text:
Q: Count and name content from a SQL Server table I have a table which is structured like this:
+-----+-------------+-------------------------+
| id  |    name     |        timestamp        |
+-----+-------------+-------------------------+
| 1   | someName    | 2016-04-20 09:41:41.213 |
| 2   | someName    | 2016-04-20 09:42:41.213 |
| 3   | anotherName | 2016-04-20 09:43:41.213 |
| ... | ...         | ...                     |
+-----+-------------+-------------------------+

Now, I am trying to create a query, which selects all timestamps since time x and count the amount of times the same name occurs in the result.
As an example, if we would apply this query to the table above, with 2016-04-20 09:40:41.213 as the date from which on it should be counted, the result should look like this:
+-------------+-------+
|    name     | count |
+-------------+-------+
| someName    |     2 |
| anotherName |     1 |
+-------------+-------+

What I have accomplished so far is the following query, which gives me the the names, but not their count:
WITH screenshots AS
(
    SELECT * FROM SavedScreenshotsLog
    WHERE timestamp > '2016-04-20 09:40:241.213'
)
SELECT s.name
FROM SavedScreenshotsLog s
INNER JOIN screenshots sc ON sc.name = s.name AND sc.timestamp = s.timestamp
ORDER BY s.name

I have browsed through stackoverflow but was not able to find a solution which fits my needs and as I am not very experienced with SQL, I am out of ideas.

A: You mention one table in your question, and then show a query with two tables.  That makes it hard to follow the question.
What you are asking for is a simple aggregation:
SELECT name, COUNT(*)
FROM SavedScreenshotsLog
WHERE timestamp > '2016-04-20 09:40:241.213'
GROUP BY name
ORDER BY COUNT(*) DESC;

EDIT:
If you want "0" values, you can use conditional aggregation:
SELECT name,
       SUM(CASE WHEN timestamp > '2016-04-20 09:40:241.213' THEN 1 ELSE 0 END) as cnt
FROM SavedScreenshotsLog
GROUP BY name
ORDER BY cnt DESC;

Note that this will run slower because there is no filter on the dates prior to aggregation.

A: CREATE TABLE #TEST (name varchar(100), dt datetime)

INSERT INTO #TEST VALUES ('someName','2016-04-20 09:41:41.213')
INSERT INTO #TEST VALUES ('someName','2016-04-20 09:41:41.213')
INSERT INTO #TEST VALUES ('anotherName','2016-04-20 09:43:41.213')

declare @YourDatetime datetime = '2016-04-20 09:41:41.213'
SELECT name, count(dt)
FROM #TEST
WHERE dt >= @YourDatetime
GROUP BY name

I've posted the answer, because using the above query can generate errors in converting the string in where clause into a datetime, it depends on the format of the datetime.

redpajama_set_name: RedPajamaStackExchange


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I can only guess that it was something like an internal tumour that ulcerated and bled or maybe an abscess (caused by a bad tooth) that burst.
I had a similar case (except I saw her die), she'd had a stroke initially and as she died started to bleed from the mouth. I, like Ann, suspected some sort of internal rupture. It could have course, been a total shock to the heart/respiratory system.


text:
Eos announces London-Dubai service
11 Feb 2008 by Mark Caswell
Eos has announced the launch of daily flights from London to Dubai, and a new service from Stansted to New York Newark.
The airline says it will begin flying daily from London Stansted to Dubai International Airport on July 6 this year. The flights will compete with rival all-business class carrier Silverjet (based in Luton), which started a daily service to Dubai in November, (see online news September 19, 2007). Details of prices and flight timings have yet to be released, but the carrier says it will begin issuing tickets for the service next month.
Meanwhile, Eos has also announced a daily London Stansted-New York Newark service, to add to its existing 44 flights between Stansted and JFK. The new route will start on May 5, shortly after American Airlines begins its second daily flight between Stansted and JFK, on April 7.
AA's two-class service on the route offers 30 angled lie-flat seats in business class (as opposed to the all-business class offering onboard Eos with 48 fully flat beds), but of course the former has advantages in terms of connections in the US, and the strength of its frequent flyer programme. Tickets for Eos Airlines' Newark route will be available from mid-February.
For more information visit eosairlines.com, americanairlines.co.uk.
Report by Mark Caswell
Tags: American Airlines, Eos
Just landed - Valentine's special
Dot2Dot adds Canary Wharf transfers

redpajama_set_name: RedPajamaCommonCrawl


text:
Presented on Thursday, January 21, 2010 .
The polio crisis of the early 20th century has been largely forgotten in the U.S. due to the creation of the Salk vaccine and the effective immunization campaigns of the 1950s. Unfortunately, the wild poliovirus (WPV) still remains a real public health threat in many corners of the world. In 1988, the World Health Assembly resolved to eradicate polio transmission by 2000, and as a result, global polio incidence declined by more than 99%, from an estimated 350,000 cases in 125 countries to 1,659 confirmed cases in 25 countries in 2008. Although there was substantial progress up to 2002 in preventing transmission of WPV, the pace of progress has since slowed or reversed. India, along with a handful of other nations with low routine immunization coverage, has served as a reservoir of wild poliovirus for importation into other countries that were once polio-free.
This session of Public Health Grand Rounds focused on current efforts to contain the remaining polio cases in India, as well as discussed measures needed to completely eradicate this public health burden globally.
Steve L. Cochi, MD, MPH
Senior Advisor, Global Immunization Division
National Center for Immunization and Respiratory Diseases, CDC
Harmid Jafari, MD
Medical Epidemiologist, Global Immunization Division
Mark A. Pallansch, PhD
Chief, Polio and Picornavirus Laboratory Branch, Division of Viral Diseases
Walter R. Dowdle, PhD
Director, Poliovirus Antiviral Initiative
Task Force for Global Health
Presentation Slide Deck pdf icon[6 MB, 100 pages]
February 2015, Vaccines
The polio crisis has been largely forgotten in the US due to the Salk vaccine and the effective immunization campaigns of the 1950s. Unfortunately, the wild poliovirus still remains a real public health threat in many corners of the world. Learn about current efforts to contain polio in India and globally.
June 2015, Vaccines
Measles is a highly contagious virus that spreads through the air when an infected person coughs or sneezes. The virus can cause serious health complications. Hear how increased focus on field and laboratory surveillance, and innovative vaccination solutions can accelerate the elimination of measles worldwide.


text:
Galveston Bay FoundationNews from the BayGalveston Bay Water Monitoring Duo Up for the Test
Dave Bulliner is a self-proclaimed "lab rat."
"I'm just like the little rats that run around in the lab," he said with a smile.
Bulliner's passion for lab work goes back to his college days at Southern Illinois University. He was a student worker in the zoology department, tasked with maintaining the channel catfish tank. His duties included routinely checking the oxygen levels and feeding the fish.
Following a 44-year hiatus from the lab to pursue a career in retail management, the Illinois-native finally donned a white coat again in 2011. He wanted to do something "a little bit more productive" in retirement and joined the Galveston Bay Area Chapter of the Texas Master Naturalists. There he learned about an opportunity to do bacteria testing as part of the Galveston Bay Foundation's volunteer water quality monitoring program.
"I said, 'Pow, that's what I want to do,'" Bulliner recalled.
The foundation first started monitoring the water quality of Galveston Bay in 1992. Originally called The Estuarine Sampling Team (TEST), the program relies on a team of volunteers to collect and test water samples from locations around the Bay each month.
The data loving Bulliner was hooked.
"I think data is one of the most important tools that scientists have to use when they go back and check things they've done and what they want to accomplish," he said.
Bulliner shared his passion for bacteria testing with Mike Petitt, a retired physician who joined the Texas Master Naturalists in 2018. Bulliner was Petitt's mentor and told him about the volunteer water monitoring team. It did not take much convincing for Petitt – a native Houstonian who grew up going to the Bay – to see the importance of the program.
"We live down here. It's an important thing to make sure our Bay is maintained," he said. "If you don't measure it, you don't know it."
At the beginning of each month, Bulliner and Petitt can be found working together in the water quality lab at Galveston Bay Foundation's headquarters in Kemah, testing samples from the Bay's brackish waters for bacteria, specifically enterococcus.
The leading cause of enterococcus in the Bay is fecal matter, most commonly from dogs. Septic tank overflow and over fertilization of yards can also cause spikes in enterococcus.
Elevated enterococcus levels are especially common after dry periods followed by heavy rainfall, when a "first flush" effect occurs.
"Think about that stuff when a heavy rain falls. Where does it go?" Bulliner asked rhetorically. "It causes problems."
To detect any potentially harmful levels of enterococcus, the two men always use the same procedure. They meticulously follow a laminated checklist – despite having done the same process countless times – to make absolutely sure they do not miss a step and have consistent results.
"Just like pilots use a checklist," Petitt said.
Arguably the most time-consuming part of their duties is the paperwork. Multiple signatures are needed when a sample is received. The sample number, location where it was taken, date and time are all recorded into a log. Labels then need to be created and matched with the appropriate sample. Even the temperature of the incubator, which is always set at 41.0 degrees Celsius (105.8 degrees Fahrenheit), is checked and recorded.
The actual testing process begins only after everything has been carefully logged and labeled. 10 milliliters from a sample is sucked up through a pipette and dispensed into 90 milliliters of ionized, enterococcus-free water. The mixture is then introverted 10 times.
A packet of Enterolert, an agent that is ingested by the bacteria and has the property to glow under a blacklight, is then added to the mix and introverted another 25 times, staining the sample a yellowish color.
After letting the solution sit for a full minute, it is then carefully emptied into a sample container with 50 square wells plus one additional at the top. Bulliner and Petitt painstakingly rid the wells of any air bubbles to ensure each contains an equal amount of solution.
The sample container is then fed through a sealer before being placed in the incubator where it remains for 24 hours.
The next day, the sample containers are removed from the incubator and placed under a blacklight. If more than 33 wells illuminate, a fresh sample from the site needs to be obtained so it can be tested again. If the bacteria level in the new sample is also elevated, authorities are alerted.
Bulliner and Petitt both noted the water quality in various parts of the Bay can quickly change, but overall, they have witnessed an improvement first-hand in recent years. The Bay's water quality grade on the Galveston Bay Report Card, published annually by the Houston Advanced Research Center (HARC) and the Galveston Bay Foundation, has gone from a B in 2015 to an A in 2021.
Bulliner admitted the experience has changed him.
"You get caught up in your work-a-day world, and you just don't think what you're doing to the wildlife," he said. "It opened my eyes."
He has since stopped fertilizing his yard, installed two rain barrels to conserve water, and planted native plants around his home.
It is his way of helping to preserve what he calls the "three W's," wind, water, and wildlife.
"We can only do so much while we're on this Earth, so we want to preserve the certain areas that we do have," Bulliner said.
Galveston Bay Foundation is currently in need of additional volunteer water monitors to take samples from sites around the Bay. If you are interested in volunteering, email us at waterquality@galvbay.org.


