text:
Nightmare holiday: Dutch tourists robbed and terrorised
Tourism and crime: Dutch tourists held up at gunpoint, robbed and terrorised in bus from airport
By Nico Gous
Image: Alon Skuy Police Minister Fikile Mbalula and the Dutch ambassador to South Africa, Marisa Gerards, were at a press briefing at a lodge in Fourways. Mbalula said the robbery of Dutch tourists was an attack on the tourism sector and the economy
Forty-nine hours after a group of Dutch tourists set foot in South Africa for a fun three-week holiday they returned battered and bruised to their home country after becoming the victims of yet another airport robbery.
"We are safe now but we are in shock and want to go back. We just landed for a 22-day trip but the fun has gone," one of the passengers, Peter Bakker, told the Dutch newspaper Telegraaf.
The group of 36 tourists, most of them over 60, were scheduled to leave South Africa on a flight on Tuesday night and arrive back in Amsterdam around 10.30am on Wednesday - spending not even 50 hours in this country.
The tourists, who arrived in South Africa on Sunday night, were robbed on their way from OR Tambo International Airport to their accommodation in Fourways.
Their bus, they said, was stopped by a vehicle with police markings. One man was dressed in a police uniform, the other five in civilian clothing.
Dutch tour bus robbery will impact negatively on local tourism: SATSA
Describing the experience as "hell", Bakker said the hands and feet of the bus driver and the tour leader were bound with cable ties. Five men boarded the bus and stole whatever they could.
"People panicked, and firearms were aimed at them. In two cases, the trigger was pulled but the gun didn't fire," said Bakker.
Several of the tourists were assaulted.
"The suitcases were taken out of the cargo area. They were busy for 20 minutes. People cried and are in shock. They took the keys from the bus and disappeared."
Spokesman for Dutch travel company ANWB, Ad Vonk, said the tourists lost their phones, bags and luggage.
"A lot of things are gone."
The Dutch ambassador to South Africa, Marisa Gerards, said the tourists had booked an organised trip instead of striking out on their own because they had safety worries.
"It was [ironic] that those who were so concerned about their safety were so brutally robbed."
Travel company continues tours to SA despite group being robbed
After the robbers left, passengers freed themselves. The driver had a spare key in the cargo area and drove the tourists to their hotel.
Vonk said most of the tourists were 60 or older, but there were a few women in their late 20s and early 30s.
Some of the passengers were treated for light injuries.
The passports of three passengers were stolen. They were issued with new passports before flying back to the Netherlands.
Vonk said some Dutch tourists had been victims of crime in South Africa before but there had been "nothing on this scale".
He said his company was not reconsidering organising tours to South Africa "because it is a beautiful country" and is popular with Dutch tourists.
The tourists' 22-day travel programme cost them about R35,000 (€2200) each.
The tour programme included visiting Johannesburg, Pretoria, Cape Town, Swaziland, Lesotho, the Wild Coast, the Kruger National Park, the Drakensberg, Route 62 and the Garden Route.
Dutch tourists attacked by bogus cops after landing at OR Tambo airport
Police Minister Fikile Mbalula said the robbery undermined South Africa as a tourism destination.
He told ambassador Gerards: "We don't have words to express our embarrassment and our regret for what happened to the people of the Netherlands."
Mbalula met the Dutch tourists on Tuesday afternoon.
Police spokesman Brigadier Vishnu Naidoo said no arrests had been made.
The Southern Africa Tourism Services Association expressed concern that the attack would harm the industry.
"It is not good for our image," Satsa's CEO David Frost said. "Exactly what impact it will have [on tourism] we don't know. It is clearly not going to have a positive impact.
"What was good is that there was a very co-ordinated response from South African Tourism, and many of Satsa members, to sort the issue out as quickly as we could.
"When an incident like this occurs we have to use it as a learning opportunity," said Frost. "The important thing is that we all need to sit down with the minister and his top team to sort out pre-emptive measures and operating procedures that will prevent these things from happening again."
According to Stats SA, the Netherlands ranked ninth on the list of countries of tourists visiting South Africa in mid-2017.
Stats SA will release its new figures on Thursday.
- Additional reporting by Penwell Dlamini


text:
How does BTS fit this definition?
by CedarBough Saeji
BTS is the textbook perfect definition of K-pop. BTS is the most popular group from Korea.
They have only Korean members, Korean management, and sing primarily in Korean. Their music and music videos are visually driven (and full of intertextuality), songs are hip-hop heavy but genre-fluid, and many are performed with fabulous, choreographed dance. These basic facts describe the core elements of contemporary idol K-pop — in fact, in some ways such as having all Korean members, they are almost more truly K-pop than some other idol groups.
The entire idea that BTS is not K-pop or trying to separate BTS from K-pop is done solely for BTS, not for the K-pop from which they emerged and which is populated with their friends and colleagues. Is the label K-pop acceptable to BTS-pop advocates when talking about TXT, Seventeen, Gfriend, and others released by BigHit? If one believes that K-pop is a negative term, does one want to "protect" these groups from that label as well? Or only TXT because that group was designed by BigHit from the start? This industry gave birth to BTS. K-pop is the social, cultural, historical context of BTS. BTS would not exist without the K-pop industry. Are they the most popular group from Korea in the USA since the Kim Sisters? Yes, they are. (And why are we giving more credence to America than Japan or Indonesia — both countries with more BTS fans than America?) Is BTS on a different level? Yes. Are they extremely special artists? Yes. But that does not mean they are not K-pop. The things that make BTS stick out, almost all of them, are also true of other Korean artists. They're not the same as BTS, because what makes BTS different is the combination of every aspect of those seven young men, their training, and their management. But the same thing could be said about every single group. No two people are the same; no two idol groups are the same. Fans often say "only BTS this" and "only BTS that" about song writing or social messages or coming from a small company, but there is greater diversity in the K-pop industry than many people know if they aren't actively following other artists. Yes, there are idol groups formed in a stereotypical attempt to uncreatively replicate the success of others, but there are also idols like IU who writes the music and lyrics for entire albums and who is one of the biggest philanthropists in Asia.

redpajama_set_name: RedPajamaCommonCrawl


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When you stump up the cash you expect a finished product, not a buggy broken mess.
The most shocking moments of the year in gaming.
The very best of 2013's gaming big bads.


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Deb Franko, a professor of counseling and applied psychology, has developed a web application called BodiMojo that helps youth improve their body image and live healthier lives.
Kathryn Nwajiaku, Oxford, gives a talk on The politics of Oil and Ethnic Nationalism in Nigeria's Niger Delta as part of the Nigeria Economy and Society section of the Celebrating Gavin Williams Conference.
Judith Heyer gives a talk in the Agrarian Societies section of the Celebrating Gavin Williams conference, held in Oxford on 9-10 July 2010.
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Lesson 280 – Literatura brasileira.
Hoje falarei da Literatura brasileira e de alguns de seus principais ícones.
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Nesta lição irei falar sobre a formação dos substantivos. Voce irá aprender a definir se os substantivos são masculino ou feminino.
SMU Political Science Professor Matthew Wilson talks with FOX 4 News of Dallas about the latest polls showing Texas Governor Rick Perry in the lead in the race for the Republican nomination for President.


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text:
"""Common command-line flags."""

from __future__ import absolute_import
from __future__ import division
from __future__ import print_function

# Dependency imports
import tensorflow as tf

flags = tf.flags
FLAGS = flags.FLAGS

flags.DEFINE_bool("registry_help", False,
                  "If True, logs the contents of the registry and exits.")
flags.DEFINE_bool("tfdbg", False,
                  "If True, use the TF debugger CLI on train/eval.")
flags.DEFINE_bool("export_saved_model", False,
                  "DEPRECATED - see serving/export.py.")
flags.DEFINE_bool("dbgprofile", False,
                  "If True, record the timeline for chrome://tracing/.")
flags.DEFINE_string("model", None, "Which model to use.")
flags.DEFINE_string("hparams_set", None, "Which parameters to use.")
flags.DEFINE_string("hparams_range", None, "Parameters range.")
flags.DEFINE_string(
    "hparams", "",
    """A comma-separated list of `name=value` hyperparameter values. This flag
    is used to override hyperparameter settings either when manually selecting
    hyperparameters or when using Vizier. If a hyperparameter setting is
    specified by this flag then it must be a valid hyperparameter name for the
    model.""")
flags.DEFINE_string("problems", None, "Dash separated list of problems to "
                    "solve.")

