Your favourite Dragon Ball Z characters get the Pop! Vinyl treatment! Standing about 3 3/4-inches tall, this Dragon Ball Super – Goku (Ultra Instinct Form) Pop! Vinyl figure is packaged in a window display box and stands approx. 10 cm tall.


text:
package com.google.template.soy.shared.internal;

import com.google.template.soy.exprtree.TemplateLiteralNode;
import com.google.template.soy.soytree.SoyFileNode;
import com.google.template.soy.soytree.SoyTreeUtils;
import com.google.template.soy.soytree.TemplateNode;
import java.util.LinkedHashSet;
import java.util.Set;

/**
 * Visitor for finding the templates called in a file that are not defined in the file.
 *
 * <p>Important: Only deals with basic callees (not delegates). Calls to delegates are not
 * applicable here because we cannot tell at compile time which delegate will be called (if any).
 *
 * <p>{@link #exec} should be called on a {@code SoyFileNode}. The returned set will be the full
 * names of all templates called by the templates in this file that that not in this file. In other
 * words, if T is the set of templates in this file and U is the set of templates not in this file,
 * then the returned set consists of the full names of all templates in U called by any template in
 * T.
 */
public final class FindCalleesNotInFile {
  public static Set<TemplateLiteralNode> findCalleesNotInFile(SoyFileNode soyFileNode) {
    Set<String> templatesInFile = new LinkedHashSet<>();
    Set<TemplateLiteralNode> calleesNotInFile = new LinkedHashSet<>();
    for (TemplateNode template : soyFileNode.getTemplates()) {
      templatesInFile.add(template.getTemplateName());
    }
    for (TemplateLiteralNode templateLiteralNode :
        SoyTreeUtils.getAllNodesOfType(soyFileNode, TemplateLiteralNode.class)) {
      String calleeName = templateLiteralNode.getResolvedName();
      if (!templatesInFile.contains(calleeName)) {
        calleesNotInFile.add(templateLiteralNode);
      }
    }
    return calleesNotInFile;
  }

