Amino acids are biologically important organic compounds composed of amine and carboxylic acid functional groups, along with a side-chain specific to each amino acid. The key elements of an amino acid are carbon, hydrogen, oxygen, and nitrogen, though other elements are found in the side-chains of certain amino acids. About 500 amino acids are known and can be classified in many ways. They can be classified according to the core structural functional groups' locations as alpha, beta, gamma or delta amino acids; other categories relate to polarity, pH level, and side-chain group type (aliphatic, acyclic, aromatic, containing hydroxyl or sulfur, etc.). In the form of proteins, amino acids comprise the second-largest component (water is the largest) of human muscles, cells and other tissues. Outside proteins, amino acids perform critical roles in processes such as neurotransmitter transport and biosynthesis.


The Producers Guild of America announced on Tuesday the five nominees for documentary theatrical motion pictures for the 2017 Producers Guild Awards.
They include the acclaimed "The Eagle Huntress," which chronicled the life of a 13-year-old girl from Asia's Altai Mountains as she defied generations of tradition by learning to hunt with fierce golden eagles.
Also nominated is "O.J.: Made in America," ESPN's 464-minute documentary that was presented both as a documentary in theaters and as a five-part series on television. Times film critic Kenneth Turan described it as "the O.J. project to watch if you are watching only one."
The nominees also include the experimental documentary "Tower," which used animated reenactments to tell the story of the 1966 shootings at the University of Texas at Austin.
Rounding out the five nominees are "Life, Animated" and "Dancer."
The PGA nominations for television series/special, children's programs, long-form television, sports and digital series will be announced Jan. 5, and nominees for the film and animated-film categories will be revealed Jan. 10.
The 2017 PGA Awards will be presented Jan. 28 at the Beverly Hilton hotel in Los Angeles, with special honors to be given to Tom Rothman, James L. Brooks and Irwin Winkler, among others.


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St Martin's is proud to be a Henshaw Trust school.
Click the logo above for more information.
Have a look at our Shine Squad page.
Shine Squad is St Martin's Christian Ethos group.
Click on the logo or CLICK HERE to go to the page.
For God has not given us a spirit of fear and timidity, but of power, love, and self-discipline.
(2 Timothy 1:7 )
Wonderful are your works;
my soul knows it very well.
"If you are not willing to learn no one can help you.
If you are determined to learn, no one can stop you."
The one who gets wisdom loves life;
the one who cherishes understanding will soon prosper.
For even the Son of Man did not come to be served, but to serve...
For each will have to bear his own load.
- Galatians 6:5
Be kind and compassionate to one another, forgiving each other, just as Christ God forgave you.
— Ephesians 4:32
But let justice roll on like a river, righteousness like a never-failing stream!
Ephesians 4:25 Therefore, having put away falsehood, let each one of you speak the truth with his neighbour.
John 8:32 The truth will set you free
"God lives in the thankful heart."
Izaak Walton
"Do to others as you would have them do to you."
At St Martin's we are grateful when parents and children donate to our charity events.
But you don't need money to be generous.
We can be "generous of spirit" by offering our time and our kind words to others.
Our school is named after St Martin who gave his cloak to a beggar.
And when you stand praying, if you hold anything against anyone, forgive them, so that your Father in heaven may forgive you your sins.
Did you know that some of our pupils in Year 6 have S.P.O.T. Bands?
S.P.O.T. stands for Special Person On Trust.
This means they can be expected to make good choices, even when no one is watching them.
...so what is courage?
See what Emily in Year 5 says in her poem:
Courage is doing a really hard test.
Courage is swimming in the deep end, even if you're not the best.
Courage is going to war even if you might die.
Courage is where you don't like something, but you still try.
Courage is stepping up for yourself if you've had enough.
Courage is trying lots of new stuff.
Courage is doing an assembly in front of lots of people.
Courage is is doing a brave deed even if you might fall or tremble.
Courage is in all of us, can't you see?
Courage is in me!
Mayoral Visit to St. Martin's
On 17th October, the pupils and staff at our school made their own Pledge to Peace in the presence of the Mayor and Mayoress of Oldham.
In the morning assembly we learned about Jelly Babies and their connection to peace. Miss Glennie, told us how Jelly Babies were first sold in 1919 at the end of the First World War as "Peace Babies".
Peace Babies were made to represent a new beginning after the war. The many colours showed how different cultures and nations could live together with respect. They were only named Jelly Babies in 1953.
During the assembly prizes were presented to two children nominated by their peers and their teachers for demonstrating peaceful conduct.
Oldham's Mayor of Peace, Cllr. Derek Heffernan said the whole school should give itself "a pat on the back".
Cllr. Heffernan to us, "Today you are going to dedicate yourselves to working for peace in the world."
Following the assembly, the Mayor and Mayoress met with the Student Council and Mrs. Glennie for a photo-call.
At St Martin's we teach British Values including tolerance as well as teaching Christian Values such as peace. You can support us by promoting peaceful conduct an tolerance in our school and in our community.
Celebrating our Christian Value, Peace.

redpajama_set_name: RedPajamaCommonCrawl


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Open-access content Thursday 24th November 2016 — updated 5.50pm, Wednesday 29th April 2020
Colin Wilson on the review of the composition of Council and how it presents an opportunity to have a say in how it serves your profession
The announcement of the joint venture with the US Society of Actuaries has led to the creation of CAA Global, the new accrediting body for the Certified Actuarial Analyst examination, which will help the qualification reach its true global potential. We are now in dialogue with other actuarial associations around the world in the hope that they too will embrace this initiative and become destination homes for qualified Certified Actuarial Analysts. This and other recent events have crystallised in my mind the importance of working together as a profession, for the benefit of the whole profession.
As our refreshed strategy document published earlier this summer states, as a profession we are small, but through working together we are stronger. That has to be right, but it doesn't just relate to how we interact with our sister organisations and other professions around the world. It is also true for the IFoA and its members. As an organisation we will be stronger if we work together, and by working together we will remain relevant too - relevant not just to you, but to those who use your services now, or will do so in the future.
Of course, Council has a critical role to play here in setting the strategic direction for our profession and ensuring we fulfil our Royal Charter objective. So we need to be confident that this body operates as effectively as possible and is constituted appropriately to meet our objectives. This is not to say that Council is not working well already - I believe it is. And it is certainly a committed and passionate group of volunteers. But there are questions over whether Council is fully representative of the diversity of our profession, and succession planning is a recurring issue.
This is why I have instigated a review of the composition of Council. I believe that it is critical that it reflects the diversity of the membership, both now and in the future. Allied to this, it is important that the governing body attracts the right people with the right skills and experience to fulfil its responsibilities.
As a Council we have already started to think about these important issues - looking to the future and what this should mean for a modern, forward-thinking, progressive governing Council.
But Council does not have a monopoly on great ideas. This is, after all, your profession. That is why I want to hear from you about what you want from your governing body, and how it should lead the profession into the future to ensure our relevance and our long-term sustainability.
All too often in life, it is easy to stand on the sidelines and criticise decisions and actions that have been taken that you don't necessarily agree with. Now I'm not saying it's wrong to hold an opposing view. On the contrary, I welcome constructive challenge and aim to promote environments that encourage this, as I firmly believe that it leads to better decisions and outcomes.
So please don't stand on the sidelines; this is your opportunity to have a say in what is important to you and in what you believe Council can do for your profession. Ask yourself, should we continue to elect the whole of Council from a vote of the membership? Should the method of election be modified to help ensure that Council is truly representative of our members and where they work? Should Council look to appoint members to some seats around the Council table, or should these be given to lay people who could add a different perspective?
I do not underestimate the challenge that I have set myself and Council. I am also sure that there is no perfect answer; no silver bullet. But it is a challenge I believe we must rise to if we are to have a Council that is best placed to deal with whatever the future holds.
Many of you belong to regional societies. I encourage you to discuss these issues and feed back your thoughts to me. If you don't belong to a regional society, then please email me directly with your thoughts.
Colin Wilson is the president of the Institute and Faculty of Actuaries
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Actor Amrya Dastur, who had earlier learnt Mandarin for Kung Fu Yoga and is now learning Tamil for her next film, Santhanam-starrer Odi Odi Uzhaikkanum, says Tamil is a difficult language to learn.
Amyra Dastur made her Bollywood debut opposite Prateik Babbar in Issaq.
Amyra Dastur can vouch for the fact that for actors, it's certainly not easy to just resemble the characters they enact on-screen but also to sound like them. The actor, who was last seen in the Indo-Chinese film, Kung Fu Yoga, alongside Jackie Chan, Sonu Sood and Disha Patani, had to speak Tamil and get the dialect right for her next film, Odi Odi Uzhaikkanum, starring Santhanam.
"I've always been fascinated with different cultures and languages. I love to memorise words and phrases in different languages. I take it as a souvenir. Tamil is not an easy language to learn. It's the pronunciation that is the difficulty. Luckily, my South team use a lot of hand gestures when speaking to me, so I get the gist of what they're trying to say to me. I've specially asked them to speak to me in Tamil only so I can get more and more familiar with the language," shares Amyra.