# data_dir is a common flag name - catch conflicts and define it once.
try:
  flags.DEFINE_string("data_dir", None, "Directory with training data.")
except:  # pylint: disable=bare-except
  pass

flags.DEFINE_integer("train_steps", 250000,
                     "The number of steps to run training for.")
flags.DEFINE_string("eval_early_stopping_metric", "loss",
                    "If --eval_early_stopping_steps is not None, then stop "
                    "when --eval_early_stopping_metric has not decreased for "
                    "--eval_early_stopping_steps")
flags.DEFINE_float("eval_early_stopping_metric_delta", 0.1,
                   "Delta determining whether metric has plateaued.")
flags.DEFINE_integer("eval_early_stopping_steps", None,
                     "If --eval_early_stopping_steps is not None, then stop "
                     "when --eval_early_stopping_metric has not decreased for "
                     "--eval_early_stopping_steps")
flags.DEFINE_bool("eval_early_stopping_metric_minimize", True,
                  "Whether to check for the early stopping metric going down "
                  "or up.")
flags.DEFINE_bool("eval_run_autoregressive", False,
                  "Run eval autoregressively where we condition on previous"
                  "generated output instead of the actual target.")
flags.DEFINE_bool("eval_use_test_set", False,
                  "Whether to use the '-test' data for EVAL (and PREDICT).")
flags.DEFINE_integer("keep_checkpoint_max", 20,
                     "How many recent checkpoints to keep.")
flags.DEFINE_bool("enable_graph_rewriter", False,
                  "Enable graph optimizations that are not on by default.")
flags.DEFINE_integer("keep_checkpoint_every_n_hours", 10000,
                     "Number of hours between each checkpoint to be saved. "
                     "The default value 10,000 hours effectively disables it.")
flags.DEFINE_integer("save_checkpoints_secs", 0,
                     "Save checkpoints every this many seconds. "
                     "Default=0 means save checkpoints each x steps where x "
                     "is max(iterations_per_loop, local_eval_frequency).")
flags.DEFINE_bool("log_device_placement", False,
                  "Whether to log device placement.")

# Distributed training flags
flags.DEFINE_integer("local_eval_frequency", 1000,
                     "Save checkpoints and run evaluation every N steps during "
                     "local training.")
flags.DEFINE_bool("locally_shard_to_cpu", False,
                  "Use CPU as a sharding device running locally. This allows "
                  "to test sharded model construction on a machine with 1 GPU.")
flags.DEFINE_bool("sync", False, "Sync compute on PS.")
flags.DEFINE_string("worker_job", "/job:localhost", "name of worker job")
flags.DEFINE_integer("worker_gpu", 1, "How many GPUs to use.")
flags.DEFINE_integer("worker_replicas", 1, "How many workers to use.")
flags.DEFINE_integer("worker_id", 0, "Which worker task are we.")
flags.DEFINE_float("worker_gpu_memory_fraction", 0.95,
                   "Fraction of GPU memory to allocate.")
flags.DEFINE_integer("ps_gpu", 0, "How many GPUs to use per ps.")
flags.DEFINE_string("gpu_order", "", "Optional order for daisy-chaining gpus."
                    " e.g. \"1 3 2 4\"")
flags.DEFINE_string("ps_job", "/job:ps", "name of ps job")
flags.DEFINE_integer("ps_replicas", 0, "How many ps replicas.")

# Decoding flags
flags.DEFINE_string(
    "decode_hparams", "",
    "Comma-separated list of name=value pairs to control decode behavior. "
    "See decoding.decode_hparams for defaults.")

redpajama_set_name: RedPajamaGithub


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text:
Q: C: How to shmat a struct? I have a struct that looks like this:
struct data {
  int code;
  char name[25], fnum[8];
};

Then I try to use it as a shared memory like this:
int dataSID = shmget(100002, sizeof(struct data), IPC_CREAT | 0666);

and attach it like this:
struct data *com = shmat(dataSID, 0, 0);

And finally I try filling the values likes this:
(*com).code = 1;
scanf("%s", (*com).name);

And I get segmentation fault on the scanf.
What am I doing wrong?
Does shmat properly allocate memory for the struct?

A: for character arrays you can always use strcpy() or strncpy(). Of which strncpy() is recommended which helps to avoid buffer overflow.

redpajama_set_name: RedPajamaStackExchange


text:
how the brain wires itself up during development, how the end result can vary in different people and what happens when it goes wrong
What is a "neurodevelopmental disorder"?
By Kevin Mitchell - June 11, 2010
This question arose at the recent, excellent meeting of the International Society for Developmental Neuroscience in Estoril, Portugal. The question came up due to some very exciting and very unexpected successes in reversing in adult animals the effects of mutations causing neurodevelopmental disorders, including neurofibromatosis, Down syndrome, Rett syndrome and tuberous sclerosis.
All of these disorders are caused by specific genetic lesions and characterised by very early deficits, variously including intellectual disability, autism, epilepsy and other psychological and neurological phenotypes. They are also associated with some degree of neuropathology, usually involving differences in the elaboration of neuronal morphology, branching and connectivity. Because of the early onset of symptoms, these disorders have traditionally been considered as being due to defects in neurodevelopment that have led to a permanently structurally compromised brain. The last thing most people would have expected is that many of the cognitive deficits in these disorders would be reversible in adults, by either restoring normal gene function or by compensating for the effects of the genetic lesion.
These lesions can be modeled in mice, often very directly, by knocking out the homologous gene, by introducing the human disease-causing mutation into the mouse gene or, for Down syndrome, by transgenically increasing the copy number of all the genes corresponding to human chromosome 21. For all these syndromes, genetically engineered mice display a range of cognitive phenotypes, correlated with alterations in the physiology of individual neurons and circuits. They thus provide invaluable models to investigate the mechanisms of disease and develop and test possible therapeutic approaches.
Rett syndrome is caused by mutations in MeCP2, which encodes a protein that binds to methylated DNA and controls gene expression. Such mutations are lethal in males, who have only one copy of this X-chromosomal gene, while girls inheriting the mutation develop a syndrome of intellectual disability, autism and sometimes epilepsy. MeCP2 mutations are also lethal in male mice, though they survive for some time and show severe behavioural defects, while females show a milder phenotype. These defects were assumed to be due to the absence of MeCP2 function during neurodevelopment and were therefore expected to be irreversible by subsequent restoration of MeCP2 activity.
However, reversal was exactly what was observed when the MeCP2 gene was turned on again from a transgene in adult mice. Lethality in males was completely rescued (not, as Adrian Bird said, after the fact), and defects in both sexes in synaptic plasticity underlying memory (long-term potentiation) and in behavioural measures of memory were all essentially completely reversed. Even more remarkable, reductions in size of particular neurons (parvalbumin-positive interneurons), in the thickness of the cortex and in the density of dendritic spines (reflecting the number of synaptic connections) were all also reversed. These results argue that Rett syndrome is less a disorder of neurodevelopment and more a disorder of aberrant neuronal maintenance.
Results for neurofibromatosis, tuberous sclerosis and Down syndrome were all similar, though in these cases, reversal was achieved in adults by pharmacologically compensating for the effects of the genetic lesions. By mutating the gene responsible for neurofibromatosis, NF1, specifically in distinct classes of neurons in mice, Alcino Silva and colleagues had found that the effects of this mutation on physiology and behaviour were only observed when NF1 was deleted in inhibitory interneurons. This led to an increase in the activity of these neurons, and a concomitant decrease in the activity of excitatory neurons. By blocking the receptors of inhibitory neurotransmission using picrotoxin, it was possible to restore the correct balance of inhibition and excitation in adults and to reverse the effects on physiology and behaviour. As with Rett syndrome, the effects of the NF1 mutation were thus not as permanent as expected.
Down syndrome model mice showed a similar effect: an increase in inhibitory neurotransmission, correlated with defects in long-term potentiation and performance in cognitive tasks. As with neurofibromatosis, blocking inhibitory receptors in adults caused remarkable rescue of these defects. Working out the nature of the biochemical defect in tuberous sclerosis also suggested a mechanism by which to reverse the effects in adults, which worked equally well.
These successes have been remarkable not just for how unexpected they were but also for how substantial the improvement is in adult animals when normal gene function is restored or the biochemical or physiological defect is compensated for. The results obviously offer great promise for effective therapeutic intervention in patients and some of the approaches are already in small-scale clinical trials. As in all such situations, however, translation to humans will be slow and difficult and miracle cures are unlikely to emerge in the near future.
Do these principles apply to all neurodevelopmental disorders? It is unlikely. Indeed, many other disorders not previously classified as neurodevelopmental may have to be re-evaluated as such. One of these is schizophrenia. While the symptoms of this disease do not usually emerge until late adolescence, there is very good evidence that they stem from earlier insults in neurodevelopment. This is supported by additional modeling of schizophrenia-associated mutations in mice, including mutations in the gene DISC1. DISC1 mutations cause a range of schizophrenia-related behavioural phenotypes in mice. Remarkably, disruption of the activity of DISC1 just during development is sufficient to induce these phenotypes in adults, even though normal gene function was subsequently restored. In this case, defects in neurodevelopment can thus not be reversed in adults.
Perhaps neurodevelopmental disorders should be defined as such not so much on when the symptoms arise but on the timing of the requirement for gene function.
EHNINGER, D., LI, W., FOX, K., STRYKER, M., & SILVA, A. (2008). Reversing Neurodevelopmental Disorders in Adults Neuron, 60 (6), 950-960 DOI: 10.1016/j.neuron.2008.12.007
Thank you for the informative report of what sounded like an excellent meeting.
I think it is clear that the fact that a given disorder emerges during development is not sufficient evidence for the disorder to be classed as developmental. But it seems that there's a flip side to the coin of reversibility (which you touched on with the DISC1 example). It seems to me that disorders that can be rescued in adulthood cannot be excluded from the developmental category straight away. It must also be established that the disease phenotype requires loss of function during development. So, even though mouse models of Rett syndrome and the other examples can be rescued by adulthood expression of the missing gene, the question remains did the mutant phenotype originate because of absence of the gene during development in the first place.
The experiment to answer this question would probably be to delete MeCP2 only after adulthood is reached, and see whether or not the disease phenotype occurs. If it does, then it's not developmental, if it doesn't, then it is developmental, but for some reason can also be rescued in adulthood.
Loss of the NMDA receptor NR1 subunit in cortico-hippocampal interneurons during postnatal development results in schizophrenia-like symptoms (Belforte et al '10 Nat Neuro). But this group tested whether adult deletion of NR1 caused the same effect, and demonstrated that indeed it did not. They did not report an adult reversal experiment, but regardless of whether or not the symptoms can be reversed by adult expression of NR1, the disorder is surely developmental. So I think your definition of the timing of requirement of gene function certainly seems the most reasonable/accurate definition.
Therefore if we can reverse these things in adults – great! and the Biotech companies will be throwing more resources at developing those solutions than any academic institution could. But either way if the disorder originates during development , then that is where fundamental science might still want concentrate a good deal of its research, and where preventative efforts by educational and health-care professionals might prove most useful for humans.
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How much innate knowledge can the genome encode?
By Kevin Mitchell - January 04, 2020
In a recent debate between Gary Marcus and Yoshua Bengio about the future of Artificial Intelligence, the question came up of how much information the genome can encode. This relates to the idea of how much innate or prior "knowledge" human beings are really born with, versus what we learn through experience. This is a hot topic in AI these days as people debate how much prior knowledge needs to be pre-wired into AI systems, in order to get them to achieve something more akin to natural intelligence.
Bengio (like Yann leCun) argues for putting as little prior knowledge into the system as we can get away with – mainly in the form of meta-learning rules, rather than specific details about specific things in the environment – such that the system that emerges through deep learning from the data supplied to it will be maximally capable of generalisation. (In his view, more detailed priors give a more specialised, but a more limited and possibly more biased machine). Marcus argues for more…
Beyond reductionism – systems biology gets dynamic
By Kevin Mitchell - September 14, 2019
Is biology just complicated physics? Can we understand living things as complex machines, with different parts dedicated to specific functions? Or can we finally move to investigating them as complex, integrative, and dynamic systems?
For many decades, mechanistic and reductionist approaches have dominated biology, for a number of compelling reasons. First, they seem more legitimately scientific than holistic alternatives – more precise, more rigorous, closer to the pure objectivity of physics. Second, they work, up to a point at least – they have given us powerful insights into the logic of biological systems, yielding new power to predict and manipulate. And third, they were all we had – studying entire systems was just too difficult. All of that is changing, as illustrated by a flurry of recent papers that are using new technology to revive some old theories and neglected philosophies.
The central method of biological reductionism is to use controlled manipulation of individual comp…
The murderous brain - can neuroimaging really distinguish murderers?
By Kevin Mitchell - July 14, 2019
A new study claims that neuroimaging can be used to distinguish the brains of murderers from non-murderers. It follows in a long tradition of attempts to find biological indicators of violent criminality, from faces to skull bumps to genes to brains. But are the data convincing? Does this study really accomplish what it claims? Is it even based on a well-founded question? And what are the ethical implications?
Here is the abstract of the paper, by Ashly Sajous-Turner and colleagues:
Homicide is a significant societal problem with economic costs in the billions of dollars annually and incalculable emotionaimpact on victims and society. Despite this high burden, we know very little about the neuroscience of individuals who commit homicide. Here we examine brain gray matter differences in incarcerated adult males who have committed homicide (n = 203) compared to other non-homicide offenders (n = 605; total n = 808). Homicide offenders' show reduced gray matter in brain areas critical for …
axon guidance
behavioural traits
biological essentialism
cell types
CNVs
common disorders
common variants
complex disorders
conectivity
congenital amusia
copy number variants
cortical areas
cross-wiring
cryptic genetic variation
developmental trajectories
dimorphism
epistasis
epistatic
face blindness
fragile X
fragility
GCTA
genetic architecture
genetic diagnosis
genetic prediction
genetic screens
genetics autism
genome-wide association studies
genotype-phenotype mapping
heritable
heterogeneity
heterotopia
hub neurons
Huntington's
laterality
leucine-rich
LRRTM
midline
multisensory integration
mutant mice
neurexin
neurobollocks
neurogenetics
neuroligin
neuronal networks
omnigenic
polygenic
polygenic scores
prenatal effects
process philosophy
prosopagnosia
psychiatric genetics
psychological traits
quantitative traits
rare disorders
rare mutations
rare variants
real genetics
replicability
self-organising
semaphorin
small-world
structural imaging
subjective experience
synapse formation
synaptogenesis
Tammett
thalamocortical
whole-genome sequencing


