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Project Love: In Service To Humanity << >>
to Humanity
Beginning of the Mission
Remember when Guru Maharaj Ji came to America? We were scattered souls. He had to pull us out of quicksand. Born in the century of the atom bomb, we grew up trying every door marked "escape" and found dead end streets. When Guru Maharaj Ji came, many of us were living on the edge. And He arrived just in time.
How can His Grace be described? He traveled half way around the world to find us. And after doing that, He gave to us the way to find Him. He changed our lives forever. Lost children were brought home. He gathered strangers and showed them they were the oldest, dearest friends. Slowly, we came to Him to see what He would have us do.
He told us to form a mission to spread love throughout the earth. He gave us the key to unlock human suffering arid ignorance. But, blinded by past actions and impressions, we had little understanding of how to use His gifts or follow Him as one family.
His patience with us was more than anyone could imagine. He taught us the consequences of noncooperation by showing us our own actions. He let us finish up our individual games. We can finally appreciate that the beginning of the Mission was for us. So we would be prepared to begin His work on earth.
Gathering the Family
Can we see the world is fast approaching that moment when human beings will open to His love? Can we sense the tide of human enterprise may soon turn and flow back to Him? Can we feel our own purpose moving toward the starting line? Can we imagine that all the separate strands of this family might now be woven into one great flag? Is now the time to gather this premie family at His feet?
A new understanding is now growing in our community. It does not matter what path we have followed to arrive at this place of understanding. It only matters that His call to us, coming from inside, be recognized and that we drop the barriers among us and join our hearts and hands.
We call this understanding Project Love. It has a simple aim. Every premie in America will be found and embraced and brought into one vast spiritual army. We'll draw ourselves in tight around Him so that even His whisper can be heard. We will dedicate ourselves to helping every brother and sister in His family connect in service and find their perfect place. So that when He speaks to us, the premies of America will be at hand to serve His will. This is Project Love.
Volunteers will form teams in over 150 communities. The teams will meet every premie and share honest satsang. They will search among the premies themselves for every suggestion that will help us come together as one family. The way to end our separation is known among us.
These community volunteer teams will open wide the Mission doors, allowing American devotees to come and contribute in any way they can. Project Love will have acheived its first goal when every brother and sister graced with Knowledge has found their own way for regular dedication, service, and satsang. Project Love is a national effort to create a perfect flow of communication and organization from Guru Maharaj Ji to every premie.
Two Steps to Perfect Love
To serve humanity, we must first find our own meeting ground and come together as a real family. Our work on earth begins with us.
There are two practical steps.
Step One: The Active Membership Program
Project Love is a call to every premie to become an active member of Guru Maharaj Ji's Mission. Before we received Knowledge, there were posters, leaflets, magazines and satsang programs explaining Knowledge. There were ashrams or centers where we could come and hear Mahatmas. If other premies had not supported these activities, we might never have received this Knowledge. Now we need a hundred thousand premie hands to support Guru Maharaj Ji's Mission in the world. We must build an active membership for this support.
There is something very practical happening when you make regular and consistent dedication. And there is something very magical happening when each expression of love is pooled with all the other donations and is multiplied many times over. The secret of dedication has to be experienced. Maybe you can't work in a DUO office full time. Or spend your weekends at a prison. Or arrange halls and sat-sang programs. But the job that occupies your time and energy can be turned into service to spread His Knowledge. What we create with our hands or earn from our jobs belongs to Guru Maharaj Ji. We can remind ourselves of the source by giving the first 10% of the fruits of every effort back to Him. The active members of Divine Light Mission are building a national tithing system. We are the foundation from which Knowledge is spreading. When every premie has gathered at the base, we will have built a platform of dedication from which Maharaj Ji can speak to all lands. This is the immediate task of every premie in the United States.
But active membership is more. It is a recognition that our family's purpose is to serve Him. Every component part of this family should be assembled into one working computer so that Guru Maharaj Ji can push the button. In the past, service has appeared to require full time availability. This is changing now. Project Love is opening a vast volunteer service program. We can serve in prisons, hospitals, mental retardation homes, camps, drug programs, food coops, schools, clinics, Divine Sales, DUO offices and ashrams. The active membership program offers service especially tailored for premies with full time or part time jobs. Social service projects are opening so that every person can give one night a week. Step one of Project Love is to bring every American premie into a direct service for Guru Maharaj Ji.
Step Two: Building the Community
Our understanding of satsang should be traced back to its roots. The word means "in the company of truth." We need each other's company on this path. If
our lives are joined together, we can better help ourselves become living satsang. Individuals can turn their homes into Info Centers. Centers will grow into communities. Communities should support schools, clinics, satsang halls, food co-ops and programs for the whole family. Help our family create the conditions for every premie to come to satsang easily. Transportation pools and babysitting clubs can be organized. But satsang is not just the meetings to hear the words. It is a way of life. It is a daily process of taking down the walls among us. Holy Company is His gift of other premies in our lives. Our lives must join like fingers on the hand for the world cries for healing, helping hands. That's why Project Love is building the community.
Will you join Project Love? Will you share this message with every brother and sister? Will you be a volunteer for this mobilization of our spiritual family? Will you participate in a growing and beautiful assembly of premies and come together to serve His plan for peace? The answer is, we will. For He has said to us: "I will come where there is love and unity for I love love and I love unity." And we are people who only want for Him to come to us and come to all the world.
Join the Active Membership Program
Programs for Service -
World Welfare Association
social service and satsang in hospitals, prisons, old age homes, drug rehabilitation centers, welfare offices, mental retardation homes, youth centers and more to come.
Performers of the Living Arts
singers, dancers, musicians, mimes, bands, actors, and performers of every kind spreading love, sat-sang and inspiration among premies and the larger community.
programs of social service and satsang for blacks, Spanish speaking people and other minority groups in the United States.
Shri Hans Humanitarian Services
programs of health treatment offered through public clinics that specialize in preventive medicine as well as human medical care.
Shri Hans Educational
teacher training and special schools based on creative expression and a consciousness of the aim of human life.
Shri Hans Food Services
food co-ops, premie farms, food relief programs, food systems for large gatherings and festivals that aims to serve the premie community and the larger public.
Divine Sales
cooperative businesses serving the larger community and supporting the programs of the Divine United Organization.
And It Is Divine
subscribe to the magic magazine to spread love and light on the earth.
Divine Times
subscribe to the premie newspaper serving the communication needs of our community.
Shri Hans Graphics
a program of design and graphics to create through art feelings of love behind every printed word.
the adaption of technology to our propagation, utilizing tapes to bring visual and audio communication to our entire community.
Shri Hans Films
a program of films on the work of Shri Guru Maharaj Ji and His Mission in the world.
For more information contact your local DIC
or DUO Director or write to:
Divine Light Mission
511 Sixteenth Street
The Satsang Project: See Site Policy for statement regarding copyrightTO TOP


EXCITING freshman Gimondi is expected to have no trouble maintaining his perfect record at Tabcorp Park Menangle tomorrow.
Winner of his two outings for trainer Blake Fitzpatrick, the trotter is a short priced favourite in a Foundation Series heat.
Pleased with the two-year-old's performances, Fitzpatrick is positive Gimondi can complete a hat-trick from barrier five.
"He goes quite well," Fitzpatrick said. "He's been a surprise packet so far. Every time he has been taken off the farm he's gotten better.
"He's always trotted nicely, but is lazy and hard to get a gauge on.
Fitzpatrick also has Fantasy Miss in the following event, with the daughter of Roll With Joe "needing luck" despite drawing the pole.
"It'll be a hard race for her," Fitzpatrick said. "She has got ability but will be better a three-year-old.
Later on the card Fitzpatrick stablemates Moment In The Sun and Pegasus Elegance will cash in the Australian Pacing Gold Trotters' Mile.
Successful when first-up from a nine-month break a fortnight ago, Moment In The Sun will begin from gate four, with Pegasus Elegance drawn six.
"Moment In The Sun is a good mare with a lot of ability," Fitzpatrick said. "She is one to follow.
"Pegasus Elegance is a consistent mare which is always and around the mark.


The Libraries Transforming Communities (LTC) team from Red Hook Public Library used their training to engage residents in their small town. They learned people were frustrated that problems in their community—even obvious ones—often went unaddressed. The only stoplight in town, which didn't work properly, was emblematic of their concerns and came up in many discussions with residents. The LTC team took action and brought officials together to figure out how to x the problem. This seemingly small act sent a signal to the community that it was possible to make things happen, which has led to people stepping forward to work together on other issues that are keeping Red Hook from being more livable and connected. The library is playing a central role in convening these groups and has become a model for how other organizations want to work in the community.
By leading Community Conversations and listening to residents, the Red Hook LTC team realized they could address a problem and improve how the community worked together by fixing the town's stoplight. Choosing to act on this was a critical decision; it sent a signal that change was possible and that people's concerns mattered.
People were so energized by the forward progress that they wanted to keep going. The library is now working with residents and other partners to establish a community center.
The LTC team's efforts have inspired residents to get involved in ways they weren't before. The library is playing a convening role, but in many cases, residents are developing solutions to problems.
The Village of Red Hook, New York—population 1,961—sits about 100 miles north of New York City near the Catskills and Hudson River Valley. It is home to Bard College, a nationally ranked liberal arts college that is the town's largest employer.
When the discussion around community aspirations began in 2014, most residents reported how much they loved living in this community. A couple of issues quickly rose to the top that needed to be addressed, including one iconic one: the town's only stoplight.
Every day, 14,000 vehicles passed through the light, which sat at the intersection of two state highways. The light's timing was off, leading to wait times as long as seven minutes. Seeking to avoid the long delay, drivers frequently chose to cut through residential neighborhoods, endangering kids and adults who were walking and riding bikes in the streets.
this story begins. For a small town, this little traffic light was a big issue. Everyone in town knew it was a problem, but the sense of urgency was never fully communicated to the state. So it sat near the bottom of a long line of state-funded public infrastructure problems.
But a seemingly small act— people coming together in 2015 to talk, share ideas and x a problem—forged a new "can-do" narrative in Red Hook in which residents take ownership of their community in a different way. This substantial change was made possible by the efforts of a small group of leaders, organized by Red Hook Public Library, a small community library with only 4,500 cardholders.
The library is working to establish a community center to give more people opportunities to come together.
And, seeing the benefits of their work, the library is helping to make connections between groups and encouraging others to take on community engagement work so more people can work toward the betterment of the community.
Being a part of changes like these also marked a big shift for the library.
Freudenberger wanted to change the perception of the library and add more value to the community.
Shortly after being named library director, she started attending local Rotary and chamber of commerce meetings and joined a group of local leaders called Red Hook Together. Started by Erin Cannan, associate director of the Bard College Center for Civic Engagement, Red Hook Together met about every five weeks "as a way to address some of the 'town and gown' issues," Cannan said.
At the first meeting Freudenberger attended, several people said they wished the community had a gathering place to help residents stay more informed about local issues.
For this reason, the library jumped at the chance to be part of the Libraries Transforming Communities (LTC) initiative, a partnership between the American Library Association and The Harwood Institute for Public Innovation, funded by the Bill & Melinda Gates Foundation. LTC aims to strengthen the role of libraries in helping communities solve problems and work together more effectively. As part of the initiative, Harwood trained and coached 10 library teams, made up of library staff and community partners. Over two years, teams learned to apply Harwood's Turning Outward approach, a practice or discipline of understanding a community in a deep way and then using that knowledge as a reference point for choices and actions.
Freudenberger said she and the rest of the Red Hook team, which included Cannan, "felt very smug" going into the LTC project, thinking they were used to working collaboratively, listening to the community and getting results.
3. How is that different from how you see things now?
Brent Kovalchik, Village of Red Hook deputy mayor and a member of the LTC team, partnered with a Bard student to do most of the canvassing.
Residents welcomed the chance to share, Freudenberger said.
People shared many common concerns. They wanted a safer, better connected community. The town's stoplight surfaced as one concrete way to help make that kind of community a reality.
The library's engagement of the community resulted in laser- like attention to this issue. With support from the mayor's office, the library encouraged people to contact their state representatives and the Department of Transportation and ask them to prioritize fixing the stoplight. The mayor made additional calls to accelerate the repair.
The library never explicitly publicized its role in harnessing community momentum to repair the stoplight.
Freudenberger said while she thought she was Turned Outward because she had been engaging with the community since starting her role at the library, she was not.
She is asking different questions now.
By the time Freudenberger and her colleagues met with other libraries at a gathering in January 2015, she realized a switch in her viewpoint had happened gradually.
She listened to other library groups describe their outreach.
Putting the community first, as opposed to focusing first on how to promote the library, has, ironically, elevated the library.
Buoyed by the momentum generated by the fixed stoplight, the LTC team is beginning to help with other issues, such as the development of a community center and offering more opportunities for young people.
Hearing residents' concerns about a lack of activities for teens and young adults in the community, the library, Bard College and Red Hook High School teamed up to provide science, technology and programming seminars in the high school, and then on a mobile program that took the program to rural areas without easy access to the library.
Bard students host summer science camps for younger students at the Red Hook library, and the library pays their stipends. The library and college were also part of a community art exhibit meant to appeal to Red Hook's young adults.
According to the LTC team, the library's knowledge of the community has become the lens through which they now evaluate their choices about how to support Red Hook.
The stoplight started a momentum that transformed the library into a critical player in solving problems throughout the community. They are continuing their efforts to help Red Hook become better connected, including work on developing a community center.
At a Red Hook Together meeting in spring 2015, nearly a year after their LTC training and more than four years after Freudenberger started promoting the library at her first meeting with this group, the conversation was very different.


