What is staffing really like? We asked Kelsie, from our San Francisco office, to give us her insight about sales & recruiting.
What did you think of "staffing" before working at Talener? — I was familiar with staffing because my parents used to own their own staffing agency for 15 years. I used to hear a LOT of phone conversations, but I had no idea what they were actually doing. Now I think I sound just like my mom.
What did you think of "sales" before working here? — I was terrified of sales. To be honest, I didn't realize that recruiting (which I had even witnessed my parents doing when I was growing up) was the same thing as sales. I started at Talener and they had told me this was a sales job. I had no idea what I was doing, but I dove in! I never wanted a "sales" job, but I'm really enjoying this.
What do you think of staffing now? — It's way more relationship-based than I thought. This job gets easier as you talk with, meet, and in some way connect with people. As long as you're putting yourself out there and meeting new people every week, you will find new clients and new candidates– matching them is up to you and your organization.
After doing this for a while, what do you think of sales? — Same as staffing, it's highly dependent on relationships you have. It's definitely high-energy and can be stressful, but again, as long as you're engaging people, whether it be over the phone or in person, you will prosper. We all have bad sales months, but those can teach you lessons that apply to life outside of work as well. For example, I've learned that it's not about your attitude when you're doing well – it's how you behave when you're not doing well that sets you apart.
Has working here changed your opinion about the bad rep that sales & staffing gets? — Yes, one of the best things about working here is that I always have help. The managers are tuned in and helping us one-on-one all day, so you never feel lost. The company is full of people that I look up to – the management is really impressive. Plus, everyone at Talener starts in the same position and moves up from there; so they understand all of the different scenarios you could be going through as a junior employee.
Kelsie is a Lead Relationship Manager in our San Francisco office. She is an expert in placing candidates in the front end space and has extensive knowledge of the current market. Looking for a front end position in the Bay area or have a position that you need to fill? Contact Kelsie, kkorn@talener.com, to get her expert opinion about market conditions, upcoming hiring trends, or to find out more about staffing.


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text:
July 28, 2010 at 1:48 PM EDT
HOUSTON, Jul 28, 2010 (GlobeNewswire via COMTEX) --
Crown Castle International Corp. (NYSE:CCI) announced today that certain of its indirect subsidiaries intend to offer, in a private transaction, up to $1.55 billion of Senior Secured Tower Revenue Notes ("Offered Notes"), as additional debt securities under the existing Indenture dated as of June 1, 2005 ("Indenture"), pursuant to which the Senior Secured Tower Revenue Notes, Series 2006-1 ("Series 2006-1 Notes"), and the Senior Secured Tower Revenue Notes, Series 2010-1, Series 2010-2 and Series 2010-3, were issued. The subsidiaries expected to issue the Offered Notes are special purpose entities that are the current issuers under the Indenture and that hold over half of the U.S. and Puerto Rico towers of Crown Castle. Crown Castle expects that all of the Offered Notes will be rated investment grade. The servicing and repayment of the Offered Notes are expected to be made solely from the cash flow from the operation of the U.S. towers that are part of the transaction.
Crown Castle expects to use the net proceeds received from this offering to repay in full the Series 2006-1 Notes, together with related prepayment premiums, fees and expenses. Crown Castle expects the offering of the Offered Notes to close on or about August 16, 2010.
This press release contains forward-looking statements that are based on Crown Castle management's current expectations. Such statements include plans, projections and estimates regarding (i) the proposed offering of the Offered Notes, including the dollar amount to be raised through such notes offering, (ii) the investment grade rating of the Offered Notes, (iii) the terms of the Offered Notes, including with respect to servicing and repayment and (iv) the use of proceeds from the Offered Notes. Such forward-looking statements are subject to certain risks, uncertainties and assumptions, including prevailing market conditions and other factors. Should one or more of these risks or uncertainties materialize, or should underlying assumptions prove incorrect, actual results may vary materially from those expected. More information about potential risk factors that could affect Crown Castle's results is included in our filings with the Securities and Exchange Commission. The term "including", and any variation thereof, means "including, without limitation".
CONTACT:  Crown Castle International Corp.

redpajama_set_name: RedPajamaCommonCrawl


BA's who purchase the PPA (formerly known as "TLA") package can take the Partner Program Sales Certification course by visiting the Dubli Network Academy (located under the "Education & Marketing" tab in the back office). TC's or higher who did not already purchase the TLA/PPA package can purchase the Certification course a la carte by proceeding to the "Partner Program" tab in the Dubli Network back office and selecting the "Buy Certificate" option. Once paid, you can access the Certification course as outlined above by visiting the Academy site.
Once you have passed your Certification course, you can then register new Partners at www.dublipartner.com by entering in your BAID in the "Sponsor" field on the registration page. It is important to note that there is no direct referral link for Partners and you must be a TLA/PPA or higher who has passed the Certification course in order for your BAID to work in the "Sponsor" field.


It's out of my hands. I've done what I can do. I showed up. I cast my vote. I said my peace. I encouraged others to do the same.
I am a workaholic. I am compelled to chase every idea I have. I feel responsible for fixing every problem for which I can perceive a solution. This is possibly my most defining characteristic - at least in my own characterization of myself.
I founded an opera company with no money, and only human resources, which, more than a decade later, continues to thrive against all odds. I've performed in operas across the world. I've designed policies and procedures, rebuilt filing systems, built databases, built accounting systems, raised money, produced concerts, written millions of words, sent and received bazillions of emails, repaired broken professional relationships for employers, collected tens of thousands of dollars in unpaid invoices, joined clubs, attended live performances, memorized millions of notes and words in foreign languages, taught countless voice lessons, and driven hundreds of thousands of miles. My life is rich for the people I've met and the experiences I've had. I am perpetually pushed to the limits of what my brain and body can handle. It's all very stimulating.
I've lost important relationships. I failed in my marriage. I have alienated friends and family members. I've shed many tears. I've missed important events in other people's lives. I've lost thousands of hours of sleep. I've made myself sick. I've had panic attacks. I've suffered from crippling depression. I've divided and subdivided my attention many times over. I've missed out on my own life because I was too focused on what came next. I've been profoundly lonely, lost, and unhappy. I've felt like I would never, ever be enough.
I am not alone in my workaholism. My struggle is not special or new or set apart from the struggles of others. But it's my struggle.