Wohlmuth, H., Smith, M. and Brooks, L.O. (2006) Essential Oil Composition of Diploid and Tetraploid Clones of Ginger (Zingiber officinale Roscoe) Grown in Australia. Journal of Agricultural Food Chemistry, 54 (4). pp. 1414-1419.
Ginger oil, obtained by steam distillation of the rhizome of Zingiber officinale Roscoe, is used in the beverage and fragrance industries. Ginger oil displays considerable compositional diversity, but is typically characterized by a high content of sesquiterpene hydrocarbons, including zingiberene, arcurcumene, â-bisabolene, and â-sesquiphellandrene. Australian ginger oil has a reputation for possessing a particular "lemony" aroma, due to its high content of the isomers neral and geranial, often collectively referred to as citral. Fresh rhizomes of 17 clones of Australian ginger, including commercial cultivars and experimental tetraploid clones, were steam distilled 7 weeks post-harvest, and the resulting oils were analyzed by GC-MS. The essential oils of 16 of the 17 clones, including the tetraploid clones and their parent cultivar, were found to be of substantially similar composition. These oils were characterized by very high citral levels (51-71%) and relatively low levels of the sesquiterpene hydrocarbons typical of ginger oil. The citral levels of most of these oils exceeded those previously reported for ginger oils. The neral-to-geranial ratio was shown to be remarkably constant (0.61 ( 0.01) across all 17 clones. One clone, the cultivar "Jamaican", yielded oil with a substantially different composition, lower citral content and higher levels of sesquiterpene hydrocarbons. Because this cultivar also contains significantly higher concentrations of pungent gingerols, it possesses unique aroma and flavor characteristics, which should be of commercial interest.
Ginger; Zingiber officinale; ginger oil; essential oil; citral; neral; geranial; tetraploidy.


The portals of Halloween are opening! A kaleidoscope of creatures will emerge as The Charter attempts to maintain control.
Meow Wolf's House of Halloween is happening throughout October with 15 performances presented by 80 artists and performers. This site-specific theater experience will activate Meow Wolf's House of Eternal Return with 3 hours worth of unexpected performance, crazy costumes, new artwork, and a special appearance by "The Family". This will be the most talked about Halloween experience in New Mexico!
First tier tickets are on sale today at $25, second tier will be $35 so get them early! Space is limited! All ages are welcome!