The new X series are the new GPRS trackers (no DTMF feature) with new functions, such as, user can program (set up APN, port, interval, dial back tele#, etc.), control the tracker (oil cutting, sleeping mode, etc.) from our GPRS tracking plateform. User can also up date the new firmware any time from that plateform. The tracker current set up status list can also be viewed from there. Beside the program from the air functions, user can also set up the tracker from the computer with the USB cable or the SMS from the phone. The new " X " series can also send the real time position to the set up dila back telephone numbers with a www hyperlink. The user can check the real time location from the smartphone with a postion on the map. Inside the tracker, there is flashmemory which allow the tracker to keep recording the positions (as a data logger function) so that user can view the tracker whole trip even there is no GSM network coverage. With the inclusive Haicom Tracking software, user can view the history position data on the user friendly map. Also, with the build in motion sensor, user can setup the tracker to go sleeping mode activaly or passively to save the tracker power consumption.
HI-603X is a multi-functional and cost-efficient communication platform ideal for all AVL applications. It integrates most recent high sensitivity GPS chipset and quad-band (850/900/1800/1900) GSM communication module with powerful microcontroller into a compact/ lightweight size.
HI-603X is secured in a solid enclosure for simple installation without sweat and hassle. Nevertheless, not only it provides user with real time GPS location and accurate vehicle status all the time on server, but also it allows user to command vehicle remotely by one-click on computer. In a word, HI-603X accomplishes advanced implementation of convenient fleet management, enhanced vehicle safety, simultaneous emergency response, and merchandise/ package transportation, etc...Unlimited advantages are come out with HI-603X extensive applications.
Quad band 850/900/1800/1900 MHz GSM system External SiRF Star III high performance GPS receiver External GSM active antennas Advanced communication via SMS/TCP/UDP/HTTP.
Program, control and update firmware the tracker from the OTA (Over the Air) web tracking platform and also USB configurations.
Build in motion sensor for power saving Support emergency/ over speed/ motion/ power lost alarm Real time location report on preferred interval and vehicle status monitoring Premium functions: Fast GPS Fixed/ A-GPS/ Geo-Fencing/ Hands free Kit/ Anti-Theft/ Data Logger function 30,000 records or more Low battery alarm and battery voltage status Optional SOS button Minimum GPRS sending interval 5~10 seconds and interval adjustable.


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Galgenfrist ist ein Album der dänischen Funeral-Doom-Band Nortt.

Geschichte
Mit Galgenfrist veröffentlichte Nortt das dritte Studioalbum. Er nahm es wie frühere eigenen Angaben zufolge in Winternächten der Jahre 2006 und 2007 alleine in seinem Heimstudio Mournful Monument ohne Produzent oder Tontechniker mit einem 8-Spur-Tonbandgerät auf. Am Mastering beteiligte er Jeppe Hasseriis.

Albuminformationen

Galgenfrist ist das dritte Studioalbum der Band.

Umfang und Gestaltung
Das am 27. Dezember 2007 erstmals veröffentlichte Album enthält sieben Stücke mit einer Gesamtspielzeit von 47:09 Minuten. Die Gestaltung übernahm Philip Rauff Nielsen. Das Album wurde über Avantgarde Music als MC, CD und LP veröffentlicht und mehrmals wiederveröffentlicht.

Stil
Mit der Musik von Galgenfrist führte Nortt das musikalische Konzept eines radikalen Funeral Doom, aus monotoner Langsamkeit in Kombination mit Elementen des Black Metal und Dark Ambient, fort. Dies Grundgefüge entwickelte Nortt auf Galgenfrist jedoch in eine atmosphärischere und introspektivere Richtung mit einer stärkeren Betonung des Dark Ambient. "Drone-Soundkaskaden verschmelzen mit rituellem Doom der Marke Esoteric und mit sphärischen Synth-Klängen. Lediglich die hochgepitchten Gitarren - eher Teppiche als strukturierte Riffs" stünden noch in der musikalischen Tradition des Black Metal. Der Hintergrund der Musik ist "durch eine sonore Geräuschkulisse" geprägt. In Relation zu den vorausgegangenen Alben verkörpere Galgenfrist atmosphärisch ein Gefühl von Apathie und Leere.

Burns spricht Nortt zu, auf Galgenfrist die Grenzen der traditionellen westlich mathematischen Kompositionen unter dem Wunsch eines auf die Romantik zurückverweisenden emotionalen Ausdrucks zu sprengen.

Wahrnehmung

Das Album wurde international positiv angenommen. Wolf-Rüdiger Mühlmann schrieb in einer für das Rock Hard verfassten Rezension, es sei "[a]bgrundtief finster, hypnotisierend – und durchweg negativ". In einer für das Webzine Metal.de verfassten Besprechung wurde angeführt, dass Galgenfrist aufzeige, "wie perfekt intonierte Leere und Hoffnungslosigkeit zu klingen" habe. Unter der Einschränkung, dass die Dark-Ambient-Stücke, die Qualität des Albums beeinträchtigten lobte Klamerin Malamov in einer für Doom-Metal.com geschriebenen Beurteilung, als Meisterwerk von einer Qualität, die Resignation "einzigartig, unvergleichlich und herausragend" transportiere. Relativiert wurde das Lob lediglich in der Reduzierung der angesprochenen Zielgruppe auf einen kleinen Kreis potentieller Hörer. Die Musik erweise sich für diese Zielgruppe allerdings als "eine wahre spirituelle Reise".

Weblinks

Einzelnachweise

Album 2007
Album (Funeral Doom)
Album (Black Doom)
Nortt-Album

redpajama_set_name: RedPajamaWikipedia


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Home > Events > World Cups and Major Sporting Events: Futures and Legacies – deadline 30 June 2021
World Cups and Major Sporting Events: Futures and Legacies – deadline 30 June 2021
4 November, All day
World Cups and Major Sporting Events: Futures and Legacies Conference
Organisers: Dr Ben Litherland, Dr Claire McCalmey, David Smith and Dr Rob Light.
In October and November 2021, the United Kingdom will host the sixteenth Rugby League World Cup tournament, with 21 different nations participating in elite men's, women's, and wheelchair competitions. It will be one of the first major international sporting events to be staged since the global Covid-19 crisis and will play an important role in shaping the future of international sporting events. In collaboration with event organisers, this conference is being held alongside the World Cup to celebrate the power of sport to bring communities and nations together, to critically reflect on the impact of the pandemic on sport and society, and to consider the position of sporting events in a world that will continue to be shaped by pandemic. Since the first modern Olympics in 1896, major sporting events have played a pivotal role in the development of sport as a global phenomenon. These events have also shaped and been shaped by broader forces like wars, colonialism and its aftermaths, health crises and other significant historical events. They are some of the most watched media spectacles, creating sporting stars and icons and fond memories for fans and spectators. But there remain ongoing questions about the economic, social and environmental impact on local and national communities, and the responsibilities of providing meaningful benefits for host cities and countries.
This joint conference, hosted by the University of Huddersfield School of Music Humanities and Media, Heritage Quay and RL Cares, aims to bring together scholars from around the world to present their research on the futures and legacies of major international sporting events, their role in the wider sporting and cultural fields, and their relationship to wider politics and society. In particular, the conference provides an opportunity to examine how we interact and engage with sporting events and their environments in a world that is still experiencing the pandemic, and how this might influence future events.
The organisers invite proposals for 20-minute papers that include but are not limited to:
o Histories and legacies of world cups and major international sporting events, particularly in times of crisis and recovery.
o Future directions for sports and international event research during and after the pandemic, including the opportunities and responsibilities elite sport will play in rebuilding social, cultural, and economic life.
o Broadcasting, documenting, and recording events, focusing on archives, material culture, museums, and exhibitions.
o The relationship between international sporting events, sporting bodies and fans, spectators and local communities.
o The importance of accessibility and inclusivity in international sport, particularly in relation to the women's, wheelchair and physical disability world cup and sporting tournaments.
Proposals for three-paper panels are also welcome. Please send a 300-word abstract by 30th June to Dr Rob Light, r.f.light@hud.ac.uk
Image credit: Commonwealth Games Opening Ceremony Brisbane 1982, Creative Commons Attribution Share-Alike 4.0 License