The KERMIT skills assessment ('Kompetenzen Ermitteln') refers to the regular learning development review by the IfBQ that takes places in all Hamburg schools. At secondary level this happens in grade 5, 7, 8 and 9.
The tasks in the assessment do not test specific areas of teaching in class. Whatever the subject, they assess at a higher level which skills students have already acquired in certain areas and provide valuable information about additional areas of development. With respect to English, our core area of specialisation, we are well above average and lead the way by a significant margin compared to other secondary schools in Hamburg.
The results of our current grade 5 and 7 students show above-average performances in all tested areas. Not only the results in English are outstanding but also their performances in math, German and natural sciences are far above the Hamburg average. Therefore, the students, who have previously attended the Phorms primary school, enter secondary school from an excellent outset. Please find a detailed overview of the results here (in German only). Please find the detailed overview of the results of the fifth and seventh grade under "Materials" in de download area of this page.
Congratulations to our grade 5 and 7 students for those excellent results.


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Die membranöse Glomerulonephritis (membranöse Glomerulopathie, epimembranöse Glomerulonephritis, membranöse Nephropathie) ist eine chronisch entzündliche Erkrankung der Nierenkörperchen. Als Ursache werden unter anderem Antikörper gegen Proteine der Podozyten angenommen, einer Zellschicht, die im Nierenkörperchen auf der Filtrationsmembran (glomeruläre Basalmembran) liegt.

Im Erwachsenenalter ist die membranöse Glomerulonephritis häufigste Ursache des nephrotischen Syndroms, das mit hohen Eiweißverlusten über den Urin, Wassereinlagerungen und Störungen im Stoffwechsel der Blutfette einhergeht.
Die Diagnose wird durch Nierenpunktion und feingewebliche sowie elektronenmikroskopische Untersuchung des gewonnenen Nierengewebes gestellt. Kennzeichnend ist die Ablagerung von Komplexen aus Antikörper und Antigen (Immunkomplexen) auf der Außenseite der glomerulären Basalmembran.

Der Verlauf der membranösen Glomerulonephritis ist sehr variabel, ca. ein Drittel der Fälle heilt spontan aus, ca. ein Drittel bleibt im Verlauf stabil und ca. ein Drittel führt zum chronischen Nierenversagen.

Die Behandlung erfolgt mit Medikamenten, welche das Immunsystem hemmen (immunsuppressive Therapie): Cortison, Cyclophosphamid, Chlorambucil, Cyclosporin und Mycophenolat-Mofetil.

Epidemiologie
In alten Nierenbiopsie-Statistiken war die membranöse Glomerulonephritis die häufigste Diagnose bei erwachsenen Patienten mit Proteinurie und nephrotischem Syndrom. Der relative Anteil membranöser Glomerulonephritiden an Nierenbiopsien hat in den letzten Jahren kontinuierlich abgenommen. Möglicherweise ist dies auf eine Zunahme der Fälle von fokal segmental sklerosierender Glomerulonephritis zurückzuführen.

Die membranöse Glomerulonephrtits kommt in allen ethnischen Gruppen und bei beiden Geschlechtern vor. Die idiopathische (= Ursache unbekannt) membranöse Glomerulonephritis ist allerdings bei Männern über 40 Jahren und weißer Hautfarbe häufiger. Bei jüngeren Frauen sollte bei membranöser Glomerulonephritis an eine Lupusnephritis gedacht werden. Bei Kindern ist die membranöse Glomerulonephritis seltener und häufig mit einer Hepatitis B assoziiert.

Pathologie

In der Lichtmikroskopie findet sich eine diffuse Verdickung der glomerulären Basalmembran in allen Nierenkörperchen (Glomeruli). Die Anzahl der Zellen ist in den Nierenkörperchen nicht vermehrt. In frühen Stadien können die Glomeruli unauffällig erscheinen, in fortgeschritteneren Stadien finden sich in der Basalmembran feine Löcher, an der Außenseite (= der subepithelialen Seite) der Basalmembranen bilden sich feine Ausziehungen, sogenannte "Spikes". Gelegentlich können in der Trichrom-Färbung subepitheliale Proteinablagerungen nachgewiesen werden. Bei weiterem Fortschreiten der Erkrankung kommt es zur Vernarbung (Sklerose) der Nierenkörperchen und Veränderungen von Nierenkanälchen (Tubuli) und Zwischengewebe (Interstitium) der Niere.

In der Fluoreszenzmikroskopie findet man feine körnelige Ablagerungen von IgG und C3 entlang der Basalmembran.

Die Elektronenmikroskopie zeigt elektronendichte Ablagerungen (Depots) an der Außenseite (der subepithelialen) Seite der Basalmembran, eine Verschmelzung der darüber liegenden Fußfortsätze der glomerulären Deckzellen (Podozyten) sowie eine Verbreiterung der Basalmembran durch Ablagerung neu gebildeter extrazellulärer Matrix zwischen den Immundepots, den "Spikes". Im Verlauf der Erkrankung werden die Immundepots schließlich vollständig von der Basalmembran umschlossen.

Der Grad der Veränderungen der Basalmembran ermöglicht eine histologische (feingewebliche) Stadieneinteilung, die aber nicht mit der Schwere der Erkrankung oder dem Ansprechen auf Medikamente korreliert.
 Stadium I: Vereinzelte kleine Ablagerungen (Immundepots) ohne Verdickung der Basalmembran.
 Stadium II: Ausgedehntere Immundepots mit Baslamembranausziehungen (Spikes) zwischen den Depots und Verdickung der Basalmembran.
 Stadium III: Die Immundepots sind vollständig von Basalmembran umgeben (intramembranöse Immundepots), die Basalmembran ist verdickt.
 Stadium IV: Unregelmäßige Verdickung der Basalmembran mit Immundepots innerhalb der Membran.

Pathogenese
Aufgrund von Befunden, die an experimentellen Modellen erhoben wurden, vermutet man, dass sich die Immundepots an Stelle der Ablagerung ("in situ") bilden. Dazu müssen IgG-Antikörper die Basalmembran passieren, und Antigene binden, die entweder an den Fußfortsätzen oder in deren Nähe exprimiert werden, oder aber aus dem Blutkreislauf an die Außenseite der Basalmembran gelangt sind.

Tiermodell Heymann-Nephritis (Megalin)
Bereits 1959 gelang es Heymann, in Ratten eine membranöse Glomerulonephritis hervorzurufen, in dem er die Tiere mit einem Extrakt von Nierentubulus-Zellen in Freund'schem Adjuvans impfte. Als Zielantigene konnten zwischenzeitlich Megalin und Cubilin identifiziert werden, die in Membran-Einstülpungen von tubulären und glomerulären Epithelzellen, den Clathrin coated pits, exprimiert werden und für die Endozytose vieler Stoffe in mehreren Gewebetypen verantwortlich sind.

Die Bildung von Immunkomplexen auf der Außenseite der Basalmembran führt zur Komplement-Aktivierung. Dadurch werden die Podozyten geschädigt. Eine Störung des Zytoskeletts führt zum Verlust der Integrität der glomerulären Schlitzmembran. Dies wiederum führt zum Übertritt von Eiweiß in den Urin, zur Proteinurie. Zudem kommt es zu einer Überproduktion von Bestandteilen der Basalmembran, Kollagen IV und Laminin, und so zur Verbreiterung der Basalmembran.

Beim Menschen war das Antigen, welches für die membranöse Nephritis verantwortlich ist, lange Zeit nicht bekannt – Megalin wird im menschlichen Glomerulus nicht exprimiert.

Vorgeburtliche (antenatale) membranöse Glomerulonephritis (Neutrale Endopeptidase)
Beobachtung an der antenatalen membranösen Nephritis legten einen ähnlichen Pathomechanismus auch beim Menschen nahe:
Im menschlichen Glomerulus exprimieren die Podozyten das Enzym neutrale Endopeptidase. Fehlt der Mutter aufgrund einer Mutation dieses Enzym, kann es während der ersten Schwangerschaft zu einer Immunisierung der Mutter kommen, wenn der Fetus dieses Enzym in der Niere exprimiert. Bei einer zweiten Schwangerschaft können mütterliche Antikörper gegen neutrale Endopeptidase die Plazenta passieren und im Fetus eine membranöse Glomerulonephritis mit nephrotischem Syndrom hervorrufen. Einige Monate nach der Geburt verschwinden die mütterlichen Antikörper aus dem kindlichen Blut, die Immundepots in der Niere lösen sich auf, das nephrotische Syndrom heilt spontan aus.

