started by cutting all the power wires and soldering them into the distribution board (the bottom plate). Unfortunately, the specific batch of distribution boards that I received were missing a solder pad for one of the ESC's. This wasn't a big deal because I was able to just branch off the other solder pads.
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Quick plays: RAINBOWS ARE FREE, ROBIN GEORGE, LOUDON WAINWRIGHT III, SALIVA, AMANDA RHEAUME
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Although some have a dated 80′s feel the production has been spruced up for the modern day and the whole album provides a fascinating insight into the start of Robin George's career. Even at this early age and stage of his career he knew how to pen a strong, melodic pop rock song. ***1/2
LOUDON WAINWRIGHT III – Haven't Got The Blues Yet
Proper Records [Released 28.07.14]
This is amazingly the 26th album from Loudon Wainwright III and a follow-up to his enjoyable 2012 album 'Older Than My Old Man Now' (which he refers to as 'my death 'n' decay opus').
What I like about Loudon Wainwright III is that he has such musical variety on each album and the topicality of his lyrics. You have the classic rock 'n' roll of 'Brand New Dance' and then a little Balkan sound on 'Spaced', an alternate parking song. 'Depression Blues' tips its musical hat to the original blues players, whilst 'The Morgue' is a lovely tune done in a Vaudeville style, entertaining stuff.
'Harmless' is a tale of everyday folk and life, it is very laid back very much like the character in the song. For a little blue grass/folk there is 'Man And Dog', marvel at the banjo playing and enjoy the harmony vocals that join in now and then like an in tune drunken chorus.
He is hard to pigeonhole which only adds to the appeal as you get a potpourri of music and themes. The album is a perfect way to while away some time and lose yourself in the music. ****
AMANDA RHEAUME Keep A Fire
It's perhaps no surprise that Ottawa songstress Amanda Rheaume has attached a clutch of Irish dates to her brief UK tour this autumn to promote her 'roots-pop-Americana' album Keep A Fire.
Her songwriting style is more popular than 'pop' and for the most part her easy on the ear style is more akin to folk than Americana, and with the mildest of Celtic undertones her delivery has many parallels with that of Mary Black.
It all adds up to a pleasant enough Radio 2 listen that is not going to offend, but neither does it break any new ground in an already over saturated field. ***
RED MOLLY The Red Album
Celebrating their 10th anniversary in 2014, Red Molly's (Laurie MacAllister(bass), Abbie Gardner(dobro), and Molly Venter (guitar) 5th studio album is their first created in Nashville, with producer Ken Coomer (original drummer for Wilco and Uncle Tupelo) pushing for more original songs and a grittier sound.
Taking their 'RedHeads' tag from Richard Thompson's iconic 1952 Vincent Black Lightning (which is covered on the album) the band's selling point is their 3 part harmony work that adds a different dimension to their brand of blues, gospel, country and bluegrass Americana.
Probably not cross over enough for those without a passing interest in the genre, but The Red Album should nevertheless move fairly well on 'merch' stand when the girls play a selection of Ireland and UK dates in October. ***
MICHAEL DES BARRES – Hot n Sticky Live
MDBimmeadia [Out now]
Following the release of 2012's critically acclaimed "Carnaby Street" album, former Detective, Chequered Past and Power Station front man Michael Des Barres releases "Hot n Sticky Live". In case you hadn't guessed from the title, this 8 track album was recorded live at Hollywood's Viper Room and features a solid band mixing the sounds of glam rock, punk and blues rock.
There is a fine line between the sexually charged swagger of say Mick Jagger or Steven Tyler, and the risk of sounding like old man Steptoe and 66 year old Des Barres in between song banter, at times, unfortunately leaves him with a foot in both camps.
Opening track "Carnaby Street" features some fine Hammond B3 organ and some excellent guitar weaving, "You're My Pain Killer" is blues soaked whilst "Please Stay" is reminiscent of Rod Stewart in his prime. "Detective Man", "Little Latin Lover" and a great interpretation of the classic "Stop, In The Name of Love" are the winning tracks here, although the 10 minute medley of "I Don't Need No Doctor/Get It On/My Baby Saved My Ass" contains some stunning guitar moments.
Michael Des Barres has enjoyed a lengthy musical career and subsequent acting career, "Hot n Sticky Live" sums up the former perfectly. ***1/2
Review by Nikk Gunns
SALIVA – Rise Up
Rum Bum Records [Released 29.04.14]
Saliva have been building a decent career for themselves in the nu metal field and appealing to fans of bands like Papa Roach. This album sees a little change in direction for the band as they have a new vocalist, Bobby Amaru, who has much more melodic sound to his vocals than the previous vocalist Josey Scott.
This album has a lot of radio friendly rock/metal tunes, full of angst and in your face guitar riffs. The title track and 'No One But Me' could even be a more metal Nickelback. They even throw in a decent ballad in the shape of 'Closer'.
Fans of the band will like this album I am sure, although when a band changes vocalist they always lose some fans who won't accept the new boy. The good thing about this album is that given its overtly melodic sound it will easily win them over new fans. ***1/2
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Power Plays w/c 7 July (Mon-Fri)
CROBOT Low Life (Mascot)
JAILBIRDS Thrill Of The Chase (Golden Robot Records)
SCARLET REBELS No One Else To Blame(indie)
WICKED STONE Unchained (indie)
JARED JAMES NICHOLS Nails In the Coffin (Listenable Records)
Featured Albums w/c 7 July (Mon-Fri)
09:00-12:00 RESTLESS SPIRITS (Frontiers)
12:00-13:00 LICENCE N.2.O.2.R (Metalapolis)
14:00-16:00 ANGELA PERLEY 4:30 (indie)
Albums That Time Forgot (Mon-Fri)
STEVE HILLAGE Open (1979)
Tweets by Get Ready to ROCK!
This entry was posted in ALBUM REVIEWS, ALBUM REVIEWS (Mobile), All Posts, Quick Plays and tagged album, Amanda Rheaume, Angel Air, classic rock, Heartline, history, Keep A Fire, Loudon Wainwright III, Mark Stanway, Michae Des Barres, night by night, Phil Lynott, Rainbows Are Free, Red Molly, review, Rise Up, Robin George, Sabbath, Saliva, stoner rock, The Red Album, Thin Lizzy, Waves Ahead Of The Ocean. Bookmark the permalink.
Album review: GLEB KOLYADIN – s/t
Album review: GAMMA RAY – Heading For Tomorrow
EP review: POWER QUEST – Face The Raven
DVD review: SUGGS – My Life Story
Album review: DARWIN – Origin of Species
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redpajama_set_name: RedPajamaCommonCrawl