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Sandy Pate Sports Bar What's On: £2 a pint during England group games!
There's plenty going on this week in the Sandy Pate Sports Bar, including £2 a pint on selected beer and cider during England's group games!
England are set to face Tunisia on Monday night in their first game of Group G.
Throughout the duration of these three games, Carling, Greene King IPA and Aspall Cyder will be available for just £2 a pint in the Sandy Pate Sports Bar!
Here's what else is happening in the bar this week...
MONDAY - Sweden and South Korea go head-to-head in the first World Cup clash of this week, kicking off at 1.00pm. England will be keeping a close eye on the 4.00pm kick-off between Belgium and Panama, which is the first game in Group G. Gareth Southgate's side then finally take to the field at the 2018 World Cup, facing Tunisia in their first game at the tournament (7.00pm kick-off).
TUESDAY - The World Cup action continues on Tuesday with Colombia facing Japan in the 1.00pm kick-off. Robert Lewandowski and Poland are the 4.00pm kick-off, before hosts Russia face Egypt at 7.00pm, following their 5-0 thrashing of Saudi Arabia.
WEDNESDAY - Cristiano Ronaldo will attempt to add to his three goals against Spain in Portugal's 1.00pm kick-off against Morocco. At 4.00pm, Saudi Arabia will attempt to find their feet at the tournament against Uruguay, having lost 5-0 in the opening fixture. In the 7.00pm kick-off, Iran take on 2010 winners Spain. We'll also have our weekly poker night which costs just £5 to buy in, and the winner could walk away with a tidy jackpot!
THURSDAY - Denmark and Australia kick-off the Thursday football at 1.00pm. France face Peru at 4.00pm, following their 2-1 win over Australia. Argentina will attempt to better their 1-1 draw with Iceland, as they face Croatia at 7.00pm in what promises to be an exciting contest. We'll also have our quiz night, where participants can put their knowledge to the test on a range of categories. There will also be our card bingo game with a prize of £537 up for grabs!
FRIDAY - Brazil are back in action on Friday, as they face Costa Rica at 1.00pm. Other games include Nigeria vs Iceland (4.00pm kick-off) and Serbia vs Switzerland (7.00pm kick-off).
SATURDAY - Saturday will bring another three games of World Cup football. The other teams in England's group, Belgium and Tunisia, face each other for a 1.00pm kick-off. Mexico will try to add to the three points they earned against holders Germany in the 4.00pm kick-off against South Korea. Germany will look to get their tournament up and running with their clash against Sweden at 7.00pm. Why not make a day of it and join us from 9.00am, when we will be serving delicious breakfasts!
SUNDAY - England are back in action for their second game of Group G against Panama at 1.00pm. The other Sunday matches are Japan vs Senegal (4.00pm kick-off), and the exciting affair between Poland and Colombia at 7.00pm. We'll also be serving our home-cooked Sunday roast from 12.00pm until 5.00pm.
We will be serving food every day in the Sandy Pate Sports Bar until 9.00pm, with the bar remaining open until late.
There is ample free parking for visitors to take advantage of in the main car park at One Call Stadium, situated outside the Sandy Pate Sports Bar to the rear of the Ian Greaves Stand.
For more information, or to book a table, please call the bar on 01623 482 482 (option 2).