Works began at the Withern Reserve on the corner of Limerick Dr and Upper Coomera Rd. Upon first site inspection it was obvious that this site required long term strategies as the weed species present were numerous. Garden escapees.
In the canopy, Chinese Celtis was the dominate species with some Camphor Laurel present as well. The World As I Am was tasked to eradicate them. Because this site has so many different varieties of weeds species throughout the understorey is was important not the clutter this understorey with debris from fallen Celtis trees.
The World AS I am was also tasked to remove every weed species within 10m on the adjoining property to the west. This included canopy, mid and understorey species along with vines.
Every weed present identified in this reserve was also present in the properties west, within this catchment line. Seed source will be ever present unless property owners manage their weed problem. Unfortunately, this reserve is only 50ms away from the Coomera river. All weeds present in this reserve have been feeding the river system.
Any weed species small enough to use loppers were cut and painted. The best technique, given this ecosystem was to frill mature trees, which will allow follow up work within the understorey.
These species include: Easter Cassia, Lantana, Creeping Lantana, Smooth Leafed Seanna, Mimosa Lily, Coral Berry, Dutchman's Pipe, Moth Vine, Tabaco Plant, Devils Fig, Ground Asparagus fern, Mist flower, Crofton Weed, Cobblers Pegs.


text:
Issue: Dec 1997
Biotech Anti-Aging Conference
In the first conference of its kind, scientific researchers and biotech companies meet to share findings on the genetics of aging and age-related diseases.
Biotech Anti-Aging Companies Convene
In one of the most comprehensive conferences on anti-aging research, new insights across the life sciences revealed novel approaches to understanding and intervening in disease and other conditions of human aging.
By Pamela Tames
In the first conference of its kind, scientific researchers and representatives of biotech companies met in Seattle, Wash., last summer to present new findings on the molecular mechanisms and genetics of aging and age-related diseases.
Aging is complex and, as was made clear at the conference, there's no one theory that "explains it all." However, scientists now have good models and tools. Many agree that genomic studies-the study of gene expression and function-is an important pathway to follow. Already it has opened up some promising lines of inquiry, giving many reasons for optimism. For example:
Some of the genes giving rise to longevity ("gerontogenes") have been isolated and characterized. Researchers believe they number in the "handful," not hundreds.
Drosophila (fruit fly) studies indicate no maximum limit to life span.
Evolutionary biology, which describes how natural selection affects which genes are passed on, is yielding clues to aging processes and candidate genes for drug discovery.
In model systems, long-lived mutants are more resistant to oxidative woes (from free radicals) and other stresses, confirming a causative role for oxidative stress in aging.
Looking for DNA sequences that code for aging is the "new gold rush," says George M. Martin, professor of pathology at the Alzheimer's Disease Research Center, University of Washington, Seattle. Martin described how concepts in evolutionary biology have led him to characterize the period when we show obvious physical signs of aging as a distinct "sageing phenotype."
Evolutionary biology theory says that the force of natural selection is directed at maximizing survival and reproductive capacity of an organism. It thus favors beneficial alleles (genes) and disfavors deleterious ones. However, the power of natural selection wanes with age, so early (reproductive) benefits are under powerful selection, while later detriments are under weak selection.
That means there's a "tradeoff," Martin explained. You get the good with the bad. Mutations and gene actions that were initially selected for because they make you more fit reproductively, paradoxically cause age-related effects later in life. And because the force of natural selection has weakened so much by then, "you can't escape them."
Martin says this indicates that aging evolved in a "non-adaptive way." That is, senescence is a phenotype that has escaped the force of natural selection, so there's "a lot of plasticity of life span," explains Martin.
Michael Jazwinski, professor, departments of biochemistry and molecular biology, Louisiana State University Medical Center, discussed his studies of longevity genes in yeast, studies that have led to the identification of mechanisms related to increased life span and stress-response. He believes these may be conserved across species and thus, are relevant to humans.
Jazwinski believes his research highlights the importance of genetic and both internal (hormonal) and external environmental factors in aging. While metabolic capacity and stress response are genetically determined functional potentials, they are also modulated by environmental factors through damage, stress and disease.
There is obvious relevance to humans, say Jazwinski. "We need to find ways to expand our metabolic capacity to improve behavior and function later in life. In order to do that we can manipulate certain genes, but there are limits in which we can operate." Exercise is a good example of those limits: just enough and the muscle fibers repair and get stronger; too much and they're damaged. This principle of limits, says Jazwinski, "applies to both physical and psychological stresses."
A lot of research has been done on Alzheimer's disease. But, says Gerard D. Schellenberg, associate director for research, veterans affairs, Puget Sound Health Care System, in Seattle, "A big part of the picture is still missing in terms of our understanding of the underlying mechanism. It might be oxidative damage."
Four million people in the U.S. are affected by Alzheimer's disease; their care costs about $90 billion to $100 billion a year. It's known that Alzheimer's disease is a neurodegenerative disease affecting the central nervous system exclusively. Its main features are: formation of B-amyloid plaques, neurofibrillary tangles, synaptic loss, and neuronal atrophy and neuronal death.
Attacking Alzheimer's at The Root
According to Schellenberg, finding good animal models with B-amyloid plaques has been a major achievement. He has been using mice to look for genes modifying Alzheimer's disease characteristics. So far, he's found a few important ones (PS1, PS2, and ApoE) and confirmed that B-amyloid is the critical molecule in all forms of Alzheimer's disease. "But these genes don't explain all the variations and features seen in Alzheimer's disease," says Schellenberg, "so we're missing some genetic risk factor. Whether it's related to aging or not, we don't know."
This year, Bryant Villeponteau, vice president of research, Jevone Pharmaceuticals, and three colleagues won the prestigious "Distinguished Inventors of the Year" award for their telomerase patent. Telomerase is an enzyme that maintains chromosome caps (called telomeres), believed to be the basis of cell "immortality." Now he's onto a new model-the heterochromatin loss model-which he thinks could potentially explain more about aging.
Villeponteau's starting point for biotech research at Jevone is the premise that aging has multiple causes, one being oxidative damage. Oxidative damage refers to the accumulated damage to DNA and/or proteins through free radicals, or reactive oxygen intermediates (ROIs). Based on recent data, ROIs have been linked to many degenerative diseases.
ROIs may also play a role in Villeponteau's heterochromatin loss model, the focus of Jevone's research effort. Heterochromatin loss describes structural changes in the chromosomal material. During embryonic development, so-called heterochromatin domains are laid down in clumps, effectively "silencing" the expression of certain genes. Because only certain genes are allowed to "talk," the cell becomes differentiated into specific tissues.
Where aging gets tied into this picture is during cell division: every time a cell divides, heterochromatin wanes, and there's a net loss. "The implication is that heterochromatin loss is a clock of cell senescence," says Villeponteau. And, there's evidence that ROIs may play a role in accelerated waning of heterochromatin, he says.
Attention also was spent on the life span of fruit flies, and the lessons they may impart. Using an approach known as QTL (quantitative trait loci) mapping and about 700,000 Drosophila flies, Jim Curtsinger, professor, ecology and behavior, at the University of Minnesota, has found evidence reversing a long-held idea about maximum life span. Specifically, there is none.
Traditionally, it's been assumed that life span is fixed. "Almost everyone," says Curtsinger, "will make it to 85, give or take seven years, and then almost everyone will check out." Working from Scandinavian church records, the most accurate human longevity data he could find, Curtsinger found two patterns: a systematically increasing maximum life span and a decreasing mortality rate. Using techniques of gene manipulation, he recreated these patterns in Drosophila, producing flies that lived twice as long as controls. "I'm not saying the flies are immortal," explains Curtsinger, "They wake up every morning and play Russian roulette- their probability of dying is about 20 percent. What's important is the probability of death is not increasing with increasing age." In other words, there's no "preordained maximum."
Moving on to a different sort of animal, Thomas E. Johnson, professor, institute of behavioral genetics, at University of Colorado, discussed C. elegans, a nematode (worm) species that has been widely used to study aging. A number of longevity genes have been found in this creature. Johnson's studies, for example, have produced long-lived nematode strains in which a single gene mutation results in a 65-percent increase in life expectancy and 110-percent increase in maximum life span.
The fact is, "a whole complex of genes in nematode can increase life span," says Johnson. "We're hoping to identify candidate genes in nematode first and then test these for prolongevity in humans."