Seth Godin's quote, which is the subject of this blog post, showed up in my inbox this morning. It spoke to me. I believe he meant it as a call to action - that we should all do what we can do, but I heard it as a call to let go, to sit still, and to have faith. It will be particularly hard to do those things today, given the stakes, but me wringing my hands will accomplish nothing. Obsessively refreshing FiveThirtyEight will not change the outcome. Posting articles on the echo-chamber that is Facebook will not sway anyone in my circles to vote any differently than they'd already planned. For me, as a citizen and not a campaign staffer, having already cast my vote, the work is done.
I want to be more present, more relaxed, more healthy, more available, and free from the pressure of self-imposed obligations. I want my artistry to be more complete and my experience of life on this planet to be more whole. I want my relationships to be as deep and as meaningful as they can be.
If I can use today, this election day, to practice non-doing, maybe that's a good start. And maybe that's enough.
My friends are all smarter than I.
I spent the evening at a friend's birthday gathering. The attendees at this event included two composers, an actor, a choreographer, a filmmaker, and me. They were all, undoubtedly, smarter than I am and I found myself humbled by my lack of knowledge about several topics of conversation - although I definitely held my own in the area of vocal style and the operatic tradition. I left the evening feeling exhilarated by stimulating conversation and embarrassed by many glaring holes in my knowledge of certain topics of great global significance. On my way home, I made a promise to myself to read more.
I also have a group of girlfriends with whom I have an ongoing group text. We hear from each other most days about both the mundane and the extraordinary events of our daily lives. Every day I am humbled by each of their strengths, insights, and commitment to personal growth. They make me strive to live a better, more meaningful, more honest life. They inspire me to let go of things that do not serve me and to think about things in new ways. I am a better person because of these ladies.
I hope you, dear reader, also have people in your life who challenge you to grow and learn.


text:
Keith Vossel
Mary S. Easton Center for Alzheimer's Disease Research at UCLA, United States
Peter S. Pressman
University of Colorado Denver, United States
Samantha K. Holden
School of Medicine, University of Colorado Anschutz Medical Campus, United States
MINI REVIEW article
Front. Neurol., 03 July 2019
Sec. Dementia and Neurodegenerative Diseases
Volume 10 - 2019 | https://doi.org/10.3389/fneur.2019.00713
Chronic Traumatic Encephalopathy: A Brief Overview
Arman Fesharaki-Zadeh*
Yale Neurology, Yale Medicine, New Haven, CT, United States
Chronic Traumatic Encephalopathy (CTE) is a debilitating neurodegenerative disease, which has been increasingly reported in athletes, especially American football players, as well as military veterans in combat settings, commonly as a result of repetitive mild traumatic brain injuries (TBIs). CTE has a unique neuropathological signature comprised of accumulation of phosphorylated tau (p-tau) in sulci and peri-vascular regions, microgliosis, and astrocytosis. As per most recent disease classification, the disease manifests itself in four different stages, characterized by widespread tauopathy. Clinically, CTE has a more subtle presentation, as patients often present with two distinct phenotypes, with one subtype initially presenting with affective changes, and the other subtype with more cognitive impairment. On a genetic basis, there are no clear risk factor genes. Although ApoE4 carriers have been reported to suffer more severe outcome post TBI. As there are no disease modifying regimen for CTE, the newly developed TBI treatments, if administered in a time sensitive manner, can offer a potential viable option. Prevention is another key strategy that needs to be implemented in various sports and military settings. Providing education for safe practice techniques, such as safe tackling and hitting, and providing ready access to full neuropsychiatric assessment by team physician could have measurable benefits. The combination of advanced of research techniques including neuroimaging, as well as increasing public awareness of CTE, offers promising vistas for research advancement.
Chronic Traumatic Encephalopathy (CTE) is a distinctive tau-protein associated neurodegenerative disease. There has been a rise of CTE diagnosis in athletes, especially American football players, as well as in military veterans in combat settings (1, 2). Although CTE has been publicly recognized relatively recently, it was first described as "punch drunk" syndrome in a classic article by Martland et al. (3). The report was focused on a number of boxers who had suffered repetitive head blows throughout their careers, and were presenting with both psychiatric symptoms as well as severe memory and neurocognitive deficits, analogous to typical dementia patients (3). The disease nomenclature evolved into "dementia pugilistica" (4), and finally CTE in 1949 (5).
CTE has a unique neuropathological characteristic, comprised of accumulation of phosphorylated tau (p-tau) in sulci and peri-vascular regions, microgliosis, and astrocytosis. These pathological changes lead to progressive debilitating neurodegeneration. Based on the pattern of pathological progression, CTE is divided into four respective stages (Figure 1). In stage I CTE, the brain grossly appears normal, but p-tau is found in a finite number of loci, often in the lateral and frontal cortices, as well as proximal to small blood vessels in the depth of sulci. There might be a scant number of neurofibrillary tangles (NFTs) and neurites in the locus coeruleus. In stage II, localized macroscopic abnormalities might be noted. On gross anatomical sections and neuroimaging, enlargement of lateral ventricles, cavum septum pellucidum with or without fenestration, as well as pallor of the locus coeruleus and substantia nigra are observed. There are multiple foci of p-tau within the depth of sulci, and there is an emergent spreading pattern. In stage III, most gross pathological sections show macroscopic abnormalities. There is global brain weight loss, mild frontal lobe and temporal lobe atrophy, and dilation of the ventricles. One half of CTE patients display septal abnormalities, including cavum septum pellucidum. P-tau pathology spreads, involving the frontal, temporal, parietal and insular cortices. In stage IV, the reduction in brain weight is dramatic, and brain weights of 1,000 g (compared to 1,300–1,400 g in normal brains) have been reported. There is profound atrophy of the frontal, medial temporal lobes, as well as anterior thalami. There is also atrophy of the white matter tracts. The majority of stage four patients have septal abnormalities. The spread of the p-tau affects most regions, including the calcarine cortex (7, 8). Abnormalities in phosphorylated 43 kDa TAR DNA binding protein (TDP-43) is also seen in most CTE patients. The parenchymal TDP-43 pathology is also progressive in nature similar to the anatomical pattern of spread of p-tau. TDP-43 immunoreactivity is found in almost all cases of stage IV disease (7).
Figure 1. The above images are depiction of the McKee's four stages of CTE [adopted from Mez et al. (6), Figures 1, 2].
