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dawn redwood tree uk
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Very young by their own standard, since they live for thousands of years, but they were only discovered and introduced to England in the 1850's. Dawn redwood is a prehistoric relic worthwhile addition to any tree collection. It has proved fast-growing: it was 1.5m tall at the time of the accession of Queen Elizabeth II to the throne in 1952, 17 metres by the time of the Silver Jubilee in 1977, and was approaching 23 metres in the Golden Jubilee year. It is the sole species of its genre. Cones and flowers insignificant, Plant range Unlike most cone-bearing trees, it loses its foliage each winter. All our Dawn Redwood tree sold online come with FREE UK MAINLAND DELIVERY and FREE 3 YEAR warranty on a sliding scale. These plants will have a lot more details displayed including an image. The dawn redwood is a living testimony to the surprises still to be found in nature. As a fast growing tree, growing up to five feet a year, you should transplant it when young. Supplied in Blue Ceramic Bonsai Display Pot 1ft by 5inch by 2inch deep . In 1946, acting on a tip-off and sight of some fragmentary material, Professor Cheng of the National Central University, China, sent an expedition to the remote village of Mo-tao-chi in Szechuan province to collect a complete set of specimens from a stand of recently-discovered, unidentified trees. They now have very restricted natural distributions – the Dawn Redwood to central China and the other two to … the RHS today and get 12 months for the price of 9, Other common names Up until then they were believed to be extinct. ... shops and churches, others in the countryside, again towering over their neighbouring trees. As a fast growing tree, growing up to five feet a year, you should transplant it when young. The dawn redwood is another fast growing, coniferous tree but it is native to China; its existence was not known until the 1940's. eBay. Family Cupressaceae. All trees certified virus free from reputable and long time established UK based fruit and ornamental tree grower and nursery. Divided into 3 groups these lists, linked below, are maintained by a team of RHS staff and are reviewed annually. Metasequoia glyptostroboides-Dawn Redwood; Current price view. A maturing Dawn redwood growing beside the car park of a leisure centre. Trees with multiple trunks are excluded. Info. Created Date Like the Coast redwood and the Giant Sequoia, it belongs to the Sequoioideae – in the Cupressaceae or Cypress family. Dawn Redwood is a deciduous conifer producing small, round 1/2" to 1" cones. More information about the dawn redwood (Metasequoia glyptostroboides). This grows in the Lake and has distinctive knobbly pneumoatophores the 'knees') rising out of the water. Metasequoia glyptostroboides or Dawn Redwood is an outdoor, deciduous tree which has bright green foliage during the summer, becoming tawny pink and gold in autumn before dropping its leaves altogether. The Dawn Redwood, the Coast Redwood and the Giant Sequoia (sometimes called the Sierra Redwood… Unlike most cone-bearing trees, it loses its foliage each winter. 2021 tickets can be booked from 23 December. They are native to China and were only recently discovered in 1944. This site refers to the three types of Redwood by the names commonly used in the U.K: Giant Redwood / Giant Sequoia / Wellingtonia Our Botanical team are working hard to increase the number of plants with detailed information. Fossil records date back to the Mesozoic era and it was thought to be extinct until a small group were discovered by the Yangtze river, China in 1944. In Japan, during 1941, a paleobotanist called Shigeru Miki was looking at fossil sequoias and he noticed a new fossil genus, which he called "metasequoia" (new sequoia). Luckily groups of people helped to collect the seeds which were then planted throughout the world. Redwood trees belong to the subfamily of Sequoioideae in the Cypress family. Seed was collected during a 1947 expedition sponsored by the Arnold Arboretum of Harvard University in Boston, USA, reaching its Director, Professor Elmer D Merrill in 1948, who then distributed the seed amongst botanic gardens in the USA and Europe for cultivation. In the UK it has fast secured itself as a useful large urban tree, as it is tough and can withstand air pollution. The dawn redwood is a coniferous tree that sheds its needles in the fall. It grows best next to water. It is one of the World's most endangered trees. Dawn redwood's preferred soil is very acidic, with a pH of 4.5 being ideal (as is the case with all American redwood species). Like the Coast redwood and the Giant Sequoia, it belongs to the Sequoioideae – in the Cupressaceae or Cypress family. Metasequoia glyptostroboides 'Dawn Redwood'. Giant Redwoods, Coast Redwoods and Dawn Redwoods in the UK - locations with pictures and descriptions. Seal the bag shut and set it on a hard, flat surface. A very large tree with a neat, pyramid crown which is a great choice for urban, avenue or specimen planting. Trees with multiple trunks are excluded. The Dawn Redwood has the familiar conical coniferous shape, with sparse upward sweeping branches giving the tree a narrow form. Girth records In this table of girth records worldwide only girth measurements made at a height between 1.30 m and 1.50 m are listed. Metasequoia glyptostroboides or Dawn Redwood is an outdoor, deciduous tree which has bright green foliage during the summer, becoming tawny pink and gold in autumn before dropping its leaves altogether. The leaves turn to a coppery red colour before falling in late autumn. Although only a relatively new introduction the Dawn Redwood has proven itself to be an excellent performer in UK conditions. Soft flaky cinnamon bark. The dawn redwood is another fast growing, coniferous tree but it is native to China; its existence was not known until the 1940's. Description: A large tree of conical habit. Try an advanced search, RHS Registered Charity no. Dawn redwood. Info. Join A deciduous conifer with lush green spring foliage and a golden yellow hue in autumn. Cold Stream Farm supplies Dawn Redwood trees which are grown as bare root seedlings and transplants and sold both wholesale and retail with no minimum order. The discovery was confirmed in 1946 after the end of the Second World War. METASEQUOIA GLYPTOSTROBOIDES – Dawn Redwood Characteristics The Dawn Redwood has the familiar conical coniferous shape, with sparse upward sweeping branches giving the tree a narrow form. The branches droop dramatically as it grows to present a unique look to the tree. All our Dawn Redwood tree sold online come with FREE UK MAINLAND DELIVERY and FREE 3 YEAR warranty on a sliding scale. Shipping $1.09. Redwood trees comprise three genera; Metasequoia, Sequoia, and Sequoiadendron. Redwoods grow best in temperatures from 20 to 90 degrees. Metasequoia honshuenensis, Family This fast growing tree requires annual or bi-annual repotting. Young twigs sla… Read more RHS Garden Hyde Hall Spring and Orchid Show, Free entry to RHS members at selected Metasequoia is a large deciduous tree with reddish-brown fibrous bark and soft, pale green linear leaves arranged in two ranks on the shoots, colouring beautifully in autumn, Details In a private conservation area with Mortimer Forest and wood/farm lands. It features an upright, narrowly conical form that enhances many landscapes. The dawn redwood tree was long thought to be extinct until it was rediscovered in 1941 by Mr. T. Kan while performing a survey in the Hubei and Sichuan provinces of China. The redwood's native habitat is warm and humid. Origin: Prior to its discovery in the Hupeh Province of China in 1941, it was only known as a fossil.It was introduced to the UK in 1948 and is now frequently planted in parks and large gardens. The strong pyramidal winter silhouette is also very pleasing, making the Dawn Redwood a strong contender for year-round interest. Thought to be extinct, it was found growing in a small grove in China in the middle of the last century. Dawn Redwood trees are straight, tall deciduous conifers originating from China. Metasequoia glyptostroboides 'Gold Rush' (Dawn Redwood) is a large deciduous coniferous tree of great beauty. Tolerant of waterlogged soils. The Dawn Redwood makes a fine tree now familiar in parks and gardens. This tree, which can grow to heights of 100 feet or more, has a tapered trunk with shredded bark. One of the most exciting discoveries in the plant world during the last century was undoubtedly that of the deciduous conifer, the Dawn Redwood, Purchase tickets for Cambridge University Botanic Garden. S & H: see site. New species are frequently discovered, although few new plant species capture people's imaginations. Amber Glow is an interesting cultivar of the Dawn Redwood, with yellow spring foliage tinged with burgundy, which turns orange in autumn before falling. Clissold Park, N4, 2012. The Dawn Redwood was first described in Japan in 1941 by Shigeru Miki, who thought he had a discovered the fossils of a tree long since extinct. If buying from October onwards please note the leaves will be more yellow/orange before shedding. Free shipping. A man demonstrates the girth size of a Redwood tree (sequoia), by the Monmouthshire and Brecon Canal near Llangattock, Brecon Beacons, Wales, UK Metasequoia glyptostroboides. Dawn Redwood Metasequoia glyptostroboides. It has a neat pyramidal shape in youth, maturing into a more rounded crown. Today, only 9 genera and 15 species exist. The Dawn Redwood is a good choice for bonsai as it can be trained into most styles, especially informal upright styles. The Dawn Redwood, the Coast Redwood and the Giant Sequoia (sometimes called the Sierra Redwood… Dawn Redwood Tree in Pot Large Outdoor Hedge Plant Conifer Bright Green Foliage. The Dawn redwood is an endangered species so additional care should be taken by owner land owners lucky enough to have them. Luckily groups of people helped to collect the seeds which were then planted throughout the world. An ancient tree that knew the dinosaurs but is well-suited to modern landscape plantings. The Dawn Redwood has the familiar conical coniferous shape, with sparse upward sweeping branches giving the tree a narrow form. This tree on the south-western edge of the Lake, then, is the first RHS members can get exclusive individual advice from the RHS Gardening Advice team. The beautiful ferny foliage is a bright, light green in spring, turning russet-brown with tints of coppery pink in autumn. Dawn Redwood . They are relics from a period 100 million years ago when they were widespread. One discovery that eventually captivated people was of a tall, deciduous, coniferous tree collected by Chinese foresters in East Sichuan (now Chongqing) in 1941. Metasequoia glyptostroboides – Dawn redwood. To give you an idea of just how acidic this … 14 inches inc. pot . The Dawn redwood is an endangered species so additional care should be taken by owner land owners lucky enough to have them. Low Maintenance, Diseases Dawn Redwood Bonsai Tree Part Trained 40cm Height. Metasequoia Dawn Redwood Bonsai Trees are an ancient species of tree, dating back many millions of years. This site refers to the three types of Redwood by the names commonly used in the U.K: Giant Redwood / Giant Sequoia / Wellingtonia trade customers: 01584 810214 enquiries@fpmatthews.co.uk GREEN NEEDLES IN SPRING, GOLDEN NEEDLES IN AUTUMN! The Dawn redwood is a prolific grower, dating back to the time of the dinosaurs when it populated regions in thick redwood forests. Minimum temperature ranges (in degrees C) are shown in brackets, Aspect South-facing or West-facing or North-facing or East-facing, MoistureMoist but well-drained, Poorly-drained, Ultimate height Grow in moist but well-drained soil. The Garden is closed 24 December–1 January, reopening 2 January 2021. It is one of the World's most endangered trees. eBay. Metasequoia glyptostroboides Hu & Cheng (Cupressaceae). Generally it is best to ensure that Dawn Redwood has plenty of room, planted in an estate, parkland or very large garden. 