Idiopathische membranöse Glomerulonephritis (Phospholipase-A2-Rezeptor Typ M; THSD7A)
2009 beschrieben Beck et al. im Serum von 70 % untersuchter Patienten mit idiopathischer membranöser Glomerulonephritis einen Antikörper vom Typ IgG4, welcher mit einem bestimmten Protein aus dem Extrakt von Nierenkörperchen reagiert. Das Protein wurde als Phospholipase-A₂-Rezeptor Typ M (PLA2R) identifiziert. Dieses Rezeptorprotein wird in normalen humanen Nierenkörperchen auf der Oberfläche der Podozyten exprimiert und kann bei Patienten mit membranöser Glomerulonephritis auch in den Immundepots nachgewiesen werden. Im Serum kann das Antigen PLA2R dagegen nicht nachgewiesen werden. Dies ist ein starker Hinweis darauf, dass bei der membranösen Glomerulonephritis die Immunkomplexe direkt an den Podozyten gebildet werden und nicht etwa zunächst im Serum mit anschließender Ablagerung an der Außenseite der glomerulären Basalmembran. Für den Phospholipase-A2-Rezeptor-Antikörper sind mittlerweile kommerzielle Tests flächendeckend verfügbar. Da der Antikörpertiter mit der Krankheitsaktivität korreliert, kann er zur Therapieentscheidung und Therapiemonitoring verwendet werden. Neben dem PLA2-Rezeptor-AK ist mittlerweile ein Antikörper gegen die Thrombospondin Typ 1 Domäne 7A (THSD7A) identifiziert worden.

Frühkindliche membranöse Glomerulonephritis (Rinderserumalbumin)
Bei Kindern im Alter zwischen 5 Monaten und 2,3 Jahren mit membranöser Nephropathie wurden im Blut hohe Konzentrationen an Rinderserumalbumin und dagegen gerichtete Antikörper vom Typ IgG1 und IgG4 gefunden. Diese Antikörper konnten zusammen mit Rinderserumalbumin auch in den epimembranösen Ablagerungen der Nierenkörperchen nachgewiesen werden. Auch im Eluat von Nierenbiopsien fand sich IgG1 und IgG4, das mit Rinderserumalbumin reagierte. Man nimmt an, dass im Darm geringe Mengen von Rinderserumalbumin aus Kuhmilch in unverdauter oder teilweise verdauter Form aufgenommen werden können. So findet man im Blut von fast allen Kleinkindern Antikörper gegen Rinderserumalbumin. Bei den Kindern mit membranöser Nephropathie waren die Serum-Spiegel von Rinderserumalbumin höher als bei gesunden Kontrollen und das Rinderserumalbumin lag in kationischer (positiv geladener) Form vor. Unter Behandlung mit Prednison, Mycophenolat-Mofetil oder Cyclosporin kam es bei allen Kindern zu einer teilweisen oder vollständigen Remission.

Sekundäre membranöse Glomerulonephritis
In Fällen, bei denen die membranöse Glomerulonephritis Folge einer anderen Erkrankung war, konnten spezifische Antigene in den Immundepots nachgewiesen werden: Doppelstrang-DNA bei Lupus erythematodes, Thyreoglobulin bei Thyreoiditis, Hepatitis B-Antigen, Treponema-Antigen und Helicobacter pylori-Antigen bei den entsprechenden Infektionen, sowie Carcinoembryonales Antigen und Prostataspezifisches Antigen bei Tumorerkrankungen.

Bei der feingeweblichen Untersuchung weisen folgende Befunde auf eine sekundäre Genese der membranösen Glomerulonephritis hin:
 Zellvermehrung in Mesangium und Kapillarknäuel (mesangiale und endikapilläre Proliferation)
 Immunhistologischer Nachweis aller Immunglobulin-Klassen und Komplementkomponente C1q.
 Nachweis anderer Immunglobuline als IgG4.
 Immundepots auf der Innenseite der Basalmembran (subendotheliale Immundepots), im Mesangium, entlang der Basalmembranen der Nierenkanälchen (tubuläre Basalmembranen) oder in Gefäßwänden.
 Elektronenmikroskopisch nachweisbare röhren- oder netzförmige (tubuloretikuläre) Einschlüsse in den Endothelzellen des Kapillarknäuels der Nierenkörperchen.

Die Rolle der T-Zellen
Neben Antikörpern sind auch T-Helferzellen an der Pathogenese der Glomerulonephritiden beteiligt.

Th1-Helferzellen überwiegen bei Glomerulonephritiden, die mit einem vermehrten Zellwachstum einhergehen, wie der rasch progressiven Glomerulonephritis. Bei der membranösen Glomerulonephritis überwiegen dagegen, wie bei der Minimal-Change-Glomerulonephritis, Th2-Helferzellen.

Genetik
Eine genomweite Assoziationsstudie an 556 Patienten europäischer Herkunft ergab eine Assoziation mit zwei Einzelnukleotid-Polymorphismen. Auf Chromosom 2q24 fand sich eine Assoziation mit dem Gen für den Phospholipase-A₂-Rezeptor Typ M (PLA2R1). Die engste Assoziation bestand jedoch zu einem Allel auf Chromosom 6p21, das für ein HLA-Klasse II Antigen (HLA-DQA1) codiert. Dieses Allel erleichtert möglicherweise eine Autoimmunreaktion gegen körpereigene Antigene, wie Varianten des PLA2R1.

Ätiologie
In etwa 75 % der Fälle von membranöser Glomerulonephritis wird keine Ursache gefunden (idiopathische membranöse Glomerulonephritis).

Für das Vorliegen einer sekundären membranösen Glomerulonephritis, die eine andere Erkrankung zur Ursache hat, sprechen:
 Tubuloretikuläre Strukturen in den Podozyten, die durch Interferon alpha hervorgerufen werden, sprechen für einen Lupus erythematodes (Abb. unter).
 Immundepots auf der Innenseite der glomerulären Basalmembran (subendotheliale Depots) oder im Mesangium (Zwischengewebe des Nierenkörperchen) sprechen für das Vorhandensein zirkulierender Immunkomplexe im Blut (z. B. bei postinfektiöser Glomerulonephritis), und gegen die in situ Bildung der Immundepots.
 Immundepots an den Basalmembranen der Nierentubuli sind bei der idiopathischen membranösen Nephritis selten, bei sekundären Formen dagegen häufig.
 Bei der idiopathischen membranösen Nephritis wird in den Immundepots vorwiegend Immunglobulin G vom Isotyp IgG4 gefunden, bei Lupus erythematodes IgG2, IgG3, IgA, IgM und Komplementfaktor C1q, bei Tumoren überwiegt IgG1 und IgG2.

Die Ursachen der sekundären membranösen Glomerulonephritis sind vielfältig:
 In etwa 10–20 % der Fälle einer Nierenbeteiligung bei Lupus erythematodes wird eine membranöse Nephritis gefunden. Die Nierenerkrankung kann der Manifestation des Lupus erythematodes vorausgehen, so dass insbesondere bei jungen Frauen mit membranöser Nephritis immer an diese Diagnose gedacht werden sollte.
 Medikamente wie Goldsalze, Penicillamin, Bucillamin, nichtsteroidale Antiphlogistika, Mercaptopropionylglycin, hochdosiertes Captopril, sowie monoklonale Antikörper wie Etanercept, Infliximab und Adalimumab können zu einer membranösen Nephritis führen.
 In Endemiegebieten kann eine membranöse Nephritis bei chronischen Trägern des Hepatitis B-Virus, insbesondere bei Kindern auftreten. In den Immundepots sind vorwiegend HBe-Antigen und Anti-HBe-Antikörper nachweisbar. Bei Kindern heilt die membranöse Nephritis bei Hepatitis B häufig aus, bei Erwachsenen kommt es dagegen häufig zu einem chronischen Verlust der Nierenfunktion. Eine membranöse Nephritis wurde auch bei Trägern des Hepatitis C-Virus beschrieben.
 Bei etwa 5–20 % der Erwachsenen mit membranöser Nephritis, insbesondere bei den über 65-Jährigen, liegt ein Tumorleiden vor, häufig Tumore der Lunge und des Magen-Darm-Traktes, seltener bösartige Erkrankungen des blutbildenden Systems.
 Eine membranöse Nephritis wurde auch infolge einer Nieren- oder Stammzelltransplantation beschrieben.
 Seltene Ursachen einer membranösen Nephritis sind Schistosomiasis, Malaria quartana, Syphilis, Sarkoidose, Sjögren-Syndrom und chronische Formaldehyd-Exposition.
 Quecksilber-Verbindungen, die z. B. in der traditionellen chinesischen Medizin oder in Haut-Bleichcremes Verwendung finden, können ebenfalls zu einer membranösen Nephritis führen.

Membranöse Glomerulonephritis und andere Nierenerkrankungen
Eine membranöse Glomerulonephritis wurde auch zusammen mit anderen glomerulären Nierenerkrankungen beschrieben.
 Fokal-segmentale Glomerulosklerose: Möglicherweise sind die sklerotischen (narbigen) Veränderungen im Nierenkörperchen Folge der membranösen Nephritis oder eines erhöhten intraglomerulären Drucks. Sklerotische Läsionen in der Nierenbiopsie weisen auf eine fortschreitende Verschlechterung der Nierenfunktion und damit auf eine schlechte Prognose hin.
 Diabetische Nephropathie: Möglicherweise enthalten die Immundepots Schweine-Insulin und Antikörper gegen Schweine-Insulin. Eine Besserung des nephrotischen Syndroms nach Umstellung auf humanes (menschliches) Insulin wurde beschrieben.
 Rasch progressive Glomerulonephritis: In etwa 5 % der Fälle kann eine membranöse Nephritis durch eine rasch progressive Glomerulonephritis überlagert werden.
 Gelegentlich treten die histologischen Veränderungen einer membranöse Glomerulonephritis zusammen mit einer IgA-Nephropathie, einem Goodpasture-Syndrom oder einer Wegener-Granulomatose auf.

Klinik
Bei etwa 80 % der Patienten besteht ein nephrotisches Syndrom mit unter Umständen erheblichen Wassereinlagerungen in den Geweben, Schwellungen von Beinen und Augenlidern, Gewichtszunahme und Abnahme der Urinausscheidung. Bei etwa 20 % der Betroffenen besteht eine Proteinurie geringeren Ausmaßes ohne zusätzliche Symptome. Im Urinsediment werden häufig ovale Fettkörperchen, Fetttröpfchen und Fettzylinder angetroffen. Bei etwa 50 % der Patienten besteht eine Mikrohämaturie, das heißt im Urin werden rote Blutkörperchen (Erythrozyten) nachgewiesen. Die Albuminurie führt zu einer Störung des tubulären Transportes, im Urin ist daher häufig Glukose trotz normaler Blutzucker-Werte nachweisbar (Glukosurie).

In der Blutuntersuchung findet sich meist eine Verminderung des Albumins und eine schwere Erhöhung der Blutfette (Hyperlipidämie)

Etwa 70 % der Patienten haben bei Krankheitsbeginn einen normalen Blutdruck und eine normale Nierenfunktion. Ein akutes Nierenversagen ist selten; Ursachen sind eine Hypovolämie durch zu aggressive Behandlung mit harntreibenden Substanzen (Diuretika), eine rasch progressive Glomerulonephritis oder der Verschluss der Nierenvene durch ein Blutgerinnsel (Nierenvenenthrombose).