The CTE clinical phenotype is yet to be clearly defined. The following paragraphs outline attempts of characterization of CTE symptoms in the various stages of the disease process (Table 1). According to McKee's classification, in stage I, a typical CTE patient is asymptomatic, or may complain of mild short term memory deficits and depressive symptoms. Mild aggression may be observed. In Stage II, the mood and behavioral symptoms could include behavioral outbursts and more severe depressive symptoms. In Stage III, patients typically present with more cognitive deficits, including memory loss, executive functioning deficits, visuospatial dysfunction, and apathy. In Stage IV, patients present with advanced language deficits, psychotic symptoms including paranoia, motor deficits, and parkinsonism.
Table 1. CTE proposed clinical classifications.
Jordan et al. (10) were one of the first to clinically characterize the disease. They divided CTE clinical presentations into three domains: behavioral/psychiatric, cognitive, and motor. The behavioral and psychiatric domain included aggression, depression, apathy, impulsivity, delusions including paranoia, and suicidality. The cognitive domain included diminished attention and concentration, memory deficits, executive functioning deficits, visuospatial dysfunction, language deficits, and dementia. Finally, the motor features consisted of dysarthria, gait abnormalities, ataxia and incoordination, spasticity, and parkinsonism features such as tremors. Based on these clinical features, as well as existent neuropathological information, four diagnostic subtypes were defined, namely "Definite," Probable," "Possible," and "Improbable" CTE.
Stern et al. (11), and related case reports (14, 15), differed in their description of a typical CTE patient, conceptualizing the clinical presentation into two distinct subtypes. The first subtype displayed mainly behavioral and mood changes, and the other presented with mainly cognitive impairment. The vast majority of the mood/behavior subtype developed cognitive deficits as the disease progressed. However, relatively few patients of the cognitive group displayed mood or behavior alterations during the course of their illness. In study by Stern et al. (11), the cognitive group patients had a significantly higher probability of developing dementia. They also were significantly older at the time of diagnosis compared to the mood/behavior group patients. The behavioral subgroup of CTE patients can resemble patients suffering from behavioral variant frontotemporal dementia (bvFTD), which makes the clinical diagnosis more challenging. However, typical bvFTD characteristic behavioral manifestations such as apathy and disinhibition are often not seen in CTE patients (11, 16). Given the inherent heterogeneity of bvFTD, as well as similar tauopathic nature of both diseases, distinguishing bvFTD and CTE poses a diagnostic challenge.
Out of the behavioral symptoms of CTE, the association between suicide and CTE remains a topic under scrutiny in the literature. Earlier studies, such as the series of five professional athletes with a confirmed diagnosis of CTE reported by Omalu et al. (17), had suggested a strong relationship between CTE and suicide. The authors further suggested that the etiology of suicidal/parasuicidal behavior in the CTE population might be partly due to tauopathy in the form of neurofibrillary tangles and neuritic threads in strategic limbic brain nuclei such as locus ceruleus. Maroon et al. (18), reviewed 153 pathologically confirmed cases of CTE published between 1954 and 2013. They reported the suicide prevalence in the CTE population and accidental deaths to be 11.7 and 17.5%, significantly higher than the general population levels of 1.5 and 4.8%, respectively (18). Proponents of opposing view suggest that suicides have been mostly reported in earlier stages of CTE, and the association between disease progression and suicide remains unclear at this time (19).
In a meta-analysis of 158 case studies by Gardner et al. (12), CTE clinical symptoms were divided into "classic" vs. "modern" CTE symptoms, to draw a distinction between an older description of CTE cases centered mostly on boxers compared to a more evolved clinical description which also applies to professional American football players. Whereas, the "classic" CTE symptoms typically included dysarthria, movement difficulties, and later progression to memory deficits, the "modern" CTE picture also included neuropsychiatric symptoms, such as depressive symptoms, paranoia, social withdrawal and isolation, compromised judgment and aggression. Cognitive deficits such as memory decline, executive dysfunction, language, and information processing deficits emerge later in the course of the disease process (12).
Since the definition of CTE primarily depends on pathological characteristics, there is a proposed alternative clinical term of traumatic encephalopathy syndrome (TES) by Montenigro et al. (13), describing the clinical sequelae of repetitive TBIs. The authors based this classification on a review of 202 published cases. TES is a more encompassing diagnosis and can be subdivided into four subcategories, including TES behavioral/mood variant, TES cognitive variant, TES mixed variant, and TES dementia. The proposed TES diagnosis was based on the existence of five general criterion, three core clinical features, and nine supportive features. Using existent biomarkers* (Table 1), additional diagnostic qualifiers were proposed, which included "Probable," "Possible," and "Unlikely" CTE (9, 13). The proposed TES diagnosis also contained temporal qualifiers and included "progressive course," "stable course," and "unknown/inconsistent course." If the clinical presentation also included motor signs such as parkinsonism, the modifier "with motor features" was also added.
As our understanding of CTE grows, there are a number of challenges and critiques that need to be addressed. One hypothesis as an alternative to the phenomena CTE, is a diminished "cognitive reserve" theory. The theory states that repetitive neurotrauma leads to a reduction in cognitive reserve and acceleration of development of an underlying neurodegenerative disorders (20, 21). If this theory held true, it would imply that CTE and AD are on the same neuropathological spectrum. This assertion deserves further analysis. Similar to AD, the Tau isoforms in CTE also consist of the mix of three-repeat (3R) and four-repeat (4R) isoforms. However, according to a recent report by Falcon et al. (22), the tau filaments extracted from the brains of CTE patients also contain a unique ß-helix region with a hydrophobic cavity, which is not present in the brains of AD patients. The cavity contains an additional cofactor that is thought to play a functional role in tau propagation. Falcon et al. (22) suggest that the location of tau inclusions in proximity to blood vessels, suggest that cofactors necessary for tau assembly may cross the blood brain barrier after head trauma. The authors further argue that the fact that brain trauma leads to CTE in only a subgroup of injured population, might be related to higher level of cofactors in the more susceptible individuals. These cofactors might provide a therapeutic target for prevention of tau assembly and development of CTE post injury (22).