The following information about Autosomal Dominant Polycystic Kidney Disease is gathered from public domain resources and is in no way medical advice. Please consult your nephrologist for all questions regarding ADPKD.
Download the free European Medical Journal ADPKD Pain Study and the latest version of the PKD Patient Handbook.
I have known for decades that I had Autosomal Dominant Polycystic Kidney Disease (ADPKD) that I had inherited it from my mother Millie. She died in childbirth from one of the side effects of polycycsic kidney disease - an hemorrhage from a brain aneurysm. She died an hour after I was born. My father was devasted by my mother's death and mourned silently the rest of his life. I recently read my mother's death certificate and realized that my parents had only been married for only a year before she died. I have no siblings.
Symptoms of my Autosomal Dominant Polycystic Kidney Disease did not begin appearing until I was 48 years old. I noticed that I woke up with a pain in my abdomen every morning. I thought that this was just a stomach ache, but after awhile I realized that the pain was from enlarged kidneys.
At at least one of my daughters (Monica) inherited ADPKD. Monica is already dealing with other multiple, unrelated disabilities.
In June 2013, Monica became ill from an infection in a hemorrhaging kidney cyst. Monica suffers from Autosomal Dominant Polycystic Kidney Disease, a genetic disease that she shares with her dad and grandmother. After the initial round of antibiotics did nothing, a second visit to the emergency room and a CT scan of her abdomen revealed the horrific condition. The test showed that a large mass including her left kidney had been developing. Surgery was scheduled for the following day. The large mass included her left kidney and part of the colon. The 4" x 10 " complex mass in her abdomen obliterated her left kidney and was removed.
Monica survived the nephrectomy and stayed in intensive care at Backus Hospital for almost 5 weeks. The staff treated her very well and helped her survival. Monica's parents stayed with her day and night since Monica is unable to express her needs and discomfort. We found that a rise in heart rate correlated with increasing pain, prompting a request for pain killers.
Recovery was excruciatingly slow and worrisome. For 2 weeks, she was unable to breath on her own so she was intubated. Fighting various infections was a constant battle. She was treated for sepsis, pneumonia, UTI, thrush, as well as the primary kidney infection.
She was able to usually sleep comfortably, waking up for a couple minutes occasionally to either play or try to take her IV out. The IV was used to provide nutrition, antibiotics and other medications. Bless her sweet heart, she did now know how sick she was.
We read to Monica or played her favorite music. A small raggedy anne doll was brought in to comfort her. Caregivers from Easter Seals visited her almost daily. Her sister Martha flew in from Denver to spend a week with her while she was recuperating in the ICU.
After 3 weeks, the internal bleeding finally stopped. Vitals were getting closer to normal except for heart rate, which was about double normal. Remaining kidney was doing fine. She still had an NG tube, respiratory tube and IV in neck.
Slowly they introduced small amounts of food in her stomach to see how her digestive system is working. She had 30 pounds of edema!
After a month of intensive care, Monica was ready to come home. She is now doing fine and her remaining kidney is working normally. She takes meals via a G-tube because her swallow mechanism never quite got back to normal (because of the prelonged use of the respiratory tube while in the hospital), despite months of speech therapy.
Almost every year, Monica, her sister Martha Price and I volunteer or contribute to the national PKD Walk Fundraiser for research - PKDcure. in association with the PKD Foundation.
PKD affects more than 12.5 million individuals worldwide, making it the most common life-threatening genetic disease. There are more persons with PKD than the combined numbers of those with cystic fibrosis, Down's syndrome, hemophilia, muscular dystrophy, and sickle cell anemia.
The advances made in Polycystic Kidney Disease (PKD) research in the past few year have been spectacular and are laying the foundation for clinical trials of possible treatments in the near future.
PKD advocates express their concern that research funding levels are inadequate and threaten the momentum generated by the promising research progress of the last year. They discussed the need for funding commensurate with the impact of the disease that afflicts approximately 600,000 Americans and costs the federal government $1.5 billion a year.
In addition, the advocates pointed out that PKD affects 20 times the number of people as cystic fibrosis, yet receives only one-tenth the federal funding. It also receives less funding than Lyme disease, a condition that is not fatal and affects 45 times fewer people.
ADPKD equally affects men and women, regardless of age, race, or ethnic origin. It typically expresses itself in adults when they are in the prime of life. ARPKD strikes children at birth and is often fatal.
In the 18th and 19th Centuries, PKD was often given the label of Bright's disease. This term encompassed any of several kidney diseases marked by high concentrations of protein in the urine.
Autosomal Dominant Polycystic Kidney Disease is a disorder in which many clusters of cysts develop primarily within your kidneys. Cysts are noncancerous (benign), round sacs that contain water-like fluid. They vary in size from tiny sacs to sacs large enough to hold several quarts of fluid.
How do the cysts grow? Cysts grow because there is a defective gene which causes abnomal growth of the epithelial cells in the kidney nephrons (filtering units inside the kidney) until a cyst develops .The cysts eventually separate from the nephrons and continue to enlarge. Over time healthy kidney tissue is replaced by cysts and we lose kidney function.
The rate of cell growth and cyst formation can be affected by several factors but it has not yet been proven that caffeine is responsible for cyst growth in humans .A study (2001) in rats, where the rats were given the equivalent of 9 cups of coffee a day, suggested that caffiene might make cysts grow. A later study in humans (2012) found no correlation between caffeine and cyst growth although it recommended further human studies studies. Caffeine does has a negative effect on BP which can affect chemical pathways leading to kidney damage and that is the primary reason why we should not drink too much of it .
The genetic mutation can cause abnormal growth in the epithelial cells and cyst development in other organs like the liver, pancreas, spleen, testes and ovaries. It is possible to have ovarian cysts that are unrelated to our PKD, either due to an hormone imalance or due to a different gentic condition called polycystic ovarian syndrome (PCOS).
Having a benign kidney cyst is common. At least half the Americans older than age 50 have one or more kidney cysts. A benign, simple kidney cyst doesn't require treatment. And having one or more kidney cysts doesn't mean you have Autosomal Dominant Polycystic Kidney Disease.
Autosomal Dominant Polycystic Kidney Disease isn't limited to your kidneys, although the kidneys usually are the most severely affected organs. The disease can cause cysts to develop in your liver, pancreas, membranes that surround your brain and central nervous system, and seminal vesicles. Other complications may occur as well.
The greatest risk for people with Autosomal Dominant Polycystic Kidney Disease is high blood pressure (hypertension) that develops as a result of their kidney disease. Complications from hypertension are the leading cause of death in people with this disease. Kidney failure also is common with Autosomal Dominant Polycystic Kidney Disease.
Autosomal Dominant Polycystic Kidney Disease affects between one in 400 and one in 1,000 people born in the United States. About 600,000 people in the United States have polycystic kidney disease. Abnormal genes are the cause, and there's no way of preventing or reversing the development of kidney cysts in affected people. But the disease varies greatly in its severity, and some complications are preventable. Regular checkups can lead to treatments to reduce damage to your kidneys from complications such as high blood pressure.
Autosomal Dominant Polycystic Kidney Disease (ADPKD). Signs and symptoms of this form often develop between the ages of 30 and 40. In the past, this type was called adult Autosomal Dominant Polycystic Kidney Disease. The illness does manifest itself in children in a small number of cases. Only one parent needs to have the disease in order for it to pass along to the children. If one parent has ADPKD, each child has a 50 percent chance of getting the disease. This form accounts for about 90 percent of cases of Autosomal Dominant Polycystic Kidney Disease.
Autosomal recessive polycystic kidney disease (ARPKD). This form is far less common than ADPKD, occurring only in one in 10,000 people in the United States. The signs and symptoms often appear shortly after birth. Sometimes, symptoms don't appear until later in childhood or during adolescence. Both parents must have abnormal genes to pass on this form of the disease. If both parents carry the genes for this disorder, each child has a 25 percent chance of getting the disease. Research has identified at least three genes that may be responsible for ADPKD and one gene that may be responsible for ARPKD.
About one person in 10 with polycystic kidney disease has no family members with the disorder. In these cases, instead of inheriting a defective gene, the gene spontaneously mutates, causing polycystic kidney disease.
It's not uncommon for people to have Autosomal Dominant Polycystic Kidney Disease for years without developing signs or symptoms and without knowing they have the disease. If you exhibit some of the signs and symptoms of Autosomal Dominant Polycystic Kidney Disease, which include high blood pressure, an increase in the size of your abdomen, blood in your urine, back or side pain, or kidney stones, see your doctor to determine what might be causing them. If you have a first-degree relative — parent, sibling or child — with ADPKD, see your doctor to discuss the pros and cons of screening for this disorder.
Several diagnostic methods are available to detect the size and number of cysts as well as to evaluate the amount of healthy kidney tissue.
Ultrasound examination. This is the most common diagnostic method. A wand-like device called a transducer is placed on your body. It emits inaudible sound waves that are reflected back to the transducer — like sonar. A computer translates those reflected sound waves into a moving image of your kidneys.
Computerized tomography (CT) scan. As you lie on a movable table, you're guided into a big doughnut-shaped device that passes very thin X-ray beams through your body. Your doctor is able to see your kidneys in two-dimensional "slices."
Magnetic resonance imaging (MRI) scan. As you lie inside a large cylinder, magnetic fields and radio waves generate cross-sectional views of your kidneys.
Genetic testing. Gene linkage analysis involves special blood tests for you and for at least three family members who are known either to have or not to have Autosomal Dominant Polycystic Kidney Disease. Then, the results of your blood tests are compared with the results of the tests of your family members. Because this test is so expensive, it's often only used when one family member is considering donating a kidney to another family member.
High blood pressure is a common complication of ADPKD. As many as 70 percent of people with ADPKD develop high blood pressure. Untreated, high blood pressure can cause further damage to your kidneys and increase your risk of heart disease and stroke.
A serious complication of Autosomal Dominant Polycystic Kidney Disease is progressive loss of kidney function. About 50 percent of people with the disease will have kidney failure by age 60, and about 60 percent will have kidney failure by age 70.
Autosomal Dominant Polycystic Kidney Disease causes your kidneys to gradually lose their ability to eliminate wastes from your blood and maintain your body's balance of fluids and chemicals. As the cysts enlarge, they produce pressure and promote scarring in the normal, unaffected areas of your kidneys. These effects promote high blood pressure and interfere with the ability of your kidneys to keep wastes from building to toxic levels, a condition called uremia.
Kidney failure and subsequent uremia can cause itching, nausea and loss of appetite. Left untreated, this toxic buildup of waste products and fluid can lead to congestion of your lungs, coma or even death.
The disease may also result in your kidneys producing less erythropoietin — a growth factor for red blood cells. Less erythropoietin results in decreased bone marrow production of red blood cells. Fewer red blood cells (anemia) mean you have less iron-rich hemoglobin to carry oxygen from your lungs through your bloodstream to the rest of your body.
As the disease worsens, end-stage kidney (renal) failure may result. When end-stage renal failure occurs, you'll need ongoing kidney dialysis or a transplant to prolong your life.
Most women — up to 80 percent — with Autosomal Dominant Polycystic Kidney Disease can have an uneventful pregnancy. Others may develop a life-threatening disorder called pre-eclampsia. Those most at risk are women who have high blood pressure before they become pregnant.
Growth of cysts in your liver. The likelihood of developing liver cysts for someone with ADPKD increases with age. About two-thirds of people with ADPKD will develop liver cysts at some point in their lifetime. While both men and women develop cysts, women often develop larger cysts and get them at an earlier age. Cyst growth appears to be aided by female hormones. They're more frequent in women who've been pregnant.
Development of an aneurysm in your brain. Localized enlargement of an artery in your brain can cause a hemorrhage if it ruptures. Aneurysms tend to run in families and are present in between 5 percent and 10 percent of people with ADPKD.
Heart valve abnormalities, such as aortic regurgitation.
Hernias, and pouches or sacs in the wall of the colon (diverticulosis).
High blood pressure. Controlling high blood pressure may delay the progression of the disease and slow further kidney damage. Combining a low-sodium, low-fat diet that's moderate in protein and calorie content along with eliminating smoking, increasing exercise and reducing stress may help control high blood pressure. However, you usually also need medications to control it.
Pain. Chronic pain, usually located in your back or your side, is a common symptom of Autosomal Dominant Polycystic Kidney Disease. Often, the pain is mild and you can control it with over-the-counter medications containing acetaminophen. For some people, however, the pain is more severe and constant. In some cases, your doctor may recommend surgery to remove cysts if they're large enough to cause pressure and pain.
Bladder or kidney infections. Prompt treatment of infections with antibiotics is necessary to prevent kidney damage.
Blood in the urine. You'll need to drink lots of fluids as soon as blood in the urine is noted, in order to dilute the urine. Dilution may help prevent obstructive clots from forming in the urine. Bed rest also may help decrease the bleeding.
Complications of cysts. Rarely, when kidney cysts cause chronic, severe pain or obstruction of other organs or veins, you may need to undergo surgery to drain the cysts.
Kidney failure. If your kidneys lose their ability to remove wastes and extra fluids from your blood, you'll eventually either need dialysis or a kidney transplant.
Liver cysts. Nonsurgical management of liver cysts includes avoidance of hormone replacement therapy. Other options in rare cases include drainage of cysts if they're not too numerous, partial removal of the liver, or even liver transplantation.
Aneurysms. If you have ADPKD and a family history of ruptured intracranial aneurysms, your doctor may recommend regular screening for aneurysms. If an aneurysm is discovered, surgical clipping of the aneurysm to reduce the risk of bleeding may be an option, depending on its size. Nonsurgical treatment of small aneurysms may involve controlling high blood pressure and elevated plasma levels of lipids — fats and fat-like substances — and smoking cessation.
As is the case with other chronic illnesses, having Autosomal Dominant Polycystic Kidney Disease may make you feel overwhelmed or helpless. But you don't have to deal with your illness alone. The support of friends and family is important in dealing with a chronic illness. In addition, a counselor, a psychologist, a psychiatrist or a clergy member may be able to help.