Our focus is to provide the best quality youth basketball development programs for boys and girls ages 6 – 13 from beginner to advanced. Our focus is on fundamentals, good sportsmanship and fair play so that our players will enjoy playing the sport and continue their development for as long as they play basketball.
Saturdays, April 20 through June 15 (excluding the May long weekend) at Rundle Academy (4330 - 16 St SW) from 12:35pm - 2:00pm. Coed, groups split on separate courts grades 1 - 4 and grades 5 - 7.
First time players will receive a SUNS basketball, reversible jersey, water bottle and cinch bag. Returning players may choose optional apparel.
Thursdays, April 25 through June 13 at the Lake Bonavista Recreation Centre (1401 Acadia Drive SE) from 4:20pm - 5:50 pm.
First time players will receive a SUNS basketbal, reversible jersey, water bottle and cinch bag. Returning players will receive their choice of optional apparel.
First time players receive a SUNS basketball, reversible jersey, water bottle and cinch bag. Returning players have their choice of optional apparel.
Fundamental skills training for beginners or players with limited competitive experience in grades 1 - 7.
Tuesdays, April 23 through June 11 from 6:30pm - 8:00pm at the Crscent Point Field House (Aldersyde). Coed, players split by age group grades 1 - 4 and 5 - 7.
First time players receive a SUNS basketball, reversible jersey, water bottle and cinch bag. Returning players will have a choice of apparel packages.
Fundamental skills training for players in grades 1 - 7 split on separate courts by age group.
Saturdays, April 27 through June 15 (excluding the May long weekend) from 3:00pm - 4:30pm at Captain Nicola Gooddard School (405 Panatella Blvd NW). Coed, players split in groups on separate courts by grades 1 - 4 and 5 - 7.
First time players receive a SUNS basketball, reversible jersey, water bottle and cinch bag.Returning players have their choice of optional apparel.
SUNS Basketball BASIC Fundamental skills session for players just start or with limited experience.
***NEW SESSION*** Thursdays, April 25 - June 13 at Hounsfield Heights/Briar Hill Community Gym (1928 - 14 Avenue NW).
Coed for players ages 6 - 12 split by age groups (6 - 9 and 10 - 12) from 5:30pm - 6:45pm. Players will receive a SUNS reversible jersey, cinch bag & water bottle.
SUNS Basketball FUNdamental Skills Camp is for beginners or players with little or no competitive basketball experience. This camp is ideal for children who want an introduction to basketball or have already started and wish to continue their skills development in a fun and no pressure environment.
Our coaches have extensive experience working with these two age groups and provide a great atmosphere for learning. We provide a coach for every 6 - 8 players so the children receive care and instruction throughout the camp.
This camp is located at Glenmore Christian School (16520 - 24 St SW) East Gym from Monday, July 22 through Friday, July 26.
Players are divided in to two ages groups, ages 6 - 9 and 10 - 13 split on separate courts.
The camp runs 6 hours per day, Monday to Friday from 9:00am - 4:00pm with a one hour supervised lunch break.
Players receive 30 hours of on-court instruction, skills development, athletic skills training, contests, fun games with scrimmage time twice per day.
Registration fee is $348 which includes a SUNS Basketball, SUNS reversible jersey, cinch bag & water bottle.
This camp is located at Glenmore Christian Academy (16520 -24 St SW) West Gym from Monday, July 22 through Friday, July 26.
Your choice of mornings from 9:00am - 12:00pm or afternoons from 1:00pm - 4:00pm. Please indicate your choice of AM or PM on your online form under program of interest.
Players receive 15 hours of on-court instruction, skills development, athletic skills training, contests, fun games with scrimmage time twice per day.
Registration fee is $189 which inccludes a SUNS Basketball, SUNS reversible jersey, cinch bag & water bottle.
Ideal for players interested in developing their skills in two different sports in the same week. Ideal for the beginner or players with some experience in either or both sports. Stryker Sports is an advocate for multi-sport athletics. We encourage all of our players to try and play as many sports as possible, especially in the younger age brackets.
This camp is located at Glenmore Christian Academy (16520 - 24 St SW) and runs from Monday, July 22 - Friday, July 26 from 9:00am - 4:00pm with a one hour lunch break each day. You would be on court for three hours of basketball each day plus three hours of volleyball each day.
This camp will be coached by members of our coaching staff who have played and/or coached both sports at a highly competitive level. You will receive fundamental skills instruction, balance, speed and agility training plus some fun games and scrimmage time each session.
The camp is divided on separate courts by age with ages 6 - 9 on one court and ages 10 - 13 on the other court.
The registration fee includes SUNS & Stars apparel, tee-shirts, water bottle and a bag.
The fee is $348 all inclusive.
Ideal for players interested in playing two different sports in the same week. Ideal for the beginner or players with some experience in either or both sports. Stryker Sports is an advociate for multi-sport athletes. We encourage all of our players to try and play as many sports as possible, especially in these younger age brackets.
This camp is located at Rundle College (7379 - 17 Avenue SW) and runs from Monday, August 12 - Friday, August 16 from 9:00am - 4:00pm with a one hour lunch break. You would be on court for three hours of basketball each day plus three hours of volleyball each day.
This camp is located at Rundle College (7379 - 17 Avenue SW) from Monday, August 13 through Friday, August 17.
Time: Afternoons from 1:00pm - 4:00pm each day.
Registration fee is $189 which includes a SUNS Basketball, SUNS reversible jersey, cinch bag & water bottle.
SUNS Fundamental skills training ideal for beginners or players with limited experience. Divided in to age groups by grades 1 - 4 and 5 - 7.
Rundle Academy (4330 - 16 St SW) from Monday, July 15 through Friday, July 19. Players on court from 9:00am - 12:00pm and 1:00pm - 4:00pm daily with a one hour supervised lunch break.
Split on separate courts by grades 1 - 4 and 5 - 7.
Players receive a SUNS basketball, SUNS reversible jersey, cinch bag and water bottle.
Rundle Academy (4330 - 16 St SW) choose mornings from 9:00am - 12:00pm or afternoons 1:00pm - 4:00pm) each day.
Players receive a SUNS Basketball, SUNS reversible jersey, socks and cinch bag.


text:
Q: How to merge changes into a Git repo? I made a mistake when copying our repo to the dev directory. After running mkdir dev && cd dev && git clone <projecturl> I decided to copy the files out of the clone dev/myproject directory into the parent directory dev. The problem is I forgot to copy over .git, so now all of my changes exist in a directory that isn't a Git repo, and I have no obvious way to commit the changes that I'm aware of.
I tried this and it did not work:
cp -RfHip production/.git dev/.git

I'm wondering how I can merge my changes without creating merge conflicts, if possible. My changes are significant enough that having to manually merge them is not feasible. I guess if all else fails I could just copy the files from dev to production and then commit it, and start removing the old files from production that don't get overwritten in the commit.

A: So funny thing, i was able to solve it myself. cp is unable to copy subdirectories. i needed to cd to the production directory and copy the files from there.
cd production && cp -rfHip .git ../dev/.git && cd ../dev && git add -A && git commit -a


Odell Beckham Jr. trade: Will NY Giants admit they're now rebuilding?
The hardest sell for the New York Giants right now will be convincing their fans — really, anyone, for that matter — that they are playing to win in 2019 after trading Odell Beckham Jr. to the Cleveland Browns.
It's OK for Big Blue to admit they are rebuilding. The question of the moment: Will they?
Because that's where we are in assessing how the Giants move forward without one of the most dynamic players in the NFL after Tuesday night's stunning deal with the Browns, in which they got in return a first-round pick, a third-round pick and hometown favorite Jabrill Peppers.
They also received back from the Browns the fourth-round pick that they flipped for a fifth-rounder last week in the Olivier Vernon-for-Kevin Zeitler swap.
From the outside, the Giants appear to be a team without direction. Ownership and general manager Dave Gettleman are taking heat — in some ways, deservedly so — for essentially gutting the current roster of its best players.
Damon Harrison. Landon Collins. Vernon. All gone from the defense that is woefully undermanned, even with promising young players like Dalvin Tomlinson, B.J. Hill, Lorenzo Carter and the newly acquired Peppers.
Then the one that left mouths agape Tuesday, shipping Beckham off to Cleveland in a move that appears to be a preemptive strike against a player whose burning desire to win could ultimately prove to be a distraction during an obvious reclamation project.
The Giants were afraid Beckham would let his frustration with Eli Manning and another year of losing get in the way of the big picture. Problem is, that big picture is cloudy at the moment, and fear is not the best way to build a winning team.
As Gettleman said at the Combine: "No guts, no glory."
There's no question managing Beckham's personality is a challenge, and the beginning of the end of Beckham's tenure with the Giants was the interview he did on ESPN with Lil Wayne seated beside him. Everything changed between Giants coach Pat Shurmur and Beckham then, even if neither allowed the divide to surface publicly.
So after living by the old Ernie Accorsi mantra of not giving up on talent, Gettleman gave up Beckham for a player who has yet to reach his potential in Peppers, and a first- and a third-round pick. The haul was far greater than what the Steelers were forced to accept in their divorce from Antonio Brown, but seemingly far less than anticipated for a star like Beckham.
The Giants are confident they have made the right move, believing Gettleman will take the group of 12 draft picks he has garnered for this year and methodically fill the holes created by the previous regime of Jerry Reese, Marc Ross, Ben McAdoo and company.
Will the Giants make a move for their next quarterback? At the moment, I'd lean toward Gettleman finding his starting right tackle — maybe Oklahoma's Cody Ford — with the No. 17 pick from the Browns, taking a defensive playmaker at No. 6. But, as we've seen, that could change in a blink.
But co-owners John Mara and Steve Tisch, more than anyone, are responsible for where the Giants find themselves. They signed off on all these moves, including the decision to give up on a talent in Beckham, whom Mara himself compared in the past to Hall of Famer Lawrence Taylor.
The Giants are caught between trying to compete with the Eagles, Cowboys and Redskins in the NFC East and finding the next quarterback to replace Manning and lead them into the next decade.
And let's be honest: they've failed in both of those quests.
Which brings us to Manning: Why would the Giants bring back the two-time Super Bowl MVP quarterback for this season when everything around him suggests a movement toward a new era of Big Blue football?
They're going to pay a 38-year-old franchise legend to hand off to Saquon Barkley 25 times a game, all the while making $23 million, a number that prevents team brass from using $17 million to improve this roster now.
That's not what a rebuilding team should do.
Let's not forget what Archie Manning told ESPN's Ian O'Connor on the night the miserable 2018 season ended: "If Eli is done playing, I'm fine with it. But if he comes back, the Giants have got to win. They can't go through another season like this."
Was that an ultimatum from the eldest Manning? Gettleman scoffed at the sentiment in his post-season news conference, saying he'd heard nothing directly from Eli's and Peyton's father. But if you're Eli Manning, does he really want to endure the same fate Kurt Warner did 15 seasons ago?
It's pretty evident that, football-wise, the Giants see Manning as their best option on the current roster to be the quarterback, especially from the perspective of a team desperate to turn the corner on five losing seasons in the past six years.
If this is truly about the long game here with the Giants, however, and Gettleman is committed to whatever is in the best interests of the franchise and his promise to "build sustained success," there needs to be serious discussion about where Manning fits.
You can't build sustained success by not being bold.
Forget sentimentality. Don't worry about what the fans or media may think about the decisions that need to be made. Win everyone over by winning again.
That's what the Giants believe they are positioned to do. Beckham's gone, and with him goes another player who was blamed for the problems of an entire organization. Is he complicit in that? You bet.
With Beckham gone, the Giants have taken yet another step toward a new future.
That future is as uncertain for the Giants today as it has been for decades.
What comes next defines the franchise for years to come.
Team brass can only hope the unknown with Barkley leading the way — and little else around him — turns out far better than what the empty reality appears to be right now.
Until the Giants reveal that plan in its entirety, the questions over exactly where the franchise is headed will remain.