  /** Non-instantiable. */
  private FindCalleesNotInFile() {}
}

redpajama_set_name: RedPajamaGithub


Mutations in MYBPC3 encoding myosin binding protein C belong to the most frequent causes of hypertrophic cardiomyopathy (HCM) and may also lead to dilated cardiomyopathy (DCM). MYBPC3 mutations initially were considered to cause a benign form of HCM. The aim of this study was to examine the clinical outcome of patients and their relatives with 18 different MYBPC3 mutations.
87 patients with HCM and 71 patients with DCM were screened for MYBPC3 mutations by denaturing gradient gel electrophoresis and sequencing. Close relatives of mutation carriers were genotyped for the respective mutation. Relatives with mutation were then evaluated by echocardiography and magnetic resonance imaging. A detailed family history regarding adverse clinical events was recorded.
In 16 HCM (18.4%) and two DCM (2.8%) index patients a mutation was detected. Seven mutations were novel. Mutation carriers exhibited no additional mutations in genes MYH7, TNNT2, TNNI3, ACTC and TPM1. Including relatives of twelve families, a total number of 42 mutation carriers was identified of which eleven (26.2%) had at least one adverse event. Considering the twelve families and six single patients with mutations, 45 individuals with cardiomyopathy and nine with borderline phenotype were identified. Among the 45 patients, 23 (51.1%) suffered from an adverse event. In eleven patients of seven families an unexplained sudden death was reported at the age between 13 and 67 years. Stroke or a transient ischemic attack occurred in six patients of five families. At least one adverse event occurred in eleven of twelve families.
MYBPC3 mutations can be associated with cardiac events such as progressive heart failure, stroke and sudden death even at younger age. Therefore, patients with MYBPC3 mutations require thorough clinical risk assessment.
Hypertrophic cardiomyopathy (HCM) is a common disease with an estimated prevalence of 1:500 in north americans . The disease may remain without clinical symptoms for many years. HCM has been recognized as the most common cause of sudden cardiac death in the young, especially in competitive athletes, where it accounts for 26% of cases . Therefore, early diagnosis is crucial for prevention of such catastrophic events. For risk stratification, an expert based recommendation exists, which is mainly based on clinical findings such as maximum wall thickness , abnormal blood pressure response during exercise and non-sustained ventricular tachycardia . Additionally, a positive family history for sudden cardiac death as a correlate of malignant gene mutations was recognized as an independent risk factor. Most cases of HCM are a result of mutations in genes encoding the sarcomeric proteins β-myosin heavy chain (MYH7), myosin binding protein C (MYBPC3), troponin T (TNNT2), troponin I (TNNI3), cardiac α-actin (ACTC) and α-tropomyosin (TPM1). Certain mutations in MYH7 and TNNT2 were ascribed a poor disease prognosis , while MYBPC3 mutations were attributed a more benign course and a low disease penetrance [6, 7].
Dilated cardiomyopathy (DCM) is a major cause of heart failure in the young and accounts for about half of all heart transplantations. It is estimated that up to 35% of DCM cases are familial, indicative of potential gene mutations . While HCM is mostly caused by mutations in sarcomeric protein genes, the genetic causes of familial DCM are more heterogeneous and hence, are less well defined. Up to 10% of familial DCM cases may be the result of mutations in sarcomeric protein genes like MYH7, MYBPC3 and TNNT2 . Besides these genes, many others encoding cytoskeletal and nuclear envelope proteins, channel proteins and those encoding factors involved in calcium cycling were identified as candidates for DCM (for review ).
Here we studied the clinical outcome of families with HCM or DCM and mutations in MYBPC3, particularly with respect to adverse events like progressive heart failure, sudden cardiac death and stroke.
Patients presenting at the catheterization laboratory, cardiac MRI or outpatient department of our university hospital with a new or previously established diagnosis of a primary HCM or DCM were recruited for the study after written consent. Patients from external cardiologists were included, if a written consent and a blood sample was available. The diagnosis of primary HCM or DCM was established according to international standards. In short, HCM was defined by left ventricular wall thickening of 13 mm or more in the absence of secondary causes . DCM was defined as depressed left ventricular ejection fraction less than 45% and a left ventricular end-diastolic diameter (LVEDD) of more than 117% of the expected value . Echocardiography was performed in all patients according national and international standards. LVEDD was obtained from M-Mode recordings, maximum wall thickness and left ventricular ejection fraction (LVEF) form two dimensional echocardiography. LVEF was classified as follows: Normal ≥ 55%, mildly impaired 45–54%, moderately 30–44% and severely < 30% . The study protocol was approved by the ethics committee of the medical faculty at the University of Heidelberg.
After exclusion of contraindications, cardiac MRI was performed in supine position at end-expiration employing an 1.5 Tesla, whole body superconducting MRI scanner system (ACHIEVA, Philips Medical Systems, Best, the Netherlands) using a five-element cardiac phased array surface coil with vector ECG-gating. Assessment of resting LV function was determined by cine images using a steady-state free precession sequence (matrix = 160/256, sense-factor = 2, flip-angle = 60°, slice thickness/gap = 8/2 mm) in continuous short axes covering the whole left and right ventricle from base to apex as well as 2-, 3- and 4-chamber views in anatomically correct heart axes. MRI data was transferred to a clinical workstation (Viewforum, Philips Medical Systems, Best, the Netherlands). End-diastolic (EDV, ml) and -systolic (ESV, ml) volumes and resulting ejection fraction (EF in %) were measured manually using short axes volumetry by determination of end-diastolic and -systolic cine images and subsequent delineation of endocardial borders excluding papillary muscles. For evaluation of myocardial mass, borders were also drawn at the interface between myo- and epicardium on end-diastolic images including papillary muscles. For anatomical description of late enhancement localisation, the AHA 17 segment model was employed consisting of six basal, six mid-ventricular, four apical segments as well as the apex. The signal intensities of late enhancement were described qualitatively (massive, normal, discrete) by consensus decision of the two observers.
All first and second degree relatives of patients with a MYBPC3 mutation were asked to participate in our family screening program. Screening always included ecg and echocardiography. If an examination had been already performed by an external cardiologist, patients were asked for permission to request the examination record from their physicians. Furthermore, all relatives were invited to participate in the genetic screening and, if screened positive for a mutation, for additional cardiac MRI. This was performed in 16 of 42 mutation carriers. If possible, a pedigree of up to four generations together with a detailed family history was recorded. Deceased family members were considered affected if a sudden unexplained death occurred at or below 35 years of age . Affection was presumed if a family member suffered from sudden death at 50 years of age or younger. Genetic counselling was offered to all participants and their families.
18 different mutations (four splice, three frameshift, seven missense, three nonsense mutations and one in-frame codon deletion), distributed throughout almost the entire gene, from intron 2 to exon 34, were found in 18 patients (Tables 1 and 2, Fig. 1). Close relatives of mutation carriers were subsequently genotyped for the respective mutation. Seven mutations were considered novel, since they were neither found in the large MYBPC3 mutation list of the "Cardiogenomics" database http://genetics.med.harvard.edu/~seidman/cg3/muts/MYBPC3_mutations_TOC.html, nor in the SNP database dbSNP http://www.ncbi.nlm.nih.gov/projects/SNP/, nor in one of recent articles found in PubMed http://www.ncbi.nlm.nih.gov/sites/entrez?db=pubmed describing novel mutations in the MYBPC3 gene. The causal role of the new mutations in the disease was underscored by their absence in 430 control individuals with normal systolic and diastolic function and, in one case (family 16), familial co-segregation with the disease. Moreover, four of the seven new mutations lead to a significant change of the amino acid sequence, namely two splice site mutations, one frameshift mutation and one nonsense mutation. A fifth mutation, p.R272C, altered one of three known phoshorylation motifs of the MYBP-C protein . Preliminary analyses revealed a threefold decrease in the phosphorylation of a mutant p-R272C MYBP-C peptide fragment compared to the wild type fragment (unpublished data), yet the functional consequence of the mutation in vivo is still unknown. The two remaining missense mutations are considered functionally relevant since both affect highly conserved amino acid positions (Fig. 2).
Pedigrees of affected families: Open and black symbols represent unaffected and affected individuals, respectively. Dashed symbols indicate individuals with presumed or borderline disease phenotype. Grey symbols denote individuals with unknown clinical status. (+) and (-) symbols indicate presence and absence of the mutation, respectively. Index patients are marked by an arrow. Slanted bars denote deceased individuals.
Conservation of mutated MYBPC3 amino acid residues throughout different species.
Two new mutations were found in DCM patients of families 5 and 16, while all other mutations were detected in patients with HCM. One patient was a double-heterozygote for two different missense mutations located in exons 7 and 28. Members of the two unrelated families 17 and 18 had the same Gln1233ter nonsense mutation, which terminates the MYBP-C protein prematurely, leading to loss of 42 of the 1274 amino acids. This mutation had been described previously as a polymorphism , based on its presence in 2% of 200 phenotypically uncharacterized control individuals. We and others, however, considered the mutation disease-causing, since it was neither detected in 350 controls in previous studies [18, 19], nor in our 430 controls. Moreover, the mutation clearly segregated with the disease in all five mutation carriers of family 17, either associated with a HCM phenotype or with borderline myocardial hypertrophy (IV-2).
To exclude mutations in other genes of high relevance in cardiomyopathies, the 18 initial mutation carriers were screened additionally for mutations in MYH7, TNNT2, TNNI3, ACTC and TPM1 encoding β-myosin heavy chain, cardiac troponin T, cardiac troponin I, cardiac α-actin and α-tropomyosin, respectively. No further mutations were found in these genes.
We observed a broad spectrum of phenotypes among the mutation carriers: from asymptomatic and phenotypically normal at the age of 85, like I-2 of family 5 (Table 2), to heavily affected at a relatively young age, like III-9 of family 16 at the age of 44 or IV-3 of family 12 at the age of 13 (Tables 2 and 3). Even close relatives carrying the same mutation displayed a striking phenotypic heterogeneity, as documented by members of families 5, 12, 16 and 17.
The index patient of family 5, II-2, initially presented with decompensated heart failure during new onset of tachyarrhythmic atrial fibrillation and arterial hypertension. In addition, cardiac catheterization ruled out significant coronary artery disease, but revealed an enlarged left ventricle with severely depressed systolic function. The patient improved, however, under guideline-conform treatment, as documented four years later by cardiac MRI displaying normalized left ventricular function and only minor pathological findings regarding wall thickness and myocardial late contrast enhancement. The son, III-2, had a mild enlargement of the left ventricle with an end-diastolic volume index of 98.1 ml/m2 during MRI (reference value 47–92 ml/m2). However, he and his father showed a marked trabeculation on cardiac MRI. This finding did not fulfil the criteria for myocardial noncompaction, but might be an explanation for the overestimated wall thickness by echocardiography. Two further mutation carriers of family 5, I-2 and II-6, were asymptomatic and, as revealed by cardiac MRI, showed no phenotypical abnormalities.
In family 16, the index patient, II-3, and his brother, II-5, had a DCM phenotype. In contrast, the other mutation carriers, all belonging to the third generation of family 16, displayed a HCM phenotype upon cardiac MRI examination, yet were otherwise asymptomatic. Only in individual III-3, a HCM was diagnosed before during a routine physical examination.
The index patient of family 12 (III-3) had a severe left ventricular hypertrophy with outflow obstruction, which required septal ablation. His son (IV-3), who suffered from sudden cardiac death at the age of 13 years, also had a severe hypertrophy. In contrast, the index patient's mother (II-6), also mutation carrier, had only a borderline hypertrophy. In this patient, a comorbidity with a coronary heart disease and previous inferior infarction was prevalent, and the ventricular arrhythmias found at holter monitoring might be a result from a myocardial scar after inferior infarction.
In family 17, the index patient, II-3, presented at the age of 72 years with a leading finding of a restrictive pathophysiology and only a mild wall thickening, which probably was a result of an end-stage HCM. Unfortunately, high-quality cardiac MRI recording was impossible due to atrial fibrillation. All three children of this patient were mutation carriers; only one was previously diagnosed with HCM, while the other son and the daughter were diagnosed with HCM in the course of our family screening.
Malignant clinical events such as unexplained sudden death, stroke or transient ischemic attack were ascertained in many members of our study families (Table 3). Based on twelve families and six single patients, a total number of 42 mutation carriers was identified, of which eleven had at least one adverse event. A total number of any adverse event occured in 23 of 45 (51.1%) cardiomyopathy cases. Eleven individuals of seven families suffered from sudden death between 13 and 67 years of age. In three families, a sudden death occurred at or before the age of 35 years. Any adverse event occurred in eleven of twelve families and in two of six single mutation carriers. The youngest, IV-3 of family 12, already diagnosed with an extreme septal hypertrophy and a dynamic outflow obstruction, died at the age of 13. Moreover, a first degree relative, II-3, of a mutation carrier in the same family experienced sudden death at the age of 33. In family 16, a sudden death occurred only shortly after the diagnosis of HCM had been ascertained in the course of family screening. The female patient, III-9, had no heart failure symptoms but a maximum wall thickness of 22 mm, moderate late enhancement during contrast MRI and mild elevated NT-proBNP. Clinical findings in the patient did not qualify for an implantable cardioverter defibrillator (ICD) based on available risk scores . A formerly asymptomatic individual, II-1, of the same family suffered from sudden cardiac death at the age of 32 in relation to exercise. This patient had not been genotyped, yet can be considered mutation carrier based on the fact that his two brothers, II-3 and II-5, and two symptomatic children, III-3 and III-6, are mutation carriers.
Five patients of four families received an ICD for primary prevention because of non-sustained ventricular tachycardia or other high-risk disease profiles. Stroke or a transient ischemic attack related to atrial fibrillation occurred in six patients of five families between 51 and 64 years of age. Two of these patients had persisting disabling symptoms. Two patients with stroke and two with a transient ischemic attack recovered without permanent impairment.
Sudden death as the most serious adverse event was prevalent in seven of 18 families (39%). However, despite missense mutations were most frequent with seven of 18 index cases, none of the sudden deaths was observed in families of these patients. In contrast, sudden deaths occurred in three of four families with splice mutations, in two of three with nonsense mutations, in one of three with frameshift mutations and in the single family with an in-frame codon deletion. All three families with sudden deaths at or before the age of 35 years were carriers of splice mutations.
Mutations in the MYBPC3 gene are among the most prevalent causes of inherited HCM. In earlier studies, e.g. [6, 7], MYBPC3 mutations were found to be associated with a low penetrance, late onset and a benign course of the disease. This notion became challenged, however, by the findings of two recent studies. In the first, carriers of a specific MYBPC3 frameshift mutation from 15 unrelated families revealed a high penetrance, early onset and malignant course of the disease . In the second, a high proportion of MYBPC3 mutations was found in children with HCM . Here we screened 87 patients with HCM and 71 patients with DCM for mutations in MYBPC3 and correlated the genotypes, phenotypes and clinical outcomes of the mutation carriers and their close relatives. We identified 18 different mutations in 18 index patients, 16 with HCM and 2 with DCM. Mutations in other likely candidate genes for HCM and DCM were excluded. In addition to the 18 index patients, 24 more MYBPC3 mutation carriers were detected among relatives, distributed over ten families. In ten of them, the presence of a cardiomyopathy was unknown before. 16 of the 24 additional mutation carriers displayed a clear disease phenotype. Additional three individuals had a borderline phenotype: One of them had hypertension as potential cause of secondary hypertrophy and coronary artery disease. Two more and still young individuals, a 17 year old male (IV-2 of family 17) and a 15 year old female (III-5 of family 18), showed also borderline signs of the disease. Only three mutation carriers, all members of family 5, had no certain pathological findings. The corresponding mutation, a novel p.R272C missense mutation, altered one of three known phoshorylation motifs of the MYBP-C protein . We found decreased phosphorylation at this position in vitro, but we don't know the functional consequences in vivo. In this family, only the index patient showed a DCM phenotype, for which a different cause, e.g. hypertensive heart disease, cannot be ruled out. The other family with a DCM phenotype of the index patient was family 16 with a novel splice site mutation. Two older mutation carriers suffered from DCM, while four younger patients from the following generation either showed symptoms of HCM or at least a HCM phenotype upon cardiac MRI examination. It is most likely that the two older patients suffered from an end-stage HCM with progression to a DCM phenotype, as had been described already in a few cases of MYH7 and MYBPC3 mutation carriers . For still unknown reasons, the number of MYBPC3 mutations associated with DCM is much lower  than the number of MYBPC3 mutations associated with HCM.
Contrasting to previous findings in families with MYBPC3 mutations , malignant clinical events were frequently observed in our families, even at a young age. Sudden cardiac deaths at or even before the age of 35 did occur in four members of families 6, 12 and 16. In accordance with current recommendations , these data underscore the importance of a close meshed risk stratification in every patient at risk due to a mutation in MYBPC3. Although the number of mutations is too small to prove statistically significant, it is remarkable that all sudden deaths at or before the age of 35 occurred in families with splice mutations, while no sudden death was observed in families with missense mutations. Stroke and peripheral embolizations had been reported by others in 6% of HCM patients, with an incidence of 0.8% per year . As several factors in HCM are favouring manifestation of atrial fibrillation, it is not surprising that in five of 18 families a history of stroke or transient ischemic attack was prevalent. Therefore, clinical care of patients and family members at risk should consider risk indicators for atrial fibrillation like left atrial enlargement, severe diastolic dysfunction or family history of stroke or high susceptibility for atrial fibrillation.
A major limitation of our study is that genotype and phenotype data of deceased family members are not available in most cases. At a more advanced age, alternative causes of sudden death like myocardial infarction and atrial fibrillation become more likely. Therefore, only a well documented sudden death below 35 years of age is an accepted criterion for familial involvement in a cardiomyopathy . In our retrospective evaluation of affected families, it cannot be excluded, that other conditions may have led to the adverse events. Nevertheless, it is noteworthy that in many of our families adverse events are prevalent in first-degree relatives at both older and younger ages, while MYBPC3 mutations initially were believed to be associated with a good prognosis. However, a referral bias in our observational study cannot be excluded, although the reason for presentation in virtually all index patients were diagnostic procedures to rule out concomitant coronary artery disease, but not adverse events. Mutations in other likely candidate genes for HCM were not found in any of the index patients. As to the heterogeneity of the genetic causes of cardiomyopathies, additional mutations in less likely or hitherto unknown disease genes cannot be excluded. Moreover, yet unknown disease modifiers might have had a modulating effect on the disease phenotype. The question of the prognostic impact of certain mutations in cardiomyopathies has to be clarified by inclusion of mutations carriers into registries.
The finding of a MYBPC3 gene mutation is not generally associated with a good prognosis. Therefore, MYBPC3 mutation carriers should be as well subjected to careful clinical risk stratification.
Philipp Ehlermann, Dieter Weichenhan contributed equally to this work.
The excellent technical assistance of Sandra Manthey and Oliver Mücke is gratefully acknowledged.
Dieter Weichenhan acknowledges support by grant NGFN 01GS0420 (project no. NHK-S02T09) from the Bundesministerium für Bildung und Forschung.
PE and DW conceived and designed the study. JZ and RZ carried out mutation detection. HS and SL were responsible for the cardiac imaging data. RP and CZ acquired and interpreted the clinical data. DW, BTI and HAK handled funding, supervised the study and manuscript drafting. The manuscript was written and approved by all authors.