Diagnose

Nierenbiopsie
Die Diagnose einer membranösen Glomerulonephritis kann nur durch eine Nierenbiopsie gestellt werden. Eine Nierenbiopsie sollte bei allen Erwachsenen mit nephrotischem Syndrom unklarer Ursache erfolgen. Bei Kindern mit nephrotischem Syndrom liegt meist eine Minimal-Change-Glomerulonephritis vor, die gut auf Glukokortikoide anspricht, so dass bei Kindern auf eine Nierenbiopsie zunächst verzichtet wird.

Labordiagnostik
Zur Identifikation einer möglichen sekundären Ursache der membranösen Glomerulonephritis sollten folgende Laboruntersuchungen erfolgen:
 Antinukleärer Antikörper. Ein hoher Titer spricht für das Vorliegen eines systemischen Lupus erythematodes.
 Komplementfaktor C3. Eine Verminderung des Komplementfaktors C3 wird bei systemischem Lupus erythematodes und Hepatitis B gefunden. Bei idiopathischer membranöser Glomerulonephritis ist der C3-Spiegel normal.
 Serologische Marker der Hepatitis B und Hepatitis C

Tumordiagnostik
Bei einigen Patienten mit membranöser Glomerulonephritis liegt ein Tumorleiden vor. In den meisten Fällen ist die Krebserkrankung zum Zeitpunkt der Manifestation der membranösen Nephropathie bereits bekannt. In seltenen Fällen manifestiert sich die Nierenerkrankung vor der Tumorerkrankung.

Die empfohlenen Krebsvorsorge-Untersuchungen sollten in jedem Fall durchgeführt werden.
Bei erhöhtem Risiko für Lungenkrebs sollte eine Röntgenuntersuchung, ggf. eine Computertomographie des Thorax erfolgen.
Eine darüber hinausgehende Tumorsuche wird nur empfohlen, wenn zusätzliche Hinweise auf eine Krebserkrankung vorliegen, wie Nachweis von occultem Blut im Stuhl, Blutarmut (Anämie) oder ungeklärte Gewichtsabnahme.

Krankheitsverlauf und Prognose
Ohne Behandlung hat die Erkrankung einen relativ günstigen Spontanverlauf:
 Bei ca. einem Drittel der Patienten kommt es innerhalb eines Jahres zu einer Normalisierung der Proteinurie (Vollremission).
 In 25–40 % der Fälle sinkt die Proteinurie unter 2 g pro Tag (Teilremission).
 Allerdings ist nach 5 Jahren bei etwa 14 % der Betroffenen ein vollständiger Verlust der Nierenfunktion (terminale Niereninsuffizienz) eingetreten, nach 10 Jahren bei 35 % und nach 15 Jahren bei 41 % der Betroffenen.

Eine Behandlung mit Medikamenten, welche das Renin-Angiotensin-Aldosteron-System hemmen, verbessert die Prognose.

Risikofaktoren für einen ungünstigen Krankheitsverlauf
In Anbetracht des oftmals günstigen Krankheitsverlaufes und der unter Umständen gravierenden Nebenwirkungen einer Behandlung sollte eine immunsuppressive Therapie nur bei den Patienten begonnen werden, bei denen ein schweres nephrotisches Syndrom oder ein hohes Risiko für eine fortschreitende Verschlechterung der Nierenfunktion besteht.

 Im Gegensatz zu anderen glomerulären Erkrankungen besteht keine Beziehung zwischen Ausmaß der tubulointerstitiellen Schädigung und Ansprechen auf eine Behandlung oder Geschwindigkeit des Nierenfunktionsverlustes.
 Ein erhöhtes Risiko der Progression besteht bei:
 Männern, über 50 Jahren mit einer Proteinurie über 8 g/Tag über mehr als 6 Monate. Auch bei sehr hoher Eiweißausscheidung werden jedoch Remissionen in ca. 20 % der Fälle beobachtet.
 Patienten mit erhöhtem Kreatinin bei Diagnosestellung.
 Asiaten habe eine bessere Prognose als Angehörige anderer Ethnien.
 Frauen, Kinder, junge Erwachsene, Patienten mit nicht-nephrotischer Proteinurie (Eiweißausscheidung < 3,5 g/Tag) und normaler Nierenfunktion drei Jahre nach Krankheitsbeginn haben eine günstige Prognose.
 Patienten mit medikamentös-induzierter membranöser Nephritis haben ebenfalls eine gute Prognose. Die Proteinurie kann allerdings bis zu einem Jahr nach Absetzen des auslösenden Medikamentes noch zunehmen und es können bis zu drei Jahre bis zu einer Normalisierung der Eiweißausscheidung vergehen.

Komplikationen
Kommt es im Verlauf der Erkrankung zu einem akuten Verlust der Nierenfunktion, ist dies meist auf folgende Komplikationen zurückzuführen:
 akute Nierenvenenthrombose (Verschluss der Nierenvene durch ein Blutgerinnsel), gelegentlich gekennzeichnet durch Flankenschmerzen
 durch Medikamente induzierte interstitielle Nephritis mit Leukozyten, insbesondere eosinophilen Granulozyten im Urin
 rapid progressive Glomerulonephritis mit Erythrozyten und Erythrozytenzylindern im Urin.

Therapie

Allgemeine Therapiemaßnahmen
Die allgemeine Behandlung der membranösen Nephropathie hat folgende Ziele:
 Kontrolle der Wassereinlagerungen (Ödeme) durch harntreibende Medikamente (Diuretika),
 Sicherstellung einer ausreichenden Nahrungszufuhr,
 Reduktion von Proteinurie und Kontrolle von Bluthochdruck durch ACE-Hemmer und AT1-Antagonisten,
 Korrektur der oft ausgeprägten Fettstoffwechselstörung (Hyperlipidämie) durch Statine, sowie
 Vorbeugung gegen thromboembolische Komplikationen durch Gabe von gerinnungshemmenden Substanzen, wenn der Albuminspiegel im Serum aufgrund der Eiweißverluste über den Urin unter 2 g/dl abgesunken ist.

Immunsuppressive Therapie
Aufgrund der günstigen Prognose der Erkrankung mit einem hohen Anteil an Spontanremissionen und der unter Umständen schwerwiegenden Nebenwirkungen einer immunsuppressiven Behandlung, wird diese nur bei Hinweisen auf eine ungünstige Prognose begonnen, nach dem der Krankheitsverlauf unter allgemeinen Therapiemaßnahmen über einen längeren Zeitraum beobachtet wurde.
 Patienten ohne Symptome, bei denen die Proteinurie unter 3,5 g/Tag liegt, werden im Allgemeinen nicht mit Immunsuppressiva behandelt, da die Langzeitprognose günstig ist. Regelmäßige Kontrollen von Nierenfunktion und Proteinurie sind jedoch erforderlich, um ein Fortschreiten der Erkrankung rechtzeitig zu erkennen.
 Patienten mit einer Proteinurie über 3,5 g/Tag, bei denen keine Symptome vorliegen, oder bei denen die Wassereinlagerungen (Ödeme) leicht mit Diuretika kontrolliert werden können, sollten vor einer immunsuppressiven Therapie über einen längeren Zeitraum beobachtet werden. In bis zu 65 % der Betroffenen kann es zu unter allgemeinen Therapiemaßnahmen zu einer Spontanremission der Erkrankung kommen, insbesondere bei Frauen, Kindern und jungen Erwachsenen mit normaler Nierenfunktion und einem prognostisch günstigen feingeweblichen Befund.
 Eine immunsuppressive Therapie wird bei Patienten mit schlechter Prognose begonnen:
 bei nicht durch einen Volumenmangel erklärter Nierenfunktionseinschränkung und prognostisch ungünstigen feingeweblichen Befunden, insbesondere Vernarbungen (Fibrose) von Nierenkörperchen (Glomeruli) und Zwischengewebe (Interstitium) der Niere,
 bei schwerem symptomatischem nephrotischen Syndrom mit ausgeprägten Wassereinlagerungen (Ödemen), schwerer Fettstoffwechselstörung (Hyperlipidämie) und einer Proteinurie über 10 g/Tag,
 bei Hinweisen in der Verlaufsbeobachtung auf eine zunehmende Nierenfunktionseinschränkung,
 bei über 50-jährigen Männern mit fortbestehendem nephrotischen Syndrom,
 bei Auftreten von Thrombosen und/oder Embolien als Komplikation des nephrotischen Syndroms.

Es werden unterschiedliche immunsuppressive Medikamente eingesetzt:
 Die alkylierenden Substanzen Cyclophosphamid und Chlorambucil werden meist in Kombination mit Prednison eingesetzt. Diese Kombination scheint die wirksamste Form der Behandlung zu sein. Allerdings sind schwerwiegende Nebenwirkungen wie eine Entzündung der Harnblase und Langzeitfolgen wie Unfruchtbarkeit und erhöhtes Krebsrisiko möglich.
 Eine Monotherapie mit Cortison ist weniger belastend, die Ansprechraten sind aber gering.
 Spricht eine Behandlung mit Alkylantien nicht an, oder wird diese von den Betroffenen wegen der möglichen Nebenwirkungen nicht gewünscht, kann mit Cyclosporin, ggf. in Kombination mit Prednison behandelt werden.
 Der selektiv gegen B-Zellen gerichtete monoklonale Antikörper Rituximab vermindert die Proteinurie. In einer italienischen Studie kam es bei zehn von fünfzig Patienten zu einer kompletten Remission der Erkrankung. Nierenbiopsien von sieben dieser zehn Patienten zeigten eine vollständige oder weitgehende Rückbildung der Immundepots. Vergleichende Studien mit ACE-Hemmern/AT1-Antagonisten und Langzeitbeobachtungen fehlen bislang. Eine seltene, schwere Nebenwirkung von Rituximab ist das Auftreten einer progressiven multifokalen Leukenzephalopathie.
 Die optimale Dauer der immunsuppressiven Behandlung ist nicht bekannt.

Alternative Behandlungsmaßnahmen
Bei Patienten, die auf konventionelle Therapien nicht ansprachen, wurden eine Vielzahl alternativer Substanzen eingesetzt:
 Eine Behandlung mit Mycophenolat-Mofetil über 12 Monate führte zu keiner Besserung der Proteinurie und war mit schweren Nebenwirkungen behaftet.
 Azathioprin hat möglicherweise keinen Effekt auf den Krankheitsverlauf, es wurden aber schwere Nebenwirkungen beschrieben.
 Nach intravenöser Gabe von Immunglobulin wurde in Einzelfallberichten eine Besserung der Proteinurie beschrieben.
 Bei Patienten, die Cyclosporin nicht vertrugen, wurde als Alternative Tacrolimus eingesetzt, vergleichende Studien fehlen bislang.
 In einer Pilotstudie führte Pentoxifyllin zu einer Besserung der Proteinurie, größere Studien fehlen bislang.
 Nichtsteroidale Antiphlogistika senken ebenfalls die Proteinurie. Da diese Substanzen jedoch die Nieren schädigen können, werden sie in der Regel nicht eingesetzt.
 In unkontrollierten Studien senkte Adrenocorticotropin die Proteinurie, Daten zum Langzeitverlauf wurden bislang nicht vorgelegt.