A key feature of all these long-lived mutants, says Johnson, is exceptional resistance to environmental stresses such as heat, oxidative damage and ultraviolet exposure. In addition, the mutants appear healthier, as evidenced by greater motion at all ages and a higher metabolic capacity. "It appears that this class of gerontogenes," says Johnson, "regulates at a molecular level (possibly mediated by increased expression of the antioxidant enzymes, SOD and catalase) multiple stress-response pathways as well as longevity." According to this stress-response hypothesis, then, genetic and non-genetic interventions that increase an organism's ability to respond to stress may prolong life. So far, the only comparable intervention in animal models, says Johnson, is dietary restriction, which has been reliably demonstrated to increase life span. He believes the underlying mechanism here may also be increased resistance to acute environmental stress. If that's the case, it may well be "we've been overly cautious about trying to minimize our exposures to things that, while hazardous at very high levels, may well be beneficial at lower exposures," says Johnson. Oxidative stress may also play a role in Alzheimer's disease. In addition, aging and Alzheimer's disease are often a linked discussion. A feature of Alzheimer's is the presence in certain brain regions of senile plaques, entities composed of B-amyloid. Using cultured neurons in vivo, Allan Butterfield, professor of chemistry and director, Center of Membrane Sciences, at the University of Kentucky, has investigated the role of B-amyloid in causing Alzheimer's disease. He's shown that B-amyloid plaques in Alzheimer's disease are areas of elevated oxidative stress, generating free radicals that destroy neuronal membrane components.
Dangerous Free Radicals
"This new finding," says Butterfield, "helps explain how one disease can have so many things go wrong."
Using a probing technique called spintrapping, Butterfield observed that B-amyloid produces free radicals, which, in turn, cause lipid (fat) peroxidation. Lipid peroxidation damages membrane lipids, proteins and nucleic acids. A by-product of this process is a very toxic molecule known as 4-hyroxy-2-trans-nonenal, or HNE, which also destroys membrane proteins, says Butterfield. The net effect of all these changes is cell death.
In studies, adding antioxidants such as glutathione and vitamin E protected neurons from HNE damage and B-amyloid-induced lipid peroxidation. This may explain findings recently reported in The New England Journal of Medicine in which high daily doses (2,000 IU) of vitamin E kept people with Alzheimer's disease out of nursing homes by at least six to seven months, explains Butterfield.
Butterfield believes it's possible to develop more powerful antioxidants than vitamin E. He's consultant for a company, Centaur Pharmaceutical, that's trying to do just that by modifying the basic structure of the non-therapeutic molecule, N-tert-butyl-alpha- phenylnitrone (PBN). In experiments, this brain-accessible free radical scavenger has extended mean life span in mice by 50 percent.
Any discussion of aging these days involves the biomarkers, or scientific measures, of aging itself. The National Institute on Aging is now in the 9th year of a 10-year mice/rat study that is seeking to establish the measures of biomarkers. Findings from this study will next be tested in humans.
Biomarkers will allow researchers to objectively measure rates of aging in all systems, such as cardiovascular, behavioral, neuroendocrine, immunologic, and others, as well as results from interventions. An important criteria for all biomarkers is that they are convenient and minimally invasive for humans.
Aging is an immensely complicated topic, perhaps even more so than AIDS. "Aging," says Calvin B. Harley, chief scientific officer at San Francisco-based Geron Corp., "is very complex. There are a multitude of effects." One way around that is to focus on a single aspect of the process, such as the difference between normal and senescent cells.
At Geron, researchers believe the difference lies in the telomeres, those DNA sequences that cap the ends of chromosomes and may function as cellular clocks. Harley discussed how Geron is applying the telomere model to developing diagnostics and treatments for such age-related diseases as cancer and atherosclerosis.
The Telomere Theory
Every time a normal (somatic) cell divides, the telomeres shorten a bit. When the telomeres have shortened to a critical point, gene expression changes: the cell stops replicating and becomes senescent. Anything that increases cell turnover-a chronic localized stress, for example-will accelerate cell replication, driving the cell into earlier senescence.
Harley believes the altered pattern of gene expression, which marks senescence, may underlie reduced tissue regeneration and ultimately age-related disease. "We are using these genes as markers of the aging phenotype," says Harley, "and targeting anti-aging therapies at the upregulated or downregulated enzymes."
Another application of the telomere model to life extension includes finding ways to stabilize the telomeres themselves. Harley believes it may be possible to do this by increasing the activity of the telomerase enzyme, which (as shown by Jevone Pharmaceuticals) appears to lengthen telomeres. Geron is cloning telomerase components in order to understand its regulation.
The flip side of this-telomerase inhibition-also has an application. Cancer cells show high telomerase activity. "Inhibiting telomerase might kill, or 'mortalize,' tumor cells," says Harley. Based on the finding that telomerase expression can be detected in a majority of tumors and this correlates to clinical outcome, Geron just launched TeloQuant, a proprietary telomere length measurement assay.
Telomere length may also serve as a potential marker of replicative aging in human vascular (endothelial) cells. Geron has shown that endothelial cells lose telomeres as they replicate. The accumulation of senescent endothelial cells in arteries may contribute to atherosclerotic plaque formation and thrombosis (excessive blood clotting). A similar process may also explain dermal (skin) atrophy.
Geron's most recent research program involves creating new tissues from primordial stem cells, unique germ-line cells that are undifferentiated and immortal (that is, show telomerase activity). Through gene manipulation, these cells can be differentiated into any and all types of cells and tissues and then transplanted in the body. Geron is using this technology to differentiate primordial cells into cardiomyocytes (heart muscle cells) for treatment of congestive heart failure, and neurons for treatment of Parkinson's disease.
Another company, Seattle-based LifeSpan BioSciences, Inc., is applying its proprietary gene discovery technology to identify the top 1 percent of therapeutic and diagnostic targets among the 100,000 genes in the human genome. "The technology," says Glenna C. Burmer, chief scientific officer at LifeSpan, "is designed to discover disease-associated genes and proteins, determine their expression, localization and function, and then identify the best targets for drug discovery."
The traditional way of discovering drugs is to test thousands of compounds from plant extracts. That takes 10 to 15 years and costs about $100 million to $300 million. "In the past five years," says Burmer, "the genomic revolution has changed the paradigm of drug discovery." An example of the success of this approach is the discovery of the protease inhibitors used in AIDS.
But finding ideal target genes is not easy. In the public domain databases, only about 10 percent of genome (10,000 genes) are fully sequenced, and only about 5 percent of those are associated with disease. So LifeSpan had to create its own gene source database. They now have a tissue bank of 100,000 human samples, taken at every decade of life, representing 175 organs and 500 diagnostic disease categories.
According to Burmer, LifeSpan is "basically a gene discovery and target validation company," and doesn't plan to develop drugs. "There's a huge opportunity in just looking for new drug targets in the human genome and many of these will lead to new drugs for aging diseases and consequently, aging in general," she says.
Death-Defying Neurons
At Cephalon Inc., researchers use neurodegenerative diseases (such as Alzheimer's disease, Amyotrophic Lateral Sclerosis (Lou Gehrig's disease), and Parkinson's as models for aging. The premise here, explains John Farah Jr., director of scientific affairs at Cephalon, "is if we can spare neurons at risk of dying in neurodegenerative diseases, maybe we can also do the same for aging." To this end, Cephalon is developing small molecule drug candidates that target intracellular kinases, proteases and transcriptional regulators.
Researchers have created rodent models with neurodegeneration leading to impaired cognition. Using an orally administered compound (CEP-3265, a vitamin D analog that has none of vitamin D's toxic effects), they showed it could reverse neuronal atrophy and may also be protective. CEP-3265 appears to work by causing expression of growth factor genes in the central nervous system.
In other studies, they have used another small molecule, CEP-1347 (a derivative of a natural product), to selectively inhibit kinases. Kinases block stress/injury-activated signaling, leading to altered gene regulation and cell death. CEP-1347 appears to promote survival of neurons affected with a variety of lesions.
Eventually Cephalon hopes to test these small molecules in people with Alzheimer's disease. "Even if we don't cure Alzheimer's disease, we may make a significant impact on disease progression," says Farah.
Another company presenting at the conference was Aquila Biopharmaceuticals. "Our program," explains Richard T. Coughlin, senior director of microbial disease research at Coughlin, "is to fight disease in advanced years through immune modulation. We're talking about vaccines." He's directing clinical trials through the National Institutes for Health, with the goal of producing a second generation pneumococcal vaccine for use in the elderly.
With age, we become more susceptible to infectious diseases and they cause greater morbidity and mortality. Pneumococcal infection, in particular, is a major cause of death in the elderly. The problem is just when we need vaccines the most, they're least effective. Even the newer conjugate vaccines are only effective in 50 to 60 percent of the elderly and there are side effects and resistance problems.
Aquila researchers believe they have overcome these obstacles by addition of the saponin adjuvant, QS-21, to the pneumococcal vaccine. In studies, QS-21 "raised a very potent antibody response, more so than any other adjuvant," says Coughlin.


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Discover another cultural reality of Quebec while enriching your vocabulary!
Are you ready to discover the words and expressions from the theater? Hurry up, the curtain rises!
Thanks to the following exercises you will learn the vocabulary regarding leisures and going out.
Are you in shape? Yes? In the shape of a heart or a square instead? If you have any doubts, we suggest a little training in the field of geometry.
» Art & Anatomy... the parts of the body.
Each color has a different meaning. What do you think? If you want to know what color can be used in the following sentences, go ahead!