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text:
Apple Retail Stores to Try New 'Table-Selling' Strategy
Chris Hauk - Mar 28, 2013
Apple is reported to be trying out a new customer service initiative across its retail stores. The new strategy, called "table selling," involves assigning employees a product table where they are responsible for working with groups of customers who have different needs, but are interested in the same device. Customers had previously been helped on a first-come, first-served basis that sometimes caused lengthy wait times, with customers walking out frustrated.
9to5Mac:
While helping multiple customers with similar purchases might help Apple sell more iPads and iPhones at a more rapid pace, the new strategy does not go without its fair share of criticism. Some Apple Store employees believe the new approach could be too transactional and take away from the personal experience the Apple Store offers.
"I don't think everyone has the same questions or needs," an Apple Store employee familiar with the strategy said. "I feel like it's slowly turning us into Best Buy. Every time I go to Best Buy, there is one guy barely able to handle and help 5 people all wanting different things. [Group selling] is taking away what used to make Apple special."
Apple is also adding a new support role called "Dibs." The "Dibber" is responsible for filtering out those with non-Apple problems. Apple wants its retail employees to provide only minor support for third-party products. The Dibber would point the customer toward the third-party's support site for those products.
The Apple Store has ranked number one in sales per square foot for the past two years, earning twice as much as its closest competitor, jeweler Tiffany's.
Apple CEO Tim Cook referenced the problem many Apple Stores face last February when he said, "Some of our stores aren't big enough. But like cash, it's a good problem to have," according to Tim Cook. "So this year we're closing 20 stores and moving them to larger spaces," the Apple CEO added.
Former SVP of Retail John Browett took what is most assuredly a unique approach to Apple's crowding problem, he fired employees, cut the number of hours for part-time employees, and froze the hiring of new workers. Let's play a game called: "Why isn't John Browett working for Apple any longer?"
I'm not sure the new selling approach will work for Apple. As a former Apple Specialist, I know that customers valued the one-on-one time I was able to spend with them. They also appreciated any help we tried to lend then no matter what the issue was. And as an employee, I knew that although I may have not sold them anything on that visit, they would most likely return to make a purchase later, in some cases asking for me by name. It was called building relationships. It worked.
What's your opinion reader? Do you think Apple's new approach to "customer service" is doomed to failure, or do you see big things for this new approach? Please share your thoughts with us in the comments section below.
Next Up If Apple Made Alcohol, This is What it Would Be Like (Video)
Topics Apple StoreapproachemployeesNewsellingspecialists
Reddit Pocket
MRonin ⚜ says:
If they can make it work then good on 'em. I for one though see this ending up with lots of bad press and PR pain for Apple.
Yes the unique approach of one person per customer is what make the shopping at apple unique (and Tiffany's by the way)
pau max duro says:
968230 837595Average In turn sends provides could be the frequent systems that offer the opportunity for ones how does a person pick-up biological, overdue drivers, what 1 mechanically increases the business. Search Engine Marketing 595697
Apple TV Screensavers and their Locations Identified
iPhone XS and XS Max Wallpapers in High Quality for Download
Boot Camp – Capture Screenshots in Windows With an Apple Keyboard
Send My Location: How to Send Your Current Location on iPhone
How To: Turn Off Receiving iMessages on Your Mac

redpajama_set_name: RedPajamaCommonCrawl


Are you looking for a fantasy baseball draft software to liven up your 2015 fantasy baseball draft?
Then you should check out the latest installment of FanDraft Baseball!
Additional information about FanDraft Baseball can be found on the official website: http://www.fantasydraftboard.com. Users (new and past) can download a near fully functional LITE version for FREE from the website. This is a great way to try FanDraft Baseball '15 out before purchasing.
FanDraft Baseball 2015 currently has an early bird rate, and can be purchased for $30 for new users, and $15 for upgrading users (prior year registered users). The early bird expires March 1st, 2015, after which the cost increases to $35(new user)/$17.50(update).


Boating is a fun summertime activity, but it's not without risks. Every year, thousands of accidents are reported across the country to the U.S. Coast Guard, many of which resulted in significant property damage, serious injuries, and even death. If you're planning to spend time on the waters of Lake Mead this summer, here are five steps you can take to avoid boating accidents, brought to you by the Las Vegas accident attorneys of GGRM Law Firm.
Weather conditions can change rapidly. If a storm suddenly strikes when you're out boating, you might find yourself trapped in the middle of the lake with no easy route to safety. Always check the weather before heading out on the water and stay alert throughout your excursion.
Any time you go out on a boat, you should let someone else know about it. Give them all of the details, such as where you are going and how long you plan to be out on the water – just like you would do when going on vacation. Make a plan to touch base with them at regular intervals to let them know you are safe and update them if your plans have changed.
Anyone who is going out on a boat should be comfortable in the water and know how to swim. Although the plan is usually to stay dry, you should always be prepared to tackle the worst case scenario.
However, one common mistake people make is putting too much faith in their swimming abilities. Even the most experienced swimmers may face unexpected obstacles that can affect their ability to swim to shore, including injuries, fatigue, cramps, or being knocked unconscious. Just like a seatbelt can protect you in a car accident, a lifejacket can mean the difference between life and death in a boating accident.
Most people know the dangers of drinking and driving, but you might be surprised to learn that alcohol is one of the leading factors in boating fatalities. No matter what you're doing, alcohol impairs your judgement, coordination, and balance. These effects are magnified when spending time outdoors in the heat and sun. Stay safe out on the water and save the cold beers until your boating trip is over.
According to a 2015 study, in over 70% of boating accidents, the operator had not received proper instruction on how to operate the boat. It is important to understand how to properly and safely operate the boat before setting sail. Taking a few boating courses can help you understand the waters.
After being injured in a boating accident, GGRM's Las Vegas accident attorneys can provide you with the advice and support you need. Give our Las Vegas accident attorneys a call today at 702-388-4476 so we can set up your free consultation.


text:
Laduma Cares
Stats Say Niang Ineffective
18 Aug 14, 2014 - 01:04 pm
By Sir Reginald
Mamelodi Sundowns graced the PSL with smooth interchanging play and an abundance of positive runs beyond opposing defences last season, all of which have disappeared at the start of the new season, for some reason.
The fluid interchanging play has been substituted for lumping balls into the head and chest of Mame Niang, while both Ajax and Maritzburg kept a high line of defence to deal with the lanky striker.
We were restricted to taking shots from range, which hasn't been as effective as Hlompho Kekana's long distance goals of last season.
Against Maritzburg we took nine shots, six of which were off target and only from inside the box. Last night against Ajax, we managed only seven shots at goal, once again six off target and none from inside the box.
Do we need target practice? Or do we need to work smaller passes and get ourselves closer to the opposition goal area?
Whatever the answers, we need to change our approach in the next match or else we may just fall victim to Swallows and Pirates next.


Book review of Television and Culture in Putin's Russia: Remote Control : written by Stephen Hutchings och Natalia Rulyova. London: Routledge. 2009.
Book review of Television and Culture in Putin's Russia: Remote Control: written by Stephen Hutchings och Natalia Rulyova. London: Routledge. 2009.