Weblinks
 Epimembranöse Glomerulonephritis Pathologie - Bilddatenbank Pathopic der Universität Basel (PathoPic - Anleitung; PDF; 2,2 MB)
 Agnes Fogo et al.: Membranous Glomerulonephritis. Atlas of Renal Pathology. In: Am J Kidney Dis, 31(3), 1998, S. E1
 S. Krautzig: Kasuistik mit Bildmaterial. 48-jähriger Chirurg mit Ödemen. In: Klinische Nephrologie, 8. Juni 2007, Medizinische Hochschule Hannover

Einzelnachweise

Krankheitsbild in der Nephrologie

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One of the sickest edits ever made!
Lots of fun in Kaunertal last week!
Some nice insights of Sage Kotsenburg's post-Olympics up and downs!
Take a look at this snowboarding African kid and his inspiring story!
Those tweaks and grabs are out of this world!
There is little snow in the UK! But they traveled and found more!


Julep has Solstice or Equinox mystery boxes available. Each box is $24.99. The solistice is a summer themed box and Equinox is fall themed box. Has anyone ordered either one or considering ordering? I used to get Julep monthly but stopped and now I'm considering signing up again but was looking at these mystery boxes instead. I really don't need a monthly sub box for nail polish but I do like the Julep polishes on my toes. The color lasts forever and doesn't chip if used with the base and/or top coat.


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Tamil » Headline News » TikTok sues US government over President Donald Trump's ban
TikTok sues US government over President Donald Trump's ban
Tuesday, August 25, 2020 • Tamil Comments
#Donald Trump #Tik Tok
Chinese video-sharing app TikTok is suing President Donald Trump's United States government in federal court over the administration's decision to ban the app in the country. TikTok has claimed that the executive order, signed by Trump on August 6th under the International Emergency Economic Powers Act, is "without any evidence to justify such an extreme action, and without any due process."
The company further added that the ban prevented TikTok from going through the due process and that the executive order ignored their effort to prove that the app isn't a national security threat as it does not share any data with the Chinese government. Trump's administration processed the impending ban worrying about US data security and data privacy with several Chinese companies, including Huawei and WeChat. TikTok reportedly mentioned in a statement, "Now is the time for us to act. We do not take suing the government lightly, however we feel we have no choice but to take action to protect our rights, and the rights of our community and employees. Independent national security and information security experts have criticized the political nature of this executive order, and expressed doubt as to whether its stated national security objective is genuine."
Claiming that the app is a security threat due to its Chinese owners, Trump's executive order that was signed earlier this month read, "TikTok automatically captures vast swaths of information from its users, including Internet and other network activity information such as location data and browsing and search histories. This data collection threatens to allow the Chinese Communist Party access to Americans' personal and proprietary information -- potentially allowing China to track the locations of Federal employees and contractors, build dossiers of personal information for blackmail, and conduct corporate espionage."
The American civil liberties union argued that banning the app is a danger to free expression. "Banning an app like TikTok, which millions of Americans use to communicate with each other, is a danger to free expression and technologically impractical. To truly address privacy concerns with companies like TikTok, Congress must ensure that ANY company that services US consumers cannot hand over our data to any government without a warrant or equivalent. Letting the president selectively ban platforms isn't the solution," ACLU posted on social media.
US President Donald Trump permanently banned from Twitter; He responds
Important report on schools reopening in Tamil Nadu to be filed today!
Mohammed Siraj reveals reason for crying ahead of Test match
Elon Musk becomes the richest person in the world; This is how he reacted to the news
Iam absolutely fine, ready to fly soon says Ganguly after discharge!
Twitter, Facebook block Trump as violent clash in US Capitol results in four deaths
WhatsApp to stop working for people who do not agree to its new privacy policy - Details
Meera Mitun sends lots of love to Nithyananda and plans to visit Kailasa
Will Jagame Thanthiram release on OTT directly? Producer's official statement!

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As always, our products are often available at various stages of maturation, from root to flower, to best suit your wants and needs. Stick around and explore some of our latest updates here on our website.
Order by Wednesday 4pm for delivery Thursday 11am-6pm.


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Home > News > Korea > More than half of criminals who fled country still at large
More than half of criminals who fled country still at large
SEOUL (Yonhap) — More than half of criminal suspects who have fled the country to escape punishment have not yet been rounded up, a report showed Wednesday.
A total of 3,890 offenders absconded overseas after committing crimes on South Korean soil in the past five years, the report showed. Of that total, 2,175, or 55.8 percent, are still on the run.
Rep. Lee Sang-min of the ruling Saenuri Party released the report based on data the justice ministry submitted to the National Assembly for an annual audit.
China was the most favored destination for fugitives with 707 choosing to go there, followed by the Philippines with 283, Japan with 223 and Hong Kong with 165, the data showed.
By the type of crime, 1,1390 were fraud suspects, followed by 247 people accused of embezzlement, 149 of drug-related crimes and 86 of theft, the data showed.
"We must continue pursuing criminal suspects who flee abroad to bring them to justice," Lee said, adding the government should come up with detailed judicial cooperation with other countries to arrest and extradite the suspects.
BLACKPINK adds 6 Guinness World Record titles
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K-pop boy group Tomorrow X Together has released several concept...
LG Electronics Q4 operating profit collapses on slumping demand, rising cost
LG Electronics Inc. said Friday its operating profit for the...
First lady meets with female PPP lawmakers
First lady Kim Keon Hee met with a group of...
DP accuses prosecution of 'witch hunt' on eve of leader's questioning
The floor leader of the main opposition Democratic Party on...
Nat'l pension anticipated to be fully drained in 2055: NPS
South Korea's state pension operator said Friday its fund is...
S. Korean baseball MVP hires top agent for potential MLB jump: source
The reigning South Korean baseball MVP Lee Jung-hoo has hired Scott Boras...
First lady Kim Keon Hee met with a group...
TXT's new EP exceeds 2 mln copies in preorders
K-pop boy group Tomorrow X Together broke its own...
Actress Yun Jung-hee's funeral to be held in Catholic church near Paris
The funeral for late South Korean actress Yun Jung-hee...


Georgia Tech's Jose Muguruza advanced to the second round of the NCAA Men's Tennis Singles Championships with his victory Wednesday afternoon at the George P. Mitchell Tennis Center in College Station, Texas on the campus of Texas A&M University.
Muguruza, the nation's 22nd ranked singles player dropped the first set of his match, but rebounded with a 3-6, 6-1, 6-4 win over No. 45 Bojan Szumanski of Texas Tech in the first round of the singles tournament. Muguruza advances to the Round of 32 where he will face fourth-seeded Catalin Gard of Ole Miss Thursday morning.
Muguruza now has a 28-13 record on the season, including 11 wins over ranked opponents this year.
In addition to Muguruza, the doubles team of David North and Marko Rajevac will begin play Thursday afternoon, facing Zach Ganger and Miron Mann from the University of Utah.
Livestats for both matches may be followed at ncaatennis.tamu.edu.


Namco Bandai just released a large batch of screrenshots of the upcoming fighting game Saint Seiya: Brave Soldiers, that will be released in North America on the PSN on November the 26th.
The screenshots showcases the Galaxian Wars custom tournament, the survival mode and the gallery modes (card gallery and toy gallery, and in addition to those there will be a special attack gallery, a BGM player and a Player Data viewer).
Online gameplay is also showcased, with the ability to gather one's data on a player card, to play in ranked leagues, to customize one's character via the Orb system, and to add special unlockable titles to your online nickname.
Finally, some characters are introduced: Unicorn Jabu, Hydra Ichi, Eagle Marin, Ophiuchus Shaina, Kanon in his Gemini cloth version, Lyra Orpheus, Gryphon Minos, Wyvern Rhadamanthys and Garuda Aiacos.
You can check all the screenies out in the gallery just below, and if you missed it, an extensive trailer was also released this morning.


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Jerricho Cotchery to sign one year deal with Steelers
Thursday, August 11, 2011 / by William DePaoli / / Inside Access, Pittsburgh Steelers / 0 comments
The Pittsburgh Steelers have finally landed a coveted wide receiver they sought, agreeing to terms with free agent wide out Jerricho Cotchery this afternoon. ESPN reports that the deal is a one year deal.
We are told that the deal has not been offically signed but an agreement between the two sides has been reached.
The Steelers emerged as the only legitimate suitor this week for Cotchery, as the Baltimore Ravens decided not to make an offer on Wednesday. Cotchery visited the Steelers on Sunday and Monday, passing a physical and the two sides remained in contact throughout the week.
What does the signing of Cotchery mean?
The Steelers go into the season with an extremely deep receiving corps with Mike Wallace, Emmanuel Sanders, Hines Ward, Antonio Brown and now Cotchery in their top five.
The addition of Cotchery does not solve the big receiver presence who could stetch the field that the Steelers sought with Plaxico Burress, but Cotchery gives them insurance and great depth in wake of injuries, sophmore slumps or for instance Hines Ward taking a step back.
The Cotchery signing means the end of the Limas Sweed era and is not good news for Tyler Grisham who probably won't make the team now.
The Steelers last year carried six receivers and if they do so again, seem destined to keep that sixth spot for Arnaz Battle due to his abilities on special teams.
The pursuit of Burress and now signing of Cotchery is another sign that the Steelers intend to open things up again like 2009 after Ben Roethlisberger and Bruce Arians had to buy into the "run" mandate from ownership last season.


EU PVSEC - Trends in PV Development – from Turmoil to Mainstream?
Trends in PV Development – from Turmoil to Mainstream?
Costs of solar electricity have fallen so rapidly that new markets, applications and business models are being unlocked faster than is generally realised. The emergence of new applications which have not been on the market development radar before now is offering exciting opportunities to expand the deployment of solar PV. At the same time, the market was driven by the boom of PV installations in China, a boom that was halted by the Chinese decision to control its market. With the largest market going down, what will happen to the PV industry and which markets will compensate this in the coming years? Will this turmoil change the path of PV development in the coming years? While the PV market was expected to continue booming, we may see some "lost" years before it grows again.
This event will first discuss the development of the PV market, and the impact of policies on the speed at which PV could develop. Will policies continue to shape the market and especially long term targets? From China to the USA and from Spain to Japan, this workshop will challenge the future evolution of PV.
The evolution of PV markets is driven by cost declines and policy pushes. But behind the apparent growth, how do policies transform the PV landscape. A review of some key aspects of PV development.
What could drive PV development in the coming years? From innovative applications in buildings to the integration in the transport sector, this session will explore innovation and forward-looking ideas.


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Ring of Honor made it official Monday that international star Marty Scurll has signed with ROH.
In a video update, ROH teased Scurll debuting on the "Reach for the Sky" U.K. tour in November.
Scurll decided to sign with ROH after being part of WWNLive/EVOLVE shows throughout 2016, including challenging for the EVOLVE Title at E64 in July.
PWTorch learned that Scurll gave his notice to EVOLVE officials around that time and he said that he was going to sign with ROH.
ROH played up Scurll's villainous persona in an introductory video released Monday on Twitter.