An alternative theory proposes that the psychiatric symptoms such as depression and anger reported in CTE patients are independent of the CTE disease process and are reported in a cofounded fashion. The proponents of this hypothesis have cited prior studies such as the one reported by Weir et al. (23), in which 1,063 former NFL players were asked whether they have experienced bouts of anger. It was reported that 30.7% of the players ages 30–49, and 29.3% of the players ages 50 or above reported bouts of anger. However, the authors also noted that the reported measures of anger was indeed lower than the one reported for the general US population, which was 54.8% for men between 30 and 49, and 47.2% for men above the age of 50 (23). Though the arguments pertaining to comorbidity of psychiatric symptoms and neurodegenerative diseases such as CTE, are difficult to verify based on neuroimaging and neuropathological findings, one can apply similar arguments to psychiatric symptoms of any neurodegenerative condition such as AD, bvFTD, Parkinson Disease (PD) or amyotrophic lateral sclerosis (ALS).
Another important source of diagnostic confusion is the clinical delineation between CTE and prolonged post-concussive syndrome (PCS), especially given prior reports indicating that ~10–20% of individuals who suffer concussions, experience prolonged symptoms. Chronic Postconcussive Syndrome (CPCS) refers to persistence of PCS symptoms leading to impaired functional and often athletic performance lasting longer than 1 year. CPCS symptoms include headache, dizziness, impaired attention, memory and executive functioning deficits, depression and irritability symptoms (10). King and Kirkwilliam coined the term, "Permanent PCS" to refer to those with PCS symptoms persisting an average of 6.9 years after the initial concussion. Furthermore, they reported that a significant number of permanent PCS patients (40–59%) also had premorbid or postmorbid neuropsychiatric conditions such as depression, anxiety, PTSD, and/or pain (24). As argued by Jordan et al. (10), CPCS is clinically distinguishable from CTE, based on its temporal relationship to the acute concussive event. A thorough and accurate temporal history remains key in the neurological assessment. Furthermore, headache is a central feature of CPCS but not commonly reported in CTE. Although arguable, McKee stages I and II patients could present with headaches, further adding the complexity of possible overlap of CTE & CPCS (9). The CPCS diagnosis remains controversial, as it is not clear whether it is tauopathic in nature. Hence, the dividing lines of CPCS and McKee's stages I and II clinical characteristics are not fully solidified.
Clear genetic predispositions to CTE have not been reported. However, the ApoE4 gene, the most well-known risk factor for Alzheimer's Disease (25), has been associated with greater cognitive deficits and a more protracted recovery period after Traumatic Brain Injury (TBI) (11). A study on a group of boxers has reported more severe outcomes in individuals carrying at least one ApoE4 allele (26). Conversely, ApoE3 might confer neuroprotection, even in the presence of a progressive CTE pathology (15). Another proposed protective factor associated with more favorable post TBI recovery is cognitive reserve, as measured by premorbid IQ and total intracranial volume (27). Other genetic candidates for further study include the microtubule-associated protein tau (MAPT) gene, the progranulin (GRN) gene, and the chromosome nine open reading frame 72 (C9ORF72) gene (11).
The pathologic synergism of tauopathy and neuroinflammation is increasingly being recognized. Extracellular secretion of hyperphosphorylated tau is thought to activate microglia and astrocytes, leading to production of pro-inflammatory cytokines such as IL1ß, and TNFa, in turn leading to activation of tau kinases such as p38 and cdk5, and further tau phosphorylation. This process creates a vicious perpetual tauopathy and neuroinflammaion cycle (28). Given the robust association between repetitive traumatic brain injuries and risk of CTE (1), timely treatment of TBI could diminish the development of CTE. The pro-inflammatory nature of TBI has been previously reported (13), and anti-inflammatory agents such as minocycline with N-Acetylcysteine, a potent anti-oxidant, administered in acute to subacute time windows post TBI, offer a promising therapeutic regimen (29, 30). The development of a time sensitive protocol, resembling the treatment algorithm for ischemic stroke, would potentially measure long term outcome in post TBI recovery and prevention of development of subsequent CTE pathology (29).
There are currently no disease modifying medications for CTE, making prevention the most effective way of combating this debilitating neurodegenerative disease (31). Given the frequency of head collisions in contact sports such as American football, prevention of head trauma will require a cultural shift in the way the sport is taught and practiced. Training for safe practice techniques, such as safe tackling and hitting, while penalizing reckless hits will offer measurable benefits. Further changes must include creating an environment of safety, in which players are encouraged to report symptoms to referees, coaches as well as to team physicians. Furthermore, establishing a baseline neurocognitive profile could be used as a clinical reference marker to track changes in players' neuropsychiatric presentation. It is incumbent upon the team physicians to remove players from the field who have suffered even a mild uncomplicated TBI for further assessment (32).
There are a number of existent CTE related challenges to address. Although the incidence of sport related concussion has been reported to range from 1.6 million to 3.8 million, the incidence and prevalence of CTE remains largely unknown (33). One explanation for this lapse of knowledge is perhaps due to the fact that athletes exposed to cumulative subconcussive hits, which exert sufficient force to confer neuronal damage but initially have no overt clinical symptoms, are often not assessed or diagnosed in a timely manner (34). Large scale prospective studies, such as tracking athletes with multiple TBIs over a predefined period, would add to our understanding of the natural course and phenomenology of the disease. CTE is increasingly reaching the public spotlight via the mass media. Continuous efforts to diagnose, assess, and treat this devastating illness are needed. The exponential advancement in neuroimaging techniques and understanding of the neuropathological mechanisms of the illness will lead to earlier diagnosis and timely treatment interventions.
The author confirms being the sole contributor of this work and has approved it for publication.
The author acknowledges the great support, guidance and mentorship of Dr. Stephen Strittmatter.
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29. Sangobowale MA, Grin'kina NM, Whitney K, Nikulina E, St., Laurent-Ariot K, Ho JS, et al. Minocycline plus N-acetylcysteine reduce behavioral deficits and improve histology with a clinically useful time window. J Neurotrauma. (2018) 35:907–17. doi: 10.1089/neu.2017.5348
30. Haber M, James J, Kim J, Sangobowale M, Irizarry R, Ho J, et al. Minocycline plus N-acteylcysteine induces remyelination, synergistically protects oligodendrocytes and modifies neuroinflammation in a rat model of mild traumatic brain injury. J Cereb Blood Flow Metab. (2018) 38:1312–6. doi: 10.1177/0271678X17718106
31. Livingston G, Sommerlad A, Orgeta V, Costafreda SG, Huntley J, Ames D, et al. Dementia prevention, intervention and care. Lancet. (2017) 390: 2673–734. doi: 10.1016/S0140-6736(17)31363-6
32. Saulle M, Greenwald BD. Chronic traumatic encephalopathy: a review. Rehabil Res Pract. (2012). 2012: 816069. doi: 10.1155/2012/816069
33. Langlois JA, Rutland-Brown W, Wald MM. The epidemiology and impact of traumatic brain injury. J Head Trauma Rehabil. (2006) 21:375–8. doi: 10.1097/00001199-200609000-00001
34. Martini D, Eckner J, Kutcher J, Broglio SP. Subconcussive head impact biomechanics: comparing differing offensive schemes. Med Sci Sports Exerc. (2013) 45:755–61. doi: 10.1249/MSS.0b013e3182798758
Keywords: TBI, CTE, tau & phospho-tau protein, concussion and sports, dementia pugilistica
Citation: Fesharaki-Zadeh A (2019) Chronic Traumatic Encephalopathy: A Brief Overview. Front. Neurol. 10:713. doi: 10.3389/fneur.2019.00713
Received: 04 March 2019; Accepted: 17 June 2019;
Published: 03 July 2019.