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Simply fill out the form below and submit it to us. We will receive this form via email and contact you for method of payment. Once payment has been received we will email your official gift certificate in a printable PDF format where you can print it and give the gift certificate to whomever the gift is for. Please allow 24 hrs.for us to receive this form and us to contact you.
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So yesterday the Middletown Division of Police was out serving the public when they found themselves "Flocked" by the Fourth Grade Middletown Youth Football Team.
From the MPD Facebook Page….
The Flamingos are part of a fundraiser for the youth football team.
Seems like money well spent to me. So where to send them next…. so many mischievous choices….
It's a really cute idea, but there's no contact information!
If you are interesting in "Flocking" your fiends in Middletown to support the Youth Football team, contact Tiffany Rupp at 513-571-2487 or by email at tiffany.rupp@ymail.com.
Would they invade my realtor? Lol Jackie and Stephan would love it.
We get home tonight and behold our front yard is full of pink flamingos!! What a fun surprise! Now, who is next?


text:
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IP Address Reputation in E-Mail Systems
Published: Dec 8, 2022
IP address reputation is a measure of the trustworthiness and reliability of a particular IP address in the context of an e-mail system. A good reputation indicates that the IP address is generally used to send legitimate, non-spam email, while a poor reputation may indicate that the IP address is used to send spam or other malicious email. Email systems often use IP address reputation as a factor in determining whether to deliver an incoming message, move to spam/junk folder and may block or filter messages from IP addresses with poor reputations in order to protect their users from spam and other unwanted email.
Reputation based on the Network your email system is on
The network that an IP address is on can affect its reputation in an e-mail system because the reputation of a network can have an impact on the reputation of the individual IP addresses that are part of that network. For example, if a network is known to be used by spammers or to host malicious content, the reputation of the IP addresses on that network may be negatively affected.
On the other hand, if a network has a good reputation and is used by reputable organizations, the reputation of the IP addresses on that network may be positively affected. Therefore, the network that an IP address is on can play a role in determining its reputation in an e-mail system.
Selecting the right network provider
Selecting the right network provider is the first step to build up a reliable email system.
Here are a few tips for selecting a network provider with a good reputation for e-mail systems:
Research the reputation of the network provider. Check online reviews, ratings, and other information to learn about the provider's track record and the experiences of other users.
Ask for recommendations from other organizations that run email systems. They may be able to provide insights into the reputation of different network providers and help you make a more informed decision.
Check the provider's policies and practices. Look for information about how they handle spam, malicious content, and other potential threats related to their network.
Consider the provider's technical capabilities and infrastructure. A provider with a strong and reliable network, robust security measures, and good support services can help ensure the integrity and performance of your email system.
By following these tips, you can help ensure that you select a network provider with a good reputation for running reliable email systems.
IP Reputation based on PTR record
The PTR (pointer) record of an IP address is a record in the Domain Name System (DNS) that maps the IP address to a domain name. This can be used to identify the owner or operator of the IP address. In some cases, the PTR record of an IP address may influence its reputation in an e-mail system. For example, if the PTR record indicates that the IP address belongs to a known spammer or a domain that is frequently used to send spam, the reputation of the IP address may be negatively affected. On the other hand, if the PTR record indicates that the IP address belongs to a reputable and trustworthy domain, the reputation of the IP address may be positively affected. Therefore, the PTR record of an IP address can play a role in determining its reputation in an e-mail system.
Changing the PTR record of an IP address can affect its reputation in an e-mail system because the PTR record is often used to identify the owner or operator of the IP address. If the PTR record is changed to point to a different domain, this can indicate a change in the ownership or operation of the IP address. Depending on the reputation of the new domain, this change may have a positive or negative effect on the reputation of the IP address.
Also often changing PTR record indicates, that the email system behind the IP may be used for malicious activity by hiding it's behaviour behind new DNS Records.
IP Reputation based on domains being hosted on the email systems
Hosted domains on an email system can impact the IP reputation of the system because the reputation of the domains can have an impact on the reputation of the IP addresses that are associated with the email system. For example, if a hosted domain is known to be used for spamming or other malicious activity, the reputation of the IP addresses used by the email system may be negatively affected. This can make it more difficult for the email system to deliver messages to other systems.
On the other hand, if the hosted domains have good reputations and are used for legitimate purposes, the reputation of the IP addresses used by the email system may be positively affected. This can help improve the deliverability of the email system. Therefore, the reputation of the hosted domains on an email system can impact the reputation of the IP addresses used by the system.
IP Reputation based on the traffic
Email traffic on an email system can impact the IP reputation of the system because the volume, content, and behavior of the email traffic can affect the reputation of the IP addresses used by the system. For example, if the email system is used to send a high volume of spam or other unwanted email, the reputation of the IP addresses used by the system may be negatively affected. This can make it more difficult for the email system to deliver messages to other systems, and can increase the risk of spam and other unwanted email being delivered to users.
On the other hand, if the email traffic on the system is legitimate and follows best practices for sending email, the reputation of the IP addresses used by the system may be positively affected. This can help improve the deliverability of the email system and protect its users from spam and other unwanted email. Therefore, the email traffic on an email system can impact the reputation of the IP addresses used by the system.
IP Reputation based on history in third party systems
You may have selected the right provider, but the history of the IP you have taken over, maybe have been used for malicious traffic. There are several ways to improve the reputation:
Dependent on the system you have bad reputation on, it is a good idea to contact the according party and ask them to reset the reputation or even the whole history. Check their postmaster website and explain, that you have taken over this domain for legitimate email traffic.
Sometimes you can simply delist a blacklisted IP address and return to normal reputation
If you have taken over a domain, which was never used for email delivery, you may slowly increase legitimate the traffic to senders. This will build up your reputation as a valid sender.
Building a high reputation IP addresses for legitimate email traffic is an essential part of an email service operator. This can't be done in one day.
Loosing reputation can be done in seconds, that's why maintaining your email service and supporting customers is essential to stay in email legit business.
Having a good reputation is essential for deliverability in email systems, but you can do a lot to gain this reputation.
Here we have some additional tips, which may help you:
Use best practices for sending email
Use authentication protocols (e.g. SPF and DKIM), and implement a feedback loop.
Monitor your sending reputation and address any issues that may affect it, such as high bounce rates or spam complaints.
Avoid sending spam or other unwanted email, and comply with email marketing regulations and best practices.
Monitor your IP and domain reputation, and take steps to address any issues that may cause your IP or domain to be blacklisted.
Mady with ♥ by Qameta Software · Imprint · Legal · Newsletter · Privacy · Whitepaper · FAQ