222879/SC038262, If you enter just a plant name, you will see results from the old RHS Plant Finder and Selector databases. The dawn redwood is a fabulous landscape or city tree with fresh green, soft needles in spring and foxy reddish brown needles in fall. Girth records In this table of girth records worldwide only girth measurements made at a height between 1.30 m and 1.50 m are listed. It is included in an evolving list of plants carefully researched and chosen by RHS experts. The branches droop dramatically as it grows to present a unique look to the tree. The leaves are feathery, a lot like ferns, and they cover the lofty branches. Growing in Cambridge University Botanic Garden, Britain's first Dawn Redwood ( Metasequoia glyptostroboides) is a fascinating tree, not least because of the way in which the species was discovered. Metasequoia glyptostroboides, the dawn redwood, is a fast-growing, endangered deciduous conifer, the sole living species of the genus Metasequoia, one of three species in the subfamily Sequoioideae. A robustly growing deciduous conifer with a narrow pyramidal crown at first that becomes broad pyramidal later. All trees certified virus free from reputable and long time established UK based fruit and ornamental tree grower and nursery. The bright green, feathery leaves … Trees should be securely staked to prevent them from rocking and … M. glyptostroboides is a vigorous deciduous conifer making a large, narrowly conical tree to 25m, with flat sprays of narrow, pale green leaves that turn brownish-pink and yellow in autumn. The photos show the … This is an Outdoor tree but will Metasequoia glyptostroboides - Dawn Redwood Metasequoia glyptostroboides is an ancient yet also relatively new tree. ... treeshop@fpmatthews.co.uk. The Garden's specimen tree by the Lake has a superb, rugged, ridged trunk unusual in cultivation. This subfamily is considered to be endangered, largely due to air pollution, climate change, and fire. We have combined these two powerful search tools into a single Find a Plant service searching over 250,000 plant records. Close-up of the fruit of the Dawn redwood, actual size 25mm long. 12.95: Available: 2154J: Dawn Redwood - Metasequoia glyptostroboides Young plug plant – Height of plant: 20/30 cm. Wildlife: The Dawn Redwood provides winter cover for birds, small mammals and deer. Dawn redwood bonsai tree against a blue background on display at a flower show. AGM plants have been through a rigorous trial and assessment programme. They turn shades of red and brown in autumn before falling; this is one of the few deciduous conifers. Branches grow in a regular pattern on the perfectly vertical main trunk. Free shipping. This tree, which can grow to heights of 100 feet or more, has a tapered trunk with shredded bark. It has a neat pyramidal shape in youth, maturing into a more rounded crown. The species is classified by the World Conservation Union as 'critically endangered' due to intensive rice cultivation and the poor prospect for regeneration in the wild. It is perfectly happy on any reasonable soil - particularly useful on constantly moist ground - and is fully hardy. Dawn Redwood Bonsai Tree Part Trained 40cm Height. Stock Status In Season: In Season and available to ... Take care not to over or under water - you can tell by feeling the soil to see if it is dry. Metasequoia glyptostroboides Dawn Redwood Common Name: Dawn Redwood Botanical Name: Metasequoia glyptostroboides Mature Height: 20m+ Key Features: Interesting deciduous conifer; v ibrant feathery foliage, colourful autumn display, attractive bark, excellent performer in range of sites. Synonyms Metasequoia honshuenensis. suppliers. The Dawn Redwood, thought of as a 'living fossil', is a deciduous conifer, the leaves of which turn tawny pink and old gold in autumn. It was judged one of the 50 'Great British Trees' in honour of the Queen's Golden Jubilee in 2002 and features in Thomas Pakenham's Meetings with Remarkable Trees (1996). $1.99. More information about the dawn redwood (Metasequoia glyptostroboides). This tree on the south-western edge of the Lake, then, is the first Dawn Redwood to grow on British soil since the Mesozoic era, when dinosaurs dominated the fauna. . They are: This plant will provide nectar and pollen for bees and the many other types of pollinating insects. Metasequoia glyptostroboides (Dawn Redwood) Redwood. The leaves which are short, broad needles, could be mistaken for those of the Yew. To help Dawn Redwood trees establish more effectively, we advise applying root grow by sprinkle in the hole. Yes, the bark on the trunk is an asset and you can prune the lower branches or limb up the tree to get this benefit. Discovered Alive: The Story of the Chinese Redwood. times, Need more criteria? Join the RHS today and support our charitable work, Keep track of your plants with reminders & care tips – all to help you grow successfully, For the latest on RHS Shows in 2020 and 2021, read more, RHS members get free access to RHS Gardens, Free entry to RHS members at selected times », Reduced prices on RHS Garden courses and workshops, General enquiries Dawn Redwood is a deciduous conifer producing small, round 1/2" to 1" cones. I don't think you would ever see root knees on a Dawn Redwood. It is not so easy to grown them from seeds, but why would you want to do that effort when you can do it much more easy: the dawn redwood is … $38.99. Beautiful Dawn Redwood Group, Compiled from 9 Trees, in Blue Ceramic Bonsai Pot. Commonly known as Dawn redwood, ... Conical tree with ascending branches and fibrous orange bark which prefers a boggy or wet area. Higher than 12 metres, Ultimate spread They are native to China and were only recently discovered in 1944. Britannia Leisure Centre, N1, 2012. It grows best next to water. Several Dawn redwoods planted at a newly constructed crossing together with other species. Mon – Fri | 9am – 5pm, Join the RHS today and support our charity. You might see them on a Bald Cypress if it is in a wet situation. We aim to enrich everyone's life through plants, and make the UK a greener and more beautiful place. Dawn redwood grows quickly into a large tree with a pyramidal shape and base that forms a wide flare. To propagate your own, simply take some seed cones and a bunch of the debris found at the base of a redwood; be sure to measure the depth of the duff under the tree, and use that as your duff depth. It grows most successfully in warmer climes and so is seen more commonly in southern areas of the UK. The grass green summer foliage turns pink and golden brown in the autumn before falling. A useful large urban tree, which can grow to heights of 100 feet or more has... Was found growing in a private conservation area with Mortimer Forest and wood/farm lands supplied in Blue Ceramic Bonsai.. Redwood a strong contender for year-round interest a genus of fast-growing deciduous trees it. Names Dawn Redwood, climate change, and they cover the lofty branches species exist,! And can withstand air pollution, climate change, and make the UK - with... 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OFF-COURT ACTION JUST AS LIVELY FOR FANS
Neil Santaniello, Staff WriterSUN-SENTINEL
As many as 450 members of the press, including reporters and photographers from a half-dozen countries, stalked their quarry on and off the main court at the Lipton International Players Championship tennis tournament Saturday.
Armed with a Nikon camera, Gary Sion, too, stalked tennis stars and others during the afternoon match between Chris Evert-Lloyd and Steffi Graf on the grounds of the Boca West Development, which hosted the tournament.
But he did not have to meet a daily deadline.
The high school student snapped several pictures for a photo essay he had to submit for a journalism class.
The idea for the class project was his and stemmed from his passion for tennis, the 15-year-old said.
"I got Chrissie and Stef coming in," the Sunrise youth said, smiling broadly. "I said 'Hi,' they looked up and I got their pictures. I also came here to capture the action and atmosphere."
Others at the tournament also sought a piece of action Saturday, but not the kind that transpires on the court.
During the Lloyd-Graf duel, merchants in booths outside the grand stand hawked cellular telephones, autographed glass etchings of players, cruises on ships and hair and skin conditioner.
The man selling the telephones, Everett Wilson, said the tournament is a prime place to market the line of phones priced from $1,300 to $2,600.
"The clients here are more affluent and successful than I find at a golf match," he said. He said jet setters like stock brokers and businessmen who attend such tournaments are big customers.
He said he sold six phones since the tournament began, including one to Boris Becker's manager, and plans to sell more.
Concession stands also tried to make a profit. They offered a variety of food that went beyond the typical frank-and-a-beer ballpark offering.
A hamburger stand dishing out charcoal-broiled burgers with all the fixings stood beside a New York-style deli touting bagel and lox, smoke ham and brie and crabmeat and seafood sandwiches.
VIP tents and others labled "Private" served up more sumptuous offerings amid a setting of white tablecloths and potted plants.
"The food is quite good, better than last year's," said Carol Lunceford, a tournament staff supervisor.
The afternoon's heat -- temperatures hit the mid-80s -- drove many spectators to seek cover.
Maurice and Marcie Semensohn, New Yorkers in town visiting their three grandchildren in Coral Springs, came to Boca West to catch the Lloyd-Graf match but retreated from their seats to a patio table umbrella outside the grandstand where they sipped ice water.
"I can't take the heat," said Marcie Semensohn, who left behind 30-degree temperatures in the Big Apple. "I thought we would just take cover."