Keith Vossel, University of Minnesota Twin Cities, United States
Peter S. Pressman, University of Colorado Denver, United States
Samantha K. Holden, University of Colorado Denver, United States
Copyright © 2019 Fesharaki-Zadeh. This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.
*Correspondence: Arman Fesharaki-Zadeh, arman.fesharaki@yale.edu
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David Ray Griffin (1939-2022)
December 3, 2022 December 3, 2022 Fran
David Ray Griffin, a moral giant among men, passed away on November 25, 2022. We in Colorado are immensely grateful for his two lectures here in 2007. He told us that one of his favorite presentations was "9/11 and Nationalist Faith," which he delivered at Denver's Iliff School of Theology, hosted by Dr. Vincent Harding, professor emeritus and speech writer for Dr. Martin Luther King, Jr. A second presentation on that trip, "9/11 Contradictions," was based on his book of the same title, and was given at the University of Colorado, Boulder.
We remember so well both presentations, but his words on nationalist faith ring especially strongly in our minds to this day:
"'People of faith' are often accused of allowing their faith to override evidence. This is often true. With regard to 9/11, the greatest obstacle to seeing the truth — that 9/11 was an inside job — is not the lack of evidence but what can be called 'nationalistic faith' — the conviction that the leaders of our nation simply would not do such a thing. Genuine faith — faith in God the creator and lover of all peoples — can help overcome the insanity created by 9/11."
Elizabeth Woodworth, who co-founded with Griffin the 9/11 Consensus Panel and worked closely with him as editor and co-author of two of his books, writes eloquently about this man whom we all love and treasure for his unceasing efforts to bring peace to our world through transparency and truth. We turn our commemoration over to her.
by Elizabeth Woodworth
How big can a mind be?
If we're lucky, we have threescore and ten years — in a very big wide world, full of history — to experience as much as we can take in.
Threescore-ten is not nearly enough, but some extraordinary people manage to encompass and give order to a lot of it.
And some even more extraordinary people manage to rise above their own lives to interpret creation and the fabric of the universe as having consistent meaning across cultures and throughout the ages.
David Ray Griffin was Professor of Philosophy of Religion and Theology, at the Claremont School of Theology and Claremont Graduate University, from 1973-2004. With his senior, Dr. John Cobb Jr., he co-founded the Center for Process Studies in 1973.
Griffin has stated that "the task of a theologian is to look at the world from what we would imagine the divine perspective, one that would care about the good of the whole and would love all the parts."
Not only was David an outstanding theologian and one of the two best-known living scholars of Alfred North Whitehead's process theology (the other being John Cobb): His books also spanned the related fields of postmodernism, theodicy (defence of God against evil), primordial truth, panentheism, scientific naturalism, parapsychology, Buddhist thought, and the mind-body interaction.
About the time that he retired in 2004, he was approached by some people who admired his candor, and pointed to evidence that the 9/11 event was highly suspicious.
At first David thought that 9/11 was simply blowback from the way America had treated the Middle East — but upon researching it more deeply he realized that there was indeed a very serious likelihood that the US had contrived 9/11 as a false flag operation to manufacture consent to occupy Afghanistan and Iraq for their oil.
This injustice fired his energy to research in depth, then write a dozen scholarly books on 9/11 — books that were not acknowledged in the media but which engaged in a cat-and-mouse game with the purveyors of the official 9/11 narrative, who continually adapted their story to cover up the weaknesses that David tracked and revealed as their tattered narrative evolved.
The first and most famous of these books was The New Pearl Harbor: Disturbing Questions about the Bush Administration and 9/11, published by his much-appreciated Interlink press in March 2004.
That best-seller was followed in 2005 by a devastating takedown of the Bush Administration's whitewash Commission titled The 9/11 Commission Report: Omissions and Distortions, which exposed 115 problems in "the 571-page lie".
Following these early 9/11 works, David was nominated for the Nobel Peace Prize in both 2008 and 2009, and was named among "The 50 People Who Matter Today" by the New Statesman, on September 24, 2009.
In November, 2008, David's seventh book about 9/11, The New Pearl Harbour Revisited, was one of only 51 books awarded as "pick of the week" that year by Publishers Weekly.
What followed was extraordinary.
As the foremost book reviewing tool in the English language, Publishers Weekly's spotlight should have led to reviews in the New York Times, the Times Literary Supplement, Library Journal, and many other top reviewing sources — but the word was out in the narrative-controlled media to give it a pass.
In 2011, David and I founded an organization called the 9/11 Consensus Panel, comprised of more than 20 professionals expert in various aspects of the 9/11 attacks. In 2018, the 51 consensus points that were developed during this unique evidence-based reviewing project were published under the title 9/11 Unmasked: An International Review Panel Investigation (2018).
During that seven-year project, David addressed the existential crisis of climate change, penning his encyclopaedic 2015 reference, Unprecedented: Can Civilization Survive the CO2 Crisis? (I took that book to the COP21 Paris climate summit in 2015, and presented it there, following up with a YouTube documentary on that enormous gathering of humanity – the largest meeting since World War II.)
David then turned his attention to US imperialism – writing Bush and Cheney: How They Ruined America and the World in 2016, and producing the incredible work of scholarship, The American Trajectory: Divine or Demonic, in 2018.
David was at last able, in 2019, to turn to his long-planned The Christian Gospel for Americans: A Systematic Theology. It is a magnum opus of enormous breadth and depth.