You may also want to consider joining a support group. Although support groups aren't for everyone, they can provide helpful information about treatments and coping. And being with people who understand what you're going through may make you feel less alone. Ask your doctor about support groups in your community.
If you use Facebook, check out the Cysterhood,The Fight to Stop PKD and Living with PKD for information and social interaction with others with Polycystic Kidney Disease.
Dietary strategies play an important role in alleviating the symptoms of renal failure or "uremia" and may, in fact, slow the progression toward renal failure as well. In light of this information, it is important to understand how nutrients play a role in maintaining a healthy "internal milieu" in a patient with kidney disease.
There are six general classes of nutrients that are essential components of a healthy diet, whether or not one is nearing renal failure: Proteins, Fats, Minerals, Carbohydrates, Vitamins, Water.
These nutrients are needed by all people, but for those on renal diets, the requirements may need to be adjusted. It is important, then, to learn how specific nutrients interact with kidneys, cysts, blood pressure, etc., and then learn how to make the necessary adjustments for an individual situation.
Salty foods can increase the retention of fluid.Even a small reduction in the amount of salt can reduce the blood pressure to a considerable extent.Ideally,the daily sodium intake should be not be more than 24oomg/day limiting the salt intake to 6g/day. Keep far away from salty foods like pickles, salted eggs, bacon, vegetables that come in cans and sauces like ketchup and soy sauce. The canned foods like canned fruits and vegetables are loaded in salt so they should be restricted. Before eating canned foods, you should rinse or boil them to get rid of extra salt. Processed foods should be avoided in ADPKD and high blood pressure as they are high in salt, preservatives, and fats. Also, ready-to-eat foods should also be limited and even avoided.
Alkaline foods are good for ADPKD. Try to go vegetarian as much as possible.
The patients with ADPKD and high blood pressure should avoid drinking alcohol.On one side,alcohol can damage the artery giving rise to hardening and marrowing of the arteries.On the other hand, it can stimulate the growth of the renal cysts.
Avoid alcohol, strong tea, coffee and any caffeine-containing drinks because caffeine can promote the cell proliferation and secretion of cystic fluids. Caffeine products should be avoided in ADPKD and high blood pressure. It can narrow arteries to rise blood pressure further and also it can worsen the renal function.
Foods with high protein usually are not suggested for Autosomal Dominant Polycystic Kidney Disease patients. When our body breaks down protein, waste product called urea will be produced, which need our kidneys to discharge. Therefore, eating too much foods with high protein means increase renal burden. Generally speaking, 0.8 grams of protein per kilo of body weight is ok. However, as the basic of our body, protein is essential for our daily life, that is to say, ADPKD patients can not limit protein strictly. Under such a condition, high quality protein can be ingested to meet the daily demand of body. High quality protein is usually contained by lean meat, egg white and fish and so on.
Kidney is responsible for keeping balance of sodium, potassium, calcium and phosphorous. Due to ADPKD, the kidneys may lose its ability to work. Therefore, ADPKD patients suffer from electrolyte disturbance more easily.
Honey contains a lot of unsaturated fatty acid such as arachidonic acid, linolenic acid which have a lot of benefits for controlling blood pressure---dilating blood vessels, increasing elasticity of the blood vessels, blood purification, reducing blood viscosity and enhancing blood activity. What is more, honey can prevent the formation of lipid peroxide and deposition of cholesterol on the vessel wall so as to maintain smoothness of blood circulation and control high blood pressure.
Add lemon juice to the water. Some fruit or vegetable juice is also suitable for these patients. For example, cranberry juice and carrot juice, containing rich vitamin C, can help improve kidney function and increase the rate of kidney filtering creatinine and blood urea nitrogen, so as to lower high creatinine level and high BUN level.
Flax seed oil has also been show to help ease the symptoms of ADPKD. Flax oil is an excellent source of a group of fatty acids (omega-3-fatty acids) that are thought to have anti-hypertensive, lipid-lowering and anti-inflammatory effects. All of these benefits can help with Autosomal Dominant Polycystic Kidney Disease. Flax seed oil, which has been show to help ease ADPKD symptoms.
Red bell peppers are low in potassium and high in flavor, but that's not the only reason they're perfect for the renal diet. These tasty vegetables are also an excellent source of vitamin C and vitamin A, as well as vitamin B6, folic acid and fiber. Red bell peppers are good for you because they contain lycopene, an antioxidant that protects against certain cancers.
A cruciferous vegetable, cabbage is packed full of phytochemicals, chemical compounds in fruit or vegetables that break up free radicals before they can do damage. Many phytochemicals are also known to protect against and fight cancer, as well as foster cardiovascular health. Sulforaphane, a phytochemical in cruciferous vegetables, may prevent or stop cancer cell growth in lung, colon, breast, bladder, prostate and ovarian cancers. High in vitamin K, vitamin C and fiber, cabbage is also a good source of vitamin B6 and folic acid. Low in potassium and low in cost, it's an affordable addition to the kidney diet.
Another cruciferous vegetable, cauliflower is high in vitamin C and a good source of folate and fiber. It's also packed full of indoles, glucosinolates and thiocyanates — compounds that help the liver neutralize toxic substances that could damage cell membranes and DNA. Serve it raw as crudités with dip, add it to a salad or steam or boil it and season with spices such as turmeric, curry powder, pepper and herb seasonings. You can also make a nondairy white sauce, pour it over the cauliflower and bake until tender. You can pair cauliflower with pasta or even mash cauliflower as a dialysis diet replacement for mashed potatoes.
Garlic helps prevent plaque from forming on your teeth, lowers cholesterol and reduces inflammation. Buy it fresh, bottled, minced or powdered, and add it to meat, vegetable or pasta dishes. You can also roast a head of garlic and spread on bread. Garlic provides a delicious flavor and garlic powder is a great substitute for garlic salt in the dialysis diet.
Onion, a member of the Allium family and a basic flavoring in many cooked dishes, contains sulfur compounds which give it its pungent smell. But in addition to making you cry, onions are also rich in flavonoids, especially quercetin, a powerful antioxidant that works to reduce heart disease and protects against many cancers. Onions are low in potassium and a good source of chromium, a mineral that helps with carbohydrate, fat and protein metabolism. For people on a kidney diet looking to add more flavors to foods, try using a variety of onions including white, brown, red and others. Eat onions raw on burgers, sandwiches and in salads, cook them and use as a caramelized topping or fry them into onion rings. Include onions in recipes such as Italian Beef with Peppers and Onions.
These tangy, tasty berries are known to protect against bladder infections by preventing bacteria from sticking to the bladder wall. In a similar way, cranberries also protect the stomach from ulcer-causing bacteria and protect the lining of the gastrointestinal (GI) tract, promoting GI health. Cranberries have also been shown to protect against cancer and heart disease. Cranberry juice and cranberry sauce are the most frequently consumed cranberry products. You can also add dried cranberries to salads or have them as a snack.
Blueberries are high in antioxidant phytonutrients called anthocyanidins, which give them their blue color, and they are bursting with natural compounds that reduce inflammation. Blueberries are a good source of vitamin C; manganese, a compound that keeps your bones healthy; and fiber, and may also help protect the brain from some of the effects of aging. Antioxidants in blueberries and other berries have been shown to help slow bone breakdown in rats made to be low in estrogen. Buy blueberries fresh, frozen or dried, and try them in cereal, topped with whipped topping, in a fruit smoothie or bake blueberry muffins, blueberry cake as blueberry crisp or blueberry pie. You can also drink blueberry juice.
Raspberries contain a phytonutrient called ellagic acid which helps neutralize free radicals in the body to prevent cell damage. They also contain flavonoids called anthocyanins, antioxidants which give them their red color. An excellent source of manganese, vitamin C, fiber and folate, a B vitamin, raspberries may have properties that inhibit cancer cell growth and tumor formation. Add raspberries to cereal, puree and sweeten them to make a dessert sauce or add them to vinaigrette dressing. You can also drink raspberry punch.
Red grapes contain several flavonoids that give them their reddish color. Flavonoids help protect against heart disease by preventing oxidation and reducing the formation of blood clots. Resveratrol, a flavonoid found in grapes, may also stimulate production of nitric oxide which helps relax muscle cells in the blood vessels to increase blood flow. These flavonoids also provide protection against cancer and prevent inflammation. Phytochemicals in grapes, wine and grape juice have been extensively studied since the discovery that the French have much lower rates of heart disease despite a diet high in saturated fat. Buy grapes with red or purple skin since their anthocyanin content is higher. Freeze them to eat as a snack or to quench thirst for those on a fluid restriction for the dialysis diet. Add grapes to a fruit salad or chicken salad. You can also drink them as grape juice or grape punch.
Buy powdered, fresh or pasteurized egg whites. Make an omelet or egg white sandwich, add pasteurized egg whites to smoothies or shakes, make deviled egg snacks or add whites of hard boiled eggs to tuna salad or garden salad to add extra protein.
Fish provides high-quality protein and contains anti-inflammatory fats called omega-3s. The healthy fats in fish help fight diseases such as heart disease and cancer. Omega-3s also help lower low-density lipoprotein or LDL cholesterol, which is bad cholesterol, and raise high-density lipoprotein or HDL cholesterol, which is good cholesterol. The American Heart Association and American Diabetes Association recommend eating fish two or three times a week. Fish highest in omega-3s include albacore tuna, herring, mackerel, rainbow trout and salmon.
Olive oil is a great source of oleic acid, an anti-inflammatory fatty acid. The monounsaturated fat in olive oil protects against oxidation. Olive oil is rich in ployphenols and antioxidant compounds that prevent inflammation and oxidation. Studies show that populations that use large amounts of olive oil instead of other oils have lower rates of heart disease and cancer. Buy virgin or extra virgin olive oil because they are higher in antioxidants. Use olive oil to make salad dressing, in cooking, for dipping bread or for marinating vegetables.
Reduce stress in your life: Stress causes cysts to grow faster by elevating adrenaline.
Avoid inflammation: Anything that causes inflammation in the kidney can accelerate the growth of cysts in ADPKD.
Eat a (mostly) vegetarian diet high in antioxidants: Diets based on plant proteins help lower blood pressure and may help slow the growth of kidney cysts. Animal studies have demonstrated that a low protein diet helps slow the progression of ADPKD.
Eat high fiber carbohydrates: Recent studies have found that people with ADPKD and other kidney diseases may be mildly or acutely insulin resistant. High insulin levels are linked with heart disease and possibly faster rates of ADPKD progression.
Vitamins are an important source of energy and 13 have been established in essential dietary constituents for humans. Essential, in this setting, means that these compounds must be in the diet because the human body cannot manufacture them. These organic compounds are necessary for normal growth and play specific roles in maintaining health. For example, retinol (vitamin A) is required not only for growth and reproduction, but also vision. Some vitamins, such as C, A and E, will accumulate in patients with diminished or absent renal function. Check with your nephrologist FIRST!
Vitamin C intake should not exceed 150-200 mg/day in patients with renal insufficiency. Excess intake results in accumulation of ascorbic acid metabolites, such as oxalate, and can predispose patients to deposition of calcium oxalate complexes in vessels and tissues.
Vitamin A also accumulates in patients with renal dysfunction because the kidney is largely responsible for its catabolism (the process where an organism reconverts living material into simpler compounds). S well, binding proteins for vitamin A increase in renal failure, causing vitamin A to stay in the body for increasing periods of time. Excess vitamin A can lead to decreased blood counts and abnormalities in lipid and calcium metabolism.
Adequate daily fluid intake is a requirement for all patients and particularly patients with ADPKD. Very early in the course of the disease, ADPKD patients lose the ability to concentrate their urine. This means that the kidneys in patients with ADPKD cannot hold on to or reabsorb water as well as other individuals. This defect is mild; nonetheless, if ADPKD patients do not have access to liquids, particularly during heavy exercise of extreme heat, significant dehydration can occur.
The amount of fluid allowed in the diet of someone receiving dialysis depends on the amount of urine made and the type and frequency of dialysis employed. If one no longer urinates, then fluid intake is usually limited to one liter each day. The amount of fluid intake that is safe should be discussed with your nephrologist and your dialysis dietitian.
Maintaining a healthy weight is important for many reasons, including protection of kidney function. Excess body weight can increase blood pressure and may lead to hormonal changes (hyperinsulinemia) that could possibly accelerate the progression of ADPKD. However, losing too much weigh or not using a healthful manner in doing so can cause other, possibly unintended complications. Before you take on any weight loss program you should consult your doctor.
Good exercises for people with ADPKD include sports where impact is minimized. These include swimming, tennis, rowing, golf, biking and, to a lesser extent, jogging or running. Clearly one's desire for a certain sport needs to be considered when recommendations about choosing a sport are made.
In general, most sports do not affect kidney function. Specifically to ADPKD, there are some issues that need to be addressed with regard to the type of exercise. Given the unique nature of ADPKD, where kidneys are enlarged and cysts can easily rupture, there are some simple precautions to take. Contact sports where the kidneys may be traumatized (flank or lower-back impact) should either be avoided or protective pads should be worn. Examples of these types of sports include football, rugby, basketball, hockey and particularly boxing or kickboxing. Horseback riding and cross-country biking are other sports where repetitive impact has resulted in hematuria or blood in the urine, in individuals with ADPKD.
For those with ADPKD who are otherwise healthy, moderate alcohol intake is okay, but please limit to two drinks per day for men and one drink per day for women.
The ability to work can vary greatly between individuals. Much of it depends on what type of work a person does and how far their ADPKD has advanced. For example, individuals who have jobs which require moderate to strenuous physical work may find it more difficult to perform their tasks as their ADPKD advances. However, even those who have positions which require less or little physical activity may also experience challenges performing their job based on how their body is reacting to ADPKD. Much of it comes down to your ability to function affectively in your position while dealing with the changes occurring in your body.
Your choice to disclose to your employer that you have ADPKD could have an impact on your work. In some instances, it may help provide more support. In others, it could unfairly affect your career opportunities. The legal requirements in relation to disclosure vary. You should check with your state's Department of Labor to learn more about the situation in your area.
"What your doctor won't tell you about Polycystic Disease but you should really know" will help you to understand changes that you will go through and how to take care of yourself. This is a must-read for anyone diagnosed (or friends and family of someone with ADPKD).