Then, just a few weeks ago, Microsoft decided it would follow suit in using deep pockets to gain an even tighter grip on the gaming industry. Microsoft announced it would be purchasing Mojang, the developer responsible for immensely popular title Minecraft, for 2.5 billion dollars. It's been an expensive summer.
It's not that exorbitantly expensive mergers and acquisitions are uncommon in this industry. Square and Enix merged in 2002 for over 764 million dollars. Electronic Arts bought Bioware/Pandemic in 2007 for 860 million dollars. Last year Activision Blizzard even bought itself out for 8.2 billion dollars. The video game industry spares no expense. However, understanding why a certain game, developer, or platform is worth a fortune is the name of the game, and what makes the recent Twitch and Minecraft purchases so intriguing.
It makes sense that Amazon bought Twitch. Amazon has had its sights on the video streaming market for some time now. Amazon Instant Video surpassed iTunes and Hulu in web traffic in the U.S. this past March. Amazon followed this success by launching its Amazon Fire TV media streamer box, which also has some gaming capabilities. Acquiring Twitch seems to be the next logical step in cornering the streaming video and gaming market. Plus, Twitch has affiliated itself with Amazon before. In addition to using Amazon Web Services such as Elastic Cloud Computing and Redshift, Twitch also sports an Amazon Fire App that has been quite popular. As more and more people cut the cord to their cable providers, streaming entertainment is really the next big thing.
The Microsoft Minecraft purchase, on the other hand, seems like yesterday's big thing. 2.5 Billion dollars is a lot to pay for a game that was released in 2011. Now to be fair, Minecraft has sold 54 million copies across multiple platforms and is a merchandising and licensing juggernaut. That doesn't count for nothing, but Microsoft has stated that it plans to break even on the deal in roughly a year. In the black or in the red, Microsoft is rolling the dice on this deal, and it may have more in common with the Twitch purchase than it seems on the surface.
Minecraft is more than a single game, or even a potential franchise of games. Like Twitch, it is a platform and a community. Gamers have been arguing for decades that gaming is an interactive and social experience and it seems that corporate America is finally getting the hint. Gaming forums, communities, channels, and networks offer an opportunity for advertising and subscription revenues that far outstrip the sticker price of any given game by itself. Maybe that is what Amazon and Microsoft are actually buying here: more direct and extensive access to their current and future target consumer base.
What does this mean for game developers? It means that games that lend themselves to online communities will be highly desirable to financers, distributors, and consumers. They don't have to have pretty graphics. They don't have to be expensive. They just have to be engrossing, entertaining, and social. The message underlying the Twitch and Minecraft acquisitions seems to be that the money is in the experience, and the experience is better when it is shared.


text:
package com.baomidou.mybatisplus.test.cache.xml;

import com.baomidou.mybatisplus.core.mapper.BaseMapper;
import org.apache.ibatis.annotations.CacheNamespaceRef;

/**
 * @author miemie
 * @since 2020-06-23
 */
@CacheNamespaceRef(XmlCacheMapper.class)
public interface XmlCacheMapper extends BaseMapper<XmlCache> {
}

redpajama_set_name: RedPajamaGithub


The Western Pennsylvanian and BearTracks are quarterly magazines produced in association with the Western Pennsylvania School for the Deaf and The Scranton School for Deaf and Hard of Hearing Children.
Each magazine provides information about student accomplishments, events on campus, and how the Schools promote the success of Deaf and Hard-of-Hearing students throughout the Commonwealth.
In this issue, enjoy articles about our students' work in the areas of academics, athletics, extra-curricular programs and community impacts. Click the covers and start enjoying the news around each campus!
Is there a friend or a family member who would benefit from the programs and services offered by our Schools and programs? Take a moment to share The Western Pennsylvanian and BearTracks with them today.
You never know the lives that you may change!


Clarence E. "Spud" Nachand, Jr., 70, of Dunmore, died peacefully surrounded by his family, Thursday afternoon at Regional Hospital, Scranton.
Born in Scranton, he was the son of the late Clarence E., Sr. and Vivian Burschell Nachand and was a graduate of Dunmore High School and the University of Scranton where he received a bachelor's degree in Science. Prior to retirement he worked as a Computer Programmer.
Affectionately known as "Spud", he was a kind man who enjoyed telling stories and was adored by all those whose lives he touched. He enjoyed riding his Harley and taking his grandchildren for rides. He was passionate about his country and was proud to be an American. Every year he rode his Harley to the Annual Rolling Thunder Rally in Washington, D.C. with his best friends. He was an "old school" Dunmore boy who lived his life to the fullest and with no regrets. He will be sadly missed by all who knew and loved him.
Surviving are daughters; Colleen Pettinato and husband Doug, Dunmore; Brigitte Baum and husband James, Scranton. A son; Brett Nachand and fiancée Kayla Jordan, Dunmore. Grandchildren; Joseph, James, Anthony, Lily, Hailey, Hannah and Brett, Jr. and cousins.
He was preceded in death by his former wife, Maureen Hinton Propst and an infant sister, Jeanell Nachand.
Services will be held on Tuesday at 6:00pm at the Carlucci-Golden-DeSantis Funeral Home, Inc., 318 E. Drinker St. Dunmore.
Friends may call from 3pm until the time of service.
In lieu of flowers, donations may be sent to The Gift of Hope Scholarship, PO Box 36 Lily Lake Rd. Waverly, Pa 18471.
Sincere sympathy for Spud's family whom he loved dearly. Spud's warmth, kindness and sweet spirit will always be remembered. He was always a gentleman. I only knew him in the last 20 years and enjoyed hearing his innumerable stories of his personal adventures, and I believe every one! He was also proud of his heritage and readily shared his remembrances. May God rest his soul.


text:
Q: Python. Overwriting new numbers into a column in a matrix I have data set which has 44000 rows and 5 columns. What I want to do is to erase all the numbers in the second column and to put new numbers there in an incremental order but every other column remains the same as before. For example, first row for that column is 0.001, second row 0.003 and the third row is 0.005 and so on.
How can I implement in Python code? I am still not quite skillful in Python.
I appreciate your helps in advance.

A: I will explain with an example, for now let me take this list. Your matrix can be thought of as a 2-d list.
let my list be L= [[0,0,0],[0,0,0],[0,0,0]]. This is 3x3 matrix. Assuming that The second column of every row needs to be uniformly incremented by 2, you can try this.
l = [[0,0,0],[0,0,0],[0,0,0]]
inc=0

for i in l:
    i[1]=inc+2
    init=i[1]

print(l)

For every row in the matrix, modify the second column. Use a variable (incr) to track the increments. At the first iteration, the second column of row 1 will be '2'. Once the value is modified, then update the increment variable to the new value, so that it will add your incremental value to this new value in the next iteration. This is what happens.
Iteration 1:
row1 before modifying is [0,0,0]. Then replace the second column with incr+2 (incr is 0 at first, so i[1] becomes 2). Then incr = i[1] will make incr = 2. So now row1 after modifying is [0,2,0].
Iteration 2:
row2 before modifying is [0,0,0] . Then replace the second column with incr+2 (incr value changed to 2 in the previous iteration. So now i[1] is incr+2 which is 2+2=4). Then incr = i[1] will make incr=4. So now, row2 after modifying is [0,4,0].
This keeps repeating.


Today is International Women's Day. The first International Women's Day was in 1911; the idea was proposed in 1910 by Clara Zetkin, leader of the 'Women's Office' for Germany's Social Democratic Party.
The day has become an occasion for women to celebrate achievements and progress in the fight for women's liberation. It is also an opportunity for renewed campaigning.
Women's liberation groups fight for the rights of women across the world. They are also part of a broader movement that tries to help the victimised and the oppressed.
In this struggle, communication is vital. One predicament of the oppressed is enforced silence. Making it possible for someone to speak and to speak out can be the beginning of liberation.
Translation and interpretation agencies will play a central role in political or personal struggles. As developing countries seek to join the major economic and diplomatic powers, more and more people will look to translators and interpreters for help in the potentially difficult and dangerous situations that they might find themselves in.
This might be at a governmental level, with consultations and negotiations taking place on women's liberation or poverty in the developing world. However, it might be at a more local level too. Councils in Britain, for example, are having to review their language provisions, to ensure that they are providing a fair and adequate service to immigrants who have fled oppressive or tyrannical regimes to live in Britain.
These people, and others all around the world, need to be able to express themselves – to tell us their needs and desires. We need to be able to understand them. Professional translators and interpreters will be the bridge-builders of the twenty-first century.


text:
Nozha Boujemaa
Research Director at Inria, Director of DATAIA Institute (Data Sciences, Intelligence & Society)
Research Director at Inria, Director of DATAIA Institute (Data Sciences, Intelligence & Society), Project leader of TransAlgo scientific platform for algorithmic systems transparency and accountability. Knight of the National Order of Merit, Founding Director of Digital Society Institute (ISN), Senior Scientific Advisor for "The AI Initiative", International Advisor for Japanese Science and Technology Agency Program "Advanced Core Technologies for Big Data Integration" , Member of the board of Data Transparency Lab, Elected Member of the Board of Directors of Big Data Value Association, General-chair of European Big Data Value Forum 2017 (Versailles), Member of the Scientific Councils of INRA, CentraleSupélec and Member of the Strategic Orientation Council of Institut Français.
Previously, Advisor to the Chairman and the CEO of Inria in Data Science with concern to the socioeconomic impact of Big Data and AI capabilities, Scientific Head of IMEDIA research group for over 10 years (till 2010) and the Director of Inria Saclay Research Center for 5 years (2010-2015).
Find more information here
2017 © Powered by BDVe
The European Big Data Value Forum 2017 has received financial support from the European Union's Horizon 2020 Framework Programme for Research and Innovation.
Pictures used in this website: "Versailles Gardens" by Jacques Postel, courtesy of Versailles Tourisme, ''Auditorium Richelieu at the Palais des Congrès de Versailles'' courtesy of VIPARIS, ''Entrance to Château de Versailles'' by Amélie Peingnez, courtesy of Versailles Tourisme, ''Facade of Palais des Congrès de Versailles'' by JC Guilloux, courtesy of VIPARIS, ''Aerial view of the Domain of Versailles, France'' by Lionel Allorge is licensed under CC Sa 3.0 and ''Aerial view of the bassin d'Apollon and the Grand Canal in the gardens of Palace of Versailles'' by ToucanWings is licensed under CC Sa 3.0
IBM and the IBM logo are trademarks of International Business Machines Corp., registered in many jurisdictions worldwide.