In it, for example, he confronts the science vs religion issue, showing that some scientists – former atheists – have been overwhelmed by the extent of exceedingly precise ratios between the chemical elements of earth that are required for life, to now saying that the universe was "fine-tuned for life," thus reflecting a "fine-tuner" (or divine creator).
In 2022, as he approached the end of his life, and following a long struggle with prostate cancer, David wrote the beautiful and crowning reflections of his maturing theology, James and Whitehead on Life after Death.
In the spirit of James and Whitehead, he explains that the universe is not separate from, but is within God, and is itself the very nature of God. This evolving world view requires a new understanding of the divine reality – panentheism, meaning "all in God".
The causal principles of the universe exist naturally, being inherent in the nature of things, because they exist in the very nature of God.
This chapter on the infinitely fine-tuned nature of the universe to support life is a transporting gift.
But he was not done yet!
Forthcoming in March, 2023, from the publisher Clarity Press, is David's America on the Brink: How the US Trajectory Led Fatefully to the Russia-Ukraine War – which was completed during the last days of his life.
In total David Ray Griffin has written 50 books and more than 200 essays. (He was once asked if he had ever had an unpublished thought!)
In all of his books – and most notably those on American imperialism – he read and cited recent scholarly investigations from top university presses, effectively overriding the propaganda that has passed down through many years.
Paul Craig Roberts wrote: "David has served truth to the hilt. He is a hero of our time."
There is no question that his body of work will go down in history as providing some of the most elegant thinking our century has witnessed.
And at some point, his chronicling of historically suppressed truths must emerge into full daylight, to allow reality-based civilization to advance.
Let us keep his work alive, so that earth's future peoples will inherit the great spectrum of wisdom he has left them: from a hopeful common-sense theology, to the exposés of imperialist propaganda and false flag operations, to the full extent of the climate crisis, to our evolving perception of the nature of the divine, to the evidence that our spirits will survive after death.
David Griffin stands with the greats – yet was quiet, humorous, down to earth, and unassuming.
Consult David Griffin's archive on Global Research.
David Griffin has passed, but his legacy will live.
Elizabeth Woodworth, a career medical librarian and author/co-author of five books, worked with David Ray Griffin in various capacities from 2006-2022. She did proof-reading/editing on about 12 of his books and many of his essays, co-authored two books with him directly, and has also written in-depth reviews of most of his books from the 2006-2022 period on Amazon.
Copyright © Elizabeth Woodworth
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David Ray Griffin, Vincent Harding. permalink.
Jonathan Cole, PE, Presents: 9/11 and the Scientific Method – Part 2: Temperature
Three Tributes to David Ray Griffin (1939 – 2022)
Two Video Interviews: Mick Harrison with George Galloway and Mick Harrison and Charlotte Dennett with Jason Goodman
New David Chandler Video – A Draft Contribution to "9/11: Crime Scene to Courtroom"
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Bibi takes White House fight to social media
By Kendall Breitman
02/10/2015 10:28 AM EST
Israeli Prime Minister Benjamin Netanyahu is taking his plight from the House floor to social media.
On Tuesday, Netanyahu tweeted, "I'm determined to speak before Congress to stop Iran. RETWEET if I have your support."
The tweet is paired with a photo image of the prime minister with the quote: "A dangerous agreement with Iran is taking shape in Munich which would risk Israel's existence. Therefore, I'm determined to travel to Washington and present Israel's position before Congress and the American people."
Unlike many of the prime minister's tweets, which are written in Hebrew, the tweets were timed for the East Coast's morning news cycle and written in English. About an hour after the tweet was first published, it had already gained about 1,600 retweets.
Netanyahu followed up on his initial tweet in the afternoon, when he tweeted, "I intend to speak in the U.S. Congress because Congress might have an important role on a nuclear deal with Iran."
Another tweetadded, "I am going to the United States not because I seek a confrontation with the President, but to speak up for the very survival of my country."
The tweets come after a week of dramatic headlineschronicling his dispute between the Obama White House, with many Democrats in Congress finding themselves forced to take sides.
The prime minister's address is scheduled for March 3, but the White House has begged off meeting with Netanyahu while he is in Washington, citing a policy of avoiding summits with foreign leaders ahead of their elections. Netanyahu is up for election on March 17.
Vice President Joe Biden announced last week that he will not attend the speech, and Sen. Bernie Sanders followed suit on Monday. On Tuesday, Sen. Patrick Leahy also saidhe will be skipping the speech.
On Tuesday, Sanders said the hosting of a foreign leader without presidential consent sets a "horrendous precedent," adding that Netanyahu "doesn't have the right to inject himself into an American political discussion."
"There are all kinds of avenues open to him to speak to members of Congress and that's fine," Sanders told CNN. "I don't think it's a good idea to have a foreign leader coming before Congress to rip apart the president of the United States."
White House,
Israel,


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As we enter a second season of protest of our country's flag by young, wealthy black NFL athletes, millions of fans will continue to turn off America's favorite past time. What could be better than waking up each morning to a panoramic view of a legend like Boston's Fenway Park, or relative newcomer, Camden Yards in Baltimore?.
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"It prompted a lot of personal growth, being away from it, and seeing what matters and what doesn years ago, Levy began using the weeks before the start of organized team activities in April as an opportunity to put some distance between himself and football.
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At the Eagles and Jaguars games, the team owners also linked arms with players. They have the potential to ruin Eli Manning's passing attack.
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Policies that allow children to be charged as adults ignore scientific research about brain development and impulse control.
13, 2017″ > >Chicago history as a sanctuary cityA look back at the leaders and laws that have shaped Chicago involvement with the sanctuary movement.
"I've never been on the world stage at any one thing," says Bobby. Broadcasting isn't for everybody. Higgins told The Daily Advertiser in Lafayette he got fed up one Sunday and "told his wife, Becca, he was through.
The latter game joined the list when the Panthers picked up cornerback LaDarius Gunter off the NFL waiver wire. I have a hard time seeing Washington compete here.
The Sports Xchange NFL roundup: WR Johnson asks Texans for trade Andre Johnson spent months away from the team and ultimately decided his future is not with the Houston Texans.
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Sadly, it is also a trait that is lacking in both the public sphere and also in the masses, especially in the current generation of recent college graduates.
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Isn about football; it about freedom, Johnson said Monday. The difference, according to the league's rookie salary scale, will cost him something between $6 million and $15 million in contract value.
Hutchinson averaged 29.9 points per game as a freshman in 1983 84. Then she got me on Ellen, where I became a regular correspondent and built my brand for mainstream, not just football.".