text:
from base import BaseClient

HUBSPOT_BROADCAST_API_VERSION = '1'

class BaseSocialObject(object):
    def _camel_case_to_underscores(self, text):
        result = []
        pos = 0
        while pos < len(text):
            if text[pos].isupper():
                if pos - 1 > 0 and text[pos - 1].islower() or pos - 1 > 0 and pos + 1 < len(text) and text[pos + 1].islower():
                    result.append("_%s" % text[pos].lower())
                else:
                    result.append(text[pos].lower())
            else:
                result.append(text[pos])
            pos += 1
        return "".join(result)

    def _underscores_to_camel_case(self, text):
        result = []
        pos = 0
        while pos < len(text):
            if text[pos] == "_" and pos + 1 < len(text):
                result.append("%s" % text[pos + 1].upper())
                pos += 1
            else:
                result.append(text[pos])
            pos += 1
        return "".join(result)

    def to_dict(self):
        dict_self = {}
        for key in vars(self):
            dict_self[self._underscores_to_camel_case(key)] = getattr(self, key)
        return dict_self

    def from_dict(self, data):
        accepted_fields = self.accepted_fields()
        for key in data:
            if key in accepted_fields:
                setattr(self, self._camel_case_to_underscores(key), data[key])

class Broadcast(BaseSocialObject):
    '''Defines a social media broadcast message for the broadcast api'''

    # Constants for remote content type
    COS_LP = "coslp"
    COS_BLOG = "cosblog"
    LEGACY_LP = "cmslp"
    LEGACY_BLOG = "cmsblog"

    def __init__(self, broadcast_data):
        self.data_parse(broadcast_data)

    def accepted_fields(self):
        return [
            'broadcastGuid',
            'campaignGuid',
            'channel',
            'channelGuid',
            'clicks',
            'clientTag',
            'content',
            'createdAt',
            'createdBy',
            'finishedAt',
            'groupGuid',
            'interactions',
            'interactionCounts',
            'linkGuid',
            'message',
            'messageUrl',
            'portalId',
            'remoteContentId',
            'remoteContentType',
            'status',
            'triggerAt',
            'updatedBy'
        ]

    def data_parse(self, broadcast_data):
        self.from_dict(broadcast_data)

class Channel(BaseSocialObject):
    '''Defines the social media channel for the broadcast api'''

    def __init__(self, channel_data):
        self.data_parse(channel_data)

    def accepted_fields(self):
        return ['channelGuid', 'accountGuid', 'account',
            'type', 'name', 'dataMap', 'createdAt', 'settings']

    def data_parse(self, channel_data):
        self.from_dict(channel_data)

class BroadcastClient(BaseClient):
    '''Broadcast API to manage messages published to social networks'''

    def _get_path(self, method):
        return 'broadcast/v%s/%s' % (HUBSPOT_BROADCAST_API_VERSION, method)

    def get_broadcast(self, broadcast_guid, **kwargs):
        '''
        Get a specific broadcast by guid
        '''
        params = kwargs
        broadcast = self._call('broadcasts/%s' % broadcast_guid,
            params=params, content_type='application/json')
        return Broadcast(broadcast)

    def get_broadcasts(self, type="", page=None,
            remote_content_id=None, limit=None, **kwargs):
        '''
        Get all broadcasts, with optional paging and limits.
        Type filter can be 'scheduled', 'published' or 'failed'
        '''
        if remote_content_id:
            return self.get_broadcasts_by_remote(remote_content_id)

        params = {'type': type}
        if page:
            params['page'] = page

        params.update(kwargs)

        result = self._call('broadcasts', params=params,
            content_type='application/json')
        broadcasts = [Broadcast(b) for b in result]

        if limit:
            return broadcasts[:limit]
        return broadcasts

    def create_broadcast(self, broadcast):
        if not isinstance(broadcast, dict):
            return self._call('broadcasts', data=broadcast.to_dict(),
                method='POST', content_type='application/json')
        else:
            return self._call('broadcasts', data=broadcast,
                method='POST', content_type='application/json')

    def cancel_broadcast(self, broadcast_guid):
        '''
        Cancel a broadcast specified by guid
        '''
        subpath = 'broadcasts/%s/update' % broadcast_guid
        broadcast = {'status': 'CANCELED'}
        bcast_dict = self._call(subpath, method='POST', data=broadcast,
            content_type='application/json')
        return bcast_dict

    def get_channel(self, channel_guid):
        channel = self._call('channels/%s' % channel_guid,
            content_type='application/json')
        return Channel(channel)

    def get_channels(self, current=True, publish_only=False, settings=False):
        """
            if "current" is false it will return all channels that a user
            has published to in the past.