text:
Carnivore, Cryptids, Canine,
Carcasses
North America (Minus Mexico and Caribbean)
Misidentified
The Turner Beast (Mutant Canine)
The Turner Beast is a creature that has been described as a husky-looking wolf with bulky shoulders, big eyes, a flat snout, short mangled ears, and a bushy tail. Before it was proven to be a hybrid, some researchers claimed that it could possibly be a Dire Wolf. It was spotted in Turner, Maine and was estimated to weigh about 120 lbs. It has been known to kill pets and livestock, mostly dogs, most likely for territorial reasons. Later DNA testing of the animal in the pictures revealed it to be a wolf-dog hybrid.
The History Channel's MonsterQuest featured the Turner Beast in the Series 1 episode "Mutant Canines."
Close up of the head
MonsterQuest Season 1 Episode 6 - Mutant Canines-0
If you visit the International Cryptozoology Museum at 11 Avon Street in Portland, Maine, you can see the foot of the Maine Mutant preserved in a jar. More information on the museum at [1]
Retrieved from "https://cryptidz.fandom.com/wiki/The_Turner_Beast_(Mutant_Canine)?oldid=100581"


These little brownies take a whole of about 5 minutes to whip up, and they are SO GOOD!
Not too sweet, with crisp crinkly edges and a rich fudgey interior.
And the best part of all is that they're really not all that bad for you. They're made of whole eggs, nutella and ground almonds. That's it!
They're flourless and gluten-free as well, though you would never know it.
Preheat oven to 350 degrees F. Grease and line the bottom of an 8-inch square pan with parchment paper.
Meanwhile, measure out 1 cup of nutella in a glass measuring cup and microwave on high power for 1 minute.
With the mixer running on medium speed, slowly drizzle in the warm nutella until just combined. Then mix in the ground almonds.
Poor the batter into the prepared pan and bake at 350 degrees for 20-22 minutes (the centre should be set).
Allow the brownies to cool completely in their pan before slicing into squares.
Enjoy! These won't last long at all!
* These can actually be made as Two-ingredient brownies, with just the eggs and nutella. However, I find that the texture is a million times better with the ground almonds. I haven't tried it, but I imagine that ground hazelnuts would be especially good as a substitute for the almonds.
I'm freaking out over these. Definitely trying them tomorrow! The only thing I don't have is Nutella and it's way too hot this evening to even think about going to the shops. Can't wait to make them and eat every single one.
Hey Lauren, I have to say, I am beyond jealous of your heat wave!
Thanks for providing such nice information to us. It provides such amazing information on care/as well Health/. The post is really helpful and very much thanks to you. The information can be really helpful on health, care as well as on Exam/tips.The post is really helpful.
Thank you for updating the information.


Sell Tickets Arizona is one of pro football's oldest franchises dating back to their days as the Chicago Cardinals, who played their first season in 1920. They're two-time NFL Champions, but have yet to hoist their first Lombardi Trophy. They play in one of the best markets in football and have a very passionate fan base, so if you're looking to catch them in person this season, be sure to find the cheapest Arizona Cardinals tickets on TickPick, where you'll save big by NEVER paying any service fees! No hidden service fees. Guaranteed lowest prices on the web.
We know that scoring cheap Arizona Cardinals tickets at University of Phoenix Stadium isn't always easy. So TickPick partners with a vast network of reputable ticket sellers to give customers tons of tickets to choose from for every single home game. Whether you are looking for the cheap seats in the upper deck or premium seats at the 50 yard line, we've got you covered with our no fee Cardinals tickets that are always guaranteed to be legitimate. There is nothing like catching a Cardinals game in Arizona, so what are you waiting for?
Whether you are searching for the cheapest Cardinals tickets or the best, TickPick is the ONLY site that has no service fees. Compare our AZ Cardinals tickets with StubHub or the Ticket Exchange and you'll see we are saving you at least 10% per sale. For the cheap seats that may mean savings of $10; for the more expensive seats that could be savings of over $100.
There are a few important dates to keep in mind if you are tracking when Cardinals tickets go on sale. The NFL releases the match ups for each upcoming season in late December, but not the specific game dates. The full Arizona Cardinals schedule is released in mid April every year. Once the schedule is released tickets for each game become available for purchase on TickPick, giving fans the fastest access to cheap Cardinals tickets. Tickets will remain on sale right up until game-time as well!
There are plenty of Cardinals fans that don't live near Arizona to catch a game in person. However that doesn't mean you can't see the Cardinals in action this season. TickPick has tickets to all the Arizona Cardinals road games too. Whether you are planning a trip to a new city months in advance or buying Cardinals tickets last minute, we guarantee the delivery and authenticity of every ticket. If you aren't familiar with the seating at one of the Cardinals opponents stadiums, feel free to give our customer support team a call at??845-538-4567.
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We know that scoring cheap Arizona Cardinals tickets at State Farm Stadium isn't always easy. So TickPick partners with a vast network of reputable ticket sellers to give customers tons of tickets to choose from for every single home game. Whether you are looking for the cheap seats in the upper deck or premium seats at the 50 yard line, we've got you covered with our no fee Cardinals tickets that are always guaranteed to be legitimate. There is nothing like catching a Cardinals game in Arizona, so what are you waiting for?
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Unlike any other ticket site, TickPick is the only place where you can sort Arizona Cardinals tickets based on the seat quality. If you are looking for the best seats within a few sections, this feature is tremendously helpful. Use the interactive seating chart to select the sections that you are interested in and then sort by Seat Quality. The relevant listings will be ordered by what are considered the best Arizona Cardinals tickets. And don't ever worry about overpaying for seats that aren't worth it. The Score Report is still available to help you decide which tickets you should buy.
There are a bunch of ways you can get cheap Arizona Cardinals tickets. First, pay no fees. Other ticket sellers and ticket marketplaces charge anywhere from 10% to 25% in hidden fees. On TickPick the price you see is the price you pay, so if you a're comparing prices, always make sure to compare the final price on the checkout page. Second, if ticket prices are still not to your satisfaction, try bidding on Arizona Cardinals tickets. Go to the tickets page for the event you want to go to and click "Bid" in the upper left hand corner. From there, you can tell sellers what you want to pay for tickets and let them scramble to meet your price rather than the other way around.
Looking for a discount on our already no fee Arizona Cardinals tickets? New users can sign up here for a $10 discount on Arizona Cardinals tickets.
All Arizona Cardinals Tickets purchased through TickPick are 100% guaranteed through our Buyer Trust Guarantee. We guarantee that your tickets are authentic and that you will receive your tickets on time. If you ever have any issues with tickets purchased through our site, we will work to replace them with tickets of equal or greater value. Furthermore, if the event is cancelled and not rescheduled, we will refund your purchase. Don't believe us? See what our customers have to say.


text:
WWF, Gov't Envisage Creation Of Ebo National Park
By Walter Wilson Nana
The World Wide Fund for Nature, WWF Cameroon Country Programme has indicated that the proposed Ebo National Park will soon become a reality. This is contained in a released issued by the WWF Cameroon Country Programme Office in Yaounde.
Game guards protecting cameronian forests
The release notes that, after two years of on-the-ground studies, research and wide consultation with local communities as well as administrative authorities, WWF through its Coastal Forests Programme, the Ministry of Forestry and Wildlife and the Dutch Government are taking one more important step towards having the biodiversity rich Ebo Forest designated as one of the country's key National Parks.
To fortify their commitment, a low altitude fly-over to fully appreciate the potentials of the Ebo forest area in Cameroon has been planned for February 2, 2010. WWF technicians, who have over the years coordinated efforts to prepare Ebo for a National Park designation, shall accompany government officials in charge of environment and conservation issues as well as a representative of the Dutch Government in Cameroon to catch a life-time glimpse of the biodiversity rich forests.
"Local community leaders and the media will be key components of the fly-over," the release states. According to Dr Atanga Ekobo, Coordinator of WWF Coastal Forests Programme; "the aim of this mission is for the key stakeholders to have a comprehensive view of the proposed park, as well as further raise awareness about the conservation importance of this Congo basin biodiversity hot spot."
Apart from forest elephants, a small but important gorilla population, and a healthy chimpanzee community, there are nine other diurnal primate species present in the proposed Ebo National Park, including important populations of species in serious decline elsewhere, such as drills and Preuss' red colobus.
From this background, WWF Cameroon Country Office posits that since the forest is facing serious threats such as poaching and illegal logging, there is need for it to be safeguarded. Ground work to enable the designation of the Ebo forest as a protected area has been coordinated by WWF Cameroon Country Programme Office, through WWF Coastal Forests (SAWA) Programme, Limbe, for the past two years with funding from WWF Netherlands.
The Ebo landscape cuts across two administrative divisions; Sanaga Maritime and the Nkam divisions in the Littoral Region. The proposed Ebo National Park is one of the most important remaining tracts of closed-canopy forest in the Littoral region. It covers a surface area 111, 2880 hectares of lowland and montane forests and contains one of the most complete populations of a wide variety of forest mammals in Cameroon.
Gov'ts Given Deadline To Submit Greenhouse Gases Reduction Plan
Envisaged Ebo Park Will Bring Development – Mayor
Niger Hosts Conference On Restoration Of Deforested Land
Over 50,000 People May Be Rendered Homeless In Bakassi
GS Nsoung Wins WWF's ESD Award


text:
Q: Class construction based on interface implementation Imagine that we have classes as such:
public abstract class WebPage
{
    public WebPage()
    { ... }
}

public class LoginOrSignUpWebPage : WebPage, ILogin, ISignUp
{
    private Info _loginInfo;
    private Info _signUpInfo;
    public readonly Info LoginInfo { get { return _meats; } }
    public readonly Info SignUpInfo { get { return _legs; } }

    public class LoginOrSignUpWebPage(Info loginInfo, Info signUpInfo) : base()
    { ... }
}

We can see that WebPages would want to have different ways of being instantiated based on different interfaces they implement.
Whilst it'd feel okay implementing individual constructions for each class, I would prefer to use inheritance to base the object construction upon. The reason for this is because another object may implement the same interfaces and would have the same way of being instantiated.
I have thought about using some sort of (abstract?) Factory method, but I'm not sure how this would work.
Question:
Right to the point, what do you think would be the best way to base construction of an object based upon what interfaces it inherits? This would include (potentially) different parameters, and at minimum different data passed depending on the implemented interface.