What Can Pilates Do for Me?
Below you will find prices for private sessions as well as all our classes. And don't forget to check out our offers page, which we regularly update with any promotions we're running.
I have struggled with pain in my lower back and shoulders for many years and especially since having my two children. I started having 1:1 studio sessions with Nic and was amazed how much my movement improved, even after my first session. Nic is an amazing instructor, always encouraging and positive which has given me confidence in my own body strength and ability.
As a specialised rehabilitation-focused studio, we offer small group classes with experienced instructors, as well as private 1-to-1, or 2-to-1 sessions. Below you will find the prices for all our actives plus introductory discounts for new clients.
You can buy a class or private session block to use at your leisure, or if you're a regular visitor you can purchase a monthly Pilates Pass and pay from only £10 per class.
If you would like some help choosing a class or want to discuss your specific requirements then please email hello@soulpilates.co.uk and we will be happy to assist you.
If you are new to Soul, we have lots of new client offers to get you started on your movement journey.
With our focus on rehabilitation, we work closely alongside osteopaths, physiotherapists, doctors and consultants to bring you back to quality, functional movement.
We use Pilates alongside other movement and chronic-pain science to work with you and give you the strategies to recover from injury or surgery, prevent injuries, to move better and to feel better. We help you realise the potential of your body and move out of pain.
If you have been referred to us, or you are currently in pain, then you must attend a private session with us first. We will assess your posture, your movement, and any specific factors surrounding your injury or condition so we can provide you with a plan for your rehabilitation. Your plan can take the form of group classes, more private sessions or a mix of both.
Many clients also choose to come to a private session before joining group Equipment Classes as this provides a solid and supportive introduction to the machines.
All our prices include VAT.
Our pricing tables should be shown below, if they do not appear please click here and you will be taken directly onto our booking portal.
Our 30 minute session are perfect for those of you who maybe come to classes regularly and wish to work on a specific issue you need more personal assistance on.
Or if you might find 60 minutes a little too much and prefer a shorter session.
If you wish to share a private session with a friend or partner, then let one instructor guide both of you through the Pilates Equipment and your bodies, in a session tailored for each of your individual needs. You don't need both be at the same level!
We run small, specialised classes with a focus on functional, rehabilitative movement led by experienced instructors. With two studios, we run classes on the Pilates Equipment, Small Mat Classes, Barre Classes and Pre-Natal Classes.
All our classes can be booked and paid for online via a class block, or a monthly Pilates pass. You are not fixed to attending the same class every week and instead can work your Pilates schedule around your availability week-by-week.
At Soul Pilates we offer classes throughout the week from 10am to 8pm. All our lunchtime classes are 45 minutes long so you can get your Pilates fix during your lunch break and our evening classes are 60 minutes long.
Our Equipment Classes are small, specialised classes on the Pilates machines (reformer, trapeze table, ladder barrel, combination chair and spine corrector).
To join an Equipment Class you need either to have previous experience across all the equipment (not just reformers), have attended a 1-to-1 private session or completed one of our 6-week Beginner Equipment courses. Full details on joining our Equipment Classes can be found here.
Can't wait for the next course? Then purchase our nee client, New to Equipment Pilates" package and get your first 1-to-1 and four classes for only £99!
Our small, specialised Mat Classes focus on functional, rehabilitative movement to enhance your body and mind, easing the body from any discomfort.
These small classes are run by highly-experienced teachers who bring a passion for movement.
Our lunchtime classes are 45 minutes long, allowing you to fit some mindful movement into your lunch break and return to the office refreshed and relaxed for the afternoon ahead – no shower needed afterwards!
If you plan to attend mat classes only at lunchtime, you can take advantage of reduced class rates.
Since we opened the studio we've seen how many of you love coming to the studio 2, 3 or 4 times per week across our range of classes, so to provide you with the best value for money and to allow you to attend any class, we've put together a discounted monthly pass that is based on a 3 month commitment.
To give you such great rates we just ask that you give us at least 14 days notice to cancel following your three month period. We can pause your pass for a minimum of one full billing month should you be away for a period of time.
All our prices include VAT.
"I have struggled with pain in my lower back and shoulders for many years and especially since having my two children. I started having 1:1 studio sessions with Nic in May and was amazed how much my movement improved, even after my first session. Nic is an amazing instructor, always encouraging and positive which has given me confidence in my own body strength and ability."
"Loving Soul Pilates...totally changing my life. Posture improved and dropped a dress size in 4 weeks. I'm now officially addicted to Pilates and have withdrawal symptoms on Friday, Saturday & Sundays!"