She said she enjoyed what she had seen of the tournament, and added, "I don't blame (Jimmy) Connors," refering to the tennis star's display of anger and subsequent withdrawal from the tournment Friday to protest a referee's call that went against him.,
Maurice Semensohn said the staging of the tourment created its only short- coming.
"I don't think Boca West is equipped to handle a tournament of this scale," he said.
Buses and trolleys ferried people back and forth from the stadium to parking lots to reduce traffic congestion inside the sprawling residential complex.
Three paramedics stationed at a white medical tent just outside the players' entrance said they did not treat any cases of sunstroke or heat-induced fainting spells.
"We've only had one person come in and lay down briefly," said Chuck Gardener, who manned a Palm Beach County Fire-Rescue golf cart painted fire- truck red and equipped with advanced life support systems.
He said players rarely came to paramedics if hurt while playing a set.
"When you got the amount of money they do invested in your arms and legs, you won't let just anyone touch them," he said.
High heels, however, had been posing a problem on the wood-chip grounds surrounding the main tennis stadium, said another paramedic, Terry Adkins. The soft turf caused a number of falls, though nothing serious, he said.
Pat Osborne, a first-time tennis tournament usher at one of the gates, said she volunteered for the job to do some people-watching and a little star- watching because she had never worked at a major tennis tournment before.
"It's fun; the whole atmosphere is fun. You get to meet players," said Osborne, who resides in a condominium complex nearby and who put in 80 hours of her own time directing people to their seats in the 13,000-capacity stadium.
Another usher, Judy Frampton, said the job had certain incentives.
"I met a lot of cute guys," the young woman said, laughing. One drawback she noted: Dealing with verbal abuse.
She said that despite the tournment's setting at the exclusive Boca West development, she had concluded most spectators were not particularly well- heeled or jet-setting.
Rather, "They are just regular shleps like us," she said.
Both ushers were among 400 volunteers who pledged their time to help the tournament in its second year (Lavers International Resort in Delray Beach hosted it the first year) run smoothly.
But no one seemed to mind working without compensation amid the chance to see world-renowned tennis stars in action.
Kids included.
Kathi Bond, a sophomore at Spanish River High School and one of 45 courtside ball boys and girls, said the tedium of standing by a baseline waiting to pounce on an out-of-play play had its advantages.
The official ball retrievers' uniforms they wore -- cap, T-shirt, windbreaker, shorts and sneakers -- are complimentary and can be taken home, she said.
She said her mother, a volunteer coordinator, recruited her, but with little difficulty because of the free outfit and the fact that "it sounded like fun."

redpajama_set_name: RedPajamaCommonCrawl


The way I feel about industrial pieces is not unlike the way I feel about jewelry. A few key pieces are unique, hip, and interesting. Add too many and you've ruined the whole look and turned into a walking hot mess version of Grandma. Adding just the right piece of vintage industrial, or urban industrial, or according to Big D...junk yard industrial, can turn an old piece of the past into a statement piece that anchors a room.
Like a kitchen. These industrial pendants are everywhere, which pretty much means I won't want them anymore. But, they really do look great in this room, don't they?


I have been involved in the evolution of mineral aggregate policy in the Town of Caledon since the first Official Plan in 1979 and the first Provincial Policy Statement.
A constant observation of the gravel strip mining industry has been the lack of any social compact with the host community. Profit is the metric of the industry. There has been and continues to be no short or long-term benefit to the host community or natural environment of a gravel strip mine. Benefits accrue to the strip mine owners and the larger GTA economy, not affected landowners who experience the loss of the enjoyment and use of their lands, threats to their safety by heavy truck traffic and health impacts.
A second constant observation is the lack of any identifiable oversight of individual strip mine operations by the Ministry of Natural Resources. References to Aggregate Resource Act licences and site plans serve a useful rhetorical crutch at approval stages to support massive, permanent changes to the Caledon landscape and communities without any termination date for mining activities.
To whom is the Town of Caledon responsible? Who is the Town supposed to represent? And who/what is it supposed to protect residents from?
And if anyone dares to speak up and ask questions, they are deemed to oppose council and its staff and summarily demonized as "grandstanders," etc. Council members assume their powers on the basis that they are best qualified to know what's best for the public, even when the public thinks differently and provides factual backup.
During the Dec. 9 council meeting, the Caledon resident delegations were professional, their numbers in attendance were high, overflowing to the cafeteria, and they took the high road. Council failed to ask key questions of the proponents and staff regarding the strip mine impacts and failed to reflect in any substantial way, the residents. Overall, council exhibited a closed mind as they voted to approve the new strip mine. What has become clear by the consistent behaviour of Caledon council is a feeling that local politics, even after courteous, professional delegations by residents, is not responding to resident's needs in terms of the enjoyment and use of their lands, threats to their safety by heavy truck traffic and health impacts.


As of this post, exactly 10 days (240 hours; 14400 minutes; 864000 seconds) remain until the start of Londonvasion, the first ever live phpBB conference. If you are planning to attend and have not yet registered, then you should do so as soon as possible. Do not wait any longer!
Londonvasion 2008 is the first ever live phpBB conference. We are very excited to present you with this unique opportunity to meet fellow community members, as well as the phpBB team.
The conference will be held on Sunday July 20th in a hotel across the street from London Heathrow Airport (UK). Doors will open at 9:00 AM and the event is scheduled to end at around 17:00 (5 PM). Presentations will be made throughout the event, and time will be provided for live Q&A.
The conference will be held at the Radisson Edwardian Heathrow Hotel which is located directly across from London Heathrow Airport (LHR). There are around a dozen other hotels in the area, so you should have no problem finding a room if necessary.
If you plan on using the London Underground for transportation, the closest station also happens to be at LHR. Travel from the airport and Underground station to the hotel is available via a £4 shuttle bus which runs every 15 minutes. The shuttle also stops at the other hotels located on Bath Rd and can therefore be used to travel between hotels ? make sure to confirm this when booking. Alternatively, taxi fare from the airport is around £7.
Community members who reside in London may be able to provide travel tips, so please feel free to ask in the discussion topic.
Several methods of live advertising are still available. If you are interested, please contact me for more information via any of the methods listed in my profile.
Wifi will be available, so you are welcome to bring any portable internet capable device. Cameras (photo, video) will be allowed.
If you bring your Bertie, make sure that he is marked in some recognisable way and differs from all the others.
On site parking is available for a fee of £10 per day. This price may differ slightly depending on the amount of space.
The hotel will be setting up a cafeteria with very reasonably priced options in the 3rd floor atrium. Items will include sandwiches of various fillings, tea, coffee, pastries, desserts and other snacks.
Important updates will be posted as replies to this topic and also emailed to all confirmed attendees. You may also wish to see the discussion topic for answers to specific questions.
Please read this post in its entirety before proceeding with registration.
There is a registration fee of £10 per person for the event. This is an unavoidable measure to prevent bogus bookings and the reselling of tickets, which is strictly prohibited. To further prevent "ticket scalping", each paid registration will have an id, name and email associated with it. All proceeds will be used to offset the cost of the event.
You can register for the event by filling out the following form: http://www.phpbb.com/londonvasion/. Upon submitting the form, you will be redirected to Paypal where you can pay the registration fee. You will receive a confirmation PM when your registration has been processed and confirmed. At this time, you will also see a confirmation page in place of the form. If you do not receive a confirmation PM, then your registration has not been confirmed. The system will only accept one registration per username, and the username will be linked to the ticket.
1) are unable to use Paypal.
2) need to register multiple tickets or a ticket for someone else.
3) do not receive a confirmation PM after payment.
4) experience any other problems.
Londonvasion will take place in the "Commonwealth" suite, which can be reached by following the signs from the lobby. We hope to start the event at around 9:00 AM. Go up the grand staircase in the lobby and over the water garden (which is directly above the lobby). The door you come to is the entrance to the suite. Again, there will be signs with arrows to indicate this path.
A badge will be given to you upon entering the event. All you need to bring is an ID or printout of the registration confirmation.
If you have not purchased a ticket, they will be available at the door for £10.


text:
About Lambton
Welcome to Sarnia-Lambton
Aamjiwnaang First Nation
City of Sarnia
Town of Petrolia
Town of Plympton Wyoming
Township of Brooke-Alvinston
Township of Dawn-Euphemia
Township of Enniskillen
Township of St. Clair
Township of Warwick
Village of Oil Springs
Village of Point Edward
Walpole First Nation
Music and Festivals
Child Care and Child Care Centres
Cultural Groups
Publicly Funded School System
Government In Canada
Immigrating to Sarnia Lambton
Permanent Resident (PR) Card
After You Arrive
Local Immigration Partnership
LIP Implementation Plan
Settlement and Language
Coordination of Services
LIP Council
LIP Settlement Strategy
Anti-Racism Campaign
Township of Dawn-EuphemialiveinlambtonJuly 24, 2013 March 13, 2018
Living in Dawn Euphemia
Dawn- Euphemia
Welcome to Dawn-Euphemia, a friendly and caring community, where residents feel a strong sense of attachment, community spirit and pride.
The Township of Dawn-Euphemia has a land area of 445 square kilometres and is located in the south east portion of Lambton County. The Township is bordered on the north by the Township of Enniskillen and the Municipality of Brooke-Alvinston, the east by the Municipality of Southwest Middlesex, the south by the Municipality of Chatham-Kent, and the west by the Township of St. Clair. The Township is characterized by many scenic roads and the meandering Sydenham River that bisects the Township.
The Township has developed around an economy based primarily on agriculture, particularly cash cropping and boosts a thriving agriculture based businesses that service the agricultural community including Cargill, Southwest Ag and Haggerty Creek.
The Township is the home to the Union Gas Dawn Compressor Station and underground storage pools and is known as the largest natural gas compressor station in North America (as measured by horsepower).