A:
We can see that WebPages would want to have different ways of being instantiated based on different interfaces they implement.

No, they wouldn't. Interfaces define how types should look like to the outside. They provide no implementation detail and also no information about the constructor or the construction process. If you have an object of the type of an interface, all you know is that you can access the properties and methods that were defined in the interface.
The way you declare your WebPage type, it is fixed to implement both ILogin and ISignUp. As such it is absolutely required to implement whatever these two interfaces specify. And every object of type WebPage will always provide what both interfaces require.
It is not necessary to make the construction of an object based on the interfaces it is implementing, simply because the interfaces you are implementing is known at compile time and can't be changed later. So for the type implementing an interface, you just specify directly how that is constructed.


text:
<mods:mods xmlns:mods="http://www.loc.gov/mods/v3">
    <mods:titleInfo>
        <mods:title>Kiøbenhavns Kongelig alene priviligerede Adresse-Contoirs Efterretninger</mods:title>
    </mods:titleInfo>
    <mods:titleInfo type="uniform" authority="Statens Avissamling">
        <mods:title>adresseavisen1759</mods:title>
    </mods:titleInfo>
    <mods:originInfo>
        <mods:place>
            <mods:placeTerm type="text">København</mods:placeTerm>
        </mods:place>
        <!--required-->
        <mods:dateIssued encoding="iso8601">1795-06-15</mods:dateIssued>
    </mods:originInfo>
    <mods:note type="noteAboutReproduction">present</mods:note>
    <mods:relatedItem type="host">
        <mods:part>
            <mods:detail type="volume">
                <mods:number>27</mods:number>
            </mods:detail>
            <mods:detail type="edition">
                <mods:number>2</mods:number>
                <mods:caption>Anden udgave</mods:caption>
            </mods:detail>
            <mods:detail type="issue">
                <mods:number>313</mods:number>
            </mods:detail>
        </mods:part>
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text:
Talk:Ireland: 2000-2004
Wouldn't this and the other Ireland: xxxx-xxxx articles be better at one of these names?
Series of articles History of Ireland (xxxx-xxxx)
History of Ireland
Timeline of Irish history
-- Chuq 23:40, 23 Sep 2004 (UTC)
Why not break them up into years as is done with the Canadian history timeline, then extra information like births and deaths can be added. - SimonP 16:54, Oct 16, 2004 (UTC)
Retrieved from "https://en.wikipedia.org/w/index.php?title=Talk:Ireland:_2000-2004&oldid=16793145"
This page was last edited on 16 October 2004, at 16:54 (UTC).


text:
Remarks by President Biden Before Meeting with House Democratic Leaders Working on the American Rescue Plan
Speeches and Remarks
THE PRESIDENT: We're going to be talking about the recovery package, but we saw the jobs report that only 6,000 private sector jobs have been created. And at that rate, it's going to take 10 years before we get to full employment. That's not — that's not hyperbole; that's a fact. We're going to be in a situation where it'll take a long, long time.
And I appreciate you all coming over because — and the urgency with which you're moving — this is about people's lives. This is not just about numbers; this is about people's lives. People are — I don't have to tell any of you, they're really hurting. People are being evicted.
Just look at all the number of people who are needing and seeking mental health services now. Suicide is up. People are very — really, really — drug abuse, violence against women. People are really feeling the hole. They don't know how to get out. You've given them a lot of hope — a lot of hope with the — as they say in southern Delaware: With the grace of God and the goodwill of the neighbor, with the crick not rising, we can really begin to do something consequentially.
So I want to thank you all. And what I — you know, President — President Obama put me in charge of the Recovery Act, and it was hard as hell to get the votes for it to begin with, and then it was hard as hell to get even the number we got. But one thing we learned is, you know, we can't do too much here; we can do too little. We can do too little and sputter.
But again, the end result is, it's not just the macroeconomic impact on the economy and our ability to compete internationally; it's people's lives. Real, live people are hurting, and we can fix it. And we can fix it. And the irony of all ironies is, when we help them, we are also helping our competitive capacity through the remainder of this decade. I mean, it's real.
So we got a chance to do something big here. And I'm — and thank you for last night, yesterday, and what you're going to be doing on this Recovery Act. And I can hardly wait to sit down with Peter DeFazio to work on infrastructure. (Laughter.) This is the next big piece.
So, thank you all for being here, and thank you for coming in. I appreciate it.
Next Post: Memorandum on Advancing the Human Rights of Lesbian, Gay, Bisexual, Transgender, Queer, and Intersex Persons Around the World Memorandum on Advancing the Human Rights of Lesbian, Gay, Bisexual, Transgender, Queer, and Intersex Persons Around the World
February 04, 2021 • Presidential Actions
https://www.whitehouse.gov/briefing-room/speeches-remarks/2021/02/05/remarks-by-president-biden-before-meeting-with-house-democratic-leaders-working-on-the-american-rescue-plan/?utm_source=link


text:
Alacamescit is a village in the Çilimli District of Düzce Province in Turkey. Its population is 598 (2022).

References

Villages in Çilimli District

redpajama_set_name: RedPajamaWikipedia


Our Stonegate Village gives you luxury living at an affordable price with dishwasher, central air, swimming pool, and private balconies. Don`t forget to ask about our remodeled units! You can`t find an apartment this size for the money anywhere in Champaign!
Enter your desired move-in date below to see what we have available!
A comfortable, well-kept, and enjoyable apartment. submitted by Meghan D. - Feb 04, 2017 |Recommended: YesI would highly recommend Royse Brinkmeyer apartments. I've never had a problem with noise or parking, the apartment itself if comfortable and exactly what I was looking for, and the staff are always friendly and eager to answer any questions I may have. I am very happy with my decision to join the Royse Brinkmeyer community.
Good with okay infrastructuresubmitted by Alex B. - Jan 04, 2017 |Recommended: YesStaff and such are great. The grounds outside and steps could use a little work. The apartment itself it fine minus a few insulation issues around sliding doors.
response from property - Jan 05, 2017 Thanks for your feedback, Alex. We agree with your assessment of our "grounds" in general and are looking forward to changing our approach with our building's appeal in 2017. Glad you hear you are enjoying your interactions with our people!
Love this placesubmitted by Michael F. - Nov 01, 2016 |Recommended: YesLove the 3rd floor high ceiling and the balcony. Its very spacious. Safe place to live.
response from property - Jan 05, 2017 Thanks Michael!
Great to staysubmitted by Gerino R. - Aug 31, 2016 |Recommended: YesClean apartments, peaceful environment and Great open parking.
response from property - Jan 05, 2017 Thanks for your feedback Gerino!
Decent for the pricesubmitted by Brian D. - Aug 24, 2016 |Recommended: YesThe apartment is in pretty decent shape. It's very spacious. Any problems I had with the apartments were quickly taken care of by maintenance. There were several outlets that don't seem to work, however.
Great property to live in for the costsubmitted by Ian C. - Jun 21, 2016 |Recommended: YesI like the overall roominess of the apartment, but I do not like the sounds I hear from my neighbors and the washers and dryers. Walls are very thin. Service and people are awesome.
response from property - Jun 22, 2016 Thank you, Ian, for your review! It's always great to hear that someone is satisfied with our service and staff--we strive to be the very best. While we recognize that multi-family housing does present some noise, we appreciate that you acknowledge this, yet would still recommend us to your friends! Thank you for your residency with us!
response from property - May 25, 2016 Thank you, Christian! We work hard to create a community that people will love being a part of! We're so glad you enjoy it!
Good environment, affordablesubmitted by Harsheel S. - Feb 28, 2016 |Recommended: YesOverall, a good place to live with affordable prices. It would be great if the the parking spots had unit numbers associated. Also, the roads within the complex needs a lot of attention especially the cracks and leveling of the ground.
response from property - Nov 19, 2015 Thank you, Samuel! We love our residents, and love hearing when they are happy! We appreciate your feedback!
Amazing place to live on submitted by Thoufeeque S. - Nov 05, 2015 |Recommended: YesI have been to different apartment building to live, but this place is amazing and peaceful.
response from property - Nov 06, 2015 Thank you, Thoufeeque! We're so glad you are happy!!
response from property - Jul 29, 2015 Thank you for the feedback, Aishwarya! We always love the suggestions of our residents, and do have the intentions of having some activities for our residents! Thank you for choosing Royse & Brinkmeyer!
Clean, Well maintainedsubmitted by Heather L. - Jun 29, 2015 |Recommended: YesOverall great. Smells from other neighbors smoking drift through the vent in the living room on occasion.
response from property - Jul 01, 2015 Thank you, Heather, for your review! We work hard to keep our properties beautiful and comfortable for the residents who live there. We're glad you enjoy Stonegate Village!
Everyone I have spoke with at the office has been pleasant and attentive to concerns.submitted by Dawna B. - Jun 23, 2015 |Recommended: YesPretty quiet, clean, and I feel safe even with the neighborhood across Springfield Ave. I do feel that the windows and patio doors should be replaced. I had ice inside my door last winter and there's heat coming in the door this summer. Overall I enjoy living here and being on the outskirts of town.
response from property - Jun 24, 2015 Thank you for the feedback, Dawna. We always appreciate our resident input, as well as any suggestions for improvements. Thank you for taking the time to let us know your thoughts!
Pet cats and/or birds permitted with appropriate deposit and addendum.