            if publish_only is set to true, then return only the channels
            that are publishable.

            if settings is true, the API will make extra queries to return
            the settings for each channel.
        """
        if publish_only:
            if current:
                endpoint = 'channels/setting/publish/current'
            else:
                endpoint = 'channels/setting/publish'
        else:
            if current:
                endpoint = 'channels/current'
            else:
                endpoint = 'channels'

        result = self._call(endpoint, content_type='application/json', params=dict(settings=settings))
        return [Channel(c) for c in result]

redpajama_set_name: RedPajamaGithub


Great location for new business! Cleared flat lot on cul de sac with curb strips. Public water and sewer, all utilities available. Adjacent to Buffalo Wild Wings and Goo Goo car wash. High traffic area ideal for restaurant, motels, office, etc.


text:
his-kyoto
Jermichael Finley: Packers should be prepared to switch to Jordan Love by Week Four
September 28, 2021 September 25, 2021 his-kyotocom
It's way too early for the Packers to panic. Former Packers tight end Jermichael Finley believes that panic could soon be justified.
Finley has been candid in the aftermath of Green Bay's embarrassing performance against the Saints regarding quarterback Aaron Rodgers.
Previously, Finley said that Rodgers "looks tired," and that it appears he has lost his "hunger" to play. Now, Finley says the Packers should be ready to pivot to Plan B, if/when it appears that Rodgers has lost it.
"If you don't see it, let's go to [Jordan] Love," Finley said.
Finley said that Rodgers should get four weeks to show that he still has it. If he doesn't, then go with Love.
"You've got one of the highest-paid backup quarterbacks sitting right behind Aaron Rodgers, so you might as well utilize him and get him going for the future," Finley said.
This presumes a lot. There's no reason to think the Packers won't turn things around on Monday night, when their intra-division punching bags known as the Detroit Lions return to town.
Now, if the Packers look in two days the way they looked six days ago, Finley may be onto something. The chances of that happening, however, seem very slim.
That said, the Packers follow their non-bye bye against the Lions with a trip to California to face the 49ers and a visit from the Steelers. At this point, a 1-3 start for a team with consecutive 13-3 seasons isn't out of the question.
Is the placebo effect just a myth?
Monkey briefly on the loose in Indiana hotel parking lot
Eagles sign Josh Sweat to three-year extension
Drug combination shows promise in treatment-resistant advanced ovarian cancer
Lagardere share price jumps 20% after Vivendi increases stake
YouTube is letting Premium subscribers experiment with downloading videos on desktop
N.C. State basketball coach helps football fan find lost wedding ring


Can Liposuction improve your health | Matthew Schulman, M.D.
Can Liposuction improve your health? High volume liposuction can eliminate health problems associated with obesity.
The common thinking among plastic surgeons is that Liposuction is for body shaping, not weight loss. This is definitely true. The best candidates are people who are not particularly overweight, but have "problem areas" that they would like corrected. Perhaps this is the outer hips ("saddlebags"), flanks, abdomen, or inner thighs. Liposuction is a great way to remove some fat, and improve the shape of your body.
But what about those people who are overweight? Will liposuction do anything? Certainly. Liposuction is designed to remove fat. If you are overweight, liposuction can remove a certain volume of fat, and can reduce your overall weight. The thing to remember is that even with significant weight reduction via liposuction, there may not be significant improvement in body shape. But there are other good consequences. The reduction in weight can reduce the risk for medical problems associated with obesity, such as high blood pressure, heart disease, arthritis, and diabetes.
When liposuction is done under close monitoring by a board certified plastic surgeon working directly with a board certified anesthesiologist, up to 5 liters of fat can be removed safely in a single operation. This is the equivalent of 12 pounds. That is a lot of weight, and is lost in a matter of a few hours. Think how long it would take you to lose 12 pounds through diet and exercise. This procedure can even be repeated at a later date to remove another 12 pounds.
I have had several overweight patients who underwent high volume liposuction. They lost significant amount of weight and this fat removal from their body helped to improve their overall health. In fact, several of these patients were diabetic before the surgery and were able to stop all diabetes medications within a few weeks after the liposuction procedure. There is scientific evidence that is surfacing that talks about high volume liposuction as a treatment for type 2 diabetes in overweight patients. As a bonus, all of my patients looked much better after the surgery too!
If you are overweight, liposuction may be a procedure for you. While it will not remove the excess skin, and in some cases, it may make the excess hanging skin worse, it may be a way to improve your overall health. You need to understand that obesity, high blood pressure, and diabetes place you at a slightly higher risk of peri-operative complications, so it is imperative that you consult with with a board certified plastic surgeon.
To learn more about Liposuction with Top New York Plastic Surgeon, Dr. Matthew Schulman, call 212-289-1851 to schedule your free consultation.


text:
POMPEY TO GO FAR
COLOMBIAN John Viafara has pledged to turn Portsmouth into Champions League contenders.
The #1.2m midfielder insists he can help Pompey emulate the success of his former club Once Caldas.
Last year, the Colombians won the Copa Libertadores, South America's Champions League - and Viafara scored the decisive penalty in the final shoot-out against Boca Juniors.
Viafara, who featured for Colombia in the 3-2 friendly defeat against England in the United States, said: "Who knew anything about Once Caldas two years ago?
"Why can't Portsmouth do the same? Joining them is a dream for me."