Within the Township are a number of small hamlets including communities of Florence, Shetland, Oakdale, Edys Mills, Rutherford, Cairo and Bentpath. One elementary school, known as the Dawn-Euphemia Public School, serves the area.
The Township provides a number of recreational/social facilities including the Florence Fairgrounds that supports the Dawn-Euphemia Community Centre – an 8,600 square foot, multi-purpose facility that comprises the Florence Library, a meeting room, a community hall and a state of the art kitchen. The Fairgrounds also support two ball diamonds; a children's playground, a tractor pull track, a picnic pavilion and an outdoor rink. Just east of the Hamlet of Shetland, on the north side of Bentpath Line, is the Shetland Conservation Area that comprises a fourteen acre park that is bisected by the Sydenham River and is popular with campers and canoeists who admire and appreciate the scenery and the quietness of the region. The Shetland Conservation Area provides 16 serviced campsites and 15 unserviced campsites, a pavilion for picnics and dock access to the Sydenham River for canoes. The Shetland Conservation Area also hosts the annual St. Clair Region Conservation Area Sydenham Canoe Race in April.
The Township is proud of the service clubs and local organizations that organize and host annual community events including Grand Ole Days, Holiday in the Hamlet, the Canada Day Barbecue, a tractor pull and the Family Fall Festival.
www.dawneuphemia.ca
Industry and Community Highlights
Average Maximum
Average Minimum
Summer 26°C 16°C
Winter -1°C -10°C
Average time to Toronto 3h 1 min
Average time to Detroit 1h 44 min
Housing Costs
House $263,400
US 1.00%
UK 2.00%
Mexico 3.00%


The latest release v0.4.4, that was made available yesterday adds in some interesting new features. A pretty important one, is the new "relative joystick camera" mode, which acts just like the Joystick Camera mode on Steam. Some games (like twin-stick shooters) don't always hold the position of your thumb on the right pad to continually fire, this mode should fix it for games where it doesn't work as expected.
You can also now map triggers to mouse movement, the trackball mode has been extended so it can now be combined with more output modes, an optional tolerance to gesture recognition, an option to map tilting on yaw axis and also per-controller configuration for default menu buttons.
It also fixes PS4 controller compatibility on Kernel versions 4.17+, enables controlling menus with the PS4 D-pad, blacklists some gamepads that have issues with HID and more.
Just like the dxvk guy, this kozec is one hell of a guy. He churns out feature after feature of this excellent product. He surely deserves praise and sponsoring.
Is it using upstream kernel driver for Steam controller now?
It probably means simply the hardware driver. To replace Steam, you still need something to upload profiles and other things that Steam does. That's what SC controller should be able to help with if I understand correctly.
This is an awesome tool for the Steam Controller. It helped me getting starting with all the various settings of the Steam Controller. Of course it's also possible to use the big picture mode of Steam, but this tools makes it much more easy to fiddle around.
Everybody who thinks the Steam Controller is hard to setup, try this tool instead of Steam's Big Picture mode.
Corben Everybody who thinks the Steam Controller is hard to setup, try this tool instead of Steam's Big Picture mode.
Also the main value is for those who aren't using Steam.
Has anyone gotten Dual Shock 3 controllers to work Sc controller? It sees my controller, but does not detect any button presses.
Is it? Because sc-controller seems to be the only way to make the steam controller work in wine steam.
I would say that in the beginning of sc-development its appeal was to better use the controller outside steam, yes. But as of today, he became so powerful that it's a vastly superior alternative to the native steam driver and configuration. I always launch it before launching steam so I can use sc-controller, and not the steam driver, to play my games. Heck, I have been using sc-controller even with FreeCAD, to do 3d modelling!


Naproxen as NAPROSYN, EC-NAPROSYN, ANAPROX, ANAPROX DS or NAPROSYN Suspension is contraindicated for the treatment of peri-operative pain in the setting of coronary artery bypass graft (CABG) surgery (see WARNINGS).
Naproxen has a molecular weight of 230.26 and a molecular formula of C14H14O3. Naproxen sodium has a molecular weight of 252.23 and a molecular formula of C14H13NaO3.
NAPROSYN (naproxen tablets) is available as yellow tablets containing 250 mg of naproxen, pink tablets containing 375 mg of naproxen and yellow tablets containing 500 mg of naproxen for oral administration. The inactive ingredients are croscarmellose sodium, iron oxides, povidone and magnesium stearate.
EC-NAPROSYN (naproxen delayed-release tablets) is available as enteric-coated white tablets containing 375 mg of naproxen and 500 mg of naproxen for oral administration. The inactive ingredients are croscarmellose sodium, povidone and magnesium stearate. The enteric coating dispersion contains methacrylic acid copolymer, talc, triethyl citrate, sodium hydroxide and purified water. The dissolution of this enteric-coated naproxen tablet is pH dependent with rapid dissolution above pH 6. There is no dissolution below pH 4.
ANAPROX (naproxen sodium tablets) is available as blue tablets containing 275 mg of naproxen sodium and ANAPROX DS (naproxen sodium tablets) is available as dark blue tablets containing 550 mg of naproxen sodium for oral administration. The inactive ingredients are magnesium stearate, microcrystalline cellulose, povidone and talc. The coating suspension for the ANAPROX 275 mg tablet may contain hydroxypropyl methylcellulose 2910, Opaspray K-1-4210A, polyethylene glycol 8000 or Opadry YS-1-4215. The coating suspension for the ANAPROX DS 550 mg tablet may contain hydroxypropyl methylcellulose 2910, Opaspray K-1-4227, polyethylene glycol 8000 or Opadry YS-1-4216.
NAPROSYN (naproxen suspension) is available as a light orange-colored opaque oral suspension containing 125 mg/5 mL of naproxen in a vehicle containing sucrose, magnesium aluminum silicate, sorbitol solution and sodium chloride (39 mg/5 mL, 1.5 mEq), methylparaben, fumaric acid, FD&C Yellow No. 6, imitation pineapple flavor, imitation orange flavor and purified water. The pH of the suspension ranges from 2.2 to 3.7.
Naproxen and naproxen sodium are rapidly and completely absorbed from the gastrointestinal tract with an in vivo bioavailability of 95%. The different dosage forms of NAPROSYN are bioequivalent in terms of extent of absorption (AUC) and peak concentration (Cmax); however, the products do differ in their pattern of absorption. These differences between naproxen products are related to both the chemical form of naproxen used and its formulation. Even with the observed differences in pattern of absorption, the elimination half-life of naproxen is unchanged across products ranging from 12 to 17 hours. Steady-state levels of naproxen are reached in 4 to 5 days, and the degree of naproxen accumulation is consistent with this half-life. This suggests that the differences in pattern of release play only a negligible role in the attainment of steady-state plasma levels.
EC-NAPROSYN is designed with a pH-sensitive coating to provide a barrier to disintegration in the acidic environment of the stomach and to lose integrity in the more neutral environment of the small intestine. The enteric polymer coating selected for EC-NAPROSYN dissolves above pH 6. When EC-NAPROSYN was given to fasted subjects, peak plasma levels were attained about 4 to 6 hours following the first dose (range: 2 to 12 hours). An in vivo study in man using radiolabeled EC-NAPROSYN tablets demonstrated that EC-NAPROSYN dissolves primarily in the small intestine rather than in the stomach, so the absorption of the drug is delayed until the stomach is emptied.
When EC-NAPROSYN was given as a single dose with antacid (54 mEq buffering capacity), the peak plasma levels of naproxen were unchanged, but the time to peak was reduced (mean Tmax fasted 5.6 hours, mean Tmax with antacid 5 hours), although not significantly.
When EC-NAPROSYN was given as a single dose with food, peak plasma levels in most subjects were achieved in about 12 hours (range: 4 to 24 hours). Residence time in the small intestine until disintegration was independent of food intake. The presence of food prolonged the time the tablets remained in the stomach, time to first detectable serum naproxen levels, and time to maximal naproxen levels (Tmax), but did not affect peak naproxen levels (Cmax).
Naproxen has a volume of distribution of 0.16 L/kg. At therapeutic levels naproxen is greater than 99% albumin-bound. At doses of naproxen greater than 500 mg/day there is less than proportional increase in plasma levels due to an increase in clearance caused by saturation of plasma protein binding at higher doses (average trough Css 36.5, 49.2 and 56.4 mg/L with 500, 1000 and 1500 mg daily doses of naproxen, respectively). The naproxen anion has been found in the milk of lactating women at a concentration equivalent to approximately 1% of maximum naproxen concentration in plasma (see PRECAUTIONS: Nursing Mothers).
The clearance of naproxen is 0.13 mL/min/kg. Approximately 95% of the naproxen from any dose is excreted in the urine, primarily as naproxen (<1%), 6-0-desmethyl naproxen (<1%) or their conjugates (66% to 92%). The plasma half-life of the naproxen anion in humans ranges from 12 to 17 hours. The corresponding half-lives of both naproxen's metabolites and conjugates are shorter than 12 hours, and their rates of excretion have been found to coincide closely with the rate of naproxen disappearance from the plasma. Small amounts, 3% or less of the administered dose, are excreted in the feces. In patients with renal failure metabolites may accumulate (see WARNINGS: Renal Effects).
In pediatric patients aged 5 to 16 years with arthritis, plasma naproxen levels following a 5 mg/kg single dose of naproxen suspension (see DOSAGE AND ADMINISTRATION) were found to be similar to those found in normal adults following a 500 mg dose. The terminal half-life appears to be similar in pediatric and adult patients. Pharmacokinetic studies of naproxen were not performed in pediatric patients younger than 5 years of age. Pharmacokinetic parameters appear to be similar following administration of naproxen suspension or tablets in pediatric patients. EC-NAPROSYN has not been studied in subjects under the age of 18.