Lynch's numbers 18 carries, 76 yards but his energetic and physical style reminded many people of his "Beast Mode" days in Seattle.
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For those players cut before June 1, all of that bonus money goes onto the current year's cap. Still, these games normally are fairly close, mostly because the network demands the champ play a formidable opponent.
The Packers can't afford to lose any more developmental time with Fackrell, a third round pick out of Utah State in 2016 whose injuries as a rookie slowed his progress.
Matt Ryan, ATL, at Chicago: Ryan had a stunning 2016 season as he led the Falcons to the Super Bowl and paced all quarterbacks in fantasy scoring.
He tweeted something and my phone went berserk. "I would like to keep moving forward in the right direction with everybody: equal rights, equal opportunity.
Baker plays with a competitive desire that leaps off the field when you watch him thanks to elite football character and above average explosiveness.
So every time Ezekiel's in a set position and he pops up and that ball is stuffed into his gut, he remembers the Frog.". I have to give props where they're due, however, and Sunday's game not only was the best defensive effort I have seen since maybe the third game of the 2014 season in a 20 9 win at home against Minnesota, but it also was the best job Sean Payton and company have done in their preparations in quite a few years.
Culpepper and Lacina didn't even end up being the biggest storyline of the season; that happened when Zeke Smith was outed as transgender by a fellow tribe member..
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It not a contradiction there.. How can they do this? Sure, it takes years of practice to become a top field goal kicker in the NFL.
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(I'm going to keep watching.) But we live in an era when more than two thirds of those surveyed believe that the league has a serious domestic violence problem.
He had been working hard. Or maybe a new structure altogether. The tweet raised the question of whether Pence actions had been planned in advance..
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text:
Q: Does serverperfmode work on Apple Silicon Macs? Is it possible to enable serverperfmode on an Apple Silicon Mac?
I am using an M1 Mac Mini on macOS 11.2, and following Apple's instructions. I enabled serverperfmode=1 in the NVRAM boot-args, and restarted the machine. However, I'm not seeing any of the values in sysctl -a, ulimit -u, or ulimit -n change.
I have successfully done this on Intel-based Mac Minis in the past. Is it possible that serverperfmode is simply not supported on Apple Silicon Macs?
Here are some of the values I'm seeing on this machine, which seem unchanged from before enabling serverperfmode.
% nvram boot-args
boot-args   serverperfmode=1
% ulimit -n
256
% ulimit -u
2666

redpajama_set_name: RedPajamaStackExchange


text:
← CN June 18, 2015
CN July 2, 2015 →
CN June 25, 2015
At last night's CPS meeting, the Board voted to borrow more than a billion dollars to keep the schools operating another year. But about 24 hours earlier pretty much nobody knew this was the plan. How did this happen?
"I don't know," says Sun-Times education reporter Lauren FitzPatrick."In the sense that it was not discussed. It looks like it's two chunks of borrowing – $200 million, andsome $935 million. But the Board of Education didn't say a word about it other than to cast a unanimous vote at the end of the meeting to approve both pieces of borrowing."
"Not even roll-call vote," adds Sarah Karp, now an investigative reporter with the Better Government Association.
Despite Interim CEO Jesse Ruiz's vow that he'd run a more transparent operation, there was no public involvement of any kind before the vote for this massive borrowing. "They did discuss a couple of other big items on the agenda," says Fitzpatrick, "So I was waiting for the money people to come out and talk about the borrowing, but it just never happened."
Any discussion that did occur at the Board level happened in a back room outside the public view. A day later there still aren't any more details about the loan – how it will be structured, when it will take effect, or when the first checks will arrive. "There's no real reason I can see why they would discuss this is executive session," asserts Karp. "This is not real estate, or personnel…the public would like to know a little more about this. My grandkids are going to be paying for it."
When we taped our discussion this morning there was optimism in the air that a new one-year placeholder contract was about to be drawn up between CPS and the CTU. But that was dashed a few hours later when the CTU published a statement that talks had broken off.
We asked how this deal would have been different from a one-year contract extension (provided for in the old, expiring contract) that both sides rejected at different times over the past few months.
"From the sound of it, this one-year deal that's being talked about right now would not have included a pay raise," explains Catalyst Chicago reporter Melissa Sanchez. "The previous one-year deal would have, if they had extended the contract. What they're getting at the moment is that CPS has backed off of their desire to stop paying the seven percent of the nine-percent pension pickup. That's what we're hearing. So for the union, they're at a standstill. They're not losing. And it gives them time to rally their troops."
Earlier this week, Mayor Emanuel said he had requested from Springfield a 40-day extension on the payment of about $635 million into the Chicago Teachers Pension Fund. But when the bill came before the House it fell well short of passage. It was mysterious. "Clearly CPS thought this was going to happen,' explains Fitzpatrick, "because Jesse Ruiz, somebody packed him up into a car and he appeared before committees and the full House, and he was there to make his case and turned up in person. I can't believe that if he thought this wasn't going to happen, he'd have taken the time to go."
It was widely believed that all the legislative leaders, the Governor, the Mayor and CPS were on board, and that the union and the pension fund were not in opposition. It seemed relatively non-controversial.
"The idea of the delay is that tax money will be coming in, in August," says FitzPatrick, "which is not in their pockets now. And if they could have forty days then they would be able to make the full pension payment as opposed to no payment or even a partial payment which was getting batted around for a while."
"I'll tell you the gossip I've heard, which is unconfirmed, that the Republicans were about to be able to take credit for solving the CPS problem. So if the deal, which was supposed to be a done deal, got pushed off, here comes Mike Madigan to the rescue a week later – and he gets to save the day, instead of the Republicans that were willing to cross the aisle in support of the 40-day delay."
Some good news this week, though, was Governor Rauner's announcement that he planned to sign that portion of the State budget that funds the schools. So, although almost everything else is still being debated, schools now know the size of the checks they'll be receiving from Springfield in August.
And finally, the Ernst and Young report. It was commissioned by CPS presumably to show the public just how bad things are.
"While we've all been waiting for the latest trick, they came in and said, there kind of aren't any," FitzPatrick tells us. I think it's page 24 of the report that lists all of the things that will pretty much all be needed to pull this off this year. Some are taxes, some is help from Springfield, some is classroom cuts. One of the other points that the report tried to make is that solving this problem can't just come from CPS alone. That there are all these other people who are gonna have to sign off on a lot of these proposals to pull them off too. The State, the City Council, retirees, unions – CPS cannot get out of this on its own, Ernst and Young was paid to say."