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Historical Figures Born in 1920 (Part 2)
Birthdays by Year
All Events Birthdays Deaths Weddings & Divorces
Weddings & Divorces
Birthdays 201 - 317 of 317
Jul 27 James Munn, Scottish educationalist and commissioner (British University) (d. 2009)
Jul 29 Neville Jeffress, Australian founder of Media Monitors Australia (d. 2007)
Jul 31 James Esdras Faust, American religious leader, born in Delta, Utah (d. 2007)
Aug 1 Henrietta Lacks [Loretta Pleasant], African-American tobacco farmer whose cancer cells are the source of the HeLa cell line, extensively used in medical research since the 1950s (1st immortalised cell line), born in Roanoke, Virginia (d. 1951)
Aug 2 Louis Pauwels, French writer and editor, born in Ghent, Belgium (d. 1997)
Aug 3 Elmar Tampõld, Estonian-Canadian architect, born in Kärdla, Estonia (d. 2013)
Aug 3 P. D. James [Phyllis Dorothy], Baroness James of Holland Park, English crime writer (Cover Her Face), born in Oxford, England (d. 2014)
Aug 4 Helen Thomas, American reporter and UPI journalist (starts press conferences), born in Winchester, Kentucky (d. 2013)
Aug 5 Sonny Montgomery, American politician (Rep-D-Mississippi, 1967-97), born in Meridian, Mississippi (d. 2006)
Aug 8 Don Cook, foreign Correspondent
Aug 9 Enzo Biagi, Italian journalist and writer, born in Lizzano in Belvedere, Italy (d. 2007)
Aug 11 Derry Jeffares, Honorary Prof of English Studies (Stirling U)
Aug 11 William Masselos, pianist/prof (Juilliard 1976), born in Niagara Falls, New York
Aug 12 Percy Mayfield, American singer (Please Send Me Someone to Love)
Aug 19 Lucila Engels-Boskaljon, Curacao-Dutch painter, born in Willemstad, Curaçao
Aug 21 Christopher R. Milne, son of Alan A. Milne (Winnie the Pooh), born in Chelsea, London (d. 1996)
Aug 22 Anthony Tuke, chairman of Barclays Bank (1973–81) (d. 2001)
Aug 22 Denton Cooley, American heart surgeon (1st artificial heart transplant), born in Houston, Texas (d. 2016)
Aug 22 Pierre A. Lauffer, Antillian poet (Patria), born in Curacao, Venezuela (d. 1981)
Aug 22 Ray Bradbury, American sci-fi author (Fahrenheit 451), born in Waukegan, Illinois (d. 2012)
Aug 22 Wolfdietrich Schnurre, German writer, born in Frankfurt (d. 1989)
Aug 25 Jef Diederen, Dutch painter, born in Heerlen, Netherlands (d. 2009)
Aug 26 Brant Parker, American cartoonist (co-created and drew The Wizard of Oz), born in Los Angeles, California (d. 2007)
Aug 27 James Molyneaux [Baron Molyneaux of Killead], MP (Ulster unionist), born in Killead, Ireland (d. 2015)
Aug 28 Frits Bernard, American clinical psychologist and pedophile activist, born in Rotterdam, Netherlands (d. 2006)
Aug 29 Joseph Lesniewski, American Easy Company, soldier in 101st Airborne, born in Erie, Pennsylvania (d. 2012)
Aug 30 Ben Cami, Flemish writer and poet (Rose from Mud), born in Durham, England (d. 2004)
Sep 1 Hubert Lampo, Flemish writer/essayist (Coming of Joachim Stiller)
Sep 4 Craig Claiborne, American food columnist (NY Times Cookbook), born in Sunflower, Mississippi (d. 2000)
Sep 4 Teddy Johnson, British singer (Pearl Carr & Teddy Johnson), born in Surbiton, Surrey (d. 2018)
Sep 5 Margaretha D. Ferguson-Wigerink, Dutch author (Anna & her Father), born in Arnhem, Netherlands (d. 1992)
Sep 7 Harri Webb, Welsh poet and nationalist, born in Swansea, Wales (d. 1994)
Sep 9 Feng Kang, Chinese mathematician (d. 1993)
Sep 9 Robert Wood Johnson III, American philanthropist (d. 1970)
Sep 10 Fabio Taglioni, Italian motorcycle engineer (Ducati), born in Lugo, Italy (d. 2001)
Sep 12 Jan W. Schulte Nordholt, Dutch poet and historian (Blossoming Stone), born in Zwolle, Netherlands (d. 1995)
Sep 14 Alberto Calderón, Argentine mathematician, born in Mendoza, Argentina (d. 1998)
Sep 14 Lawrence Klein, American economist who created computer models to forecast economic trends (1980 Nobel Memorial Prize in Economic Sciences), born in Omaha, Nebraska (d. 2013)
Sep 14 Mario Benedetti, Uruguayan journalist, novelist and poet, born in Paso de los Toros, Uruguay (d. 2009)
Sep 16 Hannie Schaft, Dutch communist resistance fighter known as the "Girl with red hair", born in Haarlem, Netherlands (d. 1945)
Sep 16 John MacKenzie, Scottish-Canadian bagpiper and pipe-major, born in Edinburgh, Scotland (d. 1996) [1]
Sep 16 Sheila Quinn, British nurse and adviser (British Red Cross), born in Blackpool, England (d. 2016)
Sep 17 Marjorie Holt, American politician (Member of the U.S. House of Representatives from Maryland), born in Birmingham, Alabama (d. 2018)
Sep 20 Jay Ward, American cartoonist (Rocky & his Friends, Bullwinkle), born in Berkeley, California (d. 1989)
Sep 22 Anders Lassen, Danish military officer (posthumously awarded Britain's Victoria Cross), born in Copenhagen, Denmark (d. 1945)
Sep 23 Gerard W Taylor, South African/British surgeon
Sep 25 Sergei Bondarchuk, Soviet director (Destiny of a Man, War and Peace), born in Belozerka, Ukraine (d. 1994)
Sep 27 J K Gill, president (Saatchi & Saatchi)
Sep 29 Peter D. Mitchell, English chemist, (1978 Nobel Prize for Chemistry), born in Mitcham, England (d. 1992)
Oct 2 William Cramond, Scottish psychiatrist and vice-chancellor (Stirling University), born in Aberdeen, Scotland (d. 2004)
Oct 5 Robert Feenstra, Dutch law historian
Oct 6 Lord Donaldson of Lymington, British judge (d. 2005)
Oct 6 Pietro Consagra, Italian sculptor (d. 2005)
Oct 7 Georg Leber, German politician (SPD), (d. 2012)
Oct 8 Frank Herbert, American sci-fi author (Dune series), born in Tacoma, Washington (d. 1986)
Oct 9 Jens Bjørneboe, Norwegian poet and writer (Dikt, Jonas), born in Kristiansand, Norway (d. 1976)
Mario Puzo (1920-1999)
Oct 15 American novelist (The Godfather, Cotton Club, Earthquake), born in NYC, New York
Oct 17 Miguel Delibes, Spanish writer
Oct 19 Pandurang Shastri Athavale, Indian philosopher (d. 2003)
Oct 20 Dov Noy, Polish-born Jewish scholar of Jewish folklore, born in Kolomyja, Poland
Oct 22 Timothy Leary, American psychologist & advocate for psychedelic drug use, born in Springfield, Massachusetts (d. 1996)
Oct 23 Ted Fujita, Japanese meteorologist (d. 1998)
Oct 24 Julius Youngner, American scientist, helped develop Salk polio vaccine, born in Manhattan, New York (d. 2017)
Oct 24 Marcel-Paul Schützenberger, French mathematician, born in Paris (d. 1996)
Oct 26 T. C. Jones, American female impersonator, born in Scranton, Pennsylvania (d. 1971)
Oct 27 K. R. Narayanan, 10th President of India (1997-2002), born in Uzhavoor, Kerala (d. 2005)
Oct 29 Baruj Benacerraf, Venezuelan-American immunologist (Nobel 1980 - discovery of genes that regulate immune responses and of the role that some of these genes play in autoimmune diseases), born in Caracas, Venezuela (d. 2011)
Oct 29 Didymos I, Catholicos of the East and Malankara Metropolitan (2005-10), born in Mavelikkara, India (d. 2014)
Oct 30 Christy Ring, Irish hurler (Cork 65 games, scoring tally [33-208], 8 x All-Ireland medals), born in Cloyne, Ireland (d. 1979)
Oct 31 Dedan Kimathi, Kenyan freedom fighter, born in Nyeri District, Central Province, Kenya (d. 1957)
Oct 31 Helmut Newton, German fashion photographer, born in Berlin, Germany (d. 2004)
Nov 2 Bill Mazer, American sports broadcaster (New York institution in sports reporting; sports trivia expert), born in Iziaslav, Ukraine (d. 2013)
Nov 3 Oodgeroo Noonuccal, Australian Aboriginal political activist and writer (We Are Going), born in Minjerribah, Queensland, Australia (d. 1993)
Nov 4 Georges Papy, Belgian mathematician (Numbers Game), born in Anderlecht, Belgium (d. 2011)
Nov 5 Douglass North, American economist, (Nobel Prize Economic Sciences 1993), born in Cambridge, Massachusetts (d. 2015)
Nov 8 Eugênio de Araújo Sales, Brazilian Roman Catholic cardinal, born in Acari, Rio Grande do Norte, Brazil (d. 2012)
Byron de la Beckwith (1920-2001)
Nov 9 American white supremacist, born in Colusa, California
Nov 9 Seymour Siwoff, American sports statistician (president and CEO Elias Sports Bureau 1952-2019), born in Brooklyn, New York (d. 2019)
Nov 10 Rafael del Pino, Spanish entrepreneur, born in Madrid, Spain (d. 2008)
Nov 11 Roy Jenkins, British MP (Labour, Liberal Democrats) and President of the European Commission (1977-81), born in Abersychan, Monmouthshire (d. 2003)
Nov 14 Johnny Desmond, singer (Your Hit Parade), born in Detroit, Michigan
Nov 15 Wayne Thiebaud, American painter
Nov 18 Bill Bedford, British test pilot (pioneered the development of V/STOL aircraft), born in Loughborough, England (d. 1996)
Nov 18 Mustafa Khalil, 40th Prime Minister of Egypt (1978-80), born in Al Qalyubiyah Governorate, Egypt (d. 2008)
Nov 23 Chalmers Wylie, American politician (Rep-R-Ohio, 1967-93), born in Norwich, Ohio (d. 1998)
Nov 23 Paul Celan [Antschell], German-Romanian poet (Collected Prose)
Nov 24 Anthony Griffin, British Royal Navy admiral (Controller of the Navy, 1971–75; chairman of British Shipbuilders 1977–80). born in Peshawar, British India (d. 1996)
Nov 24 Joseph Olutola Olu Odutola Oguntala, soho club owner
Nov 25 Tuanku Syed Putra ibni Almarhum Syed Hassan Jamalullail, King of Malaysia (d. 2000)
Nov 26 Paul Rodenko, Dutch poet and author (Stolen Lover), born in The Hague, Netherlands (d. 1976)
Nov 27 Elaine Greene, literary Agent
Nov 27 John Richard Ravensdale, historian
Nov 27 Max Tripels, Dutch politician, lawyer, and director, born in Maastricht, Netherlands (d. 1993)
Nov 29 Yegor Ligachev, Soviet politician
Dec 4 Jeanne Manford, American gay rights activist, born in Flushing, New York (d. 2013)
Dec 4 Nadir Afonso, Portuguese geometric abstractionist painter, born in Chaves, Portugal (d. 2013)
Dec 4 Ray Robinson, American magazine editor and biographer (Lou Gehrig), born in Manhattan, New York (d. 2017)
Dec 6 George Porter, British chemist (Nobel 1967), born in Stainforth, South Yorkshire, England (d. 2002)
Dec 7 Tatamkhulu Afrika, South African poet and writer, born in As Sallum, Egypt (d. 2002)
Dec 8 Ronald Gulliford, English educationalist (Dean of the Faculty of Education, Birmingham University 1979-81), born in Manchester (d. 1997)
Dec 9 Carlo Azeglio Ciampi, Italian President (1999-2006) and Prime Minister (1993-94), born in Livorno (d. 2016)
Dec 10 Clarice Lispector, Ukrainian-Brazilian writer (Laços de Família), born in Chechelnyk, Ukrainian People's Republic (d. 1977)
Dec 10 Stanko Todorov, Bulgarian communist politician, Prime Minister of Bulgaria (1971-81), born in Klenovik (d. 1996)
Dec 12 Fred Kida, Japanese-American comics artist, born in Brooklyn, New York (d. 2014)
George P. Shultz (1920-2021)
Dec 13 American economist, and statesman (Secretary of State, 1982-89; Secretary of the Treasury, 1972-74), born in New York City [1]
Dec 13 Kaysone Phomvihane, President of Laos (Pathet Lao) (1991-92), born in Na Seng village, Khanthabouli district (d. 1992)
Dec 14 Albertus Louis Meintser, Dutch office clerk and resistance fighter, born in Amsterdam (d. 1944)
Dec 15 Kurt Schaffenberger, American comics artist (Superman's Girl Friend, Lois Lane), born in Thuringian Forest, Germany (d. 2002)
Dec 17 Kenneth E. Iverson, Canadian computer scientist (d. 2004)
Dec 21 Bob Bindig, American cartoonist (d. 2006)
Dec 24 Evgeniya Rudneva, Russian World War II heroine, born in Berdyansk, Ukraine (d. 1944)
Dec 24 Stormé DeLarverie, American gay rights leader (Stonewall protests), born in New Orleans, Louisiana (d. 2014) date disputed
Dec 25 Henry Charnock, oceanographer
Dec 28 Bruce McCarty, South Bend, Indiana, architect (1982 World Fair), (d. 2013)
Dec 29 Syd Dernley, British hangman (1949-54), born in Mansfield Woodhouse, Nottinghamshire (d. 1994)
Dec 30 Aad de Haas, Dutch religious painter, graphic artist and cartoonist, born in Rotterdam, Netherlands (d. 1972)
Dec 30 Michael Allinson, London England (George Washington)
Birthdays in History by Year