Below you will find the latest online channels for Bolivar vs Blooming in April 2019. These live streams are available for your pc, smartphone, tablet or mobile device.
You must register an active account to watch today's Club Bolívar tie from Estadio Hernando Siles .
Follow the links to view the Bolivar vs Blooming match and other future ties from Bolivian Primera División in 2019.
To add to the In-Play excitement, bet365 stream over 140,000 events live to your PC every year including Bolivar vs Blooming - so you can bet as the action unfolds. Highlights include Masters Series Tennis tournaments and matches from some of the top domestic Soccer leagues in the world and other Club Bolívar home games. To use the Live Streaming service to watch Bolivar vs Blooming you will need to be logged in and have a funded account or to have placed a bet in the last 24 hours subject to geo-restrictions and t&c. Any fixture/event on our website which has the Play or Video icon next to it is scheduled to be shown via Live Streaming.


It's a goodbye for Sport At 7, as Game On is introduced to 2fm listeners from next week.
Damien O'Meara: excited by new challenge.
RTÉ WILL AIR a new one-hour sports programme, called Game On, every weeknight from 7pm starting on Monday.
The 2fm show will be presented by Damien O'Meara, who previously anchored RTÉ Radio 1′s Sport At 7, which comes to an end.
"Game On marks a big departure for both RTÉ Sport and 2fm, and to be handed the reins of the show is both an honour and the source of great excitement," says O'Meara today.
The programme will be extended to a full three-hours on nights when there's live commentary, putting it up against Newstalk's hugely-popular Off The Ball.
Email "Game On? RTÉ to take on Off The Ball with new one-hour evening sports show".
Feedback on "Game On? RTÉ to take on Off The Ball with new one-hour evening sports show".


Here we are, already at ROUND 2 for the year! Now's the time for you to write up your goals post (on your own blog, remember) and link to it in the linky below.
As you're working on defining those goals, I want you to do something for me.
I am a Type A, overachieving slavedriver. I'm incredibly goal oriented and driven, and I get more done in a day than some people do in a week. Which is all fine and good to a point, but I so often forget about taking some downtime, about resting, about recovery from Doing All The Things. Because there's an inherent danger in this constant go go go go go.
These are realities of being a writer. Particularly a professional writer. We've got deadlines and fan expectations and all the business side of what we're doing. It becomes so easy to stop thinking about the creative side of the process and start focusing on the numbers. To start thinking of ourselves as some kind of machine that can just crank stuff out at a certain pace, consistently.
There have been a number of blog posts written lately from some extraordinarily talented writers who are caught in the grip of depression. Libba Bray. Myra McEntire. Our own Dawn Montgomery wrote a post last round about suffering from burnout (which is, I think, a precursor to depression in a lot of writers). And I know half a dozen others who've skirted this state or slid into it over the last couple of years.
It's something I suffered myself while on submission a few years ago and it paralyzed me. What I wrote was flat, emotionless, and unusable. I didn't get anything publishable put together for TWO YEARS, and when I did come back to it, it took three drafts to come anywhere close to the quality of what I'd produced before. I spent a lot of time beating myself up about it. I was falling behind schedule. The writing income was falling off. And I felt the fetid breath of the hounds of desperation nipping at my heels.
Ultimately, I made some changes and decided to be kinder to myself. I revised my long term goals, my expectations and allowed for something I have never willingly accepted. A time to rest and a time to fail. Failure, as it happens, is a necessary component of mastery. I could write a whole other post on that, but I'll save it for another time. My point is, I made the decision to be kinder to myself. To realize that for all that I've accomplished in the last ten years, I am not a machine. I'm not Super Woman. And that's okay. It's taken me most of the last year to heal from that, and I feel like I'm finally back on track, where I need to be. Now, if I need a day off, I take it. If I need to refill the well, I go read or watch something awesome. And then I come back to it. If I don't reach the number of words I shoot for in a day, I don't beat myself up over it. If I got ANY words down, that's still further than I was before. And THAT is why I'm so much NOT a fan of one size fits all goals.
So as you're pondering those goals and sorting out what you'd like to accomplish, remember to be kind to yourself and remember–you're only human.
When we're kinder to ourselves, we're happier.
When we're happier, we're kinder to others.
Just a heads up that the linky is taking us to a closed list. Thought you would want to know.
Kait, thank you for including this message. I am really needing to hear it right now. Juggling All The Things has become kind of a nightmare, and I'm coming to grips with needed changes (day job changes) so I can feel successful and happy. THANK YOU for sharing.
Wow… and I was all set to beat myself up today for behind. Now I don't have any excuses. Guess i'm gonna go put on some sweatpants and take a walk. Then I'm gonna take a super-hot shower. And then I'm gonna see if that changes my outlook on getting these here words out of my head and onto the paper. Thank you. For realz.
Having "been there" all too recently (and too often), I am grateful for this, Kait. For the post itself, for this links… and for you and all of the ROW80, for the place it gives me to live and write.
Eden has said everything I planned to say. Thank you, Kait!