The CTU has said, and often repeated, that the financial mess was created by CPS, and that it's "broke on purpose". We ask if that's a credible claim.
"I don't think that they ever intended to go broke or spend themselves into oblivion," Fitzpatrick explains. "But I think the union would argue that it's a cute way of saying that they're not acting as though they're actually in financial crisis – that they're spending on big-ticket items and things that are not working, like the Aramark contract for example, the SUPES contract, which has blown up in their faces, they continue to open new schools. They're not acting like they're broke, and that they're digging themselves in even deeper."
And after last night's vote, about 1.1 billion dollars deeper.
This entry was posted in Chicago Newsroom past shows and tagged BGA, borrowing a billion, Bruce Rauner, Catalyst-Chicago, Chicago Sun-Times, CPS debt, Karen Lewis, Lauren FitzPatrick, Melissa Sanchez, Rahm Emanuel, Sarah Karp. Bookmark the permalink.


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text:
RUDN Journal of Psychology and Pedagogics
Vestnik Rossiiskogo universiteta druzhby narodov. Seriya: Psikhologiya i pedagogika
Keywords Psi Chi adaptability adaptation anxiety characteristics creativity education intercultural adaptation persistence personality personality traits self-actualization self-realization sociability social representations structure students system-functional approach tolerance values variables
Home > About the Journal > Editorial Board and Editorial Staff
Viktoriya B. Kurilenko, Peoples' Friendship University of Russia (RUDN University), Russian Federation
Assistant to the Editor-in-Chief
Irina A. Novikova, Peoples' Friendship University of Russia (RUDN University), Russian Federation
HONORARY EDITOR
Elena L. Grigorenko, University of Houston; Baylor College of Medicine; Yale University A), United States
Baptiste Barbot, Catholic University of Louvain, Belgium
Inna B. Bovina, Moscow State University of Psychology and Education, Russian Federation
Linda Jarvin, Paris College of Art, France
Anatoliy V. Kharkhurin, National Research University Higher School of Economics, Russian Federation
Hristo Kyuchukov, University of Silesia in Katowice, Poland
William S. New, Beloit College, United States
Ewa Ogrodzka-Mazur, University of Silesia in Katowice, Poland
Lilia K. Raitskaya, Moscow State Institute of International Relations (MGIMO University), Russian Federation
Grant J. Rich, Juneau, Alaska, United States
Harold Takooshian, Fordham University, United States
Eugen Tarnow, Avalon Business Systems, Inc., United States
Viktor P. Sheinov, Republican Institute of Higher School (Minsk), Belarus
Lazar Stošić, The Association for the Development of Science, Engineering and Education, Serbia
All the members are involved on the basis of a signed membership application for voluntary participation in the editorial team of the journal. The original applications are stored in the editorial.


Last week we toured three completely different Napa Valley mountain wineries. We visited Stony Hill Vineyard on Spring Mountain, Ladera Winery on Howell Mountain, and Nichelini Winery at the summit of Sage Canyon Road. This will be the first of three posts comparing these Napa Valley mountain wineries.
Stony Hill Vineyard on Spring Mountain is one of the oldest wineries in the Napa Valley and one steeped in tradition. Fred and Eleanor McCrea purchased the land in 1943 and released their first vintage in 1952. In 1972, young Mike Chelini began his stint as winemaker at Stony Hill and he continues in that position to this day. He loves his work, the land, and the owners. As he says "Life is good here at Stony Hill Vineyard." When we asked Mike Chelini if he is the Valley's longest-running winemaker, he says: "I think I am tied with Bill Sorenson over at Burgess." Bill Sorenson began his stint at Burgess in 1972, so who knows who started first. Nevertheless, these are impressive careers for both these winemakers.
The winery is presently in the hands of the McCrea's son Peter and his wife Willinda, and they continue to run the winery with the same tradition, making wines year after year that are consistent. Notably, the main character of the wines is that they are fermented and aged in neutral oak barrels. Their flagship wine, the Chardonnay, is quite different in style than most of the Napa Valley Chardonnays. No oak, but roundness in the mouth with good fruit flavors. Gewürztraminer and Riesling round out the table wine offerings, and a dessert Semillon is also offered. With a northeast exposure, the sun departs early in the day behind Spring Mountain leaving their vines in a much cooler climate than the rest of the Napa Valley. It is the ideal environment for these white wine grapes. Most of the wines are sold to their mailing list members (started in 1952) and to several local restaurants.
Something new is on the horizon at Stony Hill, a Cabernet Sauvignon. The Cabernet vineyard, planted six years ago, is located on an area of Spring Mountain that dips down and then elevates. The vines receive much more sun and the hope is that within a few years Stony Hill will produce a beautifully-aged Cabernet. For this wine Mike Chelini plans to age the grapes in new French Oak barrels. All he needs is a little more time for the roots of the vines to grow deeper into the soil. They have bottled some of this wine and for the moment have labeled it as Red Table Wine. When the character of this vineyard develops and the flavors are right, Stony Hill will release this as Cabernet Sauvignon.
A look inside the stone winery is a step back in time. I'm sure it looks very much the same as the day Mike Chelini stepped into the winery in 1972. There is no fancy equipment or a modern day crush pad or anything that smacks of high tech. It is all done the old fashioned way.
Stony Hill Vineyards is open by appointment only and guests are received at the McCrea's original home just adjacent to the winery. To get there, you must turn into the Bale Grist Mill State Park a few miles north of St. Helena. Don't go into the State Park, but turn left and head up the hill and follow the signs to Stony Hill. The climb is beautiful, sweeping through oak woodland forests. When you visit the winery, ask if you can take a walk down to the winery. Be prepared for a step back in time.


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I know you have seen it all over Facebook, the daily posts of the 25 things people are grateful for.
It is Thanksgiving and I am giving thanks for a lot, but in my own way.
Yes, I am truly thankful for my family, friends, having a roof over my head, and food to eat. I am not just a sarcastic little cookie trying to make you laugh (that is a bonus - hopefully).
I just feel like a lot of these lists are missing some truly important items. I just wanted to share my top 25 things I am thankful for that everyones Facebook posts seem to have missed.
Chocolate (although I think some people have Facebook posted their love of chocolate).
Freedom of Speech (okay minorly sappy, sorry).
Cranberry Sauce in a can!


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