The Brining Shed, also called the pickling shed, was constructed in 1907 as part of the McCurdy Smokehouse complex. This was the last operating smoked herring processing facility in the eastern United States when it closed in 1990, and is the only complex still intact. The furthest building from shore, fishing vessels would pull up to the brining shed and have their herring catch pumped out on the east side of the building. After time spent pickling in wooden brine tanks fish were strung them by their gills on long sticks which were placed on 'horses' and allowed to harden. All of the equipment associated with this process remains in the building, which as part of the McCurdy Smokehouse complex was listed in the National Register of Historic Places in 1993. Lubec Landmarks has restored and maintains the other buildings in the complex which are open to the public guided tours and talks every summer as an integral part of the town's historic waterfront The Brining Shed is the last building in the complex that Landmarks is seeking to save.
Severe damage to the building occurred during the blizzard of March 2017 and a June windstorm, requiring immediate repair and stabilization. Additionally, years of wind, tidal changes and winter storms in the channel between Lubec and Campobello Island have taken their toll on the pilings. It is essential that stabilization begin before next winter.
Lubec Landmarks is currently seeking emergency funds to repair and stabilize the brining shed, which is estimated to cost a minimum of $125,000. This funding will repair the collapsed wall as well as stabilize pier supports the shed rests on. More extensive restoration procedures will need to be implemented soon, including constructing new piers on which to rest the shed, reconnecting it to the complex. Full restoration of the building will allow it to become an integral part of the McCurdy's Smokehouse Museum.


The applications of our customers for wire bending parts are widely ranging. Metal brackets, clips and wire clips are used in almost every industry. At Schnoering we have been producing wire bending parts of different applications for our customers since 1957 demanding new challenges.
This is why we offer to our customers to cooperate during the development of these partly very complex pieces and to integrate our knowledge in wire and bending technology.
In wire and bending technology Schnoering is the specialist for thin dimensions. We process wires with diameters of approx. 0.1 to 2.5 mm. For some applications of wire bending parts just a simple carbon wire is not sufficient.
For this reason we also process special raw materials such as stainless steels and non-ferrous metals. The shape of the wire can also vary for example to make better use of a very limited installation space or to increase functionality. And so wire bending parts at Schnoering can also be produced from profile wire or square wire.
As a manufacturer of wire bending parts Schnoering specializes in the processing of thin wires. Wire diameters of approx. 0.1 - 2.0 mm are used. The different requirements of the application are realized by using special materials and different strengths. The close cooperation of our technical development departments with our customers is focused on this.


With more than 6.46 lakh companies shuttered and many others undergoing various regulatory processes, including liquidation, the total number of active entities stood at 11.16 lakh during the period.
According to official data, country has more than 18 lakh registered companies but only 62 per cent of them were actively functioning at the end of October. The majority of active companies were engaged in business services. Latest data from the corporate affairs ministry showed that there were a little over 18.10 lakh registered companies at the end of October. In terms of total registered companies, nearly 62 per cent of them were active last month.
With more than 6.46 lakh companies shuttered and many others undergoing various regulatory processes, including liquidation, the total number of active entities stood at 11.16 lakh during the period. Entities are required to get registered under the Companies Act, which is implemented by the ministry, to function as companies.
Among the total number of registered companies, 6.46 lakh entities were shut down and 39,736 companies were in the process of being deregistered. The ministry, in its information bulletin for the month of October, said "Out of the 6, 46,882 closed companies, 10,574 companies were liquidated/ dissolved; 6, 00,048 companies were declared defunct." At October end, Maharashtra had the highest number of registered companies (3.59 lakh), followed by Delhi (3.26 lakh), and West Bengal (1.98 lakh).In terms of economic activities, 3.57 lakh active companies were engaged in business services while 2.23 lakh firms were into manufacturing.


Mark Gatiss portrays the Vampire overlord, Mr. Snow, in Series 4. The name is given to him for his icy demeanor. Ironically, Gatiss's character on Sherlock (2010), Mycroft, was nicknamed "the Ice Man" by series baddie, Jim Moriarty. The two characters even share a similar wardrobe.
Annie's headstone changes from season 1-2. The first time she views it, it reads "beloved fiancée.." In season 2 when her mother is viewing it, it simply states her name. Unless of course her family had it changed when Owen confessed to killing her, but they never mention it.


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Station Bassevelde was een spoorwegstation langs de spoorlijn spoorlijn 55A (Zelzate - Eeklo) in Bassevelde een deelgemeente van de gemeente Assenede.

Van 1910 tot 1950 konden passagiers overstappen op tramlijn 375 (Bassevelde - Gent) uitgebaat door de NMVB.

Bassevelde
Assenede

redpajama_set_name: RedPajamaWikipedia


"Swimming in Deep Waters" by Deborah Schussler, Sharon Feiman-Nemser et al.
Swimming in Deep Waters. A Response to "A Review of Teaching as a Moral Practice"
The authors respond to a review of their book, Teaching as a Moral Practice: Defining, Developing, and Assessing Dispositions. The authors emphasize a vision of shared commitments for quality teaching whereby teacher-educators instill and nurture the wisdom and virtue that a moral teacher must possess in order to teach in a variety of circumstances where clear-cut answers do not exist. In addition, teacher-educators help teachers discern how, in that context, they should enact particular knowledge, skills, and commitments to reach desired ends. The key to enact this vision of teaching as a shared, moral practice is critical colleagueship.
Schussler, D. , Feiman-Nemser, S. , Diez, M. E. , Murrell, P. (2012). . Swimming in Deep Waters. A Response to "A Review of Teaching as a Moral Practice". Democracy and Education, 20 (2), Article 11.


An easement is a set of rights to use a section of land in a particular way.
It gives someone else the right to use the land for a specific purpose even though they are not the landowner. Easements over land may be granted for a specific purpose, including driveways for access to blocks that don't have suitable access, drainage, sewerage, or supply of water or gas.
A right of way is an easement that allows another person to travel or pass through your land. The most common form of right of way easement is a road or path through your land. The right of way easement road is meant to benefit a particular person or another parcel of land not owned by you. Right of way easements extend reasonable use for travel through others' lands to holders of the easements.
Easements can be created as a result of an agreement between the dominant and servient owner. The parties will determine the rights and obligations they and all subsequent owners will have with respect to the easement.
It is generally assumed easements are created to last forever, unless otherwise indicated in the document creating the easement. Despite this, an individual granting an easement should avoid any potential legal or interpretive problem by expressly providing that the easement is permanent. Although permanent easements are standard, they can be broken. Easements of limited duration are generally used to provide temporary access until completion of construction work.
Can you build on an easement?
Normally an easement will not prevent you from building over or under it. For example, if there is an access way through your property, you probably will be able to put a sewer under it or a structure over it.
Whether or not you can build over it will generally depend on whether your application causes substantial interference to someone else's rights.
For further information on and your rights in relation to easements, or if you have a situation where access to your property is an issue, please contact our Property & Commercial Law solicitors in Bendigo on 5434 6666 or Castlemaine on 5472 1588.