As the doors have been closed on ENSO 2017/18 with it officially being a weak La Niña season, it's time to move onto ENSO 2018/19! Given how last year evolved, I thought I'd hold off on making a new ENSO thread for a bit. If you do not know what happened last year, 2016/17 was a very weak La Niña season (weaker than 2017/18) beginning in July 2016 and already gone by late 2016 with ENSO neutral occurring in early 2017. The waters started to warm up with some significant warmer than average SSTs developing in the eastern side of the equatorial Pacific in Spring 2017. It was looking increasingly likely that an El Niño was in the works for 2017/18. The SOI for June 2017 was very negative with an index of -10.4, the most negative the SOI was since April 2016 at this time. A negative SOI is reflective of El Niño whilst positive is La Niña. The models, especially the CFSv2, was pinpointing at a moderate to strong El Niño for 2017/18 as a result. There were even a good few ensembles going for a very strong El Niño, just 2 years after the previous one (a very strong El Niño has occurred in only 1877-78, 1982-83, 1997-98 and 2015-16). However, a dramatic turn around occurred in July 2017 with the SOI going positive at 8.1. Not to mention, the equatorial Pacific waters cooled down a lot from Spring and June. Every month from July to November 2017 had a positive SOI whilst December went negative meaning the La Niña that was going to take place wasn't going to be particularly strong but it was stronger at least than 2016/17 which had a pathetically weak La Niña. Now with that behind us, let's get onto 2018/19.
Here is the latest SST profile from NOAA for the 24th May 2018. I have highlighted the region we're interested in, the equatorial Pacific. The La Niña signature is well and truly gone with the warm waters overtaking it.
For the central part of the Pacific (ENSO region 3.4), the CFSv2 right now is depicting a weak El Niño for 2018/19.
However, if we look towards the eastern side of the Pacific (ENSO region 1+2), the CFSv2 is showing ENSO to be neutral for 2018/19.
When you have the origins of an El Niño occurring in the central part of the Pacific, it's called El Niño Modoki. This is a rare form of El Niño where the warming is focused on ENSO region 3.4 in the equatorial Pacific - usually you see warmings beginning in the eastern part of the Pacific.
Here's a diagram showing the ENSO regions.
El Niño Modoki events tend to be favourable historically for blocked Winters in Europe. The last El Niño Modoki event occurred in 2009-10. It has also occurred in 1979-80, 1986-87, 1990-91, 1991-92, 1992-93, 1994-95, 2002-03 and 2004-05.
If this El Niño Modoki outlook continues up to the latter part of this year, it will make for some very interesting discussion on the coming Winter. Anyway, let's not worry about that for now.
The warm subsurface temperature anomalies are still there in the Pacific so El Niño is quite likely at this rate. Will it be Modoki? We'll have to see.
Not much change in terms of the SST profile of the equatorial Pacific since the last update. The waters are slowly warming up.
The CFSv2 has upgraded the chances of a weak to borderline moderate El Nino for 2018/19. Though it shows some warming reaching the El Nino threshold at regions 1+2 at the end of the ensemble, it does not look like it will reach a total of 5 tri-monthly periods, not to mention, it's well out into the end of the year and the start of 2019. Therefore, the outlook for El Nino Modoki is still there.
The Jamstec is similar to the CFSv2.
The ECM is also similar amazingly enough.
El Nino is still on the cards but perhaps less of a Modoki event.
La Nina is well and truly over, no cold SSTs shown in the equatorial Pacific or ENSO regions though there is still quite cold pool just off the coast of southwestern South America. This cold pool is making it difficult for El Nino to take over the El Nino Southern Oscillation. The warm SSTs are more focused on ENSO region 3.4 which is the sign of El Nino Modoki whilst ENSO region 1+2 is on the rather cool side (though at this moment, not exceptionally so).
Let's compare to this time in 2009 when the last El Nino Modoki was recorded. 2009 did not have that cold pool near South America and the warming at that point was in the eastern equatorial Pacific. This clearly shows that there is plenty of time for changes.
The model shows ENSO regions 1+2 reaching El Nino status by the very end of 2018 but until then, ENSO neutral conditions. The El Nino status is in the unreliable timeframe.
The models shows ENSO region 3.4 reaching El Nino status through this month, July, and reaching moderate El Nino status by the end of 2018. Previously it was looking like a weak El Nino so this is new. This would fit 2009 nicely because not only was 2009 a moderate El Nino, it was also El Nino Modoki.
A new study shows there is some sort of connection between solar minimum and ENSO since the 1960s decade, see below. 2018-19 expected to have an El Nino will continue this "connection" or coincidence.
Sryan if an el nino did happen what influence would that have on our winter,would it mean cold and dry or wet and windy?
The problem with ENSO is that its effects on Europe (not just Ireland) are unknown and vary from year to year along with each strength of the different types of ENSO events.
However, I have found that strong El Ninos tend to be followed by cold Aprils. In fact, the top 6 strongest known El Ninos on record were followed by cold Aprils including 2015/16 - April 2016 was a cold month though this could also be partly blamed on the SSW event of March that year. Could be a coincidence more than anything for all we know.
- Weak El Ninos tend to be mixed for Winter. Some are cold, some are mild. Example of a cold weak El Nino is 1977-78. Example of a mild weak El Nino is 2006-07.
- Moderate El Ninos tend to be anticyclonic in Winter and if the block ends up in a good place for cold then it'd be cold. Example of a cold moderate El Nino is 2009-10. Example of a mild but anticyclonic moderate El Nino is 1991-92.
- Strong or very strong El Ninos tend to lead to zonal winters with mild and wet conditions on the whole. One such culmination was 2015-16 with that phenomenally mild December.
El Nino Modoki (you can see above on what this type of El Nino is) events (which is the expected El Nino event for 2018-19 currently) tend to favour anticyclonic and cold Winters with wind coming in from the east. 2009-10 was also an El Nino Modoki and in fact, this was the last El Nino Modoki event we had.
Historically, moderate El Ninos and weak La Ninas are your safest bets on a cold Winter for ENSO but there are deviations as with everything. ENSO neutral also can be good but there are far too many odd ones out in that case.
So in summary, the type of El Nino that is expected (at the moment) is a moderate modoki event for 2018-19 which can be a good combination for a cold Winter especially with low solar activity and other things.
What's also funny about 1994-95's El Nino was how it was El Nino Modoki instead of classic El Nino.
So right now, we're in ENSO neutral and times are passing on the chances of this becoming an El Nino event.
UK Met Office Glosea5 in its August 2018 update is depicting El Nino (fairly weak) for 2018-19 as well and specifically, El Nino Modoki (see posts above on explanation of the difference between a classic El Nino and El Nino Modoki).
During the super El Nino events of 1982-83, 1997-98 and 2015-16, we were already in El Nino threshold (0.5c or above) by March-April-May 1982, April-May-June 1997 and October-November-December 2014 respectively. The latest tri-monthly period (May-June-July) had an ENSO anomaly of 0.1c which is textbook ENSO neutral and it's getting very late for El Nino to take place especially to what the CFSv2 is depicting. ENSO can happen late like 1976-77's El Nino did not begin 'til August-September-October but you can't help but feel extremely dubious with what the CFSv2 shows.
As shown by the subsurface temperature anomaly profile below in the Equatorial Pacific, there is quite a bit of warm water waiting to be filtered up to the sea surface but recently at the end of July over towards the eastern subsurface equatorial Pacific, some cooler waters have started to appear. Will these enlarge and make El Nino near impossible to occur or will they promote the likelihood of El Nino Modoki? We'll have to wait and see.
We're awaiting on the ECM to update for August 2018 to see what it shows for ENSO this time around. Last month, it was going for a weak to moderate El Nino Modoki.
Last but not least, here's the latest sea surface temperature anomaly map from NOAA and the equatorial Pacific doesn't look particularly favourable for El Nino with a mix of cool and warm waters over the region. Quite a strange situation with these models depicting El Nino yet the signals from the Pacific itself don't look favourable for El Nino.
If you click the play button on this graphic which gives you a timelapse of global SSTs throughout the month of August, you can really see the Modoki developing - select "SST Anomaly" from the dropdown menu. You can essentially see cool eddies appearing in the East Pacific and slowly propagating westward, while the Central Pacific becomes exceptionally warm and the Western Pacific remains generally neutral to cool throughout.
I strongly suspect that this pattern is what's causing the previously low-forecasted Atlantic hurricane season to ramp up so much according to next week's model runs. You can see this with how many Eastern Pacific storms are surviving well into the Central Pacific and maintaining hurricane status while doing so, an unusual phenomenon - not good news for Hawaii going into Autumn I'd imagine. They had a very lucky escape with Lane.