text:
Distribution of virus and viral antigen in specific pathogen-free calves following inoculation with noncytopathic bovine viral diarrhea virus
D. J. Marshall, Rodney A Moxley, Clayton L Kelling
Veterinary and Biomedical Sciences
Gross and microscopic lesions and distribution of virus were studied in specific pathogen-free calves (SPF) 10 days post-inoculation (PI) with bovine viral diarrhea virus (BVDV). To investigate possible differences in tissue tropism between BVDV isolates, two clinically and antigenically different noncytopathic (ncp) isolates of BVDV were compared in the study. Four calves were exposed to noncytopathic (ncp) BVDV 7937, and four to ncp-BVDV 126. Two additional calves that were not exposed to virus served as controls. Both ncp-BVDV 7937 and ncp-BVDV 126 induced mild disease characterized by variable fever and anorexia. Lymphoid depletion was evident in Peyer's patch of four calves and the thymus of two calves exposed to BVDV. Differences between these isolates in the distribution of BVDV or BVDV antigen in tissues of inoculated calves were not found. High concentrations of BVDV and BVDV-specific antigen were detected in the thymus, Peyer's patch, and mesenteric lymph node of all exposed calves. BVDV was shown to infect cells of the bone marrow without causing microscopic lesions. High concentrations of BVDV were recovered from the bone marrow of all calves exposed to BVDV and BVDV-specific antigen was demonstrated at this location in six of these calves. Platelet counts of calves exposed to BVDV were significantly reduced during infection, which resulted in thrombocytopenia in one calf. Focal areas of necrosis were observed in squamous epithelial cells of the tonsil and ruminai mucosa. BVDV-specific antigen was found in and adjacent to these foci. Calves exposed to ncp-BVDV 7937 had slightly more severe clinical signs than those exposed to ncp-BVDV 126.
Veterinary pathology
Bovine Viral Diarrhea Viruses
Specific Pathogen-Free Organisms
Bovine viral diarrhea virus
Peyer's Patches
lesions (animal)
tissue tropism
Antigen bovine viral diarrhea virus
Noncytopathic bovine viral diarrhea virus
Specific pathogen-free calves
veterinary(all)
Marshall, D. J., Moxley, R. A., & Kelling, C. L. (1996). Distribution of virus and viral antigen in specific pathogen-free calves following inoculation with noncytopathic bovine viral diarrhea virus. Veterinary pathology, 33(3), 311-318. https://doi.org/10.1177/030098589603300308
Distribution of virus and viral antigen in specific pathogen-free calves following inoculation with noncytopathic bovine viral diarrhea virus. / Marshall, D. J.; Moxley, Rodney A; Kelling, Clayton L.
In: Veterinary pathology, Vol. 33, No. 3, 05.1996, p. 311-318.
Marshall, DJ, Moxley, RA & Kelling, CL 1996, 'Distribution of virus and viral antigen in specific pathogen-free calves following inoculation with noncytopathic bovine viral diarrhea virus', Veterinary pathology, vol. 33, no. 3, pp. 311-318. https://doi.org/10.1177/030098589603300308
Marshall DJ, Moxley RA, Kelling CL. Distribution of virus and viral antigen in specific pathogen-free calves following inoculation with noncytopathic bovine viral diarrhea virus. Veterinary pathology. 1996 May;33(3):311-318. https://doi.org/10.1177/030098589603300308
Marshall, D. J. ; Moxley, Rodney A ; Kelling, Clayton L. / Distribution of virus and viral antigen in specific pathogen-free calves following inoculation with noncytopathic bovine viral diarrhea virus. In: Veterinary pathology. 1996 ; Vol. 33, No. 3. pp. 311-318.
@article{049c57826d8048a1b4a440a9f4391f64,
title = "Distribution of virus and viral antigen in specific pathogen-free calves following inoculation with noncytopathic bovine viral diarrhea virus",
abstract = "Gross and microscopic lesions and distribution of virus were studied in specific pathogen-free calves (SPF) 10 days post-inoculation (PI) with bovine viral diarrhea virus (BVDV). To investigate possible differences in tissue tropism between BVDV isolates, two clinically and antigenically different noncytopathic (ncp) isolates of BVDV were compared in the study. Four calves were exposed to noncytopathic (ncp) BVDV 7937, and four to ncp-BVDV 126. Two additional calves that were not exposed to virus served as controls. Both ncp-BVDV 7937 and ncp-BVDV 126 induced mild disease characterized by variable fever and anorexia. Lymphoid depletion was evident in Peyer's patch of four calves and the thymus of two calves exposed to BVDV. Differences between these isolates in the distribution of BVDV or BVDV antigen in tissues of inoculated calves were not found. High concentrations of BVDV and BVDV-specific antigen were detected in the thymus, Peyer's patch, and mesenteric lymph node of all exposed calves. BVDV was shown to infect cells of the bone marrow without causing microscopic lesions. High concentrations of BVDV were recovered from the bone marrow of all calves exposed to BVDV and BVDV-specific antigen was demonstrated at this location in six of these calves. Platelet counts of calves exposed to BVDV were significantly reduced during infection, which resulted in thrombocytopenia in one calf. Focal areas of necrosis were observed in squamous epithelial cells of the tonsil and ruminai mucosa. BVDV-specific antigen was found in and adjacent to these foci. Calves exposed to ncp-BVDV 7937 had slightly more severe clinical signs than those exposed to ncp-BVDV 126.",
keywords = "Antigen bovine viral diarrhea virus, Immunohistochemistry, Noncytopathic bovine viral diarrhea virus, Specific pathogen-free calves",
author = "Marshall, {D. J.} and Moxley, {Rodney A} and Kelling, {Clayton L}",
journal = "Veterinary Pathology",
T1 - Distribution of virus and viral antigen in specific pathogen-free calves following inoculation with noncytopathic bovine viral diarrhea virus
AU - Marshall, D. J.
AU - Moxley, Rodney A
AU - Kelling, Clayton L
N2 - Gross and microscopic lesions and distribution of virus were studied in specific pathogen-free calves (SPF) 10 days post-inoculation (PI) with bovine viral diarrhea virus (BVDV). To investigate possible differences in tissue tropism between BVDV isolates, two clinically and antigenically different noncytopathic (ncp) isolates of BVDV were compared in the study. Four calves were exposed to noncytopathic (ncp) BVDV 7937, and four to ncp-BVDV 126. Two additional calves that were not exposed to virus served as controls. Both ncp-BVDV 7937 and ncp-BVDV 126 induced mild disease characterized by variable fever and anorexia. Lymphoid depletion was evident in Peyer's patch of four calves and the thymus of two calves exposed to BVDV. Differences between these isolates in the distribution of BVDV or BVDV antigen in tissues of inoculated calves were not found. High concentrations of BVDV and BVDV-specific antigen were detected in the thymus, Peyer's patch, and mesenteric lymph node of all exposed calves. BVDV was shown to infect cells of the bone marrow without causing microscopic lesions. High concentrations of BVDV were recovered from the bone marrow of all calves exposed to BVDV and BVDV-specific antigen was demonstrated at this location in six of these calves. Platelet counts of calves exposed to BVDV were significantly reduced during infection, which resulted in thrombocytopenia in one calf. Focal areas of necrosis were observed in squamous epithelial cells of the tonsil and ruminai mucosa. BVDV-specific antigen was found in and adjacent to these foci. Calves exposed to ncp-BVDV 7937 had slightly more severe clinical signs than those exposed to ncp-BVDV 126.
AB - Gross and microscopic lesions and distribution of virus were studied in specific pathogen-free calves (SPF) 10 days post-inoculation (PI) with bovine viral diarrhea virus (BVDV). To investigate possible differences in tissue tropism between BVDV isolates, two clinically and antigenically different noncytopathic (ncp) isolates of BVDV were compared in the study. Four calves were exposed to noncytopathic (ncp) BVDV 7937, and four to ncp-BVDV 126. Two additional calves that were not exposed to virus served as controls. Both ncp-BVDV 7937 and ncp-BVDV 126 induced mild disease characterized by variable fever and anorexia. Lymphoid depletion was evident in Peyer's patch of four calves and the thymus of two calves exposed to BVDV. Differences between these isolates in the distribution of BVDV or BVDV antigen in tissues of inoculated calves were not found. High concentrations of BVDV and BVDV-specific antigen were detected in the thymus, Peyer's patch, and mesenteric lymph node of all exposed calves. BVDV was shown to infect cells of the bone marrow without causing microscopic lesions. High concentrations of BVDV were recovered from the bone marrow of all calves exposed to BVDV and BVDV-specific antigen was demonstrated at this location in six of these calves. Platelet counts of calves exposed to BVDV were significantly reduced during infection, which resulted in thrombocytopenia in one calf. Focal areas of necrosis were observed in squamous epithelial cells of the tonsil and ruminai mucosa. BVDV-specific antigen was found in and adjacent to these foci. Calves exposed to ncp-BVDV 7937 had slightly more severe clinical signs than those exposed to ncp-BVDV 126.
KW - Antigen bovine viral diarrhea virus
KW - Noncytopathic bovine viral diarrhea virus
KW - Specific pathogen-free calves
JO - Veterinary Pathology
JF - Veterinary Pathology