text:
package org.wannatrak.mobile.controller;

import org.wannatrak.mobile.model.Position;
import org.wannatrak.mobile.model.Cell;
import org.wannatrak.mobile.controller.cellid.dbclient.CellDBClient;
import org.wannatrak.mobile.controller.cellid.dbclient.CellDBOrgClient;
import org.wannatrak.mobile.controller.cellid.dbclient.OpenCellIDClient;
import org.wannatrak.mobile.controller.cellid.*;

public class CellIDFacade {

    private final CellDBClient[] dbClients;
    private final CellIDInfoProvider cellIDInfoProvider;
    private final Controller controller;

    public CellIDFacade(Controller controller) {
        this.controller = controller;

        dbClients = new CellDBClient[] {
                new OpenCellIDClient(controller),
                new CellDBOrgClient(controller)
        };

        CellIDInfoProvider defaultCellIDInfoProvider = new DefaultCellIDInfoProvider();
        CellIDInfoProvider motorolaCellIDInfoProvider = new MotorolaCellIDInfoProvider();
        CellIDInfoProvider samsungCellIDInfoProvider = new SamsungCellIDInfoProvider();
        CellIDInfoProvider nokiaS40CellIDInfoProvider = new NokiaS40CellIDInfoProvider();
        CellIDInfoProvider nokiaS60CellIDInfoProvider = new NokiaS60CellIDInfoProvider();
        CellIDInfoProvider seCellIDInfoProvider = new SonyEricssonCellIDInfoProvider();
        CellIDInfoProvider siemensCellIDInfoProvider = new SiemensCellIDInfoProvider();

        if (nokiaS40CellIDInfoProvider.doesWorkForPlatform()) {
            this.cellIDInfoProvider = nokiaS40CellIDInfoProvider;

//            controller.showLog("Nokia S40 Cell ID Provider");
        } else if (nokiaS60CellIDInfoProvider.doesWorkForPlatform()) {
            this.cellIDInfoProvider = nokiaS60CellIDInfoProvider;

//            controller.showLog("Nokia S60 Cell ID Provider");
        } else if (seCellIDInfoProvider.doesWorkForPlatform()) {
            this.cellIDInfoProvider = seCellIDInfoProvider;

//            controller.showLog("Sony Ericsson Cell ID Provider");
        } else if (motorolaCellIDInfoProvider.doesWorkForPlatform()) {
            this.cellIDInfoProvider = motorolaCellIDInfoProvider;

//            controller.showLog("Motorola Cell ID Provider");
        } else if (samsungCellIDInfoProvider.doesWorkForPlatform()) {
            this.cellIDInfoProvider = samsungCellIDInfoProvider;

//            controller.showLog("Samsung Cell ID Provider");
        } else if (defaultCellIDInfoProvider.doesWorkForPlatform()) {
            this.cellIDInfoProvider = defaultCellIDInfoProvider;

//            controller.showLog("Motorola (Old) Cell ID Provider");
        } else if (siemensCellIDInfoProvider.doesWorkForPlatform()) {
            this.cellIDInfoProvider = siemensCellIDInfoProvider;

//            controller.showLog("Siemens Cell ID Provider");
        } else {
            this.cellIDInfoProvider = null;

//            controller.showLog("Cell ID Provider not found");
        }
    }

    public boolean isCellIDAvailable() {
        return cellIDInfoProvider != null;
    }

    public Cell getCell() {
        if (!isCellIDAvailable()) {
            return null;
        }
        return cellIDInfoProvider.getCell();
    }

    public Position getPosition() {
        if (cellIDInfoProvider == null || dbClients == null || dbClients.length == 0) {
            return null;
        }

        final Cell cell = cellIDInfoProvider.getCell();
        if (!controller.setCellIfNew(cell)) {
            return null;
        }

        Position position = null;
        for (int i = 0; i < dbClients.length; i++) {
            position = dbClients[i].getPosition(
                    cell.cellID,
                    cell.mobileCountryCode,
                    cell.mobileNetworkCode,
                    cell.locationAreaCode
            );
            if (position != null) {
                if (position.getLatitude() == 0 && position.getLongitude() == 0) {
                    position = null;
                } else {
                    break;
                }
            }
        }

        return position;
    }

    public void postCellPosition(Cell cell, Position position) {
        for (int i = 0; i < dbClients.length; i++) {
             dbClients[i].postCellPosition(
                    cell.cellID,
                    cell.mobileCountryCode,
                    cell.mobileNetworkCode,
                    cell.locationAreaCode,
                    position.getLatitude(),
                    position.getLongitude(),
                    position.getTimestamp()
            );
        }
    }
}

redpajama_set_name: RedPajamaGithub


"Can I drink alcohol on low carb?" is very common question on our Low Carb Facebook page. The short answer: it depends. If you're not inAtkins induction and you've decided the occasion warrants, you can drink in moderation without negatively impacting your low carb lifestyle. Yay!
What should you consider when deciding to drink alcohol on low carb?
Alcohol provides a readily available fuel source for your body. While you're drinking, your body postpones fat burning and uses up that alcohol exclusively. If you're not using carb-laden mixers, you go right back to Ketosis once the alcohol has been burned up, but regular drinking will undeniably slow or stall weight loss efforts.
Alcohol is very calorie-dense. While most low-carbers don't have to count calories, it doesn't mean they don't exist.
Alcohol also tends to loosen inhibitions, which means you may make choices about other eating (or God knows what else!) that you wouldn't otherwise. Don't let a few drinks propel you into a junk-food-frenzy!
Alcohol on low carb is well-known for a dramatically lower tolerance level. Please be prepared for this and proceed with caution. You are officially a cheap date!
What are the best choices for drinking alcohol on low carb?
Distilled spirits straight or with sugar free mixers, dry wine (not the sweet stuff) or light beer are your best bets. Note that "tonic water" comes in at 32 carbs for a 12-oz. can, so don't let the name make you think it's a good mixer choice.
Here are some recipes for Low Carb mixed drinks. Some use ingredients like Liquid Splenda, Flavored Sugar Free Syrup, or even extracts o add the flavoring. Like everything else about this lifestyle, you'll have to determine for yourself what sweeteners, mixers or choices work for your individual situation. But whatever you choose, stay safe out there!
Do you drink on your low carb plan?
I love my glass (or 2) of wine in the evening! That said a light beer will drop my ketones down to 0.4 for a day or 2. So sad! I love beer, but it turns out drinking beer at age 52 just doesn't work for me.
Don't forget, there is Diet Tonic water for 0 cals.
Great video Bob!! You have a gift for teaching in a way easy to understand# Do you have more videos? I.could listen to you all day! Thanks for sharing your knowledge and life experience with us!!!
What about Michelob Ultra? Can I just count it into my daily carb allowance?
Sorry, kelly, just saw this. Anything like that, I'd keep it occasional but if I'm going to drink a beer, that's generally the one I pick.
What about flavor vodka over ice?
The information I'm seeing in searching on it indicates the process of flavoring doesn't add carbs to the vodka. So the same cautions as in the article apply, but not extra ones. Be safe out there!
I noticed that my sense of smell has gotten a lot more sensitive and a little alcohol goes a long way. One glass of medium priced wine….Shiraz is my favorite….is all I need for the evening. Also there are health benefits. It's been well documented that moderate amounts of alcohol can raise your good cholesterol (HDL-cholesterol) and thin your blood. This is thought to be one of the primary cardiovascular benefits from wine.
I do fairly strict low carb low calorie days and drink liquor in the evenings. Cuts the weight loss down to only 1-2 lbs/week. Provided I don't eat anything after drinking. But I'd rather have a nice evening buzz and lose gradually than not have any fun and lose weight rapidly.


text:
Press: Discovery Institute Press
Debating Darwin's Doubt: A Scientific Controversy That Can No Longer Be Denied
Edited by David Klinghoffer
In 2013, Stephen Meyer's book Darwin's Doubt became a national bestseller, provoking a wide-ranging debate about the adequacy of Darwinian theory to explain life's history. In Debating Darwin's Doubt: A Scientific Controversy That Can No Longer Be Denied, leading scholars in the intelligent design community respond to critiques of Meyer's book and show that the core challenge posed by Meyer remains unanswered: Where did the influx of information essential to the creation of new body plans come from?
In addition to several essays by Stephen Meyer, Debating Darwin's Doubt also includes contributions from biologists Richard Sternberg, Douglas Axe, and Ann Gauger; philosopher of biology Paul Nelson; mathematicians William Dembski and David Berlinski; and Center for Science and Culture research coordinator Casey Luskin. In more than forty chapters, these contributing authors explore topics such as orphan genes, cladistics, small shelly fossils, protein evolution, the length of the Cambrian explosion, the God-of-the-Gaps objection to intelligent design, and criticisms raised by proponents of theistic evolution. Anyone who wants to understand the cutting-edge of current scientific debates over modern Darwinian theory needs to read this book.