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Main Index
US Laws
US Jurisprudence
UNITED STATES SUPREME COURT DECISIONS ON-LINE
US Supreme Court Decisions On-Line> Volume 259 > SOUTH COVINGTON & C. ST. RY. CO. V. NEWPORT, 259 U. S. 97 (1922)
SOUTH COVINGTON & C. ST. RY. CO. V. NEWPORT, 259 U. S. 97 (1922)
Subscribe to Cases that cite 259 U. S. 97
South Covington & C. St. Ry. Co. v. Newport, 259 U.S. 97 (1922)
South Covington & Cincinnati Street
Railway Company v. City of Newport
Argued April 13, 1922
Decided May 15, 1922
APPEAL FROM THE DISTRICT COURT OF THE UNITED STATES
FOR THE EASTERN DISTRICT OF KENTUCKY
The bill alleged that the plaintiffs were corporations operating electric street cars and distributing electric current under perpetual franchises in the city; that, under supervision and direction of the city authorities, they constructed a high tension wire to obtain necessary additional current from another company; that, afterwards, the city council, by resolution, directed speedy removal of the wire, declaring it dangerous to life and property, contrary to the fact, and that, unless restrained, the city would forcibly remove and destroy it, thereby interfering with the operation of plaintiffs' railway, lighting, and power systems, and causing them irreparable damage, in violation of their rights under the Contract Clause of the Constitution and the due process clause of the Fourteenth chanrobles.com-red
Page 259 U. S. 98
Amendment, and prayed that the resolution be declared null and the city, it officers, etc., be enjoined from enforcing it.
Held:
(1) That the bill set up a substantial federal question and conferred jurisdiction on the district court. P. 259 U. S. 99. Des Moines v. Des Moines City Ry. Co., 214 U. S. 179, distinguished.
(2) That the jurisdiction, having attached, could not be defeated by an answer denying the city's intention to enforce the resolution except through an order of court. P. 259 U. S. 100.
Appeal from a decree of the district court dismissing for want of jurisdiction a bill to restrain the defendant city from forcible removal and destruction of an electric wire.
MR. JUSTICE McREYNOLDS delivered the opinion of the Court.
In their original bill, appellants allege: that they hold perpetual franchises over certain streets in Newport, Kentucky, for operating street cars and distributing electric current; that, in due course, it became necessary for them to obtain an additional current from another company, and that to that end, in 1915, under supervision and direction of the city authorities, they constructed a high tension wire extending from Central Bridge to their power house; that, on November 20, 1917, the board of commissioners of Newport adopted a resolution which declared this current dangerous to life and property, and directed removal of the wire not later than December 1, 1917.
The bill further alleges that,
"unless restrained by this court, defendant will forcibly remove and destroy said wire thereby interfering with the operation of the street railway
system and the electric lighting and power system above described, causing plaintiffs injury which cannot be compensated in money and to their irreparable damage,"
and that
"it is not true that said wire is dangerous to either life or property, and that said resolution is unreasonable, and in violation of the rights of plaintiffs as hereinabove set forth; that it is an impairment of the obligations of the aforesaid contracts and each of them, in violation of Article I, § 10 of the Constitution of the United States, and is a taking of plaintiffs' property without due process of law in violation of the Fourteenth Amendment to said Constitution of the United States."
The relief prayed is that the resolution be declared null, and that the city, its officers, agents, and employees be enjoined from enforcing or attempting to enforce the same.
Relying upon Des Moines v. Des Moines City Railway Co., 214 U. S. 179, the court below dismissed the bill for want of jurisdiction. The cause comes here by direct appeal, and only the question of jurisdiction is before us.
Where, as here, the jurisdiction of a district court has been invoked on the sole ground that the cause involves a federal question, and this is duly challenged, the issue must be determined by considering the allegations of the bill. If they distinctly disclose a real, substantial question of that nature, there is jurisdiction; otherwise, there is none. City Ry. Co. v. Citizens' St. R. Co., 166 U. S. 557, 166 U. S. 562; Pacific Elec. Ry. Co. v. Los Angeles, 194 U. S. 112, 194 U. S. 118; Columbus Ry., Power & Light Co. v. Columbus, 249 U. S. 399, 249 U. S. 406.
A mere formal statement that such question exists does not suffice. The allegations must show that
"the suit is one which does really and substantially involve a dispute or controversy as to a right which depends on the construction or application of the Constitution or some law or treaty of the United States."
181 U. S. 281; Hull v. Burr, 234 U. S. 712, 234 U. S. 720; Norton v. Whiteside,@ 239 U. S. 144, 239 U. S. 147.
Properly understood, Des Moines v. Des Moines City Railway Co., supra, is in harmony with these well established principles. There, the bill disclosed that the only affirmative action contemplated by the city was the institution of an orderly proceeding in court. Such action could not in any proper sense violate a right under the Constitution, laws, or treaties of the United States. The bill did not, therefore, present a substantial federal question, and for that reason jurisdiction did not exist.
Here, it is affirmatively alleged that the city intends forcibly to remove and destroy appellants' property, and thereby violate their constitutional rights. This presented a substantial claim under the Constitution.
In an amended answer, defendant denied intention to enforce the resolution except through an order of court. But, the necessary facts having been alleged by the bill, jurisdiction could not be thus defeated. The denial went to the merits of the claim. The Fair v. Kohler Die Co., 228 U. S. 22, 228 U. S. 25; St. Paul, M. & M. Ry. Co. v. St. Paul & N. P. R. Co., 68 F. 2, 10.
The judgment below must be reversed and the cause remanded for further proceedings in accordance with this opinion.
MR. JUSTICE PITNEY concurs in the result.
MR. JUSTICE BRANDEIS and MR. JUSTICE CLARKE dissent.
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"A balancing act—Telehealth cancer genetics and practitioners' experien" by Elvira Zilliacus, Bettina Meiser et al.
Telehealth is increasingly used for outreach service in cancer genetic counseling; however what occurs during the consultation and the roles practitioners adopt is largely unknown. Fifteen practitioners participated in semi-structured interviews that explored their roles within telehealth, compared to face-to-face consultations, and the relationship between practitioners during telehealth. As they were not physically present with the patient, most participants felt that telehealth altered the genetic clinician's role to one of a 'visiting specialist'. Genetic counselors described undertaking multiple roles during the telehealth process. Two models of interaction were observed. The medical model reduced the interaction to a dyadic consultation by having the genetic counselor off-screen and included minimal clinician meetings and supervision. The triadic co-facilitation model incorporated a high level of information exchange, counselor autonomy and included the counselor onscreen. The co-facilitation model offers a useful framework for telehealth genetic counselling, offering complementary roles between practitioners and efficient service delivery.


The ACTG1 gene provides instructions for making a protein called gamma (γ)-actin, which is part of the actin protein family. Proteins in this family are organized into a network of fibers called the actin cytoskeleton, which makes up the structural framework inside cells. There are six types of actin; four are present only in muscle cells, where they are involved in the tensing of muscle fibers (muscle contraction). The other two actin proteins, γ-actin and beta (β)-actin (produced from the ACTB gene), are found in cells throughout the body. These proteins play important roles in determining cell shape and controlling cell movement (motility).
γ-actin is particularly abundant in certain cells in the intestines and the inner ear. Within the inner ear, this protein is found in specialized cells called hair cells, which are essential for normal hearing.
At least six mutations in the ACTG1 gene have been found to cause Baraitser-Winter syndrome, a rare condition that affects the development of the brain, eyes, and other facial features. The known mutations change single protein building blocks (amino acids) in γ-actin. The most common mutation replaces the amino acid serine with the amino acid phenylalanine at protein position 155 (written as Ser155Phe or S155F). The mutations that cause Baraitser-Winter syndrome alter the function of γ-actin, which causes changes in the actin cytoskeleton that modify the structure and organization of cells and affect their ability to move. Because γ-actin is present in cells throughout the body and is involved in many cell activities, problems with its function likely impact many aspects of development. These changes underlie the variety of signs and symptoms associated with Baraitser-Winter syndrome.
Bryan KE, Wen KK, Zhu M, Rendtorff ND, Feldkamp M, Tranebjaerg L, Friderici KH, Rubenstein PA. Effects of human deafness gamma-actin mutations (DFNA20/26) on actin function. J Biol Chem. 2006 Jul 21;281(29):20129-39. Epub 2006 May 10.
Rendtorff ND, Zhu M, Fagerheim T, Antal TL, Jones M, Teslovich TM, Gillanders EM, Barmada M, Teig E, Trent JM, Friderici KH, Stephan DA, Tranebjaerg L. A novel missense mutation in ACTG1 causes dominant deafness in a Norwegian DFNA20/26 family, but ACTG1 mutations are not frequent among families with hereditary hearing impairment. Eur J Hum Genet. 2006 Oct;14(10):1097-105. Epub 2006 Jun 14.
Zhu M, Yang T, Wei S, DeWan AT, Morell RJ, Elfenbein JL, Fisher RA, Leal SM, Smith RJ, Friderici KH. Mutations in the gamma-actin gene (ACTG1) are associated with dominant progressive deafness (DFNA20/26). Am J Hum Genet. 2003 Nov;73(5):1082-91. Epub 2003 Sep 16.
van Wijk E, Krieger E, Kemperman MH, De Leenheer EM, Huygen PL, Cremers CW, Cremers FP, Kremer H. A mutation in the gamma actin 1 (ACTG1) gene causes autosomal dominant hearing loss (DFNA20/26). J Med Genet. 2003 Dec;40(12):879-84.


This hilltop home is the perfect, tropical paradise offering a serene lifestyle with an incredible outdoor space-perfect for entertaining. It is situated in the premier, western gated community of Lyford Cay on a cul de sac on Coral Lane. At 7,000 sq. ft. the openness of this home with its artfully designed cedar-wood high ceilings and many windows, offer an abundance of natural light. The home is furnished and features 5 bedrooms (plus a spare room) and 5 bathrooms and consists of a completely re-modelled state of the art kitchen with new appliances; it also boasts a second kitchen on the lower level along with a formal dining room and library. The home has a generator and comes with a golf cart. The terraces and outdoor patio and pool areas provide an ideal entertaining area overlooking lush tropical foliage, the golf course and sea beyond. Lyford Cay is located near to many pristine beaches, diving and snorkeling sites and the international airport. It is near the elite, gated communities of Old Fort Bay and Albany.