text:
Události
 2000–1000 př. n. l. – Dynastie Shang v Číně, Olmécká civilizace (Střední Amerika).
 2000 př. n. l. – Zhroucení třetí dynastie urské je v literární formě popsáno ve dvou Nářcích nad zkázou města Uru.
 2064–1986 př. n. l. – Válka dvou dynastií v Egyptě.
 1991 př. n. l. – Egypt: Zemřel faraon Mentuhotep IV. Konec jedenácté dynastie. Začal vládnout faraon Amenemhet I. Začátek dvanácté dynastie.
 cca 1991 př. n. l. – 1785 př. n. l. – U Beni Hasanu vznikly hrobky vytesané do skály (dvanáctá dynastie).
 1962 př. n. l. – Po Amenemhétově smrti skládá písař Cheti Poučení krále Amenemhéta I.
 1932 př. n. l. – Amorité si podrobili Ur.
 1926 př. n. l. – Za panování faraóna Senusreta I. vzniklo vyprávění o Sinuhetovi, nejvýznamnější literární dílo starého Egypta.
 1926 př. n. l. – Senusret I., egyptský faraón 12. dynastie, umírá.
 1913–1903 př. n. l. – Egyptsko–Nubijská válka.
 cca 1900 př. n. l. – Pád poslední Sumerské dynastie.

Externí odkazy

Staletí

redpajama_set_name: RedPajamaWikipedia


View full PDF of Throwdown stats for September here Video Gallery post September 2015 Fighter Matches This table displays the number of matches each fighter had for the September 2015 event. Those fighters that have not chosen a display name are listed as anonymous. If you want to update your display name please contact the website admin.
View full PDF of Throwdown stats for July here Video Gallery post July 2015 Fighter Matches This table displays the number of matches each fighter had for the July 2015 event. Those fighters that have not chosen a display name are listed as anonymous. If you want to update your display name please contact the website admin.
View full PDF of Throwdown stats for May here Video Gallery post March 2015 Fighter Matches This table displays the number of matches each fighter had for the March 2015 event. Those fighters that have not chosen a display name are listed as anonymous. If you want to update your display name please contact the website admin.


text:
The General Assembly Permanent Judicial Commission is staffed by the department of Constitutional Services.
"Church discipline is the church's exercise of authority given by Christ." (Book of Order, D-1.0101).
Judicial process is the exercise of authority for the church by the councils for the correction of:
irregularities and delinquencies taken by councils, the Mission Agency, or an entity of the General Assembly (referred to as remedial cases); and
offenses by individual members (referred to as disciplinary cases).
Judicial process is explained in the Rules of Discipline found in the Book of Order, which is Part Two of The Constitution of the Presbyterian Church (U.S.A.). The General Assembly, each synod, and each presbytery elects a permanent judicial commission composed of ministers and elders subject to its jurisdiction. The General Assembly Permanent Judicial Commission (GAPJC) is composed of one member elected by the General Assembly from each of its constituent synods (16). The General Assembly Permanent Judicial Commission issues Authoritative Interpretations of The Constitution of the Presbyterian Church (U.S.A.) through its decisions.
Staff Support
Flor Vélez-Díaz, Manager for Judicial Process
email: Flor.Vélez-Díaz@pcusa.org
Diane Minter, Manager of Administration
email: diane.minter@pcusa.org
The General Assembly Permanent Judicial Commission is a permanent commission of the General Assembly of the Presbyterian Church (U.S.A.) that exercises church discipline through judicial process.
FAQ Gracious Dismissal Policies after Tom vs. the Presbytery of San Francisco
Guide for Judicial Process
Members and Electoral Process
GAPJC Decisions
Annotated Book of Order
Handbook for Judicial Process ->
Polity Resources


Multiple sources says that the LA Galaxy have recently extended an offer to Zlatan Ibrahimovic that would bring him to the club this summer.
Multiple sources say that the LA Galaxy have recently extended an offer to Zlatan Ibrahimovic, one that would bring him to the club this summer and pay him what would easily be the highest salary in MLS history.
Ibrahimovic is 35 years old, but he has been fantastic for Manchester United this season, scoring 26 goals in all competitions. Ibrahimovic's United contract runs out at the end of this season, so it would be a free transfer, but there's a possibility he could extend for up to two more seasons with the club.
Watching Manchester United get into a bidding war with LA over Ibrahimovic would be fascinating, and sources say that LA thinks it has a realistic shot of landing him. LA did have discussions with Ibrahimovic last year before he ended up signing with the United, and his manager, Jose Mourinho, is on record recently saying that he sees Ibrahimovic, who scored the winning goals in the Community Shield and League Cup final, remaining at Old Trafford.
2017 MLS Ambition Rankings: Who strives for greatness the most?
In terms of MLS roster logistics, the Galaxy currently have their three allotted Designated Player places occupied by Giovani Dos Santos, Jelle Van Damme and winter signing Romain Alessandrini. LA could buy down the budget charge of one of the three using Targeted Allocation Money, though, which would pave the way to clear a DP place for Ibrahimovic.
Ibrahimovic is about to begin serving a three-game ban in England for elbowing Bournemouth's Tyrone Mings, which will cause him to miss an FA Cup quarterfinal vs. Chelsea and league games against Middlesbrough and West Brom.