Studies indicate that although total plasma concentration of naproxen is unchanged, the unbound plasma fraction of naproxen is increased in the elderly, although the unbound fraction is <1% of the total naproxen concentration. Unbound trough naproxen concentrations in elderly subjects have been reported to range from 0.12% to 0.19% of total naproxen concentration, compared with 0.05% to 0.075% in younger subjects. The clinical significance of this finding is unclear, although it is possible that the increase in free naproxen concentration could be associated with an increase in the rate of adverse events per a given dosage in some elderly patients.
Naproxen pharmacokinetics has not been determined in subjects with renal insufficiency. Given that naproxen, its metabolites and conjugates are primarily excreted by the kidney, the potential exists for naproxen metabolites to accumulate in the presence of renal insufficiency. Elimination of naproxen is decreased in patients with severe renal impairment. Naproxen-containing products are not recommended for use in patients with moderate to severe and severe renal impairment (creatinine clearance <30 mL/min) (see WARNINGS: Renal Effects).
In 51Cr blood loss and gastroscopy studies with normal volunteers, daily administration of 1000 mg of naproxen as 1000 mg of NAPROSYN (naproxen) or 1100 mg of ANAPROX (naproxen sodium) has been demonstrated to cause statistically significantly less gastric bleeding and erosion than 3250 mg of aspirin.
Carefully consider the potential benefits and risks of NAPROSYN, EC-NAPROSYN, ANAPROX, ANAPROX DS or NAPROSYN Suspension and other treatment options before deciding to use NAPROSYN, EC-NAPROSYN, ANAPROX, ANAPROX DS or NAPROSYN Suspension. Use the lowest effective dose for the shortest duration consistent with individual patient treatment goals (see WARNINGS).
EC-NAPROSYN is not recommended for initial treatment of acute pain because the absorption of naproxen is delayed compared to absorption from other naproxen-containing products (see CLINICAL PHARMACOLOGY and DOSAGE AND ADMINISTRATION).
NAPROSYN, EC-NAPROSYN, ANAPROX, ANAPROX DS and NAPROSYN Suspension are contraindicated in patients with known hypersensitivity to naproxen and naproxen sodium.
NAPROSYN, EC-NAPROSYN, ANAPROX, ANAPROX DS and NAPROSYN Suspension should not be given to patients who have experienced asthma, urticaria, or allergic-type reactions after taking aspirin or other NSAIDs. Severe, rarely fatal, anaphylactic-like reactions to NSAIDs have been reported in such patients (see WARNINGS: Anaphylactoid Reactions and PRECAUTIONS: Preexisting Asthma).
NAPROSYN, EC-NAPROSYN, ANAPROX, ANAPROX DS and NAPROSYN Suspension are contraindicated for the treatment of peri-operative pain in the setting of coronary artery bypass graft (CABG) surgery (see WARNINGS).
There is no consistent evidence that concurrent use of aspirin mitigates the increased risk of serious CV thrombotic events associated with NSAID use. The concurrent use of aspirin and an NSAID does increase the risk of serious GI events (see WARNINGS: Gastrointestinal Effects – Risk of Ulceration, Bleeding, and Perforation).
NSAIDs, including NAPROSYN, EC-NAPROSYN, ANAPROX, ANAPROX DS and NAPROSYN Suspension, can lead to onset of new hypertension or worsening of pre-existing hypertension, either of which may contribute to the increased incidence of CV events. Patients taking thiazides or loop diuretics may have impaired response to these therapies when taking NSAIDs. NSAIDs, including NAPROSYN, EC-NAPROSYN, ANAPROX, ANAPROX DS and NAPROSYN Suspension, should be used with caution in patients with hypertension. Blood pressure (BP) should be monitored closely during the initiation of NSAID treatment and throughout the course of therapy.
Fluid retention, edema, and peripheral edema have been observed in some patients taking NSAIDs. NAPROSYN, EC-NAPROSYN, ANAPROX, ANAPROX DS and NAPROSYN Suspension should be used with caution in patients with fluid retention, hypertension, or heart failure. Since each ANAPROX or ANAPROX DS tablet contains 25 mg or 50 mg of sodium (about 1 mEq per each 250 mg of naproxen), and each teaspoonful of NAPROSYN Suspension contains 39 mg (about 1.5 mEq per each 125 mg of naproxen) of sodium, this should be considered in patients whose overall intake of sodium must be severely restricted.
NSAIDs, including NAPROSYN, EC-NAPROSYN, ANAPROX, ANAPROX DS and NAPROSYN Suspension, can cause serious gastrointestinal (GI) adverse events including inflammation, bleeding, ulceration, and perforation of the stomach, small intestine, or large intestine, which can be fatal.
These serious adverse events can occur at any time, with or without warning symptoms, in patients treated with NSAIDs. Only one in five patients, who develop a serious upper GI adverse event on NSAID therapy, is symptomatic. Upper GI ulcers, gross bleeding, or perforation caused by NSAIDs occur in approximately 1% of patients treated for 3-6 months, and in about 2-4% of patients treated for one year. These trends continue with longer duration of use, increasing the likelihood of developing a serious GI event at some time during the course of therapy. However, even short-term therapy is not without risk. The utility of periodic laboratory monitoring has not been demonstrated, nor has it been adequately assessed. Only 1 in 5 patients who develop a serious upper GI adverse event on NSAID therapy is symptomatic.
NSAIDs should be prescribed with extreme caution in those with a prior history of ulcer disease or gastrointestinal bleeding. Patients with a prior history of peptic ulcer disease and/or gastrointestinal bleeding who use NSAIDs have a greater than 10-fold increased risk for developing a GI bleed compared to patients with neither of these risk factors. Other factors that increase the risk for GI bleeding in patients treated with NSAIDs include concomitant use of oral corticosteroids or anticoagulants, longer duration of NSAID therapy, smoking, use of alcohol, older age, and poor general health status. Most spontaneous reports of fatal GI events are in elderly or debilitated patients and therefore, special care should be taken in treating this population. To minimize the potential risk for an adverse GI event in patients treated with an NSAID, the lowest effective dose should be used for the shortest possible duration. Patients and physicians should remain alert for signs and symptoms of GI ulceration and bleeding during NSAID therapy and promptly initiate additional evaluation and treatment if a serious GI adverse event is suspected. This should include discontinuation of the NSAID until a serious GI adverse event is ruled out. For high risk patients, alternate therapies that do not involve NSAIDs should be considered.
Epidemiological studies, both of the case-control and cohort design, have demonstrated an association between use of psychotropic drugs that interfere with serotonin reuptake and the occurrence of upper gastrointestinal bleeding. In two studies, concurrent use of an NSAID or aspirin potentiated the risk of bleeding (see PRECAUTIONS: Drug Interactions). Although these studies focused on upper gastrointestinal bleeding, there is reason to believe that bleeding at other sites may be similarly potentiated.
Long-term administration of NSAIDs has resulted in renal papillary necrosis and other renal injury. Renal toxicity has also been seen in patients in whom renal prostaglandins have a compensatory role in the maintenance of renal perfusion. In these patients, administration of a nonsteroidal anti-inflammatory drug may cause a dose-dependent reduction in prostaglandin formation and, secondarily, in renal blood flow, which may precipitate overt renal decompensation. Patients at greatest risk of this reaction are those with impaired renal function, hypovolemia, heart failure, liver dysfunction, salt depletion, those taking diuretics and angiotensin converting enzyme (ACE) inhibitors or angiotensin receptor blockers (ARBs), and the elderly. Discontinuation of nonsteroidal anti-inflammatory drug therapy is usually followed by recovery to the pretreatment state (see WARNINGS: Advanced Renal Disease).
No information is available from controlled clinical studies regarding the use of NAPROSYN, EC-NAPROSYN, ANAPROX, ANAPROX DS or NAPROSYN Suspension in patients with advanced renal disease. Therefore, treatment with NAPROSYN, EC-NAPROSYN, ANAPROX, ANAPROX DS and NAPROSYN Suspension is not recommended in these patients with advanced renal disease. If NAPROSYN, EC-NAPROSYN, ANAPROX, ANAPROX DS or NAPROSYN Suspension therapy must be initiated, close monitoring of the patient's renal function is advisable and patients should be adequately hydrated.
As with other NSAIDs, anaphylactoid reactions may occur in patients without known prior exposure to NAPROSYN, EC-NAPROSYN, ANAPROX, ANAPROX DS or NAPROSYN Suspension. NAPROSYN, EC-NAPROSYN, ANAPROX, ANAPROX DS and NAPROSYN Suspension should not be given to patients with the aspirin triad. This symptom complex typically occurs in asthmatic patients who experience rhinitis with or without nasal polyps, or who exhibit severe, potentially fatal bronchospasm after taking aspirin or other NSAIDs (see CONTRAINDICATIONS and PRECAUTIONS: Preexisting Asthma). Emergency help should be sought in cases where an anaphylactoid reaction occurs. Anaphylactoid reactions, like anaphylaxis, may have a fatal outcome.
NSAIDs, including NAPROSYN, EC-NAPROSYN, ANAPROX, ANAPROX DS and NAPROSYN Suspension, can cause serious skin adverse events such as exfoliative dermatitis, Stevens-Johnson Syndrome (SJS), and toxic epidermal necrolysis (TEN), which can be fatal. These serious events may occur without warning. Patients should be informed about the signs and symptoms of serious skin manifestations and use of the drug should be discontinued at the first appearance of skin rash or any other sign of hypersensitivity.
In late pregnancy, as with other NSAIDs, NAPROSYN, EC-NAPROSYN, ANAPROX, ANAPROX DS and NAPROSYN Suspension should be avoided because it may cause premature closure of the ductus arteriosus.
Naproxen-containing products such as NAPROSYN, EC-NAPROSYN, ANAPROX, ANAPROX DS, NAPROSYN SUSPENSION, ALEVE®, and other naproxen products should not be used concomitantly since they all circulate in the plasma as the naproxen anion.