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What is Remote Viewing?
Remote Viewing Media
1I/2017 U1 (Oumuamua)
26. November 2017 Wingman Astronomy, Projects 2 comments
Category: Astronomy
Protocol: CRV (with monitor)
Coordinates: 301875 962548 655314
Number of viewers: 1 (Bernhard Reicher)
Recently, reports about an astronomical object that entered our solar system opened speculations. This "Oumuamua" named object is characterized by its extra-solar origin and unusually elongated shape. This shape, with an estimated length of several hundred meters, make you think of cigar-shaped "mothership" UFOs , which were reported in many UFO-cases. Has such a flying object entered our solar system or is it an exotic, natural structure?
One of our seminar graduates, Bernhard Reicher (from Reicher & Stark), was sent to the Oumuamua object in an operational session.
Target cue: "Describe the astronomical object Oumuamua, which was discovered on 10/19/2017, at the time of the session!"
Already in stage 3 , Bernhard received impressions that indicated an unsteady, rocky structure. He was particularly struck by two aspects: firstly, the horizon seems to be "overstepping" when one is on top of the object. Maybe it stumbles very much over its longitudinal axis? On the other hand, the overall structure of the object is strongly twisted, like a wet towel when wringed out. Furthermore, Bernhard perceived holes in different sizes and shapes on the surface. Here is the sketch with his first impressions:
Stage 4 focused on the most important sensory impressions of the object. Among them were "black", "cold" and "hard". But also brownish and silvery hues were perceived casually. Other important impressions were "twisted", "tilting horizon", "flowing, wavy shapes", "soft", "porous" and "elongated".
Stage 6 should now provide further detailed descriptions of the object. First, we tried again to summarize the basic form with its properties:
The elongated shape, the twisted surface and the porous properties stood out here. Since the drawing was still somewhat artificial, we investigated, among other things, the cause of the twisted turns to concretize the properties of the object. These emerged as a natural structure of geological origin, which was created by pressure and counter-movements. We also tried to examine the inside of the object, which was porous, but not hollow. It reminded of the construction of lava rock. Signs of life or technology could not be perceived on the object itself. Another sketch of surface impressions:
Now the question arose as to how the object was originally created and how this strange shape came about. As an origin Bernhard perceived a red-hot, spherical mass, which rotated very fast. It was about the size of the earth's moon, and seemed to have been a young, and therefore still glowing, planet or moon. The elongated object was eventually ejected from this celestial body as a liquid "splash", and when cooled it assumed its characteristic shape ("thrown out by centrifugal force and then cooled down"). We also found out that this happened more often there, although not every ejected fragment produced such an unusual shape. The ejection also caused the object's twist, which resulted in the twisted surface structure.
So obviously a normal process, which led in this case to an extraordinary shape object. Finally, there was the question for the age of Oumuamua. Although determing the exact age in cosmic periods, for lack of clear reference points, is limited. We used the origin of our solar system, the extinction of the dinosaurs, and our present time ("time of session") as anchor points:
Considering the usual stretching and compression effects in such long timelines, we estimate the emergence of Oumuamua to be a period of 500 to 1000 million years ago.
The data of this single session showed a purely natural object. No artificially constructed aspects or signs of life could be perceived there. Most likely, it's the description of a lava splatter, which is ejected as if from a volcano, then hardens in space and continues to wander through stellar or interstellar space. The impressions of a "tilting horizon" would fit quite well with the following artistic idea of the movement:
Of course, there will be the typical UFO euphoria around Oumuamua, as with many meteorites or the Ceres Lights . And who knows, maybe there's a big mothership or something like that. Extraterrestrial space traffic is often recorded during sessions on exoplanetary systems as an interesting "by-catch". In this case, however, it seems "only" to be a very unusually shaped asteroid, which began its existence long ago as a large lava splash from a glowing celestial body. Nonetheless, a discovery that fascinates because of its form and distant origin.
Update 16.10.2018: Two american viewers found some signs of life inside the porous structure of Oumuamua (like in a cave system). Maybe we do another session on Oumuamua some time, with special attention to the objects inside.
German version of this article
Interesting, I like the use of the time line. Thanks.
Wingman says:
Proxima Centauri b
Remote Viewing Crop Circles
A Short Trip To Saturn's Moon Titan (Update)
Topics examined with RV (as of 2019-11-20)
A Short Trip To Saturn's Moon Titan
Meteor of Chelyabinsk
Gliese 581c
Strange Arms at OoBE
The Garden Gnome
Monroe Institute: CHEC-Units
Smalltalk with Robert Monroe
The Lilith-Entity
Baltic Sea Anomaly (Ocean-X)
Remote Viewing Angels
Mysterious Sounds in the Sky
Viewing a non-physical Construct
Venera spacecraft on the planet Venus
Various session extracts
Wingman on 1I/2017 U1 (Oumuamua)
Mike S on 1I/2017 U1 (Oumuamua)
Aliens Archeology Astronomy Calibration Targets Consciousness CROPfm Entities Events Internet / Networking Locations Miscellaneous OBE Persons Phenomena
Eight Martinis
ONE20+
"Self experience - the difference between knowledge and wisdom."


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MS Dhoni's daughter Ziva turns makeup artist for her father's photoshoot
The 38-year-old former World Cup-winning captain was recently spotted playing volleyball in the Maldives or going on a tiger safari in Kanha National Park.
MS Dhoni and Ziva Dhoni. (Photo Source: Twitter)
He might be away from the cricket field for over seven months now but Mahendra Singh Dhoni is never really away from the headlines. The 38-year-old former World Cup-winning captain was recently spotted playing volleyball in the Maldives or going on a tiger safari in Kanha National Park.
And now, the ace wicket-keeper-batsman who is eyeing a comeback to competitive cricket in the Indian Premier League 2020, was seen in the company of his five-year-old daughter Ziva catering to him as his makeup artist.
Dhoni was in Mumbai recently for a photo shoot with his daughter and the fans relished a video of Ziva doing the makeup of her father with a brush. The video instantly went viral on social media after Dhoni's official fan clubs posted the video captioning it. In the video, Ziva was seen sitting on Dhoni's lap and doing her work diligently while the latter co-operated. Dhoni's hairstylist Sapna Bhavnani was also spotted in the background.
.@msdhoni's little makeup artist on duty!😍 #Dhoni #MSDhoni #Ziva #Mumbai pic.twitter.com/Nc3UOWEq9f
— MS Dhoni Fans Official (@msdfansofficial) February 15, 2020
Dhoni has not donned the Indian jersey since World Cup 2019
Dhoni has not played for India ever since their loss against New Zealand in the semi-final of the 2019 World Cup in England last July. The veteran, who did not have a great tournament with the bat, was India's last hope in that game after a top-order collapse but a brilliant run-out throw from Martin Guptill dashed the dream of the Men in Blue.
India played in a number of limited-over series since the showpiece event against the West Indies, South Africa, Bangladesh, Sri Lanka, Australia and New Zealand but the Ranchi cricketer did not feature in any of those. Recently, Dhoni was even dropped from the Board of Control for Cricket in India's list of central contracts fuelling speculation that the road has ended for the man who has given India two World Cups — in 2007 and 2011.
Dhoni, however, is set to begin his training in Chennai for the Chennai Super Kings — the team that he proudly leads in the Indian Premier League. The Ranchi cricketer has led the Super Kings in all editions barring the two years when they were suspended and took them to the final eight times. On three occasions, Dhoni's team lifted the crown.
Watch: Irfan Pathan's interview post his retirement
IndiaMS Dhoni
Ravichandran Ashwin has suffered for speaking his mind at team meetings: Sunil Gavaskar
Ravi Ashwin shuts down a supporter for trolling him on Champions Trophy 2017 final loss
KL Rahul jets to career-best 2nd rank in the latest ICC T20I player rankings
Top Stories: Things that made news in the cricket world today


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Rebecca Ann Latimer Felton, née le  à Decatur (dans l'État américain de Géorgie) et morte le  à Atlanta (Géorgie), est une écrivaine, conférencière, réformatrice sociale et femme politique américaine. Elle est la première femme à siéger au Sénat des États-Unis.

Biographie
Rebecca Latimer Felton est une personnalité féminine éminente dans la Géorgie de la période dite de l'Ère progressiste (Progressive era). Le droit de vote et d'éligibilité est adopté aux États-Unis en 1920. Nommée au Sénat des États-Unis, à Washington DC, Rebecca Latimer Felton prête serment le  1922, à , neuf mois et  . Elle siège pendant  seulement.

Ce bref mandat a pour origine la mort du sénateur . Candidat à sa succession, le gouverneur  cherche alors une personnalité pour occuper le siège vacant jusqu'au scrutin, et qui ne serait pas un potentiel concurrent. Il désire aussi marquer des points auprès des femmes, qui peuvent désormais voter. Cependant, le Sénat avait arrêté ses réunions jusqu'aux prochaines élections sénatoriales, si bien qu'il était peu probable que Rebecca Latimer Felton soit officiellement assermentée sénatrice. Finalement, Thomas W. Hardwick est battu par . Plutôt que d'occuper immédiatement son siège, ce dernier, grâce à la persuasion de Rebecca Latimer Felton et au soutien de féministes de l'État de Géorgie, accepte qu'elle devienne sénatrice pendant une journée. Il s'agissait d'un geste symbolique envoyé au mouvement féministe, dont elle était l'une des figures.

Jusqu'à l'élection de Kelly Loeffler en 2020, elle est également la seule femme à avoir été sénatrice de Géorgie. Auparavant, elle avait participé aux campagnes de son mari , membre de la Chambre des représentants des États-Unis et de celle de Géorgie.

Elle est particulièrement engagée sur les questions de société, notamment la réforme pénitentiaire, le droit de vote des femmes et la modernisation du système scolaire. En 1915, elle s'implique dans la rédaction d'un grand programme féministe, prévoyant notamment l'égalité de rémunération pour un travail égal.

Elle garde toutefois des préjugés racistes, notamment elle a l'obsession du « noir violeur de femme blanche », et voit dans le lynchage le seul remède à la prévention des viols. Elle est par ailleurs le dernier membre du Congrès à avoir été propriétaire d'esclaves avant la guerre de Sécession.

Publications
 .
 .
 .

Références

Articles connexes
 Liste des sénateurs des États-Unis pour la Géorgie
 Femmes au Sénat des États-Unis
 Hattie Wyatt Caraway, première sénatrice à siéger durant une longue période (1931-1945)
 Jeannette Rankin, première membre de la Chambre des représentants (1917)

Liens externes

Naissance en juin 1835
Naissance à Decatur (Géorgie)
Essayiste américaine du XIXe siècle
Essayiste américaine du XXe siècle
Suffragette américaine
Féministe américaine
Mémorialiste américain
Pionnière en politique
Sénatrice des États-Unis
Sénateur des États-Unis pour la Géorgie
Personnalité du Parti démocrate en Géorgie (États-Unis)
Décès en janvier 1930
Décès à 94 ans
Décès à Atlanta
Conjoint de personnalité politique


Do you have event planning or event production experience? Are you ready to work for a company that works together as a team to make clients and events successful? Check out Wired Production Group's available jobs.
Wired Production Group is not just an event production company. We are Wired. We are connected to our team, our clients and to our community. We are dedicated to developing the future of our company in the event industry through evolving technology and amazing teamwork.
We do work that matters. Unless you've put on an event, you probably don't understand all the planning and what happens behind the scenes at events you attend. Events are stressful — and we get it. That's why we are there for you from the event pre-planning process to the post-production wrap-up. We won't want to miss a second. Our Wired Production Team is passionate about what we do and our clients' success is our success.
We utilize technology. There is an unlimited potential for technology to be incorporated into our event productions, our daily business and our personal lives. We are connected by technology no matter where we are, making communication easy for everyone involved in our business. Call, text, email, video message, Instant Message…we're ready to connect with you.
We have the right leadership. Wired Production Group's President and CEO, Ron Rausch, grew our company with the drive and desire to provide all of our clients with amazing full-service event management solutions. With this in mind, he brought together some of the best and brightest event production leaders in the industry to form a team that is willing to go all out. Over the years our team has expanded and we have grown into the family we are today. We encourage every team member to be the best, both personally and professionally.
We are growing. With our company growing so rapidly, we are consistently looking for the right people to hire and come aboard our team. We're happy to share our benefits, full-sized kitchen and relaxed work environment with the right people. We are looking for talented, happy event professionals who have a passion for what they do.
If you're interested in working for Wired Production Group, check out our job openings below to see if you'd be a good fit. We'd love to meet you!
As a Video Production Specialist with Wired Production Group, you'll perform a variety of technical duties involved in the planning, coordination and production of various projects to support our clients' needs as part of our video production team.
Our Wired Production Group team is looking for someone with experience in basic designing lighting, operations and theatrical lighting for concerts, corporate events and/or theatrical production.
The Audio Technician position with Wired Production Group is for someone with a basic understanding of signal flow as well as experience mixing live music and bands.