Boost mailing page: Re: [boost] Boost packages?
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>> Unfortunately the link to beman's original proposal is dead :(.
>> idea to get that back into the collective consciousness.
> understand what went on.
just an idea. If you want to discuss it now, be my guest.
> happened to go right.
a few things would be useful.


The header is printed on the first page only as I need, but the header box (empty) still present on other pages. How to get rid of it?
A mathematical method based on Green`s function approach widely used, in particular, for solving problems in mathematical physics and deformable body mechanics which allows to construct controls providing approximate controllability is suggested in the present paper. Representing the solution of governing system via Green's formula and forcing the controlled state function to satisfy required terminal conditions, we obtain implicit representation for admissible controls. Choosing appropriate controls, we can provide required accuracy of approximation for terminal condition. Particularly, infinite string controlled by a concentrated force and semi-infinite rod heated by a point heat source are considered. Examples illustrating the procedure are described. Results of numerics are brought.
If I understand your setup correctly, there's actually no need to define a header block just for the first page. Instead, I suggest you do something like the following.
A mathematical method based on Green's function approach widely used, in particular, for solving problems in mathematical physics and deformable body mechanics which allows to construct controls providing approximate controllability is suggested in the present paper. Representing the solution of governing system via Green's formula and forcing the controlled state function to satisfy required terminal conditions, we obtain implicit representation for admissible controls. Choosing appropriate controls, we can provide required accuracy of approximation for terminal condition. Particularly, infinite string controlled by a concentrated force and semi-infinite rod heated by a point heat source are considered. Examples illustrating the procedure are described. Results of numerics are brought.
Since you say that the bit of text is part of the title, not actually a heading ... don't use the mechanism to generate a heading.
Do it like you would do it with any other title, add a bit of space by hand.
Not the answer you're looking for? Browse other questions tagged header-footer fancyhdr or ask your own question.
Suppress Fancy header and footer on first page only.
How to get a separate header only for the first page?


This notice is to advise the user groups identified below of a recently-discovered, /proc filesystem permission vulnerability. The issue was reported directly to Joyent Engineering by a security researcher.
This high-severity vulnerability exists in the core SmartOS platform. The exploit allows non-root users to create objects in the /proc directory within the zone. The validations for filesystem permissions have been hardened to prevent such unauthorized actions.
Joyent has created a new Platform Image (PI) containing fixes that address these vulnerabilities. This PI has been applied across the Triton public cloud.
All necessary fixes have been applied to the Triton Cloud (public cloud). No user action is required.


Out of Body Explorer Sticker | Starseed Supply Co.
The official sticker for Out of Body Explorers, Lucid Dreamers, and Astral Travelers! For those who leave their body at night to go on a spiritual quest, play around in the astral plane, or look for the deepest truths in the Universe. Whether your a dream yogi, veteran psychonaut, lucid dreamer, or casual "random" experiencer, this is for you. You can give yourself or a friend a "merit badge" of achievement if you've had at least one out of body experience in your life.
Have you ever felt the vibrational state? Or do you recognize the names of Robert Monroe, William Buhlman, or Todd Acamesis? You can use the "out of body" state to explore consciousness, access the akashic records, or discover spiritual truths for yourself. Don't take the word of any guru out there, experience this for yourself.
Or perhaps you've been having trouble trying to induce an out of body experience yourself. I wrote this guide here http://jefffinley.org/obe. If you manage to experience even a partial OBE, then you would qualify for this badge.
I'm so glad that Starseed finally offers stickers. I have a couple of their patches. Very cool! Love the stickers too!!


text:
Pohledecká skála je zeměpisné jméno skalnatého hřbetu s vrcholem 812 m n. m., významný bod a geologická lokalita v Pohledeckoskalské vrchovině. Leží na katastrálním území Pohledec, části Nového Města na Moravě, v okrese Žďár nad Sázavou náležejícím do Kraje Vysočina v České republice.

Geomorfologický útvar vytvořený mrazovým zvětráváním a typický pro krajinnou oblast dal název členité Pohledeckoskalské vrchovině. Skalní systém se nachází přibližně 2 km na severovýchod od kapličky v Pohledci a 10 km jihovýchodně od přírodní památky Devět skal. Na vrcholu oplocený vojenský objekt.

Zalesněný úzký skalnatý hřbet Žďárských vrchů situovaný SZ – JV tvoří dvojslídný migmatit až ortorula, vrcholová část se skalními útvary je zhruba 400 m dlouhá v rozsahu nadmořských výšek 790–812 m, nejvyšší skalisko je v oploceném a nepřístupném objektu. Menší izolované skály leží mimo objekt na hranici nepřístupného území I. zóny Chráněné krajinné oblasti Žďárské vrchy. Důvodem zákazu vstupu na vymezené území je nerušený průběh hnízdění chráněných ptáků a ochrana přírodního bohatství.

Skalní systém je částečně viditelný z lesní cesty na hranici nepřístupné zóny.

Odkazy

Reference

Související články
 Chráněná krajinná oblast Žďárské vrchy
 Pohledeckoskalská vrchovina
 Žďárské vrchy

Externí odkazy
 CHKO Žďárské vrchy – geomorfologie

Hory a kopce v okrese Žďár nad Sázavou
Hory a kopce v Hornosvratecké vrchovině
Kopce pod 1000 metrů v Česku


text:
Jump Community
Jump Fit
Jump Inc Leeds
Jump Inc Meadowhall
Jump Inc Rotherham
Mission Christmas: Accomplished!
Jump Community // Jump Inc Rotherham // News
Thank you to everyone who donated either a present or cash to Hallam FM's Cash for Kids appeal.
Jump Inc Rotherham acted as a drop-off point for all our generous Jumpers who donated a present this Christmas. Altogether we had over 60 presents to hand to Hallam FM!
With your help, Hallam FM has been able to support 362,694 disabled and disadvantaged children across the UK this Christmas.
One of the places that received the gifts from Hallam FM was Yorkshire-based support group Little Hiccups.
Abi Griffin from the group told Hallam FM: "Thank you so much for donating the gifts. The children really appreciate it and the parents appreciate it. It is a way to make this day extra special and they all go off with a gift, we are really grateful.
"There are families here who wouldn't be able to afford the presents. We've got some families who have three, four or five children and just for them all to receive a gift like this, is extra special.
"As a parent with a child with a disability, it's good to know that the community is behind us and supporting us."
Awesome stuff! And you can still help to make a difference by clicking here


text:
Career of Evil by Robert Galbraith
by Athena // March 25, 2016 // 2 Comments
When this book was first released last October, reviewers noted how dark it was. Indeed, this book is very heavy in tone and creepyness. Much more so than the previous two books. Even though the second book had its strange oddities too. I found some of the chapters from the perspective of the villain hard to get through. Still, I finished this book in two evenings so it was not that bad.
The other minor quibble I have with this book is the relationship between Robin and Cormoran. The constant push and pull between them is getting tiring. It is not say that I either do or do not want them together, but the constant speculation in the book from all sorts of characters is overdone now. Secondly, I am finding Robin is a bit too perfect. There is even a line in this book that says "everyone likes Robin" especially animals. I like Robin too and I guess her biggest flaw is her taste in men.
I continue to love many of the supporting or tertiary characters. In this book, I like the new side characters such as Wardle and Shanker. Rowling does have a way with dialogue and quick backstory that reveals a lot about the characters. I like all of Cormoran's associates except I do not care (and do not think we are suppose to care) for his female romantic partners.
I have notied that Rowling's writing has improved too overall over the years. I like her descriptions even more. One of the women in this book "had a look of a Bruegel peasant, with her rounded cheeks, prominent chin and wide nose." She also continues to be verbose. In this novel, some words that I rarely use, but would like to: chicanery, holdall, pierrot, and perpatetic. I am glad she is writing for adults.
Finally, one thing I love about this series is how the setting is a supporting character in itself. I know Rowling does not live in London, but she writes it extremely well especially the current modern London. I had to laugh out loud because my alma matter is mentioned and couple of scenes in the book are set in the area.
The following excerpt is one of my favourites of the city that I've read recently:
Nobody who had not lived there would ever understand that London was a country unto itself. They might resent it for the fact that it held more power and money than any other British city, but they could not understand that poverty carried its own flavor there, where everything cost more, where the relentless distinctions between those who had succeeded and those who had not were constantly, painfully visible. The distance between Elin's vanilla-coloumned flat in Clarence Terrace and the filthy Whitechapel squat where his mother had died could not be measured in mere miles. They were separated by infinite disparities, by the lotteries of birth and chance, by faults of judgment and lucky breaks. (p. 214)
The line about poverty and the relentless distinctions are very true to London. To any big city? Yes, but to a city in a country where classism still has history, the side by side of the haves and have-nots is both viscereal and subtle.
Reading this book has made me look forward to the TV adaptation that will be forthcoming. I wonder who will be cast and how and when they get the right tone for the show.
I have a knack for reading these novels during the season they are set. During this one, Easter weekend came and went too.
I enjoy reading these books, but I do know that there are issues with the style of the plotting and some other character issues. Still, I can not deny that I enjoy Rowling as a writer and have seen I was a kid. In a couple of years, I'll have said that I've been reading this woman's books for twenty years Keep at it, Ms Rowling.
Read March 21-22, 2016.
The Silkworm by Robert Galbraith
The Cuckoo's Calling by Robert Galbraith
Choose Your Own Autobiography by Neil Patrick Harris
Regency Buck by Georgette Heyer
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Geoff W says: March 25, 2016 at 10:17
I know what you mean about the back and forth with Robin and Cormoran, but not so sure about the Robin perfection. I feel like this book showed us the cracks in her facade and that she became more of a real character for me. I still need to watch the last TV adaptation of a Rowling novel (Casual Vacancy), I didn't know they were adapting this one! Are they doing the whole series?
athena says: March 26, 2016 at 13:35
Hi Geoff! I still really like Robin, and I do not think she is a Mary Sue. At the beginning of the novel, I started to realize she was a wee bit perfect. Even her cracks make her a great character because in the end, everything worked out for her even when she was rash. But as I say, I think her weakness is steadfastness to her partner. They are going to the do whole series, with one season/series per book. The script writer who adapted Casual Vacancy is adapting the first book. However, the news about the TV show officially titled "The Cormoran Strike Mysteries" has been quiet which makes me wonder if it'll be pushed forward to 2017 rather than 2016 air date that people were speculating. I'm looking forward to it because I think the books have great TV potential if they cast the right actors. Thanks for the comment. Happy Easter weekend!


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Geri Richmond is making stops in Thailand and Vietnam this month in her first official trip as a U.S. science envoy.
Richmond, a UO chemistry professor and Presidential Chair, was among four science envoys named Dec. 4 by the U.S. State Department. She left Monday on a three-week excursion aimed at strengthening U.S. science and education in her assigned region.
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