text:
Q: Intellij can't find JDK/SDK Here are my environment variables for Java:
$ echo JAVA_HOME
JAVA_HOME
$ echo $JAVA_HOME
/usr/lib/jvm/java-8-openjdk-amd64
$ echo $JRE_HOME
/usr/lib/jvm/java-8-openjdk-amd64/jre/bin

When I click Create new project with IntelliJ, it says <NO_SDK> and it gives me a link to download JDK.
Here is a screen shot of the error:

I am using Linux for the first time, can you please help?

A: Here is how I solved it:
sudo add-apt-repository ppa:webupd8team/java
sudo apt-get update
sudo apt-get install oracle-java8-installer

redpajama_set_name: RedPajamaStackExchange


If you and your partner have had a few screaming matches about raising your children, you're in good company. According to a new study, a couple with children will argue about parenting related concerns an average of eight times per month.
The study commissioned by Care.com found the average parent will argue with their other half almost 100 times per year. They also discovered the most common reasons why parents fight. And, surprisingly, money only comes into play halfway down the list.
Join us as we count down the most common arguments parents have. And revel in the fact that you are not the only one frustrated by your partner's parenting opinions.
How and when it is appropriate to reward your child.
Letting your child sleep in your bed.
Immediately seeing to a crying baby vs leaving the infant to calm down before going in.
How much to spend on the kids at Christmas or birthdays.
Whether it is okay to let a baby cry or self-soothe.
Whether one parent is shouting or being too strict.
When one parent does not follow through on a threat or punishment.
When one parents says yes to a child after the other parent has already said no.
How to discipline your child.
It's not surprising that couples will have differing opinions when it comes to parenting. More than half of parents believe the reasons behind their disagreements stem from how they were raised, while 40 per cent said it was possibly because they find different things more important than others.
How many of these arguments have you and your partner had this month? We're 8 for 10 in our household!


text:
How Eko Innovation Centre, LASG rekindle hope for data-driven smart city at AOT Lagos 2.0
by Brand Press | Dec 10, 2020
In today's world the influence of data and technology for brands and the society at large cannot be overemphasized as deployment of artificial intelligence, artificial realities and many others play a key role in the society.
Many advanced countries are well known for their data management and usage of technology to make every of their operations from governance to business seamless. Remarkably, data-driven smart cities have raised the quality of life for people around the globe.
Achieving a smart city has been the dream of many African countries in order to meet up with the rest of the world. Nigeria has also been yielding to get data management right admits various challenges that still exist; however, Lagos is making a difference with its various initiatives that it is partnering with the Eko innovation Centre, a leading innovation hub in the country.
This year, Eko Innovation Centre in partnership with the Lagos State Government (LASG) through the office of Innovation and Technology organized the second edition of Art of Technology (AOT) Lagos 2.0. The two-day conference brought together the best of tech minds from across the globe physically and virtually on the 3rd and 4th of December 2020 which is in line with its Smart City agenda of making Lagos a 21st century economy.
L-R: Babajide Sanwo-Olu, governor, Lagos State; Olatunbosun Alake, special adviser to the Lagos State governor on innovation technology and Victor Gbenga Afolabi, Curator of Art of Technology Lagos and Founder of Eko Innovation Centre at the Art of Technology Lagos 2.0 and unveiling of "Startup Lagos" in Lagos recently.
The event was themed 'Smart Data, Smarter Lagos,' and the conversation centred on the fundamental role data plays in connecting the city with its citizens and how data can be collected, collated, analysed and deployed for governance, commerce and development. The event had the Vice president, Prof. Yemi Osinbajo; the Lagos Governor, Mr. Babajide Sanwo-Olu; Commissioners and others eminent stakeholders in the private and public sector.
According to the Curator of Art of Technology Lagos and Founder of Eko Innovation Centre, Mr. Victor Gbenga Afolabi, the two-day summit was building on the success of the maiden edition which held in 2019 which recorded over 2,000 participants in attendance. He noted that last year one of the key successes was the launch of the Lagos Innovation Masterplan which has continued to guide the Lagos state government in achieving its Smart City agenda.
Meanwhile, this year's edition recorded another milestone with the launch of Startup Lagos, an initiative meant to empower Lagosians. Also, it recorded a milestone of more than 8,000 participants. The conference featured about 60 industry speakers, 50 investors and 35 experience centres.
Speaking on the choice of the theme, Advisory Board Member, Eko Innovation Centre, Doja Ekeruche said "The choice of the theme, 'Smart Data, Smarter Lagos' is because we understand the value of data, identifying and leveraging the outcome of data and its insights to develop the most populous city in Nigeria and indeed in Africa.
"The concept was to build and strengthen the capacity of stakeholders to actively participate in the development of Lagos into a smart city and then chart a vision for the next phase of development in order to cement Lagos as Africa's key tech hub.
"We do understand that data is the bedrock to accurately plan for every single segment of the city including electricity, security, transport, healthcare, education, even the creation of jobs. Every single aspect of our living requires the use of data and today, the data exist but it has not be gathered, created and it has not been analysed in a way that it delivers insights in a way that enables decision makers to take the right decision that will transform the city.
"I think the most important thing is that the discussions we have had in the last two days, we are confident that we have raised again the importance of gathering of data to indeed to drive or enhance the potential of our demography and discover the potential of the city," she stated.
Meanwhile, the Vice president, Yemi Osibanjo speaking at the event, said this administration is working harder to make Nigeria a smart country, noting that human creativity and ingenuities are major assets needed to make the society a smart place.
He maintained that President Muhammadu Buhari's administration would continue to pursue its digital and technology agenda to keep the country at pace with the rest of the world. "Data is everything. We are gathering economic data, demographic data and other data coupled with our resourcefulness to manage over two million people. So, we must leverage technology and data for better plans, policy design and decisions making. Smart data makes smart governance," he stressed.
Similarly, the Lagos State Governor, Babajide Samwo-Olu on his part restated its commitment to make the state a smart city by digitalizing its operations which is in line with his THEMES agenda
He affirmed that the state is leaving no stone unturned to give residents sound governance, this he said, can be possible through data gathering, thus, the state has proposed to capture four million Lagosians to serve the people better. He said, "We need to know ourselves, so with data obtained from Lagos State Residents Registration Agency (LASRRA) and other agencies, we shall be able to plan well, take informed decisions and make better policies."
Correspondingly, Tunbosun Alake, Special Adviser, Innovation and Technology to the Governor of Lagos State stated that government is working tirelessly to make the state better and become the nation's technology hub, adding that data is necessary to seamlessly run the affairs of the state.
He explained that as a result of COVID-19, a lot of government operations including cabinet meetings are now being done on digital platform.
Noteworthy that AOT Lagos 2.0 had series of breakout sessions and subthemes where experts charted a new course for the state with topics such as Global Cybercrime and Data Protection: Building a Smart Lagos in the face of Security breaches; Lagos 2030: Data as vehicle into creating the future; Diversity and inclusion in tech creating and others.
Experts at the event proffered various solutions to achieve a data-driven smart city. Lisa Da Silva, Principal Investment officer, IFC and keynote speaker said smart city development start with a really strong vision and thoughtful planning and in this case, Lagos is way ahead of many states with the launch of a master plan last year December."
She added that one of the foundations for Smart City development is regulation, stating that the regulatory environment needs to enable digital solutions through regulation on data management and technology.
L-R: Babajide Sanwo-Olu, governor, Lagos State; Olatunbosun Alake, special adviser to the Lagos State governor on innovation technology; Victor Gbenga Afolabi, Curator of Art of Technology Lagos and Founder of Eko Innovation Centre and Demola Shogunle, chief executive, Stanbic IBTC Holdings at the Art of Technology Lagos 2.0 and unveiling of "Startup Lagos" in Lagos recently.
Ibukun Awosika, Chairman, Board of Directors, First Bank of Nigeria Limited on her part said states need to gather data because it is key to planning and with acquire data and government's implication of it programmes for citizens, jobs would be created and other basic amenities would be provider as well.
She pointed out that there is shortage of data scientists which the state needs to enable it's aim of smarter city become a reality.
Meanwhile, Dr Demola Sogunle, Chief Executive, Stanbic IBTC and keynote speaker, when he speaking on the topic "Solving Africa's digital payment challenges: The data Infrastructure" recommended that government needs to go into partnerships with private sectors in order to deliver the required payment infrastructure.
He also called for digitize common transactions, stating that identifying and deploying a fit-for-purpose payments infrastructure for the transport sector will significantly improve adoption of digital payments and drive financial inclusion.
In the same vein, he called for the incentivize use of digital channels for transactions. "Consider providing incentives such as cash back and discounts to encourage the use of digital channels."
In addition, he also called for investment in infrastructure, "Use of self service stalls, wifi coverage offerings for transactions, etc towards driving more people to adopt digital payments. Drive development of robust IT connectivity."
Lastly, he called for inclusion of IT and financial literacy programmes in the educational curriculum and also collaborates with financial institutions to track and prosecute fraudsters, and provides a framework for reimbursing victims to improve confidence in digital.
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Q: Can I remove some code from a LGPL library when using? I'm using a javascript UI library which is released under LGPL. Since it's very big, I want to remove some unused code to make it smaller.
Is this allowed?

A: Yes it is allowed as long as you don't remove any code from the LGPL license or the credentials.


Mephedrone, or "meow meow," is a relatively new, manufactured stimulating drug. Not much is known about its effects yet, but it's important to educate yourself and your teen on what has been discovered. Here are five things you may not know about mephedrone.
Mephedrone has only been widely used since 2009 and was manufactured to be a stimulant, therefore it has no natural properties. It also wasn't explicitly illegal in many countries until recently. For example, it was banned in the UK in 2010 and scheduled under the Controlled Substances Act in the United States in 2011.
Typically, mephedrone is found as a white powder that is snorted or swallowed. In some cases it is injected, although this is associated with higher risk of overdose. Mephedrone used to be concealed and sold as plant fertilizer although it does not actually act as one.
Mephedrone users report feelings of increased energy, self-confidence and speech. Other reported effects include feeling empathetic and close to others. When snorted, users have reported feeling its effects in a matter of minutes, while consuming it orally can take over 45 minutes for effects to take place.
As mephedrone is a relatively new substance, its long term effects and dangers are not known, making it important to exercise particular caution with this drug. Addiction and withdrawal symptoms are also unclear, although users have reported urges to take the drug again while experiencing negative effects as the drug's high fades.