Unless it's someone in your own guild, it shows the last time the player logged on. If they logged on 35 minutes ago and are still logged in, it will show 35 minutes. It doesn't reset while they're logged in.
Correct, we don't show online/locations for players outside of your guild. It used to show only the logged date but now it shows how long ago they last logged in, but it doesn't mean they aren't online.


text:
Web schwicky.net
The X-Files - 2.
2.16 Colony
US Airdate: February 10, 1995
writer: Chris Carter
story by: Chris Carter & David Duchovny
director: Nick Marck
David Duchovny as Special Agent Fox Mulder
Gillian Anderson as Special Agent Dana Scully
CIA Agent Ambrose Chapel ....... Tom Butler
Bill Mulder ................ Peter Donat
Dr. Landon Prince/Gregor's ..... Dana Gladstone
FBI Agent ...................... David L. Gordon
Field Doctor ................... Bonnie Hay
Federal Marshall ............... Tim Henry
Agent Barry Weiss .............. Andrew Johnston
Samantha Mulder ................ Megan Leitch
Military Policeman ............. Michael McDonald
First Jailer ................... Capper McIntyre
Asst. Director Walter S. Skinner ..... Mitch Pileggi
Mulder's Mother ................ Rebecca Toolan
Pilot .......................... Brian Thompson
MULDER AND SCULLY INVESTIGATE THE DEATHS OF THREE IDENTICAL STRANGERS -- AND A MYSTERY FROM MULDER'S OWN PAST COMES BACK TO SHOCK HIM.
In this first part of a two-part story, Mulder and Scully are alerted to a strange coincidence: the recent murders of three abortion clinic doctors, who have nothing in common save their identical resemblance to one another. Tracing the murders, they are caught up in a web of deception, facing off against an alien assassin who has the ability to change his appearance to resemble anyone at all. A CIA agent who shares Mulder's agenda fills them in on an old Soviet cloning project, with devastating implications. Mulder's own family is dragged into the mystery, as his father calls him home to meet a stranger who claims to be his sister. Scully is confronted by the alien assassin, who enters her room and bears a perfect resemblance to Mulder.
When the fake Mulder wakes Scully, the time is 11:21.
Ambrose Chapel, the CIA agent who speaks with Mulder and Scully shares his name with a church in the Alfred Hitchcock movie 'The Man Who Knew Too Much'. In the movie, the phrase Ambrose Chapel was incorrectly assumed to be the name of a person.
Personal note: If you want to understand anything about Mulder and his search for his sister, you must see these two episodes. We also meet the Alien Bounty Hunter here, and he becomes important in later seasons. These two stand the test of time and are on my must see again list. We finally see some real concern for each other between the two agents. Personally, I was on the verge of tears a few times in these two hours, call me sentimental.
(Mulder's opening monologue)
"I have lived with a fragile faith built on the ether of vague memories from an experience that I can neither prove nor explain.When I was twelve, my sister was taken from me, taken from our home by a force that I came to believe was extraterrestrial. This belief sustained me, fueling a quest for truths that were as elusive as the memory itself. To believe as passionately as I did was not without sacrifice, but I always accepted the risks...to my career, my reputation, my relationships ... to life itself ... What happened to me out on the ice has justified every belief. If I should die now, it would be with the certainty that my faith has been righteous. And if, through death, larger mysteries are revealed, I will have already learned the answer to the question that has driven me here ... that there is intelligent life in the universe other than our own... that they are here among us ...and they have begun to colonize."
Mulder: "Oh, hey. I've been looking for you."
Scully: "I was just down the street. Someone fired more shots at the White House last night."
Mulder: "You gotta wonder about a country where even the President has to worry about drive-by shootings."
Scully: "Triplets?"
Mulder: "Nope. I can't find any blood connection between them. In fact, I can't find any records on them at all. It's as if before they died, they never existed."
Scully: "What?"
Policeman: "Dr. Prince performed legal clinical abortions. He seems to be having his own share of occupational hazards these days."
Policeman: "We're hoping for an indictment."
Mulder: "Soon as you locate Dr. Prince's remains..."
Scully: "I've got a bad feeling about this case, Mulder."
Mulder: "What do you mean?"
Scully: "Well, nothing about it makes sense. We've got 3 deaths of identical victims, no bodies, a virtual non-suspect..."
Mulder: "Sounds just like an X-File..."
Mulder: "If somebody really wanted to set us up would they give us this little to go on?"
Mulder: "How would you access that?" (voice mail)
Receptionist: "Well, you'd start by paying the bill."
Dana's computer reads:
--Date-- Mail 485723 Image enclosed
To: D_Scully@FBI.gov
From: 82974@anon.server.ca
Dr. James Dickens, Washington
(after Mulder gets hit by a car)
Scully: "How are you feeling?"
Mulder: "Like I should have used the crosswalk. (picks up remains of his cell phone) A lot better than my phone."
Scully: "Our 'friend' from the CIA is about as unbelievable as his story. As is everything about this case. I mean, whatever happened to 'Trust No One,' Mulder?"
Mulder: "I changed it to 'Trust Everyone'. I didn't tell you?"
Mulder: (to Scully) "I think *you're* paranoid."
Samantha: "Is it too late for a game of Stratego?"
Mulder: "It's twenty-two years too late."
Meet the Schwickerath's
Calvin & Hobbes
Aïkido Journal
Urlaub-Europa
(C) 1998-2013,
Jean-Pierre Schwickerath
This Site used to be
Linux From Scratch! You will gain money by a fattening action.