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How Much Does Gerd Surgery Cost
The LINX™ anti-reflux procedure is performed by minimally invasive laparoscopic (keyhole) surgery and takes less than an hour. The cost of Acid Reflux Surgery. It is important to note, however, that while these medications may reduce the symptoms associated with acid reflux, they do not treat the underlying cause.
"What we've discovered is health care really doesn't cost that much. 8,000 at Surgery Center to $37,174 at Integris.
A Medibank report suggests it pays to shop around when looking for a surgeon to perform a range of common procedures in private hospitals, including weight loss surgery. also faced average out of pocket costs ranging from $229 to $793.
This page describes about Gallstone surgery in India, cost gallstone surgery India, low cost gallstone surgery India, benefits gallstone surgery India, causes.
While residents struggled through the uncertainty of whether their homes would be replaced or repaired, suffered disappointment of learning Federal Emergency.
Apr 17, 2015. Nissen Fundoplication is usually done through laparoscopic surgery under general anaesthetic and takes about 1.5 hours – the abdomen is accessed by four incisions and inflated by gas. If clips or non-dissolvable stitches are used, they will be removed after about seven to 10 days. How much does it cost.
By chance a golf ball size tumor was found on my left kidney. Because I have had a history of Renal Failure a partial was done. (The kidney never recovered and does.
Information about non alcoholic fatty liver disease (NASH, NAFLD) caused by obesity and diabetes. Other contributing factors are poor diet, diseases, medications, and.
The course of treatment required in a case of canine liver disease will depend upon the cause of the condition. For example, if trauma was the trigger.
Nissen fundoplication is a surgical procedure to treat acid reflux ( gastroesophageal reflux disease). Acid reflux is a. What does the operation involve? The operation is. Nissen fundoplication (laparoscopic) costs are covered by most medical insurance policies, but please check with your insurer first. If you are paying for.
But I also left inspired that a community can come together and show so much compassion. Many of your officers spent. sometimes at most extraordinary costs, but in every way, putting our best foot forward; doing what's right. Thank you.
Get the facts about the symptoms and remedial treatment of hiatal hernia and GERD.
A typical botox treatment can cost $600 to. While plastic surgery is still dominated by female patients, men are.
GI Surgery. Gastro Esophageal Reflux Disease (GERD) The esophagus carries food from the mouth to the stomach. The lower esophageal sphincter is a ring of muscle at.
One out of three Americans suffer from heartburn symptoms. Many of those sufferers have gastroesophageal reflux disease (GERD) and they don't even know it. Until recently the only cure was a surgical procedure known as "Nissen fundoplication," which has the potential to cause side effects that lead many to delay or.
Oct 10, 2013. Over 15 million Americans suffer from gastroesophageal reflux disease, or GERD. While many patients manage their condition by watching what they eat and popping over-the-counter medications, a new technology can help relieve the burn for good.
Can Stomach Acid Burn Through Your Stomach Heartburn (Acid Reflux) Symptoms, Relief Medicine, – Heartburn is a sensation of burning in the chest caused by stomach acid backing up into the esophagus (food pipe). The burning is usually in the central part of the. Eric's ulcer presented itself as a gnawing or burning sensation in his upper abdomen that seemed worse when
Sep 15, 2012. I was a chronic sufferer on 40mg omeprazole before 10mg was non-presecription , andd god knows how many Rennie etc. Here's the fix:- When suffering, a big teaspoon of Manuka Honey. Use prebiotocs and probiotics to restore intestinal flora correctly. Alcohol and sugars aren't the problem – it's the waste.
Average cost of private Anti-reflux surgery (Laparoscopic) in the UK provided by Private Healthcare UK: The gateway to private healthcare in the UK.
May 28, 2013. Until recently the only cure was a surgical procedure known as "Nissen fundoplication," which has the potential to cause side effects that lead many to delay or avoid surgery. Last year, the Food and Drug Administration approved a new treatment device called the LINX Reflux Management System to offer a.
Anyone know what the current costs for this look like? I'm based in the North West and have frankly had enough of living with reflux to the point that I'm willing to raid the piggy bank. The newspaper articles seem to quote around £5k – is this still accurate? Also does anyone still do transoral incisionless – is.
Questions and Answers About the CA-125 Test: Author: Taylor Tang What is CA-125? What is a CA-125 test? What is the difference between 1 st and 2 nd generation CA-125.
FAQ: questions that are often asked about the TIF procedure, including safety, cost, eligibility, what to expect during and afterward, and more.
So how much does stem cell therapy cost? For purposes of this article, we decided to do a side-by-side comparison of a surgery and stem cell therapy so that you could evaluate for yourself if stem cell therapy is an affordable and.
The main problem continues to be the late arrival of patients well past the golden hour when much of the heart muscles can be saved," says Sunitha Viswanathan, professor and head of cardiology, MCH. MCH does not. But when it costs.
Laparoscopic Anti-Reflux (GERD) Surgery abroad: Clinics, Prices. – Results 1 – 15 of 99. MEDIGO lists clinics around the world offering Laparoscopic Anti-Reflux (GERD) Surgery procedures from $ 1204. Save up to 80% on Gastroenterology treatment abroad.
The lifetime prevalence of gallbladder polyps ranges from 1% to 4%. Overall, polyps are much less common than gallstones, and they are usually discovered as an.
Heartburn/GERD Overview. Heartburn or acid reflux symptoms include chronic cough and chest pain and burning. Knowing your triggers, such as certain foods, medications.
Not only does this. but it's much less extreme." And your skin can also stretch out again if you regain the weight, says Seth Thaller, MD, chief of plastic surgery at the University of Miami Miller School of Medicine. The number of people.
If lifestyle changes and medication don't help manage your GERD symptoms, surgery may be your best option. Learn about surgery options such as fundoplication and.
Jun 13, 2017. Through the endoscope and using special tools, he can also take tissue samples and do surgery. With a set of small tools at the tip of an endoscope, he can tightly bind the end of the esophagus to the top of the stomach. Or he could place stitches in the lower esophagus to form pleats that strengthen the.
That does not count costs of drugs or hospital visits. Daswell's plight illustrates a much. after the surgery. The Sun calculated the cost of drugs for his obesity-related sicknesses: arthritis, depression, diabetes, gastroesophageal.
Deidara (デイダラ, Deidara) was an S-rank missing-nin from Iwagakure. During his time in the.
My name is Kevin, I am 21 years old and have barretts esophagus due to bad GERD. I had a lap nissen fundoplication in order to halt any further damage to the esophagus, and the procedure was performed on December 23rd, 2011. My surgeon is very reputable from what people have told me, and he.
But that is not a sensible reason to keep a player who no longer appears to serve.
The term "ACVS Diplomate" refers to a veterinarian who has been board certified in veterinary surgery. Only veterinarians who have successfully completed the.
The most common surgery for GERD is a nissen fundoplication , in which the upper part of the stomach is wrapped around the esophagus and stitched into place. According to the Comprehensive Center for Laparoscopic Surgery, average hospital charges for fundoplication range from $10,000-$25,000 or more with a.
Stretta is a non-surgical treatment for. GERD symptoms despite medications and want to avoid surgery, Stretta is an ideal option between medications and surgery. Our team is excited about this innovative technology." Dr. Grecco.
Help Us Fight Canine Liver Disease Do you have a special business that relates to dogs and could help our readers? Click HERE to contact us about listing on the site.
Fundoplication surgery is most often used to treat GERD symptoms that are likely to be caused in part by a hiatal hernia and that have not been well controlled by medicines. The surgery may also be used for some people who do not have a hiatal hernia. Surgery also may be an option when: Treatment with medicines does.
Gallstones and gallbladder disease Highlights Diagnosis. Common symptoms of gallbladder disease include abdominal pain, nausea and vomiting, fever, and yellowing of.
Disorders of the gallbladder including gall stones and bile duct disease in dogs.
Can liver problems cause leg pain? While the two distinct body parts don't sound too awfully much alike, the answer is "Yes." When the.
Natural Treatments For Acid Reflux & GERD | Natural. – Check out the natural remedies for acid reflux, heartburn or GERD! Get natural treatments for acid reflux, GERD or heartburn & to bring symptoms under control.
May 16, 2015. Bache-Wiig said a Nissen costs between $3,000 and $5,000 at Abbott. A Linx procedure costs roughly $1,500 more, and "almost all of that differential is the cost of the device," he said. Dunn noted that Nissens require an overnight hospital stay, while the majority of Linx procedures are done as day surgery,
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MARE will be present in Tissue World, again this year, to meet its customers and update them about its latest developments in the field of yankee coating, dry and wet strength control and management of tissuemaking process chemistry.
MARE's experts will be at booth E-300 to introduce the latest success in obtaining soft and bulky paper on new state-of-the-art tissuemaking installations by innovative yankee coating programs, as well as increased performance and operational cost optimisation through control of dry and wet strength properties of tissue paper.
Mare is one of the largest privately owned manufacturer and supplier of functional chemicals to the Paper, Detergent, Concrete and Ceramics industries.
Mare was founded about 40 years ago and is now manufacturing each year a very large volume of functional chemicals, the biggest part heading for paper production. We have largely invested in our capability to supply chemicals and solutions to the tissuemaking industry, becoming one of the few companies able to stand as a global supplier for tissue producers. Come and meet us to talk about your needs and goals in this edition of Tissue World 2019.
Visit Mare at booth E-300 to find out more.
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The resource consists of a slideshare presentation with an explanation about the reported speech.
Resource consisting of a video which contains a grammar lesson about reported speech.
We have to type in the missing words to form correct sentences with the reported speech form.
In this exercise we have to complete each reported sentence by typing the verbs into the gap.
The resource consists of an explanation about the reported speech. We learn how to use it.
Activity designed to help us practice grammar. Choose the best word or phrase to complete the gap in the sentences.
The resource consists of an slideshow with an explanation about reported speech.
Set of exercises designed to help us practice Reported Speech. There is an online activity (self-evaluation) and several printable (PDF) exercises.