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Sports List
Sarlat Rugby
Uamano Ikapoté Fono
Club Sarlat Rugby
Position Flanker
Nationality Tonga
Uamano Ikapoté Fono Life and Career Highlights.
Known simply as Ika by his teammates, Uamano Ikapoté Fono is a Tongan native who has forged a solid reputation in France's fiercely competitive Pro D2 division over an entire decade. Brother to both Siaosi Fono and Ueleni Fono, both well-respected professional rugby players in their own right, there's no doubt about it: Ika has rugby in his blood.
Kicking off his Pro D2 career in 2008, Ika spent his first two seasons in France playing with Stade Aurillacois Cantal Auvergne. Here he made his mark and was extended for a second season before Oyonnax Rugby, another Pro D2 club, recruited him for the 2010/2011 season.
At Oyonnax, Ika again spent two seasons playing with the club, and continued building his reputation as a force to be reckoned with. By the time 2012 rolled around, selectors had taken note, and he was one of the 35 Tongan players preselected for the November tests that year.
From here, Ika would prove unstoppable and continued playing in the Pro D2 division for another six years. Following on from Oyonnax he moved to Stado Tarbes Pyrénées (2012-2014), then Biarritz Olympique (2014-2016), before concluding he Pro D2 career with Soyaux-Angouleme (2016-2018).
But Ika wasn't done yet, and he continues to this day playing in Frances Federal divisions. Following from his time at Soyaux-Angouleme, Ika landed at the Federal 1 club AS Mâcon Rugby (2018-2019), then Villefranche-sur-Saone (2019-2020). Finally, he was recruited by Sarlat Rugby Team where he continues to this day.
Standing at almost 2 m tall (1.95 m to be precise) and pushing 110kg on the scale, Ika is a versatile second and third-row player known for overpowering the opposition. A tower in the line out and a brick wall when tackling, Ika combines his height and strength to formidable effect on the field. Nowhere does this show more than when he takes possession of the ball, crashing through opponents as he advances up the field gaining every precious inch available.
Sarlat Rugby recently finished its first season without a single loss since the club's founding back in 1903, with a perfect 22 wins for 22 games played. The addition of players like Lucas Caneda, Hemani Paea, Ikapote Fono and Dato Okriashvili has turned a team that used to used to play the bottom brackets into a contender qualified for the French Federal 2 championship finals and a brand that has secured a global following, with new fans hailing from every corner of the world.
This performance on the field has attracted the attention of big sports brands like Samurai. Based just across the channel in the U.K., Samurai Sportswear is a leading international kit supplier to sporting clubs from Australia and New Zealand to the English Premiership Rugby division. And, in it has been doing so for the last 25 years. And, in 2021, Samurai came onboard as Sarlat Rugby Team's new kit supplier.
When word that Samurai Sportswear would potentially become the new kit sponsor for the team first spread, there was more than a little enthusiasm from some of the players. The current kit at the time left quite a lot to be desired. And, while not everyone knew who Samurai was at first, many players did.
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Ikapote Fono Sarlat Rugby's Lock
Ikapote Fono Photo Album
Ikapote Fono Social Media Feed
Partners on the field
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Eroni Tuwai
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Manuel Padrón
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Jonathan Laporte
Hemani Paea
Lucas CANEDA
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Our team of experts always keeps attaining your goals at the center of all daily tasks. Every PMI staff member actively participates in group development in order to conceive and manufacture customised metallic structural works perfectly adapted to your needs.
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Dude High Tech
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5 Reasons to Switch to Firefox and Avast Antivirus
ByHigh Tech Team
Firefox and Avast Antivirus are two of the most popular free tools out there, and if you're not using them, you're missing out on some serious technological benefits. Fortunately, Firefox and Avast Antivirus both have solid features that can enhance your browser experience and help protect you from viruses and other cyber threats. Here are five good reasons to switch to Firefox and Avast Antivirus now! 5 Reasons to Switch to Firefox and Avast Antivirus
1) Firefox better than Chrome
2) Protect your PC with Avast Antivirus
3) Save Money on Software
5) Support Freedom of Choice in the Market
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The latest version of Chrome, in addition to having a new look, is also packing some new features. But before you make the switch, here are five reasons why you might want to stick with Firefox:
1. You can still change between tabs by clicking on the tab or using the Ctrl+Tab shortcut. This is one of my favorite features in any browser because it makes switching from one thing I'm working on to another quick and easy without having to click around or open up multiple browser windows at once. 2. Tabs aren't confined to the top row of your screen anymore. Now you can drag them anywhere on your screen for easier access and better multitasking.
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3. As soon as you download Firefox, it's updated to keep you safe from security vulnerabilities that other browsers won't tell you about until they happen – like Chrome will be next month when their first major update comes out.
Avast is a powerful antivirus software that can help protect your PC against the latest viruses and malware. The best part? It's free! Plus, it's so easy to use and has such an intuitive interface – you'll be protected in no time at all.
Here are five reasons why you should switch over to Firefox with Avast Antivirus:
– You'll never have to worry about spyware again with the built-in protection features of Avast. And remember, it's completely free! Plus, it only takes one click for installation on Windows 10 computers, making it even easier than ever before.
Every year, the average person spends $120 on software. This is a huge expense that can be cut down by switching to Firefox with Avast Antivirus.
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#1 – Free Updates: You'll never have to pay again for any updates.
#2 – Free Offers: You'll never have to buy another antivirus offer ever again.
#3 – Great Customer Service: If you do have any problems, customer service is available 24/7. They are helpful and efficient when it comes to your questions. And if you're not satisfied, there's always a 30-day money back guarantee!
4) Keep All Your Data Private
Firefox is an open-source web browser that respects your privacy. Unlike Chrome, they don't use data collection tools or target you with ads based on browsing habits. In addition, it's the only major browser not using Google as its default search engine. You'll also get a free year of antivirus software from avast! If you're looking for a high-quality security product without all the bloatware, then give Firefox and avast a try! The first three reasons are listed below: -Avast offers protection against viruses, spyware, and other malware at no cost. -Avast blocks intrusive advertising while surfing the web so you can browse in peace. -There are no popups or banners to distract you when shopping online–which means less work for me so I can go buy some stuff too 😉
Mozilla is a non-profit organization that believes in the power of an open, inclusive internet. They're committed to building products for and with people who share these values. Their mission includes standing up for the user's right to privacy and having control over their lives online. When you install Firefox, you also get AVAST Free Antivirus software installed automatically. These two companies have joined forces because they believe that user privacy should be just as important as product features. The world has changed, and we want to empower people to take back control. We want them to know how they are being tracked and how data is being used against them. And if they don't like it? We'll offer them another way out: by installing our free anti-virus software on their machines.
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I was a model from age 4-35, but I wasn't the typical model. From age 7-12, I was quite large. My sister in law worked as a buyer for a department store in Pittsburgh, PA and got me jobs modeling for bridal shows. I was always the flower girl. By age 7, I was the cute and chubby side kick in the catalog shoots.
By age 12, the calls for jobs were less and less and this worked on my self esteem. My doctor told my parents I had an unhealthy cholesterol level, and the endocrinologist scared us.
Then from age 12-14 I focused on eating well and learned how to move my body for fun and self love. I took ballet and modern dance for the first time. I got my first ten speed bike. I became the youngest person ever to receive a personal trainer certificate and certified fitness trainer certificate on the East Coast, teaching a myriad of exercise classes at Elaine Powers and Bally's.
In my 40s I was running marathons and again teaching fitness classes while taking care of two children and working evenings serving food in a restaurant. I was perfect in my body proportions, but was 50 pounds heavier than I used to be. I felt like I wanted to have my outside match my inside. I felt so awesome, joyful, and abundant on the inside. I eventually made the food choices to have the outside match my inside. I lost the 50 pounds and am now helping others reach their superior selves with mindfulness, food choices, and movement. I am a research junkie, life and wellness coach, wellness workshop facilitator, author, motivational speaker, and creator of the Superior Self series. If I can do this, you can do this. If I am busy and still make the time for self love and self care every day, so can you.
It is important to me to share my stories with you. Read my older blogs. I am just like you. I still waitress at night to support my family. I help folks with lifestyle changes. It doesn't matter what shape or size one comes to me with as long as they are healthy and ok with themselves at the core. We must believe in the big WHY, use the Negative Thought Pot, and take baby steps toward a healthier life no matter what that looks like for them. It is unique for each individual.


Canada's Citizenship and Immigration Minister Jason Kenney recently announced that henceforth, Muslim women will be prohibited from covering their faces with the niqab during citizenship ceremonies. The move was met with general support and the federal opposition parties have also largely avoided commenting on the issue.
On this International Human Rights Day, I would like to reflect on the courageous role of human rights defenders.
Canadians will gather across the nation to remember the soldiers who paid the ultimate price for their country. We are reminded of the sacrifice of the men and women who fought bravely for peace and justice. We pay tribute to those who have served and who are currently serving on operations today.


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Definition - What does Monel mean?
Monel is a nickel-copper alloy with high tensile strength and resistance to corrosion. The nickel content is as high as 67 percent. The balance of Monel is made up of iron, carbon, silicon and manganese. Monel is used to make pipes, valves and piping supports which are used in trenchless construction.
Trenchlesspedia explains Monel
Monel is stronger than pure nickel and, because of the materials with which the nickel is alloyed, it readily resists the flow of saltwater, called scour. The alloy products in Monel also help prevent galvanic corrosion in a saltwater environment with the use of external sacrificial anodes.
Monel's primary drawback is its cost when used for large-scale applications. In terms of its trenchless technology applications, Monel can cost more than three times what a pipeline made from steel costs which means its uses may be limited. Its chemically resistant properties make it a good choice for applications in the chemical and refining industries where it can withstand the corrosive environment of fluorides.
While Monel readily resists the effects of saltwater scour and saltwater product flows, it shouldn't be used in saltwater applications adjacent to steel in saltwater applications. Galvanic corrosion will destroy Monel used next to or attached to steel unless zinc sacrificial anodes are attached to the Monel and replaced before half the anode dissolves.
One of the many uses of Monel in drilling is to ensure a drill string isn't affected by the Earth's magnetic field. Monel collars are used to isolate drill string instruments from the electromagnet pull of drill bits rotating as they bore a hole. If the tool is deflected by one or two degrees because of the natural magnetic field, the intended track of the bore can miss its intended target by several feet or yards, a potentially embarrassing and costly situation.
Abrasion And Scratch Resistance
Directional Boring (HDD)
Collaring
How does a tunnel boring machine (TBM) control the subsidence of the ground above the trenchless installation?
Pipe JackingOilGasTrenchless Construction
Monal, Monol