NAPROSYN, EC-NAPROSYN, ANAPROX, ANAPROX DS and NAPROSYN Suspension cannot be expected to substitute for corticosteroids or to treat corticosteroid insufficiency. Abrupt discontinuation of corticosteroids may lead to disease exacerbation. Patients on prolonged corticosteroid therapy should have their therapy tapered slowly if a decision is made to discontinue corticosteroids and the patient should be observed closely for any evidence of adverse effects, including adrenal insufficiency and exacerbation of symptoms of arthritis.
The pharmacological activity of NAPROSYN, EC-NAPROSYN, ANAPROX, ANAPROX DS and NAPROSYN Suspension in reducing fever and inflammation may diminish the utility of these diagnostic signs in detecting complications of presumed noninfectious, noninflammatory painful conditions.
Borderline elevations of one or more liver tests may occur in up to 15% of patients taking NSAIDs including NAPROSYN, EC-NAPROSYN, ANAPROX, ANAPROX DS and NAPROSYN Suspension. Hepatic abnormalities may be the result of hypersensitivity rather than direct toxicity. These laboratory abnormalities may progress, may remain essentially unchanged, or may be transient with continued therapy. The SGPT (ALT) test is probably the most sensitive indicator of liver dysfunction. Notable elevations of ALT or AST (approximately three or more times the upper limit of normal) have been reported in approximately 1% of patients in clinical trials with NSAIDs. In addition, rare cases of severe hepatic reactions, including jaundice and fatal fulminant hepatitis, liver necrosis and hepatic failure, some of them with fatal outcomes have been reported.
A patient with symptoms and/or signs suggesting liver dysfunction, or in whom an abnormal liver test has occurred, should be evaluated for evidence of the development of more severe hepatic reaction while on therapy with NAPROSYN, EC-NAPROSYN, ANAPROX, ANAPROX DS or NAPROSYN Suspension.
If clinical signs and symptoms consistent with liver disease develop, or if systemic manifestations occur (eg, eosinophilia, rash, etc.), NAPROSYN, EC-NAPROSYN, ANAPROX, ANAPROX DS or NAPROSYN Suspension should be discontinued.
Anemia is sometimes seen in patients receiving NSAIDs, including NAPROSYN, EC-NAPROSYN, ANAPROX, ANAPROX DS and NAPROSYN Suspension. This may be due to fluid retention, occult or gross GI blood loss, or an incompletely described effect upon erythropoiesis. Patients on long-term treatment with NSAIDs, including NAPROSYN, EC-NAPROSYN, ANAPROX, ANAPROX DS and NAPROSYN Suspension, should have their hemoglobin or hematocrit checked if they exhibit any signs or symptoms of anemia.
NSAIDs inhibit platelet aggregation and have been shown to prolong bleeding time in some patients. Unlike aspirin, their effect on platelet function is quantitatively less, of shorter duration, and reversible. Patients receiving either NAPROSYN, EC-NAPROSYN, ANAPROX, ANAPROX DS or NAPROSYN Suspension who may be adversely affected by alterations in platelet function, such as those with coagulation disorders or patients receiving anticoagulants, should be carefully monitored.
Patients with asthma may have aspirin-sensitive asthma. The use of aspirin in patients with aspirin-sensitive asthma has been associated with severe bronchospasm, which can be fatal. Since cross reactivity, including bronchospasm, between aspirin and other nonsteroidal anti-inflammatory drugs has been reported in such aspirin-sensitive patients, NAPROSYN, EC-NAPROSYN, ANAPROX, ANAPROX DS and NAPROSYN Suspension should not be administered to patients with this form of aspirin sensitivity and should be used with caution in patients with preexisting asthma.
NAPROSYN, EC-NAPROSYN, ANAPROX, ANAPROX DS and NAPROSYN Suspension, like other NSAIDs, may cause serious CV side effects, such as MI or stroke, which may result in hospitalization and even death. Although serious CV events can occur without warning symptoms, patients should be alert for the signs and symptoms of chest pain, shortness of breath, weakness, slurring of speech, and should ask for medical advice when observing any indicative sign or symptoms. Patients should be apprised of the importance of this follow-up (see WARNINGS: Cardiovascular Effects).
NAPROSYN, EC-NAPROSYN, ANAPROX, ANAPROX DS and NAPROSYN Suspension, like other NSAIDs, can cause GI discomfort and, rarely, serious GI side effects, such as ulcers and bleeding, which may result in hospitalization and even death. Although serious GI tract ulcerations and bleeding can occur without warning symptoms, patients should be alert for the signs and symptoms of ulcerations and bleeding, and should ask for medical advice when observing any indicative sign or symptoms including epigastric pain, dyspepsia, melena, and hematemesis. Patients should be apprised of the importance of this follow-up (see WARNINGS: Gastrointestinal Effects - Risk of Ulceration, Bleeding, and Perforation).
NAPROSYN, EC-NAPROSYN, ANAPROX, ANAPROX DS and NAPROSYN Suspension, like other NSAIDs, can cause serious skin side effects such as exfoliative dermatitis, SJS, and TEN, which may result in hospitalizations and even death. Although serious skin reactions may occur without warning, patients should be alert for the signs and symptoms of skin rash and blisters, fever, or other signs of hypersensitivity such as itching, and should ask for medical advice when observing any indicative signs or symptoms. Patients should be advised to stop the drug immediately if they develop any type of rash and contact their physicians as soon as possible.
Because serious GI tract ulcerations and bleeding can occur without warning symptoms, physicians should monitor for signs or symptoms of GI bleeding. Patients on long-term treatment with NSAIDs should have their CBC and a chemistry profile checked periodically. If clinical signs and symptoms consistent with liver or renal disease develop, systemic manifestations occur (eg, eosinophilia, rash, etc.) or if abnormal liver tests persist or worsen, NAPROSYN, EC-NAPROSYN, ANAPROX, ANAPROX DS and NAPROSYN Suspension should be discontinued.
NSAIDs may diminish the antihypertensive effect of ACE-inhibitors, ARBs, or beta-blockers (including propanolol).
Monitor patients taking NSAIDs concomitantly with ACE-inhibitors, ARBs, or beta blockers for changes in blood pressure.
In addition, in patients who are elderly, volume-depleted (including those on diuretic therapy), or have compromised renal function, co-administration of NSAIDs with ACE inhibitors or ARBs may result in deterioration of renal function, including possible acute renal failure. Monitor these patients closely for signs of worsening renal function.
When naproxen as NAPROSYN, EC-NAPROSYN, ANAPROX, ANAPROX DS or NAPROSYN Suspension is administered with aspirin, its protein binding is reduced, although the clearance of free NAPROSYN, EC-NAPROSYN, ANAPROX, ANAPROX DS or NAPROSYN Suspension is not altered. The clinical significance of this interaction is not known; however, as with other NSAIDs, concomitant administration of naproxen and naproxen sodium and aspirin is not generally recommended because of the potential of increased adverse effects.
Clinical studies, as well as postmarketing observations, have shown that NAPROSYN, EC-NAPROSYN, ANAPROX, ANAPROX DS and NAPROSYN Suspension can reduce the natriuretic effect of furosemide and thiazides in some patients. This response has been attributed to inhibition of renal prostaglandin synthesis. During concomitant therapy with NSAIDs, the patient should be observed closely for signs of renal failure (see WARNINGS: Renal Effects), as well as to assure diuretic efficacy.
NSAIDs have been reported to competitively inhibit methotrexate accumulation in rabbit kidney slices. Naproxen, naproxen sodium and other nonsteroidal anti-inflammatory drugs have been reported to reduce the tubular secretion of methotrexate in an animal model. This may indicate that they could enhance the toxicity of methotrexate. Caution should be used when NSAIDs are administered concomitantly with methotrexate.
The effects of warfarin and NSAIDs on GI bleeding are synergistic, such that users of both drugs together have a risk of serious GI bleeding higher than users of either drug alone. No significant interactions have been observed in clinical studies with naproxen and coumarin-type anticoagulants. However, caution is advised since interactions have been seen with other nonsteroidal agents of this class. The free fraction of warfarin may increase substantially in some subjects and naproxen interferes with platelet function.
Due to the gastric pH elevating effects of H2-blockers, sucralfate and intensive antacid therapy, concomitant administration of EC-NAPROSYN is not recommended.
A 2-year study was performed in rats to evaluate the carcinogenic potential of naproxen at rat doses of 8, 16, and 24 mg/kg/day (50, 100, and 150 mg/m2). The maximum dose used was 0.28 times the systemic exposure to humans at the recommended dose. No evidence of tumorigenicity was found.
Reproduction studies have been performed in rats at 20 mg/kg/day (125 mg/m2/day, 0.23 times the human systemic exposure), rabbits at 20 mg/kg/day (220 mg/m2/day, 0.27 times the human systemic exposure), and mice at 170 mg/kg/day (510 mg/m2/day, 0.28 times the human systemic exposure) with no evidence of impaired fertility or harm to the fetus due to the drug. However, animal reproduction studies are not always predictive of human response. There are no adequate and well-controlled studies in pregnant women. NAPROSYN, EC-NAPROSYN, ANAPROX, ANAPROX DS and NAPROSYN Suspension should be used in pregnancy only if the potential benefit justifies the potential risk to the fetus.
There is some evidence to suggest that when inhibitors of prostaglandin synthesis are used to delay preterm labor there is an increased risk of neonatal complications such as necrotizing enterocolitis, patent ductus arteriosus and intracranial hemorrhage. Naproxen treatment given in late pregnancy to delay parturition has been associated with persistent pulmonary hypertension, renal dysfunction and abnormal prostaglandin E levels in preterm infants. Because of the known effects of nonsteroidal anti-inflammatory drugs on the fetal cardiovascular system (closure of ductus arteriosus), use during pregnancy (particularly late pregnancy) should be avoided.