A Tamil journalist arrested on his return from exile to Sri Lanka is shocked by attempts to undermine his professional integrity by widely known Sinhalese journalists.
Punniyamoorthi Sasikaran who positively responded to an open invitation of the government by returning to the island was arrested by Sri Lankan authorities on 20 January at the country's main airport and later released on bail. He has been banned from travelling abroad for five years while being ordered to report to the notorious Fourth Floor of the Criminal Investigation Department (CID) in Colombo every month, located over 300 km away from his eastern home town.
Following his arrest former Free Media Movement (FMM) Convenor Sunanda Deshapriya has publicly called to investigate Sasikaran on human smugglers, the same charge levelled by Sri Lanka authorities.
Sasikaran 'has not been known as an activist or journalist in Sri Lanka. H (sic) has gone to Australia through human smuggler. Human smuggling rings needs (sic) to be investigated,' Deshapriya has written in his blog.
"No information has been provided re Puniyamoorthy Sasikaran's activism or journalism in Australia," it alleged.
Disturbed by this damning accusation, Sasikaran told JDS how he has learnt from bitter experience that free movement is not a right enjoyed by every returnee.
"I was forced to leave my homeland in 2012. That was not a period where Tamil journalists from the north and the east were able to safely leave the country from the Colombo airport. I had no other option than taking the dangerous boat ride to save my life. It is sad to see a fellow journalist who had to flee due to threats, betraying others."
Sri Lankan government has been severely critisized by international human rights organisations for arresting Tamil returnees at the airport.
Several Sinhala nationalist publications have also questioned Sasikaran's journalist credentials.
"Those who make such baseless accusations should be aware that facts need to be verified before making them public," he said.
A journalist and a rights activist from eastern Batticaloa, Sasikaran was forced to flee to Australia in 2012 due to threats to his life.
Before fleeing the country he worked as a provincial correspondent for Ran FM and Sudar Oli Tamil journal. He was also an activist involved with the campaigns of Sri Lanka National Fisheries Solidarity Movement (NAFSO) and Campaign for Free and Fair Elections (CaFFE).
While in exile Sasikaran has been working as a journalist for Australian public broadcasting radio SBS. A month before arriving in Sri Lanka, Journalist Sasikaran has written to President Maithripala Sirisena to assure his safety.
He has been called to appear in courts on 20 July to face charges on illegal immigration to Australia.
In addition to journalists from the Tamil speaking north, several media workers from the east were forced into exile due to threats following the unresolved killing of senior journalist Aiyathurai Nadesan in 2004.


This work is from a series I did where I re-imagined the universe as being made up of building blocks.. in the form of boats.. I call them Boat Building Blocks. Why boats, you may ask? The boats are a very simple molecular shape, but unlike regular molecules, they take our brain to other places; floating; contained in an ark; abandoned off an ark.. that sort of thing. These Boat Building Blocks are defined in how they interact with each other and in how they are able to proliferate on different surfaces. I enjoy how that restricts how much freedom the drawing can accommodate. This idea appeals to me because it is about pattern – these boats will form patterns and as a scientist, pattern is what we are always trying to see. If there is not pattern, there is chaos. This work suggests that in all things there is pattern. It was generated by sketching in pencil, while parked in my car on Bondi rd, straight onto the canvas and then the lines of that drawing became the templates from which to build the boats using ink and acrylic paint.


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AMVER of Automated Mutual-Assistance Vessel Rescue System is een wereldwijd vrijwillig rapporteringssysteem, dat wordt onderhouden door de Amerikaanse kustwacht. Het systeem wordt gebruikt bij search and rescue-operaties om schepen in de omgeving die mogelijkerwijs een hulp kunnen zijn bij de reddingsoperatie snel te kunnen vinden. De deelname aan het AMVER systeem brengt geen extra verplichting met zich mee die niet al bestaan onder de internationale wetgeving.

Types van AMVER-rapporten
 Sailing Plan – bevat de complete, geplande, route. Dit bericht moet enkele uren voor vertrek tot enkele uren na vertrek verzonden worden.
 Position Report – moet 24 uur na vertrek verzonden worden en minstens eens per 48 uur. De bestemming van het schip dient eveneens vermeld te worden.
 Deviation Report – dient verzonden te worden eens de route zodanig gewijzigd is dat AMVER de scheepspositie niet meer zou kunnen voorspellen. Wijzigingen van koers, snelheid, bestemming of andere afwijkingen van het originele plan dienen zo snel mogelijk gerapporteerd te worden.
 Arrival Report – moet verzonden worden bij aankomst.

Hulpdienst
United States Coast Guard


KORD is an FM radio station broadcasting at 102.7 MHz. The station is licensed to Richland, WA and is part of the Richland-Kennewick-Pasco, WA radio market. The station airs country music programming with the slogan "Continuous Country Favorites". KORD is owned by Townsquare Media.


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Beautiful Penang
Event & Happenings
Life In UK
Cool Cafe
Desserts & Beverage
Food / Many Choices
Food In UK
Hawker / Street
Kopitiam / Kopitiam Style
Local Malay & Mamak
Thai / Nyonya
Magazine 63, Speakeasy Hidden Bar In Penang
Nick Chan July 24, 2017
Even though this speakeasy is known as Magazine 63, most people still tends to refer it as the 'Hidden Bar' in Penang as it's one of few speakeasy bar in Penang. Speakeasy is more…
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Penang Secret Avatar Garden & Seafood Dinner At Pearl Garden
Tua Bui Teik Durian Stall With Authentic Local Penang Durian
Evening Run Along Gurney Drive (Soon, Gurney Wharf)
Dimsum At Chong Qing Lao Huo Guo, Kelawai Road
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All#Who#Are#Thirsty# WordsandMusicbyBrentonBrownandGlennRobertson ,1998& & &&&&&D& All&who&are&thirsty& &&&&&F#m&... Why Does Diabetes Cause Excessive Thirst? We've written before about the signs and symptoms of diabetes. While there are a lot of sources about what symptoms diabetes causes, and even some good information about why they're bad for you, what you don't often get are the "whys".
Why Does Diabetes Cause Excessive Thirst? We've written before about the signs and symptoms of diabetes. While there are a lot of sources about what symptoms diabetes causes, and even some good information about why they're bad for you, what you don't often get are the "whys".
Get Come All Who Are Thirsty: In Search of a God-Centered Life PDF. I'm Rachel bat Moshe ve Semha. My associates name me Ginette. i'm the great-grand-daughter of an orthodox rabbi. i've got a deep love for God. i've got a Jewish soul and i've a love for Jesus. convinced, my trip to holiness has been filled with surprises.
Strong Tower is a worship album released by the Christian rock group, Kutless and is also their third studio album. An enhanced deluxe edition of this album was released on March 20, 2007 with additional tracks and the "Strong Tower" live music video.


what techniques she uses, how she portrays the character of the narrator, Arthur Kipps and how to write spanking essays for Controlled Assessments and the Literature Exam.
NEW + How does Hill build tension in Chapter Four when we first meet the Woman in Black?
+ How is the Character of Arthur Kipps presented in the first four chapters?
+ How does Hill use gothic conventions in the first three chapters?
+symbolism of place and weather (pathetic fallacy) in the novel.
If you are studying The Woman in Black, you may also like"
The author, Melanie Kendry, is an Oxford graduate, outstanding-rated English Language and Literature teacher and of ages 10-18 in the British education system. In 2012, she was nominated for Pearson's Teaching Awards. As a private tutor, she raises grades often from C to A. Her writing is also featured in The Huffington Post. She offers private tuition in the Haywards Heath area, West Sussex.
Got a question or a comment? I'd love to hear it.
Curve Learn (c) M Kendry March 2013 - Present. Simple theme. Powered by Blogger.


When you see right, you act right.
2020Change is an organisation engendered to build the next generation of community, business and political leaders. We strongly believe that today's streetwise youth have the potential to become tomorrow's leading entrepreneurs, all they need is someone to show them how to use their past experiences as transferable skills that would cause them to thrive in the corporate world.
The wealthiest place on earth!
THE TRAILER FOR NEW LONDON BASED HANG CULTURE FILM '247365 CHANGE' FILM UNVEILED.
Being able to see the forest within the single seed is the key to constant growth.
Many of the worlds' most influential/ successful people are also some of the most "famous failures".
I've got dreams and I've got goals, so with everything that I've taken from this course. Like being aware of my mindset and goal setting. In 5 years time I see myself owning multiple businesses.
I've learned about self control, if someone does something that I don't how I react. Things like that used to be challenging to me, because how'd react before was me. In the programme I've learnt to react in a different way and how I can react.
So far from the goldmine session I've gained that to understand the progression that I'm trying to get into, it needs to be 80 percent mindset and 20 percent skill.