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Q: Image loses quality with cv2.warpPerspective I am working with OpenCV 3.1 and with Python.
My problem comes when I try to deskew (fix the tilt of) an image with text. I am using cv2.warpPerspective to make it possible, but the image loses a lot of quality. You can see here the original part of the image:

and then, here, the "rotated" image:

it is like smoothed.
I was using morphological transformation like:
kernel = np.ones((2, 2), np.uint8)
blur_image = cv2.erode(tresh, kernel, iterations=1)

and
white_mask2 = cv2.morphologyEx(white_mask2, cv2.MORPH_OPEN, kernel)

to see if it improves something, but nothing.
I saw this example here in SO, but those guys had the same problem:
 and
So, I don't have idea what can I do.
Maybe there is a way to not losing the quality of the image, or, there's another method to rotate the image without quality lost.
I know this method:
root_mat = cv2.getRotationMatrix2D(to_rotate_center, angle, 1.0)
result = cv2.warpAffine(to_rotate, root_mat, to_rotate.shape, flags=cv2.INTER_LINEAR)

But it doesn't work for me, as I have to rotate every rectangle here:

and not the whole image. It means, the best way I found to do it, was warpPerspective, and it works fine, but with quality loss. I would appreciate an advice to avoid the quality lost.

A: The problem is related to the interpolation required by the warping. If you don't want things to appear smoother, you should switch from default interpolation method, which is INTER_LINEAR to another one, such as INTER_NEAREST. It would help you with the sharpness of the edges.
Try flags=cv2.INTER_NEAREST on your warp call, be it warpAffine() or warpPerspective().
Interpolation flags are listed here.
enum InterpolationFlags {
    INTER_NEAREST = 0,
    INTER_LINEAR = 1,
    INTER_CUBIC = 2,
    INTER_AREA = 3,
    INTER_LANCZOS4 = 4,
    INTER_MAX = 7,
    WARP_FILL_OUTLIERS = 8,
    WARP_INVERSE_MAP = 16
}

redpajama_set_name: RedPajamaStackExchange


text:
Avaya to secure Bank of Ireland's online business
16 Aug 200470 Views
Business communications vendor Avaya has signed a deal with Bank of Ireland (BoI) to deploy security assessment services across the bank's direct banking business BoI 365. The value of the deal was not disclosed.
Last year 44pc of all European enterprises experienced at least one malicious security breach, the most common causes being viruses and hacking of unsecured networks. According to The Yankee Group the cost of a security breach is highest in the banking community with the average cost being €1.75m.
Avaya carried out an independent security audit on the bank's telephony system in the second half of 2003. Following the audit, Avaya's Global Services business designed and implemented new measures to further improve BoI 365's IVR security. The work was undertaken over a two week period, combining on-site and remote engineers working to ensure the security of telephony hardware and software.
David Sheehan, operations manager at BoI 365, said: "The work that Avaya has implemented ensures our customers get a fast and most importantly, a secure service when using BoI 365's phone banking service. The security audit is improving our awareness of network security –VF we have already noticed how security processes and practices have improved."
John Sharpe, director for Avaya in Ireland, added: "As communications technology continues to move towards converged networks, the security of a company's voice network becomes as important as securing data devices and applications. This is especially true for financial institutions such as BoI 365 whose reputation amongst customers would suffer should a security breach occur."
In addition to Avaya's secure communication services, BoI 365 uses a range of Avaya systems at its Dublin headquarters and Kilkenny location. In future, BoI 365 plans to migrate to Avaya internet protocol telephony at its Dublin and Kilkenny locations to enhance communication and provide greater flexibility to support its business.
Related: research, R&D, Science Gallery, software development, Bank of Ireland


text:
Archive for Red Army Faction
Imperial Storm Scientists
Posted in Ecological resilience, Influenza with tags BP, CDC, climate change, Great Recession, Minerals Management Service, oil spill, pandemic influenza, President Obama, Red Army Faction, regulation, scientists, vaccine, War on Terror on August 21, 2010 by Rob Wallace
The Red Army Faction was a communist guerilla group operating 1970-1998 in, of all places, West Germany. The RAF engaged in a variety of operations in the 1970s, including assassinations and bombings, primarily around the German government's material support of the U.S. war in Vietnam.
As depicted in the Baader-Meinhof Complex and this BBC documentary, the RAF and the West German government entered a spiral of mutual self-deception. Each told itself the lies necessary to defeat the other. However inaccurately depicted in the films, truths, spoken and lived, were sacrificed for the victory neither side could achieve:
The scope of the RAF's bombing campaign outpaced its initial public support and sharpened the fascistic reaction the group claimed its enemy. Supporters think the latter repression emblematic of RAF's success but they'd be hard pressed to claim even a moral victory as the German people acclimated to, and even openly supported, a burgeoning police state. Post-Nazi Germany meanwhile suffered a funktionshäftling's shame killing dissidents, however violent, in the name of colonial liquidations ruthlessly conducted abroad.
If only American fallacies were so rottenly principled.


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text:
Listen to WXDJ - El Zol 106.7 FM in the App
WXDJ - El Zol 106.7 FM
North Miami Beach FLUSALatinZouk and TropicalEnglish
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Leicester, Top 40 & Charts
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About WXDJ - El Zol 106.7 FM
WXDJ - El Zol 106.7 FM is a Spanish-language radio station based in Miami, Florida. The station is known for its upbeat and lively programming, which includes a mix of contemporary Latin music, news, and entertainment. The station's playlist includes a wide range of popular Latin music genres such as Salsa, Merengue, Bachata, Reggaeton and more, with a focus on the most current and popular hits. The station's DJs and presenters are renowned for keeping listeners up-to-date with the latest music, news, and events happening in Miami and the rest of the Latin community. In addition to music, WXDJ - El Zol 106.7 FM also features regular programming that focuses on lifestyle and entertainment topics that provide a glimpse into the Hispanic community's culture and artist's life. The station's on-air personalities, which include experienced and entertaining hosts, keep the audience informed and engaged with the latest happenings in the Latin music industry. The station also places a strong emphasis on community involvement, frequently participating in events and fundraisers that support local causes. They also provide a platform for local businesses and organizations to promote their products and services. Furthermore, WXDJ - El Zol 106.7 FM is easily accessible via online streaming, allowing listeners to tune in and enjoy the station's programming wherever they are. The station's website also provides additional resources and information, including past shows, podcasts, and playlists. In conclusion, WXDJ - El Zol 106.7 FM is a lively and entertaining radio station that provides its listeners with a mix of popular Latin music, informative programming, and a glimpse into the Hispanic community's culture. With its commitment to quality and diversity in programming, its community involvement, and its online presence, it's a valuable resource for Spanish-speaking individuals looking to stay informed and connected with the community, specifically in Miami and its surrounding areas.
Listen to WXDJ - El Zol 106.7 FM, WCMQ-FM - Zeta 92.3 FM and Many Other Stations from Around the World with the radio.net App
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Totally six projects were submitted to the jury. Three projects were devoted to the early diagnosis of various cancers: a screening test for stomach cancer, technology for the early detection of thyroid cancer for people working in harmful conditions and one of the methods of "liquid biopsy." Genetic engineering approach in immunotherapy was developed in one of the projects. Another group proposed a smart system that measures pain from electrical activity in a specific area of the brain using a special device. These data are transferred to an automatic device, dosing anesthetic preparations. There was also a project aimed at prevention of cancer – a vaccine that potentially prevents a certain type of breast cancer.
For ten days 16 science experts from 13 different organizations worked with young scientists: these are universities, scientific and medical institutions.
I think that this is a rational kernel for any state, when a selection of talented young people begins at the stage of forming them as professionals. Many scientific epoch-making discoveries were made when people were engaged in student work, so I am deeply convinced that the project participants will do so."— said one of the experts of the camp Andrey Ivanovich Shmak, head of the abdominal oncopathology department of the RRPC of Oncology and Medical Radiology named after N.N. Alexandrov.
Most of all we were surprised by projects maturity: searching and broad. This was evident from the way the students confidently answered the rather complicated and specific questions of the jury. The winning project was distinguished by the scientific elegance of the solution: the two existing approaches were unexpectedly integrated into one. This is a logically simple solution to a very complex problem, which is relatively simple in terms of both chemistry and the functioning of the healthcare system. All six projects have been worked out in sufficient details and each of them can be really developed to the working prototype. We very much hope that the children will have enough motivation to finalize their projects to the level of participation in the republican contests of young scientists.
Scientific session of TALENAVITA CAMP lasted 10 days, united 30 students from seven different universities from all regions of Belarus. They are future biologists, chemists, physicians who are not indifferent to the topic of scientific session: "Cancer is under control: ideas and methods of present-day oncology". For the children, participation in the camp turned out to be a unique experience of scientific work, communication with each other and experts.
Most of all I remember the very process of brainstorming, when ideas appeared massively and filled our brains. It was just fantastic!" — Oleg Mosin, second-year student of the Grodno State Medical University shared his impressions.
The summer scientific camp is organised within the framework of TalenaVita program to support talented youth, initiated by the pharmaceutical company NatiVita, with support from RRPC of Oncology and Medical Radiology named after. N.N. Alexandrov and Vitebsk State Medical University.