In rat studies with NSAIDs, as with other drugs known to inhibit prostaglandin synthesis, an increased incidence of dystocia, delayed parturition, and decreased pup survival occurred. Naproxen-containing products are not recommended in labor and delivery because, through its prostaglandin synthesis inhibitory effect, naproxen may adversely affect fetal circulation and inhibit uterine contractions, thus increasing the risk of uterine hemorrhage. The effects of NAPROSYN, EC-NAPROSYN, ANAPROX, ANAPROX DS and NAPROSYN Suspension on labor and delivery in pregnant women are unknown.
Experience indicates that geriatric patients may be particularly sensitive to certain adverse effects of nonsteroidal anti-inflammatory drugs. Elderly or debilitated patients seem to tolerate peptic ulceration or bleeding less well when these events do occur. Most spontaneous reports of fatal GI events are in the geriatric population (see WARNINGS).
Naproxen is known to be substantially excreted by the kidney, and the risk of toxic reactions to this drug may be greater in patients with impaired renal function. Because elderly patients are more likely to have decreased renal function, care should be taken in dose selection, and it may be useful to monitor renal function. Geriatric patients may be at a greater risk for the development of a form of renal toxicity precipitated by reduced prostaglandin formation during administration of nonsteroidal anti-inflammatory drugs (see WARNINGS: Renal Effects).
Gastrointestinal: inflammation, bleeding (sometimes fatal, particularly in the elderly), ulceration, perforation and obstruction of the upper or lower gastrointestinal tract. Esophagitis, stomatitis, hematemesis, pancreatitis, vomiting, colitis, exacerbation of inflammatory bowel disease (ulcerative colitis, Crohn's disease).
Incidence of reported reaction between 3% and 9%. Those reactions occurring in less than 3% of the patients are unmarked.
Carefully consider the potential benefits and risks of NAPROSYN, EC-NAPROSYN, ANAPROX, ANAPROX DS and NAPROSYN Suspension and other treatment options before deciding to use NAPROSYN, EC-NAPROSYN, ANAPROX, ANAPROX DS and NAPROSYN Suspension. Use the lowest effective dose for the shortest duration consistent with individual patient treatment goals (see WARNINGS).
After observing the response to initial therapy with NAPROSYN, EC-NAPROSYN, ANAPROX, ANAPROX DS or NAPROSYN Suspension, the dose and frequency should be adjusted to suit an individual patient's needs.
Different dose strengths and formulations (ie, tablets, suspension) of the drug are not necessarily bioequivalent. This difference should be taken into consideration when changing formulation.
Although NAPROSYN, NAPROSYN Suspension, EC-NAPROSYN, ANAPROX and ANAPROX DS all circulate in the plasma as naproxen, they have pharmacokinetic differences that may affect onset of action. Onset of pain relief can begin within 30 minutes in patients taking naproxen sodium and within 1 hour in patients taking naproxen. Because EC-NAPROSYN dissolves in the small intestine rather than in the stomach, the absorption of the drug is delayed compared to the other naproxen formulations (see CLINICAL PHARMACOLOGY).
Naproxen-containing products are not recommended for use in patients with moderate to severe and severe renal impairment (creatinine clearance <30 mL/min) (see WARNINGS: Renal Effects).
To maintain the integrity of the enteric coating, the EC-NAPROSYN tablet should not be broken, crushed or chewed during ingestion. NAPROSYN Suspension should be shaken gently before use.
In patients who tolerate lower doses well, the dose may be increased to naproxen 1500 mg/day for limited periods of up to 6 months when a higher level of anti-inflammatory/analgesic activity is required. When treating such patients with naproxen 1500 mg/day, the physician should observe sufficient increased clinical benefits to offset the potential increased risk. The morning and evening doses do not have to be equal in size and administration of the drug more frequently than twice daily does not generally make a difference in response (see CLINICAL PHARMACOLOGY).
The use of NAPROSYN Suspension is recommended for juvenile arthritis in children 2 years or older because it allows for more flexible dose titration based on the child's weight. In pediatric patients, doses of 5 mg/kg/day produced plasma levels of naproxen similar to those seen in adults taking 500 mg of naproxen (see CLINICAL PHARMACOLOGY).
The recommended starting dose is 550 mg of naproxen sodium as ANAPROX/ANAPROX DS followed by 550 mg every 12 hours or 275 mg every 6 to 8 hours as required. The initial total daily dose should not exceed 1375 mg of naproxen sodium. Thereafter, the total daily dose should not exceed 1100 mg of naproxen sodium. Because the sodium salt of naproxen is more rapidly absorbed, ANAPROX/ANAPROX DS is recommended for the management of acute painful conditions when prompt onset of pain relief is desired. NAPROSYN may also be used but EC-NAPROSYN is not recommended for initial treatment of acute pain because absorption of naproxen is delayed compared to other naproxen-containing products (see CLINICAL PHARMACOLOGY, INDICATIONS AND USAGE).
The recommended starting dose is 750 mg of NAPROSYN followed by 250 mg every 8 hours until the attack has subsided. ANAPROX may also be used at a starting dose of 825 mg followed by 275 mg every 8 hours. EC-NAPROSYN is not recommended because of the delay in absorption (see CLINICAL PHARMACOLOGY).
Store at 15° to 30°C (59° to 86°F); avoid excessive heat, above 40°C (104°F). Dispense in light-resistant containers. Shake gently before use.
EC-NAPROSYN Delayed-Release Tablets: 375 mg: white, oval biconvex coated tablets imprinted with NPR EC 375 on one side. Packaged in light-resistant bottles of 100.
500 mg: white, oblong coated tablets imprinted with NPR EC 500 on one side. Packaged in light-resistant bottles of 100.
ANAPROX DS Tablets: Naproxen sodium 550 mg: dark blue, oblong-shaped, engraved with NPS 550 on one side and scored on both sides. Packaged in bottles of 100.
These are not all the side effects with NSAID medicines. Talk to your healthcare provider or pharmacist for more information about NSAID medicines. Call your doctor for medical advice about side effects. You may report side effects to FDA at 1-800-FDA-1088 or Genentech at 1-888-835-2555.
*Vicoprofen contains the same dose of ibuprofen as over-the-counter (OTC) NSAID, and is usually used for less than 10 days to treat pain. The OTC NSAID label warns that long term continuous use may increase the risk of heart attack or stroke.
© 2013 Genentech, Inc. All rights reserved.


While we have excellent coverage in most parts of the country, we still have some areas where we do not have listings, and Flagler County is one of those counties. We're sorry for the inconvenience!
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APPRAISERSdotCOM is the oldest (since December of 1998) and best internet advertising service for Real Estate Appraisers on the Internet . Don't let your competition be first in Flagler County! You need to sign up now for a listing and you can do that by completing our Online Application and Have Your Company on the Internet for only $13.50** a month.
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Almena, d. Peter and Sally L., Jan. 27, 1842, a. 5.
Alpheus, m., s. John and Onna, Apr. 12, 1846, a. 62 y. 6 m. 23 d.
Calvin 1st, ch. John and Onnah [dup. h. Unita], Apr. 1, 1807.
Calvin 2d, ch. John and Onnah, Jan. 25, 1821.
Cornelia Ann, b. Fabeous, NY, w. Lyman A., Aug. 12, 1844, a. 28 y. 7 m. 18 d.
Horace, s. Ira and Lucy, July 27, 1830.
Ira, Mr., Sept. 12, 1836.
Jewett, s. Ira and Lucy, July 16, 1830.
John [h. Onnah], Dec. 25, 1840, a. 80.
Keziah, b. Lenox, wid. Alpheus, typhus fever, Oct. 3, 1846, a. 64 y. 1 m. 2 d.
Lyman, ch. John and Onnah, Mar. 20, 1818.
Lyman A., widr., s. Alpheus and Kezia, consumption, Mar. 13, 1846, a. 27 y. 9 m. 13 d.
Mariah, d. Alpheus and Kezia, Oct. 10, 1834.
Polly, w. Peter, May 17, 1834.
Onna, wid., b. Willington, CT, consumption, Nov. 20, 1845, a. 81 y. 1 m. 6 d.
Benson, m., inflamation of the bowels, Nov. 28, 1844, a. 62 y. 3 m. 11 d.
Hannah, ch. Domer and w., Nov. 5, 1818.
Mahaly, Miss, Dec. 3, 1814.
Mary, wid. Elijah, Mar. 22, 1840, a. 85.
Rebecca, wid., Aug. 2, 1842, a. 84.
–––––, w. Joseph, Jan. 13, 1812.
Joseph, Mr., Dec. 29, 1812.
Lucy, b. Longmeadow, w. Daniel, July 22, 1847, a. 79 y. 6 d.
Warner, s. Elias and Eliza Mary, whooping cough, Aug. 15, 1845, a. 7 m. 2 d.


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Houston, Texas | April 24 - 28, 2023
The 2023 Biennial Meeting is scheduled for April 24-28, 2023 at the Westin Galleria, 5060 West Alabama, Houston, TX 77056.
The pre-Biennial Meeting Virtual Workshop will be held on Wednesday, March 22 through Friday, March 24, 2023 from noon to 1:30 PM Eastern Time each day.
ONLINE REGISTRATION IS NOW OPEN
Access to ONLINE REGISTRATION is now available via this hyperlink to
CFP's membership and registration portal on WildApricot: CFPmemberportal.foodprotect.org
The 2023 Biennial Meeting "INFORMATION BOOKLET" is now available with details regarding the full schedule of events and registration costs. This document should provide the details you need to make travel plans.
Click HERE for a PDF of this 10-page "Information Booklet" (updated 11/2022).
From the upper right hand corner of this page, click on the various gray colored buttons to learn more about different aspects of the 2023 Biennial Meeting.
Bookmark this page! Additional information will be posted as it becomes available.
Click HERE for information about Sustaining Sponsorship and Event/Service Donation
opportunities for the 2023 Biennial Meeting.