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Who Was Chuck Taylor?
From Betty Crocker to Tommy Bahama, plenty of popular labels are "named" after fake people. But one product with a bona fide backstory to its moniker is Converse's Chuck Taylor All-Star sneakers. The durable gym shoes are beloved by everyone from jocks to hipsters. But who's the man behind the cursive signature on the trademark circular ankle patch?
As journalist Abraham Aamidor recounted in his 2006 book Chuck Taylor, All Star: The True Story of the Man behind the Most Famous Athletic Shoe in History, Chuck Taylor was a former pro basketball player-turned-Converse salesman whose personal brand and tireless salesmanship were instrumental to the shoes' success.
Charles Hollis Taylor was born on July 24, 1901, and raised in southern Indiana. Basketball—the brand-new sport invented by James Naismith in 1891—was beginning to take the Hoosier State by storm. Taylor joined his high school team, the Columbus High School Bull Dogs, and was named captain.
After graduation, instead of heading off to college, Taylor launched his semi-pro career playing basketball with the Columbus Commercials. He'd go on to play for a handful of other teams across the Midwest, including the the Akron Firestone Non-Skids in Ohio, before finally moving to Chicago in 1922 to work as a sales representative for the Converse Rubber Shoe Co. (The company's name was eventually shortened to Converse, Inc.)
Founded in Malden, Massachusetts, in 1908 as a rubber shoe manufacturer, Converse first began producing canvas shoes in 1915, since there wasn't a year-round market for galoshes. They introduced their All-Star canvas sports shoes two years later, in 1917. It's unclear whether Chuck was initially recruited to also play ball for Converse (by 1926, the brand was sponsoring a traveling team) or if he was simply employed to work in sales. However, we do know that he quickly proved himself to be indispensable to the company.
Taylor listened carefully to customer feedback, and passed on suggestions for shoe improvements—including more padding under the ball of the foot, a different rubber compound in the sole to avoid scuffs, and a patch to protect the ankle—to his regional office. He also relied on his basketball skills to impress prospective clients, hosting free Chuck Taylor basketball clinics around the country to teach high school and college players his signature moves on the court.
In addition to his myriad other job duties, Taylor played for and managed the All-Stars, a traveling team sponsored by Converse to promote their new All Star shoes, and launched and helped publish the Converse Basketball Yearbook, which covered the game of basketball on an annual basis.
After leaving the All-Stars, Taylor continued to publicize his shoe—and own personal brand—by hobnobbing with customers at small-town sporting goods stores and making "special appearances" at local basketball games. There, he'd be included in the starting lineup of a local team during a pivotal game.
Taylor's star grew so bright that in 1932, Converse added his signature to the ankle patch of the All Star shoes. From that point on, they were known as Chuck Taylor All-Stars. Still, Taylor—who reportedly took shameless advantage of his expense account and earned a good salary—is believed to have never received royalties for the use of his name.
In 1969, Taylor was inducted into the Basketball Hall of Fame. The same year, he died from a heart attack on June 23, at the age of 67. Around this time, athletic shoes manufactured by companies like Adidas and Nike began replacing Converse on the court, and soon both Taylor and his namesake kicks were beloved by a different sort of customer.
Still, even though Taylor's star has faded over the decades, fans of his shoe continue to carry on his legacy: Today, Converse sells more than 270,000 pairs of Chuck Taylors a day, 365 days a year, to retro-loving customers who can't get enough of the athlete's looping cursive signature.
Have you got a Big Question you'd like us to answer? If so, let us know by emailing us at bigquestions@mentalfloss.com.
fashion History News Sports
How Is a Sunscreen's SPF Calculated?
BY Erin McCarthy
Rawpixel/iStock via Getty Images
I'm a pale person. A very pale person. Which means that during these hot summer months, I carry sunscreen with me at all times, and apply it liberally. But I've never really understood what those SPF numbers meant, so I asked some sun care to break it down for me—and to tell me how to best apply the stuff so that I can make it through the summer without looking like a lobster.
Soaking up the sun ... safely
SPF stands for Sun Protection Factor, and it indicates a sunscreen's ability to block UVB rays. The concept was pioneered at the Coppertone Solar Research Center in 1972; in 1978, the FDA published an SPF method based on Coppertone's system, according to Dr. David Leffell, chief of Dermatologic Surgery and Cutaneous Oncology at Yale.
The numbers themselves stand for the approximate measure of time a person who has applied the sunscreen can stay out in the sun without getting burned. Say you get burned after 20 minutes in the sun without sunscreen; if properly applied (and reapplied), SPF 30 will allow you to stay in the sun 30 times longer without burning than if you were wearing no protection at all. So, theoretically, you should have approximately 600 minutes, or 10 hours, in the sun. But it's not an exact science because the amount of UV light that reaches us depends on a number of factors, including cloud cover, the time of day, and the reflection of UV rays off the ground, so it's generally recommended that you reapply sunscreen every two hours (or even sooner).
What gives a sunscreen a higher SPF comes down to the product's formulation. "It's possible that an SPF 50 might contain slightly more of one or more sunscreen active ingredients to achieve that higher SPF," Dr. Patricia Agin, president of Agin Suncare Consulting, says. "But it's also possible that the SPF 50 might contain an additional active ingredient to help boost the SPF performance to SPF 50."
No matter what SPF your sunscreen is, you'll still get a burn if it's not properly applied. So let's go over how to do that.
First, make sure you have a water-resistant, broad spectrum sunscreen—which means that it protects against both UVB and UVA radiation—with an SPF of at least 30. "Typically, you don't have to buy sunscreen that has an SPF higher than that unless you have very sun sensitive skin," Leffell says. "That's a very small percentage of the population." (Redheads, people with light eyes, and those who turn pink after just a few minutes in the sun—you'll want to load up on SPF above 30.)
Twenty minutes before you go out to the beach or the pool, begin to apply your sunscreen in an even coat. "Don't apply it like icing on a cake," Leffell says. "I see these patients and they've got the tops of their ears covered with thick, unevenly applied sunscreen, and that's not a good sign." Sunscreen sprays will easily give you that even coat you need.
Whether you're using lotion or a spray, when it comes time to apply, Leffell recommends starting with your scalp and face, even if you plan on wearing a hat. "Make sure you've covered the ears and nose and under the eyes," Leffell says. "Then, I would move down to the shoulders, and make sure that someone can apply the sunscreen on your back beyond the reach of your hands."
Other areas that are important that you may forget to cover, but shouldn't, are the tops of your feet, the backs of your hands, and your chest. "We see it all the time now—the v of the chest in women has become a socially and aesthetically huge issue when they are 50 and beyond. Because even though they can treat their faces with all sorts of cosmetics and procedures, the chest is much harder, and they are stuck with the face of a 40-year-old and the chest of a 60-year-old. You want to avoid that using sunscreen."
Another important thing to keep in mind: Water-resistant doesn't mean waterproof. "I always tell patients to reapply every couple of hours while you're active outdoors," Leffell says, "and always reapply when you come out of the water or if you've been sweating a lot, regardless of whether the label says water resistant."
Determining whether or not you've succeeded in properly applying your sunscreen is easy: "You know you're applying your sunscreen properly if, after the first time you've used it, you haven't gotten a burn," Leffell says.
Agin has a caveat, though: "It's not a good idea to think of sunscreens only as a way to extend your time in the sun," she says. "One must also understand that even before becoming sunburned, your skin is receiving UV exposure that causes other damage to the skin. At the end of the 600 minutes, you will have accrued enough UV to cause a sunburn—one Minimal Erythema Dose or MED—but there is pre-MED damage done to skin cells' DNA and to the skin's supporting structure of collagen and elastin that is not visible and happens even before you sunburn. These types of damage can occur without sunburning. So you can't measure all the damage done to your skin by only being concerned about sunburn."
Have you got a Big Question you'd like us to answer? If so, let us know by emailing us atbigquestions@mentalfloss.com.
An earlier version of this post ran in 2014.
Big Questions Health News summer
What's the Difference Between Ice Cream and Gelato?
BY Tara Rahimi
iStock/Getty Images/zoff-photo
'Tis the season for beach reads, tan lines, and ice-cold desserts. You know it's summer when going to the local ice cream or gelato shop becomes part of your daily routine. But, what exactly is the difference between these two frozen treats?
One of the key differences between the two is butterfat. While ice cream's main ingredients include milk, cream, sugar, and egg yolks, the secret to making gelato is to use much less cream and sometimes little to no egg yolk. This leads to a much smaller percentage of butterfat in gelato. The FDA rules say that ice cream cannot contain less than 10 percent milkfat (though it can go as high as 25 percent) while gelato, much like soft serve, stays in the 4- to 9-percent range.
The churning method for both also differs, which affects the treat's density. Ice cream is churned at a much faster pace, leading to more air being whipped into the mixture. Ice cream's higher butterfat content comes into play here—due to all of that milkfat, the mix absorbs the air more readily. Gelato, on the other hand, is churned at a slower pace and absorbs far less air, creating a much denser dessert.
You also might have noticed that the serving style for the two treats aren't the same, either. In order to get those perfectly stacked ice cream scoops on a cone, buckets of ice cream must be stored at around 0°F to maintain its consistency, while the softer gelato is stored at a warmer 10°F to 22°F. Ice cream is then scooped into fairly uniform balls with the round ice cream scooper, whereas a spade or paddle is best for molding gelato into mound in a cup or a cone.
You can't really go wrong with either gelato or ice cream on a sweltering summer day, but there is one more difference to keep in mind while you debate which to get: taste. If you want a bolder flavor, you'll want to go with gelato. Because of the density of the cream and because there's less butterfat to coat your taste buds, gelato can seem to have more intensity to its flavors.
Have you got a Big Question you'd like us to answer? If so, send it to bigquestions@mentalfloss.com.
Big Questions Food ice cream News summer


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Julie Gould (born 15 February 1989) is a Welsh swimmer.

Gould was born in Beveren, Belgium. As a member of the Wales team at the 2006 Commonwealth Games, she finished 7th in the 400 m individual medley with a time of 4:51.90, and 8th in the 200 m individual medley with a time of 2:20.61.

References

External links
Personal website

Female medley swimmers
Welsh female swimmers
1989 births
Living people
Swimmers at the 2006 Commonwealth Games
Commonwealth Games competitors for Wales
People from Beveren


Beginning June 1st the class payment schedule will change. The cost per student is $5.00 per class or students can purchase a 10 visit punch card for $30.00. Students must pay $5.00 or have a punch card prior to entering class. There will be NO EXCEPTIONS for non payment. All Students are required to register prior to starting the program by completing a registration form. There is an annual association fee due at sign up for basic and advanced classes. This fee is to be paid to the instructor/sensei. All uniforms must be purchased by the instructor/sensei.
For information on classes and fees please click on the flyer above this message.