My anxiety I found was shared by army personnel on the ground. My initial refutations were based on what I remembered, but after that initial publication, I thought I should do some more research. The army kindly gave me access to their records, and I was taken round some of the areas that figure in the Darusman Report, and was able to see that much of it was false.
The officer who took me round was much relieved to see me, for he was convinced that, after he moved to another job, a transfer that he thought was imminent, the records he guarded would be forgotten. Given that we have no institutional memory, that would have been the end of a trove of invaluable evidence, including the communications from the UN relating to Convoy 11, of which so much is made in the Darusman Report. These include a letter of thanks from the Head of UN Security for the excellent cooperation extended by the forces to the Convoy, a letter sent several days after the attacks by the army that Darusman alleges.
It transpired that all government had done after the publication of the Darusman Report was to send Gomin Dayasiri to tour the area. He had not produced any report on the visit, and I rather fear that, forceful opponent of LTTE terrorism as he is, he would not have been relieved to find, as I was, that the hospitals we were supposed to have deliberately shelled were largely undamaged, and indeed one of them had no signs of the shells that were supposed to have inflicted casualties within the hospital. On the contrary, Gomin would doubtless have wondered why our army had not been more aggressive in its approach.
I wrote in detail about the exculpatory evidence I had found, but it was only the Central Bank, which is doubtless more aware than most of the need to put our own case forward, that took multiple copies of the book for distribution. The Ministry of External Affairs was not interested, though the books proved most useful when I was asked by our mission in London to respond to Callum McRae of Channel 4 in a discussion at the Frontline Club. Fortunately the President also asked Arun Thambimuttu to attend, and despite a largely hostile audience, we got our message across.
But both of us are associated with government, so it is heartening that those with no such affiliations have also spent much time and effort in finding out the truth. The conclusions of the team that studied the evidence can be read in The Numbers Game, a detailed document that can be accessed on the Marga Website as well as through the writings of Michael Roberts, one of our first Rhodes Scholars who is now resident in Australia but maintains a keen interest in all things Sri Lankan. I gathered also from Michael's introduction at the last discussion at Marga that the leading light on the team is planning another report, dealing with Convoy 11.
I fear however that government is not interested in these efforts. Representatives of the armed forces were asked to the last presentation, but none appeared. Nor did the Sri Lankan journalists who had been asked, though Kath Noble, who writes more perceptively about Sri Lankan issues than many local writers, was present. So were many ambassadors and other diplomats, who seemed to appreciate the effort, though understandably enough none contributed to the discussion that took place.
The focus of the discussion was as to how these efforts could be used to promote Reconciliation. It was in this context that I explained why government might think it useless to engage in detailed refutation, since there is a pervasive belief that, whatever we do, we will still be attacked. However, as was pointed out by the critic who likened my defence of government to the excuses offered in the nineties for the intransigence of the LTTE, this is similar to the indulgence one shows to a spoilt child.
We cannot ignore allegations simply because those who make them are fraudulent. We must help our own people get over any uncertainties they have, and we owe it to them, if to no one else, to clarify matters and show that the basis of our actions was our own laws as well as international norms.


November 23, 2018: Arriving at a weekend matinee of The Prom, I eagerly anticipated seeing one of my favorite musical comedy stars, Beth Leavel in the lead role. The buzz I'd heard was really positive, so I was prepared to sit back, kick up my feet, and have a great time. Only on this occasion Leavel's understudy was playing the lead.
After all, it's a show with big name musical talent. Brooks Ashmanskas' comedic skill is so endearing here. Capturing the clichés of a slightly aging gay man, with his effeminate gestures, grinning with joy, he is like an arrow to the heart. It's he who bonds with the central character, Emma, a teenage lesbian played with a rich sweet, voice by Caitlin Kinnunen.
Even the smaller roles are character driven, with Michael Potts as the principled high school principal, Courtenay Collins as the onerous head of the PTA, and Angie Schworer as the musical comedy gal – a fully grown up version of the girl next door.
Written by Bob Martin of The Drowsy Chaperone fame, with Chad Beguelin, Prom is not as quirky. But it takes leaps at addressing social issues, in a fun-loving way. Social issues like individuality and inclusiveness "that liberal democrats from Broadway" necessarily embrace. What touches us here is the innocence and freshness of the story no matter how traditional it all clearly is.
With a consistent and even musical style Matthew Sklar's songs are easy going Broadway tuners. Simple, clear lyrics by Chad Beguelin, along with the occasional forced rhyme, sustain the energy in this fast-paced, run amok comedy. And Casey Nicholaw's choreography makes us feel like it's something we can do, too, except when the kids are on the stage. They outshine with moves no one else can do.
Scott Pask's scenery is basic, like everything else in high school. And Ann Roth and Matthew Pachtman bring years of Broadway credits to the wardrobe. With a bright red wrap around dress for our leading lady, a shirt adorned with butterflies for Ashmanksas', and plaid flannel shirts for Emma. The patchwork of wardrobe alone tells the story.
But the charm of this musical lies in its whacky characters, the diversity of its actors, and the feel-good story which ends with two girls kissing lovingly on stage, and the promise of a biracial marriage.


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Limits of Power
By Elizabeth Moon
Part of Paladin's Legacy
Category: Epic Fantasy | Military Science Fiction
Jun 11, 2013 | 512 Pages
About Limits of Power
Elizabeth Moon is back with the fourth adventure in her bestselling fantasy epic. Moon brilliantly weaves a colorful tapestry of action, betrayal, love, and magic set in a richly imagined world that stands alongside those of such fantasy masters as George R. R. Martin and Robin Hobb.
The unthinkable has occurred in the kingdom of Lyonya. The queen of the Elves—known as the Lady—is dead, murdered by former elves twisted by dark powers. Now the Lady's half-elven grandson must heal the mistrust between elf and human before their enemies strike again. Yet as he struggles to make ready for an attack, an even greater threat looms across the Eight Kingdoms.
Throughout the north, magic is reappearing after centuries of absence, emerging without warning in family after family—rich and poor alike. In some areas, the religious strictures against magery remain in place, and fanatical followers are stamping out magery by killing whoever displays the merest sign of it—even children. And as unrest spreads, one very determined traitor works to undo any effort at peace—no matter how many lives it costs. With the future hanging in the balance, it is only the dedication of a few resolute heroes who can turn the tides . . . if they can survive.
Praise for Limits of Power
"It's easy to become fully immersed in, and absorbed by, the narrative: [Moon's] great strength lies in the patient accumulation of telling detail, yielding an extraordinarily rich picture of the world's politics, philosophy, military structure, history, magic and alien cultures, where men and women stand as equals even in force of arms."—Kirkus Reviews
"Thoughtful and deeply character driven, full of personal crises as heartbreaking and hopeful as any dramatic invasion . . . Moon deftly avoids big literary explosions, preferring instead a slow boil that builds pressure without relief. There are plots within plots, but the complex story is never confusing. Fantasy fans will be delighted by this impressive foray."—Publishers Weekly (starred review)
Praise for Elizabeth Moon
"This is an excellent series, and Echoes of Betrayal is particularly well done. [Elizabeth Moon is a] consistently entertaining writer, and this book lives up to her standards."—San Jose Mercury News
"Moon's characters navigate an intricate maze of alliances and rivalries. . . . Close attention to military detail gives the action convincing intensity."—The Star-Ledger, on Kings of the North
"A triumphant return to the fantasy world she created . . . No one writes fantasy quite like Moon."—The Miami Herald, on Oath of Fealty
BONUS: This edition includes an excerpt from Elizabeth Moon's Crown of Renewal.
Also in Paladin's Legacy
Also by Elizabeth Moon
See all books by Elizabeth Moon
About Elizabeth Moon
Elizabeth Moon grew up on the Texas border, served three years of active duty in the USMC (1968–71), and now lives with her husband, also a veteran, near Austin, Texas. She has published more than twenty-five novels, including Nebula Award winner The… More about Elizabeth Moon
Published by Del Rey
Apr 29, 2014 | 512 Pages | 4-3/16 x 6-7/8 | ISBN 9780345533081
Jun 11, 2013 | 512 Pages | ISBN 9780345533074
People Who Read Limits of Power Also Read
"It's easy to become fully immersed in, and absorbed by, the narrative: [Elizabeth Moon's] great strength lies in the patient accumulation of telling detail, yielding an extraordinarily rich picture of the world's politics, philosophy, military structure, history, magic and alien cultures, where men and women stand as equals even in force of arms."—Kirkus Reviews


text:
Ö steht für:
 Ö, einen Buchstaben (Umlaut-Vokal) in der deutschen und anderen Sprachen
 Ö (Album), ein 1988 erschienenes Album von Herbert Grönemeyer
 in deutschsprachigen kirchlichen Gesangbüchern die Bezeichnung von Gesängen und Liedern, die sich in der Liste Arbeitsgemeinschaft für ökumenisches Liedgut finden

Siehe auch:
 OE
 Ø
 Ö till Ö

Abkürzung


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Butis koilomatodon es una especie de pez del género Butis, familia Butidae. Fue descrita científicamente por Bleeker en 1849.

Se distribuye por el Indo-Pacífico: Mozambique y Madagascar a China y Filipinas. Atlántico Oriental: Port Harcourt (delta del Níger en Nigeria). También encontrado en Guinea y Camerún. Introducido en Panamá, Venezuela y Brasil. La longitud total (TL) es de 10,7 centímetros. Habita en ríos, estuarios y manglares y su dieta se compone de crustáceos y pequeños peces.

Está clasificada como una especie marina inofensiva para el ser humano.

Referencias

koilomatodon
Especies descritas en 1849