BOND OFFICIAL hosts a few of New York's finest, and well suited men at Harold's in collaboration with Made to Measure suiting brand, InStitchu.
Fashion, Art, and Charity. The Frick Collection kicks off Spring 2019 with the 20th Annual Young Fellows Ball.
Arlo SoHo has officially announced the launch of their new onsite cocktail bar A.L.B. (Arlo Lobby Bar).
EXPLORER is perhaps one of Mont Blanc's most inspired fragrances and the storyline for this new scent efforts a keen appreciation for adventure, and the art of exploration - qualities the brand has possessed since its very birth.
These whiskey cocktails are simple, clean, and can be easily enjoyed anywhere. Step into your inner adventurer and go for one of these delectable tipples.
To celebrate National Margarita Day on February, 22nd, we have sought out a few must-try Margarita recipes that are perfect to make at home and share with your friends to celebrate the occasion.
Backstage with the men at the BOSS Fall/Winter 2019 Runway Show during New York Fashion week, on Wednesday, February 13, 2019.


Visiting the Zoo, the chorus (Ladies and Gentlemen of the Great British Public) find Aesculapius Carboy trying to hang himself as the father of Lætitia Grinder, the woman he loves, has rejected him because he is a mere apothecary. Eliza Smith, in charge of the Refreshment Stall, orders Carboy to desist. Thomas Brown, who has been wooing Eliza, suspects Carboy is a rival.
Lætitia arrives in search of Carboy and they sing a duet of reunion, whilst Eliza and Thomas Brown sing in counterpoint about all the refreshments Thomas has consumed while wooing Eliza. Thomas collapses; Carboy and the chorus offer conflicting medical advice, Thomas comes to and Carboy dispatches Eliza for a prescription. During the medical examination, Carboy uncovers the Order of the Garter and diagnoses that Thomas is a peer in disguise. Thomas explains that he has come here incognito in search of virtue and found it in Eliza.
Lætitia's father, Mr Grinder, appears, in search of his daughter and Carboy. They and the chorus appeal to Grinder's finer feelings, but he has none. Carboy again resolves on suicide.
Thomas Brown returns in all his ducal splendour and asks Eliza to marry him. She is reluctant to leave what she knows, but Thomas has bought all the animals for her!
As they sing of their happiness, Carboy reminds us of his intended suicide. Thomas, moved by Carboy's devotion, comes to a financial arrangement with Grinder and obtains his permission for Lætitia's marriage to Carboy.
All rejoice as the curtain falls!
As with Burnand and Sullivan's Cox and Box, The Zoo is something of a D'Oyly Carte 'cuckoo in the nest' as it has nothing to do with W. S. Gilbert whatsoever. The piece was first produced at the St James' Theatre in June 1875 some 3 months after the première of Trial by Jury, and sustained a number of revivals at various theatres until 1879. Sullivan then withdrew the piece in the hope that his friend, Andy Cole, would re-write the rather inadequate libretto by B C Stephenson. However, this was not to be and the rumour that Sullivan had reused the score in later works was finally scotched in the late 1960's when Professor Terrence Rees purchased the autograph full score at a Southerby's auction. The opera proved to be delightfully intact and apart from one number (Carboy's 'I loved her fondly' which was the obvious model for 'A wand'ring minstrel' (Mikado)), Sullivan had left the score untouched and unused.
The opera made it's first modern appearance at Fulham in 1971 partnered with a revival of Thespis and again in 1972 in a triple bill with Contrabandista and Trial by Jury. Around the same time the BBC recorded a studio performance which was subsequently broadcast on at least two occasions.
DECCA released their recording with D'Oyly Carte in 1978.
Sadly someone felt it necessary to add animal sound effects and a narration, delivered by Geoffrey Shovelton, which add nothing to the performance. Despite this nothing can detract from Kenneth Sandford's remarkable performance as Thomas Brown and there is a rare chance to hear Meston Reid (a most delightful and sadly missed tenor) as Aesculapius Carboy.


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The Geco Scorpio is a small survey ship used to carry out oceanographic survey work ??? mainly for oil exploration. Built in 2006, the large open deck are aft is used to embark containerised survey equipment and cable reels.


text:
#ifndef QUICKSTEP_PARSER_PARSE_PRIORITY_HPP_
#define QUICKSTEP_PARSER_PARSE_PRIORITY_HPP_

#include <memory>
#include <string>
#include <vector>

#include "parser/ParseLiteralValue.hpp"
#include "parser/ParseTreeNode.hpp"
#include "utility/Macros.hpp"

namespace quickstep {

/** \addtogroup Parser
 *  @{
 */

/**
 * @brief A parsed representation of PRIORITY.
 **/
class ParsePriority : public ParseTreeNode {
 public:
  /**
   * @brief Constructor.
   *
   * @param line_number The line number of "PRIORITY" in the SQL statement.
   * @param column_number The column number of "PRIORITY" in the SQL statement.
   * @param priority_expression The PRIORITY value expression.
   **/
  ParsePriority(const int line_number,
                const int column_number,
                NumericParseLiteralValue *priority_expression)
      : ParseTreeNode(line_number, column_number),
        priority_expression_(priority_expression) {}

  /**
   * @brief Destructor.
   */
  ~ParsePriority() override {}

  /**
   * @brief Gets the PRIORITY expression.
   *
   * @return PRIORITY expression
   */
  const NumericParseLiteralValue* priority_expression() const {
    return priority_expression_.get();
  }

  std::string getName() const override {
    return "PRIORITY";
  }

 protected:
  void getFieldStringItems(
      std::vector<std::string> *inline_field_names,
      std::vector<std::string> *inline_field_values,
      std::vector<std::string> *non_container_child_field_names,
      std::vector<const ParseTreeNode *> *non_container_child_fields,
      std::vector<std::string> *container_child_field_names,
      std::vector<std::vector<const ParseTreeNode *>> *container_child_fields)
      const override {
    non_container_child_field_names->push_back("");
    non_container_child_fields->push_back(priority_expression_.get());
  }

 private:
  std::unique_ptr<NumericParseLiteralValue> priority_expression_;

  DISALLOW_COPY_AND_ASSIGN(ParsePriority);
};

/** @} */

}  // namespace quickstep

#endif  // QUICKSTEP_PARSER_PARSE_PRIORITY_HPP_


text:
Check Mega Millions Winning Numbers For September 6; Jackpot Nearing $200 Million
Written By Staff on September 6, 2022
Another Mega Millions drawing tonight offers players a chance at a life-changing jackpot prize. With no big winner in the previous drawing, the prize climbs to $191 million for Sept. 6. The lump sum cash value of Tuesday's jackpot is $105.3 million before withholding.
In the previous drawing, a New York player got lucky and hit the second-tier prize by matching all five white balls drawn. That prize is usually $1 million, but the winner had the Megaplier option on their ticket. With a multiplier of 2x drawn on Friday, the prize was doubled to $2 million.
Check tonight's Mega Millions winning numbers below, after the drawing at 11:00 p.m. ET.
Winning Mega Millions numbers for Tuesday, September 6, 2022
Winning numbers: 6 – 17 – 46 – 59 – 68
Mega Millions' largest jackpots
Did you know that Mega Millions has awarded three jackpot prizes that exceeded $1 billion? It's true! In fact, two of those jackpots were hit since 2021. The most recent jackpot, won by a player in Illinois on July 29, reached $1.337 billion. Just last year, a Michigan player won $1.050 billion on Jan. 22.
Furthermore, there has been one prize larger than those two. The largest Mega Millions prize ever won was $1.537 billion, which is also the world's largest lottery prize ever won on a single ticket. That whopping prize was hit by an anonymous player in South Carolina in 2018.
Since the jackpot amount is based on ticket sales, it takes some time for the prize to reach those heights. This year, it took about three months of ticket sales to push the prize to the $1 billion mark in July.
PA Lottery tickets and games now available online
PA players can now buy Mega Millions and other tickets online at the PA online lottery. Sign up for access to your favorite PA Lottery games like Powerball, Cash4Life, Treasure Hunt, Match 6 Lotto, and the PICK games. Online players have the same odds of winning the same great prizes as tickets bought from retailers.
Get started for free with a $20 free play bonus when you sign up using our link. You'll also get a deposit match bonus up to $500.
Check Powerball Winning Numbers for January 25; Jackpot Hits $526 Million Tonight
PA Lottery Players Claimed Over $211 Million In November Scratch-Off Prizes
Mega Millions Jackpot Worth $1.35 Billion Hit In Maine January 13
Check Mega Millions Winning Numbers for November 1, Jackpot Reaches $87 Million
Single Ticket Wins World Record $2 Billion Powerball Jackpot In California
Mega Millions Jackpot Rolls To $45 Million For Tuesday, October 25; Check Winning Numbers