CFP MEMBERSHIP
Current CFP membership is NOT required to register for the Workshop or Biennial Meeting!
Membership in CFP is included with your 2023 Biennial Meeting registration and remains valid until the day before the start of the next Biennial Meeting in 2025. Membership in CFP is NOT included with the "Virtual Workshop Only" registration option.
Membership in CFP is always valid for the period between each biennial meeting. Therefore, a new membership processed today will expire the day before the 2023 Biennial Meeting begins in Houston. If you are not able to register for the April 2023 Biennial Meeting and would like to participate in CFP during the 2023-2025 biennium, we recommend waiting until AFTER the 2023 Biennial Meeting to process your membership.
Membership fees for the 2023-2025 biennium are $100 and current membership is required to participate on a CFP Committee or to serve as a Council Member. Membership for the 2023-2025 biennium will be available immediately after the conclusion of the April 2023 Biennial Meeting.
Click HERE if you would like to submit a membership application for the current biennium with the understanding that this membership will expire on April 23, 2023.
An "Issue" is a topic submitted for consideration to the Conference by any interested party which addresses an identified concern related to retail food safety and offers a recommended solution to that concern.
Issues are accepted ONLY through an automated ONLINE process. To assist in preparing an Issue for submission, the "Issue Pre- Submission Form," "Terms and Conditions for Issue Submission," and the "Issue Preparation & Review Process and Checklist" are all available on the Conference website. Click HERE to learn more about the Issue submission process.
We look forward to your participation in the 2023 Workshop and Biennial Meeting!


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Research > Research Portfolio > Research Teams > Immunotherapy Targeting Mutant KRAS
Pancreatic Cancer Collective Research Team: Immunotherapy Targeting Mutant KRAS
Grant Terms
Round 1: November 2018–December 2019
Round 2: January 2020–December 2022
Mutations in the KRAS oncogene drive the vast majority of pancreatic cancers. This research team used knowledge of the immune system and innovative bioinformatic, biochemistry and cell biology strategies to isolate T cells that can target the cancer-promoting gene. The team is studying two novel precision therapies involving highly selective white blood cells that can be given to patients with resected pancreatic cancer and will then study the most promising of these vaccines in patients with metastatic pancreatic cancer.
ABOUT THIS TEAM'S RESEARCH
The goal of the Pancreatic Cancer Collective Research Team: Immunotherapy Targeting Mutant KRAS is to develop a cellular therapy that exploits novel cell engineering to manufacture highly selective anticancer T cells on an individual patient basis. The approach is similar to T cell therapies recently approved by the FDA that target leukemia and lymphoma, except the team is targeting mutant KRAS in pancreatic cancer.
The team has been able to isolate T cells specific for mutant KRAS and has successfully grown these cells from multiple donors. In this study the researchers are isolating more KRAS T cells obtained from three groups of people: normal individuals, patients with mutant KRAS tumors, and pancreatic cancer patients who are being treated with vaccines against mutant KRAS. Based on their pre-clinical studies, the team is testing two novel vaccines in the clinic aimed at triggering mKRAS immune responses in patients with resected pancreatic cancer. In round two of funding, the team plans to use the most promising T-cell receptor identified and conduct a clinical trial of engineered T cell therapy for patients with metastatic pancreatic cancer.
This team is part of the Pancreatic Cancer Collective, an initiative of the Lustgarten Foundation for Pancreatic Cancer Research and Stand Up To Cancer.
SEE SCIENTIFIC ABSTRACT
The top scientists and researchers on the Pancreatic Cancer Collective Research Team: Immunotherapy Targeting Mutant KRAS come from a variety of backgrounds and disciplines, which leads them to great insights upon collaboration. Learn more about the Pancreatic Cancer Collective Research Team: Immunotherapy Targeting Mutant KRAS.
Robert H. Vonderheide, MD, DPhil
Beatriz Carreno, PhD
Co-leader
Elizabeth M. Jaffee, MD
Research Team Members
Robert H. Vonderheide, MD, DPhil,
Mark O'Hara, MD
Colleen Redlinger
"What we're attempting to do is to develop T cells or lymphocytes that can recognize a specific mutation that most pancreatic patients have, and that's mutated KRAS. We're aiming at KRAS because over 90 percent of pancreatic patients express mutated KRAS and it's an important target . . . The immune system is very smart. The T cell just goes after that protein and kills the whole cancer."
TEAM PROGRESS UPDATES
Stand Up To Cancer's research projects are designed to foster collaborative, swift translational research. The hallmarks of these efforts include rigorous application and selection procedures, sufficient funding to allow scientists to focus on the objectives of the grant, and reviews by senior scientists every six months. These reviews help the investigators capitalize on the latest findings, address potential roadblocks, and collaboratively evolve as the science requires. Please click below to see summaries of the research results so far for the Pancreatic Cancer Collective Research Team: Immunotherapy Targeting Mutant KRAS.
This team started its work in November 2018; links to publications will be posted when they are available.
CLINICAL TRIALS REFERRALS
Cancer clinical trials allow researchers to study innovative and potentially life-saving new treatments. The goal is to find treatments that are better than what's currently available, in fact the therapies offered to today's cancer patients were almost all studied and made possible by people participating in clinical trials. But many cancer clinical trials don't get completed because not enough people participate.
At StandUpToCancer.org/ClinicalTrials, you'll find information and answers to common questions about clinical trials. Learn more and talk to your doctor to see if a clinical trial may be the best choice for you.
You can also connect with Carebox, a free and confidential clinical trial matching service that provides access to a vast database to help you identify the clinical trials that might be right for you or your loved one.


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Updates on Social Stocks: Facebook Inc (FB)'s Hosting Tools, LinkedIn Corp (LNKD)'s Common Shares & Yelp Inc (YELP)
Published on September 6, 2013 at 9:18 am by Sachin Nigade in News
Editor's Note: Related tickers: Facebook Inc (NASDAQ:FB), LinkedIn Corp (NYSE:LNKD), Yelp Inc (NYSE:YELP), JPMorgan Chase & Co (NYSE:JPM), Morgan Stanley (NYSE:MS), Goldman Sachs Group, Inc. (NYSE:GS)
Zuckerberg: Facebook wants to take the pain out of building apps (PCAdvisor)
Facebook Inc (NASDAQ:FB) is seen as a website for connecting people. Now the company also wants to make it easier for outside developers to build their apps and connect them with users, by providing back-end hosting tools. On Thursday the social network held its first Parse Developer Day, a conference geared toward third-party app developers. Parse is a cloud service company that provides a hosted back-end infrastructure to make it easier for developers to build their mobile apps. Facebook Inc (NASDAQ:FB) announced its acquisition of Parse in April.
LinkedIn Stock Sale Could Bring In $1 Billion (CIO-Today)
With an amazing increase in share value over the past year, LinkedIn Corp (NYSE:LNKD) is finally using its success on the stock market to bring in real, usable money. According to a Securities and Exchange filing, LinkedIn plans to sell 4.17 million shares of its Class A stock to raise $1 billion, based on the company's $240.40 share value. There are few companies that experience the type of share value increase that LinkedIn has seen since its initial public offering in 2011. Since its IPO, LinkedIn Corp (NYSE:LNKD)'s share value has increased from $45 to $240, a fivefold increase in just two years.
Five Tips to Optimize Your Company's Yelp Listing (SocialBarrel)
Although most of the reviews on Yelp Inc (NYSE:YELP) focus on restaurants, other businesses are starting to join in on the Yelp conversation. Optimizing your business listing can increase traffic to your business, allow you to gain insight about your business, and give you an opportunity to engage with your consumers. Once you 'Claim your business' on Yelp, here are some tips to help you get started with Yelp Inc (NYSE:YELP) optimization:..
Is Facebook a Good Stock? (Equities)
Facebook Inc (NASDAQ:FB) posted its second quarter 2013 earnings, indicating the company far exceeded analyst expectations in both earnings and revenue, and shot up 25 percent that day. The sock continued climbing, and on Aug. 2 surpassed $38 a share, finally climbing above its July 2012 IPO price. On Sept. 4 Facebook was at $41.78 a share, not far under its all time high of $42.99. The company has been spurred by the company's ability to quickly adapt to mobile, long seen as the key for the social media behemoth's success…
LinkedIn to Raise Capital – Analyst Blog (Nasdaq)
Professional-networking behemoth LinkedIn Corp (NYSE:LNKD) is set to raise $1.2 billion by selling 5.4 million of its common shares at a price of $233.0 per share. The secondary offering is expected to close on Sep 10. JPMorgan Chase & Co (NYSE:JPM) and Morgan Stanley (NYSE:MS) will be the lead bookrunning managers while Goldman Sachs Group, Inc. (NYSE:GS) and BofA Merrill Lynch will be the joint bookrunning managers. Allen and Co. will act as the co-manager of the proposed capital-raising by LinkedIn. These financial institutions have the option to purchase another $150 million of common stock from LinkedIn Corp (NYSE:LNKD).
Facebook in fresh privacy row with new policy (GlobalPost)
Facebook Inc (NASDAQ:FB) is drawing fire from privacy activists again, after unveiling a new policy which could turn users' data and pictures into advertising. The new plan would "dramatically expand the use of personal information for advertising purposes," said a letter this week to the US Federal Trade Commission by six privacy organizations. The letter said the changes violate a 2011 consent order with the US watchdog agency and urged the FTC to "act to enforce its order."
What's next for LinkedIn (CNBC)
Bank of America Merrill LynchFacebook Inc. (FB)Goldman Sachs Group Inc. (GS)JPMorgan Chase & Co. (JPM)Linkedin Corp (LNKD)Mark ZuckerbergMorgan Stanley (MS)NASDAQ:FBNYSE:GSNYSE:JPMNYSE:LNKDNYSE:MSNYSE:YELPParse Developer DaySecurities and Exchange Commission (SEC)US Federal Trade CommissionYelp Inc (YELP)Show more...Show less
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