Editor's Note: The AMI QT Devotionals from July 11-15 are provided by Tina Hsu, who is currently serving as a preaching-intern in East Asia. Tina is a graduate of Biola University and Talbot School of Theology (M.Div.).
One of the scribes came and heard them arguing, and recognizing that He had answered them well, asked Him, "What commandment is the foremost of all?"29 Jesus answered, "The foremost is, 'Hear, O Israel! The Lord our God is one Lord; 30and you shall love the Lord your God with all your heart, and with all your soul, and with all your mind, and with all your strength.' 31 The second is this, 'You shall love your neighbor as yourself.' There is no other commandment greater than these." 32The scribe said to Him, "Right, Teacher; You have truly stated that He is One, and there is no one else besides Him; 33 and to love Him with all the heart and with all the understanding and with all the strength, and to love one's neighbor as himself, is much more than all burnt offerings and sacrifices." 34 When Jesus saw that he had answered intelligently, He said to him, "You are not far from the kingdom of God." After that, no one would venture to ask Him any more questions.
I grew up with a pretty good record of being an obedient daughter towards my earthly father. I obeyed and listened to him, but I didn't obey because I loved him. I obeyed primarily because I was afraid of him—his temper and the idea of disappointing him. God has held my hand to overcome fear and to begin obeying my father because I love him. Even though my dad's response is not in my control, this change in my heart has helped me to overcome fear. This has also revealed to me how God wants me to relate to Him. He desires that I obey His word because I love and adore Him. God calls us to obey Him because we love Him, for love is the channel through which He relates to us and desires us to relate to Him. He loved us first by sending His beloved Son to defeat sin and death, so that we could be reconciled to Him. He whole-heartedly loved us and calls us to whole-heartedly love Him in a relationship.
Jesus affirms that loving God with our heart, mind, soul, and strength is the greatest commandment, next to loving our neighbor, because the love command is the preface for the whole Law. Every command and every prohibition that comes from God teaches and guides us how to express our love for God. He is not saying all other statutes are less important, or that we should pay less attention to other laws. But He is saying that the love command is the most important, because it sums up the spirit of the Law. For this reason, Jesus was pleased to hear the scribe's response, because he seems to have understood that God delights in obedience that comes from a person's love for him. On the contrary, he does not delight in acts of obedience that are empty of love.
There comes a point in our walk with God in which we have to ask ourselves, "Why do I obey and serve God?" May God work in you and me so that the reason we obey Him is because we love Him.
Prayer: Dear God, let my obedience and sacrifice for You be an overflow of my love for You and out of a joy of being Your servant. Thank You for loving me first, so that I could experience what true and unconditional love is. In Jesus' name. Amen.
Read John 14:19-24 After a little while the world will no longer see Me, but you will see Me; because I live, you will live also.20 In that day you will know that I am in My Father, and you in Me, and I in you.21 He who has My commandments and keeps them is the one who loves Me; and he who loves Me will be loved by My Father, and I will love him and will disclose Myself to him." 22 Judas (not Iscariot) said to Him, "Lord, what then has happened that You are going to disclose Yourself to us and not to the world?" 23 Jesus answered and said to him, "If anyone loves me, he will keep my word; and My Father will love him, and we will come to him and make our abode with him. 24 He who does not love me does not keep my words; and the word, which you hear is not mine, but the Father's who sent me.
What is Jesus preparing the disciples to face?
How should the disciples expect to see Jesus after He leaves them?
What does this passage teach you about discipleship? What does it mean that you are a disciple of Jesus?
Jesus is about to leave the world by dying on the cross, rising again, and ascending to be with the Father. Jesus is preparing the disciples to trust that they will still continue to see Him.
They will receive guidance and instruction from the Holy Spirit. They can expect to experience the love of God by pursuing a mutual love relationship with Him. Jesus calls them to express their love for Him by keeping His words.
Mark 12:32-33 "…He is One, and there is no one else besides Him…" Before we can offer God anything, we must remember and absorb the truth that He is One, and there is no one else besides Him. Reflect on an area in your life that competes with honoring God and spend time praying for your relationship with God to be renewed and restored.